Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: sah/pancytopenia reason for micu admission: intubated, elevated icp s/p drain placement major surgical or invasive procedure: extraventricular drain placement central line placement arterial line placement intubation history of present illness: 33 yo female with no significant transferred from osh to for ms changes, seizures, found to have large intraparenchymal hemorrhage and midline shift/impending herniation now s/p intrcranial drain placement for elevated icp. five days ago, patient fell at home, and was evaluted at an osh with head ct without any acute bleed noted. she then presented again to an osh, with repeat hct due to persistent ha, but still no bleed seen. patient then presented today to an osh with ha, weakness on her left, and possible seizure activity, and while at osh found to have large intraparenchymal bleed. she was intubated at osh, given dilantin prior to transfer to for nsgy eval. she reportedly had reactive pupils prior to transfer, but on arrival, she had fixed/dilated pupils. in the ed, initial vs: 97.6, 78, 127/55, 100% ac/480/16/5/100%. she had a ct head which showed a 3 cm iph above thalamus with extension into third ventricle, and evidence of likely impending herniation. she was evaluted by nsgy, and had an emergent external ventricular drain placed for elevated icp (initially 45 in ed, on repeat prior to transfer 32). her labs revealed pancytopenia, decreased fibrinogen, and concern for dic. heme/onc was called in the ed, and dr. me regarding starting her on atra for likely apml. she was given 2 units prbc, 2 units of plts prior to transfer to the micu for further management. dr. from neurosurgery held family meeting and explained grave prognosis to the family. currently, she is intubated, sedated. she is not responsive to voice. ros: unable to obtain past medical history: none social history: married; works as postal worker. denies tobacco/etoh/drugs family history: unknown physical exam: bp: 110/73 hr: 72 r 17 o2sats 100% intubated; intracranial pressure 27 gen: intubated; unresponsive to verbal stimuli heent: 4 mm nonreactive pupils; intubated cv: rrr lungs: cta bilaterally abd: soft, normal bs ext: no edema neuro: no response to verbal stimuli. withdraws on right to deep painful stimuli. no response on left. toes upgoing bilaterally; clonus bilaterally. evd in place for intracranial pressure monitoring pertinent results: 07:27am blood wbc-12.8*# rbc-2.94* hgb-8.6* hct-23.2* mcv-79* mch-29.1 mchc-36.9* rdw-18.7* plt ct-56* 09:05pm blood wbc-2.4* rbc-1.36* hgb-4.2* hct-11.6* mcv-85 mch-30.8 mchc-36.1* rdw-19.9* plt ct-10* 09:55pm blood neuts-5* bands-0 lymphs-25 monos-1* eos-0 baso-0 atyps-0 metas-0 myelos-1* promyel-4* nrbc-1* other-64* 09:05pm blood neuts-3* bands-1 lymphs-14* monos-1* eos-1 baso-0 atyps-0 metas-0 myelos-0 promyel-1* nrbc-3* other-79* 07:27am blood pt-15.3* ptt-23.5 inr(pt)-1.3* 09:05pm blood pt-14.7* ptt-20.7* inr(pt)-1.3* 07:27am blood fdp->1280* 04:08am blood fdp-320-640* 01:09am blood fdp-80-160* 10:30pm blood fdp-80-160* 07:27am blood fibrino-124* 04:08am blood fibrino-155 01:04am blood fibrino-147* 09:05pm blood fibrino-115* 07:27am blood glucose-122* urean-16 creat-1.0 na-159* k-3.5 cl-122* hco3-27 angap-14 09:05pm blood glucose-138* urean-22* creat-0.9 na-137 k-4.0 cl-104 hco3-21* angap-16 07:27am blood alt-22 ast-42* ld(ldh)-529* alkphos-51 totbili-2.9* dirbili-0.7* indbili-2.2 09:05pm blood alt-19 ast-36 ld(ldh)-392* alkphos-30* totbili-0.4 07:27am blood calcium-10.0 phos-1.7* mg-2.3 uricacd-4.4 09:05pm blood totprot-6.4 calcium-8.8 phos-4.0 mg-1.8 uricacd-3.6 07:27am blood d-dimer-greater th 01:09am blood d-dimer-7895* 07:27am blood osmolal-334* 04:08am blood osmolal-332* 01:04am blood osmolal-328* 09:05pm blood pep-no specifi igg-567* iga-70 igm-50 08:45am blood type-art temp-40.0 rates-/16 tidal v-500 peep-5 fio2-50 po2-250* pco2-33* ph-7.50* caltco2-27 base xs-3 intubat-intubated 01:10am blood type-art temp-35.8 po2-499* pco2-29* ph-7.51* caltco2-24 base xs-1 intubat-intubated 10:05pm blood hgb-4.6* calchct-14 09:31am urine color-yellow appear-clear sp -1.038* 09:31am urine blood-lg nitrite-neg protein-150 glucose-neg ketone-neg bilirub-sm urobiln-1 ph-8.0 leuks-neg 09:31am urine rbc-0-2 wbc-0-2 bacteri-occ yeast-none epi-<1 04:08am urine hours-random creat-12 na-< 10 04:08am urine osmolal-224 9:30 am sputum source: endotracheal. **final report ** gram stain (final ): >25 pmns and <10 epithelial cells/100x field. 4+ (>10 per 1000x field): gram positive cocci. in pairs, chains, and clusters. 3+ (5-10 per 1000x field): gram negative diplococci. 2+ (1-5 per 1000x field): gram positive rod(s). 2+ (1-5 per 1000x field): gram negative rod(s). respiratory culture (final ): heavy growth commensal respiratory flora. staph aureus coag +. moderate growth. staphylococcus species may develop resistance during prolonged therapy with quinolones. therefore, isolates that are initially susceptible may become resistant within three to four days after initiation of therapy. testing of repeat isolates may be warranted. sensitivities: mic expressed in mcg/ml _________________________________________________________ staph aureus coag + | clindamycin-----------<=0.25 s erythromycin----------<=0.25 s gentamicin------------ <=0.5 s levofloxacin----------<=0.12 s oxacillin------------- 0.5 s trimethoprim/sulfa---- <=0.5 s blood cultures: no growth urine cultures: no growth ecg: sinus rhythm. tracing is within normal limits. no previous tracing available for comparison cxr: impression: 1. endotracheal tube tip is slightly low lying, approximately 2.6 cm from the carina. nasogastric tube in standard position. 2. no acute cardiopulmonary abnormality. ct head: impression: 1. intraparenchymal hemorrhage, largely centered over the right thalamus, with intraventricular extension and associated hydrocephalus. 2. small extra-axial hematoma overlying the left cerebral convexity, likely subdural in location. 