Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: v fib arrest major surgical or invasive procedure: iabp placement cardiac catheterization central venous catheter arterial cannulation history of present illness: the patient is a 42 yo m with a reported hx of alcoholism who was reportedly feeling poorly yesterday afternoon. he went to his friend's house where he had a witnessed arrest with associated seizure like activity. cpr was started on the scene by nurse bystander. aed was on scene and gave pt two shocks. pt subsequently went into sinus tach with rosc. pt was also given narcan and became combative. . was brought to osh and started on amio, heparin , , intubated and received 2l cool saline but not started on artic sun. ekg showed inferior ste. labs were notable for hyperkalemia of 6.5, troponin of 0.06. on way to pt went into vtach requiring another shock. . in ed ekg showed ste in inferior leads, u/s showed paradoxical septal wall motion and overall hypokinesis, worsened by dopamine. amio drip started. dopamine gtt was started while waiting for cath lab due to pressures 50s/30s. . in the cath lab, found to have circ lesion, stent placed, concern for wide open mr. oddly, mvo2 was 85%, and shunt run was negative. iabp was placed. past medical history: restless leg syndrome alcoholism social history: per mother, patient only has hx of alcoholism (30 pack of beer every day) and was feeling tired today. otherwise in usoh prior to incident. as far as mother knows, does not abuse drugs. family history: non-contributory physical exam: admission exam vs: t=afebrile bp=96/64 hr=78 rr=24 o2 sat=100% general: intubated, sedated, on arrival moving all extremities heent: ncat. sclera anicteric. perrl, op with poor dentition and numerous loose teeth neck: supple, jvp not elevated cardiac: pmi located in 5th intercostal space, midclavicular line. rrr, heart sounds distant, difficult to hear any mrg lungs: no chest wall deformities, scoliosis or kyphosis. resp were unlabored, no accessory muscle use. lungs with course sounds throughout but no crackles. abdomen: soft, ntnd. no hsm or tenderness. extremities: no c/c/e. no femoral bruits. skin: no stasis dermatitis, ulcers, scars, or xanthomas. pulses: right: carotid 2+ femoral 2+ popliteal 2+ dp 2+ pt 2+ left: carotid 2+ femoral 2+ popliteal 2+ dp 2+ pt 2+ . discharge exam vitals - tm/tc: 100.0/98.8 hr: 81 (81-98) rr: 18 bp: 104/66 (104-126/63-81) 02 sat: 91% ra (91-98%ra) weight: 91.8 kg general: well developed, well nourished male in no acute distress chest: ctab, no wheezes, no rales, no rhonchi cv: s1 s2, normal in quality and intensity, rrr, no murmurs rubs or gallops abd: soft, non-tender, non-distended, bs normoactive. no rebound/guarding, neg hsm. ext: wwp, no edema. dps, pts 2+. psych: alert and oriented x 3 pertinent results: admission labs 10:55pm blood wbc-20.2* rbc-4.72 hgb-15.7 hct-45.9 mcv-97 mch-33.3* mchc-34.2 rdw-12.6 plt ct-409 02:23am blood wbc-17.1* rbc-5.08 hgb-16.4 hct-48.3 mcv-95 mch-32.3* mchc-34.0 rdw-12.6 plt ct-400 02:23am blood neuts-84.2* lymphs-10.0* monos-5.2 eos-0.4 baso-0.1 10:55pm blood pt-13.7* ptt-72.0* inr(pt)-1.2* 10:55pm blood fibrino-361 04:28am blood fibrino-858*# 02:23am blood glucose-159* urean-19 creat-1.0 na-137 k-4.7 cl-107 hco3-19* angap-16 02:23am blood alt-58* ast-234* ld(ldh)-437* alkphos-87 totbili-0.5 05:25am blood ck(cpk)-7100* 10:55pm blood lipase-23 05:25am blood ck-mb-greater th ctropnt-2.99* 08:30pm blood ck-mb-450* ctropnt-5.64* 04:08am blood ck-mb-218* mb indx-2.2 04:28am blood ck-mb-10 mb indx-0.6 02:23am blood calcium-8.5 phos-4.6* mg-2.3 03:56am blood albumin-3.0* calcium-7.8* phos-1.6* mg-2.0 03:56am blood hapto-178 10:53am blood d-dimer-338 02:31pm blood %hba1c-5.5 eag-111 06:07am blood triglyc-170* hdl-18 chol/hd-5.4 ldlcalc-45 10:55pm blood -neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 11:04pm blood glucose-130* lactate-3.7* na-141 k-4.1 cl-109* calhco3-21 10:55pm urine bnzodzp-pos barbitr-neg opiates-neg cocaine-neg amphetm-neg mthdone-neg . discharge