Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: confusion, difficulty with speech major surgical or invasive procedure: permanent ivc filter placement history of present illness: this is an 88 year old female with past medical history of hypertension, hypothyroidism, and recurrent venous thromboembolic disease who was brought to an outside hospital on one day after a fall with confusion and word finding difficulties. per the husband the patient had been dealing with dizziness for three days prior and had sustained a fall on the day prior to presentation. the details of the fall are unknown. upon arrival the patient was not oriented and had word finding difficulties. therefore, she had a head ct, which showed intraparenchymal and subarachnoid hemorrhages. she was loaded with phenytoin, given vitamin k, and then transferred to . on arrival to pt was endorsing mild headache but no other symtpoms. past medical history: -hypertension -hypothyroid -history of dvt and pe, first seven years ago with recurrent events when taken off anticoagulation -s/p bilateral total knee replacements social history: the patient had been living independently at home with her husband. she was independent for adls but not driving and rarely leaving home. family history: non-contributory physical exam: physical exam: on admission: bp: 165/80 hr:88 r18 o2sats 90 gen: wd/wn, comfortable, nad. heent: pupils: surgical, 4mm briskly reactive eoms full neck: supple. extrem: warm and well-perfused. neuro: mental status: awake and alert, cooperative with exam, normal affect. orientation: oriented to person, place, and date. language: (+) paraphrasic errors, repetition is not intact, good comprehension; naming intact. cranial nerves: i: not tested ii: surgical pupils, reactive to light, 4mm to 2mm bilaterally. visual fields are full to confrontation. iii, iv, vi: extraocular movements intact bilaterally without nystagmus. v, vii: facial strength and sensation intact and symmetric. viii: hearing intact to voice. ix, x: palatal elevation symmetrical. : sternocleidomastoid and trapezius normal bilaterally. xii: tongue midline without fasciculations. motor: normal bulk and tone bilaterally. bilateral ue tremor (baseline per family). strength full power throughout. no pronator drift. sensation: intact to light touch. toes downgoing bilaterally coordination: mildy dysmetric bilat finger-nose-finger on discharge: vs: t97.7, bp 154/78, hr 89, rr 24, o2 98% on 50% humidified mask gen: ill appearing woman, minimally responsive and often somnolent in nad heent: normocephalic, anicteric, op appears dry but pt will not open for full exam, pupils equal round and reactive to light, dobhoff tube in right nostril cv: regular rate and rhythm, 3/6 systolic murmur along right sternal border radiating to carotids, no jvd appreciated pulm: difficult exam due to grunting and transmitted upper airway sounds, no crackles, good air movement bilaterally, no focal sounds abd: protuberant, soft, no organomegaly or masses appreciated, bowel sounds appreciated extremities: warm and well perfused, trace pedal edema, +1 dp pulses, toes unmoving in extension bilaterally neuro: minimally responsive, opens eyes to voice and tracks faces, no intelligble speech, not following axial or appendicular commands, moving all four extremities spontaneously pertinent results: =================== laboratory results =================== on admission: wbc-9.8 rbc-3.92* hgb-12.6 hct-35.6* mcv-91 rdw-13.1 plt ct-168 ----neuts-74.7* lymphs-20.0 monos-4.3 eos-0.8 baso-0.4 pt-34.5* ptt-32.3 inr(pt)-3.5* glucose-132* urean-22* creat-1.1 na-138 k-2.8* cl-102 hco3-24 calcium-10.5* phos-2.8 mg-1.8 on discharge: wbc-10.1 rbc-4.08* hgb-12.7 hct-37.3 mcv-91 rdw-12.8 plt ct-180 glucose-140* urean-23* creat-0.9 na-138 k-3.3 cl-101 hco3-26 angap-14 ============== other results ============== ecg : the rhythm is initially regular and then becomes an irregular bradycardia that may be atrial fibrillation although the rhythm cannot be definitively determined in the setting of significant baseline artifact. the q-t interval is prolonged. non-specific st-t wave changes. compared to the previous tracing of the rhythm appears to have changed ct head: impression: 1. single large round intraparenchymal hemorrhage in the right temporal lobe with vasogenic edema. cgiven size and singularity, cannot exclude an underlying mass such as avm or tumor (e.g. metastasis). an mri is recommended for further evaluation. 2. possible intraparenchymal cerebellar hemorrhage next to quadrigeminal plate subarachnoid hemorrhage. 3. several foci of subarachnoid hemorrhage along the right hemisphere and in the interpeduncular fossa. subarachnoid hemorrhage layering in the right sylvian fissure. 4. small amount of blood in the occipital of the left lateral ventricle. ct c-spine:impression: 1. no evidence of acute fracture or malalignment. 2. multilevel degenerative changes in the cervical spine with posterior osteophytes at multiple levels impinging on thecal sac anteriorly. no critical canal stenosis. cta head: impression: 1. intraparenchymal, subarachnoid and intraventricular hemorrhage as described above. 