Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: this is a 60 year old man with a past medical history significant for generalized tonic/clonic seizures since age 7 (has never been seizure free). he now presents from an outside hospital after seizure on and subsequently hitting his head on concrete and developing a subarachnoid hemorrhage. he states that his typical seizures come on without warning and he is not sure how long they last but is told that both his arms and legs shake and he recalls having bitten his tongue on occasion and loss of control of his bladder/bowel. he states that his seizures are all typically the same and he has about one per month but some are at night and he is not sure how often these occur (his wife has told him about them before.). he said that several days before admission, he was walking () down the street to his daughter's house and suddenly had one of these seizures, without warning, and hit his head on the concrete. he doesn't recall what happened after that but apparently someone called 911 and brought him to the hospital. the next thing he remembers is awakening in the hospital and he says that was his longest post ictal period. he does mention that on the day of the seizure, he had been having a sore throat and a dry cough. he had gone to his primary care physician who diagnosed him with a upper respiratory infection. since then, he has had some low grade temperatures but no congestion. he said that he has a throbbing headache in the bitemporal region currently, not relieved by tylenol and not accompanied by photophobia, nausea or vomiting. he has had this headache only since being hospitalized and it has moved from the bifrontal region to the bitemporal region over the last couple of days. he states that he has had no visual changes, no numbness or weakness and no other pain. he says that at home, he is on dilantin 100 mg three times a day, phenobarbital 120 mg q h.s. and zonegran 300 mg q h.s. per a note by dr. in , mr. has imbalance and osteoporosis from long term dilantin therapy and the plan at that time was to decrease the dilantin and gradually increase the zonegran. at the outside hospital, mr. was on dilantin 100 mg four times a day, phenobarbital 60 mg q h.s. and zonegran 200 mg q h.s. it is unclear how or why his medications were changed in this way. past medical history: seizure disorder. allergies: no known drug allergies. medications at home: 1. dilantin 100 mg three times a day. 2. phenobarbital 120 mg q h.s. 3. zonegran 300 mg q h.s. 4. multi-vitamins. 5. calcium. 6. vitamin d. social history: he is retired. audiovisual technician. he is married. he quit smoking ten years ago. no alcohol. family history: no seizures. physical examination: temperature 100.8; blood pressure 144/86; heart rate 70; oxygen saturation 96% on room air; weight 86.2 kg. general: medical examination unremarkable. neurologic: alert and oriented times three. speech fluent with no paraphasic errors. able to related detailed history of present illness with good memory of past and recent events. cranial nerves: pupils equal round and reactive to light. extraocular movements intact with end gaze nystagmus bilaterally. visual fields full to confrontation. facial sensation intact. he does have a lower motor neuron cranial nerve 7 palsy on the right. the tongue protrudes in the midline without fasciculation. sternocleidomastoid is intact bilaterally. shoulder shrug intact bilaterally. motor: normal bulk and tone throughout. no fasciculations. no pronator drift. full strength in upper and lower extremities bilaterally. reflexes symmetric throughout with toes down going bilaterally. sensation intact bilaterally to light touch, temperature, pin prick and vibration in all extremities. coordination: finger to nose, rapid alternating movements and fine finger movements intact bilaterally. gait deferred. laboratory data: on , white count was 7.7; hematocrit was 40.2. platelet count was 567. inr of 1.1. the patient did have a urinary tract infection transiently and grew group b strep; however, most recent urinalysis on was negative. cerebrospinal fluid showed 92 white cells, 139 red cells, positive xanthochromia. sodium of 135; potassium of 4.4; creatinine of 0.6; bun 16; chloride 103; bicarbonate 23; glucose 121; alt 32; ast 22; alkaline phosphatase of 115; total bilirubin of 0.2; lipase of 42; calcium 8.6; phosphate 3.0; magnesium 1.9. phenytoin was discontinued. cerebrospinal fluid protein was 63 and glucose was 66. blood cultures revealed no growth in several sets of blood culture. urine culture grew some coagulase negative staphylococcus 10,000 to 100,000 colonies and beta strep group b over 100,000 organisms per ml. cerebrospinal fluid was negative. ct on admission on showed subarachnoid in the intrapeduncular cistern; left sylvian fissure and a few frontal sulci. there is bifrontal contusion, left greater than right and probably subdural blood. there is a parenchymal hemorrhage measuring 10 by 19 mm in the left temporal lobe and a small, adjacent subdural hemorrhage along the course of the greater sphenoid . tentorium is dense and thickened, suggestive of a subdural hematoma under the tentorium. there is a slight rightward shift of structures but no sub false herniation. the ventricular size is not significantly changed since the prior study. there has been intraventricular blood, white matter differentiation is otherwise preserved. in the comparison images, extra axial hemorrhage is seen around the frontal lobes but the region of parenchymal hemorrhage is not present. also, the thickness of the tentorium on this study is more prominent than the study from four days prior at the outside hospital. therefore, there was evolution of the parenchymal hemorrhages with probable subdural under the tentorium and subarachnoid and intraventricular hemorrhage. magnetic resonance scan of the cervical spine showed at c3-4 intervertebral osteophyte formation and no canal stenosis. c4-5 osteophytic changes, especially on the right at c5-6; again osteophytic changes, resulting in mild canal narrowing and mild foraminal narrowing at this level as well, c5-6. there is left sided intravertebral osteophytic changes and moderate left sided foraminal narrowing. in addition, there may be a left sided lateral disc protrusion at this level. at c7-t1, there is a midline disc