Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: rocephin / ceftriaxone attending: chief complaint: ruq pain major surgical or invasive procedure: intubation; cholecystostomy x 2; paracentesis x 3 history of present illness: mr. is a 70 yo male with pmh of dchf, cirrhosis, cri, cad, prostate cancer, and dm who presented to today with increasing ruq abdominal pain, nausea, vomiting, and change in mental status. at the osh his family gave a history of questionable bloody emesis so he underwent an ng lavage which was negative. he was felt to be dry on exam and given 2 l of ivf. he was started on a ppi and given levofloxacin, zosyn, and morphine for pain. lfts wnl except for a tbili of 2.9 which was within his baseline. a ct abd/pelvis showed ascites and a dilated gallbladder with a thickened wall which was changed from previous studies. . in the ed at , vs were: afebrile p 99 bp 111/61 rr 23 o2 sat 94% on 2l. patient was given 1.8 l ns. surgery was consulted and recommended an abdominal ultrasound which preliminarily showed gallbladder wall thickening with nonmobile sludge at the neck. the read stated that the thickening could be related to liver disease, but was concerning for acute cholecystitis. surgery did not feel he was a surgical candidiate. hida scan was recommended for further workup and a cholecystostomy tube could be placed if he is confirmed to have cholecystitis. in the ed he also had a diagnostic paracentesis which showed 24k wbc and 21k rbc with 88% polys. . of note the family has been discussing hospice. his daughter is his health care proxy. . he was hospitalized last week for unclear reasons and is frequently hospitalized. had a recent uti and is still taking nitrofurantoin for another 4 days. last bowel movement was yesterday afternoon. has been having urinary retension for the last few days. his abdominal pain started on sunday suddenly. he states the pain is crampy and throughout his abdomen. it is and doesn't radiate. is constant. had associated nausea and vomiting x 5. denies vomiting of blood. past medical history: 1. dchf 2. htn 3. cad s/p cath in , and a second cath per cards note in , 60% (per old dc from ) 4. cirrhosis secondary to nash, followed at nvmc. complicated by hepatic encephalopathy and depemend on lactulose. denies knowledge of varices. 5. history of left hemisphere tia 6. carotid artery disease s/p left carotid endartectomy 7. dmii 8. hypothyroidism 9. djd s/p left knee arthroscopies,s/p rt. tkr x2 hx of infected knee prosthesis 10. prostate ca s/p resection complicated by incontinence, had been recieved hormonal therapy 11. histroy of + heparin antibody (listed in previous dc summ in , was sent in here and was negative) 12. osa 13. gerd social history: lives with his wife. uses a walker. denies tobacco, alcohol, or drug use. is retired. had worked as an electrician. family history: nc physical exam: vitals: t 96.6 p 99 bp 126/69 rr 24 sat 91% on 2 l nc general: alert, oriented, no acute distress heent: sclera anicteric, mm dry, oropharynx clear neck: supple, no jvd appreciated lungs: clear to auscultation bilaterally, no wheezes, rales, ronchi. decreased air movement throughout cv: regular rate and rhythm, normal s1 + s2, no murmurs, rubs, gallops abdomen: +bs, soft, obese with a positive fluid wave. diffusely tender to palpation throughout. no rebound or guarding. gu: foley present with dark urine ext: warm, well perfused, 1+ dp pulses, no clubbing, cyanosis or edema neuro: alert and oriented to person, place, and month/year but not day. cn ii-xii grossly intact. sensation to light touch intact. skin: right arm with dry skin with pigmented skin changes pertinent results: admission labs: 10:19pm type-art po2-73* pco2-35 ph-7.41 total co2-23 base xs--1 10:19pm lactate-5.0* 08:31pm glucose-423* urea n-43* creat-1.5* sodium-129* potassium-4.4 chloride-97 total co2-24 anion gap-12 08:31pm calcium-6.8* phosphate-3.5 magnesium-1.8 08:31pm plt count-39* 03:43pm wbc-6.8 rbc-3.04* hgb-10.2*# hct-31.4* mcv-103* mch-33.7* mchc-32.6 rdw-15.0 03:43pm pt-23.6* ptt-40.7* inr(pt)-2.2* 01:56pm lactate-6.5* 01:56pm o2 sat-73 01:31pm alt(sgpt)-25 ast(sgot)-48* ld(ldh)-147 alk phos-68 tot bili-3.5* labs on day of expiration: 03:07am blood wbc-11.9* rbc-2.34* hgb-8.1* hct-24.0* mcv-103* mch-34.6* mchc-33.7 rdw-19.3* plt ct-73* 03:48pm blood pt-26.9* ptt-34.6 inr(pt)-2.6* 03:48pm blood fdp-320-640* 03:48pm blood fibrino-84* 03:07am blood glucose-110* urean-167* creat-5.2*# na-142 k-5.3* cl-105 hco3-20* angap-22* 03:07am blood alt-54* ast-226* alkphos-66 totbili-18.6* 04:13pm blood d-dimer-greater th radiology: cxr: impression: 1:21 pm 1. et tube at 5.8 cm above carina. 2. ng tube traced to upper stomach; however, tip not visualized. if needed, a radiograph obtained with abdominal technique would better display the tip of the ng tube. 3. unchanged bibasilar atelectasis and pleural effusions. brief hospital course: patient is a 70 yo male with pmh of dchf, cirrhosis, cri, cad, prostate cancer, and dm who presented to an osh ed today with increasing ruq abdominal pain, nausea, vomiting, and change in mental status concerning for acute cholecystitis. . # sepsis secondary to cholecystitis: the patient presented with ruq pain and imaging which was consistent with cholecystitis. per surgery he was not a surgical candidiate. a hida scan was performed which was consistent with cholecystitis. ir placed a cholecystostomy tube to allow drainage of his infected gallbladder. he was placed on vanc and zosyn initially. intially, culture data grew gnr's; he intially started on cipro and zosyn. initally, the cultures grew e. coli and