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Interview with School Damage
My friend Farah and I sat down with School Damage for an interview recently. We talk about a lot of weird shit... check it out.
You guys tour with so many great punk bands made up of great dudes. Who are some of your favourites to play with so far?
Curtis: BOIDS!
Brad: BOIDS are good!
Jon: BOIDS are super fun.
Brad: Yeah we like going on runs with dudes we get along with.. we play with BOIDS a bunch. They're definitely on like a different skill level than we are but they're just solid dudes all around. Or like The Raging Nathans who we've toured with twice now who are just the funniest people on the planet.
Curtis: Oh yeah fuck we can't forget about our buds from Ottawa, NECK.
Brad: Oh my god NECK... we've played a lot of shows with those guys because we love those dudes.
Jon: We'll play a city with a band for the first time like we played Fort Wayne with Flamingo Nosebleed and became good friends with them so we brought them up and went on tour with them. And like Dougie Tangent and The Putz.
Brad: Yeah, The Putz!
Has there ever been a band who you thought you'd get along with on tour but ended up hating them by the end of it?
Brad: No. Not at all.
Curtis: It's been pretty smooth.
Brad: There's definitely been people who you're stoked to bring on the road but they're just different than what you expected, like they're like 'we have kids and a wife, we're just going to go to bed' and we'll be like 'whoa I just barfed'.. that kind of stuff. Or we're like 'come on smoke this!' and they're like 'no we can't'. But honestly we've never met anyone who has been shitty... except for White Lung, they were shitty.
Jon: Yeah but they were on the ass end of a long tour and they had a shitty show. They played Duffy's to a packed house but noone cared.
Brad: That's probably the only show that anyone was just like rude to us and they were probably just rude because they didn't want to be there.
Jon: We didn't want to be there!
Brad: Yeah after our set I went home and watched Saturday Night Live. That's a fact!
Curtis: Probably had more fun watching that.
Where's the creepiest place you've found love with yourself while on tour?
Battered Lives by School Damage Brad: One time I masturbated in Sherbrooke, Quebec in a toilet in this venue after a man took a shit in it. I hadn't wacked on in like 3 weeks and it was just like a wash of a show so I went in there...
Curtis: To relieve some tension..
Brad: They had free Wi-Fi! I brought my laptop into the stall and masturbated for the first time in 3 weeks!
Curtis: I think on our East Infection tour I jerked off in the van... I slept in the van that night and jerked off in a condom. And these guys think it's soooo funny like 'oh he jerked off in a condom' but at least I didn't get cum all over the seats!
Brad: Well it was only weird because we had a garbage can in the van which was facing the backseat where I sit all the time and I saw the wrapper so I was like 'who had sex in here last night' and you were eating a banana and were like 'no one, I jerked off into a condom' like it was no big deal!
Jon: All of mine are romantic instances! On the beach, at a campsite... I like to romance myself!
You guys gave me a ride back from Pouzza Fest and I kind of got the sense of what it was like on the road with School Damage which was all about sexually harassing Curtis...
Curtis: Not all of the time, but most of the time. It's all about Brad wanting to get me hard. He's all like 'what do I have to do to man get you hard' and I'm like why do you want to do this you fucking creep!
And that was like an end of a long weekend shitty drive, like a 6 hour drive..
Brad: 6 hours of trying to get Curt hard.
Curtis: It was maybe like 3 and a half hours..
Curtis did you get hard?
Curtis: No! Not at all.
Brad: And I was pulling out all the stops and everything man!
So is it always non-stop jokes like that with you guys on the road?
Brad: Here's what it's like. So day 1 is like...
Jon: Curt falls asleep 30 minutes into the drive..
Curtis: Nooo that was just our first tour!
Brad: You always fall asleep! Anyway, it usually goes by just listening to records and then next day or so it's just like we'll get into more obscure stuff in terms of music and then usually on the way home or like the way from somewhere we play that Would You Rather game, or we talk about really funny sex stuff or we pretty much just rip on each other the entire time.
Jon: Typically if we tour the states after we cross the border we get super excited and then it just gets pretty ridiculous from there. But it's always just us talking about weird shit.
Brad: The walls of that van have heard some things! But yeah we usually we just fuck with each other.
Curtis: And we get along with each other for the most part!
Jon: You have to find humour in everything when you're on the road crammed in the van for like 6-8 hour drives or else you'll just go insane.
Brad: There was this one time when we were coming back from Detroit and we stopped at one of those ONroute's and Curt was taking a poo and I just kicked open the door and he's in there pooing. I don't even know why I did it... just that kind of stuff.
That leads me to my next question, what's the most self-deprecating thing you guys have ever done as a band?
Jon: Probably wearing short shorts on stage at Pouzza! I had a crop top set up too but I didn't have the confidence to pull it off.
Brad: I was a little cowboy! That's actually kind of dope, it's not self-deprecating at all. Um, we do stuff, like I do stuff all the time to make them laugh that's self-deprecating all the time but it's never like in the public eye.
Jon: Like trying to get your buddy to get hard?
Brad: Yeah, or I'll like look at them and then pull out my dick and try to piss in my mouth..
Curtis: Oh yeah like in Halifax.
Brad: I did it in Detroit too actually...
Jon: That's more like self abusive..
Brad: No it's just funny.
Curtis: We were in Montreal and I was looking after the van for the night and I had to take a shit in the morning and everything was closed so I took a shit in an alley and wiped my butt with some leaves.
Jon: I think the most self-deprecating thing we do as a band is write songs about self-deprecation.
At your last Bovine show there was a dude who was dressed up as a clown handing out balloon hats.. what other weird shit like that has happened at your shows?
Brad: On that first real tour we had Jon got flashed like all the time.
Curtis: Yeah he was working merch for, well they thought he was working merch for Dayglo Abortions.
Jon: We were playing Barrie and some random Barrie skid just came up and flashed me her tits and said 'does this give me anything?' and I was like I think you're looking for that guy over there and she just like pivet towards him and does the same thing and he like takes her under his arm and is like 'come with me'... and then we were playing Oshawa and while we were playing someone flashed us again! It was the tour that I saw tits.
Brad: I didn't see any tits that tour.
Jon: Oh, and there was Minneapolis!
Brad: Ok, no, let's move on.. nothing but BO..
Jon: We went to Minneapolis and Brad was super stoked that we were where The Replacements were and he just got blasted like drank whiskey all day in the van..
Brad: We woke up in Madison and smoked hash and drank before we had to leave and then we left and kept drinking all the way to Minnesota and then turns out when you drink whiskey all day and smoke hash you get really drunk and we loaded into this club and I was wearing sunglasses all night and felt that was the only thing protecting me from looking like I was blackout drunk and I did some karaoke and shit at a legion before out set... I don't remember our set at all.
Curtis: He was on auto-pilot.
Jon: He was in the golden ballroom.
Brad: I was in the golden ballroom where I thought I was doing great but I wasn't doing so good. And then some guy put his finger in my mouth to make me puke.
Jon: The drummer for this first band who is this greasy fucking dude.. Brad was trying to puke outside to make himself feel better and he goes outside and offers him his help by sticking 3 fingers down his throat like to the top knuckles.
Brad: He was trying to help me.. Anywayssss.
OK we should probably talk about your album...
Brad: Yes, let's please move on.. my girlfriend is sitting beside me dude... I wonder if she's going to respect me still..
Have you sold any of the new album in Europe yet?
Brad: We just got the numbers back and yes we did sell some! It was like, 10 copies!
Do you have plans to do a Europe tour?
Brad: Next July sista!
Jon: Yeah we're working on it for next July!
Brad: We'll be there in no time.
What's the writing / recording process like with you guys?
Jon: Typically Brad or I will come up with ideas ready for practice, either verse or chorus or something then we'll flush it out as a band, work out all the kinks... and then put lyrics to it later or we'll have lyrics for it then. And for recording we try to do it all ourselves. We're just in the middle of tracking demos for a new album. We're about 10 song down, working on 5 more.. Trying to keep up with our one release a year.
Brad: When you think about it, we finished the last album and then it took a long time for it to come out..
Jon: It took about 6 months to get anyone interested in it, and then like 3 months from then it came out.
Brad: So it will be the same with this one, we're doing the demos for the new records now and it will be done in probably November, out like next November..
[Farah] Has anyone ever heard those songs yet, have you played them live?
Jon: We had one of them in our set a few times.. and we played one of them at a party. We might start peppering them into our set..
Are the new songs along the same lines as those on Battered Lives?
Brad: It's definitely a bit different, I had to solo a lot more than I used to have to solo. I dunno I'm cool with these songs man, I'm more psyched about these new stuff than some songs off Battered Lives, no offense..
Jon: But we're still super proud of that album!
Brad: We're trying to drop the whole like 1, 2, 3 kind of stuff..
Jon: Yeah, step away a little bit from the Ramones-core.
Brad: Yeah just do our own thing.
It must be weird playing songs that you wrote so long ago and are kind of over already but you have to play since the album just came out..
Brad: Yep.
Jon: We've had some of those songs for like 2-3 years... we don't play a lot of them live.
Brad: That's true there's a lot of songs on that album we never play live, and we don't even rehearse them... so if you like them on the album, don't look forward to seeing us play them!
You guys have a video for "You'll Shoot Your Eyes Out"..
Do you plan on making a video for any other Battered Lives tracks?
Brad: Yeah, I've been seriously considering doing another video.. it's just so time consuming and annoying. The last one we did was kind of a pain in the ass because I had to set it all up myself, I had to book a venue and get people to show up early and had to promise everyone beer and everyone got pissed because there wasn't enough free beer.
Jon: And we had to lose a tire..
Brad: That was pure coincidence! We actually built that video around the fact we lost a tire. But yeah we'll do one probably soon, most likely fall... but don't expect any free beer!
Catch School Damage opening up for Chixdiggit! at the Bovine on September 23rd orrrrr at Beau's Oktoberfest on the Black Forest stage on September 24th.
http://www.schooldamage.com/
Posted by I do what I want!
FREE MUSIC! MSB COMP #6
No Sex
Fifth Time's A Charm
Songs For The Shitter
Numero Threeve X$X
A(nother) Com-PUNK-ilation
A Com-PUNK-ilation
Julian interviews KJ Jansen from Chixdiggit!


