Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: s/p vfib arrest major surgical or invasive procedure: 1. intubation 2. mechanical ventilation 3. femoral line 4. cardiac catheterization with stenting to lad 5. right radial arterial line history of present illness: mr. is a 57 year old male with history of mild hyperlipidemia who presents s/p vf arrest. pt was playing basketball, and his legs buckled, and he fell to the ground, and hit his head. cpr was started at the scene until the ems arrived, ~7-10mins per report. when ems arrived, cpr was continued, and he was shocked x3, subsequent asystole, and then into pea. cpr was continued until he arrived at the ed. in the ed, she was given epi, atropine, and bicarb with return of pulse. ecg demonstrated ste in v2-v3, avl... placed on arctic sun after ct head negative for ich and sent to cath lab. urine tox positive for opioids, but negative otherwise. serum tox negative. . in the cardiac cath lab, he was found to have a mid-lad completely occluded, with des x1 placed, with return of good flow. lcx was without disease, and rca with 40-50% disease. he required levophed on low dose continued during the cath. he was loaded with prasugrel in the lab, and continued on integrillin for 1 hr. . unable to obtain complete ros given pt intubated and sedated. per his wife, prior to this, he was in his usual state of health. she denied him reporting and chest pressure, pain, or sob. his only recent complaints were a mild knee injury. otherwise he had been feeling well. he is quite active and uses an arc trainer at home. he also plays with his 6 year old son. past medical history: 1. cardiac risk factors: - diabetes, + dyslipidemia, - hypertension 2. cardiac history: -cabg: none -percutaneous coronary interventions: none -pacing/icd: none 3. other past medical history: - torn medial meniscus on right knee - elbow injury social history: he lives at home with his wife and 6 year old son. is self-employed full-time. -tobacco history: denies -etoh: denies -illicit drugs: denies family history: father with cabg at 74 or 75yo, paternal gf with heart disease no family history of early mi, arrhythmia, cardiomyopathies, or sudden cardiac death; otherwise non-contributory. physical exam: admission physical exam: vs: t=34.2c bp=127/83 hr=73 rr=14 o2 sat=98% on ventilator general: intubated, sedated, not responding to any commands or stimuli heent: large l frontal laceration, no active bleeding. sclera anicteric. pupils 1mm, reactive. neck: supple, unable to appreciate jvp given body habitus cardiac: rrr, nl s1 s2, unable to appreciate murmurs over breath sounds lungs: intubated, bilateral breath sounds anteriorly, coarse breath sounds, no wheezes abdomen: +bs, distended, but soft. arctic sun pads in place. extremities: cool at distal extremities, arctic sun pads in place skin: no stasis dermatitis, ulcers, scars, or xanthomas. pulses: right: carotid 2+ dp 2+ left: carotid 2+ dp 2+ neuro: intubated, sedated, not responding to painful stimuli, pupils 1mm, symmetric, reactive; 2-3 beats of clonus bilaterally, downgoing toes, lipsmacking movements, posturing of right arm . discharge patient died on in early afternoon. pertinent results: admission labs: . discharge labs: . pertinent labs: cardiac enzymes: a1c 5.8 lipid panel: ldl 109 total 184 tg 135 hdl 48 . studies: cardiac cath : comments: 1. coronary angiography in this left dominant system demonstrated severe single vessel disease. the lmca had no angiographically apparent disease. the lad had an eccentric 50% proximal stenosis followed by a mid-occlusion after the 1st setpal and diagonal. the 1st diagonal had 50% origin and mid stenoses. the lcx was a large, dominant vessel without angiographic cad. the rca was non-dominant and had a 50% mid-stenosis. 2. limited hemodynamics revealed a central aortic pressure of 117/82 mmhg while the patient was on 0.03 mcg/kg/min of levophed. 3. successful export thrombectomy and direct stenting of mid lad with 3.0 x 18 mm endeavor des at 12 atms, post dilated with 3.0 mm nc balloon at 16 atms. final angiogram showed 0% residual stensosis, no dissection and normal flow. final diagnosis: 1. severe one vessel coronary artery disease. 2. coma post cardiac arrest. 3. cardiogenic shock. 4. successful lad final diagnosis: 1. severe one vessel coronary artery disease. 2. coma post cardiac arrest. 