Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: s/p pea arrest major surgical or invasive procedure: none history of present illness: yof with history of htn, dm, hypothyriodism, bibems to osh ed s/p witnessed pea at home. per report, she was in her usual state of health till 2pm on , after returning from shopping all day. her husband fell onto her and caused her to fell. she banged her head against the wall on her way down. her granddaughter was present during this time and called 911. fire department arrived in 2 minutes, found patient to be non-responsive without a pulse and apnec. she received cpr via bls for 2 mins. when acls arrived noted patient to be unresponsive, in sinus brady, no shock was delivered. . once arrived to osh ed, she was given 80 of succ and 80 of propofol for rsi (7.5, 24cm at lip, position confirmed - 1 cm from carina). repeat rhythm was in nsr rate of 60-40. head ct was wnl, noted for c3-c6 fusion. abd us noted to have calcified aorta without dissection. patient was noted to be unresponsive per report of osh, and not moving her limbs. cooling was initiated at 1645 for a baseline of 35 with a goal of 33, this was done with 2l of cold ns. patient was given veccuronium, versed, propafol. ekg showed sinus, first degree av block, normal st-t waves, lad. labs were significant for wbc of 5.7, h&h of 8.8 and 26.7. chem7 was na 137, k 4.4, cl 107, hc03 21, bun 31, cr 1.2, glu 154. trop of 0.01. abg of 7.38, pco2 of 38, po2 483, hco3 23. at this point, she was transferred to for further managment. . in our ed, her inital vitals were: brady 30-40 sr, bp 130-140, sats 100% fio2 of 40%, peep of 5. temp was noted to be 32.8c with ice packs, and hr showed persistent bradycardia, cooling was temporarily stopped, then restarted. echo was performed in the ed by cardiology fellow: the left ventricular cavity size is normal. due to suboptimal technical quality, a focal wall motion abnormality cannot be fully excluded. overall left ventricular systolic function is normal (lvef>55%). the right ventricle appears slightly enlarged but is contracting well; sign is absent. the mitral valve leaflets are mildly thickened. trivial mitral regurgitation is seen. there is a very small pericardial effusion. there are no echocardiographic signs of tamponade. labs were similar to osh. ph of 7.45, pco2 26, po2 175, hco3 of 17. lactate of 0.9. access 18x2, 20. i/o 1.5l/1.25. . past medical history: 1. cardiac risk factors: diabetes (diet control), hypertension 2. cardiac history: - cabg: none - percutaneous coronary interventions: none - pacing/icd: none 3. other past medical history: - hypothyroid - s/p mvc and brain surgery for evacuation of blood (no seizure activity since) 10 yrs ago. - c3-c6 fusion - l hip surgery on . social history: - tobacco history: distant, quit 30 yrs ago. - etoh: drinks 1 drink per day (wine. - illicit drugs: none family history: - brother had early mi (30s), no other family history of arrhythmia, mi. physical exam: admission physical exam vs: t=31 bp=130/60 hr= 50 rr=16 o2 sat=100 general: nad. oriented x3. mood, affect appropriate. heent: ncat. sclera anicteric. perrl, eomi. conjunctiva were pink, no pallor or cyanosis of the oral mucosa. no xanthalesma. neck: supple with jvp of *** cm. cardiac: pmi located in 5th intercostal space, midclavicular line. rr, normal s1, s2. no m/r/g. no thrills, lifts. no s3 or s4. lungs: no chest wall deformities, scoliosis or kyphosis. resp were unlabored, no accessory muscle use. ctab, no crackles, wheezes or rhonchi. abdomen: soft, ntnd. no hsm or tenderness. abd aorta not enlarged by palpation. no abdominial bruits. extremities: no c/c/e. no femoral bruits. skin: no stasis dermatitis, ulcers, scars, or xanthomas. pulses: right: carotid 2+ femoral 2+ popliteal 2+ dp 2+ pt 2+ left: carotid 2+ femoral 2+ popliteal 2+ dp 2+ pt 2+ deceased pertinent results: admission labs 07:55pm blood wbc-5.4 rbc-2.97* hgb-8.9* hct-27.2* mcv-92 mch-29.9 mchc-32.6 rdw-14.6 plt ct-218 07:55pm blood neuts-83.4* lymphs-12.8* monos-2.8 eos-0.7 baso-0.3 07:55pm blood pt-11.0 ptt-28.1 inr(pt)-1.0 07:55pm blood glucose-125* urean-27* creat-0.9 na-142 k-3.5 cl-114* hco3-17* angap-15 12:42am blood alt-27 ast-40 ld(ldh)-243 ck(cpk)-577* alkphos-110* totbili-0.3 07:55pm blood ctropnt-0.03* 07:55pm blood calcium-8.0* phos-2.3* mg-1.7 08:11pm blood type-art rates-16/ tidal v-420 peep-5 fio2-40 po2-175* pco2-26* ph-7.45 caltco2-19* base xs--3 -assist/con intubat-intubated 08:08pm blood freeca-1.00* pertinent labs and studies 07:55pm blood ctropnt-0.03* 12:42am blood ck-mb-55* mb indx-9.5* ctropnt-0.03* 06:15am blood ck-mb-65* mb indx-11.0* ctropnt-0.02* 12:42am blood alt-27 ast-40 ld(ldh)-243 ck(cpk)-577* alkphos-110* totbili-0.3 06:15am blood ck(cpk)-591* 07:55pm blood tsh-3.5 echo there is symmetric left ventricular hypertrophy. the left ventricular cavity size is normal. due to suboptimal technical quality, a focal wall motion abnormality cannot be fully excluded. overall left ventricular systolic function is normal (lvef 65%). there is no ventricular septal defect. the right ventricular free wall is