Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: this is a 36-year-old male with a nonsignificant history was admitted 3 days ago from an outside hospital. according to his wife, the patient was in normal state of excellent health until a week-and-a-half ago, before his admission, when he noted the onset of left flank pain a few days after his return from a golf trip to . the patient notes that the day before his admission, he had developed gross blood in his urine. however, despite this symptom, his wife reports that he went to work the morning of his admission, returned around noon, seemingly to be in normal health. he was found by a neighbor around 8 p.m., on the day of admission incoherent and crawled up in a fetal position in his front lawn, he was very agitated, but highly confused leading the neighbor to contact the ems and police. police noted that he was agitated, combative, and confused resulting in his transport to hospital. while at this outside hospital, he was initially alert and oriented times 3, but due to his combative behavior, he was given 60 mg of iv ativan. by report, he was found to have a fever of 103 degrees, a negative head ct and ekg showing elevation from leads v1 through v4, and troponin level of 13. the urine toxic screen positive for benzodiazepines and cocaine. he was given iv rocephin, aspirin, nitroglycerin, and an amp of d50, was intubated. he was transported by med flight to for emergent cardiac catheterization. at presentation to er, he was found to have a blood pressure of 205/101, a heart rate of 123, saturations at 99 percent on fio2 of 0.6. his labs were significant for a white blood cell count of 13.6, platelets of 114, creatinine 2.7, serum glucose of 31, and abg of 7.21 per ph, pco2 41, po2 134. he received bedside echocardiogram, which revealed normal lv function and no valvular disease with a question of apical hypokinesis. he was sent emergently to the cardiac catheterization lab, which did not reveal any evidence of coronary artery disease. his wedge was 22 mmhg, the cardiac output of 8 liters a minute and cardiac index of 4.2; however, his ck level increased from 450 at the outside hospital to 3835 on admission to leading to suspicion of rhabdomyolysis. following catheterization, the patient was admitted to the micu. past medical history: genital herpes. muscle spasms on muscle relaxants at home. outside medications: 1. muscle relaxant that the patient cannot remember the name of. 2. xanax p.r.n. hospital medications: 1. protonix 40 mg p.o. q.d. 2. flagyl 500 mg p.o. b.i.d. 3. levofloxacin 250 mg p.o. q. 48h. 4. sevelamer 800 mg p.o. t.i.d. allergies: no known drug allergies. social history: the patient is married. he lives with his wife and a 1-year-old child. child was left locked inside when mr. was found outside forcing police to break down the door. vss is involved in this case. the patient works as an occupational therapist. his wife denies the patient had any previous tobacco history or history of alcohol use or recreational drug use. however, the patient admits to having used cocaine for a total of 6 times as well as a red pill and pill, which he is unable to mention the names of. family history: noncontributory. physical examination: temperature maximum 99.6 degrees, blood pressure 99-145 systolic over 60-74 diastolic. heart rate 75 to 97, respiratory rate 13 to 28. oxygen saturation 91 to 95 percent on room air. the patient was overall 15 liters positive upon transfer to the medicine service. generally: well-appearing, no apparent distress. heent: normocephalic, atraumatic. head, no pharyngeal erythema or exudate. sclera anicteric. neck: no jvd or lymphadenopathy. cardiac: normal. pulmonary: normal. abdomen: normal. skin: no clubbing, cyanosis or edema. neurologically: alert and oriented, mildly delayed recall process. cranial nerves intact. visual fields full to confrontation. no pronator drift. negative romberg's. rapid alternating movements intact. touch and proprioception intact. motor strength 5/5 in all extremities. laboratory data: from admission, white blood cell count 17.1, hematocrit 32.1, platelets 68. ptt 41.8, inr 3.7, fibrinogen 106, haptoglobin less than 20. chemistry profile notable for a bun of 15, creatinine is 7.9, glucose 104, calcium 7.4, phosphorous 7.2, magnesium 1.8. lfts notable for an alt of 2508, ast of 1871, ldh 1802, ck 6751, alkaline phosphatase of 81, t-bili 1.2. micu course was complicated by persistent hypoglycemia requiring 4 amp's of d50. he developed hypotension requiring a levophed infusion. his hematologic parameters continued to degrade with his hematocrit dropping to 33.6 from 44, and his platelets dropping to a low of 36,000 requiring a platelet transfusion. his thrombocytopenia was complicated by the development of coagulopathy with his inr increasing to a high of 