Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: ms. is a 63 year-old woman with a history of most significant for type 1 diabetes, coronary artery disease status post coronary artery bypass graft presented with three days of lethargy to the emergency department. history at the time of arrival was incomplete and taken mostly from conversations with the emergency room staff as the family was not immediately available. she was found in the emergency department to have blood sugar over 800 and a systolic blood pressure in the 70s to 80s. she was started on intravenous fluids and an insulin drip. physical examination was initially notable for a stiff neck and she was given 2 grams of ceftriaxone. she had a persistently low blood pressure. after 5 liters of normal saline she was started on dopamine and an arterial blood gas revealed a ph of 6.99 and the patient soon after began to tire from a respiratory standpoint and was intubated for presumed ventilatory failure and for profound acidemia. at the time of the intubation the patient vomited 200 cc of coffee grounds and additional coffee grounds were drained from the nasogastric tube and also from the et tube. negative lavage was negative for bright red blood and rectal was ob positive. she was transferred to the medical intensive care unit for further management. past medical history: 1. coronary artery disease status post coronary artery bypass graft in , three vessel. 2. tuberculosis status post upper lobectomy in the . 3. type 1 diabetes times 45 years complicated by neuropathy. 4. peripheral vascular disease. 5. high cholesterol. 6. tobacco use. medications on admission: atenolol question dose, aspirin 325 q.d., lipitor 40 q.d., neurontin question dose, fosamax question dose, wellbutrin 50 mg b.i.d., effexor 150 mg b.i.d., reglan 10 mg q.i.d., trental 400 mg t.i.d., zantac 150 mg b.i.d., prilosec 20 mg q.d., folate. allergies: no known drug allergies. social history: active smoker two packs per day. daughter is her health care proxy . family history: pertinent for premature coronary artery disease. physical examination on admission: general, intubated and sedated. heent pupils are equal, round, and reactive to light and accommodation. pupils 3 mm to 2 mm with light bilaterally. anicteric sclera. dry mucous membranes. neck somewhat stiff. no increased jvd noted. lungs rhoncerous breath sounds in upper and lower fields anteriorly. cardiac examination regular rate and rhythm. normal s1 and s2. no murmurs, rubs or gallops. abdomen slightly distended with decreased bowel sounds, nontender. extremities no clubbing, cyanosis or edema. skin no rashes. laboratories on admission: white blood cell count 24.1, hematocrit 37.8, platelets 473. differential 94 neutrophils, 4 lymphocytes, 2 monocytes, sodium 130, potassium 6.0, chloride 88, bicarbonate 8, bun 31, creatinine 1.7, blood sugar 863, alt 18, ast 11, t bili .2, albumin 2.1, amylase 18, lipase 23, magnesium 1.4, phos 4.3. urinalysis greater then 1000 glucose, ketone negative. serum acetone positive. arterial blood gas 6.99 ph, 16 pco2, 108 pao2, lactate 2.8, ionized calcium 1.16. electrocardiogram sinus tachycardia at 100. st depressions with t wave inversions in 1, avl, 2, 3, avf, v4 through v6, which were all new. chest x-ray status post endotracheal intubation with elevated right hemidiaphragm. hospital course: the patient was evaluated by gi who felt that the upper gi bleed was likely secondary due to stress gastritis from the diabetic ketoacidosis. the patient was also noted to have an elevated cpk in the 500 range by hospital day two with a troponin of 50. she was felt to have a non q wave myocardial infarction secondary to physiologic stress from the upper gi bleed and diabetic ketoacidosis. over the first few days of her micu stay her anion gap, which was initially 27 closed to 15. her blood sugars normalized on a minimal insulin drip. she had an echocardiogram, which revealed an ejection fraction of greater then 30% with no new motion abnormalities. however, her lung examination and chest x-rays continued to worsen through the early part of her micu course with worsening bilateral opacifications. she remained intubated secondary to a large aa gradient and also secondary to profound acidosis, which persisted in spite of normalization of her diabetic ketoacidosis and absence of serum acetone. she was switched to pressure control ventilation with continued difficulty of adequate ventilation. her ph remained in the 7.2 range. bicarbonate remained in the 10 to 15 range and again she continued to have an anion gap acidosis. the etiology was not entirely clear. a chest ct was performed, which revealed diffuse ground glass opacifications throughout the entire thorax. it was felt that the patient's clinical picture was consistent with ards. by micu day number five the patient's respiratory status was worsening. a pa catheter was placed as her urine output continued to drop revealing a picture most consistent with sepsis with a diminished cardiac output. levophed was started. long discussion with the family and the health care proxy is continued throughout the entire course of her hospitalization. it was felt that the aggressive level of her care was not consistent with her wishes prior to being critically ill. it was also felt that the patient would not want to be sustained on life support measures. given the fact that the patient had an extremely poor prognosis the health care proxy decided that the patient was to remain comfort measures only. levophed was removed and the patient's vent was then switched to room air. within thirty minutes the patient expired. she was pronounced dead on at 2:30 p.m. an autopsy was declined by the family. dr. the patient's primary care physician was unavailable at the time of the death. a message was left at her office. , m.d. dictated by: medquist36 procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more spinal tap incision of lung parenteral infusion of concentrated nutritional substances insertion of endotracheal tube pulmonary artery wedge monitoring diagnoses: acidosis subendocardial infarction, initial episode of care unspecified septicemia polyneuropathy in diabetes acute respiratory failure pneumonitis due to inhalation of food or vomitus diabetes with ketoacidosis, type i [juvenile type], not stated as uncontrolled diabetes with neurological manifestations, type i [juvenile type], not stated as uncontrolled other specified gastritis, with hemorrhage 

Answer: The patient is high likely exposed to