Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: ventricular tachycardia major surgical or invasive procedure: none history of present illness: 46yo woman with pmh of vfib arrest thought secondary to prinzmetal's angina, recently discharged from ccu one week ago, and htn, was transferred to from () after ventricular tachycardia. the patient was last admitted on with chest pain. she was noted to have ste in the anterior leads at an outside hospital, sent to cardiac catheterization here. at cath, she was noted to have mild irregularity of her coronary vessels but no stenosis. she was maintained on nitrates and norvasc with improvement of her chest pain, and was discharged on . this morning, she noted chest pain at 2am. ems was called; while in the field she entered vt. at the osh, she was defibrillated x 3 without success, but entered "fine vfib." in total, per osh chart, she received defibrillation x 7, was loaded with amiodarone, and returned to nsr. she was in vt/vf for approximately 30mins with some component of cpr. . upon arrival, she had minimal neurologic functioning. she was admitted to the ccu. past medical history: prinzmetal's angina coronary vasospasm hypertension social history: denies tobacco, etoh, ivda employed at 5 children family history: no cad physical exam: per neurology note, vitals: hypotensive, hr 90's, rr 16, o2 99% gen: intubated, breathing deeply, very high frequency tremor of her lower lip and tongue heart: rrr lungs: cta anteriorly abd: obese, soft ext: cannot palpate peripheral pulses ms: unresponsive to voice or sternal rub. cn: pupils 2 mm and unresponsive, absent doll's, absent corneals, absent gag, no grimace to nasal tickle. is breathing agonally spontaneously. did not test cold calorics. reflexes: trace throughout, toes mute : does not withdraw or posture to deep stim pertinent results: labs: cbc: wbc-23.6*# rbc-4.67# hgb-14.2# hct-41.3# plt ct-275# diff: neuts-80* bands-3 lymphs-10* monos-0 eos-0 baso-0 atyps-0 metas-6* myelos-1* nrbc-2* coags: pt-13.4* ptt-31.7 inr(pt)-1.2 chem10: glucose-365* urean-16 creat-1.4* na-135 k-5.4* cl-101 hco3-8* calcium-8.5 phos-7.6*# mg-2.2 enzs: alt-107* ast-329* ck(cpk)-8773* alkphos-98 totbili-0.6 albumin-4.2 card enzs: ck-mb-364* mb indx-4.1 ctropnt-4.92* tox: -neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg abg: 06:22am blood type-art temp-37.2 rates-/0 tidal v-600 peep-5 fio2-100 po2-385* pco2-26* ph-7.26* calhco3-12* base xs--13 aado2-306 req o2-57 intubat-intubated vent-controlled 08:16am po2-108* pco2-30* ph-7.16* calhco3-11* base xs--16 09:31am temp-36.7 rates-22/22 tidal v-600 peep-5 fio2-50 po2-92 pco2-36 ph-7.20* calhco3-15* base xs--12 -assist/con intubat-intubated 11:50am po2-155* pco2-34* ph-7.08* calhco3-11* base xs--19 lactate: 7.7 -> 7.8 -> 14.8 freeca-1.15 echo - there is mild symmetric left ventricular hypertrophy. the left ventricular cavity size is normal. there is severe global left ventricular hypokinesis (ejection fraction 20-30 percent). right ventricular chamber size is normal. right ventricular systolic function appears depressed. the mitral valve leaflets are structurally normal. there is a trivial/ physiologic pericardial effusion. eeg - this is a markedly abnormal routine eeg due to the presence of the absence of a clear background activity. the described low voltage fast activity could represent cable artifacts throughout the record. the underlying background activity cannot be excluded. this finding represents the midline subcortical dysfunction and is consistent with a severe encephalopathy. there were no epileptiform discharges seen. a sinus tachycardia was noted. ekg - sinus tachycardia. consider anteroseptal myocardial infarct, age indeterminate since previous tracing of same date, sinus tachycardia present brief hospital course: assessment - 46yo woman with prinzmetal's angina, htn, s/p prolonged ventricular fibrillation arrest, with minimal neurologic functioning on admission, absent brainstem reflexes except for some spontaneous agonal breathing, made cmo by family. hospital course - the patient presented s/p vfib arrest, minimal neurologic functioning. echo showed severe lv hypokinesis. the patient was maintained on pressors and a ventilator initially. she was seen by the neurology service, who assessed her poor function as a result of hypoxic injury in the setting of her arrest. status epilepticus was originally in the differential, but eeg was c/w severe encephalopathy, and had no epileptiform discharges. her prognosis was discussed with her family, who decided to make the goals of care comfort measures. she was taken off the ventilator and all medications except morphine and tylenol, and she died soon thereafter with her extended family at the bedside. the family requested an autopsy. medications on admission: lisinopril iron discharge medications: n/a discharge disposition: expired discharge diagnosis: cardiogenic shock ventricular fibrillation cardiac arrest prinzmetal's angina discharge condition: expired discharge instructions: n/a followup instructions: n/a md procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours infusion of vasopressor agent diagnoses: unspecified essential hypertension anoxic brain damage ventricular fibrillation acute myocardial infarction of unspecified site, initial episode of care acute myocardial infarction of unspecified site, subsequent episode of care prinzmetal angina 

Answer: The patient is high likely exposed to