NuWave Oven Recipe for making a Meatball Bake at home, that is similar to one sold by Marco's Pizza.
A good NuWave Oven Recipe for Raspberry Macadamia Nut Pancakes. Low carb options are provided.
A full recipe for making great Lasagne in the NuWave Oven. Low Carb options are mentioned.
Instructions for cooking a Hormel Pork Roast in the NuWave Oven.


Zionsville Community High School Royalaires and Choralaires have just completed another highly awarded competition season and will have their final performances of this year on May 17 and 18 at 7 p.m. at the Zionsville Performing Arts Center. This year, they brought home five Grand Championships, two Division Championships and many awards including Best Vocals, Best Choreography, Best Costumes, Best Combo and Best Crew. Tickets for Finale 2013 can be purchased online at www.zvilleperformingarts.org.


The brewing device, offered in extremely fashionable finish (copper, black matte, polished steel), utilizes a super functional design to bring your brewing into the next level.
The clever device allows you to enjoy a more intuitive and steady pour with Stagg's precision gooseneck pour spout. Its weighted handle allows for more control over the flow and direction of your pour like a true coffee connoisseur. You can pour at an even, slow, and controlled pace with the counterbalanced handle with moves the center of mass towards your hand for a more accurate pour.
The Stagg Stovetop Gooseneck Kettle also includes an integrated thermometer on the lid which makes its easy to gauge the temperature of brews when precision and accuracy is of utmost importance to the optimal cup. You'll never have to guess whether your coffee is at the right temperature again. The thermometer's red zone indicates the optimal coffee brewing temperature between 194 and 205 degrees going up to 203 degrees for tea.
The pour-over kettle can hold up to a liter of water which is enough to brew up seven five-ounce cups of coffee or tea.
While its design is both cutting edge and intriguing, making it a covetable object for any modern kitchen. The kettle features an attractive symmetry from the spout to the handle in a design that follows both form and function. In an ultra trendy and fashionable finish (copper, black matte, polished steel), the kettle is well suited to be a focal point of your home. The Stagg Stovetop Pour-Over Kettle also features a newly designed stainless steel lid that is both durable and stylish.
The Fellow Stagg's innovative packaging also features a "Pour Over Perfected" guide which helps users understand the right coffee to water ratios to create the ultimate pour over. Newly interested baristas can get started on their favorite brews from the moment they open the kettle's box.
The Stagg's built-in thermometer takes the guesswork out of brewing delicious coffee or tea. The kettle's lid contains a thermometer which accurately reads temperatures to indicate the optimal amount of heat for your favorite beverage. You never have to worry about over or under brewing again.
The Stagg Stovetop Pour-Over features a precise gooseneck pour spout so you can pour at an even, controlled, and slow pace serving up optimal brews with accuracy without spilling or splashing. Its weighted handle allows for more control over the flow and direction of your coffee or tea.
The Fellow Stagg can hold up to a liter of water which can accommodate seven five-ounce cups of coffee or tea. You only need to heat up one pot to enjoy a caffeine pick-me-up throughout the day. Or use its large capacity to brew up morning coffees for your entire family.
The Stagg is not only ergonomically designed to be weighted right in your hand, it also features an ultra stylish silhouette and modern design. Its bold silhouette looks elegant in even the most modern of kitchens, while its very trendy finish - copper, black matte, polished steel - is the metal of the moment.
The kettle's robust design and high-quality materials ensure that it is easy to care for and will also stand the test of time. The kettle is compatible on a variety of ranges making it easy to use wherever you are.


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Home Mint Sets
1960 U.S. Mint Set
There is a total of 10 coins. Both from the Philadelphia and Denver Mints.
Comes in the original Mint Packaging with certificates.
All sets come in original Government packaging.
This set is complete and comes in the original U.S. Mint envelope. There are ten coins from both Philadelphia and Denver Mints.
1965 Special U.S. Mint Set
This set comes in the original envelope from the U.S. Mint. There are a total of five coins. Stock Pictures
This set comes in the original box from the U.S. Mint. There are a total of five coins.
This set comes in the original box from the U.S. Mint. There is a total of five coins.
There is a total of ten coins from both the Philadelphia and Denver Mints.
All sets come in the original Government packaging. Stock Pictures.
There are a total of ten coins from both the Philadelphia and Denver Mints.
There are a total of 11 coins from both the Philadelphia and Denver Mints. Stock Pictures.
This set comes complete in the original envelope from the U.S. Mint. There is a total of eleven coins from both the Philadelphia and Denver Mints.
There are a total of 13 coins from both the Philadelphia, Denver, and San Fransico Mints.
This set comes complete with the original box from the U.S. Mint. There is a total of thirteen coins from both the Philadelphia and Denver Mints.
This set comes complete with the original envelope from the U.S. Mint. There is a total of twelve coins from both the Philadelphia and Denver Mint...
1976 3pc Silver U.S. Mint Set
This set contains a total of three coins, the Quarter, Half, and Dollar. Comes in the original envelope.
This set contains a total of 12 coins. Both P & D Mints and the original white envelope. Stock Pictures.
This set contains a total of 12 coins from both the P & D Mints. Set comes in the original white envelope. Stock Pictures.
This set comes in the original envelope from the U.S. Mint. There is a total of twelve coins from both the Philadelphia and Denver Mints.
This set contains a total of 13 coins from both the P & D Mints. Set comes in the original white envelope.
This set comes in the original envelope from the U.S. Mint. There is a total of thirteen coins from both the Philadelphia and Denver Mints.
This set contains a total of 10 coins from both the Philadelphia and Denver Mints. Set comes in the original envelope.
There is a total of ten coins from both the Philadelphia and Denver Mints. Also included is the paper inserts.

redpajama_set_name: RedPajamaCommonCrawl


It isn't easy being Mr. Wild Dingo. Because, I'm not so good with acronyms. Or names. Or numbers. He's always interpreting Wild Dingo-speak. Whenever I ask for the can of W4, Mr. Wild Dingo always replies "W4, WD40 whatever it takes."
When we moved home, we discovered our old Internet provider had a newer, faster service called N-Max, which provides 6 mbps upload and download speeds. The perfect speed needed to stream movies and TV shows. The provider came out and informed us we'd have to move our antennae from the old location to the roof of our garage so we could access the tower across the mountain range. So we spent a full, rainy Saturday afternoon, stringing RJ45 under our house, up to the utility box and back down to the garage---all through empty underground conduit piping that we (well, Mr. Wild Dingo) had good sense to install long ago for future needs.
And I confess: I just Googled RJ45 to make sure I had that acronym correct.
It wasn't too bad of a project, except for the pouring rain. And it wouldn't have taken so long, had Mr. Wild Dingo failed to make a tangled mess out of 500 feet of RJ45. Luckily one of us has the patience to untangle that mess.
As we walked the dogs that afternoon, I asked Mr. Wild Dingo, "So all I have to do now is call the Internet guys and ask them to install MI6?"
Mr. Wild Dingo only smiled, "No, they won't install MI6 now. They don't package James Bond with their service yet."
N-Max, MI6; whatever it takes.
MI6, oh dear. Must get a grip on your acronyms. Watch out for those streaming movies. It cen get addictive.
If you get an internet provider who does package James Bond with your service, please let mama know. It would be worth moving to CA for that level of service. Mama is old enough that she would like to have the original James Bond, please [Sean Connery].


text:
Back to Air Transport
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ATW announces 2019 Award Winners
William Franke, Air New Zealand, airBaltic, Spirit Airlines, Dallas Fort Worth International Airport, Airlink and the Airbus A220 airliner have been announced as among the top winners of the ATW 45th Annual Airline Industry Achievement Awards.


text:
Q: rename the categories and add the missing categories to a Series PANDAS I want to rename the categories and add the missing categories to a Series.
My code:
codedCol = bdAu['Bordersite']
print 'pre:'
print codedCol.head(10)
codedCol = codedCol.astype('category')
codedCol = codedCol.cat.set_categories(['a','b','c','d','e','f','g','h','i','j'])
print 'post:'
print codedCol.head(10)

When I do this I get NaN as the result.
pre:
0    3
1    3
2    2
3    2
4    3
5    4
6    5
7    3
8    3
9    3
Name: Bordersite, dtype: int64
post:
0    NaN
1    NaN
2    NaN
3    NaN
4    NaN
5    NaN
6    NaN
7    NaN
8    NaN
9    NaN
dtype: category
Categories (10, object): [a, b, c, d, ..., g, h, i, j]

What am I doing wrong here?
Thanks
Kheeran

A: First or creating catagories you can use .astype('category'), but categories are added from your column or Categorical with parameter categories where are defined.
You can use:
codedCol = bdAu['Bordersite']
codedCol = pd.Series(pd.Categorical(codedCol, categories=[0,1,2,3,4,5,6,7,8,9]))
print (codedCol)
0    3
1    3
2    2
3    2
4    3
5    4
6    5
7    3
8    3
9    3
dtype: category
Categories (10, int64): [0, 1, 2, 3, ..., 6, 7, 8, 9]

And then rename_categories, but number of items in categories have to be same, else error:

ValueError: new categories need to have the same number of items than the old categories!

codedCol = codedCol.cat.rename_categories(['a','b','c','d','e','f','g','h','i','j'])
print (codedCol)
0    d
1    d
2    c
3    c
4    d
5    e
6    f
7    d
8    d
9    d
dtype: category
Categories (10, object): [a, b, c, d, ..., g, h, i, j]

A: You've set the categories to the following: ['a','b','c','d','e','f','g','h','i','j']. The current values in the column in codedCat do not match any of the categories. Therefore, they get re-set to NaN. For further reading, consider this example from the docs:
In [10]: raw_cat = pd.Categorical(["a","b","c","a"], categories=["b","c","d"],
   ....:                          ordered=False)
   ....:
In [11]: s = pd.Series(raw_cat)

In [12]: s
Out[12]:
0    NaN
1      b
2      c
3    NaN
dtype: category
Categories (3, object): [b, c, d]

Since "a" is not a category, it gets re-set to NaN.

