Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: acetylcysteine attending: chief complaint: tylenol overdose major surgical or invasive procedure: picc placement and removal history of present illness: 60f history of frontotemporal dementia, complains of ingestion of ~ 70 tablets of extra strength tylenol on saturday 2 days ago at 1 pm, denies any other ingestions. reported si with superficial cutting of both wrists. she is a markedly poor historian, and is unable to provide much history. her husband states that she is significantly more confused than usual. she went to outside hospital (), received one dose of n-acetylcysteine. developed mild facial flushing for which she received benedryl. tylenol level was negative, but ast was 16,000, inr was 1.8. . per husband, patient was in her baseline (child like, longterm memory intact, short term impaired). her husband brought the tylenol 3 days prior and put it in the medicine cabin. she found it on saturday and took it. . in the ed initial vital signs were 99.5 86 122/68 18 100% 2l nc. exam was notable for icteric sclera, soft diffusely tender belly, mumbles. labs were notable for elevated ast, alt, inr was elevated to 2.7, lipase to 105, large blood in the ua. patient underwent ekg which showed nsr. patient was given n-acetylcysteine. patient was seen by toxicology: n-acetylcysteine after 1st loading dose 50 mg/kg over 4 hrs, then 100 mg/kg over 16 hrs. benadryl and/or ranitidine as needed for reaction to nac, admit to micu. patient was admitted for tylenol od. vital signs prior to transfer 99.5 90 94/44 23 97% ra. . on floor, she recognizes her husband, follows command. . review of systems: (+) per hpi past medical history: ftd hld depression gerd social history: - tobacco: none - alcohol: occassional - illicits: none family history: no family history of dementia or depression. physical exam: admission physical vitals: t:100 bp:114/62 p:90 r: 18 o2:95% general: mild distress, a&ox1, flushed red in the face heent: slight sclera icteric, dmm, oropharynx clear, poor dentition neck: supple, jvp not elevated, no lad lungs: clear to auscultation bilaterally, no wheezes, rales, ronchi cv: tachy, normal s1 + s2, no murmurs, rubs, gallops abdomen: soft, diffusely tender, mildly-distended, bowel sounds present, no rebound tenderness or guarding, no organomegaly gu: foley ext: warm, well perfused, 2+ pulses, no clubbing, cyanosis or edema discharge pe: vs: 99.1 160/90 66 18 96ra gen: calm, cooperative, aao x3 heent: anicteric sclera card: rrr, s1, s2 heard, no murmurs, rubs or gallops. pulm: lungs ctab, no crackles or wheezes appreciated abdomen: soft, nontender, nondistended, obese, normoactive bowel sounds, no hsm detected extremities: warm, well perfused, 2+ dp pulses neuro: no asterixis noted pertinent results: at admission: 08:03pm blood wbc-6.6 rbc-4.20 hgb-13.5 hct-38.5 mcv-92 mch-32.1* mchc-35.0 rdw-13.2 plt ct-49* 08:03pm blood neuts-91.4* lymphs-6.4* monos-1.3* eos-0.7 baso-0.2 08:03pm blood pt-28.2* ptt-39.1* inr(pt)-2.7* 08:03pm blood glucose-120* urean-27* creat-1.1 na-140 k-4.0 cl-111* hco3-17* angap-16 08:03pm blood glucose-120* urean-27* creat-1.1 na-140 k-4.0 cl-111* hco3-17* angap-16 08:03pm blood alt-* ast-* alkphos-90 totbili-1.0 01:00am blood alt-* ast-* ld(ldh)-* alkphos-90 totbili-1.0 08:03pm blood lipase-105* 01:00am blood albumin-3.6 calcium-9.2 phos-1.5* mg-2.3 iron-169* 08:03pm blood ammonia-90* 12:56am blood hbsag-negative hbsab-positive hbcab-negative igm hav-negative 01:00am blood afp-2.0 02:55pm blood type-art po2-74* pco2-36 ph-7.37 caltco2-22 base xs--3 intubat-not intuba imaging: ct head findings: no hemorrhage, large territorial infarction, edema, mass, or shift of normally midline structures is present. the ventricles and sulci are prominent consistent with cortical atrophy. the basal cisterns are widely patent. the visualized paranasal sinuses and mastoid air cells are well aerated. impression: no acute intracranial process; bilateral frontal more than temporal lobar atrophy, consistent with the given history. comment: findings were discussed with dr. via phone at 11 p.m. on . the study and the report were reviewed by the staff radiologist. cxr findings: upright ap and lateral views of the chest show new small bilateral pleural effusions. otherwise, the exam is unchanged. the cardiamediastinal and pulmonary structures are unremarkable. no pneumothorax. impression: new small bilateral pleural effusions. u/s indication: tylenol overdose, evaluate liver with doppler. liver and gallbladder ultrasound. comparison: none. findings: the liver appears slightly