Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: asystolic arrest major surgical or invasive procedure: endotracheal intubation placement of arterial line placement of femoral venous catheter history of present illness: note: this discharge summary should be signed by dr. , who was the attending of record; however, poe lists the only option as dr. . 83 yo f with pmh significant for cad, dvt, chf, presenting with witnessed asystolic arrest in the field to the ed. earlier in the day, she went with her husband to his pcp with dr. , who is also her pcp. was reportedly feeling well at that time around 10 am. in the taxi back to the apartment, she began to feel diaphortic and unwell. she was helped up to her apt and then at that time, her husband called dr. . he then tried to take her bp and was unable. he called ems at 10:50. they arrived at 11:00 and found the patient apneic, pulseless, and started cpr. after several minutes, she was taking agonal breaths and had weak carotid pulses. vomit was seen in her mouth. initial gcs was 3. she was intubated at 11:09 and was still pulseless. at 11;27, she had an initial rhythm of asystole, which resolved to afib. at 11:28, she had her first recorded blood pressure. therefore estimated time pulseless was 40 minutes. . in the ed, initial vitals were hr 50, bp 104/54, 16, 99%, intubated. she then became hypotensive and was started on wide open levophed and dopamine. she was given 5l ns with no urine output. she was given asa 600 pr, dopamine, levophed, vancomycin, levofloxain 500, flagyl, ativan, dilantin, calcium chloride. bedside echo was done which was "normal". cards was consulted and felt that even if this was a cardiac issue, there would be no intervention at this time because of her alarming neuro exam which was documented as pupils fixed and nonreactive. . ed was concerned for pe with acute onset sob, hypotension, and h/o dvt. discussion of lytics, but pt sent for cta which did not show pe. cta showed b/l pleural effusions/ consolidations/ ? air bronchograms. head ct negative past medical history: pcp: . hepatic cyst cad chf, diastolic mr/tr dvt (subclavian vein thrombosis) htn glaucoma hypothyroid svt, paroxysmal urinary incontinence breast ca (s/p lumpectomy and xrt) colonic adenoma hip fracture raynaud's phenomenon myeolpathy skull fracture from fall lung granuloma flu shot given social history: lives with husband in jp. visits family in frequently family history: nc physical exam: t 99, hr 78, bp 131/86 on levophed 0.2 and dopamine 10 initally. sat 80% on ac 500 (688 spont)/32 (38 spont)/10/100%. urine out - o on arrival to the . pulsus negative gen: elderly f intubated, sedated. does not open eyes to voice. posturing with myoclonic jerks heent: pupils fixed, equal at 4mm. neck: jvp flt cv: rrr. nl s1, s2. no gallops. lungs: coarse breath sounds throughout. no wheeze. + bronchial breath sounds. abd: active bs. soft. slightly distended, though appears to be muscle flexing while fighting the vent. no palpable masses. extr: mottled/purple hands and lower legs. thready dp, pt neuro: no corneals, no vor, no gag reflex. no spontanous motor movements. no withdrawal to pain. she has frequent repeative myoclonic jerks of entire body, more on the left arm. occasional bilateral tonic flexions of her arms. pertinent results: 11:57am blood wbc-22.3*# rbc-4.28 hgb-13.9 hct-42.5 mcv-99*# mch-32.5* mchc-32.8 rdw-14.1 plt ct-268# 04:05am blood wbc-14.8* rbc-4.51 hgb-14.1 hct-42.4 mcv-94 mch-31.2 mchc-33.2 rdw-13.7 plt ct-123* 04:20pm blood neuts-44* bands-35* lymphs-17* monos-4 eos-0 baso-0 atyps-0 metas-0 myelos-0 11:57am blood pt-14.0* ptt-28.4 inr(pt)-1.2* 04:20pm blood glucose-301* urean-20 creat-1.3* na-139 k-4.5 cl-109* hco3-17* angap-18 05:53pm blood alt-198* ast-172* ld(ldh)-460* ck(cpk)-104 alkphos-200* amylase-3173* totbili-0.6 11:57am blood amylase-89 05:53pm blood lipase-22 04:20pm blood calcium-9.2 phos-5.8*# mg-1.7 11:57am blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 12:03pm blood type-art po2-105 pco2-47* ph-7.11* calhco3-16* base xs--14 01:55pm blood type-art po2-68* pco2-74* ph-7.02* calhco3-21 base xs--13 01:04am blood type-art temp-36.3 rates- tidal v-600 peep-10 fio2-100 po2-77* pco2-36 ph-7.11* calhco3-12* base xs--17 aado2-606 req o2-98 intubat-intubated vent-imv 02:05pm blood type-art temp-36.7 fio2-100 po2-154* pco2-27* ph-7.17* calhco3-10* base xs--17 aado2-532 req o2-89 intubat-intubated vent-imv 11:59am blood glucose-289* lactate-10.4* na-141 k-5.1 cl-103 calhco3-17* 02:05pm blood lactate-4.7* initial cxr: right mid lung zone opacity which could represent either asymmetric pulmonary edema versus consolidation. probable skin fold on right but re-evaluate with follow up film to rule out tiny pneumothorax. initial head ct: there is no new intracranial hemorrhage, mass effect, or shift of normally midline structures, interval development of a major or minor vascular territorial infarction. the density values of the brain parenchyma are normal aside from what is likely a mild degree of bifrontal chronic microvascular infarction within the periventricular white matter. this abnormality, as well, is stable since the prior study. initial chest cta/ abdomen/pelvis ct: 1. no evidence of central or segmental pulmonary embolism. no evidence of aortic dissection or aneurysm. 