Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: code stroke major surgical or invasive procedure: none history of present illness: this is a 67 yo man with vasc rf's including cad, htn, high chol, early family hx mi (father), prostate ca s/p brachytherapy, who presented at 8:10 pm as "code stroke" with l facial droop, arm weakness and dysarthria. he had been in usoh with no recent illnesses, and was having dinner with family at 7:30pm when he got up for a glass of water. he suddenly froze for a few seconds and did not speak immediately. then he began slurring his words when he said "i want some water." his wife and family noticed a l facial droop and l-sided weakness and immediately called ems, who arrived within 15 minutes. he was brought to er where neurology was waiting; initial bp in ambulance was 156/80; in er was 203/86 but came down to 179/97 within 15 minutes. bg was 102. t was 100, for which he got tylenol. nihss score was 11 for partial gaze palsy (1), complete hemianopia (2), patial facial palsy (2), l hand drift (1), severe sensory loss on the left (2), dysarthria (1), and l inattention and extinction (2). blood was noted on rectal exam (see hx below). ct was performed and showed subtle blurring of the r insular ribbon; in retrospect, shows a hyperdensity at the distal branch of the r mca (distal m1). cta performed immediately after showed r m1 distal branch occlusion. cta of the neck was negative for severe stenosis. he received iv tpa 81 mg for weight of 200 lbs (90.1 kg), at time of needle 9:30pm (8.1 mg bolus followed by 72.9 mg infusion over 1 hr). bp was elevated just before tpa to 180s-190s systolic, and he received 10 iv labetalol x 2 with good effect. following tpa, bp was again over 185 systolic, and he received another 10mg iv labetalol - 182/86, hr 80 after total 30 iv labetalol. past medical history: 1. cad s/p mi and cabg in 2. prostate ca s/p brachytherapy , with complication of rectal ulcer from radioactive seed, now takes suppositories with "spotting" blood 3. htn 4. high chol 5. s/p appy social history: lives in with wife, retired newspaper circulation mgr; no tob (remote), occ etoh family history: father had early mi physical exam: t 100 hr 92 bp 156/80 in ambulance (later: 203/86 in hosp; rechecked 15 minutes later as 179/97) general appearance: well appearing heent: moist mucus membranes, clear oropharynx neck: supple, no bruits heart: regular rate and rhythm, no murmurs lungs: clear anteriorly abdomen: soft, nontender +bs extremities: warm, well-perfused mental status: the patient is alert and attentive, able to convey parts of history, oriented to place, date, name, situation. good knowledge for events prior to hosp. language is intact with no errors; reads only end part of word; cannot identify more than one person in cookie picture to far right. there is agnosia for l hand. cranial nerves: there is a l dense hemianopsia and visual neglect on the left. the optic discs are normal in appearance. r gaze preference, but with ocr able to abduct eyes to l fully. pupils react equally to light 4->3. sensation on the face is diminished to light touch on l compared to r, with hemineglect. l umn pattern facial droop. hearing is intact to finger rub. the palate elevates in the midline. the tongue protrudes in the midline and is of normal appearance. dysarthria for labial and palatal sounds. motor system: decreased tone lue; full strength on r. l weakness as follows: l 5- at delt; 4+ at tri; 5 at ; 4 wrist ext; 3 finger ext; 4+ finger flex 4+/5 ip, ham, foot dorsi and plantarflexion reflexes: the tendon reflexes are present on r, diminished on l at knee and biceps. the plantar reflexes are flexor on r, extensor on l. sensory: sensation is decreased to light touch over l hemibody compared to r, and there is significant l neglect. coordination: there is no ataxia on f->n. slow on l, in proportion to weakness. gait: could not be assessed pertinent results: 141 105 16 91 -------------< 4.6 24 1.1 ck: 82 mb: notdone trop-*t*: pnd ca: 9.5 mg: 2.0 