Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: chest pressure/pain major surgical or invasive procedure: cardiac catheterization history of present illness: ms. is 69 yo female with numerous cardiac risk factors (htn, hld, smoking, +fhx) as well as afib and sss s/p pacemaker who presented to ed this am with chest pressure. symptoms started around 4am, when she woke up with left-sided substernal chest pressure/tightness, non-radiating, in severity. there was no associated shortness of breath, nausea, vomiting or diaphoresis. when the pain did not go away, she called the ambulance. in the ambulance she received ntg slx3, which made the pain resolve. in the ed, she was hypertensive to >200 and started on ntg gtt for chest pain and htn. while on the ntg gtt, she reported l arm discomfort/pressure and midsternal burning, which resolved with uptitration of the drip. initial ekg showed t wave changes in avl, but repeat ekg later in am showed twi in ii,iii,avf, and v3-v6. labs significant for: trop t <0.01, 0.35, 0.46 ; ck 59, 84, 87; mb 7, 7; mbi 8,8. d-dimer elevated to 0.61 so pt had v/q scan which found low probability of pe. patient given asa 325mg, lopressor, morphine, plavix loaded. prior to transfer to for cath, she developed nausea which was treated with zofran. . on arrival to ccu, patient is hemodynamically stable, hypertensive to 160/100 on ntg gtt. she complains of persistent nausea but denies chest pain, arm/jaw pain, shortness of breath, or diaphoresis. ekg unchanged from prior. labs show trop t 0.39, ck 85, mb 7. . patient denies recent h/o anginal symptoms: no recent chest pain, dyspnea on exertion, etc. she did have a similar episode of chest pressure 2-3 years ago, for which she was worked up for pe (cta negative). she notes chronically decreased exercise tolerance since getting her pacemaker 4 years ago. per her report, she had a nuclear stress test 3 months ago for health maintenence purposes, which was completely normal. . on review of systems, s/he denies any prior history of stroke, tia, deep venous thrombosis, pulmonary embolism, bleeding at the time of surgery, myalgias, joint pains, cough, hemoptysis, black stools or red stools. s/he denies recent fevers, chills or rigors. s/he denies exertional buttock or calf pain. all of the other review of systems were negative. past medical history: -paroxysmal afib (on flecainide, off coumadin) -sss with pacemaker -htn -hld -hypothyroidism -raynaud's syndrome social history: pt is retired lab worker. divorced, lives alone at home. former smoker (1 pack/week, quit 25 years ago). drinks ~1 bottle of wine per week. denies illicits. family history: mother died of cad (age 58). aunt with stroke. no known fhx htn, hld, arrythmias, cardiomyopathies, sudden cardiac death. physical exam: admission physical exam: general: pleasant f who appears uncomfortable nausea, aaox3. mood, affect appropriate. heent: ncat. sclera anicteric. perrl, eomi. conjunctiva were pink, no pallor or cyanosis of the oral mucosa. no xanthalesma. neck: supple with jvd of 3 cm. cardiac: pmi located in 5th intercostal space, midclavicular line. rr, normal s1, s2. no m/r/g. no thrills, lifts. no s3 or s4. lungs: no chest wall deformities, scoliosis or kyphosis. resp were unlabored, no accessory muscle use. ctab, no crackles, wheezes or rhonchi. abdomen: soft, ntnd. no hsm or tenderness. abd aorta not enlarged by palpation. no abdominial bruits. extremities: no c/c/e. no femoral bruits. skin: no stasis dermatitis, ulcers, scars, or xanthomas. neuro: aaox3, cnii-xii intact, 5/5 strength biceps, triceps, wrist, knee/hip flexors/extensors, 2+ reflexes biceps, brachioradialis, patellar, ankle. pulses: right: carotid 2+ femoral 2+ popliteal 2+ dp 2+ pt 2+ left: carotid 2+ femoral 2+ popliteal 2+ dp 2+ pt 2+ . discharge physical exam: unchanged from admission exam pertinent results: admission labs: wbc-6.5 rbc-3.55* hgb-11.7* hct-34.1* mcv-96 mch-32.8* mchc-34.2 rdw-12.1 plt ct-149* pt-9.9 ptt-69.4* inr(pt)-0.9 glucose-133* urean-18 creat-0.8 na-141 k-4.0 cl-106 hco3-26 angap-13 calcium-8.9 phos-3.3 mg-2.2 alt-37 ast-41* . cardiac enzymes: 12:00 am: ck (cpk) 85, mb 7, trop t 0.39* 06:07 am: ck (cpk) 77, mb 6, trop t 0.21* . echo (): the left atrium is mildly dilated. left ventricular wall thickness, cavity size and regional/global systolic function are normal (lvef >55%). right ventricular chamber size and free wall motion are normal. the ascending aorta is mildly dilated. the aortic valve leaflets (3) appear structurally normal with good leaflet excursion and no aortic stenosis or aortic regurgitation. the mitral valve leaflets are structurally normal. there is no mitral valve prolapse. trivial mitral regurgitation is seen. the estimated pulmonary artery systolic pressure is normal. there is no pericardial effusion. impression: normal global and regional biventricular systolic function. no clinically-significant valvular disease seen. . chest x-ray (): heart size is moderately enlarged. mediastinum is unremarkable. lungs are essentially clear with no pleural effusion and pneumothorax. there is no evidence of pulmonary edema. left-sided pacemaker is placed