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nycroberts@yahoo.com
Michael Roberts (book, music, lyrics) is the composer/lyricist of GOLDSTEIN, produced Off-Broadway in 2018 at The Actors Temple Theater (Off Broadway Alliance Nomination for Best Musical).
He is best known as the author, composer and lyricist of GOLF: THE MUSICAL (2003-2004 Off-Broadway, 2006 and 2010 Off-Broadway revivals, and 40 national and international productions).
Other productions include THE FARTISTE (winner: Best Musical—2006 NY Fringe, Off-Broadway and London productions), and GREED Off-Broadway at New World Stages (2014).
Film music includes Tri-Star Pictures' LOVE WALKED IN (w/ Denis Leary). Television music includes three seasons on AMC's Emmy-winning sitcom REMEMBER WENN.
Roberts has music directed for Betty Buckley, Donna Murphy, Rupert Holmes, Lesley Gore, Stephanie Mills, and Joan Rivers, and has worked with directors Hal Prince, Susan Stroman and Kathleen Marshall.
Roberts was a nominee for a Broadway World Award, The Off Broadway Alliance Award, and is a three-time recipient of the ASCAP Plus Award.
He is published by Samuel French, Stage Rights, Leiden & Company Music Publishing, and Wendy Isobel Music.
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synom

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Macro synom::<wbr>peek

                       [−]

               [src]

macro_rules! peek {
    ($i:expr, $submac:ident!( $($args:tt)* )) => { ... };
    ($i:expr, $f:expr) => { ... };
}

Parse a value without consuming it from the input data.

Syntax: peek!(THING)

Output: THING

extern crate syn;
#[macro_use] extern crate synom;

use syn::Expr;
use syn::parse::{ident, expr};
use synom::IResult;

// Parse an expression that begins with an identifier.
named!(ident_expr -> Expr,
    preceded!(peek!(ident), expr)
);

fn main() {
    // begins with an identifier
    let input = "banana + 1";
    let parsed = ident_expr(input).expect("ident");
    println!("{:?}", parsed);

    // does not begin with an identifier
    let input = "1 + banana";
    let err = ident_expr(input);
    assert_eq!(err, IResult::Error);
}

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redpajama_set_name: RedPajamaGithub


text:
Results From Preseason Week 2
August 19, 2013 by Micheal Savoie
Are you ready for Monday Night?
On their trip to Baltimore to take on the Ravens, the Falcons played a strong 3 quarters before the Raven backups scored 20 unanswered points in the fourth quarter to win the game 27-23
Matt Ryan threw for 97 yards, while Joe Flacco threw for 118 yards. One of those included a 77 yarder to Torrey Smith for a touchdown.
In Cleveland, the Brown's QB Brandon Weeden threw two touchdown passes and a total of 117 yards in a 24-6 victory over the Detroit Lions.
The Philadelphia Eagles beat the Carolina Panthers 14-9 with both Michael Vick and Nick Foles looking good in their competition for the starting QB position.
The San Diego Chargers flew to Chicago for an evening with the Bears, but came away with a loss after losing the turnover battle. Final score Bears 33-Chargers 28.
The Minnesota Vikings fell to the Buffalo Bills 20-16 in Buffalo as Bill's rookie quarterback E j Manuel played a nearly flawless 3 series to help solidify his chances of landing the starting QB job. Bill's management reported that Manuel's knee needed a surgical procedure, however, and that he would likely miss the balance of this preseason. Stay tuned…
In a rough game between the San Francisco 49ers and the Kansas City Chiefs, the 49ers won 15-13, with former 49ers QB Alex Smith struggling to complete passes.
The Tampa Bay Buccaneers traveled to New England to face a Patriots squad that is running on all 11 cylinders as the Buccaneers offense struggled for two series before the backups came in and started moving the ball. The Patriots held on to win 25-21.
Drew Brees passed for 202 yards and a touchdown on 5 scoring drives in the first half to carry the New Orleans Saints to a 28-20 victory over the Oakland Raiders.
Meanwhile in Arizona, the Cardinals forced 6 turnovers (5 in he first half) to defeat the Dallas Cowboys 12-7. The defense of Arizona showed great promise, and the offense showed that they could move the ball, although they settled for 4 field goals out of 5 drives… so they still have some work to do.
The Cincinnati Bengals hosted the Tennessee Titans and treated them to a 27-19 defeat behind the great performances of rookie running back Giovani Bernard and veteran QB Andy Dalton.
Jacksonville Jaguars' QB Blaine Gabbert played led the Jaguars on two scoring drives to New York Jets QB Mark Sanchez' 5 scoring drives as the Jets pelted the Jags with a 37-13 win at home!
The Miami Dolphins traveled to Houston to play the Houston Texans in a game they are most likely wishing they could have over. Not only did they lose 24-17, but they lost veteran tight end Dustin Keller with a horrific knee injury.
The Green Bay Packers defeated the St Louis Rams 19-7 in a game where the touchdowns were held until the 4th quarter.
The Seattle Seahawks showed that they have improved over last year with a 40-10 defeat of the Denver Broncos. The Seahawks defense forced 3 turnovers, but the injury bug is what appears to be plaguing the Broncos after losing defensive end Derek Wolfe and right guard Louis Vasquez in Saturday night's game.
And finally, the Indianapolis Colts beat the New York Giants 20-12 against a struggling Giants red zone offense.
In tonight's Monday Night Football game, the Pittsburgh Steelers travel to Washington DC to face the Redskins.
That's it, you are up to date!
Current Football Podcasts at Blog Talk Radio with MichealSavoie on BlogTalkRadio
Filed Under: Scores and Highlights Tagged With: Monday Night Football, Preseason Football

redpajama_set_name: RedPajamaCommonCrawl


Nestled along the banks of the Concord, Sudbury and Assabet Rivers, the historic town of Concord offers residents a scenic, natural backdrop that's rarely seen in today's fast-paced, high-tech society. With sparkling ponds, dozens of historic sites and a literary and political past that few American towns can boast, it remains one of the most sought-after places to live in all of Massachusetts.
And though it might be small in size and population, this charming town is anything but boring. Just a mere 20 miles from Boston, big-city amenities are well within reach from Concord. Hop in the car, hit the road, and adventure awaits in minutes.
Not all fun is as far as car ride away, though. Within the town's boundaries, you'll find everything from museums and wildlife refuges to book stores, art houses, libraries, and recreation centers. The picture perfect town center also offers several opportunities for an outing. With everything from high-end vintage thrift stores to comforting diners, this town center is one that is constantly buzzing with activity. Aside from everyday attractions in Concord, there are community events, like Breakfast with Santa, a Thanksgiving food drive and more. It's a veritable hub of activity for the whole family.
But Concord wasn't always this booming. First settled by British colonists in 1635, the town was purchased from local Algonquian tribe members. In 1775, when the Battle of Lexington and Concord took place, it became a major part of the Revolutionary War. The legendary Paul Revere warned townspeople of the oncoming British attack, allowing them to prepare and take up arms. The town has since been immortalized in various books, songs and speeches, making it one of the most memorable names in the state.
Though Concord certainly has a rich history, its bountiful education opportunities give it a bright future, too. Students in the area attend Concord-Carlisle Regional High School, a school often regarded as one of the best in all of Massachusetts. In fact, according to Boston Magazine, it was the No. 1 school for cost efficiency and No. 3 in academic performance. It also boasts a variety of extracurricular and athletic opportunities, with organizations for fencing, swimming, skiing, football, soccer, golf, band, choir and theater.
If a town this rich in history and amenities seems like it is in demand, it is. With higher real estate costs than towns further out from Boston, Concord's housing stock is prime, and prices have climbed steadily over the past few years. While there is a possibility of owning a starter home in Concord for around half a million dollars or more, those looking for a roomier estate, sprawling properties with three, four and five bedrooms, as well as expansive lots, can expect to look for a home that will run upwards of $800k. The average home price in the area is about $482,000, which also includes condos, which go for a minimum of $250k for around 1,000 square feet.
With a seasoned history and storied past, Concord offers the perfect backdrop for growing, living and thriving.


Our activities are concentrated on the production and distribution of gold-coated substrates and thin films for the physical and life sciences.
Surrounded with excellent know-how and research expertise, PHASIS has also a strong commitment to applied research and device development.
Covering applications from microscopy to chemical sensors, PHASIS offers tailored expertise for high-tech applications using new materials, micro- and nanometric surface modifications, and thin film technology.


Biography Channel Digital Media Producer, Tien Brunelle, is in pre-production of a four-minute mini-biography segment on Judy Garland that will run on the internet. Other such segments have been done on Truman Capote, Elvis Presley, Billie Holiday, Bessie Smith, Bob Dylan, Jackie Robinson, John Galliano, Melissa Etheridge, and others. Talks are currently underway with participating interviewees. For a full listing of all the Mini-Bios that have been done at Biography Channel, go to: www.biography.com/videos/mini-bios.


The New Jersey Chapter of the Arthritis Foundation has taken a leadership role in women's healthcare and in ensuring optimal care for people living with arthritis-the majority of whom are women.
Although arthritis can affect anyone, regardless of race gender or age, the statistics are clear - over 60% of the nearly 50 million people who have arthritis are female. Women with arthritis, compared to men, account for a greater percentage of visits to physician's offices, hospitalizations, and disability claims.
Lupus is an inflammatory disease that may affect the joints, skin, kidneys and other parts of the body. It usually affects women of childbearing age. Almost 322,000 Americans-90% of whom are women-have this arthritis-related condition.
Finding ways to cope with arthritis is particularly important for women, who are often juggling work, family, care taking and a variety of social responsibilities. With all of these challenges, it is important for women with arthritis to be sure to take the time to care for themselves and stay positive.
The New Jersey Chapter is aware of these very real issues and offers a variety of educational programs, patient services, and financial assistance programs that help improve the outlook and lives of women with arthritis.
The Arthritis Foundation leads the way in helping people with arthritis live better today and create better tomorrows through new treatments, better access to healthcare and, ultimately, cures.
Helping to Find a program or information session that interests you.


text:
namespace chromeos_update_engine {

static const char* const kUpdateEngineServiceName = "com.coreos.update1";
static const char* const kUpdateEngineServicePath =
    "/com/coreos/update1";
static const char* const kUpdateEngineServiceInterface =
    "com.coreos.update1.Manager";
}  // namespace chromeos_update_engine

