Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: ventricular fibrillation arrest major surgical or invasive procedure: arterial line placement intubation history of present illness: mr is an 85m with a history of a-fib (on home coumadin) and idiopathic dilated cardiomyopathy, who presented to hospital s/p ventricular fibrillation arrest. he collapsed in his house and had been down for 2 minutes when ems arrived. he was received 120j biphasic and amiodarone 150mg iv followed by continuous infusion by ems for presumed v fib/vt and subsequently arrived at . cooling protocol was initiated sometime between presentation at ~ 7:30 am, then was medflighted to . he had reached 89f by presentation to at 10:30 am on . wife reports that patient came from bathroom this am at 7am and lied down. his wife tried to talk to him, but he was unresponsive. she contact ems ~ 5 minutes afterwards, and ems arrived in 2 minutes. they felt rhythm was ventricular fibrillation, and he received 1 shock and revereted to a narrow complex rhythm. on presentation to ed, patient had been intubated and cooled to 89 degrees fahrenheit. he received vecuronium iv and was started on fentanyl and midazolam for sedation. on arrival to ccu, vital signs were 52, 218/109, t:33 celsius, o2 sat 100% on 100% fio2. he was transiently on a nitroglycerin gtt which was turned off once patient became hypotensive to 70s systolic, and pressure returned to 130s systolic. he was experiencing several runs of wide-complex tachycardia, appearing monomorphic on telemetry, and received another 150mg iv amiodarone. unable to perform review of systems, as patient is sedated. past medical history: 1. cardiac risk factors:: (-)diabetes, (-)dyslipidemia, (+)hypertension 2. cardiac history: - dilated cardiomyopathy with lvef 25% by lv gram - atrial fibrillation, not on anticoagulation per osh records 3. other past medical history: copd (per osh discharge, obstructive and restrictive disease on pfts) social history: married, no children. retired. family history: no family history of early mi, otherwise non-contributory physical exam: admission exam- vs:bp 210/109, hr 92, fio2 100%, sats 100% gen: intubated, sedated, unresponsive to all stimuli ears, nose, mouth and throat: eyes pinpoint and nonreactive, r eye slightly open, l eye closed neck: no bruits respiratory: clear anteriorly cardiac: prominent heave, irregular, no murmurs abdominal / gastrointestinal: no bowel sounds, soft, nondistended genitourinary: (wnl, foley catheter) extremities / musculoskeletal: warm, no edema skin: wnl pertinent results: labs on admission: . 10:12am blood wbc-9.3 rbc-4.06* hgb-11.2* hct-34.2* mcv-84 mch-27.6 mchc-32.8 rdw-15.5 plt ct-209 10:12am blood neuts-84.1* lymphs-11.0* monos-3.4 eos-1.4 baso-0.2 10:12am blood pt-20.6* ptt-29.7 inr(pt)-1.9* 10:12am blood glucose-157* urean-14 creat-0.8 na-142 k-4.5 cl-109* hco3-24 angap-14 04:01am blood alt-103* ast-107* ld(ldh)-306* ck(cpk)-414* alkphos-99 totbili-0.6 03:14pm blood calcium-8.1* phos-1.7* mg-2.5 04:07am blood hapto-96 . cardiac: 10:12am blood ctropnt-0.08* 03:14pm blood ck-mb-17* mb indx-6.5 ctropnt-0.40* 04:01am blood ck-mb-28* mb indx-6.8* ctropnt-0.18* 10:12am blood ck(cpk)-127 03:14pm blood ck(cpk)-263* 04:01am blood alt-103* ast-107* ld(ldh)-306* ck(cpk)-414* alkphos-99 totbili-0.6 . microbiology bl cx - pending. . radiology: ct head (): impression: no acute intracranial hemorrhage, edema or mass. . ct head (): impression: no acute intracranial hemorrhage, edema, or herniation. . mri head (): impression: 1. no evidence of acute intracranial abnormality. evaluation of the posterior right parietal lobe is obscured by metallic artifact. 2. small vessel ischemic disease. echo the left atrium is moderately dilated. the right atrium is moderately dilated. there is mild symmetric left ventricular hypertrophy with normal cavity size. there is moderate regional left ventricular systolic dysfunction with focal akinesis of the inferior and inferolateral walls and hypokinesis of the distal inferior septum. tissue doppler imaging suggests an increased left ventricular filling pressure (pcwp>18mmhg). right ventricular chamber size and free wall motion are normal. the ascending aorta is mildly dilated. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. mild (1+) aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. there is no mitral valve prolapse. trivial mitral regurgitation is seen. the estimated pulmonary artery systolic pressure is normal. triival pericardial effusion. impression: suboptimal image quality. regional left ventricular systolic dysfunction c/w multivessel cad. elevated left ventricular filling pressures. mild aortic regurgitation. ef 30-35% most recent labs 03:15am blood wbc-14.2*# rbc-3.82* hgb-10.4* hct-31.5* mcv-83 mch-27.3 mchc-33.1 rdw-16.6* plt ct-197# 03:15am blood pt-37.9* ptt-42.2* inr(pt)-4.1* 03:15am blood glucose-105 urean-16 creat-0.7 na-144 k-4.4 cl-115* hco3-20* angap-13 03:15am blood alt-33 ast-39 ld(ldh)-293* alkphos-97 totbili-0.6 03:15am blood calcium-7.4* phos-4.3 mg-1.9 03:28am blood type-art po2-112* pco2-42 ph-7.27* caltco2-20* base xs--7 brief hospital course: in summary, mr is an 85m with htn, hl, non-obstructive cad, a-fib (on home coumadin), dilated cardiomyopathy (ischemic vs idiopathic, lvef 25%), copd, who presented s/p vf arrest at home. # v fib arrest: unclear etiology, though likely idiopathic dilated cardiomyopathy. no evidence for acute ischemic process, cardiac enzymes elevated in setting of recent defibrillation. patient underwent arctic sun cooling protocol for neuroprotection, then was rewarmed. he was intubated on transfer to and was admitted to the ccu. tte showed ef 30-35% with regional lv systolic dysfunction c/w multivessel cad. he was place on aspirin. his coumadin was held initially for elevated inr then restarted. he was monitored on tele and was in afib. # seizure: patient developed seizures after rewarming, neurology was consulted. 24-hour eeg monitoring was initiated. placed on ativan gtt and valproic acid initially, but added propofol for frequent seizures. likely in setting of anoxic brain injury s/p cardiac arrest (unclear time before pt was resuscitated), however, mri head was not confirmatory. in the morning of was noted to have unequal pupils and given manitol and sent for emergent head ct to r/o herniation, which was negative. pt continued to have seizure activity while on propofol- valporic acid was started then keppra. patient was unresponsive and continued to have abnormal pupils. extensive discussion was had with patient??????s wife expressing pt's likely poor outcome, pt was made dnr/dni. # rhythm - slow afib w pvcs. pt started on bb for rate control, but stopped due to bradycardia likely cns origin. bradycardia worsened after starting propofol and valproic acid, hr in 40s-50s. # elevated inr ?????? no known liver disease/nutritional deficiency. lfts downtrending. complete dic labs neg. concerned regarding gut function, got 1mg iv vitamin k # htn - bp was very labile, initially hypertensive. transiently used nitro gtt, then labetalol gtt. then on became hypotensive and required levo gtt to maintain pressure. once pt was levo was stopped and pressure fell to not detectable within 20 minutes #. diminished bowel sounds ?????? kub unremarkable. no bowel sounds on exam. ntg placed to suction. meds were given iv. on - after pt's wife changed pt to . morphine gtt was started. levo stopped. pt extubated at 1610. pt had asystolic arrest at 1630. admitted and attending notified. wife decline autopsy. medications on admission: #. fluticasone/salmeterol 50/250 #. furosemide 40mg daily #. losartan 50mg daily #. doxepin 25mg tid #. omeprazole 20mg daily #. albuterol prn discharge medications: none discharge disposition: expired discharge diagnosis: ventricular fibrillation with anoxic brain injury statis epilepticus idopathic dialated cardiomyopathy causing systolic heart failure, acute on chronic atrial fibrillation chronic obstructive pulmonary disease respiratory failure discharge condition: death with asystolic arrest discharge instructions: none followup instructions: none procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more arterial catheterization diagnoses: other primary cardiomyopathies unspecified essential hypertension chronic airway obstruction, not elsewhere classified atrial fibrillation other and unspecified hyperlipidemia cardiac arrest anoxic brain damage grand mal status long-term (current) use of anticoagulants ventricular fibrillation encounter for palliative care acute on chronic systolic heart failure 

Answer: The patient is high likely exposed to