Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: intubated - seizure major surgical or invasive procedure: intubation history of present illness: patient is a 30 yo m who was at work earlier today, reportedly witnessed to bend down, then have a tonic clonic seizure. ems witnessed two seizures per report in transport, observed r eye deviation. patient was take to and intubated upon arrival. per er report, prior to intubation the patient was withdrawing all 4 extremities to noxious stim. he was taken to ct where his head ct showed a punctate hemorrhage on the left posterior vertex. neurosurgery was called to evaluate. they recommended neuro c/s, eeg, dilantin loading. pt also had inr 2 and was given ffp. neuro consulted, recommended changing sedation to benzos, as thought this likely withdrawal related. labs notable for plt 50, inr 2, pos urine benzos. anion gap 17. abg after intubation was 7.36/32/453/19 on ac 100% fio2. patient given dilantin 250, banana bag, propofol, midaz, fentanyl and naloxone. ordered for 2 units ffp, did not get prior to transfer. on arrival to the micu, patient intubated and sedated, unable to provide further information. past medical history: etoh abuse, no h/o withdrawal seizures. tobacco use pancytopenia social history: + hx of etoh abuse - quit 2 months ago () - was hospitalized for detox and had not had etoh since. per pcp recs, patient originally from el . in us since . unmarried, limited english. family history: unknown physical exam: pe on admission to micu vitals: t: bp: p: r: 18 o2: general: alert, oriented, no acute distress heent: sclera anicteric, mmm, oropharynx clear, eomi, perrl neck: supple, jvp not elevated, no lad cv: regular rate and rhythm, normal s1 + s2, no murmurs, rubs, gallops lungs: clear to auscultation bilaterally, no wheezes, rales, ronchi abdomen: soft, non-tender, non-distended, bowel sounds present, no organomegaly gu: no foley ext: warm, well perfused, 2+ pulses, no clubbing, cyanosis or edema neuro: cnii-xii intact, 5/5 strength upper/lower extremities, grossly normal sensation, 2+ reflexes bilaterally, gait deferred, finger-to-nose intact pe on discharge: vs 98.7 90 120/68 18 100%ra gen: awake, alert, nad cv: rrr, no m/r/g resp: ctab abd: soft/nt/nd ext - wwp, no c/c/e pertinent results: labs on admission: 08:41am wbc-8.3 rbc-4.35* hgb-14.3 hct-42.6 mcv-98 mch-33.0* mchc-33.7 rdw-19.7* 08:41am plt count-50* 08:41am pt-21.2* ptt-39.9* inr(pt)-2.0* 08:41am fibrinoge-175* 08:41am urine bnzodzpn-pos barbitrt-neg opiates-neg cocaine-neg amphetmn-neg mthdone-neg 09:00am glucose-105 na+-141 k+-3.2* cl--111* tco2-13* 08:39pm plt count-31* labs on discharge: 08:05am blood wbc-2.0* rbc-3.27* hgb-10.7* hct-30.8* mcv-94 mch-32.6* mchc-34.6 rdw-19.4* plt ct-38* 08:25am blood pt-22.9* ptt-36.5 inr(pt)-2.2* 01:39pm blood fibrino-220 08:25am blood glucose-100 urean-7 creat-0.8 na-137 k-3.7 cl-105 hco3-26 angap-10 08:05am blood alt-20 ast-41* alkphos-54 totbili-3.1* 08:25am blood albumin-2.7* calcium-7.9* phos-2.6* mg-1.6 imaging: ct head: l posterior vertex small hemorrhage/contusion, no calvarial fracture, soft tissue swelling overlying r orbit, chronic nasal bone fx ct cspine: linear lucency through anterior aspect of c5 transverse foramen (102:164),likely congenital non-fusion and less likely fracture. no acute alignmentabnormality or prevertebral soft tissue abnormality (in setting of intubation). ct maxillo: soft tissue edema overlying right orbit. no underlying orbital fracture. chronic-appearing minimally displaced nasal bone fractures. eeg : impression: this is an abnormal continuous icu monitoring study because of low voltage and moderate diffuse background slowing with excess diffuse beta activity. these findings are indicative of moderate diffuse cerebral dysfunction which is etiologically non-specific. excessive beta activity is likely a medication effect. the focal attenuation of faster frequencies and intermittent focal slowing over the left hemisphere is less prominent on today's study than yesterday. this is indicative of focal cerebral dysfunction involving cortical and subcortical structures on the left. no epileptiform discharges or electrographic seizures are present. compared to the prior day's study, beta activity is less prominent, and the hemispheric asymmetry is also less prominent. ct head : impression: 1. slight interval increase in size of punctate hemorrhages in the left parietal lobe, without mass effect. 2. interval increase in fluid within the left mastoid air cells and left middle ear cavity, though no fracture is seen. note added in attending review: the three punctate hemorrhagic foci, located at the left frontoparietovertex -white matter junction may reflect diffuse axonal ("shear") injury, and should be correlated with the patient's neurologic status. if warranted on clinical grounds, and feasible, these might be further characterized by non-enhanced mr examination. ct head : impression: 1. no significant change in left parietal lobe intraparenchymal hemorrhages since . no new hemorrhage. if the etiology of these hemorrhages is unknown then an mr examination may be helpful. these may reflect small hemorrhagic infarctions, cortical venous thrombosis, or a neoplastic lesion. 