Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: hemoptysis major surgical or invasive procedure: bronchoscopy bronchial artery embolization pa catheter placement esophageal balloon placement cardioversion history of present illness: 65 y/o m w/cad s/p cabg, gerd, htn, pmr on steroids who presented to hospital on c/o hemoptysis. for past 2 days had been coughing up approximately 1-1.5 cups of blood. at osh, initial hct was 29.4 ct chest showed severe centrilobular/preseptal emphysema, bilateral lower lobe bronchiectasis, bilateral airspace consolidations in lul, lll, rml, and rll. bronchoscopy showed main bronchi full of blood, no no endobronchial lesions. hct dropped to 23.6, increased to 27 after 2 units prbcs. he was given vitamin k 10 mg sq and hydrocortisone 100 mg, and transferred here for further eval. past medical history: 1. gerd 2. htn 3. anxiety 4. cad s/p cabg 5. hypercholesterolemia 6. interstitial lung disease 7. pmr on steroids social history: lives with wife. + tobacco. retired machinist. family history: noncontributory physical exam: t: 96.8 p: 62 bp: 122/73 97% gen: intubated/sedated heent: ncat, plethoric face neck: supple lungs: cta anterior lung fields cv: rrr, no m/r/g abd: soft, nt/nd. +bs. ext: no c/c/e pertinent results: 07:28pm blood wbc-12.5* rbc-3.18* hgb-9.1* hct-26.9* mcv-85 mch-28.8 mchc-34.0 rdw-15.5 plt ct-302 05:12am blood wbc-9.1 rbc-3.46* hgb-9.8* hct-29.1* mcv-84 mch-28.3 mchc-33.6 rdw-15.1 plt ct-203 04:55am blood neuts-84* bands-6* lymphs-3* monos-0 eos-0 baso-0 atyps-0 metas-0 myelos-7* nrbc-1* 04:55am blood pt-18.4* ptt-33.2 inr(pt)-2.3 08:23am blood fdp-40-80 04:55am blood fibrino-524* 10:40pm blood glucose-272* urean-38* creat-2.1*# na-138 k-4.8 cl-101 hco3-25 angap-17 08:00am blood glucose-151* urean-24* creat-1.0 na-144 k-4.4 cl-109* hco3-28 angap-11 04:15am blood glucose-142* urean-22* creat-1.0 na-146* k-3.9 cl-109* hco3-29 angap-12 04:05am blood glucose-133* urean-24* creat-1.2 na-145 k-3.7 cl-108 hco3-25 angap-16 03:05am blood glucose-114* urean-18 creat-1.1 na-145 k-4.0 cl-108 hco3-28 angap-13 07:28pm blood ck(cpk)-134 07:28pm blood ck-mb-3 ctropnt-0.01 05:12am blood anca-positive 10:41am blood type-art temp-37.1 po2-50* pco2-69* ph-7.19* calhco3-28 base xs--3 06:19am blood type-art temp-36.7 rates-40/ tidal v-550 peep-15 fio2-100 po2-49* pco2-69* ph-7.21* calhco3-29 base xs--2 aado2-603 req o2-98 intubat-intubated 05:01am blood type-art temp-37.1 rates-46/ tidal v-550 peep-12 fio2-100 po2-50* pco2-64* ph-7.22* calhco3-28 base xs--2 aado2-607 req o2-98 -assist/con intubat-intubated 08:40pm blood type-art temp-36.7 rates-22/0 tidal v-700 peep-10 fio2-100 po2-96 pco2-44 ph-7.38 calhco3-27 base xs-0 aado2-591 req o2-95 -assist/con intubat-intubated 04:39pm blood lactate-0.9 08:40pm blood lactate-1.4 k-4.2 10:41am blood hgb-9.8* calchct-29 o2 sat-77 chest cta : ct of the chest without and with intravenous contrast: there are multiple lymph nodes seen within the anterior mediastinum, specifically the pretracheal region, aortopulmonary window, and prevascular spaces. no axillary or hilar lymphadenopathy is identified. there are bilateral alveolar air-space opacities located primarily centrally, however, there are also some dependent areas that are also opacified. while this patient has a history of pulmonary hemorrhage, the central distribution and symmetrical pattern of these opacities is more typical of ards or diffuse pneumonia. there is no focal source of bleeding identified. these airspace opacities are superimposed on an interstitial lung disease displaying thickened septae and mild diffuse areas of bronchiectasis. the airways are patent to the subsegmental bronchi bilaterally. coils are seen adjacent to the right main bronchus, presumably from the patient's recent angiography procedure. the patient