Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: pancreatic head mass major surgical or invasive procedure: whipple procedure, history of present illness: mrs. was a 65 year old woman with no significant past medical history who was well until approximately one month prior to admission when she presented with jaundice and weight loss to an osh. she initially underwent ercp at hospital with a plastic stent placed on a stricture but without biopsy due to her asa 81mg adily. she had a ct with visualization of the stricture as well as with dilation of both the pancreatic and common bile ducts. she was subsequently referred to for eus with fna of the cbd and pancreas however given her increased jaundice and pruritis it was thought that her current plastic stent in the cbd may not be providing adequate biliary drainage. this was removed, brushings were taken, and a self expanding metal stent was placed in the cbd. patient was discharged on with cholestyramine and augmentin. since her discharge she continued to have persistent pruritis. her urine was reportedly "gold" colored and she is was urinating about every hour, with frequent burning. repeat lab tests showed increased bilirubin (from 25 to 30). she had insomnia secondary to the pruritis and urinary frequency. she also had frequent loose stools, up to bms per day. she was readmitted to for another ercp on and scheduled for surgery on . she returned on for a whipple operation. past medical history: 1. hypertension 2. ectopic pregnancy 3. anemia, megalobastic social history: lives at home with her husband. smokes 14 cigarettes per day for many years; occasional alcohol. no current drug use or in past. retired from work in human resources. has 2 children and 5 grand children family history: son with diabetes, type ii. brief hospital course: mrs. was taken to the operating room for a whipple procedure and cholecystectomy on . the operation proceeded without complication. please refer to the operative report for additional details. mrs. was sent to the surgical floor after a brief stay in the pacu. on the morning of pod 1, mrs. was afebrile, vitals stable, alert but minimally oriented, confused and agitated. her narcotics were removed from her epidural and overall pain regimen. lab results showed significantly elevated lfts -- alt/ast 2229/2919 up from 231/336 the day prior. bilirubin was 17.8 up from 13.7. a serum ammonia level was tested and returned at 24. a uti was suspected based on ua dipstick and ciprofloxacin was started but stopped the next day after the urine cx returned no growth and no clinical signs of infection. her inr was 1.8 (from 1.9 the day prior) and her potassium was 2.9 (from 3.5 and 4.0 previously). over the course of the next few days, the patient was less agitated but her delirium was persistent with continued confusion, nonsensical speech and behavior and lack of orientation. her liver function tests, however, were consistently trending downwards with alt/ast/tbili from 1590/1450/18.2 (pod 2) to 561/157/14.3 (pod 4) to 117/42/10.8 (pod 9). her inr went down to 1.1 on pod 2 and remained at 1.1 on pod 3. from a electrolyte perspective, she was hypokalemic post operatively which gradually resolved by pod 8. her potassium levels remained persistently in the low 3s with interspersed values of 2.6 and 2.8. her phosphorous was 2.2 and 1.8 on pod 1 and 2 respectively prior to normalizing to 3.8 and eventually 3.1 on pod 9. from a nutritional perspective, she was transitioned from npo to sips on pod 4, to clears on pod 5, to fulls on pod 7 and to a regular diet with pureed consistency on pod 8. she tolerated all advances with no emesis, no apparent nausea or abdominal bloating. she was taking in low volumes but more than 400 cc of intake with her dietary advances. on the evening of pod 7, the patient was intermittently alert and oriented with markedly less confusion and able to hold a sensible conversation with family members as well as members of the healthcare team. this waxed and waned into pod 9 where she was clear most of the day, alert and oriented to person, time and place though continued to have intermittent nonsensical thought and speech. she had several loose bowel movements, sample sent for c.diff and started on empiric flaygl therapy. on pod 10, mrs. confusion returned intermittently, improved from prior but but she appearing more tired, especially after getting out of bed with physical therapy and after a 2.5 mg dose of oxycodone. oxycodone was thereafter removed from her medication list and replaced solely with tylenol.she had fewer loose bowel movements. on the morning of pod 11, mrs. was mentating, alert but not entirely oriented but interspersed with nonsensical speech. her physical exam was unchanged: the incision was clean, dry and intact, her abdomen soft, non-tender and with minimal distention, unchanged from prior exams. in the early afternoon, after lunch, mrs. had a loose bowel movement and was being cleaned by the nursing staff. after the conclusion of this process, her husband who re-entered the room noticed that the patient was not breathing and was unresponsive. he alerted the nursing staff who called a code. mrs. was found to be in pea arrest, then ventricular fibrillation. she was actively coded for 10 minutes prior to regaining a pulse with chest compressions performed throughout, receiving three shocks, epinephrine, bicarbonate and vasopressin. she ultimately regained a pulse and was transferred to the sicu. during this process, labs indicated that mrs. hematocrit was 11.9 (last checked value was pod 9 at 30.3 and had remained stable at approximately 30 during her entire post-op course) and had an inr of >21. overall, starting from the code and overnight, mrs. received 8 units of blood, 6 units of ffp, and 1 unit of cryoprecipitate. ct scan done that night showed a large intraperitoneal bleed, mostly clotted, with no active extravasation of contrast. it was decided to manage her non-operatively with close monitoring with serial hematocrits, with fluid resuscitation and a low dose neo drip. she was hemodynamically stable, responding appropriately to the blood transfusions, with her hematocrit going from 11.9-->13.7-->28.8 overnight. initially her bladder pressures were monitored frequently to monitor for abdominal compartment syndrome due to an on-going bleed. her hematocrit remained stable in the mid 20s. her bladder pressures were normal and measurements were stopped . mrs. time in the sicu was