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'Crash Bandicoot N.Sane Trilogy' Releases This June
Relive the adventures of Sony's original mascot on the PS4 this June.
Long before console developers battled over hardware specs and the potential for 1080p and 4K-quality gaming, the giants of the industry waged war using self-proclaimed mascots. Though many have fallen off the map over the past few decades, the likes of Mario and Sonic still reign supreme, even if one of their parent companies has since pulled out of the console wars. While Nintendo and Sega were butting heads through Mario and Sonic and the power of "blast processing", Sony bombastically entered the fray with the PlayStation and all-but-forgotten marsupial, Crash Bandicoot. Crash quickly became a gaming icon after a trilogy of critically-acclaimed PS1 titles, an entertaining battle-racing game, and a bombastic marketing campaign that went so far as to attack Nintendo in the most direct of ways:
Original Crash Bandicoot developer Naughty Dog has since gone on to focus on the Uncharted franchise, but that's not to say Sony's mascot has been entirely forgotten; the orange marsupial appears as a playable Easter Egg in Uncharted 4 and a Sony-exclusive character in Skylanders: Imaginators. Developer and publisher Activision announced at PSX 2016 that Crash Bandicoot would finally return to the gaming world in 2017 with a remastered compilation of the original PS1 trilogy.
Nearly seven years after his last outing- Crash Bandicoot Nitro Kart 2 for iOS- the titular character finally returns to consoles with Crash Bandicoot N.Sane Trilogy. The title features remastered versions of Crash Bandicoot, Crash Bandicoot 2: Cortex Strikes Back and Crash Bandicoot 3: Warped, complete with new music, completely remodeled stages and characters, and updated gameplay mechanics. Activision released a new gameplay trailer last week; despite the lack of new footage, the trailer finally gives the N.Sane Trilogy an official release date set for the end of June.
Crash Bandicoot N.Sane Trilogy will release exclusively for the PS4 on 6/30.
Source: Activision
Image courtesy of Activision, Sony
Categories: News, Sony, Trailers
Tagged as: Activision, Crash Bandicoot, gameplay, N Sane Trilogy, naughty dog, ps4, Release Date, remake, remaster, sony, trailer
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redpajama_set_name: RedPajamaCommonCrawl


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What Do Art Dealers Do
Ana Bambic
Ana Bambic Kostov specializes in contemporary art writing, editing, content creation and management. Connect via LinkedIn https://www.linkedin.com/in/anabambickostov/.
An art collector seeking ways to purchase new pieces or browsing the art market for novel ways of discovery is constantly in need of information and, therefore, advice. Coming across any of the collectors' tips, one seems to be getting an advice to find a dealer continuously. But what do art dealers do, that can be of such tremendous use to a collector? Don's they just sell art? Well, the truth is more complex than that, as we've tried to answer in this article.
Barbara Gladstone and Klaus Biesenbach
Art Dealer Basics
An art dealer is not always a gallerist. He/she might become a gallery owner later in their career, but this is not where they start. An art dealer is usually an educated professional, a specialist in the field of art business. Renowned dealer representatives possess significant experience in the art market and also a rich background in the industry. They can come from the world of business or art, and it is not uncommon that they were artists or curators themselves, and maybe still are. Most of the dealers specialize in one particular style or period of art. As professionals with the precious know-how of the artistic topics, dealers are happy to share what they know with their clients, both prospective and established, providing them with advice and opinions. They serve as consultants during exhibition, auction or studio visits and their judgement is taken very seriously with every new purchase, especially if a collector is interested in an emerging name. A dealer can also give legal advice in all matters art, and advice on framing, storage, conservation, cataloguing or display. Other than being closely involved in building a collection for a client, dealer can have a number of independent activities, which provide a firm foundation for the ever-important reputation of his business.
David Zwirner and Jeff Koons
Building the Art Scene
Art dealers have a strong feeling of responsibility towards building the current art scene and, consequently, art market trends. Exhibitions are one of the most important traits of art dealers' educational activities. Through them, dealers try to promote and support emerging artists, and to establish relationships between creatives in terms of style and cultural background. Placing art they promote in an art historical context is crucial for a serious dealer, in order to establish a long-term influence and relevance. When a well-known gallery has an exhibition, especially of an emerging artist, but of others as well, a collector should always attend. While the vernissage might be a good place to network, another visit to the exhibition space with an art dealer – consultant is important for the art lover to gain a deeper insight in what is up and coming in the art world, and act accordingly. The trick is that most of the well publicized exhibitions sell at the opening, or even before. Having a dealer at your side is, therefore, often crucial.
Feature image: David Hockney and entering, William Acquavella
Left: Iwan Wirth / Right: Matthew Marks
Reputation is the Cornerstone
Maintaining a professional character in the art critical waters is also significant for an art dealer. Many of them are involved in writing and publishing of exhibition catalogues, monographs and comprehensive catalogues raisonnés of their represented artists. By getting into academic studies and research, dealers get recognized from scholars as well, but more importantly, gain insight and understanding of the art world from every aspect. Advice from art dealers is not always sought by individual collectors. It's a common practice that museums, art institutions and nonprofits also seek opinions from reputable dealers, asking their help in locating certain art pieces they wish to borrow and include in their exhibitions. Occasionally, if an art dealer is well-experienced, he or she might even be appointed the show curator. For collectors, this aspect of art dealers' activities is interesting, as they themselves may be asked to lend an artwork for a show. Trusting a dealer in these instances is crucial, but it's good to know that collectors who welcome museums and institutions and happily lend their pieces are always more favored in the art circles, in general.
Sales is the Beginning is The End
The multi-purposefulness of art dealers doesn't end there. One of their most important answers to the what do art dealers do question is – appraising and selling, as well as reselling.Appraisal is something every collector needs for many purposes including insurance and all kinds of legal issues. An art dealer with an experience and reputation is sure to provide an appraisal worthy of a larger institution and as some collectors feel more comfortable working with an individual rather than with a group, this may be a great solution. Sales is the beginning and the end of an art dealer's job. With the first successful trade, a relationship with a collector is started, hopefully a long-term one. These art world intermediaries aid in buying, thus minimizing the risks of studio purchase, selling and reselling of client's artworks, managing all the undergoing consultations and papers regarding the art investment. Collectors who come to art dealer wanting to resell a piece are very much appreciated and are more likely to be offered the best artworks in the future.
Road to Fame and a Gallery
We hope to have clarified the work of dealers now, especially to all the young collectors who are still seeking the definition of their collections. To paint the picture of just how important art dealers are, let's remind ourselves of the most influential people of the trade who subsequently turned into world's most influential gallerists such as Larry Gagosian, David Zwirner, Arne Glimcher of the Pace Gallery, Iwan Wirth, Marian Goodman, William Acquavella, Barbara Gladstone, Paula Cooper, Dominique Levy and Robert Mnuchin, or Matthew Marks.
Feature image: Larry Gagosian and Jared Leto
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NYSE:KRC
Kilroy Realty News Headlines
Adding Kilroy Realty Co.
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KRC Articles
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Kilroy Realty (NYSE:KRC) News Headlines Today
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Sentiment refers to the positivity or negativity of each headline according to our language processing algorithm.
Sentiment All Sentiments
Kilroy Realty (NYSE:KRC) Raised to "Buy" at Zacks Investment Research
marketbeat.com - January 11 at 4:19 AM
Kilroy Realty Announces 2021 Tax Treatment of Its Dividend Distributions
finance.yahoo.com - January 14 at 4:45 PM
KRC Makes Bullish Cross Above Critical Moving Average
nasdaq.com - January 3 at 6:36 PM
Interesting KRC Put And Call Options For August 2022
nasdaq.com - December 16 at 3:22 PM
Investors in Kilroy Realty (NYSE:KRC) have made a return of 15% over the past year
Is Kilroy Realty Corp (KRC) Going to Burn These Hedge Funds?
finance.yahoo.com - December 11 at 6:13 PM
Finalist: Heidi R. Roth
latimes.com - December 5 at 9:26 AM
Kilroy Continues to Outperform ESG Benchmarks and Advance Sustainability Leadership Position on a Global Level
finance.yahoo.com - November 18 at 8:06 PM
KRC January 2022 Options Begin Trading
nasdaq.com - November 18 at 3:06 PM
Europe's Most Feared Investor Chris Hohn is Buying These 6 Stocks
Will a Return to Work Help This Real Estate Specialist?
finance.yahoo.com - November 11 at 7:59 AM
Kilroy Realty Corporation (KRC) Q3 2021 Earnings Call Transcript
finance.yahoo.com - October 29 at 12:38 AM
Kilroy Realty (KRC) Beats Q3 FFO and Revenue Estimates
Kilroy Realty Corporation Q3 adjusted earnings Beat Estimates
markets.businessinsider.com - October 27 at 7:38 PM
Hedge Funds Are Cashing Out Of Kilroy Realty Corp (KRC)
finance.yahoo.com - October 19 at 3:16 PM
The 33% return delivered to Kilroy Realty's (NYSE:KRC) shareholders actually lagged YoY earnings growth
finance.yahoo.com - September 29 at 12:51 AM
'We blew it': What Kilroy Realty did wrong during the pandemic — and right
finance.yahoo.com - September 23 at 10:53 PM
Kilroy Realty, L.P. Prices $450.0 Million of 2.650% Senior Notes Due 2033
finance.yahoo.com - September 23 at 5:53 PM
Kilroy Realty, L.P. to Redeem All Outstanding 3.800% Senior Notes Due 2023
Kilroy Realty Increases Common Dividend 4.0%
Kilroy's Newly Acquired Indeed Tower Awarded LEED Platinum Certification
finance.yahoo.com - September 9 at 4:53 PM
Kilroy Realty Corporation to Participate in the Bank of America Merrill Lynch 2021 Global Real Estate Virtual Conference
Kilroy Realty Stock Has 35% Upside
forbes.com - August 25 at 5:29 AM
The Office Isn't Dead, But Recovery Could Be Slow: Here's Why
finance.yahoo.com - August 6 at 1:13 PM
Kilroy Realty (KRC) Passes Through 3% Yield Mark
nasdaq.com - August 3 at 7:36 PM
With EPS Growth And More, Kilroy Realty (NYSE:KRC) Is Interesting
nasdaq.com - July 30 at 7:10 PM
Does Kilroy Realty (NYSE:KRC) Deserve A Spot On Your Watchlist?
finance.yahoo.com - July 30 at 2:10 PM
finance.yahoo.com - July 30 at 9:09 AM
What Wall Street expects from Kilroy Realty's earnings
markets.businessinsider.com - July 26 at 2:51 PM
KRC: Raising target price to $91.00
finance.yahoo.com - July 16 at 12:51 AM
Hedge Funds Are Crazy About Kilroy Realty Corp (KRC)
finance.yahoo.com - July 13 at 12:38 PM
As Companies Move to Texas, Kilroy Realty Follows
finance.yahoo.com - July 9 at 10:18 AM
First Look: Indeed Tower almost ready for delivery
bizjournals.com - July 2 at 11:40 AM
Kilroy Realty Announces Dates for Second Quarter 2021 Earnings Release and Conference Call
finance.yahoo.com - July 1 at 5:48 PM
KRC: Lowering target price to $92.00
finance.yahoo.com - June 24 at 11:58 PM
5 at 5: Your Daily Digest for Real Estate Investing, 6/18/21
finance.yahoo.com - June 18 at 7:33 PM
This Office REIT Is Expanding Its Reach Into Austin
msn.com - June 18 at 2:32 PM
Kilroy Realty to Make 3 Strategic Acquisitions Worth $670M
finance.yahoo.com - June 17 at 8:17 AM
2 Office REITs to Buy in June
finance.yahoo.com - May 29 at 9:09 AM
Political Decisions Influence Kilroy Realty KRC and Investors Should Listen
marketwatch.com - May 27 at 9:11 AM
Interesting KRC Put Options For July 16th
nasdaq.com - May 20 at 1:59 PM
Kilroy Realty's 9455 Towne Centre Drive Earns LEED Platinum Certification
finance.yahoo.com - May 19 at 5:58 PM
Kilroy Realty Corporation Common Stock (KRC)
nasdaq.com - May 15 at 12:53 AM
Kilroy Realty: What You Need to Know
fool.com - May 15 at 12:53 AM
finance.yahoo.com - April 30 at 3:26 AM
Kilroy Realty (KRC) Q1 FFO and Revenues Lag Estimates
finance.yahoo.com - April 28 at 11:00 PM
Managing Risk in Kilroy Realty #KRC by Using Price Action
marketwatch.com - April 26 at 11:08 PM
Kilroy expands revolving credit facility by $350M to $1.1B
seekingalpha.com - April 22 at 8:18 AM
Is KRC Stock A Buy or Sell?
Kilroy Realty Obtains a $1.1 Billion Sustainability-Linked Unsecured Revolving Credit Facility
finance.yahoo.com - April 20 at 6:38 PM
Kilroy Realty Earns 2021 ENERGY STAR® Sustained Excellence Award for Sixth Year in a Row


