Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: the patient is a 69-year-old male with a past medical history significant for hypertension and hypercholesterolemia who presented as a transfer from an outside hospital with respiratory failure and hypotension. per the patient's wife, the patient had been experiencing flu-like symptoms with fevers to 101, chills, and progressive malaise. the patient also reportedly had respiratory symptoms including dyspnea, cough, and hemoptysis. on the day of admission, the patient reportedly developed increasing dyspnea and acute shortness of breath and 911 was called. in the ambulance, the patient was treated with morphine, nitroglycerin, and 80 mg of intravenous lasix for congestive heart failure. the patient required intubation while in the ambulance for hypoxic respiratory failure and subsequently developed pulseless electrical activity arrest. cardiopulmonary resuscitation was initiated, and the patient received several rounds of epinephrine and atropine; with, by report, return of pulse and blood pressure. on arrival to the emergency department at the outside hospital, the patient again developed pulselessness and was treated with 1 mg of epinephrine and dopamine. lasix was also administered in the emergency department. an electrocardiogram at the outside hospital demonstrated sinus tachycardia to the 120s, new right bundle-branch block, and 1-mm to 2-mm st depressions in the precordial and inferior leads. the patient was transferred to for possible cardiac catheterization and further management. past medical history: 1. hypertension. 2. hypercholesterolemia. 3. depression. allergies: no known drug allergies. medications on admission: medications prior to admission included zoloft and antihypertensives (particular medications unknown). physical examination on presentation: physical examination on arrival to revealed temperature was 98.8, heart rate was 110, blood pressure was 80/50 (on dopamine), oxygen saturation was 93% on assist-control with fio2 of 100% and 15 of positive end-expiratory pressure. in general, the patient was intubated and sedated. head, eyes, ears, nose, and throat revealed normocephalic and atraumatic. pupils were 3 mm, equal; however, minimally reactive. sclerae were anicteric. endotracheal tube was in place. pulmonary examination revealed bilateral rales anteriorly with bronchial breath sounds at the left base. cardiovascular examination revealed tachycardic with normal first heart sound and second heart sound. no third heart sound or fourth heart sound. no significant murmurs. unable to assess jugular venous distention. abdominal examination revealed soft, mildly distended, hypoactive bowel sounds. no masses were appreciated. extremities were warm with 2+ dorsalis pedis pulses and posterior tibialis pulses bilaterally. no edema was appreciated. neurologic examination revealed sedated with occasional agitation and withdrawal to pain. pertinent laboratory data on presentation: significant laboratories and studies at the outside hospital revealed arterial blood gas with a ph of 6.8, pco2 of 71.6, po2 of 66.8, with a bicarbonate of 11.3. creatine kinase was 191, and troponin i was 4.93. complete blood count was notable for a white blood cell count of 14.3. chemistry-7 was notable for a blood urea nitrogen of 18 and a creatinine of 1.5. normal liver function tests and coagulation studies. radiology/imaging: a chest x-ray at the outside hospital demonstrated bilateral alveolar infiltrates. laboratories and studies on admission to revealed complete blood count with a white blood cell count of 18.5, hematocrit was 40.5, and platelets were 242. white blood cell count differential revealed 77% neutrophils, 13% bands, 2% lymphocytes, 4% monocytes, and 4% atypical cells. chemistry-7 revealed sodium was 134, potassium was 5.9 (hemolyzed), chloride was 102, bicarbonate was 17, blood urea nitrogen was 28, creatinine was 2, and blood glucose was 351. calcium was 7.4, phosphate was 4.9, magnesium was 1.7. liver function tests revealed alt was 62, ast was 222, alkaline phosphatase was 949, total bilirubin was 0.9, ldh was 951. the second set of enzymes revealed creatine kinase was 1229, ck/mb was 74, with an index of 6. coagulation studies revealed pt was 16.7, ptt was 94.5, and inr was 1.9. arterial blood gas revealed ph was 7.31, pco2 was 36, po2 was 77, with an oxygen saturation of 95% on assist-control, tidal volume was 600, respiratory rate was 20, fio2 was 100%, with a positive end-expiratory pressure of 15. hospital course: the patient was admitted to the coronary care unit with multisystem organ failure. the patient remained intubated with adequate ventilation; however, relatively poor oxygenation with large bilateral pleural effusions and alveolar infiltrates. the patient developed a st-elevation myocardial infarction with peak creatine kinase of greater than , and an ejection fraction of 10% to 15%, secondary to global left ventricular hypokinesis. the patient continued with persistent hypotension requiring pressor support felt secondary to a combination of cardiogenic and septic shock. the patient was started on broad spectrum antibiotics for persistent fevers to 104; however, multiple sets of cultures including sputum, blood, and urine were without growth. the source of the infection was initially felt likely to be a pulmonary process; however, given the persistent fevers despite broad spectrum antibiotics, the patient underwent head, thoracic, and abdominal cat scans for evaluation for potential additional source of infection. the head cat scan was notable for diffuse (predominantly posterior) edema with loss of /white matter distribution. the sinus cat scan demonstrated multiple air/fluid levels in the paranasal sinuses. the chest cat scan demonstrated bilateral pleural effusions with pulmonary edema, and no lymphadenopathy. the abdominal cat scan was unremarkable. neurology was consulted for evaluation of neurologic status. the patient was weaned off of sedation with minimal neurologic responsiveness to stimuli. neurology recommended electroencephalogram and nuclear brain perfusion scan. the assessment; based on the results of the studies, per neurology, was extremely poor prognosis for meaningful recovery. given this information, the family decided to withdraw care. the patient died several minutes after extubation. , m.d. dictated by: medquist36 procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified diagnoses: pneumonia, organism unspecified acidosis pure hypercholesterolemia acute kidney failure with lesion of tubular necrosis congestive heart failure, unspecified unspecified essential hypertension acute respiratory failure other shock without mention of trauma acute myocardial infarction of other inferior wall, initial episode of care 

Answer: The patient is high likely exposed to