Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: morphine attending: chief complaint: tylenol overdose major surgical or invasive procedure: none. history of present illness: -- per admitting micu resident -- 48 f with a history of depression, psa, migraines who has been abusing pain medications for several years. the history is obtained from her significant other as patient is encephalopathic. per his report patient took 100 of his percocets and took those throughout the weekend and to the best of his knowledge her last ingestion was monday. patient has a recent t11 compression fracture suffered at work which triggered increased use. denies suicide attempt. was taken to er by after she became progressivelly confused over the weekend. she was started on nac after a tylenol level of 52 at 1530. ast 2725, alt 2620, bili 2.3. cth at osh was per report negative. in the icu patient states her name but otherwise confused. abg in the er with ph 7.47. around midnight the patient had coffee ground emesis which was guiaic positive. ngt was placed and was lavaged with 2500cc, would clear but subsequently marroon liquid would be suctioned. liver fellow was present and given hd stability decision to defer endoscopy until am was made. past medical history: depression ps abuse migraines social history: smokes unknown quantity, no etoh per significant other. illicit drug use other than prescription meds. works as cna at a nursing home. family history: non-contributory physical exam: -- on arrival to floor -- vs: 99/98.7 87 bp 87-132/54-80 12 98-100% ra gen: somnolent but arousable, oriented x 3 cv: rrr s mrg resp: cta on limited exam abd: ttp on ruq > luq, no rebound, guarding, or rigidity. hyperactive bs, no distention. ext: wwp, 2+ pulses, no c/c/e neuro: could not assess asterixis as limited patient cooperativity. pertinent results: 05:23am blood wbc-6.1 rbc-2.97* hgb-8.7* hct-26.4* mcv-89 mch-29.4 mchc-33.0 rdw-18.2* plt ct-169 05:23am blood glucose-84 urean-8 creat-0.5 na-142 k-3.8 cl-107 hco3-28 angap-11 05:23am blood alt-165* ast-39 alkphos-114* totbili-0.6 05:23am blood albumin-3.0* calcium-8.2* phos-3.8 mg-1.9 05:22pm blood cryoglb-negative 07:45pm blood asa-neg ethanol-neg acetmnp-31.7* bnzodzp-pos barbitr-neg tricycl-neg 10:40pm blood acetmnp-21.7 03:44am blood acetmnp-13.3 11:38am blood acetmnp-5.5* 05:22pm blood acetmnp-neg 03:01am blood acetmnp-neg 05:22pm blood rheufac-13 10:40pm blood hbsag-negative hbsab-negative hbcab-positive hav ab-positive 11:38am blood tsh-0.30 05:22pm blood rheufac-13 10:40pm blood hcv ab-positive* 08:10pm urine blood-neg nitrite-pos protein-neg glucose-neg ketone-150 bilirub-neg urobiln-8* ph-6.5 leuks-neg urine culture (final ): escherichia coli. >100,000 organisms/ml.. presumptive identification. sensitivities: mic expressed in mcg/ml _________________________________________________________ escherichia coli | ampicillin------------ <=2 s ampicillin/sulbactam-- <=2 s cefazolin------------- <=4 s cefepime-------------- <=1 s ceftazidime----------- <=1 s ceftriaxone----------- <=1 s ciprofloxacin---------<=0.25 s gentamicin------------ <=1 s meropenem-------------<=0.25 s nitrofurantoin-------- <=16 s piperacillin/tazo----- <=4 s tobramycin------------ <=1 s trimethoprim/sulfa---- <=1 s hcv viral load (final ): this is a corrected report ( at 3pm). 1,370,000 iu/ml. - virus vca-igg ab (final ): positive by eia. - virus ebna igg ab (final ): positive by eia. - virus vca-igm ab (final ): negative <1:10 by ifa. cmv igg antibody (final ): positive for cmv igg antibody by eia. 183 au/ml. reference range: negative < 4 au/ml, positive >= 6 au/ml. cmv igm antibody (final ): negative for cmv igm antibody by eia. ruq u/s: 1. unremarkable ultrasound of the liver. 2. a 1.1 cm predominantly hyperechoic lesion incidentally noted within the upper pole of right renal cortex with features most compatible with angiomyolipoma. eeg: impression: this is an abnormal routine eeg due to a discontinuous background consisting of seconds of diffuse high voltage delta frequency slowing and triphasic waves alternating with equal periods of global attenuation approaching a burst suppression pattern. there was also 14 and 6 hz positive spikes, which is a variant pattern reported in severe hepatic failure. this eeg pattern is indicative of a severe diffuse encephalopathy. tte: due to suboptimal technical quality, a focal wall motion abnormality cannot be fully excluded. overall left ventricular systolic function is normal (lvef>55%). right ventricular chamber size and free wall motion are normal. ct head: no intracranial hemorrhage. mild diffuse cerebral edema without herniation. given patient's intoxication history, the differential includes reversible hepatic encephalopathy versus progressive hypoxic-ischemic encephalopathy. recommend mr to distinguish between these two etiologies. mr/mra head 1. subtle t2 hyperintensity and mild diffusion restriction in the bilateral basal ganglia and central thalami. diffuse mild cortical edema. these findings are suggestive of metabolic encephalopathy. 