Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: pea arrest major surgical or invasive procedure: intubation, central venous line placement, arterial line placement history of present illness: 23 y.o. male w/ h.o. benzo and heroin transferred from hospital after being found down in pea arrest likely heroin od. . per ed signout pt was found by passer by this am unconcious, ems was called and found him in pea arrest, they coded him in the field for 20 minutes which included 3 rounds of epinephrine, atropine, 8 of narcan. pt was taken to widden ed where he coded again and reecived 2 rounds of medications. he was found to be in v. fib and received 1 shock, he was then noted to be irregular wct thought to be a. fib with aberrancy. he was ?intubated at widden with an abg notable for acidosis. he was then placed on artic sun and transferred to ed. in the ed he was noted to be sinus tach, overbreathing the vent. unclear but ems thought pt did have somewhat of a gag at widden. in the ed he had an a-line placed, has 2pivs. . in discussion with the family it is unclear if this was an attempt on his life versus taking an excessive amount of heroin. per grandmother pt usually is someone who takes substances in excess. per grandmother pt has also been very depressed, 2 weeks he overdosed at his grandmother's house though it is not clear if this was an attempt. his girlfriend apparently overdosed last week. pt was interested in sobriety from his heroin habit and was being seen at connections where his urine was negative for heroin last week. . on the floor, he was noted to be on a cooling blanket, sinus tachycardia to 110s and bp 120s on levophed. pt already intubated with rr 14 which was increased to 20, peep 5, fio2 50%. . review of systems: unable to obtain. . past medical history: past medical history: (per ed and family member) h.o. heroin overdose heroin/benzo abuse bipolar disorder depression . social history: pt has history of drug abuse per family members, recently overdosed on heroin 2 weeks ago. was attending ?detox at connections with clean urine test 1 week ago. girlfriend also overdosed a week ago. family history: noncontributory physical exam: vitals: t:96.3, bp:11 p: r: 18 o2: general: caucasian male intubated in nard. heent: pupils fixed and dialted (received atropine in the field), sclera anicteric, mmm neck: supple, jvp not elevated, no lad lungs: coarse bs noted bilaterally. cv: s1, s2, distant when compared to course breath sounds. tachycardic, no murmurs, gallops or rubs abdomen: soft, non-distended, bowel sounds present ext: cool to touch on cooling blanket. no edema noted. pertinent results: labs on admission: 01:10pm blood wbc-12.3* rbc-4.43* hgb-13.8* hct-41.4 mcv-93 mch-31.1 mchc-33.3 rdw-13.5 plt ct-171 01:10pm blood neuts-89* bands-1 lymphs-5* monos-2 eos-0 baso-0 atyps-1* metas-0 myelos-2* 01:10pm blood pt-26.6* ptt-42.2* inr(pt)-2.6* 01:10pm blood glucose-154* urean-26* creat-2.3* na-141 k-3.1* cl-104 hco3-16* angap-24* 01:10pm blood alt-4815* ast-3156* ld(ldh)-4542* ck(cpk)-7605* alkphos-77 totbili-0.6 01:10pm blood ck-mb-117* mb indx-1.5 01:10pm blood totprot-5.3* albumin-3.4 globuln-1.9* calcium-7.0* phos-5.3* mg-2.1 03:21am blood osmolal-298 01:08pm blood glucose-151* lactate-10.1* na-140 k-3.0* cl-105 imaging: head mri : findings: i concur with dr. comprehensive assessment. indeed, as noted by dr. , widespread diffusion and t2 signal abnormalities of the brain indicate extensive hypoxic ischemic injury. there is no susceptibility abnormality within the brain seen to suggest the presence of a hemorrhage. there is mild prominence of the anterior communicating artery, as noted on both the axial t2 and susceptibility weighted images (see series 4, image 11 of the t2-weighted scan series), suggesting a possible aneurysm v. vascular loop. this finding could be evaluated more definitively with an mr angiogram if the patient's clinical condition permits this additional examination. there is high t2 signal within all paranasal sinuses, particularly the sphenoid and ethmoid sinuses, as well as the mastoid sinuses, which could relate to the intubated status of the patient. there is an artifact overlying the midline parietal bone area, which may relate to an eeg lead. conclusion: diffuse hypoxic ischemic injury. see above report for other findings. head ct : impression: 1. diffusely increased cerebral edema, with further sulcal effacement and slight narrowing of the lateral ventricles. no evidence of herniation at this time. 2. evolution of hypodensities in the basal ganglia and internal capsules, consistent with infarct. 3. no intracranial hemorrhage. brief hospital course: 23 yom s/p pea arrest in setting of heroin overdose transferred from hospital. pt was admitted to micu and cooled x24 hours c continuous eeg monitoring. pt did require paralysis as he was shivering when initially cooled. head ct on admission concerning for cerebral edema and pt was started on hyperosmolar saline c goal osmolality of 320. upon rewarming pt was found to be posturing, neuro agreed that there was ongoing seizure activity and started keppra and dilantin. repeat head ct as well as mri showed worsening cerebral edema and ischemic changes. neurology and micu attending met c family to discuss prognosis and the decision was made to convert pt to cmo. pt was terminally extubated on and passed away shortly after. medications on admission: depakote er 500mg daily discharge medications: discharge disposition: expired discharge diagnosis: discharge condition: discharge instructions: followup instructions: procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified insertion of endotracheal tube arterial catheterization diagnoses: acidosis urinary tract infection, site not specified acute and subacute necrosis of liver acute kidney failure, unspecified atrial fibrillation other convulsions depressive disorder, not elsewhere classified hypopotassemia opioid type dependence, continuous acute respiratory failure other specified cardiac dysrhythmias pneumonitis due to inhalation of food or vomitus cardiac arrest ventricular fibrillation suicide and self-inflicted poisoning by analgesics, antipyretics, and antirheumatics poisoning by heroin sedative, hypnotic or anxiolytic dependence, continuous 

Answer: The patient is high likely exposed to