Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: this is a 46-year-old man with a past medical history significant for alcohol and cocaine abuse who presented with rapidly increasing ascending weakness since five days prior to admission. according to his mother, whom i talked with (and whom he lives with), he was having general weakness and malaise, along with a cough (but no fevers, chills, nausea, vomiting, or diarrhea). over the weekend prior to admission and then sunday at 3:30 a.m., he got up to go to the bathroom, and his knees buckled, and he fell. he was admitted to an outside hospital that day, and since then, his weakness worsened. according to a neurologic exam at the outside hospital the day prior to admission, he had developed shortness of breath, using accessory muscles. there was a lower motor neuron right facial weakness, slurred speech, 3 out of 5 distal upper extremity weakness and 2 out of 5 proximal upper extremity weakness, and 2 out of 5 lower extremity weakness. there was also an absence of reflexes. toes were downgoing. he was transferred to the for further evaluation and management for a question of syndrome. reportedly in the first 24 hours of the process, he was having weakness in the lower extremities and paresthesias in his hands. past medical history: alcohol abuse. cocaine abuse. allergies: no known drug allergies. medications on admission: keflex 500 mg b.i.d., clonidine 0.1 mg q.week, folic acid, thiamin, multivitamin, labetalol 400 mg b.i.d., 10 mg iv p.r.n., ativan p.r.n., he was also given a dose of ampicillin, rocephin, and vancomycin prior to transfer from the outside hospital for an increasing white blood cell count at 12.7 up from 10.9 on admission. social history: alcohol and cocaine abuse. he lives with his mother. smoking. physical examination: vital signs: temperature 97.8??????, blood pressure 119/54, heart rate 82, respirations 13, oxygen saturation 100% on room air. general: the patient was in no acute distress. he appeared comfortable. heent: moist mucous membranes. oropharynx clear. no scleral icterus or injection. neck: supple. no lymphadenopathy. no carotid bruits appreciated. lungs: clear to auscultation bilaterally anteriorly. heart: regular, rate and rhythm. normal s1 and s2. abdomen: soft, nontender, nondistended. extremities: warm. there were 2+ peripheral pulses throughout. no edema. neurological: mental status exam showed the patient to be alert, awake, and following commands. he made attempts to mouth words but was intubated on admission. he became very agitated and anxious. by discharge, he had received a trach, and with his passy-muir valve, was able to talk very well. cranial nerves: pupils equal, round and reactive to light. initially his extraocular eye muscles were intact; however, during most of the hospital course, he appeared to have some extent of ophthalmoplegia. this has been recovering well. the patient also has a right lower motor neuron cranial nerve facial palsy, although facial sensation is intact bilaterally and sternocleidomastoids intact bilaterally. at the time of admission, shoulder shrug was weak but present throughout the hospital course. the patient did lose the ability to shrug his shoulders; however, that has returned as well. motor: the patient had normal bulk on admission, but extremities were flaccid. there were no fasciculations. the proximal upper extremities were 2 out of 5 bilaterally, and 3- out of 5 distally. in the lower extremities, 0 out of 5 bilaterally. through most of the hospital course, the patient did developed quadriplegia; however, he did begin to improve, and by , he could again lift his arms, and by , his biceps were 4 out of 4 bilaterally, triceps 4 out of 4 bilaterally. the distal upper extremities were 3 out of 5 bilaterally, and the lower extremities were 3 out of 5 throughout. sensation has been intact throughout the entire hospital course to pin/temp. laboratory data: on , white count was 9.1, hematocrit 30.7, the patient's hematocrit was in the low 30s through most of his admission; all of his stool guaiacs were negative; platelet count 428; inr has been 1.0 throughout admission; serial urinalysis has been negative; csf showed 1 white cell, 75 red cells; stool guaiac negative; glucose 144, bun 25, creatinine 0.6, sodium 142, potassium 4.1, chloride 107, bicarb 25; alt 37, ast 24, alkaline phosphatase 52, total bilirubin 0.4, calcium 9.5, phosphate 3.1, magnesium 2.1; iga 323; total protein in the csf was 176, glucose 76; sputum from , showed sparse growth of oropharyngeal flora and sparse growth of gram-negative rods; sputum culture from , showed staphylococcus aureus coag-positive, rare growth, and rare growth of gram-negative rods. the staph coag-positive was resistant to only penicillin; gram-negative rods also only resistant to penicillin; sputum culture from , showed moderate growth of oropharyngeal flora and staphylococcus aureus coag-positive, moderate growth; sputum culture from , showed moderate growth of oropharyngeal flora, staphylococcus aureuss coag-positive, moderate growth; sputum culture from , showed sparse growth of oropharyngeal flora; urine culture on , showed no growth; urine culture on , showed no growth; urine culture on , again was no growth; urine culture on , showed no growth; urine culture on , showed no growth; urine culture on , showed no growth; blood cultures from , , , , , were all negative, no growth; c-diff from , was negative; second c-diff from , was negative; fecal culture showed no salmonella or shigella, and campylobacter culture was negative; mrsa screens were negative; vre screens also were negative; bronchoalveolar lavage done on , grew moderate growth of oropharyngeal flora, staph coag-positive, moderate growth, and beta streptococci not group a, moderate growth; csf gram stain was negative, and fluid culture was no growth; blood fungal culture showed no fungus isolated; blood avb culture showed no macrobacteria isolated. emg revealed sensory nerve conduction studies of the left radial and right seral nerve which were normal. sensory nerve conduction studies of the left median nerves and left ulnar nerve revealed markedly reduced response amplitudes. ulnar conduction velocity was severely reduced and median conduction velocity was normal. motor nerve conduction study of the left median nerve revealed severely prolonged distal latency, normal response amplitude, and mildly reduced conduction velocity. ................. responses were absent. motor nerve conduction study of the left median nerve distal to the carpal tunnel revealed