Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: this is an 88 year old male with a history of multi-infarct dementia, cerebrovascular accident, low grade lymphoma, upper gastrointestinal bleed, coronary artery disease status post non-q wave myocardial infarction, chronic iron deficiency anemia and chronic renal insufficiency who presented with dark emesis as well as dark stools a couple of days prior to admission. the patient noted some abdominal pain. in the emergency department, he had tachycardia and hypertension with vital signs 88/63 with a pulse of 135. a right subclavian central line was placed and the patient was given two liters of intravenous normal saline. the patient was difficult to cross-match, but eventually was given three units of packed red blood cells. an esophagogastroduodenoscopy in the medical intensive care unit revealed mild gastritis and therefore plavix was held. past medical history: 1. multi-infarct dementia. 2. cerebrovascular accident with residual left upper extremity paresis. 3. history of multiple pulmonary embolisms status post ivc filter. 4. low grade lymphoma. 5. history of upper gastrointestinal bleed. 6. benign prostatic hypertrophy status post transurethral resection of prostate. 7. gastroesophageal reflux disease. 8. barrett's esophagus. 9. coronary artery disease status post non-q wave myocardial infarction. 10. diverticulosis. 11. status post cholecystectomy. 12. iron deficiency anemia. 13. chronic renal insufficiency with baseline creatinine of 1.1. 14. paget's disease. 15. history of bladder cancer. social history: the patient lives with wife in . he is a full code status. family history: noncontributory. physical examination: upon admission is temperature of 99.0 f.; with heart rate of 77 to 117, blood pressure of 98 to 130 over 29 to 51; respiratory rate of 12 to 17; saturation of 100% on room air. the patient is well appearing male in no acute distress. he is asleep but answerable. heent: his mucous membranes are dry. pupils are equal, round, and reactive to light and accommodation. extraocular muscles are intact. neck is supple with jugular venous pressure of 5 centimeters of water. cardiovascular: regular rate and rhythm, normal s1, s2. no murmurs or rubs noted. pulmonary: clear to auscultation bilaterally. abdomen soft, nontender, nondistended, with normal active bowel sounds. extremities with no cyanosis, clubbing or edema noted with two plus dorsalis pedis. laboratory: upon admission potassium 4.1 with 79% neutrophils, hematocrit of 22.9, platelets 154. sodium 143, potassium 4.0, chloride 110, bicarbonate 19, bun 50, creatinine 1.1, glucose 130. inr 1.3, iron 19, tibc 345, reticulocyte 3.0. urinalysis showed moderate leukocyte esterase with greater than 50 white blood cells, 15 red blood cells, few bacteria and 3 epithelial cells. urine cultures pending. ekg showed normal sinus rhythm of 92 with right bundle branch block. there is depression in v2 through v3 along with t wave inversions v1 through v5. compared to ekg, there is new downsloping of st and t wave inversions in v2 through v5. chest x-ray showed no cardiopulmonary changes. studies: echocardiogram revealed an ejection fraction of greater than 55%, mild to moderate mitral regurgitation and mild pulmonary hypertension. hospital course: 1. upper gastrointestinal bleed secondary to gastritis: the patient was given three units of packed red blood cells and his hematocrit bumped up appropriately from 22.9 to 30.1. his hematocrit was checked every eight hours and held steady around 30, however, when he ruled in for a myocardial infarction, he was given another unit of blood despite his hematocrit being only 29.8. his hematocrit then bumped up completely to 33.5. his hematocrit was then checked just once a day. the patient was given an h2 blocker twice a day in the form of famotidine 20 mg intravenously twice a day. his non-steroidal anti-inflammatory drugs, aspirin and plavix were held until his ruled in for a myocardial infarction and that was safely restarted once his hematocrit had been holding steady. the patient's stools remained guaiac positive throughout his hospital course. the patient's issue was going to be prepped for colonoscopy but then it was decided that this would provide no further information, so that should be deferred for an outpatient basis. by the time of discharge, the patient should be on a proton pump inhibitor twice a day orally while on his plavix and non-steroidal anti-inflammatory drugs. also, his hematocrit held steady a couple of days prior to discharge, at around 33.5 prior to discharge. a serum helicobacter pylori was sent but was still pending upon discharge. 2. coronary artery disease with non-st elevation myocardial infarction: by the third day of admission, the patient started ruling in for a myocardial infarction with cks peaking at 224 with ck mb at 11 and ck mb index of 4.9. his troponin peaked at 11 but then trended downward to 4.5. after the enzyme trended downward, cardiac enzymes were no longer checked. cardiology was consulted and it was felt that cardiac catheterization would not be an option for this patient. medical management with aspirin, plavix and a beta blocker was started. the patient was initially started on metoprolol 12.5 mg p.o. twice a day and then increased to 25 mg p.o. twice a day, however, his heart rate then became quite slow in the low 50s, so his metoprolol was then decreased back down to 12.5 p.o. twice a day. an ekg was obtained showing that he still had normal sinus rhythm at 63 beats per minute with prolonged qtc now of 493 which is new. there is persistent t wave inversions in v2 through v5. the patient was sent for an echocardiogram given that he ruled in for myocardial infarction. the echocardiogram revealed ejection fraction of 30% with anterior septal left ventricular hypokinesis. also noted was right ventricular apical hypokinesis, aortic root moderate dilation, two plus mitral regurgitation, two plus tricuspid regurgitation and moderate pulmonary artery hypertension. the tricuspid regurgitation and depressed left ventricular function is new since the echocardiogram in . given that there is left ventricular dysfunction, the patient was started on a low dose ace inhibitor in the form of captopril 4.5 mg p.o. three times a day for remodeling. discharge diagnoses: 1. upper gastrointestinal bleed secondary to gastritis. 2. anteroseptal non-st elevation myocardial infarction. 3. gastroesophageal reflux disease. 4. barrett's esophagus. 5. multi-infarct dementia. discharge medications: 1. metoprolol 12.5 mg p.o. twice a day. 2. aspirin 81 mg p.o. q. day. 3. plavix 75 mg p.o. q. day. 4. protonix 40 mg p.o. twice a day. 5. lisinopril. condition on discharge: stable. discharge status: to rehabilitation center. discharge instructions: the patient is to follow-up with his primary care physician, . , in one to two weeks. , m.d. dictated by: medquist36 procedure: other endoscopy of small intestine diagnoses: subendocardial infarction, initial episode of care esophageal reflux urinary tract infection, site not specified unspecified essential hypertension iron deficiency anemia, unspecified hemorrhage of gastrointestinal tract, unspecified esophagitis, unspecified 

Answer: The patient is high likely exposed to