text:
Film about world's oldest known Holocaust survivor Alice Herz-Sommer wins Oscar a week after her death
By Tim Lamden
Alice Herz-Sommer. Picture: Polly Hancock. - Credit: Archant
A documentary about the world's oldest known Holocaust survivor who died a week ago, aged 110, was among the Oscar winners at the 86th Academy Awards.
Filmmakers Malcolm Clarke (left) and Nick Reed in the press room with their Oscar awards for The Lady In Number 6: Music Saved My Life. Picture: PA Wire/Ian West. - Credit: PA Wire/Press Association Images
The Lady In Number 6: Music Saved My Life, exploring the life of late Belsize Park resident Alice Herz-Sommer, beat off competition from four other films to win the Oscar in the Documentary Short Subject category at last night's glitzy awards ceremony in Los Angeles.
Filmmakers Malcolm Clarke and Nick Reed picked up the award exactly a week after the death of Ms Herz-Sommer, of Belsize Grove.
Accepting the award, director Mr Clarke, who now lives in Canada but learned his trade at the BBC and Granada TV, dedicated the win to Ms Herz-Sommer's "extraordinary capacity for joy and her amazing capacity for forgiveness".
Ms Herz-Sommer spent two years with her young son Stephan in Theresienstadt concentration camp in the city of Terezin, located in what is now the Czech Republic, during the Second World War.
As an accomplished pianist, she managed to avoid deportation with her son to Auschwitz through her performances in numerous concerts during her time in Theresienstadt.
Survivors who witnessed the concerts recounted how the beauty of Ms Herz-Sommer's playing transported them away from the horrors of their daily life and offered them hope.
Ms Herz-Sommer shielded Stephan from the horrors of Theresienstadt for two years before being liberated by the Russians in 1945.
The film will now be available to audiences in the USA and Canada via the Netflix internet-streaming service as part of a distribution deal struck between Netflix and the film's producers.
Negotiations are underway to bring the film to a UK audience on Netflix.
Mr Reed, who co-produced the film with Mr Clarke, said in a statement: "Telling Alice's story was a life-changing experience for everyone involved with the film.
"We are so glad to partner with Netflix so that their viewers will be able to enjoy Alice's infectious love of life and bright spirit.
"Netflix viewers and future generations will be able to learn so many life lessons from this most amazing woman."


NORD gratefully acknowledges Maral Karalekian, NORD Editorial Intern from the Keck Graduate Institute, and Bernardo Vargas, MD, Department of Orthopedic Surgery, University Hospital of Geneva, Switzerland, for assistance in the preparation of this report.
Kohler disease is a rare bone disorder of the foot in children that may be the result of stress-related compression at a critical time during the period of growth. It is characterized by limping caused by pain and swelling in the foot. It most often occurs in children between the ages 3-7, and affects males five times more often than females. Usually, just one foot is affected so children typically walk on the side portion of the foot.
Children appear to grow out of the disorder, and the affected bones usually regain their size, density and structure within three months. Rarely, symptoms may last as long as two years.
Kohler disease is a rare bone disorder characterized by a painful swollen foot. The foot is especially tender along the length of the arch. It may include redness of the affected area. Putting weight on the foot or walking is difficult, causing further discomfort and a limp. For reasons that are not understood, the flow of blood to one of the bones in the foot (navicular bone) is interrupted, resulting in progressive degeneration of that bone. In a relatively short time, however, the bone heals itself.
Usually, symptoms will be mild, and patients may not seek treatment until the pain and swelling have persisted for a while.
The exact cause of Kohler disease is unknown.
Some orthopedic specialists think that Kohler disease may be related to an injury in the area around the navicular bone in the foot and may be the result of delayed bone formation (ossification). Bone ossification usually begins at age 18-24 months in girls and at age 24 to 30 months in boys. Structural weakness might result from an increase in the ratio of cartilage to bone. Since the navicular bone is part of the mechanism by which the foot moves (articulation), it is subject to weight-bearing pressures and stresses from twisting and turning.
Under normal circumstances, the navicular bone is served by a blood vessel from which smaller arteries supply blood to the regions of bone growth. At around the ages of 4-6, the blood supply to these regions of bone growth increases as other blood vessels reach them. If ossification is delayed and the child gains weight, the effect is to compress the blood vessels, thus causing tissue destruction (ischemia).
It has been suggested that genetic factors may play a role in the development of Kohler disease, but a specific gene has not been found to be associated with this disease.
Kohler disease is a rare bone disorder of the foot that affects males more often than females. The disorder strikes children between the ages of 1 and 10 years with a peak occurring at ages 3 to 7 years. The center of bone growth that is affected in Kohler disease develops in young girls about one year before it appears in young boys. Nevertheless, the disorder is five times more prevalent in boys than girls.
The incidence of the disorder in the population is estimated to be less than 2%.
Grierson-Gopalan syndrome is characterized by burning sensations in the feet, and aching and cramp-like pains in the soles of the feet (and possibly palms of the hands. Patients often experience a sensation like pins and needles in the feet. This condition can be related to a vitamin B deficiency.
A diagnosis of Kohler disease is suspected based on the presence of the signs of symptoms in the child. The clinical diagnosis of Kohler disease is reinforced by X-rays that show flattening, sclerosis, and fragmentation of the navicular bone, which can be compared to the unaffected side to make the diagnosis. However, these abnormalities of the navicular bone are bilateral in approximately 25% of patients and may be seen in children who do not have Kohler disease.
Kohler disease typically resolves over time with or without treatment. Symptoms can last for a few days or persist for up to two years, but symptoms usually resolve within six months. Treatment can include pain relievers or weight-bearing short-leg plaster casts. Special supportive shoes may also be considered. Staying off the foot as much as possible helps in recovery. The long-term outlook (prognosis) for people with Kohler disease is usually excellent. People affected by the condition typically recover all function of the affected foot and have no lasting consequences.
Cook RA, O'Malley MJ. Navicular Osteochondritis (Kohler Disease). In: NORD Guide to Rare Disorders. Lippincott Williams & Wilkins. Philadelphia, PA. 2003:21.
Myerson MS. Foot and Ankle Disorders. WB Saunders Co., Philadelphia, PA. 2000:793-96.
Tsirikos AI, Riddle EC, Kruse R. Bilateral Kohler's disease in identical twins. Clin Orthop. 2002;409:195-98.
Harty MP. Imaging of pediatric foot disorders. Radiol Clin North Am. 2001;39:733-48.
Toth AP, Easley ME. Ankle chondral injuries and repair. Foot Ankle Clin. 2000;5:119-33.
Bui-Mansfield LT, Lenchik L, Rogers LF, et al. Osteochondritis dissecans of the tarsal navicular bone: Imaging findings in four patients. J Comput Assist Tomogr. 2000;24:744-47.
Gips S, Ruchman RB, Groshar D. Bone imaging in Kohler's disease. Clin Nucl Med. 1997;22:636-37.
Devine KM, Van Demark RE Sr. Kohler's osteochondrosis of the tarsal navicular: case report with twenty-eight year follow-up. S D J Med.1989;42(9): 5-6.
Ippolito E, et al. Kohler's disease of the tarsal navicular: long-term follow-up of 12 cases. J Pediatr Orthop.1984;4(4):416-417.
Williams GA, Cowell HR. Kohler's disease of the tarsal navicular. Clin Orthrop. 1981;158:53-58.
www.emedicine.com/orthoped/topic410.htm Updated: Dec 18, 2017. Accessed November 7, 2018.
Kohler Disease. Genetic and Rare Diseases Information Center. https://rarediseases.info.nih.gov/diseases/6842/kohler-disease Last updated: 2/18/2014. Accessed November 7, 2018.


text:
Sweden, Turkey
Turkey is once again jeopardising Swedish accession to NATO over extradition of individuals linked to the Gülen movement from Sweden. On Wednesday Hulusi Akar, the Turkish defence minister stated that "There are commitments signed by Sweden and Finland … they must be fulfilled … the memorandum is a beginning, not an end. After these are done, the Turkish parliament will make its decision [on NATO accession]. We are also trying to help Sweden and Finland".
Mr Akar's comments follow the Swedish government's decision not to extradite to Turkey four individuals linked to the US based cleric Fethullah Gülen, after their removal was blocked by the Swedish supreme court in July last year. A memorandum signed on 28 June 2022 between the two countries agreed that Turkey would remove its veto on Sweden and Finland's accession to NATO on the basis that both countries "address Turkiye's pending deportation or extradition requests of terror suspects expeditiously and thoroughly".
Since then, the Swedish supreme court has blocked the extradition of four individuals in July 2022 and of Turkish journalist Bulent Kenes in December. The Swedish government is legally bound to follow the decision of the court on these matters. Turkey argues that this is in breach of the memorandum and so have threatened to once again block the country from joining NATO.
The Swedish Prime Minister Ulf Kristersson responded bullishly on Sunday, saying that "Turkey confirms that we have done what we said we would do, but also says that they want things that we cannot or do not want to give them". However, he tempered his comments on Wednesday claiming his previous statement had been misunderstood and that "We are doing exactly what we promised to do, that is not least in the field of fighting terrorism …. I think that has been one of the core tasks — to strengthen the Swedish legislation on counterterrorism, recognizing the fact that activities on Swedish soil can be dangerous for other countries, can be aimed at others countries, and also recognizing the fact that Turkey has been one of the countries hurt the most by terrorism."
Many Turkish extradition requests for people linked to Fetullah Gülen have been dismissed as politically motivated in foreign courts. While developments since the MOU was signed may have allayed some concerns that Ankara would prioritise its domestic political motives over Western security arrangements, the continued threat to Swedish NATO membership by the Turkish government keeps this risk very much alive.
Tags: Abuse of process, Extradition agreement, NATO Categories: Sweden, Turkey

redpajama_set_name: RedPajamaCommonCrawl


Just came back from my volunteer work. Feeling the ache in my neck after sitting on a stool chair for 4 hours straight. I don't do much other than observing in the clinic for a bit and then filling the rest of the time but I always feel super tired after I'm done.
Two weeks ago, I did so much shopping. I need to watch when I splurge. Though fortunately, I don't spend as much as other people do because I don't have that much money, haha. Hopefully, I'll get the chance to take a picture of them to show you but it's so hard taking self-photos. -_- I try to use the timer but it just makes the photo blurry. I don't know how to fix it.
I will blog a little more next time. Haven't really been in the mood and I want to keep this blog mostly just related to art and fashion. (You may have noticed that I deleted all my previous posts. Still working on the boutique. So difficult squeezing time in because of work and school.


