Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: sulfa (sulfonamide antibiotics) / penicillins / penicillins / clonidine / clonidine / azithromycin attending: chief complaint: r sided weakness major surgical or invasive procedure: tpa administration right femoral line placement intubation history of present illness: reason for consult; code stroke time code stroke called: 0850 (24h clock) time neurology at bedside for evaluation: 0855 time (and date) the patient was last known well: 0820 nih stroke scale score: 20 t-: yes time t-pa was given: 0944 i was present during the ct scanning and reviewed the images instantly within 20 minutes of their completion. nih stroke scale score was 20: 1a. level of consciousness: 0 1b. loc question: 2 1c. loc commands: 2 2. best gaze: 2 3. visual fields: 2 4. facial palsy: 2 5a. motor arm, left: 0 5b. motor arm, right: 4 6a. motor leg, left: 0 6b. motor leg, right: 2 7. limb ataxia: n/a 8. sensory: 2 9. language: 2 10. dysarthria: n/a 11. extinction and neglect: n/a hpi; yo f with hx afib not on anticoagulation, htn, hld, p/w acute-onset r facial droop, r weakness, speech difficulties this am. per home health aide (via telephone, ), last night she was hypertensive (196/82) but otherwise conversant and appeared at her neurological baseline. she went to bed and slept later than usual, waking up at 0800 instead of 0700. she hesitated to get into her wheelchair but was able to independently transfer to her chair and appeared at baseline at 0815 and said "good morning" to her hha. she went to use the bathroom and was able to independently get to the toilet and shortly after (approx 0820) her hha saw her slumped over with r facial droop, r-sided weakness, and inability to speak. no seizure activity noted. ems was called and she was transferred here. she has had some mild "stomach issues" recently per hha but no vomting, diarrhea, or bleeding, and had undergone some testing at last week but no invasive testing. she also was supposed to start an antibiotic today for an unknown infection. no known history of prior strokes, bleeds, head trauma, head surgery, or recent surgical procedures per chart review and hha. her hha states she does not take coumadin, aspirin, plavix, or any known antiplatelets or anticoagulants. ros unobtainable by patient. past medical history: -afib (self discontinued anticoagulation over 10 years ago) -htn -clonidine-induced lupus like syndrome diagnosed in and followed by rheumatology at -hyperlipidemia -hyponatremia -gerd -bilateral ovarian clear cell carcinoma -breast carcinoma -mitral regurgitation mild -positive ppd -multinodular goiter s/p rai -glaucoma -basal cell carcinoma -squamous cell carcinoma -epistaxis in setting of aspirin use -s/p tsa-bso -s/p r shoulder surgery -s/p cholecystectomy -s/p eye surgery for glaucoma social history: - born in ; moved to us in - lives alone but recently has hired a hungarian woman ms. to serve as a 24hr caregiver - retired child psychiatrist still very active in her field, attending conferences and volunteering - denies tobacco, ivda, etoh, herbal medications - only relative is her distant who is her hcp and lives in family history: noncontributory physical exam: vs; bp initially 179/76, up to 207/99 in ct scanner, s/p 10 mg iv labetalol with decrease to 170s/80s. p 62 rr 26 92% ra. gen; lying in stretcher, nad heent; nc/at, mucous membranes moist cv; irrregularly irregular pulm; cta anteriorly abd; soft, nontender extr; no edema neuro; ms; opens eyes to voice. appears to make effort to respond but is nonverbal. does not close eyes to command but appears to squeeze left hand to command. cn; perrl 3mm and minimally reactive. eyes conjugately deviated to the left and unable to passively overcome midline. does not blink to threat on the right. right facial droop. motor; able to maintain lue antigravity and moves lle in plane of bed. spontaneously moves rle as well intermittently. no movement of rue. sensory; no grimace to noxious in rue, rle. pertinent results: 08:55am blood wbc-17.6* rbc-4.19* hgb-12.6 hct-35.1* mcv-84 mch-30.0 mchc-35.8* rdw-16.5* plt ct-470* 06:40am blood wbc-9.2# rbc-3.61* hgb-10.9* hct-31.6* mcv-88 mch-30.0 mchc-34.3 rdw-17.3* plt ct-356 05:45am blood glucose-152* urean-28* creat-1.0 na-134 k-3.3 cl-91* hco3-26 angap-20 05:45am blood alt-20 ast-40 ck(cpk)-82 alkphos-83 totbili-1.4 05:45am blood ck-mb-4 ctropnt-0.04* echocardiogram: no cardiac source of embolism seen. suboptimal image quality due to body habitus. left ventricular systolic function is probably normal, a focal wall motion abnormality cannot be excluded. the right ventricle is not well seen but may be dilated/hypokinetic. mild mitral and trace aortic regurgitation. mildly elevated pulmonary artery systolic pressures. mild to moderate dilation of the ascending aorta and aortic arch. cta/p of head: 1. no evidence for intracranial hemorrhage. 