Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins attending: chief complaint: transfer for exacerbation of chronic cirrhosis major surgical or invasive procedure: none history of present illness: pt is a 41 y/o man w/ a hx of alcoholic cirrhosis c/b ascites, portal hypertension, and hepatic encephalopathy who presented w/ an exacerbation of his liver failure over the past 2 months. he also had an episode of s. viridans bacteremia in that was treated w/ 4wk of vancomycin and 2wk of gentamycin. he was admitted to an outside hospital on w/ bacteremia as above, w/ prerenal azotemia, and w/ complaints of sob and fatigue. he was found during his last admission to have a large r pleural effusion along with elevated lfts. he was treated empirically with avelox at the outside hospital and was given diuretics. a thoracentesis was planned but did not occur his elevated inr. he was transferred to for further management of his acute on chronic liver failure and for w/u of its etiology. upon arrival, the patient was mildly confused taking long time periods to answer questions and needing some redirection to focus on the question at hand. according to the patient, his liver function has been declining for approximately the past 3 months. he reports that he has had increasing edema in his le despite treatment and that he has been increasingly jaundiced of late. he states that he has not been able to think as well as he used to think. he denies any cp, sob, n/v, diarrhea, ha, palpatations, or pruritis. he denies any recent viral illness or sick contacts. has not traveled recently and has not has had any of his medications changed recently. he denies taking any herbal supplements. past medical history: 1. alcoholic cirrhosis c/b ascites and encephalopathy 2. s viridans bacteremia s/p 4wk vanco 2wk gent 3. arf aminoglycoside toxicity social history: pt is married w/ 2 children. he lives in and works as a salesman for staples. he has a hx of alcohol abuse w/ his last drink in . he denies smoking, drug use, or tattoos. family history: pt w/ diabetes in his mother and father. father died of "kidney and pancreas problems". physical exam: gen: jaundiced appearing man lying in bed in nad heent: eomi, perrla, mmm, o/p clear, + icterus skin: + jaundice, - rashes cv: rrr, s1/s2 intact, -m/r/g lungs: dullness to percussion w/out bs on the lower half of the r lung, otherwise cta abd: s/nt, distended, -hsm, +bs, mild asterixis ext: -c/c, 2+ pitting edema to the mid-thigh in the le neuro: aaox2 (not date), patient not able to do serial 7s past 86 brief hospital course: 41 y/o man with h/o alcoholic cirrhosis who presented after being admitted to an outside hospital with sob and fatigue. was found to have a r pleural effusion and worsening of his lfts. no tap was performed elevated inr. he had a long hx of cirrhosis with worsening of his condition over the past several months. he presented w/ encephalopathy and severe jaundice with unclear cause of sudden decrease in liver fxn. possible causes included infection, toxin, thrombosis of veins. blood and urine cultures were negative. an us of the liver w/ doppler revealed a cirrhotic liver without focal lesions with nearly no flow within the main and left portal veins, and no detectable flow within the right portal vein. massive varices within the abdomen with evidence of splenorenal shunting. massive splenomegaly. small amount of perihepatic ascites, which was not sufficient to tap. nondistended gallbladder with gallbladder wall edema, indicative of liver disease and right pleural effusion. labs were significant for + and igg. he was followed by the hepatology service who initiated transplant workup. the transplant service was consulted on and a transplant workup was completed. ct of the abdomen demonstrated a cirrhotic liver with no mass lesion demonstrated. patent but narrow caliber portal vessels. thin linear hypodensity within the main portal vein likley representing some nonocclusive thrombus. hepatic veins were patent. features of portal hypertension including splenomegaly, moderate amount of intra-abdominal ascites and portosystemic collaterals were described. he remained in the hospital for management of worsening liver failure with hepatorenal syndrome. he became coagulopathic requiring daily transfusions with platelets, ffp, cryo and prbcs per hepatology recommendation to keep plt>20, inr<4, hct>25, fibrin >150. encephalopathy wax and waned. this was managed with lactulose and rifaximin. he was followed by social work, psychiatry, nutrition and physical therapy. his meld score ranged in the 40s. he did not receive a liver transplant despite being at the top of the list. he was transferrred to the sicu with neurology consultation for worsening encephalopathy. he was intubated. a ct demonstrated a spontaneous subdural hematoma. he was coagulopathic and due to his contraindication to transplant, his family met with the team and decided to make him cmo. he expired on . medications on admission: avelox 400mg aldactone 25 tid ambien prn lasix 20 mvi folate discharge medications: none discharge disposition: home discharge diagnosis: end stage liver disease secondary to alcoholic cirrhosis discharge condition: expried discharge instructions: n/a followup instructions: n/a md, procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more other endoscopy of small intestine insertion of endotracheal tube enteral infusion of concentrated nutritional substances arterial catheterization transfusion of packed cells transfusion of other serum transfusion of platelets transfusion of coagulation factors diagnoses: pneumonia, organism unspecified acute and subacute necrosis of liver alcoholic cirrhosis of liver acquired coagulation factor deficiency portal hypertension acute respiratory failure hepatic encephalopathy subdural hemorrhage other and unspecified alcohol dependence, unspecified hypercalcemia portal vein thrombosis 

Answer: The patient is high likely exposed to