Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: ivp dye, iodine containing attending: chief complaint: v fib arrest major surgical or invasive procedure: intubation arterial line placement central line placement artic sun cooling blanket history of present illness: this is a 49 yom with history of type i dm, htn, cad s/p cabg, and ischemic cardiomyopathy who presents after vfib arrest. over the last several weeks has several episodes of dizzy clammy feelings with normal blood sugars. has otherwise been healthy and active. he was at the mall this evening with his wife. was feeling fine. he told his wife suddenly that he needed to sit down. he then quickly passed out, hitting the back of his head on the ground. bystanders preformed cpr. when ems arrived he was found to be in polymorphic vt. he was shocked at 7pm and became asystolic. he was given epi/atopine and returned to a narrow complex rhythm. he was intubated and taken to an osh. at the osh, head ct was reportedly negative. he was started on an amiodarone dirp and transferred by med flight to for futher care. . in our ed, his initial vitals werehr 103, bp 118/63 satting 100% on fio2 100%. he was started on a heparin gtt. he was given versed for aggitation on the vent and had a ct scan of his c spine. he also had a 1.5 cm laceration on the back of his head stapled. . upon arrival to the ccu. patient was stable. neuro was consulted for question of status epilepticus vs myoclonus. a right subclavian central line and a left a line were placed. he was started an artic sun cooling blanket at 12am. social history: significant for former tobacco use. there is no history of alcohol abuse. physical exam: vs: t 37 c bp 122/61 hr114 rr16 o2100% vent: ac 550 rr 12 fio2 60% peep 5 7.35/46/428 gen: wdwn middle aged male heent: sclera anicteric. perrl. conjunctiva were pink, no pallor or cyanosis of the oral mucosa. no xanthalesma. neck: hard c collar in place cv: pmi located in 5th intercostal space, midclavicular line. tachy, rr, normal s1, s2. no m/r/g. no thrills, lifts. no s3 or s4. chest: no chest wall deformities. ctab (anterior), no crackles, wheezes or rhonchi. abd: soft, ntnd. no hsm or tenderness. abd aorta not enlarged by palpation. no abdominial bruits. ext: no c/c/e. no femoral bruits. skin: no stasis dermatitis, ulcers, scars, or xanthomas. neuro: no corneal reflex. perrl. withdraws with purposeful movements in all 4 ext in response to noxious stimuli. exhibits spontaneous posturing movements. . pulses: right: carotid 2+ femoral 2+ popliteal 2+ dp 2+ pt 2+ left: carotid 2+ femoral 2+ popliteal 2+ dp 2+ pt 2+ pertinent results: labs on admission: 09:10pm blood wbc-9.8 rbc-5.05 hgb-11.5* hct-34.1* mcv-68* mch-22.8* mchc-33.8 rdw-14.8 plt ct-158 09:10pm blood pt-12.3 ptt-21.7* inr(pt)-1.1 11:15pm blood glucose-350* urean-25* creat-1.3* na-139 k-4.4 cl-105 hco3-15* angap-23* 09:10pm blood ck(cpk)-269* amylase-55 11:15pm blood alt-122* ast-147* ck(cpk)-271* alkphos-80 totbili-0.8 09:10pm blood ck-mb-5 ctropnt-0.10* 11:15pm blood calcium-8.8 phos-3.7 mg-1.7 09:15pm blood glucose-280* lactate-3.5* na-139 k-3.8 cl-103 calhco3-25 . ekg demonstrated sinus 94 bpm,nl axis and intervals. lvh. twi v1 , st depression in v4-6 with no significant change compared with prior dated . telemetry demonstrated:sinus tach . 2d-echocardiogram performed on demonstrated: the left atrium is normal in size. no atrial septal defect is seen by 2d or color doppler. left ventricular wall thicknesses are normal. the left ventricular cavity size is normal. there is mild-to-moderate global left ventricular hypokinesis (ejection fraction 40 percent). no masses or thrombi are seen in the left ventricle. there is no ventricular septal defect. right ventricular chamber size and free wall motion are normal. the aortic valve leaflets (3) appear structurally normal with good leaflet excursion and no aortic regurgitation. the mitral valve appears structurally normal with trivial mitral regurgitation. there is no mitral valve prolapse. there is no pericardial effusion. . cxr: large heart. right subclavian line terminates at svc. mild pulmonary edema. no ptx . ct c-spine : there is no fracture or malalignment within the cervical spine. the vertebral body and intervertebral disc space heights are preserved. mild calcification of the anterior longitudinal ligament posterior to c2 is degenerative. the odontoid process approximates well with the anterior arch of c1. the visualized outline of the thecal sac is unremarkable. . interstitial edema in bilateral lung apices. the visualized