Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no allergies/adrs on file attending: chief complaint: post-cardiac arrest major surgical or invasive procedure: none history of present illness: ms. is a 73yof with h/o bipolar d/o on lithium who is transferred from osh s/p cardiac arrest. patient is currently unable to provide history, therefore details of hpi are obtained from family and osh records. per pt's family, pt had c/o flu-like sx a week ago, with uri sx and cough. had also had progressive doe for past few weeks, to the point that she became dyspneic when walking from her apartment to parking lot (~100 feet). no h/o cardiac disease, however recently had stress test which was reportedly negative and was scheduled to see cardiologist this week. today the elevator in her apartment building was broken, and when walking up 2 flights of stairs to her apartment she became extremely sob and called for help. ems was called, started cpap but she did not tolerate it, became agitated, then became bradycardic and suffered asystolic arrest. cpr was started and she receieved epi 1mg x3 and lidocaine 100 in field then was taken to osh. at osh ed received atropine x2 and additional epi x4. total time to rosc estimated 25 minutes. she received chilled saline at osh and was transferred to , with t 33.9 en route. in the ed, initial vs were t 33f, hr 85, bp 90/40 on dopa 20mcg and epi 12.5mcg. gcs 3, not responding to commands but withdrawing to painful stimuli. labs were notable for ph 6.94, pco2 64, hco3 15, lactate 9.9. ekg showed sinus rhythm with lad and rbbb, low voltage. (of note initial ecg at osh had anterior ste, but resolved.) bedside tte showed no rv enlargement, global hypokinesis with depressed lvef, c/w post-arrest. no significant valvular disease. fast exam negative. cta showed no pe but large bibasilar consolidations c/w aspiration pna. post-arrest consult team was consulted for initiation of arctic sun cooling protocol. epi gtt was increased to max, and she was started on neo gtt. she received iv vancomycin 1g x1, iv cefepime 2g x1, and iv levofloxacin 500mg x1. she was admitted to micu for arctic sun cooling protocol. . on arrival to the micu, she is intubated and sedated. vitals t 32.2f, hr 103, bp 97/70, o2 sat 94% on 100% fio2. past medical history: -bipolar disorder -severe asthma requiring hospitalizations years ago, no exacerbations for many years (not currently treated) social history: per family, pt lives in but is very independent with adls. occ smoking (1 pack per month), no etoh or drug use. family history: notable for cad in patient's parents and siblings, but no known h/o sudden cardiac death. physical exam: admission exam: vitals t 32.2f, hr 103, bp 97/70 (on dopamine/epi/neo gtt), o2 sat 94% on 100% fio2. heent: ncat, pupils fixed at 5mm bilaterally chest: quiet breath sounds anteriorly, symmetric cardiovascular: rrr, nl s1 s2, no m/r/g abdominal: soft ntnd, ruq surgical incision scar extremities: cool, no clubbing/cyanosis/edema neuro: not following commands, not responsive to painful stimuli pertinent results: admission labs: 07:00pm blood wbc-15.6* rbc-3.80* hgb-12.1 hct-41.2 mcv-108* mch-31.8 mchc-29.4* rdw-11.5 plt ct-276 01:42am blood neuts-86* bands-5 lymphs-8* monos-0 eos-0 baso-0 atyps-0 metas-1* myelos-0 07:00pm blood pt-15.1* ptt-60.5* inr(pt)-1.3* 01:42am blood glucose-364* urean-19 creat-1.1 na-139 k-4.4 cl-112* hco3-14* angap-17 01:42am blood alt-189* ast-348* alkphos-77 totbili-0.4 09:56pm blood alt-430* ast-515* ld(ldh)-1438* alkphos-28* totbili-0.4 cardiac enzymes: 08:45am blood ck(cpk)-1424* 04:32pm blood ck(cpk)-1221* 08:45am blood ck-mb-87* mb indx-6.1* ctropnt-1.48* 04:32pm blood ck-mb-77* mb indx-6.3* ctropnt-0.96* lithium level: 08:45am blood lithium-0.4* serum tox: 07:00pm blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg imaging: ct head w/o contrast findings: there is no evidence of acute hemorrhage, edema, mass effect, or recent infarction. the ventricles and sulci are age appropriate in appearance. areas of periventricular and subcortical white matter hypodensity likely reflect sequela of chronic small vessel ischemic disease. no concerning osseous lesion or fracture is identified. there are air-fluid levels within the nasopharynx and mild mucosal thickening of the ethmoid air cells, compatible with intubation. the mastoid air cells are grossly clear. there is minimal mucosal thickening of the right maxillary sinus. impression: no evidence of acute intracranial process. cta chest findings: there are massive bilateral dependent consolidations. additionally, within the aerated portions of the lungs, there are bilateral ground-glass opacities extending from the hila. additional nodular opacities are likely related to acute process, though no prior exams are available for comparison. a small amount