Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: past medical history: 1. hypertension. 2. history of upper gi bleed from esophagitis. 3. barrett's esophagus. 4. osteopenia. 5. hyponatremia secondary to siadh. 6. history of alcohol abuse about 25 years ago. 7. carotid stenosis. 8. copd with pulmonary function tests revealing an fvc of 53%, fev1 57, preserved ratio, and decreased dlco. admission medications: 1. albuterol. 2. amlodipine 5 mg b.i.d. 3. labetalol 200 mg p.o. b.i.d. 4. protonix 40 p.o. q.d. 5. lisinopril 20 mg p.o. q.d. allergies: the patient has no known drug allergies. social history: he is a smoker with 70 pack years, quit about ten years ago. alcohol: past heavy use, quit 25 years ago. physical examination on admission: vital signs: temperature 99, blood pressure 106/40, pulse 95, respiratory rate 18, saturating 99% on a ventilator. general: the patient was an intubated gentleman, sedated. heent: the mucous membranes were moist. the neck was supple, unable to assess jvd. cardiovascular: irregular, s1, s2 with no murmurs. lungs: rhonchi in the upper lung zones bilaterally with coarse breath sounds. there were no wheezes or crackles. abdomen: soft, nontender, nondistended with hyperactive bowel sounds. extremities: there was 1+ pitting edema. laboratory/radiologic data: the initial laboratories showed a white count of 10, hematocrit 35, platelets 380,000. sodium 122, potassium 4.7, chloride 87, bicarbonate 23, bun 11, creatinine 0.7. hospital course: 1. respiratory failure: the patient's initial presentation was somewhat unclear. was treated initially for a copd flare with a combination of tachycardia, hypertension; findings of pulmonary edema on x-ray suggest most likely that he had flash pulmonary edema to explain his hypoxia. our approach, therefore, was more tailored to relieving pulmonary congestion. this was unfortunately extremely difficult as during the first several days the patient was profoundly hypotensive. he received more fluids in the context of the hypotension as well as the concern that this might be sepsis which led to worsening of his volume overload. he was initially somewhat difficult to oxygenate requiring high b pressures to maintain good 02 saturations. he then remained relatively stable for about ten days and about the middle of the second week developed a fever, a white count, and productive sputum. the sputum grew enterobacter cloacae. he was, therefore, now believed to have a ventilator associated pneumonia. he was treated for this with levofloxacin and gentamicin. after about three weeks, with improvement in his cardiovascular function and decrease of total body overload, the patient was able to do well on pressure support ventilation and was eventually extubated. he is currently requiring face mask with saturation between 90-92. of note, given the patient's copd and chronic c02 retention, he will require 02 saturations in 88-92%. 2. cardiovascular: the patient initially presented in atrial fibrillation. this is apparently a new diagnosis for him as he has no history of that. he was placed on iv heparin and multiple attempts to slow down his heart rate were unsuccessful. he was placed on esmolol and diltiazem drip and the combination of these were only able to maintain the heart rate at about 150. a consultation with ep was obtained. the patient underwent a tee as well as dc cardioversion with no success. his medical regimen was eventually maximized to a beta blocker, a calcium channel blocker and amiodarone which led to a decent control of the heart rate between 90 and 100. two echocardiograms, one transthoracic and one transesophageal, both revealed normal left ventricular function, thus making the diagnosis of diastolic dysfunction much more likely. his left atrium at 6 cm is enlarged and probably is the explanation of his continuing atrial fibrillation. there is no evidence of significant valvular disease on echocardiogram as well as no evidence for any abnormal wall motions. it is, therefore, most likely that he has diastolic dysfunction which with improvement in heart rate and diuresis led to improvement in his clinical status. 3. infectious disease: as mentioned previously, the patient had enterobacter ventilator-associated pneumonia. secondary to this, he also developed enterobacter sepsis with hypotension requiring frequent amounts of iv boluses as well as pressors. he, however, recovered without significant consequences of his sepsis. 4. neurological: during week number two, the patient was noted to have jerking myoclonal motions in his right arm and right leg as well as decreased movements in his left arm and left leg. consultation with neurology was obtained. of note, the patient has been on dilantin for ten years and this was discontinued in for unknown reasons. the neurologist's opinion was that the patient might be seizing as the antibiotics and the stress of the admission may have lowered his seizure threshold. the patient was started on dilantin and he soon dramatically improved. his jerking monoclonal movements resolved and he had symmetric movements in both extremities. he did, however, still have some weakness on the left side that his daughter attributes to a tremor in the past. he will probably require an mri to evaluate for the possibility of central nervous system lesion which may be contributing to his left-sided weakness. 5. nutrition: the patient was receiving tube feeds and tolerated this very well. 6. endocrine: on initial admission, the patient's cortisol was found to be low in the context of what was believed to be hypotension of sepsis. he was, therefore, presumably diagnosed with adrenal failure and was started on hydrocort. this is currently being weaned as he has been on this medication for about three weeks. condition on discharge: currently, the patient is sitting in a chair, although was quite deconditioned, able to talk in short sentences. he was not complaining of shortness of breath. disposition: he was discharged to a regular medical floor. discharge diagnosis: 1. chronic obstructive pulmonary disease. 2. congestive heart failure with diastolic dysfunction. 3. hypoxic respiratory failure requiring intubation. 4. ventilator-associated pneumonia. 5. enterobacter sepsis. 6. adrenal insufficiency. 7. possible seizure disorder. 8. atrial fibrillation. , m.d. dictated by: medquist36 procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more continuous invasive mechanical ventilation for 96 consecutive hours or more diagnostic ultrasound of heart insertion of endotracheal tube insertion of endotracheal tube fiber-optic bronchoscopy enteral infusion of concentrated nutritional substances percutaneous [endoscopic] gastrostomy [peg] arterial catheterization temporary tracheostomy pulmonary artery wedge monitoring bronchoscopy through artificial stoma diagnoses: congestive heart failure, unspecified hyposmolality and/or hyponatremia atrial fibrillation other convulsions acute respiratory failure chronic obstructive asthma with (acute) exacerbation streptococcal septicemia occlusion and stenosis of carotid artery with cerebral infarction other adrenal hypofunction 

Answer: The patient is high likely exposed to