Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: l visual field loss major surgical or invasive procedure: none history of present illness: 66 rhm with parkinsons ds and afib not on coumadin was brought to the er for evaluation of visual changes. code stroke was called. he lives in and was visiting with his wife. they were at mfa next door to medical area this afternoon. while looking at the paintings, suddenly he noted that he had some difficulty in seeing things on the left side. this was especially of a problem, if he looked at things that were at the top and left side. he didnt have any other symptoms and immediatley told his wife. this happened at around 1 pm. 911 was called and he was brought to the ed for eval. past medical history: parkinson's a fib not on coumadin (afib diagnosed 10 years ago, was on coumadin for 1 month and then stopped, not on asa either) social history: lives with wife in , vt. no smoking, no alcohol or drugs. independent in adls, drives. family history: negative for parkinsons, positive for stroke in brother physical exam: admission exam: vitals: temp: 98.4 hr: 71 bp: 126/64 resp: 16 o(2)sat: 99 normal general: awake, cooperative, nad. heent: nc/at, no scleral icterus noted, mmm, no lesions noted in oropharynx neck: supple, no carotid bruits appreciated. no nuchal rigidity pulmonary: lungs cta bilaterally without r/r/w cardiac: rrr, nl. s1s2, no m/r/g noted abdomen: soft, mild llq tenderness, normoactive bowel sounds, no masses or organomegaly noted. extremities: no c/c/e bilaterally, 2+ radial, dp pulses bilaterally. skin: no rashes or lesions noted. neurologic: -mental status: alert, oriented x 3. attentive. speech normal. able to read, intact repetition . able to follow both midline and appendicular commands. pt. was able to register 3 objects but recalls at 5 minutes. the pt. had good knowledge of current events. there was no evidence of apraxia or neglect. -cranial nerves: i: olfaction not tested. ii: perrl 3 to 2mm and brisk. vf show left superior field cut. funduscopic exam revealed no papilledema, exudates, or hemorrhages. iii, iv, vi: eomi without nystagmus. normal saccades. v: facial sensation intact to light touch and pain vii: symmetric viii: hearing intact. ix, x: palate elevates symmetrically. : 5/5 strength in trapezii and scm bilaterally. xii: tongue protrudes in midline. -motor: normal bulk, some ep rigidity and cogwheel tone throughout. no drift no adventitious movements, such as tremor, noted. delt bic tri wre ffl fe io ip quad ham ta edb l 5 5 5 5 5 5 5 5 5 5 5 5 5 5 r 5 5 5 5 5 5 5 5 5 5 5 5 5 5 -sensory: intact to lt, pain, position and vibration -dtrs: tri pat ach l 2 2 2 2 1 r 2 2 2 2 1 plantar response was mute on both -coordination: no intention tremor, no dysdiadochokinesia noted. no dysmetria on fnf or hks -gait: defd. discharge exam (pertinent): cv: irregular rate, no murmurs neuro: visual fields full to confrontation and with red pin examination. mild cogwheeling, stooped posture and turning en bloc, positive pull test. diminished l knee jerk. atrophy of r calf and edb. toes upgoing bilaterally. pertinent results: 02:25pm blood wbc-5.8 rbc-4.24* hgb-13.7* hct-39.7* mcv-94 mch-32.3* mchc-34.4 rdw-13.3 plt ct-160 03:59am blood neuts-75.7* lymphs-17.3* monos-4.4 eos-1.9 baso-0.7 02:25pm blood pt-15.3* ptt-27.1 inr(pt)-1.3* 02:25pm blood glucose-114* urean-20 creat-1.0 na-142 k-3.5 cl-108 hco3-26 angap-12 03:59am blood alt-8 ast-19 alkphos-61 totbili-0.9 04:12am blood calcium-8.5 phos-2.9 mg-2.0 03:59am blood %hba1c-5.8 eag-120 03:59am blood triglyc-58 hdl-44 chol/hd-3.3 ldlcalc-89 ct/cta code stroke protocol with perfusion 1. region of abnormally-increased mtt within the right pca territory, involving a large portion of the right occipital lobe with a smaller region of decreased rcbv in the same distribution is consistent with a small "core" of infarcted tissue surrounded by a penumbra of ischemic tissue. 2. there is an associated abrupt "cut-off" of the p2-p3 segment of the right pca, with "meniscus" appearance, suggestive of acute embolic occlusion. 3. no evidence of intracranial hemorrhage. 4. minimal calcified plaque at the origin of the right vertebral artery, without hemodynamically-significant stenosis. mri 1. small acute infarctions within the right pca territory, specifically within the right body/tail of the hippocampus, right temporal lobe, and right thalamus. 