A: Use add_categories to add_categories to a series.

redpajama_set_name: RedPajamaStackExchange


text:
Tag: Thrash
Album Review: Pizzatramp – Grand Relapse
Finally, there is something exiting to come out of Wales, other than the M4. Pizzatramp's 12" picture disc on TNSrecords is a win. FFO: 80's hardcore thrash crossover and swearing.
Review by Ollie Stygall.
Pizzatramp are from Wales so let's start with some facts about Wales.
Wales is the largest county in England. It is only accessible by a bridge and they make you pay to enter Wales, presumably to discourage you from bothering. Wales is owned by Prince Charles, but he doesn't live there. The Welsh have their own language but you need an overactive saliva gland to speak it, but that's OK because 99% of Welsh people don't understand it.
Wales is full of castles because they're extremely unpopular and got attacked a lot… they even attacked themselves, because they don't like each other either! Wales is full of mountains, because even the landscape dislikes the Welsh and has tried to make itself as uninhabitable as possible. Wales has a seaside resort called Barry, like the boring Brummie character from Auf Wiedersehen Pet. Wales has nothing at all to do with the mighty and majestic sea creatures that its name sounds like. Catherine Zeta Jones is Welsh – and hot – but she'd rather shag an American pensioner than Welsh blokes.
However, there is now something exiting to come out of Wales, other than the M4. A musical behemoth that does go some way towards righting the wrongs done by The Manic Street Preachers: and that is Pizzatramp.
Pizzatramp make out that they're a shambolic, pissed up mess. They're not. Well, they may well be to be honest, but that doesn't stop them being a fucking incredible band. Continue reading "Album Review: Pizzatramp – Grand Relapse"
Author Shout LouderPosted on May 8, 2019 Categories Album Reviews, UncategorizedTags Album Review, hardcore, Ollie Stygall, Picture Disc, Pizzatramp, Thrash, TNSrecordsLeave a comment on Album Review: Pizzatramp – Grand Relapse
Album Review: Thanx 4 All The Shoes – Another Ride
The intense and heartfelt third album from Italy's Thanx 4 All The Shoes is a solid skate-punk/thrash offering. FFO: Propagandhi, Strung Out, NOFX.
Review by Jake Jeremy.
Another Ride is the third album from Italian melodic hardcore outfit Thanx 4 All The Shoes, due to be released on November 25th on Disconnect Disconnect Records in the UK.
In their 11 years together, Thanx 4 All The Shoes have toured all over mainland Europe and Japan, building a reputation for their technical skill and thought-provoking lyrical messages. You can expect shades of melodic hardcore and thrash in the style of Propagandhi and Strung Out, however with a name like Thanx 4 All The Shoes I'm slightly gutted I'm not listening to a NOFX tribute band. Either way, I'm jumping in with gleefully expectant ears.
Opening track One Pen One Book instantly draws comparisons to Propagandhi, but I'm actually getting more of an 80's thrash vibe from the quickly muted guitar attack and vocal approach. Think Anthrax and Megadeth if Chris Hannah wrote their newest tracks. It is a solid opening and a promising precursor to the rest of this album.
Title track Another Ride and it's much of the same: fast and melodic with a twin guitar attack reminiscent of Propagandhi. What stands out most is the high-flying vocal harmonies and METAL AS HELL midsection where small guitar flourishes embellish another 80's thrash thrill ride. This is what I want from this band: the sections where they truly let fly and break away from what's 'expected' within standard modern hardcore are what set them apart. Continue reading "Album Review: Thanx 4 All The Shoes – Another Ride"
Author Shout LouderPosted on November 22, 2017 Categories Album ReviewsTags Disconnect Disconnect Records, Jake Jeremy, Melodic Hardcore, No Reason Records, Skate punk, Thanx 4 All The Shoes, Thrash, White Russian RecordsLeave a comment on Album Review: Thanx 4 All The Shoes – Another Ride


Want to see the Harlem Globetrotters when they come to Fresno this January?
Enter our coloring contest and that dream could become a reality. SaveMart Center has generously donated family 4-packs of tickets to the team's January 12, 2018, 7 pm performance at SaveMart Center, and we're giving them away!
Two Fresno County Public Library users will win 4-packs of tickets.
Pick up a Globetrotters coloring sheet at your nearest Fresno County Public Library branch.
Sign the back of your coloring page, with your first and last name, last four digits of your library card barcode, and (for any entrant under 18) parent/guardian's name and phone number.
By closing time on Tuesday, January 2, turn in the coloring page at a Fresno County Public Library branch.
One entry per person. Winners will be chosen by random drawing.
Upon submitting a coloring sheet that meets the guidelines established above, eligible followers will be entered for a chance to win the prize. The contest is open to all residents of Fresno County, with a Fresno County Public Library card/account, except Fresno County Public Library employees and members of those individuals' families. All contest entries must be submitted by end of business on Tuesday, January 2 (branch closure times vary).
Winners will be selected by random draw from among all eligible entries received and announced on social media by Friday, January 5, 2018. Odds of winning depend on the number of qualified entries received, but no entrant may win multiple pairs of tickets and only one prize per household will be awarded. Prizes must be accepted as awarded. No cash alternatives, substitution, or transfers of the prize will be permitted. Prizes must be picked up at a Fresno County Public Library branch selected by the winner.
Additionally, winners agree that Fresno County Public Library may use images of their coloring page and their name, for advertising and publicity purposes without further compensation (except where prohibited).
Fresno County Public Library is entitled to interpret these rules as required and its decision is final.
This prize draw is subject to all applicable federal and state laws and regulations. These incentive prize rules are subject to change without notice in order to comply with any applicable laws or the policy of any other entity having jurisdiction over the prize draw.


text:
Five Thoughts on the Wild Card Game
All wild card games – any single MLB game for that matter – is pretty close to a coin flip. When you factor in how close these two teams played – the 10-9 head to head record and shared 89-win total tells that story – the Blue Jays versus Orioles is about as much of a 50/50 proposition as you could imagine. Even the starting pitchers share similar struggles against the other team. I have no interest – and certainly lack the ability – to make any sort of educated prediction about a game this unpredictable, so here are a handful of random thoughts I have. Let's Go O's!!
The Chris Tillman Advantage
There has been quite a bit of hand wringing over the Orioles' decision to start Chris Tillman by fans of the team and there will definitely be even more of that if Tillman is ineffective early. There is certainly cause for concern. Tillman has averaged nearly 93 MPH on his fastball this season according to Brooks Baseball, but has been under 92 MPH since he began dealing with shoulder discomfort in mid-August. In his last start – last week versus Toronto – Tillman struggled to break 90 MPH, lacked command of his fastball and had to lean heavily on his changeup (which to his credit he had working) to get him through the game. On the season, Tillman throws his changeup about 15% of the time but last week he went to the off speed pitch nearly 24% of the time. There is a concern that Tillman is still hurting (he almost certainly is) and his fastball velocity and command will remain off in the wild card game.
The kicker might be that if Tillman's fastball is better – let's say an average of 92 MPH with his normal command – and he can keep the Jays hitters believing he might throw 24% changeups again (which will involve throwing a lot of changeups early) he might be at a real advantage. Players from both sides have talked in recent days how there are no secrets between the teams and that they know each other really well. The catch is that the Tillman the Jays saw last week and who pitched well against them is not the same one they were used to. Assuming his fastball comes around tonight, he has the opportunity to keep the Jays' hitters guessing in a way he would not have been able to without that changeup heavy performance from a week ago.
My Only Prediction: Liriano Will Get in Early
The one thing I am most certain will happen – maybe the only thing I am certain about at all – is that Liriano will get in the game relatively early. The Blue Jays – from their general manager on down – have stated that they expect the left-handed starter to make an impact out of the bullpen. While not a flaw unique to him, Marcus Stroman tends to stumble the third time through the order which means the Jays might want to get him out of the game sooner rather than later. Stroman also has struggled against Baltimore this season. The Orioles do not hit left-handed pitching well at all relative to right-handed pitching. With Stroman starting, the O's are starting left-handers Hyun Soo Kim and Chris Davis, and Michael Bourn. Getting Liriano in early might cause Buck Showalter to burn Kim, Bourn or Alvarez early in the game by pinch hitting for them.
There are so many reasons to get Liriano in the game early that it is difficult to believe John Gibbons won't do just that.
Does Ubaldo See Action?
If Buck was 100% certain that he was not going to pitch Ubaldo Jimenez unless he had no other options, I doubt the resurgent right-hander would have made the wild card roster. I don't believe Buck would waste a spot on a player who served no utility. I believe that Ubaldo is on the roster because Buck envisions a way in which he can help. That doesn't mean he will play – one has to think the ideal plan is for 4-5 innings from Tillman and 4-5 from the short relief group – but I do think Buck has some scenarios in mind where he would like to get Ubaldo in the game. If true and if Buck gets the opportunity it will be interesting to see how he utilizes Ubaldo. Personally, I would be leery of bringing Ubaldo out of the bullpen at all in a must-win game. That doesn't seem like the ideal role for a starter who has only recently gotten his command and mechanics under control. Buck is very, very good at using his pitchers – especially when he isn't worrying about tomorrow – so I am interested in seeing how he thinks Ubaldo could best contribute (although hopefully the game goes so well that it becomes a moot point).
The Bench(es)
The O's are carrying a six man bench comprised of Ryan Flaherty, Caleb Joseph, Pedro Alvarez, Drew Stubbs, Trey Mancini, and Nolan Reimold. Under many other situations, I would assume that most of those guys won't see significant action. Maybe Flaherty would get in the game as a defensive substitute in the later innings. Maybe Stubbs pinch runs and Reimold replaces Kim in the outfield later for defense purposes. Alvarez could definitely see an at bat against a right-handed reliever late in a normal game. However, this might not be a normal game given the possibility discussed above of the Jays using a two-handed starting pitcher approach. If Liriano does come in early, it will put pressure on Buck to swap out his lefty bats. Does he remove Kim in the fourth or fifth inning knowing that likely means a lesser hitter will face a right-handed reliever late in the game? The same goes for Bourn. Does Buck take him out in the 5th for offensive help and downgrade the outfield defense in the process? At some point I am sure we will see this game of chicken. If Buck does opt to remove a left-handed batter or two, that suddenly thrusts some of the right-handed bench players – Reimold, Stubbs, Mancini – into possibly bigger roles than they would otherwise have in this game.
How Long Could Zach Go?
Buck has used Zach Britton for more than three outs seven times so far in 2016. In fact, he just did it on Sunday against the Yankees with a 5-1 lead in what at first seemed like a curious move. Sure, the O's needed to win that game in order to control their own playoff destiny but it still seemed like an odd move. Was the primary or secondary motive behind the decision to give Britton a warm up five out save for possible wild card scenarios? I think that at least played a small role in the decision. So the question is – now that Buck has shown the willingness to use Britton for multiple innings (and relied heavily on his best reliever, Andrew Miller, in the 2014 playoffs) and maybe given a hint that he plans on doing it in the playoffs, how far will he push Zach in a winner takes all game?
A two-inning save if the Orioles are holding onto a narrow (one to two run) lead would seem to almost be a given. But what about other situations? Does Buck go to Zach to start the 7th (or during the 7th) if the Orioles have a narrow lead? Does he got to him in a tie in 8th? What about the 7th? Could Britton see action in the 7th inning (or even earlier?!?) during a big spot in the game? Or does Buck stick to what brought them to the dance and roll out his set up men one after one leaving Britton to get three outs at the end of a tight game? Of course, all of this is dependent on the situation – mainly, the O's having a lead late or being close late. The last thing any manager wants to do in a winner-take-all game is not utilize their best weapons to the fullest and Britton is clearly on the O's best weapons. It is just the meaning of fullest is a bit trickier to nail down when Buck has no way of knowing what the biggest moment in the game might be.
Written by Paul Cooke On October 4, 2016 In General Tagged playoffs, wild card
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Thomas, just 5-foot-9, has emerged as one of the NBA's top point guards.
Isaiah Thomas isn't much for subtlety.
So when the Boston Celtics guard returned to his roots recently, dedicating a Boys & Girls Club court to those kids from his hometown of Tacoma, Wash. who aren't much smaller than him, no one should have been surprised when they saw the choice of wallpaper.
For the 5-foot-9 point guard whose Celtics are second in the Eastern Conference (36-19), and who will make his second All-Star appearance in New Orleans this weekend, it has been quite the journey since he was coming up. From Tacoma to the University of Washington to his 'No. 60' status in the 2011 NBA draft to the outer edges of the MVP discussion, his is the NBA's ultimate underdog tale.
No wonder he started crying when he thought back on it.
Players this short aren't supposed to stand this tall.
Yet here is Thomas, son of a mother who is a tireless nurse and a father who has spent nearly three decades as a parts inspector for Boeing, ranked second among all NBA players in scoring (29.8 points per game) and well on his way toward fulfilling a legacy that Celtics general manager Danny Ainge dared him to dream about upon arrival.
It was Feb. 19, 2015 when everything changed: Thomas to Boston in exchange for Marcus Thornton and the Cleveland Cavaliers' 2016 first-round pick.
The Sacramento Kings team that drafted him with the final pick had let him leave in free agency in that summer before.
"They just kept trying to push me out, which they did, and I'm fine with it now," he says.
He wanted to play for the Lakers, having adopted his father's fandom while finding inspiration in Kobe Bryant's legendary work ethic, but L.A. opted for Jeremy Lin instead. The Kings, clearly unconvinced that Thomas was a good fit alongside DeMarcus Cousins, went with Darren Collison.
Enter the Phoenix Suns, who gave him a four-year, $27 million deal and had ill-fated visions of a point guard platoon approach with Eric Bledsoe, Goran Dragic and Thomas sharing time.
Minutes before the 2015 trade deadline – and not long after Dragic had been dealt to Miami – Thomas was sent to Boston. Ainge, he of the two championship rings that made him a local authority on all things Celtics, would set a very different tone.
"I sat down with Danny Ainge, and he was talking about how much of a fan he's been of mine since the draft," said Thomas, who is shooting 46.8% overall, 38.2% from three-point range and on pace to tie his career-high in assists (6.3 per game). "And when he told me I could be a Celtics legend by the way I played … I'm thinking, 'OK, now he's just excited about the trade. He's just saying stuff to make me happy, and make me feel comfortable.' But he really felt like that.
When it comes to NBA stardom, a player's confidence isn't enough to spark a rise – even when it's someone like Thomas who doesn't traffic in self-doubt. A player needs to be empowered, to be put in a position for his talents to be maximized and his weaknesses to be masked.
"Luck is what happens when preparation meets opportunity," the Roman philosopher, Seneca, once said.
But what if the right opportunity doesn't come?
And therein lies the genius of the Celtics' approach.
"(With) everybody who comes through, we're not looking at what people say they can't do, we're looking at what they can," Stevens said. "They're in the NBA for a reason. You soar with their strengths, and then you go from there.
The good? Not only is he on pace to post the second-best scoring season in Celtics history behind Larry Bird in 1987-88 (29.9 points per game), Thomas has earned his reputation as the king of the fourth quarter. His current mark (10.6 points per fourth) is the best on record since the league started keeping fourth-quarter data in 1996, and he's lapping the field in terms of 20-plus point fourth quarters (he has four – including 29 in a career-high, victorious 52-point outing against Miami on Dec. 30 – while no other players have more than one).
Even peak Kobe (9.5 per in 2005-06), the Oklahoma City Thunder's triple-double machine, Russell Westbrook (9.4 per this season), and LeBron James (9.1 per in 2007-08 while with Cleveland) didn't score at this rate late in games. It's even more impressive considering Thomas has been more efficient in the fourth (49.2% overall and 41.3% from beyond the arc).
The bad? His defense, as he is well aware, is much-maligned.
"Now that my offense is at such a high level, it's like, 'Well now his defense - he can't guard anybody,'" said Thomas, who is last in the league in defensive real-plus minus (minus 4.38) for a Celtics team that ranks 19th in defensive rating (106.2 points allowed per 100 possessions). "And that's never been the case. Like, nobody has ever picked on me or just ran every play towards me because I can't stop anybody. It is what it is.
Stevens, true to his confidence-building ways, has a word of caution for Thomas' critics.
When it comes to people like Thomas, who learned long ago that selling himself short (no pun intended) was the quickest way to welcome failure, there's really no such thing as a ceiling.
Playing in college was the first step, and he did that with aplomb for three seasons with the Huskies ("Cold…blooded!" announcer Gus Johnson hollered when Thomas' game-winner gave Washington the Pac-10 title in 2011. Getting to the pros was Phase Two.
Six years later, his production in the pros (18.7 points, 5.2 assists per game) has been enough to change the conversation that surrounds him. This is no longer about where Thomas ranks in the league's history of tiny guards, if only because he's well on his way to surpassing the Calvin Murphys, Michael Adams, Terrell Brandons, Damon Stoudamires, Earl Boykins, Muggsy Bogues, Spud Webbs and Slater Martins of this world.
So as Thomas ponders the question of where this is going, of the coming hurdles and how many of them he'll be able to clear, the same fearlessness that he uses to enthrall the masses on the court can be found in his answer.
"I want to be the best player to ever play now; and that's just what I want," he says so boldly. "Maybe I won't get there, but I put those goals in front of me so I can chase those.
Go ahead, in other words, pick him last again.