On today's episode, neuroscientist and virtual reality researcher, Dr. Mar Gonzalez Franco, talks about her work in VR, explains how avatars can help increase our empathy and reduce our biases via role play, and addresses the misconceptions that exist between the immersive experiences of virtual reality and psychedelic drugs.
Mar Gonzalez Franco: Imagine you enter this new virtual body that you look down and your hands are these other hands. They move as you move perfectly, only you're a different race, or a different gender. You can experience the life perspective of a different person from within.
Host: You're listening to the Microsoft Research Podcast, a show that brings you closer to the cutting edge of technology research and the scientists behind it. I'm your host, Gretchen Huizinga. Today, we're here with neuroscientist and Microsoft researcher Dr. Mar Gonzalez Franco. She'll discuss her research in virtual reality, the importance of avatars, and she'll clear up the misconceptions between VR and psychedelic drugs. That, and much more, on this episode of the Microsoft Research Podcast. Mar, you have a really interesting background. How did you end up doing research at Microsoft?
Mar Gonzalez Franco: I think in life, there are sort of cycles that you end up doing the same thing again. Like I started training as a computer scientist, mathematician, and then I became very interested by neuroscience. And then I came back to computer science. It kind of comes together. I would say there is always not a single road to get somewhere. And definitely you need to take it step by step. So I wouldn't be here if I didn't do my PhD with Mel Slater, and partially at MIT for sure. I mean, these – Microsoft Research really scouts for talent and in a wide spectrum, on a global scale. And you have to be there. And there is a part of being lucky to be in a very hot spot that is of interest to the company in a very specific moment.
Host: You told me that VR can trick the brain.
Host: And that people know it's fake, but they have a strong experience of reality anyway. What's good about that, and what's bad about that, and what do we take away from that? It kind of scares me, but it's kind of cool.
Mar Gonzalez Franco: Yeah, it's very interesting, because we are providing a real-time stream of information to our senses. Mainly virtual reality has been based on vision, and humans are very visual animals. We believe what we see. And virtual reality provides that. It provides our congruent sensory motor stimulation. One of the experiments I did, you put on some sort of glasses – an HMD. With virtual reality, you put your hand on top of a table, and you see a virtual hand instead of your hand. And that hand gets attacked. And what I find is that the motor cortex gets activated in the same way that it would be activated for a real attack to your real hand. You really perceive this as your hand. I think this is very interesting, because it's not only that you can enter a virtual reality. You can change your body in that reality.
Host: What are the implications of what you just said in terms of application? Outside the fact that you've observed that you can make people believe that a virtual hand is actually their hand?
Mar Gonzalez Franco: Some very interesting applications have to do with empathy. Imagine you enter this new virtual body that you look down and your hands are these other hands. They move as you move perfectly, only you're a different race, or a different gender. You can experience the life perspective of a different person from within. Philosophers and psychologists have explored this role play. And this is role play to the maximum exponential. You're basically within a different body.
Host: You're talking about avatars now?
Host: Whether it's just a hand avatar or a full body avatar. And that's the main component of virtual reality?
Mar Gonzalez Franco: I think avatars are becoming more important than ever with virtual reality. We experience reality and people experience ourselves through our bodies. It's very easy to evaluate things from the perspective of our own body. And in virtual reality, we need to have a body, an avatar that represents us.
Host: If we can have realistic experiences through avatars, what other things might we be able to do through VR? How else might it help us?
Mar Gonzalez Franco: This is a very important topic in my research, the avatar that you embody. If you embody an avatar that is of a different race, you can reduce your racial bias. And this is some experiments that have already been done. There are other experiments that have shown that if you embody an avatar that is older, so it looks like you – it's a lookalike avatar, just a little bit older – your financial decisions after you come out become more conservative. You're more likely to save money. And not only this, even more traditional fears, like fear of heights, fear of flying – virtual reality provides a very controlled platform to perform therapy interventions. And during an intervention, people may develop a crisis, and you might want to take them out. In virtual reality, it's simply a headset you remove it. You're in an airplane; you're out from an airplane. If you want to treat somebody who has a fear of flight, who has this problem, by putting them inside a real plane and flying them to New York, they might develop the crisis in the middle of the flight, and there is no way for you to stop it. In that scenario, it's much more secure. So for anybody who is treating these kind of phobias, virtual reality is clearly a very good option.
Host: Is this happening already, or is this just something that you're looking at one of the possible applications for VR in the future?
Mar Gonzalez Franco: No, people are already using it. Of course, there are only a few people that have these really, really extreme phobias that cannot be treated in a less immersive way. You could treat your phobias by going in the real world rather than in the virtual world. But it also allows you to treat your phobias from home. You have a device at home. You don't need to go to a center to be exposed to this therapy. So it also democratizes a lot, these sort of treatments or interventions.
Host: Let's talk about the illusory nature of VR for a bit. I read an article where the headline said, and I quote, "Microsoft says virtual reality could make you hallucinate in the same way as LSD." That was a rather sensational version of what you actually said, I think. But can you talk a bit about how your work in VR is like and even unlike hallucinogenic drugs?
Mar Gonzalez Franco: When Jaron Lanier started the first virtual reality company, he was faced all the time like, oh, this going to be like an LSD trip? And of course the answer is no. But I'm going to go a bit more in detail, because I do think it's legitimate for people to be concerned about how a technology is going to affect their life. Virtual reality, in the best form, provides a very strong illusory setup to create illusions. Illusions such as there is a glass on top of this table. But in reality, there is no such thing. With the reality, it has the same power. But this is an illusion in which you never lose your higher cognitive functioning. There is no chemical alteration of your brain. It's purely sensorial. You're seeing this glass of water. It looks super realistic. And you move around, and the glass is staying there. So at the end, it really feels like there is something there. And – but you take off the glasses, and there is nothing there. Compared to drugs, this person has never lost their higher cognitive function during this illusory state. And that changes completely the topic. Because higher cognitive function is the one that allows you to make conscious decisions. Decision-making processes, what's right, what's wrong. And because that's never altered, you're always aware that this is not real. Versus when people do drugs, that's completely lost. So I say that these are very 2 different ways of experiencing reality.
Host: You're really interested in the societal impacts of virtual reality. What are the best things about your work in VR?
Mar Gonzalez Franco: First of all, this democratization of the technology arriving people who have never been able to own any previous technological advancement. And now all of a sudden, they have something that includes them all. It's like one ring to rule them all. And augmented reality and virtual reality are getting there. In fact, there is still a bit the fight on whether it's going to take the form of a phone that you put on top, or it's going to be a completely new device that you can render a phone in it. But it's true that it's going to be mobile in the sense that the battery, it's going to be there. And you're going to put it on, and render from first-person perspective. So that's very exciting, because it's going to have very big implications on society. It's going to change many things of how we live.
Host: Give me an example of what you think that might be, one thing that could help us.
Mar Gonzalez Franco: Yeah, I think in particular this tendency we have to buy so many gadgets. That might actually go away, and we have one single gadget. This has not happened with any other device. Even we have tablets now, we still have a laptop, or we work on a desktop. If we're talking about a device that is going to be able to embed them all, you're really going to not have a desktop afterwards. You're just going to have this device. That's because you can render a computer in it, or you can render a tablet. Or you can render everything.
Host: Explain to me render. How does that work in virtual reality?
Mar Gonzalez Franco: In virtual reality, you have a display, a near-eye display, because it's like a glass. So inside these glasses, you digitally print an image, just like you would do in a regular screen. It's just that this screen, it's always available for you. So inside this screen, you can print different things. Printing in digital is usually referred as rendering.
Host: What I hear you talking about is like a Google Glass kind of thing.
Mar Gonzalez Franco: It's very different, because Google Glass was an annotation device. It would provide you information from the top, which is not blended with reality. This is not an annotation technique. This is a very different technological system. And this is with occurring with hollow lens. I can only imagine when we move forward, it's something more like – instead of having a computer at home, or a TV, I have – my glasses know that this – this wall has a TV on it, and when I come home, the TV is there. I don't need to put it back.
Host: But you're looking through a device, yes?
Mar Gonzalez Franco: You're looking through a device, like glasses, normal glasses.
Host: So does everyone in your family have to have them? Say like you have a family of 5. Everyone has to have the glasses in order to see the TV? The virtual TV?
Mar Gonzalez Franco: We're working on this. Because it's interesting to explore what each person will see. Because also using these glasses, I could do something very unique, which is seeing what you're seeing. We're seeing one in front of each other, and we have a chart in front of us, you're seeing one perspective of the chart, and I'm seeing another one. What if we both could see exactly the same thing despite being in different positions? I mean, we start a very interesting topic of perspective.
Host: Wow, my mind is just reeling with what you're talking about, and trying to imagine. Because my paradigms are so "right now," right? My way of writing is using a keyboard and a screen. But you're suggesting a whole new paradigm that we don't even imagine.
Mar Gonzalez Franco: If we do it well enough, you will have to change nothing from your current interaction techniques. They will be embedded in the new technology.
Host: Let me just extrapolate here. I could have a device on and render a keyboard?
Mar Gonzalez Franco: Yeah. Or you will take your pen, physical pen. The pen will be recognized by these glasses, and you'll write on a digital notebook.
Host: This is so sci-fi.
Mar Gonzalez Franco: Science fiction is a very good inspiration for science.
Host: What's the biggest limitation you face right now? You're sitting in Redmond doing research. And if you could say, hey, this would make my life way easier in what I'm trying to figure out here – what kind of things are impeding your progress?
Mar Gonzalez Franco: I think Microsoft is very good at giving all the resources you need to do whatever you want to do. Of course, you have to deliver, but that's like in every other job. So I don't think I have many – I would say it's time. I don't have time to do everything I want. But this is also good, because it gives you the unique opportunity to focus. You need to focus, and you need to pick fights that you want, the most important ones.
Host: And you actually said at one point that virtual reality can prove neuroscience.
Mar Gonzalez Franco: Yes. There are some theories that are only available because they have explored very particular pathologies, that happen very rarely in the normal population, maybe one of a million have something like that. Maybe it's something related to epilepsy, removal of an epilepsy center that has detached your frontal lobe from the motor cortex, for example. So this is very unique scenarios, and there are people who undergo this pathology of the anarchic illusion, which they cannot control their hand at will. They know it's their hand, but they cannot control it.
Host: And this is a real thing in real life?
Mar Gonzalez Franco: Yeah. It's of one hand on it.
Mar Gonzalez Franco: No, but it's like simple things like you're eating a sandwich. I'm hungry. My hand knows I'm hungry. It picks your sandwich. So these people learn to – because the other hand usually is fully functional, they learn to hold their hand. So they just hold themselves.
Host: So they keep it from grabbing my sandwich?
Mar Gonzalez Franco: Yes, for example.
Host: Is that for real?
Mar Gonzalez Franco: Yes, it is. This is a real pathology. But this is a case-study type of thing. If you want to develop a real model of this, you might want to try to produce the same kind of illusion on healthy individuals. And then you can see what happens in their brain when this happens, and then compare it to what happens to a person who has this real pathology. Of course, these healthy participants, once they go out from VR, they are perfectly well. They don't have any issue on their control of their body, just like when they enter virtual reality, they cannot control their body as they wish.
Host: But the point would be to see what comparisons you could make in a healthy brain versus an unhealthy brain, and how that might be able to treat this?
Mar Gonzalez Franco: Yes, and also to understand how the brain works in general, for both healthy and unhealthy. So there are 2 mechanisms in the brain that we use to control our experiences. The first one is a bottom up. It's purely sensorial. Whatever we feel, we touch, we see, we integrate it in a motor sensory way. And that's how we define reality. The other one is we have predictions. And sometimes they're very strong. For example, when we feel the phone vibrates, and it didn't. This is a top down illusion.
Host: Other people experience that too besides me?
Mar Gonzalez Franco: Phantom phone, yeah. Yes.
Mar Gonzalez Franco: Yeah, so this experience is very sensorial. You would swear that you felt it. This is how powerful our brain is into manipulating our sensory input. And the interesting thing about the phantom phone, is that we become aware of it. But there are millions of predictions that are being rejected without even us being consciously aware of it.
Mar Gonzalez Franco: So in virtual reality, we can kind of start separating both, and seeing how they work one with the other. And it's very interesting, because this – when you play with this, you learn a lot about the neuroscience, on things that are very hard to test in reality. So I think virtual reality is definitely a tool for neuroscience.
Host: Let's say that you're this generation of virtual reality and neuroscience exploration, if you will. What's on the horizon for students coming out of university now?
Mar Gonzalez Franco: We have both in the US and in Europe very important big projects. There is the brain project in the US, and the human brain project in Europe, both of them with over a billion dollar funding, which is huge. They are mobilizing a whole community to finally get to understand the brain. And still, we don't know it. And certainly even the people who are doing AI are very interested in understanding, because if you want to simulate something, you need to know how it works.
Host: What would you like people to know about the research you're doing?
Mar Gonzalez Franco: I think in general, one of the things from a researcher perspective that is different from a product developer perspective, is the outreach. The fact that we can communicate, that you can do these types of things, or you could use technology to explore neuroscience, can improve dramatically a whole community of science. So how from one science you can transfer to another, so you are kind of becoming that bridge. I think when we work in research, we can transfer something, show that it's useful for something else, and then empower a whole community into using it in a different way.
Host: How does doing research at Microsoft differ from doing research at say, a university, or another academic setting?
Mar Gonzalez Franco: The way Microsoft Research moves is very fast compared to academic research. For me, to pursue an idea, I need to convince my VP. So I need to pitch it. I have to think about it. But I don't need to write a grant and then ask these very big organizations of a grant funding to evaluate it. And so it's much faster. My ability to reach the people and to collaborate with them, it's incredible. And I love it.
Host: Just as we go, just say one quick thing about what's, what you're the most passionate about right now in your work. I mean, aside from your outside life.
Mar Gonzalez Franco: I'm very interested about producing tactile sensations out of the body. I know this sounds very weird, but I'm creating this system in which you're holding a virtual object, and you feel the virtual object being touched. But not directly on your hands. Somewhere else. It's kind of difficult to explain it here, but this could mean that we can perceive touch in a similar way that we're perceiving audio. And this is – I'm working with Christopher Berger on this. He's Cal Tech. And we're both super excited about it. And this has been my work during the summer. And this has a lot to do with haptics. So if you have very simple vibro-activator in your hands, and you're able to perceive touch across the space, that's even better what you can do in reality.
Host: Mar, you are fascinating. Absolutely fascinating. I want to have happy hour with you for real.
Mar Gonzalez Franco: Good. Anytime.
Host: To learn more about virtual reality and Dr. Mar Gonzalez-Franco, and the latest in Microsoft Research, explore our website at Microsoft.com/research.
Episode 20, April 18, 2018 - In a world where humans are increasingly interacting with AI systems, Dr. Mary Czerwinski, Principal Researcher and Research Manager at Microsoft Research, believes emotions may be fundamental to our interactions with machines. And through her team's work in affective computing, the quest to bring Artificial Emotional Intelligence – or AEI – to our computers may be closer than we think.
Episode 17, March 28, 2018 - Dr. McDuff talks about why we need computers to understand us, outlines the pros and cons of designing emotionally sentient agents, explains the technology behind CardioLens, a pair of augmented reality glasses that can take your heartrate by looking at your face, and addresses the challenges of maintaining trust and privacy when we're surrounded by devices that want to know not just what we're doing, but how we're feeling.


