Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: tape attending: chief complaint: diabetic ketoacidosis major surgical or invasive procedure: none history of present illness: pt is a 28 yo female with dm type i, migraines, factor inhibitor, warm autoantibodies, antral ulcer, and recently admitted in dka secondary to uti presents with 12 hours of nausea and vomiting. on night prior to admission, patient developed a headache and then decreased appetite. she then had worsening n/v x 5 episodes. in ed anion gap was found to be 34 with a bicarbonate of 10. cxr was negative. serum tox was also negative. pt was started on insulin drip in the ed. in micu insulin drip was d/cd on night of admission () and pt was started on lantus and on a humalog sliding scale. anion gap closed on night of admission as well. on transfer to the medicine floor () pt vomited x 2 and was feeling extremely nauseous. ekg on admission showed diffuse t wave inversions. ros: no ab pain/ sick contacts /dysuria /dyspareunia /vaginal discharge /f /c/ cough/ painful or red joints/ brbpr/ melena. no cp. pt had been doing well until 6 months ago with birth of first child. delivery was complicated secondary to severe hyperemesis requiring tpn and a c-section at 33 weeks. further complicated by post-op hemmorhage requiring transfusions. since delivery constant n/v and 3 other admissions for dka. is working patient up for gastroparesis. in the micu, affect noted to be flat and depressed. home life has been stressful and pt with difficulty taking care of child secondary to nausea and multiple inpatient hospitalizations. past medical history: 1. dm type 1 complicated by retinopathy (dx at 11. episodes dka secondary to infection. hga1c 7.2%) 2. depression 3. severe hyperemesis requiring tpn as above 4. s/p c-section 5. g2p1 s/p miscarriage in 6. migraines 7. factor inhibitor- treated with steroids and rituximab. 8. anti-e and warm auto-antibody 9. gerd, antral ulcer. s/p ugi bleed admission . 10. esophagitis 11. anemia social history: married with one child. no etoh/drugs/tobacco. family history: non-contributory physical exam: note: on admission to the medicine floor (day 2 of hospital admission)' vs: t:99.4; bp: 137/81; rr:10; o2: 98% on ra 16 hr i/o 4651/ gen: laying in bed, not opening eyes, flat affect. answering questions though moaning answers/ heent: pupils reactive 5->4 mm b/l. sclera anicteric. op clear though pt would not open mouth fully to visualize tonsils. neck: no lad. no jvd. cardio: tachycardia. rrr s1s2. lungs: pt not taking deep breaths. cta b/l ab: +bs. soft, nt, nd. ext: dp 2+, nt,nd. neuro: pt somewhat uncooperative. did not respons fully to all questions answered. flat, depressed affect. pertinent results: admission labs: 08:10am wbc-12.3*# rbc-4.88 hgb-15.4 hct-46.4 mcv-95 mch-31.5 mchc-33.2 rdw-13.5 neuts-86* bands-1 lymphs-11* monos-1* eos-0 basos-0 atyps-0 metas-0 myelos-1* asa-neg ethanol-neg acetmnphn-neg bnzodzpn-neg barbitrt-neg tricyclic-neg acetone-large albumin-4.9* calcium-10.4* phosphate-4.2 magnesium-1.9 lipase-11 ggt-18 alt(sgpt)-8 ast(sgot)-10 alk phos-138* amylase-36 tot bili-0.4 glucose-547* urea n-16 creat-0.8 sodium-138 potassium-4.7 chloride-94* total co2-10* hcg-<5 12:02pm blood type-art po2-97 pco2-42 ph-7.27* calhco3-20* base xs--7 ___________________________ cardiac enzymes: 02:12pm blood ck-mb-notdone ctropnt-<0.01 ck(cpk)-18* 09:30pm blood ck-mb-1 ctropnt-<0.01 ck(cpk)-20* 03:30am blood ck-mb-notdone ctropnt-<0.01 ck(cpk)-27 ___________________________ discharge labs: 05:15am blood wbc-6.0 rbc-4.01* hgb-12.1 hct-37.3 mcv-93 mch-30.2 mchc-32.5 rdw-13.6 plt ct-372 glucose-50* urean-10 creat-0.4 na-141 k-3.7 cl-102 hco3-30* angap-13 calcium-9.0 phos-5.6* mg-1.8 brief hospital course: 1.diabetic ketoacidosis pt's course in micu outlined in the hpi section of this discharge summary. pt was put on standing lantus (18 units at night) with a humalog sliding scale. electrolytes were initially checked twice a day and then once they were stable they were checked only once a day. was following pt while in the hospital. blood sugars were in the low 100s-mid 200s range. 