Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: severe, acute onset ha starting at 0800 major surgical or invasive procedure: none history of present illness: patient is 77 y.o. male with h/o myeloproliferative d/o (possibly cml) who had sudden onset "worst headache of my life." reports that he was driving to a scheduled fluoro guided lp at the va, when he had acute onset sensation of pressure and pain, as if being stabbed in the back of the neck. pain radiated up occiput and to temples bilaterally. reports no head trauma (not even slight bump while getting into car), and denies any recent trauma or falls. was in marked distress and sent to er where ct was reportedly negative. was medicatd and sent for fluor guided lp which was markedly bloody and did not clear. had 192,000 rbcs in tube #4. was evaluated by neurology/neurosurg at the va and no neurological deficits. was recommended for dilantin, nimodipine, strict bp control, cta and mri of spine. he was then transferred to for further management. patient was also febrile to 101 at the va. after transfer to he was started on vanc, ceftriazone, acyclovir for a possible meningitis. ct head showed findings c/w sah, and cta showed possibility of anterior comm artery aneurysm. the patient was admitted to the neurosurgery service in the sicu for further monitoring. scheduled for angiography this am. heme/onc consult requested to answer question of whether cns leukemia could be cause of sah. ros: ha as above. no visual changes. no weakness, numbness or tingling. no vomitting but nausea present. no seizures. past medical history: myeloproliferative disorder (possibly cml) htn cad hyperlipidemia afib s/o cardioversion not on anticoagulation gerd anxiety bph s/p cholecystectomy cataract surgery r knee surgery h/o mrsa () h/o c. diff social history: no tob, occ etoh, lives alone in . family history: no h/o stroke or hemorrhage. no h/o renal disease. mother had breast ca. physical exam: t-97.4 bp- 111-124/38-43 hr-64-71 rr-13-42 o2sat 96-100% gen: lying in bed, nad heent: nc/at, moist oral mucosa neck: no tenderness to palpation, normal rom, supple, no carotid or vertebral bruit back: no point tenderness or erythema cv: rrr, nl s1 and s2, no murmurs/gallops/rubs lung: clear to auscultation bilaterally abd: +bs soft, nontender ext: no edema neurologic examination: mental status: awake and alert, cooperative with exam, normal affect. oriented to person, place, and date. attentive, says backwards. speech is fluent with normal comprehension and repetition; naming intact. no dysarthria. and writing intact. registers , recalls in 5 minutes. no right left confusion. no evidence of apraxia or neglect. cranial nerves: pupils equally round and reactive to light, 4 to 2 mm bilaterally. visual fields are full to confrontation. extraocular movements intact bilaterally, no nystagmus. sensation intact v1-v3. facial movement symmetric. hearing intact to finger rub bilaterally. palate elevation symmetrical. sternocleidomastoid and trapezius normal bilaterally. tongue midline, movements intact. motor: normal bulk bilaterally. tone normal. no observed myoclonus or tremor. no pronator drift. tri wf we fe ff ip h q df pf te tf r 5 5 5 5 5 5 5 5 5 5 5 5 5 5 l 5 5 5 5 5 5 5 5 5 5 5 5 5 5 sensation: intact to light touch, pinprick, vibration and proprioception throughout. no extinction to dss reflexes: +2 and symmetric throughout. toes downgoing bilaterally. coordination: finger-nose-finger normal, heel to shin normal, rams normal. gait: unable to assess secondary to s/p angio romberg: not assessed pertinent results: 134 99 20 120 agap=14 ------------< 5.0 26 1.2 ca: 8.4 mg: 2.3 p: 5.2 phenytoin: 25.2 11.0 48.5 >< 410 n:83 band:3 l:2 m:3 e:1 bas:0 metas:3 32.2 pt: 13.7 ptt: 25.5 inr: 1.2 ua negative. lp at osh: 192,000 rbcs in tube 4. 127 nucleated cells with diff of 56 segs, 18 lymph, 1 mono, 12 bands. glucose 51, protein 361. serum gluc 108. imaging: cta head: probable minute anterior communicating artery aneurysm. however, such an abnormality does not likely account for the multiple areas of hemorrhage, with the largest collection seen in relation to the left side of the cervicomedullary junction of the neuraxis. while no definite enhancement is seen to suggest an alternative diagnosis for the foramen magnum lesion, such as a meningioma or leukemic deposit, after discussing the case with the attending neurosurgeon, dr. , consideration for a followup mr study using gadolinium enhancement was suggested, as well as consideration for standard catheter cerebral