Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: lisinopril / shellfish derived attending: chief complaint: chest pain major surgical or invasive procedure: cardiac catheterization history of present illness: mr. is a 46 year old man w/hx of cad s/p inferolateral mi and 2 to proximal lcx into om1 who presented to the ed with chest pain. the patient reports chest tightness which started while he was walking to work this morning. he works at and continued to walk from to the ed. by the time he reached the ed, the chest tightness was , a band sensation across his chest. no radiation to arm, jaw, or back. he denies diaphoresis or nausea but does report associated sob. he did not take ntg. he reports rare instances of chest pain since his mi but did have an episode for which he was evaluated in the ed and it was determined to be non-cardiac. he had an exercise tolerance test at that time which was normal. he does not take ntg at home and does not have any. total time of chest pain prior to arrival to ed was 20 minutes. . in the ed, initial vitals were t99.2, bp175/86, hr99 rr18 o2 sat 99%. ecg showed st elevations in ii, iii, avf. he received aspirin 325mg x 1, plavix 600mg x 1, morphine 4mg iv x 1. ntg gtt, heparin gtt and integrillin gtt were started. a code stemi was called and he was taken to the cath lab with door-to-balloon time of 40 minutes. . in the cath lab, his prior in om1 was occluded with an acute thrombus. an export wire extracted the clot and the patient became chest pain free. a balloon angioplasty was performed and ivus showed the stent to be intact. he was given prasugrel 60mg x 1 in the cath lab. . on arrival to the ccu, the patient feels well and denies chest pain, pressure or tightness, shortness of breath, nausea, vomiting, headache, abdominal pain, calf pain. of note, he admits to missing several of plavix in the last few months. his aspirin dose was recently decreased from 325mg to 81mg daily. . on review of systems, he denies any prior history of stroke, tia, deep venous thrombosis, pulmonary embolism, bleeding at the time of surgery, myalgias, joint pains, cough, hemoptysis, black stools or red stools. he denies recent fevers, chills or rigors. he denies exertional buttock or calf pain. he states that he has had a rash in his groin area recently. all of the other review of systems were negative. . cardiac review of systems on admission is notable for absence of chest pain, dyspnea on exertion, paroxysmal nocturnal dyspnea, orthopnea, ankle edema, palpitations, syncope or presyncope. past medical history: cad s/p inferolateral mi with 2 overlapping cypher stents to occluded lcx into om1 prior rca stenting angioedema after starting lisinopril , resolved pneumonectomy s/p mva social history: social history is significant for the absence of current tobacco use, quit in , 1ppd prior. there is no history of alcohol abuse. lives with his wife and 2 children. works in purchasing at . family history: there is family history of premature coronary artery disease in his father at age 41. physical exam: vs: t=97.8 bp=142/59 hr=84 rr=16 o2 sat= 99% 2l nc general: alert and oriented x 3, nad. mood, affect appropriate. heent: ncat. slight reddened appearance to face and neck area. sclera anicteric. perrl, eomi. conjunctiva were pink, no pallor or cyanosis of the oral mucosa. no xanthalesma. neck: supple with flat jvp. cardiac: pmi located in 5th intercostal space, midclavicular line. rr, normal s1, s2. no m/r/g. no thrills, lifts. no s3 or s4. lungs: no chest wall deformities, scoliosis or kyphosis. resp were unlabored, no accessory muscle use. ctab, no crackles, wheezes or rhonchi. abdomen: soft, ntnd. no hsm or tenderness. abd aorta not enlarged by palpation. no abdominial bruits. extremities: no c/c/e. no femoral bruits. skin: no stasis dermatitis, ulcers, scars, or xanthomas. no appreciable rash. pulses: right: carotid 2+ femoral 2+ popliteal 2+ dp 2+ pt 2+ left: carotid 2+ femoral 2+ popliteal 2+ dp 2+ pt 2+ right femoral cath site is clean, dry and intact with a small soft hematoma palpable. no femoral bruits. pertinent results: admission labs: 07:59am blood wbc-8.5 rbc-4.72 hgb-13.5* hct-40.6 mcv-86 mch-28.5 mchc-33.2 rdw-14.4 plt ct-310 07:59am blood pt-11.6 ptt-22.0 inr(pt)-1.0 07:59am blood glucose-143* urean-12 creat-1.1 na-141 k-4.0 cl-106 hco3-21* angap-18 07:59am blood ck(cpk)-212 07:59am blood ctropnt-<0.01 ---------------- discharge labs: ---------------- studies: . ekgs: pre-cath: nsr at 7bpm. nl axis, nl intervals. 3mm st elevations in ii, iii, avf, v4-v6 with st depression sin avl, v1, v2, v3. hyperdynamic t waves in v3, v4, v5. post-cath: resolving st elevations which are not quite as pronounced. . cardiac cath : comments: 1. coronary angiography in this right dominant system demonstrated two vessel disease. the lmca had no angiographically apparent disease. the lad had an origin 30% stenosis. the lcx had a 30-40% origin stenosis and moderate thrombus within the mid stented segment. the rca had widely patent stents and a 30-40% mid stenosis. 