Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: phenothiazines / heparin agents attending: chief complaint: massive hemoptysis, osh transfer major surgical or invasive procedure: bronchial artery embolization x 2 intubation central line placement arterial line placement multiple bronchoscopies, flex and rigid history of present illness: mr. is a 79 yo male with severe copd, who has been in hospital for the past 12 days getting treatment with steroids for a copd flare after presenting with sob and yellow sputum production, sob, wheezing, and chest tightness, now being transferred for management of hemoptysis. he was started on iv steroids (40 to 80mg during his osh stay), nebs, and antibiotics (vanc/zosyn initially). he was started on fondaparinux for history of hit. he was also diuresed. he has a very slow course of improvement and actually worsened in terms of his dyspnea in the past few days. his steroids were increased. . 24 hrs prior to transfer, mr. developed hemoptysis, initially mild, but then more significant. he was transferred to the icu and intubated for bronchoscopy, which he underwent on the morning of transfer. significant bleeding was found from the posterior segment of the rul, though there was no endobronchial lesion identified. epinephrine and saline irrigation was not successful successful in controlling the bleeding, however it subsequently bleeding improved prior to transfer. his hct was reported as stable at 35.9 on the day of transfer and he has not required transfusion. he is stable on prvc tv 450, peep 5, fio2 80%, rr 14. he was also placed got vancomycin for mrsa found in a sputum culture. bcx have been negative and he has been afebrile. before the bronch, his wbc was 18-21, afterwards it was 31, in the setting of steroids. his cxr shows an infiltrate on rul. . . on the floor, he is intubated. he opens eyes to voice and responds to simple commands. past medical history: - copd on home o2, frequent exacerbations/intubations early pfts: fev1 37%. severely reduced dlco of 18% of predicted. - cor pulmonale with peripheral edema - muliptle admission sin the past year with copd exacerbations - chf: dchf vs cor pulmonale - pulm htn - hiatal hernia - ventral hernia - prostate ca - cholelithiasis - pancreatitis social history: married, lives with his wife. has 2 sons. history of heavy etoh and tobacco use, none currently - quit tobacco 10 yrs ago. he was a professional boxer and owns his own painting business family history: nc, no history of lung disease physical exam: vitals: t: 97.6 bp: 90/53 p: 123 r: 26 o2: 95% on 80% fio2 general: intubated, sedated. opens eyes to voice and squeezes hands and wiggles toes to command heent: sclera anicteric, mmm, oropharynx clear. pupils pinpoint and minimally reactive neck: supple, jvp not elevated, no lad lungs: diffuse expiratory rhonchi, no wheezes, rales cv: tachy, normal s1 + s2, no murmurs, rubs, gallops abdomen: soft, non-tender, non-distended, bowel sounds present, no rebound tenderness or guarding, no organomegaly gu: no foley ext: + le edema. warm, well perfused, 2+ pulses, no clubbing, cyanosis pertinent results: 10:46pm blood wbc-38.6* rbc-4.27* hgb-12.9* hct-38.8* mcv-91 mch-30.2 mchc-33.1 rdw-17.7* plt ct-232 08:26am blood wbc-41.0* rbc-3.64* hgb-11.0* hct-33.0* mcv-91 mch-30.2 mchc-33.3 rdw-17.8* plt ct-184 02:25pm blood hct-29.7* 09:31pm blood hct-26.4* 03:06am blood wbc-27.4* rbc-2.82* hgb-8.7* hct-25.6* mcv-91 mch-30.7 mchc-33.9 rdw-18.2* plt ct-138* 09:05am blood hct-25.4* 04:00pm blood hct-25.9* 10:26pm blood hct-23.2* 06:01am blood wbc-24.2* rbc-3.00* hgb-9.2* hct-27.1* mcv-90 mch-30.5 mchc-33.8 rdw-18.0* plt ct-116* 04:31am blood wbc-14.3* rbc-2.22*# hgb-6.7*# hct-19.7*# mcv-89 mch-30.3 mchc-34.1 rdw-18.2* plt ct-86* 05:35am blood wbc-14.6* rbc-2.20* hgb-6.8* hct-20.0* mcv-91 mch-30.8 mchc-33.7 rdw-19.1* plt ct-77* 10:59am blood hct-32.0*# 02:37pm blood wbc-16.1* rbc-3.36*# hgb-10.3*# hct-29.6* mcv-88 mch-30.8 mchc-35.0 rdw-18.0* plt ct-99* 10:46pm blood pt-11.7 ptt-22.7 inr(pt)-1.0 04:31am blood fibrino-400 02:37pm blood fibrino-425* 10:46pm blood glucose-196* urean-30* creat-1.0 na-141 k-4.6 cl-102 hco3-32 angap-12 08:26am blood glucose-155* urean-41* creat-1.2 na-142 k-4.6 cl-106 hco3-29 angap-12 03:06am blood glucose-124* urean-48* creat-1.5* na-141 k-4.2 cl-105 hco3-29 angap-11 06:01am blood glucose-154* urean-57* creat-1.5* na-138 k-4.3 cl-104 hco3-31 angap-7* 04:31am blood glucose-137* urean-54* creat-1.2 na-141 k-4.4 cl-106 hco3-31 angap-8 02:37pm blood glucose-112* urean-51* creat-1.1 na-141 k-4.3 cl-104 hco3-31 angap-10 . chest (portable ap) study date of 10:51 pm cardiomediastinal contours are unchanged. ng tube tip is 8 cm above the carina. there is no evidence of pneumothorax. of note the lungs were not totally included in the radiograph. in the visualized portions of the lungs radiolucency of the left upper lobe is