Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no drug allergy information on file attending: chief complaint: status epilepticus major surgical or invasive procedure: lumbar puncture intubation history of present illness: mr. is a 31-year-old man with pmh significant for seizure disorder presents as a transfer from hospital after status epilepticus the day of presentation. he has had seizures since a traumatic brain injury in his teens s/p surgical treatment (after both a hiking and truck accident). initially the seizures were episodes of unresponsiveness with a recurrent dream, but 1.5-2yrs post-tbi he began to have generalized tonic-clonic seizures and has been on medications since. he was initially treated with trileptal and had seizures approximately once yearly (previously shorter duration but more severe convulsions than now), but presented to in with status (x1hr) in the setting of increased stress and hyponatremia, and was changed to dilantin. this was changed to keppra by his outpatient neurologist (dr. , back in . he had been seizure free since then until the day of presentation. he was in his usoh when his wife heard a noise around 3pm and found him sitting in his chair with arms flexed and jaw clenched. he had not vomited, which usually precedes his seizures. he had minimal convulsions (described as legs kicking and arm flopping) and this spontaneously resolved after 1-2 minutes. he got up and wanted to go to the bathroom but his wife convinced him to sit down and called 911. in this time, he was able to speak to her and knew her name. when ems came, they asked him to walk to the ambulance but he stared blankly at them, and then when in the ambulance he began to seize again. he was noted to have 2hrs of seizure activity (?intermittent) without return to baseline in between. he was given ativan 2mg x 7, fosphenytoin 1gm x 1, phenobarbital 1500mg x 1, and versed 2mg x 1 and required intubation. after the phenobarbital, he stopped seizing and has not been noted to have any seizures since transfer to . of note, his vital signs on admission to the osh were t 99.9, hr 125, bp 155/80, rr 16, and sao2 97%/2l. he was found to be febrile to 103.8 on transfer. ros is negative for fevers, chills, uri symptoms other than a couple sneezes attributed to allergies, dysuria, headache, photophobia, neck pain, abdominal pain, chest pain, dyspnea, cough. his wife thinks he has been sleeping well. she reports he has had some stress (financial) recently, but that it is not significantly more than usual (he has had more without seizures in the past). he has not had any recent medication changes. she does not think he has missed any medications. he had a cigarette the am of admission but has one daily. he had a beer the night prior, but has not been drinking heavily. he uses marijuana on occasion but she does not think he had any the day of presentation and denies any other drug use as far as she knows. past medical history: seizures secondary to traumatic brain injury as above social history: tobacco and rare etoh as above, h/o drug use with occasional marijuana use still family history: parents with hypercholesterolemia physical exam: vs: t 103.8, hr 105, bp 129/60, rr 19, sao2 100%/vent genl: nad, intubated, sedated cv: rrr, nl s1, s2, no m/r/g chest: cta bilaterally, vented bs abd: soft, ntnd, bs+ ext: warm and dry neurologic examination: mental status: sedated, grimaces to noxious, no eye opening, does not follow commands cranial nerves: pupils equal and reactive, no blink to threat, eyes midline with mild dysconjugation with outward deviation, moves past midline minimally in each direction to ocrs, +corneals bilaterally, +nasal tickle bilaterally, +cough motor: withdraws anti-gravity to noxious in all extremities, lue slightly less than others, but legs symmetric sensory: responds to noxious in all extremities dtrs: 2+ and symmetric, toes withdraw b/l pertinent results: labs: 139 109 12 120 4.0 22 1.0 ck: 679 wbc: 8.9; hct: 36.5; plt: 210 ca: 7.3 mg: 1.7 p: 2.5 alt: 19 ap: 43 tbili: 0.4 alb: 3.8 ast: 23 ldh: dbili: tprot: : lip: phenytoin: 4.2 csf: wbc 0; rbc 2; polys 1; lymphs 0; monos 0 u/a negative cultures of csf, urine, and blood negative. studies: head ct: 1. no acute intracranial pathology including no intracranial hemorrhage is detected. 2. right hyperostosis in frontal bone and right frontal craniotomy. there is a defect of the right frontal lobe, most likely related to the prior surgery in this area. cxr: the ng tube passes below the diaphragm with its tip terminating in the stomach, although the side-hole is roughly at the level of the gastroesophageal junction; thus, the advancement of at least 10 cm would be recommended. there is mildly increase in pulmonary vascularity which might be related to volume overload. no pleural effusion or pneumothorax is demonstrated. brief hospital course: mr. is a 31-year-old man with a seizure disorder secondary to traumatic brain injury who presented as a transfer from hospital following status epilepticus. his brief hospital course by problem is as follows: 1. neuro: status epilepticus. on transfer from , he was extubated and sedated. on the first morning of his hospitalization, his propofol was weaned without further evidence of seizure activity, and he was extubated. he was loaded with dilantin to a goal level of . his keppra was increased from his home 1000 mg to 1500 mg . eeg showed generalized slowing but no ongoing seizure activity or focal epileptiform discharges. ct showed no structural abnormality. mri was discussed, but it was felt that there would be no likely finding given his known tbi. his primary neurologist will consider mri at home. no precipitating cause was found for this (infectious work up revealed no source), although it's possible that the keppra dose was low for his size (90 kg). called out of the unit on and did well; sent home on . 2. pulm: he was stable on room air after extubation. 3. code: full 4. comm: primary neurologist dr. : ; wife : (c) 5. dispo: home medications on admission: keppra 1000mg allergies: nkda discharge medications: 1. levetiracetam 500 mg tablet sig: three (3) tablet po bid (2 times a day). disp:*180 tablet(s)* refills:*2* 2. phenytoin sodium extended 100 mg capsule sig: one (1) capsule po every twelve (12) hours for 2 days: take one tablet in the morning and one in the evening for the next two days, then take one tablet in the morning for two days, then discontinue this medication. disp:*6 capsule(s)* refills:*0* discharge disposition: home discharge diagnosis: -status epilepticus discharge condition: stable. neurologic examination normal. discharge instructions: please take all medications as prescribed. please attend all follow-up appointments. if you experience recurrent seizures or other concerning symptoms, call your primary care doctor or report to the nearest emergency for evaluation. note that you have had a seizure and are likely not able to drive. state laws differ with regard to how long you are not able to drive. you must call the driver's bureau to discuss this. followup instructions: please follow-up with your primary care doctor within the next two weeks. please also follow-up with your neurologist within the next two weeks. procedure: spinal tap incision of lung insertion of endotracheal tube diagnoses: tobacco use disorder grand mal status cannabis abuse, unspecified 

Answer: The patient is high likely exposed to