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Q: QML circular image "waves effect" artefacts I'm building a qml application and I would like to implement a circle with a "wave" effect inside.
I've more or less succeeded except that the result is this graphic artefact... can anyone help me?
The problem:

Source image:

Working example without circle:

I state that I've NEVER used qml in my life and this is the third day I've been working on it, of course I'm trying to read documentation etc. but i have no idea what happened, this is the code:
//QT PK
import QtQuick 2.2
import QtQuick.Window 2.0
import QtQuick.Controls 1.1
import QtQuick.Controls.Styles 1.1
import QtGraphicalEffects 1.0
//JS PK
import "componentCreation.js" as MyScript

ApplicationWindow
{
    id: window
    flags: Qt.FramelessWindowHint
    visible: true
    title: qsTr("window")
    width: 1600
    height: 900
    x: (Screen.width - width) / 2
    y: (Screen.height - height) / 2
    color: "transparent"

    property real slideValue
    signal onSlide(real value)

    Image
    {
        id: img
        source: '/home/patan/code/QML/pic/contents/images/Waves.png'
        property bool rounded: true
        property bool adapt: true

        //MAKE IMAGE CIRCULAR
        layer.enabled: rounded
        layer.effect: OpacityMask
        {
            maskSource: Item
            {
                width: img.width
                height: img.height

                Rectangle
                {
                    anchors.centerIn: parent
                    width: img.adapt ? img.width : Math.min(img.width, img.height)
                    height: img.adapt ? img.height : width
                    radius: Math.min(width, height)
                }
            }
        }
        //WAVE EFFECT
        ShaderEffect
        {
            anchors.fill: parent
            property variant source: img
            property real frequency: 1
            property real amplitude: 0.1
            property real time: 0.0
            NumberAnimation on time
            {
                from: 0; to: Math.PI*2; duration: 10000; loops: Animation.Infinite
            }
            fragmentShader: "
                            varying highp vec2 qt_TexCoord0;
                            uniform sampler2D source;
                            uniform lowp float qt_Opacity;
                            uniform highp float frequency;
                            uniform highp float amplitude;
                            uniform highp float time;
                            void main() {
                                highp vec2 texCoord = qt_TexCoord0;
                                texCoord.y = amplitude * sin(time * frequency + texCoord.x * 6.283185) + texCoord.y;
                                gl_FragColor = texture2D(source, texCoord) * qt_Opacity;
                            }"
        }
    }
}

Forgive me if this is terrible code quality, if anyone can help me get it working I would appreciate it

A: I took a different approach to your solution.
I created an SVG icon that has the wave effect applied. Then I stretch the SVG icon to fill the screen. Then I use Image.Tile to fill the wave repeatedly and then I use the NumberAnimation to shift the SVG icon x.
Then, I apply an OpacityMask to cut out a circle:
import QtQuick
import QtQuick.Controls
import Qt5Compat.GraphicalEffects
Page {
    id: window

    property real slide: 0.0

    Item {
        id: waveEffect
        anchors.fill: parent
        Image {
            height: window.height
            width: window.width + 4 * window.height
            x: -window.height * 2 + slide
            source: `data:image/svg+xml;utf8,<svg width="32" height="32">
<rect width="32" height="32" fill="#0F325B"/>
<path d="M ${wave()} 32 16 32 32 0 32" fill="#5294E2"/>
</svg>`
            sourceSize: Qt.size(height, height)
            fillMode: Image.Tile
        }
        visible: false
    }

    Item {
        id: circleEffect
        anchors.fill: parent
        Rectangle {
            width: Math.min(window.height, window.width)
            height: width
            radius: width / 2
            color: "red"
        }
        visible: false
    }

    NumberAnimation {
        target: window
        property: "slide"
        from: window.height
        to: 0
        loops: Animation.Infinite
        running: true
        duration: 8000
    }

    OpacityMask {
        anchors.fill: waveEffect
        source: waveEffect
        maskSource: circleEffect
    }

    function wave() {
        let str = [ ];
        for (let j = 0; j<32; j++) {
            let a = j * 2 * Math.PI / 32;
            let i = Math.sin(a) * 2 + 16;
            str.push(`${j} ${i.toFixed(2)} `);
        }
        return str.join(" ");
    }
}

You can Try it Online!

