Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins attending: chief complaint: seizure major surgical or invasive procedure: intubation history of present illness: yo lady with dementia, psychosis , old cva, who lives at was transfered to for evaluation form osh. she was found to have a witnessed gtc this pm lasting 'few mins". i called the nh in order to get the info but the rh on duty that time had left and hence details couldnt be obtained. after sz, she was found unrepsonsive and taken to osh. there she was found to be drowy and was intubated for airway protection. temp was 97.7 and bp was 180/120 hence given labetalol iv (20). she was loaded with 500 of fosphenytoin and atiavn 2 mgs. etomidate, succinyl, vecur amd fentanyl was used for intubation. she underwent basic labs and ct head both of which didt show any acute abn. labs: cbc : 4.7, 11/34, 241. chem 7 was normal with ca of 9.5. alb 3.1, ct head was "nil acute;. cxr didnt show anything acute. she was sent to for further eval. next, neurology was called. per chart and family, she does not have h/o seizures in the past. there has been no recent change of meds, head injury. no fevers or sick contacts. past medical history: ?r mca embolic stroke dementia gerd psychosis htn anemia lipids oa social history: lives in nursing home family history: unable to obtain physical exam: vitals were 98.1 142/68 86 20 1001l heart: irregular rhythm, 3/6 systolic murmur loudest in mitral area but audible in aortic area and left lower sternal border, does not radiate to carotids lungs: ctab abd: soft, nt, nd extrem: no swelling mental status exam oriented to self, hospital in , year. thinks it's , can't remember name of hospital. can register and remember at 3min and with multiple choice. can spell world forward and backward. can name face of a watch, calls the clasp the "wrist part" of the watch. neurologic cns acuity 20/100 when both eyes tested together. eomi bilaterally. no nystagmus. left pupil 6mm, unresponsive to light and irregular, right pupil 3 to 2. sensation to light touch equal over all 3 divisions bilaterally. smile slightly asymmetric with slight droop on left. can close eyes tightly symmetrically. tongue protrudes midline and moves laterally symmetrically. can shrug shoulders symmetrically. sternocleidomastoid strong bilaterally. reflexes: biceps , brachioradialis , triceps 0, patellar 0, babinski - minimal movement in toes motor: no pronator drift. atrophy in upper and lower extremities. tone normal. strength 5/5 in delts, in biceps and triceps, in 5th digit adduction, left forearm extensor, right forearm flexor, in quads, dorsiflexors. sensory: sensation to light touch and pinprick intact and symmetric in both arms and legs. coordination: can do rapid finger-thumb movements bilaterally pertinent results: 10:00am wbc-9.6# rbc-3.81* hgb-11.3* hct-33.0* mcv-87 mch-29.6 mchc-34.2 rdw-14.1 10:00am neuts-84.6* lymphs-9.3* monos-5.3 eos-0.1 basos-0.6 10:00am plt count-273 10:00am pt-12.9 ptt-23.3 inr(pt)-1.1 10:00am glucose-147* urea n-24* creat-0.9 sodium-138 potassium-3.9 chloride-101 total co2-28 anion gap-13 10:00am alt(sgpt)-14 ast(sgot)-19 alk phos-107* tot bili-0.6 10:00am albumin-3.7 calcium-9.4 phosphate-3.6 magnesium-1.5* 09:30pm asa-neg ethanol-neg acetmnphn-neg bnzodzpn-neg barbitrt-neg tricyclic-neg 09:30pm urea n-24* creat-0.8 09:30pm ctropnt-<0.01 imaging: nchct: no intracranial hemorrhage; left frontal centrum semiovale and subcortical hypodensity likely represents asymmetric focus of chronic small vessel disease, unchanged from outside hospital ct, less likely infarct since there is no cortical involvement and unlikely vasogenic edema from underlying mass. mri brain: no definite evidence of acute brain ischemia. chronic small vessel infarction eeg: abnormal portable eeg due to prominent focal slowing and some sharp components in the left temporal region. this suggests a marked focal subcortical dysfunction in that area. a stroke would be a common explanation. the sharp features indicate an are of cortical hypersynchrony and could be related to focal-onset seizures, but they were not diagnostic of seizures. the regular faster background activity suggested sleep or more likely, medication effect. there was no evidence of ongoing nonconvulsive status epilepticus brief hospital course: ms. is y/o woman with a past history of dementia (though her son says she is usually pretty sharp and only required placement in a nursing home about 1 month ago after she was getting recurrent utis and delirium) who lives at and was transfered to for evaluation form osh for a seizure. she was intubated at the osh for airway protection after he seizure. as she was intubated upon arrival, she was admitted to the neuro icu. she was initially loaded with dilantin, but her