labs 06:20am blood wbc-13.0* rbc-3.67* hgb-12.4* hct-34.2* mcv-93 mch-33.8* mchc-36.2* rdw-13.1 plt ct-565* 05:16am blood pt-13.8* ptt-26.3 inr(pt)-1.2* 06:09am blood glucose-91 urean-11 creat-0.8 na-142 k-3.6 cl-103 hco3-25 angap-18 04:28am blood alt-142* ast-133* ld(ldh)-635* ck(cpk)-1650* alkphos-57 totbili-0.5 06:09am blood calcium-8.4 phos-3.6 mg-1.8 . microbiology , urine culture (final): no growth , blood culture (final): no growth catheter tip culture (final): due to mixed bacterial types, no further workup performed sputum culture (final): coag + staph aureus, sparse growth clindamycin----------- r erythromycin---------- r gentamicin------------ <=0.5 s levofloxacin----------<=0.12 s oxacillin-------------<=0.25 s trimethoprim/sulfa---- <=0.5 s . imaging cardiac cath 1. selective coronary angiography of this left dominant system demonstrated two vessel coronary artery disease. the lmca was patent. the lad had minor diffuse disease. the lcx had a tubular proximal lesion of abotu 80% with possible thrombosis. the rca had a chronic occlusion mid-vessel with collaterals filling from the left coronary system. 2. limited resting hemodyanmics revealed elevated right and left-sided filling pressures with pasp of 54mmhg and mean pcwp of 35mmhg. 3. left ventriculography revealed global hypokinesis with an estimated lv ejection fraction of less than 30%. there was also + mitral regurgitation. cardiac index was preserved at 4.5 l/min/m2 using a pulmonary arterial saturation of 85% in the fick equation. no shunts were observed. 4. an intra-aortic balloon pump was placed sheathed in the right femoral artery. final diagnosis: 1. two vessel coronary artery disease. 2. severe mitral regurgitation. 3. severe systolic ventricular dysfunction. 4. acute inferior myocardial infarction, managed by acute ptca. ptca of vessel. 5. intra-aortic balloon pump placement. . echo: the left atrium is elongated. there is moderate regional left ventricular systolic dysfunction with inferior/inferolateral akinesis and anteroseptal hypokinesis/akinesis (lvef approximately 35 percent). right ventricular chamber size is normal. with normal free wall contractility. the aortic valve leaflets (3) are mildly thickened. there is no aortic valve stenosis. no aortic regurgitation is seen. the mitral valve leaflets are structurally normal. physiologic mitral regurgitation is seen (within normal limits). there is borderline pulmonary artery systolic hypertension. there is no pericardial effusion. compared to , left ventricular function appears less depressed but views are suboptimal for comparison. . cxr: mild interstitial pulmonary edema predominantly in the right upper lung is new. the large right lower lobe consolidation most likely aspiration pneumonia is stable, now accompanied by at least a small if not larger right pleural effusion. intra-aortic balloon pump has been removed. ascending swan-ganz rotation catheter ends in the right descending pulmonary artery. et tube in standard placement. nasogastric tube loops in the upper stomach. no left pleural effusion. no pneumothorax. . ct c-spine w/o contrast: there is no evidence of acute fracture or malalignment. no prevertebral soft tissue edema or hematoma is seen. the visualized portion of the aerodigestive tract is grossly unremarkable. multiple small bilateral cervical lymph nodes do not meet ct size criteria. there is at least a moderate right pleural effusion. secondary interlobular septal thickening seen in the lung apices are consistent with mild pulmonary edema. the thyroid gland is grossly unremarkable. scattered air-fluid levels are seen within left mastoid air cells. the visualized portions of the right mastoid air cells are well aerated. a mucus retention cyst is seen within the left maxillary sinus. . ankle (ap, mortise & lat) left port: three views of the left ankle were obtained. the patient is status post orif of a an old distal left fibular fracture, with lateral plate and interlocking screws. hardware is intact, in proper position and without