Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins / codeine / duragesic attending: chief complaint: abdominal pain major surgical or invasive procedure: ercp endotracheal intubation placement of right internal jugular central venouus catheter attempted lumbar puncture history of present illness: 47 yo m w/pmhx sx for alcoholic cirrhosis, hepatitis c, chronic pancytopenia above, hx sbp, hx upper and lower gi bleeding, s/p tips x5, and recurrent cholecystitis w/recurrent ruq pain who presents with sharp, cramping ruq pain, nausea, and nbnb emesis x1. he has a baseline chronic ruq abdominal pain for which he takes dilaudid 8 mg po q4, and says for 7 years there has never been a time where he has not had this pain. he was in usoh had 2 eggs and toast for breakast, then 30 minutes later had nbnb emesis. about 2 hours later, he had his first episodes of pain while lying in bed watching tv, , and lasting about 20-30 minutes, which then returned to baseline. he did not have anything to eat for the rest of the day. the next episode occurred at 11 pm last night, same nature, lasting 20-30 minutes and returning to pain, which prompted his coming to the ed. patient states that this pain is different from his baseline chronic abdominal pain in that it is like a " horse," basically in the same location, but worse. no radiation, no other associated symptoms. of note, he has recently run out of his zofran which he uses once every couple of days for nausea. he had not refilled the rx cost. . at 7am today, pt had some substernal chest pain that radiated to his ruq that he said felt like pressure similar to episodes of reflux in the past. an ekg was done, ce sent, and he was given sl ntg which completely resolved the cp. no dyspnea/diaphoresis/other assoc sxs. ruq pain returned to pain that he had before this episode began. of note, he can get up one flight of stairs and walk 1 block before getting dyspneic, notes pedal edema with prolonged standing, requires pillows for sleeping, but does not know if he would get short of breath without them, and denies pnd. . also notes similar cp last week, went to hospital, underwent a stress test, which by report was normal. . he has had no fever/chills/diarrhea/bloody stool/blood in vomit/cough/cp/sob/jaundice. he has not traveled recently. he has also noted a petechial rash on his right forearm starting yesterday, that he attributes to a kitten to which he was exposed and believes he is allergic to. he notes a recent 40 lb weight gain over 1.5 months, followed by 15 lb weight loss. ros otherwise negative. . past medical history: hepatitis c, ? transfusion after gunshot wound to r temple alcohol abuse, no etoh for 15 years. cirrhosis s/p tipsx5, 1st 12 years ago, last 6 years ago, repeated occlusion cholelithiasis cholecystitis s/p multiple ercps variceal bleed s/p banding gerd nephrolithiasis s/p appendectomy pancytopenia no cholecystectomy low platelets social history: he is married x10 years and retired x12 years, used to work with . he admits to a heavy etoh history of a 12 pack a day but quit 14 years ago and denies any etoh use at present. no smoking. he denies any history of iv drug use and has had 2 blood transfusions in the past. family history: dm. mother and father with . father with rcc, currently on hd. physical exam: vs: 98.3 bp 122/62 hr 76 rr 18 o2sat 96% ra gen: well appearing in nad, lying comfortably in bed watching tv heent: mmm. no oral ulcers. no cervical lad. hrt: rrr. no m/g/r lungs: ctab no rrw. abd: distended. soft. +voluntary guarding in ruq. no hepatomegaly. tenderness to light palpation over ruq>rlq, but able to go from lying to sitting position with no pain. no rebound. +caput medusae. ext: wwp. no c/c/e. petechial lesion and excoriations over right