Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: unresponsive s/p seizure major surgical or invasive procedure: intubation, lumbar puncture history of present illness: this is a 56 year-old male without known past medical history who presents with multiple gtc's today. he has reportedly been experiencing uri symptoms for the past several days, but without documented fever. he had a 1 minute episode of loc today, but then woke up and was back to normal except for a headache. he went to lay down and 1-2 hours later his roomate heard a gurgling noise and went in to find him unresponsive. he then had a gtc of unknown duration and ems was called. while in the ambulance he had an episode of gaze deviation to the right and continued gtc's. on arrival to the emergency room at the osh he had continued gaze deviation and was given 2 mg of ativan. he was then loaded with fosphenytoin for 2 more gtc's. at the osh he had a ct head and torso and was intubated for airway protection. a cbc showed wbc's of 20 with 5% bands. he received ceftriaxone, vacomycin and decadron and transferred here for further management. past medical history: per patient's sister, he had a "large stroke" in with residual r sided weakness. per his ct we can see this was a l mca stroke. his sister reports that he also uses marijuana daily. social history: lives with roommates, unemployed/on disability, smokes marijuana daily family history: no hx of strokes or seizures per pt's sister physical exam: admission physical exam: gen: intubated, sedated on versed and fentanyl heent: mmm, ett in place cvs: s1/s2, no murmur resp: b/l breath sounds abd: soft, non-distended, +bs ms: sedated, no spontaneous movement, gcs 3 cn: pupil pinpoint b/l, face symmetric, +gag tone: increased right lower extremity reflexes: symmetrically brisk, no clonus or crossed adductors strength: no spontaneous movement (per nursing was moving left side before fully sedated) sensation: no withdrawal to deep nailbed pressure in b/l ue and le pertinent results: admission labs: 06:11pm blood wbc-18.4* rbc-5.20 hgb-15.7 hct-45.8 mcv-88 mch-30.2 mchc-34.3 rdw-13.8 plt ct-198 06:11pm blood neuts-87* bands-0 lymphs-3* monos-10 eos-0 baso-0 atyps-0 metas-0 myelos-0 06:11pm blood hypochr-1+ anisocy-1+ poiklo-1+ macrocy-normal microcy-1+ polychr-normal ovalocy-1+ burr-1+ 06:11pm blood pt-11.7 ptt-17.1* inr(pt)-1.0 06:11pm blood glucose-195* urean-18 creat-1.7* na-139 k-3.4 cl-102 hco3-17* angap-23* 11:30pm blood alt-31 ast-29 ld(ldh)-347* alkphos-30* totbili-0.3 06:11pm blood lipase-77* 11:30pm blood ck-mb-4 ctropnt-<0.01 06:11pm blood albumin-4.3 calcium-8.6 phos-2.7 mg-3.0* 11:30pm blood tsh-0.84 06:11pm blood phenyto-13.4 06:11pm blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 06:36pm blood type-art po2-442* pco2-34* ph-7.37 caltco2-20* base xs--4 intubat-intubated 06:16pm blood glucose-173* lactate-5.7* na-138 k-3.4 cl-102 calhco3-22 06:36pm blood freeca-1.16 imaging: cxr : bedside frontal radiograph of the chest: an endotracheal tube terminates appropriately, 5.8 cm above the carina. a nasogastric tube travels into the stomach and beyond the field of view. cardiac, mediastinal, and hilar contours are normal. the lungs reveal mild left basal subsegmental atelectasis and the left costophrenic angle is beyond the field-of-view. there is no pleural effusion or pneumothorax. ct head : impression: 1. no acute intracranial hemorrhage. 2. left parietal and temporal encephalomalacia as above. ct abd : impression: 1. no definite etiology to seizures. 2. secretions in the distal trachea and plugging small left lower lobe airways with mild subsegmental atelectasis, likely related to aspiration in this patient with a mildly distended, fluid-filled esophagus 3. nodular left adrenal gland. this is incompletely characterized and could be correlated to serum adrenal function testing and adrenal-protocol ct or mri, if indicated. ct c-spine : impression: no fracture and normal alignment of the c-spine. mra : impression: sequela of subacute to chronic left mca infarct. no acute infarct seen. mra demonstrates no hemodynamically significant stenosis. brief hospital course: this is a 56 yo man prior l mca territory infarct in , who presents with multiple gtcs with r gaze deviation in the setting of a recent uri. after breaking his seizures with ativan and a phenytoin load, he was intubated for airway protection. his original labs at the osh showed evidence of a new leukocytosis with a bandemia. thus, he was broadly covered with iv antibiotics and acyclovir and transferred to the for a higher level of care. our initial examination of mr. is captured above (see physical examination). he was continued on broad spectrum antibiotics and received a lumbar puncture to investigate for possible cns infections as being a cause for his new onset seizures. the lp was unrevealing, and once all of his cultures and pcrs returned back negative, his antibiotics were discontinued. at this time, he was continued on scheduled dilantin by iv and his levels were maintained in a therapeutic level. during the first two nights of his icu stay, mr. did demonstrate quite a bit of agitation requiring high doses of ativan and antipsychotics to keep him calm. he had even