text:
Q: Something is wrong; all the RMSE metric values are missing; using caret train function I am trying to fit a gbm model using the caret package. I know other people have had the same problem, but all the solutions provided in the comments of those questions have not worked for my error. Here is my reproducible code:
library(dplyr)
library(MASS)
library(caret)
library(gbm)

Clean_winter_diff<-structure(list(Total = c(2L, 3L, 4L, 2L, 3L, 4L, 2L, 3L, 2L,
3L, 2L, 3L, 2L, 2L, 3L, 6L, 7L, 2L, 19L, 2L, 3L, 4L, 3L, 9L,
2L, 5L, 4L, 7L, 2L, 2L, 2L, 3L, 2L, 2L, 6L, 5L, 2L, 11L, 2L,
6L, 3L, 7L, 9L, 2L, 5L, 5L, 2L, 3L, 6L, 2L, 2L, 8L, 5L, 2L, 9L,
2L, 2L, 8L, 4L, 2L, 5L, 2L, 2L, 3L, 2L, 10L, 4L, 2L, 4L, 6L,
23L, 2L, 3L, 4L, 2L, 12L, 5L, 2L, 6L, 3L, 9L, 14L, 4L, 2L, 2L,
8L, 2L, 3L, 2L, 5L, 4L, 4L, 2L, 11L, 4L, 2L, 6L, 9L, 2L, 2L,
7L, 2L, 3L, 2L, 4L, 4L, 2L, 2L, 2L, 3L, 8L, 2L, 4L, 2L, 2L, 5L,
2L, 4L, 3L, 2L, 2L, 6L, 5L, 14L, 2L, 2L, 6L, 4L, 3L, 2L, 2L,
5L, 6L, 3L, 2L, 2L, 10L, 3L, 5L, 4L, 2L, 6L, 10L, 6L, 3L, 11L,
2L, 2L, 7L, 5L, 3L, 3L, 4L, 2L, 2L, 3L, 2L, 3L, 10L, 2L, 3L,
3L, 2L, 2L, 7L, 6L, 2L, 2L, 3L, 2L, 2L, 8L, 3L, 4L, 2L, 5L, 3L,
2L, 8L, 5L, 2L, 2L, 4L, 10L, 3L, 8L, 2L, 3L, 3L, 2L, 4L, 5L,
2L, 2L, 3L, 2L, 2L, 2L, 2L, 4L, 2L, 4L, 2L, 2L, 4L, 2L, 8L, 9L,
2L, 6L, 2L, 3L, 3L, 3L, 7L, 2L, 5L, 2L, 2L, 2L, 3L, 6L, 2L, 2L,
4L, 3L, 2L, 3L, 4L, 2L, 3L, 20L, 5L, 2L), Site = c(1, 1, 2, 1,
1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 2, 1, 1, 2, 1, 1, 1, 1, 2, 1,
1, 2, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 2, 2, 1, 1, 1, 1, 1, 1, 1,
1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1,
1, 1, 2, 2, 1, 1, 1, 1, 2, 1, 1, 1, 1, 1, 2, 1, 1, 1, 1, 1, 1,
1, 1, 1, 1, 1, 1, 1, 1, 2, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1,
1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 2, 1, 1, 2, 1, 1, 1,
1, 2, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 2, 1, 1, 2, 1, 1, 1, 1, 1,
1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 2, 2, 1, 1, 1, 1, 1, 2,
1, 1, 1, 1, 1, 1, 1, 1, 2, 2, 2, 2, 1, 1, 1, 1, 1, 1, 2, 2, 1,
1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 1, 2, 1, 1, 1, 1, 1, 1, 1, 1,
1, 1, 1, 1, 1, 1, 1, 1, 1, 2, 1, 1, 1, 2, 2, 1, 1, 2, 1, 1),
    Night = c(0, 0, 1, 0, 0, 0.25, 0, 0.666666667, 0.5, 0, 0,
    0, 0, 0.5, 0, 0.5, 0.428571429, 0, 0.6315789, 1, 0.666666667,
    0.25, 1, 0.333333333, 1, 0.2, 1, 0, 0, 0, 1, 0.666666667,
    0, 0.5, 0.166666667, 0, 0.5, 0.909090909, 1, 0.333333333,
    1, 0, 0.222222222, 0, 0, 0.2, 0, 0, 0.333333333, 0, 0.5,
    0.375, 0, 0, 0.222222222, 0, 0, 0.25, 0, 0, 0, 0, 0, 0.666666667,
    0, 0.2, 0.75, 0, 1, 1, 0.869565217, 0, 0, 0, 1, 1, 0.2, 0.5,
    0.333333333, 0, 0, 1, 0.25, 0.5, 0, 0, 0.5, 0, 0, 0.4, 0,
    0.75, 1, 0.363636364, 0, 1, 1, 0.222222222, 0.5, 0, 0.142857143,
    0, 0, 0, 0.25, 0, 0, 1, 0, 0.333333333, 0.25, 0.5, 0, 0.5,
    0, 0.4, 0.5, 0.25, 0, 0, 0, 0, 0, 0.785714286, 0, 0, 0.833333333,
    0, 0, 0, 0, 1, 0.5, 0, 0.5, 0, 0.6, 0, 0.2, 0, 1, 0.166666667,
    1, 0, 0.666666667, 1, 0, 0, 0.285714286, 0.2, 0, 0, 0, 0,
    0.5, 0, 0.5, 0.666666667, 0.4, 1, 0, 0, 0, 1, 0.857142857,
    1, 0.5, 0, 0.666666667, 0, 0.5, 0.5, 0, 0, 0, 0.2, 0.333333333,
    0, 0, 0.2, 1, 1, 0.25, 0.8, 0.333333333, 0.5, 0, 0.666666667,
    0.333333333, 0, 1, 0.6, 1, 0, 0.333333333, 0, 0.5, 0, 0,
    0.75, 0, 0.5, 1, 1, 0, 1, 0.375, 0.666666667, 0.5, 0.333333333,
    0, 0.666666667, 0, 0.333333333, 0.428571429, 0, 0.4, 0.5,
    1, 0.5, 0.333333333, 0.5, 0.5, 1, 0.5, 0.666666667, 0.5,
    1, 0.5, 0, 0.666666667, 0.5, 0.2, 1), Day = c(1, 1, 0, 1,
    1, 0.75, 1, 0.333333333, 0.5, 1, 1, 1, 1, 0.5, 1, 0.5, 0.571428571,
    1, 0.368421053, 0, 0.333333333, 0.75, 0, 0.666666667, 0,
    0.8, 0, 1, 1, 1, 0, 0.333333333, 1, 0.5, 0.833333333, 1,
    0.5, 0.090909091, 0, 0.666666667, 0, 1, 0.777777778, 1, 1,
    0.8, 1, 1, 0.666666667, 1, 0.5, 0.625, 1, 1, 0.777777778,
    1, 1, 0.75, 1, 1, 1, 1, 1, 0.333333333, 1, 0.8, 0.25, 1,
    0, 0, 0.130434783, 1, 1, 1, 0, 0, 0.8, 0.5, 0.666666667,
    1, 1, 0, 0.75, 0.5, 1, 1, 0.5, 1, 1, 0.6, 1, 0.25, 0, 0.636363636,
    1, 0, 0, 0.777777778, 0.5, 1, 0.857142857, 1, 1, 1, 0.75,
    1, 1, 0, 1, 0.666666667, 0.75, 0.5, 1, 0.5, 1, 0.6, 0.5,
    0.75, 1, 1, 1, 1, 1, 0.214285714, 1, 1, 0.166666667, 1, 1,
    1, 1, 0, 0.5, 1, 0.5, 1, 0.4, 1, 0.8, 1, 0, 0.833333333,
    0, 1, 0.333333333, 0, 1, 1, 0.714285714, 0.8, 1, 1, 1, 1,
    0.5, 1, 0.5, 0.333333333, 0.6, 0, 1, 1, 1, 0, 0.142857143,
    0, 0.5, 1, 0.333333333, 1, 0.5, 0.5, 1, 1, 1, 0.8, 0.666666667,
    1, 1, 0.8, 0, 0, 0.75, 0.2, 0.666666667, 0.5, 1, 0.333333333,
    0.666666667, 1, 0, 0.4, 0, 1, 0.666666667, 1, 0.5, 1, 1,
    0.25, 1, 0.5, 0, 0, 1, 0, 0.625, 0.333333333, 0.5, 0.666666667,
    1, 0.333333333, 1, 0.666666667, 0.571428571, 1, 0.6, 0.5,
    0, 0.5, 0.666666667, 0.5, 0.5, 0, 0.5, 0.333333333, 0.5,
    0, 0.5, 1, 0.333333333, 0.5, 0.8, 0), Distance_forest = c(0.527747223,
    0.680189568, 0, 0.310562619, 0.328173668, 0.278522078, 0.722954456,
    0.784333633, 0.633598813, 0.106383899, 0.525329032, 0.246038608,
    0.575318257, 0, 0.767179738, 0.443355317, 0.876859332, 0.19139315,
    0, 0.037535778, 0.432922864, 0.131314978, 0, 0, 0.093159023,
    0.128161967, 0, 0, 0.006470757, 0.30307544, 0, 0.568211372,
    0.263593171, 0.131057648, 0.168134106, 0.367657292, 0.717686941,
    0.163080941, 0, 0.202433621, 0.3842, 0, 0, 0.165167085, 0.929924705,
    2.120840521, 0.484698725, 1.078311772, 0.366644583, 0.340810601,
    0.298239859, 0.195581001, 0.02421172, 0, 0.464407271, 0.198840768,
    0.054828399, 0.489438607, 0.295818359, 0.110773002, 0.496209018,
    0.67346593, 0.214433884, 0.108712722, 0.529136166, 0.639769867,
    0, 0.396732499, 0.483450073, 0.001882719, 0.248622382, 0.925764277,
    0.175704519, 0.622952019, 0, 0, 1.142940058, 1.133076471,
    0.224133662, 1.083342909, 0.745420612, 0.377062959, 0.08050045,
    0.162178412, 1.361054023, 0.123874613, 0.49008657, 0.638751698,
    0.167293055, 0.306236508, 0.581962136, 0.269203966, 0.01981849,
    0.389124993, 0.333741945, 0.089434216, 0, 0.172470454, 0.174222306,
    0.298973407, 0.139883014, 0.455618893, 0.612636301, 0.372548564,
    0.35343891, 0.583316416, 0.291550392, 0.530795339, 0.07577014,
    0.844212848, 0.106972082, 0.992915959, 0.044859616, 0.820739224,
    0.799670156, 0.316242417, 0.319460412, 0.810118761, 0.500966406,
    0.377834056, 0.940032033, 0.151399734, 0.28102882, 0.212952188,
    0.073000622, 0.370545468, 0.872918616, 0, 0.104900131, 0.081847421,
    0.216958479, 0.008668498, 0.007014128, 0.495791646, 0.02399882,
    0.297470809, 0.490666846, 0.415433354, 0.301854897, 0.365931213,
    0.692253337, 0.165305616, 0.640148893, 0.835302988, 0.768199373,
    0.153852261, 0.134893226, 0.540233724, 0.335663076, 0.102341147,
    0.195486707, 0.362254712, 0.324739821, 1.697227338, 0.520683209,
    0.020203443, 0, 0.275300664, 0.259782193, 0.051199078, 0.217527413,
    0.550995487, 0.656144105, 0.277954065, 0.091362713, 0.769716859,
    0.817754331, 0.531972108, 0.330715097, 0.795027122, 0.818699405,
