Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: past medical history: 1. status post laparoscopic cholecystectomy, ventral herniorrhaphy . 2. catheterization on : no cad; ef 15% with severe global hypokinesis and left ventricular dilatation; right atrium 15/19/13; rv 32/14/12; pa 30/10/21; pcw 24/31/25, questionable lv 108/13/15; cardiac output equals 6.6; ci equaled 2.5. 3. hepatitis, questionable a, questionable b, questionable c, (per patient). 4. status post left lower extremity burn, status post skin graft. 5. status post spinal fusion, questionably secondary to back trauma. medications on admission: 1. coumadin 7.5 q.d. 2. ambien 5 mg q.h.s. 3. doxycycline 100. 4. lisinopril 10. 5. albuterol. 6. combivent. 7. percocet. 8. amiodarone 200. allergies: the patient has no known drug allergies. social history: the patient lives with his wife, two previous children with previous wife. one pack per day times four years. history of prior heavy alcohol use, 12 drinks per week for the past year. history of heavy cocaine/crack use. denied recent use. the patient is a mechanic involved with a lot of welding. family history: maternal grandfather with cva/diabetes mellitus. physical examination on admission: on physical examination, the patient was an alert, cooperative, well-nourished, well-developed man who looked his stated age of 44, sitting comfortably, in no acute distress. the vital signs included a temperature of 97.1, pressure 150/70, pulse 140, respiratory rate 24, saturating 96% on room air. skin: no peripheral stigmata of liver disease. lymph nodes: no lymphadenopathy. head: nc/at. eyes: anicteric. mouth: mucous membranes moist. the oropharynx was not injected. neck: supple, no thyromegaly. lungs: faint expiratory wheezes, left greater than right. no rales, rhonchi appreciated. cardiovascular: pmi not appreciated. the carotids were 2+ bilaterally and without bruits. jvd 7 cm at 30 degrees. pulse irregularly/irregular. normal s1, s2. abdomen: positive bowel sound. nt/nd. no widened aorta. liver edge felt 2 cm below the costal margin. extremities: no clubbing, cyanosis or edema. pulses 2+ throughout. neurologic: alert and oriented times three. cranial nerves, motor, sensory, and reflex examination within normal limits. laboratory values on admission: sodium 148, potassium 4.8, chloride 100, bicarbonate 31, bun 15, creatinine 0.7, glucose 114. white blood cell count 7.6, hematocrit 48, platelets 163,000. calcium 9.1, magnesium 1.9, phosphorus 3.4. pt/ptt/inr 13.8/29.5/1.3. alt/ast 233/155. ld 258, alkaline phosphatase 74, total bilirubin 1.2, albumin 4. digoxin level 0.4. cardiac biomarkers: ck 24, cardiac troponin i less than 0.3. studies: chest x-ray revealed no evidence of cardiopulmonary disease. ekg on admission irregularly/irregular with a rate of 118 with evidence of t wave inversions in i, v4-6, no prior ekg available for comparison. hospital course: as mr. had not been on warfarin continuously for six weeks secondary to interruption of his anticoagulation due to surgery, the decision was made to perform a transesophageal echocardiogram to rule out thrombus followed by dc cardioversion. the te was performed and as there was no evidence of thrombus, he was successfully dc cardioverted on and begun on heparin and coumadin for anticoagulation. he was felt to be fluid overloaded and diuresis was initiated with iv furosemide. after load reduction was initiated with increase of his lisinopril from 10-20 mg. on hospital day number three, an umbilical hematoma was noted in the setting of a dropping hematocrit accompanied by symptoms of severe abdominal pain. ct with contrast revealed a perisplenic and perihepatic fluid consistent with blood and hematoma in the pouch of . heparin and warfarin were held in the face of a 15 point drop in his hematocrit. his inr of 1.7 at the time was reversed with 1 unit of fresh frozen plasma as well as vitamin k. in the setting of his falling hematocrit, hypotension (systolic blood pressures in the 60s), anuria, rising creatinine (0.7-2.9 over 24 hours), the decision was made to transfer him to the ccu for tailored therapy. his saturations at the time were in the high 80s on 3 liters. he was started on dopamine on the floor prior to transfer. in the ccu, a swan-ganz catheter was floated revealing a wedge of 8, cvp 4, cardiac index 3, svr 494. dopamine was continued in the unit and given his low svr, pitressin was begun with a rise in svr to 800. given his low svr and white count, there was concern for a septic picture and he was begun on empiric antibiotics. post fluids, (total 3 liters nasal saline plus iv), 3 units of packed red blood cells and pressors, his cardiac output rose to 12, cardiac index 4.75, wedge 12, cvp 10, and he was weaned off pressors on . in the unit, a central line was placed. svr was found to be 494 and in the setting of a white blood cell count of 12 and expectoration of green mucus, he was pancultured and started on empiric vancomycin consisting of vancomycin, levofloxacin, and flagyl. in the unit, his central venous line was placed and cvp and wedge revealed him to be dry. fluid boluses were begun and he was transfused 3 units. with volume resuscitation, his renal function improved. his creatinine dropped back down to 0.9. his wedge and cvp rose as well as his cardiac output. his white count dropped. he was transferred back to the floor on hospital day number five. an echocardiogram was repeated on which revealed an ef of 30% and a left atrial size of 5.6 and given a rising hematocrit, the decision was made to begin anticoagulation with warfarin. he was begun on on 3 mg of warfarin (low-dose as he was on flagyl). his hematocrit remained stable despite anticoagulation. he was discharged on hospital day number seven in good condition. 