#endif  // CHROMEOS_PLATFORM_UPDATE_ENGINE_DBUS_CONSTANTS_H__


The Avaya-certified VoIP Intercom is ip65 rated and made from a robust stainless steel. Its perfect for site (Gates & Barriers) and door entry points. The Avaya VoIP Intercom (also known as a Door phone) effortlessly integrates and registers to the Avaya IP-PBX (also known as a sip server). Once converged to the Avaya network as an approved 3rd party SIP extension, you can make calls from and to the SIP Intercom. Hardware parts are minimal, only the Intercom is required. Connection to the LAN is ultra fast on a single cat5e cable to a POE port. Configuration parameters are set on its auto IP addressed web-page. True IP and VoIP Solution. On-board DTMF controlled relay, set up on Safari, IE, Firefox or Chrome. Plug and Play installation. None Keypad version is available. Also works with over 35 other IP Phone Systems.
This is the outdoor version Avaya VoIP Intercom. Made by CyberData corporation and distributed by iEntry Systems, based in Manchester. CyberData is a USA based manufacturer of SIP Secure Entry & VoIP Paging Systems. Established in 1974 they have a reputation of building high end, high quality and well thought out products. CyberData has passed interoperability testing with Avaya many years ago and has been deployed in various Avaya SIP and VoIP based door/site entry applications since. Probably one of the most popular manufacturers this solution is installed in, along with Cisco. If your IP Phone platform is not an Avaya one, Cyberdata has passed interoperability testing with another 35+. See that list here.
Just some options to operate the on-board relay: Telephone Dial Mode + Speed Dial Mode + Call Button – IP Phones and Mobile take the call and open the door/gate/barrier. Also secure PIN code entry.
Where as many Intercom manufacturers need several other parts to complete the system: (Software, Software Clients, Tied in handsets, Servers, Control Units, Separate relays, etc). CyberData only needs the Avaya VoIP Intercom. The installation is very straight forward and simple. For connecting hardware, you run a data cable into the device. Connect this back to a spare POE port in your local POE (Power Over Ethernet) switch. You have now completed the physical hardware installation. The Avaya VoIP Intercom has an integrated I/O board (relay) for triggering Doors, Gates and Barriers.
Avaya VoIP Intercom key Features.
The Avaya VoIP Intercom registers to the Avaya SIP Server. This is effortlessly set up through the intercoms auto configured ip address. From the Avaya side, you would simply set the SIP based intercom up as a new phone extension and user, that can make and receive calls.


text:
\section{Introduction}

Aspects of the $\Gr(4,n)$ Grassmannian cluster algebras~\cite{FZ1, GSV, scott2006grassmannians} have been found to play several still rather mysterious roles in the mathematical structure of scattering amplitudes in planar $\mathcal{N}=4$ Yang-Mills theory~\cite{Golden:2013xva, Golden:2014xqa}. A simple toy model which serves as a nice playground for studying features of this cluster structure is the $m=2$ version of the theory, where the momentum twistors describing the kinematic scattering data~\cite{Hodges:2009hk} are restricted to lie in a $\mathbb{P}^1$ subspace\footnote{Note that this is quite different from restricting to two space-time dimensions.} of the usual $\mathbb{P}^3$. The associated $\Gr(2,n) \cong A_{n-3}$ cluster algebra\footnote{This algebra has also been found to govern the structure of $\mathcal{N}=4$ Yang-Mills amplitudes in the multi-Regge limit~\cite{DelDuca:2016lad}.} is completely understood~\cite{FZ1}: its clusters are in one-to-one correspondence with the triangulations of an $n$-gon, and the coordinates in each cluster are in one-to-one correspondence with edges in the corresponding triangulation. The positive geometry \cite{Arkani-Hamed:2017tmz} associated to these $m=2$ amplitudes is the amplituhedron $\mathcal{A}_{n,k}^{(2)}$, which is an interesting object on its own, and its many interesting features were studied e.g.~in~\cite{karp2017decompositions,Galashin:2018fri,Ferro:2018vpf,Lukowski:2019kqi}.

In this paper we explore the conjecture~\cite{Drummond:2018dfd,Mago:2019waa} that the poles of every rational Yangian invariant are given by cluster coordinates, a property referred to as \emph{cluster adjacency} following~\cite{Drummond:2017ssj}. The full $\mathcal{N}=4$ Yang-Mills theory has a whole zoo of $n$-particle N${}^k$MHV Yangian invariants (see Chapter~12 of~\cite{ArkaniHamed:2012nw} for a discussion of their classification), and evidence supporting this conjecture is so far restricted to relatively small $n$ and $k$. In contrast, in the $m=2$ toy model, by using the amplituhedron formulation of scattering amplitudes~\cite{Arkani-Hamed:2013jha}, we are able to write down an explicit formula for all Yangian invariants for any $n$ and $k$. Each N${}^k$MHV invariant is labelled by a collection of $k$ non-intersecting triangles inside an $n$-gon, with denominator factors corresponding precisely to edges of the triangles. Consequently, the result manifestly satisfies cluster adjacency with respect to the $\Gr(2,n)$ cluster algebra.

\section{Classification of Yangian Invariants for \texorpdfstring{$m=2$}{m=2}}
\label{sec:2}

\emph{Yangian invariants} are basic building blocks for many amplitude-related quantities of interest (see for example~\cite{Mason:2009qx, ArkaniHamed:2009vw, ArkaniHamed:2009dg, ArkaniHamed:2009sx, Drummond:2010uq, Ashok:2010ie, ArkaniHamed:2012nw, Drummond:2010qh,Ferro:2016zmx}).
The classification of $\mathcal{N}=4$ Yang-Mills invariants (i.e., $m=4$) is discussed in Sec.~12 of~\cite{ArkaniHamed:2012nw}. In this section we discuss the classification of the analogous set of Yangian invariants for $m=2$. We will see that they can all be associated to the so-called generalised triangles, which are the building blocks for triangulations of the amplituhedron space $\mathcal{A}_{n,k}^{(2)}$. Then the Yangian invariants can be extracted from canonical differential forms with logarithmic singularities on all boundaries of generalised triangles.  For $k=1$  there is a unique type of Yangian invariants of the form~\eqref{omegaabc} which trivially corresponds to a triangle in $\mathbb{P}^2$~\cite{ArkaniHamed:2010gg}. For $k=2$ there are two types of Yangian invariants, \eqref{omegaabcabc} and~\eqref{omegaabcd}, which we will see correspond respectively to two non-intersecting triangles or to two triangles glued along an edge to form a quadrilateral. A general configuration of $k$ non-intersecting triangles corresponds to the general N${}^k$MHV Yangian invariant shown in~\eqref{allYangian}.

\subsection{Review}

We recall that the (tree-level) amplituhedron ${\cal{A}}_{n,k}^{(m)}$ is defined~\cite{Arkani-Hamed:2013jha} as the image of the positive Grassmannian $G_+(k,n)$ under the linear map
\begin{align}\label{eq:amplituhedronmap}
C \in G_+(k,n) \quad \to \quad
Y = C \cdot Z^{\rm T} \in G(k,k+m)\,,
\end{align}
for generic positive matrix $Z \in M_+(k+m,n)$. If $\mathcal{C}$ is a positroid cell in $G_+(k,n)$, $Z(\mathcal{C})$ is its image under the amplituhedron map, and $\Omega_{\mathcal{C}}$ is the unique canonical differential form~\cite{Arkani-Hamed:2017tmz} on $G(k,k+m)$ with logarithmic singularities (only) on the boundaries of $Z(\mathcal{C})$. Then $\Omega_{\mathcal{C}}$ provides the Yangian invariant associated to $\mathcal{C}$ defined directly in the amplituhedron space, as explained in \cite{Ferro:2016zmx}. Alternatively, Yangian invariants can be represented as certain residues, or contour integrals, of the top form on $\Gr_+(k,n)$~\cite{Mason:2009qx, ArkaniHamed:2009vw}. The connection between these two ways of representing Yangian invariants is laid out in Sec.~7 of~\cite{Arkani-Hamed:2013jha}. For our purposes, the significant advantage of using the amplituhedron construction is that it enables us to write down the completely general formula for $\Omega_{\mathcal{C}}$, given below in~\eqref{allYangian}.

In the remainder of this paper we specialise to $m=2$. The Yangian invariants considered in this paper correspond to $2k$-dimensional positroid cells in $G_+(k,n)$ whose images under the amplituhedron map are also $2k$-dimensional. We refer to the images of such positroid cells as \emph{generalised triangles}, and will denote them as $\mathcal{T}^{(n)}$. We will see that all Yangian invariants associated to generalised triangles can be labelled by collections of triangles in an $n$-gon. In the following we use $T^{(n)}_{abc}$ to denote the triangle with vertices $\{a,b,c\}$ inside a convex $n$-gon (see for example Fig.~\ref{fig:triangle}). We say that two triangles are {\it non-intersecting} if their interiors are disjoint, but we allow non-intersecting triangles to share an edge or a vertex.

\begin{figure}
\begin{center}
\includegraphics[scale=0.3]{triangle.pdf}
\end{center}
\caption{Triangle $T^{(8)}_{124}$.}
\label{fig:triangle}
\end{figure}

\subsection{\texorpdfstring{$k=1$}{k=1}}

For $k=1$ we consider the most general 2-dimensional cell in $G_+(1,n)$, which can be parametrised by a positive matrix\footnote{Throughout the following we employ the unfortunately common abuse of notation by writing \emph{positive} instead of \emph{non-negative}.} of the form
\begin{equation}
C_{abc} = \bordermatrix{
&&&a&&b&&c&&\cr
&0&\ldots &\star & \ldots & \star & \ldots & \star & \ldots&0}\,,
\end{equation}
whose only non-zero entries are located in columns $\{a,b,c\}$. The image of such a cell through~\eqref{eq:amplituhedronmap} is an actual triangle with vertices $Z_a,Z_b,Z_c$ in $\mathbb{P}^2$. The corresponding Yangian invariant is~\cite{Arkani-Hamed:2013jha}
\begin{equation}\label{omegaabc}
\Omega_{abc}=\frac{\langle abc\rangle^2}{\langle Yab\rangle \langle Ybc\rangle\langle Yca\rangle}\,,
\end{equation}
and $\Omega_{abc}\, \langle Y d^2 Y\rangle$ is the canonical form with logarithmic singularities only along the three edges of the triangle, i.e.~where one of the brackets $\langle Yab\rangle$, $\langle Ybc\rangle$, or $\langle Yca\rangle$ vanishes. We introduced the following bracket notation
\begin{equation}
\langle a_1a_2\ldots a_{k+2}\rangle=\epsilon_{A_1A_2\ldots A_{k+2}}Z^{A_1}_{a_1}Z^{A_2}_{a_2}\ldots Z^{A_{k+2}}_{a_{k+2}}\,.
\end{equation}

