Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: keflex / sulfa (sulfonamides) attending: chief complaint: tx from osh with pancreatitis major surgical or invasive procedure: intubation central line placement arterial line placement x 2 history of present illness: 52yo female, pw four day history of n/v/d, generalized weakness and decreased po. pt denied abdominal pain. on arrival to she was noted to have o2 sats of 87% on ra. in addition, she was tachycardic to 118. she was ill appearing and cachectic with "cyanotic" fingers. pt had an initial ph of 7.08, 20, po2 68. her bicarb was five. pt had elevated troponin to 1.16. she received three amps of bicarb with d5, cipro 400 iv, flagyl 500 iv. ekg demonstrated antero inferior q waves. etoh at presentation was 50. at , pt received 5 liters of normal saline. pt transferred from by pt was intubated and sedated on arrival to . she was noted to have a heart rate of 125 and a blood pressure of 97/60. her exam was notable for coarse but equal breath sounds and a distended abdomen. pt was noted to be guiac positive, she was evaluated by general surgery out of concern for necrotizing pancreatitis. they concluded that given the patient's down-trending lft's and ct changes, these were not consistent with nec pancreatitis. in the ed, the patient received another four liters of normal saline for a total-pre-arrival to the micu of 9 liters of iv fluid. at , pt received zosyn, thiamine, pt was admitted to the micu for further evaluation and treatment. past medical history: pancreatitis anemia copd social history: smokes ppd. etoh abuse, unknown ivda. family history: unknown physical exam: vs: temp: bp: / hr: rr: o2sat gen: chronically ill appearing heent: scleral icterus. neck: no supraclavicular or cervical lymphadenopathy, no jvd, no carotid bruits, no thyromegaly or thyroid nodules resp: cta b/l with good air movement throughout cv: rr, s1 and s2 wnl, no m/r/g abd: nd, +b/s, soft, nt, no masses or hepatosplenomegaly ext: no c/c/e, warm, good pulses skin: no rashes/no jaundice neuro: aaox3. cn ii-xii intact. 5/5 strength throughout. no sensory deficits to light touch appreciated. no pass-pointing on finger to nose. 2+dtr's-patellar and biceps rectal: fobt(+) pertinent results: 07:03pm blood wbc-8.3 rbc-2.78* hgb-10.5* hct-31.9* mcv-115* mch-37.7* mchc-32.8 rdw-13.3 plt ct-151 06:05am blood wbc-6.8 rbc-2.95* hgb-10.0* hct-31.2* mcv-106* mch-34.0* mchc-32.1 rdw-15.9* plt ct-784* 07:03pm blood neuts-91.8* lymphs-5.2* monos-2.7 eos-0.2 baso-0.1 06:05am blood neuts-63.0 lymphs-25.0 monos-6.4 eos-4.7* baso-1.0 07:03pm blood pt-16.4* ptt-34.2 inr(pt)-1.5* 06:05am blood pt-12.3 ptt-22.1 inr(pt)-1.0 06:05am blood glucose-85 urean-7 creat-0.5 na-137 k-4.4 cl-101 hco3-23 angap-17 07:03pm blood glucose-147* urean-8 creat-0.7 na-141 k-3.7 cl-110* hco3-11* angap-24* 07:03pm blood alt-35 ast-96* ck(cpk)-88 alkphos-181* totbili-4.9* 06:05am blood alt-70* ast-110* ld(ldh)-256* alkphos-496* amylase-56 totbili-4.3* 07:03pm blood lipase-1090* 01:44am blood lipase-663* 03:41pm blood lipase-510* 02:38am blood lipase-446* 03:58am blood lipase-51 06:05am blood lipase-130* 07:03pm blood albumin-2.7* calcium-6.0* phos-3.3 mg-1.2* 03:58am blood vitb12-971* folate-9.8 10:20am blood hapto-170 06:05am blood triglyc-210* 06:26pm blood crp-121.4* 07:31pm blood type-art rates-/24 tidal v-450 fio2-100 po2-84* pco2-30* ph-7.12* caltco2-10* base xs--18 aado2-619 req o2-98 -assist/con intubat-intubated 02:42pm blood type-art po2-69* pco2-34* ph-7.51* caltco2-28 base xs-3 07:31pm blood glucose-130* lactate-4.3* na-140 k-3.3* cl-115* ct abd: impression: 1. moderate amount of stranding within the anterior pararenal space surrounding the pancreas, which is consistent with the clinical diagnosis of acute pancreatitis. there is no evidence of complication (on this essentially unenhanced scan). 2. persistent renal parenchymal enhancement, with a somewhat striated appearance, which despite the normal serum creatinine, raises the question of acute renal dysfunction, particularly atn or other tubular abnormality. this should be correlated with clinical and laboratory information. 3. low-attenuation liver consistent with generalized fatty infiltration. 4. large consolidation in the lower lobes bilaterally and posteriorly, raises the possibility of aspiration pneumonia. 5. mild hypodensity in the wall of the cecum and ascending colon which could represent submucosal edema with mild apparent hyperemia of the mucosa, focally. this finding is nonspecific, and could related to the patient's known hypoalbuminemia; correlation with the clinical status of the patient is recommended. 6. high density within the cystic duct and common bile duct as well as intermediate density within the gallbladder. these findings, in this context, are most suggestive of vicarious excretion of contrast, also suggestive of underlying renal abnormality. : ecg: sinus tachycardia. diffuse low qrs voltage. left axis deviation. poor r wave progression - cannot rule out old anteroseptal myocardial infarction. diffuse non-specific st-t wave changes which are non-specific. no previous tracing available for comparison. : cxr: there has been interval worsening in bibasilar consolidations and effusions greater on the right side. there is no pneumothorax. et tube and ng tube remain in place echo: the left atrium is normal in size. the left ventricular cavity size is normal. there is moderate global left ventricular hypokinesis (lvef = 35-40 %). systolic function of apical segments is relatively preserved. the aortic valve leaflets (3) are mildly thickened. the mitral valve leaflets are mildly thickened. physiologic mitral regurgitation is seen (within normal limits). there is borderline pulmonary artery systolic hypertension. there is an anterior space which most likely represents a fat pad. compared with the report of the prior study (images unavailable for review) of , biventricular systolic function is now improved. ct abd/pelvis: impression: 1. no evidence of pancreatic pseudocyst or hepatobiliary obstruction. 