3. cardiogenic shock. . ct head w/o : impression: no acute intracranial process; specifically no intracranial hemorrhage. . cxr : impression: limited study. probable retrocardiac atelectasis. ett in standard position. . cxr : findings: as compared to the previous radiograph, the endotracheal tube might have been minimally advanced. otherwise, the radiograph is unchanged, with borderline size of the cardiac silhouette, partial right upper lobe atelectasis and bilateral perihilar opacities suggesting mild-to-moderate pulmonary edema. . eeg: impression: this prolonged continuous eeg showed an extremely suppressed background rhythm early on during hypothermia treatment. over the day, the background improved in voltage and frequency. by the end, it showed an encephalopathic pattern with slow frequencies in all areas. there were no prominent focal abnormalities. there were no clearly epileptiform features. . tte : conclusions the left atrium is mildly dilated. no atrial septal defect is seen by 2d or color doppler. left ventricular wall thicknesses are normal. the left ventricular cavity size is normal. there is severe regional left ventricular systolic dysfunction with mid to distal septal, anterior, distal/apical akinesis. the basal lateral wall moves best. no masses or thrombi are seen in the left ventricle. there is no ventricular septal defect. right ventricular chamber size is normal. with focal hypokinesis of the apical free wall. the diameters of aorta at the sinus, ascending and arch levels are normal. the aortic valve leaflets (3) appear structurally normal with good leaflet excursion and no aortic regurgitation. the mitral valve appears structurally normal with trivial mitral regurgitation. there is no mitral valve prolapse. there is mild pulmonary artery systolic hypertension. there is no pericardial effusion. . mri : impression: diffusion abnormalities in the biparietal cortex, predominantly in the medial parietal lobes, compatible with ischemic injury or post-ictal state. . mri : impression: bilateral parietal cortical and subcortical diffusion abnormalities are noted which indicate progression to white matter of the previously noted ischemic lesions secondary to hypoxic injury. bilateral globus pallidi lesions are also noted. no hemorrhage, mass effect or hydrocephalus. . ctap : impression: 1. 5.8 x 3.2 cm hematoma within the right pelvis may be related to recent catheterization procedure. bilateral fat stranding within the groins compatible with instrumentation. 2. areas of hypodensity and hyperdensity within the left iliacus and psoas muscles bilaterally and expansion of the left iliacus. the increased density suggests this is atleast mostly hemorrhage, however, given multifocal findings, septic emboli/abscesses are a consideration. 3. new hyperdense foreign body within the stomach. in a patient intubated with feeding tube, foreign body from dental origin is a possibility. . brief hospital course: hospital course: 56 yo m with hx of mild hld who presents s/p vf arrest, now s/p cardiac cath with des to lad and therapeutic cooling. pt treated with aed??????s for seizing s/p re-warming, and found to have anoxic brain injury. course complicated by spiking fevers, growing haemophilus and e. coli from sputum with concern for aspiration pneumonia and sinusitis. pt continued to spike fevers and antibiotics were broadened. hospital course also complicated by anemia multifactorial in origin, s/p 4 units prbc??????s. pt then developed pre-renal etiology. second neurology opinion was obtained per family request, and prognosis by two neurology teams were conflicting. his aed's were temporarily discontinued due to concern that they may be causing sedation. keppra was restarted as pt was having seizures. he continued to demonstrate poor chance of a meaningful neurologic recovery. on , mr. family elected to extubate him and focus on comfort care. . active issues: # s/p vf arrest, stemi: most likely stemi and active ischemia. ecg demonstrating diffuse st elevations in inferolateral leads. pt required several cycles of cpr, shocks x3, and epi, atropine. pt s/p des to mid-lad as below. he was placed on arctic sun with q4hr lytes for neuroprotection. he was managed for stemi. per the wife's report, pt had no previous symptoms to suggest angina, and had been very active. received 600mg asa in ed. pt