Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: confusion/aphasic major surgical or invasive procedure: none history of present illness: 81y/o f with h/o htn, af, mr who was last seen today by family at 5:30pm conversant and ambulating around house. patient then found 2 hours later by family unable to speak, sitting on bed w/ declining mental status. they called 911 and was then transferred to ed. here, in ed, patient noted to have nihss=10 as the patient was globally aphasic, with a left sided gaze preference, and did not cross midline on oculocephalic testing. also w/ hypertension. code stroke was called. she was moving ue equally and antigravity and there was a decreased response to stim in her right extremity. a stat head ct showed a massive subarachnoid hemorrhage with intraventricular extension in b/l lateral ventricles, third and fourth ventricles with no apparent sign of hydrocephalus except for ex vacuo. there was no midline shift or mass effect at this point. prior to the head ct, the patient was intubated by the ed due to her sats in 70's in the setting of decreasing alertness and depressed mental status. ngt placed, given nimodipine, dilantin, started nipride then shut off, then started labetalol then shut off, finally started on versed and propofol. neurosurgery consulted and recommended drain placement and icp monitoring/mannitol administration. given recommendations and assessment (severe brain trauma) from neurology and neursurgical service, family declined aggressive measures and moved for dnr/dni, comfort measures only. at this point neurosurgery and neurology signed off. patient was admitted to micu for extubation while family arrived and patient given last rites. past medical history: pmhx: af on coumadin, htn, mr, r knee arthritis (h/o childhood inflammatory jt disease), phtn (?: echo ' - "nml lvef"). has never had mammogram or colonoscopy. social history: lives with son. etoh/tob. family history: no known cancer. father died of cirrhosis/esld. physical exam: p: 62, bp: 106/41, r: 17, sats 97% on cmv vt 500 x 18, fio2 100% x peep 5 gen: intubated, not responsive to voice, heent: nc/at, pupils reactive from 3mm to 2mm, ett in place. cv: irreg, irreg rhythm, 2/6 sem at llsb, no r/g pulm: mechanical breath sounds, o/w cta b/l, no w/r/r abd: bowel sounds present, flat, soft, non distended ext: no c/c, +edema in both lower ext 1+ pitting, skin: b/l onychomycosis of nails, chronic venous stasis changes of skin in both lower extremities. vasc: 2+ dp/pt b/l neuro: absent corneal reflexes, pupils reactive 3mm to 2mm b/l, left gaze deviation, decorticate posturing to pain, upgoing babinski b/l. pertinent results: 10:34pm type-art o2-100 po2-449* pco2-29* ph-7.45 total co2-21 base xs--1 aado2-246 req o2-48 intubated-intubated vent-controlled 10:34pm glucose-191* lactate-2.1* k+-3.5 09:00pm glucose-173* urea n-33* creat-1.1 sodium-144 potassium-3.5 chloride-109* total co2-21* anion gap-18 09:00pm calcium-8.6 phosphate-3.0 magnesium-1.3* 09:00pm wbc-14.0* rbc-3.99* hgb-11.8* hct-35.4* mcv-89 mch-29.6 mchc-33.3 rdw-14.2 09:00pm neuts-93* bands-0 lymphs-3* monos-4 eos-0 basos-0 atyps-0 metas-0 myelos-0 09:00pm plt smr-low plt count-148* 09:00pm pt-12.6 ptt-20.3* inr(pt)-1.1 08:06pm glucose-163* urea n-33* creat-1.2* sodium-139 potassium-8.1* chloride-103 total co2-19* anion gap-25* 08:01pm ck(cpk)-92 08:01pm ctropnt-<0.01 08:01pm ck-mb-notdone 08:01pm phosphate-3.4 magnesium-1.6 08:01pm wbc-11.1* rbc-4.76# hgb-13.9# hct-41.9# mcv-88# mch-29.3 mchc-33.3 rdw-14.3 08:01pm neuts-79.7* lymphs-16.5* monos-3.1 eos-0.4 basos-0.4 08:01pm plt count-187 lplt-1+ 08:01pm pt-12.7 ptt-22.7 inr(pt)-1.1 08:01pm fibrinoge-371 = = = = = = = = = = ================================================================ chest (portable ap) 8:34 pm technique: single ap portable supine chest. findings: an endotracheal tube is in place with tip terminating 3.6 cm from the carina. there is stable cardiomegaly. the aorta is unfolded and prominently calcified. there is diffuse hazy opacity over the lung fields bilaterally, suggestive of layering bilateral pleural effusions. in addition, there is slight prominence of the pulmonary vasculature and scattered peripheral septal lines consistent with mild congestive heart failure. no focal areas of pulmonary parenchymal consolidation or air bronchograms are identified. no pneumothorax. the osseous structures demonstrate osteopenia. impression: 1) status post endotracheal intubation. endotracheal tube in satisfactory position. 2) cardiomegaly, layering bilateral pleural effusions, and mild congestive heart failure. no definite evidence of pneumonia, although followup chest x- ray is recommended after treatment to assess resolution. = = = = = = = = = = ================================================================ ct head w/o contrast 7:55 pm technique: non-contrast head ct. findings: there is massive subarachnoid hemorrhage, with extension into the lateral ventricles. hemorrhage fills the third ventricle and the fourth ventricle, as well as the basilar cisterns. there is brain edema with loss of /white differentiation, predominantly in the inferior aspect of the cerebral hemispheres. there is hydrocephalus involving the third and lateral ventricles, as well as herniation. region of hypodensity within the left posterior frontal and parietal lobe extending into the left occipital lobe is residua of prior infarction. there is no shift of the normally midline structures. slight osseous irregularity is visible along the left inferior frontal/superior temporal region. dense calcification of the supraclinoid carotid arteries is visible. the paranasal sinuses and orbits are grossly unremarkable. there is also dense calcification of the vertebral arteries bilaterally. impression: massive subarachnoid hemorrhage, likely from ruptured aneurysm. hydrocephalus, brain edema, and herniation. findings were discussed with dr. at the time of the exam at 6:25 p.m., . brief hospital course: 81y/o f with htn, afib, and mr p/w massive sah. 1. sah: ct head findings were discussed with family by neurology and neurosurgery service. they recommended drain placement and administration of manitol and blood pressure medication for icp control. they conveyed to family that patient was brain dead. given the findings, explanation from the services and the procedures to be done, the family declined any further aggressive measures and wished to make their family member dnr/ and to proceed to extubation with morphine for comfort once other family members arrived and last rites given. patient was then transferred to micu intubated, kept on mechanical ventilation until last rites given and family members arrived. was then extubated, fentanyl and versed were stopped and patient was then started on morphine for comfort. on day 2 of admission patients neurological status improved, morphine drip was stopped. stroke team and neurosurgery team discussed the improved neurological status of patient with family and readdressed the option of drain placement. family declined and expressed their wishes of continuing patient with comfort measures only. by day 3 of admission patients respiratory status began to decline with several episodes of apnea and her oxygenation began to decline as well with sats in 60's. she became more unresponsive. patient went into respiratory arrest and by 2pm on mrs. passed away. 2. fen: npo 3. code: dnr/dni, comfort measures only. medications on admission: lipitor, lisinopril discharge medications: none discharge disposition: expired discharge diagnosis: massive sub arachnoid hemorrage hydrocephalus brain edema herniation discharge condition: none, patient expired on discharge instructions: none followup instructions: none procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube diagnoses: obstructive hydrocephalus unspecified essential hypertension atrial fibrillation subarachnoid hemorrhage compression of brain cerebral edema 

Answer: The patient is high likely exposed to