Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: percocet attending: chief complaint: fever.sob major surgical or invasive procedure: bronchoscopy, intubation history of present illness: this is a 55m with mantle cell lymphoma , d+35 s/p an autologous stem cell transplant. he was in his usoh until 2 days ago when he developed worsening non-productive cough, sob and low grade fever. his temp in the clinic yesterday was 100.5. blood cxs were taken and he was started on azithromycin and vancomycin. today he was worse and atovaquone was added to his abx. however this evening he had a temp of 102 at home and he also was more dyspneic even at rest and he went to the ed. last week he had diarrhea which improved after he was started on lomotil. last week he was also noted to have transaminitis and his fluconazole was held. he denies le edema, chest pain, shortness of breath, nausea, vomiting. past medical history: 1. lipoid nephrosis at age 27, resolved with prednisone 2. lichen planus, resolved after chemotherapy, no h/o hep c 3. rectal fissures, resolved after sphincterotomy per pt 4. sciatica, resolved after exercises and stretches . past oncology history: he was diagnosed with mantle cell lymphoma by bone marrow biopsy when he was found to have an elevated white count on routine labs. his cytogenetics showed a 11:14 translocation. he has been treated with 3 cycles of hypercvad, first two were complicated by volume overload and hyponatremia. he has also received 2 doses of rituximab. his course was also complicated by splenic laceration () from splenomegally secondary to neulasta and hyperleukocytosis. underwent cycle 3a of hypercvad (cytoxan, vincristine, adramycin, decadron) in . on underwent high dose cytoxan for stem cell harvesting prior to his autologous stem cell transplantation. social history: he continues to work managing a computer database at . he quit smoking 30 years ago, at which point he had been smoking ppd for 10 years. he drinks 1-2 drinks per evening. distant h/o mj use. no ivda. he lives with his wife, no children, one dog. family history: his mother had a precancerous condition of the breast which was treated with bilateral mastectomy. his father is well. he has no children and has one healthy brother. there is no known family history of blood disorders or actual cancers. physical exam: vs: 99.1 (ax), hr:124, bp:162/80,rr:16,spo2:88 general: middle aged male, sitting in bed, in significant respiratory distress, using accessory muscles, anxious. heent: perrl eomi. no scleral icterus. mmdry op clear without thrush or lesions. neck: no jvd. no appreciable lad. lungs: +diffuse fine crackles, l>r with diffuse inspiratory and expiratory rhonchi cv: rrr s1 and s2 barely audible (above rhonchi) w/out m/r/g abd: soft, nt, nd, nabs, no masses. ext: 2+ dps. no c/c/e neuro: nonfocal, aox3 . pertinent results: cxr (): new diffuse bilateral increased interstitial markings suggestive of an atypical infection of viral etiology in this neutropenic patient . ct chest on : impression: 1. diffuse bilateral consolidation of all lobes with relative sparing of the apices consistent with multifocal pneumonia or possibly ards. 2. moderate right and small left layering pleural effusions. . u/s abd (): splenomegaly. otherwise, unremarkable abdominal ultrasound. brief hospital course: 55 y/o male on s/p autosct for mantle cell lymphoma who was initially admitted with fever and hypoxemia with bilateral infiltrates and effusions. the following events were addressed during his admission. 1. hypoxemia on arrival to the floor, patient was sating about 90% on 4 liters. abg was done that showed 7.47/34/38 on ra. a ventury mask was started and his oxygenation improved. x ray showed new diffuse bilateral increased interstitial markings suggestive of an atypical infection of viral etiology. patient was started on broad spectrum antibiotics, tmp-smx to cover pcp, steroids. pulmonary was consulted who felt that the process was more likely a viral or atypical pneumonia. in order to bronch him, they felt that he would have to be intubate at that point and after discussion with him and the family it was decided to defer it. ct scan showed diffuse bilateral consolidation of all lobes with relative sparing of the apices consistent with multifocal pneumonia or possibly ards and moderate right and small left layering pleural effusions. his respiratory status decompensated and on he had to be transferred to the icu for intubation. course to : the pt was admitted to the for intubation after developing progressive hypoxia, likely secondary to ards vs multifocal pna. his oxygen saturation prior to admission was 80% on 13l ventimask and nc. the pt was sedated and intubated by anesthesia. he underwent an a-line placement. pt had a bronchoscopy that showed evidence of prior dah, and patient was initiated on high dose solumedrol 100 mg iv bid. an tte with bubble study was negative for any pfo or asd that was causing persistent hypoxemia despite adequate ventilation. a cta was negative for any pe. his daily cxr continued to be consistent with ards of unknown etiology combined with dah. his bal cultures remained ngtd, and all other cx data remained negative. patient was