Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: sepsis respiratory failure major surgical or invasive procedure: endotracheal intubtation central venous catheter placement history of present illness: the patient is a y.o. female with pmh of hypertension and dementia who was brought in from house nh with 1 week of lethargy, poor po intake, and hypernatremia. she was given ivfs and iv cipro x 3 days for uti. urine eventually grew enterococcus. 1 day prior to admission, she is reported to have aspirated, and given nebs/o2. the morning of admission, whe was more somnolent and hypotensive to the 80s. she was sent to the ed. . in the ed, t 101.6, bp initially 70s systolic, improved to 100 after 4l ivfs. she was somnolent with rr 16 and o2 sats ranging from 86% nrb to 92% ra. she was intubated for airway protection. labs revealed leukocytosis (18.5), arf, lactate 2.6, and troponin of 0.16. ecg neg for acute ischemic abnormalities (no old). u/a was dirty and cxr with patchy bibasilar infiltrates concerning for pneumonia (? aspiration). she was given levoflox 750, vanco 1gm, flagyl 500mg, and asa 325pr. a r subclavian central line was placed. . of note, a cxr on showed clear lungs. urine from grew enterococcus but sensitivites could not be performed. labs on showed wbc 10.3, hct of 34, plt 314, na- 161, k-4.1, cl-131, co2- 21, bun 46, and cr 1.2. . ros: unable to obtain. past medical history: lung cancer s/p resection dementia w/agitation htn recent utis social history: was living with her daughter until , but then was hospitalized for agitation/worsening dementia, and has been in ever since. >50 pack year smoking hx, quit in . no etoh or illicits. at baseline, aao x1 and recognizes daughter. usually is agitated when medical care given. family history: noncontributory physical exam: vs: t 97.4 bp 104/61, hr 90, rr 23, 100% on ac/1.00/400/16/5 gen: intubated, sedated heent: dry mucous membranes neck: flat jvp lungs: bilateral rhonchi, r >l abd: soft, nd/nt ext: no edema skin: erythema over sacral region, no skin breakdown neuro: sedated but responds to pain (a-line placement) pertinent results: cxr : 1. et and ng tubes in satisfactory position. 2. patchy bibasilar consolidations, left more than right, which, in this setting, may represent aspiration pneumonitis. 3. mild chf with interstitial edema. . cxr findings: in comparison with the study of , there has been some reduction in the left pleural effusion, possibly related to a thoracentesis. little change in the effusion on the right. enlargement of the cardiac silhouette with vascular congestion is again seen. nasogastric tube has been pulled back and lies within the distal esophagus. . ct head : no hemorrhage . ct head : impression: no significant change since the previous study of . no acute hemorrhage identified. moderate-to-severe ventriculomegaly suggestive of normal pressure hydrocephalus is seen. . mri/mra head : ventricles diffusely enlarged out of proportion to the sulci with extensive deep and periventricular white matter t2 hyperintensities. above findings may represent communicating hydrocephalus versus central atrophy. there is fluid within the mastoid air cells bilaterally, and there is prominence of the adenoidal tissue, which is unusual for the patient's age. normal mra of the head. . eeg and : encephalopathy. no seizure activity. . 12:40pm wbc-18.5* rbc-3.92* hgb-10.0* hct-31.8* mcv-81* mch-25.5* mchc-31.4 rdw-17.1* 12:40pm alt(sgpt)-18 ast(sgot)-28 ck(cpk)-602* alk phos-88 amylase-60 tot bili-0.4 12:40pm glucose-327* urea n-101* creat-3.6* sodium-161* potassium-5.7* chloride-130* total co2-18* anion gap-19 12:44pm lactate-2.6* 05:02pm plt count-185 05:02pm pt-19.4* ptt-41.4* inr(pt)-1.8* brief hospital course: f h/o htn, dementia, admitted to micu given hypotension likely sepsis from urine proteus, sputum proteus; also respiratory failure requiring intubation and icu admission. pt was extubated but remained only responsive only to pain off sedation. neuro workup unrevealing and pt eventually became more responsive. pt remained hd stable and afebrile after initial resuscitation. . 1. sepsis: on admission, pt noted to be hypotensive to 70s in ed with respiratory distress, prompting intubation. pt was started on vanc for h/o vanc-sensitive enterococcal uti. urine and sputum grew proteus sensitive to pip-taz; pip-taz therefore started on . sputum grew ossa, vanc d/c'd and nafcillin started to continue abx course for pna. pt completed 10 day courses of zosyn and nafcillin on .and remained afebrile and hemodynamically stable. . 2. respiratory failure: pt initially intubated for airway protection given somnolence and decreased o2sat during episode of hypotension in ed, as well as suspected aspiration pna per cxr on admission. sedation discontinued with haloperidol prn for agitation, but pt continued to have depressed mental status. pt therefore maintained on intubation for airway protection given altered mental status, however, tolerating progressive weaning of ventilator settings. pt was extubated without difficulty. . 