Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: sob, loss of appetite, fatigue major surgical or invasive procedure: none history of present illness: 74 yo m with 3vd (inoperable), ischemic cm, chf (ef 15-25%), severe mr, s/p vt ablation and icd, cri, pvd, cva who presents with several week h/o dyspnea and decreased po intake. the dyspnea is not exacerbated with exertion. he denies fever, chills, though has occasional dry cough. he has had no recent known sick contacts and did receive his flu shot this year. no chest discomfort. no palpitations. dyspnea has been accompanied by generalized weakness and decreased po intake, with 5-6 pound weight loss over the past week. he has had difficulty sleeping for the past two nights, though denies pnd. no le edema. mr. was recently hospitalized -> with frequent episodes of vt. underwent vt ablation, and amiodarone was restarted at 400mg. also noted to have arf, which was attributed to hypotension, and improved. recent clinic notes from dr. detail similar presentation of weakness. he has had several medication changes recently, including his amiodarone and digoxin dosing. his amiodarone was originally discontinued following recent vt ablation,though was subsequently restarted at 800 mg daily. following reinitation, he noted weakness and blurred vision. his amiodarone was again d/c'd . in addition, his digoxin was d/c'd at 12/29, to see whether his symptoms improved without it. per the patient's daughter, his symptoms initially improved somewhat, though overall have not changed significantly. per family members, he has been intermittently confused, and has had bowel incontinence on several occasions. ros negative for ha, melena, brbpr, abdominal pain. has had intermittent nausea. past medical history: cad s/p cath : 3vd: 100% occlusion in prox rca, 100% mid lad and 100% intermedieus disease mi (markedly elevated tnt, negative ck) profound ischemic cardiomyopathy with an ef of 15-25% chronic atrial fibrillation s/p icd, biventricular ppm h/o monomorphic vt , s/p successful ablation of three vt circuits chf cri - baseline 1.3-1.9. 4+mr, 2+tr htn hyperlipidemia pvd cva x 2 12 years ago, 6 years ago. residual l-sided weakness he had a nephrectomy in secondary to complication of nephrolithiasis. pulm htn (tr grad 72 ). he had a lv thrombus documented in by echocardiogram depression lbp bph social history: married, lives with wife. former tobacco and etoh use. family history: nc physical exam: pe: t 95.9 bp 111/47 p 50 rr 16 97% ra patient found lying flat in bed, in nad anicteric, conj uninjected, mmm. no sores in op jvp ~, no lad regular bradycardic rhythm, iii/vi hsm at apex abd soft, nt/nd, +bs no peripheral edema, no calf tenderness follicular rash on chest awake, alert, answers questions (limited by language barrier, able to communicate with daughter). cn ii - xii in tact. strength 5+ rue/rle, 5+ lue/lle (though mildly weaker than r diffusely). pertinent results: spirometry : actual pred %pred actual %pred %chg fvc 3.07 3.16 97 fev1 2.25 2.11 107 mmf 1.59 2.11 75 fev1/fvc 73 67 110 impression: isolated reduction on diffusing capacity suggests perfusion limitation. - echo : ef25%, left ventricular cavity enlargement with global and regional systolic dysfunction c/w multivessel cad or other diffuse process. 4+ mitral regurgitation. severe pulmonary artery systolic hypertension. right ventricular hypokinesis. - p/a lat: no acute cardiopulmonary placement. stable cardiomegaly. ecg: paced rhythm, 50bpm, wide complex (qtc ~500), lad, lbb pattern. brief hospital course: a/p 74 yo m with ischemic cm, chf with biv ppm, pvd, admitted with dyspnea, weakness, decreased po intake and arf on cri. the pt was felt to be dehydrated d/t decreased po intake on initial admission, which improved gradually. he was laced on nesiritide and lasix per cardiolgoy given his chf. the pt had a history of afib, mural thrombus, and frequent vtach. coumadin was held on admission given a high inr. pacemarker was interoogated showing intermittent complete heart block and high grade av block, changes attributed to amiodarone toxicity. his pacemaker setting were changed. for his h/o cad, continued aspirin, b-blocker, statin. the pt had elevated lft's and had a ruq us which showed no e/o liver or gallbladder pathology. on , the pt underwent cardiac arrest, whcih appeared to be a pea to asystole arrest due to failure of pacing capture. code labs revealed marked acidosis with elevated k. hypotheses included ischemic bowel from severe chf, leading to lactic acidosis, leading to pacer firing but not capturing. the pt was hypotensive and was transiently on dobutamine and levophed. his renal function worsened, felt secondary to shock. the pt's ms was affected after the first code although he was noted to have a lack of movement in his lue; however, this might have been from reexpression of previous strokes. eeg showed moderate encephalopathy. the pt was extubated on . he developed septic physiology and was treated for an enterococcal uti and for group b strep in the blood with broad spectrum abx. his renal failure continued to worsen. he was placed on milrinone with improvement in cardiac index. he then had ugib with episodes of melena and was transfused. his lft's rose significantly, felt to be c/w shock liver. ruq us showed no evidence of cholestasis. the pt's skin was bright yellow/, and was biopsied by dermatology (his tbili was in the high 20's). the pt also had a coagulopathy from unclear source, resolving with vitamin k. the pt's ms began to worsen, felt to be infectious/metabolic in origin. he then developed a thrombocytopenia of unclear etiology. the pt was medically stable but his ms did not return. he was transferred to the cards floor. on , the pt went into respiratory arrest s/p possible aspiration event. he was reintubated. his bp went to the 50's despite max doses of levophed, dopamine, and milrinone. epinephrine was started. code status was discussed at length with the pt's family throughout the hospitalization. at this time, they agreed to withdraw medical support and the patient passed away. medications on admission: asa 81 mg qd, coumadin 1mg qd (2mg , ), coreg 12.5 mg , bumex 1 mg qd, digoxin 0.125 mg qd (held for past week), amiodarone 800 mg qd (held for past week), lipitor 80 mg qd, flomax .4, and detrol 2. discharge disposition: extended care discharge diagnosis: copd, chf discharge condition: deceased procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more parenteral infusion of concentrated nutritional substances insertion of endotracheal tube pulmonary artery wedge monitoring transfusion of packed cells cardiopulmonary resuscitation, not otherwise specified closed biopsy of skin and subcutaneous tissue transfusion of other serum injection or infusion of nesiritide infusion of vasopressor agent diagnoses: pneumonia, organism unspecified acidosis hyperpotassemia thrombocytopenia, unspecified acute kidney failure with lesion of tubular necrosis urinary tract infection, site not specified congestive heart failure, unspecified acute and subacute necrosis of liver atrial fibrillation hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage v or end stage renal disease atrioventricular block, complete cardiac arrest cardiogenic shock other complications due to other cardiac device, implant, and graft rash and other nonspecific skin eruption hemorrhage of gastrointestinal tract, unspecified other and unspecified coagulation defects chronic vascular insufficiency of intestine adult failure to thrive cardiac rhythm regulators causing adverse effects in therapeutic use 

Answer: The patient is high likely exposed to