Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: mrs. is an 81 year old woman with a history of type 2 diabetes mellitus and seizure disorder without known coronary artery disease presenting initially to hospital with neck pain. she was found to have an elevation and was transferred to the for primary percutaneous transluminal coronary angiography. the patient initially developed neck and jaw pain at 07:30 the morning of admission. she reported nausea as well as shortness of breath and anxiety. she denied fever or chills, lightheadedness or dysuria. at the outside hospital, she was found to be hypotensive with a blood pressure of 68/24 and a heart rate of 58. ekg showed anterior elevations. she was placed on integrilin, heparin, aspirin, and atropine, and transported to . she had also been placed on a dopamine drip at the outside hospital. on the patient's arrival, a diagnostic angiogram was notable for critical left main coronary artery disease. the patient became hypotensive, requiring an increase in her dopamine dose and placement of an intra-aortic balloon pump. emergent cardiac surgery consult was obtained, however, it was felt that the patient was a poor candidate for a coronary artery bypass graft given her integrilin bolus and cardiogenic shock. the patient thus underwent a left main intervention as well as a proximal left anterior descending artery intervention. after this the patient was stabilized hemodynamically and transferred to the coronary care unit. past medical history: 1. type 2 diabetes mellitus. 2. seizure disorder. 3. hypothyroidism. allergies: no known drug allergies. medications: 1. glucotrol 10 mg p.o. twice a day. 2. synthroid 50 micrograms p.o. q. day. 3. dph, 100 mg p.o. three times a day. physical examination: in general, the patient was an 81 year old woman, awake, alert, in no acute distress. temperature is 95.5 f.; heart rate 85; blood pressure 116/60; respiratory rate 19; oxygen saturation 97% on five liters face mask. heent: normocephalic, atraumatic. pupils equally round and reactive to light. oral mucosa was moist. the neck was supple with a 2.0 by 5.0 palpable hematoma and no bruit. the chest was clear to auscultation bilaterally. cardiovascular examination indicated distant heart sounds; normal s1 and s2, no murmurs, rubs or gallops. the abdomen was soft and nontender, not distended with normal bowel sounds. extremity examination indicated no edema; peripheral pulses were doppler-able. neurologic examination was nonfocal. laboratory: on admission indicated a white blood cell count of 20.0, hematocrit of 31.9, platelets of 246. chem-7 was remarkable for a bicarbonate of 17, bun of 35, creatinine of 1.3 and glucose of 297. magnesium was 1.5. ck was 8,301 with an mb fraction of 870 and an mb index of 10.5. electrocardiogram indicated normal sinus rhythm, elevations at leads v3 through v5. a subsequent electrocardiogram on arrival to indicated a new right bundle branch block and leftward axis. cardiac catheterization indicated a 90% proximal left main coronary artery lesion, diffusely diseased left anterior descending artery with a total occlusion beyond a small first septal. the obtuse marginal 2 was diffusely diseased with a maximal stenosis of 70% proximally. the dominant right coronary artery was diffusely diseased with a maximum stenosis of 60% proximally and 50% in the mid-vessel and it was totally occluded distally. resting hemodynamics of same after placement of the intra-aortic balloon pump revealed a mean rate atrial pressure of 7 mm of mercury with mild pulmonary hypertension with pulmonary artery pressures of 38/17. left sided filling pressures were severely elevated with a mean pulmonary capillary wedge pressure of 22 mm of mercury and a pulmonary capillary wedge pressure a wave of 26 mm of mercury. cardiac output was mildly depressed with cardiac index of 2.3 liters per minute per liter squared. echocardiogram indicated mild symmetric left ventricular hypertrophy with an apical left ventricular aneurysm, ejection fraction of 30% and severely hypokinetic anterior septum and anterior free wall. there was moderate hypokinesis of the inferior septum and lateral wall. there is extensive apical akinesis and dyskinesis. hospital course: the patient was admitted to the coronary care unit following cardiac catheterization and placement of an intra-aortic balloon pump. on hospital day number one, she was started on aspirin, plavix, heparin and integrilin drip. she was also started on lipitor. cks continued to cycle and were trending down. digoxin was held given the patient's potential arrhythmia risk. on hospital day number two, the patient was noted to have an expanding right internal jugular hematoma with a