Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: syncope major surgical or invasive procedure: upper gi endoscopy history of present illness: 76 y/o m w/htn and dm who had a couple of days of severe epigastric pain that was mildly relieved with milk of magnesia and a bowel movement. he felt better, so he went out to a restaurant with his wife where he then had a syncopal episode. he hit his head on the ground, and per his wife was unconscious for less than ten minutes. he went to the where a cxr showed ? widened mediastinum, and since they could not r/o dissection as their ct scanner was broken, they sent him here. en route, he had an episode of coffee grounds emesis. initially, in our ed he was hypotensive to the 90s/60s which responded to 300 cc ns. he got protonix and anzemet. he was guaiac negative. ng lavage revealed coffee grounds and bright red blood which did not clear after 600 cc. he was seen by gi and transferred to the micu for egd in am. of note, he had a ct torso which was negative for dissection, as well as a negative head and c-spine ct. . on the day of admission, he had an egd which revealed erythema of the esophageal mucosa with no bleeding, compatible with esophagitis. he also had - tear with no bleeding. stomach showed erythema with no bleeding c/w gastritis. he remained hemodynamically stable and was called out to the floor after the endoscopy. currently, he has no complaints other than he is very thirsty. he has no more abdominal pain. past medical history: htn dm s/p appy social history: lives in with his wife. retired from international harvester company. no tobacco or etoh. family history: non-contributory physical exam: t: 98.9 bp: 130/62 p: 66 r: 21 93% on ra gen: sleeping male in nad, awakens easily, pleasant, oriented heent: mm dry, nc/at. lungs: bibasilar crackles, no w/r/c cv: rrr, no m/r/g abd: soft, nt/nd. +bs. ext: warm and dry. no edema. . pertinent results: 09:50pm wbc-13.8* rbc-4.67 hgb-13.9* hct-39.6* mcv-85 mch-29.8 mchc-35.2* rdw-14.6 09:50pm neuts-72.9* lymphs-21.4 monos-4.9 eos-0.5 basos-0.2 09:50pm plt count-277 09:50pm pt-13.0 ptt-21.9* inr(pt)-1.1 09:50pm ck-mb-notdone ctropnt-<0.01 09:50pm ck(cpk)-63 09:50pm glucose-186* urea n-51* creat-1.9* sodium-137 potassium-4.0 chloride-104 total co2-18* anion gap-19 09:57pm hgb-14.3 calchct-43 11:50pm urine blood-neg nitrite-neg protein-neg glucose-neg ketone-15 bilirubin-neg urobilngn-neg ph-5.0 leuk-neg 11:50pm urine color-straw appear-clear sp -1.030 brief hospital course: 76 y/o m w/dm, htn, who presents with upper gi bleed, found to have gastritis and tear on egd. brief hospital course by problem below: . 1. syncope: it is unclear if this bleed is truly what caused his syncope, as he never dropped his hematocrit and didn't have much in the way of significant bleeding on his egd. possible that he had such severe pain that he passed out, but he states he had been feeling better prior to this. his episode had a prodrome only of vision dimming, no dizziness. his ecg has a rbbb, confirmed as new by pcp. test showed no reversible perfusion defects with ef 67%. his cta showed very generous pulmonary arteries, f/u tte to evaluate pa pressures was normal. in addition, head ct negative. syncope therefore with no clear etiology, possibly small bleed and decreased po fluid intake leading to dehydration, evidenced by elevated bun:cr ratio, and hemo-concentrated with hct in high 30s. . 2. gi bleed: egd revealed - tear (strange as he didn't have any prior vomiting other than in ambulance), and gastritis. pt does recall heavy straining for bm for days before episode. hct stable throughout hospital course. ppi po bid started. aspirin held due to gastritis. h.pylori negative. . 3. elevated creatinine: 1.9 --> 1.4; improved with ivf. likely due to hypovolemia in setting of gi bleed, and may be elevated at baseline diabetic nephropathy. will monitor closely given that he received dye for cta. . 4. htn: restarted felodipine, added back other home meds on day of discharge. . medications on admission: benicar 40 mg daily glyburide/metformin 5/500 felodipine 10 mg daily isosorbide mononitrate 30 mg daily aspirin 325 discharge medications: 1. pantoprazole 40 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po q12h (every 12 hours). disp:*60 tablet, delayed release (e.c.)(s)* refills:*2* 2. felodipine 5 mg tablet sustained release 24hr sig: two (2) tablet sustained release 24hr po daily (daily). disp:*60 tablet sustained release 24hr(s)* refills:*2* 3. benicar 40 mg tablet sig: one (1) tablet po once a day. disp:*30 tablet(s)* refills:*2* 4. glyburide-metformin 5-500 mg tablet sig: one (1) tablet po once a day. disp:*30 tablet(s)* refills:*2* 5. isosorbide dinitrate 30 mg tablet sig: one (1) tablet po once a day. disp:*30 tablet(s)* refills:*2* 6. vytorin 10-10 mg tablet sig: one (1) tablet po once a day. disp:*30 tablet(s)* refills:*2* discharge disposition: home discharge diagnosis: primary diagnosis: - tear with gastritis secondary diagnoses: 1. hypertension 2. diabetes mellitus discharge condition: good discharge instructions: you were diagnosed with an esophageal tear with gastritis. please return to the hospital if you begin vomiting blood, having abdominal pain, passing dark stools, or pass out. please remember to make the follow-up appointments below followup instructions: please follow up with your pcp . within one week. procedure: other endoscopy of small intestine diagnoses: unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled unspecified gastritis and gastroduodenitis, with hemorrhage hypovolemia gastroesophageal laceration-hemorrhage syndrome 

Answer: The patient is high likely exposed to