Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: sulfa (sulfonamide antibiotics) attending: chief complaint: shortness of breath major surgical or invasive procedure: whole lung lavage x 1, intubation, left ij, arterial line x 2, femoral lines x 2 history of present illness: 27yom with history of pulmonary alveolar proteinosis, dyslipidemia, obstructive sleep apnea presenting with shortness of breath. . the patient was diagnosed with pulmonary alveolar proteinosis in when he presented with progressive dysnea with minimal exertion. he was initially treated for antibiotics and nebulizers for a presumed uri vs pneumonia, but had progressive symptoms with hypoxia and underwent a ct chest that revealed bilateral ground glass opacities. he subsequently underwent a vats wedge resection of the rll on which showed pulmonary alveolar proteinosis. since his diagnosis, he has had two whole lung lavages, the first at the time of diagnosis which was complicated by hypoxia after left lung lavage. he next presented to with worsening dyspnea on exertion, and repeat ct chest showed recurrence of protein alveolar proteinosis, more prominent in the right lung. he underwent another right whole lung lavage and was weaned from 4l nc to room air prior to discharge. post-lavage, the patient was noted to be hypoxic to the mid to high- 80's on room air with ambulation and the patient was instructed to use home oxygen with exertion and and at night. of note, the patient was also diagnosed with a lobar pneumonia in that was treated with azithromycin x5 days with improvement of symptoms. he subsequently had increased productive cough and dyspnea on exertion late that was treated with another course of azithromycin x5 days with improvement of symptoms. . the patient's current symptoms began last night when he began experiencing increasing shortness of breath with a cough productive of white, stringy sputum. he denies fevers, chills, sick contacts, yellow or green sputum, diarrhea, nausea, vomiting. he presented to medical center and was found to have a normal white count. cta showed extensive bilateral ground glass opacities. he was hypoxic to the 70's**** and was placed on bipap at the osh with improvement of po2 in the low 90s. he was empirically started on levofloxacin and diuresed with ****** with improvement of symptoms, and was weaned to nasal cannula. he was made npo in anticipation of a toatl lung washout at given the high suspicion of recurrent pap and was transferred to for a whole lung lavage on friday and monday. . on arrival to the micu, the patient was comfortable breathing on nasal cannula without shortness of breath at rest in bed past medical history: - pulmonary alveolar proteinosis: dx open lung biopsy, s/p whole lung lavage in , s/p right lung lavage - obstructive sleep apnea - hyperlipidemia - pyloric stenosis as a child social history: tobacco: denies. - alcohol: drinks one to two drinks daily. - illicits: denies. lives with family. does not work. family history: father: niddm, mother: pat, sister: asthma physical exam: vitals: t: 98.1 bp: 122/76 p: 84 r: 15 po2: 96%2l nc general: alert, oriented, no acute distress heent: pupils equal and round, sclera anicteric, mmm, oropharynx clear neck: supple cv: regular rate and rhythm, normal s1 + s2, no murmurs, rubs, gallops lungs: clear to auscultation bilaterally, no wheezes, rales, ronchi abdomen: soft, non-tender, non-distended, bowel sounds present, no organomegaly ext: warm, well perfused, equal dp pulses b/l, no clubbing, cyanosis or edema neuro: moving all extremities, strength 5/5, grossly normal sensation pertinent results: admission labs: 12:26am blood wbc-5.4 rbc-5.07 hgb-15.2 hct-44.0 mcv-87 mch-30.1 mchc-34.6 rdw-13.5 plt ct-197 12:26am blood neuts-77.6* lymphs-13.0* monos-6.9 eos-2.1 baso-0.4 12:26am blood pt-10.8 ptt-32.9 inr(pt)-1.0 12:26am blood glucose-171* urean-14 creat-0.7 na-136 k-4.1 cl-100 hco3-29 angap-11 11:56am blood alt-60* ast-92* ld(ldh)-466* ck(cpk)-133 alkphos-44 totbili-0.7 11:56am blood ck-mb-3 ctropnt-0.05* probnp-295* 12:26am blood calcium-9.0 phos-2.9 mg-2.0 imaging: cxr: findings: in comparison with the study of , there is little change. relatively low lung volumes may account for the prominence of the transverse diameter of the heart. again, scattered radiation related to the size of the patient makes it difficult to properly evaluate the lungs. however, there is no definite vascular congestion or acute pneumonia. of incidental note is a right-sided aortic arch. cxr: the patient is intubated with the et tube being at the level of the clavicular heads, approximately 7.4 cm above the carina. heart size is enlarged. there is less prominence of the right mediastinal silhouette which still reflects distended superior vena cava as well as mediastinal deposition of the fat. right lower lobe consolidation is redemonstrated, slightly improved since the prior examination. there is, on the other hand, progression of the left lower lobe opacity, that might be concerning for interval development of infectious process as well. left internal jugular line tip is at the level of the junction of left internal jugular vein with brachiocephalic vein. ng tube tip is most likely in the stomach. left chest tube is in place. ct head: impression: 1. diffuse cerebral edema with effacement of the ventricles, basal cisterns, and crowding of the foramen magnum. this is worrisome for a global hypoperfusion and infarction. 