Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: iv dye, iodine containing / codeine / levofloxacin / bactrim / nafcillin attending: chief complaint: altered mental status major surgical or invasive procedure: endotracheal intubation history of present illness: 56yom with h/o esld caused by hep c and etoh cirrhosis who lives at who was found down, being transferred to the micu for hypotension. the patient was reportedly in his normal state without complaints 30 minutes prior to being found down in the bathroom by his caretaker. the patient was reportedly very somnolent and was not verbally responsive or following commands. he was subsequently brought to the ed. of note, the patient was on the transplant list until recently, with a previous meld of 18 in . however, he was taken off the active transplant list due to poor compliance and missing followups in addition to inadequate social support, poor housing, and inadequate period of sobriety. upon presentation to the ed, the patient was found to be somnolent and was given narcan with improved mental status. he was noted to have global aphasia, profound agitation, roving eye movements, and was not blinking to threat in either visual fields. his face was symmetric and he was moving all extremities. a code stroke was called. the patient was intubated with etomidate/succ for ct head and ct torso which was negative for acute hemorrhage or infarct but limited due to motion artifact. there was a concern for possible cerebral edema, and neurology recommended mri head for further evaluation, however then the patient spiked to 104.0 and became hypotensives to sbp 65 s/p ct scan. he was given vanc, ceftriaxone, flagyl. initially responded to boluses, but then was persistently hypotensive, so r-ij placed and neo/levophed ggt started. he was overbreathing the vent so he was paralyzed with vecuronium. he had difficulty maintaining bp on pressors and 5 liters of ns boluses and therefore was given stress dose decadron. foley placed for low uop. he went into a fib w rvr, and was found to have elevated troponins. cardiology said demand ischemia and hypotension contributing, and recommended trending enzymes. he was given calcium gluconate and kayexalate for hyperkalemia with widening of qrs complex. ng placed, given lactulose. there was also concern for trauma because of brusing on the abdominal wall. an og tube was placed which put out yellow/green which progressed to dark brown concerning for gi bleed. protonix/octreotide ggt ordered but not hung. . on arrival to micu, he was maxed out on levofed. a left femoral arterial line was placed and was given fluids wide open (3-4l in micu). initial abg in the micu was 7.13/69/40/24. k was 7.4, and he was given kayexalate, bicarb, calcium gluconate. started stooling w kayexalate, looked maroon. he was subsequently found to have large, unreactive pupils. neuro was consulted and recommended ct head once more stable to evaluate cerebral edema and possible herniation. . review of systems: not able to be obtained as patient is intubated. past medical history: gerd hep c genotype 3a, cirrhosis() c/b egd grade i varices, portal htn with gastropathy depression, hiatal hernia, tips for variceal bleed from alcohol abuse gun shot wound to le carpal tunnel syndrome arthritis polysubstance abuse: heroin abuse, alcohol abuse, and cocaine abuse, hepatic encephalopathy x 3, neuropathy/chronic abd pain, dm ii,acute interstitial nephritis nafcillin () social history: unable to obtain due to mental status. family history: unable