\subsection{\texorpdfstring{$k=2$}{k=2}}

For $k=2$ we consider four-dimensional cells in $G_+(2,n)$. There are three different types of such cells, corresponding to matrix representatives that can be brought, using an appropriate $GL(2)$ transformation, to one of the following three forms:
{\let\quad\thinspace
\begin{align}
C_{a_1,b_1,c_1;a_2,b_2,c_2}& =\bordermatrix{
&&&a_1&&b_1&&c_1&&a_2&&b_2&&c_2&&\cr
&0&\ldots &\star & \ldots & \star & \ldots & \star & \ldots & 0 & \ldots & 0 & \ldots & 0& \ldots& 0\cr
&0&\ldots & 0 & \ldots & 0 &\ldots & 0 & \ldots & \star & \ldots & \star & \ldots & \star & \ldots&0 },\\\nonumber
\\
C_{a_1,b_1,c_1,d_1;a_2,b_2}& =\bordermatrix{
&&&a_1&&b_1&&c_1&&d_1&&a_2&&b_2&&\cr
&0&\ldots &\star & \ldots & \star & \ldots & \star & \ldots & \star & \ldots & 0 & \ldots & 0& \ldots& 0\cr
&0&\ldots & 0 & \ldots & 0 &\ldots & 0 & \ldots & 0 & \ldots & \star & \ldots & \star & \ldots&0 },\\\nonumber\\
C_{a_1,b_1,c_1,d_1,e_1;a_2}& =\bordermatrix{
&&&a_1&&b_1&&c_1&&d_1&&e_1&&a_2&&\cr
&0&\ldots &\star & \ldots & \star & \ldots & \star & \ldots & \star & \ldots & \star & \ldots & 0& \ldots& 0\cr
&0&\ldots & 0 & \ldots & 0 &\ldots & 0 & \ldots & 0 & \ldots & 0 & \ldots & \star & \ldots&0 }.
\end{align}}

In boundary cases some indices could be repeated (for example $c_1$ could equal $a_2$ in the first matrix). Although each type of cell is four-dimensional in $G_+(2,n)$, only the first has a four-dimensional image in $G(2,4)$; it is easy to check that cells of the second or third type have images of dimension three or two, respectively. Therefore, we are interested only in cells parametrised by matrices with three non-zero entries in columns $\{a_1,b_1,c_1\}$ in the first row and three non-zero entries in columns $\{a_2,b_2,c_2\}$ in the second row. We naturally label such a cell by a pair of triangles $T_{a_1b_1c_1}^{(n)}$ and $T_{a_2b_2c_2}^{(n)}$ inside an $n$-gon. The triangles must be non-intersecting, since otherwise the matrix $C_{a_1,b_1,c_1;a_2,b_2,c_2}$ would not be positive.

\begin{figure}[h!]
\begin{center}
\begin{tabular}{ccc}
\includegraphics[scale=0.3]{t1t2.pdf}&\phantom{.}\hspace{2cm}\phantom{.}&
\includegraphics[scale=0.27]{q1.pdf}\\
a)&&b)
\end{tabular}
\end{center}
\caption{Labels for the $k=2$ generalised triangles: a) ${\mathcal{T}}_{a_1,b_1,c_1;a_2,b_2,c_2}^{(n)}$, b) ${\mathcal{T}}_{a_1,b_1,c_1,d_1}^{(n)}$.}
\label{Fig:gen.tr}
\end{figure}

There are two types of generalised triangles parametrised by $C_{a_1,b_1,c_1;a_2,b_2,c_2}$, depending on the choice of indices. If the index sets $\{a_1,b_1,c_1\}$ and $\{a_2,b_2,c_2\}$ have at most one element in common, then the two triangles $T_{a_1b_1c_1}^{(n)}$ and $T_{a_2b_2c_2}^{(n)}$ intersect at most at a single point. This type of configuration is shown in the left panel of Fig.~\ref{Fig:gen.tr}. Note that the image of this cell in $G(2,4)$, which we denote by $\mathcal{T}_{a_1,b_1,c_1; a_2b_2c_2}^{(n)}$, has six codimension-one boundaries, regardless of whether or not the triangles share a vertex.

On the other hand, the two triangles could share an edge, say $a_2 = b_1$, $b_2 = c_1$, and $d_2 = d_1$, such that they form a quadrilateral with vertices $\{a_1, b_1,c_1,d_1\}$, as shown in the right panel of Fig.~\ref{Fig:gen.tr}. The image of this cell, which we denote by $\mathcal{T}_{a_1,b_1;c_1,d_1}^{(n)}$, only has four codimension-one boundaries in $G(2,4)$. Note that there are two ways to form the same quadrilateral by joining triangles, namely $T_{a_1b_1c_1}^{(n)} \cup T_{a_1c_1d_1}^{(n)}$ or $T_{a_1b_1d_1}^{(n)} \cup T_{b_1c_1d_1}^{(n)}$. Employing a $GL(2)$ transformation, one can show that the two corresponding matrices $C_{a_1b_1c_1;a_1c_1d_1}$ and $C_{a_1b_1d_1;b_1c_1d_1}$ parametrise the same cell in $G_+(2,n)$. The fact that both the cell and its image are independent of the triangulation will become important in the following when we describe our labelling of general generalised triangles.

As a concrete example, we provide the complete list of labels for generalised triangles for $k=2$, $n=5$ in Fig.~\ref{Fig:gen.tr.n5k2}.

\begin{figure}[h]
\begin{center}
\includegraphics[scale=0.4]{n5k2.pdf}
\end{center}
\caption{The 10 generalised triangles for $n=5$, $k=2$, together with the permutations that label the corresponding positroid cells in the notation of~\cite{ArkaniHamed:2012nw}.}
\label{Fig:gen.tr.n5k2}
\end{figure}

The Yangian invariants associated to the six- and four-boundary types of generalised triangles are respectively
\begin{align}
\Omega_{a_1b_1c_1;a_2b_2c_2}&=\frac{\langle Y(a_1b_1c_1)\cap(a_2b_2c_2)\rangle^2}{\langle Ya_1b_1\rangle \langle Yb_1c_1\rangle\langle Yc_1a_1\rangle\langle Ya_2b_2\rangle \langle Yb_2c_2\rangle\langle Yc_2a_2\rangle}\,,
\label{omegaabcabc}
\\
\Omega_{a_1b_1c_1d_1}&=\frac{\langle a_1b_1c_1d_1\rangle^2}{\langle Ya_1b_1\rangle \langle Yb_1c_1\rangle\langle Yc_1d_1\rangle\langle Yd_1a_1\rangle }\,,
\label{omegaabcd}
\end{align}
where we define $\langle Y(a_1b_1c_1)\cap(a_2b_2c_2)\rangle=\langle Y_1 a_1 b_1c_1\rangle \langle Y_2a_2b_2c_2\rangle-\langle Y_2 a_1 b_1c_1\rangle \langle Y_1a_2b_2c_2\rangle$.

To summarise, the generalised triangles for $k=2$ are in one-to-one correspondence with pairs of non-intersecting triangles $T^{(n)}_{a_1 b_1 c_1}$ and $T^{(n)}_{a_2 b_2 c_2}$ that share at most a vertex, together with quadrilaterals $\lbrace a_1,b_1,c_1,d_1\rbrace$ inside an $n$-gon. Furthermore, boundaries of generalised triangles, and hence singularities of respective Yangian invariants, are in correspondence with edges of these configurations. Therefore, the set of singularities of Yangian invariants for $k=2$ correspond to a set of non-intersecting diagonals (and possible external edges) of an $n$-gon.

\subsection{General \texorpdfstring{$k$}{k}}

For general $k$ there exists a natural generalisation of the labelling we encountered for $k=1,2$. Generalised triangles are images of $2k$-dimensional positroid cells inside $G_+(k,n)$ which can be parametrised by a matrix $C_{a_1 b_1 c_1;\ldots;a_k b_k c_k}$ whose $\alpha^{\rm th}$ row has non-zero entries only in columns $\{a_\alpha,b_\alpha,c_\alpha\}$, with $\alpha=1,\ldots,k$. To this set of indices we associate the union of the $k$ non-intersecting triangles $T^{(n)}_{a_1 b_1 c_1}, \ldots, T^{(n)}_{a_k b_k c_k}$ inside an $n$-gon. If no pair of triangles share a common edge then we denote the generalised triangle associated to this configuration by $\mathcal{T}^{(n)}_{a_1b_1c_1;\ldots;a_kb_kc_k}$. If two triangles share a common edge then we combine them to form a quadrilateral. If there are more triangles sharing common edges then we remove all shared edges to further combine them into higher polygon inside the $n$-gon. Therefore, generalised triangles of the $m=2$ amplituhedron are in one-to-one correspondence with sets of non-intersecting polygons inside an $n$-gon, with no pair of polygons sharing more than a single vertex. We have checked this statement up to high values of $n$ and $k$ using the {\tt positroid} package~\cite{Bourjaily:2012gy} and we conjecture it is always true. Interestingly, we find that the intersection number (see~\cite{ArkaniHamed:2012nw, Bourjaily:2012gy}) is $\Gamma=1$ in each case. This contrasts with the situation for $m=4$ Yangian invariants, where it is not uncommon to have $\Gamma>1$.

