Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: sob major surgical or invasive procedure: ethanol septal ablation history of present illness: pt of dr. and dr. w/severe hocm w/resting lvot gradient of 20mmhg increased to 89mmhg w/valsalva, paf, ddd pm placement for high degree avb underwent etoh ablation for worsening sob. pt had had an initial etoh ablation in for hocm with improvement in sob symptoms. since last pt has noticed worsening doe, sob and fatigue. in , pt could no longer work due to his worsening doe and sob. he went on disability and has not returned to work given his severe sob. he can walk ~20feet and must stop to catch his breath, he can go up 3 stairs and must stop thereafter due to severe sob. he does not walk outside of his home, he sleeps on pillows or sleeps upright in his chair, he also has pnd. he c/o occasional palpitations, lightheadedness and dizziness more prounouced with exertion or with bowel movements. he denies any cp, no diaphoresis, no n/v. over the past 6 months he has also noticed increase in his weight ~35pound weight gain and increasing le edema. his lasix dose was recently increased but had not noticed any symptomatic improvement. . on further ros: denies any constitutional symptoms, denies f/c/cough. no n/v/diarrhea, no brbpr, no dysuria. past medical history: -hocm (dx'd )w/resting lvot 20mmhg, sever symmetric lvh w/normal lv size, ef>55%, dilated la, minimal as and ar, mild mr and mild to moderate tr -syncope (chf, mobitz ii, avb-->ddd pm ) -etoh ablation -pvd -paf/at -htn -ai -obesity social history: -pt is widowed, lives with daughter in . currently on disability, previous occupation-mechanic. -quit tob 15 years ago, smoked 1.5 ppd x30years -social etoh use 1 drink per week. denies any other drug use. family history: -f: deceased at 87 from alzheimer's disease -m: alive, years of age, no known cad physical exam: vs:afebrile, bp 111/64 hr 64 rr18 98% 2lnc gen: nad heent: mmm, perrl, anicteric sclera resp: ctabl ant'ly cv: reg (paced) nml s1, s2, hocm murmer at llsb abd: obese, soft nd/nt +bs ext: non-pitting edema at ankles to lower shins neuro: a&o x3 pertinent results: echo (pre-ablation): measurements: left ventricle - septal wall thickness: *2.0 cm (nl 0.6 - 1.1 cm) left ventricle - inferolateral thickness: *1.2 cm (nl 0.6 - 1.1 cm) left ventricle - ejection fraction: 75% to 80% (nl >=55%) left ventricle - peak resting lvot gradient: *64 mm hg (nl <= 10 mm hg) left ventricle - peak inducible lvot gradient: 110 mm hg aortic valve - peak velocity: *2.5 m/sec (nl <= 2.0 m/sec) conclusions: left ventricular systolic function is hyperdynamic (ef>75%). there is a severe resting left ventricular outflow tract obstruction. the findings are consistent with hypertrophic obstructive cardiomyopathy (hocm). right ventricular chamber size and free wall motion are normal. there is no valvular aortic stenosis. the increased transaortic gradient is likely related to aortic regurgitation. moderate (2+) aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. mild (1+) mitral regurgitation is seen. . impression: pre ethanol ablation. severe asymetric septal hypertrophy with severe resting (and inducible) lvot gradient. valvular is present. hyperdynamic lvef . echo (post ablation): measurements: left ventricle - septal wall thickness: *2.1 cm (nl 0.6 - 1.1 cm) left ventricle - inferolateral thickness: *1.2 cm (nl 0.6 - 1.1 cm) left ventricle - ejection fraction: 75% to 80% (nl >=55%) left ventricle - peak resting lvot gradient: *40 mm hg (nl <= 10 mm hg) conclusions: there is asymmetric left ventricular hypertrophy. left ventricular systolic function is hyperdynamic (ef>75%). there is a moderate resting left ventricular outflow tract obstruction. the findings are consistent with hypertrophic obstructive cardiomyopathy (hocm). the aortic valve leaflets (3) are mildly thickened. there is no valvular aortic stenosis. the increased transaortic gradient is likely related to aortic regurgitation. moderate (2+) aortic regurgitation is