3. altered mental status: slowly improved over time. ct head demonstrated no acute bleed, but moderate ventriculomegaly possibly indicating normal pressure hydrocephalus. h2 blocker removed for possible ms effects, and ppi started. pt demonstrated gaze preference to l per neuro consult exam. encephalitis was entertained, and toxic metabolic workup initiated: tsh normal, rpr nr, ua unrevealing, eeg (demonstrating encephalopathy), mri brain w/ and w/o contrast (demonstrating communicating hydrocephalus vs. volume loss, lfts (unrevealing). after discussions with family, it was decided to defer an lp at this time but to repeat eeg which did not show any new findings. pt became somewhat more alert on . it was felt that her ams is likely only slowly recovering given underlying severe dementia. . 4. melanotic stools- transient episode. pt was placed on ppi iv bid and asa 325 was held. hct remained stable. . 5. af: pt had episodes of paf with associated rvr. has responded very well to 12.5 mg po metoprolol . metoprolol was eventually increased to 25 mg tid with good effect and continued at this dose. . 6. anemia: pt noted to have decreased hct to 21.5 on , with hemolysis and iron studies nondiagnostic for hemolysis. pt received 2units prbcs and has maintained hct. in addition, episode of melanotic stools as above. however, hct remained stable around 25 without any further transfusions. . 7. acute renal failure: on admission, pt's creatinine 3.6 considered likely to either prerenal vs sepsis-related atn etiology. pt received ivf during intial resuscitaiton and arf resolved over the course of her micu stay. . 8. hypernatremia: na 161 on admission, considered possibly dehydration, and corrected with free water and intermittent d5w boluses. na trended down to low 140s. . 9. coagulopathy: on admission, inr 1.7 likely poor nutritional status given low albumin; received vit k po x 3 days with improvement. remained stable. . 10. hyperglycemia: nph titrated to maintain fs <150; hyperglycemia at night. increased qhs lantus to 12 units from 10. ....................... on the floor, the patient spiked another temperature and was restarted on vancomycin and zosyn. additional discussions were had with the family, who wished to place a feeding tube for nutrition. plans were made for ir placement on . during the procedure, after receiving 1mg iv versed, the patient's blood pressures dropped. it was initially attributed to sedation, but repeat cbc demonstrated a lower hematocrit. a sub-clavian central line was placed and a stat ct scan of the abdomen and pelvis demonstrated perforation of an abdominal aortic aneurysm. she remained stable for approximately twelve hours and was supported with transfusions and iv fluid, but on the morning of acutely dropped her blood pressure; it was felt secondary to acute rupture of the already compromised aaa, and she expired shortly afterward with her daughter at her side. the medical examiner has accepted the case. medications on admission: ciproflox 250mg po tid started , d/c'd ciproflox 400mg iv q12 started , d/c ferrous sulfate 325 po daily catapres tts 3 patch - apply 1 patch qweek on wed depakote sprinkles: 375mg po daily (9pm) lexapro taper 10mg daily ((), then 5mg daily () remeron 7.5 qhs zydis 5mg po every other day zydis 5mg qhs colace 100mg daily albuterol nebs prn asa 81mg daily mv daily trazadone 25mg po qhs prn nystatin s&s qid x 5 days () senna 2 tabs qhs fleet enema pnr bisacodyl prn mom prn acetaminophen 650mg q4h prn . allergies: nkda discharge medications: expired discharge disposition: expired discharge diagnosis: primary: septicemia secondary to urinary tract infection community acquired pneumonia hypoxic respiratory distress multifactoral encephalopathy aaa rupture acute blood loss anemia secondary: chronic blood loss anemia discharge condition: expired discharge instructions: expired followup instructions: expired procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more insertion of endotracheal tube transfusion of packed cells percutaneous (endoscopic) jejunostomy [pej] diagnoses: other iatrogenic hypotension urinary tract infection, site not specified acute posthemorrhagic anemia diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled acute kidney failure, unspecified severe sepsis atrial fibrillation personal history of malignant neoplasm of bronchus and lung other persistent mental disorders due to conditions classified elsewhere acute respiratory failure pneumonitis due to inhalation of food or vomitus blood in stool septic shock methicillin susceptible pneumonia due to staphylococcus aureus other septicemia due to gram-negative organisms metabolic encephalopathy hyperosmolality and/or hypernatremia other and unspecified coagulation defects abdominal aneurysm, ruptured foreign body in larynx edema idiopathic normal pressure hydrocephalus (inph) 

Answer: The patient is high likely exposed to