Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: chest pain major surgical or invasive procedure: percutaneous coronary intervention with bare metal stent insertion x2 to proximal and distal rca history of present illness: the patient is a 64 yo man with h/o hyperlipidemia and chronic low back pain who presented with chest pain. mr. was in his normal state of health until 3 am on the morning of his admission () when he awoke to urinate. on the way back to bed, he experienced burning substernal chest pain, radiating to his left jaw and mid sternum. he felt associated diaphoresis and shortness of breath but decided to go back to bed. he tried finding a comfortable position to relieve the pain but it was in vain. he did not experience nausea and vomiting but instead felt a sensation of 'belching'. when he got out of bed a few hours later for work, he felt weak, lightheaded and had some blurring of vision. he drove himself to for further evaluation. on the way from the parking lot to the ed, he felt increasingly weak, had to rest on a bench and had to be brought into the ed in a wheelchair. he felt more weakness than pain in the ed. . he stopped taking his aspirin on 4 days prior to admission () as he was advised to do so in order to go for a scheduled ct myelogram. he did not try any pain relief medications (especially aspirin) when he felt the chest pain as he felt he shouldn't compromise his chances on doing the scan. he has had some shortness of breath on exertion for the past 4 weeks but did not think much of it otherwise. . in , his vs were 97.3, p 49, bp 98/65, r 18, and o2 97% on 4l. he was noted to be diaphoretic and pale. ekg showed stes in ii, iii, avf, v3-v6, st depressions in i, avl, and twis in v1,v2. he was given asa, plavix, lipitor and was started on a heparin gtt. he was also given 500 cc ns and his bp incrased to 108. he was then transferred to for pci. . in the cath lab, the patient was found to have a total occlusion of the proximal rca and a distal rca lesion, which were both stented with bms. he then had a right heart catheterization, which showed elevated ra pressures (18) and mildly elevated left-sided pressures. he was then admitted to the ccu for further evaluation given the liklihood of rv infarct. . upon admission to the ccu, mr. was chest-pain free. he otherwise had no acute complaints. . on review of systems, he denied any prior history of stroke, tia, deep venous thrombosis, pulmonary embolism, bleeding at the time of surgery, headaches, myalgias, cough, rhinorrhea, hemoptysis, acid brash, abdominal swelling, black stools or red stools. he denies recent fevers, chills or rigors. he denies exertional buttock or calf pain. all of the other review of systems were negative. . cardiac review of systems was notable for absence of paroxysmal nocturnal dyspnea, orthopnea, ankle edema, palpitations, syncope or presyncope. past medical history: 1. hyperlipidemia 2. bph 3. chronic low back pain 4. tonsillectomy (age 7yo) 5. appendectomy (age 5yo) social history: the patient lives at home alone. he currently works as an it consultant for various hospitals. he is independent in his activities of daily living. he quit smoking 35 years ago (previous smoker of 51 pack years, 3 packs/day from 19-35yo), and he currently drinks one glass of wine 5 nights/week. his wife passed away two years ago. denies drug use. family history: his mother passed away at the age of 63 from pancreatic cancer. his father passed away from complications of parkinson's disease at the age of 72. his brothers and sisters are all alive and well. no family history of early mi, arrhythmia, cardiomyopathies, sudden cardiac death or strokes. physical exam: physical examination on admission: . vs: afebrile , bp 135/94, hr 95, rr 14, o2 sat 100% on 4l general: pleasant man in nad. oriented x3. mood, affect appropriate. heent: perrl, eomi. conjunctiva were pink, no pallor or cyanosis of the oral mucosa. no xanthalesma. neck: supple with jvp of 8 cm. cardiac: pmi located in 5th intercostal space, midclavicular line. rr, normal s1, loud s2. no m/r/g. no thrills, lifts. no s3 or s4. lungs: no chest wall deformities, scoliosis or kyphosis. resp were unlabored, no accessory muscle use. cta anteriorly, no crackles, wheezes or rhonchi. abdomen: soft, ntnd. no hsm or tenderness. abd aorta not enlarged by palpation. no abdominial bruits. extremities: no c/c/e. no femoral bruits. skin: no stasis dermatitis, ulcers, scars, or xanthomas. pulses: dp dopplerable on both sides, but decreased on left. radial pulses 2+ bilaterally . physical examination on discharge: . vs: 98.7 127/89 (96-133/70-80s) 82 (70-80s) 18 98%ra general: pleasant