Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: mr. is a 48 year old male who was transferred from hospital with the diagnosis of pulmonary embolism. he presented to with a two day complaint of severe right sided sharp 24 hour period. ct angiogram had been done to evaluate his symptoms revealing a right pulmonary artery embolus. he was started on a heparin drip and transferred to for further evaluation and care. of note, the patient had reported that he has experienced exactly the same symptoms approximately two weeks prior to and received a ten day course of antibiotics and no other further work-up or evaluation, however, his symptoms did spontaneously resolve. his pulmonary embolus risk factors include long sitting as a truck driver; no family history of deep venous thrombosis or pulmonary embolism. no hypercoagulable syndrome. no prior history of clot. he is not a tobacco user and he has no history of malignancy. past medical history: 1. hypertension. allergies: penicillin. medications on transfer: 1. atenolol. 2. heparin drip. social history: he is married with children. he has no significant alcohol or cigarette history. he is a truck driver. family history: noncontributory except for there is no history of clotting disorder. physical examination: on admission is notable for vital signs of temperature 102.0 f.; pulse rate of 126; blood pressure 134/85; respiratory rate was 30 with a saturation of 96% on six liter face mask. in general, he is alert and oriented times three. he was in a significant amount of pain. heent was notable for pupils being equal, reactive and round to light and accommodation. extraocular movements are intact. oropharynx was clear. mucous membranes were moist. he was having some pursed lip breathing. lungs: he had left base crackles, otherwise right was markedly decreased. he was clear at the apices. on heart examination, tachycardia, regular, no murmurs, rubs or gallops. he did have a prominent s1. abdomen is soft, nontender, nondistended. no hepatosplenomegaly. lower extremities had two plus bilateral dorsalis pedis and posterior tibial pulses. he had no cyanosis, clubbing or edema. there was no calf tenderness. sign was negative. laboratory: on admission, white blood cell count 12.1, hematocrit 42.4, platelet count 149, bun and creatinine were 12 and 1.0. his ptt on transfer was 48.9, ck of 121. his calcium, magnesium, phosphorus and remainder of his electrolyte profile were within normal limits. outside hospital just showed chest x-ray significant for a right pleural effusion. ct angiogram was consistent with a right pulmonary embolus. hospital course: hemodynamically he was stable. there was no indication for clot lysis on transfer. he was on heparin and he was started on coumadin 5 mg q. day. he was started on a hypercoagulable work-up at the outside hospital and this was continued. over the next 24 hours, the patient was on telemetry and being watched appropriately. he had a cardiogram on admission which just showed sinus tachycardia at 107 beats per minute. his pr interval was approximately 0.173 milliseconds with a qrs of 89 milliseconds. his qt interval was 283 milliseconds. a qtc value of 377 milliseconds was additionally noted. he had a slight right shifted axis at minus 23. normal p waves. he had a delayed r wave progression in v4, v5. he had no pathologic q's. he had a narrow complex qrs otherwise. no evidence of left ventricular hypertrophy; there was no evidence of heart strain. he had normal st-t segments and he did have t wave flattening in iii and avf. this was repeated, however, at the time of admission at the . at that time, he had an abnormal r wave progression upright in v3 transitioning at v5. there were no q waves. his axis had shifted to plus 60 degrees. he still had a narrow complex qrs and was in sinus tachycardia at 144. there was evidence of right heart strain with an s in i, a q in iii, and a t wave inversion in iii as well. of note on admission, the patient also was describing hemoptysis and pleuritic chest pain, tachycardia and respiratory distress and with a right strain pattern on ekg, all suggested a pulmonary embolism and consistent with the ct angiogram findings after being admitted to the medical intensive care unit. the patient remained febrile at 101.6 f., to 102.0 f. range. he was still tachypneic in the 30s, saturating approximately 94% on 10 liter face mask. he required extensive dosing of morphine to control his pain. he continued on a heparin infusion which was therapeutic with ptt values in the 55 to 60 range. a central access was required for invasive hemodynamic monitoring. on hospital day number two, the cvp was measured to be 13 with right ventricular pressures 36/5, pa pressure of 36/20, a wedge of 16. serially, the patient was supported. he underwent thoracentesis to investigate his effusion. this was completed on . an opening pressure of 7 cm of water was noted. the patient became pale and hypotensive at the end of the procedure at which time the thoracentesis was aborted. the patient was placed in trendelenburg position and resuscitated with fluids. approximately two liters of gross blood was removed at the time of the right sided thoracentesis. thoracic surgery was consulted and a right chest tube was placed without any significant