Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: visual disturbance, left sided numbness, left sided weakness major surgical or invasive procedure: cerebral angiogram with mechanical thrombectomy history of present illness: 50-year-old man with history of htn, hld, mi with vfib arrest and rca stent, prior stroke, transferred from osh with new left-sided weakness and numbness starting at 13:30 today. he reports over the past three weeks he has "not felt right." he awoke one day three weeks ago with numbness in his head, jaw, and tongue which was non-lateralizing. he has been mildly fatigued and has had two episodes of left-sided vision loss (binocular) lasting hours at a time, described as everything suddenly going black and then becoming and blurry after that. this last occurred on thursday and still reports some blurry vision in his left visual field. he has had some slurred speech and occasional difficulty finding words during this time which he describes as mild. today at 1:30 pm after carrying a television down stairs he sat down and had sudden onset of numbness and weakness of his left arm and leg and numbness in his left side of his face. he describes his limbs on the left as feeling heavy and had difficulty lifting them antigravity. onset was sudden, possibly with some slurring of speech, and has been gradually improving over the past few hours but not back to baseline. notes from also report episodes of right facial numbness, tingling in his left fingertips. he was admitted to overnight and discharged after his episode of left visual field loss. discharge paperwork stated ct head and carotid ultrasound were negative, and was discharged home without change in medications. upon return to ed today, ct head showed right pca and bilateral cerebellar subacute strokes and small hemorrhage in right pca infarct and was transferred to for further evaluation. past medical history: - cad, rca stent, vfib arrest -htn -hld social history: divorced and lives with two kids. remote tobacco hx, no etoh or drug hx. family history: no history of strokes or mi. physical exam: physical examination on admission gen; lying in bed, nad heent; nc/at, mmm, oropharynx clear neck; no bruits cv; rrr, no murmurs pulm; cta b/l abd; soft, nt, nd extr; no edema neuro; ms; a&ox3, speech fluent. naming, repetition, comprehension intact. misses left side of sentence when trying to read. follows midline and appendicular commands. no apraxia or neglect. cn; perrl 4mm-->3mm, eomi, no nystagmus. left homonymous hemianopia. face sensation intact v1-v3. left nasolabial fold flattening and mild left facial droop. hearing intact to finger-rub b/l. palate elevate symmetrically. scm and trap strong and symmetric. tongue midline. motor; normal bulk and tone, left pronator drift. strength is (r/l) delt , bicep , tricep , wre 4+/5, fe , ff , ip , ham , quad , ta , gastroc . sensory; intact to light touch, no extinction to dss. decreased to pinprick in left leg > arm, intact in face. reflexes; 1+ and symmetric, toes upgoing. coordination; no dysmetria on fnf. rams intact. gait; deferred. pertinent results: labs on admission: 05:05pm blood wbc-10.7 rbc-5.43 hgb-16.9 hct-46.8 mcv-86 mch-31.0 mchc-36.0* rdw-13.2 plt ct-232 05:05pm blood neuts-74.4* lymphs-19.9 monos-4.3 eos-0.5 baso-0.9 05:05pm blood pt-13.5* ptt-23.8 inr(pt)-1.2* 05:05pm blood glucose-116* urean-24* creat-1.4* na-140 k-3.7 cl-103 hco3-24 angap-17 05:05pm blood ck(cpk)-64 05:05pm blood ctropnt-<0.01 05:21am blood ck-mb-2 ctropnt-<0.01 05:21am blood albumin-4.3 calcium-9.6 phos-2.8 mg-1.7 cholest-135 05:05pm blood asa-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 08:16pm blood type-art po2-115* pco2-28* ph-7.58* caltco2-27 base xs-5 01:46pm urine blood-neg nitrite-neg protein-tr glucose-neg ketone-10 bilirub-neg urobiln-neg ph-5.0 leuks-neg 09:00am urine color-yellow appear-hazy sp -1.028 01:46pm urine hours-random creat-194 na-92 k-greater th cl-136 totprot-17 prot/cr-0.1 ct/a head/neck impression: 1. extensive, partly hemorrhagic infarct of the right posterior cerebral artery territory which in conjunction with further infarcts in the cerebellar hemispheres and occlusion of the proximal right vertebral artery is likely embolic in nature. 2. complete occlusion of the proximal right vertbral artery with retrograde or collateral refilling of the vessel down to c6. there is atherosclerotic disease of the intracranial vertebral arteries bilaterally. 3. occlusion of the right posterior cerebral artery at the level of the p3-p4 segment. tte the left atrium is elongated. no atrial septal defect or patent foramen ovale is seen by 2d, color doppler or saline contrast with maneuvers. there is mild symmetric left ventricular hypertrophy with normal cavity size and regional/global systolic function (lvef>55%). right ventricular chamber size and free wall motion are normal. the aortic arch is mildly dilated. