Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: sulfamethoxazole / quinolones attending: chief complaint: altered mental status major surgical or invasive procedure: none history of present illness: the patient is a 79 year old woman with a history of cerebral amyloid angiopathy s/p multiple bleeds who now presents with another intraparenchymal hemorrhage. thursday afternoon (six days prior to admission), the husband noted that she was "less focused" and more lethargic. she also had frequent non-specific head turning either side. she was still able to eat breakfast without significant difficulty. at baseline, she is aphasic and needs assistance with all adls. when watching tv, she occassionally moans or laughs. the husband her outpatient doctor who recommended increasing her neurontin. she then presented to an osh ed within 1-2 days after the onset of symptoms, where she had a cxr and urine studies (results unknown at this time) and was then discharged to home. she returned to the ed today for repeat evaluation due to persistence of symptoms. she had a head ct which showed a new right temporal bleed. she was transferred to for further evaluation/management. past medical history: - multiple intraparenchymal hemorrhages due to amyloid angiopathy. the first hemorrhage was in (presented with r hemiparesis). later had a large l fronto-parietal bleed (became aphasic). - focal motor facial seizures. previously treated with dilantin, now on neurontin. - myoclonic jerks - high cholesterol - hypertension social history: lives at home with her husband. also has a home health aide. they take 24 hour care of her. she is unable to do any of her adls. she is fairly nonresponsive at baseline, but occ says words or laughs at the tv according to her family. no tobacco, etoh, or illicit drug use. family history: h/o cad and stroke in the family physical exam: physical exam on admission: t 98.6 bp 172/107 hr 68 rr 14 general - eyes open, nad. lungs - ctab, good air movement cv - rrr, no m/r/g abdomen - non-tender, non-distended, bowel sounds present ext - warm, no edema neurologic examination: awake, alert, looking around with eyes open, not talking, does not follow simple commands, leftward gaze preference; blinks to threat b/l; perrl 2 to 1 mm, face symmetric; decreased bulk throughout, markedly increased spastic tone in all extremities, unable to formally test power all limbs in a flexed posture; reflexes 1+ in legs and 2 in arms (slightly brisker on right); toes upgoing b/l pertinent results: admit labs: 06:00pm blood wbc-5.9 rbc-4.62 hgb-14.4 hct-41.7 mcv-90 mch-31.1 mchc-34.5 rdw-13.7 plt ct-204 06:00pm blood pt-16.4* ptt-35.8* inr(pt)-1.9 06:00pm blood glucose-91 urean-9 creat-0.5 na-134 k-4.1 cl-97 hco3-27 angap-14 12:45am blood tsh-1.8 03:30am urine color-straw appear-clear sp -1.008 03:30am urine blood-mod nitrite-neg protein-tr glucose-neg ketone-neg bilirub-neg urobiln-neg ph-8.0 leuks-mod 03:30am urine rbc-* wbc-21-50* bacteri-many yeast-none epi- ---- discharge labs: 06:50am blood glucose-87 urean-8 creat-0.5 na-139 k-4.0 cl-103 hco3-25 angap-15 12:45am blood alt-56* ast-36 ck(cpk)-97 alkphos-139* amylase-73 totbili-0.8 ---- cxr:very large hiatal hernia. right lower lobe opacity, which may reflect atelectasis, aspiration, or early pneumonia. dedicated pa and lateral chest radiograph is suggested for more complete assessment. ---- head ct:right temporal hematoma. this currently measures 29 x 42 mm in cross-sectional dimension. ---- eeg:abnormal portable eeg due to the slow and disorganized background and bursts of generalized slowing as well as a background asymmetry with higher voltage slowing on the left and a lower voltage background on the right. the first two abnormalities signify a widespread encephalopathic condition affecting both cortical and subcortical structures. medications, metabolic disturbances, and infection are among the most common causes. the focal slowing on the left indicates a subcortical dysfunction there while a lower voltage background on the right suggests more widespread cortical dysfunction or material interposed between the cortex and recording electrodes. no epileptiform features were seen. brief hospital course: 79 year old woman with amyloid angiopathy, prior cerebral hemorrhages, and now new ~12 cc intraparenchymal hemorrhage presented with mental status change. the new bleed is most likely related to her amyloid disorder. she was initially admitted to the icu for blood pressure mangagement. this proved to not be an issue, and her bp was well controlled without medication throughout(was high briefly initially). she was initially more lethargic than her baseline, but quickly returned to her home level of functioning after transfer from the icu. we were concerned she may have other pathologies contributing to her symptoms given the fact that the bleed was fairly small and that it had occured 7 days prior to presentaion. an eeg was performed to rule out seizure, and although very abnormal given her longstanding pathology, it did not show evidence of seizure activity. she then had a cxr which showed questionable opacity, but this could not be well discerned given portable study and her large hiatal hernia. this may have been atelectasis. she did have a low grade temperature while here, so a ua was sent which was grossly positive. she has an indwelling foley, so the results will be clouded. she had the foley changed and given her temperature and lack of obvious other infection(pna was felt unlikely due to lack od symptoms), she was treated for 3 days with cefpodoxime. she was initially npo and had a video swallow evaluation which cleared her for a limited diet. she will obtain a formal outpatient swallow study after discharge and recovery which will hopefully clear her for a full diet. her inr was initially elevated to 1.9 on admission. she was given ffp to normalize this. however, she developed a reaction(rash) to this and it was stopped. she received vitamin k as well as this was felt to be due to a nutritional problem. inr did normalize, but stayed sligtly elevated at 1.2-1.3. dic labs were not significant(high d-dimer is non-specific). finally, her neurontin was initially held and she had an increase in the amount of myocvlonic jerking she experienced. with the reintroduction of her neurontin, this returned to baseline. she recovered well here and was back to her home baseline by discharge. she will have home services after discharge. there is no further intervention for her amyloid angiopathy. at discharge, she was alert, but non-verbal and only very occasionally addressing her environment. she is spastic in all of her limbs at her baseline. she has a significant left gaze preference and head deviation. she will follow up with dr at her scheduled appointment. medications on admission: neurontin 600mg tid discharge medications: 1. gabapentin 300 mg capsule sig: two (2) capsule po tid (3 times a day). 2. cefpodoxime 200 mg tablet sig: one (1) tablet po q12h (every 12 hours) for 3 doses. disp:*3 tablet(s)* refills:*0* 3. formal swallow study please obtain formal outpatient swallow evaluation after discharge. discharge disposition: home with service facility: , discharge diagnosis: intracranial hemorrhage uti -- myoclonic jerking discharge condition: improved to baseline functioning. afebrile. discharge instructions: please call your pcp or return to the ed if you have any change in behavior, grogginess, chest pain, shortness of breath, or if you pass out. followup instructions: provider: , m.d. phone: date/time: 4:00 provider: , m.d. phone: date/time: 1:00 procedure: transfusion of other serum diagnoses: pure hypercholesterolemia urinary tract infection, site not specified unspecified essential hypertension other convulsions intracerebral hemorrhage pressure ulcer, buttock myoclonus 

Answer: The patient is high likely exposed to