Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: s/p cardiac arrest major surgical or invasive procedure: intubation and extubation rij placement history of present illness: pt is a 77 yo f with dmii, htn, dementia, and paranoid schizophrenia who presented from an osh after a witnessed cardiac arrest at her nursing home. patient lives at x1 year. per report, she was sitting in a chair before dinner when she was noted to become blue, slumped down into her chair, and became unresponsive. no report of chest pain or shortness of breath. she was carried back to her room, and approximately 3 minutes elapsed before cpr was started. she was coded for 15 minutes, and reportedly had an aed placed with 1 shock prior to emt arrival. a perfusing rhythm was obtained, and she was intubated in the field and taken to . . at osh, pt's vs were 106/65 hr 100. ekg with st depressions in v2-v6 with no prior for comparison. she reportedly had another vf arrest 20 minutes later, and was shocked at 200 j back in sinus tachycardia. she received 300 mg iv amiodarone x1. she then received asa 325 mg po x1, plavix 600 mg po x1, heparin gtt with bolus, integrillin gtt with bolus, and benadryl 25 mg iv x1. she underwent a cardiac catherization that showed a 90% left main stenosis, 60% proximal left circumflex, 80% rca, 90% lesion followed by a long complex cork screw like 70% lesion in the proximal rca. mid to distal rca has a haze 95-99% lesion. she was transferred to for evaluation of cabg. . in the ccu, patient is unable to answer ros due to intubation and sedation. she was noted to have bleeding from groin site. angiosheath was removed and pressure held with good effect. ng tube was noted to be draining ~250 ccs of dark, frothy, coffee-ground blood. gastric lavage was + after 750 ccs of irrigation. also noted to have upward deviated gaze and seizure-like activity (first shaking of the upper extremities, and then lower extremities, then tongue fasciculations). neurology urgently consulted, recommended iv dilantin load and iv ativan (received 6 mg iv total) with good control of seizure activity. a-line, r ij placed, and patient sent for non-contrast ct and cta to assess for acute ich prior to initiating arctic sun protocol. past medical history: 1. cardiac risk factors: +diabetes, +dyslipidemia, +hypertension 2. cardiac history: -cabg: none -percutaneous coronary interventions: ( at ): -pacing/icd: none 3. other past medical history: copd dementia paranoid schizophrenia urinary incontinence sleep apnea s/p aaa repair djd/oa (lumbar back pain) social history: resident of nursing home x 1 year. ambulates with walker and 1 assistant at baseline, unable to take stairs. baseline mental status is aox2 (knows name, and date). is only truly conversant with known family members. short term memory. -tobacco history: +60 ppy hx, quit smoking 10 years ago -etoh: none -illicit drugs: none family history: sister with breast cancer, no family history of early mi. physical exam: vs: t= 96.6 bp= 153/66 hr= 78 rr= 16 o2 sat= 99% on ac 480 x 16 fio2 100% peep 5 general: intubated, sedated heent: perrla. neck: supple with jvp of 6 cm. cardiac: pmi located in 5th intercostal space, midclavicular line. rr, normal s1, s2. ii/vi late peaking systolic murmur heard best at the base. no thrills, lifts. no s3 or s4. lungs: ctab, no crackles, wheezes or rhonchi. abdomen: soft, ntnd. no hsm or tenderness. abd aorta not enlarged by palpation. no abdominial bruits. extremities: no c/c/e. skin: mild resolving bruise over l shin. pulses: right: radials 2+ dp 2+ pt 2+ left: radials 2+ dp 2+ pt 2+ neuro: opens eyes spontaneously; does not make noises; withdraws and flexes to painful stimuli; pupils reactive to light; +corneal reflexes. babinskis downgoing bl. pertinent results: admission labs: 08:12pm blood wbc-17.5*# rbc-3.60* hgb-10.7* hct-31.8* mcv-89 mch-29.8 mchc-33.6 rdw-14.4 plt ct-205 08:12pm blood pt-15.3* ptt->150 inr(pt)-1.3* 08:12pm blood glucose-171* urean-26* creat-0.5 na-143 k-3.7 cl-109* hco3-21* angap-17 08:12pm blood ck(cpk)-204* 04:55am blood alt-24 ast-37 ld(ldh)-218 ck(cpk)-196 alkphos-55 totbili-0.8 08:12pm blood ck-mb-17* mb indx-8.3* ctropnt-0.49* 04:55am blood ck-mb-24* mb indx-12.2* ctropnt-0.32* 08:12pm blood calcium-8.3* phos-3.1 mg-1.6 04:55am blood triglyc-98 hdl-34 chol/hd-3.0 ldlcalc-48 ldlmeas-<50 ----------------- discharge labs: 05:35am blood wbc-18.0* rbc-3.79* hgb-11.3* hct-34.2* mcv-90 mch-29.8 mchc-33.1 rdw-14.8 plt ct-250 02:58am blood pt-14.0* ptt-21.4* inr(pt)-1.2* 05:35am blood glucose-169* urean-23* creat-0.4 na-139 k-4.1 cl-102 hco3-31 angap-10 05:35am blood calcium-8.4 phos-3.6 mg-1.9 ------------------ studies: . cxr 1:00am: in comparison with study of , there is now an endotracheal tube in place with its tip approximately 4.8 cm above the carina. nasogastric tube extends to the upper stomach, though the side hole is in the distal esophagus. right ij catheter extends to the mid-to-lower portion of the svc. obliquity of the patient makes it difficult to evaluate the cardiac size, though it appears to be within upper limits of normal. no