Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: intraparenchymal hemorrhage major surgical or invasive procedure: intubation with mechanical ventilation eeg picc line placement history of present illness: (history based on accounts of patient's daughter, daughter-in-law, home caregiver, ems and osh records) ms. is an 85 year old woman with a history of atrial fibrillation (not on coumadin), hypertension, dvt's, and colon ca 7 years ago, who was found unresponsive lying on her right side on the day of presentation by a neighbor. she lives alone and has a neighbor who is her nurse-caregiver several mornings a week. ms. was last seen normal about 12 hours before she was found. her daughter-in-law reportedly called her the evening prior and there was no answer, and called her again in the morning, became concerned, and notified her neighbor, who called ems. ms. was found lying on her right side next to her recliner with her right arm and leg entangled in the chair. she had some edema overlying her right scalp and her right arm and leg were also edematous. she was not responsive, not talking or following commands, and not moving her right side as well as the left. she was brought to the hospital ed, where she arrived at 1:17 p.m. there her initial blood pressure was recorded as 67/45, hr 130, rr 24, t 100.8 axillary. blood glucose was 317. due to her somnolence she was intubated, for which she received lidocaine, rocuronium, etomidate, succinylcholine, ativan 4 mg and fentanyl 100 mcg at about 2 p.m. she was also given ns iv with improvement in her blood pressure. on exam there prior to intubation she was noted to have left gaze deviation and "withdrawal with all four extremities." labs were significant for a wbc of 18.5, inr of 1.32, and ua with 37 wbc. ck was elevated at 863, mb fraction was pending. she was then transferred to . upon arrival at 5 p.m. she was afebrile, with bp 220/110, hr 109, rr 20. she had received no sedation since 2 p.m., but was not responsive to voice, and did not move her right side as well as the left. neurosurgery was consulted and she received 1 gram dilantin, 50g mannitol, 10 mg decadron, and ultimately required a labetalol drip to control her blood pressures. she had a ct/cta of the head which revealed acute subarachnoid and intraparenchymal hemorrhage in the left frontal, parietal, and temporal lobes. there was evidence of edema with sulcal effacement on the left and 2 mm of midline shift. cta was not suspicious for a mass lesion. she received propofol 10 mg at 6 p.m. for the ct but has had no sedating medications since then, without any improvement in her mental status. when it was determined that neurosurgery would not be admitting her, the neurology service was consulted, 3 hours after the patient's arrival to the ed. ms. has not been feeling well for several weeks, sometimes not getting out of bed in the morning. her family are not aware of any specific recent illnesses, although her daughter thinks she may have had a mild heart attack two weeks ago. her daughter reports that she has been falling a lot lately, and hitting her head. it sounds like she loses consciousness with the falls, as she once said she woke up after falling down the stairs and didn't recall that she had fallen. review of systems: not definitively known per nurse or daughter. past medical history: receives her primary care through , dr. - hypertension, not taking her hctz recently due to side effects, baseline blood pressure 160-180 - diabetes - atrial fibrillation, not on coumadin - dvt's in both legs - per daughter was hospitalized at in past, was on heparin but they had to stop it (she doesn't know why) - colon cancer 7 years ago, s/p surgical resection; has not had follow-up colonoscopies - does not go for mammograms - cataracts - daughter says she was told she could never have any surgery because of a pinched nerve or blood vessel in her head social history: lives alone, daughter-in-law and daughter closely involved. neighbors include a nurse who is her caregiver, and her partner who is ms. lawyer. history of tobacco, alcohol, drug use. daughter: family history: not contributory. physical exam: exam on presentation to neurology service: t 99.8 hr 90's bp 160/80 rr 19 pulse ox 99% general appearance: 85 year old woman intubated in nad heent: wearing c-spine collar cv: irreg irreg, distant heart sounds, no gallops or murmurs audible lungs: clear to auscultation bilaterally. abdomen: soft, nontender extremities: bilateral pitting edema in le's with abrasions scattered over right le mental status: intubated, not sedated. eyes closed. no spontaneous eye opening, no eye opening to voice or sternal rub. grimaces to sternal rub. does not follow commands but per daughter squeezes her left hand to