Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: transfer from osh s/p vf arrest and stents to 100% occluded lad. major surgical or invasive procedure: cardiac catheterization where he was found to have a 100% occluded ostial lad and 40% rca and 40% cx. he was stented to the lad x 2 with taxus stents. history of present illness: mr. is a 61 year old man who was doing yard work today when he noted chest pressure and asked friend for . his friend came back to find mr. on the ground. he started cpr and 7 minutes later, ems arrived and found patient cyanotic, pulseless and not breathing. he was intubated and the monitor showed asystole. he was given epi/atropine. he was in vfib vs. v tach. he was shocked x 10, given amiodarone and lidocaine and went into sinus tach. he was brought to the ed of and given 325, plavix, heparin gtt and taken to cath lab where he was found to have a 100% occluded ostial lad and 40% rca and 40% cx. he was stented to the lad x 2 with taxus stents. he remained hemodynamically stable and intubated. an iabp was placed at the end of the case. post procedure he was started on integrillin, but this was stopped due to blood in his ng tube. he was subsequently transferred to for further management. past medical history: hypercholesterolemia ? htn social history: works as service tech for . lives with wife. smoked 1-1.5 ppd and quit 15 years ago. occasionally drinks. had 2 beers on day of admission family history: father died of mi, brother with mi (both in 50s). no dm. physical exam: pe: af 132/84 95 84 20 100% o2 sats vent: ac 600x20 peep 5 fio2 1 gen: intubated man rib in nad heent: clear op, mmm, perrl neck: supple, no lad, no jvd cv: rr, nl rate. nl s1, s2. no murmurs, rubs or lungs: cta, bs bl, no w/r/c (ant) abd: soft, nt, nd. nl bs. no hsm ext: no edema. trace pulses throughout. no cyanosis or clubbing. skin: no lesions neuro: sedate and intubated pertinent results: 11:18pm type-art po2-207* pco2-28* ph-7.38 total co2-17* base xs--6 10:38pm glucose-145* urea n-26* creat-1.2 sodium-140 potassium-5.1 chloride-112* total co2-15* anion gap-18 10:38pm ck(cpk)-3348* 10:38pm ck-mb-124* mb indx-3.7 ctropnt-3.07* 10:38pm calcium-7.8* phosphate-1.5* magnesium-2.0 10:38pm wbc-15.0* rbc-4.91 hgb-15.4 hct-42.7 mcv-87 mch-31.3 mchc-36.0* rdw-12.7 10:38pm plt count-252 07:07pm type-art temp-35.0 po2-345* pco2-34* ph-7.35 total co2-20* base xs--5 intubated-intubated 07:07pm freeca-1.08* 07:02pm pt-14.8* ptt-89.9* inr(pt)-1.3* 10:38am blood ck(cpk)-8630* 06:06pm blood ck(cpk)-* 04:12am blood ck(cpk)-* 11:43am blood ck(cpk)-* 04:17am blood ck(cpk)-8788* 06:50am blood ck(cpk)-2124* 06:50am blood glucose-109* urean-20 creat-0.8 na-143 k-3.8 cl-109* hco3-23 angap-15 . 10:38am blood ck-mb-222* mb indx-2.6 ctropnt-1.95* 06:06pm blood ck-mb-250* mb indx-2.5 ctropnt-1.63* 04:12am blood ck-mb-165* mb indx-1.4 ctropnt-1.40* 11:43am blood ck-mb-107* mb indx-1.0 06:25am blood ck-mb-10 mb indx-0.1 ctropnt-0.62* . 07:45am blood calcium-8.3* phos-2.4* mg-2.0 . 05:04pm blood type-art temp-38.0 o2 flow-2 po2-73* pco2-37 ph-7.44 calhco3-26 base xs-0 . echo ef 40-45% conclusions: the left atrium is normal in size. left ventricular wall thicknesses and cavity size are normal. there is mild regional left ventricular systolic dysfunction with hypokinesis of the distal half of the septum, distal anterior wall, and apex. the remaining left ventricular segments contract normally. no apical aneurysm or thrombus is seen. right ventricular chamber size and free wall motion are normal. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. no aortic regurgitation is seen. the mitral valve leaflets are structurally normal. there is no mitral valve prolapse. mild (1+) mitral regurgitation is seen. the estimated pulmonary artery systolic pressure is normal. there is no pericardial effusion. impression: regional left ventricular systolic dysfunction c/w cad (mid-lad lesion). mild mitral regurgitation. . mra brain findings: this study is of limited quality due to extensive contamination by t1 hyperintense fat. allowing for this limitation, no definite vascular pathology is seen. . cxr the increasing opacification at both lung bases can be explained by atelectasis and dependent edema, though pneumonia cannot be excluded, since there is persistent engorgement of mediastinal veins and upper lobe pulmonary vessels. small left pleural effusion is present. no pneumothorax. mild tubulated narrowing of the subglottic and upper trachea has been evident since extubation on and could be due to that or previous intubation as well as enlargement of the thyroid gland. . 