Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: the patient is a 60-year-old male status post liver transplant on complicated by mild acute cellular rejection treated with solu-medrol. he was seen yesterday in the clinic. he recently completed a steroid taper, and liver biopsy was planned for a few weeks. labs drawn as an outpatient showed platelet count to be 35. previous platelet count was 44. the patient was called to the come to the transplant center. past medical history: hepatitis c virus, alcohol cirrhosis, ascites, hepatic artery stenosis, status post stent placement, esophageal varices, umbilical hernia, inguinal hernia repair, liver transplant in , status post umbilical and left inguinal hernia repair. medications on admission: protonix 40 mg daily, bactrim single strength daily, vitamin d 400 iu daily, lasix 20 mg p.o. daily, calcium carbonate 500 mg p.o. q.i.d., dicloxacillin 100 mg p.o. daily, prednisone 15 mg p.o. daily, prograf 1.5 mg p.o. b.i.d., rapamune 3 mg p.o. daily. allergies: no known drug allergies. social history: no habits. nonsmoker. no alcohol. physical examination: vital signs: on admission t-max was 98.3, heart rate 84, blood pressure 138/77, respiratory rate 20, 97% on room air. general: no acute distress. heart: regular rate and rhythm. lungs: clear bilaterally. abdomen: soft, nontender, nondistended. rectal: normal tone. guaiac negative. hospital course: the patient was admitted to the transplant service. hepatitis c and cmv viral load were sent off. cmv was not detected. hepatitis c viral load was 23,100,000 iu/ml. hematology consult was obtained. it was noted that the patient had a bone marrow biopsy in late that showed no evidence of overt myelodysplasia and maturing trilineage erythropoiesis. findings were consistent with peripheral destruction of his hematopoietic cells. it was felt to be likely exacerbated by drug-induced affects, notably the calcineurin inhibitor was used for his immunosuppression. also suspected was bactrim and fluconazole. it was felt that the fluctuations in his platelet count was reflective of medication changes. hepatitis c infection was also felt to be a positive factor of the thrombocytopenia, as well as his liver dysfunction. heparin antibody were sent off. this was subsequently found to be negative. during his hospital course, vital signs remained stable. white blood cell count ranged between 5.3 and 8.1. hematocrit on admission was 38.8. this trended down to 36.1. platelet count was 106 on admission, and this increased to 135, subsequently decreasing to 100. creatinine was stable at 1.0. lfts on admission showed an ast of 150, alt 98, alkaline phosphatase 222, total bilirubin 1.9, with an albumin of 4.8. amylase and lipase were 77 and 18 respectively. inr was 1.2. lfts improved with an ast of 24, alt 29, alkaline phosphatase 121, total bilirubin 0.9. he remained on prograf 4 mg p.o. b.i.d. prograf levels were checked. these levels ranged between 12.5 and 15.3. of note, he did complain of diarrhea. stools were sent for c- diff x 3. all were negative. stool was also sent for o&p. this was also negative. given past history of hepatic artery stricture with stent placement, a cardiac echocardiogram was done. this demonstrated normal left ventricular cavity size, left ventricular systolic function appeared depressed along the posterior wall, which was hypokinetic. the right ventricular systolic function also appeared depressed. aortic valve leaflets were mildly thickened. the mitral valve leaflets were mildly thickened with 1+ mitral regurgitation noted. there was no pericardial effusion. on , he underwent a transjugular liver biopsy. he was given a bag of platelets pre-liver biopsy. post biopsy, he complained of some nausea and abdominal cramping. hematocrit post transjugular liver biopsy decreased to 25.7 from 27. he was transfused with 1 unit of packed red blood cells. ct scan was done to evaluate for hematoma. this demonstrated a small subcapsular hematoma and a moderate amount of hemorrhage within the abdomen and pelvis intermixed with a large amount of ascites. he was transferred to the sicu for monitoring. serial hematocrits were drawn. these were stable. he received an additional 2 units of packed red blood cells and 2 units of platelets. hematocrit increased to 33.2 and a platelet count of 94. coags were 12.9 for pt, ptt were 25.7, and inr was 1.1. his liver biopsy tissue returned with changes consistent with a recurrent viral hepatitis c, grade ii inflammation. findings were indeterminate for acute cellular rejection. he was transferred back to the medical surgical unit where his hematocrit remained stable. intravenous fluids was hep- locked, and his diet was advanced to a regular diet. his creatinine remained stable at 1.0. he remained on prograf, prednisone and rapamune. hematology followed throughout this hospital course. rapamune was discontinued. prograf continued at 2 mg p.o. b.i.d. for a level of 9.6. prednisone was continued. vital signs were stable. he was discharged home on . discharge medications: prograf 2 mg p.o. b.i.d., lasix 20 mg p.o. daily, bactrim was discontinued, metoprolol 12.5 mg p.o. b.i.d., prednisone 15 mg p.o. daily, protonix 40 mg p.o. daily, doxycycline 100 mg p.o. b.i.d. follow up: the patient was scheduled to followup in the outpatient clinic. he was instructed to call for a followup appointment within 1 week. discharge diagnosis: 1. recurrent viral hepatitis c grade ii inflammation. 2. hepatitis c virus status post liver transplant. 3. thrombocytopenia. 4. subcapsular liver hematoma status post transjugular liver biopsy. , procedure: closed (percutaneous) [needle] biopsy of liver transfusion of packed cells diagnoses: unspecified viral hepatitis c without hepatic coma hematoma complicating a procedure other specified surgical operations and procedures causing abnormal patient reaction, or later complication, without mention of misadventure at time of operation complications of transplanted liver surgical operation with transplant of whole organ causing abnormal patient reaction, or later complication, without mention of misadventure at time of operation 

Answer: The patient is high likely exposed to