Hurricane season officially began on June 1st. But the worst hurricanes of century have occurred in late summer or fall. Below is a list of steps you need to take to be prepared for those possible hurricanes yet to occur.
1) You need to have a safety kit ready. It's recommended that you keep at least enough food and water for each person in the family to last 3 days.
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GREG DAVIS OUT, STEVE ADDAZIO TO BECOME IMMORTAL
By Spencer Hall@edsbs Dec 6, 2010, 12:37pm EST
Share All sharing options for: GREG DAVIS OUT, STEVE ADDAZIO TO BECOME IMMORTAL
(Via ShaggyBevo.)
The inimitable Greg Davis, longtime assistant to Mack Brown and master of the static offense, is out at Texas along with offensive line coach Mac McWhorter and defensive line coach Mike Tolleson. These represent significant changes made in the wake of ineptitude so galling Texas fans looked at Mike Sherman and thought, "Oh, to have a dynamic charisma bomb like that running our offense!" In response Mike Sherman scratched his ear, smelled it, and judged it to be the most significant emotional experience of his life.
Meanwhile, in discussing next season's staffing, Urban Meyer said this:
There are no words. But there is an image after the jump.


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歡迎來到國家兩廳院無障礙服務,請使用快捷鍵跳至各區塊:選單 ALT+M、主文 ALT+C、搜尋 ALT+S、回首頁 ALT+H、字級 ALT+1 到 3。
國家兩廳院 - 無障礙服務
字級 小 中 大
●交通資訊
●購票優惠
●節目查詢
●環境設備
●導覽資訊
●多媒體服務
Invisible Cities - A Tale of Three Cities
2019/8/17(Sat. )2:30PM、7:30PM 2019/8/18(Sun.)2:30PM
National Theatre Institute Experimental Theater
NTCH Asia Connection Project
The Asia Connection is a three-year (2019- 2021) project connecting Asian allies to create a new vocabulary of art. We endeavor to explore Asian creativity to discover the future of Asian performing arts with the power of "We Art Together".
Dimensionality of the Theatre in Inter-Asian Exchanges: From the Interchange Project of Theatres of Eastern Asia to Invisible Cities - A Tale of Three Cities
written by Chung CHIAO,Artistic Director of the Assignment Theatre
The idea of "Inter-Asia" – a term which literally means "inside Asia" – focuses on a constantly-moving state of exchange on equal footing. When this state stagnates, the "conversation" which is based on the medium of international relations will be lost. It's a core concept for inter-Asian exchanges in the context of "cultural decolonization."
From this point of view, the Assignment Theatre from Taiwan, Experimental Theatre of Busan, Korea, and M.M.S.T. from Fukuoka, Japan, joined forces to build a platform for exchange and performance in order to enhance inter-Asian communication.
Since 2016, we have focused on theatrical exchange revolving around the theme of "cities". Curation aims to reflect upon the modernization of cities, avoiding being trapped by such myths as "art only" or "art should serve the market", and preventing creativity from being restrained by these fallacies.
The "inter-Asia" which is portrayed here is not about the pyramidal setup of power. Its foundation does not consist of firm relationships in order to keep the upper layers stable. The creation of every theatre is allegorically:
A death which turns into the past because it has lived once.
A corrosion which let us know something existed before.
So all ends well in death and corrosion!
Tomohide MOMOSE
M.M.S.T was founded in Tokyo, Japan, in order to "establish contemporary theatre aesthtics." Its work includes stage works, audio performances, and video installations. Tomohide MOMOSE is the current leader and resident director of the M.M.S.T He frequently opens up dialectical discourse about the specific status of bodies onstage in his works. At the same time, he handles various texts from the classical period to contemporary ones with his trademark distinctive sense for bodies. His work was awarded the "Best Work" award in the Competition of the Theatre held by the Foundation for Arts and Cultural Promotion of Fukuoka City.
Hyoyun YANG
Hyoyun YANG graduated from Kyungsung University with a double major in Drama and Dance. She is the playwright and director of the "Dongnyeok" Theatre. Her major works include Un ak (Best Picture Award of the 31st Korea Theatre Festival), HANARO Project Vol. 2 Drama (at the East Asia Theatre Network), and A Great Fall (the 53rd performance of the Busan City Art Organization).
Wei-lien WANG
Born in Taipei and raised in the new Hsin-Ho village near Nanjichang. Wei-lien is the grandson of the late General Cheng-chang WANG, while his father is one of the founding members of the United Bamboo Gang. After Wei-lien received his M.A. degree from the Department of Drama and Theatre at NTU, he joined the Critical Point Theater. From then onwards, he has worked avidly and innovatively in scriptwriting, directing, acting, tutoring, and lecturing for theatre. Wei- lien regards "being human" as a core tenet of the performing arts. He seeks novel and vivid theatre languages in cultural and social contexts. Recently he has used Analects of Confucius as a source of inspiration. Currently, Wei-lien is a member of the Little Theatre School, Lectures for Playgroups, Assembly at Lobby, and Weekly Mahjong Club.
Production Leader
Chung CHIAO (born 1956, Sanyi, Miaoli, Taiwan) is a playwright, theatre director, poet. He received his M.A. in Arts from Chinese Cultural University, and is currently the artistic director of Assignment Theatre.
Organizer: National Theater and Concert Hall
Special Thanks to: Assignment Theatre, Busan Cultural Foundation
◎Approx. 100 minutes without intermission
◎Post-talk: at the Experimental Theater after each performance
◎The program is subject to change
Very appreciate for your feedback! This is an online questionnaire:
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Home » Books » Lois Marshall
Lois Marshall
James Neufeld
Although she called herself "just a singer," soprano Lois Marshall (1925-97) became a household name across Canada during her thirty-four year career and remains one of the foremost figures in the history of Canadian music. She rubbed shoulders with Canada's musical aristocracy – Glenn Gould, Sir Ernest MacMillan, Jon Vickers, Maureen Forrester – but Marshall always held first place in the hearts of her adoring fans.
At the height of the Cold War, Moscow and St. Petersburg embraced her as warmly as Canada had. Yet Marshall remained true to her Canadian roots and to Toronto, her lifelong home. This first-ever biography recounts her dazzling career and paints an intimate portrait of the woman, her childhood encounter with polio, and her complex relationship with her teacher and mentor, Weldon Kilburn. Hers is a tale of a warm, courageous woman; it is also the story of classical music in Canada.
Through his painstaking research, Neufeld skillfully penetrates beneath the surface to find an inner world that is rewarding to the reader. Neufeld makes no attempt to portray Marshall as a great intellect bursting with ground-breaking new ideas.... Yet he captures the richness of a vivid life that was uncommonly rewarding, frustrating, joyous, tragic, and much else.
Literary Review of Canada
James Neufeld, author of Power to Rise: the Story of the National Ballet of Canada (1996) teaches English Literature at Trent University, Peterborough and writes regularly about the arts in Canada. He studied voice at the Royal Conservatory of Music, and has been a lifelong amateur singer.
Read more about James Neufeld
Passion to Dance
Read more about Passion to Dance
Dundurn Performing Arts Library Bundle — Musicians
Stuart Hamilton, Holly Higgins Jonas, Michelle Labrèche-Larouche and 6 others
With Mark Hernandez
Foreword by Carol Burnett
Read more about Dundurn Performing Arts Library Bundle — Musicians
Dundurn Performing Arts Library Bundle — Theatre
Mel Atkey, Charles Foster, Martin Hunter and 3 others
Read more about Dundurn Performing Arts Library Bundle — Theatre


