Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: hearing loss, visual disturbance, basilar thrombosis major surgical or invasive procedure: cerebral angiogram with mechanical thrombectomy history of present illness: 37-year-old woman with basilar clot, in the context of no known medical problems (yet known). she woke with severe headache at 3:20 am. she was near bottom of bed. asked husband for bucket, feeling as would vomit. only two drinks last night. she said she can't see and hear (but able to hear husband). vision affected on left. by time ems arrived, was vomiting, she could barely talk at this time, around 4 am. she had been gesturing as if whole head was painful, but husband thinks may have said at the back of her head. she was still able to move everything, including fingers and toes, while en route. she continued to vomit. she was never able to get out of bed and seems as if she could not walk. taken to . on arrival, deteriorated, becoming comatose. intubated, paralysed (pancuronium) and heparin (bolus 3900 units, running 800) at around 4:45 am. cta performed at with occluded left vert and cut of at midpontine basilar and reconstitution at high pontine/mesencephalic basilar. concern for basilar thrombus in situ, therefore to for further mx. labs normal at . went to chiropractor two weeks ago - scoliosis - full body adjustment. no known pregnancy (and test negative). no trauma to head or neck otherwise. no prior clots. past medical history: - no medical problems - cholecystectomy at 19 years - liposuction one year ago - vein stripping social history: lives with husband and 2 children. works as third grade teacher. no drugs, smoking, drugs. she runs and does cross-fit (last 9 am yesterday). husband says very fit. family history: her father died aaa at 71, also pvd. mother is well. pgm died 71, was diabetic. pgf cancer. mgm diabetes, heart disease. pgf heart failure (enlarged, possible rheumatic fever, died 46), emphysema, smoker. physical exam: physical exam on admission: vitals: hr 44-50, bp 110s, cmv 14 x 400 fio2 0.4, afebrile on cmv, breathing spontaneously on cpap after propofol stopped - regular, possibly apneustic. general appearance: fit and healthy appearance, no stigmata of chronic disease. heent: nc, intubated. neck: supple. lungs: cta/vent sounds cardiac: regular bradycardic. abdominal: soft extremities: cool. peripheral pulses 2+. neurologic: mental status: comatose. cranial nerves: i: not tested. ii: fixed pupils, small. iii, iv, vi: no doll's eyes. v, vii: corneal sluggish on left, later also sluggish on right. viii: hearing not tested. ix, x: gag intact. : not tested. xii: not testable. tone initially normal in arms, with extensor increase in legs. later increased in arm and more in legs, fingers flexed. power only evaluable as reflexes, pain, shiver - full in those regards. reflexes increased throughout. toes triple flexion into painful stimuli. sensation intact pain. physical exam on discharge: general: awake and alert, nad heent: ncat, mmm, sclerae anicteric cv: rrr, no murmurs lungs: ctab abdomen: benign ext: no edema neurologic: mental status: awake and alert, oriented x 3, speech fluent with moderate dysarthria, follows commands well. cranial nerves: i: not tested. ii: pupils 4mm-->2mm bilaterally iii, iv, vi: eomi with sustained nystagmus maximal on leftward gaze, +saccadic intrusions on smooth pursuit v: sensation intact bilaterally vii: r lower facial droop with weakness of r eye closure as well viii: hearing decreased on r ix, x: palate elevates symmetrically : scm and trapezius full strength b/l xii: tongue midline motor: d b t we wf fe fab | ip q h at g/ tf r 0 0 0 3 2 2 0 | 2 5 1 0 0 0 0 l 5 5 5 5 5 5 5 | 5 5 5 5 5 5 5 reflexes: 2+ and symmetric throughout. patellar response flexor on l, extensor on r. sensation: intact to light touch and pinprick throughout. coordination: intact on l, unable to assess on r gait: deferred pertinent results: 11:11pm ptt-76.6* 03:51pm ptt-47.7* 03:50pm glucose-114* urea n-11 creat-0.6 sodium-141 potassium-4.2 chloride-108 total co2-22 anion gap-15 03:50pm calcium-8.4 phosphate-4.1 magnesium-1.7 03:00pm urine hours-random 03:00pm urine bnzodzpn-pos barbitrt-neg opiates-neg cocaine-neg amphetmn-neg mthdone-neg 03:00pm urine color-yellow appear-clear sp ->1.050* 03:00pm urine blood-tr nitrite-neg protein-tr glucose-neg ketone-neg bilirubin-neg urobilngn-neg ph-7.5 leuk-neg 03:00pm urine rbc-10* wbc-0 bacteria-none yeast-none epi-0 07:56am creat-0.9 07:56am estgfr-using this 07:47am glucose-132* na+-142 k+-3.6 cl--104 tco2-26 07:40am urea n-17 07:40am hcg-<5 07:40am wbc-8.1 rbc-4.45 hgb-13.2 hct-40.0 mcv-90 mch-29.6 mchc-32.9 rdw-12.7 07:40am neuts-86.7* lymphs-10.4* monos-2.0 eos-0.5 basos-0.4 07:40am plt count-359 07:40am pt-12.2 ptt->150* inr(pt)-1.1 cta head and neck : conclusion: intraluminal thrombus occluding the basilar artery. poor and then no opacification of the distal left vertebral artery with its most distal portion apparently filling retrograde from the basilar. severe stenosis of the proximal left vertebral artery, perhaps representing a dissection. cerebral angiogram : impression: 1. technically successful intra-arterial mechanical thrombectomy of a mid basilar clot. 2. recanalization of the entire basilar segment with a possible small thrombus in the right pca which was not flow limiting. ct head : impression: new hyperdense material visualized mostly centered in the pons involving the inferior mid brain as well as superior medulla. these findings are suspicious for hemorrhagic conversion at the site of prior thrombus. close clinical followup as well as imaging followup is recommended. mra head and neck : conclusion: bilateral pontine and cerebellar infarctions. severe stenosis of the proximal left vertebral artery, worrisome for dissection. wall thickening with hyperintense material in the distal cervical left vertebral artery, also worrisome for dissection. no other vascular stenoses are detected. tte : impression: possible patent foramen ovale.no other structural heart disease or pathologic flow identified. if clinically indicated, a tee with saline contrast/maneurvers is suggested to better define the interatrial septum ct head : impression: 1. interval resolution of previously seen hyperdensity in the left paramedian pons, indicating that this was due to contrast extravasation. no new intracranial hemorrhage. 