Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: latex attending: chief complaint: s/p fall major surgical or invasive procedure: none history of present illness: 71 yo m with htn, chf, niddm, afib on coumadin, who was leaning over to pick up something, fell to the ground, hit his head on a shelf, no loc. no prodromal symptoms. brought to where a nchct showed a large r basal ganglia iph, max bp documented 151/89. tx vit k 10 mg at 11h00 and 5 u ffp. awaiting 2 vials profiline. in c-spine collar. ct c-spine at osh inconclusive so will repeat here as per radiology. labs remarkable for trop i of 0.03. now feels fine, only complaint being his significant dysarthria, no headache, and completely neglectful of his dense l hemiparesis and l sensory loss. ros: denies any fever, chills, weight loss, visual changes, hearing changes, neckpain, nausea, vomiting, dysphagia, tingling, bowel-bladder dysfunction, chest pain, shortness of breath, abdominal pain, dysuria, hematuria, or bright red blood per rectum, rash, muscle aches, joint pains. past medical history: -htn -afib -chf -niddm social history: no tobacco or etoh physical exam: vitals: t 98.1 po hr 76 bp 134/76 rr 16 so2 96%ra gluc 213 gen: lying in stretcher with c-spine collar heent: mmm neck: no lad; no carotid bruits lungs: clear to auscultation bilaterally heart: regular rate and rhythm, normal s1 and s2, no murmurs, gallops and rubs. abdomen: normal bowel sounds, soft, nontender, nondistended extremities: no clubbing, cyanosis, ecchymosis, or edema mental status: drowsy but awakens with repeated prompting, cooperative with exam, unaware of his neuro deficits, inattentive, oriented, language fluent, naming and repetition intact, comprehension intact and follows simple commands, significant dysarthria, no paraphasic errors, nml prosody, neglects his l-hemibody and l-visual field, does not recognize his l thumb as his own. cranial nerves: ii: dense l visual field cut versus neglect, pupils equally round and reactive to light both directly and consensually, 3-->2 mm bilaterally. iii, iv, vi: r gaze preference but will look to the l on command. extraocular movements intact without nystagmus. r ptosis. v: facial sensation intact to light touch. vii: l facial droop in a lmn pattern viii: hearing intact to finger rub bilaterally. ix: palate elevates in midline. xii: tongue protrudes in midline, no fasciculations. : sternocleidomastoid and trapezius normal bilaterally. motor system: normal bulk and tone bilaterally. no adventitious movements, no tremor, no asterixis. strengthb intact rue and rle. dense lue plegia, withdraws lle minimally against gravity to noxious stim. no pronator drift on rue. no rebound. reflexes: dtrs 2+ and symmetric, r plantar response extensor. sensory system: dense l hemisensory loss to light touch on lue and lle. coordination: normal fnf with rue. gait: not evaluated pertinent results: labs: 12:40pm blood wbc-8.1 rbc-4.34* hgb-13.4* hct-37.2* mcv-86 mch-30.8 mchc-36.0* rdw-12.7 plt ct-197 07:00am blood wbc-6.9 rbc-4.69 hgb-14.2 hct-40.8 mcv-87 mch-30.2 mchc-34.8 rdw-12.6 plt ct-241 12:40pm blood neuts-75.2* lymphs-19.6 monos-3.6 eos-1.1 baso-0.5 12:40pm blood pt-20.1* ptt-30.3 inr(pt)-1.9* 06:15am blood pt-13.1 ptt-25.0 inr(pt)-1.1 12:40pm blood glucose-201* urean-15 creat-0.7 na-136 k-4.2 cl-102 hco3-26 angap-12 07:00am blood glucose-218* urean-19 creat-0.7 na-138 k-4.0 cl-100 hco3-28 angap-14 12:40pm blood ck(cpk)-77 07:40pm blood ck(cpk)-83 04:16am blood alt-15 ast-17 ck(cpk)-82 12:40pm blood ck-mb-4 ctropnt-<0.01 04:16am blood ck-mb-notdone ctropnt-<0.01 04:16am blood calcium-8.8 phos-2.7 mg-2.0 cholest-146 04:16am blood triglyc-83 hdl-45 chol/hd-3.2 ldlcalc-84 04:16am blood %hba1c-8.6* 04:16am blood tsh-0.75 04:16am blood crp-2.6 09:57am urine color-yellow appear-hazy sp -1.026 09:57am urine blood-lg nitrite-neg protein-neg glucose-250 ketone-150 bilirub-neg urobiln-neg ph-5.0 leuks-neg 09:57am urine rbc-* wbc-0-2 bacteri-few yeast-none epi-<1 micro: urine cx (): no growth imaging: ecg (): sinus bradycardia at a rate of 55 with a premature beat. conducted complexes have p-r interval prolongation, left atrial abnormality and right bundle-branch block morphology. the premature beat may be an atrial premature beat. ct head (): impression: acute hemorrhage in the right basal ganglia with associated mass effect on the right lateral ventricle. no shift in midline structures or evidence of brain herniation. ct c-spine (): conclusion: 1. no acute fracture. 