dominated by neurological and cardiovascular/renal/fluid management issues. they are described in detail over her 2 week course in the icu. cardiovascular/renal: on she remained hemodynamically stable but her uop decreased after finishing transfusions of blood. the pressors were titrated accordingly to maintain increased renal perfusion and urine output. she was transfused 2units on to a post-tx hct of 28.9, her remained stable at inr 1.1, her pressors were weaned and her cr was 1.6. her pressors were turned off entirely on and she remained hemodynamically stable for the next few days and her renal function improved gradually(1.2 on to 0.6 on ). from , mrs. hemodynamic status started to worsen again. she had some low blood pressure issues (sbps in the 80s) and low urine output (20 cc/hr, cr bumped suddenly to 1.7 from 0.6) for which she received a ivf boluses including both crystalloid and albumin, eventually had pressors restarted for a short period of time then dc'd after her toes were found to be blue then restarted after her toe ischemia resolved and pressures dropped again, this time with limited change in color of the toes. her fluid overload status continued to worsen by both clinical (edema, decreased po2 on abg) and radiologic measures (worsening cxr). a lasix drip was started on and diuresis was successful (she was negative almost 800 ml as opposed to daily positive in i's/o's since icu admission) but her blood pressure did not tolerate it and it was dc'd shortly thereafter. she received a total of 3 units of blood between and into . her cr remained elevated at 1.8 on . neurological -------------- mrs. remained largely unreactive to external stimuli after the code. initially, she retracted her extremities in response to pain but this disappeared over the first day in the icu. she was thought to have had some seizure-like activity the morning of the 26th ( morning after the pea arrest) which resolved when treated with midazolam. she failed to regain any meaningful neurologic function two days post the pea event, even with sedation weaned and turned off. neurology was consulted and suggested a head ct to rule out a brain hemmorrhage which could have been an additional etiology leading to the pea arrest. they also noted that while some of the decreased alertness and activity may be the result of the poor liver function and its inability to clear sedatives, the lack of return to function two days post was a poor prognostic indicator. they additionally recommended a monitoring eeg to rule out seizure activity. the eeg revealed nonconvulsive status epilepticus, and she was treated with keppra and ativan starting . the ncse continued on eeg for the next several days, ultimately ceasing in ncse activity by and showing slight improvement on exam but with no wakefulness and continued guarded prognosis. repeat ct head showed no interval changes and no evidence of hemorrhage or hypoxia. eeg monitoring, however, showed a burst suppression pattern which was thought to be an indicator of poor prognosis. eeg monitoring was stopped on . she continued to be minimally reactive even with sedation off throughout her final days and did not improve. neurology noted slim chances at improvement and suggested that her poor neurologic function could not be accounted for by metabolic or on-going multi-organ failure issues, possibly the result of ischemic insult to the brain. a family meeting was convened on with dr. (surgery), dr. (icu) and dr. (neurology) present to discuss goals of care. the team recommended continued support and the family concurred with wishes to continue all efforts. after deteriorating clinical status over the next week and increasingly grim chances of neurological recovery, a second family meeting was held, led by dr. on . her poor prognosis was explained and the family agreed to a plan of dnr but continued life-sustaining medications until further discussions with the rest of the family for comfort-measures-only and withdrawal-of-care. mr. , the patient's husband, indicated the family's desire to withdraw life-sustaining measures and switch to comfort-measures-only on . mrs. medications were discontinued. she was extubated at approximately 4:35 pm on saturday, with morphine administration for comfort. she was pronounced at 4:42 pm. of note, on an id front, mrs. was started on flagyl on for empiric therapy of loose stools thought to be c. diff. it was dc'd on after 3 negative samples. a bal done on grew coag + staph and moraxella. initially broad based antibiotic therapy was started (vanc/cipro/flagyl) and was further refined (nafcillin/cipro) when sensitivites showed mssa on . her wbc count was 13.7 immediately post-operatively and remained in the 11-18 range before gradually increasing to the mid-to-high 20s range in the icu.` discharge disposition: expired discharge diagnosis: 1. pancreatic head carcinoma 2. post operative delirium 3. liver failure 4. renal insufficiency 5. abdominal hemmorrhage discharge condition: deceased procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more parenteral infusion of concentrated nutritional substances insertion of endotracheal tube enteral infusion of concentrated nutritional substances other electric countershock of heart other bronchoscopy radical pancreaticoduodenectomy arterial catheterization cholecystectomy closed [endoscopic] biopsy of bronchus cardiopulmonary resuscitation, not otherwise specified diagnoses: pneumonia due to other gram-negative bacteria acute kidney failure with lesion of tubular necrosis unspecified essential hypertension acute posthemorrhagic anemia acute and subacute necrosis of liver atrial fibrillation other opiates and related narcotics causing adverse effects in therapeutic use hypopotassemia hemorrhage complicating a procedure pneumonitis due to inhalation of food or vomitus cardiac arrest malignant neoplasm of head of pancreas other specified surgical operations and procedures causing abnormal patient reaction, or later complication, without mention of misadventure at time of operation grand mal status methicillin susceptible pneumonia due to staphylococcus aureus diarrhea metabolic encephalopathy drug-induced delirium examination of participant in clinical trial secondary and unspecified malignant neoplasm of intra-abdominal lymph nodes coma acute and chronic cholecystitis pressure ulcer, unspecified stage other fluid overload pressure ulcer, unspecified site 

Answer: The patient is high likely exposed to