Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: altered mental status major surgical or invasive procedure: none history of present illness: 71 yo woman with hx recent d/c from for sdh presents with new mental status change. she has at baseline a right side hemiparesis and expressive aphasia. on discharge at her last admission she was responding in dysarthric, but coherent, one or two-word phrases to some questions. she presented to the hospital today after being found unresponsive by staff in her room. a fingerstick was checked at that time and 263. she was sent to ed and underwent a ct head which was concerning for acute on chronic sdh. she was given 20 mg of labetolol at osh and transferred to . ct head showed some slight increase of left side occipital and temperoparietal sdh. pt was seen by neurosurgery in ed, who found on no focal deficits or change from baseline, and felt there was not a significant enoughg change in her imaging to warrant surgical intervention. on speaking with the patient's daughter in law she is at baseline able to answer some simple questions and commands. she was occasionally able to feed herself (though with difficulty) and attend activities. her son thought that her mental status at was close to baseline and here at a little bit worse again to the point that she was only opening her eyes on command, but not talking or following commands. of note, she does have a dnr/dni order in the chart, but her family states that she did not want to be "hooked up to machines" but think that she would want to be resuscitated. we discussed that the two usually do go together and they agreed they would discuss it further and come up with a decision re: formal code status. . in ed, initial vs: 98.5 110 sr 220/110 16 94. she was given 20mg iv labetolol and 600cc ivf. repeat vs 103 113/88 22 98/ra. ekg showed sinus tach with slight st depression in i,ii. . she was admitted to the service for toxic metabolic workup. . past medical history: 1. stroke 30 years ago (? location-images not available) with residual right hemiparesis, dysarthria, and difficulty to express herself 2. type 2 dm, insulin dependent 3. hlp 4. atrial fibrillation, not on coumadin 5. h/o recurrent uti 6. seizure disorder, unspecified 7. h/o angina 8. pvd 9. chronic thrombocytopenia 10. anemia 11. depression 12. osteoporosis 13. dementia with delusional features 14. obesity 15. bilateral cataract surgery . prior admit for b chronic sdh and acute parafalcine sdh. social history: no alcohol, tobacco. lives in nursing home. son is hcp, niece is training to be a nurse and is involved in her care. family history: could not obtain. physical exam: on medicine admission vitals - t: 99.7 bp: 140/90 hr: 96 rr: 22 02 sat: 100% on 2l general: sleeping, arousable to tactile and loud verbal stimulus. not speaking or following commands consistently. heent: ncat. limited exam as pt does not follow direction. cardiac: rrr s mrg lung: cta on limited exam abdomen: s, nt/nd, +bs, large scar on r with firm scar tissue, no guarding or rebound tenderness ext: wwp, 2+ pulses, no edema or calf pain buttocks: 2 small 1cm skin tears vs. pressure ulcers r buttock. neuro: unable to assess as pt is uncooperative. notable r-sided hemiplegia. moves l arm and leg spontaneously (full neuro exam per neurosurgery) derm: no appreciable rashes. on intial neurology consult t- 99.9 (ax) bp- 213/103 (after lopressor, 181/105) hr- 97 rr-18 o2sat 92% ra fs=293 gen: sleeping, arousable to painful stimuli. not speaking or consistently following verbal commands. heent: nc/at, dry mucus membranes. limited assessment as patient does not follow commands cv: tachycardic, appears regular with no murmurs/gallops/rubs lung: clear to auscultation bilaterally on limited exam abd: +bs soft, nontender. ext: no edema, 2+ pulses neurologic examination: mental status: not alert or arousable to voice; eyes closed during exam; moves limbs (l>r) spontaneously to sternal rub, grimaces to noxious stimuli. does not follow commands. cranial nerves: i: not tested; ii: right pupil round, reactive to light 4 to 2 mm; left pupil oblong, minimally reactive to light. consensual. does not blink to visual threat. iii, iv, vi: eom not asessed; when patient's eyes are opened by examiner, appear to move in most directions. v, vii: gross right facial droop. viii: hearing appears intact to voice ix, x, , xii: could not assess motor: normal bulk bilaterally. tone normal. no observed myoclonus or tremor pronator drift not assessed. spontaneouslty touches face with left hand, though will not open eyes or follow commands. sensation: withdraws from painful stimuli bilaterally, localizes pain by moving bilateral