Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: cc - transfer from osh for liver failure ?amiodarone toxicity major surgical or invasive procedure: liver biopsy paracentesis history of present illness: hpi - this is a 69 y/o male with a pmh significant for cad, copd, paf, dm who presented to his pcp in with symptoms of decreased appetite, fatigue, and increased abdominal girth over the last six months. there, he was found to have increased ascites, worsening liver function, and increased le edema. he was sent to for further evaluation. on initial admission to , had elevated lfts with alk p in 500's, and ast/alt in 200-300's range. amiodarone was d/c'd at that point, and medrol 8 mg was started. gi followed pt while in-house and ct of abd done showed hyperdense liver that was c/w possible amiodarone toxicity. mri was c/w ascites and gallstone w/o bile duct dilitation. he had an extensive workup for elevated lfts, and workup for autoimminue causes, viral causes, and hemachromatosis were all negative. during his admission, the lfts began to trend down to 140-150 range, 200 range for alk phos. pt was d/c'd to rehab on high-dose steroids. however, he represented to with abd fullness and sob ascites and anasarca. lfts during the second admission were unchanged with alk phos in the 400 range, with acute renal function worsening (cr 2.4 -> 3.3). this second admission was also significant for an increase in wbc from 20 to 38, pt remaining afebrile. upon review of incomplete old records that were obtained, it appears that the patient was started on amiodarone 200mg in following a valve replacement surgery c/b atrial fibrillation. he remained on this dose of amiodarone until it was d/c'd at . ros - positive for abdominal fullness, decreased appetite, recent diarrhea, and fatigue - negative for any h/a, vision changes, cough, sob, cp, abd pain, n/v, melena/hematochezia/brbpr, tremors past medical history: pmh - 1. cad 2. chf 3. paf 4. copd on chronic steroids 5. s/p bioprstetic valve placement at , complicated by afib, for which he was started on amiodarone 6. s/p right cea 7. dm on insulin 8. diverticulitis s/p partial colectomy 9. cri social history: sh - lives at home with his wife. used to smoke (30 pack-year history), but has not smoked in 30 years. occasional etoh (1 drink/2 weeks) previously, but no etoh recently. no ivda. one tatoo on right arm, done about 45 years ago. family history: fh - father died of an mi, mother passed at 80. no known family h/o liver diseases. physical exam: pe on admission: vs - t 96.3, bp 123/68, hr 78, rr 18, sats 99%/ra general - fatigued-appearing, pleasant gentleman, ao x 3, nad heent - nc/at, perrl, eomi. no scleral icterus. mm dry, op wnl neck - supple, no jvd, no thyromegaly chest - diffuse, high-pitched expiratory wheezes throughout cv - rrr s1 s2 normal, soft sm at sternal border abd - distended, firm but not tense, nontender to palpation; soft bs, alternating dullness and tympany to percussion, no discernable fluid wave; sign negative ext - 2+ pitting edema b/l up to mid-thigh; pulses 2+ b/l neuro - pt ao x 3, no asterixis pertinent results: cbc 07:32pm blood wbc-37.2*# rbc-4.95 hgb-13.9* hct-42.1 mcv-85 mch-28.1 mchc-33.0 rdw-17.5* plt ct-161 07:10am blood wbc-38.6* rbc-4.86 hgb-13.3* hct-41.5 mcv-85 mch-27.4 mchc-32.1 rdw-18.0* plt ct-166 07:00am blood wbc-38.9* rbc-4.72 hgb-12.8* hct-40.9 mcv-87 mch-27.2 mchc-31.4 rdw-17.7* plt ct-130* 06:30am blood wbc-35.4* rbc-4.07* hgb-11.0* hct-35.0* mcv-86 mch-27.1 mchc-31.5 rdw-17.9* plt ct-83* 12:25pm blood wbc-35.3* rbc-4.01* hgb-11.0* hct-34.0* mcv-85 mch-27.4 mchc-32.3 rdw-17.8* plt ct-79* diff 07:32pm blood neuts-97.3* bands-0 lymphs-1.7* monos-0.9* eos-0 baso-0 06:30am blood neuts-96.1* bands-0 lymphs-2.2* monos-1.6* eos-0.1 baso-0 coags 07:32pm blood pt-17.2* ptt-34.1 inr(pt)-2.0 07:32pm blood plt smr-normal plt ct-161 07:10am blood plt ct-166 05:19pm blood pt-15.9* ptt-33.7 inr(pt)-1.7 07:00am blood pt-16.1* ptt-35.0 inr(pt)-1.7 07:00am blood plt ct-130* 06:30am blood pt-17.2* ptt-38.3* inr(pt)-2.0 06:30am blood plt smr-low plt ct-83* 12:25pm blood plt ct-79* chemistry 07:32pm blood glucose-249* urean-118* creat-3.1*# na-136 k-4.8 cl-102 hco3-19* angap-20 07:10am blood glucose-158* urean-121* creat-3.1* na-136 k-5.3* cl-104 hco3-20* angap-17 04:50pm blood k-4.9 07:00am blood glucose-235* urean-132* creat-3.3* na-136 k-4.8 cl-103 hco3-19* angap-19 06:30am blood glucose-142* urean-132* creat-3.4* na-137 k-4.6 cl-104 hco3-18* angap-20 07:10am blood alt-123* ast-84* ld(ldh)-379* alkphos-374* totbili-1.7* 07:00am blood alt-108* ast-75* alkphos-345* totbili-1.5 06:30am blood alt-83* ast-63* ld(ldh)-281* alkphos-291* totbili-2.1* 07:32pm blood albumin-2.3* calcium-8.6 phos-5.0* mg-2.1 07:10am blood albumin-2.3* calcium-8.7 phos-5.3* mg-2.2 iron-30* 07:00am blood totprot-4.9* albumin-2.1* globuln-2.8 calcium-8.5 phos-5.5* mg-2.1 06:30am blood albumin-2.7* calcium-8.3* phos-5.8* mg-2.1 07:10am blood caltibc-100* ferritn-762* trf-77* 07:10am blood ammonia-81* 07:00am blood osmolal-336* misc 07:00am blood hcv ab-negative 