angiography. addendum: there are minor atherosclerotic calcifications involving the distal left internal carotid artery, immediately proximal to the carotid foramen. mri/mra head and neck: no official read available. no dwi abnormalities. angio: tiny several mm acom aneurysm . no abnormailty. all 4 vessels patent. brief hospital course: 77 y.o. male with h/o myeloproliferative d/o, htn, acute onset severe ha and subarachnoid hemorrhage in mulitple areas with no associated trauma. angio showed tiny 4mm acommunicating artery aneurysm. exam is currently negative. neuro: osh head ct negative for bleed however, head ct in ed showed regions of subarachnoid hemorrhage at the left cervicomedullary junction (level of foramen magnum) and bilateral posterior temporal lobes. additionally, there was a intra-ventricular hemorrhage within the right occipital . cta head showed a 1mm sessile aneurysm from the anterior communicating artery and minor atherosclerotic calcifications involving the distal left internal carotid artery, immediately proximal to the carotid foramen. differential diagnosis included meningioma or leukemic deposit, after discussing the case with the attending neurosurgeon, dr. , consideration for a followup mr study using gadolinium enhancement was suggested, as well as standard catheter cerebral angiography. dilantin was held. patient was treated with triple h therapy including elevation of the blood pressure (induced hypertension), hemodilution to improve cerebral blood flow, and maintenance of high normal circulating blood volume (hypervolemia). patient was maitained on calcium channel blocker nimodipine is given at a dose of 60 mg by mouth every 6 hours. dilantin was held. antibiotics were discontinued given negative gram stain on csf and low suspicion for infection. cvs: patient was kept on telemetry without events. on nimodipine as above. resp: no acute issues renal: increased intravasculature volume with ivf ns 100cc/hr. while on acyclovir also received fluid boluses prior to administration. endo: regular insulin sliding scale. id: iv acyclovir continued due to fever and intracranial hemorrhage at presentation and discontinued on given low clinical suspicion. prohpylaxis: heparin sc, zantac, riss medications on admission: tramadol 50mg po q6h aspirin 325mg po qd lisinopril 20mg po qd terazosin 2mg po qhs ranitidine 150mg po bid amiodarone 200mg po qd simvastatin 20mg qhs diazepam 5mg po qhs discharge disposition: home with service discharge diagnosis: primary diagnosis: subarachnoid hemorrhage hemorrhage at cervicomedullary junction on the left secondary diagnosis: myeloproliferative disease hypertension hyperlipidemia atrial fibrillation status post cardioversion gastroesophageal reflux disease anxiety history of mrsa in discharge condition: patient left again medical advice. his condition and work-up results were explained to him to date. furthermore, he was told that his work-up was not completed and that he still needed an mri of the brain with and without contrast and that neuro-oncology was asked to evaluate him. it was explained that it was unsafe for him to leave. the team spoke with his daughter in an attempt to convince the patient to stay. however, despite our efforts, patient left ama. he has a follow-up appointment scheduled with dr. in clinic in . that patient understand what was explained to him and was competent to maek this informed decision as determined by the stroke team. patient was neurologically stable at the time he left ama. discharge instructions: please take medications as prescribed. please keep your follow-up appointments. if you have any worsening headaches, weakness, falls, change in mental status or any other worrying symptoms, please call your primary care physician or return to the emergency room. followup instructions: provider: , md phone: date/time: 9:00am provider: , md (neuro-oncology) phone: please call and make an appointment within 2 weeks of discharge. patient will need repeat conventional cerebral angiogram in 3 weeks to re-evaluate for etiology of subarachnoid bleed. mri with gadolinium date/time: md, procedure: arteriography of cerebral arteries magnetic resonance imaging of brain and brain stem computerized axial tomography of head magnetic resonance imaging of other and unspecified sites diagnoses: esophageal reflux unspecified essential hypertension atrial fibrillation subarachnoid hemorrhage other and unspecified hyperlipidemia anxiety state, unspecified other lymphatic and hematopoietic tissues 

Answer: The patient is high likely exposed to