of free air is noted along the left base. while this is not clearly localized, there is suggestion on coronal images (601b:24-26) that the collection is above the diaphragm and within the pleural space. the pulmonary arterial tree is well opacified and no filling defect to suggest pulmonary embolism is seen. no evidence of acute aortic syndrome is identified. there are coronary artery calcifications. no pericardial effusion is seen. the patient is intubated with the endotracheal tube tip low lying at the level of the carina. additionally, the balloon of the endotracheal tube appears hyperinflated. there is a left-sided subclavian central venous catheter with tip reaching the svc. an esophageal catheter is in place, incompletely imaged; however, coursing to the stomach at least. no lymphadenopathy is identified. the left thyroid lobe is enlarged. limited views of the upper abdomen demonstrate at least two calcified splenic artery aneurysms. bone windows demonstrate a nondisplaced sternal fracture (602b:34), which may be related to chest compressions. impression: 1. massive bilateral dependent consolidations, compatible with aspiration. ground-glass opacities consistent with pulmonary edema. 2. air collection at the left base felt more likely above the diaphragm representing small pneumothorax vs bulla as opposed to contained within the peritoneum. in comparison with prior radiographs of the chest, it appears this was present prior to placement of central line. 3. low-lying endotracheal tube with tip at the level of the carina and hyperinflation of the endotracheal tube balloon. 4. enlargement of the left thyroid lobe. 5. non-displaced sternal fracture may be related to chest compressions. tte left ventricular wall thicknesses and cavity size are normal. there is mild regional left ventricular systolic dysfunction with distal septal hypokinesis. the remaining segments contract normally (lvef = 45%). the right ventricular cavity is moderately dilated with focal hypokinesis of the apical free wall. the aortic valve leaflets (3) appear structurally normal with good leaflet excursion and no aortic stenosis or aortic regurgitation. the mitral valve leaflets are mildly thickened. there is no mitral valve prolapse. mild (1+) mitral regurgitation is seen. there is moderate pulmonary artery systolic hypertension. there is no pericardial effusion. impression: dilated right ventricle with mild systolic dysfunction. mild regional left ventricular systolic dysfunction. mild mitral regurgitation. moderate pulmonary hypertension. brief hospital course: primary reason for hospitalization: 73yo f with history of bipolar d/o transferred from osh s/p asystolic cardiac arrest, likely secondary to respiratory etiology. brief hospital course: ms. presented after witnessed episode of respiratory distress of unclear etiology which progressed to asystolic cardiac arrest. per ems records, cpr was started in field and rosc occured after 25 minutes of pulselessness. post-cardiac arrest hypothermia protocol was initiated at osh and she was transferred to for continued management. on arrival to she was intubated, cooled to 33 degrees f and was requiring maximum dose of 3 pressors (neo, levo, vpa) to maintain blood pressure. she became progressively hypoxic and acidotic on cmv ventilation despite max vent support with 100% fio2 and peep 24. rewarming was started at 1600 on . she had continuous eeg monitoring per hypothermia protocol which showed evidence of seizure activity, and she was loaded with iv keppra. on she became bradycardic and was noted to be pulseless. a code blue was called for pulseless arrest and cpr was initiated. rhythm showed asystole, received epi 1mg x3, 1 amp nahco3. attending physician met with patient's family, who decided to terminate resuscitation. time of death was declared at 1:39am. organ bank notified, declined patient as donor. medical examiner was notified and declined autopsy. medications on admission: lithium carbonate 300 mg tab oral 1 tablet(s) -> recently changed to daily lorazepam 0.5 mg tab oral 1 tablet(s) once daily, at bedtime fluvoxamine 50 mg tab oral 1 tablet(s) once daily -> just discontinued nabumetone 500 mg tab oral 1 tablet(s) as directed propranolol 10 mg tab oral 1 tablet(s) twice daily discharge medications: n/a discharge disposition: expired discharge diagnosis: respiratory arrest cardiac arrest discharge condition: expired discharge instructions: n/a followup instructions: n/a procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified arterial catheterization diagnoses: acidosis other pulmonary insufficiency, not elsewhere classified asthma, unspecified type, unspecified pneumonitis due to inhalation of food or vomitus cardiac arrest cardiogenic shock bipolar disorder, unspecified 

Answer: The patient is high likely exposed to