Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: aspirin / heparin agents / morphine / tylenol attending: chief complaint: hypotension at hd major surgical or invasive procedure: none history of present illness: 71 f with dm, cirrhosis nash and acetaminophen toxicity, h/o gastric angioectasia (gave/watermelon stomach) with gib, esrd on hd mwf, diastolic chf, hit, seizure dx on , admitted on to the icu for low hematocrit and hypotension. she went to hd on where she was found to be hypotensive in the 70's and 80's systolic. of note, ed documentation indicates that she may have received the wrong antihypertensive prior to dialysis. patient does not recall what medications she may have received, and only vaguely recalls the events surrounding her dialysis. she had a recent hospitalization from for fall complicated by tib/fib fracture, altered mental status (with contributions from uti, hepatic encephalopathy, and hypercapnea from narcotic use), esbl enterobacter uti, and heme positive stools. in the ed, her bp was 86/p, hr 70, rr 16, sat 100%ra. she was given 1l fluid, one unit of packed red cells, and platelets. she was guaiac positive. blood cultures were drawn and stool was sent for c-diff. inferior st changes were concerning for ongoing ischemia, and she was transferred to the micu. in icu, patient was given 2 unit prbc's and 1 unit of platelets. her bp improved thereafter and hct stabilized. she was then transferred to the floor for management. past medical history: recent history includes multiple admissions in , and for confusion in the setting of lactulose noncompliance, and in for hypotension. in , she was diagnosed with gib from gastric angioectasias/watermelon stomach. she was also found to have a portal vein thrombosis on ultrasound but was not anticoagulated for h/o gave, gib, hit. other past medical history: - portal vein thrombosis but not anticoagulated for h/o gave, gib, hit - type 2 diabetes. - end-stage renal disease, on hemodialysis m/w/f - cirrhosis nash and acetaminophen toxicity. - gastric angioectasia with h/o gi bleeding in 4/. - diastolic chf. ef>55% by echocardiogram in 7/. she has a prlonged mitral deceleration time and moderate mr. - ?right sided pleural effusion: diagnosed on u/s , cxr showed a small effusion - stayed stable in subsequent imaging. - heparin-induced thrombocytopenia, ab+ in 1/. - history of seizure disorder, on . - history of infection in the left knee. - history of mrsa and clostridium difficile. - history of gram-positive rod bacteremia in 4/. - status post orif of the left distal femur fracture 12/. social history: she was recently discharged to manor in . her daughter is involved in her care. the patient currently denies alcohol use, tobacco use, and illicit drugs. family history: noncontributory. physical exam: on transfer to the floor... vitals: tm 97, tc 95.9, hr 73, bp 120/51, rr 14, 97%ra general: comfortable, in no acute distress. : sclerae icteric, op clear, mmm, eomi heart: holosystolic murmur, radiating to the axilla, audible across precordium. lungs: mild crackles at left based, decreased on right, clear anteriorly abdomen: extremely obese, soft, + bowel sounds. cannot assess hepatosplenomegaly given body habitus. 2+ dependent edema. extremities: 2+ edema bilaterally, 2+ dp pulses, lue av fistula with palpable thrill neuro: a&o x 3, +mild asterixis, tremor. pertinent results: 05:00pm wbc-2.2* rbc-2.00* hgb-6.9* hct-21.3* mcv-107* mch-34.5* mchc-32.4 rdw-21.5* 05:00pm neuts-61.6 lymphs-29.7 monos-5.6 eos-2.9 basos-0.1 05:00pm glucose-125* urea n-17 creat-2.2*# sodium-144 potassium-4.4 chloride-107 total co2-30 anion gap-11 04:45pm calcium-8.9 phosphate-3.3 magnesium-1.7 05:12pm lactate-3.0* 05:00pm ck-mb-notdone ctropnt-<0.01 blood cultures: enterococcus and coag neg staph; w/ gnr urine cx 12/ll: mixed bacterial flora u/s : no e/o ascites but pleural effusion noted cxr : stable right-sided pleural effusion brief hospital course: 71 yo h/o gastric angioectasia (gave/watermelon stomach) with chronic gib, type 2 diabetes, esrd on hd (mwf), diastolic chf, hit, seizure dx on , admitted with hypotension and anemia in setting of chronic gib and polymicrobial (proteus, enterococcus, coag neg staph) cultures from picc line and av fistula. #) bacteremia: patient w/baseline low blood pressures in 80s/90s likely