Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: the patient is a 44-year-old male with a history a seizure disorder who presented to northeast hospital on , with several days of vomiting and vague abdominal pain. the patient was found to have an increase in lfts with an ast of 11,700; an alt of 11,800. coagulopathic inr of 14.8. the patient was lethargic. alert and oriented x 3 without encephalopathy. positive asterixis. the patient also complained of right upper quadrant pain. denies a history of excessive etoh. denies a large amount of tylenol ingestion. denies iv drug abuse, or blood transfusions, or recent travel. the patient's dilantin, tegretol, and valproic acid were all in a therapeutic range. the patient was transfused 4 units of ffp and vitamin k for an increased inr. the patient was also noted to have hematemesis. serial hematocrits were obtained and monitored. the patient was intubated for increased lethargy and agitation and transferred to via medical flight. he had positive epistaxis per report, and there was an atraumatic intubation. past medical history: includes seizure disorder, gerd, hypertension, and self gunshot wound to groin as a suicide attempt in . past surgical history: none. medications at home: depakote 500 t.i.d., tegretol 300 b.i.d., dilantin 300 b.i.d., nexium 40 daily, lisinopril 10 daily. allergies: vioxx, rash. social history: no tobacco. no ethanol. no iv drug abuse. on dilantin due to a seizure disorder. physical examination on admission: vital's on admission were 95.9, 130/86, 82, 14, 100%. he was intubated on assist control of 60%, respiratory rate 14 with a peep of 5. the patient was intubated, sedated, and paralyzed. his heart was regular in rate and rhythm without murmurs, rubs, or gallops. the lungs were clear to auscultation bilaterally. the abdomen was soft, and no palpable liver edge. positive edema. mild distention on abdominal exam. imaging: imaging at the outside hospital showed an abdominal ultrasound with gallbladder small. no gallstones. chest x-ray was unremarkable. kub was unremarkable. laboratory data: the patient had an ammonia of 202. admission labs with a white count of 8.9, hematocrit of 35, platelets pending on admission. coagulation studies of 23.5, 34.7, 3.5. fibrinogen of 109. ast of 280, alt 669, amylase 55, lipase of 105, ldh of 923, alkaline phosphatase of 127, total bilirubin of 9.2, albumin of 3.3. phenytoin was 7.6, valproic acid of 4.5, carbamazepine of 7.6, and acetaminophen was 5.8. calcium of 7.5, magnesium of 2.0. sodium of 150, potassium of 4.1, chloride of 116, bicarbonate of 24, bun of 49, creatinine of 3.5, with a glucose of 115. the patient had a blood gas of 7.40, 38, 259, 25 and 0. hospital course: on hospital day 1 neurology was consulted. on hospital days 0 and 1 neurology was consulted and suggested checking levels of antiepileptic medications. suggested an eeg. neurology also suggested on hospital day 2 start versed for seizure control and overnight the patient had 3 seizures requiring large doses of ativan. a head ct showed no evidence of acute intracranial pathology with sinus opacification. abdominal ct the same day showed ascites with no focal collection, edematous appearing kidneys with no evidence of hydronephrosis or hydroureter. the distal ureters were not imaged. somewhat large edematous appearing liver with no focal lesion, parenchyma suggestive fatty replacement. gallbladder containing dense material consistent with sludge may represent biliary excretion, contrast from previous ct scan. bilateral pleural effusion, bilateral atelectasis. a liver ultrasound on hospital day 3 showed patent hepatic artery, veins small, small amount of ascites, with gallbladder sludge. the patient continued to receive large amounts of transfusions of blood products throughout hospital course, and by hospital day 3 had alt of 3802 and ast of 1300 with an inr of 2.75. because the patient was in status epilepticus, he currently was not transplantable and was clinically comatose by . progressively deteriorated by . over the course of the evening and early morning and became progressively acidotic, worsening lactate, progressive coagulopathy; unresponsive to sodium bicarbonate infusion, iv fluids resuscitation, and blood product infusion. on , was on levophed 0.5 mcg per kg per minute and neo-synephrine at 7.0 mcg per kg per minute with the most recent abg of 7.09/27/127/9/and - 20. he was on full life support measures at that time but was appearing to be futile. the patient was made cmo at the request of his wife. the patient died at 4:15 a.m. on , was asystolic on telemetry. organ bank was notified, and autopsy report showed submassive hepatic necrosis with bowel stasis most concentrated around zones 2 and 3 of the liver, soft density mild vascular congestion, mild interval thickening of the right coronary artery. , md procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours arterial catheterization transfusion of packed cells diagnoses: esophageal reflux unspecified essential hypertension acute and subacute necrosis of liver acute kidney failure, unspecified other convulsions other and unspecified coagulation defects coma 

Answer: The patient is high likely exposed to