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mendelian
HYPERLIPOPROTEINEMIA, TYPE V
A severe type of hyperlipidemia, sometimes familial, that is characterized by the elevation of both plasma CHYLOMICRONS and TRIGLYCERIDES contained in VERY-LOW-DENSITY LIPOPROTEINS. Type V hyperlipoproteinemia is often associated with DIABETES MELLITUS and is not caused by reduced LIPOPROTEIN LIPASE activity as in HYPERLIPOPROTEINEMIA TYPE I .
Genes related to Hyperlipoproteinemia, Type V
Phenotypes and symptoms related to Hyperlipoproteinemia, Type V
Decreased HDL cholesterol concentration
Decreased LDL cholesterol concentration
Increased VLDL cholesterol concentration
Increased circulating chylomicron concentration
Incidence and onset information
— Not enough data available about incidence and published cases.
Accelerate your rare disease diagnosis with us
Hyperlipoproteinemia, Type V Recommended genes panels
Panel Name, Specifity and genes Tested/covered
CREB3L3.
By Institute for Human Genetics University Clinic Freiburg in Germany.
Comprehensive Cardiology Sequencing Panel with CNV Detection.
By PreventionGenetics PreventionGenetics in United States.
HFE, TTR, GAA, COL3A1, ELN, EYA4, JAG1, DSP, SLC25A4, CRYAB, HADHA, RAF1, FBN1, GCKR, DNAJC19, SCO2, TAZ, COX15, CBS, ALMS1 , (...)
View the complete list with 144 more genes
Panel GTR000553680 complete gene list
HFE, TTR, GAA, COL3A1, ELN, EYA4, JAG1, DSP, SLC25A4, CRYAB, HADHA, RAF1, FBN1, GCKR, DNAJC19, SCO2, TAZ, COX15, CBS, ALMS1, COL5A2, DOLK, BRAF, DMD, GLA, HRAS, KRAS, LDLR, MAP2K1, NRAS, PTPN11, SHOC2, SOS1, MAP2K2, CBL, SMAD4, PRKAR1A, FKTN, SGCG, HSPB8, LMNA, APOE, FKRP, CAV3, TTN, DES, MYH7, LDB3, BAG3, FHL1, SELENON, RYR1, MYBPC3, ACTA1, TMEM43, EMD, TCAP, SGCD, KCNJ2, KCND3, LAMA2, KCNH2, SCN1B, HCN4, SCN5A, CACNA2D1, MYO6, KCNE1, KCNQ1, CACNA1C, LAMP2, NODAL, TGFB2, NOTCH1, SMAD3, TGFBR2, MYLK, FBN2, ACTA2, SLC2A10, MYH11, TRPM4, KCNJ8, ABCC9, SCN2B, SCN3B, KCNE3, CACNB2, GPD1L, PKP2, ANK2, RYR2, CALM1, TRDN, KCNE2, KCNJ5, SNTA1, CASQ2, AKAP9, SCN4B, LTBP2, EFEMP2, KCNA5, PDLIM3, ILK, ACTC1, MYLK2, TGFB3, LAMA4, PRDM16, NKX2-5, ANKRD1, TNNC1, TNNI3, MYH6, PRKAG2, PLN, NEXN, DSG2, JUP, MYPN, RANGRF, TPM1, TNNT2, GATAD1, JPH2, MYOZ2, RBM20, ACTN2, CSRP3, DSC2, MYL2, MYL3, DTNA, VCL, DPP6, SALL4, TBX5, FHL2, ABCG5, ABCG8, TMPO, ZIC3, CRELD1, CETP, LDLRAP1, APOA5, LPL, LMF1, APOC2, GPIHBP1, NPPA, GJA5, APOA4, CREB3L3, CAVIN4, MIB1, CALR3, TBX3, KLF10, ZHX3, TRIM63, SREBF2, ZBTB17
MAJOR HYPERTRIGLYCERIDEMIA.
By GENETAQ Molecular Genetics Centre and Diagnosis of Rare Diseases in Spain.
APOA5, LPL, LMF1, APOC2, GPIHBP1, CREB3L3, LIPI
By Fulgent Genetics Fulgent Genetics in United States.
Cholestasis Panel.
By Blueprint Genetics in Finland.
EPCAM, ABCB11, ABCB4, ATP8B1, TRMU, JAG1, NPHP3, NPHP1, NPHP4, SLC25A13, SMPD1, DGUOK, FAH, MKS1, CFTR, NPC1, NPC2, PEX2, SERPINA1, UGT1A1 , (...)
View the complete list with 26 more genes
EPCAM, ABCB11, ABCB4, ATP8B1, TRMU, JAG1, NPHP3, NPHP1, NPHP4, SLC25A13, SMPD1, DGUOK, FAH, MKS1, CFTR, NPC1, NPC2, PEX2, SERPINA1, UGT1A1, PEX1, TMEM216, CYP7B1, DCDC2, PEX6, TJP2, PEX26, PEX10, PEX12, PEX5, AKR1D1, LMF1, NEUROG3, VPS33B, VIPAS39, BAAT, ABCC2, TTC37, NOTCH2, HSD3B7, CREB3L3, LCT, SLC26A3, NR1H4, MYO5B, SPINT2
Hyperlipidemia Panel.
ALMS1, LDLR, LIPA, APOE, ABCG5, ABCG8, APOA1, PCSK9, LDLRAP1, APOB, APOA5, LPL, LMF1, APOC2, GPIHBP1, ABCA1, APOC3, CREB3L3
Hypertriglyceridemias Panel.
By Knight Diagnostic Laboratories - Molecular Diagnostic Center Oregon Health & Science University in United States.
APOA5, LPL, LMF1, APOC2, GPIHBP1
APOA5.
Dyslipidemia NGS panel (29 genes), Sequence & CNV analysis.
By Laboratory of genome diagnostics Academic Medical Center, University of Amsterdam in Netherlands.
CYP27A1, LDLR, LIPA, MTTP, APOE, ABCG5, ABCG8, CETP, SCARB1, APOA1, PCSK9, LDLRAP1, APOB, APOA5, LPL, LMF1, APOC2, GPIHBP1, ANGPTL3, LCAT , (...)
View the complete list with 9 more genes
CYP27A1, LDLR, LIPA, MTTP, APOE, ABCG5, ABCG8, CETP, SCARB1, APOA1, PCSK9, LDLRAP1, APOB, APOA5, LPL, LMF1, APOC2, GPIHBP1, ANGPTL3, LCAT, ABCA1, APOC3, LIPC, SLCO1B1, CYP7A1, LIPG, STAP1, MYLIP, SAR1B
APOA5. Complete sequencing.
By Instituto de Medicina Genomica Instituto de Medicina Genomica in Spain.
Hyperlipoproteinemia type 4 (sequence analysis of APOA5 gene).
By CGC Genetics in Portugal.
Obesity genetic testing.
MC4R, GNB3, APOA5, ADRB2, INSIG2, FTO
HYPERLIPOPROTEINEMIA, TYPE V.
By Laboratory for Molecular Diagnostics Center for Nephrology and Metabolic Disorders in Germany.
Hypertriglyceridemia.
Chylomicronemia.
APOE, APOA5, LPL, APOC2, GPIHBP1
Type 3 hyperlipoproteinemia.
By Unit for Cardiac and Cardiavasular Genetics Oslo University Hospital HF - Ullevaal in Norway.
Single gene testing APOA5.
By CeGaT GmbH in Germany.
Hyperlipoproteinemia, type 5.
By Asper Biogene Asper Biogene LLC in Estonia.
GPD1, APOA5, LPL, LMF1, APOC2, GPIHBP1
Inherited Cardiovascular Diseases and Sudden Death Panel.
By Health in Code in Spain.
HFE, TTR, AARS2, ACAD9, ACADM, AGL, ACADVL, ATPAF2, GAA, COL1A1, COL1A2, COL3A1, PSEN1, ELN, EYA4, GNPTAB, GLB1, PITX2, JAG1, DSP , (...)
HFE, TTR, AARS2, ACAD9, ACADM, AGL, ACADVL, ATPAF2, GAA, COL1A1, COL1A2, COL3A1, PSEN1, ELN, EYA4, GNPTAB, GLB1, PITX2, JAG1, DSP, DLD, SLC25A4, CRYAB, RAF1, FBN1, MTO1, MRPL3, LIAS, AGK, TMEM70, FAH, MRPS22, TSFM, DNAJC19, GFM1, COQ2, PDHA1, TAZ, PHKA1, COX15, SURF1, GUSB, MLYCD, CBS, COX6B1, SLC22A5, PMM2, ALMS1, COL5A1, COL5A2, DOLK, BRAF, DMD, GLA, HRAS, KRAS, LDLR, MAP2K1, NRAS, PLOD1, PTPN11, SHOC2, SOS1, MAP2K2, CBL, SMAD4, ENG, FKTN, SGCA, SGCB, CAPN3, LMNA, PSEN2, NOTCH3, FKRP, CAV3, TTN, DES, MYH7, FLNC, MYOT, LDB3, BSCL2, BAG3, FHL1, MYBPC3, ACTA1, TMEM43, EMD, TCAP, SGCD, KCNJ2, KCND3, LAMA2, KCNH2, SCN1B, HCN4, SCN5A, FLNA, CACNA2D1, TBX1, CACNA1D, KCNE1, KCNQ1, CACNA1C, LAMP2, SPRED1, TGFB2, NOTCH1, SMAD3, TGFBR1, TGFBR2, SKI, MYLK, FBN2, ACTA2, SLC2A10, MYH11, TRPM4, SLMAP, KCNJ8, ABCC9, SCN2B, SCN3B, KCNE3, CACNB2, GPD1L, PKP2, ANK2, RYR2, CALM1, CALM2, TRDN, KCNE2, KCNJ5, SNTA1, CASQ2, AKAP9, SCN4B, ACVRL1, BMPR1B, GDF2, KCNK3, CAV1, SMAD9, KCNA5, BMPR2, PDLIM3, NEBL, ACTC1, MYLK2, TGFB3, LAMA4, PRDM16, NKX2-5, ANKRD1, TNNC1, TNNI3, MYH6, PRKAG2, PLN, NEXN, DSG2, JUP, MYPN, RANGRF, TPM1, TNNT2, GATAD1, JPH2, MYOZ2, RBM20, ACTN2, CSRP3, DSC2, MYL2, MYL3, DTNA, VCL, PRKG1, TBX5, GJA1, CTF1, FHL2, TXNRD2, TMPO, GATA4, CRELD1, CETP, PCSK9, APOB, APOA5, GATA6, NPPA, GJA5, AGPAT2, ANK3, OBSL1, SCN10A, CTNNA3, KCNE5, ADAMTSL4, APOC3, CAVIN4, TBX20, MIB1, CALR3, LRP6, KLF10, TRIM63, SMAD1, FOXD4, FHOD3
Cardiovascular Diseases_General Panel.
HFE, TTR, AARS2, ACAD9, AGL, ACADVL, ATP5F1E, ATP7A, ATPAF2, B4GALT7, GAA, HNF1A, NEUROD1, PPARG, COL1A1, COL1A2, COL3A1, ELAC2, PSEN1, HNF1B , (...)
HFE, TTR, AARS2, ACAD9, AGL, ACADVL, ATP5F1E, ATP7A, ATPAF2, B4GALT7, GAA, HNF1A, NEUROD1, PPARG, COL1A1, COL1A2, COL3A1, ELAC2, PSEN1, HNF1B, ELN, EYA4, CPT2, WFS1, GNPTAB, GLB1, FOXC1, PITX2, JAG1, NPHP4, PCDH15, DSP, DLD, SLC25A4, GCK, FOXRED1, CRYAB, RAF1, OPA3, FBN1, MTO1, MRPL3, LIAS, GPD1, AGK, DNM1L, TMEM70, FAH, IDH2, CAVIN1, SLC39A13, MRPS22, SLC25A3, PNPLA2, TSFM, DNAJC19, GFM1, MEF2A, COQ2, SCO2, PDHA1, TAZ, PHKA1, COX15, SURF1, GUSB, MLYCD, CBS, PYGM, FXN, COX6B1, SLC22A5, PMM2, ALMS1, ZFHX3, COL5A1, COL5A2, SDHA, DOLK, BRAF, CHD7, CREBBP, DMD, FOXF1, GLA, HRAS, KRAS, LDLR, LEP, LIPA, MAP2K1, NRAS, PLOD1, PTPN11, SHOC2, SOS1, SOS2, RIT1, MAP2K2, CBL, BMPR1A, SMAD4, ENG, ADAMTS2, FKTN, MTTP, SGCA, SGCB, CAPN3, LMNA, PSEN2, APOE, NOTCH3, FKRP, CAV3, NF1, CEL, KCNJ11, INS, PDX1, HNF4A, TTN, DES, MYH7, FLNC, ANO5, MYOT, LDB3, BSCL2, BAG3, FHL1, RYR1, MYBPC3, ACTA1, SYNE1, TMEM43, SYNE2, EMD, TCAP, SGCD, KCNJ2, KCND3, LAMA2, KCNH2, SCN1B, HCN4, SCN5A, FLNA, KMT2D, CACNA2D1, KANSL1, TBX1, CACNA1D, KCNE1, KCNQ1, MED12, FOXP1, EHMT1, CACNA1C, CTNNB1, LZTR1, RASA1, FOXP3, LAMP2, UPF3B, ZDHHC9, SPRED1, RASA2, RRAS, A2ML1, ABCB1, NODAL, FOXH1, TGFB2, NOTCH1, SMAD3, TGFBR1, TGFBR2, SKI, MYLK, FBN2, ACTA2, SLC2A10, MYH11, FKBP14, CHST14, ACVR1, TRPM4, SLMAP, KCNJ8, ABCC9, SCN2B, SCN3B, KCNE3, CACNB2, GPD1L, PKP2, ANK2, RYR2, CALM1, CALM2, TRDN, KCNE2, KCNJ5, SNTA1, CASQ2, AKAP9, SCN4B, ACVRL1, BMPR1B, GDF2, KCNK3, CAV1, SMAD9, EFEMP2, KCNA5, BMPR2, PDLIM3, ILK, NEBL, ACTC1, MYLK2, TGFB3, LAMA4, PRDM16, NKX2-5, ANKRD1, TNNC1, TNNI3, MYH6, PRKAG2, PLN, NEXN, DSG2, JUP, MYPN, RANGRF, TPM1, TNNT2, GATAD1, JPH2, MYOZ2, RBM20, ACTN2, CSRP3, DSC2, MYL2, MYL3, DTNA, AMPD1, TOR1AIP1, SPEG, VCL, ZMPSTE24, PRKG1, MFAP5, NOS1AP, SALL4, TBX5, EP300, GJA1, FHL2, B3GAT3, ABCG5, ABCG8, CYP2D6, OBSCN, TXNRD2, TMPO, ZIC3, GDF1, GATA4, EVC, IER3IP1, NPC1L1, CITED2, LEFTY2, CRELD1, ACVR2B, CETP, SCARB1, APOA1, PCSK9, LDLRAP1, APOB, APOA5, LPL, LMF1, APOC2, GPIHBP1, ANGPTL3, ABCG1, LCAT, ABCA1, SLC2A2, AKT2, RFX6, PAX4, KLF11, BLK, GLIS3, NEUROG3, PTF1A, GATA6, EIF2AK3, PDGFRA, INSR, CFC1, XK, TDGF1, COL7A1, NPPA, GJA5, NOTCH2, TBC1D4, CIDEC, PLIN1, AGPAT2, ZFPM2, ANK3, OBSL1, FGF12, COA5, SCN10A, CTNNA3, KCNE5, ADAMTSL4, GATA5, APOC3, LIPC, PLTP, CHRM2, CAVIN4, SLCO1B1, EIF2AK4, TBX20, MIB1, CALR3, ASPH, CALM3, MYLIP, SAR1B, TFAP2B, SMAD6, TAB2, TNNI3K, CYP3A4, INSIG2, LRP6, PKP4, KLF10, TRIM63, COA6, MED13L, KCND2, MRPL44, NKX2-6, CYP3A5, MCTP2, IRX4, SMAD1, FOXD4, FHOD3, PERP, PPP1R13L, KCNK17, IRX3, SLC22A8, LPA, MYOM1, PPARA, NNT, SLC25A40, HAND2, GREM2, ISL1, TOPBP1, TRIB1, BMP10, CH25H
