Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: alcohol withdrawal major surgical or invasive procedure: none history of present illness: pt is a 47 yo chronic alcoholic w/ multiple previous ed visits and admissions for alcohol withdrawal and acute on chronic pancreatitis, most recently in . pt was brought to ed via ambulance for symptoms of alcohol withdrawal. pt states that he has been drinking ~1 quart of liquor daily, but that he has been trying to cut back. he states his last drink was 1 glass of brandy at noon on . pt states that he has been feeling tired for "a while" and was very anxious. per his most recent discharge summary from , he is a type 2 diabetic on metformin and glypizide, but he has not been taking any of his medications. pt only has mild abdominal pain, and vomited 1 x "for a few minutes" yesterday and had 1 x diarrhea. pt is currently a&o x 3, and states that he has never had any withdrawal seizures, but "had dts". denies any fevers, chills, sob, chest pain, or urinary symptoms. . in the ed inital vitals were, 96.7f, bp 180/78, hr 125, rr 20, sat 100% ra. fs glucose 487. pt was started on an insulin drip, given thiamine 100mg iv, 1l ns bolus, and given lorazepam 2mg iv x 2. pt also received morphine 4mg iv x 1 and ondansetron 4mg iv x 1 before being admitted to fiuc. serum alcohol level was 358, lipase 3131, serum sodium 126, and wbc 14.1. anion gap 42. . on arrival to the icu, pt's vital signs were: hr 130, bp 178/82, rr 18, temp 36.2, sat 100%. . pt currently appears to be intoxicated. pt is a&o x 3. reports mild epigastric pain. past medical history: anxiety diabetes, type 2, on oral medications, poorly controlled alcohol abuse hypertension hyperlipidemia chronic pancreatitis social history: patient states he owns business doing house repairs. he has had difficulty with etoh in the past and has attended aa, but not recently. he lives with his wife and has custody of his 3 grandchirlren, 10, 7,5 years old. he smokes 3 cigs/day. no illicits. family history: alcoholism. dm/htn/hl run in the family. physical exam: admission physical: vitals: t: 36.2c bp: 160/91 p: 122 r: 18 o2: 100% ra. general: sluggish responses, but a&o x 3. appears intoxicated. heent: sclera anicteric, mmm, poor dentition, perrl, eomi, cn2-12 intact. neck: supple, jvp not elevated, no lad lungs: clear to auscultation bilaterally, no wheezes, rales, ronchi cv: tachycardic rate, regular rhythm, normal s1 + s2, no murmurs, rubs, gallops abdomen: soft, mild epigastric tenderness, non-distended, bowel sounds present, no rebound or guarding, no organomegaly gu: no foley : warm, well perfused, 2+ pulses, no clubbing, cyanosis or edema neuro: cn2-12 intact, grossly 5/5 strength in upper and lower extremities bilaterally, grossly normal sensation throughout. . discharge physical exam: bp 126/83, hr 100, rr 20, 100% ra in nad op moist lungs cta b bilateral antecubital fossae contact dermatitis without edema pertinent results: admission labs: 05:30am blood wbc-11.2* rbc-4.05* hgb-11.7* hct-35.6* mcv-88 mch-29.0 mchc-33.0 rdw-13.8 plt ct-247 01:30am blood neuts-88.8* lymphs-7.8* monos-3.0 eos-0.2 baso-0.2 01:30am blood glucose-400* urean-9 creat-1.1 na-126* k-4.6 cl-83* hco3-6* angap-42* 01:30am blood alt-65* ast-89* totbili-0.5 10:47pm blood ethanol-neg 01:30am blood asa-neg ethanol-358* acetmnp-10 bnzodzp-neg barbitr-neg tricycl-neg 05:30am blood triglyc-664* 05:30am blood pt-10.9 inr(pt)-1.0 cardiac enzymes negative x 3 abg: 04:18pm blood type-art temp-37.6 po2-31* pco2-32* ph-7.43 caltco2-22 base xs--2 intubat-not intuba . lipase trend: 05:20am blood lipase-37 05:30am blood lipase-1662* 01:30am blood lipase-3131* . lactate trend: 08:43am blood lactate-0.9 06:17pm blood lactate-1.1 09:30am blood lactate-3.0* 06:25am blood lactate-3.9* . other labs: 03:20pm blood calcium-8.6 phos-2.2* mg-1.9 iron-18* 03:20pm blood caltibc-218* ferritn-886* trf-168* 08:49am blood %hba1c-11.5* eag-283* tsh 1.1 micro: urine culture (final ): mixed bacterial flora ( >= 3 colony types), consistent with skin and/or