due to emphysema. large right mid lung opacity could be hemorrhage or pneumonia. there is interstitial bilateral basal opacities larger on the right side. . echo due to suboptimal technical quality, a focal wall motion abnormality cannot be fully excluded. overall left ventricular systolic function is low normal (lvef 50-55%). the right ventricular free wall is hypertrophied. the right ventricular cavity is mildly dilated the aortic valve leaflets (3) appear structurally normal with good leaflet excursion and no aortic regurgitation. the mitral valve leaflets are mildly thickened. mild (1+) mitral regurgitation is seen. moderate tricuspid regurgitation is seen. there is moderate pulmonary artery systolic hypertension. there is a trivial/physiologic pericardial effusion. there is an anterior space which most likely represents a prominent fat pad. impression: suboptimal image quality. overall borderline normal left ventricular systolic function. right ventricular hypertrophy, dilation, at least moderate pulmonary artery systolic hypertension. . ct-a chest: mpression: 1. no evidence of pulmonary embolism to the subsegmental levels. small area of outpouching of contrast, immediately adjacent to atherosclerotic plaque is seen in the descending aorta, most likely representing an area of penetrating atherosclerotic ulceration, unchanged since . 2. significant progression of infectious process, now with extensive airspace consolidation involving the entire right hemithorax with multiple air-fluid levels, most compatible with a component of necrotizing pneumonia in addition to severe emphysema. 3. likely aspiration/hemorrhage or secretions within the right lower lobe bronchi with associated atelectasis. . cxr omparison is made with prior study performed five hours earlier. new opacities in the left lower lobe are consistent with aspiration. dense consolidations in the right mid and opacities in the right lower lobe are unchanged. right ij catheter tip is in the mid-to-lower svc. ng tube tip is out of view below the diaphragm. et tube tips are in the carina and left main bronchus. brief hospital course: mr. was transferred from an osh with massive hemoptysis and mrsa pna. he was intubated prior to transfer. on arrival to , he was sent urgently to ir for angioembolization of his bronchial artery. this was successful. he then went to the or for rigid bronchoscopy and washout of the r lung, which was filled with blood. ct-a was negative for pe, but confirmed necrotizing pna, c/w his known mrsa infection. over the next several days, he remained intubated with extremely poor oxygenation, requiring fi02 of 80-100%. on the morning of , he had a significant 8 point hematocrit drop with associated hypoxia and brb from his et tube. a flex bronch was performed, showing bubbling blood at the rms bronchus, sp[illing over in to the left lung. he was urgently switched over to a double lumen et tube to protect the l lung. he was rushed back to the ir suites were repeat embolization was attempted, but no further source could be identified. a discussion between the micu team, ir, ip, and thoracic surgery ensued, during which emergency lobectomy was considered, but ultimately considered too risky. a family meeting ensued and it was decided that the patient would be transitioned to cmo. his family gathered at the bedside and the ventilator was turned off, with ett in place to prevent hemopysis. he was kept comfortable with fentanyl and versed. he expired peacefully within an hour of the ventilator being turned off. medications on admission: medications on transfer: wellbutrin advair diskus 250/50 one puff q 12h lasix 40mg qday iss xopenix q 6hrs pnr solumedrol 40mg iv q 8 nasonex protonix 40mg iv daily tiotropium 1 puff daily vancomycin 1.25g iv q 12hr lasix . home meds: lasix 40mg qday protonix 40mg qday reglan wellbutrin potassium chloride spiriva advair albuterol neb discharge medications: n/a discharge disposition: expired discharge diagnosis: necrotizing mrsa pna, hemopysis, respiratory failure discharge condition: expired discharge instructions: n/a followup instructions: n/a procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more fiber-optic bronchoscopy enteral infusion of concentrated nutritional substances other intubation of respiratory tract closed [endoscopic] biopsy of bronchus other endovascular procedures on other vessels arteriography of pulmonary arteries diagnoses: congestive heart failure, unspecified acute kidney failure, unspecified personal history of malignant neoplasm of prostate obstructive chronic bronchitis with (acute) exacerbation other chronic pulmonary heart diseases acute respiratory failure postinflammatory pulmonary fibrosis diastolic heart failure, unspecified heparin-induced thrombocytopenia (hit) methicillin resistant pneumonia due to staphylococcus aureus 

Answer: The patient is high likely exposed to