Now it is possible to introduce a generalisation of the formulas~\eqref{omegaabc}, \eqref{omegaabcabc} and~\eqref{omegaabcd} for all Yangian invariants at general $k$. Let $s$ be the total number of polygons, and for $j=1,\ldots,s$ let $p_j$ be the number of edges of the $j^{\rm th}$ polygon. We denote the $j^{\rm th}$ polygon by $P^{(n)}_{a_{j1}, \ldots ,a_{jp_j}}$, where $a_{j1},a_{j2}, \ldots a_{jp_j}$ are its vertices. Since the total number of triangles has to be $k$ and the $j^{\rm th}$ polygon contains $p_j-2$ triangles, we must have $\sum_{j=1}^s (p_j-2)=k$. Then the Yangian invariant associated to the collection of polygons $\lbrace P^{(n)}_{a_{11},\ldots,a_{1p_1}},\ldots,P^{(n)}_{a_{s1},\ldots,a_{sp_s}} \rbrace$ is
\begin{equation}\label{allYangian}
\Omega_{a_{11},\ldots,a_{1p_1};\ldots;a_{s1},\ldots,a_{sp_s}}=\frac{\langle Y (a_{11}\ldots a_{1p_1}) \cap\ldots\cap (a_{s1}\ldots a_{sp_s}) \rangle^2}{(\langle Y a_{11} a_{12}\rangle\ldots\langle Y a_{1p_1} a_{11}\rangle)\ldots(\langle Y a_{s1} a_{s2}\rangle\ldots\langle Y a_{sp_s} a_{s1}\rangle)}\,,
\end{equation}
where we have defined
\begin{multline}
\langle Y (a_{11}\ldots a_{1p_1}) \cap\ldots\cap (a_{s1}\ldots a_{sp_s}) \rangle
\\
= \epsilon_{\alpha^1_1\ldots\alpha^s_{p_s-2}} \langle Y^\perp_{\alpha^1_1\ldots\alpha^1_{p_1-2}} a_{11}\ldots a_{1p_1} \rangle\ldots\langle Y^\perp_{\alpha^s_{1}\ldots\alpha^s_{p_s-2}} a_{s1}\ldots a_{sp_s} \rangle\,,
\end{multline}
and $Y^\perp_{\alpha_1\ldots \alpha_r}$ parametrises the orthogonal complement of a collection of $Y$'s,
\begin{equation}
Y^\perp_{\alpha_1\ldots\alpha_r}=\epsilon_{\alpha_1\ldots\alpha_k} Y_{\alpha_{r+1}} \wedge \ldots \wedge Y_{\alpha_{k}}\,.
\end{equation}

We note that if $p_j=3$ for all $j=1,\ldots,s$, i.e.~all polygons are triangles, then~\eqref{allYangian} agrees with~\cite{Arkani-Hamed:2017tmz}\footnote{Notice a typo in the numerator of formula (7.52) in~\cite{Arkani-Hamed:2017tmz}: the power should be $2$ instead of $k$.}. Moreover, the formula~\eqref{allYangian} nicely encodes the geometry of the set of polygons of our labels. The building blocks of the numerator are $s$ brackets, one for each polygon, with the $j^{\rm th}$ bracket containing all the vertices of the $j^{\rm th}$ polygon. Meanwhile the denominator has exactly $s$ factors, and the $j^{\rm th}$ factor is a product of brackets of the type $\langle Y ab \rangle$ over all edges $\lbrace a,b \rbrace$ of the $j^{\rm th}$ polygon. When the $i^{\rm th}$ and $j^{\rm th}$ polygon share an edge $\lbrace a, b\rbrace$, so that they combine to form a $(p_i+p_j-2)$-gon, then formula~\eqref{allYangian} nicely rearranges to give the Yangian invariant associated with the same set of polygons, but with the $i^{\rm th}$ and $j^{\rm th}$ polygon replaced by the merged $(p_i+p_j-2)$-gon. In particular, it can be shown that the numerator factorises and one gets an overall factor of $\langle Yab\rangle^2$, which cancels the singularities associated with the shared edge $\lbrace a, b\rbrace$ from the denominator, as expected.

To summarise, we see that in general, all singularities of a given Yangian invariant correspond to a subset of non-intersecting diagonals inside of an $n$-gon (and possibly external edges). In the appendix we provide an explicit enumeration of the number of $n$-particle N${}^k$MHV Yangian invariants for $m=2$.

\section{\texorpdfstring{$A_{n-3}$}{A(n-3)} Cluster Adjacency}
\label{sec:3}

Tree-level \emph{cluster adjacency} in $\mathcal{N}=4$ Yang-Mills theory is the conjectured~\cite{Drummond:2018dfd} property that every (rational) Yangian invariant has poles given by some collection of $\mathcal{A}$-coordinates of the $\Gr(4,n)$ cluster algebra that can be found together in common cluster. So far, evidence supporting this conjecture is restricted to relatively small $n$ and $k$. In~\cite{Drummond:2018dfd} several examples were checked by explicitly identifying a suitable cluster for several relatively simple Yangian invariants. Later in~\cite{Mago:2019waa} a computationally efficient method (first explained in~\cite{Golden:2019kks}) for testing whether two cluster coordinates belong in a common cluster was used to provide further evidence for this conjecture for various somewhat higher $n$ and $k$.

The natural generalisation of the cluster adjacency conjecture for general $m$ would posit that the poles of every Yangian invariant are given by some subset of the $\mathcal{A}$-coordinates of some cluster of the $\Gr(m,n)$ algebra. For $m=2$ this is the same as the classic $A_{n-3}$ algebra, whose structure is completely understood~\cite{FZ1}. This algebra has $\frac{1}{n-1} \binom{2n-4}{n-2}$ clusters, each containing $2n{-}3$ $\mathcal{A}$-coordinates. These numbers are respectively the number of distinct triangulations of an $n$-gon, and the number of edges (including external edges) in any such triangulation. If we label the vertices of a regular $n$-gon by homogeneous coordinates $z_1, \ldots, z_n$ of $n$ points in $\mathbb{P}^1$ and let $\langle a\,b \rangle = \epsilon_{AB}\,z_a^A z_b^B$, as usual, then the coordinates are enumerated as follows. Each cluster contains the $n$ coordinates $\langle 1\,2\rangle, \langle 2\,3\rangle, \ldots, \langle n\,1 \rangle$ corresponding to the external edges, together with precisely $n{-}3$ additional $\langle a\,b\rangle$'s that correspond to the internal edges of the triangulation.

In light of this discussion it is now essentially obvious that every Yangian invariant in the $m=2$ version of $\mathcal{N}=4$ Yang-Mills theory, whose generic form is shown in~\eqref{allYangian}, manifestly satisfies cluster adjacency. The only detail requiring comment is that, as discussed in~\cite{Arkani-Hamed:2013jha}, it is possible to replace the $(k+2)$-component $Z_a$'s appearing in a bracket of the form $\langle Y a b \rangle$ by their projections to the ``ordinary'' two-component homogeneous coordinate $z_a$ on $\mathbb{P}^1$, by setting
\begin{align}
Y = \left( \begin{matrix}
0_{2 \times k} \\
1_{k \times k} \end{matrix}\right).
\end{align}
That is, as far as the denominator of~\eqref{allYangian} is concerned, we can simply replace every $\langle Y a b \rangle$ by $\langle a\, b\rangle$, making the cluster adjacency manifest. The numerator of a Yangian invariant will in general be a rather non-trivial polynomial in the $z_a$'s and their Grassmann partners, but the numerator is of no concern to us.

The fact that the connection between cluster adjacency and Yangian invariants is strikingly simple for $m=2$ provides some circumstantial evidence in support of our hope that the same will be true for the apparently much more non-trivial case of $m=4$ (or perhaps even for general $m$). It also gives support to the suggestion made in~\cite{Mago:2019waa} that the connection between cluster adjacency and Yangian invariants might admit a mathematical explanation that is independent of the physics of scattering amplitudes, and most likely originates from the geometry of the amplituhedron.

If there is to be, one day, an analytic proof of the tree-level cluster adjacency conjecture, it is natural to speculate that it may hinge on the fact that it is known~\cite{Drummond:2010uq} that every positive Yangian invariant can be written as Grassmannian integral~\cite{ArkaniHamed:2009dn} (more specifically, over the momentum twistor Grassmannian~\cite{Mason:2009qx,ArkaniHamed:2009vw}) over a contour associated to a positroid cell. Here, then, we have access to relatively simple situations in which both the \emph{integrand} (the natural top form on $\Gr(k,n)$) and the \emph{integral} (the resulting Yangian invariant) are both \emph{rational functions}.  It would be extremely exciting to learn what property of the former is responsible, after integration, for cluster adjacency of the latter.  This could help point the way towards answering the long-standing, but much more complicated, question of how the cluster structure of integrands~\cite{ArkaniHamed:2012nw} in SYM theory is related, upon integration, to the cluster structure of the resulting polylogarithmic functions that appear in amplitudes.

\begin{table}[!ht]
\begin{center}
\begin{tabular}{c|cccccccc}
\backslashbox{$k$}{$n$}& 3&4&5&6&7&8&9&10\\
\hline
0&1&1&1&1&1&1&1&1\\
\hline
1&1&4&10&20&35&56&84&120\\
\hline
2&0&1&10&48&161&434&1008&2100\\
\hline
3&0&0&1&20&161&824&3186&10152\\
\hline
4&0&0&0&1&35&434&3186&16840\\
\hline
5&0&0&0&0&1&56&1008&10152\\
\hline
6&0&0&0&0&0&1&84&2100\\
\hline
7&0&0&0&0&0&0&1&120\\
\hline
8&0&0&0&0&0&0&0&1\\
\hline
\bf Total&\bf 2&\bf 6&\bf 22&\bf 90&\bf 394&\bf 1806&\bf 8558&\bf 41586\\
\end{tabular}
\end{center}
\caption{The number of generalised triangles for $n<11$.}
\label{table}
\end{table}

\acknowledgments

This work was supported in part by the US Department of Energy under contract {DE}-{SC}0010010 Task A (MS, AV) and by Simons Investigator Award \#376208 (AV). In addition MP would like to thank `Fondazione A. Della Riccia' for financial support, and MS and AV thank the CERN Theory Group for hospitality during the completion of this work. This work was performed in part at Aspen Center for Physics, which is supported by National Science Foundation grant PHY-1607611. TL was partially supported by a grant from the Simons Foundation.