3.5. his fibrinogen dropping to 63 and d-dimer level greater than 10,000, that was considered that this could be ttp/hus, given the initial fever, mental status change, acute renal failure, and thrombocytopenia. however, the absence of a microangiopathic process on blood smear argued for the diagnosis of dic instead. chest radiograph showed evidence of patchy opacities in the right upper lobe and left lower lobe consistent with aspiration pneumonia. infectious disease was consulted while in the icu. he was placed on flagyl, vancomycin, and levofloxacin for presumed aspiration pneumonia and acyclovir for hsv meningitis given his acute mental status changes. additionally, renal consult and gi consults were obtained while the patient was in the icu and as he had put out guaiac positive diarrhea and had rapidly progressed to acute renal failure. hospital course while on floor: cardiac. the patient ruled in for myocardial infarction by cardiac enzymes and by his ekg changes consistent with that. however, his catheterization was unrevealing in terms of evidence for cardiac ischemia. the likely explanation was that this was mostly likely a cocaine induced vasospasm causing myocyte ischemia and death. his lv function was preserved according to the echocardiogram. rhabdomyolysis. given the patient's enormous increase in his ck, the patient had evidence of heme-positive urine. again, this was mostly likely attributed to cocaine induced rhabdomyolysis. other possible etiologies could have been virally induced or possibly related to the patient's status of being found down. the patient's ck slowly began to trend down with aggressive iv fluid hydration. acute renal failure. the patient had nonoliguric acute renal failure. there was evidence of bloody-brown casts seen in his urine, which is characteristic of atn. the patient maintained adequate urinary output without requiring dialysis. liver dysfunction. the patient had evidence of hepatic involvement to his multisystem organ failure. this is mostly likely attributed to shock liver given his known development of dic and profound hypertension during his first hospital day. coagulopathy. it is most likely attributed to dic. hematology was consulted to assist in the management. there is no evidence of schistocytes on peripheral smears. mental status changes. although, the patient's mental status changes seemed highly likely to be solely to his cocaine use. it was also attributed to delirium and the onset of fever and possible sepsis. this improved after antibiotic treatment and iv fluid hydration. uremia may have also contributed to his mental status decline. hypoglycemia. the patient's initial metabolic derangements were noted in the icu, the thought was that the patient may have had an adulterated form of cocaine with quinine, which is apparently common and can cause protracted hypoglycemia. infectious disease. the patient was febrile without any obvious source of infection, felt that this may be attributed to the patient's atelectasis versus cytokine response to muscle or liver necrosis. the patient although was maintained on antibiotics for aspiration pneumonia. discharge diagnoses: acute myocardial infarction with cardiac catheterization showing no occluded coronary arteries. acute nonoliguric renal failure. disseminated intravascular coagulopathy. rhabdomyolysis. hepatitis consistent with shock liver contributing diagnosis include cocaine abuse. condition on discharge: the patient is stable without oxygen requirement, tolerating pos, mentating clearly. discharge status: the patient would be discharged to home. major surgical or invasive procedures performed: the patient had cardiac catheterization. he was intubated and he had a central line placement. follow up: the patient will follow-up with his pcp 1 week. the patient will also follow-up with gastroenterology for an elective colonoscopy given his history of bloody diarrhea as an inpatient. additionally, the patient will follow-up with intensive outpatient treatment program for substance abuse. the patient will have a follow-up renal ultrasound and follow-up with dr. nephrology to monitor his renal function. discharge medications: 1. amlodipine 5 mg p.o. q.d. 2. protonix 40 mg p.o. q.d. 3. ciprofloxacin 500 mg p.o. q.d. for 2 weeks. , md dictated by: medquist36 d: 14:52:19 t: 10:17:06 job#: procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified coronary arteriography using two catheters right heart cardiac catheterization diagnoses: subendocardial infarction, initial episode of care acute kidney failure with lesion of tubular necrosis cocaine abuse, unspecified acute respiratory failure defibrination syndrome pneumonitis due to inhalation of food or vomitus accidental poisoning by central nervous system stimulants rhabdomyolysis 

Answer: The patient is high likely exposed to