definite vascular congestion or pleural effusion. probable mild atelectatic changes are seen at both bases. . cxr 7:30am: in comparison with the study of earlier in this date, there is little change. monitoring and support devices remain in place with the side hole of the nasogastric tube just proximal to the esophagogastric junction. mild atelectatic changes persist at the bases. . cta head : bilateral atherosclerotic disease involving the carotid bifurcations with possible ulceration versus small dissection at the origin of the left internal carotid as described above. fenestration at the anterior communicating artery with small aneurysmatic formation measuring approximately 2 mm in size. areas of low attenuation are identified at the subcortical and periventricular white matter, likely consistent with chronic microvascular ischemic changes. bilateral sinus disease. . tte : the left atrium is mildly dilated. the left ventricular cavity size is normal. there is mild regional left ventricular systolic dysfunction with basal to mid inferior and inferolateral hypokinesis. the aortic valve leaflets are mildly thickened (?#). there is no aortic valve stenosis. no aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. mild (1+) mitral regurgitation is seen. there is no pericardial effusion. there is an anterior space which most likely represents a fat pad. impression: poor technical quality due to patient's body habitus. mild focal lv systolic dysfunction. mild mitral regurgitation. . eeg : impression: this is an abnormal video eeg due to slowing and disorganization of the background rhythm suggestive of a mild to moderate encephalopathy. additionally, there are rare sharp transients phase reversing in the left mid to posterior temporal region consistent with possible increased cortical irritability and potential for pileptogenesis. this telemetry captured no pushbutton activations. there was no evidence of electrographic seizures in this recording. . eeg : impression: this is an abnormal noncontinuous extended routine eeg due to mild slowing and disorganization of the background rhythm and rare sharp transients in the left anterior sylvian to midtemporal region. these findings suggests a mild to moderate encephalopathy and possible left temporal cortical irritability. this study captured no pushbutton activations and did not contain any electrographic seizurs. . cxr : a single frontal radiograph demonstrates a nasogastric tube coursing towards the stomach but the tip is not seen. endotracheal tube appears to have been removed as that is not seen clearly on this radiograph. a right ij catheter terminates in the superior vena cava. there is a small right-sided pleural effusion. there is mild bibasilar atelectasis. heart and mediastinum are within normal limits. there is mild bibasilar atelectasis. brief hospital course: 77 yo f w/ dmii, htn, hld, and dementia transferred from osh s/p cardiac arrest, complicated by upper gi bleed and generalized seizure activity. . # coronaries: patient with multiple risk factors for cad including dmii, htn, hld. she was s/p cardiac arrest at nursing home and at osh ed, likely vf requiring aed shocks x2. the etiology of her vfs were most likely ischemia-related. ekg was concerning for antero-lateral nstemi given st depressions in v2-v6 and twi in i and avl. cardiac catherization at osh which shows severe 3vd including critical left main disease. patient received heparin, aspirin, plavix, and integrillin at osh prior to transfer. by the time she arrived to , her ekg has normalized, presumably the coronary blockage responsible for the ischemia has been lysed. patient was initiated on arctic sun protocol 9 hours after her second vf arrest at the osh ed. however, it had to be discontinued after 2 hours because she developed vf x 4 episodes requiring cardioversion. ekg showed no evidence of ongoing ischemia during these vf episodes, suggesting that the new vf episodes are due to reperfusion as opposed to ischemia which were likely responsible for her first two vf arrests. patient was started on lidocaine drip for these ventricular arrhythmia, which was discontinued the next day since she had no more vf episodes. patient was off aspirin and plavix for a few days because of active gib; and when gib slowed down, aspirin and plavix were restarted. . # gi bleed: patient was noted to have ugib on admission to ccu at . this was likely ugib in the setting of anticoagulation that she received at osh, including heparin, asa, plavix, and integrillin. patient takes asa at home, but no history of gi bleeds in the past, and no history of regular nsaid use. heparin, aspirin and plavix were initially held, and gi was following. egd was going to be performed on the night of admission, but given frequent vf episodes that night in the setting of receiving arctic sun protocol, gi decided to hold off on egd. patient was hemodynamically stable and received 4u prbc transfusions during her hospital stay. she received iv protonix drip, and gib slowed down significantly. she was started on po meds on , and tf was started on . she was doing well until , when she was noted to have gib again. at that time, plavix was discontinued, and gi was re-consulted who recommended conservative management. she passed away that night from respiratory failure. . # respiratory failure: patient was intubated at osh prior to transfer to . she was successfully extubated on . she was doing well from the respiratory stand point after extubation, but on , she started to experience respiratory distress, requiring frequent suctions. multiple family meetings were held during the day to discuss prognosis, and her code status was changed to dnr/dni. she passed away on from respiratory failure. . # pump: patient underwent tte on which showed mild regional left ventricular systolic dysfunction with basal to mid inferior and inferolateral hypokinesis. ef was estimated to be 45-50%. patient required no diuresis and had no heart failure symptoms during this hospital stay. . # rhythm: patient was s/p vf arrest w/ shocks x2 and amiodarone load at osh. rhythm strip from emts shows coarse vf. as mentioned above, she developed vf episodes x 4 after being initiated on arctic sun protocol with normalizing ekg. the initial vfs x 2 at osh were thought to be ischemia-related, but the vf episodes at ccu were thought to be re-perfusion-related. patient was initiated on lidocaine gtt after she developed vfs at ccu, which was discontinued the next day. patient had no more vf episodes during the rest of her hospital stay. . # anoxic brain injury: patient had multiple episodes of vf arrests, and was shocked multiple times. she was initiated on arctic sun protocol and tolerated it for only a couple of hours. besides the generalized seizure activities noted on the day of admission, she made a remarkable recovery from the mental status stand-point. she was following commands, and, per her daughter, was at her baseline soon after extubation. . # seizure: patient was noted to have generalized seizure activity upon arrival to ccu, but a head ct was negative for acute bleed. neurology was consulted and felt that the patient's seizure was likely in the setting of hypoxia s/p vf arrest. she underwent a 48 hour eeg without evidence of seizure activity and completed a 7 day course of phenytoin for acute phase prophylaxis. she did not experience any other witnesses seizures during this hospitalization. . # htn: patient grossly hypertensive to sbp 170's on admission. have been vasoconstrictive response to hypovolemia in setting of her gib. she was placed on a nitro gtt that was slowly weaned and she was eventually transitioned to po metoprolol 25mg tid and captopril 25mg tid with resulting sbp in the 120s to 130s. . # hld: patient's home atorvastatin was increased to 80mg qhs following her cardiac arrest. . # diabetes mellitus ii: patient was maintained on an insulin sliding scale throughout this hospitalizations. . # schizophrenia: patient's home risperdal was held in the acute arrest phase, but was restarted as soon as she was able to tolerate po medications. . # dementia: patient's home namenda was held in the acute arrest phase, but was restarted as soon as she was able to tolerate po medications. . # pvd: s/p aaa repair. patient's home atorvastatin was increased to 80mg qhs when she was able to tolerate po medications. . # code: patient was admitted with a code status of full, this was changed to dnr during her hospital stay with permission to defibrillate and intubate per her hcp. code status was then changed to dnr/dni on when she started to look worse clinically. she expired on from respiratory failure. medications on admission: metformin 850 mg po qdaily colace 200 mg po daily tramadol 50 mg po tid senna 8.6 mg 2 tabs po daily:prn risperdal 4 mg po qam risperdone 6 mg po daily (8 pm) lipitor 20 mg po qhs tylenol 650 mg po q4h:prn milk of magnesia biscolax 10 mg pr:prn ipratroprium/albuterol 0.5/3 mg 1 neb inh q4h:prn tramadol 50 mg po q6h:prn pain asa 325 mg po daily detrol 2 mg po daily namenda 10 mg po bid claritin 10 mg po daily discharge medications: expired discharge disposition: expired discharge diagnosis: cardiac arrest respiratory failure discharge condition: expired discharge instructions: expired followup instructions: expired procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours enteral infusion of concentrated nutritional substances other electric countershock of heart arterial catheterization diagnoses: subendocardial infarction, initial episode of care coronary atherosclerosis of native coronary artery unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled chronic airway obstruction, not elsewhere classified other convulsions other persistent mental disorders due to conditions classified elsewhere paroxysmal ventricular tachycardia other and unspecified hyperlipidemia acute respiratory failure cardiac arrest anoxic brain damage ventricular fibrillation anticoagulants causing adverse effects in therapeutic use hemorrhage of gastrointestinal tract, unspecified hemorrhage, unspecified paranoid type schizophrenia, unspecified 

Answer: The patient is high likely exposed to