Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: past medical history: 1. myasthenia . 2. hypertension. 3. hypercholesterolemia. 4. type 2 diabetes. 5. benign prostatic hypertrophy, status post transurethral resection of the prostate. 6. spinal stenosis. 7. degenerative disk disease. 8. l5 to s1 diskectomy. admission medications: 1. flomax 0.4 mg p.o. q.h.s. 2. zocor 40 mg p.o. q.h.s. 3. lisinopril 10 mg p.o. q.d. 4. glucovance 2.5/500 mg tablets one tablet p.o. q.a.m., and two tablets p.o. q.p.m. 5. mestinon 30 mg p.o. q. four hours. 6. protonix 40 mg p.o. q.d. 7. cellcept 1,000 mg p.o. q.d. 8. prednisone 40 mg p.o. q.o.d. 9. insulin sliding scale. allergies: the patient has no known drug allergies. family/social history: details of his family and social history can be found in a previous note. physical examination on admission: vital signs: mr. is afebrile, blood pressure 165/61, pulse 84, breathing 18, 02 saturation 97% on room air. general: he is an uncomfortable appearing elderly man lying in the emergency room bed. heent: normal secretion volume, no excessive lacrimation. pulmonary: clear to auscultation bilaterally. cardiovascular: regular, no murmurs. abdomen: soft, nontender. extremities: 2+ pulses. there was no edema. neurologic: he was awake, alert. his language was fluent. there was left exotropia. his face was symmetric. he has prominent dysphonia and dysphagia for solids and liquids. his neck flexors were 4-/5, neck extensors 5-/5. his deltoids were full-strength. these muscles had been weak in the past. the remainder of his muscle groups are strong with the exception of the l5 innovator muscles in the right foot which were +/5. his reflexes are preserved. laboratory/radiologic data: white count 18.1, hematocrit 36.3, platelets 385,000. sodium 137, potassium 4.6, chloride 99, bicarbonate 25, bun 33, creatinine 1.2, glucose 219. hospital course: the patient was admitted to the neurology icu. he was given initially mestinon 45 mg p.o. q. four hours. his prednisone was discontinued and solu-medrol at a dose of 250 mg iv was instituted. his cellcept was continued. his insulin sliding scale was continued. because the patient could not take oral medication, he was given neostigmine 1-2 mm subcutaneously in order to be able to swallow his mestinon. he also received vital capacities and nebulizers q. four hours. chest x-ray, urinalysis, urine culture were performed to look for infectious sources and these ere negative. on hospital day number two, his mestinon was changed to 45, 60, 60, 45. his forced vital capacities and negative inspiratory forces remained above normal. the examination noted that he had less dysphonia and dysphagia. his neck extensors were practically full-strength and the neck flexors could be overcome with moderate resistance. mr. on . the prednisone was discontinued on under the recommendations of the neuromuscular consultation service. his prednisone was changed to 60 mg p.o. q.d. cellcept was increased to 1 gram b.i.d. the patient remained in the intensive care unit and was plasmapheresed on for the first time. he received three sessions of plasmapheresis. this resulted in improvement in his strength, particularly with his voice and with his swallowing. on , his cholinesterase inhibitors were discontinued because it was felt that these were worsening his weakness. he received another session of phoresis on the 16th. cellcept was changed to iv as he was not tolerating solids on the 16th. it seemed as though mr. had experienced a deterioration from high-dose steroids that he had been given on hospital days one to two. mr. was again plasmapheresed on and completed his final session on . the patient was called out to the neurology floor on . at that time, his dysphagia and dysphonia were much improved and his proximal muscle strength was also improved. his neck flexors remained weaker than his neck extensors. the patient has had a successful course of treatment for myasthenic crisis and because his muscle strength improved back to baseline, the decision was made to discharge him home on . discharge diagnosis: myasthenia . follow-up: dr. of the neuromuscular disease group on at 2:15 p.m. he will also follow-up with his primary care physician, . . discharge medications: 1. cellcept which will be dispensed as a syrup 200 mg per milliliter, 5 milliliters p.o. b.i.d. 2. prednisone 60 mg p.o. q.d. 3. pepcid 20 mg p.o. b.i.d. 4. flomax 0.4 mg p.o. q.h.s. 5. zocor 40 mg p.o. q.d. 6. glucovance 2.5 500 mg tablets one tablet p.o. q.a.m., two tablets p.o. q.p.m. 7. insulin sliding scale; 0-200 give 0 units of regular insulin; 201-250 give 2 units; 251-300 give 4 units; 301-350 give 6 units; 351 plus give 8 units. 8. mestinon 30 mg p.o. q. eight hours. disposition: the patient is being discharged in much improved condition. he has received definitive treatment for myasthenia . , m.d. dictated by: medquist36 procedure: venous catheterization, not elsewhere classified therapeutic plasmapheresis diagnoses: pure hypercholesterolemia unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled other specified cardiac dysrhythmias 

Answer: The patient is high likely exposed to