3. diffuse cerebral edema. 4. effacement of the basal cisterns, suggestive of early or impending central herniation. cta head: impression: 1. increased intraventricular and midbrain hemorrhage when compared to the most recent examination. small low-attenuation subdural collection overlying the left frontal lobe is not significantly changed. 2. abnormal contrast enhancement within the midbrain at the inferior margin of the hematoma could represent small foci of active extravasation, though the unusual curvilinear structure, more dorsally, may represent abnormal intravascular enhancement, either in a congested and distorted normal vein, or related to an underlying vascular abnormality such as an avm. 3. interval insertion of a right frontal ventriculostomy catheter tip terminating near the third ventricle with slight interval decrease in size of the frontal horns, though slight expansion in size of the temporal and occipital horns. 4. persistent moderate cerebral edema with effacement of the basal cisterns, indicative of central and downward transtentorial herniation. 5. while the major intracranial arteries are patent, the mild irregularity of the basilar artery suggests mild vasospasm. ct head: impression: large right thalamic hemorrhage with intraventricular extension, and the overall volume of hemorrhage appears increased from the previous studies. there is also moderate associated vasogenic edema. note added in attending review: in addition to the persistent large amount of hemorrhage and unchanged ventricular dilatation, despite the recently-inserted ventriculostomy, there is also persistent effacement of all basal cisterns, suggestive of central uncal and downward transtentorial herniation. there is no evidence of tonsillar herniation, either on this study or the sagittal reformations of the preceding cta. peripheral blood flow cytometry: interpretation: ommunophenotypic findings consistent with involvement by acute promyelocytic leukemia. brief hospital course: assessment & plan: 33 yo female s/p fall presenting with ha, left sided weakness, seizure activity now with 3 cm iph with likely herniation s/p evd placement by nsgy with elevated icp (45 initially), also found to have pancytopenia and dic labs consistent with likely early dic # iph/elevated icp: patient was initially transferred to once noted to have 3 cm iph on osh head ct. dr. (neurosurgery) evaluated patient in the ed, and and evd was placed with marked elevated pressures (45 initially). patient was at high risk for bleed given markedly low platelets, but without this procedure, she would have continued to herniate given the bleeding. she was transfused cryo, ffp, and platelets, and once the drain was secured, she was transferred to the micu. on arrival, her icp was 23. she had a central line placed, and arterial line placed, and was closely monitored for icp elevation as well as sodium and osmolarity. she was given further products to maintain her plts greater than 50k. she had q1h neurochecks, and mannitol was given to maintain na and osmolarity. she was also started on dilantin for seizure prophylaxis, and given cefazolin for evd/skin prophylaxis. neurosurgery continued to monitor her progress. despite these attempts, a few hours after admission to the micu, the patient's icp drastically elevated again (>50). neurosurgery was at the bedside to evaluate the patient, and it seemed the drain was likely clotting off, most likely due to further bleeding. she was sent for a stat repeat head ct which showed worsening of the bleeding within the ventricles. her neurologic status continued to deteriorate. after meetings with both dr. , dr. (hematology attending), the icu team, and the patient's family, it was determined that no further intervention can be performed which would reverse the patient's condition. the patient was made cmo, and she expired later that afternoon. an autopsy was not wanted by the family. # pancytopenia: patient initially presented with all depressed cell lines. given atypical cells, hematology was consulted. the intitial smear reviewed by hematology was felt to be most consistent with aml, most likely apml given the clinical scenario. the patient was started on all trans retinoic acid shortly after arrival to the micu . she was monitored for dic labs (which she did develop), and was given further blood products (ffp, plateletes, cryoprecipitate). flow cytometry was performed and was consistent with aml. given her poor prognosis with the intracranial hemorrhage, as above, the patient was made cmo. the patient's family was present and updated frequently by the primary icu team, neurosurgical team, and hematology service. social work was also involved with this patient's care and her family. autopsy was declined. medications on admission: occasional flexiril discharge medications: expired discharge disposition: expired discharge diagnosis: acute promyelocytic leukemia intracranial hemorrhage thrombocytopenia anemia -expired discharge condition: expired discharge instructions: expired followup instructions: expired procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified arterial catheterization transfusion of packed cells ventricular shunt to extracranial site nec transfusion of platelets diagnoses: obstructive hydrocephalus cerebral edema fall from other slipping, tripping, or stumbling other and unspecified intracranial hemorrhage following injury without mention of open intracranial wound, with prolonged [more than 24 hours] loss of consciousness without return to pre-existing conscious level acute myeloid leukemia, without mention of having achieved remission subarachnoid hemorrhage following injury without mention of open intracranial wound, with prolonged [more than 24 hours] loss of consciousness without return to pre-existing conscious level civilian activity done for income or pay 

Answer: The patient is high likely exposed to