Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: left sided weakness major surgical or invasive procedure: intubation for airway protection history of present illness: 73 year-old left-handed man with a history of hypertension, recurrent dvt on warfarin (also on daily baby aspirin), dyslipidemia who presents today with onset of left-sided weakness and language difficulties since ~10:30 am. he was reportedly in his usual state of health this morning, when his wife left to run errands. the patient reportedly had coffee with an acquaintance and returned home at ~10:30 am to go to his pt appointment at 11:00 am. his wife returned at 10:45 am, and found him awake, lying on the floor next to the bed. his speech was somewhat non-sensical and garbled. his left side appeared weak. there was no sign of trauma, and the patient had apparently lowered himself down to the floor. ems was called and arrived at 11:25 am. there was a discrepancy of blood pressure in each arm: 180/80 on right, 120/80 on left. pulse was 64, respiratory rate was 18, fingerstick reportedly normal. he received a 250 cc of normal saline en route to . in addition to the wife's description, he was also reported to have a left facial droop. he was noted to be bradycardic on ekg with no st-t changes. a code stroke was called on arrival to . the patient was in ct when the neurology team arrived, and scanning clearly showed a right thalamic hemorrhage with intraventricular extension, and the code stroke was cancelled. past medical history: -htn -recurrent lower extremity dvt on anti-coagulation, last reportedly (others in , ) -dyslipidemia -gerd -arthritis -gout -cervical disc disease, s/p surgery -knee surgery, unclear laterality -nephrolithiasis -glaucoma social history: retired office worker, living home with wife. rare alcohol use and reportedly a non-smoker. family history: -no stroke -mother and father died in 80s of an mi, no known thrombophilia physical exam: on admission: vitals: t 97.6 f bp 151/104 p 49 rr 15 sao2 99 ra general: nad, well nourished heent: nc/at, sclerae anicteric, mmm, no exudates in oropharynx neck: supple, no nuchal rigidity, no bruits lungs: clear to auscultation cv: bradycardic, regular rhythm, no mmrg abdomen: soft, non-tender, distended, bowel sounds present ext: warm, no edema, pedal pulses appreciated skin: no rashes neurologic examination: mental status: awake and initially alert, but then increasingly somnolent and inattentive over course of interaction, language deficits impair ability to relay history, but cooperative with exam where able, initially minimally fluent, with dysarthric speech, no paraphasic errors, simple comprehension and repetition initially intact; appears to have left-sided neglect cranial nerves: optic disc margins sharp; visual fields appear full to confrontation and blink to threat. pupils equally round and reactive to light, 3 to 2.5 mm bilaterally. right gaze deviation but can voluntarily get over to left. facial sensation intact bilaterally. perhaps mild left nlff, but facial movement normal and symmetric. hearing intact to finger rub bilaterally, though a bit reduced on left. palate elevates midline. tongue protrudes midline, no fasciculations. trapezii full strength bilaterally. motor: normal bulk , with perhaps increased tone in the left leg. + pronator drift. no tremor. d t b we fif ip q h ta edb right 5 5 5 5 5 5 5 5 5 5 5 5 5 left 4 4 5 4- 4- 4 4 5 4+ 5 5 5 5 *******please note that the patient evolved to a fairly dense left hemiplegia within the hour of presentation.**** sensation: patient has difficulty responding to questions regarding laterality of sensation; answers are non-sensical. on yes/no questioning, he seems to indicate that the left arm and leg have less sensation to light touch and pinprick. he withdraws the left-side less briskly to noxious. the other modalities of sensation are difficult to assess given language deficits and evolving somnolence. reflexes: b t br pa pl right 2 2 2 3 0 left 2 2 2 2 0 toes were upgoing bilaterally. coordination: unable to perform as patient's increasing somnolence interfered with his ability to participate. gait: deferred due to hemiplegia. in the day of discharge: pt fluctuates his mental status, remain with significant aphasia and poor comprehension, incapable to follow complex commands, however he can understand and follow simple commands. pt presents dense left hemiplegia. pertinent results: 12:24pm blood wbc-5.9 rbc-4.23* hgb-13.2* hct-37.4* mcv-89 mch-31.3 mchc-35.4* rdw-13.2 plt ct-212 02:15am blood glucose-109* urean-12 creat-0.7 na-139 k-3.6 cl-106 hco3-26 angap-11 