Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: celexa attending: chief complaint: headache, left sided neglect major surgical or invasive procedure: s/p hemicraniotomy evacuation s/p peg s/p trach conventional cerebral angio s/p excision of r arm superficial vein history of present illness: pt is a 69 yo lh male with h/o osteoarthrits who awoke with severe right sided ha this am, now presenting to ed with ich. patient provides history. patient reports waking this am with severe ha that got worse as day progressed. his wife denies seeing any change in his speech, gait, eating habits this am. he went to see him pmd who referred him to . admission bp was 184/76, hr 78, temp 98.3. he had a ct scan which showed right temporal intraparenchymal hemorrhage with extension into the subarachnoid space at 4pm. per ed resident, patient had nml neuro exam on their evaluation on admission. neurology consult eval at 6pm showed left neglect and cth repeated for change in examfindings. repeat hct showed extention of hemorrhage to right parietal lobe and ivh, 1 mm mls. per wife, patient has no ho htn and in fact reports hypotension in the past. patient reports that he has ho hypertension, thinks this was mild. no trauma. he has no ho of headache this week to suggest sentinal bleed. ros: no fever, chills, sob, ab pain, d/c. vomited 2 times in ed. no change in vision, patient has left ptosis at baseline, no dysarthria, dysphagia, diplopia. pt denies any ho transient weakness, numbness, tingling in past. past medical history: dyslipidemia insomnia h/o lumbosacral radiculopathy htn recent pancreatitis (1 month ago)- thought secondary to gallstones. social history: lives at home with wife family history: denies any h.o family with ich, vascular malformations, stroke. mother had pancreatic cancer, father and brother had heart disease physical exam: t:98.8 bp:ed 140-184/52-76 hr:76 rr:16 o2sats:99% 3lnc gen: wd/wn, comfortable, nad. heent: mmm, no dysmorphis, left ptosis cv: rrr, no m resp: cta ab: nd, nt, soft ext: wwp ms: awake and alert. names oriented x3. dow backwards, spells world backwards. reports 7 quarters as "1.50", repeats missing on transitional word. neglects/hemianopsia on exam and only describes very right corner of cookie picture ("i see a stack of plates") and sees "chair" on stroke cards (on right side). he does name some common objects such as ring, watch, book and pen when held on right field of vision. memory 0/3. cranial nerves: i: not tested ii: pupils on right 4 mm-> 3 mm and left 3.5 mm-> 3 mm. ?left hemianopsia iii, iv, vi: patient crosses ml briefly to left with stimulation. eomi. left ptosis (baseline per pt) v, vii: left smile slightly down compared to right (baseline per wife). reports no sensation of left face v1-v3. viii: hearing intact to voice. ix, x: palatal elevation symmetrical. : sternocleidomastoid and trapezius normal bilaterally. xii: tongue midline without fasciculations. motor: normal bulk and tone bilaterally. strength full power throughout. has left pronator drift. dtrs brisker on left than right in b, br, p, a. sensation: reports no sensation to lt/cold on leftside face, arm, leg. with noxious stimuli to left finger, patient appears to wince in pain. when asked where pain is coming from he reports "my head" and points to right temporal region. extinguishes on left. toes downgoing on the right and upgoing on the left. coordination: slowed on finger-nose-finger, with action tremor noted left >right. slow alt hand movements but accurate, does not perform on left but rather persists on right. gait deferred. pertinent results: admission labs: ua nml 140 103 12 agap=12 ------------<132 4.2 29 1.2 ca: 10.0 mg: 1.9 p: 2.4 wbc 8.2 hgb 14.7 plts 117 hct 40.2 n:70.1 l:25.2 m:4.2 e:0.5 bas:0.1 pt: 12.8 ptt: 23.6 inr: 1.1 ekg: nsr microbiology: urine culture- + for pansensitive enterococcus. imaging: ct head 4:09 pm: there is an area of acute hemorrhage measuring 1.8 x 3.5 cm centered in the inferior right temporal lobe. there is a small amount of surrounding vasogenic edema. the hemorrhage appears to extend the extra-axial space, and there is subarachnoid hemorrhage in sulci of the right temporal, right occipital and right posterior parietal lobe. a small amount of subarachnoid hemorrhage is also seen in the inferior right frontal cortex of the left frontal lobe could be due to artifact. there is no evidence of hydrocephalus or intraventricular blood. there is no evidence of transitory herniation. no midline shift. no ct evidence of major vascular territorial infarct. bony structures are unremarkable without fractures. ct head 6:40 pm: (wet read) dramatic progression of hemorrhage, now with multilobar extension (new in right parietal lobe and subependymal spread along right lateral ventricle. ct head w/o contrast 4:09 pm there is an area of acute hemorrhage measuring 1.8 x 3.5 cm centered in the inferior right temporal lobe. there is a small amount of surrounding vasogenic edema. the hemorrhage extends into the subarachnoid space (right temporal, right occipital and right posterior parietal lobe). there is no evidence of hydrocephalus or intraventricular blood. there is no evidence of transtentorial herniation. no midline shift. no ct evidence of major vascular territorial infarct. bony structures are unremarkable without fractures. surrounding soft tissues are unremarkable. impression: right temporal intraparenchymal hemorrhage with extension into the subarachnoid space. given the