self extubated in spite of bilateral wrist restraints and had to be emergently reintubated. ultimately, his propofol was switched to precedex and he was safely extubated to nc on . while he was quite sleepy and confused during this first day, his mentation improved quickly such that by the next day, we were able to complete a full neurological examination which revealed some mild paraphasic errors and slow speech, but normal and comprehension, impaired short term memory, right sided visual loss, mild right sided pyramidal weakness and a relatively normal gait. on repeat laboratory testing, we observed a new elevation in his liver function tests (particularly ast/alt) which was initially thought to be secondary to dilantin toxicity. ultimately, his transaminitis subsided without manipulation and he was continued on dilantin. during the days prior to his discharge, we set up follow up appointments for the patient to see his pcp and his outside neurologist, dr. . we discovered that he had at least one seizure before and had been initiated on keppra therapy which he had discontinued because of side effects (severe headaches?). he was supposed to switch to lamictal but he never filled that prescription. he was also restarted on his other home medications. we had several conversations with his sisters who we educated about the importance of him remaining on antiepileptic medications. they mentioned to us that in the past he had been driving following his seizures, in spite of a persistent right homonymous hemianopia that was only evident on formal visual field testing. he also complained of some loose stools over the last two days prior to his discharge. he had received broad spectrum antibiotics earlier and out of concern for an antibiotic associated diarrhea, we sent for c diff toxin on his stool which returned negative. it is possible that his loose stools were a consequence of his scheduled colace dosing, which was discontinued on discharge. ultimately, mr. was able to verbalize his understanding of his condition and the importance of taking an antiepileptic medication. we switched his dilantin from tid to qhs dosing and his sister was also informed about the change. he was instructed to switch back to tid dosing should he continue to have difficulties with gi distress. medications on admission: 1. aspirin 325 mg tablet sig: one (1) tablet po daily (daily). 2. omeprazole 20 mg capsule, delayed release(e.c.) sig: one (1) capsule, delayed release(e.c.) po daily (daily). 3. rosuvastatin 20 mg tablet sig: two (2) tablet po daily (daily). 4. lisinopril 20 mg tablet sig: one (1) tablet po daily (daily). discharge medications: 1. aspirin 325 mg tablet sig: one (1) tablet po daily (daily). 2. omeprazole 20 mg capsule, delayed release(e.c.) sig: one (1) capsule, delayed release(e.c.) po daily (daily). 3. rosuvastatin 20 mg tablet sig: two (2) tablet po daily (daily). 4. lisinopril 20 mg tablet sig: one (1) tablet po daily (daily). 5. dilantin extended 100 mg capsule sig: three (3) capsule po at bedtime. disp:*90 capsule(s)* refills:*1* discharge disposition: home discharge diagnosis: seizures cerebrovascular disease hypertension hypercholesterolemia discharge condition: mental status: clear and coherent. level of consciousness: alert and interactive. activity status: ambulatory - independent. discharge instructions: dear mr. , you were seen in the hospital for seizures that caused you to become unresponsive. you were initially intubated to protect your airway, and you were initiated on a medication known as dilantin to help control further seizures. you spent a few nights in the intensive care unit, and once you were stabilized we transferred you to our neurology floors. we believe that these seizures were likely a consequence of your old stroke, and perhaps your recent upper respiratory tract infection. we strongly recommend that you remain on antiepileptic medications. - we would like you to take the following medications daily: dilantin 100mg by mouth thrice daily aspirin 325mg daily lisinopril 20mg daily crestor 40mg daily prilosec 20mg daily - we have set up a follow up appointment for you to see your primary care physician and your neurologist. please make every effort to keep those appointments. - if you experience any of the below listed danger signs, please contact your doctor or go to the nearest emergency room. - it was a pleasure taking care of you on this hospitalization. followup instructions: - please follow up with your primary care provider , 1:40pm ., , ma (fax) - please follow up with your neurologist, md , 845am , , ma procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours spinal tap incision of lung arterial catheterization diagnoses: pure hypercholesterolemia unspecified essential hypertension acute respiratory failure hypotension, unspecified other late effects of cerebrovascular disease grand mal status late effects of cerebrovascular disease, hemiplegia affecting unspecified side diarrhea other malaise and fatigue nonspecific elevation of levels of transaminase or lactic acid dehydrogenase [ldh] other late effects of cerebrovascular disease, facial weakness physical restraints status 

Answer: The patient is high likely exposed to