Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: percocet / codeine / aspirin attending: chief complaint: weakness and fatigue major surgical or invasive procedure: none history of present illness: the patient is a 57 year old male with end stage liver disease secondary to hep c cirrhosis and alcohol abuse. he was discharged one day prior to this admission after being treated for hepatic encephalopathy. he slept well that night, but then became weak and lethargic. he vomited twice on the day of admission and as per his wife, he poor urine output. he has been taking his lactulose and has been having watery stools every 2 hours. denises hematemesis, hematochezia, dysuria, sob, or cp. past medical history: -hepatitis c cirrhosis (diagnosed in ) - no liver bx done; risk factors: suspected unprotected sex, tattoo; no interferon/ribavirin due to insurance reasons. variceal bleeding in (7 units required), egd revealed grade-ii varices but no acute bleeding. encephalopathy and mild ascites; egd: 4 cord varices (one was grade iii), 3 bands placed; meld score 29 (); 19 () -copd - pfts normal -surgeries for hernia (bilateral), varicose veins -cellulitis -chest pain - cath: minor cfx, lad disease social history: patient has not drank alcohol since . prior alcohol intake since adolescence, 6-7 beers/day; period of abstinence from age 34-44; does not like aa. currently denies tob, omr states 3 packs /day for 30 yrs; quit within last year. no ivd abuse tattoos + lives w/ his girlfriend ) of 13 yrs in , ma masshealth insurance; disability income - does not work currently, former painter does not drive family history: no hx of liver disease in family physical exam: vs- 97.0, 75, 105/58, 12, 100% ra gen: overweight, jaundiced, palmar erythema, spider nevi lungs: cta b/l heart: rrr, grade 2 sem heard best at apex abd: mildly tender rlq, no rebound or guarding, liver mildly enlarged, guiac negative ext: 1+ pedal edema neuro: + asterixis, cn 2-12 grossly in tact, + full body tremors pertinent results: 05:00am wbc-4.7 rbc-2.60* hgb-9.8* hct-27.9* mcv-108* mch-37.6* mchc-35.0 rdw-17.6* plt ct-74* 12:50pm wbc-5.2 rbc-2.02* hgb-7.1* hct-22.4* mcv-111* mch-35.5* mchc-31.9 rdw-17.9* plt ct-98* 07:56pm wbc-4.9 rbc-1.72* hgb-5.3*# hct-15.6*# mcv-91# mch-31.0# mchc-34.0 rdw-25.9* plt ct-50* 10:10pm wbc-4.7 rbc-2.02* hgb-6.2* hct-18.2* mcv-90 mch-30.5 mchc-33.9 rdw-20.1* plt ct-29* 11:24pm hct-25.2*# plt ct-91*# 01:00am hct-27.5* plt ct-153# 05:39pm hct-29.7* plt ct-81* 02:00am wbc-5.5 rbc-1.78*# hgb-5.4*# hct-15.4*# mcv-87 mch-30.4 mchc-35.1* rdw-16.8* plt ct-40* 02:27am wbc-4.7 rbc-1.32*# hgb-4.0*# hct-11.5*# mcv-88 mch-30.3 mchc-34.6 rdw-17.3* plt ct-27* 09:33am wbc-9.9 rbc-3.21* hgb-10.2* hct-26.6* mcv-83 mch-31.7 mchc-36.9* rdw-16.3* plt ct-46* 02:30pm wbc-6.4 rbc-1.95*# hgb-5.9*# hct-16.4*# mcv-85 mch-30.1 mchc-35.6* rdw-17.2* plt ct-86* 03:30pm wbc-6.0 rbc-1.78* hgb-5.4* hct-15.1* mcv-85 mch-30.6 mchc-36.1* rdw-17.9* plt ct-76* 05:00am pt-20.7* ptt-150* inr(pt)-3.0 03:30pm plt ct-76* 05:00am glucose-106* urean-42* creat-1.5* na-127* k-4.3 cl-98 hco3-20* angap-13 05:00am alt-87* ast-91* ld(ldh)-268* alkphos-106 totbili-7.5* 02:00am alt-311* ast-709* alkphos-72 amylase-82 totbili-6.8* 01:20am alt-819* ast-2104* alkphos-124* totbili-12.8* 05:15pm osmolal-283 05:35am ammonia-55* 12:31am cortsol-55.9* 02:35pm type-art po2-127* pco2-31* ph-7.39 calhco3-19* base xs--4 08:28am type-art po2-112* pco2-42 ph-7.35 calhco3-24 base xs--2 02:35pm glucose-50* lactate-11.7* k-4.5 cl-96* 08:28am glucose-73 lactate-6.5* k-4.5 06:06am glucose-108* lactate-6.2* brief hospital course: the patient was admitted to the hepatobiliary service on . his encephalopathy was thought to be baseline as well as due to dehydration. a ruq ultrasound revealed no portal vein thrombosis. he was afebrile with a normal wbc, but was pan-cultured