Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: hydrocodone / codeine / wellbutrin attending: chief complaint: headache. numbness and tingling in the hands. major surgical or invasive procedure: cerebral angiogram history of present illness: the patient is a 49 year old rh woman with a history of nsclc diagnosed in and resected (lul) as well, now presenting on transfer from an osh icu with a possible diagnosis of cerebral vasculitis. she presented to the osh on complaining of a headache and numbness/tingling in the hands. she was admitted and by the next day had developed a prominent left facial droop and hemi-paresis. an mri was done that showed multiple strokes "mostly in the right hemisphere, but involving the left hemisphere as well." an mra that was performed showed a vascular "beading" pattern suggestive of a vasculitis. she was begun on heparin and asa. she underwent a tee which by report did not show any evidence of a cardio-embolic source of her stroke. she also underwent a cerebral angiogram which again was suggestive of a cerebral angiitis. over the course of her hospital stay, she was noted to have a dense left-hemiparesis, confusion, slurred speech, and progressive somnulence (the onset of which is unclear from the notes). her laboratory values there showed a thrombocytosis (peak = 681), an esr of 17, negative , and unrevealing hypercoagulable workup. review of systems: -largely unobtainable past medical history: per above arthritis depression social history: the patient has been smoking 1 pack/day for 30 years. she is a psychiatric coordinator. she does not drink alcohol. family history: significant for lung cancer, emphysema, and coronary artery disease physical exam: vitals: 98.3 122/51 102 98% on ra general: middle aged woman, starring blankly out into space, non-verbal neck: supple, no bruit lungs: clear to auscultation cv: regular rate and rhythm, no murmur abdomen: non-tender, non-distended, bowel sounds present ext: warm, no edema, good pulses neurologic examination: moans to sternal rub; opens eyes wide to loud voice, will say "ouch" when pain applied but no other words spoken; not following simple one step commands, inattentive; no gaze preference appreciated; fundi not well visualized; pupils reactive 7 to 6 mm (sluggish); no blink to threat bilaterally; decreased left nlf; responds to noxious nasal stimuli; spontaneously moves right arm and leg; left arm and leg with no movement, increased tone; brisk reflexes throughout but slightly brisker on the left; bilateral up-going toes pertinent results: 09:18pm glucose-124* urea n-13 creat-0.5 sodium-135 potassium-4.2 chloride-100 total co2-23 anion gap-16 09:18pm wbc-10.9 rbc-3.50* hgb-11.0* hct-31.7* mcv-91 mch-31.4 mchc-34.7 rdw-13.9 09:18pm plt smr-very high plt count-636* 09:18pm pt-12.6 ptt-22.9 inr(pt)-1.0 09:18pm blood wbc-10.9 rbc-3.50* hgb-11.0* hct-31.7* mcv-91 mch-31.4 mchc-34.7 rdw-13.9 plt ct-636* 09:00am blood wbc-9.3 rbc-3.05* hgb-9.6* hct-28.1* mcv-92 mch-31.6 mchc-34.4 rdw-14.1 plt ct-465* 09:00am blood plt ct-465* 03:55am blood pt-12.8 ptt-22.2 inr(pt)-1.0 07:54am blood esr-7 05:35pm blood facviii-148 05:35pm blood lupus-neg 09:00am blood glucose-133* urean-17 creat-0.3* na-141 k-3.3 cl-110* hco3-21* angap-13 10:40pm blood ck(cpk)-47 12:40pm blood ld(ldh)-170 02:00am blood alt-64* ast-14 ld(ldh)-142 alkphos-95 totbili-0.3 12:40pm blood iron-92 12:40pm blood caltibc-259* hapto-303* ferritn-158* trf-199* 05:50pm blood vitb12-973* 12:40pm blood hav ab-negative igm hbc-pnd 02:00am blood anca-negative b 05:30am blood crp-0.06 02:00am blood rheufac-7 02:00am blood c3-116 c4-31 01:14pm blood hiv ab-negative 12:40pm blood hcv ab-negative 05:35pm blood lyme by western blot-pnd 03:00pm blood herpes simplex (hsv) 2, igg-pnd 03:00pm blood herpes simplex (hsv) 1, igg-pnd 05:30am blood beta-2 microglobulin-test 05:30am blood angiotensin 1 - converting -test 08:09am blood prothrombin mutation analysis-pnd 08:09am blood cardiolipin antibody- test 05:35pm blood activated protein c -pnd 01:50pm cerebrospinal fluid (csf) wbc-1 rbc-97* polys-0 lymphs-80 monos-20 01:50pm cerebrospinal fluid (csf) totprot-22 glucose-80 02:18pm cerebrospinal fluid (csf) lyme-pnd 02:18pm cerebrospinal fluid (csf) angiotensin 1 converting enzyme-pnd mri/mra brain : the diffusion images demonstrate multiple areas of restricted diffusion in both parietal-occipital regions, both frontal regions, and also along the deep watershed distributions of both hemispheres. the findings are suggestive of acute infarcts. there is also an infarct in the right posterior temporal region. there is no evidence of blood products within the infarct. no mass effect or hydrocephalus is seen. no midline shift is noted. the head mra examination is limited. flow signal is seen in both cavernous carotid arteries on the source images. no other diagnostic information could be obtained. eeg12/11:this is an abnormal portable eeg due to the presence of intermittent bursts of high amplitude delta frequency slowing that were generalized. this finding suggests deep, midline subcortical dysfunction. in addition, occasional mixed delta and theta frequency slowing was seen over the right anterior quadrant. this suggests subcortical dysfunction in this region. no epileptiform abnormalities were seen. ct chest/abd/pelvis : 1. no definite evidence for recurrence or occult malignancy. 