genital contamination. blood culture and : pending . ekg : sinus tachycardia. otherwise, normal tracing. compared to the previous tracing of t wave abnormalities are no longer present. . studies: cxr : chest, single ap portable view. slightly rotated positioning and low lung volumes. the heart is not enlarged. the aorta is slightly tortuous. no chf, focal infiltrate, or effusion is detected. minimal right basilar atelectasis is present. cxr : lung volumes are quite low. tip of the left pic line is difficult to see because of cardiac motion, approximately 3 cm below the estimated location of the superior cavoatrial junction. mild cardiomegaly has increased since , but there is no pulmonary edema or particular mediastinal venous engorgement. small left pleural effusion may be present. . discharge labs: 09:05am blood wbc-5.3 rbc-3.31* hgb-9.5* hct-28.8* mcv-87 mch-28.6 mchc-32.9 rdw-14.7 plt ct-342# 09:05am blood glucose-219* urean-4* creat-0.8 na-134 k-4.1 cl-102 hco3-23 angap-13 09:05am blood calcium-8.5 phos-2.1* mg-1.6 brief hospital course: brief course: pt is a 47 yo chronic alcoholic w/ multiple previous ed visits and admissions for alcohol withdrawal and acute on chronic pancreatitis, now admitted for alcohol withdrawal, dka, and acute on chronic pancreatitis. he was initially admitted to the icu for insulin gtt, and close monitoring. once his ag was closed and started on sc insulin, he was transitioned to the medical floor on . active issues: # hyperglycemia/dka: aic 11.5%. pt had serum glucose 400, hco3 6, and anion gap of 42 on presentation. though he is a type 2 diabetic, given high anion gap, ketones in the urine, pt thought to have dka +/- non-ketotic hyperosmolar state (can have overlap). precipitating factors thought to be alcohol use and non-compliance on home oral diabetic medications. he was started on insulin gtt and monitored in the icu. his anion gap initially closed, and was started on lantus. however, his anion gap re-opened and he was restarted on the insulin gtt. was consulted and recommended starting lantus 20 units daily in addition to the drip. once his ag closed, he was transitioned to sliding scale insulin and glargine. on the medical floor, pt expressed that he only wanted to do a regimen. therefore, recommended 15units 75/25. pt received diabetic teaching, nutritional support, and social work consult during his admission. he wishes to follow up at clinic after discharge and an appointment has been made for him. see below. ace inhibitor treatment is recommended. . # mixed ag and non-ag acidosis: ag acidosis attributed to dka and possible alcoholic ketoacidosis and lactic acidosis as above. he also developed a non-ag acidosis, attributed to aggressive normal saline resuscitation, which resolved once eating and ns stopped. . # alcohol abuse: pt was tachycardic, hypertensive, and agitated, 17 on ciwa scale upon arrival to . he was initially given iv lorazepam, but on hospital day 1, was no longer . he was transitioned to po lorazepam prn. he was seen by social work. he did not display any signs of withdrawal on the regular medical floor. pt reported that he was done with drinking due to its many complications. he was seen by sw to provide him with further resources. pt reported that anxiety was a significant contributor to his drinking and expressed interest in starting ssri therapy in the outpatient setting. . # acute on chronic pancreatitis: pt had mild abdominal discomfort on presentation with elevated lipase to 3131 on admission, likely caused by alcohol intake. he was also found to have elevated triglycerides. his abdominal pain resolved and lipase had trended down to 1662. he was advanced to a regular diet, with some diarrhea that improved. lipase normalized and pt was able to tolerate a regular diet without complications. would consider outpt gi follow up.. # tachycardia: in the icu, pt was tachycardic, possibly alcohol withdrawal initially, though continued to be tachycardic when out of range of withdrawal. dka likely initially as well given