Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: phenytoin / depakote / gabitril / zarontin / dilantin attending: chief complaint: increasing seizure frequency major surgical or invasive procedure: none history of present illness: per admitting resident: the patient is a 40-year-old rh woman with a history of mild mental retardation, cerebral palsy, who has had a worsening seizure disorder associated with language regression since . she has been followed up at epilpesy centre for management of sz by dr. since about 8 -10 years. she was recently seen in the office in and was noted to tolerate her meds well with significant side effects and per parents , this was probably the best regime so far for her. she now presents to ed with 4 episodes of seizures over 24 hrs, she called dr. who advised to be seen in ed. she had 4 grand mal seizures over last 24 hrs , while i was examining her she had grand mal seizure lasting for about 30 secs, she was given ativan 1 mg iv immediately in er. her seizures over last 24 hrs have been all similar in simiology. sudden onset jerky movements of all 4 limbs ( ul more than ll) with eyes rolling up, and tongue bites. she had incontinence in at least 1 of them . the first one was 11 am yesterday (), lasted for 45 secs with post ictal drowsiness for about an hour. second one was 7 pm on , third one 11pm on and fourth one 7.30 am on , each lasted for 30 secs, with post ictal for bout 15-20 mins. as i was examining her,(1 pm) she had an episode of gtcs , lasted for about 30 secs and she was post ictal after that. she seized again at about 1.35 pm, gtcs, lasted for 30 secs, and recieved ativan 1 mg iv again. precipitating factor history she had h1 n1 vaccine about 1 week ago, denies fevers rash coughing diarrhea. no increase in urinary frequemcy. no head trauma, or injury or fall. no change in behaviour from her usual self. no known sick contacts (though goes to day care once a week). her parents are very careful about meds and no h/o missing any doses. no recent use of antibiotics. no travels. typical seizure types and frequency grand mal - once a year others- combination of atonic, mal or myoclonic jerks very brief duration , /day past medical history: detailed sz history she is the full term born of uncomplicated pregnancy with prolonged labor and cesarean section. she was subsequently well, but noted to have an abnormal gait at age 3 and was first evaluated by neurology at age 5 for poor performance in kindergarten. at that time she had a mildly ataxic gait, mild upper extremity dystonia, poor attention, and she was felt to have mild mental retardation, cerebral palsy primarily with motor problems and gross motor function at the 3-year-old level. she was followed at with yearly scales and biyearly neuropsychological testing. her intelligence portion progressed at approximately 60 to 70 level, but with a slow decline in verbal iq scores over years. she has had difficult holding jobs due to poor spacial and attentional skills, and she has continued to have upper extremity dystonia and an ataxic gait with progressive decline over years making her dependent now on others for nealr all adls. she started having sz from . a typical seizure type is brief tonic or atonic seizures, often with the head turned to one side or the other for 5 to 10 seconds, with rapid resolution, but subsequently 5 to 10 minutes post-ictal drowsiness or confusion and decreased speech. she also gets grand mal seizures with frequency about 1/yr. a vagal stimulator was implanted in without significant benefit and has been turned off since . med history dilantin and developed an allergic reaction and rash. tegeretol phenobarbitol, depakote, zarontin (developed rash), tranxene (sedation), klonopin (poor sz control), felbatol,lamictal( small and incomplete response), neurontin (drowsiness and no significant effect on seizures), topamax were tried and discontinued wither due to poor respons or side effects. she has been extensively worked up in the past with mri and metab panel which showed increased signal intensity in a punctate distribution in the centrum semi ovale bilaterally and aa screen had amino acid screen whi social history: she lives at home with her parents. she is now wheelchair bound and requires assistance with basic adls, but can feed herself the time. her parents and a caregive at home provide most of the