Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: sulfa (sulfonamides) attending: chief complaint: seizure, arrythmia major surgical or invasive procedure: intubation history of present illness: mr. is a 29 yo male w/o significant pmhx who presents with episode of shaking to the ed. history is obtained from girlfriend and family as patient is intubated and sedated. per girlfriend, patient was in usual state of health until 11 a.m. the morning of admission when he developed sudden onset jerking movements of his body, starting in his bilateral ue, and progressing to his entire body, with fall to the ground and head trauma. during this episode his eyes rolled back, he foamed at the mouth, and he was not conscious. his girlfriend denied associated tongue biting or urinary/fecal incontinence. this episode lasted for 2 minutes, followed by 10-15 minutes where he was confused and difficult to arouse, and had no recollection of the event. at this point she called ems, and he was transported to the ed. patient's family denies any toxins or exposures. he did not have recent travel. his girlfriend noted that he did drink 8 beers the night prior while watching the super bowl but that this was not unusual for him. she denied any recent head trauma, fevers, chills, sore throat, rhinorrhea, congestion, chest pain, nausea, vomiting, diarrhea. he did have a mild upset stomach after eating a chicken the night before, but did not have n/v. she denies that he engaged in any illicit substances, including specifically no iv drugs including cocaine. he had a normal affect, and per his girlfriend, did not endorse any symptoms of depression. his family noted that he did not have any febrile seizures in childhood, and had no medical problems growing up. while in the ed, patient was noted to have another event similar to the one noted above, and was given ativan iv to break the episode. shortly afterwards, he was also noted to have an svt and given adenosine as well, and upon breaking the ekg was consistent with wpw with avrt. patient also had an lp performed which was unremarkable, as well as a head ct. he was seen by toxicology, who considered wellbutrin overdose as etiology for seizures. past medical history: none social history: patient works as a lawyer in downtown . he drinks socially. denies drugs or tobacco. family involved and live in . family history: cad and dm2. no history of sudden death, seizure, arrhythmia physical exam: vs: 97.3, 126/64, 85, 24, 96% gen: well appearing. nad heent: perrl. mmm. ncat. cor: rrr. i/vi systolic ejection murmur at rusb. no rubs or gallops. pulm: ctab no rrw. abd: soft, nt/nd. normoactive bowel sounds. no organomegaly. ext: wwp. no cce. neuro: caox3, non-focal pertinent results: 05:18am blood wbc-7.2 rbc-4.20* hgb-12.0* hct-35.4* mcv-84 mch-28.5 mchc-33.8 rdw-12.9 plt ct-204 04:21am blood wbc-7.5# rbc-4.60 hgb-13.3* hct-38.3* mcv-83 mch-28.9 mchc-34.7 rdw-12.5 plt ct-254 01:30pm blood wbc-15.8* rbc-5.11 hgb-14.3 hct-43.6 mcv-85 mch-28.0 mchc-32.8 rdw-12.5 plt ct-361 01:30pm blood neuts-69.4 lymphs-24.8 monos-4.2 eos-1.3 baso-0.4 01:30pm blood pt-12.7 ptt-23.1 inr(pt)-1.1 04:59pm blood glucose-127* urean-11 creat-1.0 na-145 k-3.8 cl-110* hco3-28 angap-11 04:59pm blood ck(cpk)-4931* 01:25pm blood ck(cpk)-4453* 05:18am blood ck(cpk)-3582* 01:00am blood ck(cpk)-2521* 06:03pm blood alt-36 ast-54* ld(ldh)-389* ck(cpk)-1821* alkphos-44 amylase-67 totbili-1.7* 03:00pm blood ck(cpk)-1341* 04:21am blood ck(cpk)-737* 01:30pm blood ck(cpk)-138 06:03pm blood lipase-25 01:30pm blood ck-mb-4 ctropnt-<0.01 04:59pm blood calcium-8.9 phos-2.7 mg-1.8 09:32pm blood triglyc-136 01:30pm blood osmolal-307 01:30pm blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 11:22pm blood type-art po2-204* pco2-35 ph-7.42 caltco2-23 base xs-0 11:22pm blood lactate-1.2 10:42pm blood lactate-2.0 01:30pm urine color-straw appear-clear sp -1.015 01:30pm urine blood-lg nitrite-neg protein-tr glucose-neg ketone-tr bilirub-neg urobiln-neg ph-5.0 leuks-neg 01:30pm urine rbc-0-2 wbc-0-2 bacteri-none yeast-none epi-0-2 01:30pm urine bnzodzp-neg barbitr-neg opiates-neg cocaine-neg amphetm-neg mthdone-neg 04:37pm cerebrospinal fluid (csf) wbc-1 rbc-1* polys-0 lymphs-86 monos-14 04:37pm cerebrospinal fluid (csf) totprot-35 glucose-87 time taken not noted log-in date/time: 4:39 pm csf;spinal fluid #3. **final report ** gram stain (final ): no polymorphonuclear leukocytes seen. no microorganisms seen. fluid culture (final ): no growth. ct head w/o contrast 1:46 pm findings: there is no intracranial hemorrhage, mass, shift of normally midline structures, edema, hydrocephalus or major vascular territorial infarct. the -white differentiation is preserved throughout. the ventricles and sulci are normal in contour and configuration. no fractures are identified. impression: no intracranial hemorrhage. chest (portable ap) 1:38 pm impression: appropriate positioning of ett and ngt. no acute cardiopulmonary abnormalities. mr head w & w/o contrast 9:14 am mr brain without and with intravenous gadolinium: there is no evidence of hemorrhage, edema, masses, mass effect or