Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins / aspirin attending: chief complaint: etoh withdrawal major surgical or invasive procedure: midline placed by ir intubation ng tube placement chest tube placement central line placement history of present illness: the patient is a 62 year old man with a history of hypertension, seizure disorder, and etoh abuse with history of withdrawl seizures, who presented with an etoh withdrawal seizure and subsequently developed left gaze deviation and right sided weakness, called as code stroke. the patient was admitted on after being found on his porch with an unwitnessed seizure with bowel and bladder incontinence. in the ed, vitals were t 97.9 p 96 bp 180/90 r 32 o2 sat 97% on 2l. he was intoxicated with etoh level 211. he was given ativan 2 mg iv x2 and 30-40 of diazepam iv. head ct on admission showed no hemorrhage. past medical history: htn emphysema childhood seizure disorder etoh abuse with h/o withdrawal seizures and dts multiple knee surgeries s/p truck accident in s/p splenectomy d/t truck accident d/t truck accident in s/p right hip fracture d/t truck accident in adrenal adenoma noted on ct during admission in liver lesion noted on ct during admission in history of unremoved ivc filter social history: heavy etoh, tobacco: 0.5 ppd family history: unknown physical exam: vitals: t: 99.0 hr: 105 bp: 142/108 r: 23 96%2l o2: general: dishevled, lethargic, arousable, alert and oriented x3, speaking slowly heent: macroglossia with whitish plaques. peerl, sclera anicteric, mmm neck: supple, jvp flat, no lad lungs: clear to auscultation bilaterally, without wheezes, rales, ronchi cv: tahcycardic, s1, s2, no m/r/g abdomen: soft, obese, non-tender, non-distended, bowel sounds present, no rebound tenderness or guarding, no organomegaly ext: warm, well perfused, 2+ pulses, no clubbing, cyanosis or edema pertinent results: labs: 11:47pm blood wbc-13.4*# rbc-4.89# hgb-14.6 hct-43.8 mcv-90# mch-29.8# mchc-33.3 rdw-15.6* plt ct-310 10:45am blood wbc-6.5 rbc-3.61* hgb-11.0* hct-32.3* mcv-89 mch-30.5 mchc-34.1 rdw-16.5* plt ct-464* 11:47pm blood neuts-86.3* lymphs-8.9* monos-4.1 eos-0.3 baso-0.3 06:00am blood neuts-67.9 lymphs-21.3 monos-7.3 eos-3.0 baso-0.6 01:15pm blood hypochr-normal anisocy-1+ poiklo-1+ macrocy-1+ microcy-normal polychr-normal ovalocy-occasional target-occasional tear dr 10:13am blood pt-12.5 ptt-32.5 inr(pt)-1.1 09:05am blood lupus-neg 09:45pm blood at iii-103 protsfn-60 09:45pm blood aca igg-41.9* aca igm-39.0* 10:13am blood protcfn-179* 09:45pm blood fibrino-554* 10:13am blood fibrino-649* 11:47pm blood glucose-159* urean-6 creat-0.9 na-144 k-3.9 cl-104 hco3-23 angap-21* 10:45am blood glucose-165* urean-15 creat-0.7 na-140 k-3.9 cl-107 hco3-24 angap-13 11:47pm blood alt-18 ast-16 alkphos-145* totbili-0.3 06:00am blood alt-20 ast-35 alkphos-92 amylase-43 totbili-0.2 02:26pm blood lipase-20 06:00am blood lipase-22 03:16am blood albumin-3.1* calcium-9.2 phos-3.8 mg-2.1 iron-28* cholest-189 03:16am blood triglyc-173* hdl-34 chol/hd-5.6 ldlcalc-120 03:16am blood %hba1c-5.5 03:22am blood hapto-240* 03:16am blood caltibc-199* ferritn-223 trf-153* 02:26pm blood tsh-2.8 10:13am blood cea-3.0 afp-2.1 10:45am blood phenyto-6.0* 11:47pm blood asa-neg ethanol-211* acetmnp-neg bnzodzp-pos barbitr-neg tricycl-neg factor v leiden () negative prothrombin (): negative 09:03am urine color-yellow appear-hazy sp -1.019 09:03am urine blood-mod nitrite-neg protein-30 glucose-neg ketone-neg bilirub-neg urobiln-neg ph-7.5 leuks-neg 11:47pm urine bnzodzp-pos barbitr-neg opiates-neg cocaine-neg amphetm-neg mthdone-neg urine cytology (): negative for malignant cells micro: blood cx (, ): no growth urine cx (, ): no growth sputum cx (): sparse growth oropharyngeal flora. sputum cx (): sparse growth oropharyngeal flora. urine cx (): pansensitive enterococcus sp catheter tip cx (): staphylococcus, coagulase negative. >15 colonies. enterococcus sp.. >15 colonies. imaging: cxr (): impression: no acute intrathoracic process. ct head (): impression: no hemorrhage. no change from , . note added at attending review: the maxillary sinuses are incompletely imaged, and no fluid is detected in the included portions of them. however, there is an apparent inferior and medial left orbital blow out fracture, of uncertain chronicity. elements of it appear new since the study of , but the absence of ethmoidal or maxillary hemorrhage argues against an acute fracture. bilateral lenis (): impression: essentially occlusive thrombus throughout bilateral common femoral, superficial femoral, and popliteal veins as well as greater saphenous vein. abdomen film (): impression: filter in lower ivc. ct head (): impression: 1. extensive new intraparenchymal hemorrhage within the left posterior parietal cortex since , extending into the subdural space along with the falx and tentorium cerebri. no intraventricular extension or midline shift. no evidence of herniation. 