Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: lethargy x 2 days major surgical or invasive procedure: none history of present illness: 82 yo m w/ h/ body dementia, parkinson's dz, htn, and h/o falls who p/w increasing lethargy x 2 days. moved to on due to progressive dementia s/p multiple falls. was stable at until the past 2 days when note was made of increasing difficulty w/ ambulation and difficulty swallowing. had swallow training today and kept coughing, per daughter. daughter and wife, no diarrhea. incontinent of urine at baseline but no foul smell recently. last fall approximately 5 days ago per wife. is nonverbal at baseline. however, family noticed he was very sleepy on arrival to ed and had an absent look about him but is now interacting with them more through touch since initial resuscitation. in ed, o2 sats dropped into 80s, and he initially required 100% nrb. sbp dropped from 120's to 90s. the patient was evaluated by the micu and placed on sepsis protocol. he received 4l ns. after receiving ivf, his bp stabilized. o2 sat eventually stabilized at 98% on 5l nc. the micu team felt that he did not require icu level of care so he was admitted to the medicine service for further observation past medical history: htn body dementia h/o falls parkinson's disease social history: lives at . dependent of all adls and incontinent of stool/urine at baseline. married and has daughter who visits him regularly. no significant h/o tob and only occasional etoh (none currently). family history: m w/ h/o alzheimer's disease physical exam: t 103.0 (rectal) hr 84 bp 119/82 rr 25 o2 98% 5l nc gen: elderly, in nad heent: perrla (3->2mm), sclera anicteric cv: rrr, no m/r/g, soft s1/s2 pulm: diminished bs at both bases, no wheeze/ronchi/rhales abd: nabs, soft, nt/nd, no masses/hsm extr: no le edema, warm/dry neuro: awake, alert, non-conversant pertinent results: cxr: tortuous aorta, no infiltrates . head ct: right subdural hemorrhage, primary isodense, suggesting subacute/chronic. no shift of midline structures. brain atrophy and areas of chronic lacunar infarction. head ct: head ct without iv contrast: again demonstrated within the right subdural space is a heterogeneous fluid collection, which is predominantly hypodense in attenuation with small foci of hyperattenuation noted. the appearance of this subdural hematoma is not significantly changed since the prior examination. there is associated mass effect upon the adjacent right cerebral cortex, which is unchanged since the prior examination, and only minimal right to left midline shift is again demonstrated, which also is unchanged since the prior examination. there are no new areas of intra- or extra-axial hemorrhage noted. the ventricles and sulci are stable in size, and there is no evidence of hydrocephalus. periventricular white matter hypodensities are again noted along with focal hypodensities within the basal ganglia, findings consistent with chronic microvascular and lacunar infarction. visualized paranasal sinuses and left mastoid air cells are clear. post- surgical changes within the right mastoid air cells are again demonstrated with an air fluid level, unchanged in the interval. impression: overall, stable appearance of subacute/chronic right subdural hematoma. no new areas of hemorrhage identified. . admission labs: 06:00pm urine rbc-* wbc-21-50* bacteria-mod yeast-none epi-0-2 06:00pm urine blood-lg nitrite-pos protein->300 glucose-tr ketone-tr bilirubin-sm urobilngn-1 ph-5.5 leuk-sm 06:00pm urine color-yellow appear-hazy sp ->1.030 06:21pm lactate-2.8* k+-4.7 06:30pm pt-15.4* ptt-27.7 inr(pt)-1.6 06:30pm plt smr-normal plt count-143* 06:30pm hypochrom-normal anisocyt-normal poikilocy-normal macrocyt-normal microcyt-normal polychrom-normal 06:30pm neuts-90.0* bands-0 lymphs-7.8* monos-2.1 eos-0 basos-0.1 06:30pm wbc-21.1*# rbc-5.52 hgb-17.0 hct-48.1 mcv-87 mch-30.9 mchc-35.4* rdw-13.6 06:30pm albumin-4.2 calcium-9.8 phosphate-2.7 magnesium-2.2 06:30pm ck-mb-6 ctropnt-<0.01 06:30pm alt(sgpt)-22 ast(sgot)-73* ck(cpk)-799* alk phos-81 amylase-23 tot bili-1.8* 06:30pm glucose-131* urea n-49* creat-1.4* sodium-142 potassium-5.4* chloride-105 total co2-20* anion gap-22* brief hospital course: he was initially stable on the floor, without fever or hypotension. then, in am of , he decompensated with temp to 101.6, rigors, and hypotension with sbp of 80's. additionally, his oxygen dropped to 78% on ra -> 97% on 100% nrb -> 97% on 3l nc. he was initially stabilized with 4l of ns boluses, but hypotension