Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: sob major surgical or invasive procedure: 1. pci 2. iabp 3. s/p right sfa patching and thrombectomy 4. swan-ganz catheter right groin and right ij history of present illness: 66 yom p/w 10 days increasing sob and le edema. in ed, found to have st elevations (v2-v4) on ekg. acs regimen started. dropped pressure with ntg was then placed on levophed and dopamine d/t him being in cardiogenic shock. . in ed, went into svt on dopamine was cardioverted and intubated with laceration of the soft palate. was also placed on insulin drip for bs 900's. . in cath lab, saw a lot of disease but did not intervene since no culprit lesion was identified. trop 3.73. echo showed global hypokinesis, iabp implanted. dopamine was changed to levophed gtt. . while on the ccu floor, patient was switched to dobutamine and dopamine with the plan to eventually add nitro gtt. diuresis not option given dka (+ketoacids in urine) and elevated cr. sugars improved from 400's to 300's. anticoagulatns were held and ent repaired soft palate lac. right foot became noticeabley cold and cyanotic. peripheral pulses had been dopplerable however became undetectable by doppler on right up to the proximal groin, femoral pulse still intact. decision was made to remove balloon pump. heparin bolus and gtt started overnight. vascular surgery consulted. . medications in ed: insulin human regular, heparin sodium 5000 units, eptifibatide 75mg, dopamine 400mg, clopidogrel bisulfate 75mg, morphine sulfate 4mg, nitroglycerin, lidocaine jelly 2%, norepinephrine, midazolam hcl, atropine sulfate, epinephrine-sodium chloride past medical history: 1. diabetes 2. hypercholesterolemia 3. hypertension social history: estranged from wife but still keeps in touch with her regularly and attends family functions. family history: nc physical exam: vs t 98.6 bp 100/70 hr 65 rr 20 spo2 100 20x650 fi02 1.00 peep 5 gen: intubated, sedated heent: perrl, blood oozing from mouth, et tube in mouth cv: nl s1 s2 rrr no m/r/g pulm: lungs clear to auscultation bilaterally, no wheezes abd: soft nontender +bs groin: no hematoma ext: 3+ to 4+ edema, dopplerable dpp 1+ pertinent results: ph 7.17/32/340/12 hgb:13.6 calchct:41 o2sat: 99 ph 7.15/37/43/14 glu:907 . na 129 cl 86 bun 39 gluc 858 agap=36 k 5.1 hco3 12 creat 2.7 ck: 712 mb: 34 mbi: 4.8 trop-*t*: 3.73 ca: 9.6 mg: 2.5 p: 8.4 wbc 13.9 hgb 15.0 plat 260 hct 46.8 n:89.7 band:0 l:6.8 m:3.3 e:0 bas:0.1 pt: 14.2 ptt: 25.8 inr: 1.4 . 6:57 pm sputum source: endotracheal. **final report ** gram stain (final ): >25 pmns and <10 epithelial cells/100x field. 4+ (>10 per 1000x field): gram positive cocci. in pairs and clusters. 2+ (1-5 per 1000x field): gram positive rod(s). respiratory culture (final ): sparse growth oropharyngeal flora. staph aureus coag +. moderate growth. sensitivities performed on culture # . . imaging: echo there is severe global left ventricular hypokinesis with only modest sparing of the inferior wall. overall left ventricular systolic function is severely depressed (ef 10-15%). mild to moderate (+) mitral regurgitation is seen. there is no pericardial effusion. . : cath results: - left dominant anatomy with three vessel disease with a tubular 50% lmca lesion. the lad was diffusely diseased proximally to occluded totally just after the s1. there is some collateralisation of the lad. the cx is dominant with a distal 80% lesion. the rca is non dominant and had proximal critical stenosis. - there was severe hypokinesia in the inferio wall. the rest of the segments and the apex were akinetic. - hemodynamics showed central aortic hypotnesion while intubated and on vasopressors. the filling pressures were markedly elevated with lvedp at 40mmhg, pcwp could not be obtained (rvedp was 28 mm hg) ci was depressed at 1.4 chest (portable ap) 7:55 am findings: the endotracheal tube continues to be 5.5 cm above the carina. the ng tube tip is in the stomach. there is a right ij line with tip in the svc. there is dense retrocardiac opacity that could be due to volume loss/infiltrate/effusion. there is increased hazy opacity projecting over the right lower lung that likely represents an effusion layering posteriorly although an infiltrate can also be present. compared to the prior film, there has been some interval clearing of the interstitial edema. brief hospital course: 66 yom presents in cardiogenic shock s/p acute mi and dka. cath showed 3vd and echo showed ef of %. intubated and iabp placed. had decreased perfusion to r le, removed iabp to or for patching of sfa and thrombectomy. patient subsequently had runs of afib w/ rvr and then vf multiple times requiring defibrillation. patient loaded on iv amiodarone and