signs of complication. an old fracture is also noted of the distal tibia, with narrowing and large osteophytosis of the tibiotalar joint. an ankle joint effusion and soft tissue swelling are seen. no new fracture or dislocation is identified. brief hospital course: 42 yo m with a reported hx of alcoholism but no know cardiac history admitted from osh with vfib arrest and stemi, already intubated for airway protection. . # v-fib arrest: it is unclear how long pt was down prior to cpr initiation and shock, but on arrival to , pt was moving all extremities and responsive, thus cooling protocol was not initiated. etiology of arrest is most likely secondary to ischemia, given pt also presented with stemi and cath lab revealed large circ lesion, as described below. pt was loaded on amiodorone and on hd1 he had several runs of nsvt, one of which was with rate in 200's and lasted for approximately 10 sec. afterwards, he was started on lidocaine drip and telemetry remained normal. he was eventually weaned off pressors and amiodarone was discontinued as pt did not have an indication for continuation of therapy. . #cardiogenic shock: pt has no known history of chf, but cath lab shows evidence of severe mr of 30% with global hypokinesis. also elevated co (consistent with mr) and elevated left sided filling pressures. there is no evidence of right sided strain and no evidence of tamponade. only unexplained factor is elevated mixed venous sats, which would raise concern for sepsis and a rll opacity was shown on cxr, concerning for pneumonia (hap v. aspiration). he was initially put on vancomycin, cefepime, and flagyl but as his sputum culture grew mssa, he was only continued on cefepime and then po cefpodoxime for a total 8 day course of antibiotics. pt initially required iabp and pressor support to maintain adequate perfusion maps but as his pneumonia was being treated, he slowly recovered his vascular resistance and was weaned off the balloon pump and pressor support. . #cad: prior to hospitalization, pt did not have known cad. cath revealed two vessel disease, stenosed lcx and there was successful pci of the proximal lcx with a 2.75 x 12 mm integrity bms. he was started on prasugril 60mg load and then transitioned to 10mg daily but this was not continued upon discharge. he was also started on aspirin 325mg, metoprolol xl 12.5mg daily, lisinopril 5mg daily, atorvastatin 80mg daily, and clopidogrel 75mg daily, and was discharged on 325mg daily, lisinopril 5mg daily, metoprolol succinate 25mg daily, simavastatin 40mg daily. . #rll pneumonia: pt had a wbc up to 20.2 with a left shift and rll pneumonia became apparent on chest x-ray from and pt was started on vancomycin, cefepime, and flagyl but as his sputum culture grew mssa, he was only continued on cefepime and then po cefpodoxime for a total 8 day course of antibiotics. wbc normalized throughout the hospital course, and was 13.0 the day of discharge. . # etoh history: pt's family reports that he drinks 30 beers every day. labs from osh showed negative serum tox for alcohol. urine tox screen was negative for other substances. pt does not have know history of cirrhosis or alc hep. he had a mild alcoholic transaminitis on admission (but this also could be explained by acute ischemia), but his liver's synthetic function was normal. initially he was requiring very high of fentanyl and versed to achieve adequate sedation. pt was started on thiamine, folate and multivitamin. ciwa protocol was not intitiated because during the period of expected withdrawal, pt was receiving benzodiazepines while he was intubated and sedated. pt reports an understanding that he will need to change his behavior and activity regarding his alcohol intake and abuse. . transitional: # recommend support system for etoh abuse medications on admission: none. discharge medications: 1. aspirin 325 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 2. thiamine hcl 100 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 3. folic acid 1 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 