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Mary Ritter, Climate-KIC's Chief Executive Officer, was presented with an OBE by Prince Charles at Buckingham Palace this week (12 February 2014).
The Queen appoints new members of the Order of the British Empire, on the advice of the UK government. Professor Ritter was appointed Officer of the Most Excellent Order of the British Empire for services to scientific research and innovation.
"It is a great honour to be recognised for my contribution, alongside that of the whole Climate-KIC team, to one of the major challenges facing mankind," Ritter said.
The long-planned ceremony coincided with one of the worst storms to hit the UK so far this winter, which has caused large areas of England and Wales to be flooded and has put tens of thousands of homes without power.
In the three years that Mary Ritter has been at the helm, Climate-KIC's public-private partnership has grown from a membership of 15 organisations with a €6 million budget, to a community of over 200 partners across Europe from business, academia and the public sector, with a budget of more than €70 million for 2014 and a mission to mitigate and adapt to climate change while creating sustainable growth and jobs.
"The citation is for 'Services to Scientific Research and Innovation' and in the context of innovation, although I have been a serial entrepreneur within the academic context, I am particularly proud of my most recent, and biggest, challenge – leading Climate-KIC," said Ritter.
Before joining Climate-KIC, Professor Ritter's research focused on how the immune system develops in the fetus and how it declines in elderly people. She also developed new research products, and established new postgraduate programmes at Imperial College London.
Professor Ritter launched the College's first Graduate School and played a key role in the establishment of the joint Imperial-NTU Lee Kong Chian medical school in Singapore. As Pro-Rector, she oversaw Postgraduate Affairs at Imperial from 2004 to 2007 and International Affairs from 2005 to 2011.


text:
News Breaking News Matthew Solorzano killed in Irvine hit-and-run on 405 Freeway
Matthew Solorzano killed in Irvine hit-and-run on 405 Freeway
Posted: January 06, 2023 EDT
Updated: January 06, 2023 04:51 AM EDT
Victim was walking on 405 Freeway in Irvine
IRVINE, CA, (January 03, 2023) - The Orange County coroner's office has released the name of a 33-year-old pedestrian killed in a hit-and-run collision Tuesday night in Irvine.
The Orange County coroner's office has released the name of a 33-year-old pedestrian killed in a hit-and-run collision Tuesday night in Irvine.
Matthew Solorzano was walking in the northbound lanes of the 405 Freeway near John Wayne Airport around 7:30 p.m. when he was struck by an unknown motorist who fled the scene. Sadly, Solorzano died at the scene as a result of injuries sustained in the crash.
Officials request individuals who may have witnessed the hit-and-run collision to contact California Highway Patrol's Santa Ana station at (714) 567-6000.
HIT-AND-RUN CRASH INVESTIGATION
Despite every warning, every memorial, every death, many drivers keep going, knowing they've hit someone.
In this incident, too, the driver fled the scene, leaving the victim to die from her injuries. Failure to remain on the collision site can attract criminal charges, and the victim can also pursue a case against the driver.
According to the California Office of Traffic Safety (COTS), in 2018, around 893 pedestrians were killed on California roadways, a 26% increase from 2014.
"Having the proper resources is extremely important to investigate a pedestrian accident case and prove negligence," said Candice Bond, an experienced hit-and-run accident attorney and a Managing Partner of Bond Legal.
"Road conditions, signage visibility, and road lighting are all possible contributing factors that are considered and must thoroughly be examined. These procedures are necessary for helping those who have lost a loved one receive the justice that they deserve."
Added Bond, the national analyst for The Legal Advocate, "The driver always has to take great care behind the wheel."
Pending the investigation, the family of Matthew Solorzano may be entitled to a wrongful death claim. Family members of wrongful death victims may seek damages for loss of companionship, pain and suffering, and emotional distress following a catastrophic accident such as this one.
"The stress and sorrow can be extremely overwhelming after losing a loved one in an accident," said Stefano Formica, Of Counsel with Bond Legal.
"We understand just what families are forced to go through, and having their rights protected is vital. A wrongful death lawyer will stay updated on the official investigation and help ensure that their legal rights and best interests are protected."
If your loved one has been killed at the hands of a hit-and-run driver and would like a no-cost consultation, please contact our national legal analysts Candice Bond and Stefano Formica today
Tags: Pedestrian Killed, Fatal Accident, Orange County, Irvine, Hit-and-run, 405 Freeway
The staff of The Legal Advocate would like to extend our thoughts and prayers to Matthew Solorzano and family
ORANGE INJURY LAWYERS


As much as we want roll out of bed with hair like J.Lo's or Beyoncé's, we just can't. It's not always that we're blessed with the luxury of time to go to the salon to blow dry and iron out our tresses properly. The worst is when we rush out of the front door and leave our hair to dry while in traffic as if we were in high school. As a result, we're left with a frizzy mane, which we're forced to conceal via a messy top knot or a loose, low ponytail. So how can we avoid this?
Here, we list down tips on how you can dry your hair (sans a blow dryer) without it going frizzy.
1. Don't get a blunt haircut.
Hair tends to create volume when it is cut evenly all around. But if you so want to sport a dead straight blunt cut, ask your stylist to layer your hair in reverse with shorter strands inside and the longer ones falling on top.
2. Apply styling cream immediately after washing.
This works well for those with curly and coarse hair, especially if you want to keep your natural texture. Simply apply the product onto damp hair and leave it to air dry.
3. Use a T-shirt to dry your hair.
Towel drying may be fast but the friction it creates is usually the number one suspect when it comes to frizz. Instead, use a T-shirt to squeeze out the excess water and allow it to air dry.
4. Comb hair while it's wet.
You've been told never to brush or comb hair while it's wet as it can lead to breakage. While that may be true, one loophole to it is taking a wide tooth comb to your strands and combing it out right after you condition. If there's still a need to do some untangling, simply run your fingers through your hair.


Welcome to the movement toward 100% graduation. Kids First!
The Office of Board President Mónica García, in collaboration with community partners, will lead education reform at the Los Angeles Unified School District to ensure that every child is prepared to read, write and think critically; to attend college or begin a successful career; and to participate as an engaged member of society.


text:
Q: .mdf created in Visual Studio my colleague created .mdf and .ldf files from Visual Studio. Now, I'm trying to access the file in SQL Server. There seems to be a problem with this. Can anybody tell me what edition of SQL Server must I have on my Windows Vista 32-bit, so I can access this database. Thanks in advance. :)

A: Your question is tagged [visual-studio-2008]. That version of Visual Studio is bundled with SQL Server 2005 (if you have the proper edition) and the builtin tools uses SQL Server 2005. However, there is nothing in Visual Studio 2008 that prohibits you from using say SQL Server 2008 if you want to. But I hope this answers your question.

A: The MDF and LDF file format is identical between all SQL Server editions that are at the same build number. Your friend's Visual Studio by default installs SQL Server Express 2008, and the Express is updated automatically by Microsoft Update and Windows Update to the lates released service pack. Since there is no supported downgrade path for MDF files, your own SQL Server has to match at least the latest Express edition pushed through Windows Update, so you'll most likely need SQL Server 2008 SP1 (build 2007.100.2531.0).
To verify what version of SQL you have and your friend has, follow the steps described in How to identify your SQL Server version and edition

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Senior Victoria Phipps loves to be involved in many extracurricular activities and lending a helping hand to people who need it. She also likes to listen to music and can spend the whole day reading a good book. She enjoys to draw and write and will use it to relieve the stress accumulated from her busy schedule.


Just in off daily rental. Fully loaded with * power windows, power locks. * tilt, cruise, 4.3LV8 8 speed engine. * Bulkhead partition. * Full Factory Warranty in effect. * Accident Free. Shelving and roof rack extra. We are located in Aurora. 15699 Yonge Street. *905-222-8234 Always nice cargo vans in stock. All certified.
All Vehicles come Certified Previous Daily Rental!! Hubcaps, keyless entry, dual air bags, side air bags, traction ctrl, vinyl floor, a/c, am/fm, mp3, reverse camera, park aid, pwr windows, pwr locks, pwr mirrors, cruise, steering wheel ctrls, tilt wheel!


A former prosecutor testified Friday that when the Manhattan District Attorney's office looked into the possibility that two men had been wrongly convicted of a 1987 murder he was never questioned for the review.
"I don't recall anyone contacting me," Michael Goldstein said in response to a question from lawyer James Henning.
The admission appeared to call into question how seriously the Conviction Integrity Program considered the claim by Eric Smokes and David Warren that they were innocent.
Asked to explain how any such review would omit feedback from the prosecutor who helped convict the men, the DA's office had no comment.
Smokes was 19 and Warren 16 when they were charged in the robbery murder of French tourist Jean Casse, 71, outside Ben Benson's steakhouse on W. 52nd St.. on Jan. 1, 1987, minutes after the ball dropped in Times Square.
Casse, who was with his wife, was surrounded by young men who pushed him to the ground. He hit his head and died later that day.
When Smokes and Warren, best friends from East New York, Brooklyn, were arrested days later they contended that they were several blocks from the scene, celebrating with friends.
They were convicted at trial of murder and robbery. Warren, who got 15 years to life, was released from prison in 2009. Smokes, who was sentenced to 25 years to life, got out in 2011.
They have been trying to clear their names ever since, aided by witnesses who now say they were pressured by police and prosecutors to falsely identify Smokes and Warren as the killers out of fear they would be charged in the case, or because they were promised leniency in unrelated criminal cases.
But Goldstein, who has been in private practice for about 30 years, testified Friday that he is confident the right men were prosecuted.
When questioned by Manhattan Assistant District Attorney Christine Keenan, who is arguing in favor of the conviction, Goldstein said he has never threatened witnesses or forced them to lie, and that he would never rely on someone he knew was lying.
When the case resumes Feb. 11, retired NYPD Det. George Delgrosso will testify and likely face the same type of questions.
In court on Friday, it was revealed that in 1978 when Delgrosso was an officer, he beat up another cop in a bar, then lied to Internal Affairs, contending neighborhood toughs were responsible. When the lie was uncovered, Delgrosso admitted what happened and was suspended for five days.
Goldstein said he was not aware of that disciplinary action when he worked with Delgrosso on the murder case. But, he said, he did not recall ever asking about the disciplinary history of cops involved in cases he prosecuted.
In recent months, with increasing attention on how the NYPD disciplines its officers, Manhattan District Attorney Cy Vance Jr. has pushed for such records.