Dyslipidemias / Early atherosclerosis.
HNF1A, NEUROD1, PPARG, HNF1B, CPT2, WFS1, PCDH15, GCK, GPD1, CAVIN1, PNPLA2, MEF2A, COQ2, PYGM, LDLR, LEP, LIPA, MTTP, LMNA, APOE , (...)
HNF1A, NEUROD1, PPARG, HNF1B, CPT2, WFS1, PCDH15, GCK, GPD1, CAVIN1, PNPLA2, MEF2A, COQ2, PYGM, LDLR, LEP, LIPA, MTTP, LMNA, APOE, CEL, KCNJ11, INS, PDX1, HNF4A, BSCL2, RYR1, FOXP3, ABCB1, CAV1, AMPD1, ZMPSTE24, ABCG5, ABCG8, CYP2D6, IER3IP1, NPC1L1, CETP, SCARB1, APOA1, PCSK9, LDLRAP1, APOB, APOA5, LPL, LMF1, APOC2, GPIHBP1, ANGPTL3, ABCG1, LCAT, ABCA1, SLC2A2, AKT2, RFX6, PAX4, KLF11, BLK, GLIS3, NEUROG3, PTF1A, GATA6, EIF2AK3, INSR, TBC1D4, CIDEC, PLIN1, AGPAT2, APOC3, LIPC, PLTP, SLCO1B1, MYLIP, SAR1B, CYP3A4, INSIG2, LRP6, CYP3A5, SLC22A8, LPA, PPARA, SLC25A40, TRIB1, CH25H
Primary familial hypertriglyceridemias.
PCDH15, GPD1, PNPLA2, APOE, APOA5, LPL, LMF1, APOC2, GPIHBP1, APOC3, SLC25A40, TRIB1
Hypertriglyceridaemia genetic screen.
By Cardiovascular Genetics Laboratory PathWest Laboratory Medicine WA in Australia.
APOE, APOA5, LPL, LMF1, APOC2, GPIHBP1
Major Hypertriglyceridemia: sequencing of APOA5 gene.
Pancreatitis Panel.
CFTR, SPINK1, PRSS1, CTRC, APOA5, APOC2, GPIHBP1, CPA1, UBR1
Familial hypertriglyceridemia.
By Bioarray in Spain.
By Molecular Diagnostic Laboratory for Inherited Cardiovascular Disease Montreal Heart Institute in Canada.
HYPERLIPOPROTEINEMIA TYPE 5.
By Laboratorio de Genetica Clinica SL in Spain.
APOA5, LPL, LMF1, APOC2, GPIHBP1, LIPI
Major Hypertriglyceridemia, Sequencing APOA5 Gene.
By Reference Laboratory Genetics in Spain.
Major Hypertriglyceridemia, Panel Massive Sequencing (NGS) 7 Genes.
APOE, APOA5, LPL, LMF1, APOC2, GPIHBP1, LIPI
LPL Sequencing.
By Cincinnati Children's Hospital Medical Center Laboratory of Genetics and Genomics Cincinnati Children's Hospital Medical Center in United States.
LPL.
LPL. Complete sequencing.
LPL. Detection of the mutation p.Gly188Glu by sequencing.
LPL. MLPA testing.
Lipoprotein Lipase De¬ciency (LPL): mutations Pro207Leu (p.Pro234Leu) and Gly188Glu (p.Gly215Glu).
By Molecular Diagnostics Laboratory Hospital Sainte-Justine in Canada.
Lipase deficiency (sequence analysis of LPL gene).
Lipase deficiency (deletion/duplication analysis of LPL gene).
Hyperlipoproteinemia, type I.
Combined hyperlipidemia, familial.
Familial Lipoprotein Lipase Deficiency via LPL Gene Sequencing with CNV Detection.
Hyperlipoproteinemia type I.
By Centogene AG - the Rare Disease Company in Germany.
Familial Lipoprotein Lipase Deficiency.
By bio.logis Center for Human Genetics Diagnosticum in Germany.
By Division Human Genetics Medical University Innsbruck in Austria.
Lipoprotein lipase deficiency: LPL gene mutation analysis (G188E).
Lipoprotein lipase deficiency: LPL gene regulatory region sequence analysis.
Lipoprotein lipase deficiency: LPL gene sequence analysis.
Major Hypertriglyceridemia: sequencing of LPL gene.
CarrierMap.
By Recombine in United States.
FMR1, MTHFR, MCCC1, MCCC2, ABCB11, ACADM, ACADS, AGL, ACADVL, ACAT1, ALDOB, PC, USH1C, ALPL, ARG1, HLCS, ATP7A, BTD, ATP7B, BCKDHA , (...)
FMR1, MTHFR, MCCC1, MCCC2, ABCB11, ACADM, ACADS, AGL, ACADVL, ACAT1, ALDOB, PC, USH1C, ALPL, ARG1, HLCS, ATP7A, BTD, ATP7B, BCKDHA, BCKDHB, BCS1L, GAA, HBB, MMACHC, TYRP1, STAR, SLC45A2, CHM, TYR, SLC37A4, ABCA12, HSD17B4, CPT2, TRMU, GNPTAB, GLB1, GNE, OCRL, ABCA4, CYP1B1, CEP290, CLRN1, TRIM32, USH2A, PCDH15, DLD, POLG, CYP11B1, SLC25A13, HADHA, OPA3, FH, RPE65, RLBP1, GUCY2D, SMPD1, CYP11B2, SLC26A4, CDH23, HPS1, PDHB, HPS4, HPS3, DHDDS, RS1, FAH, HMGCL, HSD3B2, FAM161A, TAT, G6PC, GAMT, RDH12, RARS2, HAX1, HADHB, CERKL, PCCB, PCCA, GLDC, AMT, LCA5, ALG6, TYMP, MPI, ETHE1, MYO7A, PUS1, OTC, PDHA1, ABCD1, HEXA, ALDH3A2, HEXB, CTSK, ACOX1, PTS, PAH, AGXT, TCIRG1, MPV17, CPT1A, GUSB, GALNS, GNS, HGSNAT, NAGLU, SGSH, NDUFS6, MMAB, MMAA, MUT, DBT, MAN2B1, MLYCD, GALC, IVD, SLC25A15, CBS, CYBA, PFKM, PYGM, GBE1, ETFB, ETFA, ETFDH, GCDH, GALT, GALK1, LRPPRC, SLC3A1, ASS1, CYP27A1, SLC22A5, SLC25A20, PMM2, BBS12, MKS1, BBS10, BBS1, BBS2, ASL, TPP1, CYP17A1, NR2E3, ADA, AGA, ARSB, IL2RG, AIRE, AR, ARSA, ASPA, ELP1, CFTR, FANCC, GBA, BLM, DHCR7, DMD, GJB2, GLA, IDS, IDUA, LIPA, NPC1, NPC2, RMRP, SMN1, ATM, NBN, BRIP1, ADAMTS2, CHRNE, CLN6, CLN8, COL4A3, DOK7, HBA1, LAMB3, FKTN, MTTP, G6PD, HBA2, NEB, CTNS, CYBB, PEX2, NPHS2, PROP1, NPHS1, DCLRE1C, PPT1, PEX7, SGCA, SGCB, SERPINA1, SLC7A7, SACS, SLC26A2, SLC35A3, UGT1A1, TH, GRHPR, LAMA3, MLC1, MCOLN1, MPL, PEX1, POMGNT1, RTEL1, SGCG, SLC12A6, SLC17A5, SUMF1, TTPA, WAS, ABCC8, CAPN3, CLN5, EIF2B5, LAMC2, PHGDH, PKHD1, RAPSN, TGM1, TMEM216, GJB1, NTRK1, FKRP, DYSF, LHCGR, KCNJ11, CYP21A2, COL4A4, SLC12A3, BSND, COL4A5, MTM1, VRK1, EMD, SGCD, TSEN54, MFSD8, VPS13B, VPS13A, ATP6V1B1, MYO15A, SLC4A11, PRPS1, PEX6, LOXHD1, PEX10, CHRNG, RAB23, POR, EXOSC3, CTSC, LYST, DNAI2, DNAI1, ERCC6, TECPR2, MEFV, SRD5A2, F9, F8, FANCA, FANCG, EVC, EVC2, LPL, EIF2AK3, VSX2, RAG2, ERCC8, SLC39A4, COL7A1, CIITA, EDA, TTC37, TFR2, HOGA1, HJV, CYP19A1, AMH, AMHR2, ASNS, MED17, SEPSECS, WRN, XPA, LIFR, SLC7A9, XPC, GDF5, HGD, BCHE, SLC26A3, VPS53
LPL, LMF1, APOC2, GPIHBP1
Major Hypertriglyceridemia, Sequencing LPL Gene.
Lipoprotein Lipase Deficiency , Sequencing Regulatory Area LPL Gene.
Major Hypertriglyceridemia, Deletions-Duplications (MLPA) LPL Gene.
Hyperlipoproteinemia, Type V Is also known as hyperlipidemia, type v, hyperchylomicronemia, late-onset, hyperchylomicronemia with hyperprebetalipoproteinemia, familial, hyperlipemia, mixed, hyperlipemia, combined fat and carbohydrate-induced;hlp type 5; major hyperlipidemia.
If you liked this article maybe you will also find interesting the following in-depth articles about other rare diseases, like NOVELTY SEEKING PERSONALITY TRAIT NEPHROTIC SYNDROME, TYPE 7; NPHS7 PRECOCIOUS PUBERTY, CENTRAL, 1; CPPB1
239 Old Street
EC1V 9EY London, UK
Ethics Study
Mendelian tool does not provide medical advice. It is intended for informational purposes only. It is not a substitute for professional medical advice, diagnosis or treatment. It does not diagnose, it produces a ranked list of suspected genes which provide assistance for rare hereditary disease cases. Patients should discuss their findings with their healthcare provider This service is using Human Phenotype Ontology (Build #1700 - Oct 2017). Find out more at www.human-phenotype-ontology.org.
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Yeah, that's my dog. At my ex's house. Don't ask. He comes barreling out of the house to bark at me as I walk by, and then realizes it's me and wants to know why I'm not walking him. Huh. Well sometimes I like NOT picking up poop.
This is the urban hike I sometimes do. It has a lot of weeds right now.
At least on this section. There's a big hill at one end of it. Good exercise. Takes me about an hour. Then I can come home and cook dinner and get on with things.
I graded for a while and then realized how late it was and that I was supposed to finish the binding on that quilt last night, because I have a union meeting tonight and I'll need to iron, dehair, and pack up both quilts, which is not a small amount of work for one evening. So I stopped grading (I like to finish a whole assignment if I can. I didn't.)…and started sewing binding down.
Assist not needed, Satch. Yeah, I did the whole thing. I finished just before midnight. There's a couple of embroidery stitches I need to do as well, but they won't take more than about 10 minutes. So she's done.