2. unchanged old right thalamic lacunar infarct. cxr (): the new intended right subclavian line turns abruptly lateral at the upper margin of the right clavicle. the position of the tip is not consistent with a central intravenous location. it could be in a small vein, or a pleural space. in the absence of pulmonary hematoma it is unlikely to be in the lung, though the new right pneumothorax projecting over the pleural sulcus and a small right pleural effusion suggest that the lung may have been engaged during the line placement. et tube in standard placement, nasogastric tube ends in the distal stomach. left lung grossly clear. leftward mediastinal shift probably a function of new right pneumothorax and pleural effusion. tte (): the left ventricle is not well seen. due to suboptimal technical quality, a focal wall motion abnormality cannot be fully excluded. overall left ventricular systolic function is normal (lvef>55%). right ventricular chamber size and free wall motion are normal. the number of aortic valve leaflets cannot be determined. the aortic valve is not well seen. no masses or vegetations are seen on the aortic valve, but cannot be fully excluded due to suboptimal image quality. there is no aortic valve stenosis. the mitral valve leaflets are mildly thickened. the mitral valve leaflets are not well seen. no masses or vegetations are seen on the mitral valve, but cannot be fully excluded due to suboptimal image quality. impression: poor technical quality due to poor echo windows/intubation. no cardiac source of embolism seen. left ventricular function is probably normal, a focal wall motion abnormality cannot be fully excluded. the right ventricle has normal size and function. no pathologic valvular abnormality seen. carotid series (): impression: minimal focal scattered calcific plaque, no associated ica or cca stenosis however. eeg (): impression: this 24-hour video eeg telemetry captured no pushbutton activations or electrographic seizures. no interictal epileptiform discharges were seen. the background did reach a normal alpha frequency during wakefulness, although through most of the day's recording slower background activity was suggestive of excessive drowsiness or a mild encephalopathy. mri/mrv (): impression: 1. hemorrhagic infarction of the left superior parietal lobe and small infarction of the right parietal area secondary to thrmbosed meduallry veins. 2. diffuse thrombosis of the sinuses including transverse, sigmoid, straight, superior sagittal. multiple cortical veins also are thrombosed. 3. opacification of mastoid sinuses bilaterally. lue u/s (): impression: patency of the subclavian, axillary, and brachial veins. the internal jugular vein was not identified, nor was the cephalic vein. imaging of the basilic vein could not be performed due to overlying bandaging material. ct/ctv head (): impression: 1. unchanged large hemorrhagic infarction in the left frontal and parietal lobes. 2. recanalization of the straight sinus, right transverse sinus, right sigmoid sinus, and the imaged upper portion of the right internal jugular vein. 3. the posterior half of the superior sagittal sinus, the lateral aspect of the left transverse sinus, the left sigmoid sinus, and the imaged upper portion of the left internal jugular vein remain largely occluded with small amount of recanalization. ct torso (): impression: 1. persistent, but somewhat less prominent hypoattenuating left lateral liver lesion probably indicating focal fat. mri can confirm this.. 2. additional hypoattenuating liver lesion not appreciated on prior non- contrast study in segments ii/iii of the liver anteriorly, also somewhat concerning but probably representing focal fat. again mri may be useful for further evaluation. 3. loculated-appearing right pleural effusion, with subsegmental atelectasis. subsegmental atelectasis on the left. 4. satisfactory position of ivc filter, right picc line, and gastrostomy tube. filter has central calcification indicating old, calcified clot. 5. thick-walled appearing bladder, for which correlation with any available cystoscopy may be helpful. 