cipro and vanc were d/c'd. he was continued on zosyn. as more culture data came back, he also grew e. coli that was intermediately sensitive to zosyn. he was switched to meropenem after this. unfortunately, the patient continued to spike temps and his hepatic enzymes continued to rise. through the course of his dressing changes around his cholecystostomy tube, his tube fell out. ir replaced the tube and adequate billous drainage appeared again. even after this intervention, the patient continued to have increased hepatic enzymes and worsening sepsis. patient was also found to have an anion gap acidosis, likely related to elevated lactate in the setting of hypotension and sepsis. on , the patient was intubated due to significantly increased work of breathing. it was felt intubation would improve patient's respiratory status it would help relieve the tremendous pressures on his chest and abdomen from his significant ascites. he abdomen was tapped three times during his course to help improve his respiratory status but little effect. he was unable to be weaned off of the vent, due to high pleural pressures in the setting of the high abdominal pressures pressing on the chest wall. after a prolonged course in the icu that ended in multisystem organ failure, during a family meeting on , he was made dnr by his family. shortly there after he was found to be in dic, and on , the patient was made cmo due to the unlikelihood of recovering from the multi-organ failure and dic. he was made comfortable, extubated and expired with his family present. # acute on chronic renal insufficiency: initially thought to be due to atn, also with a component of abdominal compartment syndrome, due the extremely large volume of ascites and high bladder pressures. his creatinine would transiently improve post paracentesis, but as the fluid would rapidly reaccumulate, his renal function would again worsen. as his course progressed and his liver disease worsened, his kidney function acutely worsened, which was attributed to possible hepatorenal syndrome. he was started on albumin and octreotide, while on vasopressin with no effect. he was not an appropriate candidate for cvvh, which contributed to the family's decision to make the patient cmo. . # cirrhosis secondary to nash: the patient has known cirrhosis complicated by encephalopathy requiring lactulose. on admission he was noted to have some baseline synthetic dysfunction with a mildly elevated inr, additionally it was thought that he had a component of hepatic encephalopathy despite the lactulose. with his continued infection and worsening sepsis his liver function continued to decline, with worsening coagulopathy ultimately with him developing dic. # change in mental status: per report from the ed, his family thought he had change in mental status. in the setting of his acute illness and decreased bowel movements most likely his change in mental status was due to hepatic encephalopathy, but also likely a component of sepsis. after the patient was intubated, he was sedated for some time. he sedation was weaned down, but unfortunately, the patient did not wake up. through the course of this unresponsiveness, the patient's kidney function deterioriated further causing an marked increased in his bun, also with his worsening liver function, there was concern that he was unable to clear the sedating medications from his system. . # atrial fibrillation with rvr: initially occurred post intubation, however in the setting of his hypotension, beta blockers and calcium channel blockers were unable to be used, so he was started on amiodarone. the loading dose and iv infusion controlled his rate well. . # dmii: blood sugars intially well controlled on hiss and glargine. as his infection worsened, he was requiring more insulin. he was placed on an insulin gtt due to his labile blood sugars. medications on admission: lasix 20 mg daily zocor 40 mg daily lactulose 4-5x/day nadolol 20 mg daily aldactone 50 mg daily tolterodine 4 mg po daily donepezil 10 mg po daily citalopram 40 mg po daily levothyroxine 100 mcg po daily magnesium oxide 500 mg po daily nitrofurantoin 100 mg po qid ambien 2.5 mg po qhs prn glargine 22 units sq daily - vs glargine 40 ? lispro ssi prilosec 20 mg po daily hormonal prostate cancer therapy discharge medications: none patient expired discharge disposition: expired discharge diagnosis: primary: respiratory failure secondary to sepsis and mulitorgan failure discharge condition: death discharge instructions: death followup instructions: none procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more insertion of endotracheal tube percutaneous abdominal drainage percutaneous aspiration of gallbladder diagnoses: acidosis thrombocytopenia, unspecified obstructive sleep apnea (adult)(pediatric) coronary atherosclerosis of native coronary artery esophageal reflux congestive heart failure, unspecified cirrhosis of liver without mention of alcohol diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled acute kidney failure, unspecified hepatorenal syndrome hyposmolality and/or hyponatremia severe sepsis unspecified acquired hypothyroidism hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage i through stage iv, or unspecified atrial fibrillation personal history of malignant neoplasm of prostate chronic kidney disease, unspecified acute respiratory failure defibrination syndrome other ascites hepatic encephalopathy septicemia due to escherichia coli [e. coli] acute cholecystitis peritoneal abscess chronic diastolic heart failure other specified retention of urine 

Answer: The patient is high likely exposed to