command for her. cranial nerves: pupils are equal, round and reactive to light 3>2. does not blink to threat on right. left gaze preference, can get eyes to midline but not past. unable to assess ocr due to c-spine collar. corneals present bilaterally. difficult to assess facial symmetry due to ett, grimaces bilaterally to nasal tickle, maybe a bit less on the right. does not cough/gag on ett. motor system: tone flaccid in right arm, less so in legs, paratonia with rigidity in left arm. rarely spontaneously moves left arm but not purposefully. able to wiggle fingers with left arm, withdraws to noxious stimuli. no movement of right arm, grimaces but does not move with noxious. weakly withdraws both legs to noxious, left a bit more vigorously than right. no spontaneous movement of legs. reflexes: deep tendon reflexes are 2+ and symmetric in the upper extremities, 2+ at the patellae, absent at the achilles. no clonus. plantar responses are extensor bilaterally. no . sensory: responds to noxious stimuli in all four extremities as above. coordination, gait: could not assess on discharge, appears comfortable. opens eyes spontaneously but not interactive. does not follow or fixate. pertinent results: ###lab results### at hospital: abg 7.41/20.5/435/19.1/-4.1 pt 15.2, inr 1.32, ptt 24 18.5>15.8/45.5<244 89seg, 1 band, 10 lymph 136 98 17 <271 4.2 20 1.6 ca 8.2 ua sg 1022, 37 wbc, ket 15, gluc 500 at 8:10p urine benzos, barbs, opiates, cocaine, amphet, mthdne negative colorstraw appearclear specgr1.012 ph 5.0 urobilneg bilineg leukneg bldsm nitrneg protneg glutr ketneg rbc 0-2 wbc6-10 bactmany yeastnone epi0 7:10p 137 104 19 242 agap=18 3.7 19 1.2 estgfr: 43/52 (click for details) ck: 775 mb: 10 mbi: 1.3 trop-t: 0.06 ca: 6.6 alt: 17 ap: 76 tbili: 1.5 alb: 3.0 ast: 34 ldh: dbili: tprot: : 49 lip: 54 serum asa, etoh, acetmnphn, benzo, barb, tricyc negative mcv 78 13.1 > 12.7 < 170 37.4 n:85.6 band:0 l:6.5 m:6.5 e:0.8 bas:0.5 pt: 14.2 ptt: 24.0 inr: 1.3 ###microbiology### 8:10 pm urine site: catheter urine culture (final ): escherichia coli. >100,000 organisms/ml.. presumptive identification. of two colonial morphologies. trimethoprim/sulfa sensitivity testing performed by . previously reported as sulfa x trimeth sensitive, , 9:30am. sensitivities: mic expressed in mcg/ml _________________________________________________________ escherichia coli | ampicillin------------ =>32 r ampicillin/sulbactam-- 8 s cefazolin------------- <=4 s cefepime-------------- <=1 s ceftazidime----------- <=1 s ceftriaxone----------- <=1 s cefuroxime------------ 16 i ciprofloxacin--------- =>4 r gentamicin------------ <=1 s imipenem-------------- <=1 s levofloxacin---------- =>8 r meropenem-------------<=0.25 s nitrofurantoin-------- 128 r piperacillin/tazo----- <=4 s tobramycin------------ <=1 s trimethoprim/sulfa---- r 4:57 am sputum source: endotracheal. gram stain (final ): >25 pmns and <10 epithelial cells/100x field. 4+ (>10 per 1000x field): multiple organisms consistent with oropharyngeal flora. respiratory culture (final ): moderate growth oropharyngeal flora. gram negative rod(s). rare growth. 10:04 am sputum source: induced. gram stain (final ): >25 pmns and <10 epithelial cells/100x field. 4+ (>10 per 1000x field): gram negative rod(s). 4+ (>10 per 1000x field): gram positive cocci. in pairs, chains, and clusters. 4+ (>10 per 1000x field): gram positive rod(s). respiratory culture (final ): moderate growth oropharyngeal flora. due to mixed bacterial types ( >= 3 colony types) an abbreviated workup will be performed appropriate to the isolates recovered from this site. staph aureus coag +. moderate growth. staphylococcus species may develop resistance during prolonged therapy with quinolones. therefore, isolates that are initially susceptible may become resistant within three to four days after initiation of therapy. testing of repeat isolates may be warranted. escherichia coli. moderate growth. trimethoprim/sulfa sensitivity testing available on request. beta streptococcus. moderate growth. sensitivities: mic expressed in mcg/ml staph aureus coag + | escherichia coli | | ampicillin------------ =>32 r ampicillin/sulbactam-- 16 i cefazolin------------- <=4 s cefepime-------------- <=1 s ceftazidime----------- <=1 s ceftriaxone----------- <=1 s cefuroxime------------ 16 i ciprofloxacin--------- =>4 r clindamycin-----------<=0.25 s erythromycin----------<=0.25 s gentamicin------------ <=0.5 s <=1 s imipenem-------------- <=1 s levofloxacin----------<=0.12 s meropenem------------- <=0.25 s oxacillin------------- 0.5 s penicillin------------ =>0.5 r piperacillin/tazo----- <=4 s tobramycin------------ <=1 s ###imaging### ekg: afib, twave flattening, nsst changes cta head w&w/o c & reco : findings: there is a large hypodense area in the