Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: zosyn / meropenem attending: chief complaint: nausea, vomiting, jaundice, fever major surgical or invasive procedure: post-pyloric tube placement, liver biopsy, endoscopy history of present illness: this is a 52 year-old woman with history of hypothyroidism, endometriosis admitted from outside hospital after admission there on for nausea, vomiting, abdominal pain of weeks duration and jaundice. work-up at the outside hospital revealed cholestasis of unknown etiology. after ultrasound and mrcp were unrevealing, the presumed diagnosis was alcoholic hepatitis. she was found to be bacteremic with persistent fevers and enterococcus in the urine. at presentation to , history was limited by confabulation and tangential answers. she reports increased abdominal pain, nausea and vomiting in the days preceding admission on . she stated pain preceded nausea and vomiting, and then jaundice followed. she reports dark stools, light urine. she denies dietary changes, mushroom intake, medication changes, history of ivdu or intranasal cocaine. she also noted no fevers, chills, before admission. she believes she has experienced 10 pound weight loss over weeks preceding . past medical history: hypothyroidism endometriosis social history: 50 pack year history of smoking, alcohol history-"2 shots a day of vodka, but not everyday", marijuana remote, no ivdu family history: father-lung and other cancers mother-uterine cancer physical exam: vs: bp 124/80 hr: 80 rr: 20 97%on 2 liters 98.4 gen: nad, resting comfortably, sick looking but non-toxic, obese, jaundiced heent: perlla, eomi, mmm, scleral icterus, mild conjuctival injection, no pharyngeal edema or exudate, no lymphadenopathy, no jvd, no carotid bruits lungs: cta b/l--poor air movement heart: rr, s1 and s2 wnl abd: hyperactive, high pithced bowel sounds, mild distention, obese, soft, diffusely tender without rebound or guarding, liver 4cm below costal margin extr: edema bilaterally, non-tender neuro: alert, oriented to person, knows in hospital, but not which, knows month, year and president, but not date. confabulating during exam. tangential responses, does not always answer question being asked. cerebellar with no pass pointing. decreased strength bilaterally in lowere extremities. pertinent results: admit labs: 09:00pm wbc-27.1* rbc-2.61* hgb-9.0* hct-27.7* mcv-106* mch-34.4* mchc-32.3 rdw-18.6* 09:00pm plt count-406 09:00pm pt-14.5* ptt-29.8 inr(pt)-1.3 09:00pm alt(sgpt)-29 ast(sgot)-61* ld(ldh)-124 alk phos-651* amylase-100 tot bili-1.5 09:00pm albumin-2.6* calcium-8.9 phosphate-3.4 magnesium-1.9 09:00pm lipase-130* 09:00pm glucose-98 urea n-10 creat-0.5 sodium-144 potassium-4.2 chloride-108 total co2-27 anion gap-13 09:10pm urine blood-neg nitrite-neg protein-neg glucose-neg ketone-neg bilirubin-neg urobilngn-neg ph-7.0 leuk-neg 09:10pm urine color-straw appear-clear sp -1.008 brief hospital course: 1) enterococcal bacteremia/uti: as discussed above, the patient was thought to have alcoholic hepatitis at the outside hospital and started on steroids. lft's were consistent with a cholestatic picture, but u/s, mrcp, and ct of abdomen were unrevealing with possible gallstone ileus. she was rapidly tapered off steroid on presentation to the , where the patient continued to have persistent fevers with maximum temperatures of 104 farenheit. she was started on vancomycin, gentamycin, and zosyn. urine culture was positive for vancomycin sensitive enterococcus five days after admission; antibiotics were not changed with this result given the above regimen. blood cultures were taken serially in an attempt to isolate an organism responsible for the patient's fevers, but all cultures to date were negative. with respect to diagnostic studies, several were performed and findings are as follows. ct abdomen/pelvis revealed a low density fatty liver with ascites. there was a hypoattenuating area within segment 4 of the liver which may represent an area of focal fat, edema or hemangioma, but tumor could not be excluded. there was no intrahepatic biliary dilatation and the gallbladder was unremarkable (probable tiny gallstones but no evidence of biliary obstruction). there was a left adrenal lesion consistent with a likely left adrenal adenoma. there were mildly dilated loops of small bowel without evidence of obstruction consistent with an ileus. mr abdomen revealed an abnormal heterogeneous enhancement pattern of the liver, which was interpreted as a nonspecific finding which can be seen in cases of acute viral and toxic hepatitis. there were tiny enhancing lesions in the left lobe of the liver as above. there were larger nonenhancing lesions in segment 4 of the liver without imaging characteristics typical of hemangioma or cyst. ultrasound guided paracentesis was performed as well which revealed a gradient of 1.1 consistent with cirrhosis and borderline transudative process. a hepatitis panel was negative. a difficult ultrasound guided liver biopsy was performed which revealed marked steatosis with abundant intracellular hyalin and neutrophilic reaction. trichrome