Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: s/p cardiac arrest major surgical or invasive procedure: arterial line endotracheal intubation history of present illness: 46m h/o dm1, esrd, polysubstance abuse found down at home lying in bed for unclear duration. last seen the evening prior; he had been complaining of chills, fatigue, and anorexia for a few days per his wife. upon arrival of ems, patient asystolic and received epi/atropine with return of vf followed by 200j shock into pea; after further epi/atropine (4 rounds total) return of sinus tach. he was brought to osh where he was intubated, after which he became hypotensive and was started on dopamine. med flighted to for further care. . in the ed, his vitals were t 93.2 hr 90s bp 118/75 sao2 100%. he was weaned off dopamine. noted to have fixed dilated pupils and an abscence of corenal reflexes. hypokalemic to 2.9. tox screen positive for cocaine. past medical history: dm1 c/b nephropathy, neuropathy gastroparesis esrd on hd (tuethusat at the clinic) severe gerd with h/o upper gi bleed h/o nephrotic syndrome and iga nephropathy pvd osteomyelitis hypothyroidism cad s/p non-st elevation mi () chf (ef 50%) anemia of chronic disease polysubstance abuse (crack cocaine, etoh) social history: he is on disability and lives with his brother. married, wife and children live in . he is a chronic smoker. there is a prior history of other substance abuse (crack cocaine, alcoholism). family history: his mother had an mi at the age of 54, and his father has diabetes. physical exam: t undetectable hr 85 bp 139/77 rr 13 sao2 100% on ac/1.0/450/20/5 general: intubated, cachectic heent: pupils fixed/dilated, rapid horizontal and vertical eye movements, anicteric, frothing mouth neck: supple, trachea midline cardiac: rrr, s1s2 normal, 2/6 sem pulmonary: coarse breath sounds bilaterally abdomen: soft, nondistended, flat, +bs extremities: warm, 2+ dp?pt pulses, no edema neuro: unresponsive to voice or pain, flaccid extremities, no apparent gag, toes mute, unable to illicit dtrs pertinent results: 03:45pm blood wbc-12.4* rbc-4.26* hgb-14.2 hct-41.3 mcv-97 mch-33.4* mchc-34.5# rdw-16.7* plt ct-145* 04:17am blood wbc-11.8* rbc-3.44* hgb-11.3* hct-33.8* mcv-98 mch-32.8* mchc-33.3 rdw-16.7* plt ct-131* 03:45pm blood neuts-95.0* bands-0 lymphs-1.8* monos-2.5 eos-0.4 baso-0.3 03:45pm blood pt-15.6* ptt-28.6 inr(pt)-1.4* 04:17am blood pt-18.6* ptt-37.1* inr(pt)-1.8* 03:45pm blood glucose-192* urean-20 creat-3.0* na-138 k-2.9* cl-98 hco3-29 angap-14 04:17am blood glucose-95 urean-67* creat-4.9* na-145 k-4.5 cl-104 hco3-25 angap-21* 03:45pm blood alt-24 ast-76* ck(cpk)-2462* alkphos-105 amylase-316* totbili-0.8 04:55am blood alt-34 ast-121* ld(ldh)-464* ck(cpk)-3748* alkphos-93 amylase-973* totbili-0.4 03:06am blood alt-30 ast-67* ld(ldh)-427* ck(cpk)-1429* alkphos-96 amylase-1044* totbili-0.6 04:17am blood ck(cpk)-727* 03:45pm blood lipase-15 04:55am blood lipase-9 03:06am blood lipase-10 03:45pm blood ck-mb-37* mb indx-1.5 ctropnt-0.41* 04:55am blood ck-mb-46* mb indx-1.2 ctropnt-0.52* 03:06am blood ck-mb-20* mb indx-1.4 ctropnt-0.39* 04:55am blood albumin-2.8* calcium-7.1* phos-4.5 mg-1.7 03:06am blood albumin-2.9* calcium-7.3* phos-5.6* mg-1.8 04:55am blood phenyto-16.0 03:06am blood phenyto-10.1 03:45pm blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 09:52pm blood type-art rates-26/0 tidal v-450 peep-5 fio2-60 po2-86 pco2-42 ph-7.50* caltco2-34* base xs-7 intubat-intubated vent-controlled 09:52pm blood lactate-1.2 09:11pm blood lactate-1.1 04:50am blood lactate-1.4 . ecg: sinus, 98bpm, normal axis, twi ii/iii/avf . cxr: detail limited secondary to obscuration by overlying trauma board. endotracheal tube in good position. ng tube probably in good position as well. no evidence of acute cardiopulmonary disease. . ct head: global loss of -white differentiation in bilateral cerebral hemispheres with diffuse hypodense appearance, most likely representing cerebral edema in this patient with cardiac arrest. no acute intracranial hemorrhage. brief hospital course: 46m h/o dm1, esrd found unresponsive at home in asystole. . # s/p cardiac arrest: found down in asystole for unclear duration. unclear cause, but differential