6.carotid sheath lymph node. eeg (): impression: this 24 hour video eeg telemetry captured 4 pushbutton activations without clear electrographic correlate. the telemetry during these recordings was obscurred given significant lead artifact. on video, the patient appears to have focal tremor or shaking of the left hand and arm. one automated seizure detection captured focal slowing over the left central and parietal leads with spread to the left hemispheric leads. this was correlated with a brief episode of right leg tonic clonic movements, suggestive of an electrographic seizure. automated and routine sampling also was showed significant slowing predominantly over the left central and parietal leads, consistent with a focal area of subcortical dysfunction and potential seizure focus. eeg (): impression: this telemetry captured 2 events with rhythmic activity seen over the right hemisphere and one event with generalized rhythmic activity. the first 2 events likely represent seizure activity accompanied by the behavioral changes described above; however, the eeg was significantly obscured by artifacts. the last event is consistent with a seizure with more generalized activity during which the patient had head and mouth tremor. overall, the eeg during these events were obscured by movement artifacts. the background activity was, most of the time, slow and disorganized suggestive of encephalopathy. interictal sharp and slow wave complexes were seen in the right frontal region. eeg (): impression: this telemetry captured no pushbutton activations, but did capture numerous periods of rhythmic theta activity seen broadly over the right hemisphere, and with extension to the left frontal region as well. this activity correlated with rhythmic tremor of the left hand, arm or shoulder, and impaired responsiveness. these findings are consistent with ongoing seizure activity. the background was for a substanial period of time obscured by artifacts but showed a slow and disorganized background suggestive of encephalopathy. eeg (): impression: this telemetry captured 4 pushbutton activations for episodes of left arm shaking, sometimes accompanied by unresponsiveness and rhythmic movements of the mouth, with no clear eeg correlate. there was one event when the patient turned to the left and the eeg showed semi-rhythmic right frontal delta frequency activity for about 50 seconds. the background activity showed focal slowing in the frontal regions bilaterally, right temporal region, and bursts of generalized slowing were also seen. interictal sharp waves were seen in the right temporal region. eeg (): impression: this is an abnormal portable routine eeg in the awake state due to possible sharp features seen in the right frontal area suggestive of cortical irritability in that regiona. however most of the recording was obscured by the artifacts. in addition, the background activity was slower on the left suggestive of significant structural subcortical abnormality in the left hemisphere. ct head (): impression: 1. no acute intracranial hemorrhage. 2. continued evolution of left parietal infarct . cxr (): upright ap chest: a right picc is seen, with tip overlying the subclavian/axillary vein. the tip has retracted several centimeters compared to the prior study where it was located in the distal svc. cardiac and mediastinal contours are stable. there is persistent consolidation in the right middle lobe, as before. patchy opacities are also seen in the left base. the upper lungs remain clear. there is a small right pleural effusion, unchanged. no pneumothorax. a gastrostomy tube is noted in the right upper quadrant. there is evidence of remote/healed fractures of the right proximal humerus and a right lower rib. impression: 1. right picc retraction, now overlying the subclavian/axillary vein. 2. persistent right middle lobe and left basilar opacity, possibly reflecting aspiration or infection. 3. unchanged right pleural effusion. brief hospital course: the patient was admitted on after being found on his porch with an unwitnessed seizure with bowel and bladder incontinence. in the ed, vitals were t 97.9 p 96 bp 180/90 r 32 o2 sat 97% on 2l. he was intoxicated with etoh level 211. he had minimal access (r hip iv) and had b femoral line attempts unsuccessfully for ? clots. a neck cvl could not be placed due to patient combativeness. he was given ativan 2 mg iv x2 and 30-40 of diazepam iv. ct head was negative for acute bleed but did show an orbital fracture that was felt to more likely chronic without surrounding hemorrhage. he was initially admitted to the micu. upon admission to the micu, he became "unresponsive for approximately 8 minutes while experiencing bilateral upper extremity tonic-clonic jerking." he was given ativan 2mg iv x2. his ciwa was initially 21. he was started on keppra 1000 mg (? what his home dose is). he had another episode