Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: tetanus toxoid / vasotec / neomycin / adhesive tape / levaquin attending: chief complaint: cough / shortness of breath major surgical or invasive procedure: none history of present illness: dr is an 83 year old surgeon, on disability since with a diagnosis of multiple myeloma. he presents today with worsening o2 sats both at rest and exertion (he keeps an o2 sat monitor at home), worsening cough, and increasing shortness of breath. . although he has not felt altogether well for the past six months, he had been feeling better since his original admission in , for community-acquired pneumonia, in which he was diagnosed with mm. prior to that admission he had also been having worsening cough, sob and doe. . three or four weeks ago by his account, he began having cough again, although a cxr was negative for pulmonary process. but over the last 24-48 hours, the cough "blossomed" and he also became much more short of breath. he notes that before he would have o2 sats at 90-92 after getting up and walking now they were 86-88, and he was much more uncomfortable. he was having low-grade fevers. he came to the and is now admitted for diagnosis and management. . per heme/onc fellow: "hematologic history: dr. was diagnosed in when he presented with a multilobar pna and worsening renal function and was found to have serum monoclonal iga of 2.8 gm. his renal fxn has generally been stable since admission (creat 2.6->2.1-> 2.4). a upep showed intact iga kappa and no bence proteins. anemia is mild and no hypercalceia. his skeletal survey did not show lytic lesion but no fx or impending fx. beta-2 microglobulin was elevated but hard to interpret in face of renal insufficiency. igg level 200 and igm slightly suppressed. . "he was initially treated with decadron alone and then with decadron and thalidomide, however, this was stopped in the setting of disequilibrium and peripheral edema. the peripheral edema was ultimately thought to be related to some right-sided heart failure and he was thought to be hypervolemic. he was then started on revlimid and pulse decadron in ." . past medical history: pmh (per chart and hx): iga multiple myeloma kappa type - diagnosed per outpt cards note: long-standing rbbb and left axis deviation (bifascicular heart block) cards note suggests "nqmi" - ? demand ischemia --enzymes rose in context of larger admission for shortness of breath (in which mm was diagnosed) --not mentioned as issue in d/c summary hypertension chronic renal insufficiency - (cr 2.0)- followed by dr. chronic obstructive pulmonary disease - controlled on bronchodilators, followed by dr. . per chart: on 0.5l o2 at night, 1l in the day for activities. per patient: uses o2 mainly when he takes showers, which requires additional exertion; for this he uses 1.5l. atrial fibrillation - on coumadin chronic laryngopharyngeal reflux gastroesophageal reflux disease benign thyroid enlargement gout r elbow - on allopurinol bladder ca - superficial, s/p bcg . psh: multiple right inguinal hernia repairs s/p reoccurence right nephrectomy for renal cell carcinoma . allergies: levaquin, adhesive tape, neomycin, vasotec and tetanus toxoid. social history: colorectal surgeon. he is married and lives with his wife; lives downtown near the prudential center. former pipe smoker for many years, quit 25 years ago. he drinks 1-2 drinks a week. he enjoys sailing as a hobby; none of his hobbies expose him to toxic chemicals. family history: mother died in 80s, father in his 90s. sister alive and well. physical exam: t 98.6 hr 80 rr 20 bp 126/66 o2 97% 2l ht 67.5 in wt 175.3 lbs . gen: elderly man in nad breathing with nasal cannula; sitting forward with arms on knees; ruddy face. heent: eyes: anicteric. mouth: op clear. mmm. no lesions. pulm: diffusely and significantly diminished air movement at bases. no wheezing. minimal crackles. cor: rrr. no murmurs, rubs, gallops. abd: pos bs. liver ~2 cm below costal margin. no spleen tip felt. non tender, non-distended. skin: no rashes. discoloration at the shins c/w chronic edema. no petichiae. ext: 2+ pitting edema at the ankles. neuro: strength 4+ and symmetrical at all extremities. shoulder shrug intact. mild tremor of hands when extended, less at rest. language fluent and coherent. pertinent results: 01:20pm glucose-109* urea n-75* creat-3.0* sodium-140 potassium-4.3 chloride-105 total co2-19* anion gap-20 01:20pm wbc-7.0 rbc-3.67* hgb-12.0* hct-34.5* mcv-94 mch-32.7* mchc-34.7 rdw-16.8* 01:20pm neuts-80.4* bands-0 lymphs-13.9* monos-3.7 eos-1.5 basos-0.5 01:20pm hypochrom-normal anisocyt-normal poikilocy-normal macrocyt-normal microcyt-normal polychrom-normal 01:20pm plt smr-low plt count-113* . cxr: development of patchy opacity in the right middle lobe, compatible with atelectasis and/or pneumonia. . ct head: impression: no acute intracranial abnormalities. small vessel ischemic changes and calcifications of the left temporal and occipital lobes. at least two lytic lesions of the skull which likely represent known multiple myeloma. . tte: impression: mild symmetric left ventricular hypertrophy with mild global hypokinesis. right ventricular dilation and dysfunction. at least moderate pulmonary hypertension. moderate tricuspid regurgitation. dilated thoracic aorta. . eeg: impression: this is an abnormal portable eeg due to the presence of sharply contoured waveforms seen bifrontally and more often with a rightsided predominance in the setting of a slow and disorganized background rhythm. the significance of the first finding is not entirely clear as there were no frank epileptiform