2. n/v intravenous fluids were continued while patient was unable to take good po. diet was slowly advanced to clears and then to a full diabetic diet. pt was given metoclopramide to stimulate gi motility and zofran was given for nausea. etiology of n/v could be gastroparesis. a gastric motility study was planned for but patient left against medical advice on the day prior to scheduled study. 3. cause of dka wbc was 12.3 on admission. u/a, cxr, and lfts were all normal. blood cultures were negative. continuous nausea and vomiting could be tipoff to dka (see above) and secondary to diabetic gastroparesis. unfortunantly, the gastric emptying study was not done. also, pt has been on tapering prednisone for factor vii inhibitor. on admission, dose was 5 mg qotherday. another possibility, though extremely unlikely, is that the dka is secondary to prednisone; however the dose is probably not high enough. 4. hypertension pt had newfound hypertension with tachycardia (pt noted to be tachycardic on previous admissions). diastolic blood pressure was as high as 110. pt was started on lopressor 12.5 mg po bid. head ct was done looking for bleeding or a mass. it was normal and there was no evidence of acute hemorrhage, mass effect, or shift of normally mid-line structures. another possibility of htn could be autonomic insufficiency from dm. pt's bp went down to normotensive values with the lopressor. 5. t-wave flattening and inversion on ekg on admission ekg, there was diffuse t wave flattening and inversions. cardiac enzymes were negative x 3. echo was done and was normal with ef of >55%. etiology could have been secondary to hypertension and cardiac straining. patient to discuss with pcp need for stress testing as an outpatient. 6. gerd/antral ulcer pt was on protonix . 7. social issues/depression- socialwork saw and followed patient. pt has a complicated social situation (see hpi. pt has been in hospital for a large part of her babies life). sertraline was continued. 8. factor- inhibitor- pt was s/p rituximab and on a steroid taper (prednisone 5 mg po qother day). 9. f/e/ pt's diet was slowly advanced as she was able to better tolerate po when the nausea and vomiting decreased. she was tolerating po when she left ama. medications on admission: 1. protonix 40 mg po once a day 2. sertraline 100 mg po once a day 3. reglan 10 mg qpm 4. lantus 18 units qhs 5. humalog sliding scale based on meals discharge medications: pt unfortunantly left ama secondary to childcare issues. she was d/cd on home medications with full-up at . discharge disposition: home discharge diagnosis: diabetic ketoacidosis diabetes mellitus insulin dependent hypertension depression migraines factor inhibitor anti-e and warm auto-antibody gastroesophageal refulx disease anemia discharge condition: fair. nausea and vomiting had subsided and hypertension was under control. however, gastroparesis work-up unfortunantly was not completed. discharge instructions: pt left ama. followup instructions: pt left ama. she was instructed to make a follow-up appointment at which she will hopefully do. procedure: venous catheterization, not elsewhere classified diagnoses: unspecified essential hypertension aortic valve disorders diabetes with ketoacidosis, type i [juvenile type], not stated as uncontrolled background diabetic retinopathy diabetes with neurological manifestations, type i [juvenile type], not stated as uncontrolled gastroparesis diabetes with ophthalmic manifestations, type i [juvenile type], not stated as uncontrolled peripheral autonomic neuropathy in disorders classified elsewhere congenital factor viii disorder 

Answer: The patient is high likely exposed to