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Q: linkation of excel, javascript ,python and html May I know how do I link my excel spreadsheet, java script, python and html table together when uploading through my computer? I have been doing it for three days already. the table row and column is all input so when I uploaded the file it shows the uploaded value and can be edited through the web page. I know how to upload files but the linkage is what i have problem with. I can only upload the excel spreadsheet when it's in the project folder itself.
My html code:

X	Y

<input type="text" onkeyup="" id="dataX1">	<input type="text" onkeyup="" id="dataY1">	<button onclick="remove(1)">Remove</button>

<input type="text" onkeyup="" id="dataX2">	<input type="text" onkeyup="" id="dataY2">	<button onclick="remove(2)">Remove</button>

<input type="text" onkeyup="" id="dataX3">	<input type="text" onkeyup="" id="dataY3">	<button onclick="remove(3)">Remove</button>

<input type="text" onkeyup="" id="dataX4">	<input type="text" onkeyup="" id="dataY4">	<button onclick="remove(4)">Remove</button>

<input type="text" onkeyup="" id="dataX5">	<input type="text" onkeyup="" id="dataY5">	<button onclick="remove(5)">Remove</button>

<input type="text" onkeyup="" id="dataX6">	<input type="text" onkeyup="" id="dataY6">	<button onclick="remove(6)">Remove</button>

<input type="text" onkeyup="" id="dataX7">	<input type="text" onkeyup="" id="dataY7">	<button onclick="remove(7)">Remove</button>

<input type="text" onkeyup="" id="dataX8">	<input type="text" onkeyup="" id="dataY8">	<button onclick="remove(8)">Remove</button>

<input type="text" onkeyup="" id="dataX9">	<input type="text" onkeyup="" id="dataY9">	<button onclick="remove(9)">Remove</button>

<input type="text" onkeyup="" id="dataX10">	<input type="text" onkeyup="" id="dataY10">	<button onclick="remove(10")>Remove</button>

<input type="text" onkeyup="" id="dataX11">	<input type="text" onkeyup="" id="dataY11">	<button onclick="remove(11)">Remove</button>

<input type="text" onkeyup="" id="dataX12">	<input type="text" onkeyup="" id="dataY12">	<button onclick="remove(12)">Remove</button>

<input type="text" onkeyup="" id="dataX13">	<input type="text" onkeyup="" id="dataY13">	<button onclick="remove(13)">Remove</button>

<input type="text" onkeyup="" id="dataX14">	<input type="text" onkeyup="" id="dataY14">	<button onclick="remove(14)">Remove</button>

<input type="text"onkeyup=""  id="dataX15">	<input type="text" onkeyup="" id="dataY15">	<button onclick="remove(15)">Remove</button>

My python code (can only be uploaded through project folder):
import openpyxl
from flask import jsonify

@app.route('/upload/', methods=['POST'])
def upload():
    book = openpyxl.load_workbook('Sample.xlsx') #read'Sample.xlsx'
    sheet = book.active #read
    X1 = sheet.cell(row=1, column=1) #read
    X2 = sheet.cell(row=2, column=1) #read
    X3 = sheet.cell(row=3, column=1) #read
    X4 = sheet.cell(row=4, column=1) #read
    X5 = sheet.cell(row=5, column=1) #read
    X6 = sheet.cell(row=6, column=1) #read
    X7 = sheet.cell(row=7, column=1) #read
    X8 = sheet.cell(row=8, column=1) #read
    X9 = sheet.cell(row=9, column=1) #read
    X10 = sheet.cell(row=10, column=1) #read
    X11 = sheet.cell(row=11, column=1) #read
    X12 = sheet.cell(row=12, column=1) #read
    X13 = sheet.cell(row=13, column=1) #read
    X14 = sheet.cell(row=14, column=1) #read
    X15 = sheet.cell(row=15, column=1) #read

    Y1 = sheet.cell(row=1, column=2) #read
    Y2 = sheet.cell(row=2, column=2) #read
    Y3 = sheet.cell(row=3, column=2) #read
    Y4 = sheet.cell(row=4, column=2) #read
    Y5 = sheet.cell(row=5, column=2) #read
    Y6 = sheet.cell(row=6, column=2) #read
    Y7 = sheet.cell(row=7, column=2) #read
    Y8 = sheet.cell(row=8, column=2) #read
    Y9 = sheet.cell(row=9, column=2) #read
    Y10 = sheet.cell(row=10, column=2) #read
    Y11 = sheet.cell(row=11, column=2) #read
    Y12 = sheet.cell(row=12, column=2) #read
    Y13 = sheet.cell(row=13, column=2) #read
    Y14 = sheet.cell(row=14, column=2) #read
    Y15 = sheet.cell(row=15, column=2) #read

    data = {"X1": X1.value, "X2": X2.value, "X3": X3.value, "X4": X4.value, "X5": X5.value, "X6": X6.value, "X7": X7.value, "X8": X8.value, "X9": X9.value, "X10": X10.value, "X11": X11.value, "X12": X12.value, "X13": X13.value, "X14": X14.value, "X15": X15.value, "Y1": Y1.value, "Y2": Y2.value, "Y3": Y3.value, "Y4": Y4.value, "Y5": Y5.value, "Y6": Y6.value, "Y7": Y7.value, "Y8": Y8.value, "Y9": Y9.value, "Y10": Y10.value, "Y11": Y11.value, "Y12": Y12.value, "Y13": Y13.value, "Y14": Y14.value, "Y15": Y15.value}
    data = jsonify(data)
    return data

and finally my JavaScript code:
$('#upload').on('submit', function(e){
    e.preventDefault();
    $.ajax({
        url: 'http://127.0.0.1:5000/upload/',
        data: {},
        method: 'POST',
        success: function(data) {
            var arrayX = [];
            var arrayY = [];
            var shape = "";
            var i;
            for (i = 1; i < 16; i++) {
            var Xdata = "X" + String(i)
            var Ydata = "Y" + String(i)
            arrayX.push(Xdata)
            arrayY.push(Ydata)
            var checkX = data[arrayX[i-1]]
            var checkY = 500 - data[arrayY[i-1]]
            var datatextX = "dataX" + String(i)
            var datatextY = "dataY" + String(i)
            var bug = 0
            if (checkX == null) {
                checkX = ""
                bug = 1
            }
            if (checkY == null) {
                checkY = ""
                bug = 1
            }
            if (bug == 1){
                shape += ""
            } else if (i == 1) {
                shape += checkX + ", " + checkY
                document.getElementById(datatextX).value = checkX
                document.getElementById(datatextY).value = 500 - checkY
            } else if (i < 15){
                shape += ", " + checkX + ", " + checkY
                document.getElementById(datatextX).value = checkX
                document.getElementById(datatextY).value = 500 - checkY
            }
            }
            //shape = data[arrayX[0]] + ", " + data[arrayY[0]]
            $('#uploadtxt').html(shape)
            var elem = document.getElementById('edit-star');
            elem.setAttribute("points", shape);
            let sns='http://www.w3.org/2000/svg',
            xns='http://www.w3.org/1999/xlink',
            root = document.getElementById('svg-edit-demo'),
            star = document.getElementById('edit-star'),
            rootMatrix,
            originalPoints = [],
            transformedPoints = [];
            for (let i = 0, len = star.points.numberOfItems; i < len; i++, numPoint+=1) {
               let handle = document.createElementNS(sns, 'use'),
               point = star.points.getItem(i),
               newPoint = root.createSVGPoint();

               handle.setAttributeNS(xns, 'href', uniqueClass);
               handle.setAttribute('class', uniqueID);

               handle.x.baseVal.value = newPoint.x = point.x;
               handle.y.baseVal.value = newPoint.y = point.y;

               handle.setAttribute('data-index', numPoint)

               originalPoints.push(newPoint)

               root.appendChild(handle);
             }
        }
    });
});

redpajama_set_name: RedPajamaStackExchange


Pacific Northwest National Laboratory's Christopher Mundy, Roger Rousseau, Greg Schenter, and Shawn Kathmann and IBM Research-Zurich's Alessandro Curioni were selected for a U.S. Department of Energy Office of Science INCITE award. This competitive award provides access to some of the world's most powerful supercomputers to model complex processes and analyze large data sets.
The team received a total of 1.5 million hours on DOE's Advanced Scientific Computing Research's IBM Blue Gene/P at Argonne National Laboratory and the Cray XT4 at Oak Ridge National Laboratory. On these computers, they will run calculations to explore the fundamental chemical physics of chemical reactions in complex solutions and at their interfaces.
The models the team use accurately portray reactions under real-world conditions using a combination of statistical and quantum mechanical methods. This integration of methodologies will require the processing power and speed of supercomputers. Standard desktop systems are simply incapable of running the proposed calculations to a timely completion.
"We are very fortunate to have received this award," said principal investigator Mundy.
The insights gained from these models may aid in our fundamental understanding of molecular interactions applied to materials to store hydrogen, a potential fossil fuel alternative. In addition, this new information may help scientists and engineers design better chemical processes and catalysts, which increase the speed and efficiency of important chemical reactions.
In addition, other teams including PNNL researchers received INCITE awards. PNNL's Bruce Palmer and Karen Schuchardt are part of the team working to simulate global cloudiness, Steve Ghan is a key contributor to the climate-science computational end station development team, and Glenn Hammond is part of the team modeling reactive flows in porous media.