    0.113381995, 0.73975023, 0.342823482, 0.760817657, 0.817530729,
    0.700152145, 0.88797978, 0.29428625, 0.108928974, 0.074075782,
    0.747234676, 0, 0.543069, 0.262442933, 0.262835131, 0.356383731,
    0.371421971, 0.015478187, 0.601986047, 0, 0.048889129, 0.406113218,
    0.127855407, 0.396601367, 0.294174095, 1.112770231, 0.066093385,
    0.833489821, 0.27603216, 0.261494516, 0.139170942, 0.36716509,
    0.303017066, 0.245362186, 0, 0.071559882, 0.08333732, 0.617973146,
    0.075376835, 0.778806939, 0, 0.484474765, 0.09264197, 0.605744884,
    0.568592372, 0.464302103, 0.219293483, 0.115301111, 0.636074027,
    0.69132069, 0.448515825, 0.150593216, 0, 0.668861867, 0.664099955,
    0.386919408, 0.568691441, 0.328245416, 0.441309029, 0.216574999,
    0.191497106, 0.372996079, 0.211736755), Altitude_diff = c(-0.093344147,
    -0.032953796, -0.166307236, -0.082168137, -0.074024556, 0.011625801,
    -0.035469849, 0.023688222, -0.035174545, 0.009125112, -0.148026001,
    -0.136813009, -0.140504929, -0.155278686, -0.141057312, -0.154625722,
    -0.138962751, 0.021278778, -0.112632, -0.121742996, -0.104769694,
    -0.062242187, -0.105238068, -0.118123369, -0.116926834, -0.057471783,
    -0.099749664, -0.138632839, -0.086083588, -0.086340958, -0.109178192,
    -0.09964916, -0.086616302, -0.113422317, -0.145193425, -0.139987988,
    -0.12330925, -0.062, -0.073519485, -0.0852851, -0.087, -0.041133632,
    -0.02300371, 0.145411285, 0.007278729, 0.043087274, 0.12858374,
    0.074364258, 0.444998927, -0.018522705, -0.028386627, 0.007190659,
    -0.045301581, -0.057804062, 0.132843404, 0.021017105, -0.078413605,
    -0.046420864, 0.058002304, -0.081611237, 0.079912634, -0.050522034,
    -0.024949936, -0.084849548, -0.062893188, -0.041188028, -0.051312736,
    -0.01290921, -0.072736145, -0.079543025, -0.016072741, -0.019319687,
    -0.0213343, 0.020119728, -0.071389999, -0.088737882, 0.073720496,
    -0.019645096, -0.059846527, 0.08921346, -0.027587019, -0.064136113,
    -0.06246801, -0.049053955, 0.119930542, 0.013316631, -0.060812866,
    -0.010882792, -0.072900299, -0.00263418, 0.055887116, -0.057,
    -0.152, -0.082, -0.134, -0.157, -0.117, -0.128, 0.022, -0.129,
    0.121, 0.126, 0.091, -0.075, 0.014, -0.071, 0.009, -0.137,
    -0.13, -0.131, -0.054, -0.132, -0.093, -0.134, -0.143, -0.127,
    -0.089, -0.058, -0.057, -0.057, -0.055, -0.15, -0.17, -0.106,
    -0.177, -0.009, 0.008, -0.08, 0.067, -0.131, -0.029, -0.016,
    0.048, -0.154, -0.133, -0.109, -0.056, 0.029, -0.091, 0.031,
    0.032, 0.022, 0.029, 0.06, 0.075, -0.099, 0.075, 0.202, -0.022,
    0.013, 0.118, -0.022, -0.034, 0.224, -0.003, 0.095, 0.03,
    0.04, 0.105, -0.013, 0.031, -0.038, -0.043, -0.01, 0.046,
    -0.096, -0.028, -0.033, -0.023, 0.066, 0.063, -0.041, -0.001,
    -0.005, -0.025, 0.047, -0.025, -0.028, -0.002, 0.065, -0.019,
    -0.133, -0.045, 0.0479274, -0.0969804, -0.0511209, -0.1380578,
    -0.0619915, -0.1375449, 0.028642, -0.139097, -0.0267313,
    -0.0866448, -0.0664405, -0.0098812, 0.0950015, -0.0905839,
    -0.1271573, -0.1345035, 0.0696888, 0.1161573, -0.001593,
    -0.139097, -0.139097, -0.0351609, -0.1168084, -0.0487204,
    -0.0427109, 0.0139613, -0.0361378, -0.073785, -0.0521353,
    0.0207491, -0.0398732, -0.0512241, -0.0480128, -0.0133375,
    -0.0047241, 0.0556789, -0.0389344, -0.0307192, -0.0410356,
    -0.0436031, -0.0513303, 0.0914526, 0.108031, 0.078924, -0.0482411,
    -0.0010576, -0.0543727, 0.1055158, -0.0347792, 0.0091985,
    -0.0066721), Revisits = c(0, 0, 2, 0, 0, 0, 0, 0, 0, 0, 1,
    0, 0, 1, 0, 2, 1, 0, 4, 0, 1, 2, 2, 1, 0, 2, 2, 1, 0, 0,
    1, 1, 0, 1, 2, 1, 1, 5, 1, 2, 1, 3, 3, 0, 2, 1, 0, 0, 2,
    0, 0, 1, 1, 1, 0, 0, 0, 2, 0, 0, 1, 0, 0, 2, 0, 7, 3, 1,
    1, 3, 7, 0, 0, 1, 1, 7, 1, 0, 1, 1, 2, 9, 1, 1, 0, 2, 1,
    0, 0, 0, 0, 1, 1, 3, 1, 1, 4, 2, 0, 0, 2, 0, 0, 0, 0, 0,
    1, 1, 0, 1, 2, 0, 1, 1, 0, 0, 1, 0, 1, 0, 0, 1, 1, 6, 0,
    0, 2, 2, 0, 0, 0, 3, 0, 1, 0, 0, 2, 0, 0, 1, 0, 3, 3, 2,
    2, 5, 0, 0, 4, 4, 1, 0, 1, 0, 1, 0, 1, 0, 0, 1, 0, 0, 0,
    0, 4, 3, 1, 0, 1, 0, 0, 1, 0, 0, 0, 0, 1, 0, 1, 0, 1, 1,
    0, 4, 0, 1, 1, 1, 0, 0, 1, 2, 0, 0, 0, 0, 0, 0, 0, 0, 1,
    0, 1, 0, 1, 0, 1, 1, 0, 2, 0, 0, 0, 1, 4, 1, 2, 1, 0, 0,
    0, 0, 1, 1, 0, 0, 0, 2, 2, 0, 0, 1, 2, 1), Ratio = c(0, 0,
    2, 0, 0, 0, 0, 0, 0, 0, 2, 0, 0, 2, 0, 3, 7, 0, 4.75, 0,
    3, 2, 1.5, 9, 0, 2.5, 2, 7, 0, 0, 2, 3, 0, 2, 3, 5, 2, 2.2,
    2, 3, 3, 2.333333333, 3, 0, 2.5, 5, 0, 0, 3, 0, 0, 8, 5,
    2, 0, 0, 0, 4, 0, 0, 5, 0, 0, 1.5, 0, 1.428571429, 1.333333333,
    2, 4, 2, 3.285714286, 0, 0, 4, 2, 1.714285714, 5, 0, 6, 3,
    4.5, 1.555555556, 4, 2, 0, 4, 2, 0, 0, 0, 0, 4, 2, 3.666666667,
    4, 2, 1.5, 4.5, 0, 0, 3.5, 0, 0, 0, 0, 0, 2, 2, 0, 3, 4,
    0, 4, 2, 0, 0, 2, 0, 3, 0, 0, 6, 5, 2.333333333, 0, 0, 3,
    2, 0, 0, 0, 1.666666667, 0, 3, 0, 0, 5, 0, 0, 4, 0, 2, 3.333333333,
    3, 1.5, 2.2, 0, 0, 1.75, 1.25, 3, 0, 4, 0, 2, 0, 2, 0, 0,
    2, 0, 0, 0, 0, 1.75, 2, 2, 0, 3, 0, 0, 8, 0, 0, 0, 0, 3,
    0, 8, 0, 2, 2, 0, 2.5, 3, 8, 2, 3, 3, 2, 4, 2.5, 2, 2, 3,
    2, 2, 2, 2, 4, 2, 4, 2, 2, 4, 2, 8, 9, 2, 3, 2, 3, 3, 3,
    1.75, 2, 2.5, 2, 2, 2, 3, 6, 2, 2, 4, 3, 2, 1.5, 2, 2, 3,
    20, 2.5, 2), Area = c(0.032426, 0.035282, 0.113383, 0.035693,
    0.041549, 0.058353, 0.031573, 0.057897, 0.034298, 0.075203,
    0.038044, 0.039534, 0.035463, 0.056159, 0.0319, 0.152971,
    0.063424, 0.033137, 0.184546, 0.054271, 0.043699, 0.070929,
    0.086888, 0.182135, 0.055882, 0.063176, 0.072119, 0.1096,
    0.035482, 0.040162, 0.056385, 0.042962, 0.032754, 0.062732,
    0.056648, 0.035606, 0.062001, 0.117763, 0.062311, 0.089266,
    0.078665, 0.091633, 0.065517, 0.037454, 0.060411, 0.073355,
    0.035344, 0.033497, 0.119351, 0.044972, 0.031568, 0.114325,
    0.068984, 0.061986, 0.109741, 0.033782, 0.031849, 0.105872,
    0.055202, 0.031857, 0.064647, 0.031718, 0.032588, 0.076284,
    0.036021, 0.216575, 0.100172, 0.06227, 0.060081, 0.063876,
    0.224969, 0.045917, 0.037024, 0.077219, 0.054039, 0.158028,
    0.067884, 0.034719, 0.120346, 0.044812, 0.080923, 0.171879,
    0.069136, 0.0417, 0.032867, 0.11509, 0.053077, 0.062925,
    0.033554, 0.07492, 0.114556, 0.096677, 0.049153, 0.161404,
    0.073527, 0.045258, 0.08603, 0.091654, 0.033591, 0.033243,
    0.060307, 0.048489, 0.041845, 0.031375, 0.046293, 0.034473,
    0.044909, 0.052535, 0.060832, 0.082261, 0.086662, 0.031981,
    0.053075, 0.057269, 0.031764, 0.039376, 0.061771, 0.051374,
    0.081914, 0.04886, 0.040433, 0.056631, 0.086457, 0.118001,
    0.033169, 0.033734, 0.064399, 0.065725, 0.043722, 0.062459,
    0.032385, 0.07605, 0.055818, 0.067326, 0.034017, 0.033867,
    0.052257, 0.062541, 0.073173, 0.069685, 0.034166, 0.096639,
    0.081452, 0.116512, 0.064753, 0.12308, 0.033466, 0.050626,
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    0, 0.001190210739, 0.000552443287, 0.002855486907, 0.001430594014,
    0.000594097595, 0.000339933191)), row.names = c(NA, -234L
), class = "data.frame")

mydata = transform(Clean_winter_diff, Site=Site-1)