requiring levophed on presentation from cath lab. pt loaded with prasugrel in lab and integrillin. pt's risk factors included hyperlipidemia only, on no home meds. started on atorvastatin 80mg po daily, prasugrel 10mg daily, asa 325mg daily. acei and beta blocker were initially held given hypotension initially requiring low doses of levophed. captopril was started for afterload reduction; as his bp's tolerated this he was switched to lisinopril 2.5 daily. beta blockade with metoprolol 12.5mg was started. a1c was checked for risk stratification and was 5.8; lipids were checked and showed ldl 109. he was continued on atorvastatin 80mg daily. his cardiac function appeared to have recovered appropriately; unfortunately he continued to demonstrate poor chance of a meaningful neurologic recovery. on , mr. family elected to extubate him and focus on comfort care. . # hypoxic brain injury: review of mri by both radiology and neurology concerning for extensive hypoxic injury. mri demonstrated diffusion abnormalities in the biparietal cortex, predominantly in the medial parietal lobes, that were compatible with ischemic injury or post-ictal state. family requested second neurology opinion, and per dr. , pt??????s prognosis may not be as poor as initially thought. per dr. , possibly that aed??????s could be contributing to pt??????s mental state. aed's were then held, temporarily, but restarted given continued seizure activity. repeat mri revealed progression of ischemic damage. he continued to demonstrate poor chance of a meaningful neurologic recovery. on , mr. family elected to extubate him and focus on comfort care. . # seizures: pt was found to have seizure activity on eeg after re-warming. neurology and epilepsy were consulted. he was loaded with keppra; however, he continued to seize and was loaded with fosphenytoin. dilantin levels were checked. mri was done, which showed diffusion abnormalities in the biparietal cortex, predominantly in the medial parietal lobes, compatible with ischemic injury or post-ictal state. as above, aed's were briefly held, and then restarted given continued seizure activity. rest as noted above. . # respiratory distress: pt intubated for inability to protect airway in setting of vf arrest. he was kept on mechanical ventilation with settings titrated based on abg's. his seizure activity and myoclonus would trigger breaths on the mechanical ventilator this he required propofol to facilitate ventilation. on , mr. was extubated in keeping with his living will and his family's wishes. . # fevers, leukocytosis: during re-warming, pt had elevated temperature. pt was cultured. he eventually spiked a fever, and cultures were re-sent. sputum cultures grew out haemophilus. mri also suggested sinusitis as possible source. he was treated with iv zosyn. he continued to spike fevers, and wbc climbed from 10, to 16 to 27. likely pulmonary source, possibly aspiration given now speciation of e. coli from sputum. pt also growing haemophilus, and likely sinusitis by mri. the ct scan from also suggestive of possible septic emboli, but thought to be unlikely. concern for c. diff given sig leukocytosis, but negative. he was treated with vanc/zosyn/gent to cover for vap. other possible sources included abdominal given question of septic embolic on ctap. id was consulted. gent was dc'd given worsening renal function, and cipro was started. zosyn was switched to cefepime given mic of 8 to unasyn. id signed off after goal of care transitioned to comfort care. . # anemia: multifactorial ugib on admission, possibly continued gastritis given guaiac positive stools (though no frank blood), phlebotomy, and inadequate production of rbc??????s with low retic count. pt also had a ctap to look for bleed, which showed small hematomas. pt had also been on multiple myelosuppressive medications including zosyn and phenytoin. hemolysis labs were negative. heparin gtt was dc??????d . hct continued to drop, and he was transfused 2 units prbc??????s , with appropriate bump in hct. concern for continued bleeding into abdomen given significant cont??????d drop in hct. serial hct's showed drop, with no further clear source. he was transfused an add'l 2 units of prbc's, for a total of 4 units. his hct remained stable. . # pump: pt presented in vf arrest. unknown prior ef, though pt with no symptoms per his wife of