Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: lamisil attending: chief complaint: left sided weakness/ich major surgical or invasive procedure: ng tube placement and removal history of present illness: 84 yo man with pmh of htn, dm, macular degeneration and gout and cad (s/p 3 mis and 3 stents) p/w ams. the family did not know about the events. he apparently pressed the life button and was taken to northeast () hosp at 9:48 am with: "left?? sided weakness and garbled speech per report. right facial droop. 144/ 63. fsg 255. not following commands. sleepy bur responding to verbal commands. yawning". ekg with ventricular pacing. ct cns w/o contrast showed per report a 5 cm iph temporal bleed on the leftwith vasogenic edema. received pht 1 g iv. transferred to . coags: normal. chem: na 131, glucose 169, creat 1.3, bun 37. bnp 1824. cxr: normal with ppm. past medical history: cad s/p mi (hx of inferior mi and apical akinesis) and cabg s/p 3 stents around 1 year ago. he was on plavix and asa (the later stopped 1 week ago). pacemaker (unknown reason) chf (? last ef of 25%) major depressive d/o syncope esophageal stricture social history: no toxic habits. iadls. family history: not obtained physical exam: ed examination temp 98.5f, bp 140/99, map 109, 65 bpm (paced), so2 100% in ra with 16 rr. no abnormal respiratory pattern. not on ventilator gsc: 9 gen: lying in bed, unresponsive. heent: nc/at, moist oral mucosa neck: supple, no carotid or vertebral bruit back: no point tenderness or erythema cv: nl s1 and s2, no murmurs/gallops/rubs lung: clear to auscultation bilaterally abd: soft, nontender, non-distended. no masses or megalies. percussion within normal limits. +bs. ext: no edema, no dvt data. pulses ++ and symmetric. neurologic examination: no meningismus. no photophobia. ms: drowsy. responsive to sternal rub. not to verbal command. cn: brain stem reflexes : preserved corneals + bl. pupils anisocoric: 2 to 1.5 left (surgical) and 3 to 2 on the right. looks when stimulated to rigth and left. no eom paresis. no gaze deviation. no bobbing or robbing. no nystagmus. gag +. motor: he does withdraw to pain with all limbs symmetrically. tone: dtr: 3+ throughout. patellas: postsurgical. toes: amputated on the right foot. cavus deformity in left foot with toes upgping at rest attending exam in icu vs: bp 145/52 p 65 r 15 o2 97% neuro exam: drowsy, follows commands to squeeze with the left hand, does not open eyes to verbal or noxious, moans but does not say any words cn: does not blink to threat or track (legally blind, but has some sight at baseline), perrla, right lower facial droop motor: flaccid right arm, decreased tone of the right leg, 0/5 strength of the right arm, right leg normal tone of the left arm and leg, left arm is , and left leg is . sensory: right arm has extensor posturing to noxious, right leg withdraws to noxious, left arm and leg withdraw to noxious. reflexes: 2+ biceps, 1+ knees bilaterally right foot amputation at mid foot, left toe is mute coord: unable to assess pertinent results: 01:15pm wbc-7.0 rbc-2.90* hgb-8.6* hct-27.1* mcv-94 mch-29.6 mchc-31.7 rdw-14.3 01:15pm neuts-87.9* lymphs-7.5* monos-3.0 eos-1.4 basos-0.2 01:15pm calcium-9.6 phosphate-4.3 magnesium-2.1 05:46pm glucose-133* urea n-39* creat-1.3* sodium-131* potassium-5.5* chloride-99 total co2-25 anion gap-13 01:15pm ctropnt-0.02* 03:50am blood ck-mb-4 ctropnt-0.05* 05:05am blood ck-mb-4 ctropnt-0.05* 10:34am blood %hba1c-7.0* eag-154* 06:00am blood triglyc-66 hdl-71 chol/hd-1.7 ldlcalc-34 time taken not noted log-in date/time: 5:47 pm mrsa screen source: nasal swab. **final report ** mrsa screen (final ): no mrsa isolated. ct head w/o contrast study date of 11:35 am impression: 1. essentially unchanged size of the large left temporal intraparenchymal hemorrhage. 2. interval mild increase of peri-hemorrhagic edema, with increase of the effacement of the left lateral ventricle. a new 2-mm rightward shift of midline structures. 3. no new hemorrhage or developing hydrocephalus. final attending comment: the wedge shaped configuration of the hypodensity in the left parietal lobe suggests that this represents hemorrhagic transformation of an infarct. chest (portable ap) study date of 3:31 pm findings: in comparison with study of , the dobbhoff tube and pacemaker device remains in place. moderate enlargement of the cardiac silhouette is again seen without evidence of vascular congestion. no evidence of acute focal pneumonia. brief hospital course: mr. is an 84 yo man with a history of hypertension, diabetes, and cad who presented with altered mental status and hemorrhage on outside ct scan. neuro/ischemic stroke: the patient was initially treated with dilantin for seizure prophylaxis. while his ct scan was notable for a large left sided parenchymal hemorrhage, repeat ct scans revealed this to be an infarction of the left mca with subsequent hemorrhagic conversion. dilantin was discontinued. the patinet was restarted started on plavix given his cardiac stents and for continued stroke prevention. although his stroke was likely a thrombolic event, no additional