Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: codeine / vitamin k / tagamet attending: chief complaint: found down major surgical or invasive procedure: mri/a brain history of present illness: 71 yo woman with a h/o htn, who was transferred from osh to after being found unresponsive. her husband relates the story. she was sleeping soundly at 7am on saturday when the husband left to run some errands. he returned home and found her lying on her left side beside her bed at 2:30pm. no emesis, stool incontinence or tongue biting, but she was drooling and incontinent from urine. she was unresponsive, an abrasion was on her left cheek. at osh she was occasionally responding to commands, given narcan without response. ct scan at osh showed hypodensity of the cerebral and vague hypodensities in the anterior aspects of the thalami. no hemorrhage. (all per report, no scans to assess for myself). upon admission to the ed she was completely obtunded, given narcan with minimal reponse. admitted to the icu. in the icu, she was given asa pr, extubated on , found to have a lll infiltrate on cxr with febrile to 102, started on levofloxacin . per the husband, she is active at baseline walking unaided. no recent illnesses, no complaints such as recent ha, tinnitus, diplopia, fevers/chills, cp, sob, abd pain, diarrhea, constipation, brbpr. she has low back pain x "many years" and takes darvocet prn. past medical history: 1. htn 2. low back pain - per husband, has had mri in the past that only showed osteoporosis/arthritis, takes darvocet and relafen, scoliosis 3. s/p ccy 25 ago 4. s/p rupture of the esophagus after dilation therapy for stricture in , resulting in 35 day hospitalization course 5. ovarian cyst removal 6. hysterectomy at age 28, cervical cancer 7. h/o ulcers several years ago, this is why she is not takin aspirin social history: no tob/etoh/drugs, lives with husband of 51 , walks unaided, home maker her whole life. 2 healthy daughters and 4 healthy grandsons. family history: dad passed away at age 76 of heart disease, infection. mom passed away at age 54 of a tumor or brain. no h/o stroke in the family. physical exam: upon transfer to the floor: mental status: hypersolmnolent, not following commands or answering questions, not opening eyes to voice or pain. cn: eyes midline but unable to assess doll's head with c-collar in place, pupils equal 2mm->1.5mm bilaterally, consenual to light, no rapd, wrinkles forehead symmetrically with pain, + corneal reflex bilaterally, + gag with suction motor: unable to assess at this time. dtr: 1 throughout, no clonus, no snout, upgoing toe on right, downgoing toe on left : purposefully responds to painful stimuli (reaches for her sternum with rub, purposeful movements in all 4 limbs to pain. & gait: unable to assess upon discharge to rehab: ms: poor memory, not remembering husbands name or why she's here. hypersolmnolent but less so that originally. following commands. cn: left face droop motor: no clear weakness, lifts all extrem to gravity, ambulates with pt pertinent results: mri/a: bilateral thalamic lesions, most likely acute, posterior paramedian thalamic artery distribution infarctions. diffuse imaging could not be performed on this instrument. carotid us: absence of significant plaque and carotid artery stenosis. antegrade flow in the bilateral vertebral arteries. cxr: possibly lll infiltrate. tte: no atrial septal defect is seen by 2d or color doppler. there is mild symmetric left ventricular hypertrophy. the left ventricular cavity size is normal. overall left ventricular systolic function is normal (lvef 70%). right ventricular chamber size and free wall motion are normal. the aortic valve leaflets (3) are mildly thickened but not stenotic. no aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. there is no mitral valve prolapse. there is borderline pulmonary artery systolic hypertension. there is no pericardial effusion. ngt placement: successful placement of an 8 french feeding tube with distal tip in the proximal jejunum. 