Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: slurred speech/facial droop major surgical or invasive procedure: arterial line placement history of present illness: 63 yo man with history of recently diagnosed amioderone induced thyroidtoxicosis, af on coumadin, right-sided heart failure, copd, and dm2 who presents from with slurred speech and facial droop. he was recently treated for mrsa endocarditis at . patient arrived to the ed with confusion, minimally interactive, was rapidly intubated due to poor airway protection. he subsequently went into seizure. ct scan showed small left sulcal hyperdensity, over the left parietal lobe, suspicious for subarachnoid hemorrhage. he was sedated on fentanyl and versed. neuro/neurosurg evaluated noted that the small subarachnoid hemorrhage not amenable to surgical intervention. question if there is stroke with possible hemorrhagic conversion. . patient had very varible sbp 70-140, on levo and neo, 4l fluid, foley. he was given profilnine, and vit k and ffp to reverse inr. femoral line in groin. abbroviat in chest wall. patient underwent a cta/ctv of the head (though suboptimal in quality due to lack of peripheral access) and ct of torsal. . in the ed, initial vs were: t p bp r o2 sat. patient was given patient was given naloxone, phenylephrine, levophed, midazolam, fentanyl, phenytoin, phytonadione, piperacillin-tazob, vancomycin, 4l of fluids. . on the floor, patient was intubated, sedated. . review of systems: (+) per hpi past medical history: 1. obesity s/p gastric bypass surgery 2. type 2 diabetes mellitus, on nph insulin 3. atrial fibrillation on warfarin and amiodarone 4. chf with rv dysfunction to osa 5. htn 6. gerd 7. obstructive sleep apnea--has not tolerated cpap 8. amiodarone induced thyrotoxicosis--was treated with steroids, however stopped due to weight gain, psychosis, and hyperglycemia, now on methimazole 9. moderate pulmonary htn social history: home: he lives in with his wife and teenage son. has another son living in the area. occupation: he is a retired school teacher and currently works as a rental car salesman etoh: occasional glass of wine drugs: denies tobacco: denies family history: family history of obesity and stroke. no family history of cad. physical exam: vs: t92.7, hr 93, bp 96/60, 100% on vent general: sedated, intubated heent: sclera anicteric, mmm, oropharynx clear neck: supple, jvp not elevated, no lad lungs: coarse breath sounds, without wheezes, rales, ronchi cv: tachy, regular rhythm, normal s1 + s2, no murmurs, rubs, gallops abdomen: soft, non-tender, distended, bowel sounds present, no rebound tenderness or guarding, no organomegaly gu: foley ext: warm, well perfused, 2+ pulses, no clubbing, cyanosis or edema pertinent results: admission labs 06:10pm blood wbc-11.2* rbc-3.66* hgb-9.6* hct-30.2* mcv-83 mch-26.2* mchc-31.7 rdw-20.1* plt ct-125* 11:32pm blood wbc-6.7 rbc-3.27* hgb-8.9* hct-27.1* mcv-83 mch-27.2 mchc-32.7 rdw-20.2* plt ct-115* 11:32pm blood plt ct-115* 11:32pm blood plt ct-115* 11:32pm blood pt-19.9* ptt-35.0 inr(pt)-1.8* 07:49pm blood pt-39.0* ptt-150.0* inr(pt)-4.1* 06:10pm blood pt-49.1* ptt-46.8* inr(pt)-5.3* 06:10pm blood plt smr-low plt ct-125* 11:32pm blood fibrino-433* 11:32pm blood glucose-138* urean-15 creat-1.1 na-130* k-4.4 cl-94* hco3-18* angap-22* 06:10pm blood glucose-132* urean-15 creat-1.0 na-130* k-4.8 cl-92* hco3-22 angap-21* 11:32pm blood alt-36 ast-38 06:10pm blood ctropnt-0.02* 11:32pm blood calcium-7.9* phos-3.5 mg-1.8 06:10pm blood calcium-7.9* phos-2.9 mg-1.8 11:32pm blood tsh-0.61 06:10pm blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 07:49pm blood redhold-hold 11:46pm blood type-central ve ph-7.32* 07:53pm blood lactate-6.6* 11:46pm blood o2 sat-97 11:46pm blood freeca-0.88* echo on the left atrium is moderately dilated. the right atrium is moderately dilated. no atrial septal defect is seen by 2d or color doppler. there is mild symmetric left ventricular hypertrophy. the left ventricular cavity size is normal. overall left ventricular systolic function is normal (lvef 70%). there is no ventricular septal defect. right ventricular chamber size and free wall motion are normal. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. no aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. there is no mitral valve prolapse. moderate (2+) mitral regurgitation is seen. there is moderate pulmonary artery systolic hypertension. there is a small pericardial effusion. there are no echocardiographic signs of tamponade. compared with the findings of the prior study (images reviewed) of , the findings are similar. ct head on 1. stable appearance of left parietal subarachnoid hemorrhage. 2. no new foci of hemorrhage identified (allowing for limitation, above). 3. opacification of the ethmoidal air cells and fluid layering within the nasal cavity, bilaterally, and the right maxillary sinus and sphenoid air cell. cta/ctv was not performed due to lack of iv access. ct torso on 1. suboptimal evaluation due to hand injection of iv contrast. 