included arrythmia, infection, mi, pe, arrythmia. cardiac enzymes were elevated but as the patient did receive shocks in the field this was a more likely explanation. tox screen was positive for cocaine so an arrythmogenic or ischemic event may have been precipitated by substance abuse vasospasm. based on exam and ct head, there was likely significant anoxic brain injury (see below). labs were notable for post-arrest hypokalemia which suggested the possible presence of a significant hypokalemia post-hd. he was continued on asa, but beta-blockers were held given recent hypotension, arrest, cocaine use. echocardiogram revealed global hk likely due to stunning arrest but no vegetations. infectious sources were treated (see below). hypothermia was not performed given the prolonged duration prior to presentation (unclear duration down, first presented to osh then med-flight to prior to our evaluation). . # respiratory failure: arrest. no acute process on cxr initially but then developed left-sided infiltrate likely due to aspiration event peri-arrest. he was started on levo/flayl for cap. trials of psv were attempted but unsuccessful. . # ?seizures: noted to have rhythmic eye and face/upper extremity movements at presentation. initially dilantin loaded but then dilantin stopped per neuro as eeg without cortical seizure activity. . # id: infiltrate on cxr likely aspiration and treated with levoflox/flagyl. also with 5/6 bottles positive for gpc in clusters and started on vancomycin; source may have been hd line. mildly elevated wbc count but afebrile. . # hypotension: resolved. likely sedatives and vagal stimulation during intubation vs. sepsis. at present, off dopamine gtt and maintaining good bps but became hypotensive again during dilantin loading that resolved with iv fluids. . # esrd: hd held until goals of care established (see below). continued renagel. there was no acute hd need. . # dm1: hypoglycemic initially, then improved. continued hiss. held nph for now. . # ?gi bleed: h/o severe esophagitis and upper gi bleeding. bright red blood noted return from og tube but hct remained relatively stable. . # anoxic brain injury: loss of grey-white junction on ct head and pronounced deficits on exam suggest significant injury. neurology was consulted and, after 72 hours without improvement, delivered grim prognosis for any significant recovery. at family meeting it was decided that to make the patient cmo based on his wishes. the ett was removed and the patient rapidly expired. medications on admission: asa 81 daily trazodone 100 daily humulin n 7 units daily hiss percocet prn imdur 60 daily lopressor 100 prilosec renagel 400 tid folate discharge medications: none discharge disposition: expired discharge diagnosis: cardiac arrest anoxic brain injury discharge condition: expired discharge instructions: none followup instructions: none procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube systemic arterial pressure monitoring diagnoses: other iatrogenic hypotension anemia in chronic kidney disease end stage renal disease nephritis and nephropathy, not specified as acute or chronic, in diseases classified elsewhere coronary atherosclerosis of native coronary artery tobacco use disorder congestive heart failure, unspecified severe sepsis unspecified acquired hypothyroidism other, mixed, or unspecified drug abuse, unspecified polyneuropathy in diabetes peripheral vascular disease, unspecified alcohol abuse, unspecified hypopotassemia acute respiratory failure pneumonitis due to inhalation of food or vomitus cardiac arrest anoxic brain damage cachexia old myocardial infarction ventricular fibrillation other septicemia due to gram-negative organisms infection and inflammatory reaction due to other vascular device, implant, and graft accidental poisoning by central nervous system stimulants hemorrhage of gastrointestinal tract, unspecified diabetes with renal manifestations, type i [juvenile type], not stated as uncontrolled diabetes with neurological manifestations, type i [juvenile type], not stated as uncontrolled gastroparesis diabetes with other specified manifestations, type i [juvenile type], not stated as uncontrolled 

Answer: The patient is high likely exposed to