antiseizure medication was later switched to keppra. she as extubated within 24 hours of being admitted here as she was alert and able to follow commands appropriately. after extubation, she was able to be transferred from the neuro icu to the floor. to work-up her seizure, she had basic metabolic labs checked, which were normal as well as a ua with no indication of uti. she also had a ct, mri and eeg. there were no focal lesions noted on brain imaging, but there was diffuse white matter disease noted related to small vessel infarcts; the imaging was consistent with binswanger's disease. the eeg showed prominent focal slowing and some sharp components in the left temporal region, suggesting a marked focal subcortical dysfunction in that area. for this reason, she should be maintained on keppra for seizure control. she is now taking keppra 500 mg . while she was in the hospital, she developed atrial fibrillation. she was started on metoprolol 25 mg for rate control. she occasionally would refuse to take her oral medications and would recieve iv metoprolol doses when she a. fib with rvr. she was taking plavix at home, which was continued and she was started on asa 81 mg in addition to the plavix for stroke prevention in the setting of a. fib. at this time, given her recent seizure, no coumadin was started. however, this can be considered starting in follow-up if the benefits are determined to outweigh the risks (low fall risk and seizures well controlled). her remeron and risperdal were discontinued during the hospital course. medications on admission: rameron 7.5 plavix 75 miralax risperidol 0.25 colace xalatan 0.005% left eye timolol 0.5 % left eye brimonione eye drops b12 every month discharge medications: 1. clopidogrel 75 mg tablet sig: one (1) tablet po daily (daily). 2. keppra 500 mg tablet sig: one (1) tablet po twice a day. 3. latanoprost 0.005 % drops sig: one (1) drop ophthalmic hs (at bedtime). 4. timolol maleate 0.5 % drops sig: one (1) drop ophthalmic (2 times a day). 5. brimonidine 0.15 % drops sig: one (1) drop ophthalmic q8h (every 8 hours). 6. docusate sodium 100 mg capsule sig: one (1) capsule po bid (2 times a day) as needed for constipation. 7. metoprolol tartrate 25 mg tablet sig: one (1) tablet po bid (2 times a day). 8. aspirin 81 mg tablet sig: one (1) tablet po once a day. discharge disposition: extended care facility: cape heritage, a healthcare center - discharge diagnosis: seizure binswanger's disease atrial fibrillation discharge condition: mental status: confused - sometimes. level of consciousness: alert and interactive. activity status: ambulatory - requires assistance or aid (walker or cane). discharge instructions: you were admitted with a seizure. you were initially intubated for protection of your airway, but after you became more alert, you were extubated without difficulty. an mri of your brain was performed and showed diffuse white matter disease, likely due to small strokes (this is called binswanger's disease). the exact etiology of your seizure is unknown and most seizures have no clear etiology, what is reassuring is that there were no focal brain lesions found on mri that could cause a seizure. an eeg was also performed to assess for seizure activity, this did not show any seizures but did show prominent focal slowing and some sharp components in the left temporal region indicating that there is a potential for seizures to begin in this region. for the seizure, you were started on a medication called keppra. while you were in the hospital, your heart rhythm was noted to be irregular. for this, you were started on a medication called metoprolol, to help control the rate. given the increased stroke risk with this irregular rhyhthm, you were also started on a baby aspirin to help prevent strokes as well as continued on the plavix, which you were previously taking. on your follow-up with dr. , you should discuss whether you should start a medication called coumadin. also, while you were here, your remeron and risperidal were stopped given their possible side effect risks. followup instructions: please follow-up with your pcp 1-2 weeks of discharge. please make sure that she checks your heart rate and rhythm; the medication controlling your heart rate can be increased if your pulse is too rapid. provider: , md, phd: date/time: 2:00 (neurology) procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours diagnoses: anemia, unspecified esophageal reflux unspecified essential hypertension atrial fibrillation other convulsions osteoarthrosis, unspecified whether generalized or localized, site unspecified personal history of transient ischemic attack (tia), and cerebral infarction without residual deficits presenile dementia with delusional features 

Answer: The patient is high likely exposed to