care. family history: her family history is negative for seizures except for a maternal first cousin. there is no history of developmental delay in other family members, and she has a 22 year old normal sister. she is currently living with both parents. physical exam: exam on admission: t- 98.1 bp- 144/124 hr- 110 rr- 18 o2sat 98 gen: sitting in chair, seems unconcerned with surroundings heent: nc/at, moist oral mucosa neck: supple cv: rrr, nl s1 and s2, no murmurs/gallops/rubs lung: air entry equal , no crackles/rhonchi abd: +bs soft, nontender ext: no cyanosis/clubbing/edema neurologic examination: (limited as she was given ativan twice due to her 2 gtcs in er and was post ictal and drowsy) mental status: head slumped towards left side, does not open eyes to commands, grimaces on noxious stimuli, withdraws all 4 limbs on noxious stimuli. cranial nerves: pupils equally round and reactive to light, 5 to 4 mm bilaterally (min reactive). eyes set at midline without mvmt. no nystagmus. no gross facial asymmetries. motor: normal bulk bilaterally. tone normal. no observed myoclonus or tremor ( before being post ictal, i observed semi purposeful movements of both ue esp on right, that was told to me as her 'usual' by her parents. moves all 4 limbs and withdraws to pain sensation: withdraws to noxious stim in all 4 ext. reflexes: +1 and symmetric all over on both sides. toes mute on both sides. exam at time of discharge: pertinent results: labs on admission: 11:15am blood wbc-15.4*# rbc-3.99* hgb-12.6 hct-37.1 mcv-93 mch-31.7 mchc-34.0 rdw-12.6 plt ct-287 04:40am blood wbc-8.3 rbc-3.55* hgb-11.2* hct-33.9* mcv-96 mch-31.5 mchc-32.9 rdw-13.1 plt ct-263 11:15am blood neuts-82.4* lymphs-12.5* monos-3.6 eos-0.9 baso-0.5 02:38am blood neuts-65.1 lymphs-26.9 monos-5.1 eos-2.3 baso-0.6 02:38am blood pt-12.8 ptt-28.8 inr(pt)-1.1 11:15am blood glucose-91 urean-14 creat-0.6 na-141 k-3.9 cl-105 hco3-28 angap-12 07:16pm blood alt-9 ast-19 alkphos-79 totbili-0.3 02:38am blood calcium-8.7 phos-3.0 mg-1.9 07:16pm blood albumin-4.0 07:16pm blood tsh-3.3 tox screen: 07:16pm blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 05:08pm urine bnzodzp-neg barbitr-neg opiates-neg cocaine-neg amphetm-neg mthdone-neg aed levels: 02:38am blood levetiracetam (keppra)-pnd 02:38am blood trileptal-pnd urine studies 05:08pm urine rbc-0-2 wbc- bacteri-many yeast-none epi- 05:08pm urine blood-neg nitrite-pos protein-30 glucose-neg ketone-neg bilirub-neg urobiln-0.2 ph-5.0 leuks-tr csf studies: 03:52pm cerebrospinal fluid (csf) wbc-1 rbc-1* polys-0 lymphs-84 monos-16 03:52pm cerebrospinal fluid (csf) totprot-35 glucose-64 imaging: ct head; impression study limited by motion. no large acute intracranial hemorrhage/mass effect. consider mr of parenchymal/structural causes for seizures. eeg ; impression: this telemetry captured no pushbutton activations and no clear ongoing seizure activity. interictally, in sleep, there were frequent, sometimes sharp generalized high amplitude waves with a triphasic morphology, suggestive of interictal epileptic activity. of note is that most of this recording was obtained during sleep. eeg ; impression: this 24-hour video eeg telemetry captured one pushbutton activation for an episode in which the patient was moving her legs. this did not appear to be an electrographic seizure. no change on eeg was seen around the time of this activation. however, interictally, frequent bursts of generalized spike and slow wave discharges, sometimes recurring at a 2-2.5 hz frequency for up to a few seconds at a time, were seen. these had no clinical correlate on video. these findings suggest the presence of an underlying epilepsy, possibly primary generalized in nature, although focal epilepsy with rapid secondary generalization cannot be excluded. the background was slow and disorganized throughout most of the recording suggestive of an encephalopathy. eeg ; impression: this 24-hour video eeg telemetry captured no pushbutton activations. no definite electrographic seizures were seen. however, frequent interictal bursts of generalized spike and slow wave discharges were seen, sometimes with a rightsided predominance, and sometimes occurring in runs lasting up to a few seconds with a hz frequency. these had no obvious clinical correlate. these findings suggest an underlying epilepsy, either primary generalized or focal with rapid secondary bilateral