Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: abdominal pain major surgical or invasive procedure: none history of present illness: 37 yo m w/ history of epilepsy since childhood (gtg), obesity, who presented on to osh, diagnosed with pancreatitis and is transferred to given concern for worsening hemodynamic instability. patient was in usoh until am of , when after eating breakfast, noted rapidly escalating epigastric pain, which eventually radiated to below the umbillicus and down to bilateral flanks. after several episodes of emesis, he drove himself to osh ed for an evaluation. at osh, initial vs were 97.8f 65 151/89 99% ra. he received zofran 12mg, dilaudid 3mg, 1l ns. labs were notable for lipase of > 4000, and thus admitted to the floor for management of pancreatitis. on the floor, he received ns at 1200cc bolus then 150cc/hr x 48hrs. per patient, uop has been declining over the past two days, he has developed difficulty breathing and pleuritic type chest pain. on day of transfer, he developed a fever to 102.6f, hr increased to sustained 110s and rr to 20s on ra. at osh, labs lipase > 4000 -> , amylase 363. ca 9.7 -> 6.5, cr 1.1 -> 1.6, bun 17 -> 36, na 144 -> 138, cl 107 -> 106, hco3 23 -> 23, alb 4.3 -> 2.8, ast/alt 27/12, tg 98, hct 55 -> 48, wbc 14.7 -> 19k. ua sg < 1.005, ket neg, ph 5, neg wbc/rbc. ct abd. was notable for fatty liver, edema at head and uncinate process, no necrosis, peripancreatic and mild bowel wall thickening of 2/3rd portions of duodenum. because of tachycardia, fever and worsening uop, patient was transferred to for further treatment. in icu, initial vs were 97.8f 114 139/68 22 98% 2l nc. patient appeared in nad, he was c/o of abdominal pain, orthopnea, pleuritic cp and concentrated urine. he notes that he consumes only 1-2 drinks per week, denies fhx of pancreatitis, abdominal cancers, gall stones. has not taken any new medications or ingestions. review of systems: (+) per hpi, otherwise negative in detail. past medical history: epilepsy social history: lives on . commercial driver, married, has 2 children. - tobacco - 1/2ppd - etoh - 1/wk - drug use - denies. family history: no malignancy, no etoh abuse cad/cabg in mother in sixties. physical exam: admission exam: general: obese, frustrated appearing man, alert, no acute distress heent: sclera anicteric, obese, dmm, oropharynx clear neck: supple, jvp 7, no lad cv: rr, normal s1 + s2, no murmurs, rubs, gallops lungs: decreased breaths b/l, l > r. abdomen: obese, diffusely tender to palpation in lower quadrants > upper quadrants, bowel sounds present, no organomegaly, no rebound or guarding, but reports some tenderness with passive movement of the bed. gu: no foley ext: warm, well perfused, 2+ pulses, no clubbing, cyanosis or edema neuro: alert, oriented to place and time. moyb intact. eomi, face symmetric, palate elevates symmetrically, tongue is midline. moves all extremities antiresistance. discharge exam: vs: afebrile, normal vitals gen: nad abd: soft, nontender, normal bowel sounds pertinent results: admission labs: 03:57pm blood wbc-16.7* rbc-5.17 hgb-16.4 hct-47.7 mcv-92 mch-31.7 mchc-34.4 rdw-13.4 plt ct-187 03:57pm blood neuts-86* bands-2 lymphs-5* monos-5 eos-1 baso-0 atyps-0 metas-1* myelos-0 03:57pm blood pt-15.9* ptt-34.0 inr(pt)-1.5* 03:57pm blood glucose-93 urean-30* creat-1.4* na-139 k-4.4 cl-102 hco3-18* angap-23* 03:57pm blood alt-20 ast-30 ld(ldh)-437* alkphos-41 totbili-0.8 03:57pm blood lipase-383* 03:57pm blood albumin-3.1* calcium-6.5* phos-1.4* mg-1.8 04:11pm blood type- po2-40* pco2-44 ph-7.35 caltco2-25 base xs--1 04:11pm blood lactate-2.1* discharge labs: imaging: -ruq us (): extremely limited exam. no evidence of cholecystitis or intrahepatic biliary ductal dilatation. no gallstones identified. -cxr (): there are no old films available for comparison. lung volumes are slightly low. there are compressive changes at both bases and small infiltrates can't be excluded. the heart is mildly enlarged. the mediastinal silhouette is slightly prominent, likely due to patient's size and ap technique. the left hemidiaphragm is poorly visualized which could be due to combination of overlying soft tissue, small effusion, volume loss, or small infiltrate. a lateral film would be helpful when the patient is able. ct abdomen ct abdomen: there is fat stranding surrounding the entire pancreas with fluid extending to the anterior pararenal space, lesser sac, transverse mesocolon, and root of the mesentary. there is no venous thrombosis or evidence of pancreatic necrosis. there is bibasilar atelectasis and a small left pleural effusion. the visualized portions of the heart and pericardium are unremarkable. the liver is diffusely hypodense, consistent with fatty deposition. the portal veins are patent. the gallbladder, spleen, and adrenals are normal. the kidneys enhance symmetrically and excrete contrast without evidence of hydronephrosis or mass. there is no nephrolithiasis. several undigested pills are seen in the fundus of the stomach which is otherwise unremarkable. the small bowel is normal. ct pelvis: the appendix is not visualized. the colon, rectum, seminal vesicles, prostate, and urinary bladder are normal. there is no pelvic lymphadenopathy or free fluid. osseous structures: there is no lytic or blastic lesion suspicious for malignancy. impression: 1. findings consistent with acute pancreatitis with peripancreatic effusions but no evidence of pancreatic necrosis or venous thrombosis. 