upper extremities, making moaning sounds. reflexes: b t br pa pl right 0 0 0 0 0 left 0 0 0 0 0 left toe upgoing; right toe mute coordination and gait not assessed pertinent results: 02:35pm wbc-9.5 rbc-4.12* hgb-12.8 hct-40.4 mcv-98 mch-31.1 mchc-31.8 rdw-13.6 02:35pm plt count-172 lplt-3+ 02:35pm neuts-89.3* lymphs-8.1* monos-2.2 eos-0.1 basos-0.2 02:35pm tsh-0.58 02:35pm vit b12-631 folate-15.7 02:35pm calcium-9.7 phosphate-3.9 magnesium-1.6 02:35pm ctropnt-0.01 02:35pm lipase-48 02:35pm alt(sgpt)-30 ast(sgot)-33 ck(cpk)-41 alk phos-136* tot bili-0.5 02:35pm glucose-299* urea n-13 creat-0.8 sodium-135 potassium-4.2 chloride-97 total co2-26 anion gap-16 02:45pm urine bnzodzp-neg barbitr-neg opiates-neg cocaine-neg amphetm-neg mthdone-neg 2:32 pm rapid plasma reagin test (final ): nonreactive. ua negative on admission: 02:35pm urine rbc-* wbc- bacteria-few yeast-none epi- 02:35pm urine blood-sm nitrite-neg protein-500 glucose-1000 ketone-neg bilirubin-neg urobilngn-neg ph-7.0 leuk-neg 02:35pm urine color-yellow appear-clear sp -1.011 repeat ua contaminated: 09:24pm urine color-yellow appear-cloudy sp -1.015 09:24pm urine blood-tr nitrite-pos protein-30 glucose-neg ketone-neg bilirub-neg urobiln-neg ph-5.5 leuks-lg 09:24pm urine blood-tr nitrite-pos protein-30 glucose-neg ketone-neg bilirub-neg urobiln-neg ph-5.5 leuks-lg 09:24pm urine rbc-36* wbc-929* bacteri-occ yeast-none epi-36 leuk lg bld tr nitr pos prot tr rbc 5 wbc 433 bact many yeast none epi 23 cultures: 5:27 am blood culture, routine (final ): no growth. source: line-femoral 7:15 am blood culture, routine (preliminary): staphylococcus, coagulase negative. isolated from one set only. sensitivities performed on request.. 4:34 pm blood culture, routine (final ): no growth. source: line-femoral line. 6:23 am blood culture, routine (pending) time taken not noted (log-in date/time: 7:16 pm) blood culture, routine (pending) source: venipuncture. 10:51 am urine urine culture (pending) ct head w/o contrast study date of 3:28 pm impression: 1. acute on subacute left occipital and temporoparietal subdural hematomas. 2. evolution of subacute on chronic right occipital subdural hematoma 3. no change in subacute/chronic bilateral frontoparietal subdural hematomas. ct head w/o contrast: overall, minimal change from one day prior, with redemonstration of subdural hematomas bilaterally, of varying chronicity. there is no evidence of new intracranial hemorrhage. eeg: this is an abnormal portable eeg due to multiple electrographic seizures. these findings suggest a possible right focal onset for the seizure activity. frequency and duration of seizures, as well as onset from a post-ictal background, are consistent with the state of non-convulsive status epilepticus. as a result, video eeg monitoring is recommended for further characterization, evaluation, and treatment. eeg: this telemetry captured no pushbutton activations. routine sampling as well as seizure detection files showed frequent electrographic seizures over the right hemisphere consisting of initially a hz rhythm which transitioned to a 3 hz spike and wave pattern. the frequency of the seizures decreased significantly over the course of the study with no seizures being detected after 6:30 p.m. the majority of seizures were two to three minutes or less and no seizure appeared to last longer than four minutes. eeg: this telemetry captured no pushbutton activations and no clearly ongoing seizure activity. there were interictal discharges seen in a generalized distribution as well as focally in the left temporal region and the left frontal region. in addition, there was focal slowing seen in the left temporal area and generalized slowing suggestive of mild to moderate encephalopathy. brief hospital course: ms. is a 71 yo woman with a history of stroke, subdural hematomas and seizure who presented with altered mental status which progressed to unresponsiveness with notable occular deviation. # neuro/seizures: the patient was initially admitted to medicine but neurology was consulted within hours of admission for increased unresponsiveness and repetitive movements of her head and eyes. she had not recieved her complete dilantin doses in the 24 hours prior to admission. ct of the head was without any significant change in her chronic subdurals. she was loaded with dilantin given her sub-therapeutic level (1.8) and eeg was initiated. beside eeg monitoring revealed continued seizures overnight which did not respond to therapeutic dilantin. she was started on keppra and valproate as well as a midazolam drip and admitted to the icu for status epilepticus. her seizures resolved significantly both clinically and on eeg