2. evolving infarcts in bilateral pons, and cerebellar hemispheres and right cerebellar vermis, better seen in the prior mri study of . brief hospital course: ms. was admitted to the neurology service on as a transfer from an osh due to concern for basilar artery thrombosis. on initial exam she was comatose and responding only to pain with increased tone in her lower extremities and triple flexion to noxious stimulation. repeat cta confirmed basilar thrombosis with severe stenosis of the proximal left vertebral artery concerning for dissection as well as distal left vertebral artery thrombosis. she was taken urgently to the angiography suite for clot retrieval. the procedure was successful, with recanalization of the entire basilar segment with a possible small thrombus in the right pca which was not flow limiting. she was monitored closely in the neuro-icu following the procedure. her mental status improved and she was successfully extubated. on subsequent exam she was awake and alert, speech was dysarthric but otherwise mental status was normal, and she had a residual right facial droop and right hemiparesis. mri showed left pontine infarct with some extension to the right along with bilateral cerebellar infarcts. she was transferred to the neurology floor on . neuro: she was maintained on a heparin drip with a goal ptt of 50-70 and was started on long-term anticoagulation with coumadin to treat her vertebral artery dissection. she was maintained on ivf to keep her systolic blood pressure > 110. a tte was performed which showed a possible pfo. tee was deferred as it would not change management and would require holding her heparin drip. this should be performed as an outpatient to further evaluate for possible pfo. she will also have a full hypercoagulability work-up performed as an outpatient. lipid panel was at goal and hba1c was 5.0%. she was followed by pt, ot, and speech therapy who recommended rehab placement upon discharge. cardiovascular: she was maintained on telemetry throughout her admission. bp was monitored closely as above to maintain adequate cerebral perfusion. troponin was initially slightly elevated to 0.02; this was trended and subsequently normalized. tte showed a possible pfo as above; she will need a tee as an outpatient. nutrition: she was seen by speech therapy and cleared for a regular diet with no restrictions. prophylaxis: she was maintained on a heparin drip and pneumoboots for dvt prophylaxis. she was maintained on senna and colace for bowel prophylaxis. code status: she indicated that she would like to be full code on this admission. transitional care issues: she will need to be maintained on a heparin drip with goal ptt 50-70 (checked q6 hrs) until inr is therapeutic . she will need intensive pt, ot, and speech therapy to regain her prior level of functioning. she will need a tee and a hypercoagulability work-up as an outpatient for further investigation as to the etiology of her stroke (although at this point appears to be most likely due to traumatic vertebral dissection). she has a follow up appointment with dr. in stroke clinic on . medications on admission: none discharge medications: 1. warfarin 5 mg tablet sig: one (1) tablet po once daily at 4 pm. 2. heparin (porcine) in d5w 25,000 unit/250 ml parenteral solution sig: as directed intravenous asdir (as directed): initial infusion rate: 1150 units/hr adjust for target ptt 50-70. please draw q6h ptt. to be continued until inr . 3. artificial tears ointment sig: one (1) ophthalmic 1 application both eyes as needed for dry eyes. discharge disposition: extended care facility: hospital - discharge diagnosis: basilar thrombosis left vertebral artery dissection left pontine and bilateral cerebellar strokes discharge condition: mental status: clear and coherent. level of consciousness: alert and interactive. activity status: ambulatory - with assistance. neurologic: dysarthric speech, r lower facial droop, r hemiparesis with some preserved movement of fingers and quadriceps discharge instructions: dear ms. , you were admitted to the neurology service on after experiencing hearing loss, vision problems, and facial droop. you were found to have a blood clot in your basilar artery, likely resulting from a left vertebral artery dissection that may have been related to trauma from intense exercise. you underwent angiography with clot retrieval and improved significantly. you were started on a blood thinner called coumadin and will need to remain on iv heparin until your level is high enough. you will need intensive rehabilitation to regain your strength, given your residual right sided weakness and difficulty speaking. we made the following changes to your medications: started coumadin 5mg daily started heparin iv to be continued until your coumadin level is high enough it was a pleasure taking care of you during your hospital stay. we wish you the best in your recovery! followup instructions: you have the following appointment scheduled to see dr. in our stroke clinic: provider: , .d. phone: date/time: 3:30 you should also make an appointment to follow up with your primary care physician 1-2 weeks. md, procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours arteriography of cerebral arteries procedure on single vessel endovascular removal of obstruction from head and neck vessel(s) diagnoses: ostium secundum type atrial septal defect late effects of cerebrovascular disease, hemiplegia affecting unspecified side coma occlusion and stenosis of basilar artery with cerebral infarction dissection of vertebral artery 

Answer: The patient is high likely exposed to