Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins attending: chief complaint: seizure major surgical or invasive procedure: none. history of present illness: 49y/o m transfer from osh for evaluation of possible toxic alcohol ingestion. patient was brought to osh by ems after family found him shaking/foaming at the mouth. pt reported drinking 6 beers/day x 2 days. he has a history of heavy etoh in the past however he reports being sober x~1yr. per ems report, had had been on a "2-day" binge, stopping yesterday, with prior history of withdrawal seizures. there is also report from ems and osh that the patient has hisotry of etoh abuse and prior seizures. the patient denies this. . he initially went to , where he was reported to have an anion gap of 28, and an osmolar gap of 24. his lactate was 6.8, and serum etoh of 29. no asa/apap was detected. an abg was performed 7.46/37/140. lft's with ast/alt 126/75. pcc was contact and recommended fomepazole. pt was given 15mg/kg of fomepazole (1050mg), as well as a total of 100mg thiamine, 1mg folic acid, 1gm magnesium, 30mg of iv valium, 1gm of ceftriaxone and 4mg of zofran prior to transfer. . his initial vitals in the ed were 99.4 122 142/88 100% 2l nc. toxicology was consulted and they will continue to follow. the patient denies ingestionof any other substances. pt denies f/c, ha, cp, sob, abd pain, n/v/d, tinitus, visual disturbance. he received additional 5 iv valium. he was tachycardic to 110 and this increased to 140-150 with any movement. vitals on transfer: 150/100 110 18 98 ra 99.4 . on arrival, patient is interviewed with interpreter. he again denies drinking before this current episode since . he denies fever/chills/ cough/chest pain/nausea/vomiting. he had difficulty recalling his girlfriend's phone number and his home phone number. his daughter was contact and she stated that he has been drinking chronically for at least a month. past medical history: headaches social history: lives with two daughters. , age 19, another daughter age 7. in construction. has a wife in . denies tobacco and drugs family history: non-contributory physical exam: on admission: vs: temp: 99.2 bp: 150/100 hr:115 rr: o2sat 98ra general appearance: anxious, diaphoretic cardiovascular: (s1: normal), (s2: normal), tachy peripheral vascular: (right radial pulse: present), (left radial pulse: present), (right dp pulse: present), (left dp pulse: present) respiratory / chest: (expansion: symmetric), (breath sounds: clear : ) abdominal: soft, non-tender, bowel sounds present extremities: right lower extremity edema: absent, left lower extremity edema: absent, no(t) cyanosis, no(t) clubbing skin: not assessed neurologic: attentive, follows simple commands, responds to: not assessed, movement: not assessed, tone: normal, tremulous on discharge: vs: temp: 97.0 m98.0 bp: 124/92 (100-126/72-92) hr: 80 (77-103) rr: 18 o2sat100%ra tele: 70s-80s; occasional jumps to 120s gen: nad heent: eomi, perrl, clear oropharynx neck: supple, no lad cv: nl s1, s2, rrr, no m/r/g pulm: ctab, no rhonchi, rales, wheezes abdominal: soft, non-tender, bowel sounds present extremities: wwp, 2+ dps, no edema, cyanosis, clubbing skin: no rashes, lesions neurologic: attentive, motor strength and sensation grossly intact; intact fnf, rapid alternating movements, and heel to shin; wide based ataxic gait pertinent results: on admission: 06:40pm blood wbc-7.4 rbc-3.95* hgb-13.2* hct-37.3* mcv-94 mch-33.3* mchc-35.3* rdw-16.1* plt ct-106* 06:40pm blood neuts-78.6* lymphs-14.1* monos-6.8 eos-0.2 baso-0.3 06:40pm blood pt-12.3 ptt-23.4 inr(pt)-1.0 06:40pm blood glucose-98 urean-4* creat-0.6 na-141 k-2.8* cl-99 hco3-27 angap-18 09:51pm blood alt-68* ast-100* alkphos-69 totbili-1.6* 06:40pm blood calcium-8.2* phos-3.2 mg-2.1 06:40pm blood osmolal-288 06:40pm blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 08:40pm blood type- po2-66* pco2-32* ph-7.51* caltco2-26 base xs-2 comment-green-top 07:31pm blood lactate-1.8 . on discharge from micu: 05:42am blood wbc-6.2 rbc-3.78* hgb-12.7* hct-36.4* mcv-96 mch-33.5* mchc-34.8 rdw-15.8* plt ct-132* 05:42am blood pt-11.6 ptt-23.7 inr(pt)-1.0 05:42am blood glucose-91 urean-6 creat-0.6 na-139 k-3.4 cl-102 hco3-29 angap-11 05:42am blood alt-54* ast-69* alkphos-61 totbili-1.5 05:42am blood calcium-9.1 phos-3.9 mg-1.9 . on discharge: 06:30am blood wbc-8.1 rbc-3.71* hgb-12.2* hct-36.6* mcv-99* mch-32.9* mchc-33.3 rdw-16.1* plt ct-321 06:30am blood wbc-8.1 rbc-3.71* hgb-12.2* hct-36.6* mcv-99* mch-32.9* mchc-33.3 rdw-16.1* plt ct-321 06:30am blood glucose-95 urean-13 creat-0.7 na-143 k-3.8 cl-107 hco3-29 angap-11 05:42am blood alt-54* ast-69* alkphos-61 totbili-1.5 06:30am blood calcium-9.2 phos-4.3 mg-1.8 06:20am blood vitb12-809 imaging: cxr: single view of the chest is obtained without the prior study. there is possible bilateral hilar fullness. the lungs are clear. heart is within normal limits. comparison with the prior chest x-ray would be helpful. mri brain: there is mild cerebellar atrophy. the ventricles and cerebral sulci are abnormally large for