Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins / vancomycin / lamictal / levofloxacin / benadryl attending: chief complaint: seizures major surgical or invasive procedure: intracranial electrode placement. history of present illness: 37 year old female with h/o seizures s/p anterior temporal lobectomy with recurrent seizures who presents for eeg monitoring after intracranial electrode placement. she is transferred to epilepsy from the neurosurgery service. the patient is right-handed when writing, though she believes she was intended to be left-handed and guided to switch as a child. she is left-handed with gross motor skills. when her seizures occur, they last for 2-3 days at a time. prior to stopping the lupron, she was having her period about every two weeks and therefore was having seizures equally as often. today she describes the feelings as predominantly a "foggy" feeling with nausea, a nervous feelings, and sensitivity to sounds and certain flashing lights. past medical history: began having seizures before age 1, initially with a febrile illness. she had one prolonged febrile seizure which required hospitalization. she is thought to have started having simple partial seizures around age 5, involving a "nervous funny feeling" in her chest; these seizures not fully recognized until age 9. as a teenager these became more frequent and prolonged with alteration of awareness; they also became significantly worse in relation to her menstrual cycle, with more than 20 events on the day prior to menstruation. there was a secondary generalized seizure x1 in , when a medication was changed. in , she underwent investigation at suny-. she reports she had a scalp eeg, mr which showed right hippocampal sclerosis, and which showed mixed dominance for language. she underwent right anterior temporal lobectomy. this was complicated by a post-operative infection requiring bone flap removal and replacement with an acrylic pad. eegs since then have shown spikes in the region of the right temporal lobe, and ntermittent theta frequency slowing over the right frontal and anterior temporal regions. less frequently, independent left mid-temporal sharp and slow waves or theta slowing was also seen. mris have shown stable changes from right anterior temporal lobectomy; mr pituitary also showed a small 2.3 mm aneurysm versus tortuosity at the level of the right mca bifurcation. post-operative she was symptom free until , when she tried a rapid taper off tegretol under physician . she began having foul smells "like sewage" and auditory hypersensitivity, and previously reported she was in "almost simple partial status." she went back on medications and did well until , when the frequency of events increased again. she was placed on keppra, carbatrol, and progesterone. in she had an ltm admission during which she was tapered off her meds. she had no seizures during the admission. for history of catamenial seizures and ovarian cysts, she receives hormonal treatment with progesterone. she was on lupron until , when she discontinued it. she had one period in but has not had a period since then. she continues the progesterone. she denies any chance of being pregnant. she also has a history of migraines, which can be difficult to differentiate from seizures. it is also possible that the migraines trigger the seizures or vice versa. prior seizure medications have included phenobarbital, tegretol, depakote, diamox, tranxene, topamax, and felbatol, keppra. social history: works for as a program coordinator, and works as a staff person in a group home for disabled individuals. family history: mom has high cholesterol, thyroid problems and anxiety. dad has diabetes. half sister has thyroid problems and anxiety. maternal grandmother has bipolar disorder and was treated with lithium. paternal grandmother and her sister had disease. paternal great grandmother had disease. there is a family history of alcoholism. no family history of epilepsy. physical exam: gen: awake, alert, not in distress, lying in bed. non-toxic appearance. skin: no rash. heent: depth electrodes in place with site clean and dry, no active bleeding. there is a sunken appearance in the right temporal region where the bone flap was removed, and an old craniectomy scar visible. no dysmorphic features, no conjunctival injection, nares patent, mucous membranes moist, oropharynx clear. neck: supple, no meningismus. resp: clear to auscultation bilaterally cv: regular rate, normal s1/s2, no murmurs, rubs, or gallops abd: bowel sounds present, abdomen soft, non-tender, and non-distended. no hepatosplenomegaly or masses palpable. extrem: warm and well-perfused. there is significant cracking with movement of the right shoulder. rom full. neuro: ms - awake, alert, interactive. cranial nerves ?????? eom smooth and full, no diplopia; 4 beats of nystagmus on rightward gaze; visual field full with confrontation test, intact facial sensation, face symmetric with full strength of facial muscles, hearing intact to finger rub bilaterally, palate elevation is symmetric, and tongue protrusion is symmetric and full movement. sternocleidomastoid and trapezius are strong and normal volume. tone - normal strength - delt tri wrex fex fflx io / ip quad ham gastr ta toeflex r 5 5 5 5 