2. stable post left upper lobectomy scarring. nodular pleural thickening along the left hemithorax, while nonspecific, also likely represents scarring. comparison to more remote priors of this region would also be helpful. 3. redemonstration of diffuse ground glass opacities in the right lung of uncertain etiology. more pronounced areas of ground glass opacity are appreciated in the right base which were not imaged on the prior study. this again may reflect smoking-related lung disease, pneumonitis, or asymmetric pulmonary edema. 4. emphysema. 5. nonspecific pleural-based nodules in the right lung. continued followup is recommended. brief hospital course: she was initially admitted to the micu, but subsequently transferred to the neuro-icu for close further workup for her multiple, bilateral strokes. in the micu she was placed on heparin which was shortly thereafter discontinued as there was not a clear indication for anticoagulation. mri/mra was repeated (previously done at outside hospital) and showed multiple areas of restricted diffusion in both parietal-occipital regions, both frontal regions, and also along the deep watershed distributions of both hemispheres. there is also an infarct in the right posterior temporal region. mra was limited study. eeg was done and showed no evidence of epileptiform activity, but did reveal bursts of generalized slowing. stroke workup consisted of completion of her hypercoagulable w/u (apc, factor , pt gene mutation, anti-phospholipid ab, cardiolipin ab, lupus anticoagulant were sent), possible cns angiitis. workup for hypercoag. state has been negative thus far although several tests are still pending at the time of discharge. workup for infectious causes was completed including: hiv (negative), ebv (negative igm), /c (negative), hepc (pending), hsv (pending), and lyme (pending), csf lyme (pending), vzv (pending), cmv (pending), toxo (pending). ppd was placed and was negative. lp was done to screen for vasculitis and showed no pleocytosis, glucose and protein were normal, csf cytology was negative for malignant cells, and csf ace level and csf lyme are still pending at time of discharge. rheumatology was consulted and followed the patient throughout her admission. esr was normal, c-anca , c3, c4, and crp were normal. she was started on high dose steroids, solu-medrol 1g iv qd for presumed vasculitis. she steadily began to improve with steroid treatment. on , she underwent cerebral angiogram which was consistent with vasculitis. given the characteristic appearance of her angiogram for cns angiitis and her response to treatment, it was felt that the most likely diagnosis was cns angiitis. brain biopsy was not done since she had been treated with steroids for 5 days and therefore the procedure would be low yield and would not likely change the course of treatment. prior to starting cytoxan, she had a ct of chest/abd/pelvis to screen for occult malignancy or recurrence of nsclc. no evidence of malignancy was seen. she was started on cytoxan on . she will continue treatment with cytoxan for 6 months at which time she should have a repeat cerebral angiogram. she will continue to receive steroids until the cytoxan is therapeutic per rheumatology. she will follow-up with dr. in the neurology clinic, in addition to her pcp and rheumatology. medications on admission: -keppra 500 -zithromax 250 qd -lexapro 10 qd -advair -pepcid 20 -asa 325 qd -zofran prn -ativan prn -morphine prn allergies: -hydrocodone -codeine -welbutrin discharge medications: 1. gabapentin 300 mg capsule sig: three (3) capsule po tid (3 times a day). 2. pantoprazole 40 mg iv q12h 3. promethazine hcl 25 mg iv q6h:prn 4. cyclophosphamide 50 mg tablet sig: 1.5 tablets po qam. 5. escitalopram oxalate 10 mg tablet sig: one (1) tablet po daily (daily). 6. acetaminophen 650 mg suppository sig: suppositorys rectal q6h (every 6 hours) as needed. 7. aspirin 325 mg tablet sig: one (1) tablet po daily (daily). 8. aluminum-magnesium hydroxide 225-200 mg/5 ml suspension sig: 15-30 mls po tid (3 times a day). 9. docusate sodium 100 mg capsule sig: one (1) capsule po bid (2 times a day). 10. senna 8.6 mg tablet sig: one (1) tablet po bid (2 times a day) as needed. 11. insulin regular human 100 unit/ml solution sig: one (1) injection asdir (as directed). 12. prednisone 50 mg tablet sig: one (1) tablet po once a day. 13. prednisone 10 mg tablet sig: one (1) tablet po once a day. discharge disposition: extended care facility: st hospital rehabilitation unit - , nh discharge diagnosis: cerebral angiitis discharge condition: stable discharge instructions: please return for all follow-up appointments take all medications as directed: take cytoxan only in the am. if you miss a dose do not take it at night. please drink at least 2 liters of fluids per day. please urinate right before going to bed and when you awake in the am. return to the er if any increased severity of your symptoms, nausea/vomitting, chest pain, shortness of breath, or fevers followup instructions: please call to schedule an appointment with dr. in the clinic please call to schedule an appointment with dr. in the clinic please schedule an appoint with your pcp please schedule an angiogram with dr. in 6 months md, procedure: spinal tap incision of lung arteriography of cerebral arteries diagnoses: anemia, unspecified chronic airway obstruction, not elsewhere classified personal history of malignant neoplasm of bronchus and lung cerebral artery occlusion, unspecified with cerebral infarction cerebral arteritis 

Answer: The patient is high likely exposed to