#separating training and test data
alpha<-0.7
inTrain_diff   <- sample(1:nrow(mydata), alpha * nrow(mydata))
train.set.diff <- mydata[inTrain_diff,]
test.set.diff  <- mydata[-inTrain_diff,]
winter.boost=gbm(Site~. ,data = mydata,n.trees = 10000,
                 shrinkage = 0.01, interaction.depth = 6, cv.folds = 5, verbose = F)

best.iter=gbm.perf(winter.boost, method = "cv")
best.iter
summary(winter.boost)

#Using caret to get model performance in best iteration
set.seed(123)
fitControl = trainControl(method="cv", number=5, returnResamp = "all")

model2 = train(Site~., data=mydata[complete.cases(mydata),], method="gbm",distribution="bernoulli", trControl=fitControl, verbose=F, tuneGrid=data.frame(.n.trees=best.iter, .shrinkage=0.01, .interaction.depth=1, .n.minobsinnode=1))

And this are the errors I get:
      RMSE        Rsquared        MAE
 Min.   : NA   Min.   : NA   Min.   : NA
 1st Qu.: NA   1st Qu.: NA   1st Qu.: NA
 Median : NA   Median : NA   Median : NA
 Mean   :NaN   Mean   :NaN   Mean   :NaN
 3rd Qu.: NA   3rd Qu.: NA   3rd Qu.: NA
 Max.   : NA   Max.   : NA   Max.   : NA
 NA's   :1     NA's   :1     NA's   :1
Error: Stopping
In addition: Warning messages:
1: In train.default(x, y, weights = w, ...) :
  You are trying to do regression and your outcome only has two possible values Are you trying to do classification? If so, use a 2 level factor as your outcome column.
2: In nominalTrainWorkflow(x = x, y = y, wts = weights, info = trainInfo,  :
  There were missing values in resampled performance measures

I have checked the dataframe for missing values, there are none. What is the problem?
sessionInfo():
Platform: x86_64-w64-mingw32/x64 (64-bit)
Running under: Windows 10 x64 (build 17134)

Matrix products: default

locale:
[1] LC_COLLATE=Norwegian Bokmål_Norway.1252  LC_CTYPE=Norwegian Bokmål_Norway.1252    LC_MONETARY=Norwegian Bokmål_Norway.1252
[4] LC_NUMERIC=C                             LC_TIME=Norwegian Bokmål_Norway.1252

attached base packages:
[1] stats     graphics  grDevices utils     datasets  methods   base

other attached packages:
 [1] caret_6.0-86        ggplot2_3.3.2       lattice_0.20-38     mlbench_2.1-1       gbm_2.1.8           MASS_7.3-51.4       randomForest_4.6-14
 [8] tree_1.0-40         ISLR_1.2            dplyr_1.0.0         rpart.plot_3.0.8    rpart_4.1-15

loaded via a namespace (and not attached):
 [1] tinytex_0.24         tidyselect_1.1.0     xfun_0.15            purrr_0.3.4          reshape2_1.4.4       splines_3.6.2
 [7] colorspace_1.4-1     vctrs_0.3.1          generics_0.0.2       stats4_3.6.2         survival_3.1-8       prodlim_2019.11.13
[13] rlang_0.4.7          ModelMetrics_1.2.2.2 pillar_1.4.6         glue_1.4.1           withr_2.2.0          foreach_1.5.0
[19] lifecycle_0.2.0      plyr_1.8.6           lava_1.6.7           stringr_1.4.0        timeDate_3043.102    munsell_0.5.0
[25] gtable_0.3.0         recipes_0.1.13       codetools_0.2-16     parallel_3.6.2       class_7.3-15         Rcpp_1.0.5
[31] scales_1.1.1         ipred_0.9-9          stringi_1.4.6        grid_3.6.2           tools_3.6.2          magrittr_1.5
[37] tibble_3.0.3         crayon_1.3.4         pkgconfig_2.0.3      ellipsis_0.3.1       Matrix_1.2-18        data.table_1.13.0
[43] pROC_1.16.2          lubridate_1.7.9      gower_0.2.2          rstudioapi_0.11      iterators_1.0.12     R6_2.4.1
[49] nnet_7.3-12          nlme_3.1-142         compiler_3.6.2
>

A: You are doing a classification, so you need to set the dependent variable to a factor for train in caret to work:
set.seed(123)
fitControl = trainControl(method="cv", number=5, returnResamp = "all")

mydata$Site = factor(mydata$Site)

model2 = train(Site~., data=mydata[complete.cases(mydata),], method="gbm",distribution="bernoulli", trControl=fitControl, verbose=F, tuneGrid=data.frame(.n.trees=400, .shrinkage=0.01, .interaction.depth=1, .n.minobsinnode=1))

model2

Stochastic Gradient Boosting

234 samples
  9 predictor
  2 classes: '0', '1'

No pre-processing
Resampling: Cross-Validated (5 fold)
Summary of sample sizes: 187, 187, 187, 188, 187
Resampling results:

  Accuracy   Kappa
  0.9232192  0.5550649

Tuning parameter 'n.trees' was held constant at a value

Tuning parameter 'n.minobsinnode' was held constant at
 a value of 1


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text:
Serving The Entertainment Industry
About Dinah Perez
Entertainment Attorney in Los Angeles
DINAH PEREZ attended Loyola Law School and has been in the practice of entertainment law since 1996. She practices law in the areas of film, television, new media, publishing, and music. Her clients include corporations, limited liability companies, talent management companies, motion picture and television producers/production companies, writers, composers, directors, actors, production designers, record labels, publishing companies, recording artists, Internet companies, and creators of new media content.
Ms. Perez has written legal articles for a variety of periodicals such as Story Board Magazine, Release Print, Surfview.com, Legalserviceindia.com, Mecfilms.com, and Findlaw.com. She has been quoted in Entertainment Weekly, Wired Magazine, Wired.Com. She was interviewed by John Scott Lewinski for his books, Alone In A Room and The Screenwriters Guide to Agents and Managers.
She has written chapters on the Legal 411 for Screenwriters and Producers for the Hollywood Screenwriting Directory and Hollywood Producers Directory published by F & W Media.
Ms. Perez has participated in panels and/or spoken at the Black Hollywood Film Festival, the Latin Heat Film Festival, Hawaii Songwriting Festival, ASCAP, Screenwriters World Conference, Beverly Hills Bar Association, Scriptwriters Network, Writers Digest Conference West, and Cinewomen.
She is a member of Film Independent, the Association of Independent Music Publishers (AIMP), the Copyright Conference (CCC), the Beverly Hills Bar Association (Executive Committee, Entertainment Section), and the California State Bar. She was a founding board member of Cinewomen in Los Angeles, CA.
Ms. Perez is fully bi-lingual in Spanish and services clients from Spain, South America, and Mexico.
Warning: include(/var/www/vhosts/dinahperezlaw.com/httpdocs/inc/index.php): failed to open stream: No such file or directory in /var/www/vhosts/dinahperezlaw.com/httpdocs/entertainment-attorney/dinah-perez-lawyer.html on line 57 Warning: include(): Failed opening '/var/www/vhosts/dinahperezlaw.com/httpdocs/inc/index.php' for inclusion (include_path='.:') in /var/www/vhosts/dinahperezlaw.com/httpdocs/entertainment-attorney/dinah-perez-lawyer.html on line 57
© 2019 Dinah Perez : Attorney at Law • Entertainment Lawyer for Film, Music & Television in Los Angeles

redpajama_set_name: RedPajamaCommonCrawl


Background - Amygdala-orbitofrontal cortical (OFC) functional connectivity (FC) to emotional stimuli and relationships with white matter remain little examined in bipolar disorder individuals (BD). Methods - Thirty-one BD (type I; n = 17 remitted; n = 14 depressed) and 24 age- and gender-ratio-matched healthy individuals (HC) viewed neutral, mild, and intense happy or sad emotional faces in two experiments. The FC was computed as linear and nonlinear dependence measures between amygdala and OFC time series. Effects of group, laterality, and emotion intensity upon amygdala-OFC FC and amygdala-OFC FC white matter fractional anisotropy (FA) relationships were examined. Results - The BD versus HC showed significantly greater right amygdala-OFC FC (p = .001) in the sad experiment and significantly reduced bilateral amygdala-OFC FC (p = .007) in the happy experiment. Depressed but not remitted female BD versus female HC showed significantly greater left amygdala-OFC FC (p = .001) to all faces in the sad experiment and reduced bilateral amygdala-OFC FC to intense happy faces (p = .01). There was a significant nonlinear relationship (p = .001) between left amygdala-OFC FC to sad faces and FA in HC. In BD, antidepressants were associated with significantly reduced left amygdala-OFC FC to mild sad faces (p = .001). Conclusions - In BD, abnormally elevated right amygdala-OFC FC to sad stimuli might represent a trait vulnerability for depression, whereas abnormally elevated left amygdala-OFC FC to sad stimuli and abnormally reduced amygdala-OFC FC to intense happy stimuli might represent a depression state marker. Abnormal FC measures might normalize with antidepressant medications in BD. Nonlinear amygdala-OFC FC–FA relationships in BD and HC require further study.


Together with Bacardi. Polska we organised the very first Instagram exhibition in Poland in cooperation with beginning | creative collaboration agency, Państwomiasto, FUTU Magazine and LOT Polish Airlines.
Exhibition took place in Państwomiasto from 31/07 to 1/09/2014.
Set of pictures of ONLINE#OFFLINE - first polish Intagram photo exhibition.


text:
Q: How to configure AVCaptureSession for high res still images and low res (video) preview? I'd like to capture high resolution still images using AVCaptureSession. Therefore AVCaptureSession preset is set to Photo.
This is working well so far. On an iPhone 4 the final still image resolution is at its maximum of 2448x3264 pixels and the preview (video) resolution is 852x640 pixels.
Now, because the preview frames are analyzed to detect objects in the scene, I'd like to lower their resolution. How can this be done?
I've tried to set AVVideoSettings with a lower width/height to AVCaptureVideoDataOutput, but this leads to the following error message:
AVCaptureVideoDataOutput setVideoSettings:] - videoSettings dictionary contains one or more unsupported (ignored) keys: (AVVideoHeightKey, AVVideoWidthKey

So it seems this is not the right approach to configure the size of the preview frames received by AVCaptureVideoDataOutput / AVCaptureVideoDataOutputSampleBufferDelegate. Do you have any ideas how the resolution of the preview frames can be configured?
Any advise is welcome,
Thank you.

