Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: acute paralysis major surgical or invasive procedure: none history of present illness: 75yo korean gentleman awoke this morning, talked to the bathroom and felt sudden onset back and abdominal pain after which he lost functioning of bilateral lower extremeties. taken to osh where abdominal ct scan thought to show dissection of thoracic aaa, pt xferred to for possible surgical intervention but on review of outside ct, no aneurismal rupture noted. past medical history: gerd htn social history: previously heavy smoker, quit 1.5 yrs ago. no alcohol family history: non contributary physical exam: vs: afeb 130/60 72 general: wnwd, nad heent: anicteric, mmm without lesions, op clear neck: supple, no lad, no carotid bruits, no thyromegaly cv: rrr s1s2 no m/r/g resp: ctab no r/w/r abd: soft/distended ext: no c/c/e, distal pulses intact skin: no rashes, petechiae ms: a&o x 3, interactive, appropriate, following all commands speech fluent w/o paraphasic errors, +naming of wholes & parts, +repetition, +comprehension no evidence of neglect with visual or tactile stimulation no apraxia: able to comb hair, screw in light bulb cn: i - not tested, ii,iii - perrl, vff by confrontation, optic discs sharp, visual acuity od, os; iii,iv,vi - eomi, no ptosis, no nystagmus; v- sensation intact to lt/pp, responds to nasal tickle, masseters strong symmetrically; vii - no facial weakness/asymmetry; viii - hears finger rub b; ix,x - voice normal, palate elevates symmetrically, gag intact; - scm/trapezii b; xii - tongue protrudes midline, no atrophy or fasciculations motor: nl bulk and tone, no tremor, rigidity or bradykinesia. no pronatordrift. no asterixis. deltd bicep tricp ecr/u exdig fldig dorsi opppb c5 c5-6 c7 c6-7 c7 c8 t1 c8-t1 l 5 5 5 5 5 5 5 5 r 5 5 5 5 5 5 5 5 ilpso qufem hamst tiban l1-2 l3-4 l5-s2 l4-5 s1-2 l5 dtrs: (c56) br (c6) tri (c7) pa (l34) ac (s12) plantar l 2 2 2 0 0 mute r 2 2 2 0 0 mute sensory: lt intact throughout; temperature, vibration, pin decreased from t10 level down. coord: fnf intact. gait: unable to perform. pertinent results: wbc-7.4 rbc-4.79 hgb-15.2 hct-44.7 mcv-93 mch-31.7 mchc-34.0 rdw-13.7 plt ct-103* pt-14.1* ptt-26.2 inr(pt)-1.2* glucose-139* urean-46* creat-1.1 na-139 k-4.3 cl-107 hco3-24 angap-12 alt-62* ast-20 ck(cpk)-95 alkphos-62 totbili-2.1* calcium-8.1* phos-3.2 mg-2.3 urine color-yellow appear-clear sp -1.011 urine blood-tr nitrite-neg protein-neg glucose-neg ketone-neg bilirub-neg urobiln-1 ph-5.0 leuks-neg urine rbc-* wbc-0-2 bacteri-few yeast-none epi-0-2 urine hours-random creat-43 na-less than totprot-13 prot/cr-0.3* 11:13 am renal u.s. port technique: portable renal ultrasound with doppler studies. findings: the right kidney measures 10.9 cm in length, the left 11.6 cm. within the upper pole of the right kidney, two simple cysts, each measuring 2.7 cm in diameter, are visualized, as well as a 2.1 cm simple cyst in the lower pole of the left kidney. these correspond to a hypoattenuating foci seen on the recent ct. in the lateral mid pole of the right kidney, there is a region of cortical echogenicity, which likely corresponds to an area of relative perfusion defect on the recent ct. the appearance may represent a small evolving renal infarct. doppler studies show normal flow in the right main renal artery and vein, as well as normal arterial flow in interlobar arteries among the upper, middle, and lower poles. the resistive indices range from 0.63-0.70 on the right. on the left, the main renal artery and vein are also patent, but interlobar arteries show slight parvus et tardus waveforms, particularly when compared to the opposite side. the resistive indices among the interlobar arteries range from 0.63-0.86. in the setting of portable technique, the doppler studies of the left kidney are somewhat suboptimal, but the findings suggest that there is likely somewhat decreased perfusion to the left kidney compared to the right. impression: 1. small echogenic region involving the cortex in the right mid pole, which correlates with a region of relative decreased perfusion on the recent ct. this appearance may represent an evolving infarct within a portion of the right mid pole. 2. patency of flow to both kidneys. however, doppler studies are suggestive of somewhat decreased perfusion to the left compared to the right 2:01 pm chest (portable ap) single portable chest radiograph demonstrates no interval change in the cardiomediastinal silhouette. there is increased perihilar opacity involving the bilateral hila and mild diffuse increased airspace opacity representing mild-to-moderate pulmonary edema. there is blunting of the left costophrenic angle representing a small effusion. the right costophrenic angle is sharp. the trachea remains in the midline. cardiomegaly is unchanged. impression: cardiomegaly, unchanged. worsening chf. brief hospital course: pt admitted stat lumbar drain placed - to decrease csf pressure less then 10 / pt transfered to the sicu a-line placed stroke service consulted / ct - reveals aortic dissection no acute compression or infarct noted / the diseection and low bp is thought to be responsible by decreasing the blood flow to the spinal cord. it is noticed if pts bp elevated, paralysis improves pt bp is kept elevated ( pt also has arf on admission ) / the increase bp is probably due to decreasse blood flow to the kidneys. / steroids started for ? acute cord infarction. lumber drain stops working / replaced troponin is increasing / pt started on beta blockers. the increase troponin is thought to be due to hypoperfusion syndrome. echo done increase creat / bun - renal consulted stable diovan added for bp control creat improves pt symptoms gradually improve with bp control pt consult / fails voiding trial foley replaced pt stable for dc medications on admission: protonix bp med discharge medications: 1. albuterol sulfate 0.083 % solution sig: one (1) inhalation q6h (every 6 hours) as needed. 2. acetaminophen 325 mg tablet sig: one (1) tablet po q4-6h (every 4 to 6 hours) as needed. 3. pantoprazole 40 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po q24h (every 24 hours). 4. bisacodyl 10 mg suppository sig: one (1) suppository rectal hs (at bedtime) as needed. 5. valsartan 80 mg tablet sig: one (1) tablet po daily (daily). 6. tamsulosin 0.4 mg capsule, sust. release 24hr sig: one (1) capsule, sust. release 24hr po hs (at bedtime). 7. hydralazine 20 mg/ml solution sig: one (1) injection q6h (every 6 hours) as needed for sbp>160: prn. 8. amlodipine 5 mg tablet sig: two (2) tablet po daily (daily). 9. labetalol 200 mg tablet sig: one (1) tablet po bid (2 times a day). 10. heparin (porcine) 5,000 unit/ml solution sig: one (1) injection (2 times a day). discharge disposition: extended care facility: - discharge diagnosis: paralysis with decrease bp aa dissection arf discharge condition: stable discharge instructions: bp control 140-180 moniter bun creat followup instructions: please follow-up with neurology (dr. provider: . phone: date/time: 3:30 **this appointment is on the of the building. you will need to call ahead of time to update your registration. please call . thank you. please call dr office at procedure: arterial catheterization other exploration and decompression of spinal canal diagnoses: esophageal reflux unspecified essential hypertension acute kidney failure, unspecified dissection of aorta, thoracoabdominal paraplegia vascular myelopathies 

Answer: The patient is high likely exposed to