Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins / iodine attending: chief complaint: headache major surgical or invasive procedure: angiogram history of present illness: 56 yo f with a hx of all s/p whole brain rt, essential tremor, syndrome, ocular myasthenia, various types of headaches, l sided ataxia, and a hx of episodes of near syncope, presented 2 days ago with a sah. she describes the event as a very sudden worst headache of her life while changing out of her business clothes after dinner. she did not fall at that time either before or after the onset of the headache. she also denies any hx of head trauma in the preceding days. she called her husband to call 911 and was taken to . she recived a head ct showing sah and was transferred to . here, she received a repeat confirmatory head ct and an angiogram which was negative for aneurysm. she has been treated with analgesics and antiemetics. this sah has occurred in the context of an ongoing history of episodes of lightheadedness and headaches have been worked up by drs. and of neurology. she states that these episodes have been going on for 8-9 years, ever since she had the whole brain rt. she states they occur roughly weekly, although she can go for longer periods without one. she states they were worse when she had been simultaneously on chemotherapy until , and then presented sometimes as frank syncope. more recently however, she denies any frank syncopal episodoes. she states that sometimes these episodes are accompanied by a band-like headache around her head, she she states that if she gets that headache first, it is a cue for her to sit down. she states that these headaches are different that her migraines, which are preceded by an aura of funny smells and flashing lights. she received a brain mri in after presenting to dr. with a complaint of one of these spells, which was negative. in early summer of she also underwent tilt table testing at , which was positive per her report. she does not have any history of seizures, and she denies any focal motor movements or loss of bowel/bladder continence during these episodes. she denies any other recent focal weakness, parasthesias, vertigo, or hearing changes. she does state that she has a history of intermittent diplopia c/w her ocular myasthenia. past medical history: all s/p whole brain rt essential tremor syndrome ocular myasthenia various types of headaches including migraine l sided ataxia hx of episodes of near-syncope social history: married, works in corporate banking, no tobacco or drugs, rare etoh family history: father with cad, sister with syndrome physical exam: t- 98.5 f bp- 121/74 hr- 82 rr- 21 o2sat 96%ra gen: lying in bed, nad heent: nc/at, moist oral mucosa, o/p clear neck: no tenderness to palpation, normal rom, able to touch chin to chest, but states that it is painful of she lingers in this position, no carotid or vertebral bruit back: no point tenderness or erythema cv: rrr, nl s1 and s2, no murmurs/gallops/rubs lung: clear to auscultation bilaterally abd: +bs soft, nontender ext: no edema neurologic examination: mental status: awake and alert, cooperative with exam, normal affect. oriented to person, place, and date. attentive, says dow backwards. speech is fluent with normal comprehension and repetition; naming intact. no dysarthria. and writing intact. registers , recalls in 5 minutes, and w/ semantic prompting. no right left confusion. no evidence of apraxia or neglect. cranial nerves: pupils equally round and reactive to light, 3 to 2 mm bilaterally. visual fields are full to confrontation. extraocular movements intact bilaterally, no nystagmus. right retina showing sharp disc margin and no hemorrhages, left retina poorly visualized. no double vision to extended upgaze. sensation intact v1-v3. facial movement symmetric. hearing intact to finger rub bilaterally. palate elevation symmetrical. sternocleidomastoid and trapezius normal bilaterally. tongue midline, movements intact motor: normal bulk bilaterally. tone normal. no observed myoclonus. no pronator drift tri wf we fe ff ip h q df pf te tf r 5 5 5 5 5 5 5 5 5 5 5 5 5 5 l 5 5 5 5 5 5 5 5 5 5 5 5 5 5 sensation: intact to light touch, pinprick, vibration and proprioception throughout. no extinction to dss reflexes: +2 and symmetric throughout. toes downgoing bilaterally coordination: (+) tremor on finger-nose-finger bilaterally with some dysmetria on the right (mildy misses to her right of mark and corrects), normal heel to shin, rams normal, finger tap normal. gait: deferred by pt pertinent results: 02:28pm glucose-133* 02:28pm albumin-3.9 02:28pm phenytoin-0.9* 02:28pm hct-34.1* 04:42am glucose-134* urea n-12 creat-0.7 sodium-144 potassium-3.6 chloride-108 total co2-27 anion gap-13 04:42am calcium-8.7 phosphate-4.1 magnesium-1.7 04:42am wbc-8.5 rbc-4.21 hgb-13.3 hct-36.4 mcv-87 mch-31.6 mchc-36.5* rdw-13.6 04:42am plt count-309 04:42am pt-12.7 ptt-20.1* inr(pt)-1.1 01:25am glucose-116* urea n-13 creat-0.7 sodium-144 potassium-4.1 chloride-105 total co2-31 anion gap-12 01:25am estgfr-using this 01:25am calcium-9.3 01:25am wbc-9.6 rbc-4.61 hgb-13.9 