2. opacification of the ethmoidal air cells, sphenoid sinuses, and right mastoid air cells. brief hospital course: 27yom with history of pulmonary alveolar proteinosis, dyslipidemia, obstructive sleep apnea presenting with shortness of breath and white sputum, now admitted for lung lavage complicated by hypoxia, pea arrest # increased icp/cerebral edema: noted on eeg and recent ct scan, herniation is imminent. temporizing measures were performed with hyperventilation, mannitol and hypertonic saline initially until all of family arrived. neurology consulted and patient had minimal cerebral reflexes and his prognosis remained poor. on the morning of at 9:40 am the patient expired. death note: called to see patient for seizure and inability to ventilate plus hypoxia. on exam the patient did not respond to verbal or physical stimuli. absent heart and breath sounds. absent peripheral pulses. pupils are fixed and dilated. no corneal reflexes. patient pronounced dead at 0940. dr. notified. , and were notified via phone and arrived within minutes. both wanted an autopsy. medical examiner dr. declined examination. organ donation spoke to patient's family and agreed to undergo organ donation. # s/p pea arrest: etiology either acidosis, hypoxia, or pneumothorax. down time is not exact, but appears to be anywhere from 15-30 minutes. pt was placed on arctic sun cooling protocol and was rewarmed. neurology and post arrest team followed patient. has been off sedation with no response. initially with increased myoclonic jerks and seizure activity and started on keppra and high doses of versed. ultimatley, eeg developed flatlining pattern consistent with cerebral edema which is very poor neurologic prognostic sign. confirmed with ct head. demise as above. pea arrest events: - prior to procedure 95-100% 6 liters with c/o cough, general anesthesia induced and his single lumen ett moved to double lumen, ett procedure aborted due to difficulty ventilating the left lung with ventilation only returning volumes of 30-55 cc with tv of 600cc tv delivery and rapid hypoxia recovered when both lungs were ventilated. patient then awoke and was extubated but immediately was gasping with sats in 80s-90s on 100% fio2, reintubated without incident. then became more hypoxic to 50-60% and initially hypertensive and tachycardic, then within < 10 minutes became hypotensive and bradycardic -> pea arrested. - no breath sounds appreciated to left lung during code, thoracics placed left sided chest tube with bloody output. copious frothy pink secretions from ett during code. - per notes: 10:05 -> had saturations around 70-80% and then became hypotensive and bradycardic and lost pulse. 0959 pea arrest and cpr initiated. - had rounds of epi, 2 mg of atropine, 100 meq of bicarbonate, left chest tube placed, calcium chloride given. tee in or showed htn/tachycardia in setting of global biventricular dysfunction given 80 mcg iv ntg due to concern for pulmonary edema. - circulation recovered and patient was transferred to the micu for further care as above. . # seizures: noted on eeg and evidence on initial cooling. also noted myoclonic jerks on initial evaluation. eeg has shown high amplitude bursts, keppra was uptitrated. # possible pneumothorax: noted to have uneven breath sounds during the code. unclear if actual pneumothorax as no air leak, however chest tube remained and ultimately placed to water seal without incident. #. dyspnea/pulmonary alveolar proteinosis: the patient was diagnosed with pulmonary alveolar proteinosis by ct scan showing bilateral ground glass opacities. iniitially presented with shortness of breath and white sputum concerning for recurrence of pulmonary alveolar proteinosis, and is s/p lavage x 1 friday, and extubated post procedure. he did have difficulty with ventilation on friday and the procedure had to be terminated prior to full completion. he was quite acidemic during this episodie, but recovered on his own without incident. his lung lavage on the right was to occur , but underwent pea arrest during intubation. further management deferred. gm-csf was ordered prior to death and was pending at time of death. medications on admission: - simvastatin 20mg po qhs - vitamin b, e, and d - fish oil discharge medications: none discharge disposition: expired discharge diagnosis: pulmonary alveolar proteinosis hypoxic respiratory failure leading to death discharge condition: death discharge instructions: death followup instructions: none md procedure: insertion of intercostal catheter for drainage continuous invasive mechanical ventilation for less than 96 consecutive hours continuous invasive mechanical ventilation for 96 consecutive hours or more other intubation of respiratory tract cardiopulmonary resuscitation, not otherwise specified other operations on lung diagnoses: pneumonia, organism unspecified acidosis hyperpotassemia obstructive sleep apnea (adult)(pediatric) pulmonary collapse other and unspecified hyperlipidemia other specified procedures as the cause of abnormal reaction of patient, or of later complication, without mention of misadventure at time of procedure iatrogenic pneumothorax cardiac arrest anoxic brain damage cerebral edema morbid obesity surgical or other procedure not carried out because of contraindication central nervous system complication acute respiratory failure following trauma and surgery generalized convulsive epilepsy, without mention of intractable epilepsy body mass index 50.0-59.9, adult pulmonary alveolar proteinosis 

Answer: The patient is high likely exposed to