Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: fall with closed head injury major surgical or invasive procedure: none history of present illness: 63 year old male +etoh s/p fall from standing position with +loc. he went to an osh and performed a ct scan of his head which revealed a large right frontal sah with extension to the left frontal lobe as well. the patient received ativan at the osh and was transferred here for further evaluation. the patient is awake and following commands here. he had a repeat ct scan which is stable compared to the osh. the patient is not able to provide further history due to his intoxicated state. past medical history: +etoh abuse social history: heavy etoh use family history: unknown physical exam: physical exam: t:98.1 bp:142/84 hr:102 rr:14 o2sats:98% gen: intoxicated, lying on stretcher. heent: pupils:perrl eoms-intact neck: no tenderness, in cervical collar. lungs: cta bilaterally. cardiac: rrr. s1/s2. abd: soft, nt, bs+ extrem: warm and well-perfused. neuro: mental status: drowsy, participates with simple commands. orientation: oriented to person only. language: dysarthric. cranial nerves: i: not tested ii: pupils equally round and reactive to light, 5 to 4 mm bilaterally. iii, iv, vi: extraocular movements intact bilaterally without nystagmus. v, vii: facial strength and sensation intact and symmetric. viii: hearing intact to voice. ix, x: palatal elevation symmetrical. : sternocleidomastoid and trapezius normal bilaterally. xii: tongue midline without fasciculations. motor: normal bulk and tone bilaterally. no abnormal movements, tremors. moves all 4 spontaneously and is antigravity but will not participate with strength testing. unable to test drift. sensation: appears intact to light touch bilaterally. toes downgoing bilaterally pertinent results: ct head w/o contrast findings: there is a large approximately 5.1 x 3.5 cm right frontal intraparenchymal hemorrhage. there is perilesional edema with effacement of overlying sulci. there is associated subarachnoid blood. this represents a significant increase from the prior study. there is apparently new left-sided subdural hematoma (series 2a, image 14). again there is hematoma also too in the inferior aspect of the left frontal lobe. there is now approximately 4 mm of leftward shift, new from prior. the right lateral ventricle is slightly effaced. basal cisterns are preserved. there is no depressed skull fracture. mastoid air cells are clear. visualized paranasal sinuses are unremarkable. impression: significant increase in right frontal intraparenchymal hematoma. left frontal hematoma small but increased from prior. subdural and subarachnoid components also demonstrated. ct head w/o contrast findings: again seen are bifrontal hematomas, larger on the right measuring approximately 4.7 x 2.2 cm similar to prior with similar perilesional edema and leftward shift to 8 mm. there is a component of the subarachnoid hemorrhage involving the right frontal lobe. there is a stable effacement of the overlying sulci. left frontal subdural hematoma appears unchanged. a component of a left parietal subarachnoid hemorrhage is more prominent and likely represents re-distribution. ventricles, sulci and cisterns are similar to prior with mild effacement of the right frontal of the lateral ventricle. basal cisterns are preserved. mastoid air cells and visualized paranasal sinuses clear. impression: overall similar appearance of bifrontal intraparenchymal hematomas left subdural hematoma. left parietal subarachnoid is more prominent likely representing re-distribution. ct c-spine w/o contrast findings: there is no acute cervical fracture or malalignment. the prevertebral soft tissues are within normal limits. there are multilevel degenerative changes, most marked at the c4/5 and c6/7 levels. the visualized lung apices are unremarkable. the visualized mastoid air cells are clear. impression: 1. no acute fracture or malalignment of the cervical spine. 2. multilevel, multifactorial degenerative changes most marked at the c4/5 and c6/7 levels, where disc-endplate sponylotic ridges efface the ventral theca and flatten the cord (3:36,46). this confers an increased risk of spinal cord injury even in the setting of slight trauma. if clinical suspicion is high, recommend mri. ct head w/o contrast findings: again seen are bifrontal parenchymal hematomas, likely hemorrhagic contusions, larger on the right measuring approximately 4.8 x 2.2 cm, with similar perilesional edema and leftward 9 mm shift. there is stable effacement of overlying sulci. the thin subdural hematoma layering over the left frontal convexity and measuring only 3.5mm in maximal thickness (3:17), appears stable. however, a new subdural hemorrhage has emerged, layering over the right frontal and temporal convexity, measuring 8.5mm maximally, with mild effacement of subjacent gyri (3:). the component of left parietal subarachnoid blood appears to have resolved or may be obscured by motion artifact. the ventricles, sulci, and cisterns are similar, with mild effacement of the frontal of the right lateral ventricle. basal cisterns are preserved. mastoid air cells and visualized paranasal sinuses are clear. impression: similar appearance of bifrontal hemorrhagic contusions and thin left subdural hematoma, with emergence of right frontal subdural hematoma, having only minimal mass effect. 