text:
FORMER Brocklesby footballer Tony Armstrong will create AFL broadcasting history on Sunday. The 29 year-old will become the first Indigenous person to call a match on commercial radio. Armstrong will be on the ball-by-ball team for Triple M at the Richmond-West Coast match at the MCG. "Hopefully I'm at the start of a long journey, I'm really rapt at being given the opportunity," Armstrong said. The former Adelaide, Sydney and Collingwood player, who played 35 games from 2010 to 2015, has been calling for the National Indigenous Radio Service. His start in commentating came on the Queen's Birthday weekend last year when he was asked by Brisbane premiership player Chris Johnson to do special comments. "I didn't have the on-field career to last just doing specials, so I started doing calling," Armstrong said. "Once I realised how much fun it was, I thought why not just be as good as I can be." Armstrong's Triple M gig follows Gilbert McAdam, who played at St Kilda, this year becoming the first Indigenous member of the Seven AFL broadcast team. While not wanting to compare himself directly to the Marngrook Footy Show host, Armstrong said it was important to create firsts to encourage others. "Hopefully Gilly is out there inspiring young Indigenous boys and girls that they can go on telly and hopefully this weekend, and if Triple M gives me more of an opportunity in the future, I can do the same thing," Armstrong said. Triple M AFL chief Ewan Giles said: "Tony has impressed with his dedication and commitment to his craft. "While he has been play-by-play calling with NIRS over the past 12 months, he has been working with us to fine-tune his skills and is now ready to make his mark as a caller on Triple M footy." Armstrong grew up at Brocklesby and played senior football for the Roos before embarking on his AFL career. His mother lives at Burrumbuttock.
https://nnimgt-a.akamaihd.net/transform/v1/crop/frm/XJLgPnEdnKaFugZzKyL6Sw/6cd5c593-3475-46d5-be03-6125b22eb2fa.jpg/r0_92_4261_2499_w1200_h678_fmax.jpg
August 15 2019 - 12:50PM
Past AFL player from Brocklesby, Tony Armstrong, to break new broadcasting ground on Triple M radio
Anthony Bunn
On the team: Tony Armstrong, who used to call himself as Sydney champion Michael O'Loughlin as a boy running around the hallway of his home, will be describing AFL play for Triple M on Sunday. Picture: MARTIN KEEP/AFL PHOTOS
FORMER Brocklesby footballer Tony Armstrong will create AFL broadcasting history on Sunday.
The 29 year-old will become the first Indigenous person to call a match on commercial radio.
Armstrong will be on the ball-by-ball team for Triple M at the Richmond-West Coast match at the MCG.
"Hopefully I'm at the start of a long journey, I'm really rapt at being given the opportunity," Armstrong said.
The former Adelaide, Sydney and Collingwood player, who played 35 games from 2010 to 2015, has been calling for the National Indigenous Radio Service.
His start in commentating came on the Queen's Birthday weekend last year when he was asked by Brisbane premiership player Chris Johnson to do special comments.
"I didn't have the on-field career to last just doing specials, so I started doing calling," Armstrong said.
"Once I realised how much fun it was, I thought why not just be as good as I can be."
Armstrong's Triple M gig follows Gilbert McAdam, who played at St Kilda, this year becoming the first Indigenous member of the Seven AFL broadcast team.
While not wanting to compare himself directly to the Marngrook Footy Show host, Armstrong said it was important to create firsts to encourage others.
Welcome move: AFL general manger inclusion and social policy Tanya Hosch and AFL chief executive Gillon McLachlan congratulate Tony Armstrong on his elevation to the Triple M ball-by-ball team. Picture: MARTIN KEEP/AFL PHOTOS
"Hopefully Gilly is out there inspiring young Indigenous boys and girls that they can go on telly and hopefully this weekend, and if Triple M gives me more of an opportunity in the future, I can do the same thing," Armstrong said.
Triple M AFL chief Ewan Giles said: "Tony has impressed with his dedication and commitment to his craft.
"While he has been play-by-play calling with NIRS over the past 12 months, he has been working with us to fine-tune his skills and is now ready to make his mark as a caller on Triple M footy."
Armstrong grew up at Brocklesby and played senior football for the Roos before embarking on his AFL career.
His mother lives at Burrumbuttock.
This story Former Riverina footballer to create AFL radio history first appeared on The Border Mail.


Purple Square Tablecloth - TOP 10 Results for Price Compare - Purple Square Tablecloth Information for April 22, 2019.
The Remedios 70-inch Square Polyester Tablecloth can complete any table service and is durable enough to last. DURABLE 100% premium polyester, durable and stain resistant. Each is one-piece design and finished with surged edges. The tablecloths become softer every time they're washed. FUNCTIONAL Multi-size and multi-color for your choice. Perfect for many occasions, such as wedding, party, reataurant and more. EASY CARE Machine wash in cold water, gentle cycle. Low heat iron as needed. Line dry recommended to avoid shrinkage. SIZE INFORMATION A full drop is regarded as a formal look while the half drop is considered more casual. A puddle drop can be amazingly elegant, depending on the setting and mood you're trying to convey. Please refer to our tablecloth size chart to choose the tablecloth size for your table. KINDLY NOTICE As we are updating our product service, we have 2 different packing bags of these tablecloths. We promise that the cloth fiber and the material quality of Remedios Tablecloths are the same as before. We will experiment with a technology of the material components to give you value-driven tablecloths at wallet-friendly prices.
Deconovo Solid Color Water Risestant Table Cloth Slub Polyester Tablecloth These Deconovo solid water risestant tablecloth is made of high quality slub polyester material. Their good texture and attractive color bring a beautiful addition to any table to impress your guests. They are waterproof and wrinkle resistant, suitable for indoor and outdoor use. However, due to differences in computer monitors, the actual products colors can slightly differ from the ones on. 1.Deconovo solid color waterproof table cloth is made of high quality slub polyester material, the bright and fresh color dresses your table lively. 2. This slub polyester tablecloth is water repellant and wrinkle resistant, suitable for indoor and outdoor use. 3. Three sizes to choose from 54W x 54L inch, 54W x 72L inch. 4. A beautiful addition to any table, and match with a variety of DECONOVO decors. 5. 100% polyester. Machine washable. 6. Our products pictures are real and shot by professionals. However, due to differences in computer monitors display, there can be color variations between the actual product and your screen.
This extra-long lasting, 6 mil thick, solid color vinyl tablecloth is perfect for your heavy use tablecloth needs. The Galaxy Vinyl Tablecloth by Fairfax Collection is twice as heavy as most vinyl tablecloths. These vinyl tablecloths have durable sewn edges. The quality flannel backing for extra durability, weight and slip resistance help keep the tablecloth in place. The vinyl surface makes clean up a snap and keeps your table protected from everyday spills and accidents. Perfect for indoor and outdoor dining. Great for picnics, birthdays, dinner, BBQ's, gatherings, parties, occasions, everyday meals and more! For best results, use a mild soap solution with a damp cloth or sponge on vinyl surface. Do not iron. This is a 52" x 52" square tablecloth and seats 4.
Don't satisfied with search results? Try to more related search from users who also looking for Purple Square Tablecloth: Transitional Linear Chandelier, Topiary Form, Standard 4 Channel, Striking Jet, Video Audio Capture Card Software.
Purple Square Tablecloth - Video Review.


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Rose-Margaret Ekeng-Itua, Ph.D.
For influence and passion extending far beyond the classroom; for vision, leadership, and mentoring that attracts and retains young women; and for advancing engineering as an instrument for social change.
Rose-Margaret Ekeng-Itua, Ph.D., is a professor of engineering at Ohlone College in Fremont, California. She is also a TED-Ed author with more than 20 years of experience in engineering and STEM education. At Ohlone College, she is principal investigator for the National Science Foundation Advanced Technological Education Grant for Smart Advanced Manufacturing Education in the Silicon Valley. She also leads various apprenticeship/internship initiatives for engineering and advanced manufacturing in the Silicon Valley region.
She deeply believes in humanitarian engineering as a force for social change. She created a culturally responsive pedagogy that has increased retention of women and other historically underrepresented engineering students, and her efforts were instrumental in more than doubling enrollment in Ohlone's engineering department within her first two years at the college.
Dr. Ekeng-Itua's strong commitment to students' socioeconomic upward mobility motivated her to launch several academia-collaborative projects with Fortune 500 companies, including Vodafone, Orange, T-Mobile, Tesla, and other companies such as Scottish Energy, London Underground, Thames Water, and Ericsson. These projects resulted in and ensured a 100% student graduate employability.
A thought leader in engineering and engineering technology education, Dr. Ekeng-Itua shares best practices at conferences, speaker events, and workshops. She has also been a two-time visiting research fellow at the University of California, Berkeley, where she led research and investigations on factors influencing manufacturing productivity and Smart Microgrids in the U.S. and in emerging economies.
Dr. Ekeng-Itua is widely recognized for her work in engineering education and advocacy for women, including, among others, by SWE's 2018 Advocating Women in Engineering Award, the 2017 American Society for Engineering Education Outstanding Teaching Award; and the Multicultural Women Executive Leadership Foundation 2022 Courage in Leadership Award.
Within SWE, Dr. Ekeng-Itua has served as an FY21 special director on the board of directors, FY21 Africa task force board liaison, FY20 president of the Mt. Diablo Section, founding lead for the African American affinity group, and founding faculty advisor for one of the first SWE collegiate interest groups. Currently, she is founding co-lead for the community college affinity group and an advisor on the SWE Africa advisory board.
In her community, Dr. Ekeng-Itua is a commissioner on the economic development advisory board for the city of Fremont, California, and an appointee of the California State Senate Rules Committee for the Associate Degree for Transfer Intersegmental Implementation Committee.
Dr. Ekeng-Itua holds a B.S. in engineering with honors in electrical and electronic engineering from Federal University of Technology Owerri in Nigeria; an M.S. in mobile and satellite communications engineering from the University of Surrey, U.K.; and a Ph.D. in cybernetics from the University of Reading in the U.K. She enjoys traveling, painting, reading, listening to music, and spending time with family and friends.


text:
Q: Force Link With Editor to be activated by default in Eclipse RCP I am trying to get the Eclipse force activate Link With Editor option by default . The solution doesn't necessarily need to be programmatically , but it would be good. So , I could use an .epf setting too or any other option.
Till now , I found this link : "link with editor" for FormEditor

A: As far as I know, there is no preference for this setting which is persisted in <workspace>/.metadata/.plugins/org.eclipse.e4.workbench/workbench.xmi (as <sharedElements ... elementId="org.eclipse.ui.navigator.ProjectExplorer" ...> ... <persistedState key="memento" value="...CommonNavigator.LINKING_ENABLED="0"..."/> or ...="1"..."/>).
Programmatically, the linking enablement can be set in the class org.eclipse.ui.navigator.resources.ProjectExplorer via the method setLinkingEnabled.


