Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: latex attending: chief complaint: abdominal distension, jaundice major surgical or invasive procedure: paracentesis egd history of present illness: mrs. is a 48 yo female with 18 year h/o of etoh abuse presenting with jaundice, abdominal discomfort and bright red blood per rectum. mrs. reports a 9 month h/o of fatigue, abdominal pain and distension for which she saw her pcp in . she was told she likely had alcoholic hepatitis and was advised to stop drinking for which she has been struggling for the past several months. husband reports noting scleral icterus and worsening of patient's baseline fatigue 10 days prior to presentation. she had been celebrating at a niece's wedding and may have been drinking more etoh. 4 days prior to admission, she experienced brisk bright red blood per rectum during a bowel movement and continued to bleed for 34 hours. 3 days prior to admission, she reports new onset cough and low grade fever to 100.7 with nbnb emesis x 2. she was convinced by her husband and pcp to come into the ed for evaluation. patient denies further episodes of brbpr, emesis, fevers, chills, confusion, nausea, denies acetaminophen use. + pruritis. past medical history: gastritis () alcoholic hepatitis () corneal transplants, pyelonephritis () rib fracture () hemorrhoids bulimia genital herpes hypothyroidism social history: patient splits her time between and , where her husband of 10 years works. she is currently not working. denies tobacco use, illicit drug use. etoh: >2 bottles of wine/day x 18 years. + for cage (cge), has occasional blackouts according to husband. reports that last drink was . longest period of abstinence was 90 days. no h/o of delerium tremens, seizures. family history: paternal grandfather: etoh father, sister, paternal grandfather: ankylosing spondylitis physical exam: pe: t 99.2 bp 120/80 hr 96 rr 16 weight 61.5 kg height 5'7'' general: pleasant female non toxic, nad, jaundiced, appropriate and aox3 heen: sclera icteric, perrl, eomi, opc, mmm neck: no jvd lungs: cta bilaterally heart: rrr, 2/6 sem on left sternal border 2ics abdomen: distended, firm and tender to deep palpation with maximal tenderness in epigastric area. liver span 5 cm below rib cage. tympanic to percussion. extremities: no edema, flapping tremmor. palmar erythema, skin: jaundiced, multiple spider angioma on chest and back. neuro: aox3. + asterixis. able to repeat months of year backwards. pertinent results: 04:30pm pt-23.9* ptt-35.6* inr(pt)-2.4* 06:50am alt(sgpt)-31 ast(sgot)-116* ld(ldh)-173 alk phos-205* tot bili-37.8* 10:45am hbs ab-negative hav ab-positive 10:45am hcv ab-negative 10:45am afp-4.8 06:50am wbc-7.8 rbc-2.75* hgb-10.3* hct-30.2* mcv-110* mch-37.4* mchc-34.0 rdw-16.9* brief hospital course: 48 yo female with long history of alcohol use and previous episode of etoh hepatitis and likely etoh cirrhosis presenting with acute etoh hepatitis who presented with jaundice, abdominal pain and bright red blood per rectum. . 1.) etoh cirrhosis/etoh hepatitis: patient likely has chronic etoh cirrhosis with acute etoh hepatitis and possible acetaminophen toxicity. she was found to have a tbili of 37.8, inr of 2.2 and albumin of 3.2 and slightly elevated transaminases. these were all felt to be consistent with etoh cirrhosis with an acute etoh hepatitis. she was started on pentoxifylline for the hepatitis. she was also treated with a full course of acetylcysteine for the question of acetaminophen toxicity as it was unclear the timeframe of her acetaminophen ingestion. she was given thiamine, folate, vitamin b12 and vitamin k. patient developed signs of hepatic encephalopathy on hospital day 3 and was started on lactulose titrated to bowel movements. rifaximin was added on day 10 due to inability to tolerate diarrhea and continued encephalopathy. on hospital day 15, patient experienced one episode of hemetemesis (500cc). patient was given 1.5 l ns, typed, screened and crossmatched and sent to the ccu for further management. portal hypertensive gastropathy was found on egd. after return to the floor, patient developed hepatorenal syndrome with a creatinine of 3.9. she was started on midrodrine and octreotide. she developed an aspiration pneumonia with an o2 requirement of 10l facemask. after discussion with patient and family, all medications and treatments were discontinued and she was provided comfort measures only with morphine drip. patient expired on . . 2.) abdominal pain/distension: patient complained of abdominal pain thought to be due to hepatitis and abdominal distension. given patient's low grade temperature, there was a concern for sbp. three diagnostic paracenteses, however, all showed no sbp. patient's pain was well controlled by oxycodone 5-10 mg q4-6h. . 3.) bright red blood per rectum: patient had no further episodes of bright red blood per rectum during her hospital course. her reported episode was most likely due to hemorroidal bleeding. her coagulopathy (inr 2.2) was felt to be the result of her chronic liver disease and decreased hepatic synthetic function. she was given vitamin k sq with no improvement in liver function. . 4.) elevated wbc: on hospital day 5, patient developed an elevated white blood count, cough, and low grade temperatures. extensive workup with blood cx, urine cx, cxr, ct torso and c. dif toxin assays failed to reveal a source of the leukocytosis. sbp was ruled out by diagnostic paracentesis. this was felt to be due to hepatitis and a systemic inflammatory process. . 5.) nutrition: due to the poor nutritional intake and increased caloric demand, a post pyloric dobbhof nasointestinal tube was placed by ir on . on , patient vomitted up feeding tube. after two failed attempts of placing a nasogastric tube on the floor and one further attempt to place a nasointestinal tube by fluorscopy, the tube was replaced by fluoroscopy under conscious sedation with versed on . patient was restarted on tube feedings and tolerated well. after transfer to the ccu, tubefeedings were stopped. . 6.) hyponatremia: patient developed hyponatremia to serum sodium of 128 believed to be secondary to cirrhosis and resultant hyperdynamic state. she was fluid restricted to 1000 cc per day without improvement in her serum sodium. . 7.) renal failure: patient's creatinine became elevated to 1.5 from a baseline of 1.0 on admission. this was concerning for hepatorenal syndrome vs. intravascular volume depletion. patient's renal function improved with administration of ns bolus and was felt to be due to dehydration. patient's renal function continued to fluctuate, but continued to be responsive to ns bolus. after episode of hemetemesis and transfer to ccu, patient developed hepatorenal syndrome with worsening of renal function. nephrology was consulted who felt that dialysis was not an option given her tenuous state. medications on admission: 1. acamprosate 333 mg tablet, delayed release (e.c.) sig: two (2) tablet, delayed release (e.c.) po tid (3 times a day). 2. hexavitamin tablet sig: one (1) cap po daily (daily). 3. folic acid 1 mg tablet sig: one (1) tablet po daily (daily). discharge medications: discharge disposition: expired discharge diagnosis: alcoholic cirrhosis with acute alcoholic hepatitis discharge condition: . discharge instructions: . followup instructions: . procedure: other endoscopy of small intestine enteral infusion of concentrated nutritional substances percutaneous abdominal drainage diagnoses: alcoholic cirrhosis of liver acute kidney failure, unspecified hepatorenal syndrome hyposmolality and/or hyponatremia pneumonitis due to inhalation of food or vomitus other ascites other and unspecified alcohol dependence, continuous acute alcoholic hepatitis 

Answer: The patient is high likely exposed to