continued on cefepime and vanco for treatment of his neutropenic fever, although no etiology to his fevers could be found. eventually, patient's wbc count returned to , and his fevers resolved. he self-extubated himself on and was transitioned initially to bipap and then to face mask as tolerated. he was eventually weaned down to 6l nc on transfer to the floor. his steroid dose was quickly tapered down given his immunocompromised state, and at time of transfer, was on solumedrol 100mg iv daily. at that point he had completed a 10 day course of cefepime and vancomycin on . night prior to transfer to the floor patient spiked fevers and it was decided to continue with empiric antibiotic coverage. his steroids were decreased upon transfer. on the floor, his respiratory status was tenous and his fevers continued. his given ? of new infiltrates in x ray prior to transfer a ct was ordered. it showed marked improvement of consolidations and septal wall thickening in comparison to the previous ct, but rapid worsening during the last three days in comparison to the chest x-ray suggest pulmonary edema. infectious process should also be included in the dx. on he had an episode of low sats to 88% and increase shortness of breath. chest x ray showed worsening bilateral pulmonary opacities with progression to frank alveolar consolidation. report suggested recurrent pulmonary hemorrhage or pulmonary edema. he was given lasix however his respiratory status did not improved much. echocardiogram was also done that showed no change lvef >55%, and moderate pulmonary hypertension. basically unchanged from prior. given his persistent fevers and unclear etiology of pulmonary infiltrates pulmonary was re-consulted and infectious disease service. given lack of clear of positive data from blood cx, urine cx and bal, it was decided to continue treating empirically for cap, pcp, diffuse alveolar hemorrhage. patient was continued on cefepime, vancomycin, clindamycin and primaquine were added for pcp coverage, ambisome, and steroid dose was increased. next day x ray was checked and showed marked improvement, however o2 requirement still present with sats 92-94% on 40%fio2. given this rapid changes on x ray infiltrates, id and pulmonary feel at this point that is less likely to be infectious. despite being on broad antibiotic coverage, his respiratory status again declined on . his steroids were increased. he was diuresed with lasix. abx and steroids were continued. unclear what was causing his episodes of respiratory distress. transferred back to oncology floor. on the am of , he became more hypoxic and was again transferred to the . continued to diurese. continued on abx. ct revealed worsening disease, b/l airspace opacities. he was then started on bipap. he was intubated around 3pm on . bronchoscopy was attempted at 4:30pm. pt became bradycardic and hypotensive during bronchoscopy. code blue was called and cpr was initiated. family decided to change his code status to dnr/dni. pt expired at 6:25pm. 2. fevers: as stated above. blood cx, bal and urine cx all negative. 3. elevated lft/alk phosph: slightly elevated on admission. trending up over the course of hospitalization. u/s was done on that was normal. hep b and c was negative in . repeated serologies were sent and were negative. thought to be medication related. medications on admission: : famvir 500mg qd, protonix 40mg qd, azithromycin (started on ), vancomycin (started on ), atovaquone (started on ), ativan prn, tylenol#3 prn . meds on transfer: . acetaminophen 650 mg po x1 prn prior to prbc tx hydrocodone-acetaminophen tab po q4-6h:prn acetaminophen 500 mg po q6h:prn lorazepam 0.5-1 mg po/iv q4-6h:prn azithromycin 250 mg po q24h methylprednisolone sodium succ 100 mg iv q 12h ceftriaxone 1 gm iv q24h pantoprazole 40 mg po q24h order date: @ 0228 diphenhydramine hcl 25 mg po/iv x1 prn prior to prbc tx famvir *nf* 500 mg oral daily sulfameth/trimethoprim 420 mg iv q8h guaifenesin-codeine phosphate ml po q6h:prn cough vancomycin hcl 1000 mg iv q 12h discharge medications: pt expired. discharge disposition: expired discharge diagnosis: pt expired. discharge condition: pt expired. discharge instructions: pt expired. followup instructions: pt expired. md procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more parenteral infusion of concentrated nutritional substances insertion of endotracheal tube enteral infusion of concentrated nutritional substances closed [endoscopic] biopsy of bronchus closed [endoscopic] biopsy of bronchus transfusion of packed cells cardiopulmonary resuscitation, not otherwise specified transfusion of platelets diagnoses: pneumonia, organism unspecified unspecified pleural effusion congestive heart failure, unspecified other specified disorders of pancreatic internal secretion adrenal cortical steroids causing adverse effects in therapeutic use hyposmolality and/or hyponatremia other malignant lymphomas, unspecified site, extranodal and solid organ sites peripheral stem cells replaced by transplant acute respiratory failure cardiac arrest cachexia raynaud's syndrome nonspecific elevation of levels of transaminase or lactic acid dehydrogenase [ldh] 

Answer: The patient is high likely exposed to