12:24pm blood alt-22 ast-21 ld(ldh)-203 ck(cpk)-93 alkphos-95 totbili-0.4 12:24pm blood ctropnt-<0.01 05:30pm blood ctropnt-<0.01 02:15am blood ctropnt-<0.01 12:24pm blood albumin-4.0 02:15am blood calcium-8.5 phos-2.7 mg-1.8 cholest-147 02:15am blood triglyc-131 hdl-42 chol/hd-3.5 ldlcalc-79 12:24pm blood tsh-2.9 12:24pm blood pt-29.0* ptt-36.6* inr(pt)-2.9* 05:55am blood wbc-9.4 rbc-4.68 hgb-14.7 hct-41.8 mcv-90 mch-31.4 mchc-35.1* rdw-13.4 plt ct-313 07:00am blood wbc-10.3 rbc-4.66 hgb-14.3 hct-41.3 mcv-89 mch-30.7 mchc-34.7 rdw-13.4 plt ct-293 06:50am blood wbc-10.6 rbc-4.59* hgb-14.1 hct-40.6 mcv-88 mch-30.7 mchc-34.8 rdw-13.3 plt ct-292 07:15am blood wbc-9.0 rbc-4.39* hgb-13.5* hct-38.7* mcv-88 mch-30.8 mchc-34.9 rdw-13.3 plt ct-257 03:15pm blood wbc-8.6 rbc-4.22* hgb-13.5* hct-37.4* mcv-89 mch-32.0 mchc-36.1* rdw-13.4 plt ct-242 02:04am blood wbc-9.4 rbc-4.19* hgb-13.1* hct-37.0* mcv-88 mch-31.2 mchc-35.2* rdw-13.4 plt ct-247 05:55am blood neuts-67.6 lymphs-22.4 monos-7.5 eos-2.2 baso-0.4 06:50am blood neuts-72.1* lymphs-18.6 monos-7.8 eos-1.3 baso-0.2 05:55am blood plt ct-313 05:55am blood pt-14.9* ptt-30.2 inr(pt)-1.3* 07:00am blood plt ct-293 07:00am blood pt-15.1* ptt-30.0 inr(pt)-1.3* 06:50am blood pt-14.3* ptt-26.7 inr(pt)-1.2* 07:15am blood plt ct-257 05:55am blood glucose-111* urean-33* creat-0.7 na-138 k-4.3 cl-102 hco3-24 angap-16 07:00am blood glucose-113* urean-35* creat-0.8 na-140 k-4.5 cl-105 hco3-25 angap-15 06:50am blood glucose-116* urean-28* creat-0.7 na-137 k-4.6 cl-102 hco3-25 angap-15 07:15am blood glucose-152* urean-24* creat-0.7 na-139 k-4.2 cl-104 hco3-25 angap-14 03:15pm blood glucose-152* urean-24* creat-0.6 na-138 k-4.3 cl-104 hco3-26 angap-12 02:04am blood glucose-155* urean-23* creat-0.7 na-140 k-4.5 cl-106 hco3-23 angap-16 05:55am blood alt-53* ast-31 alkphos-93 totbili-0.7 06:50am blood alt-60* ast-39 totbili-0.5 02:15am blood ck(cpk)-43 02:15am blood ck(cpk)-73 05:30pm blood ck(cpk)-87 02:15am blood ck-mb-notdone ctropnt-<0.01 02:02pm blood ck-mb-notdone ctropnt-<0.01 11:00am blood ck-mb-notdone ctropnt-<0.01 05:55am blood calcium-9.1 phos-4.3 mg-2.2 07:00am blood calcium-9.3 phos-4.4 mg-2.3 06:50am blood calcium-9.2 phos-4.3 mg-2.1 07:15am blood calcium-9.0 phos-3.4 mg-2.1 02:15am blood %hba1c-6.4* 02:15am blood triglyc-131 hdl-42 chol/hd-3.5 ldlcalc-79 12:24pm blood tsh-2.9 02:49am blood type-art po2-112* pco2-37 ph-7.43 caltco2-25 base xs-0 08:26pm blood type-art po2-395* pco2-41 ph-7.42 caltco2-28 base xs-2 12:34pm blood glucose-98 na-141 k-4.4 cl-105 calhco3-24 ekg: sinus bradycardia. tracing is normal except for rate. nchct at noon: 4 x 2.3 cm parenchymal hemorrhage centered in the right thalamus with intraventricular extension. no hydrocephalus or evidence of herniation. repeat nchct at 2:30 for increased somnolence: 1. increased size of right thalamic hemorrhage. 2. increased amount of intraventricular hemorrhage with extension into the bilateral occipital horns and the fourth ventricle. 4. 4 mm leftward shift of normally midline structures. 5. no evidence for transtentorial herniation. head ct the right thalamic hemorrhage, with intraventricular extension is unchanged in comparison to yesterday's ct. associated edema and compression of the right lateral ventricle are stable. no areas of new hemorrhage are identified. small focus of high attenuation within the sulcus within the left parietal lobe is stable. opacification of the left sphenoid sinus and probable mucosal retention cysts are not significantly changed. impression: 1. unchanged right thalamic hemorrhage, with intraventricular extension and surrounding edema, and compression of the right lateral ventricle. 2. stable small focus of subarachnoid blood within the left parietal lobe, likely representing redistribution of blood. 3. no new hemorrhage identified. mri is more sensitive for the detection of acute stroke. brief hospital course: patient is 73 year-old left-handed man with a history of hypertension, recurrent dvt on warfarin (also on daily baby aspirin), dyslipidemia who presents today with onset of left-sided weakness and language difficulties since ~10:30 am. on general examination, he was hypertensive and bradycardic. his initial examination was notable for a fluent aphasia, left-sided neglect, and worsening left-sided weakness in an upper motor neuron pattern of distribution. he was initially anti-gravity on the left, but susequently became plegic. there is also evidence of loss of sensation to light touch and pinprick. inr is 2.9. ct head showed an acute right thalamic hemorrhage with extension into the ventricles, without hydrocephalus