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Health benefits of Grape Seed Extract
Human case reports and results from basic research provide preliminary evidence that grape seed extract may affect heart diseases such as hypertension, high levels of blood cholesterol, cancer, platelet aggregation or inflammation. Some of these effects have been demonstrated in vivo in animal models. According to the American Cancer Society, "there is very little reliable scientific evidence available at this time that drinking red wine, eating grapes, or following the grape diet can prevent or treat cancer in people".
A polyphenol contained in grape seeds is resveratrol, which is under study for its possible effect on cancer cell growth, proliferation or apoptosis, among other potential chemopreventive mechanisms.
Other preliminary research on disease models include:
skin and wounds -- OPCs induced vascular endothelial growth factor and accelerated healing of injured skin in mice
teeth -- seed phenolics may inhibit oral sugar metabolism and retard growth of certain bacteria that cause dental caries
bones -- grape seed extracts enhanced bone density and strength in experimental animals
in vitro cancer studies -- grape seed proanthocyanidins decreased tumor numbers and reduced the malignancy of papillomas
ultraviolet damage -- dietary proanthocyanidins are under study for mechanisms against carcinogenesis and sunscreen protection
anti-viral effects
antibacterial properties
blood flow and fluid balance
There are 13 clinical trials as of January, 2012, assessing potential effects of grape seed extracts on human diseases, such as breast cancer, blood estrogen levels in postmenopausal women, and coronary artery disease.
One clinical trial with adults having coronary disease or cardiac risk factors concluded that: "Four weeks of muscadine grape seed supplementation in subjects with increased cardiovascular risk did not produce a statistically significant increase in brachial flow-mediated vasodilation or a significant change in other biomarkers of inflammation, lipid peroxidation, or antioxidant capacity. However, the muscadine grape seed supplement did result in a significant increase in resting brachial diameter. The clinical significance of the effect on resting diameter is not yet established."
A meta-analysis of randomized controlled trials concluded that "grape seed extract appears to significantly lower systolic blood pressure and heart rate, with no effect on lipid or C-reactive protein levels."
The US National Center for Complementary and Alternative Medicine reported that oral administration of grape seed extract was well tolerated in people over 8 weeks. In one completed clinical trial, grape seed extract did not alleviate the hardening of breast tissue in female patients undergoing radiation therapy to treat breast cancer.
Labels: Health benefits of Grape Seed Extract
High Fructose Syrup and Diabetes-A study-STOP all products that has high fructose HFCS.
There was a time when "food" was something you ate or drank to nourish your body. It's a necessity of life -- like air and water -- and you could usually depend on food to be nourishing and healthy. But then something happened. Someone decided that food didn't have to be nutritious or healthy -- as long as they could make a profit from selling it. Early in human civilization, the limited diets of local farming and food production were supplemented by food specialists who imported things like spices, grains and sugar, making our consumption of food more interesting and varied. Farmers and fishermen sold their products in local markets, making it easier for the average human to obtain and eat a well balanced and varied diet. Consumers had always trusted that their food supply was healthy.But something has changed.Over the past year we have seen hidden cameras that documented Chinese bakeries who substituted bacon grease and cardboard as the filler of meat dumplings. We've had stories of anti-freeze and brake fluid being used as a cheap sweetner in everything from candy to cough medicine and toxic chemicals used to fake the protein analysis of baby and pet food -- all to make a profit. These high profile examples of greed have made people seriously ill and have even killed dozens of people. But a more sinister product of greed and poison is probably in your body right now -- killing you softly with sweetness.High Fructose Corn Syrup -- killing you softly Have you had a soft drink today? How about a candy bar, a stick of gum or some fruit juice. These are just the most obvious foods that contain high fructose corn syrup or HFCS. It's in just about everything that's sweet and things you don't normally think of as sweet. Things like bread, cereal, coffee creamer, ketchup and salad dressing all contain High Frustose Corn Syrup. It's now implicated as the main source of obesity, high blood pressure, elevated triglycerides and diabetes -- conditions that cause mysery and death around the globe.The GreedHFCS is a useful ingredient because it's sweet and saves the food producers money. Real sugar is very expensive because of sugar tariffs and import quotas. Even with all the chemistry and processing, HFCS is still the cheapest way to sweeten food. It's also said to extend the shelf-life of products and is much easier to mix in food because it's a liquid. Over the past 40 years, as methods for producing HFCS improved, food and beverage companies have replaced all other sweeteners with HFCS.What is HFCS?HFCS is not natural. It's manufactured from cornstarch, using a process developed in Japan in 1971. HFCS is made up of glucose and fructose molecules, unlinked. The most common form in the American food supply is HFCS-55, which contains 55% fructose and 45% glucose. The fact that the fructose and glucose molecules are not linked is very significant and we'll discuss that later.Fructose, the main ingredient of HFCS, is the villain in this story. Fructose is a simple sugar normally found in fresh fruits and vegetables. Fuctose is unique in that it does not require the body to make insulin in order to use it as fuel. Ingesting fructose doesn't produce that "sugar rush" you get when the body is pumping insulin to the bloodstream to digest other sugars, like glucose. This "rush" is connected with your feeling of "satisfaction" or "fullness," so after ingesting fructose you may not feel satisfied -- even though your caloric intake is the same as other sugars. This causes you to consume more.If you received your fructose only from vegetables and fruits (where it originates) as most people did a century ago, you'd consume about 15 grams per day -- a far cry from the 73 - 100 grams per day the typical adolescent gets from drinking HFCS sweetened soft drinks. In vegetables and fruits, it's mixed in with fiber, vitamins, minerals, enzymes, and beneficial phytonutrients, all which moderate the negative metabolic effects.Food manufacturers often compare the calorie of HFCS to normal table sugar. A teaspoon of both HFCS and table sugar is only 16 calories. Even accounting for the elevated consumption of fructose in HFCS, the increase in calories is not that significant. 15 grams of fructose (1 tablespoon) contains about 16 calories. 100 grams of fructose (6 tablespoons) contains about 105 calories. This seems hardly enough calories to cause a health problem, yet research confirms the problems are very real.
For years scientists and nutritionists have been warning that more studies needed to be done to determine the complex metabolism of how our livers process and deal with fructose. While the exact biological process remains unknown, studies continue to link HFCS with many unhealthy global epidemics.2010: The Princeton Study reveals the terrible truthA Princeton University research team has demonstrated that all sweeteners are not equal when it comes to weight gain: Rats with access to high-fructose corn syrup gained significantly more weight than those with access to table sugar, even when their overall caloric intake was the same. In addition to causing significant weight gain in lab animals, long-term consumption of high-fructose corn syrup also led to abnormal increases in body fat, especially in the abdomen, and a rise in circulating blood fats called triglycerides. The researchers say the work sheds light on the factors contributing to obesity trends in the United States.
When rats are drinking high-fructose corn syrup at levels well below those in soda pop, they're becoming obese -- every single one, across the board.
The Princeton ExperimentThe first study showed that male rats given water sweetened with high-fructose corn syrup in addition to a standard diet of rat chow gained much more weight than male rats that received water sweetened with table sugar, or sucrose, in conjunction with the standard diet. The concentration of sugar in the sucrose solution was the same as is found in some commercial soft drinks, while the high-fructose corn syrup solution was half as concentrated as most sodas. The second experiment -- the first long-term study of the effects of high-fructose corn syrup consumption on obesity in lab animals -- monitored weight gain, body fat and triglyceride levels in rats with access to high-fructose corn syrup over a period of six months. Compared to animals eating only rat chow, rats on a diet rich in high-fructose corn syrup showed characteristic signs of a dangerous condition known in humans as the metabolic syndrome, including abnormal weight gain, significant increases in circulating triglycerides and augmented fat deposition, especially visceral fat around the belly. Male rats in particular ballooned in size: Animals with access to high-fructose corn syrup gained 48 percent more weight than those eating a normal diet. In humans, this would be equivalent to a 200-pound man gaining 96 pounds!
"Some people have claimed that high-fructose corn syrup is no different than other sweeteners when it comes to weight gain and obesity, but our results make it clear that this just isn't true, at least under the conditions of our tests," said psychology professor Bart Hoebel, who specializes in the neuroscience of appetite, weight and sugar addiction. "When rats are drinking high-fructose corn syrup at levels well below those in soda pop, they're becoming obese -- every single one, across the board. Even when rats are fed a high-fat diet, you don't see this; they don't all gain extra weight." "These rats aren't just getting fat; they're demonstrating characteristics of obesity, including substantial increases in abdominal fat and circulating triglycerides," said Princeton graduate student Miriam Bocarsly. "In humans, these same characteristics are known risk factors for high blood pressure, coronary artery disease, cancer and diabetes." The Princeton researchers note that they do not know yet why high-fructose corn syrup fed to rats in their study generated more triglycerides, and more body fat that resulted in obesity. [Results published online March 18 by the journal Pharmacology, Biochemistry and Behavior.]Unusual metabolism of HFCSDiscoveries about the unusual metabolism of HFCS were announced in a report published in the Archives of Internal Medicine by Julie Palmer and her colleagues at Boston University. This study showed that fructose metabolized differently from glucose, common table sugar.
"The public should be made aware that these drinks are not a healthy alternative to soft drinks with regard to risk of type 2 diabetes..."
The findings came from an assessment of 44,000 black women in the United States who were checked from 1995 through 2005. Those who said they drank two or more non-diet soft drinks (sweetened with HFCS) a day had a 24 percent increased risk for developing type 2 diabetes than those in the study who drank fewer than one regular soft drink per month, the research team said.Women who drank two or more sweetened (HFCS) fruit drinks per day had a 31 percent increased risk compared to those who drank fewer than one such fruit drink a month. Diet soft drinks, grapefruit juice and orange juice (which contain no HFCS) were not linked to a higher diabetes risk.While pure orange and grapefruit juices also contain natural sugars, including fructose, they may have a different metabolic effect or may be more likely to be consumed as part of a meal, the investigators said.Those who develop type 2 diabetes will need a blood sugar monitor and will need to change their diet for the rest of their lives.A related study found that a low-fat diet does not seem to change the risk of diabetes. Thus becoming obese and becoming diabetic does not seem to be linked purely to weight gain from fat, but rather from the obesity specifically induced by the HFCS.
"The common denominator that appears clear is that calories trump everything. And certain nutrients, like high fructose corn syrup, make it easier to overeat. If you keep the calories low, you can probably eat almost anything, which is what the low-carb diets show us. Specific metabolic issues aside, an important reason that low carb works is because you don't eat a lot of calories." -- Dr. Mark Feinglos of the Duke University Medical Center in North Carolina.
So, what's so different about HFCS?Natural sources of fructose include fruits, some vegetables, honey, sugar cane and sugar beets. In a normal diet, fructose would only be ingested in these natural forms. It would remain a relatively low source of sugar. Today, the inclusion of fructose in virtually every food product dramatically increases the proportion of fructose in our diet.For many years, Dr. Meira Fields and her coworkers at the US Department of Agriculture investigated the harmful effects of dietary sugar on rats. They discovered that when male rats are fed a diet deficient in copper, with sucrose as the carbohydrate, they develop severe pathologies of vital organs. Liver, heart and testes exhibit extreme swelling, while the pancreas atrophies, invariably leading to death of the rats before maturity. Sucrose is a disaccharide composed of 50 percent glucose and 50 percent fructose. Dr. Fields repeated her experiments to determine whether it was the glucose or fructose moiety that caused the harmful effects. Starch breaks down into glucose when digested. On a copper-deficient diet, the male rats showed some signs of copper deficiency, but not the gross abnormalities of vital organs that occur in rats on the sucrose diet. When the rats were fed fructose, the fatal organ abnormalities occured.Pure fructose contains no enzymes, vitamins or minerals and robs the body of its micronutrient treasures in order to assimilate itself for physiological use. While naturally occurring sugars, as well as sucrose, contain fructose bound to other sugars, high fructose corn syrup contains a good deal of "free" or unbound fructose. Research indicates that this free fructose interferes with the heart's use of key minerals like magnesium, copper and chromium. Among other consequences, HFCS has been implicated in elevated blood cholesterol levels and the creation of blood clots. It has been found to inhibit the action of white blood cells so that they are unable to defend the body against harmful foreign invaders.In studies with rats, fructose consistently produces higher kidney calcium concentrations than glucose. Fructose generally induces greater urinary concentrations of phosphorus and magnesium and lowered urinary pH compared with glucose.In humans, fructose feeding leads to mineral losses, especially higher fecal excretions of iron and magnesium, than did subjects fed sucrose. Iron, magnesium, calcium, and zinc balances tended to be more negative during the fructose-feeding period as compared to balances during the sucrose-feeding period.According to a recent article in the American Journal for Clinical Nutrition, high fructose corn syrup (HFCS) is the likely culprit for the obesity epidemic in America, type 2 diabetes, cardiovascular disease and breast cancer.How much HFCS do we eat?The average American consumes about 28.4 kg (62.48 pounds) of HFCS annually, versus 26.7 kg (58.74 pounds) of sucrose (common table type) sugar. Surprisingly many other countries have quite a sweet tooth! Sucrose consumption per person is higher than the USA in the following examples (note also the diabetes rates): * USA: 26.7 kg, 58.74 lbs. -- diabetes rate 7.8%