3-mm retrolisthesis of c4 on c5 and associated degenerative changes at c3-c4 and c4-c5 levels. 2. scarring at the lung apices with 11 x 10 mm opacity in the right apex, these lung changes can be assessed with a non-emergent ct chest. tte (): the left atrium is mildly dilated. the right atrium is moderately dilated. left ventricular wall thickness, cavity size and regional/global systolic function are normal (lvef >55%). right ventricular chamber size and free wall motion are normal. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. trace aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. trivial mitral regurgitation is seen. the pulmonary artery systolic pressure could not be determined. there is no pericardial effusion. impression: normal global and regional biventricular systolic function. ct abdomen/pelvis (): conclusion: 1. 45 x 24 mm pleural-based oval opacity at the right lung base may represent round atelectasis, however, other etiologies are not excluded and comparison with any prior imaging would be invaluable. otherwise, a followup chest ct in one month is recommended to ensure stability/resolution. 2. right tenth rib irregularity may represent prior partial resection. 3. bilateral renal hypodensities, some of which are clearly cysts and others are too small to characterize. cxr (): impression: no acute abnormality on limited portable exam. ecg (): sinus rhythm at a rate of 70. right bundle-branch block. cxr (): since yesterday, right upper lobe thickening is unchanged, could be related to old tuberculosis exposure and should be evaluated by non-emergent chest ct. comparison with old chest x-ray would also be helpful. lungs are otherwise clear. clips are projecting over the right hilum and the left upper abdominal quadrant. the cardiomediastinal silhouette and hilar contours are normal. there is no pleural effusion. mr (): impression: large parenchymal hemorrhage with surrounding vasogenic edema, centered in the right basal ganglia, but extending into the medial temporal lobe, not much changed in size, configuration or degree of mass effect, with effacement of the ipsilateral lateral ventricle, since the ct of some 17.5 hours earlier. of note, there is no evidence of "microhemorrhage" elsewhere to specifically suggest underlying amyloid or hypertensive angiopathy, and no pathologic enhancement in or around the lesion, to suggest an underlying mass or other structural abnormality; however, the flair images do demonstrate bihemispheric subcortical and periventricular and central pontine white matter abnormalities, seen in chronic microvascular disease from hypertension, which may be the underlying etiology of the bleed. ecg (): irregular tachy-arrhythmia at a rate of 143 of uncertain mechanism but may be atrial fibrillation with rapid ventricular response. right bundle-branch block. st-t wave changes are probably primary and are not specific. ecg (): atrial fibrillation with rapid ventricular response at a rate of 101. right bundle-branch block. st-t wave changes are probably primary and are non-specific. ecg (): atrial fibrillation with ventricular premature beat at a rate of 94. right bundle-branch block. st-t wave abnormalities are primary and are non-specific. brief hospital course: the patient is a 71 year old man with a history of atrial fibrillation on coumadin, hypertension, and niddm, who presented s/p a fall, and was transferred from an osh with a large right basal ganglia iph. he received vitamin k 10 sq and 5 u ffp at osh (unsure what inr was on admission to osh), his inr was 1.9 at so was given prophylnine 2 vials and 1 u ffp. his coumadin was held. neurosurgery was consulted on admission and suggested reversing inr. he was initially in the neuroicu, but was then transferred to the neurology floor. ct head showed acute hemorrhage (4.7 cm x 2.7 cm) in the right basal ganglia with associated mass effect on the right lateral ventricle, no shift in midline structures or evidence of brain herniation. mri head showed large parenchymal hemorrhage with surrounding vasogenic edema, centered in the right basal ganglia, but extending into the medial temporal lobe, not much changed in size, configuration or