4. famotidine 20 mg tablet sig: one (1) tablet po bid (2 times a day). disp:*60 tablet(s)* refills:*2* 5. lisinopril 5 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 6. metoprolol succinate 25 mg tablet extended release 24 hr sig: 0.5 tablet extended release 24 hr po daily (daily). disp:*30 tablet extended release 24 hr(s)* refills:*2* 7. clopidogrel 75 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 8. simvastatin 40 mg tablet sig: one (1) tablet po once a day. disp:*30 tablet(s)* refills:*2* discharge disposition: home discharge diagnosis: ventricular fibrillation arrest st elevation myocardial infarction cardiogenic shock aspiration pneumonia wtih methacillian sensitive staph aureus discharge condition: mental status: clear and coherent. level of consciousness: alert and interactive. activity status: ambulatory - independent. discharge instructions: you had a heart attack and a fatal heart rhythm called ventricular fibrillation that required a shock. you were brought to and a stent was placed in your heart artery to open it. it is extremely important that you take aspirin and plavix every day to prevent the stent from clotting off. do not stop taking aspirin and plavix or miss unless dr. tells you it is ok. you will need to stop smoking and drinking all alcohol in order to let your heart heal from the heart attack. you will need to take new medicines to help your heart recover and prevent another heart attack in the future. physical therapy reviewed your recommended activity for the next month at home. your heart is weaker after the heart attack and you will need to monitor yourself for fluid buildup at home. weigh yourself every morning before breakfast, call dr. if weight goes up more than 3 lbs in 1 day or 5 pounds in 3 days. you need to avoid salt in your diet, a teaching packet was given to you about a low salt diet. your memory is only slightly impaired during the evaluation by occupational therapy. you can call dr. at if you notice that you are having any trouble with your memory or thinking. . we have started you on the following medicines: 1. start taking aspirin and plavix every day to prevent the stent from clotting off and causing another heart attack. 2. start taking metoprolol to prevent another heart attack 3. start taking lisinopril to help your heart pump better 4. start taking simvastatin to lower your cholesterol and prevent another heart attack. 5. start taking thiamine and folic acid to help your nutrition 6. start taking famotidine to protect your stomach from the plavix and aspirin. followup instructions: name: dr specialty: cardiology , , ma phone: when: friday, octber 7th at 2:15pm procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of non-drug-eluting coronary artery stent(s) combined right and left heart cardiac catheterization coronary arteriography using two catheters angiocardiography of left heart structures enteral infusion of concentrated nutritional substances implant of pulsation balloon cranial or peripheral nerve graft insertion of one vascular stent excision of lingual thyroid percutaneous transluminal coronary angioplasty [ptca] procedure on single vessel diagnoses: other primary cardiomyopathies acidosis hyperpotassemia thrombocytopenia, unspecified coronary atherosclerosis of native coronary artery tobacco use disorder mitral valve disorders unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled severe sepsis methicillin susceptible staphylococcus aureus septicemia other and unspecified hyperlipidemia acute respiratory failure pneumonitis due to inhalation of food or vomitus cardiogenic shock septic shock acute myocardial infarction of other inferior wall, initial episode of care methicillin susceptible pneumonia due to staphylococcus aureus ventricular fibrillation unspecified accident other disorders of neurohypophysis other and unspecified alcohol dependence, continuous restless legs syndrome (rls) other sprains and strains of ankle 

Answer: The patient is high likely exposed to