Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: "heart racing" major surgical or invasive procedure: electrophysiology study, icd placement history of present illness: 19yo man with pmh sig for v tach in , worked up at osh and told due to "bulge in heart," had ep study at time but unaware of results or ablation, briefly on bb but d/c'd , presented to complaining of dizziness and heart racing that came on while playing basketball. pt denies chest pain, sob. no family hx of suddent cardiac death. episode possibly viral syndrome related per pt. . in ed ekg showed v tach, as bp dropped to 75/50 pt was sedated and cardioverted to sr with 200j. past medical history: ventricular tachycardia social history: male student, sexually active with girlfriend. + marijuana ppd tobacco + etoh family history: father mi @ 50 years age brother with muscular dystrophy physical exam: pe: 96.9 hr 120s 127/60 16 97ra nad no carotid bruits rrr nl s1s2 no mrg lungs clear abd soft nt nd nabs groin with 2+ pulses, no bruits dp 2+ no edema pertinent results: max ck 365, max trop 1.09 - these were shortly after dc cardioversion . tsh 1.0 . 10:23pm wbc-16.0* rbc-4.62 hgb-15.2 hct-44.0 mcv-95 mch-32.9* mchc-34.6 rdw-13.0 10:23pm glucose-100 urea n-14 creat-1.2 sodium-141 potassium-4.6 chloride-106 total co2-24 anion gap-16 10:23pm calcium-9.3 phosphate-1.6* magnesium-1.9 . ekg on admission: regular wide complex rhythm at 252 beats per minute. left superior qrs axis with right bundle-branch block configuration most likely representing antidromic atrio-ventricular reciprocating tachycardia. although, cannot exclude ventricular tachycardia. . cardiac mr: structure and function- the myocardium had two focal areas of mid myocardial fibrofatty infiltration in the mid lateral left ventricular wall. the more proximal area was larger. there was normal epicardial fat distribution. the pericardium appeared normal in thickness. there was a trace pericardial effusion. there were no pleural effusions. the origins of the left main and right coronary arteries were identified in their customary positions. the diameters of the ascending, arch, and descending thoracic aorta were normal. the main pulmonary artery diameter was also normal. the left atrial ap dimension was normal. the right and left atrial lengths in the 4-chamber view were normal. the coronary sinus diameter was normal. the left ventricular end-diastolic dimension index was normal. the end- diastolic volume index was mildly increased. the calculated left ventricular ejection fraction was normal at 70% with normal regional systolic function. the left ventricular mass index was normal. the right ventricular end- diastolic volume index was mildly increased. the calculated right ventricular ejection fraction was mildly reduced at 41%, with mild free wall hypokinesis. there was no focal thinning or fatty infiltration seen in the rv free wall. there were no aneurysms seen in the rv free wall or right ventricular outflow tract. the aortic valve was trileaflet with normal valve area. quantitative flow- there was no significant intra-cardiac shunt. aortic flow demonstrated no significant aortic regurgitation. the calculated mitral valve regurgitant fraction was consistent with mild mitral regurgitation. the resultant effective forward lvef was borderline reduced at 59%. the right ventricular stroke volume and pulmonic flow demonstrated no significant pulmonic or tricuspid regurgitation. viability- there were two focal areas of scarring in the mid lateral ventricular wall which corresponded to the areas of fatty infiltration described above. coronary mr imaging- normal origins and orientations of the left main and right coronary arteries. the left main coronary artery had normal signal characteristics and caliber with bifurcation into the lad and lcx. the lad and lcx had normal caliber and signal characteristics. the rca also had normal caliber and signal characteristics. impression: 1. focal fibro-fatty infiltration of the left ventricle in the mid lateral wall. 2. mildly increased left ventricular cavity size with normal regional left ventricular systolic function. the lvef was normal at 70%. the effective forward lvef was borderline reduced at 59%. 3. mildly increased right ventricular cavity size with mild global hypokinesis. the rvef was mildly reduced at 41%. no mr evidence of right ventricular fatty infiltration. 4. mild mitral regurgitation. 5. the diameters of the ascending aorta, arch, and descending thoracic aorta were normal. the main pulmonary artery diameter was normal. 6. normal coronary artery origins with no evidence of anomalous coronary arteries, and normal signal characteristics of all visualized vessel segments. . echo: 1. left ventricular wall thickness, cavity size, and systolic function are normal (lvef>55%). regional left ventricular wall motion is normal. 2. the right ventricular cavity is mildly dilated. there is moderate global right ventricular free wall hypokinesis. right ventricular systolic function appears depressed. 3. there is a small pericardial effusion with significant fibrin deposits on the surface of the heart. the pericardium appears thickened. . ep study with ventriculat tachycardia ablation: after informed consent was obtained, the patient was brought to the electrophysiology laboratory in the fasting state, and was prepped and draped in the usual sterile fashion. both groins were anesthetized with lidocaine and sheaths and catheters were placed as reported above. heparin 5000 units iv was given and an iv heparin drip was started to maintain act>250 s throughout the case. the results were as follows: signal averaged ecg performed demonstrating: std qrs 76 ms; filtered qrs 130 ms; las 57 ms rms 2 mcv (positive by all 3 criteria) 1. the baseline rhythm was sinus with scl 1050ms, hv 42, and ah 77 ms. 420 ms . no va conduction was present at pcl of 600 ms. 2. double ves were delivered from the rvot, inducing multiple morphologies of vt (up to 7) falling into 4 major categories as following: vt # induction cl morphology axis termination hemodynamics 1 90 atyplbbb ls burst from rvot tolerated 2 15 lbbb ls catheter pvc tolerated 3 550/250/210 220 lbbb ls burst rvot tolerated 4 burstpace #3 265 lbbb inferior burst rvot tolerated progressive fusion was documented during tachycardia #1 proving re-entry as the mechanism for #1. 