right maxillary sinus is completely opacified, and there is mild mucosal thickening in the left maxillary sinus. mastoid air cells are clear. . mri/mra brain : there is no slow diffusion to indicate an acute infarct. there is a left parietal subgaleal hematoma present. the brain parenchymal signal is normal with no evidence of midline shift or herniation. there is opacification of all of the paranasal sinuses which could be due to intubation. the normal vascular flow voids are present. . tte : the left atrium is mildly dilated. there is mild symmetric left ventricular hypertrophy. the left ventricular cavity size is top normal/borderline dilated. overall left ventricular systolic function is mildly depressed with inferolateral hypokinesis/akinesis. right ventricular chamber size and free wall motion are normal. the aortic valve leaflets (3) appear structurally normal with good leaflet excursion and no aortic regurgitation. the mitral valve leaflets are mildly thickened. trivial mitral regurgitation is seen. there is mild pulmonary artery systolic hypertension. there is no pericardial effusion. . mr : no signal abnormality is identified within the cervical spinal cord. alignment of the vertebral bodies is anatomic. mild cervical spondylosis is unchanged. there remains a small rim of fluid posterior to the anterior longitudinal ligament between the c2 and c5 levels. the anterior longitudinal ligament itself appears intact on these images. also posterior to the c2 and c3 levels, there is a sliver of fluid just anterior to the posterior longitudinal ligament; the ligament itself appears intact. a small amount of fluid is also seen at the lateral atlantodental joints bilaterally. no soft tissue abnormalities are appreciated. the visualized cerebellum and brainstem appear unremarkable. . brief hospital course: pt is a 49 yom with known cad/cm who presents after v fib arrest. . 1) cad/v fib arrest: patient collapsed in mall and was found to be in vfib when ems arrived. he received bystander cpr with estimated time between collapse and ems arrival of 15 minutes. was shocked and became asystolic and then was given epi/atropine and returned to narrow complex rhythm. has h/o cad s/p 4 vessel cabg and ischemic cardiomyopathy. ekg done afterwards did not look ischemic, however may have had acute occlusion of one of his grafts causing his arrythmia without ekg findings currently. had signs of anoxic brain injury on arrival to the ccu including myoclonus and posturing which are poor prognostic indicators. neurology was consulted. he was loaded with iv valproic acid for sz ppx and placed on versed drip. given the patient had an hospital arrest and he was within 6 hours of his arrest the decision was made to treat with therapeutic hypothermia to reduce cerebral metabolic needs. he was placed on vecuronium to reduce shivering during the cooling process. he was cooled down to 89 degrees for 18 hours per protocol and then slowly rewarmed. given the possibility of an ischemic event he was placed on iv heparin and continued on asa. he had been started on amiodarone prior to arrival which was stopped due to prolonged qt interval. he was continued on bb which was uptitrated. . 2) resp: patient was intubated in the field for airway protection. has no history of lung disease. was found to have an aspiration pna which was treated as below. during the hospitalization he did not tolerate his ett, specifically, when attempting to wean him off of sedation he would cough violently and become very agitated. given his poor neurologic status and likely need for prolonged intubation interventional pulmonology was consulted regarding trach placement. a trach was placed on . he continued to have a strong cough despite anesthetics and cough suppressants. he was changed to ps ventilation and tolerated this well and was then weaned to trach mask. with improvement in his pna he was able to be weaned to nc and his trach was capped. . 3) dm: normally on inuslin pump at home. sugars initially elevated to 400 with gap acidosis. likely dka in setting of arrest and no insulin pump. bolused with 1l ns and placed on insulin gtt. his anion gap resolved and he was maintained on the insulin gtt for tight glucose control. was consulted for assistance in transition to sc insulin. once tfs were stabilized he was switched to glargine and riss. his long-acting insulin was uptitrated to 50u to be given at lunchtime daily. . 4) renal insufficency - on admission the patient's cr was slightly elevated to 1.3. etiology was likely poor perfusion in setting of cardiac arrest. his cr returned to baseline and remained stable. . 