Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: the patient is a 52 year-old female with a history of hydrocephalus status post right posterior occipital shunt placement in a prior seizure disorder who presented to an outside hospital for seizures. according to her prior records the patient had the onset of a focal seizure possibly involving the right arm for which she presented to an outside hospital. she was treated with up to 20 milligrams of prn ativan as well as additional doses of her prior anti-epileptic drugs without improvement. she was subsequently intubated and started on an ativan drip and was transferred to the for further management. at the outside hospital her particular treatment had including fentanyl 50 micrograms, ativan 4 milligrams, phenobarbitol 300 milligrams bolus, ativan drip, valium 5 milligrams, ativan 2 to 4 milligrams up to eight doses, neurontin 400 milligrams, trileptal 300 milligrams. the patient had also been given versed prior to her transfer. review of systems: unable to assess. past medical history: 1. seizure disorder described previously as a focal seizure with occasional generalization. 2. hypercholesterolemia. 3. normal pressure hydrocephalus. 4. siadh. 5. hypertension. allergies: include 1. penicillin. 2. codeine. 3. aspirin. 4. dilantin. medications on admission: 1. the patient had recently been started on trileptal. 2. neurontin. social history: the patient is college educated. she has occasional alcohol use, no tobacco use. physical examination: on initial physical exam her heart rate was 100, blood pressure 139/87, respiratory rate 15, fio2 60, assist control at 12 and tidal volume of 600. in general the patient appeared older than her stated age and was lying in bed intubated with minimal spontaneous movement. heent was normocephalic, atraumatic with white sclerae. her neck was supple without jvd or bruits. her lungs were clear to auscultation with vented breath sounds bilaterally. cardiovascular exam revealed a regular rate and rhythm with normal s1, s2 and no murmurs, rubs, or gallops. her abdomen had normal bowel sounds. it was soft, nontender, nondistended. extremities were warm without cyanosis, clubbing or edema. neurologic examination: the patient was not responsive to voice and had no spontaneous ability to open her eyes. cranial nerves - her face appeared symmetric and her eyes were conjugate with forward gaze. pupils were fixed at 1 mm and nonreactive to light. ocr was not present and corneal reflexes as well as blinking to threat were also absent. gag reflex was present. funduscopic demonstrated small ectatic retinal vasculature but sharp disc borders bilaterally. on motor and sensory exam the patient withdrew from painful stimuli in the upper extremity with triple flexion response in the lower extremities. she grimaced to pain in all extremities. reflexes were equal and symmetric bilaterally with upgoing plantar responses. there was no ankle clonus. laboratory data: initial labs demonstrated a head ct scan obtained at the outside hospital that showed bilateral hydrocephalus with the presence of a right posterior occipital shunt. brain mri obtained in had a previous of a right frontal craniotomy, bilateral craniotomies. there was right frontal encephalomalacia. initial ekg showed sinus tachycardia with a normal axis, a q wave in the inferior leads and early r wave progression. her initial labs of note include a wbc 15.5, with 87% neutrophils, and a hematocrit of 36.6 and a normal mcv. her urine tox screen was negative. her coags as well as her lfts were normal. her initial sodium was slightly decreased at 134 and her chloride was slightly elevated at 134. her bicarb was decreased at 19. the patient was admitted to the neuro icu and continued initially on the ativan drip while she was loaded with depakote. summary of hospital course: a neurosurgery consult was obtained to assess the possibility of a shunt malfunction. on review of her prior images it was felt that there was no significant change in the size of her hydrocephalus according to the neurosurgical consult. it was subsequently determined that dr. in the department of neurosurgery had been her surgeon in the past and the consult was deferred to dr. who saw the patient later in her hospital course and also felt that it was unlikely that her subsequent ataxia was related to shunt malfunction. in the intensive care unit the patient continued to have decreased mental status presumably from ativan and postictal stated that required continued intubation until approximately . the patient was then transferred to the general neuro medical service. during her icu stay she had an eeg that demonstrated occasional left temporal and generalized burst of beta slowing. she also had a wide spread faster beta rhythm background particularly in the frontal regions. this is felt to be most likely a medication affect probably due to her ativan. no clear epileptiform features were documented. on transfer to the general neurology service the patient was noted to be quite ataxic both trunkly and in her extremities. the etiology of this was unclear but the patient reported that she had prior episodes of gait instability related to neurontin which she was being continued on. it was decided to taper her neurontin and continue depakote for the time being. after discussing her anti-epileptic regimen with her outside neurologist, dr. , at the decision was made to start the patient on trileptal and maintain her on depakote until her trileptal had reached a therapeutic range. otherwise the patient has continued to do well. discharge diagnosis: 1. focal status epilepticus. 2. gait ataxia of unclear etiology possibly from medication affect. 3. history of hydrocephalus status post shunt placement. discharge diet: low sodium diet, low cholesterol diet. discharge activities: as defined by physical therapy. disposition: the patient is to be discharged to a rehabilitation facility. discharge medications: 1. neurontin 100 milligrams po tid. this could be tapered to one day after transfer and then q day and then off. 2. trileptal 300 milligrams po bid which should be increased after seven days of treatment by 600 milligrams to trileptal 600 milligrams po bid. trileptal was started on . while the patient is on trileptal her sodium levels need to be followed closely given her history of siadh. she has a prior adverse affect from tegretol which has included hyponatremia. 3. robitussin dm 30 cc po q six hours prn. 4. depakote 250 milligrams po q a.m., 250 milligrams po q noon, 500 milligrams po q p.m. the patient's depakote level should be followed as well as her cbc and lfts. 5. heparin 5000 units subcutaneous . follow up instructions: on arrival to the rehab facility the patient's neurologist described above should be contact to inform him of her status. the patient should follow up with her neurologist on an outpatient basis. , m.d. dictated by: medquist36 procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified diagnoses: obstructive hydrocephalus unspecified essential hypertension unspecified essential hypertension hyposmolality and/or hyponatremia acute respiratory failure grand mal status personal history of noncompliance with medical treatment, presenting hazards to health presence of cerebrospinal fluid drainage device lack of coordination 

Answer: The patient is high likely exposed to