Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: demerol / codeine attending: chief complaint: 66f w/ esophageal cancer s/p ivor- esophagogastrectomy with complicated post-operative course including cardiac arrest, unit myopathy, and vent dependence. major surgical or invasive procedure: s/p ivor- esophagogastrectomy tracheostomy muscle biopsy insertion of feeding j-tube multiple central line placements history of present illness: on patient underwent an open ivor- esophagogastrectomy following neoadjuvent therapy for esophageal cancer. past medical history: s/p bilat tkr bulbar polio as a child htn not currently being treated cholecystectomy esophageal cancer, s/p neoadjuvent therapy vad placement prior j-tube placement social history: pt is a pharmacist and she and her husband run a local pharmacy. one of their several daughters is also a pharmacist. she has never smoked. she drinks socially on occasion. family history: her father had head/ neck cancer and was cured of it, but no other family history of cancer. physical exam: 5'3" 150lb t98.2 122 134/54 98% well developed female in nas, a&ox3 perrl, cn ii-xii intact lungs clear bilaterally cardiac exam unremarkable abdomen soft, well healed ccy inciosn. j tube site noted in place, c/d/i le warm well perfused pertinent results: 03:34pm wbc-9.2 rbc-2.38* hgb-7.9* hct-23.1* mcv-97 mch-33.2* mchc-34.1 rdw-15.1 03:34pm pt-13.6 ptt-30.1 inr(pt)-1.2 09:02am glucose-123* lactate-1.3 na+-137 k+-3.8 cl--106 09:32am type-art po2-89 pco2-46* ph-7.38 total co2-28 base xs-0 -assist/con intubated-intubated 03:34pm glucose-141* urea n-18 creat-0.8 sodium-141 potassium-3.6 chloride-110* total co2-23 anion gap-12 brief hospital course: on , patient underwent an uneventful open ivor- esophagogastrectomy, apparently without complications (ebl <300). post-op, patient had chest tubes in place and was transferred to the intensive care unit kept intubated for pain control. on pod #1, patient received 2 units of prbcs for low hct and persistent hypotension. later that day, patient went into af with fast ventricular response for the first time, ultimately responsing to an amiodorone bolus and ongoing drip, she returned to nsr shortly thereafter. by pod #2, patient had progressive respiratory decomensation, and could not be extubated. over the next two days, oxygenation improved and vent wean was once again tried. as oxygenation deteriorated, cxr became more consistent with ards. patient was started on empirically on zosyn for febrile episode to 102.5. sputum cultures would ultimately grow out pan sensitive klebsiella, and the patient was switched to levofloxacin. tpn was then started, as were tube feeds through the feeding j. on pod #6, an addittional chest tube was placed on the patient's right side for effusion. starting on pod #7, patient had to sedated and paralyzed for agitation and poor oxygenation. sedation/paralysis was achieved through various combinations of propofol, fentanyl, and cis-atrocurium drips. as ards continued to worsen, fio2 and peep were increased. patient continued to have signifigant pressor and fluid requirments and there was ongoing concerns for possible sepsis. on pod#11, steroids were started for presumed adrenal insufficiency. this seemed to help the intermittent hypotension, and progress was made weaning inotropic support. over the next several days there was growing concern about reduced mental status. initially this was thought to be over sedation in the setting of rebal failure, however, this failed to improve with weaning the sedation. ultimately, a ct scan was performed, showing no acute processes. on pod #16, a trachesotomy was placed at the bedside. by pod #17, patinet's weakness had become complete flaccid paralysis. a neuro consult suggested the possibility of anoxic brain injury, but no other etiologies could be isolated. mr of the brain confirmed no evidence of anoxic brain injury. emg was inconclusive, while eeg showed no specific cause. focus then turned towards unit myopathy and other complications of sedation. several attempts at weaning steroids resulted in increased bp lability and steroids were ultimately left alone to be weaned over a slower course. patient was thne fitted with a pmv which she tolerated well. over the next several days, patient made progress with trach mask, however, by pod 30, she again had increasiung need for ventilatory support and was once again placed on the ventilator. shortly after a trip to radiology for ng manipulation, patient had a brady cardic event, evolving into full asystolic arrest. she was succesfully resuscitated and started on a levo drip. empiric coverage for sepsis was once again started. once again, only pathogen that was isolated was kelbsiella pneumonia from sputum samples. after consulting with neurology, a muscle biopsy was obtain from the right qudriceps. microscopic examination of this confirmed unit myopathy. the remainder of the unit course was comp - discharge diagnosis: resolving unit myopathy vent dependence addisonian crisis (secondary to adrenal suppression) esophageal cancer resolving klebsiella pneumonia ards vent dependence s/p full arrest intermittent af discharge condition: stable remains steroid dependence discharge instructions: continue treatments as described. rehabilitation for unit myopathy. followup instructions: as directed by rehabilitation facility. should follow-up with dr. within 6-8 weeks. procedure: insertion of intercostal catheter for drainage venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more parenteral infusion of concentrated nutritional substances other total gastrectomy insertion of endotracheal tube fiber-optic bronchoscopy enteral infusion of concentrated nutritional substances arterial catheterization temporary tracheostomy closed [endoscopic] biopsy of bronchus pulmonary artery wedge monitoring transfusion of packed cells open biopsy of soft tissue infusion of vasopressor agent diagnoses: long-term (current) use of steroids acute kidney failure, unspecified atrial fibrillation pneumonitis due to inhalation of food or vomitus cardiac arrest pressure ulcer, lower back secondary and unspecified malignant neoplasm of intrathoracic lymph nodes pneumonia due to klebsiella pneumoniae critical illness myopathy malignant neoplasm of cardia 

Answer: The patient is high likely exposed to