Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: transfer for intracranial hemorrhage major surgical or invasive procedure: intubation ventricular drain placement right ij placement mri angiography history of present illness: 55 yo woman with a history of hepatitis c and cirrhosis, bactermia, osteomyelitis and epidural abscess who presented from osh after identifying an intracranial hemorrhage. per the family she uses methamphetamines. she was shopping on late at night, at felt "disoriented" and weak on the right. she made it home and beeped her . her mother and husband, who were at home, helped her get inside. she was noted to be agitated, talking, but not making sense and had right arm and leg weakness. she went to sleep. her mother called 911 on am of admission when she was brought to an osh. head ct there showed a large left parieto- occipital hemorrhage. she was given 1g dilantin for seizure prophylaxis and transfered here for further management. at she was admitted to the neuro icu service for q 1hour neuro checks. she initially was awake and oriented, became obtunded, thus intubated, started on mannitol, hyperventilated, and vent drain placed . she was slow to improve but eventually sedation was weaned and she was extubated on . ventricular drain was also removed on . regarding the etiology of the bleed, angiography was performed on and showed only unrelated frontal lobe venous anomaly but no clear cause of her bleed. it is likely the methamphetamine use (as per history obtained later by family) is the culprit of the bleed. she needs an mri in a few weeks though to r/o masses. ct torso was obtained to r/o masses and was unrevealing (see labs and studies). bp was controlled with po metoprolol. serial cts showed improving blood resorption. glucose control via insulin drip while in the icu. she was covered with cefazolin while drain was in place and for 3 doses after drain was removed. she developed a uti with fever and was treated with 6 days of levofloxacin. she was given nutrition via tube feeds. keppra was started upon arrival for seizure prophylaxis, but several days into her admission she was noted to have increased gaze preference to the left, thus dilantin was started and keppra was weaned to off. 2 eegs did not show epileptiform activities. the patient currently complains of some mild trouble breathing but is doing well otherwise. she is confused and unable to relate a coherant story. past medical history: 1. hepatitis c chronic, biopsied in with b stage four cirrhosis with inflammation. s/p pegylated interferon and ribavirin. ribavirin and interferon was stopped secondary to anemia. 2. esophagogastroduodenoscopy on evaluated no varices. 3. anemia. 4. micu admission for bacteremia with mental status changes, requiring intubation. 5. osteomyelitis and epidural abscess s/p laminectomy and drainage of l4/5 , also with osteo of 5th metatarsal s/p surgery also in at time of laminectomy. 6. tachycardia and frequent pvcs with normal ef, cta neg for pe in 7. cholelithiasis 8. multilobar pneumonia after intubation social history: nurse at a nursing home, acquired hcv via needle stick at work, + h/o heavy etoh use, + h/o amphetamine use per family family history: per previous d/c summ, patient is adopted physical exam: t 98.8 bp 137/87 hr88 rr18 o2 sat 92% gen: on ed stretcher, c/o ha neck: supple, +pain on passive fexion, back: +lumbar lami scar, no cv: rrr, nl s1 and s2, 2/6 sem lung: clear to auscultation bilaterally abd: +bs soft, nontender, +hepatomegaly (non-tender) ext: no edema neurologic examination: mental status: awake and alert, cooperative with exam, oriented to person, place, and date. she is inattentive, cannot say backwards. speech is fluent with impaired comprehension and intact repetition; naming impaired - makes many semantic paraphasic errors. no dysarthria. intact. unable to do calculations. + left-right confusion. no evidence of apraxia or neglect. cranial nerves: i: not tested ii: pupils equally round and reactive to light, 3 to 2 mm bilaterally. appears to have a mild right field cut (worse inferiorly than superiorly), but she does not cooperate well with exam. iii, iv, vi: extraocular movements intact bilaterally without nystagmus. v: sensation "different" on left. vii: mild righ nlf flattening. viii: hearing intact to finger rub bilaterally. ix, x: palate elevation symmetrical : sternocleidomastoid and trapezius normal bilaterally. xii: tongue midline without fasciculations, intact movements motor: normal bulk bilaterally. tone increased on right ue and le. no observed myoclonus or tremor tri wf we fe ff ip h q df pf te tf r 0 0 0 0 0 0 0 0 0 0 0 0 1 1 l 5 5 5 5 5 5 5 5 5 5 5 5 5 5 sensation: decreased to light touch, pinprick on right. difficult to evaluate vibration