07:00am blood pep-no specifi 07:00am blood -negative 07:00am blood hbsag-negative hbsab-negative hbcab-negative hav ab-negative 07:32pm blood tsh-2.8 urine 01:12am urine osmolal-500 11:40pm urine u-pep-pnd ife-pnd 04:37pm urine osmolal-429 01:12am urine hours-random urean-871 creat-90 na-137 04:37pm urine hours-random urean-761 creat-43 na-less than 01:12am urine eos-negative 01:12am urine rbc->50 wbc-* bacteri-few yeast-none epi- transe- 06:44pm urine rbc-0-2 wbc->50 bacteri-few yeast-none epi-0 04:37pm urine rbc-0 wbc->50 bacteri-few yeast-none epi-0 06:44pm urine blood-lg nitrite-neg protein-tr glucose-neg ketone-neg bilirub-neg urobiln-neg ph-5.0 leuks-mod 04:37pm urine blood-mod nitrite-neg protein-neg glucose-neg ketone-neg bilirub-neg urobiln-neg ph-5.0 leuks-mod 01:12am urine color-yellow appear-clear sp -1.016 06:44pm urine color-yellow appear-hazy sp -1.015 04:37pm urine color-yellow appear-clear sp -1.014 peritoneal fluid 01:58pm ascites totpro-0.8 albumin-less than 01:58pm ascites wbc-66* rbc-26* polys-33* lymphs-24* monos-0 macroph-43* brief hospital course: this is a 69 y/o man with pmh significant for cad, dm, copd, a fib, who was on chronic amiodarone since for rhythm control of a fib, who was transferred to from osh after an extensive workup for liver disease of unknown origin. workup there was notable for hyperdense liver, ascites; and negative for any autoimmune, viral/infectious, hemachromatosis causes. pt also here with arf on cri. 1. liver failure/renal failure - most likely multifactorial, also likely that it was amiodarone toxicity, based on his prior workup. workup at osh for autoimmune and infectious causes for liver failure were all negative. his hepatitis serologies were also negative during this hospital stay. therapeutic tap on negative for sbp at the osh with normal cytology. his paracentesis at this hospital was also negative for sbp. he underwent a transjugular liver biopsy which showed significant amiodarone toxicity with severe fibrosis and incomplete nodule formation. his venous pressure measurements revealed a hepatic venous pressure gradient of 28mmhg consistent with portal hypertension. it is unclear as to whether the liver biopsy findings were entirely related to amiodarone toxicity or whether the amiodarone toxicty was superimposed on a background of cirrhosis. risk factors for cirrhosis include diabetes and nash. he was initially continued on the high-dose steroids that he was transferred here with, but was started to be weaned off as they had no clear benefit. a ruq u/s with dopplers showed no liver masses, + ascites, findings c/w cirrhosis and portal hypertension, hepatofugal flow in splenic and portal veins, nl flow in hepatic veins. given worsening creatinine, he was started on octreotide/midodrine for suspected hepatorenal syndrome, along with albumin. in addition, he developed an enterococcal uti for which he was begun on ampicillin . an ng tube was placed for tube feeds as the patient had poor po intake secondary to decreased appetite. given his liver failure and probable hepato-renal syndrome, he was evaluated for potential liver transplant by dr. . based on the patient's cardiac risk factors/cardiac history and his poor functional status, it was decided that the patient would not be a good liver transplant candidate. on he became hypotensive and was transferred to the micu for further management. following transfer to the micu, the patient received 2u prbc, 2u ffp, and vit k with stabilization of his blood pressure. his renal function/coagulopathy continued to worsen, attributed to hepato-renal syndrome. following a family meeting on , the decision was made not to pursue dialysis, as the patient is not a liver transplant candidate. per this family meeting, other medical treatment (octreotide, midodrine, antibiotics, albumin) was decided to be continued, although the patient was made dnr/dni from a full code on admission. his blood pressures stabilized while in micu and he was transferred back to the floor as cmo per patient's and family's wishes. he eventually expired on . medications on admission: meds (on transfer) protonix 40mg po qam flagyl 500mg po tid (started ) medrol 8mg po bid insulin glargine 15 u qhs advair 2puffs inh combivent 2puffs inh fosamax 90mg po qweek discharge disposition: expired discharge diagnosis: expired discharge condition: expired procedure: enteral infusion of concentrated nutritional substances closed (percutaneous) [needle] biopsy of liver percutaneous abdominal drainage transfusion of other serum diagnoses: urinary tract infection, site not specified long-term (current) use of steroids cirrhosis of liver without mention of alcohol diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled acute kidney failure, unspecified hepatorenal syndrome portal hypertension chronic airway obstruction, not elsewhere classified atrial fibrillation long-term (current) use of insulin heart valve replaced by transplant cardiac rhythm regulators causing adverse effects in therapeutic use 

Answer: The patient is high likely exposed to