2. limited resting hemodynamics revealed normaly systemic arterial blood pressure with sbp 103mmhg and dbp 69mmhg. 3. successful thrombectomy and ptca of the om stent thrombus with a 3.5mm balloon. 4. successful closure of the right femoral arteriotomy site with a 6f perclose device. final diagnosis: 1. two vessel coronary artery disease. 2. thrombus within mid lcx stent successfully treated with thrombectomy and ptca. . tte : the left atrium is mildly dilated. no atrial septal defect is seen by 2d or color doppler. left ventricular wall thicknesses and cavity size are normal. there is mild regional left ventricular systolic dysfunction with infero-lateral hypokinesis. there is no ventricular septal defect. right ventricular chamber size and free wall motion are normal. the diameters of aorta at the sinus, ascending and arch levels are normal. the aortic valve leaflets (3) appear structurally normal with good leaflet excursion. there is no valvular aortic stenosis. the increased transaortic velocity is likely related to high cardiac output. no aortic regurgitation is seen. the mitral valve appears structurally normal with trivial mitral regurgitation. there is no mitral valve prolapse. there is mild pulmonary artery systolic hypertension. there is no pericardial effusion. compared with the report of the prior study (images unavailable for review) of , no change. brief hospital course: mr. is a 46yo m with cad s/p prior inferolateral mi and to om1 who presented to the ed with chest pain, was found to have a stemi and taken to the cath lab were om1 stent showed acute thrombus. . # stemi: patient presented with inferior stemi, and door-to-balloon time was 40min. in the cath lab, patient was found to have thrombosis in the om1 stent. an export wire extracted the clot and the patient became chest pain free. a balloon angioplasty was performed and ivus showed the stent to be intact. this acute thrombosis in the stent may be due to missed plavix dosing; however it is also possible that he has failured plavix. as a result, plavix was switched to prasugrel. pt was given 60mg loading dose in cath lab, and was kept on 10mg po qday. patient was also continued on aspirin 325mg daily, metoprolol 25mg po bid, toprol xl 25mg daily and lipitor 80mg daily. patient came back from the cath lab on nitro gtt which was promptly turned off, and he was chest pain free during the rest of his hospital stay. . # pump: no evidence of heart failure; prior echo showed posterolateral hypokinesis with ef 50%. repeat tte was done on , which showed ef 50-55% and mild regional left ventricular systolic dysfunction with infero-lateral hypokinesis, not significantly different compared to the one from approximately 2 years ago. . # rhythm: patient was in sinus rhythm. toprol 25mg daily was continued. . # fen: patient received cardiac, heart-healthy diet, and he tolerated pos well. # ppx: patient was on sc heparin for dvt prophylaxis. . # code: full, confirmed on admission. . # comm: wife : (cell); (home) medications on admission: lipitor 80mg po qday plavix 75mg po qday toprol xl 25mg po qday asa 325mg po qday discharge disposition: home discharge diagnosis: primary diagnoses: - myocardial infarction - coronary artery disease discharge condition: afebrile, hemodynamically stable, chest pain free mental status:clear and coherent level of consciousness:alert and interactive activity status:ambulatory - independent discharge instructions: it was a pleasure to be involved in your care, mr. . you were admitted to after having chest pain. you underwent a cardiac catheterization and a blood clot was removed from the stents supplying blood to your heart. your plavix was changed to prasugrel 10mg by mouth once a day to help prevent a clot from reforming. it is very important that you take this medication, along with your aspirin, every day. please do not miss . as prasugrel was not available at your home pharmacy in today, a prescription was sent to cvs at in that you can pick up when you are discharged. your other medications have not been changed. please continue to take lipitor, toprol xl, and full dose aspirin (325mg daily). followup instructions: you need to see dr. , your cardiologist, within the next two weeks. we will try to make an appointment for you this weekend, and please call the cardiology office at ( on monday to confirm your appointment. if for any reason there is no appointment made for you over the weekend, please make one with the receptionist at that time. please see you primary care physician, . , v., within 1-2 weeks after discharge. please call to make an appointment. procedure: coronary arteriography using two catheters injection or infusion of platelet inhibitor left heart cardiac catheterization excision of lingual thyroid percutaneous transluminal coronary angioplasty [ptca] procedure on single vessel diagnoses: coronary atherosclerosis of native coronary artery percutaneous transluminal coronary angioplasty status old myocardial infarction acute myocardial infarction of other specified sites, initial episode of care chronic total occlusion of coronary artery 

Answer: The patient is high likely exposed to