This new laboratory is located in Saudi Arabia, Qassim, Unaizah, King Abdul-Aziz road in front of Al Waha Hotel, above Saadeddin Pastry.
The laboratory will be serving its clients with the highest standards of customer service and will provide a complete range of laboratory testing. The laboratory will be operating Saturdays to Thursdays and the opening hours are 8:00 AM to 9:00 PM but during the Holly month of Ramadan the working hours are 10:00 AM to 6:00 PM, and 9 PM to 12 AM.


text:
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CP Rail designates first woman to head railway's board of directors
Canadian Pacific Railway locomotives are shuffled around a marshalling yard in Calgary on May 16, 2012. , THE CANADIAN PRESS/Jeff McIntosh
CALGARY - Canadian Pacific Railway (CP.TO) has selected the first woman to head the board of directors of the country's second-largest railway.
Isabelle Courville, 56, has been designated to replace Andrew Reardon at the Calgary-based company's annual meeting in May, according to a proxy circular.
An independent director since 2013, Courville is one of four women on CP's nine-member board. The former Hydro-Quebec executive is also currently a member of the boards at SNC-Lavalin and Laurentian Bank.
She received $303,504 in compensation from CP Rail last year - half to be paid in cash, the rest in share-based awards. As chairman, Reardon received $514,344 in total compensation in 2018.
CP Rail CEO Keith Creel received $12.5 million in compensation last year, down from $20.1 million in 2017, the year he was appointed president and CEO.
The decrease was mainly the result of a reduction in option-based awards.
These top Canadian CEOs face higher tax bills on stock options
CN and CP Rail appealing minister's order for use of handbrakes on stopped trains
CP Rail CEO not supportive of Notley's crude-by-rail deal


Australia: Abandoned Formaldehyde Great White #Shark Exhibit In Melbourne.
Posted byMIMIC SHARK SUITS	 February 12, 2019 Leave a comment on Australia: Abandoned Formaldehyde Great White #Shark Exhibit In Melbourne.
Posted byMIMIC SHARK SUITS February 12, 2019 Posted inMIMIC SHARK SUITSTags: MIMIC SHARK SUITS, SharkLeave a comment on Australia: Abandoned Formaldehyde Great White #Shark Exhibit In Melbourne.
California: Baby Swell #Shark Egg Returned To The Ocean.
Posted byMIMIC SHARK SUITS	 February 12, 2019 Leave a comment on California: Baby Swell #Shark Egg Returned To The Ocean.
Posted byMIMIC SHARK SUITS February 12, 2019 Posted inMIMIC SHARK SUITSTags: MIMIC SHARK SUITS, SharkLeave a comment on California: Baby Swell #Shark Egg Returned To The Ocean.
Australia: Mick Fanning Gives Advice To Another Surfer Attacked By A #Shark.
Posted byMIMIC SHARK SUITS	 February 12, 2019 Leave a comment on Australia: Mick Fanning Gives Advice To Another Surfer Attacked By A #Shark.
Posted byMIMIC SHARK SUITS February 12, 2019 Posted inMIMIC SHARK SUITSTags: MIMIC SHARK SUITS, SharkLeave a comment on Australia: Mick Fanning Gives Advice To Another Surfer Attacked By A #Shark.
Cayman Islands: Rare Sighting Of A Whale #Shark.
Posted byMIMIC SHARK SUITS	 February 12, 2019 Leave a comment on Cayman Islands: Rare Sighting Of A Whale #Shark.
Posted byMIMIC SHARK SUITS February 12, 2019 Posted inMIMIC SHARK SUITSTags: MIMIC SHARK SUITS, SharkLeave a comment on Cayman Islands: Rare Sighting Of A Whale #Shark.
Australia: #SharkAttack On A Spear Fisherman Off Umbakumba In The Northern Territories.
Posted byMIMIC SHARK SUITS	 February 11, 2019 February 14, 2019 Leave a comment on Australia: #SharkAttack On A Spear Fisherman Off Umbakumba In The Northern Territories.
Posted byMIMIC SHARK SUITS February 11, 2019 February 14, 2019 Posted inMIMIC SHARK SUITSTags: MIMIC SHARK SUITS, Shark, SharkattackLeave a comment on Australia: #SharkAttack On A Spear Fisherman Off Umbakumba In The Northern Territories.
Reunion: Article In French Asking If The Crisis Is A Man Crisis Or #Shark Crisis? #Requin.
Posted byMIMIC SHARK SUITS	 February 11, 2019 Leave a comment on Reunion: Article In French Asking If The Crisis Is A Man Crisis Or #Shark Crisis? #Requin.
Posted byMIMIC SHARK SUITS February 11, 2019 Posted inMIMIC SHARK SUITSTags: MIMIC SHARK SUITS, Requin, SharkLeave a comment on Reunion: Article In French Asking If The Crisis Is A Man Crisis Or #Shark Crisis? #Requin.
Italy: Article In Italian Of Possibly A Dead Basking #Shark Carcass Found Near Messina. #Squali #Squalo.
Posted byMIMIC SHARK SUITS	 February 10, 2019 Leave a comment on Italy: Article In Italian Of Possibly A Dead Basking #Shark Carcass Found Near Messina. #Squali #Squalo.
Posted byMIMIC SHARK SUITS February 10, 2019 Posted inMIMIC SHARK SUITSTags: MIMIC SHARK SUITS, Shark, Squali, SqualoLeave a comment on Italy: Article In Italian Of Possibly A Dead Basking #Shark Carcass Found Near Messina. #Squali #Squalo.
Florida: https://www.sharkallies.com/ Pushing For Florida Ban On The #Shark Fin Trade.
Posted byMIMIC SHARK SUITS	 February 10, 2019 February 14, 2019 Leave a comment on Florida: https://www.sharkallies.com/ Pushing For Florida Ban On The #Shark Fin Trade.
Posted byMIMIC SHARK SUITS February 10, 2019 February 14, 2019 Posted inMIMIC SHARK SUITSTags: MIMIC SHARK SUITS, SharkLeave a comment on Florida: https://www.sharkallies.com/ Pushing For Florida Ban On The #Shark Fin Trade.
USA: Footage Of A Great White #Shark & A Turtle Taken From A Fishing Charter. Shark Seems Split Between The Two.
Posted byMIMIC SHARK SUITS	 February 9, 2019 Leave a comment on USA: Footage Of A Great White #Shark & A Turtle Taken From A Fishing Charter. Shark Seems Split Between The Two.
Posted byMIMIC SHARK SUITS February 9, 2019 Posted inMIMIC SHARK SUITSTags: MIMIC SHARK SUITS, SharkLeave a comment on USA: Footage Of A Great White #Shark & A Turtle Taken From A Fishing Charter. Shark Seems Split Between The Two.