2. atherosclerosis in the intracranial arterial vasculature with post-stenotic dilatation of the mid course of the vertebral artery measuring 5.4 x 5.4 mm. 3. focal outpouching in the m2 segment of right mca likely represents an infundibulum. conventional angiography may be performed to assess for an intracranial vascular abnormality to account for the intracranial hemorrhage as per clinical need. ct head: impression: 1. stable appearance of right temporoparietal intraparenchymal hemorrhage and diffuse subarachnoid hemorrhage, as detailed above. mild interval increase in ventricular hemorrhage likely due to redistribution of blood products. 2. ill-defined hyperdensity adjacent to interpeduncular fossa represents post-stenotic dilatation of the basilar artery, better characterized on cta of the same date. cxr: lungs are low in volume but aside from mild right basal atelectasis, essentially clear. the heart is at least moderately enlarged, and the thoracic aorta is generally dilated but not focally aneurysmal. there is no appreciable pleural effusion, and no pneumothorax or displaced rib fracture. eeg: impression: this is an abnormal routine eeg due to the presence of focal right hemisphere slowing suggestive of subcortical dysfunction on that side. in addition, the generalized bursts of slowing and background slowing indicate the presence of a moderate encephalopathy. no epileptiform features were seen. mri (formal read pending): prelim read per neuroradiology is of an evolving right sided intraparenchymal hemorrhage with central resolution. to fully rule out underlying mass or vascular malformation would need imaging after hematoma resolves. : ir ivc filter placement: impression: 1. inferior venacavogram demonstrating single patent ivc with no filling defects or duplication. the lower most renal vein was noted to be the left renal vein. 2. successful placement of a permanent vena tech ivc filter below the renal veins via right common femoral venous access. brief hospital course: this 88 y.o. female with history of hypertension and hypothyroidism presenting with two separate intracranial hemorrhages causing severe encephalopathy. 1) encephalopathy, precumed due to intraparenchymal and subarachnoid hemorrhages: the patient presented with imaging revealing a right sided intraparenchymal hemorrhage as well as a subarachnoid hemorrhage. per husbands report she had been complaining of dizziness for two days, sustained a fall on the preceding day, and then developed confusion and word finding difficulties. at presentation she had an expressive aphasia but an otherwise intact neurological exam by report. over the first night in the hospital, however, she developed agitation and received haloperidol and olanzpaine and was noted to be less responsive. the following morning her exam continued to be notable for her being dramatically less responsive and essentially not following commmands, only responding with nonsensical utterances, and being somnolent much of the time. neurology was consulted and recommended an eeg to evaluate for seizure as well as an mri head in order to better evaluate for underlying lesions that could predispose her to an intraparenchymal bleed. eeg showed no evidence of seizure and mri showed no likely source of spontaneous bleeding. repeat head cts showed no growth of her hemorrhages or increased edema. etiology of intraparenchymal hemorrhage was unclear as this is not in a typical location for hypertensive hemorrhage. her subarachnoid hemorrhage was thought most likely due to trauma of her fall. unfortunately, her mental status remained stablely poor for the remainder of her hospitalization from that first morning with her not following commands or responding in a meaningful way though she would track with her eyes. generally she was very somnolent but definitely less so when she had not received olanzapine for some time. neurosurgery thought no further acute management and her deficits were a result of evolution of her traumatic brain injury. she will be discharged to an ltac with dobhoff feeds and medication for blood pressure control. sbp's were managed under 160 with her home metoprolol and amlodipine as well as hydralazine prn. 2) history of venous thromboembolic disease: per family the patient has history of multiple dvts and pes and thus was on lifelong anticoagulation. this was not considered appropriate in the setting of her intracranial hemorrhages and therefore a permanent ivc filter was placed on . 3) delirium/agitation: the patient was noted to have periods of acute delirium/agitation on her underlying encephalopathy. no fever or signs of acute infection. she received occasional doses of olanzapine for this agitation with resultant worsened somnolence. she was also intermittently kept in soft wrist restraints to avoid forcible removal of her dobhoff given the need for it to maintain nutrition. 4) bradycardia/ likely tachy-brady syndrome: the patient had occasional bouts of bradycardia to the 30s while sleeping in the context of increased beta blocker doses. ecg showed bouts of irregular atrial tachycardia and then bradycardic periods. these were managed by titrating dose of beta blocker back down and patietn had been stable on discharge metoprolol dose for two days without concerning bradycardia at the time of discharge. 