Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins attending: chief complaint: seizure major surgical or invasive procedure: ivc filter placement central line placement/removal picc line placement history of present illness: mr. is a 22 yr old male who was transferred from on for further management of seizure, altered mental status, and fever. pt was in his usual state of health until 3 1/2 weeks prior to admission when he developed a viral syndrome including rhinorrhea, body aches, warmth, night sweats which lasted for 2 weeks. this happened approx a week after he returned from spring break in . he was fine for a week after the illness. on he developed olfactory hallunications and seized. was taken to . ct scan at the time was reportedly negative, and he was given an outpatient neuro appt. on he again had an episode of generalized seizure and was taken back to the lgh where a ct head was neg for any acute bleed. an lp was done that showed 679 wbc with lymph predominance, tp of 51 and glucose of 81. was given a dose of ceftriaxone/vanc/acyclovir and transferred to . on he had a tmax of 102 where he was started on ceftriaxone/vanc for possible bacterial meningitis, acyclovir for aseptic meningitis, and bactrim to cover for listeria. he was also started on methylprednisone as ct showed edema and was admitted to . past medical history: 1.)asthma 2.)h/o ebv infection social history: mr. lives with his girlfriend and her parents. he works at a desk job for an insurance company. occasional etoh. no tobacco. no drugs. family history: --father htn --mother arthritis --grandparents - mi, dm, chf, lung ca, prostate ca all died at age 70s --brother had generalized seizure in physical exam: t=103, bp=130s/80s, p=103, rr=28, o2sat=99% ra gen: lying in bed, nad heent: eomi, perrl, mmdry, no lymphadenopathy, abrasions on nose/lip cv: rrr, nl s1/s2, no m/r/g pulmo: ctab abd: soft, bs+, nt, nd, no masses ext: warm, 2+ dp/pt, no c/c/e skin: abrasions on knuckles, no rashes neuro: 5/5 strength upper/lower equal bilaterally; 2+ reflexes biceps/patellar equal bilaterally; sensation to pain and light touch intact throughout; cn 2-12 intact; toes downgoing mental status: aphasia (receptive and expressive), anomia, apraxia. for example, pt has halting speech without much intonation and has difficulty producing spontaneous speech, naming, and repeating. he also has episodes where he speaks in jibberish and has difficulty finding words. when asked to write "i love chocholate" he wrote "i tool like", and when asked to write "my name is " he wrote "my name closet" he was unable to subtract 7s from 100, first saying "7, 6, 5, . . ." then when asked again, saying "100, 99, 98, 97" when asked to read my name tag he would say some words that were on the tag then say words that were not on the tag like "international" along with gibberish. when attempting to ask him to remember 3 objects, he was unable to repeat the objects immediately back. he would say car, the first word, then go into a gibberish-laden story that did not make sense. pertinent results: cbc: 02:50am blood wbc-8.5 rbc-4.02* hgb-12.9* hct-35.7* mcv-89 mch-32.0 mchc-36.0* rdw-12.2 plt ct-237 06:30am blood wbc-5.7 rbc-3.55* hgb-11.3* hct-31.9* mcv-90 mch-31.9 mchc-35.6* rdw-13.2 plt ct-232 05:42am blood wbc-5.9 rbc-3.22* hgb-10.3* hct-29.1* mcv-90 mch-31.9 mchc-35.3* rdw-12.4 plt ct-273 chem panels: 02:50am blood glucose-123* urean-9 creat-0.8 na-131* k-3.8 cl-95* hco3-28 angap-12 05:42am blood glucose-96 urean-12 creat-0.8 na-135 k-4.2 cl-99 hco3-30* angap-10 anemia: 06:30am blood caltibc-195* vitb12-364 folate-9.7 ferritn-261 trf-150* csf: 08:50pm cerebrospinal fluid (csf) wbc-1500 rbc-225* polys-2 lymphs-92 monos-6 08:50pm cerebrospinal fluid (csf) totprot-258* glucose-54 micro: csf hsv pcr: positive for hsv cryptococcal antigen (final ): cryptococcal antigen not detected. rapid respiratory viral antigen test (final ): respiratory viral antigens not detected. culture confirmation pending. specimen screened for: adeno,parainfluenza 1,2,3 influenza a,b and rsv. imaging: mri: findings are consistent with encephalitis and meningitis. the pattern of temporal lobe involvement is characteristic for herpes encephalitis, with increased signal intensity along the cortical margin of the left and right insula, more on the left than the right, and also along the cortical margin of the anterior and medial left temporal lobes, likely reflecting an ischemic component of these areas, and not simply t2 shine-through. eeg: this is an abnormal portable eeg obtained in drowsiness due to the presence of disorganized severe focal and continuous delta frequency slowing in the left central temporal region. this finding suggests subcortical dysfunction in this region and is