Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins / ciprofloxacin attending: chief complaint: chest pain major surgical or invasive procedure: cardiac catheterization with des to rca intubation intra aortic balloon pump placement. history of present illness: hpi: 51 yo m with iddm, htn, transferred from with stemi. patient originally presented to with exertional chest pain nausea that started at work at 9 am. had 2-3 episodes of moderate chest pressure with numbness down the back of both arms, as well as, diaphoresis, nausea, and lightheadedness while at work. no shortness of breath, headache, visual changes, back pain, or abdominal pain. no history of prior episodes of chest pain. admission ekg at was negative for ischemic changes, however, he later developed ekg changes consistent with an inferior stemi. the pt was given lopressor 50 mg po, morphine 2 mg, heparin 5000 u x 1, plavix 600 mg po, aggrastast bolus 44cc/hour over half hour, then 11cc/hour iv, and aspirin 325 mg x 1. initial labs were significant for cpk 109 and troponin of .17 (positive). <br> the pt was med flighted to the cath lab where a left heart cath revealed 3vd with to of rca. access for iabp was obtatined when cath was started. while trying to enagage the rca, deploy the distal des and remove the guidewire, patient deveoped asymptomatic hypotension with vt. the guidewire and stent balloon were removed but he had continued vt and so was shocked. lidocaine was started and aibp was inflated. the rca then reoccluded. vessel was opened again via balloon - initially unable to place des at most distal location to recurrent vt progressiing to vf (requiring multiple shocks). then started on amiodarone 150 mg bolus followed by infusion; he was also intubated at that time for airway protection. remainder of his cath revealed: l main w/out sig disease; lad diffuse, prox 70 % lesion; l circ prox 80% lesion; om1 diffuse 70%. wiggle wire was used to deliver distal, overlapping stents to rca for a total of 3. he was started on lidocaine gtt, amiodarone gtt and dopamine gtt while in the cath lab and then was transferred to the ccu intubated and sedated. past medical history: 1. iddm 2. htn 3. hyperlipidemia social history: lives with his wife. at a paper mill. former smoker - smoked for 20 yrs, quit 23 yrs sgo. family history: mother - cad (mi), dm father - colon cancer physical exam: vs on transfer to ccu: t 97.8 bp 94/56 hr 79 rr 12 sat 100 ac fio2 100 tv 600 rr 12 peep 5 wt: 212 lbs gen: intubated heent: perrla resp: clear anteriorly cv: reg, systolic murmur (likely iabp) abd: + bs, non-tender ext: dp 1 + bilat, no edema neuro: sedated pertinent results: studies: cxr - - satisfactorily positioned et tube and og tube. bilateral basilar opacities most likely representing atelectases but consolidations cannot be excluded. probable bilateral small pleural effusions. . ekg: - 11:52 am at osh - nsr, hr 70s, st seg elevation in ii, iii, avf, v4-v6, st seg dep i, avl . - 3:33 pm - nsr, hr 80s, st seg elevations in ii, avf, ? qs in ii and avf, resolution of st seg elevation in v4-v6, st seg dep in i, avl .. tte: - ef 35-40% regional lv and rv systolic dysfunction c/w cad brief hospital course: a/p: 61 yo m with insulin dependent dm, htn, hyperlipidemia who presents with inferior stemi s/p ptca complicated by hypotension requiring balloon pump and da gtt as well as vt/vf resulting in amio and lidocaine gtt. . 1. cv: a. stemi: 3vd as per cath with to of rca which was felt to be the culprit lesion. he is now s/p 3 overlapping des to the rca. patient was monitored on telemetry and was followed with daily ecgs with no further events. he was discharged on aspirin, atorvastatin, metoprolol, lisinopril and clopidogrel. he did not have any additional episodes of chest pain while in-patient. his blood pressure was well controlled at time of discharge. patient may need further intervention to deal with other areas of significant blockage, however, no intervention will be done acutely. he may need stress-mibi as out-patient prior to further intervention (pci vs cabg). he will follow-up with his pcp . in one week and dr. (cardiologist) in one month. he will also see ct surgery in to discuss cabg. . b. chf: in the cath lab the patient had hypotension as a result of the ischemia as well as arrhythmia. this may reflect a rv infarct physiology given the proximal rca lesion. as per pcwp and ra pressure from cath lab, he was effectively volume resuscitated. tte on showed an ejection fraction of 35-40%, symmetric lvh, mild rv free wall hypokinesis. the current hypotension may therefore reflect some amount of myocardial stunning and the heart may recover function at a future date. iabp dced on . also weaned off dopamine and extubated on . in an effort to keep volume status even patient was given furosemide as needed. he was not discharged on furosemide. he will need a repeat echo in three months. . c. rhythm: the pt had episodes of vt and vt/vf in cath lab during the acute intervention. this may be secondary to reperfusion or due to involvement of conduction system. he is now s/p loading with both lidocaine and amiodarone. lidocaine was not continued after transfer to ccu; amiodarone was continued for 18 hours but was subsequently discontinued. patient was monitored on telemetry. no further episodes of vt/vf. he was not discharged on an anti-arrhythmic. . d. valve: no evidence of acute valvular insufficiency on physical exam or by echo. . 