Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: sulfa (sulfonamides) attending: chief complaint: leukocytosis major surgical or invasive procedure: none history of present illness: 71 yo mandarin-speaking man who was admitted to the in for evaluation of a three-week history of blurred vision and was subsequently found to have a wbc of 300,000 and was diagnosed with aml. he was twice leukopheresed and then was treated with idarubicin for 3 days and a continuous infusion of ara-c for 7 days. a follow-up bone marrow on showed remission. of note, the hospital course at that time was complicated by the findings of interstitial pulmonary abnormalities and pulmonary nodules on imaging studies; these were ultimately thought to be a chronic finding, but because of his rll lesion, he was treated empirically for aspergillosis with voriconazole, and he was also given a course of levofloxacin. further investigation following his discharge from the hospital demonstrated that he had been treated with gatifloxacin and azithromycin at an osh in for presumed rhodococcus equi pneumonia (the organism was cultured from bal fluid at that time); the significance of this finding and the relative adequacy of this treatment remains unclear. a sputum culture obtained after readmission in for dyspnea and hemoptysis showed mycobacteria gordonae (a known contaminant and not likely to cause disease). a chest ct done demonstrated a persistent spiculated nodule in his right lower lobe, multiple opacities in the right and left upper lobes that were more prominent than on previous examinations, unchanged calcified mediastinal lymphadenopathy, and stable low-attenuation liver lesions. since his diagnosis with aml he has been followed as an outpatient the division of infectious diseases. in summary, his voriconazole was discontinued based upon the presumption that he appears too well to have persistant infection and the lack of any definitive evidence (e.g. culture data) of infection. he was scheduled to be seen in pulmonary clinic for follow-up of the persistent collapse vs. atelectasis of his right middle lobe out of concern for another primary malignancy. he was seen last week in oncology clinic and was noted to have a wbc of 19,000 without any blasts. follow-up today in clinic showed an asymptomatic wbc of 64,000 with 42% blasts. after discussion the matter with his family, the patient agreed to be admitted for treatment of relapsed aml. he was given 3 grams of hydroxyurea orally and 300 mg of allopurinol orally in the clinic and was then admitted to the bmt floor. past medical history: 1. aml: diagnosed in , status-post remission induction with 7+3 chemotherapy 2. pulmonary disease as per hpi, s/p treatment for aspergillosis 3. chronic micronodular pulmonary disease due to pneumoconiosis (retired coal miner) 4. bleeding peptic ulcer disease in 5. left knee surgery 6. prolonged (two month) course of gatifloxacin and azithromycin for possible rhodococcus equi pneumonia 7. ppd negative social history: he worked at a coal mine for about 20 years in . former smoker, with a 100 pack-year smoking history; he quit smoking in . no etoh consumption. he currently lives with his wife, daughter and son-in-law. has 5 children. family history: his mother, who passed away 3 years ago, and his brother, whom he has not seen for 7 years, both had tuberculosis. physical exam: temp 97.7, bp 126/78, hr 73, rr 14, spo2 94% ra gen: mandarin-speaking only, very pleasant, comfortable, papers in bed and ambulating around room, non-toxic heent: ncat, no sinus tenderness, pinpoint pupils bilaterally, conjunctivae clear, op clear, moist oral mucosa neck: soft, supple, shotty submandibular adenopathy cv: rrr, normal s1 and s2, no m/r/g pulm: diminished bibasilar breath sounds but otherwise clear to auscultation bilaterally abd: soft, non-tender, mildly distended with tympany to percussion, active bowel sounds, no organomegaly back: no cva or paraspinal angle tenderness ext: trace bilateral lower extremity pitting edema, 2+ dp pulses, warm nodes: no palpable cervical, axillary, or inguinal adenopathy pertinent results: wbc-64.5 (n-35 band-6 l-10 m-4 e-1 meta-2 blast-42) hct-37.3 mcv-86 plt-173 pt-12.9 ptt-32.2 inr-1.1 fibrinogen-322 na-142 k-4.2 cl-109 bicarb-27 bun-16 cr-1.3 ca-9.3 mg-2.2 phos-3.9 alb-4.1 alt-14 ast-38 alk phos-72 tbili-0.1 ldh-817 uric acid-7.4 brief hospital course: 71 yo man with aml, status-post idarubicin and ara-c in , initially in remission by bone marrow biopsy at that time, now with relapsing aml/acute leukemic crisis (wbc 64,000 with 42% blasts) and mild acute renal failure. 1. relapsing aml: patient presented with recurrence of his aml. he was managed with first hydrea, then re-induction with mec chemotherapy. he tolerated this well. he developed neutropenia and spiked temperatures. his initial infectious work up was unrevealing, with the exception of bilateral basilar lung disease on chest xray. sputum cultures only revealed sparse oropharyngeal flora. he was managed with empiric cefepime/vancomycin. 2. pulmonary: elaborate and complicated history of pulmonary disease with possible superimposed infectious disease (recent fungal infection? remote rhodococcus pneumonia?) as detailed in the hpi. has completed course of antifungal therapy for aspergillus, as well as antibiotic therapy for rhodococcus. he has a history of coal-worker's pneumoconiosis as well. 3. acute renal failure: admission creatinine elevated to 1.3 from a baseline of 0.9. likely etiologies include an effect of tumor lysis vs. pre-renal azotemia. this returned to baseline with hydration. on (hospital day 20), the pt. was transferred to the icu for persistent tachycardia (uncontolled atrial fibrillation) and hypotension, presumed to be related to sepsis. shortly after arrival to the icu, he was intubated and placed on a ventilator for hypoxic respiratory failure. over the course of the next three hospital days, the pt's. clinical status deteriorated despite treatment with multiple antibiotics, pressors and mechanical ventilation. the pt. became profoundly acidemic on the 23rd hospital day and progressively hypotensive despite increasing doses of pressors. a family meeting was held given his deteriorating status but it was decided to continue all current treatment, however, it was determined that cpr was not indicated. shortly thereafter, the pt. went into asystole. he was pronounced deceased at 2:02pm on . medications on admission: none. discharge medications: n/a discharge disposition: expired discharge diagnosis: n/a discharge condition: deceased discharge instructions: n/a followup instructions: n/a procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified parenteral infusion of concentrated nutritional substances insertion of endotracheal tube other bronchoscopy transfusion of packed cells injection or infusion of cancer chemotherapeutic substance injection or infusion of oxazolidinone class of antibiotics diagnoses: congestive heart failure, unspecified unspecified essential hypertension acute kidney failure, unspecified unspecified septicemia severe sepsis atrial fibrillation acute respiratory failure pneumonitis due to inhalation of food or vomitus septic shock diastolic heart failure, unspecified acute myeloid leukemia, without mention of having achieved remission unspecified diseases of conjunctiva due to viruses 

Answer: The patient is high likely exposed to