coarse in echotexture. within the left lobe of the liver is a septated cyst measuring 2 x 1.4 x 1.6 cm. in the right lobe is a simple-appearing cyst measuring 1.5 x 1 x 1 cm. no solid lesions are identified within the liver. there is normal hepatopetal flow within the main portal vein. hepatic vasculature including the portal veins, and the hepatic veins are patent. the hepatic arteries are patent. direction of flow is normal. the ivc is patent. the splenic vein is patent and the superior mesenteric vein is patent. there is trace abdominal ascites. the spleen is normal in size measuring 10 cm. the right and left kidneys are normal in echotexture measuring 12.2 and 12.1 cm, left and right respectively. there is marked gallbladder wall thickening, particularly along the contour of the liver with the thickness of the gallbladder wall measuring up to 1.1 cm. however, there is no son elicited and no stones within the gallbladder. impression: 1. patent hepatic vasculature. 2. marked gallbladder wall thickening likely related to hepatic dysfunction in the setting of tylenol overdose. 3. trace abdominal ascites. 4. liver cysts. tte the left atrium is normal in size. no atrial septal defect is seen by 2d or color doppler. left ventricular wall thickness, cavity size and regional/global systolic function are normal (lvef >55%). there is no ventricular septal defect. right ventricular chamber size and free wall motion are normal. the diameters of aorta at the sinus, ascending and arch levels are normal. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. no aortic regurgitation is seen. the mitral valve appears structurally normal with trivial mitral regurgitation. there is no mitral valve prolapse. the estimated pulmonary artery systolic pressure is normal. there is no pericardial effusion. ecg sinus rhythm. normal tracing. no previous tracing available for comparison. tracing #1 micro: urine urine culture-pending inpatient blood culture blood culture, routine-pending inpatient blood culture blood culture, routine-pending inpatient blood (toxo) toxoplasma igg antibody-final; toxoplasma igm antibody-final inpatient blood (ebv) - virus vca-igg ab-pending; - virus ebna igg ab-pending; - virus vca-igm ab-pending inpatient blood (cmv ab) cmv igg antibody-final; cmv igm antibody-final inpatient serology/blood varicella-zoster igg serology-final inpatient serology/blood rubella igg/igm antibody-final inpatient serology/blood rapid plasma reagin test-final inpatient mrsa screen mrsa screen-pending inpatient urine urine culture-final emergency blood culture blood culture, routine-preliminary {staphylococcus, coagulase negative}; anaerobic bottle gram stain-final emergency blood culture blood culture, routine-pending emergency discharge: 05:58am blood wbc-7.0 rbc-3.20* hgb-10.6* hct-30.0* mcv-94 mch-33.1* mchc-35.4* rdw-13.8 plt ct-254 01:00am blood neuts-91.8* lymphs-6.8* monos-0.8* eos-0.4 baso-0.2 05:58am blood pt-11.8 ptt-24.1 inr(pt)-1.0 05:58am blood glucose-90 urean-9 creat-1.0 na-143 k-3.5 cl-113* hco3-20* angap-14 05:58am blood alt-934* ast-135* alkphos-93 05:58am blood calcium-9.1 phos-2.7 mg-2.0 12:56am blood hbsag-negative hbsab-positive hbcab-negative igm hav-negative 12:56am blood ama-negative smooth-negative brief hospital course: 60f with frontotemporal dementia admitted after tylenol overdose from suicide attempt. complains of ingestion of 70 tablets of extra strength tylenol on saturday at 1 pm, now with ams, worsening lfts, concerning for fulminent liver failure. . . #tylenol overdose/acute liver failure: patient admitted to ingesting 70 tablets of extra strength tylenol. she was admitted to the icu with evidence of hepatic necrosis and synthetic liver dysfunction with encephalopathy and coagulopathy but without hyperbilirubinemia. she was treated with nac protocol with improvement in mental status and coagulopathy. to rule out other causes of acute liver failure a ruq us was obtained which showed normal hepatic vasculature and an afp was wnl. serologic workup for viral hepatitis and autoimmune liver diseases were negative. her antipsychotic and anxiolytic medications were held in the setting of alf but were restarted at low doses when liver chemistries, inr, and mental status improved. her chemistries were not normal on discharge, but had been downtrending for a number of days and pcp was to recheck at follow up visit. # coag negative staph bacteremia: this was felt to be a contaminant as it grew from only bottles and patient did not have clinical evidence of infection. she was started on antibiotics during transplant evaluation