text:
Psychological Medicine (3)
CNS Spectrums (1)
Development and Psychopathology (1)
Twin Research and Human Genetics (1)
International Soc for Twin Studies (1)
Neuroscience Education Institute (1)
Royal College of Speech and Language Therapists (1)
Dentate gyrus volume deficit in schizophrenia
Soichiro Nakahara, Jessica A. Turner, Vince D. Calhoun, Kelvin O. Lim, Bryon Mueller, Juan R. Bustillo, Daniel S. O'Leary, Sarah McEwen, James Voyvodic, Aysenil Belger, Daniel H. Mathalon, Judith M. Ford, Fabio Macciardi, Mitsuyuki Matsumoto, Steven G. Potkin, Theo G. M. van Erp
Journal: Psychological Medicine , First View
Published online by Cambridge University Press: 03 June 2019, pp. 1-11
Schizophrenia is associated with robust hippocampal volume deficits but subregion volume deficits, their associations with cognition, and contributing genes remain to be determined.
Hippocampal formation (HF) subregion volumes were obtained using FreeSurfer 6.0 from individuals with schizophrenia (n = 176, mean age ± s.d. = 39.0 ± 11.5, 132 males) and healthy volunteers (n = 173, mean age ± s.d. = 37.6 ± 11.3, 123 males) with similar mean age, gender, handedness, and race distributions. Relationships between the HF subregion volume with the largest between group difference, neuropsychological performance, and single-nucleotide polymorphisms were assessed.
This study found a significant group by region interaction on hippocampal subregion volumes. Compared to healthy volunteers, individuals with schizophrenia had significantly smaller dentate gyrus (DG) (Cohen's d = −0.57), Cornu Ammonis (CA) 4, molecular layer of the hippocampus, hippocampal tail, and CA 1 volumes, when statistically controlling for intracranial volume; DG (d = −0.43) and CA 4 volumes remained significantly smaller when statistically controlling for mean hippocampal volume. DG volume showed the largest between group difference and significant positive associations with visual memory and speed of processing in the overall sample. Genome-wide association analysis with DG volume as the quantitative phenotype identified rs56055643 (β = 10.8, p < 5 × 10−8, 95% CI 7.0–14.5) on chromosome 3 in high linkage disequilibrium with MOBP. Gene-based analyses identified associations between SLC25A38 and RPSA and DG volume.
This study suggests that DG dysfunction is fundamentally involved in schizophrenia pathophysiology, that it may contribute to cognitive abnormalities in schizophrenia, and that underlying biological mechanisms may involve contributions from MOBP, SLC25A38, and RPSA.
Interactive impact of childhood maltreatment, depression, and age on cortical brain structure: mega-analytic findings from a large multi-site cohort
Leonardo Tozzi, Lisa Garczarek, Deborah Janowitz, Dan J. Stein, Katharina Wittfeld, Henrik Dobrowolny, Jim Lagopoulos, Sean N. Hatton, Ian B. Hickie, Angela Carballedo, Samantha J. Brooks, Daniella Vuletic, Anne Uhlmann, Ilya M. Veer, Henrik Walter, Robin Bülow, Henry Völzke, Johanna Klinger-König, Knut Schnell, Dieter Schoepf, Dominik Grotegerd, Nils Opel, Udo Dannlowski, Harald Kugel, Elisabeth Schramm, Carsten Konrad, Tilo Kircher, Dilara Jüksel, Igor Nenadić, Axel Krug, Tim Hahn, Olaf Steinsträter, Ronny Redlich, Dario Zaremba, Bartosz Zurowski, Cynthia H.Y. Fu, Danai Dima, James Cole, Hans J. Grabe, Colm G. Connolly, Tony T. Yang, Tiffany C. Ho, Kaja Z. LeWinn, Meng Li, Nynke A. Groenewold, Lauren E. Salminen, Martin Walter, Alan N Simmons, Theo G.M. van Erp, Neda Jahanshad, Bernhard T. Baune, Nic J.A. van der Wee, Marie-Jose van Tol, Brenda W.J.H. Penninx, Derrek P. Hibar, Paul M. Thompson, Dick J. Veltman, Lianne Schmaal, Thomas Frodl, 'for the ENIGMA-MDD Consortium'
Published online by Cambridge University Press: 14 May 2019, pp. 1-12
Childhood maltreatment (CM) plays an important role in the development of major depressive disorder (MDD). The aim of this study was to examine whether CM severity and type are associated with MDD-related brain alterations, and how they interact with sex and age.
Within the ENIGMA-MDD network, severity and subtypes of CM using the Childhood Trauma Questionnaire were assessed and structural magnetic resonance imaging data from patients with MDD and healthy controls were analyzed in a mega-analysis comprising a total of 3872 participants aged between 13 and 89 years. Cortical thickness and surface area were extracted at each site using FreeSurfer.
CM severity was associated with reduced cortical thickness in the banks of the superior temporal sulcus and supramarginal gyrus as well as with reduced surface area of the middle temporal lobe. Participants reporting both childhood neglect and abuse had a lower cortical thickness in the inferior parietal lobe, middle temporal lobe, and precuneus compared to participants not exposed to CM. In males only, regardless of diagnosis, CM severity was associated with higher cortical thickness of the rostral anterior cingulate cortex. Finally, a significant interaction between CM and age in predicting thickness was seen across several prefrontal, temporal, and temporo-parietal regions.
Severity and type of CM may impact cortical thickness and surface area. Importantly, CM may influence age-dependent brain maturation, particularly in regions related to the default mode network, perception, and theory of mind.
D2 receptor occupancy following lurasidone treatment in patients with schizophrenia or schizoaffective disorder
Steven G. Potkin, David B. Keator, Marilyn L. Kesler-West, Dana D. Nguyen, Theo. G. M. van Erp, Jogeshwar Mukherjee, Nikunj Shah, Adrian Preda
Journal: CNS Spectrums / Volume 19 / Issue 2 / April 2014
Objective/Introduction
Lurasidone is an atypical antipsychotic medication approved for the treatment of schizophrenia over a dose range of 40–160 mg/day. This study examined D2 receptor occupancy and its association with clinical improvement and side effects in patients with schizophrenia or schizoaffective disorder following repeated doses of 80, 120, or 160 mg/day of lurasidone.
Twenty-five patients with The Diagnostic and Statistical Manual of Mental Disorders, 4th ed. (DSM-IV) diagnoses of schizophrenia or schizoaffective disorder were washed out of their antipsychotic medications (5 half-lives) and randomly assigned to 80, 120, or 160 mg/day of lurasidone. Subjects were imaged with 18F-fallypride at baseline and at steady-state lurasidone treatment to determine D2 receptor occupancy.
Blood lurasidone concentration (plus major metabolite), but not dose, significantly correlated with D2 receptor occupancy. D2 receptor occupancy in several subcortical structures is associated with positive but not negative symptom improvement or the presence of movement symptoms.
Blood concentrations greater than 70 ng/mL may be required to achieve a 65% occupancy level in subcortical areas. Intersubject blood concentrations at fixed dose were highly variable and may account for the lack of dose correlations.
Positron emission tomography (PET) occupancy data suggest that greater than 65% occupancy can be achieved across the dose range of 80–160 mg/day and that some patients require higher doses to achieve antipsychotic efficacy; this finding supports prior randomized clinical trial results.
Heritability of Multivariate Gray Matter Measures in Schizophrenia
Jessica A. Turner, Vince D. Calhoun, Andrew Michael, Theo G. M. van Erp, Stefan Ehrlich, Judith M. Segall, Randy L. Gollub, John Csernansky, Steven G. Potkin, Beng-Choon Ho, Juan Bustillo, S. Charles Schulz, FBIRN, Lei Wang
Journal: Twin Research and Human Genetics / Volume 15 / Issue 3 / June 2012
Published online by Cambridge University Press: 15 June 2012, pp. 324-335
Structural brain measures are employed as endophenotypes in the search for schizophrenia susceptibility genes. We analyzed two independent structural imaging datasets with voxel-based morphometry and with source-based morphometry, a multivariate, independent components analysis, to determine the stability and heritability of regional gray matter concentration abnormalities in schizophrenia. The samples comprised 209 and 102 patients with schizophrenia and 208 and 96 healthy volunteers, respectively. The second sample additionally included non-ill siblings of participants with and without schizophrenia. A standard voxel-based analysis showed reproducible regional gray matter deficits in the affected participants compared with unrelated, unaffected controls in both datasets: patients showed significant gray matter concentration deficits in cortical frontal, temporal, and insular lobes. Source-based morphometry (SBM) was applied to the gray matter images of the entire sample to determine the effects of diagnosis on networks of covarying structures. The SBM analysis extracted 24 significant sets of covarying regions (components). Four of these components showed significantly lower gray matter concentrations in patients (p < .05). We determined the familiality of the observed SBM components based on 66 sibling pairs (25 discordant for schizophrenia). Two components, one including the medial frontal, insular, inferior frontal, and temporal lobes, and the other including the posterior occipital lobe, showed significant familiality (p < .05). We conclude that structural brain deficits in schizophrenia are replicable, and that SBM can extract unique familial and likely heritable components. SBM provides a useful data reduction technique that can provide measures that may serve as endophenotypes for schizophrenia.
Developmental disruptions in neural connectivity in the pathophysiology of schizophrenia
Katherine H. Karlsgodt, Daqiang Sun, Amy M. Jimenez, Evan S. Lutkenhoff, Rachael Willhite, Theo G. M. van Erp, Tyrone D. Cannon
Journal: Development and Psychopathology / Volume 20 / Issue 4 / Fall 2008
Published online by Cambridge University Press: 07 October 2008, pp. 1297-1327
Print publication: Fall 2008
Schizophrenia has been thought of as a disorder of reduced functional and structural connectivity. Recent advances in neuroimaging techniques such as functional magnetic resonance imaging, structural magnetic resonance imaging, diffusion tensor imaging, and small animal imaging have advanced our ability to investigate this hypothesis. Moreover, the power of longitudinal designs possible with these noninvasive techniques enable the study of not just how connectivity is disrupted in schizophrenia, but when this disruption emerges during development. This article reviews genetic and neurodevelopmental influences on structural and functional connectivity in human populations with or at risk for schizophrenia and in animal models of the disorder. We conclude that the weight of evidence across these diverse lines of inquiry points to a developmental disruption of neural connectivity in schizophrenia and that this disrupted connectivity likely involves susceptibility genes that affect processes involved in establishing intra- and interregional connectivity.
Memory and verbal learning functions in twins with bipolar-I disorder, and the role of information-processing speed
TUULA KIESEPPÄ, ANNAMARI TUULIO-HENRIKSSON, JARI HAUKKA, THEO VAN ERP, DAVID GLAHN, TYRONE D. CANNON, TIMO PARTONEN, JAAKKO KAPRIO, JOUKO LÖNNQVIST
Journal: Psychological Medicine / Volume 35 / Issue 2 / February 2005
Background. Euthymic bipolar-I disorder (BP I) patients and their siblings have shown impairments in verbal learning and memory functions compared with controls, suggesting that these impairments may be genetic in origin. Reduced information-processing speed has been associated with impaired memory in the elderly, and recently in schizophrenia. The authors compared verbal learning and memory functioning in twins with BP I and co-twins to control twins, and examined whether the observed deficits are related to information-processing speed.
Method. Finnish Medical and Population Registers and Twin Cohorts were used to identify the BP I and control twins. Neuropsychological tests assessing verbal learning and memory, working memory, facial recognition, visual memory, and information-processing speed were administered to 26 BP I twins, 19 non-bipolar co-twins, and 114 controls. Group differences were analyzed by generalized estimation equation modeling.
Results. BP I patients, but not co-twins, showed impairments in all memory tests compared with controls. Female co-twins showed impairment in verbal learning and memory. Information-processing speed had a significant effect on encoding and learning efficiency.
Conclusions. This study showed for the first time that information-processing speed is related to memory functioning and verbal learning in BP I in a population-based, representative and euthymic sample. Furthermore, the data support the view that defects in verbal memory may be related to the genetic factors predisposing to BP I in females.
13 - Investigating gene–environment interaction in schizophrenia using neuroimaging
from III - The genetic epidemiology of schizophrenia
By Theo G. M. Van Erp, Timothy L. Gasperoni, Isabelle M. Rosso, Tyrone D. Cannon
Edited by Robin M. Murray, Institute of Psychiatry, London, Peter B. Jones, University of Cambridge, Ezra Susser, Columbia University, New York, Jim Van Os, Universiteit Maastricht, Netherlands, Mary Cannon, Institute of Psychiatry, London
Book: The Epidemiology of Schizophrenia
Print publication: 28 November 2002, pp 254-270
View extract
Applications of classical epidemiological methods have led to general acceptance of a multifactorial threshold model of schizophrenia, whereby multiple genetic influences and environmental factors aggregate together, additively and/or interactively, to form a continuum of disease liability. Studies that make use of clinical diagnostic categories are limited in what they can tell us about the precise nature of the aetiological factors involved in schizophrenia because of the inherent limitations of categorical measures in reflecting a continuous liability scale. We propose that new methodologies that can represent this liability continuum on a quantitative scale are needed to aid in the search for schizophrenia susceptibility genes and in elucidating the pathophysiological mechanisms by which these genes and environmental factors act. Here we review evidence concerning structural brain abnormalities as markers of aetiological influences in schizophrenia, concluding that use of quantitative neuroanatomical measures will facilitate the detection of predisposing gene loci and help to clarify whether nongenetic influences contribute independently of or interactively with genetic factors in influences in schizophrenia, concluding that use of quantitative neuroanatomical measures will facilitate the detection of predisposing geneloci and help to clarify whether nongenetic influences contribute independently of or interactively with genetic factors in influencing disease liability. The eventual discovery of such genes will spawn a new era of epidemiological research, in which the effects of specific predisposing genotypes and specific environmental factors on schizophrenia phenomenology and pathophysiology can be studied.
Investigating gene-environment interaction in schizophrenia
Family, twin, and adoption studies have demonstrated that schizophrenia is a substantially heritable disorder. These studies are reviewed in Chapter 10. Still, attempts at isolating specific genes that confer vulnerability to schizophrenia have so far been only moderately successful (Moldin, 1997).


