Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: r sided weakness, n/v, aphasia major surgical or invasive procedure: none history of present illness: pt. is a 50 year old woman, listed in osh records as having no pmh, who is transferred for further management of ich. husband reports that pt. had been in her usoh all day today. she did not complain of any headache, and she was not confused or lethargic, or not herself. around 7:00 he was in the house and she came in from the back yard and said "i can't move my right arm. i think i'm having a stroke." she slumped down on a chair. he called ems right away. by the time they got there he felt that she was slurring her speech somewhat. per osh records, pt. was brought in to their ed at 7:30. ems described that they saw a r facial droop, r sided weakness, and aphasia on their exam. she vomited en route to the hospital. ot the hospital ct scan showed a large l bg hemorrhage with midline shift. she was intubated at 8:30 (with etomidate, lidocaine, succ) and given 50 g mannitol and 80 mg iv lasix. her exam is listed at pipoint pupils bilaterally, r facial droop, r hemiparesis, and aphasia. her initial bp was 240/150 and she was given labetalol 20 mg iv x 2, then started on a labetalol gtt at 1 mg/min for bp control. she was transferred here for further management. past medical history: none per husband, but 't seen a doctor in 15 years social history: lives with husband (retired ob/gyn), not working, no tobacco, drinks 2-3 glasses of wine/night family history: nc physical exam: t- bp- 186/101 hr- 67 rr- 12 o2sat- 99% on vent gen: lying in bed, intubated heent: nc/at, moist oral mucosa neck: supple, no carotid or vertebral bruit cv: rrr, nl s1 and s2, no murmurs/gallops/rubs lung: clear to auscultation bilaterally abd: +bs soft, nontender ext: no edema neurologic examination: mental status: intuted, doesn't open eyes to voice or sternal rub cranial nerves: pupils pinpoint, nr bilaterally. no eom with dolls or cold calorics. + corneal on l, absent on r. no gag. motor/sensation: no movement r arm with pain, withdraws l arm with pain, triple flexion in both legs with pain. reflexes: +2 and symmetric throughout. toes upgoing bilaterally pertinent results: ct head without contrast. large left basal ganglia intraparenchymal hemorrhage with intraventricular extension with associated midline shift, vasogenic edema and mass effect upon the pons and third ventricle. mr : large hemorrhage is again seen in the region of the left basal ganglia and left thalamus. this is associated with diffuse cerebral edema and effacement of sulci. again seen is approximately 1.4 cm rightward shift of normally midline structures. left uncal herniation is redemonstrated, along with cerebellar tonsillar herniation. there are punctate cerebellar hemorrhages that were not appreciated on ct. infarction is again seen in the left parietal lobe in the distribution of the mca. there is also infarction in the left occipital lobe in a watershed distribution. there is a region of infarction in the left basal ganglia which is not involved by the hemorrhage. there is a region of infarction in the right frontal lobe anterior watershed distribution and several small areas of infarction of the right deep white matter consistent with deep watershed distribution. impression: 1. multiple regions of infarction as described above. 2. large hemorrhage in the left basal ganglia and thalamus is redemonstrated. there are also punctate hemorrhages in the cerebellum. 3. subfalcine herniation with 1.4 cm shift to the right. left uncal herniation is again seen, along with cerebellar tonsillar herniation. 4. due to distortion of the basal ganglia and arterial contamination, the deep cerebral veins are not well visualized and thrombosis cannot be ruled out. it is possible that the left internal cerebral vein may be compressed or distorted by hemorrhage versus thrombosed. brief hospital course: the patient was admitted to the neuroicu. her exam deteriorated over the course of her stay. her right pupil became enlarged and unresponsive to light. based on the catastrauphic bleed and subsequent ischemia due to left mca compression by the bleed the patient's family decided to refocus the goals of care on comfort. the patient passed away shortly after discontinuing life prolonging measures. medications on admission: baby asa, mvi discharge medications: - discharge disposition: expired discharge diagnosis: hemorrhagic storke. discharge condition: expired discharge instructions: none followup instructions: none md, procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube intravascular imaging of intrathoracic vessels transfusion of platelets diagnoses: pneumonia, organism unspecified obstructive hydrocephalus unspecified essential hypertension intracerebral hemorrhage cerebral artery occlusion, unspecified with cerebral infarction aphasia 

Answer: The patient is high likely exposed to