Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: cc: major surgical or invasive procedure: none history of present illness: 66 m recently discharged to from neurosurg after being struck by car while on bike and sustaining head trauma (frontal and parietal contusion, subarachnoid, ivh but no fracture). intubated and monitored. trach'd and peg'd then d/c to . hosp course complicated by e. coli pneumonia for which was treated and a r superficial thrombus. per discussion with was discharged only responsive to noxious stimuli, but ? alert at rehab yesterday. developed tachy 10 107/tachypnea to 30 yesterday. started on levoquin . suctioning thick mucus. got hypoxic to low 90's on trach mask and transferred here to r/o pe. . here unable to obtain access for cta contrast, but cxr showed rml infiltrate. never hypotensive. past medical history: pmh: special needs mental retardation high functioning has been holding a job and taking care of his father - bike versus car s/p blunt head trauma with multiple brain contusion and subarachnoid, ivh --> intubated -- trach and peg -- baseline mental status on discharge only responsive to noxious stimuli social history: had taken care of father before accident, has special needs, has been at family history: nc physical exam: pe: vs: tm 100.6, tc 98.8, rr 20, 150/87 (130-150), 100% on 50% tm (7l o2) gen: no acute resp distress neuro: good gag pupils 3mm --> 2mm, blinks to threat in c-collar - doll's not tested unresponsive even to noxious stimuli withdrawl to deep pain bilateral lower ext, not upper dtr 2+ biceps/triceps, absent lower clonus bilat ankles, toes up on r, down on l upper ext tone normal heent: mmm, trach nonerythematous, no erosions lungs: cta b anteriolaterally, good aeration cv: s1/s2, tachy, no m/r/g abd: obese, nabs, peg in place looks good, no grimace with palpation ext: ecchymosis lle, dp 2+ bilat, no edema access: l picc brachial vein - no erythema or pus pertinent results: 04:37pm blood wbc-12.1* rbc-3.31* hgb-9.4* hct-29.4* mcv-89 mch-28.4 mchc-32.0 rdw-15.1 plt ct-636* 04:37pm blood neuts-72.5* lymphs-19.3 monos-5.8 eos-1.8 baso-0.6 07:42pm blood pt-16.6* ptt-33.4 inr(pt)-1.9 04:37pm blood glucose-142* urean-22* creat-0.6 na-143 k-4.2 cl-107 hco3-27 angap-13 04:37pm blood alt-69* ast-40 alkphos-161* totbili-0.5 10:39am blood type-art temp-38.7 rates-/40 fio2-94 po2-116* pco2-40 ph-7.47* calhco3-30 base xs-4 aado2-520 req o2-86 intubat-not intuba comment-trach mask 04:34pm blood lactate-1.1 . right upper extremity venous ultrasound : no evidence for deep vein thrombosis in the right upper extremity. cxr : a single semiupright view is compared to previous examination of . there is new right basilar atelectasis. the heart size remains stable. the small focus of consolidation in the left mid lung has resolved. there is also better aeration of the left lung base. there is no evidence of pulmonary edema. again, note is made of tracheostomy tube and left subclavian picc line with the tip in proximal svc. . cxr :there is a faint airspace opacity in the right middle lung zone, which is new in comparison to prior study, and may represent aspiration or pneumonia. there is bibasilar atelectasis, and small bilateral pleural effusions, greater on the left. no pneumothorax is seen. a left- sided picc line is seen, with the tip in the upper svc. a tracheostomy tube is seen with the tip approximately 4 cm above the carina. the pulmonary vasculature is stable in appearance. degenerative changes are seen within the mid thoracic spine. the mediastinal and cardiac contours are stable in appearance. impression: faint new airspace opacity within the right mid lung zone. as this is a new finding in a short time period, this is concerning for aspiration, though pneumonia should also be considered. again seen are small bilateral pleural effusions. . head ct :ct head without iv contrast: no new intracranial hemorrhage, hydrocephalus, shift of normally midline structures, or mass effect is identified. a small amount of hyperattenuating fluid consistent with hemorrhage is layering within the occipital horns of the lateral ventricles bilaterally, unchanged from the previous exam. bilateral frontal areas of hypoattenuation as well as within the splenium of the corpus callosum consistent with diffuse axonal injury and/or contusions are unchanged. partial opacification of the left maxillary, sphenoid, ethmoid, and frontal air cells is again identified. impression: 1. no acute intracranial hemorrhage or edema. 