On Valentine's Day, it's all about showing that special someone in your life that they're, well, special. Here at LiquidPlanner, we'd like to think that our customers are our Valentines, which is why we want to give one of you a knock out Valentine's Day gift this year.
It's no secret that we love our jobs at LiquidPlanner. We have a passionate team, an innovative product, a dog-friendly office, and a snack cart that just won't quit. Now we want to know why YOU love YOUR job.
Maybe you have a great boss, or your company has a dedicated Pinball Machine Room (invite us over!), or your office is a hammock on a beach (again – invite us over!). Maybe you love that your team does something good in the community or for its employees. We want to hear about it!
Just leave us a comment on this blog post telling us why you have the best job around, and you could win 10 LiquidPlanner licenses for your team for one year. The answer that gets our hearts racing the most will win, so get creative. This prize can go to a new or existing customer, and it's just the type of gift that might make your boss love you (even more).
This could be the start of a life-long relationship with your favorite online project management software (and could make you think about February 14th in a whole new way).
*Terms and conditions apply. Contact sales@liquidplanner.com for details.


text:
Roelof Jansz. van Vries (Haarlem, ca, 1630 - Amsterdam, na 1681) was een Nederlands kunstschilder uit de periode van de Gouden Eeuw. Hij specialiseerde zich in het vervaardigen van landschappen, in veel gevallen voorzien van architectuur.

De geboortedatum van Van Vries wordt afgeleid uit het gegeven dat hij op 11 oktober 1659 in ondertrouw ging met de Haarlemse Marytje Adriaens; hij was toen 28 jaar oud. Zijn getuige bij deze gebeurtenis was Reinier Hals, een zoon van Frans Hals. Zijn eerste gedateerde werk stamt uit 1651, het laatste uit 1681. Schilder en schrijver Vincent van der Vinne vermeldt hem in een aantekening van het Sint-Lucasgilde in 1702 als overleden.

Rond 1653 was Van Vries actief in Leiden en vanaf 1659 in Amsterdam. Onbekend is wie zijn leermeester was. Hij was in zijn stijl een navolger van de vermaarde landschapsschilder Jacob van Ruisdael. Ook liet hij zich inspireren door het werk van onder anderen Meindert Hobbema, Jan Wijnants en Philips Wouwerman.

Externe links

Afbeeldingen in de Wikigallery
Vermelding in het Biographisch Woordenboek der Nederlanden als Jan Renier de Vries
Landschap met valkenier in het Rijksmuseum Amsterdam
Landschap met bleekveld, Bespreking van het werk van Van Vries

Nederlands kunstschilder
Persoon in de Nederlanden in de 17e eeuw

redpajama_set_name: RedPajamaWikipedia


Still the Enemy Within is an award winning film that tells the story of the great miners strike. Thatcher called the 160,000 miners, their families and community 'The Enemy Within' and went to war with their union, the NUM, for a year from 1984 to 1985.
Join People's Assembly Nottingham for a beer before hand in the cafe before we go to watch the film together. We'll meet up afterwards to talk about the film.
The film has proven so popular the cinema have moved it to the biggest screen! Look forward to seeing you all tomorrow!


BO-PEEP was conceived, written and partially recorded during an extended weekend of solitary recluse at the Waikiki of Denmark. One man. One guitar. One kick drum. One microphone. Two bottles of absinthe. Once back in the big, bad, Jutlandian metropolis of Aarhus Kix added more guitars, drums, organs and his old secret weapon: The tripple bass. He even met up with his Railthin Brother Bo who laid down a few dope beats on a couple of songs for Christ and old times' sake. Mixing was achieved through the divine intervention of Mr. Nik Heyman, and a final layer of dirt was shoveled into the woodchipper by Emil Thomsen at ET Mastering. 17 songs in total. The ones too strong for unlimited release were chosen to comprise the 7-inch 45rpm wonder that is PEEK-A-BO. Released in 500 copies on Record Store Day 2018.
2018 is the year of the SOLO. The Atomic Child that so many many years rode alongside Brother Kix on a horse called Mule and defeated audiences across the Globus with his mighty axe decided to take a load off and lay his barking dawgs to rest by the fire place at Casa Railthin. Enjoying his PowerOtium looking at all the platinum records, Grannies and other prestigious awards and sparkling trophies from two decades of confrontational badassery on stage and record both, Brother Beau deserves nothing but our utmost respect and praise for his lifelong dedication to rock'n'roll and beyond.
But The PowerSolo forges on. A juggernaut through the big, black narcotic night, Kim Kix and his merry band is once again setting sail towards the clubs, festivals, dives, concert halls, your mother's house, bars, ball rooms and other entertainment establishments. And this year they're armed with kick-face material taken from the new PowerSolo unlimited edition long player album BO-PEEP and it's Record Store Day un-unlimited edition 4 song companion-EP PEEK-A-BO. Both releases will be available on all futuristic megapixel bitcoin platforms and the LP will even come disguised as a CD in countries where such a thing is still a thing.
So much for the sound. The beautiful artwork was done by Martin Wild of The Courettes - a fine band that Kim Kix has produced 2 records for. The colours are bold and the designs even bolder. The final result defies description. But we'll try: This is a fine record. Damn fine. It's what the doctor ordered. It's what your Father warned you against. It is highly toxic but strangely addictive. It is BO-PEEP. And PEEK-A-BO. A double whammy like you've never heard before. 17 inches of pure indiluted PowerSolo. Can you dig it?


