Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: dyspnea major surgical or invasive procedure: intubation left femoral cvl left subclavian cvl arterial line history of present illness: mr. is a 79 year old man with a history of metastatic non-small cell lung cancer who presented to the er today with dyspnea for the past 2 days. he had recently undergone thoracentesis on with 700 cc of fluid drained. he has been on bactrim for treatement of moraxella found on bal culture on . in the emergency department, initial vitals: 97.7 88 132/52 28 90% 4l nc. us and cxr showed a large left-sided pleural effusion. he was seen by ip who performed a thoracentesis at the bedside which drained 2l of bloody fluid. post-thoracentesis cxr showed persistent collapse of left hemithorax. he was treated emperically with levofloxacin for concern for infection. he was then admitted to the oncology floor. past medical history: past oncologic history: - known left lung pulmonary nodule since , followed with serial imaging. - developed dyspnea with exertion, dry cough, left sided chest discomfort and fatigue - : imaging showed left-sided pulmonary mass, mediastinal/hilar adenopathy, left pleural effusion and impending left airway obstrcution - : admitted to for evalution, ct showed complete obstruction of the left upper lobe with post obstructive upper lobe collpase with small to moderate left pleural effuison, paraesophageal lymph node, mulitple prevascular lymph nodes and aortopulmonary lymph nodes. there was also a lytic lesion in the lateral aspect of the left 6th rib and focal lucent area in the right t11 vertebra. - : bronchoscopy and thoracentesis with 700 ccs of ser-sanguinous fluid. the pleural fluid, lymph nodes stations 7, 4r, 4l and 11 showed adenocarchioma post-obstructive pneumonia with moraxella catarrhalis. he was treated with supplemental oxygen and antibiotics (levofloxacin - to complete the course within the next few days). tumor cells on pleural effusion cell block s11- were positive for 31, b72.3 and ck7, and negative for cd68, ttf-1, p63, wt-1 and calretinin. - : pet scan showed fdg avid large left hilar tumor causing compression of the left upper lobe bronchus with lul collapse, extensive fdg avid mediastinal adenopathy a loculated moderate left pleural effusion and extensive fdg avid osseous metastasis. - : mri brain negative for brain mets past medical history: hypertension hypercholesterolemia cad s/p cabg , depressed ef per report ckd with creatinine > 1.5 nephrolithiasis hernia repair social history: lives in with wife; previously in . he worked as a maintenance worker in various roles.quit smoking at age 42. started smoking at age 12 and smoked 1 and pack-per day until age 42. this places him at an approximate 45-pack-year history of smoking. the patient denies chronic alcohol use/abuse. the patient denies significant exposures to asbestos or chemicals in prior work. no exposure to radiation. family history: the patient's father died from unknown causes. mother died from sepsis (toxemia). there is no other history of cancer in the family. physical exam: vs t97.1 bp 110/70 hr 85 rr20 92% on 4l general: alert and oriented, nad heent: no scleral icterus. perrla/eomi. mmm. op clear. neck supple, no lad. cardiac: rr. normal s1, s2. no m/r/g. lungs: decreased breath sounds on the left. thoracentesis drain in place with bloody fluid draining. abdomen: nabs. soft, nt, nd. no hsm extremities: no c/c/e, 2+ dorsalis pedis/ posterior tibial pulses pertinent results: labs: on admission: 09:25am blood wbc-16.7* rbc-3.79* hgb-10.7* hct-32.0* mcv-85 mch-28.3 mchc-33.5 rdw-13.8 plt ct-302 09:25am blood neuts-78.3* lymphs-16.0* monos-4.3 eos-0.9 baso-0.6 09:25am blood pt-13.6* ptt-25.9 inr(pt)-1.3* 09:25am blood glucose-158* urean-47* creat-2.3* na-138 k-4.0 cl-99 hco3-23 angap-20 09:13pm blood alt-51* ast-33 ld(ldh)-355* ck(cpk)-44* alkphos-65 totbili-0.2 during pea arrest: 09:13pm blood wbc-13.6* rbc-2.96* hgb-8.5* hct-27.6* mcv-93# mch-28.8 mchc-30.9* rdw-14.1 plt ct-232 09:13pm blood