location and the absence of history of trauma, anticoagulation or hypertension, this is most likely related to amyloid angiopathy, or less likely, underlying mass lesion or vascular malformation. ct head w/o contrast 8:45 am findings: appearance of right temporoparietal craniotomy is little changed from prior exam. the right temporoparietal intraparenchymal hemorrhage and surrounding vasogenic edema are also little changed, with continued effacement of right hemispheric sulci, and frontal of the right lateral ventricle. approximately 3 mm of rightward subfalcine herniation is unchanged. several tiny foci of pneumocephalus persists. extra-axial blood in the right frontal and temporoparietal region is little changed, still measuring approximately 4 mm in greatest axial dimension. impression: 1. unchanged appearance of right temporoparietal craniotomy, with areas of intraparenchymal hemorrhage, surrounding mass effect, and slight leftward subfalcine herniation. 2. unchanged right frontal, and temporoparietal extra-axial fluid collection. ct head w/o contrast 11:06 am findings: again post-surgical changes consistent with right temporoparietal craniotomy are noted. no significant change is observed in the pattern of the intraparenchymal hemorrhage and the vasogenic edema. persistent effacement of the sulci involving the right cerebral hemisphere, unchanged subdural hemorrhage as well as the midline shifting. there is no evidence of perimesencephalic, uncal or transtentorial herniation. the paranasal sinuses demonstrate larger mucosal thickening involving the ethmoidal air cells as well as the sphenoidal sinus, and mild mucosal thickening is detected on the left maxillary sinus. impression: no significant change is observed in the overall configuration of the right intraparenchymal hemorrhage with associated soft tissue mass effect. persistent and unchanged right frontal and temporoparietal extra-axial subdural collections. increase in the pattern of mucosal thickening observed in the ethmoid and sphenoidal sinuses as described above. mr head w & w/o contrast; mra brain w/o contrast 12:06 am comparison: multiple non-contrast head cts are available from to . conventional cerebral angiogram, ; cta head . mr of the brain without and with iv gadolinium: allowing for differences in modalities, the appearance of the brain is not significantly changed compared to the most recent study of . again demonstrated is high t1- weighted signal and associated areas of susceptibility artifact consistent with hemorrhage involving the parenchyma of the right temporal and parietal lobes. high flair signal surrounding the areas of hemorrhage corresponds to low attenuation areas on the prior head ct indicating surrounding vasogenic edema, not changed. a thin subdural collection layering around the right cerebral convexity is stable. mass effect evidenced by effacement of the right cerebral sulci, shift of the septum pellucidum to the left by approximately 5 mm, and effacement of the occipital of the right lateral ventricle has not changed. overall, the size and configuration of the ventricular system is stable. as before, there is a thin extra-axial collection near the craniotomy site. no underlying enhancing mass lesion is seen. mild leptomeningeal enhancement of the right parietotemporal region is probably related to subarachnoid hemorrhage. on the diffusion weighted images there is expected high signal associated with the areas of hematoma, but no evidence of acute infarction elsewhere. mucosal thickening and small fluid levels of the sphenoid sinus, opacification of a few left ethmoid air cells and mild left maxillary sinus mucosal thickening are unchanged. mra of the brain including circle of : the vertebrobasilar and carotid circulations including circle of are patent without evidence of aneurysm, dissection, vascular malformation or hemodynamically significant stenosis. incidental note is made of early branching of the left middle cerebral artery. there is a fetal posterior cerebral artery on the right. diminutive appearance of the basilar artery is probably related to partial supply of the posterior brain by the anterior ciruclation via large posterior communicating arteries. impression: 1. overall appearance of the brain is similar to at 11:26. 2. no change in extent or related mass effect of right parietotemporal intraparenchymal hemorrhage or small right subdural hematoma. 3. no underlying enhancing mass lesion or evidence of vascular malformation. 4. stable paranasal sinus mucosal disease as described. brief hospital course: mr. is a 69 yo lhm found to have spontaneous, acute ich of right temporal lobe that extended to right pariental lobe and right lateral ventricle. on admision the patient's exam was notable for right ptosis (baseline per pt/family), profound left neglect, possible left hemianopsia, left pronator drift, brisk dtrs on left, left upgoing toe, left extinction. 