consistent with a mild and diffuse encephalopathy. no lateralization or epileptiform abnormalities were seen. ct head: hyperdense material consistent with acute intraparenchymal hemorrhage located in the left temporal and right frontal lobes with surrounding edema that on retrospective view of limited brain images from the ct scan of the sinuses dated also present. there is an additional stable low attenuation lesion in the region of the right sylvian fissure that appears to correspond to a region of increased t2 signal on the previous mri. there is slight left to right shift with mild compression of the left lateral ventricle. if clinically indicated, further evaluation with an mri/mra of the brain with gadolinium may be performed. cta: 1. small pulmonary embolus within a subsegmental branch of the right pulmonary artery supplying the right lower lobe. mild bilateral lung base air space consolidation, left greater than right. no air bronchograms are identified. small bilateral pleural effusions, right greater than left. 2. small amount of free fluid within the pelvis. brief hospital course: 22 y/o male with asthma presented with seizures following 3 weeks of uri symptoms. found to have hsv meningoencephalitis, whose course was complicated by intermittent fever, headaches, and a pulmonary embolism. 1.)meningoencephalitis: when mr. presented, his constellation of symptoms and findings lead everyone to believe this was hsv meningoencephalitis. he came from the outside hospital with lp results consistent with an aseptic meningitis (plus red cells, already making hsv more likely). here, at , he had a repeat lp, again with a lymphocytosis and red cells. multiple studies were sent off. an mri was performed showing findings consistent with this presumed diagnosis (see results section). the patient was empirically started on iv acyclovir and continued on the the ceftriaxone and vancomycin that were started at the outside hospital until their culture data came back negative, at which point he was transitioned to acyclovir only. in terms of his neurologic status, his main deficit was cognitive, most specifically word-finding, with a receptive and expressive aphasia. over the course of his hospitalization, this slowly improved, though the expressive deficit persisted, though to a lesser extent. the meningoencephalitis course was complicated by seizures at presentation and intermittent headaches, as described below. at the time of discharge, his adenovirus titer was negative, cryptococcal negative, rxv negative, adenovirus negative, vzv negative, west and ebv were pending. he will likely need outpatient speech therapy. he will be followed by neurology and id as outpatient. 2.)seizures: this was the presenting symptoms, preceded by olfactory and gustatory hallucinations. an abnormal portable eeg obtained in drowsiness due to the presence of disorganized severe focal and continuous delta frequency slowing in the left central temporal region. this finding suggests subcortical dysfunction in this region and is consistent with a mild and diffuse encephalopathy. no lateralization or epileptiform abnormalities were seen. he was initially started on phenytoin, that made him excessively somnolent, and this was weaned off while oxcarbezapine was started. the patient tolerated this medication well, and remained seizure free throughout the rest of the hospitalization. he occasionally experienced odd smells and tastes; this was discussed with neurology, who felt that as long as he remained alert and oriented, this did not represent a seizure, but more likely an effect from the temporal lobe irritation. 3.)headache and intracerebral/intraventricular bleed: during the hospital course, mr. began developing severe frontal headaches. a ct of the head was peformed, showing hyperdense material consistent with acute intraparenchymal hemorrhage located in the left temporal and right frontal lobes with surrounding edema. this was felt to be mostly unchanged when compared to a sinus ct from three days prior, yet these cuts were felt to be inadequate to fully comment. for the bleed, an mra was performed that was limited to the arteries just off the circle of , but did not show any abnormalities. a follow-up head ct was performed two days later with no significant change. in discussions with id and neurology, this was felt to be consistent with the disease process and the plan was to have this followed as an outpatient; no further imaging was felt necessary as his neurologic exam did not change. 