Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: neurosurgery was consulted for an abnormal head ct major surgical or invasive procedure: intubation and subsequent extubation picc line placement and removal history of present illness: 79 year old female reportedly found down at the bottom of her stairs by her husband. she was vomiting upon arrival to the er. initially her sbp was in the 130s and then it dropped to the 70s. she was paralyzed, intubated and started on a neo drip. the er reported that her left pupil was 3mm and her right was 1mm. her head ct showed bilateral sdhs so a neurosurgery consult was called. past medical history: - dm2 w/retinopathy and neuropathy - arthritis - right hip fracture social history: previously lived at home with her husband, one -two drinks per night, no tobacco, walked with a walker family history: non-contributory physical exam: physical exam upon admission: t:97.9 bp:99/51 hr:85 rr:18 o2sats:100% vented (examined initially just after the patient was intubated and paralytics were still on board. the following exam reflects my second exam after paralytics wore off.) gen: intubated, sedated heent: pupils: left 1mm, surgical, right 2-1mm eoms- unable to test has an open laceration on the occipital region of her head. neck: in cervical collar. does not appear to have tenderness. lungs: cta bilaterally. cardiac: rrr. s1/s2. abd: soft, nt, bs+ extrem: warm and well-perfused. neuro: mental status: no eye opening, not following commands. (+) gag with suctioning cranial nerves: i: not tested ii: pupils: left 1 mm, surgical, right 2-1mm iii-xii: unable to test motor: normal bulk and tone bilaterally. no abnormal movements, tremors. localized bilaterally with upper extremities. withdraws bilateral lower extremities to noxious. sensation: appears to be intact bilaterally. toes upgoing bilaterally pertinent results: head ct : findings: right frontal parenchymal hemorrhage is noted which measures 2.0 x 1.4 cm on axial view. a second focus of right frontal parenchymal hemorrhage closer to the vertex measures 10 x 8 mm. there is a small amount of subarachnoid hemorrhage of both frontal lobes. also noted are bilateral subdural hematomas, which appear to be acute on chronic, with a hyperdense component indicating acute blood. these layer around both cerebral convexities. on the left maximal thickness of the subdural collection is 6 mm and on the right maximal thickness also 6 mm. subdural blood layers along the tentorium. there is no appreciable shift of normally midline structures or mass effect. the basilar cisterns are not effaced. there is an acute fracture of the right temporal bone with extension through the right mastoid air cells, which contain blood. there is mild depression of the squamous portion of the right temporal bone. the temporal bone fracture extends into the lesser sphenoid on the right. there is also a fracture through the right occipital bone with over-riding of bone fragments by about 6 mm. there is subcutaneous and intramuscular emphysema as well as a small amount of pneumocephalus near the temporal fracture sites. there is a small amount of blood layering in the right maxillary sinus. the nasal cavity is opacified, probably due to blood. the sphenoid sinus is opacified with heterogeneous material suggesting chronic opacification secondary to inspissated secretions. however, there is probably a small amount of blood in the sphenoid sinus as well. the globes are intact. there is soft tissue swelling of the right periorbital region. the patient is intubated with terminus out of view. impression: 1. parenchymal contusion of the frontal lobes, right greater than left. 2. bifrontal subarachnoid hemorrhage. 3. bilateral subdural hematomas with mix densities. 