Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: this is a 65 year old man with past medical history notable for coronary artery disease, congestive heart failure, type 2 diabetes, paroxysmal atrial fibrillation who was found in a temporary residence at a local by his attorney who found the patient in his own stool and urine in bed, conscious but disoriented. the patient has not had any clear memory of any preceding symptoms such as chest pain, shortness of breath, seizures, head trauma, chills, fever, melena or bright red blood per rectum. the patient denies any alcohol or illicit drug consumption. he did admit to taking percocet, quantity is unclear. had obtained this percocet from the emergency department. he was given this by the emergency department on after he fell. he tripped at his daughter's house in . chest x-ray was negative for rib fractures. when the patient was found, by report there were no pills left of 45 tablets of percocet and he was brought to the emergency department. yet, the patient only admits to taking two to perhaps five tablets of percocet. he claims that he has been staying at the while he settles a 65 million dollar estate claim. past medical history: notable for coronary artery disease, three vessel disease including 30% left main, 95%, 85% left anterior descending and 100% right coronary artery. also he has a history of congestive heart failure with 10% ejection fraction. type 2 diabetes, paroxysmal atrial fibrillation, an supraventricular tachycardia, splenic hematoma status post inferior vena cava filter placement in secondary to a possible motor vehicle accident, status post motor vehicle accident with multiple rib fractures, prior cerebrovascular accidents and status post right hip arthroplasty, status post motor vehicle accident, also bilateral knee surgery. social history: the patient denies smoking, drinking ever or any illicit substances. he is married. family history: unclear, the patient is not aware of any family history of any diseases. medications on admission: the patient is uncertain but he says he takes one tablet of lasix mg unknown. he takes "a heart medication," and he claims he takes insulin 57 units of some sort of insulin split over two doses daily. physical examination: on examination he had a temperature of 97.2, heartrate 89, blood pressure 130/85, respirations 30 and breathing 95% on room air. in general appearance he was a disheveled, groggy middle-aged man. he was notable for dry tongue, poor dentition with possible bite injury of right upper lip and cheek, notable scleral jaundice but no jugulovenous distension and no adenopathy. heart was mildly tachycardiac on examination. lungs were notable for bibasilar crackles with the left greater than right. the abdomen was obese, soft, nontender. there was no organomegaly. extremities were notable for 1+ pitting and lower extremity edema. they were cold to the touch. the patient was neurologically alert and oriented times three with some groggy speech. he has three beat lateral nystagmus. cranial nerves ii through xii were intact, he moved all extremities. there was noted, however, asterixes and decreased pinprick sensation of both calves bilaterally anteriorly. laboratory data: laboratory studies were notable for white blood cell count of 12.1, hematocrit 48, platelets 162. chem-7 was notable for a sodium of 140, potassium 5.7, chloride 103, bicarbonate 21, bun 82 and creatinine 3.6 with a baseline approximately 1.0 and glucose of 286. calcium was 9.6, magnesium 2.6, phosphate 4.7. his transaminases were found to be markedly elevated. the alt was 1,233, ast 2,119, alkaline phosphatase 92, total bilirubin 4.2, amylase 54, lipase 44, albumin 3.3, creatinine kinase was 1795, mb 21 and mb index 4.2 and troponins were 2.7. urine cultures were also drawn. chest x-rays were showing stable cardiomegaly with increased opacities of both lung bases, suggesting pneumonia. no pleural effusions were present and pulmonary vasculature were unremarkable. right upper quadrant ultrasounds were performed to rule out, obstruction series, normal gallbladder, no cholecystitis, no observable biliary obstruction. hospital course: given the missing percocet and the empty bottle on the patient's bedside as well as the transaminases, the immediate concern was of acetaminophen toxicity secondary to percocet overdose. the patient was immediately put on intravenous acetycysteine with initial loading dose followed by a continuous infusion of the intravenous mucomyst solution. the patient continued to be somewhat tachypneic during this hospital course. he was being admitted to the intensive care unit because of expected toxicity. he was also empirically treated with antibiotics, in particular he was treated with clindamycin, levofloxacin to cover a community-acquired or aspiration pneumonia. the patient was also tachycardiac. he had been given both intravenous and p.o. lopressor in the emergency department. here on the floor he was put initially on the diltiazem drip for rate control. cardiac enzymes were continued to be cycled. he had borderline positive ckmbs and troponins during his course. he was put on lactulose because of his noted asterixis. his inr continued to rise during his hospitalization. he was started on vitamin k, potassium and acute renal failure was believed to be secondary to dehydration given his status and condition when found in his room. he was given boluses of 250 cc of normal saline, smaller boluses were given given the patient's known congestive heart failure with ejection fraction of approximately 10%. however, during the second day of the patient's hospitalization he became increasingly tachypneic and less responsive. he was also found to be hypotensive. extremities were cool on touch. this was believed to be possibly evidence of cardiogenic shock at which point the patient was electively intubated for impending respiratory failure. over the courses of the evening, the patient developed cardiac arrest. it should be noted that prior to that his urine output dropped to 0. the patient's hypotension progressed. a cuff blood pressure was unable to be obtained as well as wide complex bradycardia. he received 1 mg of atropine. dopamine was started 10 mcg/min. asystole followed. cardiopulmonary resuscitation was started. he went into ventricular fibrillation and was shocked at 200 joules and given 1 mg of epinephrine, shocked subsequently at 360 joules with another mg of epinephrine and 360 joules again with 1 mg of epinephrine. he was developed atrial fibrillation with wide complex. he was given amiodarone, bolus of 150 mg. asystole reappeared and cardiopulmonary resuscitation was resumed. the patient was given 1 amp of bicarbonate. his ventricular fibrillation developed. he was given a shock at 360 joules and then returned to sinus rate in the 60s. a femoral arterial line was obtained and the blood pressure was measured to be in the 30s to 40s systolic. vasopressin was started and blood pressure was then in the 70s to 80s systolic with a mean atrial pressure in the 60s. he was given a second amp of bicarbonate and 2 amps of potassium chloride. attempts were made to contact his wife at the time. the patient was pseudostabilized on vasopressor two hours with arterial blood gases which were improving. echocardiogram was obtained to rule out tamponade, there was no tamponade effusion or global akinesis. at 3 am despite maximum persistence the patient developed hypotension and heartrate to the 40s. again he coded and received atropine times two, epinephrine one with improvement in pulse and respiration. vasopressin was discontinued with continuation of levophed and addition of epinephrine. repeat arterial blood gases post the arrest was ph of 7.1, pco2 20 and po2 of 215 on an assist control vent with a title volumes of 600 and positive end-expiratory pressure of 5 and respiratory rate 30 with an fio2 of 50%. the patient's wife was finally contact the subsequent morning and it was her expressed understanding of the patient's wish because he had prior myocardial infarctions that he did not want to be continued on mechanical ventilation. in discussion with the team as well as consultation with the patient's daughter it was decided at that point to cease artificial ventilation. the patient expired at 3:30 pm on . the patient's daughter and the patient's wife were notified. , m.d. dictated by: medquist36 d: 17:32 t: 16:39 job#: procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube cardiopulmonary resuscitation, not otherwise specified diagnoses: other primary cardiomyopathies acidosis congestive heart failure, unspecified acute and subacute necrosis of liver acute kidney failure, unspecified atrial fibrillation acute respiratory failure poisoning by other opiates and related narcotics 

Answer: The patient is high likely exposed to