Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: codeine / morphine attending: chief complaint: aicd firing major surgical or invasive procedure: none history of present illness: 84 yo male with hx of ischemic cm, polymorphic vt s/p icd, esrd who presents after aicd firing. pt reports not feeling well for the past month with decreased energy but no chest pain, chest tightness or increasing sob. he has lost lbs and has decreased appetite with dysphagia, and his meals consist almost entirely of oatmeal. he awoke today in his usoh and went to dialysis which he tolerated well. while waiting for his daughter to pick him up he felt a sudden flushing feeling in his face but no chest pain, tightness or palpitations. this resolved spontaneously. then while sitting at home watching tv he felt presyncopal with the same warm feeling and then woke up to his icd firing. he was found by his daughter slumped in his chair but was alert by the time she questioned him. she called ems and he felt well until arrival of ems. while on the ride to the hospital he again felt the "warm feeling" and the icd fired twice in the ambulance. this recurred twice in the ed at . labs revealed a potassium of 2.4 so he was given 10 meq of iv potassium and 20meq po and he was started on a lidocaine gtt at 2mg/min after an initial bolus of 50mg. plan was then made for transfer to for further management. past medical history: 1. esrd, dialysis dependant qtrs followed by dr. 2. htn 3. hyperlipidemia 4. anemia 5. factor v leiden heterozygous, s/p superficial lower ext venous thrombosis. on coumadin with goal inr 1.3 6. prostate cancer - s/p bilateral subcapsular orchiectomy 7. depression 8. polymorphic vt noted with presyncopal and syncopal episodes with a suspected ischemic component; pcm and defib placed and replaced, sp firing after dialysis started on mexilitine, had been on amiodarone that was stopped early for concern of pulm toxicity 9. cad s/p pci with ef 45% 10. lupus anticoagulant 11. hypothyroidism social history: former restaurant owner in , currently lives with daughter. etoh use, history of heavy tobacco use but quit 30 years ago. family history: mother with cardiomyopathy and ? blood clots physical exam: vs: t 97.9 bp 135/75 hr 85 rr 16 o2 98% 2l nc gen: elderly male in nad. oriented x3. mood, affect appropriate. heent: ncat. hemorrhagic conjunctiva on rt eye, sclera anicteric. perrl, eomi. conjunctiva were pink, no pallor or cyanosis of the oral mucosa. no xanthalesma. neck: supple with jvp of 5 cm. cv: pmi located in 5th intercostal space, midclavicular line. rr, normal s1, s2. difficult to hear heart sounds with loud referred bruit. no s3 or s4. chest: no chest wall deformities, scoliosis or kyphosis. resp were unlabored, no accessory muscle use. cta no crackles, dullness to percussion on rt side 2/3 up. abd: soft, ntnd. no hsm or tenderness. abd aorta not enlarged by palpation. no abdominial bruits. ext: no c/c/e. no femoral bruits. lue fistula with good thrill skin: no stasis dermatitis, ulcers, scars, or xanthomas. . pulses: right: carotid 2+ femoral 2+ popliteal 2+ dp 2+ pt 2+ left: carotid 2+ femoral 2+ popliteal 2+ dp 2+ pt 2+ pertinent results: 10:15pm blood wbc-6.1 rbc-2.62* hgb-8.1* hct-23.3* mcv-89 mch-30.8 mchc-34.6 rdw-17.6* plt ct-251 05:21am blood wbc-5.1 rbc-2.39* hgb-7.4* hct-20.6* mcv-86 mch-31.1 mchc-36.0* rdw-17.9* plt ct-250 03:56pm blood hct-25.1* 10:15pm blood pt-28.5* ptt-38.3* inr(pt)-3.0* 05:21am blood pt-29.7* ptt-40.8* inr(pt)-3.1* 10:15pm blood glucose-131* urean-14 creat-2.6*# na-142 k-3.5 cl-98 hco3-36* angap-12 05:21am blood glucose-98 urean-20 creat-3.3* na-141 k-4.8 cl-99 hco3-37* angap-10 03:56pm blood ld(ldh)-151 ck(cpk)-27* 05:21am blood ck-mb-4 ctropnt-0.06* 03:56pm blood ck-mb-notdone ctropnt-0.07* 10:15pm blood calcium-8.0* phos-2.2* mg-2.0 05:21am blood iron-32* 05:21am blood caltibc-165* hapto-301* ferritn-* trf-127* 05:15am blood wbc-5.1 rbc-3.21* hgb-9.9* hct-28.4* mcv-89 mch-30.7 mchc-34.6 rdw-17.2* plt ct-218 05:15am blood gluc-81* urean-46 creat-2.6*# na-1362 k-5.25 cl-101 hco3-25, ca: 8.7 mg: 2.2 p: 3.2 - chest, pa and lateral: comparison is made to . dual lead pacemaker demonstrates a similar configuration. cardiac and mediastinal contours are unchanged. although a left-sided pleural effusion is resolved, there is a markedly increased right-sided pleural effusion, occupying most of the right hemithorax. given the lack of mediastinal shift, there is probably considerable associated collapse of the lower portions of the right lung. the right apex is aerated. the pulmonary vascularity is similarly prominent. impression: interval marked increase in right-sided pleural effusion. . - echo conclusions: the left atrium is mildly dilated. left ventricular wall thicknesses are normal. the left ventricular cavity size is normal. overall left ventricular systolic function is severely depressed with global hypokinesis and akineis of the distal septum, apex and infero-laterla walls. right ventricular chamber size and free wall motion are normal. the aortic valve leaflets are mildly thickened. there is a minimally increased gradient consistent with minimal aortic valve stenosis. mild (1+) aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. moderate (2+) mitral regurgitation is seen. the tricuspid valve leaflets are mildly thickened. moderate tricuspid regurgitation is seen. there is moderate pulmonary artery systolic hypertension. there is no pericardial effusion. compared with the prior study (images reviewed) of , the lvef appears lower and the severity of mr are now more significant. -- cardiac catheterization 1. coronary angiography in this right dominant system demonstrated an lmca, lad, lcx and rca free of angiographically significant cad. the previously placed stents (lad with 2.5x18 cypher, diagonal with 2.5x10 bms and om1 with 3.5x8 and 3.5x6) were patent. 