Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: doe, chest pressure major surgical or invasive procedure: none history of present illness: pt is a 35 year-old man who has a h/o palpitations/svt for over 10 years s/p ablation of two left sided accessory pathways in who now presents with svt with chest pressure and sob. . per dr. last omr note in , "these were documented to be a long rp tachycardia, which turned out to be in a left-sided accessory pathway. (from to the pt underwent a pt activated recorder that made note of episodes of afib that occurred right after runs of rapid svt with brief conversion to sinus. prominent st depressions were noted during these episodes also. the majority of the episodes were a long rp tachycardia that occasionally degenerated into atrial fibrillation.) he underwent a mapping and ablation of his pathway in , localized to two locations on the left side of the mitral annulus. these were ablated. for the following seven months, he was free of symptoms whatsoever. he then began to develop a recurrence of palpitations, however, these were distinctly different than his supraventricular tachycardia. they were less intense and shorter in duration. in retrospect, he felt a similar feeling after some of his more typical svt episodes prior to his ablation. further monitoring () found that he is having runs of paroxysmal atrial fibrillation. he occasionally has a narrow complex tachycardia preceding this, which looks like an atrial tachycardia, perhaps the pulmonary vein etiology. in general, he is doing quite well with these and only has enduring periods of heightened stress. when relaxed, he seems to be very quiescent from any arrhythmia standpoint." . pt. had been in his usual state of health until last night before admission when he couldn't sleep, feeling subjectively hot and cold. he developed chest pressure when lying on left chest starting roughly around mn. he also notes doe, feeling winded when climbing one flight of stairs. he took atenolol 50 mg prn (he takes prn); however, symptoms persisted until he saw his pcp 6pm, who noted svt with rate of 170, and sent him to ed. possible triggers recently include several stressors in his life, uri symptoms (earache), recent etoh on saturday, 2 cups of coffee daily chronically. . in ed, had unsuccessful cardioversion attempted with ibutilide, successfully converted with 200j without complication. cxr showed mild pulmonary edema. ep consulted and recommended atenolol 50 mg qd and observation. during obs, his hr increased to 150s with oxygen sats in 90-93% on room air. this rhythm was noted to be aflutter. he received propafenone 600 mg x1 and was cardioverted again (200j) to sinus rhythm. his cxr is suggestive of mild pulmonary edema and resting sats 92% on 8l nc. per ep, he will continue propafenone 150mg q8hours and possible ablation in am. . in ccu, he reports feeling slightly better. he is still c/o mild left chest pressure only when he leans on l side. +mild sob with talking. no sensation of palpitattions, lhd, dizzyness. he does feel very tired as he has not slept in 48 hrs. . on review of symptoms, he denies any prior history of stroke, tia, deep venous thrombosis, pulmonary embolism, bleeding at the time of surgery, myalgias, joint pains, cough, hemoptysis, black stools or red stools. he denies exertional buttock or calf pain. all of the other review of systems were negative. . cardiac review of systems is notable for absence of paroxysmal nocturnal dyspnea, orthopnea, ankle edema, palpitations, syncope or presyncope. past medical history: svt paf right inguinal hernia at age of 1 social history: patient is married and works as a sales engineer. social history is significant for the absence of current tobacco use. there is no history of alcohol abuse. there is no family history of premature coronary artery disease or sudden death. family history: father: hx premature atrial fibrillation mother: mvp physical exam: vs: t 99 , bp 131/95 , hr 104, rr 18, o2 92% on 5lnc gen: wdwn young male in mild resp distress. oriented x3. mood, affect appropriate. pleasant. heent: ncat. sclera anicteric. perrl, eomi. conjunctiva were pink, no pallor or cyanosis of the oral mucosa. mm dry. neck: supple with jvp at 10 cm (under jaw) cv: tachycardic, regular, normal s1, s2. no s4, no s3. no murmurs chest: no chest wall deformities, scoliosis or kyphosis. scarce crackles l>r 1/3 up bilaterally abd: soft, ntnd, no hsm or tenderness. no abdominial bruits. ext: no c/c/e. skin: no stasis dermatitis, ulcers, scars, or xanthomas. pulses: right: carotid 2+ without bruit; 2+ dp left: carotid 2+ without bruit; 2+ dp pertinent results: transesophageal echocardiogram: no spontaneous echo contrast or thrombus is seen in the body of the left atrium/left atrial appendage or the body of the right atrium/right atrial appendage. no atrial septal defect or patent foramen ovale is seen by 2d, color doppler or saline contrast with maneuvers. the ascending, transverse and descending thoracic aorta are normal in diameter and free of atherosclerotic plaque to 45 cm from the incisors. the aortic valve leaflets (3) appear structurally normal with good leaflet excursion. no masses or vegetations are seen on the aortic valve. no aortic regurgitation is seen. the mitral valve leaflets are structurally normal. no mass or vegetation is seen on the mitral valve. mild (1+) mitral regurgitation is seen. impression: no thrombus, masses, or vegetations identified. no pfo/asd. mild mitral regurgitation. . . transthoracic echocardiogram the left atrium is mildly dilated. the right atrium is moderately dilated. the estimated right atrial pressure is 0-10mmhg. left ventricular wall thicknesses are normal. the left ventricular cavity size is normal. overall left ventricular systolic function is normal (lvef>55%). right ventricular chamber size and free wall motion are normal. the aortic valve leaflets (3) are mildly thickened. no aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. mild (1+) mitral regurgitation is seen. the pulmonary artery systolic pressure could not be determined. there is no pericardial effusion. . . mri/mri head and neck 1. acute infarct of the medial right posterior temporal and occipital lobes. small acute infarct of the right thalamus. 