redpajama_set_name: RedPajamaStackExchange


text:
Celebrate The Debut Album Of Metal Giant THOR With A Special 3 Disc Reissue Plus & A Spectacular NY Concert Event!
Los Angeles, CA – Get ready for one of the biggest metal reissues of the YEAR – a super deluxe edition of the legendary, underground classic, the 1977 debut album Keep The Dogs Away from metal icon THOR! This landmark release has become revered among music aficionados of every generation as one of the most explosive debut albums of any era with such ultra catchy rock anthems as "Keep The Dogs Away," "Sleeping Giant," "Thunder" and more. The CD version comes in a deluxe 6-panel digipak CD with alternate cover art, liner notes from longtime fan Brian Kehew (co-author of Recording The Beatles), 22 rare and unreleased bonus cuts on a separate disc PLUS a vintage 1980 concert DVD. The vinyl version meticulously recreates the original pressing with a foldout poster and printed inner sleeve. Both formats will officially hit the streets on May 13, courtesy of Deadline Music.
To celebrate this landmark reissue, THOR, in association with famed radio personality and metalhead Eddie Trunk and Fabio Productions, has put together a spectacular 4-hour multi-media event at the Highline Ballroom in New York City on May 14th. The event includes a first time EVER full concert performance of the entire KTDA album, plus a theatrical screening of the I Am Thor documentary, a special THOR Museum display, as well as another concert performance of THOR's most popular songs with tons of special guests! This is the ULTIMATE THOR experience – a metal extravaganza unlike any BETHOR! https://www.facebook.com/THOR-A-Rock-Odyssey-Live-in-New-York-1016591135046207/
1. Keep The Dogs Away
2. Sleeping Giant
3. Catch A Tiger
4. I'm So Proud
5. Tell Me Lies
6. Military Matters
7. Superhero
8. Wasted
9. Rosie
10. Thunder
BONUS TRACKS (CD ONLY)
1. Asgardia
2. All Nite
3. Atomic Vibrations
4. Do The Muscle (1975 Version)
5. Away From You
7. You Tried My World (No Time)
8. Do The Musicle
9. We Got The Power (Brian Kehew Mix)
10. Hey Tonight (Brian Kehew Mix)
11. Everybody Needs A Hero
12. We Are Body Rock / Start The Show
13. You Have Too Much
14. Just For You
15. Give Me The Green Light
16. She Sings The Body Rocker
17. Tears In My Soup
18. Who's Better? (You Haven't Got A Chance)
19. Best Built Man Alive (Live)
20. Land Lover
21. Open Sky
22. Keep The Dogs Away (Demo)
Buy the CD: http://flyt.it/ThorKTDACDAmazon
Buy the Vinyl: http://flyt.it/ThorKTDALP
Download the digital version: http://flyt.it/ThorKTDADig
Billy James
glassonyonpr@gmail.com
CLEOPATRA RECORDS, Inc.
11041 Santa Monica Blvd #703
Los Angeles CA 90025
http://www.CleopatraRecords.com
This entry was posted on Friday, April 22nd, 2016 at 19:22	and posted in Press Release, Thor.	You can follow any responses to this entry through the RSS 2.0 feed.
« YES Legends Billy Sherwood and Tony Kaye's "Live In Japan" 2CD/1DVD OUT NOW!
Italian Prog Ensemble Armonite Releases Debut Album "The Sun is New Each Day" »


Following the reveal of the PS4, Microsoft's reply is expected to happen in April.
Unspecified "industry developers" have said that the next Xbox iteration will be announced at a one-off event, which will take place in April, according to CVG.
In addition to the rumours surrounding Microsoft's unannounced reveal event, a senior Sony official, who asked to remain anonymous, said that the company's PS4 announcement "caught Microsoft off-guard" and they "definitely ruffled some feathers".
Looks like the next-gen console race has begun – and Sony's reveal has left everyone wondering what Microsoft's next move will entail.


text:
<?php

namespace PHPCodeBrowser\Tests\Plugins;

use DOMDocument;
use PHPCodeBrowser\File;
use PHPCodeBrowser\Issue;
use PHPCodeBrowser\IssueXml;
use PHPCodeBrowser\Plugins\ErrorPMD;
use PHPCodeBrowser\Tests\AbstractTestCase;

/**
 * ErrorPMDTest
 *
 * @category   PHP_CodeBrowser
 * @package    PHP_CodeBrowser
 * @subpackage PHPUnit
 * @author     Simon Kohlmeyer <simon.kohlmeyer@mayflower.de>
 * @copyright  2007-2010 Mayflower GmbH
 * @license    http://www.opensource.org/licenses/bsd-license.php  BSD License
 * @version    Release: @package_version@
 * @link       http://www.phpunit.de/
 * @since      Class available since  0.9.0
 */
class ErrorPMDTest extends AbstractTestCase
{
    /**
     * The object to test.
     *
     * @var ErrorPMD
     */
    protected $errorPmd;

    /**
     * The xml string to test the plugin against.
     *
     * @var string
     */
    protected $testXml = <<<HERE
<?xml version="1.0" encoding="UTF-8" ?>
<pmd version="0.2.6" timestamp="2010-07-17T02:38:00-07:00">
  <file name="/some/file">
    <violation beginline="3"
               endline="4"
               rule="Rule1"
               ruleset="Ruleset 1"
               priority="1">Description 1</violation>
    <violation beginline="5"
               endline="5"
               rule="Rule2"
               ruleset="Ruleset 1"
               class="SomeClass"
               method="someMethod"
               priority="3">Description 2</violation>
  </file>
  <file name="/other/file">
    <violation beginline="15"
               endline="15"
               rule="The third rule"
               ruleset="Ruleset two"
               priority="3">Description 3</violation>
  </file>
  <file name="/has/no/violation">
  </file>
</pmd>
HERE;

    /**
     * (non-PHPDoc)
     * @see tests/cbAbstractTests#setUp()
     */
    protected function setUp()
    {
        parent::setUp();
        $issueXML = new IssueXML();
        $xml = new DOMDocument('1.0', 'UTF-8');
        $xml->loadXML($this->testXml);
        $issueXML->addXMLFile($xml);
        $this->errorPmd = new ErrorPMD($issueXML);
    }

    /**
     * Test getFileList
     *
     * @return  void
     */
    public function testGettingFileList()
    {
        $expected = array(
            new File(
                '/some/file',
                array(
                    new Issue(
                        '/some/file',
                        3,
                        4,
                        'PMD',
                        'Description 1',
                        'error'
                    ),
                    new Issue(
                        '/some/file',
                        5,
                        5,
                        'PMD',
                        'Description 2',
                        'error'
                    )
                )
            ),
            new File(
                '/other/file',
                array(
                    new Issue(
                        '/other/file',
                        15,
                        15,
                        'PMD',
                        'Description 3',
                        'error'
                    )
                )
            ),
            new File(
                '/has/no/violation',
                array()
            )
        );
        $actual = $this->errorPmd->getFileList();
        $this->assertEquals($expected, $actual);
    }
}

redpajama_set_name: RedPajamaGithub


text:
Research Fellow Profile: Dr. Ian DeVolder
Myotonic has awarded a 2016-2017 postdoctoral fellowship to Dr. Ian DeVolder, Ph.D., a Graduate Research Assistant in the Department of Psychiatry at the University of Iowa Carver College of Medicine.
Dr. DeVolder's research proposal is titled "Structural and Functional Connectivity in the Brains of Patients with Adult- and Late-Onset Myotonic Dystrophy Type 1 (DM1): A Potential Biomarker for Disease Progression." In this study, Dr. DeVolder and his colleagues will evaluate brain structure and function in DM1 and correlate these with measures such as neurocognitive functioning and disease duration. The investigators will study 30 patients with classic adult-onset or with late-onset DM1, ages 21 to 65 years old, and compare them to 30 age-matched healthy controls.
Dr. DeVolder received his doctorate in neuroscience from the University of Iowa in 2015 and is a graduate research assistant in the laboratory of Peg Nopoulos, M.D., at the University of Iowa Carver College of Medicine. His work at Iowa has focused on the structure and function of the brain in children with clefts of the lip or palate and in children at risk for Huntington's disease.
"If we can know how and when myotonic dystrophy type 1 affects the brain," DeVolder says, "we can better time treatment so as to have a neuroprotective effect and try to prevent these brain changes from happening in the first place." We recently talked with Dr. DeVolder to learn more:
Myotonic: Your previous work was focused mainly on the brain abnormalities that can accompany clefting disorders, such as cleft lip and cleft palate. [See DeVolder, I., et al., Abnormal cerebellar structure is dependent on the phenotype of isolated cleft of the lip and/or palate, The Cerebellum, April 2013.] How did you move from there into myotonic dystrophy?
ID: It's definitely been a shift in terms of the clinical population that I've been working with. Clefting abnormalities and myotonic dystrophy are not directly related. However, in terms of the basic practice, the basic study we did, they're actually not that far removed. It's the same type of imaging techniques, the same type of neuropsychological evaluation.
And, even though my thesis work was with the clefting community, I actually have had a large role in a number of different studies in my lab. Importantly, one of those was our study on Huntington's disease, which can be thought of as a sister disease to myotonic dystrophy. They're both trinucleotide repeat disorders, and both previously were thought of as primarily neuromuscular diseases.
The Huntington's study was focused on children who were at risk for developing HD. These children had either a parent or grandparent who was affected by the disease. We did a full neuropsychological evaluation, MRI and genetic testing. We were comparing children with the expanded repeat, who, in 30 years or so, will likely develop HD, with those who don't have the expanded repeat. We were looking at Huntington's from a developmental perspective to see whether, at an early age, there is something being set in motion in terms of neurodevelopmental changes. Results from this study should start being published within the coming year.
It's been an interesting shift into myotonic dystrophy. We wanted to model our DM1 study after our Huntington's study, looking at kids who were not yet showing symptoms but who were at risk for DM1. But we underestimated the role of anticipation in DM1. This is a phenomenon that is seen in Huntington's but not nearly as frequently and not nearly as severely as in myotonic dystrophy.
We discovered that families with DM1 oftentimes don't know that they have it or that their children are at risk until they have a child that's born with an extremely expanded repeat and the congenital-onset or childhood-onset form of the disease. So it's much harder to identify children with pre-DM1 than children with pre-HD. Therefore, we shifted our focus to adult-onset and late-onset myotonic dystrophy.
There have been some neuroimaging studies in myotonic dystrophy, but they've typically focused on the childhood-onset, adult-onset and congenital-onset forms all together in one group.
We really wanted to focus on one type of DM1, because the congenital-onset and childhood-onset forms seem to be so different in terms of the symptoms they show. We wanted to completely remove that confounding factor. We're looking at a pretty big age range – 21 to 65 – but it's still adult-onset DM1. We cut off the age for this study at 65 because we didn't want to introduce aging effects as confounding factors.
We're combining concepts from a lot of previous neuroimaging studies. We're using several neuroimaging techniques and we're combining those with a neuropsychological evaluation. We're also making it a longitudinal study, where participants will come back once a year for three years. The study is unique in that sense. It's the first neuroimaging study in DM1 to combine all of these elements.
Myotonic: What kinds of brain abnormalities are you looking for?
ID: The brain changes in myotonic dystrophy have been primarily found to be white matter-related. We expect to find some of the things that have already been seen, such as increased numbers of white matter hyperintensity lesions. White matter refers to the myelinated fibers that connect different regions of the brain, and there are variants that you can see on an MRI scan. They're a little bit unclassified, but basically they're considered to be abnormal white matter.
We're also using diffusion imaging, which looks even more specifically at white matter structural integrity. Diffusion imaging measures the movement of water molecules in tissues. It's a way to see if water is moving along the axon versus going out. From that we can get an idea of the actual shape and structure of the white matter. Typically, white matter in the brain forms tight fiber bundles and tracts, so healthier and better-myelinated white matter would lead to an increase in water movement along the axons, rather than out into the brain. This can be measured by diffusion imaging.
There's been a fair amount of neuroimaging work in myotonic dystrophy, but there's been hardly any functional neuroimaging. That's something I've worked with in our studies and something I've really wanted to focus on for this population as well.
I was really excited and somewhat surprised when I saw the 2014 paper on functional brain connectivity in DM1. [See Serra, L., et al., Abnormal functional brain connectivity and personality traits in myotonic dystrophy type 1, JAMA Neurology, May 2014.]
They found that in patients with DM1 there was increased functional connectivity in certain parts of the brain compared to the control group. Specifically, they found increased network connectivity between the left and right posterior cingulate cortex and the left parietal node when the participant was in a resting state – in other words, not engaged in any specific task. They also found that the DM1 group was more likely to show certain personality traits, such as the presence of fixed ideas, rigidity of thought, and an acute sensitivity to anger or hostility in others, than the control group.
In our study, we're looking at the resting state, and we're looking at functional connectivity, but we're also looking at the developmental component, whether these networks are changing over time and with disease duration.
In resting-state functional connectivity analyses, we're examining low-level changes in blood flow throughout the brain. You can look at the time course of these blood-flow changes at each individual voxel [volume element] in the brain, and then can compare that time course to all other voxels of the brain. From this you can discover areas of the brain that are showing the same levels of blood-flow changes, with the idea being that those areas that are functionally connected to each other would show a more similar type of pattern to each other in terms of blood-flow. With this data we can examine functional networks in the brain, and how they may be changing in DM1.
In some of the questionnaires that we're administering, we're also looking for personality traits that may be typical. We'll see whether or not we capture the same types of findings as the 2014 study.
Myotonic: If you do find brain abnormalities, are they necessarily the cause of the cognitive and personality differences sometimes seen in DM1? Could it be that focus on certain thoughts or activities could change the brain? Or could it be that respiratory or cardiac impairment associated with DM1 affect the brain?
ID: I think that if there's a common pattern of brain abnormalities seen in a population, I would argue that it's more neurobiologically based rather than the other way around. But it's a hard thing to parse out.
In another part of your question, you asked about whether what we're seeing might not be primary but secondary to some of the respiratory or cardiac issues. It's an issue that we've run into, particularly in the clefting studies that I've been involved in.
A fair critique of that study is that some of the changes we measured may actually be secondary, a response to the things these kids experience at really young ages – like anesthesia during reparative surgeries when they're not even one year old yet. They are facing these environmental insults at this critical developmental time point. It's a potential caveat to some of our studies.
I think with myotonic dystrophy it won't be quite as big an issue. In our screening process, we automatically exclude individuals who have a pacemaker installed, because they can't go into the MRI scanner. As a result, I think those individuals who would be the most severely affected in terms of the cardiorespiratory symptoms are automatically being excluded from the study.
I think it's going to be more reasonable in this study to really try and parse out the abnormalities that are directly caused by the gene expansion as opposed to other factors.
Also, we do get a pretty extensive medical history from all our study participants, so potentially we could create within the myotonic dystrophy group some separate subgroups, such as those that are most severely affected by arrhythmias, and see whether or not we are getting the same patterns of brain changes.
Myotonic: Would finding brain abnormalities in study participants with DM1 have therapeutic implications?
ID: We're focusing on the longitudinal aspect in these studies. What we're hoping to find is essentially biomarkers for the disease. These do have important therapeutic implications, but they're not going to be immediately obvious.
As for the current drug trials that are going on with Ionis, they potentially could have a lot of therapeutic benefit. However, the drug they're testing cannot cross the blood-brain barrier unless delivered intrathecally -- via spinal infusion. Right now, the potential drug treatments, which are delivered subcutaneously, won't actually get into the central nervous system. [Ionis Pharmaceuticals is testing its antisense-based drug IONIS-DMPK-2.5Rx, which targets the abnormally expanded RNA from the DMPK gene in DM1.]
The thing is, we don't have a good idea of the developmental component of the brain abnormalities in terms of the disease progression itself. Before drug discovery can start moving into that area, we have to know what's actually happening in the brain. If we can get a better idea of when these changes are occurring and what the changes actually are, we can track disease progression much better, potentially having much better timing of when drug delivery should happen. With optimal timing of drug delivery, these drugs could have a neuroprotective effect and ideally prevent these brain changes before they happen.
Myotonic: Is your study still open to recruitment?
ID: The study is well under way, but yes, it's still open. We will continue to recruit new participants over the next few years.
Note: For details about this and other DM studies, go to Myotonic's Study and Trial Resource Center and select the Current Studies and Trials tab. The study discussed in this article is Brain Structure and Function in Adults with a Family History of DM1.
Home / Research Fellow Profile: Dr. Ian DeVolder