4.)pulmonary embolism: mr. began complaining of right upper quadrant pain during the admission. lft's were normal, as was a ruq ultrasound. he initially improved with the relief of his constipation, but as the pain did not fully resolve a ct of the thorax with cta was performed, demonstrating a subsegmental pe. as this demonstrated that he was at increased risk of further clots, an ivc filter was placed by interventional radiology, as he was felt to be too high of a bleeding risk, given the aforementioned intracerebral bleed. at no point did he experience any related hemodynamic compromise. to note, his ivc filter needs to be removed in the future. this should be arranged by his pcp. 5.)blurred vision: although this was probably from dehydration and orthostatic changes, with his cns infection, there was concern for extension to optic nerve or retinal involvement, although this seemed less likely. however, given his risk, we asked ophthamology to evaluate the patient, who felt there was nothing on exam to suggest any abnormality, but that he should follow-up non-urgently in the clinic. the symptom resolved without recurrence. 6.)fevers: for approximately five days, mr. daily and, occasionally, twice daily fevers, up to 102, with drenching night sweats. he was frequently cultured, a ppd placed, had chest x-rays, leni's, a ruq ultrasound, and an abdominal ct, all without findings that could explain the fever. the two major theories left were drug fever (especially phenytoin) versus central irritation for his intracerebral bleed. the intracerebral bleed seems to be the most likely etiology, yet the fevers subsided without a clear cause and did not recur. 7.)anemia: there was no clear etiology, although the main theory was decreased production from inflammation and viral infection. it remained relatively stable throughout the course. his labs (fe studies, b12, and folate) were consistent with this theory; fe studies showed more of an anemia of inflammation picture. a cbc should be checked again as an outpatient when he is through the more acute phase of this process. medications on admission: fluticasone-salmeterol 100-50 1 puff albuterol prn discharge medications: 1. fluticasone-salmeterol 100-50 mcg/dose disk with device sig: one (1) puff inhalation (2 times a day). disp:*1 diskus* refills:*2* 2. oxcarbazepine 300 mg tablet sig: two (2) tablet po qpm (once a day (in the evening)). disp:*60 tablet(s)* refills:*2* 3. oxcarbazepine 300 mg tablet sig: one (1) tablet po qam (once a day (in the morning)). disp:*30 tablet(s)* refills:*2* 4. oxycodone hcl 5 mg tablet sig: 1-2 tablets po q3h as needed for pain. disp:*30 tablet(s)* refills:*0* 5. pantoprazole sodium 40 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po q24h (every 24 hours). 6. docusate sodium 100 mg capsule sig: one (1) capsule po bid (2 times a day). 7. magnesium hydroxide 400 mg/5 ml suspension sig: thirty (30) ml po q6h (every 6 hours) as needed. disp:*1 bottle* refills:*0* 8. senna 8.6 mg tablet sig: one (1) tablet po bid (2 times a day). disp:*30 tablet(s)* refills:*2* 9. bisacodyl 5 mg tablet, delayed release (e.c.) sig: two (2) tablet, delayed release (e.c.) po bid (2 times a day) as needed. disp:*30 tablet, delayed release (e.c.)(s)* refills:*0* 10. morphine sulfate 2 mg/ml syringe sig: two (2) mg injection q4h (every 4 hours) as needed for headache: if oxycodone fails. 11. iv flush heparin and saline flush per pharmacy protocol 12. outpatient speech/swallowing therapy outpatient speech therapy as directed 13. acyclovir sodium 500 mg recon soln sig: five hundred (500) mg intravenous three times a day for 4 days: last day is sunday . discharge disposition: extended care facility: tcu - discharge diagnosis: herpes simplex virus meningoencephalitis seizures intracerebral hemmorrhage pulmonary embolism secondary: asthma discharge condition: good, with improving sx, afebrile discharge instructions: please call your pcp or return to the ed for seizures, worsening confusion, if the patient becomes unarousable, severe headache that does not respond to pain medication or is associated with confusion, fevers/chills, or other concerning symptoms. follow-up as below. take medications as prescribed. followup instructions: clinic at 10 am with dr. medical building - basement. , provider: , md where: lm disease phone: date/time: 10:00 neurology provider: , md where: ks building (/ complex) date/time: 10:30 allergy - dr. you have an appointment on , 1pm with dr. but you need to call your primary care doctor (dr.) for a referral ( ) pls call to re-schedule this appt at ( - procedure: venous catheterization, not elsewhere classified spinal tap incision of lung interruption of the vena cava diagnoses: anemia, unspecified asthma, unspecified type, unspecified other convulsions intracerebral hemorrhage other specified visual disturbances aphasia other pulmonary embolism and infarction orthostatic hypotension herpetic meningoencephalitis 

Answer: The patient is high likely exposed to