A: If you want to specify the settings manually, you need to set activeFormat on the AVCaptureDevice. This will be implicitly set the session preset to AVCaptureSessionPresetInputPriority.
The activeFormat takes a AVCaptureDeviceFormat but you can only take one from the list of AVCaptureDevice.formats. You'll need to go through the list and find one that fits your needs. Specifically, check that highResolutionStillImageDimensions is high enough for desired still capture and formatDescription (which needs to be inspected with CMFormatDescription* functions, e.g., CMVideoFormatDescriptionGetDimensions) matches your desired preview settings.

A: Just for the records: I ended up configuring AVCaptureSession in preset Low while aiming the camera. As soon as the shutter is triggered, the app switches to preset Photo, performs a focus run and takes the picture. This way it takes between 1 and 2.5 seconds to take a picture, which isn't that great, but it's at least a workaround.

A: To lower the size of the output of AVCaptureVideoDataOutput you can set the bitrate to be lower thus producing a small sample size.
commonly used keys for AVCaptureVideoDataOutput are:
AVVideoAverageBitRateKey
AVVideoProfileLevelKey
AVVideoExpectedSourceFrameRateKey
AVVideoMaxKeyFrameIntervalKey

For example:
private static let videoCompressionOptionsMedium = [AVVideoAverageBitRateKey : 1750000,
                                                    AVVideoProfileLevelKey : AVVideoProfileLevelH264BaselineAutoLevel,
                                                    AVVideoExpectedSourceFrameRateKey : Int(30),
                                                    AVVideoMaxKeyFrameIntervalKey : Int(30)]

redpajama_set_name: RedPajamaStackExchange


Second place Heinrich Haussler is called up with GC leader Peter Sagan.
Mark Cavendish and Heinrich Haussler crashed spectacularly in the finale of stage four at the Tour de Suisse.
Heinrich Haussler will be the marquee name at IAM Cycling next year and the Australian is hoping that he will discover the same kind of esprit de corps on the squad that he found at Cervélo Test Team during its maiden season in 2009.
After competing in the WorldTour with Garmin-Sharp in recent years following its merger with Cervélo at the end of 2010, Haussler has no qualms about moving to a Pro Continental team next season, pointing out that he found himself in the same boat when he joined Cervélo, even if the presence of reigning Tour de France champion Carlos Sastre acted as something of an insurance policy when it came to wildcard invitations to the biggest races.
"I don't think it's necessarily a big step; I made the same step four years ago with Cervélo Test Team," Haussler told Cyclingnews. "Maybe the team doesn't have big names like Carlos Sastre and Thor Hushovd but it's still a great team. There's a good bunch of guys who have been brought together. The team has a lot of potential. We might have to depend on invites for some of the big races but if we perform well and start winning, we'll get some good starts."
So often relegated to a supporting role in the classics at Garmin, Haussler is enthusiastic about the challenge of acting as outright leader next spring. "That's something really important to me, to go into a team where I'm a captain," he said, admitting: "The last two years I've got to that level but I haven't performed well and got the big results. Now I want to try something different and I'm ready to open up a new chapter of my career and get back to that level."
Cervélo's successful debut campaign in 2009 was marked by a strong start to the season and Haussler is looking for something similar this time around, both on a personal and a collective level. "There were times [at Garmin] when I was on form but I didn't get the results. Maybe the confidence wasn't there – sometimes you just need one or two wins to get the confidence back and that just puts you on a whole different level and you're riding on a wave from race to race."
In recent years, Haussler has wintered in Australia, but ahead of the 2013 season, he has decided to stay in Europe and include cross training in his preparation. "This winter I'll be training the way I want to do. I'll be doing a lot of cross-country skiing and spending the winter in Europe. I'm pretty confident that I'm going to come out strong in my first couple of races."
Australian "just wants to be good again"


text:
ZoZo debut "Close to Falling Asleep" video
ZoZo
Dream pop singer and producer ZoZo has just released her sophomore single "Close to Falling Asleep", which premiered on March 12th. Today she has debuted the brand new video for the track. The song is characterised by the sweeping synth soundscapes and chilled electronic beats typical of ZoZo's previous music. After the success of her debut single 'I Won't Leave Your Bed Tonight', she has been playing gigs all around London and working on the music video.
ZoZo on "Close to Falling Asleep" video
"It's a simple carefree concept which portrays saying goodbye and really not caring at all!
I wanted to make a stripped back video where my expressions changed as much as my feelings about the situation in the song did."
Bad Pop debuts video for "On Your On"
"Number Crunching" by Ginger Snaps
Slow Riot release new single "Absent Dreams"
"Wake In Fright" by Desert Planes


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text:
Home Statistics Others Zaria Mosley Height, Weight, Age, Body Statistics
Zaria Mosley Height, Weight, Age, Body Statistics
Zaria Mosley Quick Info
Date of Birth June 29, 1999
Zodiac Sign Cancer
Eye Color Dark Brown
Zaria Mosley is an American YouTuber and social media influencer who has earned more than 100k followers on Instagram where she documents her daily life and often shares cute pictures with her daughter. She also has a YouTube channel with more than 10k subscribers, where she shares motherhood tips, beauty tutorials, life stories, vlogs, skincare routines, etc. Zaria also has more than 20k followers on Twitter.
Zaria S. Mosley
Zaria Mosley in an Instagram selfie as seen in July 2019 (Zaria Mosley / Instagram)
Durant, Mississippi, United States
Zaria is a high school graduate and passed school with honors. She then went to college to pursue a degree in nursing.
Mother – Leah Mosley
Siblings – She has 2 brothers and a sister.
YouTuber, Social Media Influencer
She self-manages her YouTube channel.
5 ft 2 in or 157.5 cm
Zaria Mosley with her daughter as seen in December 2019 (Zaria Mosley / Instagram)
Zaria has dated –
Kirk (2016–2018) – Zaria began dating Kirk in 2016 and had even co-started a YouTube channel with him titled Zaaryyy & Kirrk in 2017. She has a daughter with him named Za'Riah A. Mosley (b. August 12, 2018). However, the couple got separated before the birth of their daughter due to personal differences.
She is of African-American descent.
Zaria Mosley in a selfie in April 2019 (Zaria Mosley / Instagram)
Has a mole above her right eyebrow
Has a tattoo on her left wrist
Curvaceous figure
Has her daughter's name tattooed near her right collarbone
Zaria Mosley Favorite Things
Role Model/Inspirational Figure – Queen Naija
Chocolate Brand – Twix
Singer – Jacquees
Rapper – Cardi B
Songs – Girls Need Love (2018) by Summer Walker and Drake, Worth It (2019) by YK Osiris, 4 Walls (2007) by P-Dub & Pretty Willie, Please Me (2019) by Cardi B and Bruno Mars, It Ain't My Fault (2018) by Yella Beezy, Chi Chi (2019) by Trey Songz and Chris Brown, 7 Rings (2019) by Ariana Grande, Caramel (2018) by Lloyd, Shot Clock (2018) by Ella Mai
Hobbies/Interests – Listening to Music, Dancing, Traveling
Source – YouTube, YouTube, YouTube, YouTube, YouTube, YouTube
Zaria Mosley in an Instagram post as seen in May 2019 (Zaria Mosley / Instagram)
Zaria Mosley Facts
There have been many speculations that Zaria is social media star De'arra Taylor's sister but Zaria has stated in a YouTube video that even though she admires De'arra, the two are not related at all.
Her channel was initially named Zaaryyy & Kirrk and was started in 2017, in collaboration with her then-boyfriend. After their breakup though, Zaria re-named the channel to Zaria Mosley and started handling the content on her own.
Soon after giving birth to her daughter in August 2018, she got her first job outside of her social media work.
In November 2018, she met with a serious car accident. She had returned home after her office supervisor had given her an unexpected afternoon break. On her way back to the office, a car slammed into hers at a busy traffic signal. Fortunately for her, she sustained only minor injuries and felt that the slow speed of her car helped her survive the incident.
She revealed in a YouTube video that she was kicked out of the house by her grandmother when she was 17 years old. She had gone to live with her grandmother at the age of 12 and stayed with her for about 5 years but was disowned by her grandmother after she mistook her meeting with a friend for her meeting with a sperm donor. The incident left Zaria homeless for a few days until she moved back with her mother and siblings.
Featured Image by Zaria Mosley / Instagram
Zaria Mosley
Bow Wow Height Weight Body Statistics
Nick Sandow Height, Weight, Age, Body Statistics


ok so i have just rebuilt the valve train and installed a new k03 turbo. however after the car runns for about 10-15 mins, she starts knocking (it sounds horable), Ive had this car for a little over a year now and i am died to drive it but with issues like this i dont see it happening. Im wondering if i need a new oil pump or what? When i last pulled the oil pan to check to see if anything was in it, I did find metal that i have mark up to "old metal that was apart or the old turbo when it blew up.
Metal in the pan is never good. Where does the knocking come from? Head? Block? Have you checked oil pressure? Compression?
What was the need for rebuilding the valve train? Other failure?
well when i had bought it, it had a bad turbo that the seller didnt disclose to me. so when it blow i saw white smoke and went stright to the head gasket and later found out about the turbo. so with a new turbo in haul, i had the cylinder head decked and pressure checked (all good) installed and now im here.
You can be pretty certain it's the tensioner if the noise magically goes away if you rev it above 2,000 rpms or so.
This failure happens much more often on the VVT (variable valve timing) version of the cam chain tensioner on 2001+ models. The VVT version is more correctly called a "Cam Chain Adjuster" due to this added purpose. That one has more moving parts and tends to have the clanking failure more than the simpler non-VVT version. The problem happens 15 minutes into run time because that is when the oil warms up and oil pressure at idle comes down. Cold oil pressure at idle is probably 60-80psi. When its all warmed up it comes down to 15-20psi. When the tensioner is worn out, it hemorrhages oil pressure through loose tolerances of the piston. When pressure is high (either because cold temp or higher revs) it can keep up with the leaks. But at hot idle it cannot and it operates basically on spring pressure alone. The piece requires spring pressure in addition to oil pressure to obtain full tension.
yeah i know exatly what the cam tensioner is. I do have another one(new) but i dont have the cam tension tool. I'd really hate to pull the cams just to change it out. However The sound does go away when i step on the gas.
Tool is only like 5 bucks, only have to move the intake cam, no need to remove both cams, it's really an easy job that only takes 20-30 minutes start to finish. Pull the valve cover and you should be able to tell. When mine started to fail I could see wear on the aluminum where the chain was slapping around, not to mention the awful noise it made.
The new ones usually come with the tool. It holds them together in transit. But if you don't have one they are 5 or 6 bucks shipped on ebay.
I think 20-30 minutes is possible, if this is a race between mechanics or something. A first timer will be lucky to have the valve cover off in 20 minutes, so figure about 2-3 hours the first go-round.
yeah i agree it shouldnt take me but maybe 30-45 mins tops if i Dont F*** off a little while doing it to. But thanks for the advise i never even thought about that. How long do the shoes on those last, and are the black shoes "better" than the other ones?


