Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: shortness of breath major surgical or invasive procedure: intubation and mechanical ventilation central line placement history of present illness: hpi: 21 previously healthy male presents dyspnea. he developed a mild frontal headache and mild nonproductive cough at noon on the day prior to admission. at about 9 pm he developed worsening ha and severe, "hacking" nonproductive cough. the cough continued today, and he found he was too short of breath to walk to his dorm after work. he felt "terrible" overall, so presented to the ed for evaluation. . no recent travel, +sick contact: roommate recently ill with "bronchitis" x ~2-3 weeks, did take antibiotics. no hiv risk factors (see sh, below) ros: +anorexia/poor po intake x 1 day. no n/v/d. no stiff neck/meningeal signs. no rash. . ed course: initial vitals: t101.6, hr 120s, bp 130/70s satting 88% ra, then 94% 6l, and 99% nrb. wbc 19.7; cxr with b/l reticular opacities as well as focal consolidation in the lingula and left upper lobe. given ceftriaxone, azithromycin, 1l ivf. past medical history: none social history: lives with: roommate. student at college (biomedical electrical engineering); employed at . tobacco: none etoh: drinks/ 1 night per week drugs: denies ivdu ever, no illicits 2 prior sexual partners, both female, used condoms. family history: father: alive and well mother: obesity physical exam: vs 98.3 100/72 110 18 97% ra gen: comfortable, nad, resting in bed heent: nc, at, mmm pulm; ctab, air movement significantly improved cv: tachy, rrr, no m/r/g abd: +bs, soft, nt/nd ext: no c/c/e pertinent results: admission labs: 01:50pm wbc-19.7* rbc-5.75 hgb-17.6 hct-49.4 mcv-86 mch-30.7 mchc-35.7* rdw-12.9 01:50pm neuts-92.2* bands-0 lymphs-2.7* monos-3.0 eos-1.7 basos-0.3 01:50pm plt count-224 01:50pm glucose-121* urea n-15 creat-1.3* sodium-141 potassium-3.7 chloride-103 total co2-23 anion gap-19 02:28pm lactate-1.5 . studies: chest (pa & lat) impression: diffusely increased reticular opacities bilaterally with more focal region of consolidation noted within the lingula and peripheral left upper lobe. findings may relate to a bacterial or atypical pneumonia (ie. mycoplasma or viral, including cmv or even varicella); etiologies such as pneumocystis jiroveci should be considered, if patient is immunocompromised. . tte (complete) done the left atrium is normal in size. left ventricular wall thicknesses are normal. the left ventricular cavity size is normal. due to suboptimal technical quality, a focal wall motion abnormality cannot be fully excluded. overall left ventricular systolic function is normal (lvef>55%). tissue doppler imaging suggests a normal left ventricular filling pressure (pcwp<12mmhg). right ventricular chamber size and free wall motion are normal. the aortic valve leaflets (3) appear structurally normal with good leaflet excursion and no aortic regurgitation. the mitral valve appears structurally normal with trivial mitral regurgitation. the estimated pulmonary artery systolic pressure is normal. there is a very small pericardial effusion. there are no echocardiographic signs of tamponade. . had 18 chest x-rays in total, put on disk for outpatient pulmonolgist. last cxr - - near resolution of left lower lobe opacity. improving small pleural effusions. brief hospital course: 21m presents with sob, found to have ards likely secondary to acute eosinophilic pneumonia. . # respiratory distress/ards/acute eosinophila pneumonia: pt met criteria for ards and was initially intubated for concern of respiratory fatigue. this was initially thought to be community acquired pneumonia, and he was started on azithro/vanc/zosyn for 7 day course. given the results of the bal (negative culture and 25% eosinophils), pt was felt to have acute eosinophilic pneumonia and was started on steroids. he had an extensive id workup inc. influenza (neg), resp. viral antigen panel (neg), legionella (neg), chlamydia ab (neg), mycoplasma (pending), hiv ab (neg), hiv vl (neg), bal for pcp (neg), galactomannan (neg), histo ab (neg), b-glucan (neg), blasto ab (neg), coccidio ab (neg), and hantavirus ab (neg). he was also initially worked up for dah: (neg), anca (neg), anti-gbm (neg). he was maintained on ards net protocol in regards to ventilation. during ventilation, pt had acute episodes of agitation 2-3x/day, requiring high doses of sedation. after his bal results came back with 25% eosinophils, he was started on solumedrol. after one week of intubation his oxygen requirements decreased and radiographic improvement was seen on his cxr. his ventilation setting were decreased to pressure support and after a breathing trial was successful he was extubated without event. he remained on steroids, and was tapered to 40mg prednisone q daily at time of discharge with no supplemental oxygen requirement. . #. septic shock ?community acquired pneumonia: it is unclear if pt was actually septic. he became hypotensive after intubation and receiving propofol and was hypotensive in the setting of high peep, requiring levophed for 24 hrs. cvp had been slightly