5) fever: patient developed high fevers on hd#2 up to 102. initially felt to be either central fevers vs. infection, however he then developed bandemia suggestive of acute infection. he was pan-cultured. cxr was consistant with either aspiration pneumonitis vs. pna so he was started on ceftriaxone and flagyl. sputum culture grew out pan-sensitive klebsiella so his flagyl was discontinued. he had recurrent fevers a week into treatment and was recultured. cxr showed worsening of his pna and given the concern for hospital acquired pathogens and vap his antibiotics were broadened to vanc/zosyn. repeat sputum culure grew out mssa and the vanco was discontinued. all urine and blood cultures were ngtd. zosyn, which was started on , needs to be continued for a total of two weeks. . 6) fen: npo initially given patient was paralyzed. tfs were initiated and peg was placed for permanant feeding. . 7) ppx: heparin gtt transiently, then heparin sc, ppi . 8) access: r subclavian, left a line . 9) code: dnr/i medications on admission: niacin 500 toprolxl 50 diovan 80 plavix 75 crestor 5 insulin pump discharge medications: 1. artificial tear with lanolin 0.1-0.1 % ointment sig: one (1) appl ophthalmic prn (as needed). 2. acetaminophen 160 mg/5 ml solution sig: ten (10) ml po q6h (every 6 hours) as needed. 3. valsartan 80 mg tablet sig: one (1) tablet po daily (daily). 4. heparin (porcine) 5,000 unit/ml solution sig: 5000 (5000) units injection tid (3 times a day). 5. ipratropium bromide 17 mcg/actuation aerosol sig: six (6) puff inhalation q4-6h (every 4 to 6 hours) as needed. 6. ferrous sulfate 300 mg/5 ml liquid sig: five (5) ml po daily (daily). 7. metoclopramide 10 mg tablet sig: one (1) tablet po qid (4 times a day) as needed for high residuals. 8. albuterol 90 mcg/actuation aerosol sig: six (6) puff inhalation q4h (every 4 hours) as needed. 9. codeine-guaifenesin 10-100 mg/5 ml syrup sig: 5-10 mls po q6h (every 6 hours) as needed. 10. beclomethasone dipropionate 80 mcg/actuation aerosol sig: one (1) puff inhalation (2 times a day). 11. aspirin 325 mg tablet sig: one (1) tablet po daily (daily). 12. albuterol sulfate 0.083 % (0.83 mg/ml) solution sig: one (1) inhalation q6h (every 6 hours) as needed. 13. metoprolol tartrate 50 mg tablet sig: 1.5 tablets po bid (2 times a day). 14. levetiracetam 100 mg/ml solution sig: one (1) po bid (2 times a day). 15. insulin regular human 100 unit/ml cartridge sig: as dir injection as dir. 16. pantoprazole 40 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po once a day. 17. lorazepam 2 mg/ml syringe sig: one (1) injection q4h (every 4 hours) as needed for seizure activity. 18. piperacillin-tazobactam 4.5 g recon soln sig: one (1) recon soln intravenous q8h (every 8 hours): day 1 = . to be continued for a total of 4 weeks. discharge disposition: extended care facility: - discharge diagnosis: ventricular fibrillation with cardiac arrest. anoxic brain injury. discharge condition: awake, does not follow commands, frequent myoclonic jerks. discharge instructions: you are being discharged after treatment for ventricular fibrillation with cardiac arrest. please note the changes in your medications. you will need to continue antibiotics until the course is completed. followup instructions: follow-up with your cardiologist when your neurological rehab is completed. procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more enteral infusion of concentrated nutritional substances percutaneous [endoscopic] gastrostomy [peg] arterial catheterization temporary tracheostomy closure of skin and subcutaneous tissue of other sites hypothermia (central) (local) diagnoses: hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage i through stage iv, or unspecified aortocoronary bypass status other chronic pulmonary heart diseases open wound of scalp, without mention of complication paroxysmal ventricular tachycardia other specified forms of chronic ischemic heart disease acute respiratory failure pneumonitis due to inhalation of food or vomitus anoxic brain damage long-term (current) use of insulin unspecified disorder of kidney and ureter methicillin susceptible pneumonia due to staphylococcus aureus syncope and collapse pneumonia due to klebsiella pneumoniae other thalassemia diabetes with ketoacidosis, type i [juvenile type], uncontrolled myoclonus insulin pump status other accident caused by striking against or being struck accidentally by objects or persons 

Answer: The patient is high likely exposed to