to obtain due to mental status. physical exam: on admission to micu general: intubated and sedated heent: bilateral 5 mm, unreactive pupils. dry mm neck: supple, jvp not elevated, no lad lungs: clear to auscultation bilaterally, no wheezes, rales, ronchi cv: regular rate and rhythm, normal s1 + s2, s3 present. no murmurs, rubs, gallops abdomen: soft, obese, distended, small ascities skin: multiple areas of ecchymoses across chest ext: cool and cyanotic pertinent results: 11:20pm blood wbc-9.7# rbc-5.85 hgb-19.4*# hct-56.7* mcv-97 mch-33.2* mchc-34.3 rdw-17.1* plt ct-73* 07:35am blood wbc-13.3* rbc-3.85* hgb-13.0* hct-38.6* mcv-100* mch-33.8* mchc-33.8 rdw-17.8* plt ct-32* 07:55pm blood wbc-13.6* rbc-3.69* hgb-12.7* hct-37.5* mcv-102* mch-34.3* mchc-33.8 rdw-19.6* plt ct-36* 04:00am blood wbc-16.1* rbc-3.47* hgb-11.9* hct-35.9* mcv-103* mch-34.3* mchc-33.3 rdw-19.6* plt ct-56* 11:20pm blood neuts-88.2* bands-0 lymphs-7.8* monos-3.2 eos-0.4 baso-0.5 06:10am blood neuts-79.6* lymphs-10.6* monos-8.4 eos-0.7 baso-0.7 07:35am blood neuts-83.0* lymphs-10.3* monos-5.6 eos-0.6 baso-0.4 04:00am blood neuts-65 bands-10* lymphs-9* monos-9 eos-0 baso-0 atyps-0 metas-7* myelos-0 nrbc-5* 06:10am blood pt-28.4* ptt-85.5* inr(pt)-2.7* 08:18am blood pt-44.2* ptt-136.5* inr(pt)-4.6* 12:50am blood pt-27.5* ptt-60.4* inr(pt)-2.6* 10:55am blood pt-38.3* ptt-50.8* inr(pt)-3.9* 07:35am blood fibrino-67*# 08:18am blood fibrino-71* 08:18am blood fdp-320-640* 10:44am blood fibrino-93* 10:55am blood fibrino-126* 07:55pm blood esr-2 11:20pm blood glucose-116* urean-29* creat-1.9* na-146* k-5.8* cl-106 hco3-13* angap-33* 07:35am blood glucose-175* urean-34* creat-3.0* na-147* k-5.7* cl-115* hco3-17* angap-21* 10:44am blood glucose-60* urean-35* creat-2.8* na-149* k-4.9 cl-118* hco3-14* angap-22* 07:55pm blood glucose-255* urean-41* creat-3.3* na-144 k-5.7* cl-106 hco3-7* angap-37* 10:55am blood glucose-199* urean-40* creat-4.0* na-145 k-5.4* cl-102 hco3-12* angap-36* 11:20pm blood alt-259* ast-918* ld(ldh)-1400* ck(cpk)-* alkphos-185* totbili-4.3* 06:10am blood alt-203* ast-862* ld(ldh)-1335* ck(cpk)-* alkphos-110 totbili-3.0* 10:44am blood ck(cpk)-* 04:00am blood alt-1571* ast-6577* ld(ldh)-6440* ck(cpk)-* alkphos-106 totbili-6.9* 11:20pm blood ctropnt-0.33* 06:10am blood ck-mb-70* mb indx-0.3 ctropnt-1.19* 10:44am blood ck-mb-86* mb indx-0.4 ctropnt-1.89* 07:55pm blood ck-mb-248* mb indx-0.7 ctropnt-1.93* 12:50am blood ck-mb-293* ctropnt-2.11* 04:00am blood ck-mb-310* mb indx-0.7 ctropnt-1.86* 11:20pm blood albumin-3.2* calcium-10.0 phos-2.2* mg-1.8 06:10am blood calcium-7.7* phos-6.9*# mg-1.9 07:35am blood calcium-7.4* phos-5.9* mg-1.7 12:50am blood calcium-6.6* phos-9.9*# mg-2.1 10:55am blood calcium-6.6* phos-9.5* mg-1.9 07:35am blood hapto-<5* 08:18am blood d-dimer-grearter t 04:29am blood ammonia-326* 02:17pm blood tsh-1.7 10:44am blood vanco-8.0* 11:57pm blood type-art temp-38.8 tidal v-550 peep-10 fio2-100 po2-416* pco2-34* ph-7.39 caltco2-21 base xs--3 aado2-263 req o2-51 -assist/con intubat-intubated 01:54am blood po2-422* pco2-30* ph-7.36 caltco2-18* base xs--6 06:23am blood type-mix po2-82* pco2-62* ph-7.14* caltco2-22 base xs--8 07:01am blood type-art po2-40* pco2-69* ph-7.13* caltco2-24 base xs--8 07:41am blood type-art rates-22/15 tidal v-500 peep-10 fio2-100 po2-320* pco2-47* ph-7.20* caltco2-19* base xs--9 aado2-350 req o2-63 intubat-intubated 08:38am blood type-art rates-22/16 tidal v-500 peep-10 po2-73* pco2-47* ph-7.18* caltco2-18* base xs--10 intubat-intubated vent-controlled 10:59am blood