Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: codeine attending: chief complaint: hematemesis major surgical or invasive procedure: egd history of present illness: 52 y.o. male transferred from where he presented with hematemesis. he has a history of upper gi bleeding and was recently discharged from 1 day prior to this admssion for upper gi bleed. he has a h/o dieulafoy lesion in the cardia and peptic ulcer disease. he underwent egd () which revealed a small dieulafoy in the cardia of the stomach, which was injected with epinephrine, resulting in vigorous bleeding. he underwent multiple transfusions, and was stabilized. repeat egd revealed no active bleeding. he was transferred the the medical floor at , but the following day had massive hematemesis again. he was transfused 5 units prbcs, and transferred to for further management. . initially admitted to the micu, the patient was seen by gi in consult. he was hemodynamically stable with no further hematemesis, and underwent repeat egd here on , revealing clips and clots, but no active bleeding. he is called out to the floor . past medical history: morbid obesity hypertension hypercholesterolemia non-insulin dependent diabetes mellitus renal insufficiency - baseline cr 1.7-1.8 coronary artery disease - ptca in c/b spiral dissection of rca successfully stented - 50% mid lad - diffuse 80% mid lcx - 80% proximal om2 - 90% ostial rpda congestive heart falure - ef 30% s/p pacemaker implant in for sick sinus previous alcoholic w/ h\o dts at least twice, last ~ 25 yrs ago copd obesity hypoventilation chronic low back pain depression s/p fractured skull in childhood social history: patient is married, lives with his wife in . has 2 children. current 1 ppd smoker x 30 years. previous alcoholic with h/o dt's, current etoh 2-3 beers/day. now disabled, former construction worker. family history: mother alive with h/o cva; father also alive. maternal grandfather died of an mi in his 60s. physical exam: upon admission to the medical floor: vitals: t 98.8 bp 147/85 hr 84 rr 18 99% ra, fs 160 general: morbidly obese, comfortable, nad heent: perrl, eomi, mmm, op clear neck: jvd difficult to assess given body habitus, heart: distant hs, but rrr and w/o murs lungs: distant lung sounds, but ctab, no wheezes or rhonchi abdomen: obese, nabs, soft, nt/nd; no stigmata of liver disease extremities: l arm with 2+ bilat lower extremity pitting edema neuro: a&ox3, strength 5/5, no focal defecits pertinent results: 02:11pm pt-12.6 ptt-25.1 inr(pt)-1.1 02:11pm plt smr-low plt count-95*# 02:11pm neuts-93.5* bands-0 lymphs-5.1* monos-1.2* eos-0.3 basos-0 02:11pm wbc-9.4 rbc-3.63* hgb-11.3* hct-31.7* mcv-87 mch-31.0 mchc-35.5* rdw-15.8* 02:11pm hcv ab-negative 02:11pm hbsag-negative hbs ab-positive hbc ab-negative 02:11pm albumin-2.4* calcium-7.2* phosphate-4.8* magnesium-1.5* 02:11pm lipase-161* 02:11pm alt(sgpt)-57* ast(sgot)-47* ld(ldh)-317* alk phos-41 amylase-108* tot bili-0.6 02:11pm glucose-183* urea n-67* creat-1.6* sodium-134 potassium-4.0 chloride-104 total co2-21* anion gap-13 03:18pm urine blood-neg nitrite-neg protein-neg glucose-100 ketone-neg bilirubin-neg urobilngn-neg ph-5.0 leuk-neg 03:18pm urine color-yellow appear-clear sp -1.010 10:09pm hct-29.4* . egd : blood in the fundus two clips in the fundus ulcer in the duodenal bulb otherwise normal egd to second part of the duodenum recommendations: no active bleeding or sites of bleeding seen on this exam. . pcxr : portable ap, without comparison, demonstrates pacemaker leads within right atrium and right ventricle on single view. lungs are clear. costophrenic sulci are sharp. heart, mediastinal contours, and pleural surfaces are unremarkable, given technique. impression: no cardiopulmonary process. . . abd u/s : 1. echogenic liver consistent with fatty infiltration, however, other forms of liver disease including significant hepatic fibrosis and cirrhosis cannot be excluded on the basis of this exam. 2. no ascites. 3. cholelithiasis without evidence of cholecystitis. 4. normal appearance of the kidneys. . l upper extremity u/s : l cephalic vein dvt bilat leg venous ultrasound (osh ): no evidence of deep venous thrombosis. . ecg ( 18:53) rrr @ 77, nl axis, intervals, no st/t changes (no change brief hospital course: 52 y.o. male with h/o dieulafoy lesion and pud p/w hematemesis. . # hematemasis - the patient has a h/o peptic ulcer disease, as well as dieulafoy's lesion. he had egd at the outside hospital with hemostatis achieved. he then rebled requiring multiple more blood transfusions and underwent egd on at with no evidence of active bleeding. he was given protonix . his hct remained stable. we considered the possibility of bleeding varices given his etoh history, but liver u/s w/o evidence of portal hypertension, showing only fatty liver. no varices were noted on multiple egds. his diet was advanced to regular and tolerated well. he will f/u with his pcp. . # l cephalic vein clot: the pt had a picc line placed at hospital in his l arm, and was found to have a l