Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: iodine attending: chief complaint: s/p brady arrest major surgical or invasive procedure: endotracheal intubation history of present illness: mr. was a 66 year old male with h/o cad s/p cabg (atatomy not known) as well as pci with stent in in who was in his usoh until 4 pm on when the patient collapsed after lifting heavy boxes. by report, cpr was initiated immediately and 911 called with rapid ems response. per report, ems found pt in wct likely vt and pulseless. pt received a total of 9 shocks and lidocaine push during the transport to osh ed. on arrival in the ed the patient was unreponsive and without a pulse, s/p two more shocks and intubated for airway protection. ekg with wct and he was given amio bolus x 2 and started on a drip. the patient also was given epi and atropine during the code. the patient remained hypotensive and was started on dopamine/levophed for pressure support. the patient was then transferred to for ongoing care. echo at osh by report demonstrated an ef of 40% with global hypokinesis, no focal wall motion abnormalities, but was a limited study. ct of the head showed no acute changes. meds on transfer included amio gtt and plavix. past medical history: - cabg yrs ago, anatomy: lima to lad, svg to high lateral - pci w/two des to mid and distal rca - pci w/des to svg . cardiac risk factors: dyslipidemia, hypertension social history: lives in , was here in the , in area. family history: not obtained physical exam: per dr. : vs: t 99 bp 118/63 hr 70 rr 20 o2 100% on ac 500/15 gen: intubated and sedated. heent: ncat. sclera anicteric. perrl, eomi. bleeding gums. neck: supple with jvp flat cv: pmi located in 5th intercostal space, midclavicular line. rr, normal s1, s2. no m/r/g. no thrills, lifts. no s3 or s4. chest: intubated, b/l coarse crackles, ?rib fracture abd: soft, ntnd. no hsm or tenderness. ext: no c/c/e. no femoral bruits. pertinent results: admission labs: 145 112 38 --------------< 174 4.0 19 1.5 ck: 9097 mb: >500 trop-t: 9.87 ca: 10.1 mg: 2.8 p: 4.0 . 15 20.1 >----< 319 44.2 09:29am blood wbc-11.7* rbc-3.11* hgb-10.0* hct-28.4* mcv-91 mch-32.1* mchc-35.1* rdw-14.6 plt ct-189 04:21pm blood wbc-10.1 rbc-3.10* hgb-10.1* hct-28.2* mcv-91 mch-32.7* mchc-35.9* rdw-14.5 plt ct-135* 05:39am blood wbc-17.8* rbc-1.47*# hgb-4.6*# hct-14.9*# mcv-101* mch-31.6 mchc-31.1 rdw-14.4 plt ct-269 04:45am blood fibrino-822* 04:21pm blood glucose-126* urean-26* creat-0.9 na-143 k-3.7 cl-114* hco3-23 angap-10 05:56am blood glucose-112* urean-27* creat-0.9 na-150* k-3.2* cl-112* hco3-26 angap-15 08:02am blood alt-143* ast-216* ld(ldh)-839* alkphos-39 totbili-0.6 04:45am blood alt-111* ast-165* ld(ldh)-799* alkphos-36* totbili-0.7 05:44am blood alt-74* ast-96* ck(cpk)-694* alkphos-40 totbili-0.6 01:40am blood ck-mb-greater th ctropnt-9.87* 02:25pm blood ck-mb-282* mb indx-3.1 09:14am blood ck-mb-13* mb indx-0.6 05:31am blood ck-mb-13* mb indx-0.8 05:44am blood ck-mb-5 09:14am blood hapto-143 02:29am blood type-art po2-445* pco2-39 ph-7.39 caltco2-24 base xs-0 01:53pm blood type-art po2-72* pco2-30* ph-7.48* caltco2-23 base xs-0 05:27am blood type-art temp-38.7 tidal v-500 peep-5 po2-138* pco2-35 ph-7.46* caltco2-26 base xs-2 intubat-intubated 12:43pm blood lactate-1.6 . stool clostridium difficile toxin assay-neg stool clostridium difficile toxin assay-neg stool clostridium difficile toxin assay-neg urine urine culture-ng urine urine culture-ng sputum gram stain (final ): >25 pmns and <10 epithelial cells/100x field. no microorganisms seen. respiratory culture (final ): rare growth oropharyngeal flora. klebsiella pneumoniae. rare growth. identification and sensitivities performed on culture # 232-0962f (). blood culture ng blood culture ng sputum gram stain (final ): >25 pmns and <10 epithelial cells/100x field. 1+ (<1 per 1000x field): gram negative rod(s). respiratory culture (final ): oropharyngeal flora absent. klebsiella pneumoniae. sparse growth. trimethoprim/sulfa sensitivity testing available on request. sensitivities: mic expressed in mcg/ml _________________________________________________________ klebsiella pneumoniae | ampicillin/sulbactam-- 4 s cefazolin------------- <=4 s cefepime-------------- <=1 s ceftazidime----------- <=1 s ceftriaxone----------- <=1 s cefuroxime------------ 2 s ciprofloxacin---------<=0.25 s gentamicin------------ <=1 s imipenem-------------- <=1 s meropenem-------------<=0.25 s piperacillin/tazo----- <=4 s tobramycin------------ <=1 s catheter tip-iv ng urine urine culture-ng blood culture ng blood culture ng sputum gram stain (final ): >25 pmns and <10 epithelial cells/100x field. 