2. multifocal pneumonia. 3. featureless sigmoid colon may represent c. diff infection. recommend correlation with serum c. diff toxin markers. 4. fatty liver, unchanged since . 5. anasarca, unchanged since . brief hospital course: this is a 52 year old female transferred from osh with pancreatitis, likely etoh, intubated with possible ards and aspiration pna. # pancreatitis: nausea, vomiting, decreased po, ct scan, and lipase of > 1000 demonstrate pancreatitis on admission. there was no evidence of hemorrhage or pseudocyst on ct scan. she was transferred from and intubated at in the ed and then transferred to the micu. she was extubated successfully and transferred out to the medical floor. patient admits to drinking whiskey daily which is the likely cause of her pancreatitis. her pancreatitis has now resolved and she has been advanced to a po diet. she is to follow up with her primary care physician as an outpatient. she was educated on the harms of alcohol abuse and was told to refrain from any etoh use in the future as it may precipitate another episode of pancreatitis. # acute on chronic systolic heart failure: tte on with lvef 25%, with severe global hypokinesis. f/u tte showed improvement in systolic function. patient denies any history of chf. chf is likely etoh cardiomyopathy. she was started on aspirin, toprol and lisinopril. # pneumonia: patient was admitted with pna and ards thought to be aspiration pneumonia. she was started on levofloxacin and flagyl for treatment. during her micu stay, she continued to spike fevers and her antibiotics were broadened to vancomycin and zosyn iv. she was treated for 10 days of antibiotics and was afebrile on discharge. # nsvt: ms. had a few 5-10 beat runs of nsvt while in the hospital. she has no history of cad but may have etoh cardiomyopathy based on tte done here. she was started on toprol for treatment of her chf and nsvt. this should be followed up by her pcp as an outpatient. # elevated lfts. an obstructive pattern. no evidence cholecystitis, biliary tree abnormality on ruq u/s x2 and ct torso. given history, likely, etoh hepatitis. hepatology was consulted and recommended ercp if lft's rose again, however they remained stable and the patient was discharged home. she should be follow up by her pcp regarding this matter. # respiratory failure- initially intubated for ards, likely aspiration pna. she was extubated successfully and was breathing at baseline on discharge with good oxygen saturations. # etoh abuse: social work was consulted for etoh abuse. patient was referred to local programs for help. medications on admission: folic acid discharge medications: 1. lisinopril 5 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 2. toprol xl 25 mg tablet sustained release 24 hr sig: one (1) tablet sustained release 24 hr po once a day. disp:*30 tablet sustained release 24 hr(s)* refills:*2* 3. aspirin 81 mg tablet sig: one (1) tablet po once a day. tablet(s) discharge disposition: home with service facility: care discharge diagnosis: primary: - hypovolemic/septic shock - aspiration pneumonia - acute respiratory distress syndrome - necrotizing pancreatitis - etoh hepatitis - non-thrombotic troponin elevation - acute systolic heart failure - svt with aberrancy - nsvt - multifactorial anemia - rash nos - malnutrition, moderate degree secondary: - etoh abuse - copd discharge condition: good. pancreatitis resolved. discharge instructions: you were admitted for pancreatitis to the intensive care unit. your pancreatitis has now resolved. you were also found to have heart failure which will need to be addressed with your cardiologist. . please refrain from drinking alcohol. . please take your medications as prescribed. . follow up as below. followup instructions: please follow up with your primary care physician 1 week. , f. . please follow up with your cardiologist to address your heart failure. . please follow up with your gastroenterologist to address you abnormal liver function and for continuing care of your pancreatitis. procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more parenteral infusion of concentrated nutritional substances insertion of endotracheal tube enteral infusion of concentrated nutritional substances thoracentesis diagnoses: anemia, unspecified unspecified pleural effusion congestive heart failure, unspecified chronic airway obstruction, not elsewhere classified acute respiratory failure pneumonitis due to inhalation of food or vomitus dermatitis due to drugs and medicines taken internally acute on chronic systolic heart failure acute pancreatitis acute alcoholic hepatitis 

Answer: The patient is high likely exposed to