low, and pt was likely intravascularly volume depleted, requiring ivf boluses. overall, he had good urine output and then became hypertensive off pressors. . # pancreatitis-while receiving intitial high dose steroids he had abdominal pain, lfts as well as amylase and lipase were checked. his amylase and lipase were elevated, and his pancreatitis was felt to be secondary to steroids. his tube feeds were held and his symptoms as well as his amylase and lipase improved. . # elevated lfts-after extubation, his abdominal pain prompted evaluation of his lfts, which were found to be elevated. a ruq u/s was done to evaluate for acalculous cholecystitis, which was negative for stones or evidence of infection. his lfts were monitored and trended down without intervention. . # hallucinosis- the patient developed hallucinations after extubation, a few days after receiving steroids. also, this was in the context of cessation of large dose midazolam, which he had been receiving while intubated. the etiology was felt to be steroid induced vs benzodiazepine withdrawal. however, over the next few days his symptoms seemed to be more consistent with steroid induced hallucinosis and his symptoms were controlled with haldol prn. he did not require more than 2 doses of haldol. his symptoms had completely resolved prior to discharge . # tachycardia/atrial fibrillation with rvr: pt went into rapid a. fib in the evening of ; he was otherwise hemodynamically stable. ekg had no evidence of ischemia. this was likely secondary to pulmonary disease or increased cathecholamines. he was started on a diltiazem gtt with good response and converted to sinus in am of . echo was obtained and was normal. pt was noted to be tachyacrdia throughout his hospitalization. his intitial presentation with tachycardia was likely due to his infection. with the exception of the episode of afib, the patient was in sinus rhythym. his symptoms were triggered by exertion. he was completely asymptomatic denying shortness of breath or palpitations. at time of discharge, his resting heart rate was in the 80s but would increase with physicial exertion. given his negative echo and prolonged hospital course with intubation and mechanical ventilation it is believed that there is an element of deconditionning to his tachycardia. . #. acute renal failure: slightly elevated creatinine on admission was likely prerenal as pt had poor po intake x 1 day and insensible losses with fever. fena of 0.6%. improved with ivfs. . #. hyperglyemia-pt with fs in 200s likely steroids, was covered with strict insuling sliding scale. . #. code: full medications on admission: none discharge medications: 1. omeprazole 20 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po once a day. disp:*30 tablet, delayed release (e.c.)(s)* refills:*0* 2. bactrim 80-400 mg tablet sig: one (1) tablet po once a day. disp:*30 tablet(s)* refills:*0* 3. prednisone 20 mg tablet sig: two (2) tablet po daily (daily). disp:*60 tablet(s)* refills:*0* discharge disposition: home discharge diagnosis: acute respiratory distress syndrome acute eosinophilic pneumonia tachycardia acute pancreatitis, likely steroid-induced discharge condition: improved discharge instructions: you were admitted to the hospital for pneumonia and subsequently developed acute respiratory distress syndrome requiring you to be intubated. your pneumonia was treated with antibiotics and steroids. you will need to continue to take these steroids as directed until you see your pulmonologist in follow up who will prescribed a slow taper of this medication. you were also started on a medication called bactrim that you will need to take while you take the steroids to prevent an additional infection. you have also been precribed a medication, omeprazole, that will help with acid reflux and indigestion while you are on the steroid as well. please take all medications as prescribed and please follow up with your pulmonologist in as scheduled below. . should you experience any increased shortness of breath, heart palpitations, worsening cough or any other symptoms that are new or of concern to you, please contact your primary care physician immediately or return to the emergency department. followup instructions: please follow up with dr. in pulmonology at hospital on thursday, at 9:20 am. please take your cd with your chest x-rays with you to this appointment. if there is a problem with this appointment, please contact their office at (. you should also receive information regarding this appointment in the mail. md, procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified insertion of endotracheal tube closed [endoscopic] biopsy of bronchus diagnoses: anemia, unspecified other specified disorders of pancreatic internal secretion adrenal cortical steroids causing adverse effects in therapeutic use acute kidney failure, unspecified unspecified septicemia severe sepsis acute respiratory failure septic shock hypoxemia accidents occurring in residential institution tachycardia, unspecified pulmonary eosinophilia acute pancreatitis 

Answer: The patient is high likely exposed to