5 5 5 5 5 5 5 5 5 5 5 l 5 5 5 5 5 5 5 5 5 5 5 5 5 5 5 reflexes - biceps triceps brachioradialis patellar ankle r 2+ 2+ 2+ 2+ 2+ l 2+ 2+ 2+ 2+ 2+ plantar responses flexor bilaterally sensation - itact to light touch, temperature, vibration, throughout. coordination - no dysmetria and smooth finger to nose. gait - deferred. pertinent results: labs on admission: 08:10am blood wbc-4.7 rbc-3.71* hgb-11.5* hct-34.5* mcv-93 mch-31.0 mchc-33.3 rdw-12.9 plt ct-226 08:10am blood pt-12.3 ptt-27.3 inr(pt)-1.0 08:10am blood glucose-65* urean-8 creat-0.7 na-133 k-3.8 cl-94* hco3-30 angap-13 01:22pm blood calcium-8.9 phos-4.5 mg-2.1 06:18am blood carbamz-8.2 monitored hct: 08:10am blood wbc-4.7 rbc-3.71* hgb-11.5* hct-34.5* mcv-93 mch-31.0 mchc-33.3 rdw-12.9 plt ct-226 01:22pm blood wbc-4.4 rbc-3.58* hgb-10.9* hct-33.4* mcv-93 mch-30.3 mchc-32.5 rdw-13.3 plt ct-231 06:18am blood wbc-8.2# rbc-3.24* hgb-10.0* hct-30.3* mcv-94 mch-30.8 mchc-32.9 rdw-13.0 plt ct-205 mr : there is unchanged encephalomalacia involving the anterior right temporal lobe from prior right frontotemporal craniotomy. the brain is otherwise normal on the limited sequences without new abnormality. the ventricles are normal. the configuration of the right optic tract as well as the cerebral peduncle is unchanged. mr : two anterior electrode leads terminating in the medial temporal and superolateral right temporal lobe. the posterior electrode lead terminates in the antero-medial tip of the right temporal lobe. mild edema noted along the electrode leads. ct stereotaxis head : unchanged ct appearance since the recent study. status post right frontotemporal craniotomy with craniectomy and right anterior temporal lobectomy,with extensive cystic encephalomalacia redemonstrated. ct head : limited post procedural study. no acute intracranial hemorrhage. extensive post-procedural pneumocephalus. eeg : multifocal interictal epileptic activity is found with the most active sites residing in the more anterior electrodes of the right hippocampal chain. discharges are seen at 1, at 2, and at 3. these take the form of spike and wave and short runs of polyspike activity. additionally, discharges are occasionally noted from the deepest contacts of the r and f (right medial placed frontal electrode). here, there are isolated discharges mainly from contact 1 but occasionally contact 2. an additional fairly active interictal epileptic activity is noted from the rlf or the more lateral frontal electrode residing in the cut edge of the right superior temporal gyrus. there, the activity is mainly from 1 but is also seen frequently in 2. there are bursts of spindle-like activity noted from 4 and 5 which may represent more normal physiology. additionally, there is widespread slowing that is most prominently noted in the rh electrodes and at their deepest sites. eeg : multifocal interictal epileptiform activity, again primarily from the region in the periremoval site from prior operation in the right temporal lobe. eeg : there was one pushbutton that did not reveal any significant sustained activity but was for minor symptoms. eeg continues to show multifocal independent interictal activity that, if anything, appears to be more and more frequent, probably in relationship to the patient's medication withdrawal. eeg : there continues to be extremely frequent paroxysmal interictal epileptiform activity in the form of isolated and short runs of epileptic activity from the deeper contacts of rh maximum at 1, 2, and 3 but occasionally seen at more distance sites 6, 7, and 8. activity is also extremely frequent in the rlf contacts 1, 2 and also sometimes extending into higher contacts 3, 4, 5, and 6. isolated spike and spike wave discharges are seen from the deepest contact of also. there is a considerable amount of slowing also from rh and, to a lesser degree, from rlf at their deeper sites. the automated spike detection algorithm and seizure detection algorithm captured some of this activity as false detection. eeg : continued multifocal independent interictal epileptiform activity still most prominently noted from the deeper contacts of rh to a slightly lesser degree from the deeper contacts of rlf and more infrequently from the deeper regions of . there is also continous slowing noted from the deeper regions of rh suggesting cortical structural pathology. eeg : this depth electrode recording captured no pushbutton activations; however, there was frequent sharp activity seen in the anterior contacts of the hippocampal chain as well as in the middle and inferior contacts of the lateral frontal chain with additional runs of polyspike activity seen predominantly at the rlf5 contact. however, there was no ongoing seizure activity seen. the time samples captured mixed frequency activity. brief hospital course: neuro: after placement of intracranial electrodes by neurosurgery, the patient was transferred to neurology for intracranial eeg monitoring. po progesterone was discontinued in the hospital to increase seizure frequency; progesterone cream had been discontinued 2 days prior to admission. carbamazepine was weaned off as well, and the patient had sleep deprivation and electrode stimulation to further evaluate seizures. intracranial eeg revealed abundant sharp activity as well