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text:
The New Turkey > Economy > Finance , Turkey , Society , Opinion | December 14, 2016
Securing a Future for Syrian Women in Turkey
By Nergis Dama
Turkey is home to the highest refugee population in the world, having adopted an open door policy for people who come from Syria to Turkey. The number of registered Syrians in Turkey has reached 2,783,617. Of these, 1,301,026 are Syrian women.
Turkey is home to the highest refugee population in the world, having adopted an open door policy for people who come from Syria to Turkey. The number of registered Syrians in Turkey has reached 2,783,617 according to data from the Ministry of Interior Directorate General of Migration Management for December 2016. Of these, 1,301,026 are Syrian women.
While all Syrians – women, men, children, and elders – have been affected by violence and conflict in Syria and their life has been upended, the group mostly affected by affairs are women and children.Even when there is no war or conflict, women and children are regarded as a disadvantaged group. Taking on the burden of chaos and conflict has deepened their usually disadvantaged situation. Analyzing news about Syrian refugees in written and visual media illustrates the fact very well. Sometimes, the coverage shows a Syrian girl sleeping in front of traffic lights after begging; sometimes it shows that Syrian women are being taken as second wives, and sometimes it reflects success stories. Considering that the issue of Syrian refugees has been used as a political tool, this depiction, which symbolizes the extremes of either "happiness" or "poverty," does not reveal much about Syrian women's future.
It is important that observations and experiences give an idea about problems. Commentary, which has been used as a political slogan, will contribute only to ideological propaganda. In order to improve the economic and social conditions of Syrians at the macro level, and at the micro level the present and future of Syrian women, it is necessary to identify the problems facing them, and develop a road map by giving up ideological obsessions and subjective reasoning.
Why Should Syrian Women Be Selected As Target Group for Support?
In general, women have more gender-oriented problems than men when participating in economic, political and social life. This is valid for women living not only in developing but also developed countries, where they confront difficulties and challenges to varying degrees. Despite their disadvantages, women take on a role being a leader for a family and society to change and advance. Because of their powerful effect on the family, women have come into prominence as an actor that makes easier the process of adaptation. For this reason, Syrian women are responsible for the Syrian population's efforts to participate in the economic and social life in Turkey.
When analyzed in terms of their age groups, the female Syrian population offers ideas for new policies.
Syrian women are almost half of the Syrian refugee population in Turkey, and the 5-18 and 19-34 age groups include the most Syrian women. These groups have 829,255 persons, which is almost 64 percent of total Syrian female population in Turkey. The youngest age group, those less than five years old, includes a large number of women, 189,814. This indicates that the fertility of Syrian women has not been affected by problems such as living in a foreign country, not having a stable life, and an uncertain future. At the same time, this indicates Turkey has to take into consideration these groups to determine programs such as education and health support. But a further policy needs direct communication with Syrian mothers. For this reason, education, care and health policies including the under-five age group, girls and boys, can be used as a tool to solve problems of Syrian women and satisfy their needs.
On the other hand, the ratio of Syrian women in the 19-34 age group is almost 30 percent. If this group actively participates in economic and social life, it might be easier for them to adapt to life in Turkey, and any negative impacts of the Syrian refugee presence on society would diminish.
In making policies to address the issues facing them, certain aspects, like the traditions, culture, and habits of Syrian women must be taken into consideration.
A basic mistake seen in developing policies is the assumption that Syrian women are the same as women in Turkish society, since both groups are predominantly Muslim, and they come from neighboring countries. But Syrian society has a somewhat different culture, life style and customs, and the war in Syria has created new social practices that have problematic aspects for those trying to settle in Turkey.
One example is the presence of polygamy in Syria, where the law allows for a man to take a second wife after getting permission from the first. In Turkey, the law prohibits a second wife altogether.
Polygamy has increased inside Syria since the outbreak of the war, because more and more widowed women find themselves looking for ways to provide for their children's needs. Data on the practice is not clear, but some research examining the ratio of polygamy has shown that it has increased since the start of the conflict.
According to figures, polygamous relationships accounted for 30 percent of marriages registered in Damascus in 2015, up from just five percent in 2010. The popularity of the practice has apparently been driven by a situation in many parts of Syria where women find themselves far outnumbering men, as they have either gone to join rebel groups, or been killed or captured in combat. A large proportion of young men have fled the country fearing the regimes expanding policy of conscription as well.
Syrian women are more likely to face economic and social pressure to enter a polygamous marriage in order to secure their future. There are also practical problems for a Syrian woman in a polygamous relationship. Under Turkish law, for instance, such a marriage is not recognized. A whole range of things are therefore unavailable, everything from obtaining a proper residence permit to making sure an ex-husband provides financial support after a divorce.
While Syrian women need to be empowered in terms of social and economic aspects, a "modernist" approach that looks for a complete break from religious and cultural habits needs to be avoided.
Good implementation taking into account unexpected difficulties can result in positive outcomes in the long term. A more realistic and rational implementation is needed. Considering the reality related to cultural features of Syrian women, such as polygamy, childbearing (high fertility rate), early marriage, and perception of women and family, they should be encouraged to participate in education and training programs, and macro policies should be enacted to help them become working professionals.
Younger girls, aged between 4 and 17, should have an education, and an effective communication method should be generated between their mothers and educational actors such as teachers and school principals. Otherwise, an uncertain and gloomy future might be in store for Syrian women and girls. Moreover, the social dynamic of Turkish society will be affected as well. The economic and social participation of the Syrian population can be ensured through the leadership of Syrian women.
Syrian Women
Is a Common Currency Backed by the Gold Standard Achievable?
Omar Khalid Bhatti
A Look into Turkey's Agricultural Policies
Fahri Yavuz
The US Went Too Far Against Huawei
Mevlüt Tatlıyer
Nergis Dama
Nergis Dama is Assistant Professor at Faculty of Political Sciences, Ankara Yıldırım Beyazıt University and conducts researches on welfare studies, social aids, economic and social rights of disadvantages groups. Dama holds a BA in Mathematics, earned her MA in Economics and finished her PhD in Social Policy.
Melian Dialogue and Six Years of Civil War in Syria
Shockwaves of the Syrian Conflict in Europe: The Price of Standing Aside
Turkey urges EU action on Syria
Over 100 Tanzanians killed for witchcraft since January


I was born in Detroit, Michigan to Verl and Aimee Sparling. Throughout my youth, I lived in Detroit, Ferndale, and Owosso. As a child, summer vacations were always spent in the "Thumb" and to this day I feel emotionally drawn to the area. I still have family in Michigan and it will always be home to me.
For as long as I can remember, my relatives told stories about the "Dying Sparlings." This unsolved mystery has puzzled me since childhood and I've always wanted answers. I never even considered writing a book, but the opportunity to combine my love of family history and historical research appealed to me.
This is my first book. I also enjoy reading, volunteer work, playing the piano, and especially being a proud wife, mother and grandmother. I currently reside in the Shenandoah Valley of Virginia with my husband, Bob.
J.W. and Carrie Sparling were happy newlyweds in 1882 with a promising life ahead of them. They lived and farmed in the rural "Thumb" of Michigan. Nine years later their family had grown to include one daughter and four sons. By all accounts, the family was happy and prosperous.
Starting in 1908, however, the Sparling family was plagued by the unexplained deaths of four family members, one by one.
What was happening to the Sparlings?
This was my question as I gathered information. After researching the story through court records and testimony, newspaper articles, medical records, insurance forms, and oral history, I had accumulated the foundation for this book. "The Thumb Pointed Fingers" can best be described as historical fiction.