5:08 am urine source: catheter. **final report ** urine culture (final ): escherichia coli. >100,000 organisms/ml.. proteus mirabilis. >100,000 organisms/ml.. sensitivities: mic expressed in mcg/ml __________________ escherichia coli | proteus mirabilis | | ampicillin------------ <=2 s <=2 s ampicillin/sulbactam-- <=2 s <=2 s cefazolin------------- <=4 s <=4 s cefepime-------------- <=1 s <=1 s ceftazidime----------- <=1 s <=1 s ceftriaxone----------- <=1 s <=1 s cefuroxime------------ 4 s <=1 s ciprofloxacin---------<=0.25 s <=0.25 s gentamicin------------ <=1 s <=1 s imipenem-------------- <=1 s levofloxacin----------<=0.25 s <=0.25 s meropenem-------------<=0.25 s <=0.25 s nitrofurantoin-------- <=16 s piperacillin---------- <=4 s <=4 s piperacillin/tazo----- <=4 s <=4 s tobramycin------------ <=1 s <=1 s trimethoprim/sulfa---- <=1 s <=1 s . 5:43 am sputum source: endotracheal. gram stain (final ): >25 pmns and <10 epithelial cells/100x field. 4+ (>10 per 1000x field): gram negative rod(s). 2+ (1-5 per 1000x field): gram positive cocci. in pairs and clusters. 1+ (<1 per 1000x field): gram positive rod(s). consistent with corynebacterium and propionibacterium species. respiratory culture (preliminary): sparse growth oropharyngeal flora. haemophilus influenzae, beta-lactamase positive. heavy growth. beta-lactamase positive: resistant to ampicillin. staph aureus coag +. moderate growth. staphylococcus species may develop resistance during prolonged therapy with quinolones. therefore, isolates that are initially susceptible may become resistant within three to four days after initiation of therapy. testing of repeat isolates may be warranted. gram negative rod(s). moderate growth. further identification to follow. gram negative rod #4. sparse growth. sensitivities: mic expressed in mcg/ml _________________________________________________________ staph aureus coag + | gram negative rod(s) | | clindamycin-----------<=0.25 s erythromycin----------<=0.25 s gentamicin------------ <=0.5 s levofloxacin----------<=0.12 s oxacillin------------- 0.5 s penicillin------------ =>0.5 r . brief hospital course: assessment: the patient is a 61 year old man transferred from osh for vt/vf arrest and subsequent intubation and transfer s/p cath revealing 100% lad lesion which was stented. upon arrival, he was placed on a cooling protocol with arctic sun machine with body temp cooled to 94f for 18 hours to preserve long term neurologic functioning. he was subsequently extubated and mental status improved daily. . # cad: pt s/p stent to 100% occluded lad. he has no known history of cad but does have a hx of hyperlipidemia. we monitored on tele throughout admission. we continued , statin, bb (and titrated up as bp tolerated), plavix and started ace-i once bp improved. he was temporarily on integrillin gtt s/p cath for 18 hours. echocardiogram on hd #1 revealed 40-45% ef with hypokinesis of the distal half of the septum, distal anterior wall, and apex. regarding his mi, he did quite well post-stent with little ectopy on telemetry. he was extubated on hd #1 and remained cp free. troponins steadily decreased throughout the admission (from a peak of 3.07). he will follow at cardiology. since he has two taxus stents, he will need plavix with aspirin for at least 6 months. . # vt/vf: his vt and vf was probably secondary to his large mi due to a 100% occluded lad. he had no residual ectopy since catheterization except for occasional pvcs. we monitored on tele and continued bb. echo results as per above. he had no further vt after his stents to the lad. . # neurologic status: pt s/p cooling protocol with arctic sun machine to maintain total body temp of 34c for 18 hours upon admission (which was within 4-6 hour window where cooling has been shown to have best effects). this has been demonstrated to improve long term neurologic deficits secondary to hypoxic damage in patients who suffer vf arrest and were subsequently recussitated. per protocol, we monitored electrolytes q6 hours while on machine. we maintained sedation while paralyzed with vecuronium bromide 0.05-0.14 mg/kg/hr iv (used to titrate to no shivering). he tolerated this protocol well and regained some residual neurologic function by hd #1. he was seen by neurology