Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: gtc seizure and r occiptal hemorrhage major surgical or invasive procedure: cerebral angiogram history of present illness: the pt is a 58 year-old man (handedness unknown) who was transferred from . this history is obtained from his transfer records and his girlfriend as he was intubated for agitation prior to transfer. it appears from the record that mr. had presented to the ed with complaints of seeing "flashing lights" associated with a headache today. it is documented that he would only "look to the left with the head turned". he also complained of nausea and vomiting. per his girlfriend, the symptoms started around 9am this morning. he waited all day and she was finally able to convince him to go to the ed. on arrival he had a "grand mal" seizure in triage for which he was given 3mg of ativan. his bp at the osh was recorded as 175/117. the nursing notes are difficult to read, however it appears that he had vomiting while there prior to his ct. the image showed a 2cm r occipital bleed and a repeat study 3 hours later was stable. over the next few hours he was given ativan 1mg, 2mg, 2mg, dilantin 1gm and then another ativan 2mg. in one of the notes, he was described as unresponsive but moving extremities and head but not purposefully, "thrashing in bed". he was reportedly in restraints at this time and his behavior was attributed to a post-ictal state. at 2200 he was given versed 1mg x 2 and then another dose of ativan (?). prior to transfer he was given another dose of versed and intubated. his labs at showed a metabolic acidosis with a low hco3 and an anion gap of 19. his inr was 1.1 and his platelet were 197. his tox screen was positive for thc. these symptoms occur in the context of a fall 3 weeks ago. he was sitting and fell forward striking his forehead. he had loc for a few seconds but was reportedly back to baseline immediately without evidence of being post-ictal. past medical history: hepatitis c - reports receiving treatment but details unknown social history: -heavy and longstanding tobacco use -no etoh or drug use per girlfriend -works from home as a currency exchanger family history: father with cancer. physical exam: vitals: t: af p: 80 r: 20 bp: 132/75 map: 91 sao2: 100% on eg general: intubated, sedated heent: nc/at, no scleral icterus noted, et in place neck: supple, no carotid bruits appreciated. no nuchal rigidity pulmonary: lungs cta bilaterally cardiac: nl s1, s2, regular abdomen: soft, nt/nd, normoactive bowel sounds, no masses or organomegaly noted. extremities: no peripheral edema skin: + petechia on chest and shoulders, no splinter hemorrhages neurologic: -mental status: intubated, sedated, does not follow grimaces to nox stim but does not have consistently purposeful movements cn i: not tested ii,iii: pupils 1mm but reactive, unable to visualize fundi due to pupil size iii,iv,v: no dolls v: + corneals with head turning away to stimulus, + v1-v3 to lt vii: face grossly symmetrical w/ et in place viii: ua ix,x: + gag : ua xii: ua motor: normal bulk and tone; withdraws symmetrically to nox stim in all extremities reflex: no clonus tri bra pat an plantar c5 c7 c6 l4 s1 cst l 1 1 1 2 0 flexor r 1 1 1 2 0 flexor -sensory: withdraws to nox stim in all extremities -coordination: na -gait: na physical exam at discharge was essentially normal, with normal visual field to confrontation test. gait was stable. pertinent results: 01:00am blood wbc-9.6 rbc-4.13* hgb-13.4* hct-37.6* mcv-91 mch-32.5* mchc-35.6* rdw-13.3 plt ct-167 01:00am blood glucose-106* urean-11 creat-1.0 na-141 k-3.5 cl-108 hco3-24 angap-13 01:00am blood alt-27 ast-29 ld(ldh)-276* ck(cpk)-762* alkphos-54 amylase-30 totbili-0.8 01:00am blood tsh-2.5 03:47pm blood phenyto-13.6 04:45am blood hbsag-negative hbsab-borderline hbcab-negative hav ab-positive igm hbc-negative igm hav-negative 04:45am blood hcv ab-positive mr/mra/mrv of head: 1. hyperacute parenchymal hemorrhage with small amount of surrounding edema in the right occipital lobe as described above. no findings are detected to explain the etiology. if the hemorrhage was secondary to a small avm, this would not be detectable on mri, and if clinical concern present, a conventional arteriogram is recommended. 2. intensely enhancing lesion with irregular borders in the atrium of the left lateral ventricle with associated cystic component indenting into the superior ventricular. given that similar enhancement characteristics are noted and a focus of choroid plexus appears to extend towards this region, this most likely represents a focus of ectopic choroid plexus. correlation with ct and continued followup is recommended. 3. sinusitis. cerebral angiogram official report pending. brief hospital course: patient was admitted to nsicu and successfully extubated the next day - initially was quite agitated requiring several doses of haloperidol, possibly due to the r occipital hemorrhage but soon improved with redirection provided per 1:1 sitter. he was also loaded with dilantin and was continued on dilantin for seizure prophylaxis. his mri with gadolinium was unrevealing. he underwent angiogram per dr. on without complications, which raised the possibility of an occipito-temporal arteriovenous fistula. a repeat angiogram on under anesthesia was negative, however. during the admission he was noted to have progressive ck elevations, and had mild rash around his neck. it was felt to be due to dilantin hypersensitivity syndrome. dilantin was dicontinued and keppra was started. ck went down. he tolerated well keppra. he was discharged with plans for repeating his mri with gadolinium in 4 weeks. consideration for additional studies, such as ct torso, will be determined after his repeat mri by . & upon follow up. dr. will also discuss the results of his angiogram with dr. , our interventional vascular neurosurgeon, who is currently out of the country when he returns. medications on admission: none discharge medications: 1. acetaminophen 325 mg tablet sig: one (1) tablet po q6h (every 6 hours) as needed. 2. levetiracetam 500 mg tablet sig: two (2) tablet po bid (2 times a day). disp:*120 tablet(s)* refills:*0* 3. phenol-phenolate sodium mouthwash sig: one (1) spray mucous membrane q4h (every 4 hours) as needed. disp:*1 1* refills:*0* discharge disposition: home discharge diagnosis: right parietal-occipital intracerebral hemorrhage seizures discharge condition: stable with mild residual visual changes discharge instructions: you were admtted to this hospital because you developed visual changes and headache, while in the outside hospital you had a generalized seizure. you underwent a head ct which showed bleeding in the posterior aspect of your brain. to evaluate the cause for this bleeding you had a brain angiogram, the first test showed a possible fistula in your vessels but because of the amount of the bleeding and movement artifact a second angiogram was required. to prevent further seizures you need to continue taking keppra 1000mg twice a day. the most common side effect is behavior changes. while in the hospital your muscles enzymes levels were levated, and this might be related to the shaking movements during the seizure. before your discharges the ck level was going down. please call dr or return to the emergency department if you have visual changes, persistent headache, mental status change, weakness, dizziness or seizures. you will need to call dr to obtain a referal to see neurology on followup instructions: provider: mri phone: date/time: 3:15 provider: . & phone: date/time: 1:30 please call the office to provide general information md, procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours arteriography of cerebral arteries arteriography of cerebral arteries diagnoses: tobacco use disorder unspecified viral hepatitis c without hepatic coma other convulsions intracerebral hemorrhage cerebral edema dermatitis due to drugs and medicines taken internally other nonspecific abnormal serum enzyme levels hydantoin derivatives causing adverse effects in therapeutic use other visual distortions and entoptic phenomena 

Answer: The patient is high likely exposed to