Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: sepsis. major surgical or invasive procedure: none. history of present illness: 78m with parkinson's disease, cad s/p cabg and multiple pci's with stents, chf with ef 30%, osteoporosis, and history of multiple falls who is being admitted to micu for sepsis from presumed urinary source. patient with multiple falls in past, the last 2 days prior to this admission felt to be mechanical (tripped on shoe) with head laceration treated with staples. pt unsure if he loss consciousness during this but knows that he hit his head. denied lh, dizziness, cp, palps, sob, tongue biting, incontinence surrounding event. head ct nml and c-spine with t3 compression fracture, old. patient was cleared in ed and sent home. this a.m. he was getting oj out of refrigerator and lost balance, falling backwards. no vertigo, loc, cp, sob, palps, lh, dizziness preceding event ('unsteady on feet'). he called for help and was brought to the ed this a.m. in ed had temp to 102.8, hr 118, bp 138/42. bp dropped transiently to 92/60 but he was repositioned and bp increased to 115/73. recieved 3l ns in ed, was pancultured, and was given levofloxacin 500, vancomycin 1g, and flagyl 500 (for abd pain and h/o diarrhea x 4 months), asa, ticlid. ce's cycled and monitored on telemetry. patient and family do not want central line placed. also had inferolat st depressions on ekg while tachycardic, and cardiology was curbsided and felt they were rate related. upon arrival to the floor he states that he is comfortable and without pain. this a.m. patient noted dysuria and frequency, no urgency. on ros mouth is dry, he is fatigued, and he has been having diarrhea x 4 months (w/u as outpt unrevealing - improved after stopping ppi). denies fevers, chills, night sweats, weight loss, ha, vision changes, uri sxs, chest pain, sob, palpatations, abdominal pain, melena, hematochezia, nausea, hematemesis. no focal motor or sensory deficits. past medical history: 1. cad s/p cabg and nstemis (: lima-lad, svg->diag, om1, om2, svg->pda); s/p pci of proximal svg-d1-om 1-om2 with des in and pci of svg-om/d with des in . 2. chf: ef 30% 3. parkinson's disease 4. hypercholesterolemia 5. htn 6. h/o tia 7. bladder ca 8. osteoporosis 9. s/p right hip fracture, orif in social history: former prof at . the patient lives in . he lives with his wife on the same street as his daughter. has another daughter who lives in . he smoked until , smoking one pack a day for fifteen years. family history: positive for father, who died of a stroke, mother who had a stroke in her 90s and one brother had disease. physical exam: vitals: 97.6, 102, 137/86, 23, 99%2l gen: diaphoretic well nourished male lying flat in nad, pleasant, communicative. heent: perrl, eomi, anicteric sclera, mm dry, op clear but difficult to visualize d/t inability to open mouth fully with c-collar in place. dysarthric speech with accent. neck: cervical collar in place cardiac: tachycardic, regular rhythm, nl s1 and s2, no mrgs lungs: ctab ant, no wheezes, rhonchi, crackles abd: soft, ntnd, nabs, no hsm, no rebound or guarding ext: warm, 2+ dp pulses, no c/c/e neuro: cn: unable to gaze laterally to left with left eye (may not be cooperating with exam), cn iv,x,vi intact. easy to open eyes bilaterally when closed tight. cn ix, x, xii intact. unable to assess d/t c-collar. motor throughout, sensory intact. + rigidity in ue, l>r. toes upgoing bilaterally. no clonus. no tremor noted. pertinent results: lab data: cbc: 06:40am blood wbc-13.2* rbc-4.17* hgb-11.8* hct-35.3* mcv-85 mch-28.3 mchc-33.4 rdw-16.7* plt ct-279 coags: 09:20am blood pt-13.6* ptt-26.7 inr(pt)-1.2* chemistries: 06:40am blood glucose-100 urean-15 creat-0.8 na-131* k-4.3 cl-99 hco3-22 angap-14 cardiac enzymes: 09:20am blood ctropnt-0.07* 08:04pm blood ck-mb-76* mb indx-15.3* ctropnt-1.01* 03:04am blood ck-mb-55* mb indx-13.8* ctropnt-1.60* 12:19pm blood ck-mb-34* mb indx-11.6* ctropnt-1.44* 04:54am blood ck-mb-11* mb indx-8.1* ctropnt-0.88* anemia labs: 04:54am blood caltibc-222* vitb12-602 folate-14.5 hapto-279* ferritn-30 trf-171* 04:54am blood ret aut-2.0 misc: 09:30am blood lactate-4.0* ct of c-spine (): there is a anterior wedge compression fracture of the t3 vertebral body. the presence of adjacent osteophytes raises the possibility that the fracture is chronic, but this observation requires clinical correlation. ct head (): no intracranial hemorrhage, no fracture. 