forearm. rle ~1 cm healing excoriated lesion over lower shin. neuro: alert and oriented. +asterixis. pertinent results: 02:48am urine blood-sm nitrite-neg protein-neg glucose-neg ketone-tr bilirubin-sm urobilngn-12* ph-7.0 leuk-neg 02:48am urine rbc-0-2 wbc- bacteria-rare yeast-none epi- 02:48am wbc-3.0* rbc-3.74* hgb-12.0* hct-32.4* mcv-87 mch-32.0# mchc-36.9* rdw-16.0* 02:48am neuts-60.0 lymphs-27.8 monos-5.5 eos-6.6* basos-0.2 02:48am alt(sgpt)-26 ast(sgot)-48* alk phos-118* amylase-33 tot bili-2.1* 02:48am albumin-3.5 02:48am glucose-122* urea n-12 creat-0.6 sodium-138 potassium-3.7 chloride-106 total co2-24 anion gap-12 09:20am pt-17.3* ptt-37.3* inr(pt)-1.6* 02:48am plt count-31* 09:40am blood wbc-6.8# rbc-3.53* hgb-12.4* hct-31.8* mcv-90 mch-35.1* mchc-37.3* rdw-16.5* plt ct-58* 12:54pm blood wbc-7.3 rbc-3.14* hgb-10.2* hct-27.2* mcv-87 mch-32.6* mchc-37.7* rdw-16.4* plt ct-50* 03:33am blood wbc-16.8* rbc-3.04* hgb-10.1* hct-27.2* mcv-89 mch-33.2* mchc-37.2* rdw-18.5* plt ct-72* 03:01am blood wbc-13.4* rbc-2.80* hgb-9.2* hct-25.2* mcv-90 mch-32.9* mchc-36.5* rdw-19.0* plt ct-70* 07:58am blood wbc-19.4* rbc-2.74* hgb-9.0* hct-25.7* mcv-94 mch-32.9* mchc-35.1* rdw-19.6* plt ct-61* 05:50am blood neuts-79.2* lymphs-15.5* monos-4.8 eos-0.5 baso-0 03:01am blood neuts-79* bands-1 lymphs-10* monos-8 eos-0 baso-1 atyps-0 metas-1* myelos-0 nrbc-49* 09:40am blood pt-18.9* ptt-31.4 inr(pt)-1.7* 02:53am blood pt-18.6* ptt-33.2 inr(pt)-1.8* 08:27am blood pt-20.0* ptt-36.4* inr(pt)-1.9* 03:01am blood pt-20.5* ptt-35.6* inr(pt)-2.0* 07:58am blood pt-22.4* ptt-34.0 inr(pt)-2.2* 05:45am blood glucose-92 urean-10 creat-0.7 na-139 k-3.8 cl-104 hco3-29 angap-10 05:50am blood glucose-135* urean-15 creat-0.7 na-139 k-4.2 cl-106 hco3-22 angap-15 09:40am blood glucose-215* urean-18 creat-0.8 na-138 k-3.3 cl-110* hco3-15* angap-16 09:25pm blood glucose-223* urean-28* creat-1.2 na-145 k-4.5 cl-113* hco3-13* angap-24* 02:59pm blood glucose-139* urean-80* creat-3.6* na-150* k-4.5 cl-117* hco3-17* angap-21* 03:01am blood glucose-162* urean-78* creat-3.2* na-148* k-4.6 cl-115* hco3-16* angap-22* 07:58am blood glucose-175* urean-78* creat-2.7* na-146* k-6.8* cl-119* hco3-12* angap-22* 02:48am blood alt-26 ast-48* alkphos-118* amylase-33 totbili-2.1* 09:20am blood alt-27 ast-51* ck(cpk)-579* alkphos-108 totbili-2.5* 05:30am blood alt-28 ast-55* ld(ldh)-280* alkphos-106 amylase-30 totbili-2.5* 01:39am blood alt-55* ast-149* ld(ldh)-428* ck(cpk)-5069* alkphos-88 amylase-67 totbili-4.0* 03:33am blood alt-131* ast-211* ck(cpk)-5075* alkphos-68 totbili-3.1* 03:01am blood alt-146* ast-260* ld(ldh)-1217* ck(cpk)-* alkphos-61 totbili-3.6* 05:30am blood lipase-17 05:46am blood ck-mb-9 ctropnt-<0.01 09:20am blood calcium-8.6 phos-3.5 mg-1.9 03:01am blood albumin-2.3* calcium-5.9* phos-7.4* mg-1.9 09:40am blood vitb12-640 folate-13.6 02:59am blood ammonia-922* 08:23am blood osmolal-322* 12:48pm blood osmolal-327* 01:30pm blood ammonia-430* 12:20pm blood ammonia-279* 03:01am blood ammonia-299* 09:40am blood prolact-44* tsh-0.36 02:59pm blood vanco-19.0* 12:21pm blood cortsol-28.6* 01:07pm blood cortsol-31.6* 03:01am blood phenyto-13.2 phenyfr-3.8* %phenyf-29* 09:40am blood asa-6 ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 10:37am blood type-art po2-338* pco2-22* ph-7.47* caltco2-16* base xs--4 08:04am blood lactate-9.4* 09:44pm blood lactate-14.7* 04:27pm blood freeca-0.99* . ct head : there is no evidence of acute intracranial hemorrhage. compared to the two examinations of yesterday, particularly in the left frontal and parietal regions, and within the posterior cerebral hemispheres. the lateral ventricles have become more compressed. the fourth ventricle may be slightly smaller in size. there is also loss of the normal -white