stain showed prominent sinusoidal fibrosis, without cirrhosis. iron stain showed no definite stainable iron. there was evidence of tumor seen. ciprofloxacin was added and later discontinued for sbp prophylaxis. despite these studies, the patient continued to spike fevers accompanied by mental status changes, but blood cultures continued to be negative. ct abdomen was repeated which again revealed no evidence of intra-abdominal abscess. at this point, the id service was consulted and it was recommend that tee be performed looking for endocarditis. the patient continued to spike and blood cultures were positive for gram negative rods. caspofungin was started for possible fungal coverage. vancomycin, flayl, and zosyn were started, gram negative rod coverage. unfortunately, the patient continued to spike and the patient developed a pruritic back/truncal/lower extremity rash. this was presumed to be related to zosyn and the drug was stopped and substituted with ceftriaxone. the patient's blood pressure simultaneously dropped and she was unable to defend her blood pressures with aggressive fluid boluses. the patient was sent to the intensive care unit on . in the intensive care unit, the patient did not require pressors to maintain her blood pressure because she responded well to several liters of iv fluids. antibiotics were continued, including ceftriaxone, levofloxacin, caspofungin, and vancomycin. tee was performed which was negative for endocarditis. because of the patient's drug reaction to zosyn the strategy was to limit antibiotic exposure. flagyl was discontinued when c.diff was negative. repeat ct was performed on which showed no abscess. liver lesions were unchanged from prior study. there was some cecal and splenic flexure colonic thickening. ultracound was repeated which showed diffuse fatty infiltrate of the liver, without significant ascites. there were small lesions in segment 2 of the liver. mental status continued to wax and wane as it did on the floor and this was attributed to toxic metabolic complications. post pyloric feeding tube was placed because the patient was unable to tolerate pos secondary to nausea. by discharge from the unit, the patient remained afebrile while only on vancomycin, flagyl, meropenem. flagyl was discontinued with the thought of possible relation to mental status changes. the patient returned to the floor on with persistent low grade fevers, but mental status improved to where she was alert and oriented times three. 2) hypothyroidism: getting synthroid iv 112.5 at osh--very difficult to control 3)?ms change/wernicke-korsakoff: could be sepsis, vs. alcohol will: thiamine, mvi, folate, possible mri head-mamillary bodies? 4)full code 5)dispo: pending resolution of fevers/etiology of cholestasis ***needs follow-up in months with ct or mri for ?adrenal mass. medications on admission: protonix vancomycin gentamycin zosyn prednisone 20 discharge disposition: extended care facility: - discharge diagnosis: endometriosis mastoiditis hypothyroidism alcoholic hepatitis liver steatosis liver failure gallstones ascites colonic perforation heterotopic pancreatic tissue internal hemorrhoids lll pneumonia postop atelectasis pulmonary edema pleural effusions ards hypovolemia swan ganz monitoring blood loss anemia acute renal failure anasarca hypokalemia hyperkalemia hypocalcemia hypophosphatemia hypomagnesemia hypernatremia hyperalimentation cellulitis sepsis septic shock infected central venous catheter bacteremia (enterococcus, b fragilis) candiduria 2.6cm incidental adrenal adenoma discharge condition: stable discharge instructions: 1) pt/ rehab. 2) trach care 3) vac change every three days. keep vac at continuous vacuum per routine. followup instructions: contact dr. office to arrange a follow up appointment in about 1 month. procedure: venous catheterization, not elsewhere classified venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more parenteral infusion of concentrated nutritional substances open and other left hemicolectomy enteral infusion of concentrated nutritional substances closed (percutaneous) [needle] biopsy of liver percutaneous abdominal drainage percutaneous abdominal drainage temporary ileostomy temporary tracheostomy colonoscopy esophagogastroduodenoscopy [egd] with closed biopsy other gastrostomy replacement of gastrostomy tube open biopsy of liver injection or infusion of oxazolidinone class of antibiotics local excision of lesion or tissue of small intestine, except duodenum diagnoses: cellulitis and abscess of trunk acquired coagulation factor deficiency severe sepsis perforation of intestine accidental puncture or laceration during a procedure, not elsewhere classified septic shock other specified disorders of biliary tract infection and inflammatory reaction due to other vascular device, implant, and graft streptococcal septicemia other and unspecified alcohol dependence, unspecified other chronic nonalcoholic liver disease acute alcoholic hepatitis infection of gastrostomy mechanical complication of gastrostomy anomalies of pancreas 

Answer: The patient is high likely exposed to