neuts-50.3 lymphs-43.2* monos-5.1 eos-0.9 baso-0.5 09:13pm blood glucose-309* urean-41* creat-2.1* na-136 k-4.0 cl-110* hco3-10* angap-20 09:13pm blood ck-mb-2 ctropnt-<0.01 09:13pm blood albumin-2.3* calcium-7.8* phos-6.6*# mg-2.0 09:18pm blood type- ph-6.93* comment-green top post-arrest trends: cbc 10:16pm blood wbc-14.0* rbc-2.64* hgb-7.8* hct-24.3* mcv-92 mch-29.5 mchc-32.0 rdw-14.4 plt ct-186 01:49am blood wbc-17.2* rbc-4.37*# hgb-12.8*# hct-37.8*# mcv-87 mch-29.3 mchc-33.9 rdw-14.0 plt ct-209 05:57am blood wbc-15.7* rbc-4.12* hgb-11.9* hct-35.4* mcv-86 mch-28.8 mchc-33.6 rdw-14.1 plt ct-190 02:56pm blood wbc-16.7* rbc-4.11* hgb-12.2* hct-35.3* mcv-86 mch-29.8 mchc-34.6 rdw-14.5 plt ct-178 coags: 10:16pm blood pt-17.3* ptt-31.4 inr(pt)-1.6* 01:49am blood pt-15.5* ptt-28.3 inr(pt)-1.5* 05:57am blood pt-16.2* ptt-29.5 inr(pt)-1.5* 02:56pm blood pt-18.3* ptt-150* inr(pt)-1.7* chem 10: 10:16pm blood glucose-253* urean-40* creat-2.0* na-139 k-3.2* cl-111* hco3-15* angap-16 01:49am blood glucose-254* urean-43* creat-2.1* na-141 k-3.9 cl-109* hco3-16* angap-20 05:57am blood glucose-287* urean-45* creat-2.4* na-139 k-4.3 cl-106 hco3-22 angap-15 02:56pm blood glucose-162* urean-44* creat-2.6* na-140 k-3.9 cl-107 hco3-20* angap-17 10:16pm blood calcium-8.2* phos-8.9*# mg-1.9 01:49am blood calcium-8.0* phos-6.0*# mg-1.6 05:57am blood albumin-2.4* calcium-7.8* phos-5.0* mg-1.6 02:56pm blood calcium-7.9* phos-4.9* mg-1.5* lfts: 10:16pm blood alt-114* ast-115* ld(ldh)-472* alkphos-54 totbili-0.2 01:49am blood alt-231* ast-231* alkphos-90 totbili-0.6 05:57am blood alt-205* ast-198* ck(cpk)-100 alkphos-83 totbili-0.8 imaging: ct chest: 1. at least partially loculated large left pleural effusion, stable in size since study but progressed since . new left pigtail catheter appears appropriately coiled deep within the left costophrenic angle. 2. known left hilar mass causing left bronchial compression with complete collapse of the left upper lobe, stable, and near complete collapse of the left lower lobe, progressed since . 3. lytic lesions involving the left lateral sixth rib and vertebral body t12, most consistent with bony metastatic disease. multiple other bony sites of disease are better evaluated on the pet-ct. post-intubation cxr: the endotracheal tube is in standard placement. large left pleural effusion developed in the setting of left upper lobe collapse is larger now than it was at 1:00 p.m. shifting the mediastinum further to the right and collapsing the remainder of the left lung as before. nasogastric tube ends in the stomach. new right infrahilar consolidation is presumably atelectasis. echo: the left atrium is normal in size. the coronary sinus is dilated (diameter >15mm). left ventricular wall thicknesses are normal. the left ventricular cavity size is normal. left ventricular systolic function is hyperdynamic (ef 75%). the right ventricular free wall is hypertrophied. the right ventricular cavity is dilated with severe global free wall hypokinesis. there is abnormal septal motion/position consistent with right ventricular pressure/volume overload, with marked ventricular interaction. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. mild (1+) aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. there is no mitral valve prolapse. mild (1+) mitral regurgitation is seen. the tricuspid valve leaflets are mildly thickened. moderate to severe tricuspid regurgitation is seen. the tricuspid regurgitation jet is eccentric. there is severe pulmonary artery systolic hypertension. the end-diastolic pulmonic regurgitation velocity is increased suggesting pulmonary artery diastolic hypertension. the main pulmonary artery is dilated. the branch pulmonary arteries are dilated. there is no pericardial effusion. compared with the findings of the prior study (images reviewed) of , severe right ventricular pressure and volume overload with marked ventricular interaction are now present. findings are consistent with acute-on-chronic right ventricular strain. bilateral lenis: impression: findings consistent with deep vein thrombosis within the preliminary reportbilateral popliteal and posterior tibial veins and right peroneal vein. brief hospital course: mr. is a 79 year old man with a history of stage iv nsclc with recurrent pleural effusions and left-sided collapse who presented for dyspnea and was s/p a thoracentesis with 2l pleural fluid removed which was bloody. upon admission, he was sent immediately to radiology for ct chest, and at that time was feeling dyspneic but was in no distress. on arrival back up to the floors, he was found to be hypoxic, and shortly thereafter lost his pulse. a code blue was called. on arrival, chest compressions had been started. rhythm was analzyed and pt was found to be in pea arrest. he was given 2 rounds of epi 1mg with ~ 10mins of cpr, with recovery of pulse. he was intubated and transferred to the unit. on arrival to the icu, vs were temp 96.0 hr 104 bp 118/64 rr 23 o2 sat 67%. vent settings cmv fio2 100% tv 550 rr 20 peep 5. pt appeared mildly uncomfortable and was started on fentanyl/versed. within a few minutes of arrival, he lost pulse and was coded again. he was given 2 amps of bicarb, 1 calcium gluconate, and 1mg epi with return of pulse. he was transfused prbc's. his initial lactate during resuscitation returned at 10.8. bedside echo was performed and showed right sided volume overload with underfilling of lv. bedside bronchoscopy was performed and showed severe extrinsic compression of left bronchus from known hilar mass, but there were no secretions or mucous plugs. he was also started on empiric vancomycin and zosyn in case sepsis was playing any role in his acute decline. he was aggressively resuscitated with iv fluids and phenylephrine and norepinephrine were started to help support blood pressures. he was also transfused 4 units of prbcs for 6pt drop in hct, and empiric anticoagulation was deferred. he stabilized overnight on these supportive measures, and latate trended down. in the morning , heparin gtt was started. formal echo confirmed rigth heart strain with under filling of the left ventricle, and bilateral dvts were found on lenis. he required uptitration on his pressors throughout the morning, suggesting worsening shock. though it was medically indicated due to his hemodynamic instability, the team decided to speak with the family first about goals of care prior to starting lysis therapy. a family meeting was held with the patient's son (hcp), daughter-in-law , dr. from the icu, dr. from oncology, and the rest of the members of the icu team. upon discussion of all risks and benefits of treatment and his overall poor prognosis, the family decided to forgo clot lysis and change his management to comfort focused care. he was started on a morphine drip, pressors were withdrawn, and he was extubated. he passed away peacefully with family at his side around 6:20 pm. medications on admission: atorvastatin 40 mg po daily lorazepam 0.5 mg po bid prn anxiety metoprolol 25 mg po bid nifedipine xl 30mg po daily ntg 0.4 mg po prn chest pain omeprazole 20 mg po daily aspirin 81 mg po daily vitamin d 400 iu po daily discharge medications: expired discharge disposition: expired discharge diagnosis: stage iv lung cancer pea arrest discharge condition: expired discharge instructions: expired followup instructions: expired procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified insertion of endotracheal tube thoracentesis other bronchoscopy arterial catheterization diagnoses: hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage i through stage iv, or unspecified coronary atherosclerosis of unspecified type of vessel, native or graft aortocoronary bypass status chronic kidney disease, unspecified pulmonary collapse other and unspecified hyperlipidemia cardiac arrest malignant neoplasm of bronchus and lung, unspecified encounter for palliative care precipitous drop in hematocrit secondary malignant neoplasm of bone and bone marrow malignant pleural effusion 

Answer: The patient is high likely exposed to