Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: fevers, abd pain major surgical or invasive procedure: picc line history of present illness: 29 y.o. male with hx of mental retardation, s/p kidney transplant 21 years ago on immunosuppressants and hep c x20 years presents with mental status changes, anorexia and fevers. went to pcp's office for evaluation, found to have fever to 102 and tender abd. has hx of ascites liver failure and hypoalbuminemia. mother also reports that pt had brbpr 2-3 days ago with bowel movements. stools have been brown with no diarrhea. pt denies n/v. doe snot report abd pain but mother states that he does not always say when is bothering him because he does not like hospitals. pt gets liver care at . mother also reports that pt has become tranfusion dependent over past 2 years and last transfusion . baseline hct 27-33. in , pt was guiac + with no bright red blood. pericentesis revealed 6500 wbc with 88% pmns. pt received one dose of ctx in ed. pt was also hypotensive to 80's in ed which resolved with fluids. past medical history: cirrhosis hep c s/p kidney transplant- living related at age 7. dandy walker cyst/ mental retardation depression blindness social history: lives at home with parents. pt is able to understand everyting. sometimes with delayed responses. family history: non-contributory physical exam: vs t:101.1, p:86, bp:85/60->129/92 rr16 o2sat: 100% general: mildly pale male, nad. no jaundice heent: pupils equal, scleral injection on the l, pale conjunctive, no icterus. neck: supple with no lad. cardiovascular: rrr no murmurs. lungs: ctab abdomen: mild distension. no fluid wave. no tenderness to deep palpation. extremities: mult ecchymoses on legs. no edema, no jaundice. neuro: , pt responding yes and no to all questions. does not respond to questions about location and date. mother feels that ms is as baseline. pertinent results: 07:48pm ascites tot prot-0.8 glucose-90 ld(ldh)-82 albumin-less than 07:48pm ascites wbc-6850* rbc-525* polys-88* lymphs-0 monos-3* mesotheli-1* macrophag-8* 04:22pm lactate-3.3* 04:10pm urine color-amber appear-clear sp -1.014 04:10pm urine blood-lg nitrite-neg protein-neg glucose-neg ketone-neg bilirubin-neg urobilngn-1 ph-5.0 leuk-neg 04:10pm urine rbc-* wbc-0-2 bacteria-mod yeast-none epi-0-2 04:10pm urine hyaline-* 04:06pm glucose-95 urea n-37* creat-1.0 sodium-137 potassium-4.9 chloride-106 total co2-22 anion gap-14 04:06pm alt(sgpt)-64* ast(sgot)-100* alk phos-113 amylase-90 tot bili-4.0* 04:06pm calcium-8.5 phosphate-4.0# magnesium-1.6 04:06pm wbc-11.7*# rbc-2.53* hgb-9.7* hct-27.0* mcv-106*# mch-38.1* mchc-35.8* rdw-23.1* 04:06pm neuts-88* bands-7* lymphs-4* monos-1* eos-0 basos-0 atyps-0 metas-0 myelos-0 04:06pm hypochrom-normal anisocyt-1+ poikilocy-2+ macrocyt-3+ microcyt-occasional polychrom-1+ ovalocyt-occasional schistocy-occasional burr-1+ teardrop-occasional acanthocy-1+ elliptocy-occasional 04:06pm plt count-113* 04:06pm pt-14.7* ptt-30.2 inr(pt)-1.5 : cxr: lordotic positioning. the heart is not enlarged. there is no chf. there is a small right base effusion. the left costophrenic sulcus is clear. aside from some atelectasis at the right base, the lungs are grossly clear. no free air is detected beneath the diaphragm. clips are noted over the mid abdomen. a/p: 1. colonic wall thickening involving the ascending colon and hepatic flexure, consistent with colitis. 2. no evidence of bowel obstruction. 3. cirrhosis with ascites, splenomegaly, patent portal vein. 4. bilateral pleural effusions, right more than left. : no evidence of cholecystitis. the gallbladder is distended as it was previously in , but the gallbladder is otherwise unremarkable in appearance. no intra- or extrahepatic biliary ductal dilatation. brief hospital course: 29 y.o. male with hx of mr and kidney transplant presents with fevers and ascites. found to have sbp and c.diff colitis. . # sbp: tap showed 6850 with 88% pmn's. met criteria for sbp. bandemia on diff. this was felt to be likely spontaneous as no other recent abd procedures. pt hypotensive on admission but resolved with fluid. no abd tenderness. elevated tbili but no evidence of obstruction on u/s, likely cirrhosis. pt remained hemodynamically stable, and was initially maintined on cipro 500mg po bic for 5 days. however, the decision was made by the liver service to contine on cipro. patient' cultures remained negative. . # anemia: on admission pt's mother reported that he had had some slight blood in stools recently. hct was initially 27 but then dropped to 23 and pt was transfused 2 units. hct initially came up to 28.9 but then on the 3rd day of admission he had a black tarry stool with slight amount of blood. gi was consulted who felt that egd/colonoscopy was indicated once patient stable. hct again stabilized in 29-30 range, but on , again fell to 21. patient's inr also noted to be 2.2. patient transfued 1u prbc, and am hct 30. patient also maintained on vitamin k. however, on the morning of , patient with 100cc of brbpr. he remained hemodynamically stable, but was transferred to the micu for