Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: nausea, vomitting, abdominal pain. major surgical or invasive procedure: abdominal aortic aneurysm repair with tube graft history of present illness: 51 y/o m w/no medical care in many years, who was in his usoh on thursday morning, getting ready for work, when he suddenly developed acute onset of nausea and vomiting. he states that he vomited approximately 20 times a day from thursday through saturday. he did not have any abdominal pain at this point. his vomiting was somewhat mucous-like but he didn't notice any blood in it. he states that on saturday, he had some "reddish brown" material in his vomit. this resolved on its own, but on sunday he developed bilateral lower quadrant abd pain. the pain was severe but not the worst pain he has ever experienced. it didn't radiate. nothing made it better or worse, although he was unable to eat so unclear if this made a difference. his nephew bought him some antacid, and he reports he took more doses per day than was recommended on the bottle, but this resolved his pain. he has also not had a bowel movement for the past 10 days. he also notes that since the onset of his nausea and vomiting, he has had "unsteadiness". he has not fallen but has felt that his legs are weak and that he had difficulty going up the stairs. no vision changes, dysarthria, dysphagia. he denies any fevers, chills, headache, neck pain or stiffness, chest pain, shortness of breath, diarrhea, numbness or tingling on any extremities, or focal weakness. . in the ed, his vitals were 99.6, 96, 145/94, 16, and 100%ra. his labs revealed a mildly elevated amylase and lipase, as well as hyponatremia and hypokalemia. he had an abd ct scan which revealed a 9.3 cm infrarenal aortic aneurysm, and a distended gallbladder with sludge. he was seen by vascular surgery who recommended further w/u of his other issues prior to an elective repair. he had a head ct which showed a hypodensity in the pons which may represent an old lacune. he was then seen by neurology who recommended an mri/mra. because of the sludge in his gallbladder on ct scan, he was seen by general surgery, who recommended a ruq u/s but had a low suspicion for gallstone pancreatitis. his ecg revealed ? st elevations vs j point elevation in the anterior leads, so it was faxed to cardiology who recommended cycling enzymes and aspirin if vascular was okay with it. he was admitted to the micu for further monitoring in the setting of multiple ongoing issues past medical history: - s/p fall 2 months ago, hit back of head, no loc - s/p back surgery social history: lives by himself in . works as a criminal defense attorney. states he drinks a "couple of drinks a few times a week". smoked 1 ppd x 30 yrs. recently divorced. family history: colon cancer and cad in father (alive at age 82), dm in mother physical exam: t: 99.4 bp: 154/94 p: 99 r: 16 96%ra gen: awake, alert, oriented male in nad, fidgeting frequently in bed heent: nc, at. perrl (was reported to have unequal pupils by neuro but appear equal on my exam), eomi. mm dry. poor dentition. neck: supple, no cervical lad or thyromegaly, no carotid bruits lungs: cta anteriorly. pt refused posterior exam. cv: rrr, ii/vi systolic murmur at lusb abd: soft, protuberant but nondistended, nontender, with hyperactive bowel sounds, no abd bruit ext: warm/dry, no edema, ? petechiae over anterior shins, 2+ dp pulses bilaterally, muscle wasting evident in thighs/arms neuro: cn ii-xii intact, strength 5/5x4, unable to elicit dtr in patellar area, sensation intact to light touch throughout, finger to nose with dysmetria bilaterally, no pronator drift, wide-based gait pertinent results: ct head on : impression: 1. no evidence of acute intracranial hemorrhage. mri with diffusion-weighted images is more sensitive in the evaluation for acute ischemia/infarct and for vascular detail. 2. left paramedian pontine lacune. ct abdomen : large saccular infrarenal aortic aneurysm extending to the level of the iliac bifurcation, measuring approximately 9.3 cm in greatest axial dimension. "hyperattenuating cresent" sign seen in the anterior aspect of the mural thrombus, suggesting relatively acute intramural hemorrhage. fat-stranding also seen adjacent to the aneurysm. these findings are concerning for impending rupture of the aneurysm. abdominal ultrasound : 1. no gallstones identified. no evidence of acute cholecystitis. 2. echogenic liver consistent with fatty infiltration. however, other forms of liver disease and more advanced liver disease including significant hepatic fibrosis/cirrhosis cannot be excluded. brief hospital course: 51 year-old male with no prior medical care who presents with nausea/vomitting/abdominal pain, all of which have currently resolved, but subsequently found to have infrarenal abdominal aortic aneurysm as well as numerous electrolyte abnormalities. . # aaa: seen by vascular surgery. per radiology, at risk for rupture. undergoing cardiovascular evaluation pre-operatively. echocardiogram and carotid ultrasounds ordered. trying to maintain systolic blood pressure less than 120mmhg with metoprolol 75mg po tid. patient is typed and crossmatched 6 units. will need to discuss best stress test imaging with cardiology. for pre-operative work-up, on , he had a persantine mibi wich showed a moderate anterior reversible defect and an ejection fraction of 25%. on , he had a