continuous drip. went into vfib and was dnr and passed away. . # r le decreased perfusion: iabp was discontinued and taken to or for sfa patch and thrombectomy. now with dopplerable dorsalis pedis pulse. on heparin drip perioperatively then discontinued. . # cardiac: ischemic: cath showed diffuse disease no clear stentable target. trop-*t* peaked 11.28. cardiac were consulted and would consider ht transplant when improved. pump: pt w/severely depressed global lv systolic function with regional wall motion abnormalities. upon presentation, difficult to treat diabetic ketoacidosis because requires fluid resuscitation however in setting of cardiogenic shock, we were unable to aggressively increase intravascular volume. started on dopamine and dobutamine drip. patient returned from pacu on levophed, dopamine and dobutamine drips. patient's dka resolved and a lasix drip was started because of overload, elevated pa diastolic pressure (33) and cvp(17). patient was weaned off dopamine and dobutamine and sedation. patient was continued on lasix drip and added aldactone and diuryl to improve diuresis since he was net 4-5 liters positive over length of stay. attempted to wean patient weaned off levophed however still required some pressor support. . rhythm: patient had runs of afib with rvr to 140s. attempt at cardioversion x 1 at 200j but did not convert. pt was loaded w/1g amiodarone gtt, started amio 400mg po bid and 1g digoxin, start digoxin 0.125mg po qd in am . patient then required 5 defibrillated shocks 360j for pulseless vt that would not stay in sinus rhythm. he was loaded with 300mg iv amiodarone then continued on 1mg/min amio drip for 6hrs then 0.5mg/min indefinitely. digoxin was discontinued. . # acidosis: combination of decreased perfusion (lactic acidosis) and diabetic ketoacidosis with now closing gap. used iabp to increase cardiac perfusion and decrease preload, now discontinued d/t difficulties with r le perfusion. tight insulin control with insulin gtt and boluses as needed to decrease ketoacids. started d5w and nph 20u after fs 160 to prevent hypoglycemia and widely varying bs's. anion gap acidosis resolved. continued insulin gtt and tube feeds as tolerated. . # id: persistent low grade fevers concerning for infection. on vanco/levo day #, broad-spectrum antibiotics in setting of possible sepsis. flagyl d/c on . changed coverage to hospital acquired pna -> vanco/zosyn day 2, dc levo (). vanco dosed q24 given decrease crcl. dc'd swan right neck and send tip for culture -> triple lumen placed. sent surveillance cultures daily/cis. . # mouth laceration: s/p packing of lac x3days, packing was removed and required re-packing for another 3 days. ent was following. . # intubation: continued intubation and continued to wean tidal volume and fio2 per serial abgs. . # code: wife changed patient from full code to dnr on which was documented. medications on admission: 1. warfarin 2-3mg 2. avandia 4mg 3. zocar 20mg 4. gemfibrozil 600mg 5. lisinopril 20mg 6. furosemide 20mg 7. glipizide 10mg 8. atenolol 50mg discharge disposition: expired discharge diagnosis: 1. acute mi/cardiogenic shock 2. r femoral vein thrombus 3. dka 4. vfib discharge condition: deceased discharge instructions: none followup instructions: none procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more combined right and left heart cardiac catheterization coronary arteriography using two catheters angiocardiography of left heart structures insertion of endotracheal tube enteral infusion of concentrated nutritional substances other electric countershock of heart aortography implant of pulsation balloon incision of vessel, lower limb arteries pulmonary artery wedge monitoring repair of blood vessel with synthetic patch graft infusion of vasopressor agent diagnoses: pneumonia, organism unspecified acidosis subendocardial infarction, initial episode of care coronary atherosclerosis of native coronary artery unspecified essential hypertension accidental puncture or laceration during a procedure, not elsewhere classified paroxysmal ventricular tachycardia other specified procedures as the cause of abnormal reaction of patient, or of later complication, without mention of misadventure at time of procedure acute respiratory failure cardiac arrest cardiogenic shock diabetes with ketoacidosis, type i [juvenile type], not stated as uncontrolled other complications due to other vascular device, implant, and graft cardiac catheterization as the cause of abnormal reaction of patient, or of later complication, without mention of misadventure at time of procedure acute diastolic heart failure open wound of palate, without mention of complication 

Answer: The patient is high likely exposed to