1) ich the patient was admitted to the neuro icu for close monitoring and bp control. initial labs were unrevealing for thrombocytopenia or coagulopathy. the patient is likely to have had a history of hypertension. however the location of the hemorrhage is not commonly seen with primary hypertensive hemorrhage. no history of trauma. conventional angiogram was performed by dr. of the neurosurgery service without evidence for avm or aneurysm. antiplatelet therapy was held. an mri with gradient echo, with and without gadolinium was done and did not show any evidence of amyloid angiopathy nor underlying mass lesion. the patient's neurologic status declined on hospital day #3 with evidence by ct for enlarged hemorrhage with mass effect. the patient's mental status became somnolent from a baseline of conversant and easily arousable with worsening left neglect and left hemiplegia. eeg negative for seizure. he was started on keppra 500mg which has since been discontinued. he was taken for emergent hemicraniectomy and evacuation of the left parietal portion of the hemorrhage. the patient was severely inattentive following the procedure with profound withdrawal from his environment. extubation was successful for several days, but the patient did not protect his airway due to depressed mental status and required re-intubation. he went for tracheostomy and peg placement on hd #10 given need for prolonged period of rehabilitation. 2) infectious disease- pt had fevers on hd #8, had right forearm phlebitis, that underwent i and d on hd #10. started on empiric vancomycin. noted to have a uti with growth of pansensitive enterococcus. vancomycin was discontinued and he was continued on amoxil to complete a 7-day course. he has had some residuals since starting peg tube feeds so we have started him on reglan and decreased his peg tube feeds to half strength. kub was unremarkable. he has a history of a bout of pancreatitis one month ago attributed to gallstones. his lipase has been stable aroun 100 for the past few days. he also had pulled out his foley with the ballon inflated and has some hematuria and decreased urine output likely due to a bladder clot so a foley catheter was reinserted. his wbc count is increasing to 13 so we are resending urinalysis, cxr, and sputum culture. the wound care nurse has been monitoring his r forearm incision where he had the thrombophlebitis, so it needs to continue to be checked to ensure there is no cellulitis. hematocrit is rising as was 31 on the day of discharge. ---- **** on the day of discharge, his ua was normal, sputum culture is pending, and a cxr was repeated which was read as "there is very mild region of heterogeneous opacification in the left mid lung very similar to the appearance on , which developed after . this could be a small region of pneumonia, but has not worsened. lungs otherwise clear. heart size normal. tracheostomy tube in standard placement. no pneumothorax or pleural effusion.". given the stability of the lesion in his left lung, we felt it was not likely infectious, however, if he does develop fever or other signs of infection, would consider repeating this study with focus on this area. *** medications on admission: asa 81 mg qday atenolol 20 mg qday clonazepam 0.5 mg qhs simvastatin 20 mg qhs vitamin b12 multivitamin omeprazole omega-3 fatty acids/vitamin e discharge medications: 1. polyvinyl alcohol-povidone 1.4-0.6 % dropperette : drops ophthalmic prn (as needed). 2. acetaminophen 325 mg tablet : 1-2 tablets po q6h (every 6 hours) as needed for fever/pain. 3. heparin (porcine) 5,000 unit/ml solution : 5000 (5000) units injection tid (3 times a day). 4. metoclopramide 10 mg tablet : one (1) tablet po qidachs (4 times a day (before meals and at bedtime)). 5. lansoprazole 30 mg tablet,rapid dissolve, dr : one (1) tablet,rapid dissolve, dr daily (daily). 6. labetalol 100 mg tablet : 1.5 tablets po tid (3 times a day). 7. albuterol sulfate 2.5 mg/3 ml solution for nebulization : one (1) inh inhalation q6h (every 6 hours) as needed. 8. senna 8.6 mg tablet : one (1) tablet po bid (2 times a day). 9. docusate sodium 50 mg/5 ml liquid : one hundred (100) mg po bid (2 times a day). 10. lactulose 10 gram/15 ml syrup : thirty (30) ml po q8h (every 8 hours) as needed for until bowel movement. 11. bisacodyl 10 mg suppository : one (1) suppository rectal daily (daily) as needed. 12. insulin nph human recomb 100 unit/ml suspension : five (5) units sc subcutaneous twice a day. 13. insulin regular human 100 unit/ml solution : sliding scale sliding scale injection acmeals & qhs. 14. amoxicillin 125 mg/5 ml suspension for reconstitution : five hundred (500) mg po tid (3 times a day) for 3 days. 15. zocor 20 mg tablet : one (1) tablet po once a day. 16. multivitamin capsule : one (1) capsule po once a day. discharge disposition: extended care facility: - discharge diagnosis: 1)large r temp-parietal ich with ivh 2) pancreatitis 3) thrombophlebitis discharge condition: neurologically impaired but stable, dense l hemiparesis discharge instructions: you have had a large bleed into the right side of your brain. we have been unable to find the underlying cause of this. followup instructions: neurology: , md date/time: 9:00 radiology: phone: provider: scan phone: date/time: 11:30 neurosurgery: provider: , md phone: date/time: 1:30 md, procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube enteral infusion of concentrated nutritional substances percutaneous [endoscopic] gastrostomy [peg] arteriography of cerebral arteries temporary tracheostomy other incision with drainage of skin and subcutaneous tissue other incision of brain diagnoses: urinary tract infection, site not specified unspecified protein-calorie malnutrition subarachnoid hemorrhage intracerebral hemorrhage cerebral artery occlusion, unspecified with cerebral infarction phlebitis and thrombophlebitis of upper extremities, unspecified acute pancreatitis other diseases of respiratory system, not elsewhere classified 

Answer: The patient is high likely exposed to