2. fatty liver. 3. bibasilar atelectasis and small left pleural effusion. cxr: cardiomediastinal silhouette and hilar contours are unremarkable. lung volumes are low. an opacity projecting over the left hemithorax can correspond to atelectatic areas seen on ct on . the right lung also has areas of atelectasis, but no focal opacities concerning for an infectious process. no pleural effusion is noted on this radiograph. kub: supine and upright abdominal radiographs demonstrate dilated loops of small bowel measuring up to 5 cm with air-fluid levels on upright imaging. there is residual gas and oral contrast throughout the colon and rectum. there is no evidence of free intraperitoneal air. bibasilar atelectasis is again noted. the osseous structures are unremarkable. impression: likely ileus. brief hospital course: 37 m with history of epilepsy since childhood, obesity, who presented on to osh, diagnosed with acute pancreatitis of unclear etiology and was transferred to given concern for worsening hemodynamic instability, and clinically stabilzed. # acute pancreatitis, severe: unclear etiology. pt initially admitted to the micu given his severe pancreatitis and concern for hemodynamic instability. on admission, apache ii score 10 and approximately 10% tbw volume depleted. etiology of pancreatitis is unclear, minimal etoh use, normal tg. no biliary colic on history, and ruq ultrasound negative for stone although it was a limited study. he has not had exposures to common medications that cause pancreatitis, nor has he had hypercalcemia. triglycerides were 131 at admission. patient was repleted with 5 liters lr overnight after admission, but continued to have significant tachycardia. pain was controlled with iv dilaudid. he was initially kept npo and diet was advanced and he tolerated a regular diet as of . gi plans on evaluating his biliary tree and pancreas within 4-6 weeks of discharge. given the fact that he has been a stable dose of lamictal for over a year and it has effectively controlled his seizures, we did not pursue switching this medication. # ileus: acute, found on kub done for leukocytosis and increased abdominal girth. he is passing liquid stool and gas, but appeared bloated. pt kept npo on pm and advanced to clears the following afternoon. ileus resolved on and his diet was slowly advanced. # leukocytosis: wbc was 16 at admission, thought to be pancreatitis. bcx and ucx were sent which showed coag negative staph, so vancomycin which was emperically started for gpcs on the gram stain were stopped. however, his wbc continud to rise to 24 on . he had a clear cxr (other than atelectasis), a negative ua, improving abdominal pain, and an abdominal ct on showing pancreatitis without findings suggestive of acute infection, and negative cdiff on culture. a potential source includes a red and warm area from a prior peripheral iv site in his r antecubital fossa, though there was no streaking, purulence, or large area of cellulitis. repeat cultures were sent on and were no growth to date at the time of discharge. his wbc on discharge was 20.5 # anion gap acidosis. resolved after admission with aggressive fluid resuscitation. likely related to the mild lactate elevation at admission, this is further supported by the fact that his acidosis improved with volume resuscitation. # : cr at admission was 1.4 and improved to baseline of 0.9-1.0 after fluid resuscitation. likely pre-renal azotemia in the setting of volume depletion from pancreatitis with significant third spacing of fluid. # epilepsy: no seizures this admission, continued on home lamictal. # coagulopathy: inr 1.5 at admission and he is not anticoagulated. thought to be due to recent poor po intake. no evidence of bleeding on exam. # transitional issues: gi outpatient follow-up mrcp repeat cbc to make sure wbc normalizes follow-up final blood culture results medications on admission: home: -lamictal 300mg on transfer: - lamictal 300mg po bid - dilaudid 1-2mg q1h prn - zofran 4mg q6h prn - tylenol discharge medications: 1. lamotrigine 100 mg tablet sig: three (3) tablet po bid (2 times a day). discharge disposition: home discharge diagnosis: pancreatitis, acute ileus hypokalemia acute renal failure epilepsy leukocytosis bacteremia (coag neg staph) discharge condition: mental status: clear and coherent. level of consciousness: alert and interactive. activity status: ambulatory - independent. discharge instructions: you were hospitalized for pancreatitis. you were also found to have ileus. please avoid alcohol, fatty foods. please continue to take all of your medications. followup instructions: name: , g address: , , phone: appointment: thursday 10:00am 03:45p , ( gi) (, ma), gi procedure: enteral infusion of concentrated nutritional substances diagnoses: acidosis abnormal coagulation profile acute kidney failure, unspecified hypopotassemia constipation, unspecified bacteremia paralytic ileus obesity, unspecified staphylococcus infection in conditions classified elsewhere and of unspecified site, other staphylococcus acute pancreatitis leukocytosis, unspecified other respiratory abnormalities other and unspecified anticonvulsants causing adverse effects in therapeutic use other fluid overload generalized convulsive epilepsy, without mention of intractable epilepsy 

Answer: The patient is high likely exposed to