2. global atrophy, more than expected for patient's age. pending: eeg brief hospital course: 30 yo m with hx of etoh abuse, found down at work, reported to have witnessed seizure activity and cerebral hemorrhage that required intubation. active issues: #acute respiratory failure-resolved: pt required intubated given altered mental status in the setting of seizures. pt extubated after one day on the vent, without issue. he was discharged off of supplemental oxygen and clinically stable from a respiratory perspective. pt was transferred to the floor where he remained stable through discharge. #cerebral hemorrhage: pt most likely hit his head during his seizing episodes. he was seen withdrawing extremities to noxious stimuli only. head ct showed 2 small left punctate hemorrhages in posterior vertex. neurosurgery was consulted and recommended no acute interventions. pt received vitamin k and ffp to normalize his elevated inr. also, received 1 unit plts in setting of thrombocytopenia. pt was only mildly responsive to ffp and vit k. it is thought that this is due to significant cirrhosis (see below). # witnessed seizures: pt had 2 witnessed seizures with right eye deviation that required intubation for airway protection for one day without complications. without history, it was assumed that pt was seizing due to etoh withdrawal. however, upon stabilization and transfer to the floor, pt reports that he has not had anything to drink for the last two months and was detoxed while hospitalized. pt also reports seizure one month prior to admission for which he went to outside hospital, was "scanned" and "they said i could leave." neurology re-consulted and recommended starting keppra 1g for possible new-onset seizure disorder. pt to follow-up with neuro as outpt. #c-spine trauma: pt reported neck pain s/p seizure and c-collar placement. during his micu stay, we were unable to safely clear his c-spine clinically. his c-spine ct did not show any acute bone pathology. upon transfer to the floor, c-spine was able to be cleared clinicially without any further studies. c-spine ct showed linear lucency through the anterior aspect of the right c5 transverse foramen, likely congenital nonfusion, less likely, fracture. no acute alignment abnormality or prevertebral soft tissue abnormality. this abnormality was discussed with neurosurgery who felt there was no acute issue to be addressed and recommmended follow-up in the clinic with dr. . #thrombocytopenia: most likely due to hypersplenism from etoh cirrhosis seen on imaging from osh. he received plts x1. etoh abuse-last admission for withdrawal in , h/o withdrawal seizures (questionable), has been going to aa (corroborated by family report to rn); essential hypertension not on meds; cirrhosis-had been on spironolactone, nadolol for varices, lasix as of discharge but not on med list from . #anemia: thought initially to be dilutional anemia and the setting of multiple ivf boluses. there was no clinical evidence of acute blood loss. trended his h/h daily, followed his hemodynamics and no transfusions were necessary. h/h remained stable through discharge. chronic diagnoses #cirrhosis: pt has a significant history of etoh abuse and ultrasonographic evidence of cirrhosis. he would benefit subsequent management for underlying liver disease (nadolol, lasix, spironolactone, etc.) with pcp and hepatology jointly managing (per pcp ). #h/o etoh abuse: continued encouragement of attendance at aa, family support. sw was consulted during admission who recommended same. ***********transitional issues**************** #outstanding issues: pending - eeg f/u with pcp who may consider referral to hepatology for cirrhosis. #family: contacts (step-father, speaking) ; (mother, spanish-speaking but is decision-maker if needed) ( medications on admission: folic acid 1 mg daily multivitamin thiamine omeprazole 20 mg daily discharge disposition: home discharge diagnosis: seizure presumed cirrhosis discharge condition: stable. discharge instructions: dear mr. , you were admitted after having had a seizure. when you came into the emergency room by ambulance, you required a breathing tub (you were intubated) and were monitored in the icu. they were soon able to remove the breathing tube and you were transferred to the floor. because you described another episode of seizure a month prior to this admission, the neurology service (doctors in the brain and seizures) was consulted. they recommended that you start a medication to help prevent seizures and also an additional scan of your brain to be done as an outpatient. additionally you were found to have a possible infection of your urinary tract. you were started on antibiotics to clear this infection. finally, your blood counts were found to be low. this is likely due to damage to your liver from your previous alcohol intake. please continue to abstain from alcohol. you primary care doctor might want you to see a hepatologist (a doctor s in the liver). new medications: start keppra 1g po bid start ampicillin 500mg po q6hrs x 4 days followup instructions: name: , location: community health center address: , , phone: when: thursday, :00 pm we are working on follow up appointments in the neurology department with both dr. and dr. . you will be called at home with appointments. if you have not heard or have questions, please call for dr. and dr. at . . procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube diagnoses: tobacco use disorder urinary tract infection, site not specified unspecified essential hypertension alcoholic cirrhosis of liver other and unspecified alcohol dependence, in remission acute respiratory failure epilepsy, unspecified, without mention of intractable epilepsy streptococcus infection in conditions classified elsewhere and of unspecified site, streptococcus, group d [enterococcus] cervicalgia fall resulting in striking against other object cerebellar or brain stem contusion without mention of open intracranial wound, with loss of consciousness of unspecified duration hypersplenism other pancytopenia 

Answer: The patient is high likely exposed to