Not only that sweet potatoes are healthier than regular ones, but they cook faster, being a perfect ingredient for quick and easy dishes. So, if you want a saving time breakfast recipe, try this sweet potato and bacon gratin. It's comforting enough thanks to the mozzarella, Emmental cheese and bacon!
Grease with vegetable oil a round baking tray.
Place the sweet potato slices on the bottom of the tray.
Drizzle some more vegetable oil on it. Season with salt and pepper.
Add a layer of shredded mozzarella (half of the whole quantity), then add the Emmental slices, and the bacon slices.
Add the mushroom slices on the edges.
Top with tomato sauce and the remaining shredded mozzarella.
Crack the egg and add it in the center.


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Die Liste der Einträge im National Register of Historic Places im Iron County in Michigan führt alle Bauwerke und historischen Stätten im Iron County auf, die in das National Register of Historic Places aufgenommen wurden.

Die meisten Objekte sind Teil der Iron County MPS, einer als Multiple Property Submission (MPS) zusammengefassten Gruppe von historischen Objekten im gesamten County.

Legende

Aktuelle Einträge

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Einzelnachweise

Iron
!

redpajama_set_name: RedPajamaWikipedia


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Using Yamaha's screen-flow technology, you can play even the longest piece of sheet music without ever having to turn a page. This keeps your eyes on the music and your hands on the keys where they belong!
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Easily transpose any song from the NoteStar library!
Increase the note size with a single tap. No zooming is ever required!
Hands-free page turning in portrait or landscape view offers a seamless playing experience!
NoteStar now offers 4 types of songs so you can choose the experience that's best for you.
All NoteStar songs feature automatic, hands-free page turning in time with the music, so you can keep your hands on the keys where they belong.
The songs in this series feature separate reference audio tracks for keyboard left and right hands. Each audio track can be independently muted for hands separate or hands together practice and performance.
The songs in this series feature a realistic audio backing track and a separate keyboard reference audio track. Each audio track can be independently muted for practice or to provide a unique 'in the band' performance experience.
The songs in this series feature a professional audio backing track and separate keyboard reference and lead vocal audio tracks. Each of the aforementioned tracks can be independently muted for practice or to provide a unique in-the-band performance experience, with or without vocals.
This series combines classical songs and exercises from composers such as Czerny, Beyer and Hanon into a perfect lesson for the practicing pianist. Like Piano Solo songs, Lesson Collections feature reference audio tracks for keyboard left and right hands. Each audio track can be independently muted for hands separate or hands together performance.
[Note] Please refer to "Related Products" in the Overview section for compatible Yamaha instruments.


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Daniel Amokachi Biography and Net Worth
Daniel Owefin Amokachi popularly known as Daniel Amokachi is a famous Nigerian professional football player, entrepreneur and former assistant manager of the Nigeria national football team (Super Eagles). He has been a technical director since 2006. He played as a forward striker for Nigeria's national team (Super Eagles) before retiring from international football.
He was born on 30th December 1972 in the city of Kaduna, Kaduna State in the northern part of Nigeria. He is currently 45 years old. He is originally from Abia State in the south eastern part of Nigeria, but was born and bred in Kaduna State in the northern part of Nigeria.
During his performances at the World Cup, Daniel Amokachi was nominated three times and was placed at third position in the African Footballer of the Year Awards. As a forward striker, Daniel Amokachi was known for his speed, technique and physical strength, which earned him the nicknames Black Bull and Black Train.
Daniel Amokachi who is also very popular and well known in his country, has contributed to Nigeria's top two tournaments in the FIFA World Cup along with his fellow attack partner Emmanuel Amunike. On 25th November 1992, Daniel Amokachi scored the first goal of the UEFA Champions League with Club Brugge, beating CSKA Moscow 1–0.
Daniel Amokachi was discovered while playing for Ranchers Bees by Nigerian national team coach Clemens Westerhof, who brought him to the 1990 African Nations Cup, after which Daniel Amokachi moved to play for Club Brugge in Belgium. He competed in the new format of the Champions League, and became the first player to score in the competition, after which he scored a goal and secured a 1–0 win for his team Club Brugge in the opening match in the group stage against CSKA Moscow.
After his impressive performance in Club Brugge in Belgium and his amazing performance for the Nigeria's Super Eagles at the 1994 FIFA World Cup, Everton became interested in him and signed him for a contract fee worth £3,000,000 ($4,700,000).
In 1995, he went on to win the FA Cup with Everton after scoring two goals in the semi-final against Tottenham Hotspur. Daniel Amokachi remained at Everton until the end of the 1995 to 1996 season, when he was later transferred to Besikta of Turkey for a fee of £1.75 million.
In 1995, he spent six weeks in prison during the autumn of 1995 for an offence committed in Scotland 18 months earlier. In 1999, he signed a contract with 1860 Munich, but the contract was cancelled after he failed a medical test. Unfortunately, he was rejected by Tranmere Rovers for the same reason.In 2002, American club Colorado Rapids signed him, but seeing that he wasn't fit enough they released him before a single match was played. He went to play in the United Arab Emirates, but was denied again due to his medical condition.
Daniel Amokachi has played many international matches for Nigeria, and was part of the team that participated in the 1994 FIFA World Cup and 1998 FIFA World Cup. In 1994, he helped his country Nigeria to win the 1994 African Nations Cup. He also helped his country Nigeria to win the Olympic gold medal in 1996.
Daniel Amokachi has worked as the manager of Nigerian club Nasarawa United and later Enyimba of Aba. In April 2007, Daniel Amokachi quit his role as assistant coach of the Nigeria national team (Super Eagles). On 10th April 2008, Daniel Amokachi was re-appointed as the assistant coach of the Nigeria's national team (Super Eagles). He worked as an assistant to both Shuaibu Amodu and Stephen Keshi.
In 2015, Daniel Amokachi managed Ifeanyi Ubah and later resigned after spending five weeks in the club. In January 2016, Daniel Amokachi was named as manager of JS Hercules, a club which he is currently coaching till date.
Daniel Amokachi is currently married to a Tunisian woman and the marriage is blessed with two twin sons named Kalim and Nazim, who are currently following their father's footsteps in becoming professional footballers. His two sons are currently in the Besiktas youth academy.
Daniel Amokachi is currently one of the richest and most influential football player in Nigeria with an estimated net worth of $5 million.
Tags: ahmed musa, alex iwobi, daniel amokachi, daniel amokachi biography, daniel amokachi net worth, Didier drogba, jay jay okocha, kanu nwankwo, kelechi iheanacho, michael Jordan, mikel obi, okocha, samuel eto'o, tiger woods, victor moses


Well of course this would happen in New Jersey, home of the Sopranos and long known as a corruption hot-bed. This is a pretty standard money laundering case. $97,000 in Apple Jacks Box. Check. Illegal sale of body parts. Check. Mayors Takng Bribes. Check. In all 44 people were indicted, along with three NJ mayors, two NJ state assemblymen, and five rabbis.


This week for those of us in the United States is about joining with family and giving thanks. It also is a way for those in the obstacle racing industry to snag some incredible deals. Annually Mud Run Guide puts together a Black Friday and Cyber Monday page. Our aim is to help you get the best deals in OCR.


Medium coverage bottom that features a classic silhouete with crochet details on the edges. With this reversible garment you can choose between an elegant print or a bright burgundy color.
Intricate forms create symmetry within the interesting scenarios of this print, that has the timeless mix of colors for swimwear: black and white.


