Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: amiodarone / univasc attending: chief complaint: hypotension/rapid afib major surgical or invasive procedure: femoral a-line history of present illness: 77f with history of atrial fibrillation, htn and depression is admitted to micu with hypotension and rapid afib. history was taken from both patient and family, however details from both were very limited. . pt was in her usoh until sunday of this week when at this time she had an episode of chest pain. unclear about characteristics, duration, or circumstances of complaints. pain resolved but for remainder of the week she felt very fatigued. then 24 hours prior to admission she developed nausea, vomting, diarrhea. no obvious blood in the emesis, no melena. denied any fevers. she also noted increasing dyspnea on exertion. . in the ed she was found to be in rapid afib in the 150's. they attemptd to decrease rate with diltiazem however patient became hypotensive and required levophed. a vbg was noted to be 7.09/65/57/21. she had acute renal failure with a creatinine of 2.1. her lactate was 3.8. she was empirically given levofloxacin and vancomycin for possible infection. a central line was placed and she was given 6 l of fluid. her cvp was in the high teens after this resuscitation. she was then transferred to the micu with a rapid heart rate. past medical history: 1. atrial fibrillation 2. interstitial lung disease; reticular pattern bilateral bases with hypodensity in liver potentially presumably secondary to amiodarone 3. asthma 4. htn 5. depression social history: lives with daughter. history of smoking, etoh use family history: non-contributory physical exam: vitals: 99.1(in ed) bp 80/56 hr 120's-130's rr 20 98% nrb attempted pulsus however was unable to hear bp sounds gen: awake, slightly agitated, initially a&o times 3 heent: perrl, + arcus senilus, eomi, anicteric, dry mm neck: unable to assess jvd as patient was laying flat in slight trendelenberg for lbp lung: breath sounds heard throughout, slightly diminshed in left base. no rhonchi, rales, wheezes heart: tachy, irregularly irregular, heart sounds heard well abd: soft, mild ruq tendern3ess, no , + bs per ed no stool in vault, guaiac negaitve ext: cool, palpable dp bilaterally, no edema pertinent results: 01:08pm blood wbc-11.0# rbc-4.73 hgb-14.3 hct-44.9 mcv-95 mch-30.2 mchc-31.9 rdw-13.6 plt ct-132* 01:08pm blood neuts-79.4* bands-0 lymphs-15.6* monos-4.4 eos-0.4 baso-0.2 03:15pm blood pt-22.3* ptt-53.2* inr(pt)-3.6 01:08pm blood glucose-177* urean-34* creat-2.1*# na-142 k-5.2* cl-103 hco3-19* angap-25* 01:08pm blood ck(cpk)-64 01:08pm blood calcium-9.2 phos-5.3* mg-2.2 09:33pm blood wbc-12.7* rbc-3.65* hgb-11.1* hct-36.3 mcv-100*# mch-30.3 mchc-30.5* rdw-13.5 plt ct-109* 11:41pm blood wbc-16.8* rbc-3.94* hgb-11.8* hct-37.0 mcv-94 mch-30.0 mchc-31.9 rdw-13.4 plt ct-103* 03:20pm blood pt-15.5* ptt-28.1 inr(pt)-1.7 09:33pm blood glucose-131* urean-32* creat-1.9* na-146* k-5.5* cl-120* hco3-11* angap-21* 09:36pm blood glucose-144* urean-33* creat-2.0* na-145 k-5.9* cl-119* hco3-15* angap-17 11:41pm blood glucose-74 urean-32* creat-2.1* na-153* k-6.7* cl-119* hco3-20* angap-21* 03:15pm blood alt-37 ast-37 alkphos-57 amylase-38 totbili-0.6 09:36pm blood alt-45* ast-48* ld(ldh)-184 ck(cpk)-60 alkphos-54 totbili-0.5 01:08pm blood ctropnt-0.06* 03:15pm blood ctropnt-0.04* 09:33pm blood ck-mb-notdone ctropnt-0.09* 09:36pm blood ck-mb-6 ctropnt-0.09* 09:33pm blood calcium-6.0* phos-7.2*# mg-1.7 11:41pm blood calcium-7.5* phos-8.3*# mg-2.4 03:26pm blood type- po2-57* pco2-65* ph-7.09* calhco3-21 base xs--11 09:43pm blood type-art po2-119* pco2-63* ph-7.01* calhco3-17* base xs--16 10:13pm blood type-art po2-210* pco2-54* ph-6.91* calhco3-12* base xs--23 11:14pm blood type-art po2-227* pco2-40 ph-7.01* calhco3-11* base xs--21 11:47pm blood type-art fio2-100 po2-133* pco2-67* ph-7.04* calhco3-19* base xs--13 aado2-519 req o2-86 intubat-intubated comment-manual bag 12:33am blood type-art po2-90 pco2-52* ph-6.97* calhco3-13* base xs--21 03:26pm blood lactate-3.8* 09:43pm blood lactate-4.9* 10:13pm blood lactate-8.4* 11:14pm blood lactate-8.0* . ct abd impression: 1. marked inflammatory stranding around the pancreatic head with poor definition of the pancreatic parenchyma in this region. these findings are most consistent with pancreatitis. please note that pancreatic necrosis cannot be excluded on this non contrast enhanced scan. 2. marked gallbladder wall edema, this could be due to pancreatitis. if cholecystitis is of clinical concern ultrasound could be performed. 3. moderate amount of intraabdominal ascites. 4. right pleural effusion. 