2+ (1-5 per 1000x field): gram positive cocci. in pairs. respiratory culture (final ): sparse growth oropharyngeal flora. klebsiella pneumoniae. sparse growth. trimethoprim/sulfa sensitivity testing available on request. sensitivities: mic expressed in mcg/ml _________________________________________________________ klebsiella pneumoniae | ampicillin/sulbactam-- 4 s cefazolin------------- <=4 s cefepime-------------- <=1 s ceftazidime----------- <=1 s ceftriaxone----------- <=1 s cefuroxime------------ <=1 s ciprofloxacin---------<=0.25 s gentamicin------------ <=1 s imipenem-------------- <=1 s meropenem-------------<=0.25 s piperacillin/tazo----- <=4 s tobramycin------------ <=1 s blood culture ng urine urine culture-ng blood culture ng sputum gram stain-final; respiratory culture-final {klebsiella pneumoniae} inpatient blood culture ng blood culture ng urine ng . chest (portable ap) 11:10 am two portable views. comparison with the previous study done earlier the same day. there is streaky density at the lung bases consistent with subsegmental atelectasis as before. the patient is status post median sternotomy and cabg. mediastinal structures are unchanged. an endotracheal tube and nasogastric tube remain in place. impression: subsegmental atelectasis. . portable semi-upright chest 7:56 a.m. compared with at 10:44 p.m., no obvious interval change in the pulmonary vascular engorgement centrally. the patchy streaky opacities at the right lung base are slightly more prominent and confluent suggesting pneumonia. . cardiology report ecg study date of 1:59:54 am sinus rhythm, rate 76. technical artifacts are seen. an indeterminate axis is noted. right bundle-branch block pattern is seen. ther is likely an anteroseptal myocardial infarction of undetermined age. no previous tracing available for comparison. . echo measurements: left atrium - long axis dimension: *4.8 cm (nl <= 4.0 cm) left atrium - four chamber length: *5.5 cm (nl <= 5.2 cm) right atrium - four chamber length: *5.9 cm (nl <= 5.0 cm) left ventricle - septal wall thickness: 0.8 cm (nl 0.6 - 1.1 cm) left ventricle - inferolateral thickness: 0.8 cm (nl 0.6 - 1.1 cm) left ventricle - diastolic dimension: *6.5 cm (nl <= 5.6 cm) left ventricle - ejection fraction: 20% (nl >=55%) aorta - valve level: *4.0 cm (nl <= 3.6 cm) aorta - ascending: *3.5 cm (nl <= 3.4 cm) aortic valve - peak velocity: 1.4 m/sec (nl <= 2.0 m/sec) mitral valve - e wave: 0.7 m/sec mitral valve - a wave: 0.7 m/sec mitral valve - e/a ratio: 1.00 mitral valve - e wave deceleration time: 174 msec pulmonic valve - peak velocity: 1.0 m/sec (nl <= 1.0 m/s) interpretation: findings: left atrium: mild la enlargement. right atrium/interatrial septum: mildly dilated ra. normal interatrial septum. no asd by 2d or color doppler. dilated ivc (>2.5cm) with <50% decrease during respiration (estimated rap 16-20 mmhg). left ventricle: normal lv wall thickness. moderately dilated lv cavity. no lv mass/thrombus. severely depressed lvef. no resting lvot gradient. no vsd. right ventricle: focal apical hypokinesis of rv free wall. paradoxic septal motion consistent with conduction abnormality/ventricular pacing. aorta: moderately dilated aortic sinus. mildly dilated ascending aorta. aortic valve: mildly thickened aortic valve leaflets (3). no as. no ar. mitral valve: normal mitral valve leaflets. no mvp. trivial mr. tricuspid valve: tricuspid valve not well visualized. indeterminate pa systolic pressure. pulmonic valve/pulmonary artery: no ps. pericardium: no pericardial effusion. general comments: suboptimal image quality - poor echo windows. echocardiographic results were reviewed by telephone with the md caring for the patient. conclusions: the left atrium is mildly dilated. there is an echodensity associated with the left atrial of the posterior mitral annulus ( vs artifact/tissue?). no atrial septal defect is seen by 2d or color doppler. the estimated right atrial pressure is 16-20 mmhg. left ventricular wall thicknesses are normal. the left ventricular cavity is moderately dilated. no masses or thrombi are seen in the left ventricle. overall left ventricular systolic function is severely depressed with severe global hypokinesis and akinesis (thinned) of the basal inferior and lateral walls. there is very apical dyskinesis. there is no ventricular septal defect. there is focal hypokinesis of the apical free wall of the right ventricle. the aortic root is moderately dilated at the sinus level. the ascending aorta is mildly dilated. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. no aortic regurgitation is seen. the mitral valve leaflets are structurally normal. there is no mitral valve prolapse. trivial mitral regurgitation is seen. the pulmonary artery systolic pressure could not be determined. there is no pericardial effusion. impression: severely depressed lvef with regionality c/w cad. possible vs artifact. if clinically indicated, a tee may better characterize . . mr head w/o contrast 9:33 pm mr head w/o contrast reason: please assess for bleed, please asses for thromboembolic cva medical condition: 66 year old man with brady arrest requiring 11 shocks by dc-cardioversion. reason for this examination: please assess for bleed, please asses for thromboembolic cva, please assess neck for cord compression and soft tissue injury. indication: cardiac arrest, requiring shocks by cardiac conversion. technique: multiplanar t1- and t2-weighted sequences were obtained through the brain with diffusion-weighted imaging. findings: evaluation of the adc map demonstrates diffuse cortical low signal. this corresponds to increased signal on the diffusion-weighted sequence within the cortex. these findings represent diffuse cortical slow diffusion. this would represent diffuse cortical injury from anoxia. there is a tiny focus of abnormal magnetic susceptibility at the -white matter junction in the posterior right frontal lobe consistent with petechial hemorrhage. there is no midline shift, mass effect, or hydrocephalus. the normal vascular flow voids are present. there is paranasal sinus disease due to the patient's intubated status. impression: findings are consistent with diffuse anoxic brain injury. . mr cervical spine w/o contrast 9:33 pm mr cervical spine w/o contrast reason: now patient with c-collar needs to be cleared. medical condition: 66 year old man s/p brady arrest and fall. reason for this examination: now patient with c-collar needs to be cleared. indication: brady arrest and fall. the patient with c collar needs to be cleared. technique: multiplanar t1- and t2-weighted sequences were obtained through the cervical spine with sagittal stir sequence. findings: the alignment of the cervical spine appears normal. there is no abnormal bone marrow edema. the intrinsic cord signal appears generally normal although it is poorly evaluated due to some motion. at the level of , there is a small focus of abnormal magnetic susceptibility within the left-sided cord. this is suspicious for an intramedullary hemorrhage. there are multilevel posterior osteophytes causing mild spinal canal narrowing. there are areas of moderate bilateral neural foraminal narrowing associated with these osteophytes. given the patient's history and the presence of abnormal susceptibility within the cord, the concern is for a cord injury. impression: small area of abnormal magnetic susceptibility within the cord at the level of c5-6 is concerning for a petechial hemorrhage. this could be a secondary finding associated with cord injury. the intrinsic cord signal is poorly evaluated due to patient motion artifact on the stir sequence. there however is no bone marrow edema. . object: bedside side eeg with viedo, . the heart was monitored because disorders of heart rhythms produce neurological complaints as described above or neurological disorders such as seizures, when symptomatic, produce cardiac arrhythmias. referring doctor: dr. findings: routine sampling: a low voltage hz disorganized posterior background rhythm is seen with frequent electrode artifacts seen at the bilateral temporal leads with a very rhythmic alpha frequency quality that is limited to these leads; however, at other times, it is also seen in the right central region. there was also electrode artifact seen in the left central leads. when these artifacts were at their lowest, a very slow hz low voltage rhythm was noted with no clear regions of focal slowing and no clear epileptiform discharges noted. sleep: there were no normal sleep/wake transitions seen. cardiac monitor: a generally regular rhythm was noted with an average rate of 96 bpm. however, frequent premature ventricular contractions were seen. automatic spike detection files: there were 259. these consisted primarily of electrode artifact, particularly at the bilateral temporal leads. there also seemed to be superimposed electrical artifact of low voltage and high frequency. no true epileptiform features were noted. automatic seizure detection files: there were 43. these consisted of the above-noted electrode or electrical artifact seen in the bilateral temporal leads as well as multiple other leads. no true electrographic seizures were recorded, however. pushbutton activations: there were none. impression: this is an abnormal 24-hour video eeg telemetry in the waking and sleeping states due to the low voltage suppressed slow and disorganized background rhythm with much superimposed electrical artifact. nonetheless, no true electrographic seizures or epileptiform features were noted. there were no pushbutton activations. this slow low voltage and disorganized background is suggestive of a severe encephalopathy which may be seen with medication effect, toxic metabolic