orthopnea, sob, etc. cxr looked like fluid overload, which would not be surprising given stemi in lad region. tte showed severely depressed ef of 15% with severe lv dysfunction. pt had been s/p several cycles of shocks, and was thought to at least in part have stunned myocardium. he was started on heparin gtt given apical akinesis and concern for stroke. captopril was started, which he tolerated, and was switched to lisinopril 2.5mg daily. metoprolol 12.5mg was started as above. pt auto-diuresed initially, but required ivf given fevers as above. repeat tte showed improved lvef of 50%. . # rhythm: as above, presented s/p vf arrest. pt in sinus with prolonged pr interval s/p cath. pt intermittently going into vtach after cath. however, rhythm switched to afib on repeat ecg overnight. on morning of hospital day 1, pt continued to be in afib with hypotension, cardioverted with conversion to sinus. however, pt was then bradycardic, likely cooling. ep was consulted. he was started on lidocaine and amiodarone gtt. however, lidocaine was discontinued given that amiodarone was preferred in the context of reperfusion. he was switched to po amiodarone load. dopamine was initially given for bradycardia and hypotension, but weaned off. he continued on po amiodarone and remained in sinus rhythm. . # : pt??????s cr bumped from 1.1 to 1.6 on the morning of after continued fevers. thought to be pre-renal in etiology given diaphoresis and fevers. ulytes showed fena <1%. his uop was monitored, and he continued to put out good urine. his creatinine improved. gentamycin was dc'd as above. his cr was trended and remained stable. . # ugib: pt had 150cc dark red blood coming up from ogt on admission, concerning for ugib. most likely to integrillin, prasugrel and possible gastritis. gi was consulted. pt had ng lavage with clearing of blood. hct's were checked frequently, and remained stable. he was placed on ppi drip initially, which was switched to iv bid ppi once hct stable. h. pylori was sent, and was negative. gi was consulted prior to starting heparin gtt. given that he was not stable for endoscopy, recommended proceding with heparin as long as pt didn't rebleed. rest as above under "anemia." . # head laceration: the patient sustained a head laceration on his original fall prior to admission. plastics was consulted, and placed several deep and superficial sutures. bacitracin was applied for one day and then dry dressing applied. sutures were removed. medications on admission: - glucosamine - chondroitin - mucinex - advil prn discharge medications: patient expired discharge disposition: expired discharge diagnosis: patient expired discharge condition: patient expired discharge instructions: patient expired followup instructions: patient expired procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more coronary arteriography using two catheters left heart cardiac catheterization insertion of endotracheal tube enteral infusion of concentrated nutritional substances arterial catheterization arterial catheterization closure of skin and subcutaneous tissue of other sites insertion of drug-eluting coronary artery stent(s) cranial or peripheral nerve graft insertion of one vascular stent excision of lingual thyroid percutaneous transluminal coronary angioplasty [ptca] procedure on single vessel diagnoses: acidosis hypocalcemia coronary atherosclerosis of native coronary artery acute posthemorrhagic anemia acute and subacute necrosis of liver acute kidney failure, unspecified atrial fibrillation other convulsions unspecified fall paroxysmal ventricular tachycardia open wound of forehead, without mention of complication other specified forms of chronic ischemic heart disease other and unspecified hyperlipidemia acute respiratory failure pneumonitis due to inhalation of food or vomitus cardiac arrest anoxic brain damage cardiogenic shock pneumonia due to escherichia coli [e. coli] encounter for palliative care do not resuscitate status hemorrhage of gastrointestinal tract, unspecified acute myocardial infarction of unspecified site, initial episode of care hyperosmolality and/or hypernatremia precipitous drop in hematocrit chronic total occlusion of coronary artery coma other agents affecting blood constituents causing adverse effects in therapeutic use activities involving basketball 

Answer: The patient is high likely exposed to