seen. there is systolic anterior motion of the mitral valve leaflets. mild (1+) mitral regurgitation is seen. there is no pericardial effusion. impression: post ethanol ablation. asymetric septal hypertrophy with a moderate resting lvot gradient c/w hypertrophic obstructive cardiomyopathy (hocm). valvular . compared to the prior study of the same day, the resting lvot gradient has signficantly decreased. . ecg: a-v paced rhythm since previous tracing of , no significant change . labs: wbc rbc hgb hct mcv mch mchc rdw plt ct 06:05am 6.7 4.12* 12.5* 35.3* 86 30.4 35.4* 13.6 112* 04:17am 8.9 4.33* 13.3* 37.3* 86 30.6 35.5*# 13.6 126 . glucose urean creat na k cl hco3 angap 06:05am 87 14 0.9 141 3.7 103 29 13 04:17am 98 15 0.9 139 4.0 104 27 12 03:44pm 4.5 . ck(cpk) alkphos amylase totbili dirbili 06:05am 192* 03:10pm 381* 04:17am 591* 09:09pm 654* 03:44pm 541 . ck-mb mb indx 06:05am 11* 5.7 03:10pm 35* 9.2* 04:17am 76* 12.9* 09:09pm 93* 14.2* 03:44pm 84* 15.5 brief hospital course: ap: 64 yo m w/severe hocm, paf, ddd pm, s/p etoh ablation with improved gradient immediately after ablation. . #. hocm: s/p etoh ablation with improvement in lvot gradient. pt's ck were cycled until peaked at 654 and trended down to 192 on day of discharge. pt had some chest heaviness after the procedure but resolved without any ekg changes. pt had some bleeding when hemoband removed but re-applied hemoband per interventional fellow and had no bleeding or further complications of l radial artery. pt remained comfortable and sob improved dramatically. pt was continued on asa 325, restarted coumadin. his home regimen of amio, ccb, lipitor and lasix was restarted. his verapamil dose was decreased from 360 to 120 in am and fr/120 to 90 in pm for persistent low sbp range 90s-low 100s. . #. cad: pt with clean coronaries on previous cath. he was continued asa, statin,and ccb for optimization of bp control . #. rhythm: pt av paced and did not have any dysrhythmias post ablation . #. htn: continued ccb . #. hyperlipidemia: continued home regimen, lipitor . #. code: full medications on admission: -lasix 40mg qam and 20mg qpm -atenolol 75mg -amiodorone 200mg daily -verapamil 300qam and 120qpm -lipitor 10mg daily -coumadin 2.5mg daily (held since ) -colace discharge medications: 1. atorvastatin 10 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 2. amiodarone 200 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 3. atenolol 25 mg tablet sig: three (3) tablet po bid (2 times a day). disp:*90 tablet(s)* refills:*2* 4. docusate sodium 100 mg capsule sig: one (1) capsule po bid (2 times a day). 5. warfarin 2.5 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 6. furosemide 20 mg tablet sig: one (1) tablet po bid (2 times a day). disp:*30 tablet(s)* refills:*2* 7. verapamil 240 mg tablet sustained release sig: one (1) tablet sustained release po qam. disp:*30 tablet sustained release(s)* refills:*2* 8. verapamil 180 mg tablet sustained release sig: tablet sustained release po qpm. disp:*30 tablet sustained release(s)* refills:*2* discharge disposition: home discharge diagnosis: 1. hocm 2. s/p ethanol ablation 3. afib 4. pvd 5. htn discharge condition: stable discharge instructions: please take all your medications as directed and keep all your follow up appointments. . if you experience any significant cp, shortness of breath, palipations, please call your pcp or go to er. . please change your lasix dose to 20mg twice daily. your verapamil dose was changed to 240mg in the morning and 90mg at night followup instructions: provider: . phone: date/time: 9:30 procedure: left heart cardiac catheterization coronary arteriography using a single catheter excision or destruction of other lesion or tissue of heart, endovascular approach angiocardiography of right heart structures diagnoses: unspecified essential hypertension atrial fibrillation rheumatic heart failure (congestive) multiple involvement of mitral and aortic valves cardiac pacemaker in situ 

Answer: The patient is high likely exposed to