Download and complete the Entrance Class Application Form. Page 1 of the application includes important information regarding the required documents that must be submitted with the application form.
If you submit your application digitally, please attach all documents as PDF files.
See JFKS Application Periods. Once your complete application has been received, you will be sent a confirmation email within 10 weeks. Applications are processed in the order they are received within the application periods, however, the date of submission has no effect on your application.
The language test, administered by an experienced teacher or a JFKS administrator, determines the child's dominant language ("Mother Tongue") and into which lottery group the child will be placed.
You will receive a confirmation by e-mail that your application has been received, followed by a more detailed letter including information about which contingent and/or lottery group your child belongs to according to the parents' and child's nationalities and whether your child will have to take a language test as well as when.
According to the JFKS Admissions Regulations certain applicants receive preference (siblings of JFKS students, students with two American parents, US Embassy families, JFKS teachers' children). All remaining available spaces will be filled according to the results of the three respective public, district-supervised lotteries in April.
A reserve list of 10 children will also be drawn.
You may attend the lotteries in person but you will be informed of the results by e-mail. If your child has received a place in the JFKS Entrance Class, you will receive a provisional letter of admission.
Upon receiving the provisional letter of admission for your child, please respond immediately stating whether you will accept the space for your child or not.


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Home > News and Publications > Newsroom > News Releases
Drugs Create Balancing Act for Patients with Non-Small Cell Lung Cancer
Credit: iStock
The results of a large, retrospective study of patients who received a form of immunotherapy for non-small cell lung cancer (NSCLC) revealed that patients may get more than one immune-related side effect, and identified a correlation between these multisystem immune-related adverse events (irAEs) and improved patient survival. In fact, patients who developed two irAEs did better, in terms of delaying the time to cancer progression and overall survival, than those who developed only one irAE. According to the researchers, this new information will be helpful in discussing with patients the spectrum of immune side effects that may occur from immunotherapy and the implications for their survival.
The findings were published online Oct. 29 in the journal JAMA Oncology.
The researchers analyzed data from 623 patients with NSCLC from five academic medical centers: The Johns Hopkins Hospital, Baltimore, Maryland; East Carolina University, Greenville, North Carolina; the Ohio State University-James Comprehensive Cancer Center, Columbus, Ohio; the University of Perugia, Perugia, Italy; and Sendai Kousei Hospital, Miyagi, Japan.
Patients diagnosed with stage III/IV NSCLC and treated with anti-PD-1/anti-PD-L1 immune checkpoint inhibitors (alone or in combination with other treatments) between January 2007 and January 2019 were included in the study. Of the 623 participants, 148 (24%) developed a single irAE. The most common four irAEs were inflammation of either the lungs, thyroid, liver or skin (pneumonitis, thyroiditis, hepatitis and dermatitis, respectively). Fifty-eight patients (9.3%) sequentially developed two single-organ irAEs, dubbed "multisystem irAEs." Patients with higher health scores — in the best overall health — who were treated with immune checkpoint inhibitors tended to be treated for a longer period of time, and were more likely to develop multisystem irAEs.
"While it makes sense that people who are fitter may be treated longer and have a higher risk of irAEs, this study accounted for treatment duration, and the association between irAEs and survival remains statistically robust," says Jarushka Naidoo, M.B.B.Ch., an adjunct professor at the Johns Hopkins Kimmel Cancer Center and a Bloomberg~Kimmel Institute for Cancer Immunotherapy investigator. Naidoo was an assistant professor of oncology at the time of the study.
The higher risk seems to come with a benefit, as patients showed incrementally improved survival outcomes based on the number of irAEs they developed. Those with no irAEs lived a median of 8.7 months after diagnosis versus 12.3 months and 21.8 months for those with one or two irAEs, respectively. A similar trend was seen in patients' progression-free survival (PFS) time, which is the time from diagnosis until tumor growth as seen on a CT scan. The median PFS for those with no irAEs was 2.8 months versus 5.1 months and 10.9 months for those with one and two irAEs, respectively.
Immunotherapy is a relatively new type of drug therapy that targets the interactions between cancer cells and cells from the immune system, causing immune cells to kill cancer cells. A subset of these drugs is called immune checkpoint inhibitors. These drugs bind to specific molecules — in this study, PD-1 and PD-L1 — found on the surface of cancer cells. When PD-1 and PD-L1 connect with receptor molecules on immune cells, the immune cell response to the cancer is shut down. Anti-PD-1/PD-L1 immune checkpoint inhibitors break the communication between PD-1 and PD-L1, reigniting the immune response against the cancer.
Immune checkpoint inhibitors generally have fewer side effects than standard chemotherapeutic drugs, but a subset of patients develop irAEs, which can be complicated and difficult to manage, the researchers report. Since immune checkpoint inhibitors work by temporarily releasing the brakes on the immune system, this heightened immune response can sometimes cause an attack on healthy organs and tissue.
"IrAEs are tricky because they are characteristically unpredictable. They can develop within days — but also after many years of treatment, so patients and oncologists have to always be on the lookout for symptoms," says Naidoo. They usually respond well to steroids, but can occasionally become chronic and even be fatal, she says. "At the Kimmel Cancer Center, we started noticing that some patients developed multiple irAEs, so I thought we might learn something helpful by characterizing them, seeing what patterns might exist and the implications on survival," she says.
The incidence of severe immune side effects depends on the type of immunotherapy received or what it combined with, and may be as low as about 5%–10% in patients receiving a single immune checkpoint inhibitor or as high as 35%–40% in those who receive a combination, says Naidoo. "We know that the outcome for most advanced cancers is poor, so the balance usually favors treatment with immune checkpoint inhibitors. However, it is important for patients to be aware of immune side effects, that they may experience multiple immune side effects, and the implications on survival," Naidoo says.
NSCLC is the most common type of lung cancer, comprising 80% of cases. It is also the most common cancer in men and the leading cause of cancer deaths in men and women worldwide. Naidoo says that, until five or six years ago, the average survival for those diagnosed with NSCLC was about one year. Two new treatment options — targeted therapy and immunotherapy — have led to dramatic improvements in survival for the patients in whom these treatments are effective.
In addition to Naidoo, researchers participating in the study include co-lead authors Bairavi Shankar, Jiajia Zhang, Patrick Forde, Josephine Feliciano, Kristen Marrone, David Ettinger, Christine Hann and Julie Brahmer of the Sidney Kimmel Comprehensive Cancer Center and the Bloomberg Kimmel Institute for Cancer Immunotherapy; Abdul Rafeh Naqash and Paul Walker of East Carolina University, Greenville, North Carolina; Biagio Ricciuti of the University of Perugia, Perugia, Italy, and the Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts; Dwight Owen, Gregory Otterson and Sandip Patel of The Ohio State University–James Comprehensive Cancer Center, the Ohio State University, Columbus, Ohio; and Yukihiro Toi and Shunichi Sugawara of Sendai Kousei Hospital, Miyagi, Japan.
Funding for the study was provided by the Bloomberg-Kimmel Institute for Cancer Immunotherapy, the National Institutes of Health and the Ohio State University Center for Clinical and Translational Science.
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Johns Hopkins researchers have identified correlation between multisystem immune-related adverse events and improved patient survival.
amone@jhmi.edu
Valerie Mehl
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Cancer, Science Research
Bloomberg~Kimmel Institute for Cancer Immunotherapy
Johns Hopkins Medicine International
Sidney Kimmel Comprehensive Cancer Center


Photographing food is something we do on a regular basis, mostly for restaurants or food manufacturers. Many of our food photographs end up on websites and the key to taking a good shot of food is fresh preparation, and good natural lighting.
I can't remember what this delicious dish was called, but the Chef let me finish it off after the shoot.
Captured for Quarry Road Wines.
Captured for Brooklands Country Estate for a recent 'Hi Tea' promotion.
Image of Nuts photographed for Prolife Foods Hamilton.
Dessert photographs are the hardest of all, you want to eat them before you shoot them.


Our contractors are always ready to serve residential, commercial, and industrial customers. Most of the time we are ready for same day dispatch. Call today to schedule a leak detection expert. We serve all of 84044, 84081, 84104, 84118, 84119, 84120, 84123, 84125, 84126, 84127, 84128, 84129, 84130, 84131, 84170, and all other zip codes in West Valley.


After all the glitz and glam of holiday lights and garland are gone, your room can seem empty and boring, but there are some easy ways you can update your decor and bring some of that pizzazz to your home all year round!
Your red and green twinkle lights may have come down, but candles are a great way to keep the mood of your home bright and cheery and create a focal point to awe your guests. Candles are also cost efficient and easy to swap out whenever you're in the mood for a change of scenery or scent!
Try mixing different sizes as well as jar and tin candles in exciting designs with a few pieces you already own, like vintage frames or a small clock, to create a mantle or table display that really wows. Tip: Be innovative—try using books or even old wine glasses as risers.
The right pillows can bring a drab couch to life, and are a simple way to bring color and pattern into your living room.
With a few more months of chilly nights in store, a nice warm blanket is a must for cuddling up on the couch. The right throw can keep you warm and bring a pop of color and texture to your sofa.
Hang a large piece of art that speaks to you or a fun patterned rug to create a unique statement wall that is sure to get as much attention as your dazzling holiday tree did. Dual-purpose wall hangings are trending this year, plus next year when you refresh your home you can take down the rug and use it under your living room upholstery for a brand new look.
Have you started redesigning for the New Year? Do you have any other design ideas that you just love? Let us know in the comments!
For more inspiration on how to use accent pieces to glam up your home check out our Pinterest board by clicking the link below.


