Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no drug allergy information on file attending: chief complaint: left sided weakness, slurred speech. major surgical or invasive procedure: intubation and mechanical ventilation. history of present illness: patient is a 75 year old right handed male with past medical history of coronary artery disease s/p nqwmi , s/p lad angioplasty and stent , dementia, hyperlipidemia, aortic stenosis, atrial fibrillation on coumadin who presented from his eye doctor's office wtih acute onset left sided weakness and dysarthria. patient was in his usual state of health until around 12:15 on day of admission. was at this doctor's office and complained of severe headache. thereafter, wife noted development of left sided weakness, facial droop, slurring of speech. sent to for evaluation emergently. ed course: arrived at at 12:30. finger stick blood glucose 120. rest of vitals bp 112/70, hr 60. blood pressure up to 230s systolic. seen emergently by stroke team. initial nih stroke scale of 33. head ct showed large right intracerebral hemorrhage. intubated for airway protection at 13:35 after fentanyl, etomidate, vecuronium and succhinylcholine given. started on labetalol and then nipride drip for bp control; discontinued as goal bp in 130-160s. received 10 mg sc vitamin k and 2 units ffp while in ed. pupils noted to be fixed and dilated at 14:10. loaded with dilantin and 50 grams mannitol. transferred to intensive care unit. past medical history: 1. atrial fibrillation, on coumadin 2. coronary artery disease status post nqwmi , s/p lad angioplasty and stent . known non-dominant 90% rca lesion not intervened upon. 3. dementia 4. mild-moderate aortic stenosis 5. hyperlipidemia 6. colon cancer 7. obstructive sleep apnea 8. anxiety 9. diabetes mellitus, on oral medications social history: married. lives with wife. one son, in area. social alcohol. no tobacco, drug use. family history: non-contributory. physical exam: physical exam (examined off of propofol): tc: 96.7 bp: 120/48 hr: 61 rr: 14 o2sat.: 100%, ventilated. gen: wd/wn male, comfortable appearing, intubated, not sedated. heent: nc/at. anicteric. mmm. +endotracheal tube. neck: supple. no masses or lad. no jvd. no thyromegaly. no carotid bruits. lungs: coarse breath sounds anterolaterally. cardiac: irregularly irregular. s1/s2. no m/r/g. abd: soft, nt, nd, +nabs. no rebound or guarding. no hsm. extrem: warm and well-perfused. no c/c/e. neuro: mental status: no response to voice, sternal rub. grimaces to nasal tickle. cranial nerves: pupils fixed at 7mm bilaterally. very weak flicker of inner canthus on left with corneal stimulation. no oculocephalic reflex. left facial droop. no gag provoked, but biting ett at times. tongue midline. motor: normal bulk. increased tone x4 with les>ues. no withdrawal to noxious stimuli. bilateral feet externally rotated. sensation: no withdrawal to noxious stimuli. reflexes: absent in lue, left ankle. striking left patellar tendon results in sustained clonus of left leg. toes upgoing bilaterally. rue, absent right ankle. coordination: unable to assess. gait: unable to assess. pertinent results: 12:39pm wbc-9.1 rbc-4.29* hgb-14.1 hct-38.7* mcv-90 mch-32.8* mchc-36.4* rdw-14.7 12:39pm plt count-147* 12:39pm pt-20.0* ptt-32.4 inr(pt)-2.5 12:39pm glucose-101 urea n-25* creat-1.0 sodium-142 potassium-4.2 chloride-106 total co2-27 anion gap-13 12:39pm ck(cpk)-200* 12:39pm ctropnt-<0.01 12:39pm ck-mb-7 09:45pm ck(cpk)-196* 09:45pm ck-mb-3 ctropnt-<0.01 03:30am blood ck(cpk)-312* 03:30am blood ck-mb-5 ctropnt-<0.01 01:55pm urine blood-sm nitrite-neg protein-100 glucose-neg ketone-neg bilirubin-neg urobilngn-neg ph-6.5 leuk-neg 11:16am blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 10:17am urine bnzodzp-neg barbitr-neg opiates-neg cocaine-neg amphetm-neg mthdone-neg ct head without contrast : findings: there is a large region of high attenuation within the right frontal lobe and the region of the right basal ganglia. this is consistent with intraparenchymal hemorrhage, more likely hypertensive in etiology. no significant subarachnoid or intraventricular hemorrhage is appreciated. there is mass effect present with shift of the normally midline structures towards the left. there is also left sided subfalcine herniation. there is no hydrocephalus. the visualized osseous structures and paranasal sinuses are unremarkable. impression: large intraparenchymal hemorrhage within the right frontal lobe. this most likely relates to hypertensive hemorrhage, as opposed to a ruptured right middle cerebral artery aneurysm. brief hospital course: patient was a 75 year old right handed male with past medical history of atrial fibrillation on coumadin, coronary artery disease, hyperlipidemia, dementia with sudden onset left sided weakness, dysarthria. head ct demonstrated a large right intracerebral hemorrhage, centered around basal ganglia and putamen. we felt this was likely secondary to hypertension. while still in the emergency department, he had rapid decompensation necessitating intubation for airway protection. examination post intubation but off intubations showed clinical evidence of elevated intracranial pressure and brainstem compression. the overall prognosis was guarded. he was admitted to the neurology/neurosurgery icu. he was loaded with mannitol and dilantin in the emergency room. he was evaluated by the neurosurgery service and they did not feel a surgical intervention or ventriculary drain was warranted. after arrival in the icu, patient was followed with neuro checks every 1 hour. sedation was held to allow for frequent exams. head of bed was kept at 30 degrees. labetalol continuous infusion was titrated for systolic blood pressure of 130-160. mannitol and dilantin were continued. after lengthy discussion, the patient's wife opted to make him dnr on afternoon of . overnight, fresh frozen plasma was given for goal inr of <1.4. on serial exams by neurology and icu attendings in the morning on , patient had no clinical signs of brainstem activity. namely, he was unresponsive. pupils were fixed and dilated. no corneal, gag, oculocephalic, and oculovestibular reflexes were elicited. he was not overbreathing his ventilator. at this point, patient was felt to have clinical signs consistent with brain death. plans were made to proceed with apnea testing. however, after discussion with patient's wife, this was postponed with test planned on . full medical care and managment continued overnight . at approximately 06:00 on , the patient became bradycardic. he lapsed first into a junctional rhythm and then had very infrequent ventricular escape beats on telemetry. finally, he went asystolic. he was not resuscitated as per previous conversations with his wife. was pronounced dead at 06:59 am. the patient's wife was notified. the attending was notified as well. medications on admission: 1. coumadin 2. lipitor 3. toprol xl 4. asa 5. zoloft 6. glucophage 7. aricept 8. namenda 9. artificial tears discharge medications: not applicable. discharge disposition: extended care facility: patient deceased. discharge diagnosis: not applicable; patient deceased. discharge condition: not applicable. discharge instructions: not applicable. followup instructions: not applicable. md, procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube arterial catheterization transfusion of other serum diagnoses: coronary atherosclerosis of native coronary artery unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled atrial fibrillation aortic valve disorders percutaneous transluminal coronary angioplasty status intracerebral hemorrhage other persistent mental disorders due to conditions classified elsewhere other and unspecified hyperlipidemia old myocardial infarction long-term (current) use of anticoagulants personal history of malignant neoplasm of large intestine 

Answer: The patient is high likely exposed to