7mm rounded lymph node in the right submental region. ct head (): 1. no definite acute intracranial abnormality. 2. mild atrophy and left more than right basal ganglia chronic lacunes, with some volume loss. 3. chronic-appearing ethmoid inflammatory disease with extensive opacification of bilateral ethmoid air cells, right more than left. cxr (): interstitial pulmonary edema. renal us (): no renal stones or hydronephrosis on either side. echo (): the left atrium is mildly dilated. left ventricular wall thicknesses and cavity size are normal. there is regional left ventricular systolic dysfunction with akinesis of the basal 2/3rds of the inferior and inferolateral walls. the remaining segments are mildly hypokinetic. no masses or thrombi are seen in the left ventricle. right ventricular chamber size is normal with mild free wall hypokinesis. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. no aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. severe (3+) mitral regurgitation is seen. tricuspid regurgitation is seen. there is mild pulmonary artery systolic hypertension. there is no pericardial effusion. brief hospital course: 1. sepsis: patient with sirs based on tachycardia and fever with evidence of end organ damage with lactate of 4.0, qualifying for sepsis. was not hypotensive. cxr without evidence of infiltrate. ct scan did show sinusitis. overall, the source was presumed to be urinary as had wbc on ua - urine culture did not grow any organisms. in the unit, the patient was treated with ivf. hr and urine output were followed. broad spectrum antibiotics were used initially levofloxacin and vancomycin for urinary and nasal pathogens. flagyl was started initially with d/c as there was no evidence of aspiration or bowel pathogen. the patient did well in the unit and was called out to the floor. treatment was a planned 10 days of levaquin. on the day of discharge, the patient had an episode of diapheresis. oral temperature was 97 with a rectal temperature of 100. his finger stick was ~170 and an ekg showed no changes. given that his wbc had increased slightly from the prior day (12.3->14.8), blood and urine cultures were drawn. at the time of discharge, the patient felt well. 2. cad: in the setting of sepsis, the patient had an ekg with inferolateral st depressions with and a troponin that peaked at 1.6. cardiology saw the patient and felt this was demand as opposed to an acs. as such, they did not feel that an acute cath was needed. the patient was placed on heparin and integrillin on the 17th - the heparin was stopped two days later and the integrillin was stopped the next day. the patient was followed by cardiology - cardiac cath was considered, but not pursued. plan was for outpatient stress test once the patient was improved s/p sepsis. an echo was repeated showing an ef of 30% (unchaged from ) with mod/severe mr tr. regarding cardiac meds, the patient was treated with asa, ticlopidine (per home regimen), beta-blocker (metoprolol 75mg ), lisinopril 10mg daily, statin (came in on simvastatin; atorvastatin used while in-house with plan for resumption of simvastatin). in addition to the above, he was transfused 2 units of prbcs on for hct <30. thereafter, his hct remained >30. 3. chf: initially, cxr with pulmonary edema; did not require oxygen and was breathing easily with no le edema. lasix was held at the onset, given sepsis. as his bp remained stable, he was gently diuresed. thereafter, he was continued on acei and lasix. 4. fall: combination of loss of balance and neurologic d/o (parkinson's). no loc, did not sycopize. head ct negative for bleed. c-spine film showed no evidence of cervical spine fracture and a stable wedge deformity of t3 dating back to . b12 and folate were normal with a negative rpr. scalp staples from admission were still in place with plan for removal at rehab. 