Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of the present illness: the patient is an 82-year-old woman with a past medical history of cardiomyopathy, nonischemic, chf, a fib, admitted to the ccu from for v tach at approximately 180 beats per minute at roughly 1:30 a.m. on . the patient began at midnight. she received a bolus of 150 mg of amiodarone beginning at 12:20 a.m. and then had an amiodarone drip placed in the unit. her systolic blood pressure dropped to 75. she complained of being sweaty, shortness of breath. a fluid bolus was given. she was 94% on 2 liters nasal cannula. the patient converted from v tach to a paced rhythm at roughly 90 beats per minute spontaneously while bearing down prior to second dose of amiodarone and prior to paging anesthesia for conscious sedation for cardioversion. her symptoms resolved which returned to her baseline rhythm. initially in the afternoon of , the patient complained of palpitations. she called ems and was found to be in v tach by the ems team. in route to the hospital, her rhythm remained in v tach despite 13 shocks at 100 joules. the patient converted to sinus rhythm, bundle branch block, conduction delay. she was loaded with amiodarone during the cardioversion. she also received 100 mg of lidocaine, adenosine 6 mg prior to amiodarone. at , she went to the ep laboratory where v tach was noninducible and she was found to have an infra his block. a ddd pacer was placed. the patient complained of shoulder pain secondary to shocks. in the ccu she was stable and loaded with amiodarone on 1 mg per minute iv. past medical history: 1. nonischemic cardiomyopathy. 2. chf. 3. a fib. 4. hypercholesterolemia. 5. squamous cell skin cancer, status post removal on left arm. at-home medications: 1. digoxin 0.125 mg q.d. 2. lasix by weight, baseline dry weight; she normally takes lasix when her weight is greater than 145 pounds. cardiologist is dr. . allergies: she has an intolerance to statin. social history: sixty-year pack history of one alcohol drink per week and no drugs. she is a widow. she has one son who is involved in her care. we discussed her case with her pcp who said that the patient was unable to tolerate an ace inhibitor and had trouble tolerating beta blockers in the past. physical examination on admission: vital signs: status post the episode of vt on , in the morning, roughly at 2:00 a.m., the patient's vital signs revealed a pulse of 91, blood pressure 120/53, 98% on 2 liters nasal cannula, breathing at 15-22, not in any acute distress. general: the patient was elderly, younger appearing than stated age. heent: eomi. perrla. anicteric. jvd approximately 9 cm. heart: regular rate and rhythm. there was a iii/vi holosystolic murmur at the apex. lateral pmi. crackles at the left base laterally and anteriorly. abdomen: soft, no distention, no tenderness, normoactive bowel sounds. extremities: there was dorsalis pedis pulses bilaterally. no clubbing, cyanosis or edema. neurologic: a&o times three. cranial nerves ii through xii intact. pleasant. right groin bruit, slightly ecchymotic. hospital course: the patient is an 82-year-old woman with a history of nonischemic cardiomyopathy, chf, who presents to the ccu status post an episode of v tach, status post multiple shocks that were unable to resolve the v tach, status post ep study without inducibility of the rhythm, although the patient was found to have an infra his block resulting in a ddd pacer being placed. the patient received boluses of amiodarone 150 mg times one and was loaded on iv amiodarone for the v tach. 1. cardiology: rhythm/v tach: after the load of 1 mg per minute and vagal bang down broke into normal sinus rhythm. the amiodarone was initially decreased to 0.5 mg per minute. she was converted to amiodarone 400 mg p.o. q.d. and the amiodarone was discontinued after the patient had been loaded sufficiently. the patient was started on low-dose of beta blocker 25 p.o. b.i.d. the patient had a run of sustained v tach on at approximately 4:00 p.m. and was given iv 2.5 lopressor and the rhythm spontaneously reverted back to normal sinus rhythm. she had a 50 plus beat run of v tach at 8:55 a.m. on , again nonsymptomatic and then the ectopy tended to decrease, having three to four beats of nsvt on and four beats on . the lopressor was converted to toprol xl 50 mg q.d. at discharge. of note, the patient