protrusion which is focal; however, it does not touch the cord. question artifact limits full evaluation for ligamentous injury but none is seen. repeat head ct most recently on showed no change, simply evolution of the hemorrhage. chest x-ray on showed only some left base atelectasis. magnetic resonance scan of the thoracic spine showed no acute fractures but compression fracture at t12 with a possible signal change to suggest an acute component. there are also compression fractures of l1 and l3 but no signal changes in the vertebral bodies to suggest acute fractures. the alignment is maintained at l3-l4. there is a mild paracentral disc protrusion, left greater than right, with mild indentation of the thecal sac. there is a right sided foraminal narrowing at this level; however, no impingement on the exiting nerve root sleeve. there are numerous compression fractures in the thoracic spine, most of which are old. hospital course: the patient was admitted to the neurology service. he received a head ct upon arrival and after the discovery that there was new blood on his head ct, he was briefly admitted to the intensive care unit. he remained stable there and repeat head ct on discharge was unchanged. he required no medications to control his blood pressure. during his hospital course, he became very lethargic and unmotivated which was attributed namely to the bifrontal hemorrhages; however, there was a point when he started having temperature spikes to 102 and once as high as 103. it was discovered that he was growing group b strep in his urine. infectious disease consult was called, who did not feel that the urine was causing this high of a fever but did suggest antibiotic regimen. the patient received a course of clindamycin and aztreonam and eventually the aztreonam was discontinued, as the patient stopped having fevers and there was no other evidence of infection found. the patient also continued to have severe head pain due to the blood, as well as due to his occipital skull fracture and perhaps also due to compression fractures in the spine. pain control remained an issue. we attempted not to place him on any ansaid. he was started on scheduled tylenol plus oxy-contin around the clock and prn oxycodone. he did well on that, yet became quite somnolent and, as this was decreased, he began to awake. pain consult was called and suggested continuing the oxy-contin 10 mg q. 8 hours with prn oxycodone. they also suggested starting clonidine .1 mg twice a day as well as vioxx 25 mg q. day. the vioxx should not have any affect on platelet function or cause any chance of increased hemorrhage. in respect to his seizures, the patient did have several seizures during the admission. initially, he was on dilantin and when his dilantin level dropped to 6.8, he was reloaded on the dilantin. he had no further seizures until we attempted to decrease the dilantin. the patient's outpatient neurologist who had wanted, prior to this hospitalization, to stop the dilantin all together, had increased his zonegran and so this was done as an inpatient. the patient was gradually tapered off the dilantin and increased on the zonegran. the patient's most recent seizure was a very brief seizure on in the evening, generalized tonic/clonic, lasting for less than 30 seconds, which quickly resolved without incident. the patient was given just prn ativan for seizures at this point, while waiting for his blood levels of zonegran to rise. the patient was also kept on eeg 24 hour monitoring for several days. the eeg showed a push-button event on where there was a generalized fashion and progression and prominent generalized delta frequency slowing; no clear epileptiform changes that appeared to be a generalized seizure, although there were no frank spike discharge seen at the time of the push-button event. the background was mildly slow, suggestive of encephalopathy. in addition, there was right more than left bifrontal mixed delta and beta frequency slowing seen. there were some sharp features in the right frontal region as well. the patient also had a lower motor neuron cranial nerve 7 palsy. the etiology of thought to be secondary to his trauma and skull fracture. however, the patient did seem to sustain a corneal abrasion perhaps and he was placed on lacrilube ointment was well as eye patch for his right eye. the cranial nerve 7 palsy did not change throughout the admission. the patient also seemed to have no other focal neurologic deficits. the patient was also seen by physical therapy who recommended acute rehabilitation. the patient was otherwise well and says that his pain is now much better controlled. discharge medications: 1. colace 100 mg p.o. twice a day. 2. dulcolax 10 mg p.o. q. day prn. 3. calcium carbonate 500 mg three times a day. 4. vitamin d 400 i.u. p.o. q. day. 5. phenobarbital 120 mg p.o. q h.s. 6. salt tabs, one gram p.o. three times a day. 7. senna one tablet p.o. twice a day. 8. protonix 40 mg p.o. q. day. 9. polyvinyl alcohol 1.4% eye drops, one to two drops prn o.d. 10. oxycodone 10 to 15 mg q. four to six hours as needed for pain. 11. zonegran 500 mg p.o. q h.s. 12. tylenol 650 mg p.o. q. four hours. 13. oxycodone 10 mg p.o. q. eight hours. 14. erythromycin 5 mg per gram ointment, one drop o.d. four times a day. 15. clonidine 0.1 mg p.o. twice a day. 16. vioxx 25 mg p.o. q. day. 17. artificial tear ointment, one drop o.d. prn. 18. ativan 2 mg intravenous prn for seizure more than two minutes and please keep an eye patch on his right eye at all times. follow-up: the patient has follow-up appointments with in the neurology office on at 11:15 a.m. and with dr. on at 11:15 a.m. the patient is discharged in good condition to rehabilitation. , md dictated by: medquist36 d: 03:26 t: 15:52 job#: procedure: spinal tap incision of lung diagnoses: pneumonitis due to inhalation of food or vomitus osteoporosis, unspecified closed fracture of dorsal [thoracic] vertebra without mention of spinal cord injury epilepsy, unspecified, without mention of intractable epilepsy other disorders of neurohypophysis superficial injury of cornea fall from other slipping, tripping, or stumbling closed fractures involving skull or face with other bones with subarachnoid, subdural, and extradural hemorrhage, with loss of consciousness of unspecified duration 

Answer: The patient is high likely exposed to