matter differentiation. the findings are in keeping with cerebral edema. the left arachnoid cyst is unchanged. the visualized paranasal sinuses and mastoid air cells remain clear. metallic structures near the right lateral orbit are noted. findings of cerebral edema, with a worsened appearance of the brain compared to yesterday. metallic structures near the right orbit preclude mri evaluation. the findings were discussed with dr. at the time of the exam. note added at attending review: this change may represent post ictal swelling, rather than edema. correlation with follow up studies is recommended. . ct head : there is again noted diffuse brain edema. however, the mass effect and the edema has progressed since the prior study. the previously seen arachnoid cyst in the left temporal fossa is now compressed. there is also complete effacement of the cerebral cisterns and downward displacement of the cerebellar tonsils into the foramen magnum. it has progressed since the prior study. there is no shift of midline structures. there is again noted opacification of the posterior ethmoid cells, which appears to be similar when compared to prior study. there is also a small amount of fluid in the right sphenoid sinus, which has increased since the prior study. impression: interval progression of diffuse brain edema. brief hospital course: 47 yo w/hx alcoholic cirrhosis s/p tips, esophageal varices s/p sclerosis, hepatitis c, pancytopenia, and hx recurrent cholecystitis, who presents with worsening ruq, nausea, and vomiting. brief hospital course as follows: . course on the floor: . for his ruq pain, mrcp was initially considered, but due to metal close to his eye, the patient underwent ercp on . during ercp, limited exam of the esphagus, stomach and duodenum was without varices. major papilla was normal with plastic stent in situ. biliary duct was cannuized with successful and deep sphincterotome using a free-hand technique. contrast medium was injected resulting in complete opacification. the procedure was moderately difficult. plastic stent was removed from the major papilla. biliry cannulation and cholangiogram revealed a non-dilated common bile duct, with a single mobile radiolucent gallstone present. a sphincterotomy was performed in the 12 o'clock position using a sphincterotome over an existing guidewire. the stone was extracted successfully using a 15 mm balloon. the patient received a dose of levoflox in the ercp suite. patient returned to the floor in stable condition, afebrile, mentating well. . soon after ercp, temp rose to 101.0, bcx were taken. that night (day before micu admission), the patient was found to be pacing around the floor. the patient received 4mg scheduled iv dilaudid and reportedly slept all night without incident. the morning of micu admission, the patient was found to be acutely delirious, very tremulous, unable to communicate with the staff. temp was noted to be 99.9, bp 128/80, sats 94-95% on ra. 30 mins later, the patient was found in bed, unresponsive, still tremulous with dilated but reactive pupils, temp of 95.2 axillary; bp 120/61. pt was taken to the micu and intubated for airway protection. pt received etomidate for intubation and started on propofol for sedation. pt rapidly decompensated and started dropping bps and urine output dramatically fell off. cvl was placed and fluids were started wide open. . course in micu . after transfer to the micu, he underwent a repeat ercp where he was found to have bleeding at the sphincterotomy site. for this procedure he was given 12.5mg of demerol, 10mg of cisatracurium at approximately 6:30pm. at 9:15pm, he was noted by staff to have head deviation to the right and clonic jerking of the right arm. this lasted roughly ten minutes and ceased