Warrington letting agent Hamlet Homes Warrington is an independent family ran property services company offering original and dedicated expertise to landlords, tenants and the professional investor. We manage single-lets, HMOs, flats and 2up 2downs all day long, and have been doing this since 1992, we purchased, converted and tenanted a HMO long before it became a fashionable investment strategy.
We work with local, national and international investors on a range of property strategies from bog standard BTL to the more innovative and creative R2R, Lease Options and will also JV with our investor landlords.
Letting property these days is not rocket science, however, matching tenants to your property and your investment strategy requires some outside of the box thinking. The team at Hamlet Homes are ALL dedicated and committed property investors themselves, we'll carefully match tenants to align with your own investment goals. Doing this right gives you a long term tenant and the tenant a long term home. If you want to maximize and grow your returns from your property investment then you really need to speak to us.
Give us a call on 01925 235 338 or pop into our office opposite Iceland/Aldi and lets have a chat over a cup of tea.


text:
Q: Run Cypress.io on Azure Pipelines Hosted Linux Agent I'm trying to run Cypress tests on the Hosted Linux Pool for Azure Pipelines. Unfortunately, the Hosted Agent doesn't have all the dependencies for Cypress installed.
Running the documented apt-get doesn't work:
2018-05-18T21:03:14.7423331Z ##[section]Starting: Install cypress dependencies
2018-05-18T21:03:14.7474742Z ==============================================================================
2018-05-18T21:03:14.7488281Z Task         : Bash
2018-05-18T21:03:14.7501148Z Description  : This is an early preview. Run a Bash script on macOS, Linux, or Windows
2018-05-18T21:03:14.7513088Z Version      : 3.127.0
2018-05-18T21:03:14.7524823Z Author       : Microsoft Corporation
2018-05-18T21:03:14.7537179Z Help         : [More Information](https://go.microsoft.com/fwlink/?LinkID=613738)
2018-05-18T21:03:14.7549730Z ==============================================================================
2018-05-18T21:03:15.0174503Z Generating script.
2018-05-18T21:03:15.0535056Z Script contents:
2018-05-18T21:03:15.0547355Z apt-get install xvfb libgtk2.0-0 libnotify-dev libgconf-2-4 libnss3 libxss1 libasound2
2018-05-18T21:03:15.0656822Z [command]/bin/bash --noprofile --norc /opt/vsts/work/_temp/cac4d3f9-42e7-49f3-94f6-7d0444827d83.sh
2018-05-18T21:03:15.6040707Z Reading package lists...
2018-05-18T21:03:15.6085335Z Building dependency tree...
2018-05-18T21:03:15.6153815Z Reading state information...
2018-05-18T21:03:15.6186788Z Package libgconf-2-4 is not available, but is referred to by another package.
2018-05-18T21:03:15.6198707Z This may mean that the package is missing, has been obsoleted, or
2018-05-18T21:03:15.6211380Z is only available from another source
2018-05-18T21:03:15.6216969Z
2018-05-18T21:03:15.6229592Z E: Unable to locate package xvfb
2018-05-18T21:03:15.6242128Z E: Unable to locate package libnotify-dev
2018-05-18T21:03:15.6254440Z E: Package 'libgconf-2-4' has no installation candidate
2018-05-18T21:03:15.6268141Z E: Unable to locate package libxss1
2018-05-18T21:03:15.6370826Z ##[error]Bash exited with code '100'.
2018-05-18T21:03:15.7283047Z ##[section]Finishing: Install cypress dependencies

Running apt-get update takes forever (25 minutes and still not done).
Any clues on how to get Cypress working quickly on the Hosted Agent is welcome.
Note: I tried running on Windows, that works, fortunately, so I'm not completely blocked. But to use the Windows Agent I now have 2 agent phases which adds overhead due to artefact downloads and npm install overhead.

A: Solved the issue using a pre-pended Shell (Bash) task in VSTS agent phase prior to running Cypress:
apt-get -qq -y update
apt-get -qq -y install xvfb libgtk2.0-0 libnotify-dev libgconf-2-4 libnss3 libxss1 libasound2


Jun 22, 2018 WPC decking material is a new type of environmentally, eco friendly and energy-saving composite WPC DIY Decking is a kind of outdoor flooring which highlights the Ask: What can you use wood plastic composite for?
Jun 24, 2015 Outdoor decks create the perfect space for summer grilling and add visual appeal to any yard. From wood to composite, there are a ton of variables including color, texture, There are three main types of wood you can use:.
2017 new type outdoor WPC bi-color composite decking mixed 2 colors 140 x Natural wood grain make the decking looks more authentic. 2. Application:.
Different Types of Composite Decking: Hollow vs Solid Core Boards Once you decide to use composite decking material for your home's or business's deck, you JF Outdoor Composite Interlocking Deck Tiles - Gray 12" x 12" / interlockin .
Dec 31, 2011 Composite decking is made from a combination of natural wood fiber and resins. Home Hacks & Answers · Outdoor Building · Decks & Patios barbeque grill mats over composite decking, some types of backing materials If you use too much pressure, the color can be stripped from the decking boards.
Feb 18, 2018 If you are dreaming of an outdoor deck to enjoy neighborhood barbeques pine will hold up for many seasons of use with the proper care and maintenance. Wood/plastic composite materials are a great choice for decks.


Will I receive a fair share of our assets?
What will my future look like?
Will I be able to live reasonably?
How much time will I have with my children?
A clear understanding of the divorce process can go far to reduce this anxiety. At our first meeting, we will discuss your concerns, how the courts look at specific issues, and how the legal process will unfold.
Part of my role as your attorney is to explain the range of likely outcomes, given your particular situation, on each issue the court must decide. With a good understanding of these factors, you are in the best position to start deciding how to share the time with your children and how best to separate finances.
When emotions run high, I act as a buffer to help defuse those passions. My job is to help you focus on the things that matter — developing the evidence on each issue that the court must decide: dividing assets and debts, alimony, the parenting plan, and child support. Emotional reactions and mudslinging may feel good in the short run, but often sabotage your best interests.
I believe the judges and commissioners appreciate a reasonable advocate. They face people badmouthing each other on a daily basis. Rather than making a lot of noise without substance, I work with you to support our position with receipts, photos, records, and testimonies from neutral professionals.
Finally, in a divorce it is important to disrupt the children's lives as little as possible. It is critical that your children not feel placed in the middle, and not be asked to take sides. Part of our job as attorneys and as parents is to protect the children from the conflict as much as possible and not interfere with the love they have for their parents.
Mediation: I also work as a family law settlement conference mediator. As a settlement conference mediator, I help the attorneys for both sides work toward an agreement by giving neutral, experienced feedback on the strengths and weaknesses of both sides' positions, as well as working to defuse conflict and explore common interests.
If you would like to know more about Mark, read the blog post Meet Mark Alexander.
You can contact Mark directly at mark@seattledivorceservices.com.
Mark Alexander graduated with honors from Harvard College and attended the Backus School of Law of Case Western Reserve University, where he was a legal research, advocacy, and writing instructor for first-year law students. Licensed as an attorney since 1979 and admitted in five states, he has been practicing family law in the Puget Sound area since 1990. He has served as Managing Attorney for the Family Law Mentor Program of the King County Bar Association, as Chair of its Family Law Section, and as a member for over 15 years of its Local Rules committee. In 2013, Mark was elected to the statewide Family Law Executive Committee of the Washington State Bar Association. In addition to volunteering at the Greenwood and Eastside legal clinics and the Volunteer Settlement Conference program, Mark has been a speaker on divorce and family law at local schools, at seminars for attorneys, and at the University of Washington's business school.


The page provides the exchange rate of 2012 Euro (EUR) to Polish Zloty (PLN), sale and conversion rate. Moreover, we added the list of the most popular conversions for visualization and the history table with exchange rate diagram for 2012 Euro (EUR) to Polish Zloty (PLN) from Friday, 19/04/2019 till Friday, 12/04/2019. Latest update of 2012 Euro (EUR) exchange rate.
Convert currency 2012 EUR to PLN. How much is 2012 Euro to Polish Zloty? — 8615.33 Polish Zloty.
2012 Euro to Polish Zloty, convert 2012 EUR in PLN.