2. dm: based on the findings of digami ii, there is no evidence to support use of glucose/insulin gtt in the setting of acs to improve mortality or morbidity in diabetics. however the recent findings by dr. , and timi study group in the journal of american college of cardiology suggests a "u" shaped relationship between admission glucose and mortality in acs. the admission glucose of 271 therefore is not a favorable finding. patient was initially placed on an insulin gtt for better control of dm. he was then transitioned to nph and a regular insulin sliding scale. . 3. respiratory: the pt was intubated during the cardiac arrest in the cath lab for airway protection. given the discomfort of the invasive monitoring in the icu at this time (swan, arterial and femoral sheath, iabp), sedation and intubation was continued until the iabp was removed on . the patient was extubated without difficulty. medications on admission: medications at home: 1. lantus with sliding scale insulin 2. quinipril 20 mg qd 3. lipitor 10 mg qd . allergies: pcn - hives, shortness of breath, cipro - hives discharge medications: 1. aspirin 325 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po daily (daily). disp:*30 tablet, delayed release (e.c.)(s)* refills:*2* 2. clopidogrel 75 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 3. atorvastatin 80 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 4. lantus 100 unit/ml solution sig: 35 units subcutaneous qhs. disp:*qs qs* refills:*2* 5. lisinopril 10 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 6. toprol xl 50 mg tablet sustained release 24hr sig: three (3) tablet sustained release 24hr po once a day. disp:*90 tablet sustained release 24hr(s)* refills:*2* discharge disposition: home discharge diagnosis: primary diagnosis stemi * secondary diagnoses dm - insulin dependent htn hyperlipidemia discharge condition: good, tolerating medications and ambulating without difficulty discharge instructions: you were diagnosed with a myocardial infarction or heart attack, which was treated with stents that were used to open an artery in your heart. as a result you were started on several new medications. it is very important that you take all of your medications as prescribed. one of them called clopidogrel (or plavix) and is used to keep the stent open. you should not stop your medications unless you discuss this with your cardiologist. * please call your doctor or return to the emergency room if you have chest pain, shortness of breath, you cannot eat drink or take your medications, or you develop any other symptoms that are concerning to you. followup instructions: please follow-up with your pcp . in one week. he should check your blood pressure to make sure it is adequately controlled. please continue to follow-up your blood sugars at and discuss your insulin regimen with your pcp. should also provide you with referrals for your appointments with drs. and . * please follow-up in the cardiology clinic with , m.d. phone: date/time: 3:20. you will need a referral for this appointment from your pcp. * please follow-up with dr. in the cardiac surgery department on at 1:30 pm to discuss having open heart surgery to correct your blocked arteries. procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours combined right and left heart cardiac catheterization coronary arteriography using two catheters insertion of endotracheal tube insertion of temporary transvenous pacemaker system atrial cardioversion implant of pulsation balloon nonoperative removal of heart assist system insertion of drug-eluting coronary artery stent(s) excision of lingual thyroid percutaneous transluminal coronary angioplasty [ptca] procedure on single vessel insertion of three vascular stents diagnoses: coronary atherosclerosis of native coronary artery unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled cardiac complications, not elsewhere classified paroxysmal ventricular tachycardia other and unspecified hyperlipidemia long-term (current) use of insulin acute myocardial infarction of other inferior wall, initial episode of care ventricular fibrillation other second degree atrioventricular block 

Answer: The patient is high likely exposed to