HYDERABAD: Sindh Chief Minister Syed Murad Ali Shah has said that the inefficient federal government [of PTI] has completely failed to deliver and has brought a tsunami of price hike, unrest and has pushed the poor people to abject poverty by reducing employment opportunities.
He was speaking on the occasion of the 12th death anniversary of former Sindh chief minister Syed Abdullah Shah organised at his native village Wahur-Murad Shah, Sehwan. The death anniversary programme was attended by Sindh PPP President Nisar Khuhro, provincial cabinet members, special assistants, MNAs, MPAs, senators, PPP workers and people from different walks of life.
The CM said on the occasion that the inefficient federal government has no capacity to rule the country and improve condition of masses because they have no roots among the people of Pakistan. He added that everyone knew very well how they had been brought into power.
Shah said that people of the province were asking him about launching of development works in their areas but "I want to tell them that the inefficient federal government has failed to release the Sindh government's due share from the federal divisible pool, therefore, we are facing a shortage of funds and cannot start development as we had started last year," he said and vowed that the provincial government would start development schemes from its own resources from the next financial year.
Murad Ali Shah said that we had gathered here last year to observe Syed Abdullah Shah's 11 death anniversary and then went to contest the general election.
"Our opponents formed big alliances and groups but the people of Sindh rejected them and voted the PPP to power with more seats and more strength," he said.
The CM said that the PPP government in Sindh would be serving the people of Sindh because service to people was a tradition of PPP leadership.
Recalling the recent train march of PPP Chairman Bilawal Bhutto Zardari, the CM said that he [Bilawal Bhutto] was just going to Naudero on April 4 via train and people living in every nook and corner of central and northern Sindh, exhibited their love and respect for the son of Shaheed Benazir Bhutto, as they thronged the railway stations and gave him a warm and historic welcome.
"This historic and crowded welcome of the people of Sindh has caused a turmoil in corridors of power in the federal government," he said and added they were scared of him (Bilawal) because they know he had the ability to deliver for this country and its people.
DERA ISMAIL KHAN: The district administration has imposed Section 144 to prevent the nomadic population from entering into the district unless they get vaccinated their children against polio.


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The Internet Says This Is The Evidence That Khloe Kardashian Is Carrying Kim & Kanye's Baby
Astrid Stawiarz, Getty Images
It wouldn't be the craziest story from the Kardashian Kamp, but is Khloe carrying her sister's baby?
With the circulating reports of Khloe Kardashian's pregnancy, a KUWTK fan theory has been given new life. Is Khloe Kardashian carrying Kim and Kanye's baby? Before you roll your eyes and close this story, keep in mind that's what most people did when tabloids suggested Olympic legend Bruce Jenner was leaving his wife to become a woman.
Now, to the "receipts."
Numerous fans of Keeping Up With The Kardashians point back to the show's season finale back in June where Kim made this suggestion to Khloe:
I have a proposition for you. I'll give you the embryo; you carry the baby, then you give it back to me.
Khloe even went as far as seeing a doctor about it.
Ultimately she revealed that her boyfriend wanted kids of his own.
[Tristan] wants to have kids now. He wants to have like five or six kids with me, and that's lovely. We could start at one and we could grow from there. But now knowing I'm not on birth control is scary. It's like a really big step.
The math certainly lines up and you know the Kardashians love a good storyline no matter what it takes but did Khloe (or Kylie) decide to take Kim up on that surrogacy offer?
Let us know what you think of the far-fetched theory.
[via Marie Claire]
KHLOE KARDASHIAN REPORTEDLY PREGNANT WITH FIRST CHILD
Filed Under: kanye west, keeping up with the kardashians, khloe kardashian, kim kardashian, kylie jenner
Categories: Entertainment News


Blue . Challenge . Christmas . Holidays . Nail Art . Nail Polish . nailcraziesunite . Stamping . Zoya .
There are three movies named after my favourite Christmas carol "silent night", but I've seen none of those! So, my manicure is only inspired by the song.
As the carol, my manicure is simple, a blue canvas with stars and snow flakes. That's all. I used the lovely Zoya Pixie Dust in Waverly (from the recent Enchanted Collection for Holidays), which is a gorgeous blue, as a base. I then stamped a star and added some tiny dots with white. Finally I added a very thin french line to represent snow. That was it!
I hope you like this manicure even though it is a very simple one.
Do not forget to pay a visit to my challenge mates listed down below!
Now, I'd like to know, do you watch Holiday movies? What are your favourites? what would you recommend me? Have a wonderful weekend!!
P.S. I'm joining a new challenge from today and as this manicure happens to fit the first prompt of the Winter nail art challenge, I'm submitting it there too!


It's Sunday again, which means it's time for this week's smorgasbord. I have a variety of links to share with you- I hope you enjoy it. If you have anything you think would make a good addition to a smorgasbord, drop me a line at carrie@edbites.com. I will, of course, credit you with your suggestion.
I really wish people would stop referring to compulsive overeating as an addiction. I feel that the definition of addiction has been watered down so much that it has lost all meaning.
Overeating is a compulsion. It may have traits in common with adictions but I'm not sold on the idea that it is a a true addiction. Call me paranoid, but I think this is just a backdoor way to classify certain foods as drugs and bolster the cause to ban them and to pathologize and monitor people who use these "drugs."
I've been to a health class where a drug was defined as "anything that changed the body's chemistry." Well, ANYTHING could be classified as a drug under that definition! Water could be defined as a drug because less ADH is produced. Vegetables could be classified as drugs because their nutrients stimulate and enhance various processes in the body.
Listening to music, going on a rollercoaster (all that adrenalin), all could be drugs. Again, addiction is about to lose all meaning because of the way the word is thrown around.
I like the article about attitudes and healthy lifestyles. A lot of the so-called lifestyle diseases, such as high blood pressure, are actually stress responses. The ironic thing is that we stigmatize people with chronic illnesses, especially, again, the "lifestyle" illnesse such as diabetes.
ED cutoffs DO miss some of the sickest patients. One of my pet peeves is when people see anorexic patients and feel sorry for them. Then they proceed to engage in eating disordered behaviors such as counting calories, "making up" for the calories they consumed by overexercising, etc. The point of images like that is to let people know how serious an illness anorexia is, the drawback is that a lot of people who are sick or who are at risk, who engage in unhealthy behaviors, don't think they're in danger when they are.


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9/11 First Responders Face Uncertain Future as Cancer Continues to Plague Victims
Posted on September 11, 2017 September 28, 2020 by Sokolove Law Team
Sixteen years on, and for some, memories of the deadliest terrorist attack in world history are as vivid as ever.
The unprecedented damage from September 11 lives on in lingering economic ruin, the war on terror, life-altering physical and mental injuries, and most painfully, emotional wreckage from the loss of almost 3,000 innocent lives.
Most of those who died in World Trade Center buildings or surrounding areas were civilians and financial district workers trapped and struck by tumbling debris as they prepared for a day of work. Scores of first responders also perished in their brave attempts to rescue people. Still others, including civilians, survived or dedicated hundreds of hours to rescue, recovery, and cleanup efforts. But in the last camp are those who risked their lives to save others or gave their all to help pick up the pieces, only to die years later.
Exposure to toxic chemicals, including carbon monoxide, crystalline silica, and known carcinogens such as asbestos, has been a major health crisis that first responders and firefighters have fought ever since. These people, already suffering trauma from that day's tragic events, are also suffering and even dying at the hands of poisonous substances. Within 5 years, in fact, the number of those killed by exposure will exceed the number of those who died during the attacks.
Incomprehensible though it is, 9/11 is still ruining a growing number of lives. Are we doing all we can to stop this?
Devastation to Last a Lifetime
In the days after the attacks, the aim at Ground Zero – known by rescue personnel as "the pile" – was to rescue the living and recover the dead. The 1.5 million tons of toxic debris that transformed the streets of New York City was the least of anyone's concerns.
But 1 of those toxic substances was asbestos, a lethal mineral once popular for use in building construction. As the North and South towers of the WTC fell, the crash released a poisonous cloud of toxin-containing dust and ash. This cloud shrouded Lower Manhattan, Brooklyn, and the Long Island Sound, entering homes and offices, sticking to clothes and interiors, and making its way undetected into people's bodies.
The danger of asbestos is common knowledge. When airborne, its miniscule particles are health risks in any capacity. Asbestos is known as a "silent killer," the cause of debilitating and fatal diseases. This was a mineral that had already killed thousands of people through mesothelioma, a condition caused exclusively by asbestos exposure for which no cure exists. But that didn't stop these brave men and women from rushing into a chemical nightmare unprotected, putting aside their own health to save others.
The Bill That Changed It All
On January 6, 2006, a New York Police Department Officer called James Zadroga died of a respiratory disease aged 34. He became the first NYPD officer whose death was ruled a direct result of the Ground Zero cleanup.
This tragic death led to the birth of the James Zadroga 9/11 Health and Compensation Act, signed into law by President Barack Obama in 2011 after a 5-year political battle in Congress. The Act sought to provide medical benefits and other financial aid for 9/11 survivors sickened or dying from cancers and illnesses linked to the attacks through the World Trade Center Health Program and Victim Compensation Fund (VCF). In October 2015, the Act expired, and was finally extended days before Christmas to guarantee survivors lifelong care.
Can the Program Help?
Today, the number of heroes certified sick as a result of 9/11 is estimated to be 50,000. Over 1,000 have already died. Respiratory and digestive disorders are among the most common causes of death, but cancer diagnoses have multiplied. And sadly, the so-called "next wave" of illnesses hasn't fully surfaced yet. Post-traumatic stress disorder is considered a precursor to dementia; mesothelioma is a cancer whose symptoms remain dormant until up to 20-50 years after asbestos exposure.
For the 77,500 people enrolled, and others who join in the future, the World Trade Center Health Program is a life-changer. The Program helps to pay for medical monitoring and care, which would be an impossible task for many – especially cancer patients, whose medical costs often reach a quarter of a million dollars. The WTC Health Program also funds medical research to identify and treat new WTC-related health conditions, and the VCF has awarded more than $3 Billion to claimants.
Firefighters, law enforcement officers, paramedics, area workers, students, residents, and volunteers, both young and old at the time of 9/11, who inhaled poisoned air in their efforts to save lives from senseless terrorism – these are the people we should think about this year. By supporting these efforts to help first responders, we can at least limit the number of years taken from their life spans as thanks for their unrivaled bravery.
Posted in Mesothelioma
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The PDSL supports researchers and clinicians by providing high quality, specialized laboratory assays, services and materials that are optimized for detection and quantitation of non-human primate proteins and nucleic acids. Please see our services in the links below and contact us to determine the best and most economical solutions for your testing needs. Download Virology Specimen Submission Form.


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