8:10 pm blood culture site: arm **final report ** aerobic bottle (final ): reported by phone to , -fa5- @ 20:50 . staphylococcus, coagulase negative. of two colonial morphologies. isolated from one set only sensitivities performed on request.. anaerobic bottle (final ): no growth. blood cx from (2/2 bottles) and (4/4 bottles): no growth 08:10pm glucose-133* urea n-15 creat-0.7 sodium-143 potassium-4.3 chloride-107 total co2-24 anion gap-16 08:10pm ast(sgot)-23 ck(cpk)-97 alk phos-64 08:10pm ctropnt-<0.01 08:10pm ck-mb-notdone 08:10pm calcium-9.5 phosphate-3.7 magnesium-1.7 08:10pm asa-neg ethanol-neg acetmnphn-neg bnzodzpn-neg barbitrt-neg tricyclic-neg 08:10pm urine color-straw appear-clear sp -1.007 08:10pm urine blood-lg nitrite-neg protein-neg glucose-neg ketone-neg bilirubin-neg urobilngn-neg ph-5.0 leuk-neg 08:10pm urine rbc-0-2 wbc-0 bacteria-none yeast-none epi-0 hct : 36 chol 200, ldl 79, hdl 92, tg 145 brief hospital course: 1. cerebral infarction of the bilateral thalami - she was started on a statin, modafinil to decrease solmnolence, and aggrenox for antiplatelet therapy. carotid us and tte were unrevealing re: a source of embolus. antihypertensives were initially held. later, sbp rose to the 180's so ramipril was started. this drug has been shown to be beneficial in stroke patients. her glucose was well controlled. she worked well with pt/ot. 2. left lower lobe pneumonia, febrile to 102 - she completed a 7 day course of levofloxacin and was afebrile for the remainder of the course. 3. bacteremia - she grew one out of two positive blood cultures on , gpc prs and clusters. started on vancomycin until specificities returned. found to be coag neg staph aureus, likely a contaminant. subsequent blood cultures (6 bottles) were all no growth. 4. normocytic anemia: iron studies consistent with iron deficiency anemia, all stools were guaiac negative. she was transfused a total of 2 u prbcs ( - hct bumped from 26->31, and for hct 27->35). she should recieve an outpatient colonoscopy at some point. fen: she eventually passed a video swallow soft foods, thickened liquids, however her dobhoff tube should remain in place for additional nutritional needs. ppx: sc heparin for dvt proph, ppi given h/o ulcers on aspirin full code medications on admission: 1. relafen 500mg qd (nsaid) 2. amitriptyline 25mg qd 3. prinovil 5mg qd 4. hctz 25mg qd 5. evista 60 mg 6. darvocet 100/650, takes 4-6 tabs qd, for low back pain discharge medications: 1. heparin sodium (porcine) 5,000 unit/ml solution sig: one (1) 5000 units injection tid (3 times a day): for dvt prophylaxis. 2. acetaminophen 650 mg suppository sig: one (1) suppository rectal q4-6h (every 4 to 6 hours) as needed: may give po in custard. 3. modafinil 100 mg tablet sig: one (1) tablet po qam () as needed for bilateral thalamic infarcts. 4. lansoprazole 30 mg capsule, delayed release(e.c.) sig: one (1) capsule, delayed release(e.c.) po qd (once a day). 5. ramipril 1.25 mg capsule sig: one (1) capsule po bid (2 times a day): hold for sbp<130, hr<55. 6. dipyridamole-aspirin 200-25 mg capsule, multiphasic release sig: one (1) cap po bid (2 times a day). 7. atorvastatin calcium 10 mg tablet sig: one (1) tablet po qd (once a day). discharge disposition: extended care facility: st. hospital rehab discharge diagnosis: bilateral thalamic strokes of unclear etiology. left lower lobe pneumonia s/p 7 days of levofloxacin bacteremia, likely contaminant iron deficiency anemia discharge condition: improved - able to swallow (see swallow eval) but getting supplemental tube feeds, ambulates with pt. discharge instructions: please take all medications. please return to the ed if you experience weakness, numbness, or other concerning symptoms. followup instructions: please call the stroke clinic when you leave rehab for a followup appointment. . please call your pcp for followup appointment after you leave rehab. . you should have an outpatient colonoscopy at some point to evaluate for colon cancer given your iron deficiency anemia. procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube enteral infusion of concentrated nutritional substances arteriography of cerebral arteries transfusion of packed cells magnetic resonance imaging of brain and brain stem diagnoses: unspecified essential hypertension personal history of malignant neoplasm of other parts of uterus pneumonitis due to inhalation of food or vomitus iron deficiency anemia, unspecified cerebral artery occlusion, unspecified with cerebral infarction lumbosacral spondylosis without myelopathy facial weakness memory loss 

Answer: The patient is high likely exposed to