Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: confusion major surgical or invasive procedure: none history of present illness: m w/ h/o chf, cad s/p nstemi, afib on dabigatran, brought in by his son due to confusion lethargy and markedly slurred speech for approximately one week. at baseline, he has clear speech, is oriented, and can walk independently for very short distances. per the patient's son, the patient has had no specific complaints, however for the last week and a half he has had slurred speech and dizziness. he was recently changed from plavix to dabigatran. he was seen in the ed for hypoxia and discharged on an increased dose of lasix 120mg daily. his son called his cardiologist's office with a complaint of dizziness. the next day he was noted to be hypotensive with a pressure 70/60. his lasix was held, but he continued to have symptoms. this morning, his son held all his medications except for asa 81 and dabigatran. he has had a couple of mild episodes of epistaxis. he continued to have slurred speech and hypotension, so he was brought in to the ed. . in the ed, initial vs were: 86.9 50 120/48 18, on a non-rebreather. noted to have an intact neuro exam except for incomprehensible speech. started on bair hugger and given warmed fluids. found to have guaiac positive brown stool. sbps down to the 70s, so a r ij was placed. head ct negative. cxr showed chronic r sided effusion. soon after 98% on ra. patient was given vanc, zosyn and 3l iv fluids. he was given 1 unit prbc's. lactate 2.0. . on the floor, patient is coherent, though his speech remains slurred. he denies pain and is unable to provide further ros. past medical history: - cad s/p nstemi in , , and treated medically - chf ef 25% to 30% on tte - aortic stenosis (valve area 0.8cm2, peak gradient 38, mean gradient 24 in tte) - hypertension - chronic kidney failure (baseline cr 1.7-2.0) social history: the patient lives with his wife of 72 years and his son. has a visiting nurse come once per week and nurses aid several times per week. he is able to walk up the stairs by himself, but his son always walks with him. -tobacco history: denies -etoh: drinks occasional etoh at temple services. -illicit drugs: no drugs. family history: there is no family history of premature coronary artery disease or sudden death physical exam: general: alert, oriented to self and month, but not place heent: sclera anicteric, r pupil reactive, dry mucous membranes, oropharynx clear neck: supple, jvp just above the clavicle, no lad lungs: clear to auscultation anteriorly, diminished at right base. cv: regular rate, distant, faint, harsh systolic abdomen: soft, non-tender, non-distended, bowel sounds present, no rebound tenderness or guarding, no organomegaly gu: no foley ext: warm, well perfused, 1+ pulses, mild le edema bilaterally neuro: a&ox1.5, cnii-xii intact except for left eye and dysarthric speech, sensation and strength grossly intact in all extremities pertinent results: 03:30pm blood wbc-5.4 rbc-3.14* hgb-8.6* hct-27.7* mcv-88 mch-27.5 mchc-31.1 rdw-15.6* plt ct-130*# 03:30pm blood pt-35.7* ptt-120.3* inr(pt)-3.6* 03:30pm blood glucose-209* urean-76* creat-2.3* na-137 k-5.1 cl-98 hco3-30 angap-14 03:30pm blood alt-25 ast-36 ck(cpk)-29* alkphos-60 totbili-0.3 03:30pm blood ctropnt-0.03* 03:30pm blood calcium-9.1 phos-4.9* mg-2.5 echo the left atrium is moderately dilated. there is mild symmetric left ventricular hypertrophy. the left ventricular cavity size is normal. there is severe regional left ventricular systolic dysfunction with anteroseptal/anterior hypokinesis/akinesis with apical akinesis/dyskinesis. right ventricular chamber size and free wall motion are normal. the aortic root is mildly dilated at the sinus level. the aortic valve leaflets are severely thickened/deformed. there is aortic valve stenosis which is probably severe (valve area 0.8-1.0cm2) but the gradient is low; since left ventricular systolic function is severely depressed is possible that the aortic valve area would be larger with a higher stroke volume. trace aortic regurgitation is seen. trivial mitral regurgitation is seen. mild to moderate (+) mitral regurgitation is seen. the tricuspid valve leaflets are mildly thickened. severe tricuspid regurgitation is seen. there is severe pulmonary artery systolic hypertension. there is an anterior space which most likely represents a prominent fat pad. brief hospital course: m w/ h/o chf, cad, afib on dabigatran, brought by his son due to confusion lethargy and markedly slurred speech for approximately one week. he was found to have a pneumonia and was treated with antibiotics. he also had an anemia, likely secondary to gi blood loss secondary to decreased clearance of dabigaran in the setting of renal impairment. despite antibiotics, he worsened and the decision was made to transition to comfort-oriented care. he passed away comfortably at 11:05 pm on . medications on admission: - dabigatran 75mg - furosemide 120mg daily (being held) - isosorbide mononitrate 30mg daily - lidocaine 5% patch daily - metoprolol 50mg daily - nitroglycerin 0.4mg sl tabs prn - polyethylene glycol 17gm/dose prn - simvastatin 40mg qhs - aspirin 81mg daily discharge medications: none discharge disposition: expired discharge diagnosis: pneumonia discharge condition: expired discharge instructions: expired followup instructions: none procedure: venous catheterization, not elsewhere classified diagnoses: pneumonia, organism unspecified hyperpotassemia thrombocytopenia, unspecified subendocardial infarction, initial episode of care pure hypercholesterolemia urinary tract infection, site not specified congestive heart failure, unspecified acute kidney failure, unspecified unspecified septicemia iron deficiency anemia secondary to blood loss (chronic) severe sepsis hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage i through stage iv, or unspecified atrial fibrillation coronary atherosclerosis of unspecified type of vessel, native or graft aortic valve disorders chronic kidney disease, unspecified septic shock old myocardial infarction chronic systolic heart failure anticoagulants causing adverse effects in therapeutic use hemorrhage of gastrointestinal tract, unspecified hypovolemia hyperosmolality and/or hypernatremia other and unspecified coagulation defects shock, unspecified hypothermia not associated with low environmental temperature 

Answer: The patient is high likely exposed to