as spike and wave rhythmic activity especially at the anterior hippocampal contacts (, 2) and the middle contacts of the lateral frontal chain (rlf4, 5) which sometimes spread to all the lateral frontal electrodes. there was no clear correlation between the clinical events and changes in the eeg activity. id: continued post-operative prophylaxis with clindamycin while depth electrodes are in place, due to multiple medication allergies. the patient did have fevers 24-48 hours after electrode placement, which resolved without medication changes. fen: the patient was noted to have dropping na several days after electrode placement, likely due to siadh. this was treated with salt tabs. prior to discharge she was able to come off of her salt tabs with stable sodium. endo: tsh was sent in the context of siadh, and noted to be low. free t4 was low as well. endocrine was consulted and felt this was most likely due to withdrawal of the carbamazepine, which changes thyroid metabolism and thyroid binding globulin. they recommended rechecking tfts in weeks. prolactin was noted to be slightly elevated (37); this was thought to be due to treatment with ranitidine. endocrine recommended rechecking prolactin levels when the patient was off ranitidine for at least 2 weeks. pulm: portable cxr, checked in the context of fever, showed a small right mediobasal opacity, likely representing fat but could not rule out posterior mediastinal mass on portable film. ap and lateral cxr revealed no posterior mediastinal mass. gi: continued home dose zantac. received senna, colace to prevent constipation. pain: received tylenol, codeine prn head pain. patient preferred not to receive morphine if possible because after her surgery she became concerned that she was becoming addicted to percocet, and has a family history of "addictive personality." after electrodes removed no pain issues. medications on admission: carbamazepine - 400 mg qam, 300mg qpm, 500mg qhs progesterone micronized 100mg po tid lorazepam - 1 mg prn seizures ranitidine hcl - 150 mg daily advair discharge medications: 1. acetaminophen 325 mg tablet sig: two (2) tablet po q6h (every 6 hours). 2. claritin 10 mg tablet sig: one (1) tablet po once a day. 3. carbatrol 100 mg cap, multiphasic release 12 hr sig: three (3) cap, multiphasic release 12 hr po once a day. 4. carbatrol 100 mg cap, multiphasic release 12 hr sig: four (4) cap, multiphasic release 12 hr po qam. 5. carbatrol 100 mg cap, multiphasic release 12 hr sig: five (5) cap, multiphasic release 12 hr po at bedtime. 6. advair diskus 100-50 mcg/dose disk with device sig: one (1) inhalation twice a day. 7. lorazepam 1 mg tablet sig: one (1) tablet po q24 prn () as needed for seizure. 8. codeine sulfate 30 mg tablet sig: 0.5 tablet po q6h (every 6 hours) as needed for pain. disp:*30 tablet(s)* refills:*0* discharge disposition: home discharge diagnosis: seizure discharge condition: stable. no focal neurological deficits discharge instructions: you were admitted for placement of intracranial electrodes, to determine the source of your seizures. your progesterone and carbamazepine were stopped to increase seizure frequency. eeg monitoring revealed r temporal seizures. during your hospital stay you were noted to have slightly low sodium, most likely due to siadh (syndrome of inappropriate release of the anti-diuretic hormone), which can occur due to intracranial electrode placement. this was treated with salt tabs, and should resolve when the electrodes are removed. you were also noted to have altered thyroid studies (low tsh and free t4); you were seen by endocrine who thought this was due to changes in the carbamazepine, and expect it to resolve when you restart the carbamazepine. you should have your thyroid function checked by your primary care doctor 6-8 weeks after leaving the hospital. the level of the hormone prolactin was also noted to be high in your blood, most likely due to the ranitidine (zantac) you take. this can be rechecked at a time when you are off ranitidine for at least two weeks. you received clindamycin to help prevent infection while the intracranial electrodes were in place. you had fevers early in the hospital course, which resolved with continued clindamycin; no other changes were made to your antibiotic regimen. followup instructions: 1) please see dr. on thursday at 12:30 pm. his office may call you to schedule an earlier appointment. 2) please call your pcp, . , , for a follow-up appointment. please have tsh, free t4, palpable thyroid exam checked by pcp as an outpatient in 6 weeks to confirm total resolution of your hypothyroidism. procedure: implantation or replacement of intracranial neurostimulator lead(s) removal of intracranial neurostimulator lead(s) diagnoses: esophageal reflux cerebral aneurysm, nonruptured long-term (current) use of other medications stricture of artery migraine, unspecified, without mention of intractable migraine without mention of status migrainosus other abnormal blood chemistry tracheoesophageal fistula, esophageal atresia and stenosis personal history of surgery to other organs localization-related (focal) (partial) epilepsy and epileptic syndromes with complex partial seizures, with intractable epilepsy 

Answer: The patient is high likely exposed to