but this was discontinued when liver function normalized. #frontotemporal dementia: the patient's home medications were held and then restarted at lower dose (including seroquel) when liver function normalized. #+ua: patient was initially started on ctx but this was discontinued when ucx grew mixed flora and felt not to have true uti. #depression: given severe depression with suicide attempt the patient was kept on suicide precautions. she was evaluated by inpatient psychiatry who felt that patient would benefit from inpatient psychiatry admission. given acute liver failure, home medications were held and were not restarted when liver function normalized as these would be managed by the patient's inpatient psychiatry facility to which the patient was discharged. transitional issues: # htn: while hospitalized on the medical floor, the patient had sbps 150s-170s. before hospitalization, the patient was not on any anti-hypertensives medications. she was started on metoprolol tartrate 25 mg with fair control of bps and discharged on metoprolol succinate 50mg daily. please follow her blood pressures and discontinue them if not necessary. it is possible some of her hypertension is related to anxiety and taking less than her home dose of ativan. #liver injury: the patient should have lfts checked weekly until normal. medications on admission: ativan 1mg five times a day omeprazole 40mg citalopram 40mg qd premarin 0.3 qod simvastatin 40mg qd gabapentin 800mg tid rivastigmine 3mg tid seroquel 100mg tid discharge medications: 1. quetiapine 25 mg tablet sig: one (1) tablet po tid (3 times a day). 2. omeprazole 40 mg capsule, delayed release(e.c.) sig: one (1) capsule, delayed release(e.c.) po twice a day. 3. lorazepam 1 mg tablet sig: one (1) tablet po at bedtime as needed for insomnia. 4. metoprolol succinate 50 mg tablet extended release 24 hr sig: one (1) tablet extended release 24 hr po once a day. 5. outpatient lab work please get liver function test including: ast, alt, alkaline phosphatase, total bilirubin, albumin, and inr weekly discharge disposition: extended care discharge diagnosis: primary diagnosis: tylenol toxicity depression secondary diagnosis: frontotemporal dementia discharge condition: mental status: clear and coherent. level of consciousness: alert and interactive. activity status: ambulatory - independent. discharge instructions: dear ms. , it was a pleasure taking care of you while you were hospitalized at . you were admitted to the hospital with tylenol toxicity. you were initially admitted to the intensive care unit and given a medication that helps treat the effects of tylenol toxicity. your liver function tests were continually improving and will continue to be followed as an outpatient. our transplant surgery evaluated you and felt that you did not need to have a liver transplant because your liver was improving on its own. because of your improvement, you were transferred out of the intensive care unit and came to the medicine floor. while on the medicine floor, your mental status continued to improve and your liver function also started to get better. we also noted that your blood pressures were a little high while you were here and we started you on a blood pressure lowering medicine. we also started giving you some ativan at night before bed to help you sleep. you will be transferred to a psychiatric rehabilitation facility where you will continue to receive care. the psychiatry team there will assess you and restart the rest of your medications when it is safe to do so depending on your liver enzymes. the following changes were made to your medications: 1) start metoprolol succinate 50mg daily 2) decrease seroquel to 25mg three times daily 3) decrease ativan to 1mg at night as needed for sleep 4) stop citalopram 5) stop premarin 6) stop simvastatin 7) stop gabapentin 8) stop rivastigmine followup instructions: please make sure patient follows up with her outpatient dr. . procedure: central venous catheter placement with guidance diagnoses: acidosis thrombocytopenia, unspecified esophageal reflux unspecified essential hypertension acute and subacute necrosis of liver other persistent mental disorders due to conditions classified elsewhere other and unspecified hyperlipidemia major depressive affective disorder, single episode, unspecified diarrhea encephalopathy, unspecified poisoning by aromatic analgesics, not elsewhere classified suicide and self-inflicted poisoning by analgesics, antipyretics, and antirheumatics home accidents generalized anxiety disorder 

Answer: The patient is high likely exposed to