Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: nifedipine / penicillins / codeine / sulfonamides / bactrim / lisinopril / naproxen attending: chief complaint: unresponsiveness major surgical or invasive procedure: intubation history of present illness: 53 yo rhw pmh anterior communicating artery aneurysm s/p clipping in , hypertension and heavy alcohol use who presents after decreased responsiveness earlier this morning. history provided by patient's husband and hcp . was in her usual state of health yesterday evening and this morning when she got out of bed to go to the bathroom. upon her return to bed, she told her husband that she was not feeling right. she said that she needed to go back to the bathroom but then was having difficulty getting up again. at that point, he noted she had urinary incontinence, slurred speech. her husband called 911 immediately. upon arrival to ed, patient was unresponsive with decerebrate posturing. she was intubated and taken for head ct which showed a large basal ganglia bleed on the left with spread to the lateral, third and fourth ventricles. nsurg was consulted and did not think surgical intervention would improve outcome. past medical history: htn cerebral aneurysm as above chronic neck pain history of - tear history of angioedema social history: per recent omr note in , current smoker and recent h/o of alcohol. per eldest daughter, patient drinks heavily. has seven healthy children, divorced and remarried. pt is on disability. family history: father died of mi. physical exam: t- 99.0 bp- 232/104 hr- 72 rr- 14 97 o2sat ra gen: lying in bed unresponsive, nad heent: nc/at, moist oral mucosa cv: rrr, nl s1 and s2, no murmurs/gallops/rubs lung: clear to auscultation bilaterally abd: soft, nontender ext: no edema neurologic examination: mental status: not responsive to verbal or tactile stimuli. decerebrate posturing bilaterally. cranial nerves: pupils 4mm bilaterally and fixed, nonreactive to light. does not blink to threat. wall eyed, no eye movements with oculocephalic maneuver. intact corneals b/l. grimaces and postures extremities to nasal tickle. motor/sensory: normal bulk. increased tone legs>arms throughout. no spontaneous movements only decerebrate posturing to noxious stim. reflexes: +2 and symmetric throughout except in ankles unable to illicit due to tonic plantar flexion. toes mute bilaterally coordination/gait/romberg: unable pertinent results: 10:25pm sodium-145 potassium-3.5 10:25pm magnesium-3.6* 09:03pm type-art temp-38.3 po2-216* pco2-33* ph-7.56* total co2-30 base xs-8 -assist/con intubated-intubated 09:03pm freeca-1.03* 05:43pm glucose-267* urea n-13 creat-1.3* sodium-135 potassium-2.4* chloride-92* total co2-23 anion gap-22* 05:43pm calcium-9.0 phosphate-2.4*# magnesium-1.5* 05:43pm osmolal-320* 05:43pm wbc-8.7 rbc-3.33* hgb-11.6* hct-32.7* mcv-98 mch-34.7* mchc-35.3* rdw-15.1 05:43pm plt count-200 05:43pm pt-13.6* ptt-24.1 inr(pt)-1.2* 05:43pm fibrinoge-219 04:30pm urine hours-random 04:30pm urine bnzodzpn-neg barbitrt-neg opiates-neg cocaine-neg amphetmn-neg mthdone-neg 03:28pm urine hours-random 03:28pm urine gr hold-hold 03:28pm urine color-yellow appear-clear sp -1.015 03:28pm urine blood-neg nitrite-neg protein-neg glucose-tr ketone-tr bilirubin-neg urobilngn-0.2 ph-8.0 leuk-neg 01:57pm urea n-15 creat-1.3* total co2-30 01:57pm estgfr-using this 01:57pm tot bili-1.1 01:57pm lipase-32 01:57pm triglycer-241* 01:57pm tsh-1.5 01:57pm asa-neg ethanol-neg acetmnphn-neg bnzodzpn-neg barbitrt-neg tricyclic-neg 01:57pm wbc-6.3 rbc-3.74* hgb-12.8 hct-36.9 mcv-99* mch-34.2* mchc-34.6 rdw-14.8 01:57pm plt count-216 01:57pm pt-12.7 ptt-24.1 inr(pt)-1.1 01:54pm glucose-164* na+-140 k+-2.8* cl--92* ct angiography of the head history: altered mental status. question cerebral bleed. technique: non-contrast head ct scan followed by ct angiography of the intracranial vasculature. comparison study: ct scan. findings: there is a huge hemorrhage within the left basal ganglia/internal capsule and thalamic region extending into the mesencephalon. there is a prominent degree of intraventricular hemorrhage as well extending throughout the supratentorial ventricular system and likely the aqueduct of sylvius as well. the hemorrhage creates prominent mass effect upon the left lateral ventricle and there is a prominent degree of rightward subfalcine herniation. the hyperdense right-sided posterior temporal/parietal lesion, felt to be a meningioma on prior studies is redemonstrated. there are streak artifacts arising from aneurysm clips in the region of the anterior communicating artery. there is a left frontotemporal craniotomy. the ct angiogram discloses several small vascular structures along the lateral aspect of the left lentiform nucleus portion of this hemorrhage. the remaining vasculature appears within normal limits. obviously, vascular detail is obscured by streak artifacts arising from the anterior communicating artery aneurysm clips. conclusion: a few small vascular structures along the lateral aspect of the large left lentiform nucleus component of this hemorrhage raise the question for an underlying vascular lesion, such as a vascular malformation. portable ap cxr: findings: the left lateral aspect of the lung field is excluded from the film. the cardiomediastinal silhouette is stable. the mediastinal and hilar contours are unremarkable. the lungs are clear. there is no pleural effusion. a newly placed endotracheal tube terminates approximately 4.4 cm above the carina in good position. soft tissues and osseous structures appear grossly unremarkable. impression: endotracheal tube in standard position. no acute cardiopulmonary process. brain flow scan: radiopharmeceutical data: 25.0 mci tc-m neurolite (); history: evaluate for brain death. findings: following injection of tracer, multiple planar images of the brain were obtained and show no evidence of perfusion to the cerebral hemispheres on both flow and static imaging. the perfusion abnormalities noted above are consistent with no brain activity. impression: no cerebral flow or activity within the brain. brief hospital course: given the severity of the intracranial hemorrhage, the patient was admitted to the neurologic icu for further evaluation and management. her prognosis was poor. her exam was notable for loss of brain stem reflexes, including pupillary, oculocephalic, corneals, and gag. cold calorics initially performed on showed no response from the eyes. the patient subsequently failed an apnea test on , but the decision was made to repeat the test with the brain death examination on , after metabolic disturbances including hypernatremia were appropriately corrected. with these metabolic disturbances appropriately corrected, the patient failed again failed the apnea test on . a brain scan showed no cerebral flow or activity in the brain. care was subsequently withdrawn and the patint expired later that evening. medications on admission: hydralazine 50mg tid pepcid 20mg qd metoprolol 225mg hctz 25mg qd benadryl 25mg q6h discharge medications: not applicable discharge disposition: expired discharge diagnosis: intracranial hemorrhage discharge condition: expired discharge instructions: not applicable followup instructions: not applicable md procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours diagnoses: intracerebral hemorrhage hyperosmolality and/or hypernatremia 

Answer: The patient is high likely exposed to