esld and generalized low systemic vascular resistence. cortisol stim from : 10.1->15.6. patient also with picc cultures growing pansensitive enterococcus faecalis and coag negative staph , corynebacterium species , proteus mirabilis , and gnrs , , and vanc resistant enterococcus faecium. surveillance cultures since has ngtd until on . ct scan without clear abdominal source. other source could be gi fistula (no fluid collection per us). tte negative for vegetations and no tee obtained as pt was not a surgical candidate given comorbidities. antibiotics were planned for full four week course. id followed her during her stay, and in the days preceeding her death, she was on a four drug pathogen-specific regimen. despite these treatments, ms. continued to have progressively poor hemodynamic stabily, until, despite pressors, her blood pressure declined causing cardiopulmonary arrest. #) hypotension: likely to sepsis and low svr due to esld. required pressor support throughout her stay. many family discussion were held, and, given her poor prognosis, it was decided that pressor support would not be escalated. thus, despite these treatments, ms. continued to have progressively poor hemodynamic stabily, until, despite pressors, her blood pressure declined causing cardiopulmonary arrest. #) cirrhosis secondary to nash/acetaminophen. hepatologist dr. . liver disease was end-stage upon admission. total bili and inr were monitor and continued to increase during her stay. associated illnesses included coagulopathy, thrombocytopenia, hypotension and chronic gi bleed. per liver consult obtained while inpatient, she was not a transplant candidate. she was continued on rifaximin, ursodiol, and lactulose. nadolol was held given hypotension. . #) anemia/gi bleed. has known chronic gi bleed from gave as well as insufficient erythropoeitin in setting of esrd. transfused for hct less than 21. #) thrombocytopenia. chronic, most likely from splenomegaly and esld with poor thrombopoetin levels. also has h/o hit so heparin products were avoided. transfused for platelets of < 10k for spontaneous bleed. #) esrd on hd. upon admission was requiring dialysis three times weekly, but becamed too hypotensive to tolerate hd. per renal, cvvh would be the next step, but this is not indicated given patient's continued hypotension and poor prognosis. #) type 2 diabetes: well controlled while inpatient with insulin sliding scale and qid glucose monitoring. #) diastolic chf. extravascular hypervolemia but continued to need pressor support to maintain adequate blood pressure. thus, diuretics were held while inpatient. #) seizure disorder. no observed seizures while in patient. continued on at home dose. #) s/p l tib-fib fracture: first noted upon follow-up appointment with orthopedics s/p orif. admitted with external brace. orthopedics followied while inpatient and determined no additional interventions were required. despite these treatments, ms. continued to have progressively poor hemodynamic stabily, until, despite pressors, her blood pressure declined causing cardiopulmonary arrest. medications on admission: pantoprazole 40mg q12h sevelamer 800mg tid with meals urosdiol 300mg rifaximin 400mg tid levetiracetam 500mg daily lactulose 30ml po qid propranolol 10mg discharge medications: patient expired. discharge disposition: expired discharge diagnosis: primary: sepsis secondary: esld, esrd, diastolic chf, chronic anemia discharge condition: expired. discharge instructions: expired. followup instructions: expired. md, procedure: venous catheterization, not elsewhere classified enteral infusion of concentrated nutritional substances hemodialysis transfusion of platelets diagnoses: end stage renal disease mitral valve disorders congestive heart failure, unspecified cirrhosis of liver without mention of alcohol iron deficiency anemia secondary to blood loss (chronic) severe sepsis other convulsions unspecified fall other specified septicemias septic shock other sequelae of chronic liver disease hepatic encephalopathy glucocorticoid deficiency chronic diastolic heart failure closed fracture of unspecified part of fibula with tibia angiodysplasia of stomach and duodenum with hemorrhage diabetes with renal manifestations, type ii or unspecified type, uncontrolled 

Answer: The patient is high likely exposed to