How To Boost Your Sales With The Right Videos For many business owners, frustration has a unique feeling – the one you get when a qualified lead drops out on you at the last possible minute, deciding not to buy whatever you're selling.... So, how good are you at earning new clients and closing sales? How often does what you have in pending actually close? If you don't know, or you you would like to improve your closing ratio, than I'd like to suggest you apply the following best practices.
The sales world is pretty cluttered with advice. In fact, there are hundreds of different selling techniques we could share with you. Here at New Breed, we most often turn to industry leaders in the field.... Sales pro Grant Cardone shares his rules for helping small business owners close more deals. Sales pro Grant Cardone shares his rules for helping small business owners close more deals.
The dream of every sales professional is to close a sale the very day of the first contact with a business. This is an awesome experience when the sales cycle is an hour, the sale … how to change the registered color of a vehicle alberta As a marketing speaker and marketing coach, my clients often ask me for advice on sales. Naturally, this makes a ton of sense because the MORE and BETTER marketing you do, the FASTER and EASIER your sales process becomes.
If you get a strong buying signal, and you feel it's appropriate, it might be a good time to use a trial close. If the prospect responds well, you can proceed to the close. If the prospect responds well, you can proceed to the close.
On-the-spot coaching: I will be sharing with you the Ultimate Sales Problem Identifier so that you can pinpoint where exactly is your challenge in your sales so you can begin taking action immediately.
The spot price is the current price in the marketplace at which a given asset—such as a security, commodity, or currency—can be bought or sold for immediate delivery.
Wouldn't it be great to close jobs on the spot? Have you ever wanted to try but didn't want to be over bearing, a hard closing sales person?


An event quotation provides information about the price of the items, products, materials, services, and work force that an event host or organizer needs to pay to assure the completion of the event. The content of event quotations depends on the special requests of clients and the items expected from the suppliers.
This post is a collective list of downloadable Quotation Templates that are used in transactions for event planning and execution. If you want to have more references of other kinds of Formal Quotations, the templates and samples in the link provided are also available for download.
Set a meeting with the client, whether in person or through a digital platform to assure them that you can fully provide them with what they need.
Identify the specific kind of event where the event quotation will be used.
Suggest items for the client's approval.
Use any of our Quote Templates to create and/or edit the format and layout.
Place the important details in the event quotation, including the items needed and their respective prices.
Properly allocate the number of pieces that you should provide per item.
Get the sum of the prices of all the items so you can identify the total amount of the transaction.
Review the final event quotation and assure that there are no errors present in the document especially in terms of price summation.
Other than our event quotation templates, we can provide you with samples of Price Quotations in the specified link.
Event planning quotations are created by event planners to list down the rates of the services that their clients would like to acquire from them.
Corporate event quotations are given to businesses and companies who would like to hire a team of event specialists to assure that the event that they will host will smoothly run from the processes of planning up to its overall execution.
An event photography quotation contains the price of the photography deal that a photographer is offering to his/her possible clients. This document contains the number of post-processed shots that the photographer can provide, the time duration that the services of the photographer will be provided and other information depending on the photographer's rate and the photography needs of the client.
If you want to have more references in creating quotations that are used not only for events, kindly browse through our downloadable Quotation Samples and Word Format Quotation Templates.


I seem to be pinning lots of outfits involving trainers lately. So when I finally got my hands on a new pair of converse I had lots of inspiration on how to style them up. I remember buying my first pair when I was about 14/15 and wearing them until they had holes in a practically fell off of my feet.In The 'band tee' – something I always wore as a teenager. I had a drawer full of them. Mostly because I was at 3 gigs a week minimum back in high-school and I bought one at every merch stand I could. Most of these have had to go to charity as I'm not the size I was at that age.
I like the way band tees look with denim so I decided to style it up with a simple denim skirt. I wanted to make sure I could still wear these pieces in a more sophisticated way, as I'm actually not far away from 30 now (no way I hear you cry).
I feel like a piece like this can elevate any outfit. Of course I finished off my look with THOSE statement earrings from H&M. And the straw bag I can't seem to put down lately.
What pieces have you resurrected from your past wardrobes?
"Victoria Jackson | Part-time Magazine Editor and Lifestyle Blogger"
"How I Achieve My Rose Gold Hair"
I absolutely love your jacket, it's seriously to die for!


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Q: Problema en el envío de un registro en CRUD Laravel 8 Tengo un array con una lista de empresas y su localidad, ambas con sus tablas individuales. Las recorro  con un foreach, cada empresa tiene su enlace editar con la ruta correspondiente pero al momento de enviar, manda todos los registros de todas las empresas en vez de la que corresponde al boton.
Con el codigo queda mas claro :
index.blade.php :

       @foreach ($misEmpresas as $Empresa)

{{$Empresa->nombre}}

                        Editar

                        Eliminar

       @endforeach

Al hacer clic en editar una empresa tomando de ejemplo la empresa con id=9 el controller recibe una colección de todas las empresas, como si en la vista anterior hubiese enviado la variable $misEmpresas en vez de la variable.
Puede que la funcion index tenga algo que ver por eso la publico, pero en el while me muestra correctamente las 4 empresas que tengo con el usuario logeado.
$Empresas
Controller :
public function index()
{
        $misEmpresas=Empresa::join('communes','communes.id','=','empresas.commune_id')
                              ->where('empresas.user_id',auth()->user()->id)
                              ->latest('empresas.id')
                              ->get();

        return view('empresas.misempresas',compact('misEmpresas'));
}

public function edit(Empresa $Empresa)
{
        // Return para comprobar los ratos recibidos
        return $miEmpresa->all();

        $communes=Commune::orderBy('name','asc')->pluck('name','id');
        return view('empresas.edit',compact('Empresa','communes'));

}

** Ruta :**

Route::resource('misempresas',EmpresaController::class);

A: la manera correcta en editar un dato es la siguiente
id)}}" class="btn btn-warning btn-block ">  Editar

 public function edit($Empresa_id){
            // Return para comprobar los ratos recibidos
            //return $miEmpresa->all();
            $Empresa = Empresa::findOrFail($Empresa_id);

            return view('empresas.edit', ['empresa' => $Empresa]);
            //$communes=Commune::orderBy('name','asc')->pluck('name','id');
            //return view('empresas.edit',compact('Empresa','communes'));

    }


Did you know that Robin Meade is the LONGEST running morning news anchor on a National news show? That is quite an accomplishment, and one recognized in a recent AJC article for #WomensHistoryMonth. Executive Speakers Bureau has had the extreme pleasure of working with Robin Meade for multiple speaking engagements.
Robin takes her audience on a journey, telling her story about overcoming obstacles and empowering others to believe in themselves and work toward their dreams. This is what makes her an excellent choice if you are looking for a women's empowerment keynote speaker. She is very personable and has a way of connecting with the attendees. She loves people and it shows not only in her presentation but in how she interacts with attendees. And did we mention she can sing? Robin often incorporates song into her keynote and accepts entertainment bookings as well!
For more information on journalist and women's motivational speaker Robin Meade, contact Executive Speakers Bureau at (901) 754-9404.