6.5 mm of leftward shift of midline structures is similar to prior. note added in attending review: the presence of abundant subarachnoid hemorrhage in the cistern of the lamina terminalis, present on the series of studies dating to the original nect ( hosp, 2042h, ) is unusual in the setting of trauma, and was associated with extensive sah in the suprasellar cistern at presentation, much of which has either redistributed or resorbed. this raises the possibility of underlying aneurysmal bleed (with secondary traumatic hemorrhage, related to the patient's fall), particularly from an acom aneurysm. the findings should be closely correlated with nature of the patient's original trauma (ie. truly mechanical?), as well as any premonitory symptoms, and consideration might be given to dedicated cta. cta head w&w/o c & recons there has been a slight interval increase in edema and mass effect along the superior aspect of the right frontal hemorrhage. accounting for differences in slice selection and angulation, there is no interval increase in the size of the right frontal hemorrhage. there is stable left inferior frontal hemorrhage with surrounding edema, unchanged. there is slightly increased effacement of the overlying right frontal sulci. there is a 9-19 mm right subfalcine herniation, without significant change. there is stable subarachnoid hemorrhage within the suprasellar cistern. there is stable acute on chronic left frontal/temporal subdural hemorrhage. there is stable acute on chronic, although predominantly acute, right frontal/temporal subdural hemorrhage. no intraventricular hemorrhage is identified. no hydrocephalus is seen. cta head: anterior circulation: there is a 1 mm triangular focal outpouching pointing inferiorly in the mid anterior communicating artery, which may represent a small aneurysm or infundibular origin of a penetrating vessel. no other evidence of an aneurysm is identified. the bilateral mcas and acas are unremarkable. there is calcification involving the bilateral intracranial carotid arteries. posterior circulation: the left vertebral artery is dominant. the right vertebral artery demonstrates a small focus of calcified plaque just distal to the origin of the right pica origin, causing mild narrowing. the remainder of the posterior circulation is unremarkable. there is no evidence for aneurysm, avm, or stenosis. impression: 1. 1 mm triangular focal outpouching from the mid aspect of the anterior communicating artery, which may represent a small aneurysm or an infundibular origin of a penetrating vessel. 2. stable suprasellar subarachnoid hemorrhage. stable acute on chronic bilateral frontal/temporal subdural hemorrhage. 3. stable right greater than left frontal lobar hemorrhage with surrounding edema, likely reflecting hemorrhagic contusions. slight increase in mass effect and edema surrounding the superior aspect of the right frontal hemorrhage. brief hospital course: mr. was admitted to the trauma icu on . c-spine was cleared by the trauma service. repeat imaging showed some evolution of contusions as expected. he remained stable. he was on a ciwa protocol and required a 1:1 sitter on for confusion. ct imaging on this date suggested aneurysmal origin but cta ruled out vascular anomaly. the patient was transferred to the neurosurgical floor on . he no longer required a sitter while on the floor. pt and ot evaluated him and determined that he would need rehab. on he was febrile to 101 f and he was pancultured. chest x-ray and lens were without significant finding. all cultures were negative as of . he had some hyponatremia on and salt tabs were started on for a na of 129. they were increased on for na of 128. he continued to be screeend for rehab and was discharged to an appropriate facility on . medications on admission: none discharge medications: 1. senna 8.6 mg tablet sig: one (1) tablet po bid (2 times a day) as needed for constipation. 2. docusate sodium 100 mg capsule sig: one (1) capsule po bid (2 times a day). 3. bisacodyl 10 mg suppository sig: one (1) suppository rectal daily (daily) as needed for constipation. 4. potassium & sodium phosphates 280-160-250 mg powder in packet sig: one (1) powder in packet po bid (2 times a day). 5. phenytoin sodium extended 100 mg capsule sig: one (1) capsule po q 8h (every 8 hours). 6. famotidine 20 mg tablet sig: one (1) tablet po bid (2 times a day). 7. thiamine hcl 100 mg tablet sig: one (1) tablet po daily (daily). 8. folic acid 1 mg tablet sig: one (1) tablet po daily (daily). 9. haloperidol 5 mg tablet sig: 0.5 tablet po q6h (every 6 hours) as needed for agitation. 10. menthol-cetylpyridinium 3 mg lozenge sig: one (1) lozenge mucous membrane q2h (every 2 hours) as needed for throat irritation. 11. acetaminophen 325 mg tablet sig: two (2) tablet po q6h (every 6 hours) as needed for fever/pain. 12. sodium chloride 1 gram tablet sig: two (2) tablet po tid (3 times a day). discharge disposition: extended care facility: discharge diagnosis: left frontal hematoma right frontal intraparenchymal hematoma perilesional cerebral edema sah ddd c4/5 and c6/7 levels discharge condition: neurologically stable discharge instructions: general instructions ?????? take your pain medicine as prescribed. ?????? exercise should be limited to walking; no lifting, straining, or excessive bending. ?????? increase your intake of fluids and fiber, as narcotic pain medicine can cause constipation. we generally recommend taking an over the counter stool softener, such as docusate (colace) while taking narcotic pain medication. ?????? unless directed by your doctor, do not take any anti-inflammatory medicines such as motrin, aspirin, advil, and ibuprofen etc. ?????? you have been prescribed an anti-seizure medicine, take it as prescribed and follow up with laboratory blood drawing in one week. please have results faxed to . ?????? call your surgeon immediately if you experience any of the following ?????? new onset of tremors or seizures. ?????? any confusion, lethargy or change in mental status. ?????? any numbness, tingling, weakness in your extremities. ?????? pain or headache that is continually increasing, or not relieved by pain medication. followup instructions: follow-up appointment instructions ?????? please call ( to schedule an appointment with dr. , to be seen in 4 weeks. ?????? you will need a ct scan of the brain without contrast. procedure: alcohol detoxification diagnoses: hyposmolality and/or hyponatremia cerebral edema alcohol abuse, continuous fall from other slipping, tripping, or stumbling degeneration of cervical intervertebral disc cortex (cerebral) contusion without mention of open intracranial wound, with loss of consciousness of unspecified duration 

Answer: The patient is high likely exposed to