type-mix po2-42* pco2-33* ph-7.31* caltco2-17* base xs--8 11:06am blood type-art temp-38.7 rates-22/11 peep-10 fio2-80 po2-200* pco2-26* ph-7.35 caltco2-15* base xs--9 aado2-348 req o2-62 intubat-intubated vent-controlled 02:56pm blood type-art rates-22/13 peep-10 fio2-50 po2-145* pco2-22* ph-7.19* caltco2-9* base xs--17 intubat-intubated 04:00pm blood type- 06:19pm blood type-art temp-36.3 rates-22/12 peep-8 o2 flow-50 po2-154* pco2-20* ph-7.11* caltco2-7* base xs--21 intubat-intubated 07:03pm blood type- temp-36.3 08:13pm blood type-art temp-36.3 peep-8 fio2-50 po2-146* pco2-21* ph-7.13* caltco2-7* base xs--20 intubat-intubated 01:11am blood type-art temp-37.0 peep-8 fio2-50 po2-104 pco2-27* ph-7.06* caltco2-8* base xs--21 intubat-intubated 02:17am blood type-art temp-38.3 peep-8 fio2-50 po2-106* pco2-28* ph-7.10* caltco2-9* base xs--19 intubat-intubated comment-axillary 04:35am blood type-art po2-91 pco2-28* ph-7.13* caltco2-10* base xs--18 06:28am blood type-art temp-37.7 peep-8 fio2-50 po2-90 pco2-25* ph-7.18* caltco2-10* base xs--17 intubat-intubated 11:07am blood type-art rates-22/36 peep-8 fio2-50 po2-pnd pco2-pnd ph-pnd caltco2-pnd base xs-pnd -assist/con intubat-intubated 11:39pm blood glucose-103 lactate-5.5* na-144 k-5.8* cl-109* calhco3-19* 04:05am blood lactate-4.5* k-6.9* 06:23am blood lactate-6.0* k-7.4* 07:01am blood lactate-6.1* k-6.5* 07:41am blood lactate-7.0* 10:59am blood lactate-7.5* 11:06am blood lactate-7.8* 02:56pm blood lactate-11.2* 06:19pm blood lactate-14.8* k-5.7* 08:13pm blood lactate-14.9* 01:11am blood lactate-15.9* 02:17am blood lactate-16.0* 04:35am blood glucose-230* lactate-15.7* na-142 k-5.0 cl-109* 07:01am blood freeca-0.95* 04:35am blood freeca-0.75* 06:28am blood freeca-0.77* 01:30am urine color-dkamb appear-hazy sp -1.025 01:30am urine blood-lg nitrite-neg protein-600 glucose-neg ketone-tr bilirub-sm urobiln-neg ph-6.0 leuks-tr 01:30am urine rbc-76* wbc-7* bacteri-few yeast-none epi-0 01:30am urine castgr-28* casthy-39* 01:30am urine amorphx-rare 01:30am urine mucous-occ 04:50am urine hours-random urean-661 creat-289 na-20 k-88 cl-32 01:30am urine hours-random 04:50am urine osmolal-566 myoglob-presumptiv 01:30am urine gr hold-hold 04:50am urine bnzodzp-neg barbitr-neg opiates-pos cocaine-neg amphetm-pos mthdone-neg brief hospital course: is a 56-year-old male with history of chronic hepatitis c as well as alcohol-induced liver cirrhosis complicated by hepatic encephalopathy, previous variceal bleeds, status post tips procedure, who was admitted to the micu for ams, hypotension, and fevers and who passed away on hospital day 2. . # shock: etiology was uncertain even at the time of death although looked most likely to be septic shock given high fevers and white count. however no clear infectious source was identified. he received broad empiric coverage with vanc, cefepime, and azithromycin. toxic ingestion such as amphetamine overdose or serotonin syndrome was also on the differential because he had fever, ams, and rhabdo, however it was unclear why he would have had hypotension if that was the explanation. amphetamine overdose and seratonin syndrome were considered because he was prescribed adderall and multiple seratonergic medications. however according to his alf he was actively abusing drugs in addition to adderall and therefore he could have had almost any toxidrome. a cardiac component to his shock was initially considered as he had elevated troponins and ck-mb however cardiac output was as measured by nicom. scv02 was high. pe was considered