discharges seen. the second finding is consistent with a moderate encephalopathy suggesting the presence of deeper midline dysfunction. medication, metabolic disturbances, infections, or anoxia are among the most common causes but there are others. there were no clearly epileptiform discharges noted. no electrographic seizure activity was seen. brief hospital course: dr. is an 83 yo surgeon with h/o multiple myeloma s/p decadron/revlimid therapy, copd, chf, afib on coumadin, who initially presented on with fatigue, low grade fever, and increased productive cough from his baseline. he noted decreased 02 saturations at home on his pulse ox with increased doe (92% -> 88%). he had previously been treated for cap indergoing a hospital stay. he denied any chest pain, pleurisy, orthopnea, n/v/d, or increased le swelling during that time. moreover, he had recently finished a decadron taper for his mm. in the ed, he was found to have a rml infiltrate on cxr. cultures were obtained and the patient was started on ceftriaxone. he was subsequently transferred to the bmt service for further care. . on the bmt service, his ceftriaxone was continued, and was started on vancomycin on for + blood cultures for coag neg staph. additionally, he was given ivig on for his mm, which he tolerated well. . on the morning of transfer, the patient was found unresponsive in his room at 7am with blood coming from mouth. code team was activated. patient was found to be in asystolic arrest. cpr was initiated and the patient was intubated. he received epi 1mg x3, atropine 1mg x1 where he regained pulse. abg was 7.04/49/252. . he then presented to the after suffering cardiac arrest in the setting of rml pna and staph bacteremia. course by problem is as follows: . # cardiac arrest: patient was found to be unresponsive for an unclear duration (most likely < 10 minutes as the nurse had seen him prior to code and he was mentating normally) and was intubated, as above. most likely the cause of arrest was felt to be secondary to a poor conduction system given periods of prolonged asystole without capture rhythms and spontaneous return to nsr on telemetry in the micu. ich and large stroke was unlikely causes given a neg. head ct. mi was unlikely given tte findings of mild global hypokinesis without focal deficits. pe was unlikely given that the patient was therapeutic on coumadin. infection was also in the differential as the patient had a rml pna and coag neg. staph bacteremia. on admission to the the patient did not complete artic cooling protocol s/p arrest given periods of prolonged asystole (up to ~10seconds). on an eeg was performed, which showed evidence of moderate encephalopathy consistent with deeper midline dysfunction. the patient did not meet criteria for brain death. neurology was consulted to further assess encephalopathy and long-term prognosis, and they corroborated a poor prognosis with low likelihood of a meaningful neurologic recovery. . # renal failure: rising cre from baseline of 2.0 to 3.3 to 3.9 to 4.6, most likely due to hypoperfusion - either from the initial asystolic arrest or subsequent periods of asystole. there was concern for arrhythmias given increasing potassium and acidemia. . # pna: has r base consolidation, with 2+ gpc in sputum. also found to have coag neg. staph bacteremia = mrse. the patient was continued on vanco and zosyn, renally dosed, and azithromycin. . # bacteremia: the patient was found to have coag neg. staph (=mrse) on admission blood cultures, which was concerning for endocarditis given the patient's immunocompromised state. the patient was continued on vancomycin and zosyn. surveillance cultures were negative to date. a tee was held until long-term prognosis could be determined and per family wishes . # htn: the patient's bp was borderline following intubation, and required levophed upon admission to the micu. this was weaned as tolerated, with anti-hypertensives held. . # afib: hr was well rate-controlled during micu course. rate controlling agents were held given the patient's tenuous status. the patient was maintained on heparin gtt for prophylaxis. . # code: as the patient valued quality of life and given that a meaningful neurological recovery was not likely per the assessment of the micu team and neurology, the patient was made dnr with no further escalation of care during a discussion with the family. he was extubated on and passed away due to cardiopulmonary arrest. . # communication: wife medications on admission: advair allopurinol 100 mg tab every other day (odd-numbered days) combivent (as rescue, rarely used) albuterol (as rescue, not often used) coumadin 5 mg daily, 7.5 mg monday flomax 0.4 mg daily hydralazine 100 mg pindolol 2.5 mg spironolactone/hctz 25/25 daily discharge disposition: expired discharge diagnosis: primary: cardiopulmonary arrest secondary: multiple myeloma, a fib, chronic renal insufficiency, copd discharge condition: expired procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube cardiopulmonary resuscitation, not otherwise specified injection or infusion of immunoglobulin diagnoses: pneumonia, organism unspecified esophageal reflux acute kidney failure, unspecified severe sepsis chronic airway obstruction, not elsewhere classified hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage i through stage iv, or unspecified gout, unspecified atrial fibrillation chronic kidney disease, unspecified long-term (current) use of anticoagulants multiple myeloma, without mention of having achieved remission other staphylococcal septicemia 

Answer: The patient is high likely exposed to