This research was actually a byproduct of Russian-American competition in Moon exploration.'The issue with Chronology' is the first volume in the series "History: Fiction or Science?", the fundamental oeuvre that exposes and expounds the numerous inveracities of the traditional version of history.
In "The Issue with Chronology" we are reminded of when the contemporary chronological scale was created and by whom, with the culprits named as the XVI-XVII century clergy that was in charge of all matters historical in that age.
We also learn that the consensual model of history had prominent critics ever since its creation – among them such names as Sir Isaac Newton and Jean Hardouin, chief librarian of Louis XIV, the Sun King of France and collector of Louvre museum.
The author dissects every historical age and analyses the data from every source imaginable – Roman and Egyptian chronology take a good beating, and it goes rapidly downhill from there. Poggio Bracciolini and Petrarch take the blame for creating the legend of a mythical Classical age that never was. Our perception of history begins to change dramatically even before we're through with "The Issue with Chronology".


text:
Charlie Sheen-less 'Two and a Half Men' season debut draws record audience
By Lisa de Moraes
Record crowds turned out Monday night to watch Charlie Sheen be killed off on one television network and resurrected on another.
Nearly 29 million people caught Monday night's expunging of Sheen from the CBS sitcom "Two and a Half Men." That's the long-running show's largest audience — ever. It's also the biggest season-debut audience for any scripted program on television since 2005.
Also making money off Sheen on Monday night: Comedy Central, as 6.4 million tuned into the first airing of a Sheen roast. That's the franchise's biggest audience ever, edging out the record holder: the Jeff Foxworthy roast of 2005 (6.2 million). Comedy Central is particularly happy that more than half of the throng were 18-to-34-year-olds, the network's target audience.
The "Men" season opener also clocked the show's biggest-ever audience among CBS's target audience: 18-to-49-year-olds. In the episode, Sheen's Charlie Harper is described as having died when he slipped on a Paris Metro station platform, fell in front of a train and exploded like "a balloon filled with meat."
Nearly 11 percent of the country's audience in the 18-to-49 age bracket tuned into the show to see how creator Chuck Lorre would introduce Ashton Kutcher, who joined the show Monday as a lovelorn Internet billionaire named Walden Schmidt, who buys Charlie's Malibu pad and yet somehow wants Charlie's impecunious brother, Alan (Jon Cryer), and son, Jake, to remain.
(The answer: He's emotionally immature.)
A 10.7 rating in that demographic group for an eight-season-old series is the very definition of ginormous ratings.
Previously, the biggest audience "Men" had snared was the 24 million who watched an episode that aired May 16, 2005, immediately after the series finale of "Everybody Loves Raymond."
In much the same way the demise of "Raymond" helped "Men," the demise of Charlie Harper was a gift to the new CBS sitcom that followed: "2 Broke Girls" nabbed 19.4 million viewers — the biggest crowd for a fall comedy premiere since September 2001.
Monday night was probably CBS's last chance to make some coin off the Sheen saga.
(In case you missed it, back in January, Warner Bros. TV shut down production on the network's hit sitcom so that Sheen, whose partying ways kept TMZ in material, could get treatment for substance abuse. Sheen insisted that he was good to go weeks later, but Warner and Lorre disagreed. Sheen took to the airwaves to rant. Warner Bros. shut down the show for the rest of the TV season. Sheen ratcheted up the rhetoric. The studio sacked him. Sheen filed a $100 million suit. A judge said it would have to be arbitrated privately because of the actor's contract with the studio. Sheen launched his Torpedo of Truth national tour. Warner Bros. cast Kutcher to replace Sheen. Everybody took a breather.)
Anyway, all these Sheen-fueled ratings are very good news for Lionsgate's Debmar-Mercury, which is developing a new comedy series starring Sheen and loosely based on the "Anger Management" flick.
But there was other, non-Sheen ratings news on the first night of the 2011-12 TV season, as well.
At 10 p.m. Monday, attention shifted to the season debut of ABC's "Castle," which handily won the hour as nearly 14 million fans tuned in — the show's biggest season-premiere audience yet.
And for those of you wondering what NBC was thinking of when it threw its new, much-ballyhooed "Playboy Club" into the intense Monday-at-10 competition, which includes "Castle" and CBS's "Hawaii Five-O," your answer may be "the season's first cancellation."
The drama about characters who frequent and work in the original Playboy Club in Chicago in the '60s attracted a mere 5 million viewers — and only a 1.6 rating in the plum age bracket that advertisers lust after.
On the bright side, that would be a monster number were the show on, say, AMC, as is "Mad Men" — the stylish '60s-set Madison Avenue drama that is credited with inspiring "Playboy Club" and ABC's new "Pan Am."
But, sadly, "Playboy Club" is not. That said, the show appears to have a close commercial relationship with the Playboy company — ka-ching — and if it's run like "Mad Men," other product placements will abound in coming episodes. In which case, ratings aren't the only factor that will determine this show's fate.


text:
#ifndef itkHistogramToIntensityImageFilter_h
#define itkHistogramToIntensityImageFilter_h

#include "itkHistogramToImageFilter.h"

namespace itk
{
/** \class HistogramToIntensityImageFilter
 * \brief The class takes a histogram as an input and produces an image
 * as the output. A pixel, at position I,  in the output image is given by
 *
 * \f[
 * f(I) = q_I
 * \f]
 *  where  \f$q_I\f$ is the frequency of measurement vector, I.
 *
 *  \sa HistogramToProbabilityImageFilter, HistogramToLogProbabilityImageFilter,
 *  HistogramToImageFilter, HistogramToEntropyImageFilter
 * \ingroup ITKStatistics
 */

namespace Function
{
template< typename TInput, typename TOutput = SizeValueType >
class HistogramIntensityFunction
{
public:

  //Intensity function returns pixels of SizeValueType.
  using OutputPixelType = TOutput;

  HistogramIntensityFunction():
    m_TotalFrequency(1) {}

  ~HistogramIntensityFunction() = default;

  inline OutputPixelType operator()(const TInput & A) const
  {
    return static_cast< OutputPixelType >( A );
  }

  void SetTotalFrequency(SizeValueType n)
  {
    m_TotalFrequency = n;
  }

  SizeValueType GetTotalFrequency() const
  {
    return m_TotalFrequency;
  }

private:
  SizeValueType m_TotalFrequency;
};
}

template< typename THistogram, typename TImage=Image< SizeValueType, 3 > >
class HistogramToIntensityImageFilter:
  public HistogramToImageFilter< THistogram, TImage,
                                 Function::HistogramIntensityFunction< SizeValueType, typename TImage::PixelType > >
{
public:
  ITK_DISALLOW_COPY_AND_ASSIGN(HistogramToIntensityImageFilter);

  /** Standard class type aliases. */
  using Self = HistogramToIntensityImageFilter;

  /** Standard "Superclass" type alias. */
  using Superclass = HistogramToImageFilter< THistogram, TImage,
                                 Function::HistogramIntensityFunction< SizeValueType, typename TImage::PixelType > >;

  //using FunctorType = typename Function::HistogramIntensityFunction;
  using Pointer = SmartPointer< Self >;
  using ConstPointer = SmartPointer< const Self >;

  /** Run-time type information (and related methods).   */
  itkTypeMacro(HistogramToIntensityImageFilter, HistogramToImageFilter);

  /** Method for creation through the object factory. */
  itkNewMacro(Self);

protected:
  HistogramToIntensityImageFilter() = default;
  ~HistogramToIntensityImageFilter() override = default;
};
} // end namespace itk

#endif


text:
<?xml version="1.0" encoding="UTF-8"?>
<!--

  Copyright (C) Christian Schulte, 2005-206
  All rights reserved.

  Redistribution and use in source and binary forms, with or without
  modification, are permitted provided that the following conditions
  are met:

    o Redistributions of source code must retain the above copyright
      notice, this list of conditions and the following disclaimer.

    o Redistributions in binary form must reproduce the above copyright
      notice, this list of conditions and the following disclaimer in
      the documentation and/or other materials provided with the
      distribution.

  THIS SOFTWARE IS PROVIDED "AS IS" AND ANY EXPRESS OR IMPLIED WARRANTIES,
  INCLUDING, BUT NOT LIMITED TO, THE IMPLIED WARRANTIES OF MERCHANTABILITY
  AND FITNESS FOR A PARTICULAR PURPOSE ARE DISCLAIMED. IN NO EVENT SHALL
  THE AUTHORS OR COPYRIGHT HOLDERS BE LIABLE FOR ANY DIRECT, INDIRECT,
  INCIDENTAL, SPECIAL, EXEMPLARY, OR CONSEQUENTIAL DAMAGES (INCLUDING, BUT
  NOT LIMITED TO, PROCUREMENT OF SUBSTITUTE GOODS OR SERVICES; LOSS OF USE,
  DATA, OR PROFITS; OR BUSINESS INTERRUPTION) HOWEVER CAUSED AND ON ANY
  THEORY OF LIABILITY, WHETHER IN CONTRACT, STRICT LIABILITY, OR TORT
  (INCLUDING NEGLIGENCE OR OTHERWISE) ARISING IN ANY WAY OUT OF THE USE OF
  THIS SOFTWARE, EVEN IF ADVISED OF THE POSSIBILITY OF SUCH DAMAGE.

  $JOMC$

-->
<module xmlns="http://jomc.org/model"
        xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance"
        xsi:schemaLocation="http://jomc.org/model http://jomc.sourceforge.net/model/jomc-1.2.xsd"
        name="${project.name}" version="${project.version}" vendor="${project.organization.name}"
        modelVersion="1.2">

  <documentation defaultLanguage="en">
    <text language="en">${project.description}</text>
  </documentation>

  <implementations>
    <!-- *********************************************************************************************************** -->
    <implementation identifier="org.jomc.sequences.it.SequenceDirectoryTest"
                    class="org.jomc.sequences.it.SequenceDirectoryTest" classDeclaration="true"
                    name="${project.name}" vendor="${organization.name}" version="${project.version}"
                    createDate="2009-07-27T19:56:45Z">

      <documentation defaultLanguage="en">
        <text language="en">Testcase for SequenceDirectory implementations.</text>
      </documentation>
      <authors>
        <author name="Christian Schulte" version="1.0" mail="schulte2005@users.sourceforge.net"
                createDate="2005-07-25T19:56:45Z"/>

      </authors>
      <dependencies>
        <dependency name="SequenceDirectory" identifier="org.jomc.sequences.SequenceDirectory" version="1.0"
                    optional="true"/>

      </dependencies>
      <properties>
        <property name="sequenceNameMinLength" type="int" value="1">
          <documentation defaultLanguage="en">
            <text language="en">Minimum required length of a sequence name.</text>
          </documentation>
        </property>
        <property name="sequenceNameMaxLength" type="int" value="200">
          <documentation defaultLanguage="en">
            <text language="en">Maximum allowed length of a sequence name.</text>
          </documentation>
        </property>
      </properties>
    </implementation>
    <!-- *********************************************************************************************************** -->
    <implementation identifier="org.jomc.sequences.it.SequenceOperationsTest"
                    class="org.jomc.sequences.it.SequenceOperationsTest" classDeclaration="true"
                    name="${project.name}" vendor="${organization.name}" version="${project.version}"
                    createDate="2009-07-27T19:56:45Z">

      <documentation defaultLanguage="en">
        <text language="en">Testcase for SequenceOperations implementations.</text>
      </documentation>
      <authors>
        <author name="Christian Schulte" version="1.0" mail="schulte2005@users.sourceforge.net"
                createDate="2005-07-25T19:56:45Z"/>