abnormalities, or infections as well as global ischemic disease. of note, there were frequent premature ventricular contractions noted throughout the tracing. . neurophysiology report ep study date of object: cardiac arrest. assess neurologic function. referring doctor: dr. findings: brain stem auditory evoked potential (07-085): after stimulation of the right ear there was no discernible evoked potential at any position. this can often come from lesions in the viiith cranial nerve. the patient was reported to have an earlier and severe hearing loss on the right. after stimulation of the left ear there was a very poorly formed and faint peak at position i and another poorly formed peak at position v with a normal latency. this suggests some conduction from the periphery to the mid-brain, and with a normal latency. median nerve somatosensory evoked potential (07-086): after stimulation of the right median nerve there was an evoked potential peak at erb's point with a normal latency. subsequent peaks were not discernible. this suggests a defect in the large fiber somatosensory conducting system after right median stimulation, with the defect proximal to the brachialplexus. this can be at the root level or centrally. after left median nerve stimulation there were no discernible evoked potential peaks at any position. there was no peak at erb's point. this suggests a defect in the large fiber somatosensory conducting system peripherally. this can be due to peripheral neuropathies, body habitus, and sometimes to technical factors. brief hospital course: 66 m with h/o cad s/p cabg and pci who presented from osh s/p cardiac arrest, reportedly down for ~10 hrs, pulseless, s/p multiple shocks, intubated & sedated on amiodarone and heparin gtt's. hospital course by problem: . #) cad: pt was s/p cabg with unknown anatomy (done in ), also with recent pci in . ami per ekg. due to an unkown etiology for his arrest, thought seconsary to scar rather than acute mi, in combination with his tenuous clinical status and questionable nuerologic recovery - an acute cardiac catheterization was not performed. . #) rhythm: his amiodarone drip was continued for several days. he had only small runs of nsvt and a malignant arhythmia did not return. his amiodarone drip was discontinued. he remained in sinus rhythym while monitored on telemetry. . #) pump: ef was reportedly 40% at osh with global hk. a repeat echo here showed an ef of 20%. . #) resp: he remained intubated up until the point he was made comfort measures only at which point he was taken off the ventilator. . #) neuro: neurology was involved in this patient's care and an mri was obtained. the mri showed diffuse cortical injury. he did not recover meaningful cortical activity. he developed epileptiform partialis continuium is his right arm and was initially started on a dilantin load. this was discontinued after eeg showed no epileptiform activity. a family meeting was held to discuss the neurologic prognosis and ultimately the family decided that given his poor prognosis, they would change his care to comfort measures only. . #) febrile illness - unclear source. infectious vs. central fever. the patient appeared septic early in the course of his hospitalization and was broadly covered with vanc and zosyn. this was changed to levoquin for 2 days, but high spiking fevers to 102 returned and he was re-started on vanc/zosyn. sputum cultures were not initially definitive for a source, though eventually grew klebsiella (cukture data above. . #) dispo: the patient was made comfort measures only and expired on . medications on admission: diovan 120 mg daily asa 325 plavix 75 lipitor 30 folic acid discharge medications: n/a discharge disposition: expired discharge diagnosis: anoxic brain injury following cardiac arrest. discharge condition: expired. discharge instructions: not applicable followup instructions: not applicable procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more diagnostic ultrasound of heart enteral infusion of concentrated nutritional substances transfusion of packed cells diagnoses: pure hypercholesterolemia urinary tract infection, site not specified congestive heart failure, unspecified unspecified essential hypertension acute kidney failure, unspecified unspecified septicemia severe sepsis aortocoronary bypass status percutaneous transluminal coronary angioplasty status other specified procedures as the cause of abnormal reaction of patient, or of later complication, without mention of misadventure at time of procedure acute respiratory failure other specified cardiac dysrhythmias anoxic brain damage acute myocardial infarction of unspecified site, initial episode of care flail chest pneumonia due to klebsiella pneumoniae 

Answer: The patient is high likely exposed to