2. intact intracranial circulation, with possible infundibulum at the origin of the right pcomm. brief hospital course: briefly, the patient presented with ams and was started on nac given a h/o ingestion of "100 percocets" per patient's so. tylenol level found to be 52 in ed with ast/alt + and bili 2.3. ct head @ osh negative. patient was admitted to micu on nac drip and had a hospital course complicated by coffee ground emesis leading to ngt placement. with lavage the patient cleared, but she did require blood transfusion. began to develop decerebrate posturing, and a ct head showed e/o cerebral edema. eeg showed evidence of diffuse encephalopathy with no epileptiform activity. neuro recommended mannitol and hyperventilation, after which patient's neuro exam improved. mri of the head was consistent with metabolic encephalopathy. she was continued on nac/lactulose and had interval improvement in awareness, mental status. incidentally, pt was found to have uti and completed a 3 day course of macrobid. at time of transfer, patient was oriented x 3 but still significantly drowsy. . she was continued on lactulose on the floor and her mental status cleared. as her liver enzymes were returning to normal, the n-acetylcysteine drip was discontinued. . the patient was placed under section 12 by psychiatry with plan to transfer to inpatient psychiatry unit after discharge. at time of discharge, patient is medically stable with evidence of resolving liver damage warranting no further inpatient workup or management. her mental status has cleared with no current evidence of ongoing encephalopathy. her upper gi bleed earlier in her hospital course spontaneously resolved and her hematocrit remained stable. she will need to continue a proton-pump inhibitor for at least 4-8 weeks. as an outpatient, she will need an egd to further work up the source of the bleed, but as there is no evidence of ongoing bleeding for several days, this does not warrant inpatient workup. . she was also noted to be hep c positive during this admission with an viral load of 1,370,000 iu/ml. this should be followed up as an outpatient. medications on admission: trazodone 100mg methadone 10mg po bid valium percocet abuse discharge medications: 1. pantoprazole 40 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po q12h (every 12 hours). discharge disposition: extended care discharge diagnosis: primary diagnosis: 1. tylenol-induced hepatitis 2. substance abuse 3. suicidal ideations 4. metabolic encephalopathy discharge condition: mental status:clear and coherent level of consciousness:alert and interactive activity status:ambulatory - independent discharge instructions: you were seen at for altered mental status thought due to an overdose. you were placed on a detoxification regimen and admitted to the intensive care unit. you had evidence of brain dysfunction, but this resolved after several days in the unit. you also had a bleed from your stomach which spontaneously resolved, but you required a blood transfusion. the following medications were changed; added pantoprazole to treat you after a history of bleeding from your stomach followup instructions: please follow up with your primary care physician, . , four weeks after you are discharged. you can contact his office at . procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube diagnoses: urinary tract infection, site not specified chronic hepatitis c without mention of hepatic coma acute and subacute necrosis of liver other, mixed, or unspecified drug abuse, unspecified cerebral edema hematemesis hepatic encephalopathy poisoning by aromatic analgesics, not elsewhere classified migraine, unspecified, without mention of intractable migraine without mention of status migrainosus major depressive affective disorder, recurrent episode, unspecified accidental poisoning by aromatic analgesics, not elsewhere classified 

Answer: The patient is high likely exposed to