onservation. patient's bleeding felt likely c-diff colitis in setting of negative ng lavage. patient also reconfirmed to have an elevated inr (> 2) and a falling fibrinogen from 103 to 192 on the medical floor to 59 in the icu. patient supported with prbc to keep hct > 28, platlets with goal > 50 in setting of bleeding, ffp with goal inr < 1.7 and cryoprecipitate with goal fibrinogen > 100. . # diarrhea: mother stated this has been present for several months and pt has been taking imodium almost every other day to help control it. on hd#2 stool cultures turned positive for c.diff and pt was started on flagyl. plan to continue on flagyl for 14 days, but during admission, patient with worsening diarrhea. hence, cholestyramine added to regimen. patient remained afebrile, but course was complicared by both guiac positive stool and on , 100cc of bright red blood per rectum. patient remained hamodynamically stable, but was transferred to the micu. there, patient had a negative ng lavage. patient had a ct of a/p, which revealed colitis consistent with c-diff infection. patient supported with blood products (see below) and sent to the floor on . . # s/p kidney transplant: pt has never had problems with kidney. creat remained stable throughout hospitalization. patient was on prednisone outpatient as part of his immunosuppresant regimen, and felt to have adrenal insufficiency in setting of beirf hypotension on admission, and so patient mainteined on stress dose steroids with dexamethasone at 4mg . this was initially tapered on the floor, but dose increased to 4mg in icu. . # weakness/ataxia: pt has developed worsening ataxia over the past few years per mother. he is very weak. ? of whether secondary to steroid mcypathy vs adrenal insufficiency. again, patient maintained on stress dose steroids, but was too weak during his hospitalization to ambulate or participate with pt. . # cirrhosis: pt's child- class is c. patient also with grade i esophageal vricies. patient initially on nadalol and spirolactone, but both stopped in setting of gib. however, spironolactone gradually added back in setting of blood product support. liver service consulted and followed throughout hospitalization. . # septic shock: on morning of , pt developed worsening abdominal pain found to be febrile. peritoneal fluid with 70 wbc, but grew yeast (c. albicans). blood cx from grew yeast as well and pt was started on caspofungin with id following. on am pt was increasingly hypotensive on the floor and pt transferred to micu where he required pressors to maintain adequate blood pressure. pt required intubation and continued hemodynamic support. despite maximal medical management pt's condition continued to detoriate. after discussions with family and icu attending, decision made to withdrawl care and concentrate on comfort. pt died from respiratory failure secondary to septic shock shortly thereafter. medications on admission: fosamax spironlolactone 25mg qd nadolol 20 qd prednisone 5mg qd imuran 75mg qd discharge medications: none discharge disposition: home with service discharge diagnosis: primary diagnoses: c-diff colitis spntaneous bacterial peritonitis dic lower gi bleed fungemia septic shock secondary diagnoses: hep c cirrhosis ascites discharge condition: deceased discharge instructions: none followup instructions: none procedure: venous catheterization, not elsewhere classified venous catheterization, not elsewhere classified insertion of endotracheal tube injection or infusion of thrombolytic agent percutaneous abdominal drainage percutaneous abdominal drainage arterial catheterization insertion of other (naso-)gastric tube gastric lavage transfusion of packed cells insertion of (naso-)intestinal tube transfusion of platelets control of epistaxis by anterior nasal packing diagnoses: acidosis thrombocytopenia, unspecified unspecified essential hypertension other specified disorders of pancreatic internal secretion adrenal cortical steroids causing adverse effects in therapeutic use cirrhosis of liver without mention of alcohol acute posthemorrhagic anemia acute kidney failure, unspecified severe sepsis portal hypertension unspecified viral hepatitis c without hepatic coma infection with microorganisms resistant to penicillins other convulsions methicillin susceptible staphylococcus aureus septicemia acute respiratory failure defibrination syndrome hypotension, unspecified other shock without mention of trauma septic shock intestinal infection due to clostridium difficile other complications due to other vascular device, implant, and graft other sequelae of chronic liver disease disseminated candidiasis hemorrhage of gastrointestinal tract, unspecified dehydration other complications due to other internal prosthetic device, implant, and graft epistaxis profound impairment, both eyes, impairment level not further specified unspecified intellectual disabilities spontaneous bacterial peritonitis esophageal varices without mention of bleeding congenital hydrocephalus purulent endophthalmitis, unspecified 

Answer: The patient is high likely exposed to