Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: aspirin / bactrim attending: chief complaint: slurred speech, l sided weakness major surgical or invasive procedure: endotracheal intubation. history of present illness: mrs. is an 89 y/o with htn, hyperlipidemia, type ii dm, hx amarosis, who presents as code stroke for acute onset slurred speech and l sided weakness today. she reports that she was at the dentist's office today from 1:00-1:30, and was in her usoh before the appointment. around the time the appointment ended (1:30-2:00) she started noticing that her speech was slurred. shortly thereafter (around 2:00 - 2:30) she started noticing that her left side was weak. she called dr. , her pcp, his office and he directed her to come directly to the ed. code stroke was called at 3:05 and neurology was at the bedside within 5 minutes. head ct was performed and showed a r frontoparietal hemorrhage, therefore tpa was not pursued. past medical history: oa in hips and knees r hip replacement htn hyperlipidemia type ii dm amarosis fugax, on coumadin for awhile but this was stopped after a fall and shoulder fracture many years ago hashimoto's thyroiditis mi s/p angioplasty to lad diverticulosis osteoporosis social history: in , widowed, remote tobacco (quit > 30 yrs ago) family history: unknown physical exam: t- bp- 170/100 hr- 92 rr- 17 o2sat- 98% on ra gen: lying in bed, nad, thin, frail heent: nc/at, moist oral mucosa neck: no tenderness to palpation, normal rom cv: rrr, nl s1 and s2, no murmurs/gallops/rubs lung: coarse transmitted upper airway sounds throughout abd: +bs soft, nontender ext: no edema neurologic examination: mental status: awake and alert, cooperative with exam. oriented to person, place, and date. speech is fluent with normal comprehension and repetition, naming intact, but markedly dysarthric, difficult to understand. intact. + l sided neglect. cranial nerves: r pupil irregular, surgical, 5 mm, but briskly reactive. l pupil 4 -> 3. extraocular movements intact bilaterally, no nystagmus. r gaze preference. l umn facial droop. palate elevation symmetrical. shrug symmetric. tongue midline, movements intact motor: diminished bulk throughout. tone normal. no observed myoclonus or tremor. lifts r arm and leg off bed easily. no movement of l arm with pain. attempts to lift l leg on command, just barely lifts off bed for a second, contracts at ips and bends knee but not anti-gravity. sensation: intact to light touch throughout. + extinction to dss in l arm and leg. reflexes: +1 and symmetric throughout. toes downgoing on r, upgoing on l coordination: not assessed gait: not assessed pertinent results: labs: trop-t: <0.01 141 103 13 ------------< 136 4.2 27 0.7 ck: 65 ca: 9.5 mg: 2.1 p: 3.1 wbc 8.5 hgb 13.8 plt 206 hct 40.5 mcv 93 n:62.3 l:30.7 m:3.9 e:2.6 bas:0.5 pt: 12.2 ptt: 25.0 inr: 1.0 imaging head ct : ct head: there is a large, acute right temporoparietal intraparenchymal hemorrhage measuring 4.4 x 3.6 cm with fluid-fluid levels, a small amount of surrounding edema, and with small regions of adjacent subarachnoid hemorrhage. there is some mass effect along the right lateral ventricle with minimal, 3-mm leftward shift of normally midline structures. there is no hydronephrosis and the basal cisterns are preserved. regions of periventricular and subcortical white matter hypodensities likely relate to chronic small vessel ischemic change. otherwise, the -white matter differentiation is preserved. a 5-mm density along the inner table of the left frontal region likely represents a meningioma. no fracture or region of bony destruction is seen in the skull. bilateral lens replacements are noted. a small mucous retention cyst is noted in the left maxillary sinus. otherwise, the visualized paranasal sinuses and mastoid air cells are well aerated. impression: large right temporoparietal intraparenchymal hemorrhage with fluid-fluid level and adjacent small subarachnoid hemorrhage. there is associated minimal leftward shift. <br> head ct : ct head without iv contrast: compared to the study performed 4.5 hours prior, there is interval increase in size of large right temporoparietal intraparenchymal hemorrhage. a surrounding rim of edema, fluid-fluid levels, and adjacent regions of small subarachnoid hemorrhage are again seen. there is increased compression of the right lateral ventricle, with increased leftward shift of normally midline structures, from 4 to now 9 mm. in addition, there is increased loss of the right basal cistern, consistent with early uncal herniation. the remainder of the exam is largely unchanged. impression: interval enlargement of the large right temporoparietal intraparenchymal hemorrhage with increased leftward shift and early uncal herniation. <br> head ct : findings: again demonstrated is a large right temporoparietal intraparenchymal hemorrhage, with a slight increase in associated edema. this overall collection measures approximately 7 cm x 6 cm, largely unchanged in size from . leftward shift of midline structures, with an 8- mm subfalcine herniation, is unchanged. diffuse subarachnoid hemorrhage in the cortical sulci and along the interhemispheric fissure, and layering blood within the occipital horns of the lateral ventricles, are more apparent from prior study, likely reflecting redistribution. apparent thickening of the tentorium is likely artifactual rather than representing a subdural component. there is no evidence of a major vascular territorial infarction. the ventricular system is unchanged without evidence of hydrocephalus. patchy mucosal thickening of the ethmoidal sinuses is seen. atherosclerotic calcification of the cavernous portion of the carotid arteries is seen. osseous structures are unremarkable. impression: 1. large right intraparenchymal hemorrhage, not significantly changed in size from . however, associated edema is more apparent. 2. diffuse subarachnoid hemorrhage and intraventricular blood is more apparent, likely reflecting redistribution of blood. brief hospital course: mrs. was admitted initially to the neuro icu for close monitoring and blood pressure management. as documented in the ct reports, her bleed got significantly worse after initial presentation despite tight control of her blood pressure. after many family discussions with careful explanations of her likely disabilities if she were to survive and the severe complications that were likely to occur in the acute and subacute periods, her family decided to make her code status comfort measures only (cmo). she was extubated and given prn morphine for comfort. she was transferred to the floor and passed away shortly thereafter. cause of death was cerebral herniation due to intracerebral hemorrhage, likely precipitated by amyloid angiopathy. time of death was 8:47 pm on . autopsy was offered and declined by the family. medications on admission: zocor 10 mg qd triamcinolone 0.025% cream glucovance 5-500 mg synthroid 0.112 mg qd lisinopril 2.5 mg qd metoprolol 25 mg folic acid 1 mg qd provigil 100 mg qd plavix 75 mg qd miacalcin 200 iu qd all: asa (rash), bactrim (rash) discharge medications: none discharge disposition: expired discharge diagnosis: intracerebral hemorrhage amyloid angiopathy discharge condition: expired discharge instructions: n/a followup instructions: n/a md procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube diagnoses: unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled intracerebral hemorrhage compression of brain other and unspecified hyperlipidemia osteoporosis, unspecified hip joint replacement transient retinal arterial occlusion 

Answer: The patient is high likely exposed to