Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: zoloft / bupropion attending: chief complaint: anteroseptal st elevation myocardial infarction major surgical or invasive procedure: central line: right internal jugular vein history of present illness: 86m anteroseptal stemi x 13hrs without intervention at nursing home, found to have unintervenable 3vd, now cardiogenic shock (new ef 15-20%, ci 1.7, pcwp17). usoh at nursing home until last night when began to have substernal cp, no interventions performed at the time. morning of admission, presented to cardiologist () office, where ekg revealed 3-4mm st elevations in anteroseptal leads, echo 20-25%. sent to ed (chest pain free, 12 hrs after onset of cp), ck 1043, mb 123, tni 45.17. ekg revealed q in v1-v3, deep twi in v2-v3, v4-5. in cath lab, 3vd, no interventions performed: lmca 60% lad 90% prox, 100% mid w/ r->l collat lcx 60% mid, 70% inf branch large ramus rca 60% prox, 90% mid, 80% distal, 80% origin pda past medical history: seizure disorder cva circa '(residual l sided weakness) paroxysmal atrial fibrillation (not anticoagulated, but on amio) remote h/o gi bleed cad - stable angina w/ "small mi" in past, medically mgmt aaa (no imaging available here) etoh abuse anemia social history: lives in nursing home family history: non-contributory physical exam: t98, hr99, bp115/57, rr17, o2sat 100% on 3liters gen: nad, lying in bed heent: mmm, op clear, adentulous heart: irreg, s1s2, no g/m/r, no jvd, no carotid bruits lungs: scattered crackles abdomen: benign ext: no c/c/e, no femoral bruits, pulses dopplerable pertinent results: 05:29pm blood ck(cpk)-858* 05:49am blood ck(cpk)-591* 04:00am blood ck(cpk)-261* 03:46am blood ck(cpk)-94 . 05:29pm blood glucose-99 urean-8 creat-0.6 na-137 k-4.3 cl-103 hco3-25 angap-13 04:15am blood glucose-189* urean-11 creat-0.7 na-144 k-3.4 cl-107 hco3-30 angap-10 . 05:29pm blood pt-13.5* ptt-29.1 inr(pt)-1.2 11:30am blood pt-14.2* ptt-51.9* inr(pt)-1.3 05:29pm blood wbc-8.3 rbc-3.70* hgb-11.5* hct-33.8* mcv-91 mch-31.2 mchc-34.2 rdw-14.4 plt ct-336 04:15am blood wbc-10.3 rbc-3.92* hgb-11.8* hct-35.4* mcv-90 mch-30.1 mchc-33.3 rdw-14.6 plt ct-436 brief hospital course: 1. anterior st elevation myocardial infarction patient presented to approximately 16 hours after the intial onset of substernal chest pain. however given the patient's age, the extent of coronary artery disease, and late presentation, the patient was not deemed a suitable candidate for cardiac catherization and was tranferred to the ccu for medical management. at the time of tranfer the patient's pap was 40/17, pcwp was 17, ci was 1.7 and pa sat was 49%. enzymes from the osh were notable for ck 1043, mb 123, mb 11.8 and trop i 45.2. ekg from the osh showed t wave inversions and q waves in v1-v3. an echo done at the , showed an ef of 15-20%, and depressed left ventricular systolic function. (an echo a year prior had been completely normal). throughout his course patient was medically managed for his coronary artery disease with aspirin, lipitor and plavix. cardiac enzymes trended downward and with gentle diuresis patient's cardiac index improved to 3.15. . 2. aflutter the patient's initial course was complicated on the day of presentation by atrial flutter, with hr>150. early in the hospital course the patient was asymptomatic during these episodes and would revert back into normal sinus rhythm. the patient was medically managed with amiodarone. on the patient reverted back into atrial flutter while going to the bathroom. thus, despite being on amiodarone, it became apparent that with the slightest exertion the patient was unstable. the decision was made cardiac ablation. this intervention was a temporary one because the patient reverted back into aflutter approximately 2-3 hours following the procedure.. the final decision has been to give the patient an amiodarone load over the course of one month, 400 mg / day. pt was also started on coumadin. however this was later held due to elevated inrs. . 3. hypotension on , the patient became acutely hypotensive, sbp's 70s-80s. an echo revealed no cardiac effusion (s/p day 1 cardiac ablation). blood cultures were sent which later came back negative. an ekg was done which showed no significant changes. the patient responded to 2 liters of fluids. sbp improved to the 120s. pressors were never administered. the patient returned to the ccu for overnight monitoring. . given the patient's poor p.o. intake throughout his course, this hypotensive episode may have resulted from the patient being intravascularly depleted. the patient was started on maintenance fluids. . 5. nutrition the patient has had poor p.o. intake throughout hospitalization. the patient has a history of aspiration pneumonia and esophageal strictures requiring dilatation. to better assess the patient's aspiration risk, a speech and swallow was done. the patient did not show evidence of aspiration during the study. the patient was noted to have an extremely discoordinated oral phase. a gi consult was then obtained to rule out the possibility of a stricture. a barium study was done. due to the limitations of the study, gi recommended an outpt eval in 2-3weeks. . in order to improve the patient's nutritional status, the patient has been receiving tube feeds through a nasogastric tube. . 6.hypoxia .on , the patient became acutely hypoxic, desatting to 89% on 4l. a cxr showed bilateral effusions, left lower lobe collapse, consolidation, and mild background of chf. the patient was readmitted to the ccu for diuresis. the patient was started on on ceftriaxone for presumed pneumonia. the patient was already on flagyll for c.diff. . 7. infection . on the day of admission the patient was continued on flagyll due to a diagnosis of aspiration pneumonia from the nursing home. stool cultures later came back positiove for c. diff. the decision was made to continue the antibiotic for a 14 day course. . ua . on , a family meeting was called to address the goals of care for mr. . his children, dr. , and dr. with rn were present. as the patient's cardiac and pulmonary status continued a tenuous course, a decision was made to ask the family whether they agreed that a swan ganz pulmonary artery catheter should be placed. the family unanimously agreed that the patient would not have wanted continued invasive monitoring, and the request was made to make mr. dnr/dni status, and then finally, cmo status. he was made comfortable with a morphine iv drip after palliative care was consulted. the patient expired within the next 36 hours secondary to respiratory arrest/hypoxia. his family was notified, and elected not to undergo autopsy. medications on admission: advair amiodarone 100mg asa 81 dilantin 100mg , qod, alternate w/ 150mg florinef 0.4mg kcl mg oxide 500mg daily mvi paxil 40 ntg patch 0.1mg neutrophos compazine flagyll 400 tid colace 100mg discharge medications: patient was not discharged. he passed away in the ccu. discharge disposition: expired discharge diagnosis: n/a discharge condition: n/a discharge instructions: n/a followup instructions: n/a md, procedure: combined right and left heart cardiac catheterization coronary arteriography using two catheters enteral infusion of concentrated nutritional substances catheter based invasive electrophysiologic testing excision or destruction of other lesion or tissue of heart, endovascular approach transfusion of packed cells diagnoses: pneumonia, organism unspecified subendocardial infarction, initial episode of care coronary atherosclerosis of native coronary artery pure hypercholesterolemia urinary tract infection, site not specified congestive heart failure, unspecified other convulsions atrial flutter hypotension, unspecified pneumonitis due to inhalation of food or vomitus cardiogenic shock intestinal infection due to clostridium difficile encounter for palliative care abdominal aneurysm without mention of rupture stricture and stenosis of esophagus 

Answer: The patient is high likely exposed to