2. bilateral pleural effusions, right greater than left with associated compressive atelectasis and non-specific ground-glass opacity in the lungs could represent inflamation or infection. 3. hypodensity in the right kidney is incompletely evaluated and could represent a cyst or other lesion, but infarct cannot be excluded. 4. umbilical and right-sided inguinal bowel-containing hernias without evidence of obstruction/incarceraction. the study and the report were reviewed by the staff radiologist. brief hospital course: hypotension/shock: multifactorial - thought secondary to possibly septic picture (given known source of visa endocarditis) and possible cardiogenic factor. an echo was done and the patient required 4 pressors in order to keep maps in the 60s. he received 1 uprbcs which helped transiently. he was bolused a total of 9l ns as well between the ed and the floor. the patient was started on stress dose steroids given his long standing h/o steroid use. his home medications for htn and afib were held in the setting of hypotension, and cultures were taken for the possible infectious cause. his abx coverage was expanded to linezolid, cefepime, and flagyl while on the floor to cover broadly. an id consult was called, and an svo2 was checked in an effort to differentiate between the possible septic/cardiac etiologies. ultimately, after much of the workup could be completed, the patient's wife opted for comfort care, and he was taken off of the four pressors and made comfortable. he expired shortly thereafter. . &#9658; endocarditis (visa): was on synercid at osh due to lsided lesion, and as above, his coverage was increased to broad spectrum abx. also, he was put in the for the echo to evaluate his valves and overall cv status. as previously mentioned, when he was transitioned to cmo, his abx were stopped. . &#9658; acidosis, metabolic: likely due to hypoperfusion of the end organs given elevated lactate. we continued to trend his lactate, however it continued to rise from 6.6 in the ed to >12 before his death. bicarb was given given the increasing trend, and iv hydration was maintained for perfusion pressures. . &#9658; seizure vs stroke: imaging suggest subarachnoid hemorrhage, not for surgical intervention. the patient was continued on the phenytoin on the floor as he was loaded with dilantin in the ed. a neuro and neurosurgery c/s were called in the ed, and the patient was not a surgical candidate. an eeg was ordered, however this study could not be completed after he was made cmo. . # ventilation: the patient was kept sedated while mechanical ventilation was used. his ct chest notable for b/l pleural effusions with rll segmental collapse. he was extubated when he was made cmo. . &#9658; atrial fibrillation (afib) on coumadin with high inr - given subarachnoid bleed, he was reversed with profilnine, vitamin k, and ffp. we held any anticoagulation and decided to trend inr. . &#9658; anemia, chronic - currently at baseline, we continued to trend his hct. he did receive one u prbc in an effort to increase end organ perfusion. . #) wounds/ulcers ?????? wound care c/s was called . #) code: pt was dnr/dni after a conversation with his wife on . was made cmo after this conversation and expired shortly thereafter. medications on admission: zolpidem 10mg daily digoxin 0.25mg po daily metoprolol tartrate 25mg po bid lisinopril 5mg po daily warfarin 1.5mg po dialy furosemide 60mg po daily prednisone 20mg po dialy epo sq 1/wk (wednesday) synercid 750mg iv q8h daptomycin 650mg x1 insulin ss neutrophos 2 po tid alumina/mag/simethicone po q6h prn kcl 40meq po x1 petrolatum white 1 app topical q2h prn and qhs miconazole powder 1 app topical q shift multivitamin 1 tab po daily ferrous sulfate 325 po bid pantoprazole 40 mg po bid morphine sulfate 2mg iv push q6h prn nepro full strenght daily ibuprofen prn acetaminophen prn discharge medications: none discharge disposition: expired discharge diagnosis: deceased discharge condition: deceased discharge instructions: n/a as pt is deceased followup instructions: none md procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified insertion of endotracheal tube arterial catheterization diagnoses: acidosis thrombocytopenia, unspecified obstructive sleep apnea (adult)(pediatric) esophageal reflux congestive heart failure, unspecified unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled unspecified septicemia severe sepsis chronic airway obstruction, not elsewhere classified atrial fibrillation other chronic pulmonary heart diseases other convulsions subarachnoid hemorrhage acute respiratory failure long-term (current) use of insulin septic shock acute and subacute bacterial endocarditis other and unspecified coagulation defects bariatric surgery status cardiac rhythm regulators causing adverse effects in therapeutic use thyrotoxicosis of other specified origin without mention of thyrotoxic crisis or storm 

Answer: The patient is high likely exposed to