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. trace aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. physiologic mitral regurgitation is seen (within normal limits). there is borderline pulmonary artery systolic hypertension. there is no pericardial effusion. there is an anterior space which most likely represents a prominent fat pad. impression: no atrial septal defect identified. nchct impression: unchanged interval appearance of infarcts at the left mca-pca junction, left cerebellar hemisphere, and right pca distribution, the latter with hemorrhagic transformation. the distribution is suggestive of shower emboli, superimposed on posterior circulation insufficiency (as demonstrated on cta). the hyperdensity in right pca region likely represents petechial hemorrhage. mr impression: acute right posterior cerebral infarct, left cerebellar infarct and punctate foci of acute infarcts in the left occipital lobe. acute infarcts are also seen in the right thalamus. blood products are seen in the right posterior cerebral artery infarct. mra neck impression: mra and the fat-suppressed images demonstrate absence of flow signal in the proximal right vertebral artery indicating occlusion or slow flow. there is also appearance of slow flow seen in the distal right vertebral artery in the distal v2, v3 and v4 segments. irregularity of the flow in the distal vertebral arteries in v4 segment indicates atherosclerotic disease. eeg impression: this is an abnormal eeg during wakefulness due to intermittent generalized theta occasionally intermixed with delta slowing of background during wakefulness. this finding may represent an underlying mild encephalopathy or diffuse vascular pathology. no epileptiform activity or electrographic seizures were present. cta head/neck impression: 1. the right pca territory infarction with hemorrhagic transformation, the right superior cerebellar infarction, and the left inferior cerebellar hemisphere infarction are grossly unchanged. 2. increased effacement of the right lateral aspect of the fourth ventricle, without dilatation of the third and lateral ventricles. persistent compression of the temporal and occipital horns, as well as the body, of the right lateral ventricle. 3. persistent occlusion of the right vertebral artery origin, with increased extent of occlusion in the v2, increased narrowing of the v3 segments, and new occlusion of the v4 segment. new filling defects in the basilar artery with an approximately 50% stenosis in its midportion. new right aica narrowing. 4. persistent right distal pca occlusion. persistent right pica nonvisualization. 5. approximately 40% stenosis of the proximal cervical right internal carotid artery due to noncalcified plaque. mr impression: 1. acute infarcts in bilateral posterior cerebral artery territories, bilateral cerebellar hemispheres, bilateral thalami and midbrain. new infarcts are noted in bilateral thalamus and left posterior cerebral artery territory and in the midbrain. consider dedicated mra if necessary. 2. increased hemorrhage in right posterior cerebral artery territory infarct and blood products are also noted in the midbrain infarct. 3. mucosal thickening and fluid are noted in all the paranasal sinuses and mastoid air cells. sputum culture - coagulase positive staphylococcus aureus (pan-sensitive) blood culture - coagulase negative staphylococcus (one bottle) brief hospital course: see above for a more extensive history of present illness. briefly, mr. initially presented with left-sided weakness, left-sided numbness, and dysarthria to an outside hospital. he was transferred to for further care and was outside the window for intravenous tpa. he was additionally on exam found to have a dense left homonymous hemianopia. the investigation at the osh was negative, but at he was found to have a r pca infarction with an associated r vertebral artery occlusion. although not having a known history of af, he did experienced af with rvr early during this hospitalization. he was started on a heparin infusion to prevent further propagation of the clot. while on heparin, he was doing well. he passed a speech/swallow evaluation, and was eating well except for intermittent nausea. his strength significantly improved and was only left with some slight depressed mood and left field cut. on the morning of , he was noted to have a new right facial droop, fixed and dilated pupils bilaterally, and depressed loc with subsequent obtundation. a noncontrast head ct showed no evidence of bleed. he was transferred to