5. diarrhea: a chronic issue for the patient. while an inpatient, c.diff was checked and negative x2. 6. parkinson's: stable while an inpatient; continued home regimen of sinemet and comtan. 7. htn: antihypertensives held initially given sepsis. slowly added back. metoprolol and lisinopril were continued. 8. hyponatremia: patient has a long history of hyponatremia. sinemet may be contributor. acutely, hypovolemic hyponatremia may have been playing a role. at the time of discharge, serum sodium was 133. 9. osteoporosis: continue calcium and vitamin d. outpatient aldendronate was to be resumed upon discharge. 10. hyperlipidemia: as above, simvastatin at home with atorvastatin while in-house. cholesterol panel from showed tc 167, tg 233, hdl 62 and ldl 58. plan was for resumption of simvastatin upon discharge. dni/dni. medications on admission: 1. carbidopa-levodopa 25-100 mg tablet 8 times a day 2. comtan 200mg 5 five times a day 3. aspirin 325 mg tablet 4. metoprolol tartrate 50 mg 5. ticlopidine 250 mg tablet 6. lisinipril 10mg daily 7. furosemide 20 mg po daily 8. zocor 40 mg tablet 9. calcium plus vitamin d 10. alendronate 70mg qweek on sunday 11. multivitamin discharge medications: 1. aspirin 325 mg tablet sig: one (1) tablet po daily (daily). 2. ticlopidine 250 mg tablet sig: one (1) tablet po bid (2 times a day). 3. metoprolol tartrate 25 mg tablet sig: three (3) tablet po bid (2 times a day). 4. furosemide 40 mg tablet sig: one (1) tablet po once a day. 5. lisinopril 10 mg tablet sig: one (1) tablet po daily (daily). 6. hexavitamin tablet sig: one (1) cap po daily (daily). 7. calcium carbonate 500 mg tablet, chewable sig: one (1) tablet, chewable po tid (3 times a day) as needed for with meals. 8. cholecalciferol (vitamin d3) 400 unit tablet sig: one (1) tablet po daily (daily). 9. carbidopa-levodopa 25-100 mg tablet sig: one (1) tablet po 8x/d (). 10. entacapone 200 mg tablet sig: one (1) tablet po 5x/day (). 11. simvastatin 40 mg tablet sig: one (1) tablet po once a day. 12. acetaminophen 325 mg tablet sig: one (1) tablet po q4-6h (every 4 to 6 hours) as needed. 13. insulin regular human 100 unit/ml solution sig: one (1) injection asdir (as directed). 14. cholestyramine-sucrose 4 g packet sig: one (1) packet po daily (daily). 15. ferrous sulfate 325 (65) mg tablet sig: one (1) tablet po daily (daily). 16. levofloxacin 500 mg tablet sig: one (1) tablet po q24h (every 24 hours) for 4 days. 17. heparin (porcine) 5,000 unit/ml solution sig: one (1) injection tid (3 times a day). 18. alendronate 70 mg tablet sig: one (1) tablet po once a week: each sunday. discharge disposition: extended care facility: - discharge diagnosis: primary: 1. sepsis / uti 2. coronary artery disease. secondary: 1. parkinson's disease 2. hyperlipidemia 3. hypertension discharge condition: good; improved. discharge instructions: you were admitted after a fall and found to have an infection. you will be sent home with antibiotics which you should take, as directed, for the full course. given your history of heart failure, you should be sure to weigh yourself every morning and call your pcp if your weight > 3 lbs. there were no changes made to any of your current medications. followup instructions: you have the following appointments scheduled: , m.d. phone: date/time: 2:40 , md phone: date/time: 9:30 in addition to the above, you should call your pcp to be seen within 1-2 weeks. procedure: transfusion of packed cells diagnoses: subendocardial infarction, initial episode of care mitral valve disorders urinary tract infection, site not specified congestive heart failure, unspecified unspecified essential hypertension unspecified septicemia hyposmolality and/or hyponatremia aortocoronary bypass status percutaneous transluminal coronary angioplasty status sepsis paralysis agitans osteoporosis, unspecified personal history of malignant neoplasm of bladder hypovolemia diseases of tricuspid valve 

Answer: The patient is high likely exposed to