monitoring. she was transferred to the neurology service. on neurology, in an effort to streamline the medication regimen, the valproate was discontinued and keppra dose was increased. the patient continued phenytoin, and levels were monitored closely to remain therapeutic. # id: the patient had one blood culture drawn from her femoral line which revealed coag negative staph, treated with vancomycin. blood cultures from a different site at the same time revealed no growth (final), repeat blood cultures no growth (preliminary), so the vancomycin was discontinued upon discharge. the femoral line was removed on the day of discharge. a urinalysis prior to discharge appeared contaminated, but the patient was asymptomatic; repeat ua was still contaminated with wbc 433 and epi 23. she also had no fever or leukocytosis. we therefore opted not to treat her further for this ua. her urine cultures will need to be followed up after discharge. please call ( to obtain these results from the lab. # hypertension: in the icu, the patient received iv metoprolol and enalaprilat for blood pressure control. she was transitioned back to her home dose lisinopril on the floor. # hyperglycemia: the patient had significantly elevated blood sugars on admission (200-300), which may have been due to her not receiving her normal dose of glargine. she has been relatively well controlled here on her insulin regimen (glargine 10 units qhs, humalog sliding scale). # fen: the patient received tube feeds and medication by ng tube while antiepileptic medications were adjusted and she remained sleepy. ng tube was removed on the day prior to discharge. the patient passed a swallow evaluation on the day of discharge. labs pending at discharge: blood cultures , urine culture and medications on admission: verapamil 240 mg daily simvastatin 20 mg phenytoin sodium extended 200 mg qam/ 100mg qpm verapamil 120 mg tablet qpm metoprolol tartrate 75 mg tid lisinopril 40 mg daily glargine 10 units qhs, humalog sliding scale trazodone 50 mg qhs actonel 35 mg daily discharge medications: 1. verapamil 120 mg tablet sig: two (2) tablet po q24h (every 24 hours). 2. verapamil 120 mg tablet sig: one (1) tablet po qpm (once a day (in the evening)). 3. simvastatin 10 mg tablet sig: two (2) tablet po daily (daily). 4. levetiracetam 500 mg tablet sig: 1.5 tablets po bid (2 times a day). 5. lisinopril 20 mg tablet sig: two (2) tablet po daily (daily). 6. phenytoin 50 mg tablet, chewable sig: four (4) tablet, chewable po twice a day. 7. insulin glargine 100 unit/ml solution sig: ten (10) units subcutaneous at bedtime. 8. insulin lispro 100 unit/ml solution sig: sliding scale subcutaneous qachs: 2 units for every 40mg/dl that blood glucose is >120. if blood glucose is >400, notify md. 9. actonel 35 mg tablet sig: one (1) tablet po once a day. 10. trazodone 50 mg tablet sig: one (1) tablet po at bedtime as needed for insomnia. discharge disposition: extended care facility: rehabilitation and nursing center - discharge diagnosis: seizure due to chronic/subacute subdural hemorrhage. discharge condition: awake, smiling, blows kisses to examiner. says a few words (ie, 'love you') but does not speak in sentences. spontaneously moves left arm and leg. does not move right arm or leg. discharge instructions: you were admitted after having a seizure, most likely due to your history of subdural hemorrhages in your brain. your medication regimen was adjusted to maximize seizure control while keeping you from being too tired; keppra (levetiracetam) was increased, dilantin (phenytoin) was continued, and depakote (valproic acid) was discontinued. you received nutrition through a nasogastric tube while you were not awake enough to eat on your own; after you were less tired, the tube was removed an you passed a swallow evaluation. followup instructions: provider: scan phone: date/time: 10:15 provider: , md phone: date/time: 11:00 provider: . & phone: date/time: 4:30 procedure: enteral infusion of concentrated nutritional substances diagnoses: thrombocytopenia, unspecified coronary atherosclerosis of native coronary artery atrial fibrillation other persistent mental disorders due to conditions classified elsewhere other and unspecified hyperlipidemia long-term (current) use of insulin osteoporosis, unspecified grand mal status diabetes mellitus without mention of complication, type ii or unspecified type, uncontrolled obesity, unspecified dehydration encephalopathy, unspecified subdural hemorrhage late effects of cerebrovascular disease, aphasia hemiplegia, unspecified, affecting unspecified side delusional disorder benign essential hypertension 

Answer: The patient is high likely exposed to