as a possibility as tte showed rv dysfunction and worsened pulm htn, but he had rv dysfunction in the past. bilateral lenis were negative for dvt. he was not stable enough for cta or v/q scan and was unlikely to be able to tolerate anticoagulation given he was also in dic with active bleeding. . the patient was severely ill on arrival to the micu and continued to rapidly deteriorate despite aggressive resuscitation efforts. his blood pressure was not able to be maintained despite fluids and multiple pressors. lactic acid was high on presentation and continued to rise up to 16. he had respiratory failure requiring intubation. his laboratory findings were suggestive of dic and he required cryo, ffp, and blood transfusion. during the hospitalization he developed bleeding from the rectum, bladder, and mucous membranes. he had severe acute kidney injury and associated electrolyte derangements. he also had evidence of shock liver. rhabdomyolysis was presents as well which could be explained by toxidrome but unusual for septic shock. . despite aggressive resuscitation efforts the patient continued to decline. after discussion with the patient's son and also his brothers it was determined that the patient would not want prolonged intubation or resuscitation if he had a small chance of returning to his previous level of functioning. a decision was made to make the patient cmo and take the patient off of the ventilator. he passed away shortly thereafter. . # respiratory failure/hypoxia: most likely this was ards from shock. ct chest showed some small peripheral wedge-shaped infiltrates, which could have been infarcts. he was not stable enough for vq scan or cta. . #altered mental status: infection (cns vs. pulmonary) vs. encephalopathy vs. toxic ingestion. has tox screen positive for amphetamines/opioids, however he was on adderall and opioids at home. nchct did not show any acute process. neurology was consulted and recommended mri although patient was never clinically stable enough to be taken for mri. . # gi bleed: maroon stool, was thought to be possibly from a watershed infarct of colon in setting of profound hypotension. the patient also had known varices but there was only minimal blood-tinged fluid in ng tube. medications on admission: ( alf) acetaminophen 750mg po bid albuterol 90mcg 1puff q6h prn adderall 15mg po bid prn clotrimazole 1% cream to feat vit d 50,000 u wweek fluticasone 110mcg inh 2 puffs folate 1mg once daily thiamine 100mg po daily tums 500mg po bid humalog 75/25 45 units in am 30 units in evening klor-con 20meq po daily mvi po daily omeprazole 20mg po daily lactulose 30ml po qid sertraline 50mg po daily tramadol 50mg po tid ibuprofen 400mg po q6h prn rifaximin 550mg po bid discharge medications: expired discharge disposition: expired discharge diagnosis: expired discharge condition: expired discharge instructions: expired followup instructions: expired procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube arterial catheterization central venous catheter placement with guidance diagnoses: acidosis acute kidney failure with lesion of tubular necrosis chronic hepatitis c without mention of hepatic coma diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled acute and subacute necrosis of liver alcoholic cirrhosis of liver unspecified septicemia severe sepsis acute respiratory failure defibrination syndrome septic shock other sequelae of chronic liver disease acute vascular insufficiency of intestine rhabdomyolysis 

Answer: The patient is high likely exposed to