Explore Gallery of Ivory Painted Dining Tables (Showing 1 of 25 Photos)Bentleyblonde: Diy Farmhouse Table & Dining Set Makeover With Annie Inside Famous Ivory Painted Dining Tables | Find the Best Interior Design Ideas to Match Your Style.
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06/03/2016 21/03/2016 Blogging, Books, Environment, Feature, Health, History, Life, Non-fiction, Planet, Planet, Psychology, Religion, Social problems, Society, Writing AfricaAfrica's UnderdevelopmentAfrican citizensAfrican leadersAggressionAids/HIVAmericaAmerican journalistsArticlesBelgiumBibleBio-weaponsBrusselsColonizationCongoDictatorsDocumentEbolaEuropeEvidenceFoundationGaddafiGambiaHistorical factsHollandHumblenessIntegrityInvestigationKing Leopold IIKoranLecturerLibyaMalariaMediaMenMen sleeping with menPerseverancePhysically strongPrinciplesPsychologically strongResearchRobert MugabeSame Sex MarriageSlaveryStatueSuicideToughnessTruthWhitman's graveWomenWomen sleeping with womenYahya JammehZimbabwe
Prevent Africa To Become A Continent Of Sodomites
There are evidence of hundreds of historical articles, facts and documents, revealing that Europe and America haven't any good plans for Africa. Slavery, colonization aggression, colonial underdevelopment, looting, killing of powerful African leaders, to mention a few, proved they were all planned to chronically weakened the foundation of Africa.
Whatever evil plans Europe and America hatched against Africa, seemed fruitless. The tough nature of Africans, living like ancient cave men, had trained them both physically and psychologically strong that, they could resist everything including malaria, when the vaccine of the disease hasn't been discovered yet.
The Europeans thought they could challenge Africans, proving they are strong, tough and could stand the jungle's fever and malaria. Unprepared, malaria killed thousands of them and the few escaped back to Europe, after naming the continent 'The White man's Grave.' Instead of learning how to survive on that continent, they planned to destroy the people and the entire continent.
Hence, after slavery and colonization, the creation of Aids, Ebola, Lassa fever and other deadly diseases, as bio-weapons to depopulate Africa emerged. Those medical crimes were written as a book by Holland's Micro-surgeon and scientist, Johan Van Dongen, after forty years of research and investigation. He suffered gravely for revealing the truth. He lost his job as a lecturer in one of the universities in Holland.
Despite all that he went through, Johan Van Dongen still has Africa in his heart. Together, we have co-written many articles warning African leaders that the intention to destroy Africa by Europe and America, is not yet over. If I were to be a journalist in living in Africa, I wouldn't have known the truth, because no European and American journalist is prepared to write this truth.
The leaders of Western Europe and America think African are fools and want African leaders to listen to whatever they tell them. Yet Africa is the least respected continent on earth. They don't even have respect for African leaders, let alone its citizens. If they do, Belgium wouldn't have built a statue of king Leopold II in Brussels. That king killed over ten million Africans, including children and women in Congo, while there isn't any statue of Adolf Hitler for killing six million Jews.
It is time for African leaders to stand firmly behind Robert Mugabe of Zimbabwe, to prevent the continent to become land of the Sodomites. After the killing of Gaddafi in Libya, Mugabe and Yahya Jammeh of Gambia are now the biggest threat to Europe and America, because they don't tolerate their nonsense. The media usually called those African leaders they can't stand 'Dictators,' but the real dictators are European and American leaders. They shouldn't tell Africa to legalize same sex marriage. We don't want to enjoy the 'back' of men, when the continent is full of pretty women.
Nobody cares about Africa, yet European and American leaders continue to interfere in African politics, telling African leaders what to do, yet many lack intelligence. If they are intelligent enough, then they shouldn't swear with the Bible and do the opposites of the Bible. The faith of Africans in the Bible and the Koran has taken us far, building us impenetrable, so we are not going to kowtow to European and American leaders to begin sleeping with men and women sleeping with women.
African may be poor, but we have something Europe and America can never take away. That is toughness, respect, principles, humbleness, perseverance, integrity and above all the strength to prevent committing suicide. Not even Aids and Ebola as bio-weapons could destroy Africa. I will end this article by asking Europe and America leaders to ask their citizens: Why everyday Europeans and Americans commit suicide but Africans aren't?
27/02/2016 Blogging, Feature, Health, History, Life, Review, Society, Writing AfricaAfricanAidsAmericaAmericanArticlesAutismBad associationBelgian journalistsBibleBitternessCIACocaineCrimeDeathDecayed societyDiscomfortDr. Jeff BradstreetEbolaEuropeEuropeanExperimentFamilyFriendsHashishHollandHolland's Professor Johan Van DongenHypocrisyJoel SavageJournalistJournalistsJudiciary SystemLassa FeverLazinessLecturerLiesMediaMedical CrimeNew York TimesPainPrecious stonePrinciplesProstitutionResponsibilitySarah KershawSuicideThe NetherlandsTruthUniversityUseful habitsViolence
You Know Where To Find Me If You Want To Kill Me
Bad association spoils useful habits. The reason many like to keep away from lies, hypocrisy, crime, violence, prostitution etc. But in today's world, everything has changed. If you are not a bad influence you can't get friends. Looking for a true friend now, is like a search for a precious stone on a large acre of plot.
Nevertheless, those that want to live in truth, will continue to search, in spite of the pain, agony, humiliation, threat etc, that follow them. I was raised in a family that believed everything opposite to the principles in the Bible, including laziness, is a sin. Therefore I find it extremely hard to smile, when it's time to frown or praise when time to criticize.
The decay and corruption of the society have rapidly affected every sector, including our judiciary system. Truth will not set you free, instead it may give you a lot of discomfort and sometimes may lead to your death, as it happened to Dr. Jeff Bradstreet, for opposing autism vaccine he considered dangerous for the health and former New York Times journalist, Sarah Kershaw, exposing the CIA's mind control program of illegal program of experiments on human subjects.
Reading such articles in this dangerous world don't scare me, instead they encourage me. The reason I teamed up with Holland's Micro-Surgeon/Scientist Johan Van Dongen, to write every necessary article, to let the world know that a medical crime is being covered up by European and American leaders.
Just as I said, the truth will not always set you free, some may give you a lot of worries. Following his forty-years of research, fully and satisfied that Aids and Ebola were medical crimes used as bio-weapons against Africa, Professor Dongen published his findings, but little did he know that his native country, The Netherlands (Holland) will punish him for publishing the truth. He was betrayed by some of his scientist friends and eventually it cost him his job as a lecturer at the university.
The same hatred is generated towards me by some of the Belgian journalists, due to my uncensored raw articles. It's like I'm exposing them. It's like I have them to be recognized as liars, rather than self-dedicated honest journalists. If you are an European or American journalist and you find it hard to write openly that Aids, Ebola, Lassa fever etc; are medical crimes against Africa, I 'm an African, I will do that and please don't hate me for this. Hate yourselves for being liars and hypocrites.
About the fear of dying, that's not in my agenda. Truth is always bitter than the bile. If you want that bitterness to taste sweet in your mouth, try to cultivate the habit of speaking the truth, because it's our responsibility to restore out nation's dignity, not to ruin it.
Should in case I die today, I will be very happy to go down my grave, but I want the truth to be published. The Belgian media shouldn't write I committed suicide or drug related incidence, because Joel Savage doesn't know what suicide is, above all I haven't seen hashish before let alone cocaine.
11/02/2016 Blogging, Environment, Feature, Life, Planet, Planet, Society, Writing AccidentAids ExpertsAlberto BeharArizona State UniversityCervical CancerChinaDr. Anne SzarewskiElectrical EngineerFlight InstructorGlenn ThomasHelicoptersInternational Aids CoferenceJet Propulsion LaboratoryJoep LangeKashif KhusheedMelissa KetunutiMH17NASAPlanePlanesScientistScientistsSingaporeSuicideSuspicious DeathUS SecurityVan Nuys AirportWeatherWorld Health Organization
Why NASA Scientists Dying Mysteriously?
Friday January 12th 2015, renowned scientist Alberto Behar crashed his small plane in the streets of L.A. shortly after he had taken off. Alberto worked at NASA's 'Jet Propulsion Laboratory' and Arizona State University; he was one of the leading scientists responsible for discovering that there was once water on Planet Mars. Behar was an expert pilot; he was a flight instructor for both planes and helicopters – the weather conditions were very clear and for some unknown reason his plane just started to lose altitude. The Van Nuys Airport pilot Kashif Khursheed stated "I can't see what would be the cause of something like this. He was very knowledgeable, competent and thorough."
This tragic event is just one of the most recent cases of a prominent key scientists suspicious death, over the last two years 74 leading medical and NASA scientists have died, almost 'ALL' are officially labelled suicide or an accident. (all 74!?) Glenn Thomas was a World Health Organisation spokesperson; he was reportedly an expert on Ebola and AIDS, he was aboard MH17 when it was shot down with approximately 100 other researchers.
Other strange deaths of leading scientists are: Joep Lange, a leading AIDS researcher and former president of the International AIDS Society (IAS) was also aboard MH17. They were all on their way to an international AIDS conference. With one fell swoop a large portion of the leading AIDS experts were killed before they could attend a global conference.
Melissa Ketunuti, 35, was a paediatrician who specialised in cancer; she also worked on an AIDS fellowship in Botswana. She was hogtied and set on fire in the basement of her home in Philadelphia. Dr Anne Szarewski, 53, was a cervical cancer expert; she pioneered the cervical cancer vaccine.
Perhaps one of the most controversial scientist deaths in the last so many years was that of Shane Truman Todd, 31. In June 2012 he was found dead, his body was in a morgue in Singapore, he was a rather important electrical engineer.
He was working on an EXTREMELY top-secret "one of a kind" machine for the Chinese that was believed to be some kind of a defence weapon. Before his death Shane had told his family that he was not happy with what he was doing and he feared for his life, he was allegedly being asked to compromise U.S security!!
Shane had quit his job and was due to leave China and fly home but sadly died a week before his flight after his last day at work building this 'unknown machine'. Foul play was most definitely suspected and his family began campaigning straight away for the truth, Chinese official said they would look into the matter and try and determine if it was murder or suicide but guess what!? Yes, as of yet there are no answers….Hmmm!?!?!?!? –
Continue reading: http://truthcdm.com/another-dead-nasa-scientist-list-of-dead-scientists/#sthash.35gxxKHg.dpbs
13/01/2016 Contemporary Women, Feature, History, Life, Non-fiction, Review, Society, Writing 3 Comments 1st and 2nd World WarsAfro-BrazilianBlack HistoryBlacksColonizersCommunityDandaraDutchEnemiesFighterOverthrownPalmaresPortugueseResistanceResponsibilitySlaverySuicideWarriorWhite SoldiersWomenZumbi
Dandara: A Great Black Woman That Made History
Dandara, the great fighter.
It's our responsibility to dig out great stories of black history. Many are racists because of your colour black, but during the First and Second World Wars, White soldiers painted their faces black to avoid detection by enemies. Many blacks survived because enemies hardly had a glimpse at them. This is short story of one of the greatest black women fighters in black history.
Dandara was an Afro-Brazilian Woman, Warrior who lived in the 1600s. She was co-founder of Palmares, a run-away slave community (quilombo) that thrived for almost a century.
Bravely she fought alongside Zumbi and others defending the freedom of her people and her community. Palmares was eventually overthrown by Dutch and Portuguese colonizers, but rather than return to slavery, Dandara took her own life as an act of resistance.
19/12/2015 20/07/2016 Books, Feature, Health, History, Interviews, Life, Non-fiction, Review, Society, Writing 3 Comments AfricansAidsAmericaAutismBookCenter For Diseases ControlContinentDepopulateDestructionDiseasesDr. Jeff BradstreetEbolaEuropeEvil DeedsFishermanFranceHollandHumanHypocrisyImpunityJusticeLawLawsuitsLecturerMediaMedical CrimeMedical TerrorismMicro-SurgeonMilitaryNaziPanicPerpetratorsResearchRussiaScientistSuicideTerrorismThe Lying SyndromeTruthUniversityVictimsWorld Health OrganizationWriter
Aids And Ebola Are Medical Terrorism Against Africa by Europe And America
The resting place of poor victims of Ebola in Africa.
Without any support from the European and American media, since the beginning of this year, I have been working very hard with Joel Savage, a writer, who also advocate truth, to let the whole world, including Africa, know that Aids and Ebola were man-made diseases or created in laboratory and used on poor innocent defenseless Africans by Europe, Russia, and America.
In fact, just like the fight against terrorism continues unabated, we (Dongen and Savage) will also continue to establish the truth that Aids and Ebola, were indeed medical crimes against Africa, for military defending purposes, to de-populate and cause mass human destruction and panic in the continent of Africa.
We are asking European and American leaders to show concern to Africans, the same way they showed concern over terrorism in France or in any part of this world. It's pure hypocrisy for Europe and America to haunt perpetrators of terrorism when they are free after committing the same medical terrorism in Africa.
Again it's pure hypocrisy for Europe and America, to hunt down the perpetrators of Nazi crime when those that committed the medical crimes are enjoying their lives with impunity. Do we have to ask, is it because that medical terrorism against Africa was committed many years ago, while terrorism is freshly taking place in Europe and America? If the answer is yes, then there is no law and if there is, then the law is an ass or raped.
