Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: headache major surgical or invasive procedure: brain biopsy history of present illness: 68 yo woman c/o "worst headache of her life" on day of admission, called ems, en route became unresponsive, intubated in ed. sister visited with patient x 3 weeks, left on . during that visit, they both went to . sister is healthy. the only complaint the patient had (per sister) was headache, "off and on x several weeks", no visual complaints, able to go about daily activities. no personality changes, no c/o weakness/numbness. unaware of any alternative medication use. no h/o prior neurologic illness such as shaking (seizure activity), no hearing loss. past medical history: htn hyperchol social history: lives alone, only family is a sister in , , never married, no children, used to work in a factory, retired. no tob/etoh/drugs. family history: sister has severe osteoporosis, no other family. physical exam: vitals: tm 101.2, tc 100.6, bp 156/78, hr 78, rr 17, o2 sat 100% on cpap + ps 5/5, vt 380, fio2 0.4. i/o: -1.8l los. gen: intubated, sedated, obese woman skin: no rash heent: mmm,ett in place neck: supple chest: normal respiratory pattern, cta bilat cv: regular rate and rhythm without murmurs abd: nontender, softly distended, + bs extrem: no edema neuro: mental status: patient is alert, awake when propofol weaned. unable to assess speech or verbal answers to questions as is intubated. nods head to questions "are you cold?" "are you in pain?" unclear how accurate her answers are. cranial nerves: i: deferred ii: unable to assess visual acuity or fields. fundoscopic exam: discs flat, fundi clear, no hemorrhages or exudates. pupils: 3mm- >2mm bilat, consenual constriction to light. iii, iv, vi: eoms full, gaze conjugate. no nystagmus or ptosis. v: unable to assess sensation on face, + corneal reflex (tested when more sedated) vii: unable to assess facial strength viii; unable to reliably test hearing ix, x: gag reflex present (gagging on ett occasionally) : unable to assess xii: unable to assess sensory: unable to assess fully, patient withdrawls right sided limbs only from pain, nods head "yes" when asked if she can feel me touching her on right arm and leg only. motor: normal bulk, tone. no fasciculationst. no adventitious movements. lifts right leg partially to gravity. wiggles left toes (leg falls when lifted). reflexes: 2+ throughout, toes downgoing bilaterally. coordination: unable to assess gait:unable to assess pertinent results: 08:32pm wbc-14.1* rbc-4.57 hgb-14.3 hct-40.1 mcv-88 mch-31.4 mchc-35.8* rdw-14.4 08:32pm plt count-271 08:32pm glucose-134* urea n-18 creat-0.7 sodium-141 potassium-2.9* chloride-100 total co2-29 anion gap-15 08:32pm asa-neg ethanol-neg acetmnphn-neg bnzodzpn-neg barbitrt-neg tricyclic-neg 08:32pm pt-12.0 ptt-19.1* inr(pt)-1.0 brain mr: 1. innumerable nodular enhancing foci predominantly in a watershed distribution in the cerebrum but also within the cerebellum with associated edema. a focus of restricted diffusion in the right posterior parietal region without evidence of minimal hemorrhage appears to represent a small associated infarction. given the watershed distribution, small vessel vasculitis should be considered in the differential diagnosis. the appearance and distribution is not classical for acute disseminated encephalomyelitis. the differential diagnosis also includes an unusual encephalitis, sarcoidosis, or other granulomatous disorder. 2. normal circle of mra. brief hospital course: the patient was admitted and managed for her headache and loss of consciousness. she was initially intubated and sent to the nicu. her head mri showed multiple white matter lesions consistent with a leukoencephalopathy of unclear etiology. an lp was done which was unrealing. a rheumatology consult was obtained to help us work up a possible cerebral vasculitis/angitis. an eeg showed bursts of generalized slowing. a brain biopsy was performed which showed "acute hemorrhagic leukoencephalopathy". she was started initially on iv steroids and then switched to po steroids in addition to seizure prophylaxis with phenytoin. her neurologic exam improved over the course of her stay to where she was more alert and oriented. her speech became more fluent. she still remained with a mild left hemi-neglect and weakness in the distal left extremities. medications on admission: amoxacillin lipitor atenolol triamterene/hctz kcl reglan zofran ibuprofen caltrex tramodol celebrex discharge medications: lisinopril 40 qd levofloxacin 500 qd x 5 more days metoprolol 50 protonix 40 qd phenytoin 100 qd prednisone 60 qd vitamin d 400 qd calcium 500 discharge disposition: extended care facility: - discharge diagnosis: 1. leukoencephalopathy 2. hypertension discharge condition: stable discharge instructions: please take medications as prescribed. md, procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more spinal tap incision of lung insertion of endotracheal tube enteral infusion of concentrated nutritional substances non-invasive mechanical ventilation open biopsy of brain diagnoses: pneumonia, organism unspecified unspecified essential hypertension hemorrhage complicating a procedure other alteration of consciousness unspecified causes of encephalitis, myelitis, and encephalomyelitis 

Answer: The patient is high likely exposed to