profoundly hypovolemic from osmotic diuresis. pt had low-grade temps, but no fevers, and no sources of infection. he was comfortable in no pain. his hr trended downward. pe was considered, but pt had no desats in oxygenation and no chest pain, making this unlikely. this improved on the medical floor. would consider outpatient echo to evaluate for etoh induced cardiomyopathy. tsh was normal. . #normocytic anemia: hct has fallen from 39 on admission to 27, 32.1 on day of dc. unclear what hct is at baseline as pt frequently presents with intoxication and hemoconcentrated hct. mcv chronically low-normal. pt denies melena, hematemesis, coffee ground emesis. iron studies c/w chronic disease. stools ordered for guaiac. this can be further monitored in the outpatient setting. . inactive issues: # hypertension: unclear baseline but per prior dc summaries, generally sbp 140s-150s. pt was previously on lisinopril 10mg po daily per report, but patient was not taking this medication recently. . # hyperlipidemia: held statin given initially elevated lft's. . transitional care: 1. f/u for continued reinforcement of dm regimen and teaching 2.consideration of ssri for anxiety with psychotherapy 3.outpatient sw for etoh/anxiety if pt willing 4.consideration of echo for what appears to be chronic, asymptomatic tachycardic 5.reinitiation of acei, statin, ?starting of asa for dm. deferred on starting at time of discharge given concern for repeat etoh abuse and recent acute illness. medications on admission: home medications (per dc summary, pt states he's not taking): - metformin - glipizide - pioglitazone - lisinopril - simvastatin discharge medications: 1. humalog mix 75-25 100 unit/ml (75-25) suspension sig: fifteen (15) u subcutaneous twice a day. disp:*1 month's supply* refills:*1* 2. folic acid 1 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 3. thiamine hcl 100 mg tablet sig: one (1) tablet po once a day. disp:*30 tablet(s)* refills:*2* 4. other you reported that you were on lisinopril and simvastatin some time ago. please discuss with your pcp and whether you should restart these medications 5. cortisone 1 % cream sig: one (1) appl topical qid (4 times a day) for 5 days: to antecubital fossa. disp:*1 tube* refills:*0* 6. insulin syringe 1 ml 29 x syringe sig: one (1) syringe miscellaneous twice a day: use as directed for insulin injection. disp:*1 month's supply* refills:*1* discharge disposition: home discharge diagnosis: alcohol intoxication alcoholic acute pancreatitis diabetes, type ii metabolic acidosis discharge condition: mental status: clear and coherent. level of consciousness: alert and interactive. activity status: ambulatory - independent. discharge instructions: you were admitted with alcohol intoxication, abdominal pain that was found to be due to pancreatitis related to alcohol use and high blood sugars. because of your high blood sugar and dehydration on admission, you were initially admitted to the medical icu. you improved on insulin and with iv fluids. . it is very important that you receive treatment for your anxiety and stop drinking as this could be very dangerous to your health. it is also important that you take your insulin as directed and follow a diabetic diet. you are being discharged on new medications: you are starting insulin therapy and you expressed understanding of your regimen. 75/25 mg twice a day, 15 units. followup instructions: diabetes follow up: , np at 8:30am 1 pl , ( name: , md specialty: internal medicine when: thursday at 10:20am location: medical ctr-general medical assoc. address: , , phone: procedure: central venous catheter placement with guidance diagnoses: acidosis anemia, unspecified tobacco use disorder unspecified essential hypertension hyposmolality and/or hyponatremia other and unspecified hyperlipidemia chronic pancreatitis acute pancreatitis other and unspecified alcohol dependence, continuous alcohol withdrawal diabetes with ketoacidosis, type ii or unspecified type, uncontrolled 

Answer: The patient is high likely exposed to