hypertrophied. the right ventricular cavity is dilated with borderline normal free wall function. the mitral valve leaflets are mildly thickened. trivial mitral regurgitation is seen. the tricuspid valve leaflets are mildly thickened. moderate tricuspid regurgitation is seen. the pulmonary artery systolic pressure could not be determined. there is a very small pericardial effusion. there is an anterior space which most likely represents a prominent fat pad. cxr single supine ap portable view of the chest was obtained. endotracheal tube is seen, terminating approximately the 3.3 cm above the level of the carina. nasogastric tube is seen coursing below the level of the diaphragm, distal aspect not included on the image. right perihilar opacity may relate to atelectasis or consolidation from aspiration, less likely infection. the left lung is clear. no large pleural effusion is seen, although a trace effusion may be difficult to exclude. the cardiac silhouette is mildly enlarged. the aorta is calcified and tortuous. deceased brief hospital course: yo f with history of htn, dm, hypothyroidism presents s/p cardiac arrest on cooling protocol, now deceased. #pea arrest - patient was found to be pulseless after her husband fell on top of her prior to admission. she received cpr in the field but no medications or shocks. she was brought to an osh where she was intubated and received 2l of chilled ns for post-arrest cooling protocol, she was transferred to for further management. the precise etiology of her arrest is not known, however it is not thought to be ischemic. here, she was started on dopamine for hypotension and cooling protocol was continued for 24 hours via arctic sun. she was rewarmed and sedation was weaned, her mental status began to slowly improve. tte after cooling showed a preserved ef with no wall motion abnornalities. there was briefly concern for pe on her repeat tte, but cta chest did not show a pe. #neurologic status - she was reported to be not moving her extremities in the field prior to arrival at the osh, which is why the cooling protocol was initiated after her arrest. after rewarming was complete, versed and fentanyl were weaned and her mental status slowly improved, although she had movement in all 4 extremities. head ct did not show any acute changes. continuous eeg during and after cooling did not show evidence of seizure. neurology was consulted and they felt that she would have low likelihood of having improvement in her mental status given her prolonged state of altered mental status due to severe anoxic brain injury. the patient's family expressed their wish to transition the patient to comfort measures only. the patient was extubated and continued to spontaneously breath until her death on at 11:33am. #potential aspiration and leukocytosis - there was initial concern for aspiration with subsequent respiratory arrest after her fall. she subsequently developed a worsening opacification on her cxr but, in alignment with her family's subsequent wishes for comfort measures only, the patient was not started on abx. #pneumomediastinum - there was a small amount of air in the mediastinum noted on cxr and ct chest. etiology is not entirely clear, this may have been introduced during right ij line placement. thoracic surgery was contact and felt that there was no intervention required. #anemia - her baseline hct is unknown, but she was found to be anemic with a normal mcv and hct in the 26-29 range during this admission. iron studies were normal. b12 and folate were 1009 and 9.6. there was no evidence of bleeding on exam. #htn - home lisinopril was held because of her hypotension at admission #hypothyroidism - her home levothyroxine was initially converted to the equivalent iv dose and was continued. #code status this admission: full code, transition to dnr/dni, transition to cmo. #transitional issues: deceased. medications on admission: - lisinopril 30 mg qd - famotidine 20 mg qd - synthyroid 88 mg qd discharge medications: none discharge disposition: expired discharge diagnosis: primary: pneumonia, respiratory failure, anoxic brain injury, cardiac arrest discharge condition: pt expired. discharge instructions: pt expired. followup instructions: pt expired. procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more arterial catheterization central venous catheter placement with guidance diagnoses: anemia, unspecified unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled unspecified acquired hypothyroidism other chronic pulmonary heart diseases other specified procedures as the cause of abnormal reaction of patient, or of later complication, without mention of misadventure at time of procedure acute respiratory failure other specified cardiac dysrhythmias pneumonitis due to inhalation of food or vomitus cardiac arrest anoxic brain damage encounter for palliative care do not resuscitate status emphysema (subcutaneous) (surgical) resulting from procedure accidents occurring in residential institution diseases of tricuspid valve arthrodesis status other fluid overload 

Answer: The patient is high likely exposed to