text:
Long-Acting Therapy for Malaria Prevention: Toward a Malaria-Free World
Resources TAGline TAGline October 2021 Long-Acting Therapy for Malaria Prevention: Toward a Malaria-Free World
By Joelle Dountio Ofimboudem
Malaria is a life-threatening disease prevalent in the world's poorest countries, particularly in sub-Saharan Africa. Preventing malaria hits close to home for me. I am a mother to two children, aged three and five. We are originally from Cameroon, where we all have contracted malaria multiple times. As an intellectual property lawyer and global public health advocate, I know that geography — the fact that we live in the "neglected" part of the world in terms of research and development (R&D) priority — is the main reason for the constant battle with malaria. It is appalling to know that the multiple trips to the hospital and hospitalizations resulting in my kids missing school or not playing with their friends can be avoided.
Today, 14 of the 15 countries — which account for 80% of the malaria burden — are in sub-Saharan Africa. Yet, elimination efforts do not focus on these countries.1 While malaria remains a leading cause of death in sub-Saharan Africa, the disease was eradicated in the United States and Europe in 1951 and 1975 respectively.2 This is a stark reminder of the inequities in global health.
Malaria is a common comorbidity for people living with HIV. HIV infection can increase the risk and the severity of malaria, and the prevalence of symptomatic malaria is high among people who are coinfected with HIV.3 Moreover, malaria increases HIV plasma viral load and decreases CD4+ T cells,4 and both diseases cause complex activation of immune cells and disregulated production of cytokines and antibodies.5 Coinfection can also accentuate the progression of anemia.
As we explore long-acting technologies (LATs) for preventing malaria and HIV, many of the communities with whom we work are dramatically impacted by both and face similar access and linkage to care barriers (see Johnson). As a partner on the LONGEVITY project, which aims to develop LATs for preventing malaria and tuberculosis and curing hepatitis C, Treatment Action Group (TAG) applies our technical and community engagement expertise to follow and translate the science of LATs to benefit patients, affected communities, providers, researchers, and other stakeholders across these diseases.
Current Malaria Prevention Measures and Challenges
Malaria persists because current prevention measures do not go far enough, hence, the need for more comprehensive approaches in the battle against malaria. Currently, prevention revolves around three main interventions: (1) sleeping under insecticide-treated nets; (2) spraying insecticides indoors; and (3) providing malaria prevention medications to people traveling to malaria endemic countries, pregnant individuals, and infants living in high-transmission areas.7 Yet, each of these approaches presents challenges. People do not always use insecticide-treated nets because they increase heat,8 reduce airflow and cause breathing difficulties.9 The chemicals on these nets sometimes cause skin reactions; and the nets can be inconvenient for daily set-up in homes that lack space.10 Moreover, these nets are not typically replaced or re-treated in time to provide continuous protection against mosquito bites. With respect to indoor spraying, Pyrethroid — a chemical contained both in insecticide spray and insecticide-treated nets that kill mosquitoes— although affordable and having a long residual action, has developed resistance across most of malaria endemic sub-Saharan Africa due to increased use.11 The high cost of alternative insecticide sprays and their low community acceptance have also contributed to minimal spraying of insecticides indoors across sub-Saharan Africa.12 Lastly, insufficient capacity and funding for malaria prevention constrains countries' ability to set their own agendas for reducing the disease burden.13
Current Malaria Treatment and Challenges
While there are several approved Artemisinin-based Combination Therapies for malaria treatment which are recommended by the World Health Organization,14 malaria treatment also faces many challenges Historically, due to high malaria prevalence in endemic countries such as Tanzania, health providers treated people with suspected cases of the disease with relatively cheap antimalarial medications, a practice known as presumptive treatment.15
In Cameroon, due to insufficient health staff, people stock and self-administer anti-malarial medications at the onset of any fever, especially in remote areas. The failure of authorities to effectively regulate medicines imports and quality floods the market with unverified/low quality antimalarials. Ineffective regulation provides patients with antimalarials through street vendors and even at pharmacies without prescriptions. There are also high costs: transportation, hospital consultation, and laboratory exams which, combined, cost more than the full treatment course for malaria from street vendors.
Due to these factors, and the frequency of symptoms, adults and parents self-administering malaria medication when they, or their children, have a fever is a common practice, despite healthcare guidelines and providers' dissuasion. They often use unverified, low quality antimalarials that fuel drug resistance, or seek traditional herbal treatments at the onset of symptoms, and present too late at healthcare facilities with severe illness.
The LONGEVITY Project and Malaria Elimination
A long-acting malaria preventive — if proven safe, effective, acceptable, and accessible — could address many of the shortcomings of the current approaches to both treatment and prevention. By offering an easier way to protect people living in endemic regions against malaria, a long-acting formulation for malaria prevention would indirectly address drug resistance by sparing people from taking medications and eventually eliminating the market for unverified antimalarials.
Given the shortage of healthcare staff in most sub-Saharan African countries, a long-acting preventive for malaria would significantly reduce the workload on health providers and enable them to focus on other health problems — hence, enhancing public health overall. A long-acting malaria prevention therapy would also drastically reduce adult and infant mortality. More children could live more fulfilling and healthy lives, and parents like myself and their children would no longer have the burden of frequent visits to the hospital and hospitalizations. Apart from the health benefits, malaria elimination would reduce economic loss in the form of disability-adjusted life years in endemic countries.16
A long-acting formulation will hopefully eliminate the need for insecticide-treated nets, which cause skin reactions and exacerbate the already warm, tropical climates where malaria is endemic. People could sleep better, enhancing their overall well-being.
Malaria endemic countries usually have very fragile healthcare systems. Malaria prevention could help avert complications that arise with the onset of malaria, including anemia, miscarriage, liver failure, kidney failure, premature and stillbirth, and cerebral malaria. Given the high out-of-pocket cost, and the limited number of specialists available to provide care for these complications, especially in rural areas, prevention would be a tremendous public health win for malaria endemic countries.
The LONGEVITY Project – Redefining Pharmaceutical Research and Development
The pharmaceutical industry's status quo has failed to develop new antimalarials and antibiotics because these medications have a lower return on investment than treatments for conditions that turn higher profits in high-income countries, such as cancer.17 The access-to-medicines movement has called for "push," "pull," and "pool" mechanisms to incentivize pharmaceutical developers and donors to invest in R&D for neglected diseases, such as malaria. The LONGEVITY Project encapsulates both the "push" (i.e., incentivizes industry by reducing R&D costs and absorbs upfront risks) and "pull" (i.e., creates incentives for private sector engagement by creating viable market demand) mechanisms. The project invests in R&D for long-acting injectables of existing treatments; in the case of malaria, it investigates atovaquone. The LONGEVITY Project also involves community engagement and advocacy to ensure accessibility and acceptability of long-acting formulations and prepare stakeholders for eventual introduction.
The high prevalence of malaria in LMICs over 50 years after its eradication in high-income countries is a glaring example of global health inequity. Malaria eradication in low- and middle-income countries would ensure a "malaria-free" world for all and enhance global health equity. Long-acting technologies offer a critical opportunity for advancing that goal.
1 Ranson H. Malaria: key challenges and potential solutions. High-Quality Technical Assistance for Results [Internet]. 2018 Apr 20 [cited 2021 Sept 6]; [about 11 a.].
https://www.heart-resources.org/reading_pack/malaria-key-challenges-potential-solutions/
[2] Thomson Reuters Foundation. Timeline: Seven decades of efforts to eradicate malaria. Reuters [Internet]. 2016 Oct 31 [cited 2021 July 20]. https://www.reuters.com/article/us-health-malaria-timeline/timeline-seven-decades-of-efforts-to-eradicate-malaria-idUSKBN12W2TS.
3 Hochman S, Kim K. The impact of HIV and malaria coinfection: what is known and suggested venues for further study. Interdiscip Perspect Infect. 2009 Aug 9:617954. doi: 10.1155/2009/617954.
4 Alemu A, Shiferaw Y, Addis Z, Mathewos B, Birhan, W. Effect of malaria on HIV/AIDS transmission and progression. Parasites Vectors. 2013 Jan 17;6:18. doi: 10.1186/1756-3305-6-18
5 Ibid.
6 World Health Organization. Guidelines for Malaria. Geneva (CH): World Health Organization. 2021 July 13. pp. 23-70. https://www.who.int/publications/i/item/guidelines-for-malaria.
7 Ahorlu CS, Adongo P, Koenker H, et al. Understanding the gap between access and use: a qualitative study on barriers and facilitators to insecticide-treated net use in Ghana. Malar J. 2019 Dec 12;18(1):417. doi: 10.1186/s12936-019-3051-0.
8 Ngwibete A, Ozoms J. Attitudes toward utilization of insecticide-treated bed nets among pregnant women and care-takers of under-five. InfectionControl.tips [Internet]. 2016 Aug 14 [cited 2021 July 20]. https://infectioncontrol.tips/2016/08/14/insecticide-treated-bed-nets-731/
11 Oxborough RM. Trends in US President's Malaria Initiative-funded indoor residual spray coverage and insecticide choice in sub-Saharan Africa (2008-2015): urgent need for affordable long-lasting insecticides. Malaria Journal. [Internet]. 2016 March 8 [Cited 2021 July 20]; 15:146. Available from https://malariajournal.biomedcentral.com/articles/10.1186/s12936-016-1201-1.
12 Namuganga JF, Epstein A, Nankabirwal JI, et al. The impact of stopping and starting indoor residual spraying on malaria burden in Uganda. Nat Commun. 2021 May 11;12:2635. doi: 10.1038/s41467-021-22896-5.
13 Ranson H. Malaria: key challenges and potential solutions.
14 World Health Organization. Guidelines for Malaria. pp. 65-72.
15 Reyburn H, Mbatia R, Drakeley C, et al. Overdiagnosis of malaria in patients with severe febrile illness in Tanzania: a prospective study. BMJ. 2004 Nov 12;329(7476):1212. doi: 10.1136/bmj.38251.658229.55. Phillips V, Njau J, Li S, Kachur P. Simulations show diagnostic testing for malaria in young African children can be cost-saving or cost-effective. Health Aff (Millwood). 2015;34(7):1196–203. doi: 10.1377/hlthaff.2015.0095.
16 World Health Organization Regional Office for Africa. A Heavy Burden: The indirect cost of illness in Africa. Republic of Congo: World Health Organization. 2019. https://www.afro.who.int/publications/heavy-burden-productivity-cost-illness-africa
17 Plackett B. No money for new drugs. Nature Antimicrobial Resistance Outlook [Internet]. 2020 Oct 22 [cited 2021 July 20]; 586. https://media.nature.com/original/magazine-assets/d41586-020-02884-3/d41586-020-02884-3.pdf.


Add your favourite topping for Lunch…Snacking…Cheese Boards….even good enough to eat on their own.
Wheat Flour (Contains Vitamins; Thiamine & Folic Acid), Unsalted Butter, Non Fat Milk Solids, Poppy Seeds, Sesame Seeds, Sugar, Salt.
Contains Gluten, Milk and Milk Products, Sesame Seeds. May contain traces of Soy. No Added Yeast. Made In Australia .


Vibrant steel-blue globes of summer color. Silvery, strong foliage is spiny with downy undersides. Very colorful and drought tolerant. When Echinops Blue Glow is combined with Achillea or daylilies it will provide an interesting contrast of shape and color.
Echinops are not thistles despite their common name, Globe Thistle. Provides carefree late summer color. Does well in any soil. Provides good color and contrast in the perennial border, especially toward the back, or in the cut flower garden.
Deadhead initial blooms and several smaller shorter blooms will appear. Once done blooming, cut all flower stalks to the basal foliage and leave to overwinter. Clean up in spring. These plants are slow to recover after being moved or divided. Can be divided in early spring by carefully splitting a few offsets from the base of an established clump.


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Culturebox
Houellebecq: A Boocq
Feb 04, 199911:55 AM
Michel Houellebecq is a 40-year-old computer programmer who emerged from a French mental hospital a decade ago, started publishing poems, and branched out. His second novel, Les particules elementaires, concerns two desperately lonely half-brothers. One is a sex maniac, the other a genetic researcher who seeks through cloning to make sex obsolete. The two have more in common than it sounds–which is Houellebecq's point, sort of. The book has sold several hundred thousand copies and launched the first real French literary spat in years.
Particules has already got two write-ups you might have seen: a New Yorker piece on the affaire by Adam Gopnik and a driblet in the TLS. Both seek the source of the book's controversy in its politics. Houellebecq was kicked out of his Paris literary coterie, Les Perpendiculaires, on those grounds. But if his politics are right-wing, it's not in the French anti-dreyfusard-poujadist-lepenist line. No, these are '90s issues. Houellebecq rails at materialism–both consumerist and sexual. He especially damns the places where the two overlap, like drugs, abortion, and penile and breast implants. But the reader soon notices that these diatribes are being delivered in the course of lengthy gang-bang descriptions–which are lovingly, even excitedly rendered. A reviewer who, like this one, is shaky on his French idioms, will learn all sorts of words, like se branler for "whack off," partouze for "orgy," and branlette espagnole for … Sorry, that's where I draw the line. But finish this book and you'll have no trouble reading the menu in any French whorehouse.
How does Houellebecq reconcile his paeans to random sex with his lament over the world it leaves in its wake? By viewing both as stages on the way to a eugenicist paradise. Houellebecq even reads Huxley's Brave New World as a u-, not dys-, topia. (And defends his point with considerable documentation and brilliance on pp. 193-201.) Here's a sum-up of Houellebecq's thinking on the matter:
(1) without God and/or a belief in progress, life's a torture;
(2) still, sexual liberation's worth trying as a consolation;
(3) but it doesn't ultimately satisfy, so forget about it. Let's clone a new
race and get the hell out of here.
The critic Frederic Badre calls Particules "the first 21st-century novel" and Houellebecq "the first novelist who is not a humanist." One hopes (and suspects) that Houellebecq is describing not la condition humaine but la condition francaise.
(To order Particules, or indeed any French book, go to www.lemonde.fr/livres, www.fnac.fr, or www.alapage.com.)


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Q: How to clear a hashmap in Android Below is the reference code
public static Map<String, Long > map1 = new HashMap<String, Long >();

if i call map1.clear() , will all elements of hashmap become eligible for GC or do i need to set every element of the map explicitly to null ?

A: hashmap.clear() just clears all the data of the HashMap to make it reusable again.Here reusable means the same object can be reused again.So I dont think there is any role of GC here.