drainage. despite agressive resuscitation, he continued with hemodynamic instability and was taken to the or for urgent exploration to manage the hemorrhage and drain a potential empyema. during positioning he suffered an asystolic arrest and cpr immediately initiated. silmultaneously, he underwent a emergent left resuscitative thoracotomy and right thoracotomy (clam-shell incision) and an abdominal exploration. after internal cardiac message, futher resuscitation and epinephrine/atropine he recovered his own rhythym. operative findings included a right empyema and fresh blood arround the liver, but no ongoing bleeding. his right lung was decorticated and chest and abdomen closed with a silo because of edema. the next day he was taken back to the or and his chest and abdomen closed. he slowly came off pressures and the vent was weened down. however, he was very slow to wakeup. because of concern over possible hypoxic brain injury during his code neurology was consulted. it was felt that he likely suffered some degree of hypoxic brain injury and that improve. however, a brain mri did not show this. due to his persistent ventilatory needs, he underwent a tracheostomy and peg placement on . he subsequently developed a line sepsis. he then deveoped a severe allergic reaction to a ceflosporin used to treat this, including an allergic nephritis with renal insufficiency. after recovering from this he weaned off the ventilator and was discharged to rehab. he was interactive with family, but not back to baseline. at the time of discharge he was postoperative day number 22, 20 and 7, from the three previously mentioned procedures. he was off of fluconazole, still on vancomycin, still taking ciprofloxacin. his temperature maximum was 100.2 f.; his blood pressure was ranging 100 to 130 systolic with a diastolic pressure of 60, heart rate of 110, respiratory rate of 15. he was saturating at 95% on tracheostomy mask 50%. his gas on that was significant for a ph of 7.45, paco2 of 36, pao2 of 81, base excess of minus two, saturating at 96%, otherwise looking comfortable. his hematocrit at this time was 37.2; he had a bun and creatinine of 20 and 0.8. he still had a left picc which is day number three, a left radial arterial line which was day seven, a peg, a tracheostomy and continued to make some improvements neurologically. he was just being supported to start with physical therapy and occupational therapy. he was placed back on his atenolol 100 mg q. day to combat blood pressure and cardiac issues. he was on ciprofloxacin, day 9 of 14, continued on subcutaneous heparin and protonix for prophylaxis. given this disposition, on overall plan, the patient looked stable and appropriate for discharge. he was on the following. discharge medications: 1. impact at 90 cc per hour. 2. atenolol 100 mg p.o. q. day. 3. sliding scale insulin as needed. 4. subcutaneous heparin 5000 units three times a day. 5. reglan 10 mg intravenously q. six. 6. colace 100 mg p.o. twice a day. 7. vancomycin, day 21 of 21, completed on . 8. ciprofloxacin, day number 9 of 14, to be continued until . 9. albuterol and atrovent inhalers as needed. 10. actigall 300 mg p.o. twice a day for some biliary stasis issues. 11. being offered p.r.n. tylenol. at this time, he was deemed appropriate and stable for discharge to a vented rehabilitation. he will be on his tracheostomy collar and continue to get aggressive pulmonary toilet. he will continue ciprofloxacin, as stated, for the pseudomonal organism that had grown out from either tracheobronchitis or pneumonia. he has already completed his vancomycin therapy for the strep milleri as well as for his methicillin resistant staphylococcus aureus in his sputum; note that the strep milleri was from his thoracic wound. discharge instructions: 1. wound care for this patient can be just a dry dressing applied to the wound twice a day. 2. he will continue on aggressive physical therapy, out of bed, ambulating. 3. pulmonary toilet including coughing and deep breathing, incentive spirometry, tracheostomy collar and suctioning as indicated. if he were to become tachypneic or to tire, he should go back on a ventilator and be supported accordingly with the rehabilitation physicians titrating the settings for appropriate arterial blood gases. 4. the patient should follow-up with dr. in one to two weeks from time of discharge. , m.d. dictated by: medquist36 d: 13:57 t: 14:21 job#: \ procedure: venous catheterization, not elsewhere classified diagnostic ultrasound of heart fiber-optic bronchoscopy enteral infusion of concentrated nutritional substances thoracentesis exploratory laparotomy percutaneous [endoscopic] gastrostomy [peg] decortication of lung temporary tracheostomy excisional debridement of wound, infection, or burn other incision of pleura trocar cholecystostomy open chest cardiac massage excision or destruction of lesion or tissue of mediastinum diagnoses: other postoperative infection unspecified pleural effusion acute kidney failure, unspecified accidental puncture or laceration during a procedure, not elsewhere classified hemorrhage complicating a procedure cardiac arrest other shock without mention of trauma other pulmonary embolism and infarction empyema without mention of fistula 

Answer: The patient is high likely exposed to