age, consistent with mild cerebral atrophy. there is no acute infarction. there are scattered foci of high t2 signal in the supratentorial white matter, as well as a focus of high t2 signal in the midline pons and a focus of high t2 signal in the right cerebellar hemisphere, consistent with small chronic infarctions. the major arterial flow voids are preserved. there is no evidence of parenchymal blood products. there is a small focus of polypoid mucosal thickening in the inferior left maxillary sinus. impression: 1. mild cerebellar and cerebral atrophy, abnormal for age. 2. scattered small chronic infarctions in the supratentorial white matter, pons, and right cerebellar hemisphere. no acute infarction. brief hospital course: 49 y/o with confirmed history of chornic alcohol use (though patient denies) presented to osh with seizure and transferred here for eval of possible toxic alcohol ingestion and treatment of withdrawal. . # alcohol withdrawal. patient had witnessed seizure secondary to etoh withdrawal. patient denies etoh ingestion before two days ago but daughter confirms chronic drinking. patient was admitted to the micu and treated agressively with valium and transferred to the floor on once requirement decreased to q4hours. he received close to 500 mg of valium during his hospital stay. after he was no longer on ciwa, he remained ataxic and tachycardic with movement. was seen by pt who felt that his ataxia was related to his chronic alcohol abuse and would not benefit from further pt/rehab. patient treated with thiamine, folate and mvi. . # ? toxic alcohol ingestion: received fomepizole x 1 at osh. transferred here for tox eval. seen by tox here. on review of osh labs, his osmolar gap was accounted for by alcohol and lactate and it has resolved. there was minimal concern for toxic alcohol ingestion and no indication for further fomepizole. . # h/o lactic acidosis: 6.8 at osh - resolved. likley seizure. . # ataxia- patient note to have broad based ataxic gait even after no longer on ciwa. cerebellar exam was otherwise intact and non-focal. b12 level was checked and within normal limits. he underwent mri brain to assess for cerebellar lesions- this was notable for age advanced global atrophy and scattered chronic small infarcts. was seen by pt who felt that his deficits were not likely to be improved by further physical therapy and rehab and were more likely chronic in nature secondary to his long standing alcohol abuse. he was felt safe for discharge. . # social: patient initialy denied chronic alcohol use though family confirms. patient was seen by social work and continued to deny use of alcohol and necessity of detox/rehab. eventually admitted use of alcohol and voiced desire to quit but wanted to do so on his own without rehab. we emphasized to the patient through an interpreter that he puts his life at risk by drinking and that his seizures, ataxia and brain atrophy were directly related to his use of alcohol. we asked him to establish care with a pcp through or in his home town if more convenient. medications on admission: denies discharge medications: 1. thiamine hcl 100 mg tablet sig: one (1) tablet po once a day. disp:*30 tablet(s)* refills:*2* 2. folic acid 1 mg tablet sig: one (1) tablet po once a day. disp:*30 tablet(s)* refills:*2* 3. multivitamin tablet sig: one (1) tablet po once a day. disp:*30 tablet(s)* refills:*2* discharge disposition: home discharge diagnosis: seizure alcohol withdrawal discharge condition: mental status: clear and coherent. level of consciousness: alert and interactive. activity status: ambulatory - independent. discharge instructions: dear mr. , you were admitted to the hospital because you were having a seizure. we believe the seizure was due to withdrawal from alcohol. you were treated with medications and monitored closely in the medical intensive care unit and then transferred to the general medicine floor when your condition improved. you were seen by social work and physical therapy who offered you resources on alcohol abuse and assessed your physical condition. you had an mri of your head which showed shrinkage of your brain which we believe is related to your use of alcohol. we strongly recommend you stop drinking alcohol as you put your life and the lives of others in danger when you drink. we also recommend you establish care with a doctor who can help manage your health conditions (see below). we have started you on the following medications: - folic acid - thiamine - multivitamin please take them as directed. we wish you a speedy recovery. followup instructions: please call to establish care with a primary care doctor. procedure: alcohol detoxification diagnoses: acidosis other convulsions abnormality of gait other and unspecified alcohol dependence, unspecified volume depletion, unspecified alcohol withdrawal jaundice, unspecified, not of newborn unspecified persistent mental disorders due to conditions classified elsewhere alcoholic liver damage, unspecified primary cerebellar degeneration 

Answer: The patient is high likely exposed to