Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: vfib arrest major surgical or invasive procedure: cardiac catheterization history of present illness: mr. is a 42 yo male with a history of hyperlipidemia who collapsed while playing soccer on the evening of admission. 911 was called at approx 9:30pm after the patient had collapsed. the fire department arrived first, and initiated cpr. acd was applied, and reportedly the patient was shocked x 1 for vfib (strip not available). ems team arrived at 9:40 pm, pt in asystole at that time. cpr continued, acls started and pt given epi x 2 and atropine x 1 at which point he developed sinus brady at 57 and sbp 110/70. he was intubated in the field and transported to osh. . the patient's wife reports that the patient had complained of chest pain during exercise over the past few months. no reports of pain today. wife reports he had no recent complaints of fever/chills at home. no complaints of dyspnea. no significant pmh with the exception of hyperlipidemia for which he took medication. . at the osh: initial vitals: hr 50-60, bp 100-110/60-70s, afebrile. wbc: 11, hct 43.4, plt 211, na 138, k 3.5, cl 100, co2 17, glc 272, bun 31, cr 1.4, ck 426, mb 3.8, i 0.8, tropi 0.09 (nl 0-0.3). started on lidocaine gtt, propofol gtt, given asa 325 pr, ativan 1 mg iv, 1 l ns. gcs = 4 per note. . to ccu at request of family. . ros: unable to obtain. past medical history: hyperlipidemia no history of hypertension or diabetes social history: social history is significant for the absence of current tobacco use. there is no history of alcohol abuse or drug use. family history: there is no family history of premature coronary artery disease or sudden death. father died of mi at age 60s, grandfather age 80s. history of arrhythmia. no recent travel. pt moved to the us at age 20. physical exam: admission physical exam vs: t 99.8, hr 80 bp 117/75, rr 19, o2 100% on ac 500x20, 60%, peep 5. gen: intubated, sedated, nonresponsive. heent: ncat. sclera anicteric. perrl 4->2 b/l. conjunctiva were pink, no pallor or cyanosis of the oral mucosa. neck: supple without jvd cv: pmi located in 5th intercostal space, midclavicular line. rr, normal s1, s2. no s4, no s3. chest: no chest wall deformities, scoliosis or kyphosis. resp were unlabored, no accessory muscle use. no crackles, wheeze, rhonchi. abd: obese, soft, nd, no hsm. no abdominial bruits. ext: cool to touch. no c/c/e. no femoral bruits. skin: no stasis dermatitis, ulcers, scars, or xanthomas. pulses: right: carotid 2+ without bruit; femoral 2+ without bruit; 2+ dp left: carotid 2+ without bruit; femoral 2+ without bruit; 2+ dp neuro: perrl. moves b/l le in response to pain. did not see any ue movement, (though ue movement documented in osh note). babinski mute b/l. no clonus. +cough, uncertain if gag reflex. pertinent results: osh ekg: nsr at 63, tw depression v3, tw depression v4 v5, unable to interpret leads avr, avl, avf. ekg on admission to ccu: nsr at 67, nl axis, nl intervals, qtc = 374. twi iii, q in iii. ?j point elevation in v2, v3. biphasic t waves v3, v4. no prior available for comparision. . 02:30am wbc-10.7 rbc-4.76 hgb-15.1 hct-42.6 mcv-89 mch-31.6 mchc-35.4* rdw-12.8 02:30am neuts-82.9* lymphs-11.5* monos-5.2 eos-0.3 basos-0.1 02:30am plt count-203 02:30am pt-13.5* ptt-23.5 inr(pt)-1.2* 02:26am glucose-114* lactate-1.8 na+-138 k+-5.0 02:26am freeca-1.19 02:30am asa-neg ethanol-neg acetmnphn-neg bnzodzpn-neg barbitrt-neg tricyclic-neg 02:30am triglycer-74 hdl chol-49 chol/hdl-2.9 ldl(calc)-80 02:30am albumin-4.0 calcium-8.8 phosphate-3.2 magnesium-2.1 cholest-144 02:30am ck-mb-41* mb indx-6.1* ctropnt-2.43* 02:30am alt(sgpt)-115* ast(sgot)-126* ld(ldh)-278* ck(cpk)-670* alk phos-96 tot bili-0.4 02:30am glucose-116* urea n-31* creat-1.4* sodium-140 potassium-5.6* chloride-106 total co2-27 anion gap-13 03:53am urine rbc-* wbc-0-2 bacteria-rare yeast-none epi-0-2 trans epi-0-2 03:53am urine blood-tr nitrite-neg protein-neg glucose-neg ketone-neg bilirubin-neg urobilngn-neg ph-5.0 leuk-neg 03:53am urine bnzodzpn-pos barbitrt-neg opiates-neg cocaine-neg amphetmn-neg mthdone-neg 08:06am ck-mb-49* mb indx-7.5* ctropnt-0.76* 08:06am ck(cpk)-651* 04:39pm ck-mb-80* mb indx-9.1* ctropnt-1.11* 04:39pm ck(cpk)-877* . tte: the left atrium is normal in size. there is mild symmetric left ventricular hypertrophy with normal cavity size. there is severe global left ventricular hypokinesis. quantitative (biplane) lvef = 20%. no masses or thrombi are seen in the left ventricle. right ventricular chamber size is normal. with mild global free wall hypokinesis. the aortic root is mildly dilated at the sinus level. the aortic valve leaflets (3) appear structurally normal with good leaflet excursion and no aortic regurgitation. the mitral valve leaflets are structurally normal. there is no mitral valve prolapse. mild (1+) mitral regurgitation is seen. the estimated pulmonary artery systolic pressure is normal. there is no pericardial effusion. impression: mild symmetric left ventricular hypertrophy with severe global systolic dysfunction. mild right ventricular systolic dysfunction. no asymmetric septal hypertrophy, marked regional variability in lv wall motion, or significant valvular disease seen. . ct head: there is no evidence of