4. fractures through the right temporal and occipital bones. head ct : findings: the right frontal parenchymal hemorrhage has significantly worsened over the short interval, now measuring approximately 5.6 x 3.7 cm, previously 2.0 x 1.4 cm. there is new mass effect with shift of the anterior midline structures to the left by approximately 5 mm and partial effacement of the frontal of the right lateral ventricle. there has also been worsening in left frontal subarachnoid hemorrhage and parenchymal contusion. redemonstrated are bilateral subdural hematomas, which layer around both cerebral convexities and along the tentorium, which are similar in volume to the prior study. the basilar cisterns are not effaced. there is no new major vascular territorial infarction. the temporal and occipital bone fractures are similar to the prior study. there remains blood in the right mastoid air cells. there remains small fluid levels in both maxillary sinuses and the left sphenoid sinus air cell. opacification of the right sphenoid sinus air cell is probably due to a combination of acute blood and chronic secretions. several of the ethmoid air cells are opacified. impression: 1. significant short interval worsening in right frontal intraparenchymal hemorrhage with new mass effect, which causes shift of the anterior midline structures to left by about 5 mm and mild effacement of the frontal of the right lateral ventricle. 2. worsening in left frontal subarachnoid hemorrhage and contusion. 3. no appreciable change in subdural hematomas of the bilateral cerebral convexities and along the tentorium. 4. redemonstration of temporal and occipital fractures. ct head : findings: the right frontal mixed attenuation parenchymal hemorrhage appears relatively stable in size measuring approximately 5.4 x 3.6 cm with increased effacement of the ipsilateral frontal of the lateral ventricle. no change in shift of anterior midline structures by approximately 5 mm detected. there is relatively stable appearance to bilateral subdural hematomas layering around both cerebral convexities and along the tentorium. there is no evidence of uncal/transtentorial or tonsillar herniation. there is large amount of intraventricular hemorrhage within the occipital horns of the lateral ventricles bilaterally with unchanged degree of mild hydrocephalus. the temporal and occipital bone fractures are similar to prior study. there is stable opacification in bilateral mastoid air cells, right maxillary sinus and sphenoid sinuses. several ethmoid air cells are also opacified. impression: 1. large right frontal mixed attenuation parenchymal hemorrhage with increased effacement of the ipsilateral frontal however size and associated midline shift appears stable. 2. no appreciable change in bilateral subdural hematomas. there is no evidence of uncal/transtentorial or tonsillar herniation. 3. re-demonstration of temporal and occipital fractures. 4. unchanged opacification of sinus opacification. 5. large amount of intraventricular hemorrhage with unchanged degree of mild ventricular dilatation. ct head : findings: redemonstrated is the large right frontal intraparenchymal hemorrhage which is similar in size compared to at 9:12 a.m. there has been interval evolution with the periphery of the hemorrhage now hypodense relative to brain parenchyma consistent with edema/infarction. effacement of the frontal of the right lateral ventricle is similar to the prior study. there remains slight shift of the anterior midline structures to the left by about 5 mm, not changed. left frontal parenchymal contusion and subarachnoid hemorrhage has not appreciably changed. subdural hematomas which layer around the bilateral cerebral convexities and along the tentorium are similar to the prior study. the volume of blood layering within the occipital horns of the lateral ventricles has diminished. the size and configuration of the ventricular system is stable. there is no effacement of the basilar cisterns. there remains a fluid level in the sphenoid sinus. the right temporal and occipital fractures are similar to the prior study. impression: 1. evolution of right frontal intraparenchymal hemorrhage with surrounding edema/infarction. no appreciable change in mass effect with effacement of the frontal of the right lateral ventricle and shift of the anterior normally midline structures to the left by about 5 mm. similar appearance of left frontal parenchymal contusion and subarachnoid hemorrhage. 2. no significant change in bilateral subdural hematomas. 3. diminished amount of blood layering within the occipital horns of the lateral ventricles. no change in ventricular size or configuration. 