5) urinary retention: the patient had her foley catheter removed on but developed urinary retention again on and had this replaced. ua was sent given cloudy urine but showed no pyuria or signs of acute infection. most likely etiology of retention was thought to be olanzapine and opiate use and these were both discontinued. bowel regimen was also increased as constipation was thought to potentially contribute to urinary retention. she should have a voiding trial in days if feasible. 6) hypertension: hctz held on admission and her hypertension was conrolled with metoprolol, amlodipine, and prn hydralazine. amlodipine dose was increased prior to discharge in hopes of avoiding need for further hydralazine. 7) nutrition: given somnolence and inability to follow commands dobhoff was placed for feeds. given uncertainty about ultimate recovery and goals of care peg placement was deferred and patient will be discharged on dobhoff feeds. 8) goals of care: conversations were held with the patient's huband and daughters about the patient's poor functional status and unlikely significant recovery. the issues of aggressive care going forward and potential options including dnr/dni status and minimizing hospitalizations were discussed but the patient's husband did not feel ready to make decisions at this time. therefore, patient remains full code. 7) hypothyroidism: she was continued on her home levothyroxine dose 8) ostopenia vs osteoporosis: alendronate was stopped given lack of ability to speak or follow commands was considered high concern for pill esophagitis. the patient had ivc filter placed for pe prophylaxis given she is not considered an anticoagulation candidate. she remains on dobhoff feeds while the family gets an idea of potential for improvement and discusses ideal of goals of care. she remains full code pending further discussions with her husband. medications on admission: synthroid 50 mcg daily simvastatin 80 mg daily hydrochlorothiazide 12.5 mg daily metoprolol succinate er 25 mg daily alendronate 35 mg daily magnesium oxide 400 mg daily aspir-81 81 mg daily coumadin 2 mg daily discharge medications: 1. levothyroxine 50 mcg tablet sig: one (1) tablet po daily (daily). 2. atorvastatin 80 mg tablet sig: one (1) tablet po once a day. 3. metoprolol tartrate 25 mg tablet sig: 1.5 tablets po tid (3 times a day). 4. docusate sodium 50 mg/5 ml liquid sig: one hundred (100) mg po bid (2 times a day). 5. senna 8.6 mg tablet sig: 1-2 tablets po twice a day as needed for constipation. 6. magnesium oxide 400 mg tablet sig: one (1) tablet po daily (daily). 7. multivitamin tablet sig: one (1) tablet po daily (daily). 8. olanzapine 5 mg tablet, rapid dissolve sig: 0.5 tablet, rapid dissolve po twice a day. 9. polyvinyl alcohol-povidone 1.4-0.6 % dropperette sig: drops ophthalmic prn (as needed) as needed for dry eyes. 10. cholecalciferol (vitamin d3) 400 unit tablet sig: one (1) tablet po daily (daily). 11. amlodipine 10 mg tablet sig: one (1) tablet po once a day. 12. hydralazine 10 mg tablet sig: one (1) tablet po every four (4) hours as needed for sbp>160. 13. bisacodyl 10 mg suppository sig: one (1) suppository rectal once a day as needed for constipation. 14. milk of magnesia concentrated 2,400 mg/10 ml suspension sig: thirty (30) ml po three times a day as needed for constipation. discharge disposition: extended care facility: discharge diagnosis: primary diagnoses: -subarachnoid hemorrhage and intraparenchymal hemorrhage complicated by significant encephalopathy -history of venous thromboembolic disease on chronic anticoagulation secondary diagnoses: -hypertension -hypothyroidism discharge condition: mental status: confused - always. level of consciousness: lethargic but arousable. activity status: bedbound. discharge instructions: you were admitted after a fall with bleeding into your skull and brain. unfortunately, the bleeding was not in a place that was amenable to drainage. therefore, your blood pressure was controlled and you were monitored. due to the bleeding in your brain your blood thinning medication (coumadin) was stopped. we had a filter placed to keep clots from traveling to to your lungs now that your blood thinner was stopped. your blood pressure medications have been changed and your warfarin has been stopped. your other medications have not been changed. followup instructions: your rehabilitation facility should call ( to schedule an appointment with dr. , to be seen in 4 weeks. you will need an mri of the brain with and without contrast prior to your appointment. this can be scheduled when you call to make your office visit appointment. procedure: interruption of the vena cava enteral infusion of concentrated nutritional substances diagnoses: unspecified essential hypertension unspecified acquired hypothyroidism unspecified fall intracerebral hemorrhage cerebral edema long-term (current) use of anticoagulants retention of urine, unspecified personal history of venous thrombosis and embolism sinoatrial node dysfunction encephalopathy, unspecified aphasia subarachnoid hemorrhage following injury without mention of open intracranial wound, with no loss of consciousness physical restraints status other antipsychotics, neuroleptics, and major tranquilizers causing adverse effects in therapeutic use 

Answer: The patient is high likely exposed to