Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: shortness of breath. major surgical or invasive procedure: none. history of present illness: this history is taken from patient and her home aide, dodi. . this is an 88 yo woman not followed at with history of chf who started having dyspnea 2-3 days ago. she says she was diagnosed with chf less than one year ago, and is followed by dr. in cardiology at . says that her symptoms have been progressive over the last couple days and include doe, fatigue, orthopnea, and lower extremity swelling. she says she has also gained weight, but when asked to quantify the amount says that her weight has only increased pounds over the last six months. . on ros, she denies fever, cough or sputum production. no sick contacts. she lives in in . no history of recent prolonged travel or immobilization. no history of asymmetric leg swelling, redness or tenderness. denies recent history of chest pain, syncope or lightheadness, or palpitations. as above, she endorses symptoms of orthopnea (sleeps on pillows), bilateral leg swelling, abdominal fullness, and generalized fatigue/malaise. . at baseline, she can walk several hundred feet before becoming dyspneic. she seldom uses stairs anymore. she is not on home oxygen. she takes care of most adls, but has health aide and to help with iadls. . in the emergency department she was satting 93% on 2l. cxr showed pulmonary edema. bnp was 4281. first set of enzymes showed tropt of 0.03 with ck 98 and creatinine 1.1. cards was consulted and did not feel this was acs; they recommended against starting heparin drip. she was given 10 mg iv lasix x1 after which she had good urine output and was admitted to medicine for further management. past medical history: history of remote tuberculosis exposure hypothyroidism hypertension mitral valve prolapse cataracts/macular degeneration congestive heart failure with lvef 65% history of crohn's dz for which she took prednisone, sxs resolved severe obstructive pulmonary disease (fev1 55% expected--) echo (): la enlargement, severe mv prolapse social history: lives at ("boyleston"). competent in adls. has home health aide. remote smoking history of 20 pk-yrs. family history: nc. physical exam: vital signs: 98.9, 122/50 (pulsus was <8 mmhg), 82, 24, 95% on 2l weight: 66.5 kg general: well-appearing elderly woman, in mild respiratory distress with labored breathing; able to speak in full sentences heent: eomi, perrla, no conjuntival pallor, mmm neck: jvd to angle of mandible cardiac: rrr, normal s1/s2. 3/6 systolic murmur radiating to carotids bilaterally, also radiating to axilla. lungs: bibasilar crackles to 1/3 up posterior lung fields; scattered faint expiratory wheezes abdomen: distended; normal bowel sounds; non-tender extremities: no pitting edema, 1+ dp pulses bilaterally neuro: aaox3. moving all extremities. sensation grossly intact. pertinent results: labs at admission: 09:50am blood wbc-9.8 rbc-4.30 hgb-11.5* hct-33.8* mcv-79*# mch-26.8*# mchc-34.1 rdw-18.1* plt ct-217 09:50am blood neuts-84.6* lymphs-8.5* monos-4.0 eos-2.6 baso-0.4 09:50am blood glucose-198* urean-25* creat-1.1 na-133 k-4.8 cl-97 hco3-26 angap-15 09:50am blood calcium-9.2 phos-3.6 mg-2.2 01:30am blood type-art temp-36.4 fio2-97 o2 flow-4 po2-82* pco2-49* ph-7.39 caltco2-31* base xs-3 aado2-590 req o2-93 intubat-not intuba comment-nasal . cardiac enzymes: 09:50am blood ck(cpk)-98 06:55pm blood ck(cpk)-83 02:09am blood ck(cpk)-70 09:50am blood ck-mb-notdone probnp-4281* 09:50am blood ctropnt-0.03* 06:55pm blood ck-mb-notdone ctropnt-0.04* 02:09am blood ck-mb-notdone ctropnt-0.03* . cxr (): enlarged cardiac silhouette with prominence of interstitial markings suggestion of airspace opacities in the lower lung field, which may all represent pulmonary edema, although the chronicity of this process is unclear. correlation with presentation and other comparison to outside priors for continued radiographic followup is recommended. it is difficult to exclude an infectious process in the lower lung fields. . cxr (): although respiratory motion slightly limits the assessment, there has been apparent interval worsening of perihilar and basilar opacities attributed to pulmonary edema in the setting of cardiomegaly and vascular engorgement. small right pleural effusion is unchanged, but there is apparent new small left pleural effusion. . tte (from hosp): : normal lv size and wall thickness, without obvious regional wall motion abnormalities. overall left ventricular systolic function is normal. the estimated lvef is 65%. mild left atrial enlargement. mitral valve is myxomatous and the mitral valve is moderately diffusely thickened. degree of prolapse is severe. aortic valve is tricuspid and sclerotic. there is mild aortic regurgitation. mild-moderate tricuspid regurgiation is present. estimated right ventricular systolic pressure is 60 mmhg with an ra pressure 5 mmhg. . echo the left atrium is mildly dilated. there is mild symmetric left ventricular hypertrophy with normal cavity size and regional/global systolic function (lvef>65%). right ventricular chamber size is normal with mild global free wall hypokinesis. the diameters of aorta at the sinus, ascending and arch levels are normal. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. no aortic regurgitation is seen. the mitral valve leaflets are mildly thickened with moderate/severe systolic prolapse/partial posterior leaflet flail. torn mitral chordae are present. an eccentric, anteriorly directed jet of at least moderate (2+) mitral regurgitation is seen. the tricuspid regurgitation jet is eccentric and may be underestimated. there is moderate pulmonary artery systolic hypertension. there is no pericardial effusion. impression: mitral valve prolapse/partial flail with at least moderate mitral regurgitation. pulmonary artery systolic hypertension. right ventricular free wall hypokinesis. mild symmetric left ventricular hypertrophy with preserved global and regional systolic function. brief hospital course: an 88 yo woman with h/o diastolic chf and severe mitral valve prolapse presenting with progressive shortness of breath, fatigue, now with new oxygen requirements. . her symptoms at admission were most consistent with a chf exacerbation, especially given her elevated bnp and cxr showing pulmonary edema. slight elevation of tropt (with flat ck) also supported diagnosis of chf exacerbation. also considered in ddx were acute worsening of mitral regurgitation, pericardial effusion, acs, pulmonary process (pe seemed less likely given history of progressively worsening symptoms, no signs/symptoms of dvt). . cardiac enzymes were cycled x3. ck was flat and tropt remained elevated but stable at 0.03 to 0.04. she was diuresed overnight with iv boluses of lasix, for total of 60 mg. she had good urine output and symptoms improved on first hospital day, but there was concern about persistent oxygen requirement and crackles on exam. she was started on metoprolol for slowing of heart rate in setting of diastolic dysfunction and need for increased diastolic filling time. she was then transferred to cardiology service for initiation of lasix gtt. . on the medicine floor, received a cummulative total of 80mg of iv lasix and total net output was 950cc. a dose of 40 resulted in a bp drop to 92/40, she had previously been in the 110s on the floor and 140s at home. diuresis of 950cc resulted in little clinical improvement as the pt was still markedly dyspneic, tachpneic and reliant on 3l nc with sat of 93%. she was therefore transferred to the cardiology service to try diuresis with a lasix drip. . while on the cardiology floor she was started on hydralazine for afterload reduction. she was placed on a lasix drip but progressively became more oliguric, putting out only 400cc urine daily, and her creatinine trended up from 1.1 to 2.3 over 5 days. she was started on imdur and metolazone on . in addition, she developed a leukocytosis to 23 along with diahrrea, and was therefore begun on flagyl. . echo showed a torn mitral chordae. she was not a candidate for mitral valve replacement. she was then transfered to the ccu for tx with lasix gtt and milrinone in attempt to reduce afterload. her mental status worsened and she eventually became obtunded. then the lasix and milirnone were stopped and nirtoprusside was started instead. her blood pressure continued to deteriorate. the attending met with the health care proxy and the decision to change the goals of care to comfort measures only was made. she was started on a morphine gtt and her other medications were stopped. she remained unresponsive and was transfered to the floor. she passed away on the night of at approximately 11:58 pm. medications on admission: vitamin d cereforin fish oil caps vitamin c calcium citrate 1000 mg qday vitamin a aspirin 81 mg qday benicar 40 mg qday bupropion 100 mg qday paxil 20 mg qday levoxyl 75 mcg qday actonel 35 mg qweek on sat spiriva inh albuterol inh omeprazole 20 mg qday discharge medications: deceased. discharge disposition: expired discharge diagnosis: deceased. discharge condition: deceased. discharge instructions: deceased. followup instructions: deceased. md procedure: diagnostic ultrasound of heart diagnoses: mitral valve disorders urinary tract infection, site not specified congestive heart failure, unspecified acute kidney failure, unspecified unspecified acquired hypothyroidism chronic airway obstruction, not elsewhere classified hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage i through stage iv, or unspecified other chronic pulmonary heart diseases chronic kidney disease, unspecified acute on chronic combined systolic and diastolic heart failure 

Answer: The patient is high likely exposed to