territory of the left mca involving the temporal as well as frontoparietal regions. there is associated intraparenchymal hemorrhage. the findings are most consistent with hemorrhagic conversion of an infarct. there is no herniation present. the cta demonstrates that the left vertebral artery is dominant. the internal carotid arteries appear normal. their origins are not visualized. there are areas of atherosclerosis within the cavernous portions of both internal carotid arteries. the basilar artery is small. there is a fetal origin to both posterior cerebral arteries. there is a suggestion of an area of stenosis in the p2 segment of the right posterior cerebral artery. both posterior inferior cerebellar arteries are present. both middle and anterior cerebral arteries appear normal. there are no aneurysms. the volume rendered images of the circle of demonstrate inferior displacement of the distal and middle cerebral artery branches on the left due to the mass effect from the intraparenchymal hemorrhage infarct. no filling defects are noted. impression: hypodensity in the territory of the left middle cerebral artery with areas of intraparenchymal hemorrhage. the cta does not demonstrate any evidence of an aneurysm or filling defects. ct c-spine w/o contrast : findings: there is no evidence of acute fracture. evaluation of the prevertebral soft tissues is limited by endotracheal tube. there is exaggerated lordosis of the cervical spine with relative widening of the anterior disc spaces seen at c3-4 and c4-5 levels. ct does not provide intrathecal detail comparable to mri, however, the thecal sac appears grossly intact. degenerative changes noted within the cervical spine. impression: 1. no evidence of acute fracture. exaggerated cervical lordosis with relative widening of the anterior disc spaces at the c3-4 and c4-5 levels. ligamentous injury cannot be excluded and if clinically indicated, mr could be helpful for further evaluation. 2. degenerative changes noted within the cervical spine. eeg : this is an abnormal eeg due to the suppressed background and intermittent low voltage frontal alpha activity. the anteriorly predominant intermittent alpha activity may represent an alpha coma, although this is usually more persistent. the low voltage suppressed background suggests a severe encephalopathy, which may be seen with infections, toxic metabolic abnormalities, medication effect or ischemia. no epileptiform features were noted. a repeat study would be beneficial to better assess the posterior background rhythm and to assess for interval change. ct t-spine: no fracture or malignment within the thoracic spine. ct l-spine: 1. no fracture or malalignment within the lumbar spine. 2. 1.5 cm nonobstructing left proximal ureteral stone. 3. atherosclerosis. head ct : 1. no significant mass effect or evidence of herniation. 2. stable or slight interval increase in left frontoparietal temporal acute intraparenchymal hemorrhage with acute adjacent subarachnoid blood. 3. slight interval worsening in surrounding edema. mr : there is no abnormal bone marrow edema. there is no disc edema. there is minimal prevertebral soft tissue edema along the entire cervical spine. this could be due to ligamentous sprain. in addition, mild edema is also noted within interspinous ligament posteriorly to c3-4 and 5. again, this could indicate ligamentous strain. the craniocervical junction is normal. the cervical spine alignment is also preserved. at c4-5, there is a tiny central disc protrusion indenting the ventral thecal sac causing no significant spinal canal narrowing. impression: no evidence of disc or bone marrow edema. prevertebral interspinous ligament edema could indicate ligamentous strain. no abnormal signal within the cord or spinal canal stenosis. mr brain with contrast : findings: the mrv does not demonstrate any evidence of venous sinus thrombosis. there is a large area of slow diffusion associated with the large left frontoparietal hematoma. these findings are most consistent with hemorrhagic infarct. an additional acute infarct is noted within the left posterior inferior cerebellar artery territory. there is no associated hemorrhage within this infarct. there are moderate amounts of confluent periventricular white matter t2 hyperintensity consistent with moderate amounts of chronic microvascular ischemic change. no enhancing masses are identified. areas of increased signal on the post-gadolinium images were present on the pre-gadolinium axial t1 imaging. there is no midline shift or herniation. there is fluid within the paranasal sinuses as well as both mastoid air sinuses due to intubation. impression: hemorrhagic left middle cerebral artery territory infarct. additional acute infarct within the posterior inferior