Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: status epilepticus major surgical or invasive procedure: nil history of present illness: 55yo m nursing home resident s/p r craniectomy, h/o etoh abuse, hepatic cirrhosis with esophageal varices, korsakoff's dementia who was noted to have seizure around 5:05pm, when found in bed by nursing staff with generalized shaking and eyes open. after 1 min it stopped,then started again. this continued until ems came; he was put on a breathing mask (sat in lower 90's). pb level was 19. per report, he was seizing for about 15 min before arriving to the ed. in the ed, no iv access could be established for some 20 min (18.20). the patient continued to receive ativan im. this did not stop his seizure activity. per ed staff he had l-gaze deviation with twitching of the eyes and occasional shaking, but no seizure. at some point he apparently made some purposeful movements, before his eye would deviate again. he also vomited while in the ed, with likely aspiration. he was intubated, loaded on pht 750mg (80kg), and labs were drawn. he was then brought to ct. at baseline he has gtc seizures. last one in , lasting 10 min. at baseline he is "confused", cannot memorize anything and gets lost in the nursing home all the time, walking around in the wrong direction. he is able to walk, but his gait is unsteady. past medical history: known seizures: gtc's per nursing home, last in x 10min -+ppd -pna -ethoh abuse -hepatic cirrhosis -esophageal varices -korsakoff's psychosis -hypokalemia -dementia with delusions -s/p craniotomy r social history: lives in nursing home. walks unassisted but unsteady, gets lost daily in the nursing home. family history: unknown physical exam: (shortly after intubation on arrival) vitals: t afebr hr117 bp143/82 rr21 so2 95 gen: lac on l knee heent: mmm neck: no lad; neck supple lungs: vented bs heart: obese, normal bowel sounds, soft, nontender, nondistended extremities: edema l foot mental status: intubated; eyes closed; not following commands; no purposeful movements cranial nerves: ii: no blink to threat; perl 2-->1.5; no hippus iii, iv, vi: vor intact; no gaze deviation; no skew; very subtle twitching of the eyelids v: corneals and nasal tickle intact vii: no facial droop. viii: - ix: gag intact xii: - : - motor system: decreased tone; triple flexion in both le, more so on the r than on the l. reflexes: b t br pa pl right - - - - - left - - - - - toes: upgoing bilaterally. sensory system: triple flexion to nox in both le; no response in ue coordination: - gait: - pertinent results: discharge labs 142 109 4 97 agap=10 3.5 27 0.5 ca: 8.3 mg: 2.0 p: 2.9 alt: 19 ap: 55 tbili: 0.8 alb: 3.7 ast: 21 ldh: 188 dbili: tprot: : 48 lip: 26 phenoba: 28.4 91 2.7 10.5 61 29.8 pt: 14.0 ptt: 31.3 inr: 1.2 admission rbc-* wbc-0-2 bacteria-few yeast-none epi-0-2 plt count-111*# wbc-9.5# rbc-4.20* hgb-14.2 hct-38.4* mcv-92 mch-33.7* mchc-36.9* rdw-15.3 bnzodzpn-neg barbitrt-pos opiates-neg cocaine-neg amphetmn-neg mthdone-neg asa-neg ethanol-neg acetmnphn-neg bnzodzpn-neg barbitrt-pos tricyclic-neg phenobarb-13.6 glucose-171* urea n-8 creat-0.8 sodium-139 potassium-4.0 chloride-104 total co2-20* anion gap-19 alt(sgpt)-18 ast(sgot)-23 ld(ldh)-249 ck(cpk)-108 alk phos-71 tot bili-0.9 pt-14.6* ptt-36.6* inr(pt)-1.3* ammonia-112* tsh 0.85 (csf) wbc-1 rbc-2* polys-38 lymphs-26 monos-36 protein-29 glucose-90 eeg: this is an abnormal eeg due to the frequent right frontocentral slowing and rare sharp waves and the 5 hz slow and disorganized background rhythm. the first abnormality suggests right frontocentral subcortical dysfunction with an overlying region of potential epileptogenesis. the second abnormality suggests a moderate encephalopathy, which may be seen with infections, toxic metabolic abnormalities or medication effect. no electrographic seizures were recorded. nccthead 1. no evidence of acute bleed or traumatic injury. 2. several regions of encephalomalacic change, perhaps related to old traumatic contusion, which may provide substrate for seizure activity. 3. global cerebral atrophy. 4. evidence of old right frontal craniotomy, perhaps related to old trauma. us 1. no ascites. 