Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: this is an 86-year-old male nursing home resident with advanced dementia, coronary artery disease, cerebrovascular accident since , peg placed in in the setting of pneumonia and sepsis who presents status post pea arrest. the patient was noted to be lethargic on the and chest x-ray was done at that time which showed a question of a right lower lobe pneumonia. he was started on levaquin. by report, the patient improved the next day and became more verbal. on the morning of admission, the patient was found to be again lethargic and less responsive. at that time, temperature was normal. his blood pressure is 104/76, heart rate 118, respiratory rate was increased with an oxygen saturation of 86% on room air. fingerstick was 326 and he was given insulin and ems was called. he was brought to the emergency room. on arrival, the patient was unresponsive and cyanotic. his temperature was 98 and his oxygen saturation was 30% and he had no blood pressure. rhythm was pulseless electrical activity. cpr was started. atropine and epinephrine were given with restoration of his pulse. pressors were started for hypotension. patient was intubated. there is an unclear duration of arrest prior to the code being called. the code itself lasted nine minutes. ct angiogram of the chest revealed no pulmonary embolism. hematocrit was noted to be 20 and he was transfused 1 unit of packed red blood cells, and was admitted to the intensive care unit for further management. past medical history: 1. coronary artery disease with coronary artery bypass graft in . 2. dementia. 3. cerebrovascular accident with left sided weakness resulting. 4. diabetes mellitus type 2. 5. peptic ulcer disease. 6. atypical psychosis. 7. prostate cancer. 8. hypercholesterolemia. 9. ejection fraction of 40-50% with left ventricular hypertrophy, moderate mitral regurgitation, and moderate as with global hypokinesis. 10. avr for aortic insufficiency. 11. peg tube for feeding placement . 12. aspiration pneumonia and sepsis with no identified source in . 13. upper gastrointestinal bleed. 14. abdominal aortic aneurysm. 15. seizure disorder. 16. gout. medications: 1. norvasc 5 mg po q day. 2. prevacid 30 mg po q day. 3. risperdal 0.25 mg po bid. 4. allopurinol 100 mg po q day. 5. aspirin 81 mg po q day. 6. dilantin 300 mg po q am, 400 mg po q pm. 7. nph insulin 3 units q am, 4 units q pm. 8. cardura 4 mg po q day. 9. lipitor 10 mg po q day. 10. trazodone 25 mg po bid prn. 11. tramadol 25-50 mg po q6h prn. 12. lactulose 20 cc po prn. 13. levaquin 500 mg po q day since . 14. promod with fiber at 95 cc per hour. allergies: no known drug allergies. social history: nursing home resident x2.5 years at the . no tobacco or ethanol. pcp is . and dr. from group. family history: noncontributory. review of systems: not able to obtain. vital signs on admission: temperature 95.0, blood pressure 109/43 with a map of 65, heart rate is 97, and oxygen saturation of 99%. he was mechanically ventilated on simv plus pressure support of 600 cc tidal volume with a respiratory rate of 12 and a fio2 of 1.0. no spontaneous respirations, peep of 5, and pressure support of 10. he was on dopamine at 21 mcg/kg/minute. physical examination: in general he was unresponsive and intubated. pupils were fixed and dilated. there is no response to confrontation. jugular venous pressure was not seen. the chest was clear anteriorly. heart has a normal s1, s2 without murmur, and is regular, rate, and rhythm. abdomen was obese with a g tube in place with no surrounding erythema or pus. he is guaiac positive according to the exam in the emergency room. extremities were without edema and were warm. distal pulses were not felt in the feet, but there were 1+ radial pulses. skin was intact without rash. there is no response to voice, and he did withdraw to pain. toes were upgoing bilaterally. tone was increased with flaccid tone noted on the left and decreased but flaccid on the right. pertinent laboratories: hematocrit was 20.5, white count 7.0, platelets 182. inr is 1.3. sodium is 139, potassium 4.4, chloride 103, bicarb 13, bun 62, creatinine 1.1, and glucose of 609. anion gap was 23. transaminases were normal. amylase and lipase 103 and 22. ck was 44 with a troponin of 0.3. albumin 1.9. calcium 7.1, phosphate 6.3, magnesium 2.2. arterial blood gas showed a ph of 7.13, pco2 of 40, and a po2 of 252. lactate was 5.7. chest x-ray showed et tube 7 cm above the carina with heart size within normal limits and low lung volumes. there is normal pulmonary vasculature and a widen mediastinum. ct angiogram of the chest showed a right lower lobe aspiration pneumonia. no pulmonary embolism. a right lower lobe mass 3.2 x 2.9 cm encasing the right lower lobe bronchus and pulmonary artery. mediastinal lymphadenopathy including pericarinal and ap window lymphadenopathy. an anterior 8 mm nodule and ascites with intraabdominal hemorrhage. electrocardiogram showed atrial fibrillation at 127 beats per minute with st depressions 1 mm in leads v3 to v6 with t-wave inversions in those leads. there was also t-wave inversions seen in leads i, avl, ii, iii, and avf. ct scan of the abdomen showed a layering hematoma adjacent to the liver extending down the right pericolic gutter. a 5.1 x 4.8 cm exophytic simple cyst in the right kidney in the lower pole, a large 8.8 x 12 cm infrarenal abdominal aortic aneurysm just above the bifurcation concerning for recent expansion and no obvious liver disease or injury. ct scan of the head showed a large chronic right middle cerebral artery territory infarct that was felt to be old as well as right cerebral watershed infarct also felt to be old. there is also an old left caudate lacune. there was no new mass effect or intracranial hemorrhage. impression: this is an 86-year-old male with advanced dementia, abdominal aortic aneurysm, coronary artery disease, who presented with pulseless electrical activity, cardiac arrest, and was successfully resuscitated, but now with examination suggestive of anoxic brain injury. hospital course: the cause of the patient's pea arrest was not clear. it was felt to most likely be multifactorial secondary to anemia, pneumonia, and hypovolemia. pe and tamponade were effectively ruled out on ct angiogram. the patient's troponin rose to over 50, which was felt to be consistent with the patient's cardiac arrest. there is no intervention that was felt to be required according to the cardiology consult service. in terms of the patient's abdominal aneurysm, there was radiographic evidence of recent expansion, but rupture was ruled out by the abdominal ct scan. patient received packed red blood cells for a hematocrit less than 28. blood sugar was managed with insulin drip initially and changed to regular insulin-sliding scale. the patient's new lung mass was not known prior to this admission and this was felt to worsen the patient's overall prognosis. this was communicated to the family, who understood. it was felt that the appearance of the mass was most consistent with malignancy. in terms of the patient's pneumonia, he was given levaquin and flagyl. the neurology service was consulted and agreed to the intensive care unit's assessment that the patient had a very poor prognosis given his multiple comorbidities and the prolonged arrest. on , the patient developed new anisocoria and repeat ct scan of the head revealed massive left sided edema with subfalcial herniation and probable uncal herniation. mannitol was given as per the neurology and neurosurgery consultants. vancomycin was added to the patient's antibiotic regimen once blood cultures returned positive for gram-positive cocci. on , the family meeting was held with patient's wife, son, daughter, and several of the physicians. the grave prognosis was communicated to the family. the family decided to withdraw the ventilator which was done. morphine was given and titrated for comfort. the patient died that night at 11:35 pm. , m.d. procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube diagnoses: acidosis other convulsions acute respiratory failure pneumonitis due to inhalation of food or vomitus cardiac arrest anoxic brain damage malignant neoplasm of other parts of bronchus or lung 

Answer: The patient is high likely exposed to