p: 3.8 alt: 45 ap: 56 tbili: 0.8 alb: 4.3 ast: 40 serum etoh 23 serum asa, acetmnphn, , , tricyc negative mcv 95 wbc 8.7 h/h 14.7/41.4 plt 175 pt: 10.7 ptt: 24.0 inr: 0.9 imaging: neck cta with vessels patent cta head with likely distal m1 (vs prox m2) clot, associated vascular changes on r ct with distal "dense mca" and blurring of insular ribbon ekg: nsr with q's in ii, iii, avf, v1-v3 twi's in ii, iii, flat in avf st elev 1.5 mm in v1-v3 rbbb l axis brief hospital course: 67 yo man with multiple vascular risk factors including cad, htn, hyperlipidemia, and prostate cancer s/p brachytherapy, who presented as a "code stroke" with left facial droop, arm weakness and dysarthria, with a left field cut and inattention on exam as well, and was found to have a right mca distal m1 occlusion (and very proximal m2) on cta. he was given iv tpa 2 hours after the onset of his symptoms. he was admitted to the icu for close monitoring. his bp was controlled initially with labetolol, with the goal sbp 130-185. a repeat hct after the tpa showed hyperintensity around the stroke consistent with dye extravasation, then another the next morning showed a small amount of hemorrhagic conversion. he could not get an mri/a due to agitation. he continued to improve clinically. he ruled out for an mi with cardiac enzymes and was monitored by telemetry. an echo showed symmetric lvh with mild regional left ventricular dysfunction and an lvef of 45-50%. his risk factors were evaluated with fairly good lipid control; nonetheless, a statin was started. hba1c was 5.3. he was treated with plavix since he had been on asa previously. other notable events included brbpr in the ed after tpa which resolved; hct remained stable. he had known rectal ulcer. he had a fever and was found to have a uti and cellulitis vs thrombophlebitis of his arm; he was started on ceftriaxone . he was able to pass his swallow evaluation with honey-thickened liquids and pureed solids. he had an episode of bradycardia while sleeping but with normal cardiac enzymes. he was given ativan per ciwa scale for his alcohol use, as well as thiamine and folate. prior to discharge, he was off ciwa scale and without any signs of withdrawl. his improvement was significant with resolution of dysarthria and improvement in left arm/leg strength. medications on admission: flomax aleve prn asa 81 ?atenolol lipitor mvi **incomplete list discharge medications: 1. thiamine hcl 100 mg tablet sig: one (1) tablet po daily (daily). 2. folic acid 1 mg tablet sig: one (1) tablet po daily (daily). 3. metoprolol tartrate 25 mg tablet sig: 0.5 tablet po bid (2 times a day). 4. atorvastatin 10 mg tablet sig: one (1) tablet po daily (daily). 5. tamsulosin 0.4 mg capsule, sust. release 24hr sig: one (1) capsule, sust. release 24hr po hs (at bedtime). 6. indomethacin 25 mg capsule sig: one (1) capsule po tid (3 times a day). 7. dipyridamole-aspirin 200-25 mg cap, multiphasic release 12 hr sig: one (1) cap po bid (2 times a day). 8. ceftriaxone-dextrose (iso-osm) 1 g/50 ml piggyback sig: one (1) gram intravenous q24h (every 24 hours) for 6 days. 9. protonix 40 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po once a day. discharge disposition: extended care facility: rehab unit at - discharge diagnosis: right mca infarct high blood pressure high cholesterol discharge condition: good, more strong on left side, no slurred speech discharge instructions: please take all medications as prescribed. keep all follow-up appointments. please return to the nearest ed if symptoms of weakness and slurred speech return. md, procedure: injection or infusion of thrombolytic agent diagnoses: coronary atherosclerosis of native coronary artery pure hypercholesterolemia urinary tract infection, site not specified unspecified essential hypertension personal history of malignant neoplasm of prostate cellulitis and abscess of upper arm and forearm cerebral artery occlusion, unspecified with cerebral infarction ulcer of anus and rectum 

Answer: The patient is high likely exposed to