of "tonic ue and eye rolling" on or , for which he was given ativan 2 mg im with ressolution in 10 minutes. he was also treated with valium ciwa. he was transferred to the floor on . he was called out to the floor on . on the floor, he was found to have extensive b le clots. he was also discovered to have an infrarenal ivc filter in place. he was not anticoagulated given suspicion that clots were chronic. he had recurrent seizures on the floor treated with iv ativan. on , patient noted to have further seizing with gaze preference, nystagmus, and was unresponsive with flaccid paralysis of rue. at that time he also had respiratory arrest. ct head at that time showed new intraparenchymal hemorrhage within the left posterior parietal cortex and code stroke was called. neurology felt this was most c/w an mca ischemic stroke with hemorrhagic transformation. on , the patient was noted to be more somnolent than before, but was otherwise conversant with the house officer team. the patient was last noted to be normal at approximately 3:00pm on by his nurse. at 4:45 pm, the medicine attending evaluated the patient and found him to be less responsive with left gaze preference. they were initially concerned for seizure, and gave him ativan 1 mg im x2. upon re-evalution he was concerned that the patient had developed a right hemiparesis with flaccid tone in the rue. a code stroke was called at 16:52, and neurology arrived at approximately 17:00. neurology: the l mca territory was likely from hemorrhagic conversion of ischemic stroke or from venous sinus thrombosis. no embolic source identified on echo. carotid u/s unrevealing. repeat ct scans showed stability of his hemorrhage with expected progression of infarction. he worked with pt and ot and improved during his hospitalization. speech improved. he was cleared for a modified diet on swallow evaluation. deficits include expressive aphasia, rue weakness with intermittent strength upto anti-gravity, and l gaze preference and intermittent nystagmus. with regards to his orbital blow out fracture seen on ct head at admission, it appeared chronic. discussed with plastics. no further intervention needed at this time as at least >5 days out from injury and no evidence of decreased eye rom, impingement of eom, or other concerning findings. regardless, would be poor surgical candidate. with regards to his seizures, patient had a h/o childhood seizures and etoh withdrawal seizures. unclear etiology of initial seizures on admission. however, following hemorrhage, he had recurrent seizures where were felt to be due to irritable focus from hemorrhage as well as new stroke. he had multiple dilantin loads until his corrected dilantin level was in therapeutic range of 15-20. he was also maintained on keppra. he had recurrent seizures despite therapeutic dilantin levels. typical seizure activity was lue rhythmic shaking, sometimes with jaw twitching and l superior gaze preference. at times his seizure would become more generalized and he would have alteration of his mental status. his seizures always broke with 2 mg of iv ativan. his keppra dose was uptitrated with decreased frequency of seizures. of note, he was strictly npo after his peg tube was placed so his po dilantin was changed to fosphenytoin. however, once his tube was ready for use he was changed back to oral dilantin. he is currently on keppra 2g , phenytoin 200mg tid, zonegran 200mg/500mg, and trileptal 1200mg plus standing ativan but continues to have intermittent clinical seizures responsive to 2mg to 6mg of iv ativan or 10mg im diazepam. he was discharged on diastat 15mg per rectum as needed for seizures. at time of discharge, the goal phenytoin level was 20-25 with a normal albumin. he should have his anti-seizure medication levels checked every 6 months. his altered ms/agitation was likely due to stroke, venous sinus thrombosis, recurrent seizures, and toxic metabolic encephalopathy in the setting of infection, prolonged hospital stay. has improved with discontinuation of tele and foley. after peg placement can discontinue lue restraint. initially required 1:1 sitter but able to be safe with bed alarm only. frequently has outbursts of yelling but may be post-ictal confusion. hematology: with regards venous sinus thrombosis, it is likely due to underlying hypercoaguable state. anticoagulation complicated by poor access and difficult blood draws. he was initially treated with heparin but due to difficult blood draws this was transitioned to lovenox. lovenox was transitioned back to heparin for 24 hrs for peg tube placement and he was restarted on lovenox 4 hours after tube placement. ct venogram