2. intraventricular hemorrhage again identified within the dependent portions of the lateral ventricles bilaterally, unchanged from the previous exam. 3. changes from diffuse axonal injury/parenchymal contusions again seen. . left lower extremity venous ultrasound : no evidence of left lower extremity deep vein thrombosis. brief hospital course: upon admission mr. was started on ceftaz, vanco, and flagyl for a possible aspiration pna. admission cxr showed rml infiltrate. he had been on levofloxacin since discharge on for continued treatment of pansensitive e. coli bacteremia and e. coli and enterobacter positive sputum, which was d/c'ed on arrival to the floor. overnight he required frequent suctioning of thick mucous and was transferred to the for closer monitoring. in the , mr. continued to be tachycardic and have copious secretions. he was continued on ceftazidime and vancomycin. flagyl d/c'ed on . in efforts to determine the etiology of his tachycardia (up to 120s), rue, rle and lle u/ss were done, which showed no evidence of thrombosis (despite pt's recent h/o rue superficial vein thrombosis). piv was attempted, but despite several attempts, could not be placed and, thus, cta to r/o pe could not be done. since no dvts were appreciated, coumadin was d/c'ed. a morphine trial was done , which resulted in no slowing of hr. his hr responded moderately to ivf, but remained somewhat tachycardic. a similar protocol was carried out on , with hr to 130s, not responding to morphine or lopressor, but did decrease to 90s after 500ml ns bolus. metoprolol was titrated up to 75mg tid. tachycardia possibly autonomic instability as result of his accident, possibly with mild overlying hypovolemia. due to nonspecific t-wave changes in anterior leads, was ruled out with negative trops x 3. mr. fio2 was weaned from 0.5 to 0.35 trach mask, and nursing noted that suctioning was required only intermittently (approximately 4 times yesterday), with less thick secretions than at admission. his temperatures, which were initially elevated, normalized, and he was afebrile by time of discharge. he was sent out on a somewhat altered regimen, with significantly increased metoprolol dose (25mg to 75mg tid). coumadin was also d/c'ed, as u/s's showed no evidence of thrombosis. he was discharged back to hospital for further management. medications on admission: rx: 1. docusate sodium 150 mg/15 ml liquid sig: one (1) po bid 2. bisacodyl 10 mg suppository sig: one (1) suppository rectal daily (daily) as needed. lansoprazole 30 mg susp,qd polyvinyl alcohol 1.4 % drops sig: 1-2 drops ophthalmic prn (as needed). albuterol 90 mcg/actuation aerosol sig: 1-2 puffs inhalation q6h (every 6 hours) as needed. acetaminophen 160 mg/5 ml solution sig: one (1) po q4-6h (every 4 to 6 hours) as needed. oxycodone-acetaminophen 5-325 mg/5 ml solution sig: 5-10 mls po q4-6h (every 4 to 6 hours) as needed. ferrous sulfate 300 mg/5 ml liquid sig: one (1) po daily (daily). miconazole nitrate 2 % powder qid prn metoprolol tartrate 25 mg levofloxacin 500 mg po qd (started ) ipratropium bromide albuterol sulfate 0.083 % warfarin 5 mg tablet sig: one (1) tablet po once (once) for 1 doses. was using hydralazine in neuro sicu for bp control, none at rehab discharge disposition: extended care facility: - discharge diagnosis: pneumonia tachycardia discharge condition: stable. decreased secretions, sao2 stable on 35% trach mask, afebrile. on iv antibiotics. discharge instructions: please take your medications as prescribed. . you should return to the hospital if you develop worsening o2 saturations, weakening cough reflex, significantly increased secretions, fever, or any other concerning problems. followup instructions: you will be followed at facility, where your care will be further managed. procedure: enteral infusion of concentrated nutritional substances diagnoses: anemia, unspecified unspecified essential hypertension infection with microorganisms resistant to penicillins other specified cardiac dysrhythmias pneumonitis due to inhalation of food or vomitus alkalosis methicillin susceptible pneumonia due to staphylococcus aureus gastrostomy status tracheostomy status 

Answer: The patient is high likely exposed to