2. acute thromboembolism of the p2 segment of the right posterior cerebral artery. 3. normal mra of the neck. . . ct angiogram of the chest 1. no evidence of pulmonary embolism or pulmonary edema. 2. bilateral pleural effusions with associated atelectasis. 3. patchy areas of airspace disease involving both upper lobes suspicious for pneumonia. 4. the tip of the endotracheal tube is seen at the superior edge of the clavicles. brief hospital course: the following issues were dealt with on this admission: . # rhythm: on the evening of the patient went into rapid atrial flutter with a rate in the 150's. he did not respond to a diltiazem drip, so he was started on procainamide following cardioversion, and then propafenone. he continued to have tachyarrhythmias on this regimen, and was started on amiodarone and esmolol drips on . he did quite well on this regimen, and converted to sinus rhythm, with intermittent bouts of atrial fibrillation that were not sustained. he was transitioned to a po regimen of amiodarone and metoprolol on , which he tolerated well. . # pump: patient presented with signs and symptoms of pulmonary edema, confirmed on cxr and ct, which was thought to be secondary to a tachycardia-induced cardiomyopathy in the setting of his arrhythmia. an echocardiogram was ordered, and was wnl, and this was followed up with a cardiac mr (read pending on discharge). the edema was severe enough to require a brief period of intubation electively on . the patient was extubated without any complications on the morning of . . # cva: patient was found to have an acute right posterior cerebral infarction on ct head. mri following showed an acute thromboembolism of the p2 segment of the right posterior cerebral artery, and acute infarction of the medial posterior temporal and occipital lobes, and a small acute infarct of the right thalamus. a tee was negative for thrombus or asd/pfo. neurology was consulted, and recommeded anticoagulation with warfarin, with a heparin bridge to an inr of 2.5, and lipitor. he was discharged with follow up in coumadin clinic. . # pna: the patient was found to have sputum cultures postive staph aureus, pan-sensitive, and resistant to penicillin. he was initially managed with vancomycin, and was transitioned to po dicloxacillin once his sensitivities confirmed the absence of mrsa. medications on admission: atenolol discharge medications: 1. warfarin 2.5 mg tablet sig: as directed by your coumadin clinic at dr. office tablet po once a day: until your follow up with dr. , continue to take 5mg each day, which is two 2.5mg tablets. disp:*60 tablet(s)* refills:*2* 2. amiodarone 200 mg tablet sig: two (2) tablet po tid (3 times a day). disp:*180 tablet(s)* refills:*2* 3. dicloxacillin 500 mg capsule sig: one (1) capsule po q6h (every 6 hours) for 3 days. disp:*12 capsule(s)* refills:*0* 4. metoprolol tartrate 50 mg tablet sig: one (1) tablet po every twelve (12) hours. disp:*60 tablet(s)* refills:*2* 5. atorvastatin 40 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* discharge disposition: home discharge diagnosis: atrial fibrillation stroke discharge condition: stable discharge instructions: you were admitted because you had an irregular heart rhythm. we controlled this with intravenous medications, and eventually transitioned you to oral medications called metoprolol and amiodarone. we will be discharging you with a monitor for your heart rhythm. this will be followed up by dr. . . you also suffered a stroke during this admission, which required us to thin your blood with an iv medication called heparin. we transitioned you to an oral blood thinner called coumadin. you are also on a cholesterol drug called lipitor for stroke prevention. . you will need to follow up with your primary care physician . to monitor your coumadin. when you are on coumadin, we closely monitor a level in your blood called your inr, which measures how thin your blood is. this will be more frequent initially. please see below for your follow up information. . you also need to follow up with dr. for management of your abnormal heart rhythm. please see below for follow up information. . . please take all of your medications as indicated below. . . if you experience any concerning symptoms, please return to the emergency department. followup instructions: 1. dr. office will be in touch regarding follow-up for your coumadin 2. dr. nurse practitioner will contact you regarding follow up for your rhythm. procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified diagnostic ultrasound of heart insertion of endotracheal tube other electric countershock of heart diagnoses: other iatrogenic hypotension congestive heart failure, unspecified atrial fibrillation personal history of tobacco use atrial flutter other and unspecified hyperlipidemia other specified cardiac dysrhythmias methicillin susceptible pneumonia due to staphylococcus aureus cerebral embolism with cerebral infarction cerebral thrombosis with cerebral infarction secondary cardiomyopathy, unspecified 

Answer: The patient is high likely exposed to