* EU: 40.1 kg, 88.2 lbs. -- diabetes rate 7.0%
* Brazil: 59.7 kg, 131.34 lbs. -- diabetes rate 15.7%
* Australia: 56.2 kg, 123.64 lbs. -- diabetes rate 8%American Indians hit hard with diabetesAccording to government health officials, not only do the American Indian and Alaska Native populations suffer have the highest diabetes rates among ethnic groups, but the disease is increasing in the young American Indian population.Native American Indians have lived an ethnically isolated existence for tens of thousands of years. They have survived and evolved in a natural environment, free from processed and chemically altered food. As such, Native American Indians represent an example of a human anatomy in harmony with what is healthy and nutritious. European vices, such as alcohol and sugar, have a dramatic effect on these people and so demonstrate the dangerous and unhealthy aspects of our modern diet.
"In some communities, the prevalence rate [of diabetes] is as high as 60 percent among adults," Charles Grim, the head of the Indian Health Service, told the Senate Indian Affairs Committee.Diabetes increased 128 percent among teens ages 15 to 19 between 1990 and 2004. The disease increased 77 percent among young people younger than 15 during the same time frame.Another possible hint at culture or diet as a factor can be seen in the statistics for culturally diverse New York City. According to the city's health commisioner, Dr. Yhomas R. Frieden, in the city 12.2 percent of Hispanics have diabetes, as do 10.8 percent of non-Hispanic blacks. Whites who are not Hispanic have the lowest rate -- 5 percent -- and Asians are second to last, with 6.8 percent. The Bronx leads the city in diabetics, with 11.5 percent of residents having the illness, while 4.6 percent of Staten Island residents have it.Ignoring the factsWhile the exact physiology of the link to diabetes has yet to be understood, several human and animal studies confirm that high levels of fructose, as found in HFCS, can be deadly. Beverage and food manufacturers don't care about these studies because the causal link is not there to make them culpable. They continue to reap the profits and claim that HFCS is totally safe. In the 40 years since the introduction of high-fructose corn syrup as a cost-effective sweetener in the American diet, rates of obesity in the U.S. have skyrocketed, according to the Centers for Disease Control and Prevention. In 1970, around 15 percent of the U.S. population met the definition for obesity; today, roughly one-third of the American adults are considered obese, the CDC reported. High-fructose corn syrup is found in a wide range of foods and beverages, including fruit juice, soda, cereal, bread, yogurt, ketchup and mayonnaise. On average, Americans consume 60 pounds of the sweetener per person every year. Here are some studies which further implicate HFCS:
High Fructose causes diabetes in laboratory.
High-fructose feeding elicits insulin resistance, hyperinsulinism, and hypertension in normal mongrel dogs.FJ Martinez, RA Rizza and JC Romero
Department of Physiology and Biophysics, Mayo Clinic, Mayo School of Medicine, Rochester, Minn. 55905.ABSTRACT
To determine whether chronic high-fructose feeding causes insulin resistance and hypertension in normal dogs, we fed 10 male dogs a normosodic diet containing 60% of the calories as fructose for 20 to 28 days; a control group of 8 dogs was fed a similar diet containing dextrose instead of fructose. In the fructose-fed group, (1) fasting triglyceridemia increased from 35.3 +/- 0.63 to 91.9 +/- 11.55 mg/dL after 25 days (P < .001); (2) fasting insulinemia increased from 19.0 +/- 1.9 to 58.9 +/- 7.22 microU/mL after 25 days (P < .001); (3) insulin resistance, which was estimated by steady-state glycemia during an insulin suppression test, increased from 105.8 +/- 21.5 to 187.8 +/- 32.6 mg/dL after 15 days (P < .001), whereas steady-state insulinemia did not change; (4) mean arterial pressure increased from 100.4 +/- 1.6 to 122.6 +/- 2.3 mm Hg after 28 days (P < .01); and (5) cumulative sodium balance was increased on days 7 through 11 (111.60 +/- 4.44 mEq on day 8, P < .01), returning to normal for the rest of the experiment. All these parameters were similar between the fructose-fed and dextrose- fed groups before the diets were started and remained constant in the dextrose-fed group. Neither group showed any change in body weight, fasting plasma glucose, atrial natriuretic factor, or endothelin-1 levels. We conclude that chronic high-fructose feeding elicits hypertriglyceridemia, insulin resistance, hyperinsulinemia, hypertension, and a transient sodium retention in dogs without fostering fasting hyperglycemia or weight gain.(ABSTRACT TRUNCATED AT 250 WORDS)
Fructose, weight gain, and the insulin resistance syndrome.
Sharon S Elliott, Nancy L Keim, Judith S Stern, Karen Teff and Peter J HavelLaboratory evidence that high fructose is associated with insulin resistance and diabetesABSTRACT
This review explores whether fructose consumption might be a contributing factor to the development of obesity and the accompanying metabolic abnormalities observed in the insulin resistance syndrome. The per capita disappearance data for fructose from the combined consumption of sucrose and high-fructose corn syrup have increased by 26%, from 64 g/d in 1970 to 81 g/d in 1997. Both plasma insulin and leptin act in the central nervous system in the long-term regulation of energy homeostasis. Because fructose does not stimulate insulin secretion from pancreatic ß cells, the consumption of foods and beverages containing fructose produces smaller postprandial insulin excursions than does consumption of glucose-containing carbohydrate. Because leptin production is regulated by insulin responses to meals, fructose consumption also reduces circulating leptin concentrations. The combined effects of lowered circulating leptin and insulin in individuals who consume diets that are high in dietary fructose could therefore increase the likelihood of weight gain and its associated metabolic sequelae. In addition, fructose, compared with glucose, is preferentially metabolized to lipid in the liver. Fructose consumption induces insulin resistance, impaired glucose tolerance, hyperinsulinemia, hypertriacylglycerolemia, and hypertension in animal models. The data in humans are less clear. Although there are existing data on the metabolic and endocrine effects of dietary fructose that suggest that increased consumption of fructose may be detrimental in terms of body weight and adiposity and the metabolic indexes associated with the insulin resistance syndrome, much more research is needed to fully understand the metabolic effect of dietary fructose in humans.
Fructose, insulin resistance, and metabolic dyslipidemia
Heather Basciano, Lisa Federico and Khosrow Adeli, Clinical Biochemistry Division, Department of Laboratory Medicine and Pathobiology, Hospital for Sick Children, University of Toronto, Toronto, Ontario, CanadaHigh fructose linked to insulin resistance Abstract
Obesity and type 2 diabetes are occurring at epidemic rates in the United States and many parts of the world. The "obesity epidemic" appears to have emerged largely from changes in our diet and reduced physical activity. An important but not well-appreciated dietary change has been the substantial increase in the amount of dietary fructose consumption from high intake of sucrose and high fructose corn syrup, a common sweetener used in the food industry. A high flux of fructose to the liver, the main organ capable of metabolizing this simple carbohydrate, perturbs glucose metabolism and glucose uptake pathways, and leads to a significantly enhanced rate of de novo lipogenesis and triglyceride (TG) synthesis, driven by the high flux of glycerol and acyl portions of TG molecules from fructose catabolism. These metabolic disturbances appear to underlie the induction of insulin resistance commonly observed with high fructose feeding in both humans and animal models. Fructose-induced insulin resistant states are commonly characterized by a profound metabolic dyslipidemia, which appears to result from hepatic and intestinal overproduction of atherogenic lipoprotein particles. Thus, emerging evidence from recent epidemiological and biochemical studies clearly suggests that the high dietary intake of fructose has rapidly become an important causative factor in the development of the metabolic syndrome. There is an urgent need for increased public awareness of the risks associated with high fructose consumption and greater efforts should be made to curb the supplementation of packaged foods with high fructose additives. The present review will discuss the trends in fructose consumption, the metabolic consequences of increased fructose intake, and the molecular mechanisms leading to fructose-induced lipogenesis, insulin resistance and metabolic dyslipidemia.
Journal Reference for Princeton article cited: Miriam E. Bocarsly, Elyse S. Powell, Nicole M. Avena, Bartley G. Hoebel. High-fructose corn syrup causes characteristic of obesity in rats: Increased body weight, body fat and triglyceride levels.Pharmacology Biochemistry and Behavior, 2010; DOI: 10.1016/j.pbb.2010.02.012
From viewzone
Labels: high fructose corn syrup and diabetes, मधुमेह /Diabetes
List of Ayurvedic products for various ailment
Baba Ramdev Medicines
Patanjali products by Divya Pharmacy which is also known Baba Ramdev Medicines is really healing many numbers of illness / diseases / health issues that many of the allopathy medicines may not work. No gurantee for cure as these are herbal supplement and not classifies by FDA as to cure disease ,so remember to discuss with your doctors before you buy or use it as many prescription medications interact with herbal medicines or supplements.
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Labels: ayurvedic herbs, Herbal medicine for brain, herbal medicines for ailments
Natural flouride free toothpaste
HiOra Toothpaste
Toothpaste for inflamed and spongy gums
Reduces gingival inflammation
Reduces gingival infection
Reduces gum bleeding and improves gum health
Pilu and Triphala reduce gingival indices in patients with gingivitis
Presence of proteolytic enzymes (Bromelain and Papain) help reduce plaque formation
Babbula, Amra, Lavanga and Nimba are effective against periodontal pathogens
Polyphenols in Triphala exhibit astringent action that tighten the gums and checks bleeding
Brush twice daily till symptoms subside or as directed by your doctor.
Labels: herbal toothpaste, TOOTHPASTE
Herbal treatment for Fatty Liver-LIV 52
Liv.52
Unparalleled in liver care
Hepatoprotective action: The natural ingredients in Liv.52 exhibit potent hepatoprotective properties against infective hepatitis.
Improves appetite: In less than optimal growth and weight gain, Liv.52 normalizes the basic appetite-satiety rhythm. As a daily health supplement, Liv.52 improves appetite, digestion and assimilation processes and promotes weight gain.
For the prevention and treatment of infective hepatitis and loss of appetite.
The Caper Bush (Himsra) contains p-methoxy benzoic acid, which is a potent hepatoprotective. It prevents the elevation of malondialdehyde (biomarker for oxidative stress) levels in plasma and hepatic cells. Caper Bush also inhibits the ALT and AST enzyme levels and improves the functional efficiency of the liver and spleen. Flavonoids present in the Caper Bush exhibit significant antioxidant properties, as well.
Chicory (Kasani) is a potent antioxidant, which can be seen by its free radical scavenging property. The hepatoprotective property of Chicory suppresses the oxidative degradation of DNA in tissue debris.
Please consult your physician to prescribe the dosage that best suits the condition.
Available as drops.
Liv.52 is not known to have any side effects if taken as per the prescribed dosage.
For information on clinical research Click here
The information on this page is not intended to be a substitute for professional medical advice. Do not use this information to diagnose or treat your problem without consulting your doctor.
Labels: Fatty Liver, liv 52
Natural Herbal Immune enhancing Germ Killers , Antibiotics.
Builds the body's own defense mechanism
Enhances immunity: Septilin's immunomodulatory, antioxidant, anti-inflammatory and antimicrobial properties are beneficial in maintaining general well-being. It increases the level of antibody-forming cells, thereby elevating the body's resistance to infection. Septilin stimulates phagocytosis (elimination of bacteria through ingestion) by macrophage (white blood cells) activation, which combats infection.
Other benefits: Septilin possesses antipyretic (reduces fever) properties. It is also beneficial in respiratory tract infections, including chronic tonsillitis, pharyngitis, chronic bronchitis, nasal catarrh (mucous membrane inflammation of the respiratory tract) and laryngitis.
As an immunomodulator in the management of upper and lower respiratory tract infections, allergic disorders of the upper respiratory tract, skin and soft tissue infections, dental and periodontal infections, ocular infections, bone and joint infections and urinary tract infections
For early recovery in postoperative conditions
To reduce recurrence in infection-prone individuals
As an adjuvant to anti-infective therapy
Resistance to antibiotic therapy
Tinospora Gulancha (Guduchi) is a potent antimicrobial that has immunostimulatory properties, which helps in increasing the level of antibodies. This helps in building up the body's resistance to infections.
Licorice (Yashtimadhu) enhances immunostimulation and increases macrophage (white blood cells that ingests antibodies) function in vitro. It is also an antiviral agent and an expectorant, which is beneficial in asthma, acute or chronic bronchitis and chronic cough.
Indian Bdellium (Guggulu) has anti-inflammatory properties, which soothe sore throat and help reduce inflammation. As an antioxidant, Indian Bdellium helps in maintaining overall health.
Available as tablets or as a syrup.
Septilin is not known to have any side effects if taken as per the prescribed dosage.
From http://www.himalayawellness.com
Labels: natural antibiotic, septilin
High Fructose Syrup and Diabetes-A study-STOP all ...
Natural Herbal Immune enhancing Germ Killers , Ant...