1. cardiovascular: 1) atrial fibrillation: dc cardioverted on . the decision was made not to initiate antiarrhythmic agents. warfarin was begun post cardioversion as well as heparin but was discontinued on secondary to expanding hemoperitoneum. restarted on coumadin 3 mg on once hematocrit had stabilized. discharged with of hearts monitor for two weeks. 2) chf: on admission, he was felt to be fluid overloaded and diuresis was initiated with furosemide. furosemide was discontinued secondary to hypotension on . his hypotension resolved with fluids and pressors, as did his cardiac output, cvp, and his wedge. he was discharged on hospital day number seven on 20 of lasix with follow-up in the heart failure clinic with dr. on . 3) cardiomyopathy: etiology was felt to be most likely tachycardia-induced. the tsh was within normal limits (1.9), ruled out thyroid-induced. hiv was negative. other possible etiologies such as alcohol-induced or cocaine-induced were felt to be somewhat unlikely given that the patient reports abstinence from cocaine in the past year as well a moderation of alcohol intake over the past year. his medical regimen was optimized including beta blocker and ace inhibitor. his ef increased from 10% to 30% prior to discharge. 2. id: on hospital day number five, the patient became hypotensive and was found to have an svr of 494 in the setting of a white count of 12 and expectoration of purulent mucus. gram's stain of sputum revealed 4+ gnr. preliminary cultures grew oropharyngeal flora. he had a negative u/a. he was begun on empiric antibiotics, vancomycin, levofloxacin, flagyl. the vancomycin was discontinued on secondary to negative blood cultures. he was continued on levofloxacin and flagyl for a total of a 12 day course for presumed pneumonia (ct showed increase in bibasilar air space opacity suggestive of aspiration). 3. renal: on hospital day number five, the patient became anuric in the setting of hypotension. creatinine bumped from 0.7 to 2.9 in 24 hours, but following fluids and pressors in the unit, his creatinine dropped back down to 0.9. his acute renal failure was thought to be secondary to prerenal azotemia. 4. pulmonary: at the outside hospital there was apparently concern for copd and the patient was started on nebulizers and questionable steroid taper. we did not feel that copd was an active part of his disease. pfts were performed and were unremarkable. nebulizers were held secondary to concern for beta 1 agonist activity. 5. gi: lfts were elevated on admission, as noted above. hepatitis a and b serologies were sent and were found to be negative. as lfts decreased following diuresis, the picture was felt to be most consistent with hepatic congestion. hepatitis c serologies were not sent. 6. hematology: the patient's admission hematocrit of 48 dropped to 28 at its nadir. his hematocrit stabilized off anticoagulation. once his anticoagulation was resumed, his hematocrit nevertheless rose to 36 prior to discharge. discharge: the patient was discharged on hospital day number seven in good condition. medications on discharge: 1. levofloxacin 500 mg p.o. q.d. times seven days. 2. flagyl 500 mg p.o. q.d. times seven days. 3. oxycontin 10 mg p.o. q. 12. 4. oxycodone 5-10 mg p.o. q. 4-6 hours p.r.n. 5. warfarin 3 mg (patient on flagyl). 6. lisinopril 10 mg p.o. q.d. 7. klonopin 0.5 mg t.i.d. p.r.n. (anxiety). 8. carvedilol 3.125 t.i.d. 9. lasix 20 mg p.o. q.d. follow-up: the patient is to follow-up with dr. in clinic in two weeks. the patient is to follow-up with dr. at on . condition on discharge: good. discharge diagnosis: 1. atrial fibrillation with rapid ventricular response, status post dc cardioversion. 2. dilated cardiomyopathy. 3. acute renal failure. 4. congestive heart failure. 5. intraperitoneal bleed. , m.d. dictated by: medquist36 procedure: diagnostic ultrasound of heart other electric countershock of heart pulmonary artery wedge monitoring diagnoses: pneumonia, organism unspecified other primary cardiomyopathies acute kidney failure with lesion of tubular necrosis atrial fibrillation hemorrhage complicating a procedure other shock without mention of trauma acute hepatitis c without mention of hepatic coma viral hepatitis b without mention of hepatic coma, acute or unspecified, without mention of hepatitis delta 

Answer: The patient is high likely exposed to