with two leads, one of them terminating most likely in the right atrium and the other one in the right ventricle. the right ventricle lead makes a loop most likely within the posterolateral aspect of the right atrium. no pneumothorax. brief hospital course: # chest pain: pt with multiple cardiac risk factors including htn, hld, cigarettes and +fhx presenting with new-onset substernal chest pressure. symptoms improved with sl ntg and resolved with initiation of ntg gtt + morphine bolus. troponins peaked at 0.46, ck 87, mb 7. initial ekg without changes at osh, but repeat ekg showed twi in ii,iii,avf and v3-v6. per osh records she was hypertensive to sbp>200 in the ed. patient's timi score was 5, putting her at 12% risk of death/mi at 2 weeks, and 26% risk of death/mi/urgent revascularization at 2 weeks. she was plavix loaded and started on heparin gtt and ntg gtt at osh. prior to cath, she was also treated with home metoprolol tartrate 100mg po bid (goal hr 60-70), atorvastatin 80mg po daily, asa 325mg po daily, and plavix 75mg po daily, and she was weaned off ntg gtt. she underwent cardiac cath on the morning of , which showed mild cad, lvedp of 22, and anatomic anomoly (bronchial arteries take off from rca with av malformation). no interventions were performed. patient did well after cath, with no recurrence of chest pain or nausea. it was felt that given that she had had sbp>200 at osh, her chest pain was most likely hypertensive emergency. therefore, on discharge she was started on lisinopril in addition to her home metoprolol for better control of blood pressure. she will also continue her home dose of atorvastatin 40mg daily and asa 325mg daily. patient agreed to purchase a bp cuff and monitor her blood pressure regularly at home. . #.nausea: patient c/o persistent nausea starting 2-3 hours prior to arrival at ccu. no evolving ekg changes or increasing enzymes. nausea most likely morphine and ntg. resolved with zofran + ativan, and did not recur once ntg discontinued. . #.afib: per patient, she is no longer on coumadin as her cardiologist found that she only has paroxysmal afib. her home flecainide was held in the setting of concern for nstemi; home metoprolol and asa were continued. once nstemi had been ruled out via cath, her home flecainide was restarted. heart rate well-controlled throughout hospitalization. . #.sick sinus syndrome: patient has pacemaker, and is sinus paced on ekg. pacer interrogation was normal. . #.htn: patient hypertensive to 160 on ntg gtt and home metoprolol on arrival. given that her chest pain was most likely hypertensive emergency ( problem #1), she was discharged on lisinopril in addition to her home metoprolol 100mg . . #.hld: patient on lipitor 20mg at home. she is discharged on atorvastatin 40mg daily. . #.hypothyroidism: continued home levothyroxine. . transition of care: 1. needs chem 10 checked in 1 week because started lisinopril 2. please note rca avm found on cardiac cath. medications on admission: -asa 325mg po daily -simvastatin 20mg po daily -metoprolol tartrate 100mg po bid -flecainide 100mg po bid -levothyroxine 100mcg po daily -fish oil -vitamin c -calcium+d discharge medications: 1. aspirin 325 mg tablet sig: one (1) tablet po daily (daily). 2. metoprolol tartrate 100 mg tablet sig: one (1) tablet po twice a day. 3. levothyroxine 100 mcg tablet sig: one (1) tablet po daily (daily). 4. atorvastatin 20 mg tablet sig: one (1) tablet po daily (daily). 5. lisinopril 5 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 6. flecainide 100 mg tablet sig: one (1) tablet po twice a day. 7. outpatient lab work please check a1c and potassium on tuesday at 2:30p at dr. office discharge disposition: home with service facility: vna discharge diagnosis: chest pain hypertensive urgency hyperlipidemia discharge condition: mental status: clear and coherent. level of consciousness: alert and interactive. activity status: ambulatory - independent. discharge instructions: you had chest pain that did not result in a heart attack. we are not sure why you had chest pain but it might be because of high blood pressure. an echocardiogram showed normal heart function and a cardiac catheterization did not show any acute blockages. we have started a new medicine to lower your blood pressure further which is called lisinopril. this medicine can sometimes raise your blood potassium level so we would like you to get your potassium checked at dr. office next week. a prescription was written for this, please bring it to your appt. . followup instructions: name: , address: phone: fax date/time: please call the office on monday for an appt . name: , md specialty: internal medicine when: tuesday at 2:30p address: , , phone: md, procedure: coronary arteriography using two catheters left heart cardiac catheterization diagnoses: coronary atherosclerosis of native coronary artery unspecified essential hypertension unspecified acquired hypothyroidism atrial fibrillation other and unspecified hyperlipidemia family history of ischemic heart disease cardiac pacemaker in situ sinoatrial node dysfunction raynaud's syndrome 

Answer: The patient is high likely exposed to