is intubated, and an ng tube is seen coursing into the stomach, terminating in the gastric antrum. the visualized lung, spleen, and pancreas are unremarkable. no dissection within the aorta or pulmonary embolus within the pulmonary artery is identified. impression: 1. bilateral, central, primarily symmetrical air-space opacity with some dependent component which is most likely pulmonary hemorrhage with a possible ards or diffuse pneumonia component. this process is superimposed on a background of interstitial lung disease as seen on the prior ct. 2. coils seen adjacent to the right main bronchus, as patient is status post pulmonary angiography procedure. no focal source of bleeding identified on the ct scan pulmonary angio:bronchial artery embolization of second and third order branches off the right bronchial artery to the right upper and middle lobes, embolized with coils. brief hospital course: he was admitted to the micu and placed on antibiotics for pneumonia. it was felt that his hemoptysis was due to his bronchiectasis and interstitial lung disease. the morning after admission, he began having spontaneous bleeding into the ett. he had a bronchoscopy that demonstrated bleeding from the rul and rll. he was taken to ir and underwent embolization of 2nd and 3rd order branches of the r bronchial artery. he developed ards. he was paralyzed to minimize the possibility of coughing episodes that would cause subsequent bleeding. an esophageal balloon was placed to monitor transpulmonary pressure. he remained hypoxemic. he underwent 2 subsequent bronchoscopies on and which revealed thick secretions with blood, but no active bleeding. he then developed afib with rvr and hypotension. he was cardioverted with transient response to nsr. amiodarone was loaded and he was cardioverted again, this time staying in nsr. he had a pa catheter placed to further evaluate his hypotension, which revealed rv pressures 49/17, pa pressures 57/39, and wedge 22. svr 450, co 4.5. he developed sq air felt secondary to barotrauma, so his peep was decreased. cxr did not demonstrate tension ptx. because of worsening hypoxemia, he was proned, with mild improvement. he developed arf, creatinine went from 1.0 to 2.3. on , a family meeting was held regarding goals of care given his overwhelming respiratory failure. his fiance agreed that he would want to be kept comfortable. he was made cmo, and died one hour later with his family by his side. medications on admission: atenolol zetia protonix prednisone alprazolam discharge medications: none discharge disposition: expired discharge diagnosis: bronchiectasis ards renal failure discharge condition: expired discharge instructions: none followup instructions: none procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more fiber-optic bronchoscopy fiber-optic bronchoscopy injection or infusion of other therapeutic or prophylactic substance arteriography of other intrathoracic vessels atrial cardioversion closed [endoscopic] biopsy of bronchus closed [endoscopic] biopsy of bronchus pulmonary artery wedge monitoring other endovascular procedures on other vessels transfusion of packed cells infusion of vasopressor agent diagnoses: polymyalgia rheumatica esophageal reflux pure hypercholesterolemia unspecified essential hypertension long-term (current) use of steroids acute kidney failure, unspecified unspecified septicemia iron deficiency anemia secondary to blood loss (chronic) severe sepsis atrial fibrillation coronary atherosclerosis of unspecified type of vessel, native or graft aortocoronary bypass status acute respiratory failure other shock without mention of trauma postinflammatory pulmonary fibrosis other and unspecified coagulation defects bronchiectasis without acute exacerbation 

Answer: The patient is high likely exposed to