Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: s/p collapse major surgical or invasive procedure: intubation central venous line placement history of present illness: yo f hx iddm, htn who presented after she collapsed in the lobby of her building while awaiting her son to pick her up for appointment to see her pcp. was reportedly feeling well recently according to son, no infectious symptoms. she came down to the lobby and sat in a chair while waiting and then collapsed. pt had cpr initiated from bystanders (rn and aide in lobby), per report ems on presentation noted vfib, was defibrillated 200j x1. review of provided strip appears to demonsrate a nsr with artifact followed by shock and resumption of nsr. fs was 188, unresponsive, intubated in the field. pt hypotensive to 60's on arrival to ed, rec'd 1.5 l ns, started on levophed transiently. pt had head, chest/abd cts performed which were unrevealing. past medical history: iddm c/b retinopathy, neuropathy htn h/o frontal lobe meningioma - resected in s/p hemorroidectomy s/p t and a social history: lives by herself, independently, no prior hx of tobacco. family history: nc physical exam: vs: t 93.4, bp 156/60, hr 58 , rr 16, o2 % on gen: elderly female, sedated, intubated, unresponsive. heent: pupils 2mm, nonreactive. cv: rrr nl s1, s2, no m/r/g chest: breath sound b/l abd: soft, nd, no hsm ext: 2+ r pedal edema, palpable dp and pt pulses b/l skin: no stasis dermatitis, ulcers, scars, or xanthomas. pertinent results: 02:46pm blood wbc-7.4 rbc-3.00* hgb-9.6* hct-28.7* mcv-96 mch-32.2* mchc-33.6 rdw-15.1 plt ct-265 02:46pm blood pt-12.2 ptt-25.8 inr(pt)-1.0 07:15pm blood glucose-216* urean-27* creat-0.9 na-141 k-4.1 cl-108 hco3-22 angap-15 07:15pm blood ck-mb-10 mb indx-5.2 ctropnt-<0.01 06:00am blood ck-mb-9 ctropnt-0.02* 07:15pm blood ck(cpk)-192* 06:00am blood ck(cpk)-131 04:10pm blood alt-12 ast-13 alkphos-41 totbili-0.5 02:34am blood calcium-8.8 phos-4.1 mg-2.6 09:55pm blood caltibc-226* vitb12-325 ferritn-158* trf-174* 04:10pm blood tsh-0.80 04:10pm blood free t4-1.0 04:10pm blood cortsol-42.3* 02:46pm blood asa-neg ethanol-neg acetmnp-14.5 bnzodzp-neg barbitr-neg tricycl-neg 02:48am blood type-art po2-88 pco2-35 ph-7.34* caltco2-20* base xs--5 cxr: no priors available for comparison. today's examination is markedly limited by low lung volumes causing crowding of the bronchovascular structures. the right hemidiaphragm is asymmetrically elevated in comparison to the left hemidiaphragm. there is marked distention of the stomach and abdominal bowel likely related to resuscitative effort. slightly increased spacing between the gastric bubble and the left hemidiaphragm may suggest a component of subpulmonic effusion. there is an ill-defined retrocardiac opacity which may reflect atelectasis and/or sequelae of aspiration. endotracheal tube terminates 4.7 cm from the carina. the aorta is slightly ectatic and calcified and there is multilevel degenerative changes of the spine. no pneumothorax or large effusions are identified. impression: 1) appropriately positioned endotracheal tube. gaseous distention of stomach and bowel likely related to resuscitative efforts. ngt may be of benefit. 2) ill-defined retrocardiac opacity may represent atelectasis and/or sequelae from aspiration. cta torso: 1. no etiology for acute arrest identified. no pe or aortic dissection. mild dilatation of the right main pulmonary artery may suggest underlying pulmonary arterial hypertension. 2. moderate amount of secretions distal to the endotracheal tube within the trachea proximal to the carina may place the patient at risk for aspiration. 3. non-obstructive left renal calculi and simple left renal cyst. 4. incompletely characterized right adrenal lesion, likely benign on patient of this age. hypoattenuating right thyroid lesion also likely benign in a patient's age. 5. cholelithiasis without evidence of acute cholecystitis. 6. extensive vasculopathy. is there a history of diabetes? tte: the left atrium is elongated. the interatrial septum is aneurysmal. left ventricular wall thicknesses are normal. the left ventricular cavity is small. left ventricular systolic function is hyperdynamic (ef 70-80%). tissue doppler imaging suggests an increased left ventricular filling pressure (pcwp>18mmhg). there is no ventricular septal defect. right ventricular chamber size and free wall motion are normal. