Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: iph major surgical or invasive procedure: none history of present illness: mr. is a 77 yo rh man with pmh of cad s/p cabg (4 times), htn, dlp p/w acute cns bleed. he was attending a football game and per report (ems) felt something popped in his head. he then started with garbled speech 5 minutes later. unable to follow commands and confused. he was noted to have right sided weakness (arm and leg). his fsg was normal. he was transferred to osh. he received a ct cns without contrast at 2:15pm which showed an extensive left occipital and parietal bleed with midline shift (5 mm) and edema in compressing wernicke's area. in addition, there was midbrain mild compression. no ivh. he was awake and did not require ett at that time. his inr was 1.1, normal ptt. he received lev 500 mg and was transferred to in the ed. once at , we discussed the case with the family. dr. came in-house and informed the family. once they agreed he was fc, he vomited and was intubated for airway protection given his declining ms episode. his vomit had coffee grounds contents. past medical history: cad (+) s/p cabg 4 times, af (-) htn (+), hld (+), osa (-), dm (-), seizures (-), migraine (-), cns tumors (-) strokes (-), procoagulant conditions (-) social history: lives with his wife. a lawyer. exercises (-) tobacco (-) etoh (-) drugs (-) family history: hx of early strokes (-) or cns bleeds. seizures (-) cns tumors (-) demyelinating conditions (-) autoimmune conditions (-) procoagulant conditions (-) cad (-) htn mother, father (unknown type) physical exam: pe: 9 at 16:10 70 150/80 16 99 gen: lying in bed, nad. heent: nc/at, moist oral mucosa neck: supple, no carotid or vertebral bruit back: no point tenderness or erythema cv: nl s1 and s2, no murmurs/gallops/rubs lung: clear to auscultation bilaterally abd: soft, nontender, non-distended. no masses or megalies. percussion within normal limits. +bs. ext: no edema, no dvt data. pulses ++ and symmetric. . neurologic examination: no meningismus. no photophobia. . ms: general: alert, awake, opens his eyes when saying his name. not consistently following commands. no meningismus. no photophobia. ms: responsive to noxious stimuli, but not localizing on the right hemibody. cn: brain stem reflexes : preserved: corneals + bl. pupils 2 to 1 bl and symmetrically. dolls eyes +. there is a left gaze deviation, does not cross to the right volitionally. there is roving. no nystagmus. gag +. motor: left arm antigravity. left leg antigravity. right hemibody: withdraws to pain (not localizing it). tone: dtr: 2+ throughout in arms. cannot elicit dtrs in patellas or ankles. toe downgoing on the left upgoing on the right . normal bulk. tone: normal. no tremor or myoclonus pertinent results: labs on admission and discharge: import 10:39pm glucose-190* urea n-14 creat-0.8 sodium-141 potassium-3.2* chloride-101 total co2-31 anion gap-12 import 10:39pm ck(cpk)-121 import 10:39pm ck-mb-7 ctropnt-<0.01 import 10:39pm calcium-8.7 magnesium-2.0 import 10:39pm osmolal-306 import 08:04pm type-art rates-10/ tidal vol-550 peep-5 po2-496* pco2-49* ph-7.40 total co2-31* base xs-4 -assist/con intubated-intubated import 04:45pm urine hours-random import 04:45pm urine gr hold-hold import 04:45pm urine color-straw appear-clear sp -1.014 import 04:45pm urine blood-sm nitrite-neg protein-neg glucose-100 ketone-neg bilirubin-neg urobilngn-neg ph-8.0 leuk-neg import 04:45pm urine rbc-* wbc-0-2 bacteria-rare yeast-none epi-<1 import 04:45pm urine amorph-rare import 03:15pm glucose-152* urea n-19 creat-0.8 sodium-142 potassium-3.4 chloride-102 total co2-30 anion gap-13 import 03:15pm estgfr-using this import 03:15pm calcium-8.8 phosphate-2.3* magnesium-2.1 import 03:15pm wbc-10.4 rbc-4.78 hgb-15.3 hct-43.6 mcv-91 mch-32.0 mchc-35.1* rdw-13.7 import 03:15pm neuts-80.5* lymphs-12.2* monos-5.0 eos-2.1 basos-0.2 import 03:15pm plt count-172 import 03:15pm pt-12.7 ptt-24.6 inr(pt)-1.1 imaging: ct on admission: large left parietal-occipital parenchymal hemorrhage. 8mm rightward midline shift. compression of left lateral ventricle. 4th ventricle open. mass effect on left brainstem. cta - patent vessels ct : note added in attending review: the true measurement of the large left temporoparietoccipital hemorrhage, with more acute bleeding superimposed on layering less-acute blood, is probably more like at least 8.5 (ap) x 4.4 cm (trv) . more important, there is severe uncal and early downward transtentorial herniation, with marked effacement of the suprasellar and worsening effacement of the left perimesencephalic cisterns and severe mass- effect upon the left midbrain (e.g. 2:13). the degree of displacement and effacement of the pontomesencephalic junction has progressed over the interval roughly 10 hours. brief hospital course: 77 year old man with cad, htn, hl presented w/ sudden onset large left occipital-parietal hemorrhage with subfalcine and uncal herniation. his examination on admission is noted above. give the lifethreatening hemorrhage, a discussion was held with family and neurosurgery regarding potential for intervention. dr. and dr. said that if the patient were to survive the acute phase of this massive left parietal-occipital hemorrhage, that he would likely have the following neurological deficits: severe language impairment, right homonymous hemianopsia, severe right hemiparesis or hemiplegia, right hemisensory loss, dysarthria, and difficulty swallowing. neurosurgical intervention might enable the patient to survive this acute episode, but it would not reverse any of the neurological deficits and in fact the neurosurgical intervention may make these deficits worse. based on this discussion, a decision by family was made to pursue only medical management. patient was admitted to neuro icu, he was started on mannitol, labetalol drip, hob > 30, sbp goal of 120-140 in addition to other supportive care. the etiology could be due to amyloid angiopathy, vascular malformation, or underlying malignancy. by 2am on , patient's examination deteriorated significantly. he became unresponsive, had an impaired corneal, impaired dolls and flexor withdrawal to noxious stimuli. a repeat ct showed a worsening midline shift, severe uncal and transtentorial herniation and effacement of the perimesencephalic cisterns with mass effect on the midbrain. a discussion was held with family and patient was made cmo. he was extubated in the late morning of and died that day. medications on admission: asa (81 ng qd), coumadin (- ) antihypertensives (diuretic) fish oil, mvi, vit d. statin (unknown which one) discharge medications: not applicable discharge disposition: expired discharge diagnosis: intraparenchymal hemorrhage discharge condition: deceased discharge instructions: not applicable followup instructions: not applicable procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube diagnoses: unspecified essential hypertension aortocoronary bypass status intracerebral hemorrhage compression of brain other and unspecified hyperlipidemia cerebral edema 

Answer: The patient is high likely exposed to