cardiac catheterization which demonstrated occlusion of the lad at d1, a collateralized distal lad, 70% occlusion of the cx and 70% occlusion of the om1. cardiology saw him on and recommended peri-operative metoprolol. on , he had an abdominal aortic aneurysm repair with a tube graft. please see operative note for details. he was transferred to the vicu in stable condition. on pod 1, his pain was controlled with an epidural. he got out of bed to a chair. his neosynepherine was weaned off. he made adequate urine output. his hematocrit was stable. on pod 2, his diet was advanced. his epidural was removed. on pod 3, his central venous line was removed. he was transferred to the floor. he was able to ambulate without much pain. on pod 4, he was discharged home in good condition. . # neuro: if he truly is an alcoholic, this could be due to wernicke's encehalopathy. he does have a wide-based gait on exam but does not have any ocular findings currently. patient also became somnolent after receiving 10mg diazepam on micu admission. pontine lacune on admission head ct, likely old. patient's neurological exam is non-focal. he did have ophthalmoplegia on admission that seems to have resolved, and he denies diplopia. will defer further imaging, per neuro, as patient does not have any current focal manifestations. continue thiamine and folate supplementation. . # n/v/abd pain: unclear etiology although given the elevation in amylase and lipase, it may be due to pancreatitis (? alcoholic). he became very uspet when we questioned him further regarding his alcohol use, and denied that he would ever have drinks per day, stating it was only a "couple" per day. per surgery, gall bladder findings not concerning. no abnormalities in the bowel seen on ct scan to suggest an alternative diagnosis. his symptoms of n/v/abd pain have all resolved and he is tolerating po diet. . # ? alcohol abuse: this would provide a unifying diagnosis for his pancreatitis and ataxia, although pt denies history of heavy drinking. however, his physical exam findings of muscle wasting, slight agitation, hypertension, tachycardia, as well as his hyponatremia are suggestive of alcohol abuse. will obtain psychiatry consultation to see if patient is suffering from primary depressive disorder vs. secondary to substance use. . # st elevations: unclear significance in the absence of any symptoms. biomarkers are negative which is reassuring. will hold on aspirin for now, given above. 2 sets of enzymes negative x 12 hours apart lipid panel remarkable only for ldl 134. . # hyperglycemia: has elevated glucose on fingersticks, likely due to stress from current illness vs new diabetes. hemoglobin a1c = 7.6%. qid sliding scale, but may consider changing to oral regimen as outpatient. . # hyponatremia: likely due to volume depletion in the setting of 4 days of vomiting and decreased po intake. appears dry on exam. other possibility would be beer potomania, although would rehydrate first and see if his sodium improves before making this diagnosis. correct slowly with normal saline, given risk of central pontine myelinolysis with rapid correction with alcohol history. . # hypokalemia: likely due to alkalosis from vomiting. replete. . # fen: ivf, replete k, check calcium/mg/phos. resume diet. . # ppx: will start ppi given history of alcohol use, hold off on heparin sq given aaa, pneumoboots . # communication: with patient and his mother : () (cell) . # code: full. . # dispo: called out to medical floor, since hemodynamically stable with stable hematocrits. medications on admission: loratadine prn. discharge medications: 1. oxycodone-acetaminophen 5-325 mg tablet sig: 1-2 tablets po q4-6h (every 4 to 6 hours) as needed. disp:*40 tablet(s)* refills:*0* 2. metoprolol tartrate 25 mg tablet sig: one (1) tablet po bid (2 times a day). disp:*60 tablet(s)* refills:*2* discharge disposition: home discharge diagnosis: abdominal aortic aneurysm discharge condition: good discharge instructions: please call or come to the ed with any fever > 101, nausea, vomiting, redness from your incision, increased abdominal pain, lightheadedness, or any other issues that may arise. you may shower. activity as tolerated. take all medications as directed. you were started on metoprolol. please make an appointment with your pcp about this medication change. followup instructions: please call the office of dr. for a follow-up appointment in 1 week at ( please make an appt with your pcp 1 wk to evaluate your medication changes procedure: left heart cardiac catheterization coronary arteriography using a single catheter other repair of aneurysm transfusion of packed cells insertion of catheter into spinal canal for infusion of therapeutic or palliative substances systemic to pulmonary artery shunt diagnoses: coronary atherosclerosis of native coronary artery tobacco use disorder hyposmolality and/or hyponatremia alcohol abuse, unspecified abnormality of gait hypopotassemia other specified disorders of biliary tract abdominal aneurysm without mention of rupture hypovolemia other and unspecified alcohol dependence, unspecified other nonspecific abnormal serum enzyme levels alcohol withdrawal delirium diabetes with unspecified complication, type ii or unspecified type, uncontrolled other and unspecified manifestations of thiamine deficiency unspecified disorder of eye movements 

Answer: The patient is high likely exposed to