3. biosense carto electroanatomic mapping of the left ventricle performed, demonstrating normal lv voltage and lv volume of 135 cc. no adequate pace-maps were obtained by pacing from the lv. 4. biosense carto electroanatomic mapping of the right ventricle performed during sinus rhythm, demonstrating moderate size area of decreased voltage, including areas of dense scar, late potentials and widely split potentials in the basal subvalvular inferior area of the rv (rv volume of 98 cc). 5. good pacemaps were obtained for tachycardia #1 at the subvalvular area of the scar. a total of 24 rf ablations using a 4 mm biosense catheter were performed in the area of scar during sinus rhythm as well as vt, guided by late and diastolic potentials. two of the ablations during vt #4 terminated vt during ablation. 6. following ablations, the ventricular stimulation protocol was repeated. tight triple ves (400/230/210/200) induced vt #5 (similar morphology to vt #4 with shorter cl - 210 ms) which was poorly tolerated and terminated by burst pacing. conscious sedation and appropriate monitoring was provided by the ep nursing staff. the patient tolerated the procedure well and left the laboratory in stable condition. after the act was less than 180, the sheaths were pulled from the groins. ablation- a total of 24 rfa using a 4 mm biosense catheter were performed in the area of scar in the inferobasal part of the right ventricle during sinus rhythm and vt for a total duration of 1527 seconds and target temperature of 60 degrees, guided by substrate map, pace-map, presence of diastolic potentials. stable catheter position, good impedance drop and power were observed during most of the burns. vt #4 was terminated twice during the burn. conclusions- 1. area of decreased voltage, including areas of dense scar in the rv inferobasal wall compatible with arvc; normal lv voltage. 2. multiple morphologies of hemodynamically tolerated vts, induced by double ves. 3. substrate- and pacemap-guided ablation of vt, with partial success (inducible vt at the end of the ablation using aggressive stimulation protocol). . icd placement: local anesthetic was delivered to the left pectoral region, and an incision was made in the left deltopectoral groove. the incision was extended inward by blunt dissection and the cephalic vein was identified. the vein was cannulated and a 9f peel away sheath was placed in the vein over a retaining wire. a pacing/defib lead #6949-65 was advanced in rv under fluoroscopic guidance. multiple attempts were made to obtain the best parameters, but sensing was poor in most typical locations. finally, a location in the mid septum was obtained with good parameters after multiple interrogations over >30 minute period. the lead was sutured to the underlying pectoralis muscle using 2-0 ethibond sutures. final sensing and pacing thresholds were checked and were good. a subcutaneous pocket was created using blunt dissection, and was copiously irrigated with a saline solution containing gentamicin and vancomycin. the lead was connected to a vr icd, and the entire system was implanted into the pocket. propofol was then administered by a member of the anesthesia staff. ventricular fibrillation was induced with a t wave shock, and was successfully recognized and treated with an 18j internal shock from the icd. the subcutaneous tissues were then closed with a layer of interrupted 2-0 vicryl suture and a running 2-0 vicryl stitch. the skin was closed with a running 4-0 vicryl subcuticular stitch. dermabond was then applied to the incision. the wound was covered with a dry sterile dressing. the patient tolerated the procedure well and left the laboratory in good condition. conscious sedation was provided and appropriate monitoring performed by members of the ep nursing staff. pacing parameters: vvi at 40bpm icd therapies were programmed as follows: vf (<=240ms): 1. shock 20j x 1 2. shock 35j x 5 fvt (<=300ms): 1. burst 8 pulses at 69% x 2 sequences 2. cv 20j x 1 3. cv 35j x 4 vt (<=340ms): 1. burst 10 pulses at 72% x 3 sequences 2. burst 10 pulses at 84% x 3 sequences summary 1. successful implantation of a vr icd technical data summary pulse generator information manufacturer model name model # serial # location vr 7232cx prn105547h single chamber left pectoral lead information chamber date implanted manufacturer model # fixation serial # 6949 active lfj053132v threshold measurements chamber p/r stimulation threshold impedance amplitude at 0.5 ms rv lead 18.1 mv 0.9 v 819 ohms defibrillation testing episode # induction induced shock shock configuration conversion impedance method rhythm energy waveform 1 t-wave shock vf 18 biphasic ax > b yes 41 defibrillation threshold: 18 j brief hospital course: 1)ventricular tachycardia: *please see full electrophysiology reports in "pertinent results" section of discharge summary for full details.* pt underwent ep mapping, ventricular tachycardia ablation on and returned to the ep lab for a vr icd placed . findings by echo and cardiac mr could be consistent with arvd. in further work-up of abnormal cardiac findings, ace level sent to evaluate for sarcoid. medications on admission: none discharge medications: 1. oxycodone-acetaminophen 5-325 mg tablet sig: 1-2 tablets po every 6-8 hours as needed for 4 days. disp:*14 tablet(s)* refills:*0* 2. keflex 500 mg capsule sig: one (1) capsule po four times a day for 2 days. disp:*8 capsule(s)* refills:*0* discharge disposition: home discharge diagnosis: ventricular tachycardia discharge condition: stable discharge instructions: please return to the ed if you develop chest pain, shortness of breath, palpitations. followup instructions: follow up with dr. with in one month . . provider: clinic where: cardiac services phone: date/time: 1:00 procedure: other electric countershock of heart catheter based invasive electrophysiologic testing excision or destruction of other lesion or tissue of heart, endovascular approach implantation or replacement of automatic cardioverter/defibrillator, total system [aicd] cardiac mapping diagnoses: paroxysmal ventricular tachycardia 

Answer: The patient is high likely exposed to