      </authors>
      <dependencies>
        <dependency name="SequenceDirectory" identifier="org.jomc.sequences.SequenceDirectory" version="1.0"
                    optional="true"/>
        <dependency name="SequenceOperations" identifier="org.jomc.sequences.SequenceOperations" version="1.0"
                    optional="true"/>
      </dependencies>
    </implementation>
  </implementations>
</module>


text:
2012 October 25 - 27 Vintage Sports Collectibles Signature Auction- DallasAuction #7065
1880's Jake Kilrain vs. Jem Smith Original Cabinet Photograph....
1880's Jake Kilrain vs. Jem Smith Original Cabinet Photograph. The rather charming original cabinet photograph belies the agonizing brutality of the historic December 19, 1887 between Jake Kilrain and Jem Smith. In a cold, drizzling rain, Kilrain and Smith scrapped with bare knuckles through an ungodly 106 rounds in a bout that lasted nearly three hours. No more than seventy-nine spectators, each of whom had paid the princely sum of $200 for his ticket, were in attendance, including the Marquis of Queensberry who was among the overwhelming British crowd favoring Smith. In the 106th round, Kilrain was able to knock Smith off his feet, seemingly for good, but before the referee could declare victory for the American, the crowd forced him to declare the fight a draw due to darkness. The original image measures at 4.25x6.5" and it exhibits Good condition, with a good amount of creasing throughout.
25th-27th Thursday-Saturday
Heritage auctions is more than just an auction business. They are in the 'preserving history' business as well.
Alicia Kramer, Daughter of Jerry Kramer, five-time NFL World Champion,


Figueroa Street is a major north-south street in Los Angeles County, California, spanning from the Los Angeles neighborhood of Wilmington north to Eagle Rock.
The street is named for General José Figueroa (1792 – September 29, 1835), governor of Alta California from 1833 to 1835, who oversaw the secularization of the missions of California.
Figueroa Street (left), Harbor Freeway (center), and S Broadway (right) night aerial from the south, with downtown Los Angeles in the distance. Figueroa Street turns and become less visible at the University of Southern California.
One of the longer streets in the city, it runs in a north/south direction for more than 30 miles (48 km) from its southern terminus at Harry Bridges Boulevard in the Wilmington neighborhood to Chevy Chase Drive in the city of La Cañada Flintridge at the north end.
From its south end at Harry Bridges Boulevard to Downtown Los Angeles, Figueroa Street runs north parallel to the Harbor Freeway (Interstate 110) in South Los Angeles. The only portion of this segment of Figueroa Street that lies outside Los Angeles city limits is in the city of Carson. South of the Los Angeles Financial District, Figueroa Street passes well-known locations including the University of Southern California, the Los Angeles Convention Center, and Staples Center/L.A. Live.
After passing through downtown Los Angeles near Bunker Hill and South Park, the southern portion of Figueroa Street ends near the overcrossing of Sunset Boulevard over the Arroyo Seco Parkway (SR 110). The street resumes at San Fernando Road in Cypress Park. An early routing of Figueroa Street in this area was originally part of U.S. Route 66, today a part of the Arroyo Seco Parkway (State Route 110). The noted Figueroa Street Tunnels were once a part of that same stretch of roadway.
After resuming at San Fernando Road, Figueroa continues to run roughly parallel to the Arroyo Seco Parkway until it reaches York Boulevard in Highland Park. Afterwards, it heads north to its terminus with the Ventura Freeway (State Route 134). A short, unconnected continuation of Figueroa runs from just south of Marengo Drive in Glendale to end at Chevy Chase Drive just over the city limit line in La Cañada Flintridge.
Early maps produced by the Automobile Club of Southern California measured distances to Los Angeles from the club's headquarters at the intersection of Figueroa Street with Adams Boulevard.
On April 2, 2011, a portion of Figueroa Street at Jefferson Boulevard was blocked off for the "Orange Carpet" and the grandstand for the broadcasting of the 2011 Kids' Choice Awards.
Figueroa was originally called Calle de los Chapules (or Grasshopper Street). Later in the 1880s it became known as "Pearl Street". The section of what is now Figueroa in Highland Park above Avenue 39 was known as "Pasadena Avenue" until Figueroa was extended through Elysian Park. The portion of what is now Figueroa between the Los Angeles River and Avenue 39 was originally known as Dayton Avenue until the Arroyo Parkway went through.
Metro Local Line 81 bus stop at Figueroa and 65th Streets, Los Angeles, California. Metro Local Line 81 runs on Figueroa Street.
The Metro Green and Silver lines operates a station underneath Interstate 105 at Figueroa Street. Metro Local Line 81 operates on Figueroa Street between Colorado Boulevard and Interstate 105 and Torrance Transit Line 1 between Interstate 105 and the Harbor Gateway Transit Center. The Metro Expo Line has 5 stations nearby. Two of which are shared with the Metro Blue Line, one being a major hub connecting to the Metro Red and Purple lines. The Metro Silver Line runs on Figueroa Street in Gardena and between 23rd and 6th Streets in Downtown: Northbound Silver line trips to El Monte Bus Station continue north on Figueroa Street to serve the 7th Street / Metro Center and finally turn right on 6th street, leaving Figueroa Street. Southbound Silver Line trips to Harbor Gateway Transit Center or San Pedro run south on Flower Street from 5th Street to the Harbor Transitway. There are 6 Silver line street stops located on Figueroa Street: Figueroa/190th/Victoria; Northbound only: Figueroa/23rd, Figueroa/Washington, Figueroa/Pico, Figueroa/Olympic, and Figueroa/7th . In addition, there are 7 Metro Silver Line Stations served on the Harbor Transitway and Harbor Freeway close to Figueroa Street: 37th Street/USC, Slauson, Manchester, Harbor Freeway, Rosecrans, Carson, and Pacific Coast Hwy. The Harbor Transitway is located on the I-110 freeway, between Figueroa Street and Broadway. The Lincoln/Cypress Station for the Metro Gold Line on Avenue 26 at its intersection with Lacy Street is about a 5-minute walk from Figueroa Street.
^ Lucas, Greg (2011-09-29). "The Streets of Los Angeles". California's Capitol. Retrieved 2017-05-16. Grasshopper Street – Calle de los Chapules – is now Figueroa which, in turn, is a tip of the hat to the former Mexican Governor Jose Figueroa.
^ "California Proposition 1C, Bonds for Housing (2006)". Retrieved 2017-05-16.
^ Barragan, Bianca (2016-06-10). "Bikeable, Walkable Figueroa Actually Is Coming Soon". Curbed. Retrieved 2017-05-16.
^ "What is MyFig?". myfigueroa.com. Retrieved 2017-05-16.
^ "Stage 1: Vision, Games Concept and Strategy" (PDF). la24.org. 2016-02-12. Retrieved 2017-05-16.
Wikimedia Commons has media related to Figueroa Street.


text:
On View Events Shop Visit Learn Donate
We are open today from 11 am to 8 pm.
Vaccination proof required.
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Christian Dior (French, 1905–1957). Bar suit, afternoon ensemble with an ecru natural shantung jacket and black pleated wool crepe skirt. Haute Couture Spring–Summer 1947, Corolle line. Dior Héritage collection, Paris. (© Katerina Jebb)
Member Morning: Christian Dior: Designer of Dreams
10–10:45 am
Beaux-Arts Court, 3rd Floor
Enjoy a Members-only morning viewing of the exhibition.
Andy Warhol (American, 1928–1987). The Last Supper (Detail), 1986. Screenprint and colored graphic art paper collage on HMP paper, 315/8 × 233/4 in. (80.3 × 60.3 cm). The Andy Warhol Museum, Pittsburgh; Founding Collection, Contribution The Andy Warhol Foundation for the Visual Arts, Inc., 1998.1.2125. © 2021 The Andy Warhol Foundation for the Visual Arts, Inc. / Licensed by Artists Rights Society (ARS), New York
Member Morning: Andy Warhol: Revelation
Morris A. and Meyer Schapiro Wing, 4th and 5th Floors
Art History Happy Hour: Modern Gothic
6–7:30 and 8–9:30 pm
Virtual Open House: Adult Literacy at the Brooklyn Museum
10 am–12 pm
Artist's Eye: Modern Gothic
Studio Art: Printmaking Inspired by Andy Warhol
Sat, February 5, 2022
Brooklyn Talks: White Shoes with Nona Faustine
Fri, February 11, 2022
Art-Making
The Brooklyn Museum stands on land that is part of the unceded, ancestral homeland of the Lenape (Delaware) people.
About Collection Contact Us Press FAQ Copyright


Mabima Metal Quarry – Environment Foundation (Guarantee) Limited.
This is a revision application against an action for public nuisance which was instated under Section 98 of the Criminal Procedure Code Act upon a complaint made to the Sapugaskanda Police Station about the operation of a metal crushing machine in a metal quarry in the Mabima area. The Petitioner states that the Magistrate ordered the Central Environmental Authority (CEA) to produce a report on the matter and that it disclosed that the metal quarry was capable enough to be continued without causing a nuisance to the residents nearby. Parties entered into a settlement before the magistrate. The Petitioner stated that the Plaintiff in this matter has sold his land which was situated closer to the metal quarry and therefore no nuisance was caused to him by the operation of this quarry and sought for an order for the settlement of terms to be dispensed with. The learned Magistrate refused the application and accordingly, the Petitioner filed this revision application.
The Environmental Foundation Limited (EFL) provides legal assistance to the Respondents of this matter and stated that the public nuisance still continues and that the continuous metal quarry operation using crushing machines would cause severe environmental damage as well. Therefore the application was sought to be dismissed and the case was brought before the High Court of Gampaha. The High Court of Gampaha declared that the matter be returned to the Gampaha Magistrates Court. The Gampaha Magistrates Court decided in favour of the Petitioners and ordered the Metal Quarry to cease operation.


Now instead of buying a textbook and signing up for the other tools, students just have to register for an edX Edge account to get started on the physics course.
At STLHE 2015, Dr. L. Dee Fink discussed five high impact teaching practices that have the most potential to transform teaching and learning in higher education.
Learn how community-based experiential learning and team-based learning approaches are being used in the Faculty of Land and Food Systems.
Catch up on all the latest STLHE commentary!
Come see how flexible learning is enhancing learning opportunities at our new website. On it you will find information and resources related to how the UBC teaching community is transforming teaching and learning at the University.
Registration is Now Open for the CTLT Spring Institute!
Join us May 26-27, 2015, at the annual CTLT Spring Institute! The theme for this year's Institute is inquiry-based experiential learning, with sessions focusing on flexible learning, community-based experiential learning, and open education.