man in nad. oriented x3. mood, affect appropriate. heent: perrl, eomi. conjunctiva were pink, no pallor or cyanosis of the oral mucosa. no xanthalesma. neck: supple, no jvd cardiac: pmi located in 5th intercostal space, midclavicular line. rr, normal s1, loud s2. no m/r/g. no thrills, lifts. no s3 or s4. lungs: no chest wall deformities, scoliosis or kyphosis. resp were unlabored, no accessory muscle use. cta anteriorly, no crackles, wheezes or rhonchi. abdomen: soft, ntnd. no hsm or tenderness. no abdominial bruits. extremities: trace pretibial edeema groin: soft, non-tender, no femoral bruits. skin: no stasis dermatitis, ulcers, scars, or xanthomas. pulses: dp dopplerable on both sides, but decreased on left. radial pulses 2+ bilaterally pertinent results: admission lab results ( 08:50am): wbc-14.1* rbc-4.48* hgb-14.1 hct-40.2 mcv-90 mch-31.5 mchc-35.2* rdw-14.0 plt ct-234 glucose-224* urean-22* creat-1.4* na-141 k-4.0 cl-108 hco3-15* angap-22* ck-mb-21* mb indx-4.1 ctropnt-0.19* ck(cpk)-507* %hba1c-5.7 eag-117 albumin-4.0 . hospitalisation labs ( 05:09pm) ck-mb->500 ctropnt-13.44* ck(cpk)-6944* (peaking) calcium-8.5 phos-3.9 mg-2.0 urean-27* creat-1.6* k-4.3 . ( 07:36am) ck-mb-187* mb indx-3.9 ctropnt-8.17* ck(cpk)-4798* (enzymes trending down) triglyc-220* cholest-152 hdl-35 chol/hd-4.3 ldlcalc-73 (lipid profile) calcium-8.9 phos-3.7 mg-2.1 urean-34* creat-1.6* k-4.3 (urean and creat trending down) pt-12.1 ptt-22.2 inr(pt)-1.0 . ( 04:30pm) ck-mb-109* mb indx-3.2 ctropnt-7.60* ck(cpk)-3382* (enzymes trending down) . ( 07:05am) ck-mb-32* ctropnt-8.82* (ck-mb down, ctropnt plateuing) urean-33* creat-1.5* k-4.1 (urean and creat stable) wbc-11.4* rbc-4.25* hgb-13.4* hct-38.9* plt ct-250 . peak cardiac markers: trop: 13.44 ck: 6944 mb > 500 . discharge labs: 06:50 . wbc hgb hct mcv mch mchc rdw plt ct 10.6 12.8 36.1* 88 31.3 35.4* 14.0 225 . glucose urean creat na k cl hco3 angap 113 30* 1.5* 141 4.2 107 25 13 . hga1c: 5.7 . cholest triglyc hdl chol/hd ldlcalc 152 220 35 4.3 73 . radiology: c.cath (): 1. coronary angiography in this right dominant system demonstrated one vessel disease. the lmca had no angiographically apparent disease. the lad had a less than 50% stenosis after s1. the lcx had no angiographically apparent disease. the rca had a total proximal occlusion with collaterals from the lca. 2. resting hemodynamics revealed elevated right sided filling pressures with rvedp of 20 mmhg. the pasp was slightly elevated at 32 mmhg. the cardiac index was slightly reduced at 2.33 l/min/m2. the systemic arterial pressure was normal with a central aortic pressure of 122/73 mmhg. . echo ( 3:54:12 pm): - mild symmetric lvh. - moderate regional left ventricular systolic dysfunction with severe hypokinesis of the septum and inferior wall. - rv mildly dilated with mild global free wall hypokinesis. - aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. the mitral valve leaflets are mildly thickened. there is no mitral valve prolapse. trivial mitral regurgitation is seen. the estimated pulmonary artery systolic pressure is normal. there is no pericardial effusion. . impression: mild symmetric lvh with regional lv systolic dysfunction and rv dysfunction consistent with rca ischemia/infarction. no significant valvular abnormality seen. ef 30-35%. . ecg: 1. ( 9:50:28 am) sinus rhythm. acute inferior myocardial infarction with deep q waves in inferior leads as well as tall r wave in lead v2 suggesting posterior myocardial infarction. no previous tracing available for comparison. . 2. ( 12:13:50 pm) sinus rhythm. compared to the previous tracing st segment elevation in the inferior leads is reduced. findings are consistent with evolving inferior-posterior myocardial infarction. . 3. ( 8:03:24 am) sinus rhythm. more pronounced stes and q waves in ii, iii, avf, v4-v6. brief hospital course: mr is a 64 yo man with h/o hyperlipidemia who presented with chest pain and was found to have an inferolateral stemi who subsequently underwent pci with bms x 2 insertion to rca with uncomplicated post-cath course. . 1. stemi: the patient presented with ss chest pain, diaphoresis, lightheadedness, and sob at home. r/i for large stemi with peak ck 6944 and trop 13.44. his cardiac catheterization demonstrated two occlusions of the rca, for which he had two bms placed, and his hemodynamics were concerning for a rv infarct. patient monitored closely post-procedure. follow-up tte demonstrated mild symmetric lvh with regional lv systolic dysfunction, rv dysfunction consistent with rca ischemia/infarction and no significant valvular abnormality. patient remained hemodynamically stable and chest pain free post-pci. patient medically managed with metoprolol 200mg xl, lisinopril 5mg, atorvastatin 80mg daily, plavix 75 mg daily and asa 325 mg daily with plan to fully anticoagulate for at least 1 month's duration. physical therapy was agreeable for home discharge. outpatient issues -- patient to follow-up with dr. in 1month. . 