moderately prolonged dl with normal ra. motor nerve conduction study of the left ulnar nerve revealed markedly prolonged vl, mildly reduced ra, and mildly slowed cv throughout the length of the nerve with more severe focal slowing of the elbow, ............. responses were absent. motor nerve conduction study of the bilateral tibial nerves revealed moderately prolonged distal latency, markedly reduced ras and normal cv in the right tibial nerve with mildly reduced cv in the left tibial nerve. ... responses were absent in both tibial nerves. partial conduction block was identified in both tibial nerves by decreased in ra at 55% in the right tibial nerve and 62% in the left tibial nerve. bilateral blink reflexes were absent. emg of selected muscles in the right lower extremity representing the l1-s2 myotomes revealed markedly reduced recruitment of normal motor unit potentials in tibialis anterior and no motor unit activity in gastrocnemius and vastus lateralis. poor activation was seen in the right iliopsoas, and evaluation of the motor unit potentials was suboptimal. nonspecific increase insertional activity was seen in the right tibialis anterior and right vastus lateralis. emg of the left tibialis anterior and left iliopsoas revealed moderately to markedly reduced recruitment of normal motor unit potential. no motor units were activated in the left deltoid. the impression was that this was an abnormal study. there is electrophysiologic evidence for a moderately severe acute generalized demyelinating polyneuropathy as in syndrome. hospital course: the patient was admitted in transfer from an outside hospital on . he remained in the intensive care unit until . on presentation, he had already been intubated, and he remained quadriplegic through most of the admission. he initially underwent a 5-day course of ivig which was begun on the day of admission, and following this, the patient received supportive care and close monitoring for his neurologic condition. respiratory wise, he developed what was felt to be an aspiration pneumonia with sputum cultures growing staphylococcus aureus coag-positive, and a chest x-ray revealed left retrocardiac opacity, and he was treated with seven days of zosyn and also received several days of vancomycin. the patient continued to spike fevers throughout the course of antibiotics, and so on day #7, the antibiotics were stopped. the patient spiked fevers until , and since has been afebrile. his last chest x-ray on , showed marked improvement in the basilar patchy opacities, and there was no evidence of congestive heart failure. the patient's blood cultures and urine cultures revealed no growth. on , the patient underwent placement of a tracheostomy and a peg tube for further support. by , the patient had been weaned off the vent and was maintained by trach collar. cardiovascularly the patient had very difficult-to-control hypertension throughout the hospital stay. his heart rate also tended to be in the 80s to low 100s throughout the hospital course. initially the patient was receiving enalapril, metoprolol, and would be sedated with propofol, which also seemed to lower his blood pressure. as the days went on, he was changed to p.o. labetalol and given a clonodine patch to attempt to wean his sedation, as well as keep his blood pressure down. the enalapril was also increased, and he was continued on the hydralazine. he required ativan in quantities of 25-30 mg/day after the propofol was finally discontinued when the patient was weaned from the vent. in an attempt to decrease the amount of ativan that was required, the patient's clonodine patch was increased to three patches, and the patient was changed on , to klonopin, which is currently at 2.5 mg t.i.d., which has also helped reduce the amount of ativan needed. furthermore, the patient was started on zoloft on , for a question of depression and anxiety. the electrolytes have been mostly stable throughout his hospital course. initially the patient was having decreasing sodium, and this was felt to be due to siadh, likely related to his .................. syndrome. the patient was started on salt tablets 3 g t.i.d. per the neurology sicu service, and this was eventually weaned off, as his sodium became stable. for nutrition, the patient was maintained on tube feeds throughout the hospital course and also given protime pump inhibitor and bowel regimen for prophylaxis. he additionally has been getting regular fingersticks and a regular insulin sliding scale to control blood sugars, which have ranged typically from 100 to 140 or 150. renally the patient has not had any issues. on , the patient was called out of the intensive care unit to the floor. he has not been febrile since . his respiratory status is currently stable, and trach collar requires suctioning approximately every four hours. neurologically the patient has dramatically improved and is now again able to lift all of his extremities, antigravity, and is somewhat stronger than that in the proximal upper extremities. he has received his passy-muir valve and is talking well and is interactive. the plan will be to monitor him on the floor until he is well enough to go to rehabilitation. he has been seen by physical therapy throughout his hospital stay, and this will continue when he is on the floor. he is also continued on thiamin, folate, and a multivitamin in regards to his history of alcohol abuse. as far as his hypertension goes, he will be continued p.o. labetalol, enalapril, and for now the hydralazine as needed; however, we will attempt to increase labetalol in order to stop the hydralazine, and the clonodine patches are also stopped. the patient is on klonopin for anxiety, and this should only be weaned very slowly to avoid withdraw if this is necessary. the remainder of the discharge summary will be dictated by the oncoming resident on the patient's discharge. , m.d. dictated by: medquist36 procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more spinal tap incision of lung percutaneous [endoscopic] gastrostomy [peg] temporary tracheostomy closed [endoscopic] biopsy of bronchus injection or infusion of immunoglobulin diagnoses: urinary tract infection, site not specified unspecified essential hypertension dysthymic disorder acute respiratory failure pneumonitis due to inhalation of food or vomitus staphylococcus infection in conditions classified elsewhere and of unspecified site, other staphylococcus quadriplegia, unspecified acute infective polyneuritis other protein-calorie malnutrition 

Answer: The patient is high likely exposed to