the neuro icu for further management with the interventional team notified and activated. he was intubated in the neuro icu as he showed signs of extensor posturing in all extremities and myoclonic jerks. he was brought to the angiography suite where he was found to have a basilar artery occlusion which was removed via mechanical clot retrieval. he was brought back to the neuro icu for further management. his exam improved with regards to his motor function, but several of his brainstem reflexes were lost (pupillary reaction, oculocephalic reflexes) and he did not follow commands or demonstrate awareness. multiple discussions were held with his father and his sister who together opted to deescalate care. he was made cmo on : he was extubated and placed on a morphine infusion for control of pain and air hunger. at this time, he was transferred to the neurology floor for formal cmo level care and was switched to sublingual morphine. his picc line was discontinued prior to discharge. note that after being intubated, the patient was noted to have thick secretions which were sent for culture in the setting of low-grade fevers. he was started on empiric antibiotic treatment with vancomycin, cefepime, and tobramycin. these returned from as coagulase-positive staphylococcus aureus. following the switch to cmo status, his antibiotics were discontinued. medications on admission: -aspirin 325 mg daily -plavix 75 mg daily -atenolol 50 mg -fish oil 1g tid -hctz 25 mg daily -zestril 10 mg daily -zocor 40 mg qhs discharge medications: 1. morphine concentrate 100 mg/5 ml (20 mg/ml) solution sig: 15mg po q4h (every 4 hours). 2. morphine concentrate 100 mg/5 ml (20 mg/ml) solution sig: 10mg po q2h (every 2 hours) as needed for tachypnea rr>25. discharge disposition: extended care facility: nursing and rehabilitation center - discharge diagnosis: coma secondary to ischemic stroke paroxysmal atrial fibrillation pneumonia history of hypertension history of hypercholesterolemia history of coronary artery disease s/p mi and pci discharge condition: discharge condition: eyes closed at baseline, breathing approximately 20 breaths/min, left hemiplegia, some intermittent spontaneous movements on the left that are not purposeful. he does not arouse to painless or painful central or peripheral stimulation. brainstem examination is significant for no pupillary response, no gag, positive corneal reflex and he does breathe on his own. discharge instructions: mr. was admitted to the neurology wards of the on for an acute stroke that left him with new left sided weakness and a left sided visual field deficit. he was started on iv heparin and initially did well, until approximately five-six days later when his neurological examination deteriorated and required the mechanical retrieval of an expanding intracranial clot. he was intubated at that time and transferred to the icu where he remained in a comatose state with poor brainstem reflexes and mechanically ventilated. on he was made cmo by his father and subsequently transferred back to the floor. he was placed on an iv morphine drip initially and transitioned to scheduled doses of sublingual morphine for comfort. he can receive additional . he remains comfortable with these medications, breathing regularly. at this time, he is without iv fluids or peg/ng/og source of nutrition. followup instructions: none procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more insertion of endotracheal tube arteriography of femoral and other lower extremity arteries arteriography of cerebral arteries procedure on single vessel endovascular removal of obstruction from head and neck vessel(s) central venous catheter placement with guidance diagnoses: coronary atherosclerosis of native coronary artery pure hypercholesterolemia unspecified essential hypertension acute kidney failure, unspecified atrial fibrillation intracerebral hemorrhage nausea with vomiting other specified procedures as the cause of abnormal reaction of patient, or of later complication, without mention of misadventure at time of procedure acute respiratory failure old myocardial infarction retention of urine, unspecified personal history of transient ischemic attack (tia), and cerebral infarction without residual deficits encounter for palliative care dysarthria dehydration ventilator associated pneumonia accidents occurring in residential institution hemiplegia, unspecified, affecting unspecified side long-term (current) use of aspirin coma occlusion and stenosis of vertebral artery with cerebral infarction facial weakness dissection of other artery homonymous bilateral field defects long-term (current) use of antiplatelet/antithrombotic 

Answer: The patient is high likely exposed to