Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins attending: chief complaint: l sided weakness major surgical or invasive procedure: none history of present illness: pt is a 71 year old r handed male with htn and cardiomyopathy of unclear origin who presents from with l sided weakness and dysarthria. he was well until today around 10:30 am when he suddenly felt nauseous and dizzy. he says he slumped over to the ground and need to go to the bathroom but could not get up to do so. he lost continence on the ground. he says he noticed then that his l arm and leg were weak. no loc. his landlord found his confused, on the floor of his house, 1 hour later and called ems. pt was taken to hosptial where a head ct showed r mca infarct. he then was sent to for further management. he arrived in our ed at 5:30 pm, 7 hours after onset of symptoms. ros: negative for recent illness, head or neck trauma, travel, change in mental status, headache, focal neuro deficits. of note, pt had laparascopic polypectomy last tuesday for benign polyps, and since then has had diarrhea. past medical history: htn cardiomyopathy, per pt recent echo and ekg were "stable" s/p l nephrectomy for renal tumor, pt did not know it's identity social history: lives on with a significant other, has 4 children, works as a police officer denies tobacco or drugs, occasional etoh family history: noncontributory physical exam: vs: t afeb hr 66 bp 156/83 rr18 sat 95% on room air pe: sleepy but arousable, no acute distress heent at/nc, mmm no lesions neck supple, no thyromegaly, no , no bruits chest cta b cvs rrr w/o mgr abd soft, ntnd, + bs ext no c/c/e, distal pulses full, no rashes or petechiae neuro ms: awake, dysarthric, oriented x3. speech: fluent w/o paraphasic error, repetition, high frequency naming intact. writing not tested. neglects l side intermittently cn: i--not tested; ii,iii-- perrla, vff by confrontation, seems to neglect l visual field. optic discs sharp; iii,iv,vi-eomi w/o nystagmus, no ptosis; v-- sensation intact to lt/pp, masseters strong symmetrically; vii-- l facial weakness; viii--hears finger rub bilaterally; ix,x-- voice normal, palate elevates symmetrically, gag intact; -- scm/trapezii ; xii--tongue protrudes midline, no atrophy or fasciculation. motor: normal bulk and tone, no tremor, dense hemiparesis of l rm with minimal mvmt even on deep painful stimulation. able to ift l leg above bed for 2-3 seconds, then drops it. wiggles toes n l. cannot overcome resistence on l leg. r side full strength. coord: rapid alternating and point-to-point (fnf, hts, ttf)movements intact on r, cannot perform on l given hemiparesis. refl: | |tri |bra |pat | |toe | l | 2+ | 2+ | 2+ | 2+ | 2+ | up | r | 2 | 2 | 2 | 2 | 2 | dn | : decreased sensation to all modalities on l, a bit difficult to assess b/c of neglect. extinguishes on l to double stimulation consistently. pertinent results: ct of chest/abdomen/pelvis results pending ct and cta head w&w/o c & recons; ct neck w/contrast impression 1. large right mca infarct involving almost the entire territory supplied by this artery. 2. almost total thrombosis of the origin of the right m2 segment with reconstitution of the distal branches. a filling defect is noted in the right m2 segment. ct head w/o contrast 7:49 am impression 1. evolving large right mca distribution infarct. 2. no acute intracranial hemorrhage identified. 3. these results were called to dr of neurology at the time of interpretation (10:30 a.m.). ct head w/o contrast impression: again seen is a right mca infarct, without evidence of new infarction or hemorrhage. carotid series complete impression: no evidence of stenosis in either carotid artery. tte impression: moderate inducible mid-ventricular cavity gradient. mild aortic regurgitation with normal valve morphology. mild mitral regurgitation. no definite cardiac source of embolism identified. based on aha endocarditis prophylaxis recommendations, the echo findings indicate a moderate risk (prophylaxis recommended). clinical decisions regarding the need for prophylaxis should be based on clinical and echocardiographic data. tee conclusions: the left atrium is dilated. mild spontaneous echo contrast is seen in the body of the left atrium. no mass/thrombus is seen in the left atrium or left atrial appendage. no spontaneous echo contrast is seen in the left atrial appendage. the right atrium is dilated. no spontaneous echo contrast or thrombus is seen in the body of the right atrium or the right atrial appendage. no atrial septal defect or patent foramen ovale is seen by 2d, color doppler or saline contrast with maneuvers. there is symmetric left ventricular hypertrophy. the left ventricular cavity size is normal. overall left ventricular systolic function is normal (lvef>55%). no masses or thrombi are seen in the left ventricle. right ventricular chamber size and free wall motion are normal. the ascending aorta is moderately dilated. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. mild (1+) aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. there is probably systolic anterior motion of the mitral valve leaflets. mild (1+) mitral regurgitation is seen. there is no pericardial effusion. impression: no cardiac source of embolism identified. cxr impression: left subclavian central venous catheter with its tip in the superior vena cava. no pneumothorax. cardiomegaly without evidence of congestive failure. cea, psa, esr, fibrinogen all pending. wbc-12.4* hct-44.3 plt ct-178 wbc-8.3 hct-39.2* plt ct-139* neuts-87.6* lymphs-8.7* monos-3.3 eos-0.3 baso-0 pt-14.0* ptt-26.8 inr(pt)-1.2 pt-13.5 ptt-49.5* inr(pt)-1.1 pt-13.2 ptt-38.1* inr(pt)-1.1 esr-5 glucose-121* urean-22* creat-1.0 na-137 k-4.2 cl-103 hco3-24 glucose-103 urean-20 creat-1.1 na-136 k-4.6 cl-103 hco3-29 alt-21 ast-22 ck(cpk)-292* alkphos-59 amylase-87 totbili-0.8 06:20pm blood ck-mb-3 ctropnt-<0.01 02:27am blood ck-mb-2 ctropnt-<0.01 10:00am blood ck-mb-2 ctropnt-<0.01 calcium-8.9 phos-2.9 mg-1.8 calcium-7.5* phos-2.0* mg-1.9 calcium-8.4 phos-3.2 mg-1.8 triglyc-46 hdl-50 chol/hd-2.9 ldlcalc-87 09:10pm urine blood-mod nitrite-neg protein-tr glucose-neg ketone-neg bilirubin-neg urobilngn-neg ph-6.5 leuk-neg 09:10pm urine rbc-* wbc-0-2 bacteria-rare yeast-none epi-0 06:20pm asa-neg ethanol-neg acetmnphn-neg bnzodzpn-neg barbitrt-neg tricyclic-neg brief hospital course: 71 y.o. male presented w l hemiplegia and neglect of injury admitted for r mca stroke. a summary of his pertinent hospital course by system follows: neuro pt presented from w l hemiplegia, dysarthria and neglect of injury. inital ct/cta on showed a large right mca infarct involving almost the entire territory supplied by this artery as well as almost total thrombosis of the origin of the right m2 segment with reconstitution of the distal branches. the patient was not a candidate for tpa because of time elapsed since onset of symptoms. follow-up head ct on showed evolving large right mca distribution infarct, but no acute intracranial hemorrhage. because of a persistent headache, pt was reimaged on and the head ct showed no evidence of new infarction or hemorrhage. the patient's stroke is currently of unknown etiology -- workup included carotid u/s, tte, and tee all of which were unrevealing for source of thrombus. with no known etiology as of , an oncologic w/u was initiated. esr, cea, fibrinogen, psa and a ct of the chest/abd/pelv were ordered and were pending as of 5pm. patient's l hemiplegia and l facial droop persisted through his entire hospital course. he did show improved understanding that he was not moving his l side, though he still maintained that he was capable of moving them. cardiac patient ruled out for mi. patient developed tachycardia and an irregular rhyhtm on . a cardiology consult was called that concluded the patient was demonstrating pvcs and svt likely secondary to effects of the stroke in the context of preexisting heart disease/cardiomyopathy. they recommended reinitiating bb and long term acei,as well as making sure ca/k/mg were properly repleted, and these recs were followed. pt continued to have intermittent pvcs through his hospital course. fen swallowing eval cleared the patient for soft solids and thin liquids. proph patient was initially given heparin 5000 u sc tid, pneumoboots, ppi. pt had isolated ptt rise from normal range to 49.5. heparin sc was d/c'd, ptt fell started to trend downward, ? causation. dispo pt/ot evaluated patient. pt was d/c'd to rehab facility. medications on admission: 1. atenolol 75 mg po qd 2. asa 325 mg qd 3. lipitor 20 mg qd discharge medications: 1. aspirin 325 mg tablet sig: one (1) tablet po daily (daily). 2. lipitor 20 mg tablet sig: one (1) tablet po once a day. tablet(s) 3. metoprolol tartrate 25 mg tablet sig: 0.5 tablet po bid (2 times a day). disp:*60 tablet(s)* refills:*0* 4. lisinopril 5 mg tablet sig: one (1) tablet po once a day: please start taking lisinopril on . disp:*60 tablet(s)* refills:*0* 5. pantoprazole sodium 40 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po q24h (every 24 hours). discharge disposition: extended care facility: rehab at med ctr discharge diagnosis: 1. right mca territory infarction 2. hypertension 3. cardiomyopathy discharge condition: stable. patient has l facial droop, unable to move l arm or l leg, l leg has postural response to noxious stimulus, patient exstinguishes to double simultaneous stimulation at lle and lue. patient has some neglect of injury: thinks he is capable of moving his l arm and l leg, does realize that he has not moved them. discharge instructions: patient may need ace inhibitor in about a week or so after bp stabilizes. keep all appointments. take all medications as prescribed. please call your doctor and return to emergency department for increased weakness, visual changes, or worsening confusion. followup instructions: please follow-up with your pcp 1 week after discharge from the hospital. patient will need to follow up with dr. in the clinic after discharge from rehab. call for an appointment. md, procedure: venous catheterization, not elsewhere classified diagnostic ultrasound of heart diagnoses: other primary cardiomyopathies gout, unspecified other specified cardiac dysrhythmias personal history of malignant neoplasm of kidney other pulmonary embolism and infarction other late effects of cerebrovascular disease, facial weakness cerebral thrombosis with cerebral infarction unspecified hypertensive heart disease without heart failure other specified hemiplegia and hemiparesis affecting nondominant side 

Answer: The patient is high likely exposed to