Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: the patient is a 43 year-old male with a history of ethanol use, history of remote intravenous drug abuse approximately six months ago with no known liver problems who presents with hematemesis and fall. he was in his usual state of health until the day prior to admission when he states that he had decreased ethanol intake due to not buying enough for his trip back from . his girlfriend noted that he started to shake and fell to the ground at south station. he presented to the emergency department for evaluation of fall with normal head ct when the patient had witnessed hematemesis of approximately 700 cc. the patient had previous care at and is not aware of his primary care physician. patient states he has a history of hematemesis, but smaller amounts then recently. he denies any history of colonoscopy or esophagogastroduodenoscopy. he has no history of blood transfusions and no abdominal pain, but he does have some back pain and shoulder pain since recent motor vehicle accident. he denies cough or changes in bowel habits. he denies melena, black tarry stools or bright red blood per rectum. in the emergency department his vital signs were temperature 99.4, heart rate 128, blood pressure 155/86, respiratory rate 20, o2 sat 92% on room air. head ct was conducted, which was within normal limits with no acute changes or intracranial hemorrhages, however, subsequently patient had bright red hematemesis of approximately 700 cc and nasogastric tube was attempted, but not tolerated. he was given ativan and protonix 40 intravenous times one and octreotide 50 gtt and he was given 1 unit of fresh frozen platelets, platelets, 1 unit of packed red blood cells, 2 liters normal saline and 1 gram of ceftriaxone intravenous and 10 mg of vitamin k subq on a banana bag. given the patient's gross hematemesis on presentation the patient was transferred to the micu and admitted for treatment and evaluation. past medical history: 1. ethanol abuse, history of delirium tremens per patient. 2. cirrhosis. 3. hepatitis c. 4. nephrolithiasis. 5. lumbar compression fracture of l1 and l3. 6. right shoulder injury status post motor vehicle accident in with fracture to the humeral head. medications: vicodin for pain. allergies: no known drug allergies. social history: significant for ethanol abuse, primarily beer. he denies any history of seizures, although he has had delirium tremens in the past. he smokes less then one pack per day for thirty years. he has a history of intravenous drug abuse in the past, but quit six months ago and he is hiv negative one month ago. he was recently in jail for a dwi approximately 30 days prior to presentation. physical examination: the patient on presentation had a temperature of 99.4, heart rate 120, blood pressure 135/86. respiratory rate 20. o2 sat 95% on room air. generally, the patient was a white middle aged male with rhinophyma and smelling of ethanol. heent examination pupils are equal, round, and reactive to light and accommodation. oropharynx was clear with dried blood. mucous membranes are moist. neck examination supple, no left anterior descending coronary artery. jvp flat. cardiovascular examination tachycardic, normal s1 and s2. no murmurs appreciated. lung examination clear to auscultation bilaterally. back examination no costovertebral angle tenderness. abdominal examination soft, slightly distended, slightly tympanic to percussion, nontender and hyperactive bowel sounds. extremities decreased range of motion and some tenderness on the right shoulder and he has positive for palmar erythema. he had no clubbing in his extremities. neurological examination positive asterixics. cranial nerves ii through xii intact and strength grossly intact. laboratory: his initial cbc showed a white blood cell count of 5.5, hematocrit 30.1, platelets 41, inr 1.6, pt 15.7, and ptt 37.5. his chem 7 was within normal limits except for a potassium, which was 3.1. his alt was 31, ast 135, ld 287, and alkaline phosphatase was 126. amylase and lipase was within normal limits. his albumin was 3.2, total protein was 7.3., total bilirubin was 1.2. his head ct showed no hemorrhage and small soft tissue attenuation, but most likely status post trauma, but no acute changes. his ct of the abdomen and pelvis showed diffuse low attenuation with fatty uptake. there was some attenuation densities in the right hepatic area measuring 2.4 by 2.1 cm, tiny stones were also elucidated and the left kidney with soft tissue attenuation on the right paralumbar region and s7, osteal units. his electrocardiogram showed normal sinus rhythm at 84 beats per minute and normal axis and intraventricular rhythm conduction delay and no st-t wave changes. hospital course: in summary this is a 42 year-old male with a history of alcohol and intravenous drug abuse and no known history of liver disease who presents with coagulopathy and hematemesis with abdominal ct revealing fatty liver and question of hepatomas. 