5. cardiomegaly. brief hospital course: 77 yo f with h/o afib, htn, asthma who presents with rapid afib, pancreatitis, and acute renal failure. when the patient arrived to the floor she was in rapid afib at a rate of 150-160's. she was mentating well at this time with no complaints of chest pain, shortness of breath, or dizziness. here blood pressure on arrival was in the 90-110. she was given another bolus of 1l ns given the concern for her pancreatitis. when her bp was above 105 she was given lopressor 5mg, her heart rate came down to the 110-120 range. however her bp dropped again in response to the rate control and she required levophed. the levophed was titrated up with little effect however her hr began to increase again on the levophed. she was then switched to phenylephrine. this had little effect on her bp. she was givena second bolus, thus at this point she had gotten 8 l of fluid. her cvp was measured in the high 20's a-line was placed by surgery and correlated well with her non-invasives. despite pressors and fluids her bp remained in the 80's and her heart rate in the 120's. at this time she also began having mental status changes and agitation. restraints were placed for patients safety. on admission to micu labs were redrawn which demonstrated stable creatininedecrease bicarb, hypernatremia. her lft's and amylase/lipase were still normal despite evidence of pancreatitis on ct and possibler cbd dilatation. abg at this time was 7.01/63/119. in the setting of her mental status changes her she had difficulties protecting her airway, it was decided to proceed with endotracheal intubation. just prior to intubation, the patient went into pea. she was given epinephrine and responded with bp increaing into the 180's and hr 150-160's. immediately after intubation abg was 6.91/54/210, free ca 0.97, lactate 8.4, ag 11, k 5.9. she was given calcium gluc, magnesium, bicarb. increased her minute ventilation to help with her acidosis. follow up abg was 7.01/40/227. her heart rate was still in the 130's but her pressures were stable so attempted dilt gtt. she was also given bolus fluids. this time both her heart rate and blood pressure decreased. once again she went into pea. she was given atropine and epinephrine. she responded to this again. after resusitation her hr was back into the 120's with stable pressures. repeat labs now showed k 6.7, na 153, ag 14, cr 2.1, abg 7.04/67/133. she was given dextrose and insulin. her pressure then continued to fall and repeat gas about 45 minutes later was 6.97/52/90. at this time i had a discussion with the family in regards to her grave state. after discussion with the daughter and (the is in the health care field) it was decided to make the patient comfortable and withdraw care. she quickly deteriorated and passed away at 0130. during the evaluation and work up of the patient ecg showed rapid afib. we were concerned about possible st changes in leads v1- elevations and v4- depressions. throughout this event her ck's were flat and trop peaked at 0.09. cardiology was consulted first to do possible echo however they felt based on story that pericardial effusion was unlikely and that the afib was her problem. they recommended digoxin load at a lower dose given renal failure. after the ecg changes were noted the ecg was faxed to the on call cardiologist dr. who felt that there was no significant ischemic changes compared to old ecg's. also contact ercp given pancretitis and cbd dilatation. given the normal lft's and normal amylase and lipase they did not feel that this was the acute issue. they were more concerned about ischemic bowel. thus we reconsulted surgery, who was to see patient to re-evaluate patients abdominal status, however patient died before she was seen by them again. when patient intially arrived to the floor our first concern was her rapid heart rate and hemodynamics. possible causes for the patients condition include myocardial ischemia(however had no significant trop leak, and no significant ecg changes per cardiology), pe(however she was with inr of >2 from recent labs and was 3.6 then 1.7 in the ed), ischemic bowel(no bowel wall thickening on ct, but did have ag acidosis and elevated lactate), sepsis(empirically given vanco and levofloxacin however no identifiable source was found on intial eval.) # code- initially full and has patient condition worsened had extensive discussion with patients daughter and . they understood the gravity of the situation and decided to make the patient comfort and dnr/dni. medications on admission: advair 1p albuterol 2p 2-4x/day coumadin per inr diovan 320mg qhs flovent 2p verapamil 240mg daily discharge medications: n/a discharge disposition: expired discharge diagnosis: pancreatitis shock rapid afib sepsis cardiac arrest discharge condition: expired procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube diagnoses: unspecified essential hypertension atrial fibrillation depressive disorder, not elsewhere classified acute respiratory failure hypotension, unspecified dehydration 

Answer: The patient is high likely exposed to