anticoagulation was considered in light of his hemorrhage. the patient's mental status improved significantly. he was initially fluently aphasic with relatively speech but retained comprehension. he has right sided weakness which vastly improved. of note, the patient was legally blind at baseline and did not blink to threat on exam. while his speech output has improved significantly, he continues to have a prominant fluent aphasia with dysarthria (also possibly related to his edentulous state). he was cleared by speech and swallow for a diet of nectar thickened fluids and pureed solids. he should continue to undergo speech and physical therapy. he is enrolled in a dysphagia study at under the direction of . he is scheduled for follow up in the neurology clinic in with dr. . cadiovascualr/hypertension/hyperlipidemia: the patient's antihypertensive regimen was initially held in the setting is his stroke. he was initially agressively hydrated and in the setting of his diminished ef, he did require subsequent diuresis with good results. his home antihypertensive regimen was reinstituted over the course of his hospitalization with the exception of his nifedipine as the patient could not take continuous release pills (all medications were to be crushable). he was started, instead, on diltiazem at 30mg tid and this can be uptitrated as needed for blood pressure control. alternatively, the patient may resume nifedipine 60mg daily once he is able to take whole pills. the patient's lipid panel was within goal range and no changes where made. the patient was monitored on telemetry and no events were seen. there was a noted increase in troponin without ekg changes and relatively flat ck. this was felt to be related to subendocardial ischemia and renal failure. no interventions where made. endocrine/diabetes: the patient's a1c was 7.0. during this hospitalization, he was treated with sliding scale insulin and routinely required upwards of 10units of insulin daily. standing insulin was not started in the setting of his transition from tube feeds to oral diet but oral hypoglycemic agents should be considered. pulm/tachypnea: the patient was noted to be tachypneic with rales. chest x-ray was without evidence of infection or effusion, no frank chf. it was felt his tachypnea was related to volume overload and he was diureased with good effect. renal/chronic renal failure: the patient has a history of renal insufficency with a baseline creatinine of 1.3. there was acute elevation to 1.5 in the setting of diuresis but this improved to his baseline prior to discharge. code status: dni. not dnr. medications on admission: plavix 75 mg daily lisinopril 20mg zocor 40mg metoprolol 12.5mg allopurinol 100mg lasix 20mg daily iron 325mg vitamin d 400u mvi protonix 40mg nifedipine 60mg daily discharge medications: 1. simvastatin 40 mg tablet sig: one (1) tablet po daily (daily). 2. allopurinol 100 mg tablet sig: one (1) tablet po daily (daily). 3. lisinopril 20 mg tablet sig: one (1) tablet po daily (daily). 4. furosemide 20 mg tablet sig: one (1) tablet po daily (daily). 5. clopidogrel 75 mg tablet sig: one (1) tablet po daily (daily). 6. metoprolol tartrate 25 mg tablet sig: 0.5 tablet po bid (2 times a day). 7. diltiazem hcl 30 mg tablet sig: one (1) tablet po qid (4 times a day). 8. iron 325 mg (65 mg iron) tablet sig: one (1) tablet po once a day. 9. vitamin d 400 unit tablet sig: one (1) tablet po once a day. 10. multivitamin oral 11. protonix 40 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po once a day. discharge disposition: extended care facility: discharge diagnosis: left mca infarct with hemorrhagic conversion hypertension diabetes, type ii chronic renal insufficiency hyperlipidemia blindness discharge condition: the patinet was hemodynamically stable. neuro exam was notable for blindness (no blink to threat), fluent aphasia with frequent neologisms and impaired but some retained comprehension, slight right facial droop but good strength in extremities (exam limited by ability to follow commands). discharge instructions: you were admitted for evaluation of altered mental status. you were found to have a stroke affecting the left side of your brain and this stroke had bled. you are being discharged to a rehabilitation facility for further treatment. followup instructions: provider: , md phone: date/time: 2:00 procedure: enteral infusion of concentrated nutritional substances diagnoses: congestive heart failure, unspecified diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage i through stage iv, or unspecified gout, unspecified aortocoronary bypass status percutaneous transluminal coronary angioplasty status chronic kidney disease, unspecified other and unspecified hyperlipidemia cerebral edema old myocardial infarction unspecified disorder of kidney and ureter cardiac pacemaker in situ dysarthria hyperosmolality and/or hypernatremia aphasia legal blindness, as defined in u.s.a. cerebral thrombosis with cerebral infarction foot amputation status flaccid hemiplegia and hemiparesis affecting unspecified side 

Answer: The patient is high likely exposed to