hematocrit drop of 31 to 28. she was transfused one unit of packed red blood cells. dopamine was weaned and integrilin drip was discontinued after 18 hours. on hospital day number three, the patient's urine output was noted to drop to 18 cc per hour, however, her urine output improved in response to fluid boluses. her fractional excretion of sodium was found to be 1.1% and she was thought to have a possible type 4 renal tubular acidosis. her intra-aortic balloon pump was changed from 2 to 1. on hospital day number four, the patient was noted to have a sudden episode of hypoxia, hypotension and increased pulmonary artery pressure to the 70s over 30s as well as st elevations above her baseline in the interior leads consistent with acute pulmonary edema. she was given lasix, morphine, intravenous nitroglycerin and sublingual nitroglycerin, all without relief. arterial blood gas indicated a ph of 7.05, paco2 of 31, pao2 of 8.2. her potassium was found to be 7.4. ekg indicated new wide-complex tachycardia consistent with aivr. the patient was emergently intubated and started again on dopamine drip as well as levophed drip. she was given calcium chloride, insulin and bicarbonate. after being evaluated by the interventional cardiology fellow, the patient was taken emergently to the catheterization laboratory with a concern over stent re-stenosis and left anterior descending dissection. an emergent echocardiogram obtained prior to the re-catheterization, indicated left ventricular dysfunction without any acute mechanical event. catheterization indicated a patent left main stent as well as a 25 mm dissection in the left anterior descending artery. the left circumflex artery presented for a dissection and flap at the origin. the patient had timi-iii flow in her left anterior descending and left circumflex arteries, and intra-aortic balloon pump was reinserted. ck was found to be 665 with an mb fraction of 21. the patient was maintained on pressors and intra-aortic balloon pump and continued on pressors. lasix was continued for diuresis. on hospital day number five, the patient became febrile with a temperature of 101.3 f.; she was started on vancomycin, metronidazole, and levofloxacin. chest x-ray indicated a possible left lower lobe infiltrate. blood cultures were sent and found to be positive in one out of four bottles for gram positive pairs and clusters. sputum culture was sent and found to be growing out moderate staphylococcus aureus. repeat ekg indicated a new left bundle branch concerning for possible new ischemia. on hospital day number five, the patient was noted to have an asystolic arrest on telemetry. she was given 1 mg of atropine and transcutaneous pacing with restitution of heart rate to 100 and systolic blood pressure of 150. subsequent ekg indicated an ivr with a rate of 85. her blood pressure stabilized and she was weaned off of transcutaneous pacing. dopamine drip was reduced. the asystolic arrest was thought to be secondary to respiratory compromise and possible mucous plugging. a family meeting was held on hospital day number six and given the patient's extremely poor prognosis, the decision was made to discontinue pressors and reduce the intra-aortic balloon pump to 1 to 4. the patient was also made "do not resuscitate". on hospital day number seven, the patient had a second asystolic arrest for a total of ten minutes. she was unresponsive to voice and painful stimuli. her pupils were not reactive to light. no heart sounds or breath or lung sounds were heard for one minute and the patient was pronounced deceased at 10 p.m. on . the family and attending physician were notified. the family declined a post-mortem examination. discharge diagnoses: 1. coronary artery disease. 2. large anterior wall myocardial infarction. 3. respiratory failure secondary to cardiogenic shock. 4. type 2 diabetes mellitus. 5. seizure disorder. 6. hypothyroidism. condition at discharge: deceased. dr., 12-462 dictated by: medquist36 procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of non-drug-eluting coronary artery stent(s) combined right and left heart cardiac catheterization combined right and left heart cardiac catheterization coronary arteriography using two catheters injection or infusion of platelet inhibitor implant of pulsation balloon implant of pulsation balloon diagnoses: acidosis congestive heart failure, unspecified acute kidney failure, unspecified hematoma complicating a procedure accidental puncture or laceration during a procedure, not elsewhere classified acute myocardial infarction of anterolateral wall, initial episode of care pneumonitis due to inhalation of food or vomitus cardiogenic shock 

Answer: The patient is high likely exposed to