Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no drug allergy information on file attending: chief complaint: transfer for cirrhosis major surgical or invasive procedure: intubation, a line placement history of present illness: 57-year old man with pmh of alchoholic cirrhosis with portal hypertensive gastropathy and esophagitis who initially presented on to with weakness and lightheadedness who is now presenting with worsening liver failure, renal failure, hypotension and encephalopathy. briefly, at the osh, he was admitted for above. he was also acting confused and had epistaxis. he was newly jaundiced and had pain in his r shoulder and r knee. during his hospitalization he was found to have mssa bacteremia, probable endocarditis, with septic emboli to brain, arf, and worsening bilirubinemia. first, respiratory-wise, he was intubated on for an ac joint washout. he was diuresed and extubated. his respiratory status stayed stable. as for his infection, he was started on azithro/ceftriaxone for pna initially. then blood cx on grew mssa. he was switched to vanco. then again switched to oxacillin/levoflox for eye penetration of his vitreous infection. ophtho followed. a tte was performed and showed no vegetation, and a tee was deferred because of risks and the mortality of cardiac surgery would be so high, that the management would not change. he did have a shoulder washout with cultures that grew mssa. his wbc trended upward. he had worsening pancytopenia and multiple transfusions while at osh. he also had worsening arf. he became oliguric and the thought was that it was secondary to nsaids and sepisis. he was dialyzed once and his cr improved and his urine output increased. over the last few days, his bun and cr have continued to rise again. he also was thought to have septic emboli to his brain. he had asterixis but was conversational until the day of transfer when his lethargy worsened. on the floor, initial vs were t 98.6, p 90, bp 119/42, r 12, o2 sat 97% on 2l. patient was intubated for airway protection as he was gurgling and not responding. he was posturing. he withdrew to pain only minimally. he was continued on his levofed gtt. propofol was tried for sedation, but not tolerated because of hypotension. fentanyl and versed was started for sedation. unable to obtain ros. past medical history: etoh cirrhosis with ascites portal gastropathy htn social history: married, 3 children and works as police detective. - tobacco: none - alcohol: hx of etoh abuse, quit after detox at , was drinking approximately - illicits: none family history: non-contributory, unable to obtain, per report no liver dx in family physical exam: vitals: t: bp: 97/53 p: 97 r: 15 o2: 95% @ 50% general: unarousable heent: sclera severely icteric, mmm lungs: coarse crackles and rhonchorous noises in all fields bilaterally cv: rrr, almost unable to hear over coarse breath sounds abdomen: soft, non-tender, distended, hyperactive bowel sounds ext: 4+ pitting edema bilaterally skin: bright yellow, sporadic spider angiomas across upper torso pertinent results: labs: 11:56pm urine color-yellow appear-clear sp -1.012 11:56pm urine blood-mod nitrite-neg protein-tr glucose-neg ketone-neg bilirubin-lg urobilngn-neg ph-5.5 leuk-tr 11:56pm urine rbc-81* wbc-9* bacteria-mod yeast-none epi-0 11:56pm urine granular-1* 10:39pm type-central ve po2-50* pco2-23* ph-7.38 total co2-14* base xs--9 10:39pm lactate-2.4* na+-127* k+-4.2 10:39pm freeca-1.08* 10:22pm glucose-91 urea n-138* creat-4.5* sodium-126* potassium-4.4 chloride-94* total co2-12* anion gap-24* 10:22pm estgfr-using this 10:22pm alt(sgpt)-21 ast(sgot)-48* ck(cpk)-27* alk phos-101 tot bili-23.7* 10:22pm ck-mb-notdone ctropnt-0.25* 10:22pm albumin-2.2* calcium-8.0* phosphate-9.1* magnesium-2.5 10:22pm wbc-20.7* rbc-2.82* hgb-8.4* hct-23.8* mcv-84 mch-29.6 mchc-35.2* rdw-18.7* 10:22pm neuts-91.3* lymphs-5.1* monos-2.9 eos-0.4 basos-0.2 10:22pm plt count-117* 10:22pm pt-37.7* ptt-107.9* inr(pt)-3.9* ct head: large intraventricular and probable left thalamic hemorrhage with moderate hydrocephalus. brief hospital course: 57m with etoh cirrhosis and worsening liver failure, encephalopathy, renal failure, leukocytosis and known mssa septic emboli transferred from . # etoh cirrhosis/acute liver failure: has liver failure with new encephalopathy, jaundice, and coagulopathy. not improving. per liver consult the patient is not a transplant candidate. initially treated with with octreotide, midodrine, blood products and lactulose. individual problems discussed below. # hypotension: likely septic shock in the setting of rising wbc. transferred on low dose levofed and tolerating well. did not tolerated propofol because of hypotension. levofed gtt started initially. infectious workup as below. # hyperbilirubinemia: liver failure. # coagulopathy: worsening coagulopathy and is oozing from nose and ng tube. inr is 3.9 and ptt over 100. initially given ffp and vitamin k via ngt. # altered mental status: ich found on ct. possibly also with encephalopathy from liver failure and contribution from uremia. patient intubated for airway protection. extubated as discussed below. # acute renal failure: has worsening renal failure with rising bun and cr. likely has uremia, although electrolytes are stable. was dialyzed once at osh. is not oliguric. initially treated with ivfs/levofed for kidney perfusion. renal consulted, however patient not considered suitable candidate for hd line and dialysis. # anion gap acidosis: likely from combination of renal failure and lactic acidosis. would not benefit from hco3 because worsens encephalopathy when broken down across the bbb. # leukocytosis/mssa bactermia: rising leukocytosis and known mssa bactermia. have new source of infection. etiologies could be line infection, sbp, or c. diff. cultures pending. initially started on broad antibiotics which were stopped given decision to make patient cmo. # hyponatremia: likely from hypervolemic hyponatremia in the setting of cirrhosis and ascites. # gi bleeding: ongoing bleeding from ng tube, guiac positive. hemodynamically stable on night of admission. initially transfused with rbcs and ffp. # respiratory failure: intubated for altered mental status and inability to protect his airway. patient terminally extubated after decision to make cmo as discussed below. # code status/goals of care: family meeting on second day of hospitalization to discuss poor prognosis. patient has intracranial bleed, end stage liver and renal failure. family made aware that he will not recover from these illnesses. decision was made to become comfort measures. dr. was present for the meeting, as was a social worker and the patient??????s nurse . the family will visit right now and plan for extubation later today. the patient was extubated in the early afternoon of and he slowly became bradycardic, hypotensive, bradypneic, and expired at 16:05. medications on admission: calcium acetate capsule 1334mg oxacillin sodium 2gm hydromorphone lansoprazole 30mg norephinephrine 4mg per criteria discharge medications: n/a discharge disposition: expired discharge diagnosis: terminal extubation liver failure intraventricular hemorrhage discharge condition: pt. expired discharge instructions: pt. expired followup instructions: pt. expired md procedure: insertion of endotracheal tube diagnoses: acidosis obstructive hydrocephalus unspecified essential hypertension acute and subacute necrosis of liver alcoholic cirrhosis of liver acute kidney failure, unspecified hyposmolality and/or hyponatremia severe sepsis other and unspecified alcohol dependence, in remission portal hypertension methicillin susceptible staphylococcus aureus septicemia intracerebral hemorrhage acute respiratory failure septic shock other ascites hepatic encephalopathy hemorrhage of gastrointestinal tract, unspecified esophagitis, unspecified embolism and thrombosis of other specified artery other specified disorders of stomach and duodenum other and unspecified coagulation defects septic arterial embolism 

Answer: The patient is high likely exposed to