cerebral artery territory on the left. lenis bilateral : no evidence of any dvt in either lower extremity cxr : endotracheal tube and nasogastric tube are in standard positions. cardiac silhouette is mildly enlarged, and there is new slight engorgement of the pulmonary vascularity accompanied by perihilar haziness. moderate left and small right pleural effusions have developed. left retrocardiac opacity may reflect a combination of atelectasis and effusion, but underlying infectious process is not excluded in the appropriate setting. brief hospital course: 85 year old woman with history of afib not on coumadin, hypertension, dvt's, colon cancer, multiple recent falls with possible loc, now found unresponsive with left intraparenchymal and subarachnoid hemorrhage. exam revealed right sided weakness, left gaze preference and decreased blink to threat from the right. possible etiology for bleed included hypertension, amyloid angiopathy, contusion/fall, underlying mass or hemorrhagic emoblic infarct. also, given the history of recent falls, question was raised of possible seizure activity. hospital course is listed below by system. notably, on , by the family's decision, the goals of care were changed to comfort measures only. all antibiotics and antihypertensives were discontinued, all ivs removed, and no further labs or vital signs were taken. her cervical collar was removed but kept at bedside to replace for any movements that might cause neck pain without a collar in place. she was started on sublingual ativan and morphine as needed, scopolamine and levsin for secretions, and a suppository for constipation as needed. hospital course: neuro: left intracranial hemorrhage was most likely secondary to hemorrhagic conversion of emoblic stroke. patient was evaluated by neurosurgery, and received mannitol, decadron and dilnantin. head cta was negative for aneurysm or vascular malformation. an mri brain showed again left mca hemorrhagic infarct and an additional acute infarct with in the left pica territory further supporting the etiology of emoblic phenomenon. eeg initally showed slowing with alpha bursts concerning for alpha coma but no epileptiform activity; repeat showed findings consistent with encephalopathy, still no epileptiform activity. ct spine was negative for fracture and mr showed prevertebral interspinous ligament edema that could indicate ligamentous strain. as a result, she was placed in a hard collar. cv: acute myocardial infarction was ruled out. hbaic and fasting lipid panel were sent, and aspirin held. her heart rate and bp were controlled with a beta blocker and prn hydralazine. pulm: patient was extubated without difficulty. id: she had leukocytosis and gram stain of sputum positive for coag positive staph aureus and e. coli. she was started on empiric antibiotics (vancomycin and cefepime). endo: treated initially with iss then started on glyburide . ppx: treated with ppi, sc heparin, lipitor, bowel regimen, ssi. medications on admission: hydrochlorothiazide 1 daily lisinopril 10 mg lopressor 50 mg glyburide 1.25 mg daily ?"sanctura 10 mg daily" discharge medications: 1. morphine concentrate 20 mg/ml solution sig: mgs po q1-2h () as needed for discomfort. 2. lorazepam 0.5 mg tablet sig: 0.5-2 mg po q4-6h (every 4 to 6 hours) as needed: sublingual. 3. scopolamine base 1.5 mg patch 72hr sig: one (1) patch 72hr transdermal q72h (every 72 hours) as needed for secretions. 4. hyoscyamine sulfate 0.125 mg tablet, sublingual sig: one (1) tablet, sublingual sublingual q4h (every 4 hours) as needed for secretions. 5. bisacodyl 10 mg suppository sig: one (1) suppository rectal hs (at bedtime) as needed for constipation. 6. acetaminophen 650 mg suppository sig: one (1) suppository rectal q4-6h (every 4 to 6 hours) as needed for fever or pain. discharge disposition: extended care facility: - discharge diagnosis: left intraparenchymal hemorrhage with associated subarachnoid hemorrhage pneumonia hypertension atrial fibrillation with rapid ventricular rate hypokalemia discharge condition: unresponsive to voice; not interactive. opens eyes spontaneously but does not fixate or track. discharge instructions: take medications as needed for comfort. followup instructions: none md procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more enteral infusion of concentrated nutritional substances diagnoses: pneumonia, organism unspecified urinary tract infection, site not specified diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled atrial fibrillation subarachnoid hemorrhage intracerebral hemorrhage hypopotassemia personal history of malignant neoplasm of large intestine personal history of venous thrombosis and embolism encounter for palliative care apnea 

Answer: The patient is high likely exposed to