or shift. the location of the hemorrhage was suggestive of a non-traumatic etiology; the risk factors of hypertension and anti-coagulation are the most likely etiology. reversal treatment with vit k 10mg iv, profilnine 4 vials with 2 units ffp was done. given worsening exam, he was intubated for airway protection and head ct was repeated which showed increased hemorrhage with ~4mm leftward midline shift. neurosurgery was consulted and evd was considered but patient stabilized with repeat/3rd ct showing no significant changes hence no neurosurgical intervention was undertaken.patient was initially admitted to neuro icu and. he remained bradycardic with hr in 50's and occasionally requiring iv hydralazine to maintain sbp < 160~180. another head ct was performed in hd#2 which again had no significant changes to previous 2 ct's and his exam remained stable as well with l hemiplegia and neglect. he was successfully extubated on hd #3. on (hd#4) around 4~5am, patient developed afib with rvr which was refractory to metoprolol 5mg iv x4 hence dilatizem drip was started after bolus of 25mg iv. ekg showed afib rhythm without st changes and cardiac enzymes were negative including trop <0.01 x3. surface echo showed that he had preserved systolic function (lvef>55%) but both atria were dilated and he had mild pulmonary htn. no thrombus was seen. patient was not deemed to be a candidate for direct cardioversion given that he couldnot be anticoagulated after the intervention. he was started on asa 81mg daily and heparin sc 5000 tid for dvt ppx on and he was transferred to step down while still on diltiazem drip plus oral diltiazem for afib rate control. in the following days he remained stable. medications on admission: -warfarin 5 mg daily -asa 81 mg daily -lescol 80 mg daily -norvasc 10 mg daily -atenolol 50 mg daily -betimol 0.5% drip -protonix 40 mg daily -glucosamine -multivitamin -indocin, recently -lunesta discharge medications: 1. docusate sodium 50 mg/5 ml liquid sig: one (1) po bid (2 times a day). 2. timolol maleate 0.5 % drops sig: one (1) drop ophthalmic daily (daily) as needed for glaucaoma. 3. atorvastatin 10 mg tablet sig: one (1) tablet po daily (daily). 4. aspirin 81 mg tablet, chewable sig: one (1) tablet, chewable po daily (daily). 5. polyvinyl alcohol-povidone 1.4-0.6 % dropperette sig: drops ophthalmic prn (as needed). 6. diltiazem hcl 60 mg tablet sig: one (1) tablet po q6h (every 6 hours). 7. famotidine 20 mg tablet sig: one (1) tablet po bid (2 times a day). 8. senna 8.6 mg tablet sig: one (1) tablet po bid (2 times a day). 9. docusate sodium 100 mg capsule sig: one (1) capsule po bid (2 times a day). 10. bisacodyl 10 mg suppository sig: one (1) suppository rectal daily (daily). 11. lisinopril 10 mg tablet sig: one (1) tablet po daily (daily). 12. nystatin 100,000 unit/ml suspension sig: five (5) ml po tid (3 times a day). discharge disposition: extended care facility: - discharge diagnosis: right thalamic hemorrhage secondary diagnosis: atrial fibrilation discharge condition: stable, fluctuating mental status and very dense left hemiplegia. discharge instructions: you were admitted to this hospital because you had subtle onset of left sided weakness and changes in your mental status. you underwent a headt ct and brain mri which showed signs of hemorrhage in the deep structure of the right side of the brain, called thalamus. you were treating deep venous thrombosis with anticoagulant therapy and you also have hypertension and this combination seems to be the cause of this hemorrhage. you received treatment to revert the anticoagulant therapy. during this admission you presented irregular cardiac rhythm called atrial fibrillation, and this condition is not new to you. you will need to follow up with your cardiology. please contact your primary care doctor or return to the emergency department if you have weakness, loss of consciousness, persistent headache, dizziness, change in vision or any concerning symptoms. followup instructions: provider: ,md (vascular ) provider: (cardiologist ) provider: , md phone: date/time: 3:00 please contact the office to update some of your information md procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube enteral infusion of concentrated nutritional substances diagnoses: esophageal reflux unspecified essential hypertension gout, unspecified atrial fibrillation intracerebral hemorrhage other and unspecified hyperlipidemia acute respiratory failure cerebral edema long-term (current) use of anticoagulants personal history of venous thrombosis and embolism 

Answer: The patient is high likely exposed to