persisted to 70's systolic. dopamine was started. hypoxia persisted with sats in 80's on 100% nrb. a respiratory code was called, and he was intubated by anesthesia. abg done at codewhile on 100% nrb was 7.43/31/132, lactate of 2.0. he was transferred to the ccu for further treatment. . in the ccu he required one pressor for less than 24 hours. his hypotension was felt to be due to vasodilation from sepsis in combination with hypovolemia. his culture data only revealed gram negative rods in his urine, and vanc, levo and flagyl were continued. he was intubated for hypoxic respiratory failure. the etiology of his hypoxemia was unclear. his rapidly deteriorating neurolgic function from parkinsons' he may have aspirated, although, he did not have radiologic evidence of a pneumonia. his hypoxemia may have been due to acidosis and v/q mismatching. lenis did not reveal evidence of dvt. he was extubated successfully on . he had moderate secretions and received aggressive chest pt, but maintained adequate o2 sats on shovel mask. additionally, he was started on low dose beta-blocker for hypertension and treated with free water boluses for hypernatremia. during his icu stay he was less responsive to stimuli and a repeat ct scan revealed unchanged size and mass effect from subdural hematoma. his mental status improved to baseline according to family after weaning sedation. he developed lue edema and on a lue doppler was performed which showed occlusive thrombosis of both brachial veins and the axillary vein, and nearly completely occlusive thrombosis of the subclavian vein. pt. had not been receiving sc heparin because of his known subdural hematoma and risk for increased bleeding and mass effect, and a decision was made not to anti-coagulate him, even given the doppler results. stim test was performed, with decreased cortisol response, and pt. was pulsed with 7 days of fludrocortisone and hydrocortisone. during his ccu stay, he was started on tube feeds. a discussion was begun with the family regarding long-term nutrition and possible peg placement. he was about 8 l positive on day of transfer, and had been receiving lasix qd. pt was transfered from ccu to the floor on with resolved urosepsis. pt remained afebrile but un-responsive. he finished vanc/levo/flagyl x 10 days. his wbc continued to be elevated. family discussion was held and patient was made cmo. pt was started on morphine drip titrated to comfort. pt's breathing became more agonal over next few days and he peacefully expired on @ 10:18 am. family was notified and were able to say their final goodbyes while patient was still in the hospital. . # sdh: - appears to be subacute to chronic. stable. repeat ct last: unchanged subdural hematoma/hypodense collection, unchanged/persistent mass effect on right cerebelar cortex, minimal right to left midline shift. no hydrocephalus, no new hemorrhages. chronic lacunar and microvascular infarcts. neurosurg was re- consulted but unable to say whether burr hole procedure will reverse mental status changes, drainage in ~ 4 weeks is still recommended, once acute events stabilized. . # ue dvt - hold off on anticoagulation for now given sdh . # failed stim - initially tx with 7 d steroids; stable electrolytes/bp. . # ppx: pneumoboots, ppi . # fen: ngt, tube feeds were continued until pt was made cmo; supplemented with ivfs d5 1/2ns with 20 k. # full code: family met with the providers and decided to make cmo. . # communication: (daughter): cell (no messages), home , work medications on admission: mvi sinemet 25/100 1 tab po bid discharge medications: none discharge disposition: expired discharge diagnosis: 1. urosepsis 2. end stage parkinson's disease discharge condition: deceased discharge instructions: none followup instructions: none procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more insertion of endotracheal tube enteral infusion of concentrated nutritional substances diagnoses: urinary tract infection, site not specified unspecified essential hypertension acute kidney failure, unspecified unspecified septicemia severe sepsis unspecified fall personal history of tobacco use paralysis agitans acute respiratory failure septic shock encounter for palliative care dementia in conditions classified elsewhere without behavioral disturbance other and unspecified coagulation defects subdural hemorrhage following injury without mention of open intracranial wound, with no loss of consciousness dementia with lewy bodies 

Answer: The patient is high likely exposed to