2. low back pain: the patient has a history of lbp, for which he is undergoing surgical evaluation later this month. was taking ibuprofen, changed to standing tylenol dut to chronic kidney disease. during admission, pain controlled with po acetaminophen prn and oxycodone prn. . 3. hyperlipidemia: atorvastatin increased from 20mg daily to 80mg daily. lipid profiling done showed ldl at goal, hdl 35. . 4. bph: patients takes alfuzosin at home, but was stopped during hospitalization since was at risk for developing hypotension in the setting of his rv infarct. medication held at time of discharge with plan to readdress. outpatient issues -- follow-up blood pressure as outpatient; if stable and patient without symptoms of orthostasis can be restarted or may consider alternative bph medication without side effect profile of hypotensive. . code status: full during this admission medications on admission: lipitor alfuzosin ibuprofen 800 mg -tid asa 162 mg daily (until ) discharge medications: 1. aspirin 325 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po daily (daily). 2. clopidogrel 75 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 3. atorvastatin 80 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 4. acetaminophen 500 mg tablet sig: two (2) tablet po q6h (every 6 hours) as needed for back pain. 5. nitroglycerin 0.3 mg tablet, sublingual sig: one (1) tablet, sublingual sublingual prn (as needed) as needed for cp: take one tablet and wait 5 min. you can take one more tablet but call dr. or 911 for any chest pain. disp:*25 tablet, sublingual(s)* refills:*0* 6. lisinopril 5 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 7. metoprolol succinate 200 mg tablet extended release 24 hr sig: one (1) tablet extended release 24 hr po once a day. disp:*30 tablet extended release 24 hr(s)* refills:*2* discharge disposition: home discharge diagnosis: st elevation myocardial infarction dyslipidemia discharge condition: mental status: clear and coherent. level of consciousness: alert and interactive. activity status: ambulatory - independent. discharge instructions: mr , it was a pleasure taking care of you during your hospital stay. you had a heart attack and required emergency cardiac catheterization and 2 bare metal stents were placed in your right coronary artery. these stents will stay in place forever and keep the artery open. it is very important that you take a 325 mg aspirin and clopidogrel (plavix) every day to prevent the stent from clotting off and causing another heart attack. do not stop taking aspirin and plavix or miss unless dr. says that it is ok. your heart is weak after the heart attack and we have started new medicines to help your heart pump more effectively and weigh yourself every morning, call dr. if weight goes up more than 3 lbs in 1 day or 5 pounds in 3 days. . we made the following changes to your medicines: 1. start taking aspirin and clopidogrel (plavix) to prevent the stent from clotting off 2. start taking metoprolol to slow your heart rate and help your heart beat effectively 3. start lisinopril to lower your blood pressure and help your heart beat more effectively 4. increase your lipitor to 80 mg daily 5. start taking nitroglycerin as needed for chest pain. call dr. if you have chest pain at home. 6. stop taking ibuprofen, take tylenol instead for back pain 7. stop taking alfuzosin until seen in follow-up and your blood pressure is stable. followup instructions: primary care: name: , j. address: 695 hwy, , , phone: appointment: monday 2:30pm . cardiology: name: , md location: address: , , phone: appointment: at 10:30am procedure: insertion of non-drug-eluting coronary artery stent(s) combined right and left heart cardiac catheterization coronary arteriography using two catheters injection or infusion of platelet inhibitor insertion of drug-eluting coronary artery stent(s) transposition of cranial and peripheral nerves insertion of two vascular stents excision of lingual thyroid percutaneous transluminal coronary angioplasty [ptca] procedure on two vessels diagnoses: coronary atherosclerosis of native coronary artery hypertrophy (benign) of prostate without urinary obstruction and other lower urinary tract symptom (luts) other and unspecified hyperlipidemia lumbago acute myocardial infarction of inferoposterior wall, initial episode of care 

Answer: The patient is high likely exposed to