degree of mass effect, with effacement of the ipsilateral lateral ventricle, since the ct of some 17.5 hours earlier. there was no evidence of "microhemorrhage" elsewhere to specifically suggest underlying amyloid or hypertensive angiopathy, and no pathologic enhancement in or around the lesion, to suggest an underlying mass or other structural abnormality; however, the flair images do demonstrate bihemispheric subcortical and periventricular and central pontine white matter abnormalities, seen in chronic microvascular disease from hypertension, which may be the underlying etiology of the bleed. tte showed normal global and regional biventricular systolic function, lvef >55%. ces: ck 77-83-82, tropt <0.01 x2; hga1c 8.6%; flp chol 146, tg 83, hdl 45, ldl 84; tsh 0.75; crp 2.6. he should hold his coumadin for 4 weeks, and at that time it can be restarted. he has a follow up appointment with dr. in neurology as an outpatient. on , he developed atrial fibrillation with rvr to 140-150. he required metoprolol iv and diltiazem iv, and he converted to sinus rhythm. he was started on diltiazem 60 mg tid. he has a history of niddm, and received an iss during the hospitalization. his metformin was added back at half his home dose at the time of discharge, and can be increased to his home dose of 1000 as tolerated. he has been scheduled for an outpatient ct chest in 1 month for scarring at the lung apices with 11 x 10 mm opacity in the right apex found on ct c-spine, and 45 x 24 mm pleural-based oval opacity at the right lung base may represent round atelectasis, however, other etiologies are not excluded on ct abdomen/pelvis. medications on admission: coumadin 5 mg po qhs metformin 1000 mg po bid (he mentions taking a 3rd medicine, but it is unintelligible) discharge medications: 1. diltiazem hcl 60 mg tablet sig: one (1) tablet po tid (3 times a day). 2. metformin 500 mg tablet sig: one (1) tablet po bid (2 times a day): *** can be increased to home dose of 1000 as needed ***. 3. medication humalog insulin sliding scale 4. senna 8.6 mg tablet sig: one (1) tablet po bid (2 times a day) as needed for constipation. 5. docusate sodium 100 mg capsule sig: one (1) capsule po bid (2 times a day). 6. bisacodyl 10 mg suppository sig: one (1) suppository rectal hs (at bedtime) as needed for constipation. 7. acetaminophen 325 mg tablet sig: 1-2 tablets po q6h (every 6 hours) as needed for pain, fever. discharge disposition: extended care facility: of discharge diagnosis: primary: acute right basal ganglia hemorrhage atrial fibrillation secondary: hypertension diabetes mellitus discharge condition: triple flexes left leg to noxious, does not wiggle left fingers, flattening of left nlf, no sensation to light touch on left side, dysarthria. discharge instructions: you were admitted to the hospital after a fall, and were found to have a large hemorrhage in the right side of your brain. the cause of your hemorrhage is likely from high blood pressure. you were initially admitted to the neuroicu, and were transferred to the neurology floor when stable. your coumadin was discontinued and should not be added back until 4 weeks from now. you also developed atrial fibrillation with a rapid heart rate, and were started on diltiazem 60 mg three times a day. your metformin was initially held during this hospitalization and were given insulin. your metformin was added back at half your home dose (500 twice daily) and can be increased to your home dose as needed (1000 twice daily). if you develop increased confusion, weakness or numbness, difficulty speaking or swallowing, decreased vision or blurry vision, fevers/chills, or any other symptoms that concern you, call your pcp or return to the ed. followup instructions: you have a repeat ct chest as an outpatient () on at 1:30 in the center, . you should not eat or drink for 3 hours prior to this test. you have a follow up appointment with dr. in neurology () on at 1:30 pm in the center, . md, procedure: other electric countershock of heart diagnoses: congestive heart failure, unspecified unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled atrial fibrillation intracerebral hemorrhage pneumonitis due to inhalation of food or vomitus long-term (current) use of anticoagulants other pulmonary embolism and infarction 

Answer: The patient is high likely exposed to