Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: multiple ich, status epilepticus major surgical or invasive procedure: none history of present illness: 55y male known to have seizure disorder, cocaine abuse, etoh abuse, transferred from caritas (chfh) with status epilepticus. he was known to be a daily cocain sniffing abuser, and seemed that he abused it last night, too. according to the family, they heard large thump approximately 3am this morning, and found that the patient was down on the floor under the stairs. he was unresponsive, mumbling (?). family took him to couch, which he could cooperate partially. they called his older sister to ask to come in. around 4:30am, sister came and thought that he might have one of his regular seizure (once monthly gt seizure). he was snoring loudly. since he did not wake up over several hours, family called emt. upon arrival, emt suctioned small amount of vomit from his mouth and transfered to chfh. there he was found to have continuous acitivity of generalized tonic seizures and in status. he was paralyzed, intubated and sedated. he was loaded with dilantin. head ct showed bilateral thalamic hemorrhage, midbrain hemorrhage, right frontal ich, traumatic? sah with collection of blood in the lateral ventricles and brain contusion at bilateral temporal lobes. patient was transferred to for further treatment. at arrival to ed, he was persistently seizing on and off, received propofol to stop seizure. the seizure stopped but dropped bp to 80/40, propofol was discontinued. but afterwards, no visible seizure at ed besides, occasional tonic posturing when he was touched for exam. patient was admitted to icu neurology service for further seizure and ich management. ros: cocaine abuse daily. once per month base regular seizure. no fever, no cough, diarrhea, recent sickness. past medical history: pmh: seizure disorder burns (from fire, aprox 80%, skin grafted at ) etoh abuse cocain abuse (daily basis, last intake last night?) per family, admitted to psych hospital. social history: sh: history of etoh and cocain abuse as above. family history: fh: unknown. brief hospital course: 55y male with (1) multiple ich (bil thalamus, r frontal, sah, ivh (2) status epi mechanism: (1) cocaine abuse (2) fall (3) neuroleptic malignant syndrome? pmh: seizure disorder, burns (from fire, aprox 80%, skin grafted at ), etoh abuse, cocain abuse (daily basis, last intake last night?) per family, admitted to psych hospital. #1 bilateral thalamic hemorrhage, r frontal lobe homorrhage. mechanism likely to be hypertensive 2ndary to htn by cocaine abuse. control bp (sbp 120-160) and control seizure. lp showed intracranial htn (op 32.5), blood. control seizure, withdrawal to avoid icp elevation. neurosurgery consulted at ed, will follow up pt. - stable repeat ct. continue to be rigid, unresponsive, unstable vs, trendeling up ck (983). suspected for neuroleptic malignant syndrome (nms). started on dantrolene 0.25mg/kg/dose (15mg/dose) q12h. - titrate up dantrolene to 0.5mg/kg/dose (30mg/dose) q12h, started bromocriptin 2.5mg q6h. patient rigid, shiverring, febrile--> withdrawal (etoh, cocaine?) +/- nms: midazolam drip titrate up (2.5mg/h) mri : stable or improved bithalamic hemorrhage, r frontal hemorrhage. no stroke. mra unremarkable. # neuroleptic malignant syndrome +/- etoh withdrawal pt continuous to be rigid, febrile, unstable vs/hr, elevated ck. suspected for nms, started on dantrolene. - toxicology consult: likely to be nms due to severe rigidity (more than etoh withdrawal). but certainly coexists. use sedative propofol (x due to hypotension) or midazolam drip (safely upto 110mg/h). will see pt (dr., ) - psych consult:malignant hyperthermia? - dantrolene increased to 3mg/kg/dose q12h (6mg/kg/dose) - dantrolene increased to 4mg/kg/dose q12h (6mg/kg/dose), bromocriptine 5mg q6h. - dantrolene redistributed to 2mg/kg/dose q6h #2 sah, ivh, bilateral temporal lobe contusion mechamism likely to be traumatic. same as above. #3 status epilepticus loaded with dilantin, with current pht level 13.2. will continue on 100mg q8h iv. midazolam prn seizure. eeg continuous bedside monitor was requested to neurophysiology team. decide further aed management based on eeg, clinical findings. - eeg no seizure activity (occasional spike at bitemporal lobe)--> will continue pht 100q8. - pht level daily - eeg portable : l frontotemporal discharge, disorganized bg - pht 5.7 loaded 500mg, increased to 150tid. - pht 200mg q8h # on ciwa protocol. - will manage withdrawal symptom by midazolam drip (upto 10mg/h) and dc ativan. - dc midaz drip. started on ativan 2mg q6h. go slow for benzo weaning. #5 fever start on antibiotics (vancomycin, ceftriaxone, acyclovir). bcx, ucx, csf cx need to be followed up. - fever got better after dantrolene and bromocriptin were started. - 101f, cultured. started on clindamycin for pansinusitis. #6 respiratory intubated. on vent. keep on vent for tonight. possible aspiration (emt suctioned vomitus from mouth at the scene). - cxr normal. #7 cv bp control sbp 120-160. cez neg x3. - very unstable bp, hr. sedated with propofol and prn neo. - stabilized after started and increased dantrolene, bromocriptine #8 id - pansinusitis, started on clindamycin #9 hem n.p. #10 metabolic/fen started tf - 45ml/h #11 abdomen famotidine ppx. on , the patient was made comfort measures only after speaking with the family and he expired. medications on admission: risperdal, topamax, effexor discharge medications: expired discharge disposition: expired discharge diagnosis: intracerebral hemorrhage discharge condition: expired discharge instructions: expired followup instructions: expired md, procedure: insertion of intercostal catheter for drainage venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more spinal tap incision of lung insertion of endotracheal tube enteral infusion of concentrated nutritional substances diagnoses: unspecified essential hypertension unspecified fall alcohol abuse, unspecified intracerebral hemorrhage iatrogenic pneumothorax cocaine abuse, continuous epilepsy, unspecified, with intractable epilepsy acute sinusitis, unspecified other and unspecified cerebral laceration and contusion, without mention of open intracranial wound, with prolonged [more than 24 hours] loss of consciousness without return to pre-existing conscious level neuroleptic malignant syndrome 

Answer: The patient is high likely exposed to