showed partial recannalization of thromboses suggesting response to anticoagulation. given suspected hypercoaguable state, he will likely require lifelong anticoagulation. however, due to extremely difficult blood draws and poor access, the decision was made to continue long term lovenox therapy. he was eventually transitioned to 1.5 mg/kg dosing for once daily administration. he was started on calcium and vitamin d for osteoporosis prophylaxis given suspected lifelong lmwh requirements. he should have bone density testing performed as an outpatient. if worsening density despite calcium and vitamin d, could consider addition of bisphosphonate or transition from lovenox to fondaparinox or coumadin. hematology was consulted and of the labs ordered, he does have elevated anticardiolipin antibody. to establish antiphospholipid antibody syndrome, he will need repeat testing in 6 weeks. he has follow-up appt in heme clinic set up as outpatient. his hypercoaguable state as evidenced by multiple clots in les and diffuse venous sinus thrombosis as well as suspected ischemic stroke all suggest hypercoagulable state. hypercoaguable work up limited by patient's anticoagulation requirement. bilateral ue ultrasounds were negative for clots. he has an infrarenal ivc filter in place which was old, likely placed for chronic le clots. lupus anticoagulant was negative. factor v leiden was also negative but anticardiolipin ab positive although not conclusive for antiphospholipid syndrome as noted above hence will follow-up in heme clinic as outpatient. likely needs life-long anticoagulation. pulmonary in the setting of acute ich and status epilepticus, he had an aspiration pna s/p multiple intubations and multiple central line attempts without success. r subclavian cvl attempt failed and was complicated by a pneumothorax. he is s/p ct x 3. last ct was removed without evidence of recurrent pneumothorax on repeat cxrs. he self extubated twice and remained extubated after the second. he was treated with 10 days of clindamycin and 8 days of vancomycin for presumed aspiration pna. following completion of his treatment he had no fevers, leukocytosis, or other evidence of pneumonia. his hypoxia with o2 requirement likely had multiple potential etiologies including emphysema, recent pneumonia, recurrent aspiration, and possible pes given suspected thrombophilia. he was maintained on anticoagulation as above. he should have pfts performed as an outpatient. gastroenterology: a peg tube was placed for nutrition and medication administration. he was started on tube feeds per nutrition recs. as mental status improved, he passed a swallow evaluation for a modified diet with supervision. he still receives cycled nutrition via peg overnight. iv access: patient had l midline which was changed to r picc at the time of peg placement by ir. this picc fell out on , requiring replacement in ir. picc line was subsequently discontinued prior to discharge as antiepileptic levels and labs had been within normal limits and stable. prophylaxis: therapeutic lovenox. famotidine. bowel regimen. social: code is dnr/dni communication: patient had no healthcare proxy on admission. he had family consent for all procedures through his sister, (sister) (w), (c), (h). after patient's mental status improved, he agreed to make his sister his healthcare proxy. medications on admission: ? keppra 750 mg po daily (1000 mg per pharmacy) discharge medications: 1. acetaminophen 325 mg tablet sig: 1-2 tablets po q6h (every 6 hours) as needed for fever or pain. 2. senna 8.6 mg tablet sig: one (1) tablet po bid (2 times a day) as needed for constipation. 3. docusate sodium 50 mg/5 ml liquid sig: one hundred (100) mg po bid (2 times a day). 4. albuterol sulfate 2.5 mg /3 ml (0.083 %) solution for nebulization sig: one (1) neb inhalation q4h (every 4 hours) as needed for shortness of breath or wheezing. 5. ipratropium bromide 0.02 % solution sig: one (1) neb inhalation q4h (every 4 hours) as needed for shortness of breath or wheezing. 6. amlodipine 5 mg tablet sig: one (1) tablet po bid (2 times a day). 7. multivitamin tablet sig: one (1) tablet po daily (daily). 8. folic acid 1 mg tablet sig: one (1) tablet po daily (daily). 9. thiamine hcl 100 mg tablet sig: one (1) tablet po daily (daily). 10. calcium carbonate 500 mg tablet, chewable sig: one (1) tablet, chewable po qid (4 times a day). 11. cholecalciferol (vitamin d3) 400 unit tablet sig: two (2) tablet po daily (daily). 12. famotidine 20 mg tablet sig: one (1) tablet po q12h (every 12 hours). 13. levetiracetam 500 mg tablet sig: four (4) tablet po bid (2 times a day). 