We appreciate so much the faithfulness and confidence our friends put in us by giving to the college through the Sustainer's Club. As the college expands, these gifts are more and more appreciated. The Sustainer's Club is a group of very special friends, and we really could not make it financially without them.
The goal of the Club is to support our scholarship fund for missionary kids, foreign students, and bus kids from churches across the country and world. Although you may not have been able to be a Sustainer in the past, many of you would particularly like to help these types of students in the college.
In order to allow more to support the work of the college, we have created several different levels.


A Chicago-based trio with rock energy dedicated to the fearless exploration of group improvisation. Featuring Chris Weller on saxophone, Cole DeGenova on keys, and Devin Drobka on drums.
Thank you for taking the time to visit our page on ScratchBang! We are humbled and extremely greatful for the support we recieve here. A lot of time and money goes into composing new music, rehearsing (living in different cities), recording, mixing, mastering, the pressing of CDs and LPs, booking shows, promoting shows, touring, and everything else that contributes to getting our original music to your ears. Thank you for helping us share our art with the world!
Hanging Hearts is a Chicago-based trio with rock energy dedicated to the fearless exploration of group improvisation. Described by critics as "hammering the walls between genres," their music bridges the gap between jazz, rock, and experimental music. The band features Chris Weller on tenor saxophone, Cole DeGenova on keyboards, and Devin Drobka on drums.
Recorded March 14, 2015. Composition by Devin Drobka.
Thank you for supporting artists through Scratchbang and taking the time to visit our page! The process of creating new music, recording it, mastering it, pressing it into CD's, booking shows, promoting shows, and touring internationally is an expensive one. We appreciate all of our backers (YOU) and the opportunity to continue creating and spreading our art around the world.


text:
Siebershof war ein Wohnplatz im Ortsteil Greiffenberg der Stadt Angermünde im Landkreis Uckermark (Brandenburg). Er wurde 1825 aufgebaut und wurde wahrscheinlich erst nach dem Zweiten Weltkrieg abgerissen. Die ursprüngliche Schreibweise Sibershof änderte sich im Laufe des 19. Jahrhunderts zu Siebershof.

Lage
Siebershof lag ca. 1,5 km südwestlich des Rittergutes Peetzig und 3,5 km südöstlich von Steinhöfel und ca. 5 km südwestlich von Greiffenberg. 400 Meter östlich liegt der Kleine Peetzigsee, etwa 300 Meter südöstlich der kleine Faule See. Der Wohnplatz lag auf .

Geschichte
Das im Hochmittelalter entstandene Dorf Peetzig fiel im 15. Jahrhundert wüst. Ende des 16. Jahrhunderts wurde zunächst eine Schäferei, dann auch ein Vorwerk auf der Feldmark aufgebaut. Im Dreißigjährigen Krieg wurden Schäferei und Vorwerk zerstört. Bis 1705 war wieder ein Vorwerk auf der Feldmark Peetzig aufgebaut worden, das Rittergutsqualität erhielt.

1818 verkaufte der von Karl Friedrich von Wedel-Parlow (1765–1832) Landrat des Kreises Angermünde das Rittergut Peetzig an Carl Ernst Ludwig Rehfeldt und Friedrich Wilhelm Siber. Siber war mit der Schwester von Rehfeldt, Johanna Ernestina Wilhelmina Rehfeldt, verheiratet. Von 1819 bis 1825 besaßen Friedrich Wilhelm Siber und der Carl Ernst Ludwig Rehfeld(t) gemeinsam das Rittergut Peetzig. 1825 teilten sie die Gemarkung. Rehfeld behielt das Rittergut Peetzig und die östliche Hälfte der Feldmark Peetzig. Friedrich Wilhelm Siber errichtete auf seiner, der westlichen Hälfte der Feldmark Peetzig ein neues Vorwerk. 1826 heißt es im Amtsblatt der Königlichen Regierung zu Potsdam: Dem auf der getheilten Feldmark des Rittervorwerks Peetzig bei Greifenberg im Angermündeschen Kreise neu erbauten Vorwerke, ist die Benennung Sibershof beigelegt worden. Siber besaß das Gut noch bis 1834. Von 1834 bis 1835 war es im Besitz des Ernst Wilhelm Hoefer. 1835 bis 1840 soll es der Ökonom Winterfeldt besessen haben. Nach der Topographie der Untergerichte der Kurmark Brandenburg von 1837 gehörte der Sibershof zu dieser Zeit jedoch dem Ritterschaftsrat Albrecht Otto von Wedell-Parlow zu Neu-Günterberg. 1840 bestand das Gehöft Sibershof aus vier Wohnhäusern und einer nicht genannten Anzahl von Wirtschaftsgebäuden. Der Wohnplatz hatte damals immerhin 52 Einwohner. 1840 bis 1841 war es dann im Besitz des Georg Carl von Scheven, der es an einen Herrn Büring verkaufte. Auch Büring behielt das Vorwerk nicht lange und verkaufte es 1845 an Graf Carl von Moltke, der es bis 1852 behielt.

Dann wechselte es in den Besitz des Grafen Friedrich Wilhelm von Redern (1802–1883) auf Görlsdorf und Greiffenberg. 1861 bestand der Sibershof aus drei Wohngebäuden und sieben Wirtschaftsgebäuden und hatte 56 Einwohner. Insgesamt war das Gut 1583 Morgen groß, davon entfielen 17 Morgen auf das Gehöft selber, 1400 Morgen waren Acker, 126 Morgen Wiesen und 40 Morgen Wald. Der Tierbestand ist mit 12 Pferden, 23 Stück Rindvieh und 500 Schafen angegeben. Siebershof gehörte damals zum Schlossgut Greiffenberg. Adolf Frantz gibt dagegen für 1863 die Gesamtgröße mit 1631 Morgen an, davon 1543 Morgen Acker, 126 Morgen Wiese und 63 Morgen Forst. 1871 war Siebershof ein eigener Gutsbezirk mit drei Wohngebäuden und 43 Einwohnern. 1874 wurde der Gutsbezirk Siebershof dem Amtsbezirk 20 Görlsdorf des Kreises Angermünde zugewiesen. Das General-Adressbuch der Ritterguts- und Gutsbesitzer im Deutschen Reiche von 1879 nannte erstmals Hektarzahlen: Gesamtgröße 388,83 ha, davon 343,70 ha Acker, 32,29 ha Wiesen, 7,28 ha Weiden und 5,56 ha Wasser. Der Graf von Redern hatte das Gut an einen Oberamtmann Bündiger verpachtet. Der Grundsteuerreinertrag betrug 2868 Mark. 1884 war Sieberthof an den Grafen Heinrich von Redern (1804–1888) übergegangen. Er hatte die beiden Güter Peetzig und Siebershof an den Amtmann August Kayser verpachtet. Die Gesamtgröße war nun mit 412 ha angegeben, davon 357 ha Acker, 33 ha Wiesen, 7 ha Weiden und 15 ha Wasser. Der Grundsteuerreinertrag betrug 2865 Mark. 1888 verstarb Graf Heinrich von Redern; ihm folgte sein Sohn Wilhelm Heinrich (1842–1909) nach. 1896 hatte das Vorwerk Siebershof nur noch 9 Einwohner. 1900 war nur noch ein Wohnhaus vorhanden. 1896 und 1903 waren die beiden Güter Peetzig und Sieberthof an Odo Bartels verpachtet. 1907 war der Siebershof mit dem Gut Peetzig zusammen gelegt worden. Pächter war Odo Bartels, der auch das Dampfsägewerk Marienmühle betrieb. Die Marienmühle lag beim Bahnhof Greiffenberg.