2. areas of ischemic penumbra in the left mca territory along with areas of acute infarction. 3. decreased flow in the left internal carotid artery in the petrous segment with no flow in the left cavernous and supraclinoid segments to the termination, likely from extensive thrombosis, less likely embolic. minimal flow in the left mca and its branches. short segment narrowing of right vertebral artery at the origin and v4 segment. significant atherosclerotic disease- aortic arch, brachiocephalic trunk, common caortid artery bifurcation and proximal internal carotid arteries bilaterally with moderate stenosis. 4. bilateral pleural effusions. correlate with dedicated imaging. 5. heterogeneous enlarged thyroid gland with nodules in both lobes. ultrasound is recommended when the patient is clinically stable if not done previously. 6. diffuse osteopenia and degenerative changes in the cervical and thoracic spine. ct head - follow-up scan: increased hypodensity and sulcal effacement within the left mca territory, consistent with evolving infarct. abdominal x-ray: a non-obstructive bowel gas pattern is visualized. a large amount of stool is present in the distal colon. there is no evidence of free intraperitoneal air. note is made of a feeding tube, with tip terminating in the body of the stomach. brief hospital course: in brief, this is a yo f with hx afib not on anticoagulation, htn, hld, p/w acute-onset r facial droop, r weakness, speech difficulties this am. per home-health aide she was last seen at baseline around the morning of . exam on admission notable for aphasia, left gaze deviation, right facial droop, plegic rue, weak rle, concerning for large left mca stroke, possibly cardioembolic in setting of afib without anticoagulation. cta head showed l ica cutoff and decreased flow in l mca. given high nih stroke score and early infarct signs on ct head, her hcp was to discuss the risks and benefits of thromboylsis. concern for poor outcome regardless of treatment as well as high risk for hemorrhage were discussed, but after discussion between stroke fellow, dr. and hcp via telephone, it was decided to proceed with iv-tpa. . after tpa, her head ct showed a complete l mca infarct without hemorrhage. she had no improvement in her mental status and remained aphasic. she was hemodynamically stable and transferred to 11. overnight it was noted that her abdomen was large, hard and distended. the decision was made to obtain a ct abdomen with contrast to better characterize. . while in the ct scanner, she began vomiting copious coffee ground emesis. her airway was obstructed and she became pulseless. consistent with her full code status, chest compressions were started. chest compressions continued were continued for 20 minutes, and the patient was intubated with a right femoral line placed. the hcp, was , and he stated that the patient would not want prolonged resuscitation. chest compressions were stopped. at that time, the patient was noted to have regained a pulse. levophed was started and the patient was transported to the sicu. . the hcp was again , and on hearing the patient's poor prognosis, indicated that he would like her to be made cmo. on arrival to the sicu, the patient was hypotensive to 70/30 and her pupils were non-responsive. she was extubated and appeared peaceful. the chaplain was . declared dead 12pm . hcp and pcp . medications on admission: -senna qhs -docusate 100 mg -tylenol prn -omeprazole 20 daily -diltiazem 120 mg sr tid -losartan 50 mg -prednisone 5 mg daily discharge medications: none discharge disposition: expired discharge diagnosis: mca stroke aspiration pea arrest atrial fibrillation discharge condition: expired discharge instructions: patient expired followup instructions: n/a md, procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified insertion of endotracheal tube enteral infusion of concentrated nutritional substances injection or infusion of thrombolytic agent cardiopulmonary resuscitation, not otherwise specified diagnoses: abdominal pain, unspecified site esophageal reflux unspecified essential hypertension atrial fibrillation personal history of malignant neoplasm of breast other and unspecified hyperlipidemia personal history of other malignant neoplasm of skin pneumonitis due to inhalation of food or vomitus cardiac arrest anoxic brain damage cerebral embolism with cerebral infarction aphasia leukocytosis, unspecified hemiplegia, unspecified, affecting unspecified side facial weakness physical restraints status personal history of malignant neoplasm of ovary 

Answer: The patient is high likely exposed to