Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: hypoxia major surgical or invasive procedure: intubation history of present illness: 57m with ild (?nsip) on prednisone 20 mg daily, afib s/p recent ablation, cad, osa; transfer from osh after admission for dyspnea, hypoxia, and hemoptysis. recent admit to osh from to for tee and afib catheter ablation. discharged on coumadin and lovenox until inr >2. prior to discharge, had small volume hemoptysis x few episodes, attributed to intubation for procedure. on the day following discharge, patient presented to in with c/o dyspnea and more episodes of small volume hemoptysis. cta was performed and was negative for pe. transferred to hospital (had ablation there). ct reviewed and thought to have diffuse ground glass opacities. admitted to the medicine floor, continued on o2 by simple fm with gradual increase up to 10 l/min. continued having hemoptysis. given several doses of lasix. on steroids increased from home 20 mg po pred to solumedrol 80 mg iv q8h. abg today 7.41/37/73 on 10 l fm. transferred to given that his pulmonologist () located here. . currently continues to have both hemoptysis and dyspnea. thinks hemoptysis may be slightly improved, but dyspnea overall worsening. other ros: + epistaxis on day of osh admission, + chills. negative for fever, ha, vision changes, cp, abdominal pain, diarrhea, constipation, vomiting/nausea, dysuria, bleeding from other sites, weight gain, change in baseline le edema. past medical history: - cad s/p bms to lad in , subsequent caths without significant obstructive disease - ild (early ipf vs. nsip) - afib s/p ablation/pvi x 2, first and second . previously dofetilide (not tolerated due to side effects); currently on sotalol. - mild pulmonary hypertension (pap 38/19 seen on past rhc; no pa htn on cpet ) - obesity - osh note of "pfo with shunting" - sleep apnea (intolerant of cpap) - type ii dm - nafld - dyslipidemia - htn - bilateral torn rotator cuffs - bph - gerd c/b barrett's esophagus - anxiety - severe spinal stenosis - s/p ccy - s/p multiple back surgeries (for disc herniation) - s/p hernia repair social history: social history is significant for the absence of current tobacco use. 50 pk year history of smoking. prior h/o etoh abuse - 7 years ago cut down significantly now occasional etoh use. last drink was 2 weeks ago. married w/ 2 children, on disability due to back problems. ambulates with crutches at baseline family history: father w/ mi in 50s or 60s, had a cabg. mother: type diabetes and hypertension. physical exam: on transfer from the icu to the medicine service: . t:96.0 hr: 75 bp: 96/47 02: 96% on 5l and 90% on 5l while getting up to bathroom. gen: obese male, nad heent: perrl, eomi neck: full lungs: decreased breath sounds in lower of lungs l>r, mild rhonchi. abd: soft, non-tender, obese ext: no edema, dp pulses 2+ pertinent results: admission labs: 01:30am blood wbc-6.8 rbc-2.97*# hgb-10.0*# hct-28.8*# mcv-97 mch-33.7* mchc-34.7 rdw-15.6* plt ct-237 01:30am blood pt-20.6* ptt-38.4* inr(pt)-1.9* 01:30am blood glucose-202* urean-23* creat-0.9 na-135 k-4.4 cl-100 hco3-21* angap-18 cxr: there is probably a region of consolidation in the right suprahilar lung, new since prior study. conventional radiographs would be more definitive. heart size is mildly enlarged, augmented substantially by mediastinal fat, also responsible in part for widening of the mediastinum in the right paratracheal region and at the thoracic inlet. pleural effusion, if any, is minimal. the upper esophagus is mildly distended with air. ct chest: newly developed areas of ground-glass opacities in addition to areas of chronic subpleural minimal interstitial chronic changes. the chronic interstitial abnormalities are most likely consistent with nsip. the rapid development in last two weeks of the diffuse homogeneous ground glass opacities in combination with severe hypoxia and radiological appearance of the findings are highly concerning for several possibilities: acute exacerbation of interstitial lung disease, infection (for example opportunistic infection), acute interstitial pneumonia, hypersensitivity reaction to a new drug/ and less likely hemorrhage. bal: rare atypical group of squamous cells. background pulmonary macrophages, neutrophils, and red blood cells. brief hospital course: 57m with ild on prednisone, afib s/p recent ablation, cad; transfer from osh with dyspnea, hypoxia, and hemoptysis with high o2 requirements. . # dyspnea/hypoxia: this patient had multiple potential causes of dyspnea including ild/nsip, potential cardiac causes (chf, cad, afib s/p recent ablation - potential for pulmonary vein stenosis, ?history of pfo - potential for shunt), and pulmonary artery hypertension. he was also thought to have component of chronic dyspnea unrelated to ild, with a significant hyperventilatory response to exercise. pulmonary vein stenosis was considered in light of recent ablation, but thought by cardiology to be unlikely after imaging pre and post ablation was reviewed with radiology. instead, they felt that pulmonary contusion as sequelae of ablation was possible explanation. . on , he began having tachypnea in the evening once bipap mask applied, which progressed to tachypnea to the 50s with oxygen desats. he underwent urgent intubation after discussion with him and his wife. then required high level of sedation and airway pressure release ventilation (aprv) mode prior to getting oxygen sats from mid-80s post intubation back to 90. he was subsequently very agitated, requiring extra sedation. he underwent bronchoscopy showing diffuse alveolar hemorrhage. infectious workup from bal was negative for pcp, , afb, legionella. vasculitis was a potential cause, and he was started on solumedrol 1g q24h. he was extubated successfully shortly thereafter. an attempt was made to diurese him, but this was limited by blood pressure. on , due to low-grade fever and persistent copious thick secretions was started on on empiric vanc and zosyn. mini-bal culturs were negative. he continued to desaturate with movement to and from chair, but was otherwise with o2 sats low 90s on 5-6l nc. given negative blood and sputum cultures, antibiotics were stopped. he was called out of the icu to the general medicine wards. he transitioned from iv steroids to an oral prednisone taper. (60 mg x 10 days followed by 40 mg for a total of 7 days, to be followed as an outpatient by 20 mg daily for 7 days, and then 15 mg ongoing.) also, cyclophosphamide therapy was initiated (250 mg daily for 7 days followed by 150 mg ongoing). there, he continued to desaturate with exertion and intermittently at night. bipap was placed, but the patient found it uncomfortable and didn't wear it. . liver function tests were trended while on cyclophosphamide. prior to discharge, transaminases were mildly but stably elevated, and tbili rose to 1.7. these should be trended every 3 days, and the dose of cyclophosphamide reduced if needed. . prior to discharge, the patient was requiring 50% venti mask to maintain o2 sat ~94%, with a respiratory rate in the mid 20s. he would desaturate with exertion to the low 80s, with respiratory rate increasing to ~35. the patient and his family understand that this will continue to improve as his alveolar hemorrhage resolves and the steroids and cyclophosphamide exert their effect on his underlying lung disease. he will follow up with his pulmonologist in 2 weeks. . # hemoptysis: on coumadin for afib with recent ablation. differential for bleeding initially included bronchitis, airway trauma from intubation, pneumonia (including bacterial, pcp, , or other atypical), neoplasm, bronchiectasis, ?pneumonitis exacerbation. bronchoscopy demonstrated diffuse alveolar hemorrhage. all anticoagulation was held. . # atrial fibrillation: he was s/p recent ablation and now in sinus rhythm. on anticoagulation at admission, which was stopped when alveolar hemorrhage found. sotalol was continued. atenolol was stopped because of borderline blood pressures. . # cad: he was ruled out for mi by enzymes on admission. asa, statin, beta blocker, and ace were continued. . # htn: continue home regimen. . # dm: fsbg were elevated in the setting of high-dose steroids. he was initially covered with sliding scale insulin. given persistently high , long-acting insulin was added. after transitioning to oral steroids and being called