Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: all allergies / adverse drug reactions previously recorded have been deleted attending: chief complaint: ruq abdominal pain major surgical or invasive procedure: laparoscopic cholecystectomy history of present illness: 47m with long standing history of alcholism presented to hospital with 3-4 days history of continuous ruq pain. patient states this pain has been a long problem for several years and comes after eating or drinking too much. the pain typically is dull and resolves after several hours, sometimes days. he presented to the hospital last evening because the pain was constant, sharper and was associated with cramping. patient also reports some mild nausea but denies vomiting, change in stool, melena, hematocchezia, fevers, chills, weight loss or fatigue. past medical history: pmh: anxiety, depression, alcholism, chronic back pain psh: left inguinal hernia repair 20 years ago social history: on disability. 1 case of beer weekly, 1.5 ppd smoking, occ mj distant cocaine. tattoos obtained from family member. family history: fh: noncontributory physical exam: admission exam vs: 97.4 127/97 96 20 general: extremely tan male in nad heent: nc/at, perrla, eomi, sclerae anicteric, mmm, op clear. neck: supple heart: irregularly irregular, normal s1 s2 lungs: cta bilat, no r/rh/wh, good air movement, resp unlabored. abdomen: soft, no masses noted. ttp in ruq, sign is present extremities: wwp, no c/c/e, 2+ peripheral pulses. skin: no rashes or lesions. lymph: no cervical lad. neuro: awake, a&ox3, cns ii-xii grossly intact. pertinent results: imaging: ecg: () sinus tachycardia. ecg (): atrial fibrillation. ecg (): sinus rhythm. poor r wave progression. non-specific diffuse low amplitude t waves with t wave inversions in leads v1-v2. low qrs voltage in the limb leads. compared to the previous tracing of atrial fibrillation has been replaced by sinus rhythm. ruq ultrasound with dopplers: 1. cholelithiasis with gallbladder distention. if there is persistent clinical concern for acute cholecystitis, hida scan could be performed. 2. increased echogenicity of the liver compatible with diffuse fatty infiltration. please note that more advanced forms of liver disease such as cirrhosis or fibrosis cannot be excluded. 3. reversal of flow in the portal vein, splenomegaly and multiple splenic varices. 4. right-sided pleural effusion. hida scan 1. no evidence of acute cholecystitis. 2. delayed tracer clearance secondary to hepatic dysfunction. ct abdomen with contrast impression: 1. portal vein patency cannot be assessed without a technically adequate portal venous phase image. the patient can be rescanned using bolus tracking technique if clinical concern for portal vein thrombus persists. 2. portal adenopathy, splenic vein tortuosity, and anasarca consistent with liver cirrhosis. 3. distended gallbladder with pericholecystic fluid, most likely secondary to third spacing and liver dysfunction rather than cholecystitis. 4. age indeterminant compression fracture of t11. echo: the left atrium is mildly dilated. there is mild symmetric left ventricular hypertrophy with normal cavity size. overall left ventricular systolic function is moderately depressed due to severe hypokinesis of the distal two-thirds of the left ventricle (lvef= 30-35 %). there is no ventricular septal defect. the right ventricular cavity is moderately dilated with moderate global free wall hypokinesis. the diameters of aorta at the sinus, ascending and arch levels are normal. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. no aortic regurgitation is seen. the mitral valve leaflets are structurally normal. an eccentric, posteriorly directed jet of mild (1+) mitral regurgitation is seen. the estimated pulmonary artery systolic pressure is normal. there is no pericardial effusion. impression: normal lv cavity size with severe hypokinesis of the distal of the ventricle. dilated and hypokinetic right ventricle. mild, posteriorly directed, mitral regurgitation, likely due to leaflet tethering. cxr: as compared to the previous radiograph, the monitoring and support devices, including the swan-ganz catheter, are unchanged. moderate cardiomegaly with bilateral pleural effusions and signs of moderate to severe pulmonary edema. the changes are stable since the previous examination. no evidence of pneumothorax. stable appearance of the mediastinal and hilar contours. echo: the left atrium is mildly dilated. no spontaneous echo contrast or thrombus is seen in the body of the left atrium or left atrial appendage. the right atrium is moderately dilated. no spontaneous echo contrast is seen in the body of the right atrium or right atrial appendage. no atrial septal defect is seen by 2d or color doppler. the left ventricular cavity size is top normal/borderline dilated. overall left ventricular systolic function is severely depressed (lvef= 15-20 %). the right ventricular cavity is mildly dilated with severe global free wall hypokinesis. the ascending, transverse and descending thoracic aorta are normal in diameter and free of atherosclerotic plaque t. there are three aortic valve leaflets. the aortic valve leaflets (3) are mildly thickened. there is no aortic valve stenosis. no aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. mild to moderate (+) mitral regurgitation is seen. the tricuspid valve leaflets are mildly thickened. moderate tricuspid regurgitation is seen. there is no pericardial effusion. brief hospital course: the patient was admitted to the hospital to the medical service with abdominal pain and a distended and large gallbladder. he underwent a number of imaging studies that demonstrated a distended gallbladder with stones. he also underwent a hida scan that did not demonstrate any evidence of cystic duct obstruction. additionally he underwent additional medical workup which revealed hep c, cirrhosis with ascites, as well as poor cardiac function with an ef 30-35%. he was watched for several days, but over the course of the weekend of , he developed an increase in abdominal pain despite antibiotics. at that point we elected to take him to the operating room for laparoscopic cholecystectomy. postoperatively the patient was unable to extubate and was admitted to the icu. he was oliguric postoperatively and extubated uneventfully. he was conservatively resuscitated with ivf on pod 1 but continued to be oliguric. echo was performed which showed worsening biventricular heart failure with ef estimated between 15-35% and associted tr. a swan ganz catheter was floated for better hemodynamic monitoring. over the course of the next days he had had increasing right heart failure, poor oxygenation requiring reintubation, atrial fibrillation requiring chemical and electrical cardioversion as well as volume overload requiring cvvh. he remained hypotensive throughout his sicu course and he had increasing pressor support. there was suspicion for pulmonary embolus but the patient was never stable enough to go to the ct scanner. on he was on maximal pressor support. a family meeting was held and the decision was made to make the patient comfort measures only. he expired shortly thereafter at 18:10 on . medications on admission: none discharge medications: none discharge disposition: expired discharge diagnosis: primary diagnosis: chronic colelithiasis cirrhosis congestive heart failure cp arrest secondary diagnosis: alcohol abuse discharge condition: none discharge instructions: none followup instructions: none procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified insertion of endotracheal tube hemodialysis closed (percutaneous) [needle] biopsy of liver venous catheterization for renal dialysis arterial catheterization laparoscopic cholecystectomy pulmonary artery wedge monitoring diagnoses: acidosis thrombocytopenia, unspecified tobacco use disorder congestive heart failure, unspecified alcoholic cirrhosis of liver acute kidney failure, unspecified unspecified septicemia hyposmolality and/or hyponatremia severe sepsis portal hypertension unspecified viral hepatitis c without hepatic coma atrial fibrillation dysthymic disorder acute respiratory failure cardiogenic shock septic shock other ascites acute systolic heart failure calculus of gallbladder with acute cholecystitis, without mention of obstruction other and unspecified alcohol dependence, continuous mixed acid-base balance disorder cannabis abuse, unspecified hereditary hemochromatosis 

Answer: The patient is high likely exposed to