Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: sob, doe major surgical or invasive procedure: vats history of present illness: 82 yo man with history of prostate ca, hyperlipidemia, recently diagnosed afib (incidentally found while doing an elective hand surgery)several mos ago, who presented to several weeks ago () with pleuritic chest pain and cough with productive green/yellow sputum; had a rul pna and stemi (troponin peak at 70 with an ekg that showed 2 mm st segment elevation in v2-v5); med management. was d/c'ed on asa and coumadin. in house, pt had adenosine mibi, which showed anterior apical and inferoapical fixed defect with peri-infarct, a reversible defect; ef 65%. cxr showed likely pna (prior xray done at wh showed interstitial markings c/w fibrosis). echo showed mild mr of 63%, mod dilated rv with depressed rvsf and mild pah, moderately dilated ra, mod depressed lv function. since discharge, pt has been doing poorly, and was re-admitted to the c/o sob and doe. was in ccu from till when was transferred to micu for management of ? underlying pulm process. his initial presentation was felt to be consistant with subacute mi and perimi chf as well as pna. he has been on nrb from admission till pm when he failed a trial of bipap and was intubated. he was net > 3l negative without improvement in oxygenation. pt was initially on dopa and vasopressin; now vasopressin weaned off. was initially on levofloxacin; switched to zosyn/vanco on . initial cxr showed diffuse alveolar opacities. ct shows diffuse ground glass, multifocal consolidations, loculated effusions. after intubation, an attempt was made to place cordis (unsuccessful); needs sgc to r/o decompensated chf; then bronch and possible vats. past medical history: prostate cancer 3 yrs ago afib on coumadin stemi 1 mos ago: never cath'ed tte ef decreased to 40%, o/w unchanged from previous social history: occasional etoh, never smoked; lives with family family history: n/c pertinent results: 12:50pm wbc-16.4* rbc-3.59* hgb-11.4* hct-33.8* mcv-94 mch-31.7 mchc-33.7 rdw-13.5 12:50pm plt count-145* 12:50pm neuts-84.1* lymphs-11.7* monos-4.0 eos-0.1 basos-0.1 12:50pm pt-33.5* ptt-46.1* inr(pt)-7.1 . 12:50pm glucose-123* urea n-25* creat-0.6 sodium-138 potassium-4.2 chloride-102 total co2-26 anion gap-14 . 12:50pm cortisol-40.6* 04:53pm lactate-1.0 12:50pm ck(cpk)-69 12:50pm ck-mb-notdone ctropnt-0.12* . 01:11pm urine color-ltamb appear-clear sp -1.023 01:11pm urine blood-sm nitrite-neg protein-30 glucose-neg ketone-neg bilirubin-neg urobilngn-neg ph-6.5 leuk-neg 01:11pm urine rbc-* wbc-* bacteria-rare yeast-none epi-0-2 trans epi- 04:53pm po2-65* pco2-44 ph-7.42 total co2-30 base xs-3 brief hospital course: 1. respiratory failure: initial differential diagnosis included (and was felt likely a combination of these): chf in the setting of a recent mi, pna, ards, interstitial disease or pe given rle dvt. there was a question of iatrogenic pneumothorax during l subclavian line placement, which resolved on serial chest x-rayds. pt was clinically diagnosed with ards and put on ards net ventilation (6cc/kg). pt required increased peep of at least 10 to maintain oxygenation. esophageal balloon was placed for a period of time to measure more accurate peep. fio2 was titrated down to 0.50. he was empirically treated for pneumonia with broad spectrum antibiotics of vanco, zosyn, levo. pt was found to be hypotensive requiring pressors and it was unclear if he was in heart failure vs sepsis. a swan ganz catheter was placed, which was more consistent with septic physiology. pt was unable to be diuresed secondary to his persistently lowish blood pressures both on and off pressors. bronch was performed on which was negative for legionella, fugus, viruses, dfa, pcp. fluid was exudative by light's criteria. sputum culture from found 2+ gnr and all other culture data including blood and urine remained negative during the hospitalization. pt's esr was 84 and was anca and negative. serial cxrs remained unchanged showed patchy alveolar and interstitial opacity of the left chest greater than the right, consistent with ards. cta was negative for pe and showed biltaeraly effusion, persistent diffuse ground glass opacity with multifocal parenchymal consolidation in an asymmetric pattern consistent with diffuse multifocal pneumonia, ards, and less likely asymmetric congestive heart failure. repeat echo was unchanged with slightly depressed lv systolic function (>50%), 3+ tr, and no pericardial effusion. pt had a vats procedure performed by thoracic surgery on with a lung biopsy which showed chronic fibrosing lung disease predominantly of end stage lung tissue with honeycomb change. very little alveolar tissue was present in the biopsy but showed focal evidence of organizing pneumonitis, raising the possibility of a superimposed acute process such as infection. some of the histologic features of the chronic fibrosing lung disease raised the possibility of usual interstitial pneumonia. pt was give high dose pulse steroids for 3 days without improvement in his oxygenation and ventilator settings. it was felt that pt did not have steroid responsive interestitial lung disease. given the fact that pt's clinical status showed no improvement over the past several weeks and it was highly unlikely that the pt could ever come off the ventilator, a family discussion was held and the decision was made to make the pt . pt was extubated and he expired shortly thereafter. . 2. cv a. rhythm: pt remained in afib, which was adequately rate-controlled on digoxin. he was anticoagulated with heparin. pt was noted to be in brachycardia in afib and digoxin was discontinued. b. ischemia: pt is s/p stemi with slightly depressed lv systolic function. pt was continued on aspirin, statin. antihypertensives were held in the setting of his hypotension. c. pump: ischmemic cardiomyopathy s/p mi. swan not consistent with cardiogenic shock; more consistent with hypovolemia, sepsis. severe tr could make swan readings misleaded. pt was bolused for cvp>12. very little diuresis was attempted/accomplished since he was likely clinically dry despite total body volume overload. . 3. rle dvt: a large rle dvt dx'd on le ultrasound. pt was continued on heparin drip, which he was already on for afib. cta was negative for pe . 4. anemia: most likely chronic disease from fe studies; mcv 95. folate and b12 both normal. . 5. fen: ngt placed by fluoro. given tube feeds. lytes repleted prn . 6. proph: ppi, heparin gtt discharge medications: none discharge disposition: expired discharge diagnosis: ards, chronic interstitial lung disease, afib, dvt discharge condition: expired discharge instructions: none followup instructions: none procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more insertion of endotracheal tube fiber-optic bronchoscopy enteral infusion of concentrated nutritional substances non-invasive mechanical ventilation closed [endoscopic] biopsy of bronchus pulmonary artery wedge monitoring transfusion of packed cells open biopsy of lung transfusion of other serum infusion of vasopressor agent diagnoses: pneumonia, organism unspecified congestive heart failure, unspecified atrial fibrillation acute and chronic respiratory failure cardiac arrest postinflammatory pulmonary fibrosis other and unspecified coagulation defects systemic inflammatory response syndrome due to noninfectious process with acute organ dysfunction subendocardial infarction, subsequent episode of care 

Answer: The patient is high likely exposed to