out to the floor, the long-acting insulin was stopped and metformin restarted. his fingersticks were ~150 on this regimen. . the patient remained full code throughout this admission. . medications on admission: meds on transfer: lisinopril 5 mg po daily acetaminophen 325-650 mg po q6h:prn multiple vitamins liq. 5 ml po daily atorvastatin 80 mg po daily nystatin oral suspension 5 ml po qid:prn thrush bisacodyl 10 mg po/pr daily:prn constipation ondansetron 4 mg iv q8h:prn nausea calcium carbonate 1000 mg po daily paroxetine 40 mg po daily colchicine 0.6 mg po daily piperacillin-tazobactam na 4.5 g iv q8h cyanocobalamin 1000 mcg po daily prednisone 60 mg po daily order date: @ 1009 cyclophosphamide 250 mg po daily ropinirole 0.25 mg po hs docusate sodium (liquid) 100 mg po bid senna 2 tab po bid haloperidol 2 mg po tid:prn agitation heparin 7500 unit sc tid sotalol 160 mg po bid ibuprofen 400-800 mg po q8h:prn sulfameth/trimethoprim ds 1 tab po 3x/week (mo,we,fr) insulin sc (per insulin flowsheet) vancomycin 1000 mg iv q 12h lansoprazole oral disintegrating tab 30 mg po daily vitamin d 800 unit po daily lactulose 30 ml po q8h:prn constipation . medications at home: atenolol 50 mg tablet daily atorvastatin 80 mg daily celecoxib 200 mg once a day colchicine 0.6 mg tablet daily fexofenadine 180 mg daily lisinopril 5 mg tablet - daily metformin 1000 mg qam, 500 mg qpm omeprazole 20 mg twice a day paroxetine hcl 50 mg daily prednisone 20 mg tablet daily (recently decreased from 30 mg daily) propoxyphene n-acetaminophen 100 mg-650 mg q4h prn pain ropinirole 0.25 mg at night sotalol 160 mg tamsulosin 0.4 mg daily trimethoprim-sulfamethoxazole - 800 mg-160 mg three times a week warfarin lovenox 140 aspirin 81 mg daily calcium 500 + d cyanocobalamin 1,000 mcg daily multivitamin daily discharge disposition: extended care facility: & rehab center - discharge diagnosis: primary: diffuse alveolar hemorrhage, interstitial lung disease secondary: type 2 diabetes mellitus, coronary artery disease, anemia discharge condition: stable, on 50% venti mask with o2 sats in the mid 90s falling to mid 80s with exertion discharge instructions: you were transferred to because of trouble breathing. this was due to bleeding in your lungs. you were intubated in the intensive care unit, but after that your oxygen saturation improved and you came to the regular hospital floor where you had physical therapy and monitoriing. the following medications were changed: cyclophosphamide was added prednisone was changed atenolol was stopped celecoxib was stopped fexofenadine was stopped warfarin was stopped lovenox was stopped please call your doctor or return to the emergency department for worsening shortness of breath, chest pain, fevers and chills, or other symptoms that are concerning to you. followup instructions: please follow up with dr. as below: provider: function lab phone: date/time: 9:30 provider: ,interpret w/lab no check-in intepretation billing date/time: 9:30 provider: . phone: date/time: 10:00 procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more diagnostic ultrasound of heart insertion of endotracheal tube non-invasive mechanical ventilation arterial catheterization laryngoscopy and other tracheoscopy closed [endoscopic] biopsy of bronchus diagnoses: other iatrogenic hypotension obstructive sleep apnea (adult)(pediatric) coronary atherosclerosis of native coronary artery adrenal cortical steroids causing adverse effects in therapeutic use acute posthemorrhagic anemia diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled unspecified acquired hypothyroidism atrial fibrillation personal history of tobacco use hypertrophy (benign) of prostate without urinary obstruction and other lower urinary tract symptom (luts) percutaneous transluminal coronary angioplasty status candidiasis of mouth other and unspecified hyperlipidemia anxiety state, unspecified acute respiratory failure postinflammatory pulmonary fibrosis long-term (current) use of anticoagulants spinal stenosis, unspecified region barrett's esophagus other chronic nonalcoholic liver disease other specified diseases of pulmonary circulation 

Answer: The patient is high likely exposed to