cephalic vein clot by u/s, which was persued to investigate ue swelling. the picc was removed, but no anticoaggulation given due to recent gi bleeding. this is a peripheral vein clot and was treated with removal of the offending line. . # thrombocytopenia - unclear etiology, likely due to alcohol use/fatty liver disease, but more likely consumptive in setting of recent bleeding. hit was considered low probability as plts remained stable. . # elevated lfts: hepatitis serologies indicate immunity to hbv, but no active infection. abdominal u/s was performed and revealed fatty liver, but no evidence of portal hypertension or cirrhosis. nash or alcoholic hepatitis are possible diagnoses. in addition, the patient reported he was recently started on a statin, so this may be the cause. his zocor and tricor were held. he was instructed to follow up with his pcp for repeat lfts, and referral to hepatology if they remain elevated. . # cad: the patient has known 3vd, but no evidence of active ischemia> his asa was stopped to due gi bleeding, and should be restarted by his pcp when appropriate. after he was stabilized for >48 hours, his b-blocker was re-started, which he tolerated well. he will be discharged on toprol xl, and his pcp can titrate the dose as needed. . # chf: ef 30% by report. his diuretics and b-blocker were held in the icu for possiblity of hemodynamic instabiltiy with recent bleeding. upon transfer to the medical floor, the pt was volume overloaded on exam. his lasix and b-blocker were restarted and doses should be titrated by his primary care physician. . # copd: advair & nebulizers were continued. . # renal failure: etiology is unknown, presumed from hypertension vs diabetes. according to osh records, he is near his baseline (cr 1.6) his nephrocaps and phoslo were restarted. . # chronic back pain: morphine initially, then percocet prn. . fen: regular, monitor lytes . proph: no heparin given gi bleed; pneumoboots, ppi . full code medications on admission: advair 250/50 rocaltrol 0.25 po qd phoslo 667 po bid prozac 40 po qd trazodone 100 po qhs prn insomnia nephrocaps 1 po qd zocor 40 po qd tricor 145 po qd lasix 80mg qam, 40mg qpm omeprazole 20 po bid toprol xl 150mg daily discharge medications: 1. calcium acetate 667 mg capsule sig: one (1) capsule po tid w/meals (3 times a day with meals). disp:*90 capsule(s)* refills:*2* 2. fluticasone-salmeterol 250-50 mcg/dose disk with device sig: one (1) disk with device inhalation (2 times a day). 3. fluoxetine 20 mg capsule sig: two (2) capsule po daily (daily). 4. lasix 20 mg tablet sig: three (3) tablet po twice a day. 5. toprol xl 100 mg tablet sustained release 24 hr sig: one (1) tablet sustained release 24 hr po once a day. disp:*30 tablet sustained release 24 hr(s)* refills:*2* 6. folic acid 1 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 7. b complex-vitamin c-folic acid 1 mg capsule sig: one (1) cap po daily (daily). disp:*30 cap(s)* refills:*2* 8. oxycodone-acetaminophen 5-325 mg tablet sig: 1-2 tablets po q4-6h (every 4 to 6 hours) as needed for pain for 7 days. disp:*20 tablet(s)* refills:*0* 9. pantoprazole 40 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po q12h (every 12 hours). disp:*60 tablet, delayed release (e.c.)(s)* refills:*2* 10. thiamine hcl 100 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 11. zolpidem 5 mg tablet sig: two (2) tablet po hs (at bedtime) as needed. 12. albuterol 90 mcg/actuation aerosol sig: 1-2 puffs inhalation prn as needed for shortness of breath or wheezing. discharge disposition: home discharge diagnosis: 1. upper gi bleeding 2. upper extremity cephalic clot secondary to picc 3. transaminitis discharge condition: stable, hct stable, no signs of bleeding discharge instructions: * take medications as prescribed. several medications were held, and you should discuss restarting any other medications with your primary care physician. * do not take aspirin until directed by your primary care physician. * call your doctor if you have blood in your vomit or stool, light-headedness, or any other concerning symptom. * please note that you have elevated liver enzymes. this may be due to your cholesterol medication, alcohol, or fat infiltration of your liver. you must follow up with your primary care physician and seek referral to a liver specialist if this does not resolve. followup instructions: please call your doctor, dr. () for an appointment in the next week. he needs to monitor your hematocrit and should check your liver enzymes and if they remain elevated, refer you to a liver specialist. md procedure: other endoscopy of small intestine diagnoses: coronary atherosclerosis of native coronary artery pure hypercholesterolemia congestive heart failure, unspecified unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled acute kidney failure, unspecified percutaneous transluminal coronary angioplasty status other complications due to other vascular device, implant, and graft morbid obesity hematemesis 

Answer: The patient is high likely exposed to