2. extensive bilateral pulmonary opacities, slightly improved compared to . 3. the nasogastric tube should be advanced further into the stomach. 4. peripancreatic fat stranding, which is concerning for pancreatitis. correlation with pancreatic enzymes is suggested. 5. acute left lateral rib fracture. cxr: 1. right lower lobe pneumonia. 2. improving pulmonary edema. 3. stable small left effusion and left lower lobe atelectasis. brief hospital course: the etiology of mrs. asystolic arrest and resultant anoxic brain injury remained unclear. to the rapid onset and prodrome of diaphoresis and sob, it was most likely to have been cardiogenic in nature. after initial evaluation and stabilization in the icu, spoke with mrs. husband about the gravity of the situation, explaining that due to her physical findings, serious anoxic brain injury was likely. placed on dilantin, due to extensive myoclonic activity. valium was also used prn when thought to be experiencing greater myoclonic and possible seizure activity. also placed on broad spectrum antibiotics for elevated wbc count and possible pna on cxr. initially required dopamine and norepinephrine for maintenance of map>65; these were weaned off over the first 12-24 hours. neurology was consulted, who agreed with primary team's findings, and recommended the addition of valproic acid for myoclonus and seizure prophylaxis. multiple discussions were held with mr. and his daughter regarding the grave nature of mrs. condition. they requested that aggressive treatment be maintained until her second daughter could fly in from . this was done, and her clinical situation continued to decline, with worsening renal and hepatic function, along with difficulty in finding a ventilatory strategy that provided adequate oxygenation without very high pressures, though she remained in stable, though critical, condition. once mrs. second daughter arrived, her family agreed first to make her dnr, and ultimately to change the focus of care to comfort measures. mrs. pcp was also aware of the evolving situation, and came by to speak to her family. social work also provided emotional support. at 8:30am on , mrs. endotracheal tube was removed. she was placed on a morphine drip and ativan drip, to be titrated to goals of maximal comfort and minimal respiratory distress and myoclonic activity. one hour later, housestaff was called to the bedside. mrs. was unresponsive, with no visible chest wall expansion. she had no pulse, and had no heart or breath sounds after two minutes of palpation and auscultation. she was pronounced dead at 9:30am. her husband and two daughters were present, and declined an autopsy. her pcp was notified. medications on admission: aspirin 81mg--one by mouth every day detrol la 4 mg--1 capsule(s) by mouth qam levothyroxine sodium 88mcg--one by mouth every day lipitor 20 mg--1 tablet(s) by mouth at bedtime toprol xl 50 mg--1.5 tablet(s) by mouth once a day verapamil 80 mg lumigan eye gtts both eyes nightly timoptic eye gtts gtts both eyes timolol eye gtts both qam citracal centrum baby asa vit e discharge medications: n/a discharge disposition: expired discharge diagnosis: anoxic brain injury resulting from asystolic arrest discharge condition: deceased discharge instructions: n/a followup instructions: n/a md procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified arterial catheterization infusion of vasopressor agent diagnoses: acidosis coronary atherosclerosis of native coronary artery acute kidney failure with lesion of tubular necrosis congestive heart failure, unspecified unspecified essential hypertension acute and subacute necrosis of liver unspecified acquired hypothyroidism personal history of malignant neoplasm of breast other convulsions hypotension, unspecified pneumonitis due to inhalation of food or vomitus cardiac arrest anoxic brain damage personal history of venous thrombosis and embolism closed fracture of one rib chronic diastolic heart failure 

Answer: The patient is high likely exposed to