hemorrhage, masses, shift of normally midline structures, hydrocephalus, or major vascular territorial infarct. no abnormalities are seen in the globus pallidi, hippocampal region, or watershed areas of the cortex. ventricles and sulci are normal in contour and configuration. no fractures are identified. air-fluid levels are seen in the sphenoid, ethmoid, and maxillary air cells. endotracheal intubation is noted on the scout film. impression: no intracranial abnormalities. cardiac catheterization 1. selective coronary angiography of this right dominant system demonstrated a single vessel cad. the lmca had a 20% distal stenosis. the lad had a 90% stenosis at the bifurcation with a moderately sized d1. the mid lad had a 30% stenosis. the lcx was patent. the rca had a 20% distal vessel stenosis. 2. limited resting hemodynamics revealed an elevated left sided filling pressure with an lvedp of 19 mmhg. 3. left ventriculography was deferred. 4. successful pta of the first diagonal with a 2.5x12mm voyager balloon. successful ptca and stenting of the proximal lad with a 3.0x18mm cypher stent which was post-dilated to 3.25mm. final angiography revealed 0% residual stenosis in the lad, 20% residual stenosis in the ostium of d1. the patient left hemodynamically stable and back to the ccu for further treatment. brief hospital course: acute myocardial infarction the patient had a myocardial infarction while playing soccer on . he also suffered from vfib arrest and was given cpr on the field. on , he underwent cardiac catheterization and was found to have a 90% stenosis of the lad; he received a cypher stent in this vessel. he was started on atorvastatin 80 mg qd as well as aspirin and plavix. echo on showed an ef of 20% with global hypokinesis, but repeat echo on showed an ef 60% likely reflecting recovery of the stunned myocardium. there was no evidence of structural heart disease. during his event, he was intubated for airway protection and likely suffered some degree of anoxic brain injury from the vfib arrest. he was initially very confused and agitated after his extubation, but these symptoms resolved by the time of discharge. he did seem to have short term memory deficits at the time of discharge, and it was thought he would benefit from ot and pt in short term rehab before returning home. hypertension his high blood pressure was initially managed with metoprolol 37.5 mg tid; he was switched to metoprolol xl 100 mg qd with good effect and discharged on that medicine. he was also started on lisinopril 5mg qd. fevers during his hospitalization, he had several low grade temperatures and was found to have heavy growth of mssa in his sputum. he was also found to have vials of coagulase negative staph in his blood cultures, likely a contaminant. he was started on a seven day course for the mssa in his sputum beginning on and defervesced. medications on admission: unknown medication for high cholesterol discharge medications: 1. aspirin 325 mg tablet sig: one (1) tablet po daily (daily). 2. atorvastatin 80 mg tablet sig: one (1) tablet po daily (daily). 3. clopidogrel 75 mg tablet sig: one (1) tablet po daily (daily). 4. lisinopril 5 mg tablet sig: one (1) tablet po daily (daily). 5. metoprolol succinate 100 mg tablet sustained release 24 hr sig: one (1) tablet sustained release 24 hr po daily (daily). 6. trimethoprim-sulfamethoxazole 160-800 mg tablet sig: one (1) tablet po bid (2 times a day) for 4 days: continue through . discharge disposition: extended care facility: hospital discharge diagnosis: acute myocardial infarction discharge condition: stable-- with improved cardiac function; not short of breath or dyspneic. still with some short term memory deficits from likely anoxic brain injury but overall improved. discharge instructions: you should not do any running or exercise for the next several weeks and until further advised by your cardiologist. this is because you had a mild heart attack should be reevaluated before returning to your normal activity level. there are several medications for your heart that you have been started on because you had a heart attack. you need to take these until told to do so otherwise by your cardiologist. followup instructions: you should make an appointment to see your primary care doctor in the next 1 - 2 weeks. you should make an appointment to see a cardiologist at hospital in the next 2 - 3 weeks-- you can call ( to make an appointment. procedure: coronary arteriography using two catheters angiocardiography of left heart structures insertion of endotracheal tube insertion of drug-eluting coronary artery stent(s) cranial or peripheral nerve graft insertion of one vascular stent excision of lingual thyroid percutaneous transluminal coronary angioplasty [ptca] procedure on single vessel diagnoses: coronary atherosclerosis of native coronary artery unspecified essential hypertension other and unspecified hyperlipidemia ventricular fibrillation family history of ischemic heart disease nausea alone acute myocardial infarction of unspecified site, initial episode of care accidental fall from bed accidents occurring in residential institution other and unspecified superficial injury of face, neck, and scalp, without mention of infection 

Answer: The patient is high likely exposed to