Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: the patient is a 70-year-old woman with a past medical history significant for parkinson disease, shy- syndrome, and a history of recurrent syncope. four days prior to admission, the patient visited her primary care doctor who noted about a 12-pound weight gain over the prior week, along with increasing edema of the hands and feet. over this period, she denied any chest pain, shortness of breath, or palpitations. she subsequently witnessed a syncopal episode while sitting in a chair just following this visit. she electively admitted herself to for fluid management. the patient was found to have a junctional escape rhythm on electrocardiogram and was taken to the catheterization laboratory to have a pacemaker implanted. in the catheterization laboratory, the atria would not capture the pacemaker's rhythm, and the ventricle eventually captured the rhythm after some time. the patient arrived on the floor awake and slightly confused. her pulmonary arterial pressures were 60s/30s. we began aggressive fluid treatment for what we felt was intraventricular water depletion. subsequently, her hematocrit fell from 39 to 26. two units of fresh frozen plasma were ordered along with 2 units of packed red blood cells. her other past medical history is significant for increased cholesterol, 1 to 2+ aortic regurgitation, 3+ mitral regurgitation, and 1 to 2+ tricuspid regurgitation. medications on admission: (the medications that she is on at home included) 1. amantadine 100 mg by mouth three times per day. 2. atenolol 25 mg by mouth once per day. 3. florinef 0.1 mg by mouth every day. 4. levothyroxine 75 mcg by mouth once per day. 5. midodrine 20 mg by mouth three times per day. 6. pravachol 40 mg by mouth once per day. 7. sinemet. 8. tricor. 9. premarin. allergies: impression and plan: our impression at the time was for a woman with a 2-week history of weight gain, fluid retention, and syncope. the exact reason the patient initially went into heart block remained unclear; however, the effect of the heart block combined with 4+ mitral regurgitation likely produced a state where there was inadequate forward flow of her blood leading to increased peripheral edema. 1. cardiovascular system: in terms of her cardiovascular status, we decided to monitor her cardiac index and output via a swan-ganz catheter. we were also monitoring her pulmonary arterial pressure. 2. hematologic issues: in terms of her hematologic status, we were monitoring serial hematocrit levels and transfusing 2 units of packed red blood cells when necessary. given a possible disseminated intravascular coagulation picture, we were prepared to transfuse fresh frozen plasma. physical examination on presentation: on physical examination on admission, her blood pressure was 111/33, her heart rate was 80, respiratory rate was 14, and she was saturating 100% on 2 liters. her pupils were equal, round, and reactive to light. she had moist mucous membranes. her neck was supple. her chest was clear to auscultation bilaterally. there were decreased breath sounds at the bases. her cardiovascular examination demonstrated a regular rate and rhythm. there were no murmurs, rubs, or gallops. her abdomen was slightly obese. her bowel sounds were positive. her extremities were warm. she had tense 2+ edema of the hands and feet, possible acrocyanosis. pertinent laboratory values on presentation: her laboratory values on admission were significant for a white blood cell count of 11.4, hematocrit was 26.9, and platelets were 133. her creatinine was 2.1. her lactate dehydrogenase was 460. her inr was 1.8. her d-dimer level was greater than . pertinent radiology/imaging: a chest x-ray showed a normal heart, small bilateral pleural effusions, and patchy opacities in the left lower lobe. hospital course: on hospital day two, her hematocrit was found to be 23.7. a hematology consultation was obtained. steroids were given for a possible hemolytic anemia; 100 mg of methylprednisolone. she was transfused with 2 units of packed red blood cells. her liver enzymes were markedly elevated with an ast of 466 and an alt of 233. her lactate dehydrogenase was 419. the elevated liver enzymes were thought to be the result of decreased cardiac perfusion or shocked liver. in terms of her cardiovascular status, we continued to monitor her pulmonary arterial pressures. her cardiac indices remained approximately 4.5. with regarding to her renal function, her creatinine slightly decreased from 2.1 to 1.7. on day three in the coronary care unit, the patient obtained an upper extremity doppler which showed an old deep venous thrombosis which was cannulized. her systemic vascular resistance levels remained in the range. a nipride drip was started, and her systemic vascular resistance titrated down to 1300. on coronary care unit day four, the patient was given several doses of lasix 80 mg; putting out 100 cc to 150 cc of urine per hour. she was also started on a nesiritide drip, and her urine output increased to 200 cc to 300 cc per hour. her hematocrit remained stable in the low 30s. her cardiac output on coronary care unit day four, was 5.6. her index was 3.4. the decision was made to titrate the patient off the nipride drip and start her on a oral regimen of hydralazine. during the day, on hospital day four, the patient was transfusion from a nipride drip to a nitroglycerin drip. oral hydralazine was given. her indices remained around 1.4 and were slowly coming up. hydralazine 10 mg was given intravenously, and her blood pressure dropped to the 50s systolic. one liter of normal saline was given and two 500-cc boluses, and she was started on a dopamine drip for 30 minutes. her blood pressure responded appropriately, coming up to the 100s/60s. a chest x-ray was obtained at that time which showed increasing amounts of pulmonary edema. she was subsequently placed back on the nipride drip and continuous positive airway pressure with 10 of pressure support and 5 of positive end-expiratory pressure. on coronary care unit day five, the patient was back on the lasix and the nesiritide drip. her hematocrit remained stable in the low 30s. a cardiothoracic surgery consultation was obtained, and they decided that she was not a good surgical candidate secondary to her other underlying medical conditions. it was again decided that we would try to switch the patient from the nipride drip to an oral regimen of hydralazine, and 40 mg of oral hydralazine were given in the morning. at approximately 4:30 in the afternoon on the day of , the patient began dropping her blood pressures into the 60s systolic. the patient was placed on a dopamine drip and intravenous fluids running wide open. a short while later, the patient went into a ventricular tachycardia rhythm. her blood pressure dropped, and she became pulseless. she was shocked three times with increasing amounts of joules. she was given epinephrine times one. she went from a ventricular tachycardia to asystole after the third shock. rounds of epinephrine and atropine were given sequentially times two. bicarbonate and magnesium were also given. the patient was found to be pulseless electrical activity. the patient's family was outside the room witnessing the cardiac code and requested that we stop the code. the patient was pronounced dead at 4:58 p.m. on . condition at discharge: deceased. discharge status: not applicable. discharge diagnosis: cardiopulmonary failure. medications on discharge: not applicable. discharge instructions/followup: no applicable. , m.d. dictated by: medquist36 procedure: initial insertion of dual-chamber device initial insertion of transvenous leads [electrodes] into atrium and ventricle pulmonary artery wedge monitoring injection or infusion of nesiritide diagnoses: acidosis acute kidney failure, unspecified hyposmolality and/or hyponatremia mitral valve insufficiency and aortic valve insufficiency paroxysmal ventricular tachycardia paralysis agitans other specified cardiac dysrhythmias diseases of tricuspid valve other degenerative diseases of the basal ganglia 

Answer: The patient is high likely exposed to