1. hematemesis/coagulopathy: initial evaluation suggested the cause to be liver failure causing a decrease in vitamin k and coagulation factors. therefore he was given platelets and fresh frozen platelets for coagulopathy and vitamin k as well. he was also transfused with 2 units of packed red blood cells to stabilize his hematocrit. he underwent an esophagogastroduodenoscopy, which showed varices, however, he started having alcohol withdraw at the time so the esophagogastroduodenoscopy was halted and the patient was intubated and treated for alcohol withdraw. a repeat electrocardiogram showed grade three esophageal varices, which was successfully banded. he was on sbp prophylaxis, which was then discontinued secondary to no evidence of ascites. he was extubated on , and gradually woke up. he was transferred to the floor from the micu on to the service for further management of his hematemesis and gastrointestinal bleed. hepatology service followed throughout and made sure that the hematocrit remained stable and banding was successful. he was continued on octreotide to prevent further bleeding and the patient denied any further reports of gross hematemesis during the hospital course. the patient was also treated with nadolol 40 mg q.d. and he continued to show no signs of ascites and no evidence indicating sbp, therefore he was not started on fluoroquinolone. he was also continued on his proton pump inhibitor with good results. 2. acute blood loss: decrease in hematocrit and acute blood loss was thought to be secondary to esophageal varices, which on esophagogastroduodenoscopy was grade three. banding of the varices, however, stabilized his hematocrit and his transfusion requirement. his hematocrit remained stable throughout his hospital course status post banding. 3. coagulopathy: this was likely secondary to liver failure. the platelets were at 41 on presentation and with acute gastrointestinal bleed status post 1 unit of platelets and fresh frozen platelets as needed on the micu. he was given vitamin k, fresh frozen plasma and platelets. subsequently he did well without further need of coagulation factors. 4. ethanol and drug abuse: his serum tox and urine tox indicated the patient had been recently on alcohol and he was started on a ciwa scale with ativan p.r.n. for withdrawal. no diazepam was given, given his liver failure and addiction consult was consulted to help the patient deal with his alcohol abuse. 5. fen: the patient had hypokalemia on admission and given his chronic ethanol abuse the patient's calcium, magnesium and phosphate and other electrolytes were repleted prn. thiamine, folic acid were also given he was initially npo given the gastrointestinal bleed, but following his banding he was restarted on a house diet. 6. liver mass: an mra was scheduled to further evaluate the mass that was seen in the ct for a possible hepatocellular carcinoma mass. the mri revealed two focal hepatic lesions with imaging characteristics most consistent with hemangiomas concerning the presence of cirrhosis were quite evident in the mri and dilatations and sequences; follow imaging in approximately four months is to be considered. discharge status: good. he was discharged to home with addiction consult services, information given. on discharge the patient was given instructions on continuation of nadolol and proton pump inhibitor. his follow up plan was to have a repeat banding of his esophageal varices on at 1:00 p.m. at the gi unit on the with dr. . , m.d. dictated by: medquist36 procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube prophylactic administration of vaccine against other diseases endoscopic excision or destruction of lesion or tissue of esophagus alcohol detoxification diagnoses: tobacco use disorder chronic hepatitis c without mention of hepatic coma alcoholic cirrhosis of liver hypopotassemia acute respiratory failure esophageal varices in diseases classified elsewhere, with bleeding other and unspecified coagulation defects alcohol withdrawal delirium acute alcoholic intoxication in alcoholism, continuous 

Answer: The patient is high likely exposed to