Nach dem Tod des Wilhelm Heinrich 1909 folgte ihm sein Sohn Wilhelm von Redern (1888–1914 gef. bei Ypern) als Besitzer nach. 1914 hatte der Graf Redern die Landwirtschaft der beiden Güter Peetzig und Siebershof Julius Schultze zur Administration übertragen, den Betrieb des Dampfsägemühlenbetriebs Reinhold Newrzella. Letzterer war auch für die Forsten des Grafen Wilhelm Heinrich von Redern zuständig.

1921 und 1929 war Victoria Marie Fürstin zu Lynar Gräfin von Redern (1889–1981), Tochter des Grafen Wilhelm Heinrich von Redern (1842–1909) und der Marie Caroline Gräfin Lichnowski (1861–1933) und verheiratet mit Ernst Fürst zu Lynar (1875–1934), Besitzerin des riesigen Grundbesitzes der von Redern im Barnim und der Uckermark, und damit auch der Güter Peetzig und Siebershof. Die Landwirtschaft der beiden Güter wurde 1923 und 1929 von Ernst Holzkamm administriert. Für das Dampfsägewerk war 1923 weiterhin Reinhold Newrzella zuständig. 1929 hatte Ferdinand Marquardt die Leitung des Sägewerkes übernommen.

1928 wurde der Gutsbezirk Siebershof mit der Gemeinde Greiffenberg vereinigt. Der westliche Teil des Gutsbezirks kam an die neugebildete Gemeinde Neuhaus. 1931 war Siebershof ein Wohnplatz der Gemeinde Greiffenberg. Wann der Siebershof schließlich abgerissen wurde, ließ sich bisher nicht ermitteln, wahrscheinlich aber erst nach dem Zweiten Weltkrieg.

Einzelnachweise

Geographie (Angermünde)
Wüstung im Landkreis Uckermark

redpajama_set_name: RedPajamaWikipedia


text:
Q: ad hoc scanner just wont work So far i have this code that is suppose to be an ad hoc scanner, im not sure where exactly the problem is because i get a very odd error in the output.txt file. it all starts when the program hits the left parenthesis, is there somthing wrong with my checkforoperators function? i have looked but have found little and have even asked others, but no reall luck as to what the problem is.
the input file contains this
read A1
read A2
sum:=(A1-33)+A2*5.5
write sum
write (A1+A2)/2

and the output file is SUPPOSE to look like this
read
A1
read
A2
Sum
:=
(
A1
-
33
)
+
A2
*
5.5
write
sum
write
(
A1
+
A2
)
/
2

heres the main file
#include <iostream>
#include <string>
#include <fstream>
using namespace std;

// This program is an ad-hoc scanner.
// It will recognize tokens and seperate them.
// this language will NOT DISTINGUISH between uppercase or lowercase.

    void stripSpacing(string &);
    void checkForCommands();
    void checkForLetters();
    void checkForOperators();
    void checkForAssignment();
    void checkForDecimal();
    void checkForDigit();

    int x1 = 0;
    int x2 = 0;
    int vars[10];
    // Only for demonstration purposes, we could use a dynamic array for larger purposes.

    string holder;
    // used to hold the current string before processing.

    fstream handling;
    // file handling from other libraries.
    fstream output;

    char temp1[1];
    char temp2[1];
    int x = 0;
    int xtemp = 0;

int main()
{
    output.open("output.txt");
    handling.open("input.txt");

    if (handling.is_open())
    {
        while (!handling.eof())
        {
            dcom:
            getline (handling,holder);
            if(holder[x] == '/' && holder[x+1] == '/')
            {
                cout << "This line has only comments.... REMOVED :)" << endl;
                //do nothing :)
                goto dcom;
            }
            cout << endl;
            stripSpacing(holder);
            cout << "The line is this long after removing spaces: " << holder.length() << endl;
            cout << "The line contains: " << holder << endl;
            cout << endl;

            while(holder.length() != x)
            {
                // let's check for commands, such as read write or sum.
                checkForOperators();
                checkForAssignment();
                checkForDecimal();
                checkForDigit();
                checkForCommands();
                checkForLetters();
            }
            output << "\n";

            x=0;
        }

    handling.close();
    output.close();
    return 0;
    }
}

void stripSpacing(string &str)
{
    for (int i=0; i<str.length(); i++)
        if (str[i]==' ')
        {
            str.erase(i,1);
            i--;
        }
}
void checkForOperators()
{
    if(holder[x] == '(' || holder[x] == ')'|| holder[x] == '+' || holder[x] == '-' || holder[x] == '*' ||holder[x] == '/')
    {
        output << holder[x] + "\n";
        x++;
    }
    cout << "checkForOpertors" << endl;
}

void checkForCommands()
{
    xtemp = x;
    if(holder[x] == 'w')
    {
        x++;
        if(holder[x] == 'r')
        {
            x++;
            if(holder[x] == 'i')
            {
                x++;
                if(holder[x] == 't')
                {
                    x++;
                    if(holder[x] == 'e')
                    {
                        x++;
                        output << "write\n"; goto stop;
                    }else{x=xtemp; goto stop;}
                }else{x=xtemp; goto stop;}
            }else{x=xtemp; goto stop;}
        }else{x=xtemp; goto stop;}
    }

    if(holder[x] == 'r')
    {
        x++;
        if(holder[x] == 'e')
        {
            x++;
            if(holder[x] == 'a')
            {
                x++;
                if(holder[x] == 'd')
                {
                    x++;
                    output << "read\n"; goto stop;
                }else{x=xtemp; goto stop;}
            }else{x=xtemp; goto stop;}
        }else{x=xtemp; goto stop;}
    }

    if(holder[x] == 's')
    {
        x++;
        if(holder[x] == 'u')
        {
            x++;
            if(holder[x] == 'm')
            {
                x++;
                output << "sum\n"; goto stop;
            }else{x=xtemp; goto stop;}
        }else{x=xtemp; goto stop;}
    }

    stop:
    cout << "checkForCommand" << endl;
}

void checkForLetters()
{
    if(isalpha(holder[x]))
    {
        output << holder[x];
        x++;
    }
    cout << "checkForLetters" << endl;
}

void checkForAssignment()
{
    if(holder[x] == ':')
    {
        x++;
        if(holder[x] == '=')
        {
            output << ":=\n";
            x++;
        }
        else
        {
            cout << "ERROR!!! NO : BEFORE =" << endl;
        }
    }
    cout << "checkForAssign" << endl;
}

void checkForDecimal()
{
    if(holder[x] == '.')
    {
        x++;
        if(isdigit(holder[x]))
        {
            output << '.';
            x--;
        }
    }
    cout << "checkForDeci" << endl;
}

void checkForDigit()
{
    if(isdigit(holder[x]))
    {
        output << holder[x];
        x++;
    }
    cout << "checkForDig" << endl;
}


My parents immigrated to the United States believing in the American dream. They came over with a little more than hope, optimism and determination. Once they settled into the Atlanta area, they spent their money wisely, worked inordinate hours and taught all four sons to give their best in every effort. Dr. John shares, "Their guidance certainly influences how I treat my patients. It's important to know my patients and their satisfaction is what matters. In fact, my team and I strive to do more than please our patients. We want to go beyond their expectations and earn their trust and friendship. Consideration is of high value in my family. Consideration begins with respecting a patient's concerns and desires and making sure the dental care we provide is the best available to them. That's what drives us.
After receiving his undergraduate degree from the Medical College of Georgia, Dr. Ananthasane continued his studies at Boston University receiving his degree as a Doctor of Dental Medicine (General Dentist). Knowing the field of Periodontics was advancing and offering phenomenal options to patients with cosmetic dental needs, Dr. John continued his studies at the Medical College of Georgia where he received his Masters in Oral Biology and completed MCG's Periodontal Residency Program. Upon finishing this 3 year program, he fulfilled the requirements becoming Board Certified in Periodontics.


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July 18, 2019 – Welcome to "This Week in Blood," a weekly snapshot of the hottest studies from each week's issue of Blood, the official journal of the American Society of Hematology (ASH), hand-picked by Blood Editor-in-Chief Bob Löwenberg, MD, PhD, and Deputy Editor Nancy Berliner, MD.
Cover Figure: Vitamin K antagonist impairs hematopoiesis and stem cell function. See the article by Verma et al.
R-CHOP preceded by blood‐brain barrier permeabilization with engineered tumor necrosis factor-α in primary CNS lymphoma
The investigators report the feasibility and safety of using engineered tumor necrosis factor-a to increase blood‐brain barrier permeability in patients with relapsed/refractory primary central nervous system (CNS) lymphoma. The drug targets CD13 on tumor blood vessels and increases permeability of the blood‐brain barrier, improving the radiographic response to R-CHOP (rituximab, cyclophosphamide, doxorubicin, vincristine, and prednisone) chemotherapy.
Plasmin-mediated fibrinolysis enables macrophage migration in a murine model of inflammation
Plasminogen is necessary for macrophage migration. This study identifies a critical role of fibrinolysis in macrophage migration, presumably through the alleviation of migratory constraints imposed by the interaction of leukocytes with fibrin(ogen) through the integrin αΜβ2 receptor.
EFL1 mutations impair eIF6 release to cause Shwachman-Diamond syndrome
This work describes the identification of loss-of-function germline mutations in EFL1 that account for a subset of "gene‐negative" Shwachman-Diamond syndrome and explores the mechanistic link between mutations and disease pathogenesis using multiple complementary model systems.
Benefits and challenges with diagnosing chronic and late acute GVHD in children using the NIH consensus criteria
In a prospective multicenter trial described in this month's CME article, the investigators evaluated for the first time the performance of the National Institutes of Health (NIH) consensus criteria for the diagnosis of chronic graft-versus-host disease (GVHD) in pediatric patients.
Hepatic leukemia factor is a novel leukemic stem cell regulator in DNMT3A, NPM1, and FLT3-ITD triple-mutated AML
The investigators explore the molecular interaction between mutations affecting the genes DNMT3A and NPM1 and internal tandem duplications (ITDs) of FLT3 in acute myeloid leukemia (AML). They show that the 3 co‐occurring mutations synergize to drive expression of the transcription factor HLF (hepatic leukemia factor), and establish HLF as a novel leukemic stem cell regulator that mediates the adverse phenotype in this high-risk AML genotype.
Vitamin K antagonism impairs the bone marrow microenvironment and hematopoiesis
This study provides direct evidence that the widely used vitamin K antagonist warfarin impairs hematopoiesis and hematopoietic stem cell function in both mice and humans.
The enhancer RNA ARIEL activates the oncogenic transcriptional program in T-cell acute lymphoblastic leukemia
This work demonstrates that ARID5B-inducing enhancer associated long noncoding RNA (ARIEL) is a novel enhancer RNA that amplifies oncogenic transcriptional loops in a subset of T-cell acute lymphoblastic leukemia.
View this week's complete table of contents
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Blood (www.bloodjournal.org), the most cited peer-reviewed publication in the field of hematology, is available weekly in print and online. Blood is the official journal of the American Society of Hematology (ASH) (www.hematology.org), the world's largest professional society concerned with the causes and treatment of blood disorders.
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