WHO IS JOHAN VAN DONGEN?
Dongen is a Dutch Micro-Surgeon/Scientist and a former lecturer in one of the universities in Holland. At the age of 48, he lost his job because of revealing the truth about Aids and Ebola as man-made viruses, which were created in the laboratory, as a means to depopulate Africa. Decades of his research, he immediately published a book about those medical crimes in the native language, in Holland.
Since Europe, America, Russia, World Health Organization and Center for Diseases Control want to cover up those medical crimes, Holland didn't take Professor Dongen's publication likely. What was his 'crime?' He was considered a whistle-blower. After losing his post as a lecturer, Micro-Surgeon Dongen stood tall, brave and firm, to fight the authorities in his native Holland, since his research had been confirmed by other brave scientists that Aids and Ebola were indeed medical crimes against Africa.
After many lawsuits against his professional life, the Dutch authorities crippled him financially, as one of the measures to stop him from bringing out the truth. "Joel Savage, I lost everything, but not my brain and my wife," says Professor Johan Van Dongen.
At his ripe age, Dongen still continues to fight for the truth, penniless, by using the social media. Whatever wrong happening to us today, including terrorism, were caused by man. The wages of sin is death and whatever a man sows, the same he shall reap. We are against terrorism and other crimes, but many have failed to realize that whatever is happening to us today, were the results of the evil deeds planted by Europe and America.
Professor Johan Van Dongen's research over Aids and Ebola medical crimes in Africa was first published in Dutch, before translated.
The media knows this very well, but they will not publish it to 'tarnish' their image or someone to lose his job, instead they keep on killing and making things difficult for those standing up for the truth. Since Aids, Ebola, and other medical crimes occurred, many scientists have lost their lives in strange circumstances. The latest is Dr. Jeff Bradstreet: After claiming that vaccines caused autism, the doctor was found dead.
The media claimed in an apparent suicide in North Carolina, Bradstreet died of what seems to be a self-inflicted gunshot wound to the chest. Found in Rocky Broad River in Chimney Rock on June 19, by a fisherman. In a world of white-collar liars and criminals, that's what we often hear and the European and American media have also inherited the same disease: The Lying Syndrome.
We are never pleased over terrorism taking place around the world because violence doesn't pay. In fact, like every human being, we feel the pain for victims of terrorism, even though no one cares about Africans, yet we shall use the social media to discourage this act of violence some extreme groups are using to get their voice heard. But while we are showing concern over the lives of innocent victims killed by terrorists, the world should also show concern over the plight of Africans to bring those responsible for facing the law or justice.
http://www.amazon.com/s/ref=nb_sb_noss?url=search-alias%3Dstripbooks&field-keywords=aids+johan+van+dongen
09/11/2015 24/08/2016 Books, Feature, Health, History, Life, Non-fiction, Review, Society, Writing AfricaAgentsAidsAids PandemicAnimalsAutismAutisticBlack PrisonersCelebratingClinicControversiesCriminalsDeathsDisappearanceDoctorsDonald TrumpEbolaEnvironmentalEstablishmentFDAGermanGhanaGraduationHealthHigh SchoolHistoryHorrificInterviewJapaneseJeffery BradstreetJokingKenyaLand of evilLaughingLuc MontagnierMedicalMicro-SurgeonMMR Vaccine CorporationMysteriousNamibiaNazi CriminalsOptimisticOtmar von VerchuerPatientsPharmaceuticalPlantationsPresidentProtectionScientificSonSouth AfricaSuicideThree Piece SuitsTransmissionUgandaUnited States of AmericaUniversityVaccinesWolff Geisler
Donald Trump: Why Aren't You Killed Like Jeffrey Bradstreet Or Other Opponents Of Vaccinations Which Not Only Cause Autism But Also Aids And Ebola?
This article is dedicated to Jeffrey Bradstreet: By Johan Van Dongen
The above question is raised by me and many others, because recently in the United States of America, over seven doctors have been murdered or found dead in strange circumstances. I find it very necessary to ask Donald Trump this question, because of the statement he made that: "Vaccines Do Cause Autism." and also to find an answer to why the disappearance from the medical scene or mysterious deaths of alternative health doctors who have real cures, but opposed and disapproved by the FDA?
When I saw the interview of Dr. Jeff Bradstreet, nothing shows a possible suicide as mentioned by US governmental institutions. During this interview, Bradstreet showed his engagement, for he was not a person who would give up easily. It was a very optimistic person with a normal attitude. He was laughing, joking and celebrating because his autistic son just graduated from high-school.
The supposed reason for his "suicide" – an FDA raid on his clinic – is nothing new for him. There was nothing new in his life that would have been a reason for him to want to end his own life, only new things that would encourage him to live his life and perhaps encourage others that want to take their lives. Now we have to wait and see what happens to Donald Trump.
The overall conclusion
"Dr. Jeffrey Bradstreet has been under attack by big pharma for his success during all his professional life, so there is no way he would have committed suicide for just another attack. He was murdered; the FDA were clearly involved, and the other suspect is the MMR vaccine corporations, who work with the FDA. Dr. Bradstreet loudly published the fact we all know: The MMR jab, which makes billions of profit, causes autism."
It isn't only autism which is caused by vaccines but also Aids and Ebola. For decades Africa is used as a bio-warfare laboratory of German and Japanese war criminals under the guidance of the USA because they protect these Nazi bastards. This makes the USA the land of evil, so once Donald Trump becomes president of the United States, there is a lot work to do by replacing black prisoners with white pharmaceutical criminals in three piece suits.
The horrific Aids pandemic tremendously has generated scientific controversies within and outside the scientific establishment. Only a minority of scientists and other engaged people have access to inside information concerning (bio-warfare) Aids and Ebola research.
As an experimental micro-surgeon in the early seventies, almost at the beginning of the multiple organ transplantation eras, I have carried out thousands of experimental organ transplantations. In order to deal with organ rejection, I administered, radiation and sera for diminishing the immunity of the organ receiver. Besides that, I also administered uncountable agents to recipients of organs in order to trigger, diminish or completely wipe out the immune capacity which can be compared with Aids.
During my university and hospital appointments in the early seventies, and later undercover in the pharmaceutical industry, I discovered at that time that animals didn't die because of rejection of the transplanted organ but because of multiple infections which can be compared with human Aids victims. So, I noticed that Aids can be induced by radiation, aflatoxins, Immuran/prednisolone combination, anti-lymphocyte sera and many other bio-warfare agents.
Dormant HIV virus
As head of the Department of Experimental Microsurgery, and involved in all transplantation and immunological experiments, I also have been involved in many controversies. Especially the connection of my work and the polemic concerning the transmission of HIV in many ways. I discovered not only in experiments but also in the extensive scientific literature the role of an obligatory co-factor that trans-activates the "Dormant" virus HIV in specific human cells.
This obligatory co-factor which trans-activates the "Dormant" virus in specific human cells are deliberately introduced into mostly black-skinned African people, governed by massive environmental factors as you can read in our book: "Aids and Ebola the greatest crime in medical history against mankind," in order to depopulate Africa and other parts of the world.
This article is to enlighten readers about the real origin of Aids and the true nature of famous international researchers as Robert Gallo. As far as Gallo is concerned, Ricardo Veronesi, professor of the Faculty of Medicine at the University of Sao Paulo, was personally informed about the true nature of Gallo's research long before this controversy turned into a public scandal and as a consequence thousands of scientific Aids dissidents.
It was no less than Francoise Barré-Sinoussi of the French Pasteur Institute who revealed the criminal intention of Gallo. Not only she became an Aids dissident? but also the discoverer of the HIV virus Luc Montagnier disputed Gallo, the fake discoverer of the HIV virus.
In their opinion, the major bursts in the common scientific approach of lies in its ignoring that the pathogenicity of the HIV indeed is governed by multiple deliberate environmental factors and one of these determinant factors are the PCR test (Polymerase Chain Reaction Test).
Using and extrapolation of these kinds of techniques we can conclude that people who have HIV in their bodies, were purposely infected with this virus which can lead to Aids. Bio-warfare scientists are able to make black-skinned people 'Africans) artificially susceptible for HIV or Ebola by using controllable diseases as a cover-up.
Most of the biowarfare research using viruses which cause Aids and Ebola is predominantly carried out in Germany and Japan until 1945 and since then mainly in the USA and France, using Nazi and Japanese (military) scientific war criminals.
Autism/Aids/HIV Theory Dissidents Like Jeffrey Bradstreet
The official scientific origin of the diverse HIV-strains has been placed somewhere between 1938 and 1948 when scientist T.F. Smith et al, published an article in the authoritative medical journal Nature, is 1988, captioned: "The phylogenetic history of immunodeficiency virus."
He wasn't the only scientists who revealed the true nature of the HIV virus. Smith's efforts to reveal the real origin of HIV was followed, to name a few, by Sharp et al with his article: "Understanding the origins of Aids viruses," also in Nature, followed by Meyers et all with: "The phylogenetic analysis of the HIVs." But the most important article is described in the top of the bill of medical journals the Lancet, by scientist L.A. Evans et al who discovered the; "Simultaneous isolation of HIV-1 and HIV-2 from an Aids patient".
All these mentioned scientists agreed that the distribution of the HIV virus was an intentional action. Their findings make it very conceivable that this distribution was intentional, because sometimes both the new viruses HIV-1 and HIV-2, respectively HTLV-IV, are existing in one and the same person according to Evans. And because his publication is checked by the editing and scientific boards of the Lancet, the outcome of his investigation was true. This counts also for thousands of publications in other medical journals as described in our book "Aids and Ebola the greatest crime in medical history against mankind."
According to the famous Aids/HIV theory dissident Wolff Geisler, further evidence of the intentional distribution, out of the mentioned simultaneous infection of the same persons, it was described as a second Aids epidemic in the same black-skinned population, by an inefficient transmission of the HIV virus. The appearance of this extreme rare retrovirus among the African Aids patients is so conspicuous that some world famous scientists uttered a sentence about it. They alleged this to be; "Only another acquired opportunistic infection but rather an additional death sentence." But is it?
In Africa the probability of an early death of HIV patients is three times higher than elsewhere when HIV patients are simultaneously infected with HTLV-1 as described in the Lancet by Page et al in his scientific publication: HTLV-I/II seropositivity and death from Aids among HIV-Seropositiveintravenous drug users (Lancet, 1990; 335: 1439-41), an even more extremely important publication for the Aids/HIV theory dissidents. Because, especially HTLV-I, among many other HIV viruses, was only demonstrated in Uganda, Ghana, South Africa, and Namibia.
Only in these countries, HIV patients appear simultaneously up till now. According to Wolff Geisler, the concomitant existence of HTLV-I and HIV produces the observed rate of Aids patients in Uganda, Kenya and black-skinned people in Florida, USA and some Caribbean Islands, even though in general black people are by nature more resistant against HIV-infection than pale-skinned persons (see below). This means the HIV viruses are genetically engineered as describe in our book.
No less than Luc Montagnier et al, the discoverer of the HIV virus stated that this virus is made out of the Nazi eugenics and a genetically engineered experiment as well as the development of Aids-causing viruses in horses. In a very talked about an article he described in the authoritative Annals of Virology: "A new type of retrovirus from patients presenting with lymphadenopathy and acquired immune deficiency syndrome": Structural and anti-genetic relatedness with Equine Infectious Anemia Virus EIAV (horse Aids), 1984; 135E: 119-31.
Equine Infectious Anemia Virus EIAV (HIV/Horse-Aids) made by Nazi Germany
If we compare these findings to our references in:"Aids the greatest crime in medical history against mankind" the book now available at Amazon, the HLA-A, B, C, DR3 and DR5 loci, is examined by the Nazi's led by Otmar Verschuer.
In 1956, he joined the American Eugenics Society and worked under auspices of the Rockefeller-fund. He was also head of the Department of the Kaiser Wilhelm Institute in Germany.
Furthermore, we have to take into account that within people who have blood type HLA-DR3 Aids, it is much less common than in people who have the HLA-DR5 type. Under the Nazi's research, it is important to note that precisely the HLA-DR5 type occurs mainly in Jews. The HLA-DR3 type contrast is most common in dark-colored Africans.
The two evidence or references are enough to let you know vividly what took place. In general you can say that it is harder for blacks to get Aids than as it is for whites, but blacks have been made susceptible for a broad spectrum of brand new diseases caused by Germans, partly under the auspices of the South African Apartheid regime, and after the war under the guidance of the U.S.A.
Nowadays we now know that monkeys do not get Aids when infected with the human Aids virus. The same goes for tuberculosis until the moment that monkeys in a laboratory made receptive. Therefore black-skinned people are under no circumstances contaminated with Aids by monkeys with or without eating them. That is so to speak a criminal WHO/ CDC / FDA scientific fairy tale.