Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins attending: chief complaint: left parietal bleed major surgical or invasive procedure: cerebral angiogram history of present illness: 49 yf p/w severe ha and right hemiparesis to osh, found to have large high-parietal bleed and avm on ct head. pt had intact mental status, but was intubated for airway protection because of repeated emesis. pt was then trasnferred to for further care. past medical history: depression and gerd social history: married with 3 children family history: unknown physical exam: bp:131/80 hr:101 r:23 o2sats: 98%on 100%ett gen: wd/wn, comfortable, nad. heent: pupils: errl eoms neck: supple. lungs: cta bilaterally. cardiac: rrr. s1/s2. abd: soft, nt, bs+ extrem: warm and well-perfused. neuro: intubated, mildly sedated, limited cooperation with exam, follows commands motor: normal strength on left side, dense hemiparesis on right side multiple beats of clonus bilat pertinent results: 05:55am blood wbc-6.4 rbc-3.81* hgb-12.3 hct-35.1* mcv-92 mch-32.4* mchc-35.2* rdw-14.2 plt ct-470* 05:55am blood plt ct-470* 05:55am blood glucose-83 urean-15 creat-0.5 na-140 k-3.9 cl-105 hco3-24 angap-15 07:00am blood estgfr-using this 03:06am blood alt-15 ast-19 alkphos-52 totbili-0.2 05:55am blood phenyto-2.3* 04:21pm blood type-art mri head findings: as noted on the head ct of , there is a large posterior left frontal lobe intraparenchymal hemorrhage with minimal adjacent vasogenic edema. there are multiple areas of low signal superficial to this region, in the left frontoparietal region consistent with flow voids due to an avm. as described previously on the cta, this appears to be predominantly supplied from the ipsilateral anterior middle cerebral artery branches and drains into the superior sagittal sinus. there is no evidence of herniation. there is mild mass effect upon the ipsilateral lateral ventricle. the remainder of the brain parenchyma is normal. there is no slow diffusion to indicate an acute infarct. impression: known left frontoparietal arteriovenous malformation with an the adjacent large intraparenchymal hematoma. mra: technique: 3d time-of-flight mra of the circle of . findings: the circle of shows no evidence of aneurysms or intracranial stenoses. the previously noted arteriovenous malformation of the left frontoparietal region is not identified completely as the mra does not image high enough. the superior division, as well as the m2 segment of the left middle cerebral artery, are enlarged as there is increased flow through these vessels to supply the arteriovenous malformation. impression: known left frontoparietal arteriovenous malformation with enlargement of the left m2 and superior segments of the middle cerebral artery due to supply of the arteriovenous malformation. angiogram: findings: left internal carotid artery: the cervical bifurcation did not demonstrate any significant atherosclerotic disease. the selective left internal carotid artery injection demonstrated a 4 cm left parietal arterial venous malformation with rapid shunting through the superior sagittal sinus. this avm was supplied by both the aca and mca distal branches. no intranidal or feeding pedical aneurysm was seen. sluggish avm drainage through a dilated cortical vein was noted, possibly due to the hematoma induced mass effects. no other vascular lesions were identified. left external carotid artery: the left external carotid artery injection demonstrated brisk filling of the distal branches with normal arterial runoff. no arterial venous shunting or vascular lesions were identified. left vertebral artery: the left vertebral artery injection demonstrated some arterial contribution to the left parietal avmby the distal branches of the parietal occipital branch of the left posterior cerebral artery. no other vascular lesions were identified. the cervical proximal left vertebral artery and its origin were without any significant atherosclerotic disease or stenosis. right internal carotid artery: the right internal carotid artery injection demonstrated brisk filling of the distal branches from normal arterial runoff. brisk filling of the contralateral anterior cerebral artery and the middle cerebral artery, with subsequent filling of the left parietal avm, was noted. no other vascular lesions were identified. right external carotid artery: the right external carotid artery injection demonstrated brisk filling of the distal branches with normal arterial runoff. no arterial venous shunting or vascular lesions were identified. right