after administration of 8mg of iv ativan. he remained without obvious seizure activity until 9:45pm when he experienced 30 minutes of the head and right arm jerking. he was given an additional 8mg of iv ativan, loaded with 1g of iv dilantin, and started on a versed gtt. . he continued in status epilepticus for much of the ensuing night, despite being loaded with dilantin, receiving 25mg of iv ativan over the course of the night, maxing out on a versed drip, propofol drip. finally, after consultation with the neurology / epilepsy service, he was started on a pentobarb drip, and a coma state was induced. he had an eeg that confirmed seizures, and then there was suppressed state one pentobarb was started. repeat imaging showed worsening cerebral edema. this was thought to be due to fulminant hepatic encephalopathy (ammonia level 922), or edema from the post-ictal state. . an lp was attempted on his first night in the micu, without success in obtaining csf due to body habitus. broad spectrum antibiotics were started and continued at that time. he spiked temps to 104, but that was in the setting of prolonged seizure activity. . attempts were made to decrease cerebral edema. neurosurgery was consulted, and decided that surgical intervention would not improve the situation, as it was diffuse edema. mannitol was given twice, but the team was unable to continue giving as his serum osmolality remanined >330. . the family was consitently with him, and well aware of his grave prognosis. on , a family meeting was held with the micu team, sw, nurses, and neurology. they wanted to attempt to wean the pentobarb and reevaluate for signs of brain activity; they also requested a repeat head ct to assess for decreased swelling. after weaning the pentobarb, there was no further brain activity. the repeat head ct was done and showed worsening edema with evidence of herniation. . on , the decision was made by the wife and her sister, in consultation with the remainder of his family, to withdraw life support. he was extubate and vasopressors were stopped. he remained comfortable and he passed away shortly thereafter with the medical team, wife, sister-in-law, and social worker at the bedside. . the family declined an autopsy. parents were notified in . medications on admission: dilaudid 8 mg q4h lasix 80 mg qd aldactone 100 mg qd paxil 40 mg qd ambien 10 mg qhs prevacid 30 mg qd zofran prn, takes 1 every couple of days lactulose prn (used for constipation 2-3x/month) discharge medications: na discharge disposition: expired discharge diagnosis: epilepsy hepatitis c cirrhosis s/p ercp branistem herniation discharge condition: deceased discharge instructions: na followup instructions: na procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more insertion of endotracheal tube endoscopic removal of stone(s) from biliary tract endoscopic sphincterotomy and papillotomy endoscopic control of gastric or duodenal bleeding transfusion of packed cells transfusion of other serum transfusion of platelets infusion of vasopressor agent diagnoses: esophageal reflux unspecified essential hypertension acute and subacute necrosis of liver chronic hepatitis c with hepatic coma alcoholic cirrhosis of liver acute kidney failure, unspecified acquired coagulation factor deficiency other and unspecified alcohol dependence, in remission compression of brain hemorrhage complicating a procedure acute respiratory failure cerebral edema grand mal status mechanical complication due to other implant and internal device, not elsewhere classified obesity, unspecified personal history of urinary calculi rhabdomyolysis calculus of gallbladder and bile duct with other cholecystitis, with obstruction cerebral cysts 

Answer: The patient is high likely exposed to