received amiodarone 400 mg b.i.d. for one week and then was transitioned to 400 q.d. hemodynamics: on the morning of , when she first presented to the ccu, roughly 45 minutes after going into normal sinus rhythm, the patient's blood pressure dropped into the 70s. she was bolused iv fluid with her blood pressure going up to the 90s and then would drop again to the 70s when her iv fluid was not running. she received approximately 600-700 cc with her blood pressure going to 90-100, heart rate in the 70s, pulsus paradoxus at the time was 5 mmhg. repeat hematocrit showed 31.2. it had been 37 the day before. therefore, she received 1 liter of iv fluid. repeat hematocrit was 31.8, stable. the cardiology fellow was called. he did an echocardiogram at the bedside which showed none/trace pericardial effusion, global lvhk. the left ventricle was very enlarged, more so than the right ventricle. no ra collapse. the blood pressure, status post 1 liter of iv fluid, was stabilized in the 110s/50s with heart rate in the 70s and afebrile at 98.2 and breathing at 14-21, 97% on 2 liters nasal cannula. 2. congestive heart failure: the patient has the cardiomyopathy considered to be nonischemic. she had a repeat echocardiogram done on a nonemergent setting with an ejection fraction of 20%. tr gradient of approximately 30 mmhg. the left atrium was mildly dilated. the right atrium was normal in size. left ventricular wall thickness was normal. left ventricular cavity was mildly dilated with severe global lvhk. no resting lv outflow tract obstruction. no mass or thrombi seen in the left ventricle. right ventricular wall thickness was normal. right ventricular chamber size was normal. right ventricular systolic function appeared depressed. aortic root was normal in diameter. there were focal calcifications in the aortic root. the aortic valve was mildly thickened and not stenotic. mitral valve leaflets were mildly thickened. no mvp. mild annular calcification. there was mild thickening of the mitral valve chordae. there was a systolic anterior motion on mitral valve leaflets. there was no significant mitral stenosis, moderate to severe 3+ mr was seen. tricuspid valves were mildly thickened. mild tricuspid regurgitation was seen, +1. mild pulmonary systolic hypertension. the patient's weight rose to 147. normally, her lasix was initiated with a weight greater than 145. she received actually 80 mg p.r.n. we started her on 20 mg p.o. q.d. dose for more even diuresis and fluid shifts. she had very good urine output and negative 1.6 liters on the first day of this regimen. her weight went from 147 to 143. , losartan, was also added for afterload reduction as the patient was unable to tolerate an ace inhibitor as an outpatient. 25 q.i.d. was started. the patient initially had reported some orthopnea and pnd when her weight was higher and dyspnea on mild exertion with the physical therapist at the scene. on the date of discharge on this regimen, she was able to ambulate in the hallway without any complaints, any shortness of breath, and her pnd was reduced from earlier in the week. she was placed on a 2 gram sodium diet. 3. infra-his block: the patient had a ddd pacer placed as previously mentioned. status post her pacemaker placement, the patient developed a right groin bruit secondary to a pseudoaneurysm. an ultrasound of the pseudoaneurysm showed it to be greater than 2 cm. interventional radiology embolized the aneurysm with good result. 4. hematology: the patient's hematocrit was 35 on admission. status post vigorous hydration and blood draws, it dropped to 31.2 and then was stable, roughly 31 throughout her stay. 5. electrolytes: her electrolytes, especially her potassium, magnesium, and calcium, were vigorously monitored and supplemented secondary to the vt. , m.d. dictated by: medquist36 procedure: catheter based invasive electrophysiologic testing initial insertion of dual-chamber device initial insertion of transvenous leads [electrodes] into atrium and ventricle injection or infusion of other therapeutic or prophylactic substance diagnoses: other primary cardiomyopathies pure hypercholesterolemia congestive heart failure, unspecified atrial fibrillation paroxysmal ventricular tachycardia peripheral vascular complications, not elsewhere classified aneurysm of artery of lower extremity 

Answer: The patient is high likely exposed to