anyway. ua was negative. hematocrit was stable at 30. ceftriaxone was started empirically for possible peritoneal infection. lactulose was continued. he was aggressively hydrated. he also had a createnine of 2.1 (baseline 1.5) and a sodium of 124 (baseline 132). his fena was < 1%, so this was likely pre-renal. he could not have a feeding tube placed due to recent bleeding, and required supervised oral feeding. his meld on admission was 31. ceftriaxone was discontined hd 2 (no signs of infection). lactulose was titrated so as to get bms daily. on hd 2, createnine was down to 1.6. physical therapy saw him and felt he should progress to be discharged home. on hd 5, he became lightheaded and dropped to his feet while walking. denied loss of consciousness. thought to be due to orthostasis. his bp was 60/palp and hr was 110. he was helped back to bed and his hr and bp returned to (sbp 100, hr 90). his cmv viral load was negative. his hct was 22, so he was transfused 2 units rbcs for loss anemia. his inr was 3.0, so he was transfused 3 units of ffp to correct his coagulopathy. a ct scan revealed a right-side retroperitoneal hematoma. at that time, he was transferred to the trnasplant surgery serivce and sent to the icu. on hd 6, the patient was intubated. a central venous catheter and arterial line were placed. from this point on, this patient required aggressive transfusion of rbcs, ffp, platelets and cryo. over the night, his hct dropped to 15, then rose to 18, then 25, then 27. his platelets went fro 50 to 29 to 91 to 153. his inr went from 3.3 to 2.8 to 2.5 to 0.6. he required 11 units of rbcs, 2 of platelets, 4 of ffp, 1 of cryo and 1 of factor 7. on hd 7, his hct dropped from 28 to 11 over 3 hours. he did not have guiac + stools and hig ng tube outpuit was , his loss anemia was though to be entirely due to his retroperiotneal hematoma. tips and surgery were considered, but the mortality was thought to be too high. over the day, he reuired 9 units of rbcs, 2 platelets, 4 ffp, 2 cryo and 1 of factor 7. a dialysis catheter was placed for cvvhd. vancomycin, levofloxacin, and fluconazole were begun empirically. the goal was to transfuse products to keep hct > 27, plt > 75, and inr < 2.0. he required maximum pressor support (neosynepherine, vasopressin and levophed). despite extremes in transfusion of products, we were not able to stop his bleeding. a family meeting was arranged and the decision was made to hold all pressor support and products. he died later that night. medications on admission: atrovent, albuterol, flovent, protonix, flonase, quinine, lactulose, lasix, aldactone discharge disposition: expired discharge diagnosis: cirrhosis, retroperitonel hematoma, death discharge condition: dead procedure: venous catheterization, not elsewhere classified hemodialysis venous catheterization for renal dialysis arterial catheterization arteriography of other intra-abdominal arteries transfusion of packed cells other irrigation of (naso-)gastric tube transfusion of other serum transfusion of platelets transfusion of coagulation factors infusion of vasopressor agent diagnoses: acute kidney failure with lesion of tubular necrosis unspecified pleural effusion acute posthemorrhagic anemia acute and subacute necrosis of liver chronic hepatitis c with hepatic coma alcoholic cirrhosis of liver acquired coagulation factor deficiency unspecified septicemia hyposmolality and/or hyponatremia other and unspecified alcohol dependence, in remission chronic airway obstruction, not elsewhere classified acute respiratory failure other shock without mention of trauma hepatic encephalopathy dehydration hypovolemia systemic inflammatory response syndrome due to noninfectious process with acute organ dysfunction hemorrhage, unspecified orthostatic hypotension 

Answer: The patient is high likely exposed to