and proprioception due to inattension. reflexes: b t br pa ach right 2 2 2 0 1 left 2 2 2 0 1 toes were upgoing on right, downgoing on left coordination: normal on finger-nose-finger with left hand. pertinent results: wbc 9.8 (peaked at 16 on ) hct 37 (nadired at 34 on mcv 94 plt 209 inr 1.1 ptt 36.9 (previous ptts were normal) ua : sm le, mod bld, tr prot, wbc, 0 epis, rbcs csf ( after drain) 2 wbc, 305 rbcs, prot 27, gluc 81 csf ( prior to drain removed) 30 wbcs, 1140 rbcs na 142, k 4.0, cl 109, bicarb 25, bun 19, cr 0.6, gluc 126 ck 239, 266 () with mb 3, trop <0.01 x 2 cal 9.1, phos 3.1, mag 2.1 hba1c 5.4 tg 56 hdl 57 ldl 97 chol 163 dilantin 10.2 on stox neg abg : 7.43/38/82 micro: blood cx: ng, ng, pending urine cx: gnr, contam, contam csf cx: ng, ng sputum cx: & sparse op flora cath tip cx: ng nchct's: at 7:30pm: 1. moderate-to-large intraparenchymal hemorrhage within left parietal lobe. 2. surrounding edema and sulcal effacement. at 11:30pm - no significant change : there is no gross change in the left parietal intracerebral hemorrhage. 3. there has been mild interval decrease in size of the right frontal subdural hematoma compared to . : 1. no change in the size of the intraparenchymal left parietal hemorrhage. 2. slightly more mass effect in the left cerebral hemisphere with slightly more shift of the midline structures and more effacement of the suprasellar cistern. : the extent of the hyperdense component of the large left parietal lobe hemorrhage has reduced since the prior study of , consistent with partial resorption of the high density components. both studies show what is likely a small right frontal subdural hematoma, hyperdense relative to spinal fluid and therefore likely relatively acute in age. the most superior portion of the hemorrhage is the thickest and causes slight impression on the adjacent frontal lobe gyri. the left parietal lobe hemorrhage has an extensive zone of surrounding edema within the white matter, essentially unaltered in extent but causing considerable compression of the adjacent left lateral ventricle. there is a few mm rightward shift of the septum pellucidum and likely the 3rd ventricle as well. no other interval changes are appreciated. angiogram on : 1. no evidence of definite dilated arterial structure, nidus, or dilated veins to indicated arterial venous malformation. however, it should be noted that ct angiography is not sensitive for subtle arterial venous malformation,or changes from vasculitis, which can be detected with conventional angiography. 2. no aneurysm is identified. 3. a small somewhat dilated venous structure in the left frontal lobe could be secondary to a developmental venous anomaly. 4. it should be noted that the volume-rendered images are not available for interpretation. if new findings are seen on those images when they are available, an addendum will be given to this report. 5. mri would be helpful for further evaluation. cxr : no cp dz : report pending, possibly some effusion on the left ? mri brain with gad on : 1. no change in the large left parietal hemorrhage and its mass effect. 2. there is increased vascularity noted in the left temporal region. a conventional angiogram is recommended for further evaluation to exclude an underlying vascular malformation. 3. no change in the size of the right frontal subdural hematoma. ct torso on : 1) diffuse patchy nodular or pulmonary opacities greater posteriorly. this appearance may be due to infection, aspiration, or inflammatory changes. 2) right ij cvl tip in right atrium. 3) nodular appearance of liver consistent with patient's history of cirrhosis. two low attenuation right hepatic foci are unchanged in appearance and may represent cysts. 4) cholelithiasis without evidence of cholecystitis. 5) bilateral nonobstructing renal stones. 6) likely pancreatic cysts. eeg on : this is an abnormal portable eeg due to the presence of a slow and disorganized background rhythm in the hz delta and theta frequency ranges. in addition there is moderate to high amplitude generalized delta frequency slowing, often with a triphasic morphology. these findings suggest deep midline subcoritcal dysfunction and are consistent with an encephalopathy. no clear lateralizing or epileptiform abnormalities were seen. sinus tachycardia was noted on the cardiac monitor. : impression: markedly abnormal portable eeg due to the prominant focal delta slowing seen over much of the left hemisphere with some sharp components but no overtly epileptiform abnormalities, and due to the bursts of generalized slowing and surpression and slowing of the background. the first abnormality suggests a focal structural lesion in the left hemisphere. this had sharp features but no overtly epileptiform abnormalities. this does not