Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: mr. is a 60-year-old gentleman with no prior medical history who was in his usual state of health until three days ago when he noted upper respiratory infection symptoms consisting of cough, rhinorrhea, and a sore throat. then he noted the sudden onset of chest ache at 4 a.m. on . this chest ache was initially dull but increased with inspiration and was relieved by sitting forward. the patient was able to go back to sleep, and when he woke up at 7 a.m., he noticed that the pain was somewhat decreased in its intensity. he went to work as a carpenter (as he would do routinely), and he noticed that the chest pain was recurring and becoming more intense throughout the day. he denies an increase in the pain with exertion or any shortness of breath. he also denies any diaphoresis and denies any nausea or vomiting. that night, he reported to emergency department complaining of chest pain. his temperature at that time was 99.6, and he was found to be in atrial fibrillation with a heart rate in the 150s. he was given lopressor, cardizem, and an esmolol drip with his blood pressure decreasing to 80/palpitation. he was also given sublingual nitroglycerin and aspirin with improvement of his chest pain from an intensity of to . he was then transferred to for further management. en route from to , he received intravenous morphine with almost complete resolution of the chest pain. at , and electrocardiogram was done which showed atrial flutter and st elevations in leads i, ii, avl, and v3 through v6. cardiology was consulted, and the patient was initially started on heparin and aggrastat and was admitted to the coronary care unit for possible acute coronary artery syndrome. past medical history: none. medications on admission: none. allergies: no known drug allergies. social history: he is a carpenter. he is divorced and lives with his girlfriend. has three grown up children who live nearby. he has a 40-pack-year history of tobacco, and he drinks about three beers per day. family history: his mother died of a heart attack at the age of 65. physical examination on presentation: physical examination on presentation revealed temperature was 99.8, heart rate was 90, blood pressure was 101/68, oxygen saturation was 96% on 2 liters on 6 cm of water. in general, a middle-aged male in no acute distress. very pleasant. head, eyes, ears, nose, and throat examination revealed pupils were equal, round, and reactive to light. the oropharynx was clear. mucous membranes were moist. the neck revealed jugular venous pressure around 9 cm of water. positive bulky 1-cm to 2-cm anterior submandibular lymphadenopathy; mostly on the right side. the chest was clear to auscultation bilaterally. cardiovascular examination revealed a regular rate and rhythm. positive soft diastolic murmur loudest at the left sternal border. no friction rub. the abdomen was soft, nontender, and nondistended. positive bowel sounds. extremities revealed no edema. good dorsalis pedis pulses bilaterally. neurologically, a nonfocal examination. pertinent laboratory values on presentation: complete blood count revealed white blood cell count was 24.9, hematocrit was 46.4, and platelets were 325 at the outside hospital. chemistry-7 at revealed sodium was 135, potassium was 4.8, chloride was 105, bicarbonate was 19, blood urea nitrogen was 14, creatinine was 0.9, and blood glucose was 155. creatine kinase was 114. troponin was less than 0.3. radiology/imaging: electrocardiogram #1 at the outside hospital revealed atrial fibrillation with a heart rate of 168. st elevations in v5 and v6. electrocardiogram #2 at the outside hospital revealed atrial fibrillation with a heart rate of elevations in leads i, avl, and v2 through v6. p-r elevation in lead avr. electrocardiogram #1 upon arrival to revealed atrial fibrillation/atrial fibrillation with a heart rate of 93. st elevations in leads i, avl, v2 through v6; q waves in leads ii, iii, and avf. hospital course: 1. cardiovascular system: (a) rhythm: mr. is a 60-year-old gentleman with risk factors for coronary artery disease including a long history of tobacco use, sex, age, and family history. also with electrocardiogram with q waves in the inferior leads consistent with old inferior myocardial infarction who presented with chest pain and st elevations in the lateral leads; initially suspicious for lateral ischemia. the patient was initially started on aggrastat and heparin for treatment of possible acute coronary syndrome. however, upon further questioning and examination, it became evident that the acute pain was caused by pericarditis. an echocardiogram was performed which showed atrial fibrillation which prevented the determination of the ejection fraction and a small pericardial effusion with no tamponade. heparin and aggrastat were stopped. the patient was started on motrin for relief of symptomatic pain. the patient remained tachycardic and in atrial fibrillation. therefore, he was started on diltiazem and beta blocker for rate control, and he was put on amiodarone in the hopes that he would cardiovert; however, this did not happen, so the patient underwent electrocardioversion with electricity at 200 joules. he was rapidly converted. he converted the first time and remained in a sinus rhythm for approximately 30 seconds; after which he went back into atrial flutter. electrocardioversion was attempted three more times at 300 joules, 300 joules, and 360 joules without success. therefore, the patient opted for fibrillation which was performed with success. the patient was then continued on amiodarone 400 mg p.o. t.i.d., and diltiazem was stopped. (b) coronary artery disease: the patient had evidence of old inferior myocardial infarction on electrocardiogram and hypokinesis in the inferolateral walls on echocardiogram consistent with old inferior myocardial infarction. the patient was started on aspirin and lopressor. the patient was instructed to follow up as an outpatient and discuss with is primary care physician the possibility of a stress test for further workup of his coronary artery disease. (c) anticoagulation: in the hospital, the patient was started on heparin and coumadin at the same time. inr on the day of discharge was 1.8. therefore, the patient was discharged on lovenox 100 mg subcutaneously q.d. until his inr was between 2 and 3; which is therapeutic. 2. bilateral phlebitis: the patient developed bilateral phlebitis, and he was treated with keflex with improvement of the phlebitis while in the hospital. medications on discharge: 1. aspirin 81 mg p.o. q.d. 2. metoprolol 25 mg p.o. b.i.d. 3. amiodarone 400 mg p.o. t.i.d. (for four days); then 400 mg p.o. b.i.d. (for one week); then 400 mg p.o. q.d. (for one week); and then 200 mg p.o. q.d. (for two weeks or until he sees his electrophysiology cardiologist). 4. coumadin 5 mg p.o. q.d. (for one month if he remains in sinus rhythm). 5. lovenox 100 mg subcutaneously q.d. (until inr is 2 or higher than that). 6. keflex 500 mg p.o. q.i.d. (for five days). discharge status: discharge status was to home with to administer lovenox injections until inr is therapeutic. condition at discharge: condition on discharge was good. discharge diagnoses: pericarditis with atrial fibrillation. discharge instructions/followup: 1. the patient was to follow up with his primary care physician within the next two weeks who was to set him up with a cardiologist in the area where he lives. 2. the patient also to follow up with electrophysiology cardiologist, dr. , within four weeks. 3. the patient was discharged with of hearts monitor. , m.d. dictated by: medquist36 procedure: injection or infusion of platelet inhibitor diagnostic ultrasound of heart other electric countershock of heart catheter based invasive electrophysiologic testing excision or destruction of other lesion or tissue of heart, endovascular approach cardiac mapping diagnoses: tobacco use disorder congestive heart failure, unspecified atrial fibrillation old myocardial infarction acute idiopathic pericarditis 

Answer: The patient is high likely exposed to