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. trace aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. there is no mitral valve prolapse. trivial mitral regurgitation is seen. the left ventricular inflow pattern suggests impaired relaxation. the tricuspid valve leaflets are mildly thickened. the supporting structures of the tricuspid valve are thickened/fibrotic. the pulmonary artery systolic pressure could not be determined. there is no pericardial effusion. eeg: this is an abnormal eeg due to the existence of an active area of epileptogenesis in the left posterior frontal to anterior temporal region. though this activity did not meet criteria for focal status epilepticus, there were periods of apparent electrographic seizures without evident clinical correlate. the presence of a slow and disorganized background is consistent with a moderate encephalopathy of toxic, metabolic, or anoxic etiology. ct c-spine w/o contrast 9:17 pm 1. no fracture or malalignment is detected. 2. diffuse calcification of the transverse ligament adjacent to the dens causes severe canal narrowing and cord impingement. the thecal sac measures approximately 5 mm at this area. 3. hyperdense focus within the lower pole of pons and upper medulla, might represent intraparenchymal hemorrhage or cavernoma. 4. bilateral thyroid nodules which can be further evaluated on nonemergent basis. ct head w/o contrast 9:15 pm small intraventricular hemoorhage in the occipital horns and small focus of hemorrhage in the upper medulla. however, the cause of this is uncertain from the present study. a close follow-up examination or mr head without and with iv contrast, would be helpful. mri head/cspine 1-cm hemorrhagic non-enhancing lesion of the caudal medulla with expansion and edema. this finding may represent a hemorrhagic infarct versus a cavernoma. given the findings on the concurrent mr cervical spine, a hemorrhagic infarct is favored. hemorrhagic neoplasm is thought to be less likely given the lack of contrast enhancement and no prior history of cancer. multiple extra-axial partially calcified enhancing masses as described above consistent with meningiomas. intraventricular blood as before. brief hospital course: the patient presented with syncope of unknown etiology, but likely secondary to ischemia/hemorrhage in her caudal medulla and also found to have cervical lesion and edema causing canal stenosis and compression. she was initially treated with cooling protocol due to possible cardiac arrest, but found to be in sinus rhythm. patient was intubated and not breathing spontaneously. initially, patient evaluated by ct head on that showed no intracranial hemorrhage or mass. unable to do mri as patient with staples from prior meningioma surgery from . we did daily neurologic assessments to follow recovery s/p cooling protocol, showing patient was awake, sometimes tracking with her eyes, with some facial movements, but not moving any extremities. repeat ct head/spine on showing multiple hemorrhages including a lesion in her caudal medulla and edema/mass around her cervical spine. neurology was following and after speaking to neuroradiology, patient was deemed safe to have mri evaluation. on , patient evaluated by mri which showed same findings. read of mri showing two hemorrhagic/ischemic lesions of the caudal medulla and cervical cord with severe cord compression. no indication for any surgical intervention per neurosurg and neurology.patient trialed twice on psv with no spontaneous respirations. one week after insult, family meeting arranged to discuss poor prognosis given lack of recovery and goals of care. family made decision not to remove care but to place patient on spontaneous breathing trial without reinstating intubation. the patient failed the the sbt and expired 8 minutes afterwards. medications on admission: klonopin insulin discharge medications: expired discharge disposition: expired discharge diagnosis: expired discharge condition: expired discharge instructions: expired followup instructions: expired procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more diagnoses: unspecified essential hypertension diabetes with neurological manifestations, type ii or unspecified type, not stated as uncontrolled polyneuropathy in diabetes intracerebral hemorrhage acute respiratory failure hypotension, unspecified ventricular fibrillation diabetes with ophthalmic manifestations, type ii or unspecified type, not stated as uncontrolled background diabetic retinopathy quadriplegia, unspecified late effects of cerebrovascular disease, other paralytic syndrome, bilateral vascular myelopathies 

Answer: The patient is high likely exposed to