Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: respiratory failure, acute stroke syndrome major surgical or invasive procedure: intubation history of present illness: dr. is a 78 yo male with a h/o atrial fibrillation, dementia, and idiopathic thrombocytopenia who is transferred from after acute onset of facial droop and dysarthria on the morning of . per information gathered from medical record and from family, patient awoke with slurring of speech and right facial droop. he was immediately transferred from the rehab facility to out of concern for cva. per ems record, patient had equal grip and strength and denied any headache or chest pain at time of transfer. . on arrival to bp 121/81, hr 103-112 (afib), t 95.3, bg 160, respirations were unlabored on 2l. he was documented to be alert but with difficulty word finding, with right facial droop, and slurred speech. he was able to swallow a dose of coreg 6.25 mg po which was administered for rapid afib. he subsequently received 5 mg iv lopressor for hr in the 130's. per family's report, he became increasingly agitated and confused and attempted to climb out of bed. he is next documented to have developed a 20 second period of apnea with - breathing and inability to speak. he was reported to be cyanotic in his lips and extremities at this time and was intubated with versed 4 mg, ativan 2 mg iv, and succ 200 mg. he was transported via to . . on arrival to the ed, t 97.9, bp 72/42, rr 14, spo2 99%. levophed drip was started. bedside fast exam was performed and lij sepsis catheter was placed. ct head was negative for acute intracranial process. ct torso was negative for evidence of occult infection. he received 4 liters ns, ceftiraxone 2 gram, vancomycin 1 gram, protonix 40 mg iv, thaimine 100 mg iv, and decadron 10 mg iv. neurology was consulted. past medical history: atrial fibrillation, s/p failed cardioversions x 2 at thrombocytopenia h/o nasal polyp dementia of the alzheimer's variant cardiomyopathy with globally dilated heart, ef 40% (adenosine sestamibi stress from mild early senial demential of the alzheimer's type anemia gout cad s/p bilateral hip and knee replacements nsvt social history: patient's family reports he smoked a pipe; he has no history of cigarette smoking. he drank 2+ alcoholic beverages everynight. he is married and has 8 children. he is a retired internist. he has temporarily been living at in , nh prior to this admission, recovering from recent medical illness. family history: father with lung cancer (smoker), mi, cva. mother with emphysema (smoker). his eight children are all healthy. there is no other significant family history of cva, mi, or malignancy. physical exam: vs: t 97.9, hr 120, bp 127/91, rr 20, spo2 100% gen: intubated, minimally responsive without sedation cv: irregularly, irregular resp: lungs cta abdomen: obese, soft, nt/nd extrem: cool to touch in all four extremities; 2+ lower extremity pitting edema to shins; well-healed midline scars over both knees skin: non-blanching purpura over lower extremities; no rashes neuro: no obvious facial droop; pinpoint pupils with sluggish reaction to light; opens eyes to stimuli; moves left upper extremity, left lower extremity & right lower extremity; flacid tone in rue without any purposeful or nonpurposeful movements; areflexive in patellar tendons; upgoing toes bilaterally; no clonus. pertinent results: 05:15pm wbc-11.6* rbc-3.03* hgb-9.5* hct-31.3* mcv-103* mch-31.3 mchc-30.3* rdw-16.2* 05:15pm neuts-90.6* bands-0 lymphs-5.5* monos-3.4 eos-0.3 basos-0.1 05:15pm plt smr-very low plt count-41* 05:15pm pt-21.0* ptt-39.1* inr(pt)-2.0* 05:15pm glucose-131* urea n-38* creat-1.2 sodium-140 potassium-4.5 chloride-111* total co2-20* anion gap-14 05:15pm alt(sgpt)-197* ast(sgot)-95* ld(ldh)-315* ck(cpk)-45 alk phos-69 tot bili-2.9* 05:15pm lipase-44 05:15pm ctropnt-0.69* 05:15pm ck-mb-notdone 05:15pm albumin-2.9* calcium-6.9* phosphate-4.2 magnesium-2.4 05:15pm haptoglob-117 05:15pm tsh-2.0 05:15pm asa-neg ethanol-neg acetmnphn-neg bnzodzpn-neg barbitrt-neg tricyclic-neg . studies: * torso ct : 1. bilateral pleural effusions, right greater than left. 2. mesenteric fat stranding, pericholecystic fluid, periportal edema and small amount of ascites which may be due to low albumin state or third spacing. 3. bilateral hypodense lesions, some of which are too small to characterize, others which are simple cysts. 4. infrarenal abdominal aortic aneurysm measuring up to 4.8 x 6.2 cm in widest dimension. 5. calcifications within the head of pancreas. . * head cta : 1. no evidence of acute intracranial process on ct head. 2. ct perfusion shows no evidence of abnormal perfusion in the visualized portions of the brain. 3. cta head shows persistent trigeminal artery between the basilar artery and the right cavernous carotid artery. related narrowing of the basilar artery as well as the vertebral arteries, which feeds the pica. no region of focal stenosis or occlusion is seen. 4. a region of apparent narrowing of the left ica distal to its origin is likely artifactual due to a large amount of streak artifacts from dental filling at this level. . * brain mri : 1. findings consistent with acute infarct in the deep white matter of the left centrum semiovale. findings suggest watershed infarct which may be related to hypotensive episode. 