vertebral artery: the right vertebral artery injection again demonstrated filling of the left parietal avm through the distal left pca branches. no other vascular lesions were identified. impression: 4 cm left parietal arterial venous malformation, supplied by left anterior cerebral artery, left middle cerebral artery, and left posterior cerebral artery distal branches as described above. rapid shunting through the superior sagittal sinus was noted. slugging venous drainage through a dilated cortical vein was also noted, likely secondary to hematoma induced mass effects. no nidal or feeding pedicle aneurysms were detected. brief hospital course: ms was admitted to the surgical icu her blood pressure was kept strictly less than 140. she underwent an immediate cerebral angiogram on 4 cm left parietal arterial venous malformation, supplied by left anterior cerebral artery, left middle cerebral artery, and left posterior cerebral artery distal branches as described above. rapid shunting through the superior sagittal sinus was noted. slugging venous drainage through a dilated cortical vein was also noted, likely secondary to hematoma induced mass effects. no intranidal or feeding pedicle aneurysms were detected. post angiogram she was extubated on hospital day 2 following commands on the left side and withdrawing right side. frequent cts showed left sided blood resolving. she was transferred to the neurostep down on hospital day 4. she was continued to watch closely with strict blood pressure parameters. her neurological exam improved daily with right side plegia and word finding difficulty. a mri was done on which showed known left frontoparietal arteriovenous malformation with enlargement of the left m2 and superior segments of the middle cerebral artery due to supply of the arteriovenous malformation. it was decided since her avm was less than 3cm she could be treated with radiation therapy which will be started in the next 2 weeks. on discharge she was tolerating a dysphagia diet, she was being bladder trained. she was awake, alert and orientated x3 with speech finding difficulty and right sided plegia. medications on admission: prozac, ativan, prilosec, verapamil discharge medications: 1. insulin regular human 100 unit/ml solution sig: one (1) injection asdir (as directed): ssi as directed. 2. heparin (porcine) 5,000 unit/ml solution sig: one (1) injection tid (3 times a day). 3. docusate sodium 100 mg capsule sig: one (1) capsule po tid (3 times a day). 4. bisacodyl 5 mg tablet, delayed release (e.c.) sig: two (2) tablet, delayed release (e.c.) po daily (daily) as needed. 5. magnesium hydroxide 400 mg/5 ml suspension sig: thirty (30) ml po q6h (every 6 hours) as needed for constipation. 6. senna 8.6 mg tablet sig: one (1) tablet po daily (daily). 7. verapamil 40 mg tablet sig: one (1) tablet po q8h (every 8 hours). 8. fluoxetine 20 mg capsule sig: three (3) capsule po daily (daily). 9. acetaminophen 650 mg tablet sig: one (1) tablet po q4-6h (every 4 to 6 hours) as needed. 10. oxycodone-acetaminophen 5-325 mg tablet sig: 1-2 tablets po q4-6h (every 4 to 6 hours) as needed for pain: please give cautiously with tylenol. 11. butalbital-acetaminophen-caff 50-325-40 mg tablet sig: tablets po q4-6h (every 4 to 6 hours) as needed for ha: please use cautiously with tylenol. 12. diazepam 5 mg tablet sig: one (1) tablet po q6h (every 6 hours) as needed for muscle spasm. 13. pantoprazole 40 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po q24h (every 24 hours). 14. labetalol 200 mg tablet sig: one (1) tablet po qid (4 times a day). 15. phenytoin sodium extended 100 mg capsule sig: two (2) capsule po bid (2 times a day). discharge disposition: extended care facility: - discharge diagnosis: avm s/p bleed discharge condition: neurologically stable discharge instructions: return to er if you experience any worsening headaches, vision changes, nausea, vomitting, mental status changes or fevers continue agressive pt/ot. followup instructions: follow up with dr from radiation oncology. his office will be callng you next week with an appointment. if you don't hear from his office please call at . follow up with dr in 4 weeks procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified enteral infusion of concentrated nutritional substances arteriography of cerebral arteries diagnoses: esophageal reflux unspecified essential hypertension depressive disorder, not elsewhere classified intracerebral hemorrhage anomalies of cerebrovascular system 

Answer: The patient is high likely exposed to