hct-39.9 mcv-87 mch-30.0 mchc-34.7 rdw-13.3 01:25am neuts-80.2* lymphs-16.6* monos-2.5 eos-0.2 basos-0.6 01:25am plt count-343 01:25am pt-12.2 ptt-22.6 inr(pt)-1.0 brief hospital course: 56 yo f with hx of of all s/p whole brain rt, essential tremor, syndrome, ocular myasthenia, various types of headaches, l sided ataxia, and a hx of episodes of near syncope admitted with a spontaneous sah that was negative for aneurysm (negative angiogram). her complaints of dizziness and syncope are consistent with the diagnosis of autonomic disorder (positive tilt-table test from caritas was requested and exam performed on revealed "patient was progressively tilted from lying down to 60 degrees. after 17 minutes of tilt at 60 degrees, her bp dropped to 74/52. she developped a severe headache and began to "feel funny". her hr was maintained. the table was tilted to the trendelenburg position. her bp rose over the next minute from 74/52 to 108/72 and eventually to 131/78". neurosurgery recommended the use of dilantin 100mg tid prophylatically for 1 week. patient also developed a rash on the back that was thought to be due to dilantin (which was stopped on ; she had supratherapeutic levels for 2 days.. she was oriented to seek medical support in case the rash does not improve and importantly, if it is on the face. no seizure or abnormalities on the neurological exam ocurred during admission. during admission patient complained of headache, frontal, dull in character, constant, without worsening factors that was controlled with oxycodone. patient also had an echocardiogram which was a normal study (lvef >55%). ct head report (): 1. right sided subarachnoid hemorrhage without hydrocephalus or mass effect. 2. small amount of left frontal subarachnoid blood is likely due to redistribution. repeat cta on : 1. bilateral subarachnoid hemorrhage less conspicuous with no new intracranial hemorrhage identified. potential new focus of edema in the right frontal lobe of unclear etiology though mri can be considered for further evaluation. 2. no aneurysm identified. brain mri (): bilateral subarachnoid hemorrhage reidentified with a small amount of subdural hemorrhage noted along the right convexity. the previously noted right frontal hypodensity seen on ct, most likely represents hemorrhage within the subarachnoid space. however, if this lesion is in fact intraparenchymal, contusion or subacute infarct are other diagnostic possibilities. recommend continued attenuation on followup. patient's headache improved over time and no change in the neurological examination occured. this case was also discussed in dr. rounds who suggested studies of hypercoagulability (which are pending upon discharge). he also suggested the performance of csf studies in a month to investigate for the possibility of the diagnosis of vasculitis. medications on admission: celexa 20 mg po qday imitrex prn mvi qday fish oil tab qday vitamin d qday discharge medications: 1. senna 8.6 mg tablet sig: one (1) tablet po bid (2 times a day) as needed: you should take it while on oxycodone to prevent constipation. disp:*60 tablet(s)* refills:*0* 2. citalopram 20 mg tablet sig: one (1) tablet po daily (daily). 3. oxycodone 5 mg tablet sig: one (1) tablet po every 4-6 hours as needed for headache. disp:*60 tablet(s)* refills:*0* 4. nimodipine 30 mg capsule sig: two (2) capsule po q4h (every 4 hours) for 15 days: please take every 4 hours through . disp:*120 capsule(s)* refills:*0* 5. bisacodyl 5 mg tablet, delayed release (e.c.) sig: two (2) tablet, delayed release (e.c.) po daily (daily) as needed. disp:*30 tablet, delayed release (e.c.)(s)* refills:*0* 6. atenolol 25 mg tablet sig: 0.5 tablet po daily (daily). disp:*50 tablet(s)* refills:*2* 7. acyclovir 200 mg capsule sig: two (2) capsule po tid (3 times a day) for 2 days. disp:*12 capsule(s)* refills:*0* 8. camphor-menthol 0.5-0.5 % lotion sig: one (1) appl topical qid (4 times a day) as needed. 9. diphenhydramine hcl 25 mg capsule sig: one (1) capsule po q6h (every 6 hours) as needed. disp:*60 capsule(s)* refills:*0* discharge disposition: home discharge diagnosis: sah discharge condition: good discharge instructions: you were admitted with a very severe headache. your brain imaging studies revealed the presence of subarachnoid hemorrhage. in an exam called angiogram, no aneurysms were found that would explain this hemorrhage and, therefore, no neurosurgical intervention was made necessary. your headache was better controlled with pain medications. contact your physician in case you have new symptoms or if your headache deteriorates or if the rash you have does not improve or start involving the face. followup instructions: neurology: , md phone: date/time: 4:00 neurosurgery: , md phone: date/time: 1:30 procedure: arteriography of cerebral arteries diagnoses: subarachnoid hemorrhage dermatitis due to drugs and medicines taken internally disorders of bilirubin excretion hydantoin derivatives causing adverse effects in therapeutic use essential and other specified forms of tremor acute lymphoid leukemia, in remission 

Answer: The patient is high likely exposed to