2. mra again shows persistent trigeminal artery with decreased size of the basilar artery as well as the vertebral arteries which feed the pica. no region of focal stenosis or occlusion is seen. 3. tissue loss is again demonstrated in the left inferior frontal lobe. old ischemic changes also seen in the pons. . * abd u/s : 1. gallbladder remains nondistended without evidence of stone. note is made of wall thickening and pericholecystic fluid which is non-specific and may be seen in the setting of liver dysfunction, congestive heart failure or hypoalbuminemia. please correlate clinically. 2. trace perihepatic ascites. 3. pleural effusion. . * eeg : this is an abnormal portable eeg due to the disorganized, low voltage and slow background admixed with bursts of generalized mixed frequency slowing. this constellation of findings is consistent with a mild to moderate encephalopathy and suggests dysfunction of bilateral subcortical or deep midline structures. medications, metabolic disturbances, infection, and hyoxia are among the common causes of encephalopathy, but there are others. there were no areas of prominent focal slowing, although encephalopathic patterns can sometimes obscure focal findings. there were no clearly epileptiform features. the beta activity likely reflects medication effect. note is made of the abnormal cardiac tracing. . * echo : dilated left ventricle with severe regional and global systolic dysfunction. dilated right ventricle with moderate systolic dysfunction. mild aortic regurgitation. moderate mitral regurgitation. moderate pulmonary hypertension. dilated thoracic aorta. . * head mri : 1. new large vascular territory infarct involving left mca territory and superimposed on previously seen left watershed infarct. this could be embolic or thrombolic in etiology. no hemorrhage or shift of normally midline structures is seen. although this is not an adequate evaluation of intracranial vessels, the normal vascular flow voids are demonstrated. brief hospital course: 78-year-old man with a history of atrial fibrillation, idiopathic thrombocytopenia, and dementia who presented with left-sided watershed infarct and depressed ef, with hospital course complicated by a pea arrest requiring 7-minute cpr, epi x 1, re-intubation, and death. . # cva: mri on admission revealed a left centrum semiovale stroke. the patient displayed right-sided neglect. he was intermittently responsive to stimuli. his respiratory status improved temporarily and he was briefly extubated before being re-intubated again after a pulseless electrical activity arrest. repeat head mri then showed a left mca embolic stroke. the patient's clinical status deteriorated with hypotension requiring pressors. with continued clinical decline, and following extensive discussions with family members (including wife and hcp), all in agreement for dnr status, and the patient quietly and comforably died in the presence of his family on . . # coagulopathy: he had thrombocytopenia and required platelet and cryoprecipitate infusions. there was no evidence for ttp. concerning for itp, he was given steroids. the etiology was his coagulopathy was unclear. . # acute blood loss: patient had hematemesis and epistaxis, likely precipated by his coagulopathy. he was transfused with prbcs. . # atrial fibrillation: he received digoxin and diltiazem prn. . # transaminitis: lfts elevated throughout the hospital stay. unclear etiology. medications on admission: allopurinol 150 mg daily carvedilol 6.25 mg colchicine 0.6 mg daily (d/c'd ) colace 200 mg aricept 10 mg daily lisinopril 10 mg daily prednisone taper 25 mg po daily x 1 week nacl nasal spray tid zocor 20 mg qam lasix 40 mg qam zosyn 3.375 g iv q 8h (started ) arixtra 2.5 mg sc daily (d/c'd ) zolpidem 10 mg qhs oxygen via nc discharge disposition: expired discharge diagnosis: stroke discharge condition: expired discharge instructions: expired followup instructions: expired md procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more enteral infusion of concentrated nutritional substances transfusion of packed cells transfusion of other serum transfusion of platelets transfusion of coagulation factors diagnoses: anemia in chronic kidney disease congestive heart failure, unspecified hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage i through stage iv, or unspecified atrial fibrillation personal history of tobacco use chronic kidney disease, unspecified acute respiratory failure family history of ischemic heart disease cerebral embolism with cerebral infarction metabolic encephalopathy alzheimer's disease dementia in conditions classified elsewhere without behavioral disturbance hemorrhage of gastrointestinal tract, unspecified acute myocardial infarction of unspecified site, initial episode of care other and unspecified coagulation defects other nonspecific abnormal serum enzyme levels family history of malignant neoplasm of trachea, bronchus, and lung 

Answer: The patient is high likely exposed to