2. no evidence of intracranial hemorrhage. echo no cardiac source of embolism seen. normal global and regional biventricular systolic function. negative bubble study. brief hospital course: neuro: 1. r pca stroke the patient presented with a left superior quadrantanopsia. a code stroke was called and the ct/cta/ct perfusion showed a r p2 cut-off, with large penumbra in that vascular territory. given the visualized blockage, the large size of at risk brain tissue, and the potential for debilitating visual loss, the decision was made to give tpa. the patient received tpa in the ed, and was admitted to the neuro icu for monitoring. after the tpa infusion was complete, his visual deficit remained, but the following morning it had resolved. mri was done 24 hours after tpa, there was no bleed, and 3 small areas of infarction in the r medial temporal lobe and thalamus, consistent with pca stroke. the etiology was most likely atrial fibrillation. cta showed no other vascular abnormalities in the posterior circulation. transthoracic echo showed mild left atrial enlargement, but normal global systolic function, and bubble study was negative for asd/pfo. the patient was started on dabigatran (pradaxa) for stroke with atrial fibrillation. fasting lipid panel showed cholesterol 145, tg 58, hdl 44, ldl 89. he was started on 10 mg simvastatin for secondary stroke prevention. he was seen by pt/ot, and given a prescription for outpatient pt for his parkinsons. there are no restrictions on his daily activities, including driving. 2. ?radiculopathy the patient had a diminished l knee jerk compared to the right side. he then mentioned a concern about r calf atrophy over the past 1 year. his right calf and foot muscles did in fact look atrophied compared to the left side. strength and sensation was intact. he has a history of chronic low back pain, though denies any radiating pains down the legs. he likely has bilateral radiculopathy. he may warrant mri of the l-spine as an outpatient, however there is likely no treatment indicated given he is asymptomatic. atrial fibrillation: patient was in continuous af during his hospital course. he had 1 episode of rapid ventricular response at rest that resolved with 1 dose of iv metoprolol. of note, his verapamil had been held to allow his blood pressure to autoregulate post-stroke. the day of discharge, he ambulated and climbed stairs with pt. he did have tachycardia to 140-160s during this exercise, but remained asymptomatic. we will therefore restart his verapamil at discharge and have him follow up with his cardiologist in the next 2 weeks. medications on admission: sinement -0.5 amantidine 100 mirapex 1-1-0.5 verapamil 360 mg daily discharge medications: 1. senna 8.6 mg tablet sig: one (1) tablet po bid (2 times a day) as needed for constipation: over the counter. 2. docusate sodium 50 mg/5 ml liquid sig: one (1) po bid (2 times a day) as needed for constipation: over the counter. 3. outpatient physical therapy evaluate and treat diagnosis: parkinsons 4. amantadine 100 mg capsule sig: one (1) capsule po bid (2 times a day) as needed for parkinson's disease. 5. carbidopa-levodopa 25-100 mg tablet sig: one (1) tablet po qam and q noon () as needed for parkinson's disease. 6. carbidopa-levodopa 25-100 mg tablet sig: 0.5 tablet po qpm (once a day (in the evening)). 7. pramipexole 0.5 mg tablet sig: one (1) tablet po qpm (once a day (in the evening)) as needed for parkinson's disease. 8. pramipexole 1 mg tablet sig: one (1) tablet po noon (at noon) as needed for parkinson's disease. 9. pramipexole 1 mg tablet sig: one (1) tablet po qam (once a day (in the morning)) as needed for parkinson's disease. 10. simvastatin 10 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 11. dabigatran etexilate 150 mg capsule sig: one (1) capsule po bid (2 times a day). disp:*60 capsule(s)* refills:*2* 12. verapamil 360 mg cap,ext release pellets 24 hr sig: one (1) cap,ext release pellets 24 hr po once a day. discharge disposition: home discharge diagnosis: r pca stroke atrial fibrillation discharge condition: mental status: clear and coherent. level of consciousness: alert and interactive. activity status: ambulatory - independent. discharge instructions: you were admitted with visual difficulties. you had a small stroke caused by a blocked blood vessel. you received a clot-busting medication called tpa to break up the clot. the stroke was caused by atrial fibrillation. you were started on a blood thinner called pradaxa. you were started on simvastatin for cholesterol and stroke prevention. followup instructions: you should follow up with your primary care doctor and your cardiologist within 2 weeks. md, procedure: injection or infusion of thrombolytic agent diagnoses: atrial fibrillation paralysis agitans cerebral embolism with cerebral infarction lumbago visual field defect, unspecified 

Answer: The patient is high likely exposed to