infarction. the ventricles and sulci are normal in caliber and configuration. no diffusion abnormalities are detected. the hippocampi are symmetric in morphology, signal intensity, enhancement characteristics. there are no foci of abnormal enhancement post contrast. a moderately large air- fluid level is seen within the right sphenoid air cell. there is moderate mucosal thickening in bilateral ethmoid air cells. slow flow within the right jugular bulb is incidentally noted. the remaining vascular flow patterns are within normla limits. a small thornwaldt cyst measures 5 mm. impression: unremarkable mri brain. sinusitis. tte : conclusions the left atrium and right atrium are normal in cavity size. left ventricular wall thicknesses and cavity size are normal. regional left ventricular wall motion is normal. overall left ventricular systolic function is low normal (lvef 50-55%). transmitral and tissue doppler imaging suggests normal diastolic function, and a normal left ventricular filling pressure (pcwp<12mmhg). there is no left ventricular outflow obstruction at rest or with valsalva. right ventricular chamber size and free wall motion are normal. the aortic valve leaflets (3) appear structurally normal with good leaflet excursion and no aortic regurgitation. the mitral valve appears structurally normal with trivial mitral regurgitation. there is no mitral valve prolapse. there is no systolic anterior motion of the mitral valve leaflets. the pulmonary artery systolic pressure could not be determined. there is no pericardial effusion. impression: normal left ventricular cavity size with low normal systolic function. eeg study date of impression: abnormal portable eeg due to the occasional bursts of irregular slowing in the left temporal region and due to the single burst of sharp theta slowing. the left temporal slowing suggests a focal subcortical abnormality in the left hemisphere, but the tracing cannot specify its etiology. the burst of sharp activity reflects a deeper midline or generalized process. it included sharp features but no overtly epileptiform abnormalities. the background often reflected drowsiness. the cardiac monitor showed an intermittently irregular rhythm. brief hospital course: a/p: mr. is a 29 yo m w/o significant pmhx who presents with tonic-clonic seizure activity and avnrt with wpw. 1. generalized tonic-clonic seizures - patient without a clear etiology including no history of significant alcohol use, other ingestions, cns masses, or traumatic brain injury. urine and serum tox screens negative, including a negative etoh level. osmolal gap wnl (3.5). lp performed in the ed did not show evidence of infection. - neurology consultation - eeg/mri as above - seizure precautions were used in house, but no further seizures - keppra 1000 mg load, 500 mg per neurology recommendations, until then to 750 until with 1000mg - toxicology consultation - rhabdomyolysis presumably from seizure - patient informed he can not drive for 6 months from last seizure. 2. altered mental status - intubated in ed for airway protection in setting of tonic-clonic seizures. no evidence of respiratory failure. - propofol gtt for sedation while intubated - weaned 3. -parkinson-white with avnrt. - patient with episode of avnrt requiring adenosine while having a tonic-clonic seizure. likely developed arrhythmia in setting of stress. - post resolution of avnrt noted with delta-waves and short pr interval - monitored on telemetry, with episodes of bradycardia with wpw morphology - repeat ekg with no changes - planned ablation with dr. - ep consultation medications on admission: none discharge medications: 1. keppra 250 mg tablet sig: two (2) tablet po twice a day: increase to 3 tabs on , then to 4 tabs . disp:*120 tablet(s)* refills:*2* 2. toprol xl 25 mg tablet sustained release 24 hr sig: one (1) tablet sustained release 24 hr po once a day. disp:*30 tablet sustained release 24 hr(s)* refills:*0* discharge disposition: home discharge diagnosis: generalized seizure -parkinson-white syndrome arrythmia rhabdomyolysis discharge condition: good discharge instructions: return to the hospital with seizures, confusion, fevers/chills, palpitations, chest pain. you should not drink any alcohol, as this can start a seizure you should not engage in activities which either a seizure or arrythmia could be dangerous such as sports, standing on heights. under state law you may not drive a car for 6 months from your last seizure. you will be returning to the medical center to have a cardiac arrythmia ablation procedure. followup instructions: provider: , md phone: date/time: 8:00 8 () dr. 2/21/08@0 for cardiac ablation ( - you will be contact for details) procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours spinal tap incision of lung insertion of endotracheal tube diagnoses: acidosis other convulsions personal history of tobacco use other specified cardiac dysrhythmias encephalopathy, unspecified hyperosmolality and/or hypernatremia rhabdomyolysis unspecified sinusitis (chronic) anomalous atrioventricular excitation 

Answer: The patient is high likely exposed to