Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of the present illness: mr. was a 22-year-old gentleman who had no significant past medical history who was involved in a motor vehicle accident at high speed after losing control of his vehicle involving a prolonged extrication from the accident site with agonal respirations and transport via to the emergency room where he arrived as a trauma plus. during the transport, he was intubated and the airway was secured and his vital signs were stabilized. upon arrival to the er, he was further resuscitated and standard atls protocol was instituted with all supporting services informed and neurosurgery present. upon arrival, his gcs was 3 as it was at the scene and his initial examination findings were significant for a right pupil 2 mm and a left pupil of 6 mm, right facial swelling and ecchymosis with obvious facial fractures, multiple. the chest and pelvis demonstrated no obvious signs of injury or deformity; however, he had an obvious right arm deformity. after the patient was stabilized, ct scans and radiology imaging demonstrated a left subdural hematoma, subarachnoid mass, subarachnoid hemorrhage, and intraventricular blood as well as left mandibular fracture, multiple right orbital fractures, zygomatic fractures, sphenoid and maxillary sinus fractures. a ct of the c spine demonstrated no fracture of the neck and chest ct was negative except for pulmonary contusions and a small left pneumothorax which was followed by chest x-ray and found not to be progressing and neither was it evident on chest x-rays. there was no evidence of abdominal injuries and workup of his humerus fracture showed a displaced midshaft humeral fracture with no evidence of vascular compromise in that extremity. upon evaluation, the neurosurgical team felt that the patient was unsalvagable. they did not place a ventriculostomy drain for two reasons; first, the ventricle was not accessible for adequate drainage of csf and that any drainage of csf would be of no utility. we then instituted optimal medical management to minimize secondary brain injury from the trauma and we did place a komino icp monitor. at the insertion site, the icp was 60. we instituted standard protocols including increasing the systolic blood pressure to maintain cerebral perfusion pressure as best as we could as close to 70 and we ensured adequate volume resuscitation through central monitoring. upon arrival into the trauma intensive care unit, under the full supervision of multiple senior residents and with constant contact with the attending surgeons as well as all consult services, it was noted that the patient was bradycardiac and the pressure fell to 68/24. he was again resuscitated to a pressure of 130/140 with an adequate heart rate of initially 140, stabilized to 80-90, cvp of 13 at that time. we continued maintaining his systemic blood pressure with an epinephrine drip which was then added to a levophed drip. eventually, a pitressin drip after the epinephrine was stopped. this was done in an attempt to maximize cerebral perfusion; however, his icp continued to increase despite all of our medical management efforts and his icp peaked out at 138. it was noted that between the hours of 8:00 and 9:00 a.m. on , the patient's urine output had increased significantly and it was most likely diabetes insipidus. we conducted replacement of all fluid losses and continued to best support him as we could. at this point, the clinical examination demonstrated fixed and dilated pupils on both sides indicating severe brain injury and there was no gag reflex. the patient had a gcs of 3 and no oculovestibular reflex. there was no evidence of any kind of neurological activity with regards to the neuromotor system. the organ bank had been informed earlier during the hospital stay and were involved in approaching the family during the course of this admission. neurology was involved and the medical intensive care unit attending to conduct brain death examinations and a cerebral perfusion study was conducted on the afternoon of to confirm brain death which demonstrated no cerebral flow. in light of this fact with the clinical examination demonstrating significant neurological injury indicative of brain team, an adequate mechanism to lead to brain death, and irreversibility demonstrated by the nuclear study showing no cerebral flow, the patient was declared dead at 4:45 p.m. on . the attending, the intensive care team, the organ bank, and all consulting services were informed and agreed upon this decision and the family was with great regret informed of this unfortunate outcome despite our best efforts. the family declined a postmortem and declined organ transplantation. the case was referred to the medical examiner's office and an autopsy was declined at that time. the attending staff had constant involvement with the case as well as all consult teams. , m.d. 2923 dictated by: medquist36 procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours other diagnostic procedures on brain and cerebral meninges diagnoses: traumatic pneumothorax without mention of open wound into thorax closed fracture of malar and maxillary bones closed fracture of orbital floor (blow-out) closed fracture of other facial bones motor vehicle traffic accident due to loss of control, without collision on the highway, injuring driver of motor vehicle other than motorcycle closed fracture of shaft of humerus closed fracture of base of skull with subarachnoid, subdural, and extradural hemorrhage, with prolonged [more than 24 hours] loss of consciousness, without return to pre-existing conscious level closed fracture of mandible, ramus, unspecified 

Answer: The patient is high likely exposed to