rule out the possibility of seizures at other times. the other abnormalities indicate a widespread encephalopathy. medications, metabolic disturbances and infection are among many possible causes. brief hospital course: 55 yo woman with large left parietal-occipital lobe hemorrhage likely secondary to drug use although other causes must be ruled out. ** please hpi for complete icu course including workup thus far ** on exam upon discharge, is awake, alert, oriented to self only, thinks she's in , +repeats, + follows commands, + fluent, realizes she has had a stroke and her right side does not work well. no neglect, but does have right visual field cut, right hemiplegia. neuro - lobar bleed, likely secondary to amphetamines, angiography negative, ct torso and mri showed no metastases. mannitol d/c'd. on dilantin for sz prophylaxis although plan is to wean this over the next 5 days, 2 eegs without frank epileptiform activity. - bp control with po lopressor - multipodus boot for rle - vent drain d/c'd and extubated on confusion - likely secondary to pneumonia, bleed, possibly from methamphetamine withdrawl causing prolonged intubation. - levo x 10 days for aspiration pna (to finish ). also has atelectasis and needs inspiration spirometer use qd. id: - finished course of cefazolin for vent drain bp control: - metoprolol 25 tid hyperglycemia (likely from tube feeds which have now been stopped): riss fen: - ppi, thiamine/folate/mvi - passed video swallow, ground food + thin liquids ppx: ppi, sc heparin, is, oob dispo: seen by pt/ot, needs rehab. medications on admission: none discharge medications: 1. acetaminophen 325 mg tablet sig: 1-2 tablets po q4-6h (every 4 to 6 hours) as needed. disp:*30 tablet(s)* refills:*0* 2. heparin sodium (porcine) 5,000 unit/ml solution sig: one (1) injection, 5000 units injection tid (3 times a day). disp:*90 injection, 5000 units* refills:*2* 3. clonidine hcl 0.1 mg/24 hr patch weekly sig: one (1) patch weekly transdermal qthur (every thursday). disp:*4 patch weekly(s)* refills:*2* 4. bisacodyl 5 mg tablet, delayed release (e.c.) sig: two (2) tablet, delayed release (e.c.) po daily (daily) as needed. disp:*60 tablet, delayed release (e.c.)(s)* refills:*0* 5. senna 8.6 mg tablet sig: one (1) tablet po bid (2 times a day) as needed. disp:*60 tablet(s)* refills:*0* 6. nystatin 100,000 unit/ml suspension sig: five (5) ml po tid (3 times a day). disp:*450 ml(s)* refills:*2* 7. quetiapine fumarate 25 mg tablet sig: 0.5 tablet po bid (2 times a day). disp:*30 tablet(s)* refills:*2* 8. levofloxacin 500 mg tablet sig: one (1) tablet po q24h (every 24 hours) for 4 days. disp:*4 tablet(s)* refills:*0* 9. multivitamin capsule sig: one (1) cap po daily (daily). disp:*30 cap(s)* refills:*2* 10. folic acid 1 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 11. thiamine hcl 100 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 12. phenytoin sodium extended 30 mg capsule sig: per taper schedule, below capsule po once a day for 5 days: : 3 capsules : 2 capsules : 2 capsules : 1 capsule : 1 capsule : none. disp:*9 capsule(s)* refills:*0* 13. lansoprazole 30 mg capsule, delayed release(e.c.) sig: one (1) capsule, delayed release(e.c.) po daily (daily). disp:*30 capsule, delayed release(e.c.)(s)* refills:*2* 14. docusate sodium 100 mg capsule sig: one (1) capsule po bid (2 times a day) as needed. disp:*30 capsule(s)* refills:*0* 15. metoprolol tartrate 50 mg tablet sig: one (1) tablet po tid (3 times a day). disp:*90 tablet(s)* refills:*2* 16. insulin regular human 100 unit/ml solution sig: per sliding scale units, regular insulin injection qachs: for bg 151-200: 2 units 201-250: 4 units 251-300: 6 units 301-350: 8 units 351-400: 10 units >= 401: 12 units and md. . disp:*qs units, regular insulin* refills:*2* discharge disposition: extended care facility: - discharge diagnosis: left parieto-occipital hemmorhagic stroke discharge condition: stable discharge instructions: pt/ot take all meds. attend all followup appointments. return to the ed if you experience worsening weakness or unresponsiveness. followup instructions: with acute rehabilitation facility, and with dr. in the clinic at the hospital: , 3 pm, , building. md, procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more enteral infusion of concentrated nutritional substances arteriography of cerebral arteries intravascular imaging of intrathoracic vessels arterial catheterization diagnoses: urinary tract infection, site not specified cirrhosis of liver without mention of alcohol chronic hepatitis c without mention of hepatic coma pneumonitis due to inhalation of food or vomitus unspecified intracranial hemorrhage amphetamine or related acting sympathomimetic abuse, continuous 

Answer: The patient is high likely exposed to