14. enoxaparin 120 mg/0.8 ml syringe sig: one y (120) mg subcutaneous qday (). 15. oxcarbazepine 600 mg tablet sig: two (2) tablet po bid (2 times a day). 16. lorazepam 0.5 mg tablet sig: one (1) tablet po bid (2 times a day). 17. quetiapine 50 mg tablet sig: one (1) tablet po tid (3 times a day). 18. olanzapine 5 mg tablet, rapid dissolve sig: one (1) tablet, rapid dissolve po qhs (once a day (at bedtime)) as needed for agitation, insomnia. 19. camphor-menthol 0.5-0.5 % lotion sig: one (1) appl topical tid (3 times a day) as needed. 20. phenytoin 125 mg/5 ml suspension sig: two hundred (200) mg po three times a day. 21. zonegran 100 mg capsule sig: two (2) capsule po qam. 22. zonegran 100 mg capsule sig: five (5) capsule po at bedtime. 23. diastat acudial 12.5-15-17.5-20 mg kit sig: fifteen (15) mg rectal once a day as needed for focal motor seizure: may repeat in 4 to 12 h if necessary. 24. diazepam 5 mg/ml solution sig: ten (10) mg injection prn:mr1 as needed for focal motor seizures: in ambulance only. discharge disposition: extended care facility: - discharge diagnosis: primary: - left frontal/parietal mca hemorrhagic stroke. - right small parietal infarction - cerebral venous thrombosis - bilateral femoral and popliteal dvt. - general seizure - right pneumothorax - aspiration pneumonia - acute renal failure - thrush - reactive thrombocytosis secondary: - copd/emphysema - etoh abuse: withdrawal seizures and delirium tremens. - childhood seizure disorder - right thalamic lacunar infarct. - recurrent vte - ivc filter - 2.4 x 2.0 cm right adrenal adenoma. - 4.6 x 3.4 x 2.1 liver lesion nos - left orbital blow-out fracture - mva : s/p splenectomy, s/p right hip fracture. discharge condition: fluent aphasia, pupils equal, right arm hemiplegia, moves left side spontaneously and purposefully. discharge instructions: you were admitted to the hospital for alcohol withdrawal. your hospitalization was complicated by a hemorrhagic stroke due to clots in the veins around your brain. you had levels checked for factors that can make you hypercoaguable, and were found to have elevated anticardiolipin. you were started on lovenox daily. you had a central line placed during your icu stay, which led to a pneumothorax requiring chest tube placement. you developed frequent seizures, requiring multiple medications for seizure control. please take all medications as prescribed. recheck anti-seizure medication levels (dilantin, zonegram, trileptal, keppra) every 6 months or sooner as indicated by your outpatient epileptologist. please call your doctor or return to the hospital if you have worsening of your speech or vision, new numbness, new weakness, worsening seizures, fevers, chills, chest pain, shortness of breath, or any other concerns. followup instructions: please follow up with your pcp . once you are discharged from rehab. please call to schedule an appointment. you have an outpatient repeat mri and mrv of your head on at 9:00 am in the , building, . you have a follow up appointment with dr. in neurology/stroke () on at 3 pm in the center, . you have a follow up appointment with dr. a pcp () on at 10:10 am. you have a follow up appointment with dr. in epilepsy/neurology () on at 1:00 in the center, . you have a follow up appointment with dr. in hematology clinic () on at 11:00 in the center, . procedure: insertion of intercostal catheter for drainage venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more insertion of endotracheal tube enteral infusion of concentrated nutritional substances percutaneous [endoscopic] gastrostomy [peg] alcohol detoxification electroencephalogram diagnoses: urinary tract infection, site not specified acute kidney failure, unspecified chronic airway obstruction, not elsewhere classified other specified procedures as the cause of abnormal reaction of patient, or of later complication, without mention of misadventure at time of procedure acute respiratory failure pneumonitis due to inhalation of food or vomitus iatrogenic pneumothorax hypoxemia streptococcus infection in conditions classified elsewhere and of unspecified site, streptococcus, group d [enterococcus] cerebral embolism with cerebral infarction accidents occurring in residential institution accidents occurring in unspecified place closed fracture of orbital floor (blow-out) alcohol withdrawal other specified disorders of liver other accidents acute alcoholic intoxication in alcoholism, continuous cerebral thrombosis with cerebral infarction other forms of epilepsy and recurrent seizures, without mention of intractable epilepsy other specified forms of effusion, except tuberculous 

Answer: The patient is high likely exposed to