synchrony. the background was mostly slow and disorganized throughout the day suggestive of an encephalopathy. brief hospital course: 40 yo rhf with complicated h/o sz, mr, cp, presented to er with 4 epiosodes of gtc at home and 2 in er in last 24 hrs. there was no evidence on history of infection or medication non-adherence, however she did have an elevated wbc count. given the significant increase in frequency of her gtcs and somnolence, she was admitted to the neuro icu for monitoring and further evaluation. the patient was started on ativan 1mg q4h for seizure control given gtcs in the ed, her keppra was increased to 3g daily and continued on home dose of oxcarbamazepine. she was evaluated with a continuous eeg which showed interictal spikes but no generalized convulsions. her infectious evaluation revealed a positive ua, for which she was started on bactrim. the ultimate reason for patient's decompensation was unclear, although a uti predisposing to increasing seizure activity was suspected. the remainder of evaluations, including lp, cxr, u/serum toxicology screens and bmp were unremarkable. a head ct was performed that showed no ich or other large acute pathology. the patient did not have any further clinical seizures during her hospital course. she was somewhat tachycardic with a heart rate of 100-120 (sinus). this had mild response to iv fluid and it was thought she may have been mildly dehydrated at the time of admission. the patient had a temperature of 101.5 on the evening of and a repeat infectious workup has been negative to date. her ativan was slowly tapered and patient was noted to appear back at her baseline as per her parents at the time of discharge. continuous eeg was notable for frequent interictal bursts of generalized spike and slow wave discharges, sometimes with a rightsided predominance, and sometimes occurring in runs lasting up to a few seconds with a hz frequency. these had no obvious clinical correlate. she was discharged home on the increased dose of keppra and otherwise maintained her home medications. medications on admission: oxcarbazepine- generic- 2400/d, keppra- generic- 2500/d, 1000/1500 calcium supplements lorazepam 1 mg po prn anxiety (uses /month) discharge medications: 1. oxcarbazepine 600 mg tablet sig: one (1) tablet po qam (once a day (in the morning)). 2. oxcarbazepine 600 mg tablet sig: one (1) tablet po qpm (once a day (in the evening)). 3. oxcarbazepine 600 mg tablet sig: two (2) tablet po qhs (once a day (at bedtime)). 4. levetiracetam 500 mg tablet sig: three (3) tablet po bid (2 times a day). disp:*180 tablet(s)* refills:*2* 5. calcium acetate 667 mg capsule sig: two (2) capsule po tid w/meals (3 times a day with meals). 6. cholecalciferol (vitamin d3) 400 unit tablet sig: one (1) tablet po daily (daily). discharge disposition: home with service facility: all care vna of greater discharge diagnosis: primary: generalied tonic clonic seizures secondary: epilepsy, mr, cp discharge condition: hemodynamically stable. patient is arousable to voice. nonverbal but tracks with eyes. moves all extremities equally against gravity. discharge instructions: you were admitted to with increasing frequency of your generalized tonic clonic seizures. this was felt to be most likely due to a urinary tract infection which you received antibiotics for. you required a temporary stay in neurological intensive care unit and were monitored on continuous eeg monitoring without any further seizures during your hospital course. please continue your medications as prescribed and follow up with your neurologist and pcp as directed. should you experience more generalized tonic clonic seizures (grand mal), please call your neurologist immediately or go to the emergency room. should you experience any of the following concerning symptoms, please call your doctor or go to the emergency room. followup instructions: neurology: provider: , m.d. phone: date/time: 10:00 procedure: spinal tap incision of lung enteral infusion of concentrated nutritional substances video and radio-telemetered electroencephalographic monitoring diagnoses: urinary tract infection, site not specified epilepsy, unspecified, without mention of intractable epilepsy dehydration infantile cerebral palsy, unspecified mild intellectual disabilities other nonspecific findings on examination of urine 

Answer: The patient is high likely exposed to