4. right temporal and occipital fractures redemonstrated. ct head : findings: there is a large right frontal intraparenchymal hemorrhage of similar size with a similar moderate amount of surrounding edema exerting mild leftward shift of the midline approximately 4 mm, unchanged compared to prior study. there is an unchanged right temporal subdural hematoma and unchanged bifrontal subdural hematomas. there is an unchanged left frontal hypodensity consistent with contusion. subarachnoid blood within the left frontal and posterior temporal lobe are stable. there is no hydrocephalus. blood is layering within both occipital horns to a small degree, right greater than left. there is hyperdense fluid within the sphenoid sinuses with right skull base fractures again demonstrated that are unchanged. impression: 1. unchanged head ct. 2. unchanged large right intraparenchymal hemorrhage. 3. unchanged bifrontal and right temporal subdural hematomas. 4. unchanged left frontal and posterior temporal subarachnoid hemorrhage. 5. unchanged left frontal contusion. 6. similar blood layering within the occipital of ventricles with no evidence of hydrocephalus. 7. no change in mild leftward shift of the midline . pa & lateral view, chest : mild bilateral pleural effusions and vascular plethora more prominent at lung bases. there is also evidence of retrocardiac opacity suggestive of either left lower lobe atelectasis or aspiration. old rib fracture in the posterior 9th left rib is noted. there is no pneumothorax, hilar contours are normal. pulmonary vasculature demonstrates mild engorgement. cardiomediastinal silhouette and heart size is within normal limits. impression: interval development of small bilateral pleural effusion and mild vascular plethora, suggestive of volume overload. retrocardiac opacification could be due to left lower lobe partial atelectasis or aspiration. . eeg study date of mpression: abnormal eeg due to the mildly slow and disorganized background and due to the occasional left hemisphere sharp wave discharges (very rarely on the right). the first abnormality signifies a widespread encephalopathy affecting cortical structures broadly. metabolic disturbances, infection, and medications are among the most common causes. there were no areas of prominent and persistent focal slowing. sharp waves indicate cortical hypersychrony and raised the possibility of an epileptogenic focus, but there were no simple discharges to suggest ongoing seizures. brief hospital course: a/p: 79 yo female with dm found down with bilateral subdural hemorrhage, also developed dka during her stay. . # subdural hemorrhage: the patient was admitted to the sicu under the neurosurgery service for bilateral sdhs and a right frontal contusion. she was moving all extremities when her sedation was off but she was intubated initially. on her ct showed increased hemorrhagic contusion, but her neuro exam remained stable. she had an mri of the c-spine which showed no injury. she had multiple cts during the remainder of her stay, all of which were stable. she was initially managed on dilantin, but because of a rash, was transitioned to keppra. she had an episode of seizure like activity, after which her keppra was titrated up to her discharge dose. she has follow-up with dr. of neurosurgery on at 3pm. dr. office will call with date/time of ct head and mr head (w and w/o contrast). dr. phone: . # dm/dka: the patient was extubated on and was transferred to the floor. on the patient became more lethargic and she was hyperglycemic with a glucose of 408. she had another ct scan which was stable with no increase in hemorrhage. her sugars continued to be elevated overnight an consult was obtained on . she was transferred to tsicu team for dka, and then transferred to the micu. her anion gap closed while in the micu and she was tranferred to the floor where continued to follow and titrate her standing and sliding scale insulin. her blood sugar should be followed and titrated as needed. she may benefit from an ace-i as an outpatient, but was not started during this admission. please call dr. with for any questions on her blood sugar control. , phone: . # pna: on the patient's cxr revealed "small bilateral pleural effusion and mild vascular plethora, suggestive of volume overload. retrocardiac opacification could be due to left lower lobe partial atelectasis or aspiration." she was started on vanc and zosyn for nosocomial pna, which she received a 7 day course and finished on . . # uti-ucx enterococcus sensitive to vanc. difficult to assess symptoms given mental status. initially on cipro, d/c'd on 9th. finished vanc course as above. . # urinary retention: foley was discontinued during her stay, but had to be replaced given residual