2. limited resting hemodynamics revealed normal systemic arterial pressures. final diagnosis: 1. coronary arteries are normal with patent previously placed stents. brief hospital course: #) cad:the patient has a known history of cad and initially this event did not seem to be caused by ischemia. an echocardiogram, however, showed a worsening ef to 25%, raising ischemia as a possible cause. his cks were flat and troponins were mildly elevated in the setting of renal failure. cath on revealed clean coronaries with patent stents. we continued his asa, b-blocker, ace-i, statin. . #) rhythm: telemetry strips revealed polymorphic vt likely due to severe hypokalemia as has occurred in the past. the patient's potassium was repleted to 4.0. mexilitine was continued at his home dose. the patient had no events until the 3 day of admission when his icd fired after returning from dialysis. potassium was 4.1 and free calcium was slightly low at 1.04. the electrophysiology service was involved in the patient's care. the pacer was interrogated and fired appropriately. a resting thalium mibi was performed to eval for possible scar and an ablatable focus for the vt. this was negative. as above, cardiac cath revealed clean coronaries with patent stents. . #) pump: pt was not clinically volume overloaded except for isolated rt sided pleural effusion. the effusion was thought to be related to inadequate uf with dialysis, but we also considered other processes such as worsening chf and malignancy. cytologic examination of the pleural fluid was negative for malignant cells. #) pleural effusion: a thoracentesis was performed on with 900cc of bloody fluid removed. the procedure was terminated early patient discomfort and a cxr showed a large residual volume of effusion. a repeat thoracentesis was performed on with 700cc serosanguinous fluid removed. procedure was continued despite pt complaining of cp again, but was terminated when needle drew air from pleural cavity. f/u cxr revealed small r basilar pneumothorax, which was treated with 100% o2 by face mask x 24 hours without decrease in size by cxr. pneumothorax remained stable in size and patient was asymptomatic for the remainder of his hospital stay. he was evaluated by interventional pulm and thoracic surgery who both agreed that conservative management was the best approach. . #) hypothyroidism: tsh was checked and was within normal limits. we continued the patient on his out-patient levothyroxine dose. . #) hypercoagulable state: we held the patient's coumadin in preparation for a thoracentesis and cardiac catheterization... . #) esrd: the patient was followed by the renal service and was dialyzed while in the hospital. medications on admission: lisinopril 5 mg daily toprol 25 mg daily mexiletine 150 mg b.i.d. aspirin 81 mg daily levoxyl 0.2 mg daily lipitor 20 mg daily magnesium 400 mg daily celexa 20 mg daily trazodone 100 mg daily coumadin 3 mg daily renagel discharge medications: 1. hospital bed please provide patient with a hospital bed at home. 2. mexiletine 150 mg capsule sig: one (1) capsule po q12h (every 12 hours). 3. lisinopril 5 mg tablet sig: one (1) tablet po daily (daily). 4. metoprolol succinate 25 mg tablet sustained release 24 hr sig: one (1) tablet sustained release 24 hr po daily (daily). 5. aspirin 81 mg tablet, chewable sig: one (1) tablet, chewable po daily (daily). 6. atorvastatin 20 mg tablet sig: one (1) tablet po daily (daily). 7. trazodone 100 mg tablet sig: one (1) tablet po hs (at bedtime) as needed. 8. citalopram 20 mg tablet sig: one (1) tablet po daily (daily). 9. levothyroxine 100 mcg tablet sig: two (2) tablet po daily (daily). 10. b complex-vitamin c-folic acid 1 mg capsule sig: one (1) cap po daily (daily). 11. magnesium 200 mg tablet sig: two (2) tablet po once a day. 12. coumadin 2 mg tablet sig: one (1) tablet po once a day. discharge disposition: home with service facility: discharge diagnosis: polymorphinc ventricular tachycardic arrest with icd discharge discharge condition: stable discharge instructions: weigh yourself every morning, md if weight > 3 lbs. adhere to 2 gm sodium diet fluid restriction: followup instructions: please follow-up with dr. at the interventional pulmonology clinic on at 11am. please call your pcp, . , after your appointment with interventional pulmonology. please follow up with the ep device clinic and dr. on thursday, , at 4 and 4:20p respectively. procedure: coronary arteriography using two catheters angiocardiography of left heart structures left heart cardiac catheterization hemodialysis thoracentesis automatic implantable cardioverter/defibrillator (aicd) check diagnoses: anemia of other chronic disease coronary atherosclerosis of native coronary artery unspecified pleural effusion congestive heart failure, unspecified unspecified acquired hypothyroidism personal history of malignant neoplasm of prostate depressive disorder, not elsewhere classified hypopotassemia hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage v or end stage renal disease other specified forms of chronic ischemic heart disease other and unspecified hyperlipidemia iatrogenic pneumothorax long-term (current) use of anticoagulants personal history of venous thrombosis and embolism primary hypercoagulable state conjunctival hemorrhage paroxysmal supraventricular tachycardia 

Answer: The patient is high likely exposed to