who recommended mra brain which was unremarkable. his mental status and overall neurologic function improved throughout this hospitalization and he passed a speech and swallow eval and ambulated well, conversed coherently and could follow commands well by time of discharge. some slugglishness and a resting posture which resembles decerebrate posturing has been noted in the past day. nonetheless, the patient remains neurologically intact, and has follow up planned with behavioral neurology (dr. at . . # chf: pt had interstitial pulmonary edema on cxr on arrival. by , cxr read "the increasing opacification at both lung bases can be explained by atelectasis and dependent edema, though pneumonia cannot be excluded, since there is persistent engorgement of mediastinal veins and upper lobe pulmonary vessels. small left pleural effusion is present." he remained afebrile yet sputum cx grew out h. influenza and s. aureus. he was started on vancomycin and zosyn which was subsequently switched to levofloxacin when culture data became available. . # elevated ck: this was presumed to be due to the cooling protocol (see above). cks trending down at time of discharge to 2124 from peak of 11,444. this should be followed closely at rehab. . # uti: on hd #2 urine grew pansensitive e.coli and proteus and ciprofloxacin was initiated. when the pt was placed on levofloxacin for his sputum culture data (see below) on , this was discontinued. his urine cultures can be repeated at rehab in the coming week. he denies dysuria and his urine appears clear at time of discharge. . # respiratory: pt was intubated and sedated upon admission. after the cooling protocol (see above), we weaned ventillation without event and he was extubated on hd #1 and remained off the vent for the duration of the admission. o2 mask was weaned completely by hd #2. . # hypothyroidism: levothyroxine continued throughout admission . # hyperlipidemia: holding statin until ck is <1000 (2124 and trending down on day of discharge). recommend 40mg po atorvastatin daily and rechecking ck daily once restarting. . # code: full . # comm: with sons (and wife in am) . # disp: to rehab . medications on admission: levoxyl lipitor discharge medications: 1. clopidogrel 75 mg tablet sig: one (1) tablet po daily (daily). 2. insulin regular human 100 unit/ml solution sig: one (1) injection asdir (as directed). 3. heparin (porcine) 5,000 unit/ml solution sig: one (1) injection tid (3 times a day): until pt ambulating frequently. 4. aspirin 325 mg tablet sig: one (1) tablet po daily (daily). 5. pantoprazole 40 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po q24h (every 24 hours). 6. levofloxacin 500 mg tablet sig: one (1) tablet po q24h (every 24 hours) for 12 days. 7. lisinopril 20 mg tablet sig: two (2) tablet po daily (daily). 8. metoprolol succinate 100 mg tablet sustained release 24hr sig: two (2) tablet sustained release 24hr po daily (daily). 9. levothyroxine 50 mcg tablet sig: one (1) tablet po once a day. discharge disposition: extended care facility: rehab unit at - discharge diagnosis: primary: vt/vf arrest s/p mi due to 100% occluded lad anoxic brain injury secondary: htn hyperlipidemia discharge condition: stable discharge instructions: please take all medications as prescribed. seek medical attention immediately if you experience new symptoms including shortness of breath, chest pain, loss of sensation, dizziness, fatigue, nausea/vomiting, fainting, palpitations, or any other new symptoms. follow up as per below. followup instructions: call pcp , md to arrange a follow up appointment within the next 2 weeks. cardiology group (. they will call with an appointment time. please verify this. dr. (neurology) @ 10:30am building () . ( procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more nonoperative removal of heart assist system diagnoses: acidosis urinary tract infection, site not specified congestive heart failure, unspecified unspecified acquired hypothyroidism percutaneous transluminal coronary angioplasty status acute myocardial infarction of anterolateral wall, initial episode of care other and unspecified hyperlipidemia acute respiratory failure pneumonitis due to inhalation of food or vomitus anoxic brain damage cardiogenic shock methicillin susceptible pneumonia due to staphylococcus aureus rhabdomyolysis pneumonia due to hemophilus influenzae [h. influenzae] 

Answer: The patient is high likely exposed to