urine over 400cc. an attempt should be made to take her foley catheter out again while in . . # fever/leukocytosis: ? cdiff given recent antibiotics. she was cdiff negative x 3 during her stay. no witnessed aspiration and tube feeds without residual, so doubt aspiration. antibiotics d/c'd on . her wbc trended down during the last week of her stay and nadired at 12-14. . #hct- stable around 25, though continues to be inconsistent. hapto wnl. guaiac positive stools. s/p overall normal egd when peg tube placed. iron studies consistent with chronic disease. . # fen: she was given a peg tube and her tube feeds were titrated to goal per nutrition recs. . # access - picc, peg . # ppx: she should have sc heparin and bowel regimen. . # code: per family wishes, she was transitioned to dnr/dni during her stay. . # dispo and follow-up: she was transferred to a rehab facility on for further pt/ot, nutrition feedings, and ongoing neurological recovery. she will f/u with her pcp as needed after discharge from rehab. follow-up with neurosurgery as above. medications on admission: insulin (nph and sliding scale regular) fosamax discharge medications: 1. senna 8.6 mg tablet : one (1) tablet po bid (2 times a day) as needed. 2. docusate sodium 50 mg/5 ml liquid : ten (10) ml po bid (2 times a day). 3. heparin (porcine) 5,000 unit/ml solution : 5000 (5000) units injection tid (3 times a day). 4. bisacodyl 5 mg tablet, delayed release (e.c.) : two (2) tablet, delayed release (e.c.) po daily (daily). 5. acetaminophen 325 mg tablet : 1-2 tablets po q6h (every 6 hours) as needed. 6. calcium carbonate 500 mg tablet, chewable : one (1) tablet, chewable po daily (daily). 7. cholecalciferol (vitamin d3) 400 unit tablet : one (1) tablet po daily (daily). 8. metoprolol tartrate 25 mg tablet : one (1) tablet po bid (2 times a day). 9. lansoprazole 30 mg tablet,rapid dissolve, dr : one (1) tablet,rapid dissolve, dr daily (daily). 10. insulin regular human 300 unit/3 ml insulin pen : as per sliding scale below units subcutaneous every six (6) hours. 11. levetiracetam 100 mg/ml solution : five (5) ml po qam (once a day (in the morning)). 12. levetiracetam 100 mg/ml solution : ten (10) ml po qpm (once a day (in the evening)). 13. lantus 100 unit/ml cartridge : thirty (30) units subcutaneous at bedtime. 14. loperamide 2 mg capsule : one (1) capsule po qid (4 times a day) as needed. discharge disposition: extended care facility: - discharge diagnosis: bilateral sdhs right frontal contusion sah dka now resolved nosocomial pneumonia discharge condition: stable for discharge to her rehab facility discharge instructions: you were admitted to the hospital after a fall and developed bleeding in the fronal lobes bilaterally. you also developed diabetic ketoacidosis and are now on a stable insulin regimen. you were treated for a full course of hospital acquired pneumonia. and you also had a feeding tube placed for nutrition. . please take medications as instructed below. . if you develop fevers, worsening cough, abdominal pain, vomiting or diarrhea, or any other concerning symptoms, please call your doctor or report to the nearest er. . followup instructions: you have follow-up with dr. on at 3pm. dr. coordinator will call with times for a follow-up mri (with and without contrast) and ct head. dr. phone: md procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more insertion of endotracheal tube enteral infusion of concentrated nutritional substances percutaneous [endoscopic] gastrostomy [peg] transfusion of packed cells diagnoses: anemia of other chronic disease urinary tract infection, site not specified other convulsions polyneuropathy in diabetes pneumonitis due to inhalation of food or vomitus accidental fall on or from other stairs or steps retention of urine, unspecified dermatitis due to drugs and medicines taken internally background diabetic retinopathy diabetes with neurological manifestations, type ii or unspecified type, uncontrolled closed fracture of base of skull with subarachnoid, subdural, and extradural hemorrhage, with loss of consciousness of unspecified duration family history of diabetes mellitus diabetes with ketoacidosis, type ii or unspecified type, uncontrolled closed fracture of vault of skull with subarachnoid, subdural, and extradural hemorrhage, with loss of consciousness of unspecified duration 

Answer: The patient is high likely exposed to