2. cholelithiasis. brief hospital course: patient is a 55yo man with seizure disorder, hx of etoh abuse with alcoholic cirrhosis and korsakoff's, who was brought to the ed on from his nursing home for continued seizure activity. the seizure described at the nh was a gtc sz, the activity seen in the ed was likely focal or postictal. the phenobarbital level was low at 13.6. he was intubated in the ed. exam was limited as it was done shortly after intubation. subtle twitching of the eyelids could be observed. he was loaded on dilantin. based on l-gaze deviation, the focus may have been in the r hemisphere. this would fit with decreased response to noxious stimuli in the lle, and with a possible brain injury on the r (given a large scar). . neuro: he was admitted to neuro-icu. there was no acute infection seen in the csf. eeg showed right frontocentral slowing and generalised encephalopathy with no active seizures. ct head showed changes consitent with right craniotomy and l-sided encephalomalacia. on he had left arm tonic flexion off propofol (sma focus?). dlantin was loaded again and 100mg tid commenced. keppra dose was increased to 1500mg . phenobarbital boluses were given to maintain levels and dilantin ceased on . maintenance phenobarbital was increased to 60mg/135g and last level was 28.4. neurontin was continued. he was extubated on and transferred to floor . gi: liver chirrhosis there wer no signs of ascites on abdominal ultrasound. spironolactone and lactulose were continued. last lfts in results section. proton pump inhibitor was prescribed for prophylaxis. . id: cxr showed bilateral subsegmental atelectasis with no focal consolidation. the patient was closely observed and repeat cxr improved. there was no evidence of urinary tract infection. csf cultures were negative and blood cultures negative to date. . heme: pancytopenia was observed during the admission. old results show some evidence of low counts and it was thought possible that he has longstanding bone marrow suppression and was dehydrated on arrival with an effect of hemodilution. iatrogenic causes including dilantin and hit were considered. dilantin was ceased on . heparin was ceased and hit antibodies were negative. platelets at lowest were 46 on now improved to 61. hemoglobin is at lowest today 10.5 and hematocrit 29.8. white cell count is at low of 2.7. . fen: the patient was treated with thiamine and folate given korsakoff's dementia. he was commenced on tube feeds and transitioned to oral intake when awake. . discharge examination was consistent with his baseline with mental status changes, frequent chuckling, limited spontaneous speech. no focal motor deficits. asterixis. medications on admission: spironolactone 50 phenobarb 45/45/90 (last level 19 on ) keppra 1000/1000 neurontin lactulose kcl milk of magnesium discharge medications: 1. gabapentin 300 mg capsule sig: three (3) capsule po tid (3 times a day). 2. spironolactone 25 mg tablet sig: two (2) tablet po daily (daily). 3. lactulose 10 g/15 ml syrup sig: thirty (30) ml po tid (3 times a day). 4. pantoprazole 40 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po q24h (every 24 hours). 5. phenobarbital 20 mg/5 ml elixir sig: 135mg po q pm (). 6. phenobarbital 30 mg tablet sig: two (2) tablet po qam (once a day (in the morning)). 7. docusate sodium 100 mg capsule sig: one (1) capsule po bid (2 times a day). 8. levetiracetam 500 mg tablet sig: three (3) tablet po bid (2 times a day). 9. thiamine hcl 100 mg tablet sig: one (1) tablet po daily (daily). 10. folic acid 1 mg tablet sig: one (1) tablet po daily (daily). discharge disposition: extended care facility: discharge diagnosis: status epilepticus korsakoff's dementia cirrhosis discharge condition: improved. eating and drinking and mobilising independently. altered mental status consistent with known korsakoff's dementia. discharge instructions: you have had a prolonged seizure requiring intensive care. you should take all as directed. please keep follow up appointments and seek further medical advice for any further seizures or unwellness due to infection which may lower the seizure threshold. followup instructions: please arrange to see your pcp in the next week for clinical review and review of medications. procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube diagnoses: alcoholic cirrhosis of liver pneumonitis due to inhalation of food or vomitus grand mal status dehydration alcohol-induced persisting amnestic disorder aplastic anemia, unspecified 

Answer: The patient is high likely exposed to