Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: unresponsive major surgical or invasive procedure: none history of present illness: 78 yo m w/ h/o seizure disorder, parkinson's, dementia, recent hospitalization for aspiration pneumonia and encephalopathy/delirium presents to the ed with altered mental status and hct drop from 28.4 upon discharge to 21.7 (baseline appears to be 33-35). . he was recently dischared from hospitalization for aspiration pneumonia and encephalopathy/delirium and went to nh from there. at the nh this am, rn report had acute onset ams and was found to be hypoxic with o2 sats in the mid 80s which responded to o2 via nasal cannula (nml. o2 sats high 90s on ra). his wife reports that 1 day pta, she noted that at the nh, he had multiple large black stools; no bright red blood. upon tx to the ed, medics found him to have "low blood sugar" although exact number is not known. he received d50 en route to the hospital. . in the ed, he was found to have the above mentioned hct drop and had melanotic stool. gastric lavage was not performed in the ed. he received 2u prbcs. he was found to have diffuse pulmonary crackles and o2 requirement and cxr revealed e/o pulmonary edema and b/l pleural effusion c/w heart failure. bnp was 1193. vitals on presentation revealed hypothermia, bp 94/65 rr 16 o2 sat was 100% (unclear in documentation on what settings). bear hugger was applied and he was started on bipap. blood cultures were sent and he received vanco, ceftaz and flagyl x1. he received 3l ivf for persistent hypotension into 70s systolic to which his bp only transiently responded. discussion was had with his family by the ed staff and although he is dnr/dni, pressors are exceptable although they do not want central line placement. thus, he was started on levophed and was transferred to the for further management. . during his most recent hospitalization , he was treated with unasyn for aspiration pneumonia. he was discharged on augmentin to complete the course on . given his probable aspiration pneumonia, he was evaluated by speech and swallow at the bedside who recommended pureed solids and nectar thick liquids. he was also started on flagyl for c. diff colitis which he is to complete on . additionally, pt. had movements that were originally thought c/w seizure. neurology evaluated him and thought movements were likely dyskinesis from parkinson's. with the restarting of parkinson's medication, he became more responsive. past medical history: 1. parkinsons dx in 2. ? h/o stroke 3. gerd 4. bph 5. ? seizures 6. fatigue 7. atypical chest pain, noncardiac social history: lives at . children and wife live locally. from originally, cantonese speaking. denies tobacco, etoh, and drugs family history: non-contributory physical exam: vitals: t 95.0 ax bp 121/61 hr 61 rr 14 100% on 2l nc gen: unresponsive, nonverbal, does not follow commands heent: perrl, mmm, shallow wound on chin. neck: supple, +jvd to angle of jaw cv: rrr, no mrg resp: rhonchorus anteriorly with intermittent exp wheeze right anterior chest abd: decreased bowel sounds. thin. no guarding. no organomegaly. gastric lavage w/ sl. pink fluid ext: b/l upper extremity decorticate posturing. able to bend arms but w/ resistance. 2+ b/l le edema to mid shins. when attempt to bend left arm at elbow, pt. moans. neuro: 1+ patellar reflexes. unable to get biceps reflexes to stiffness and extensor posturing. pertinent results: ekg: nsr with rate 67. nml axis. 1st degree av delay. no acute st-t wave changes appreciated. . cxr: 1. bilateral pleural effusions, fluid overload with interstitial edema. more confluent suprahilar opacities may be due to dependent atelectasis component of edema or a superimposed process such as acute aspiration. 2. likely left humeral fracture. dedicated shoulder radiographs could be performed for further characterization. . left shoulder x-ray: read pending . head ct: no acute intracranial hemorrhage or mass effect . mra brain: 1. 5-mm low signal intensity in the white matter of the right frontal lobe consistent with hemosiderin and could represent a cavernoma or trauma. 2. diffuse abnormal bone marrow signal in the cervical spine could represent anemia or other diffuse infiltrative process. 3. normal circle of mra. . ct brain: no intracranial hemorrhage or mass effect. unchanged appearance from . . ct cspine: 1. no evidence of fracture or spondylolisthesis. 2. multilevel degenerative change. . cxr: left basal and right infrahilar opacity concerning for evolving pneumonia or aspiration. brief hospital course: # gi bleed: significant hct drop from 28.4 on to 21.7 in the ed on presentation, with elevated bun where it was previously was normal. unclear source. lavage with minimal pinkish tinge p 200cc, but ngt at g-e junction. family did not wish currently to pursue aggressive measures (egd/). he was transferred to the fiu as a dni and after discussion with family, he was made dnr/dni. . # sepsis: probable pulmonary source given recent hospitalization for aspiration pneumonia. patient was hypothermic with wbc >12 (12.3) w/ probable aspiration pneumonia as source. he was not tachycardic nor tachypneic. he was hypotensive despite 3l fluid resuscitation and required pressors to maintain his bp. currently on peripheral pressors started in the ed, but family requested no central line placement. he was continued on zosyn to cover anaerobes, vanco given recent hospitalization, and flagyl for known c. diff. above, family discussion was had regarding their wishes and he was made dnr/dni. despite peripheral levophed, his bp continued to drop and he expired within hours of transfer. family was at bedside. . # altered mental status: likely toxic metabolic in the setting of sepsis and gib. ct head was negative for acute intracranial process. . # coagulopathy: elevated inr to 1.9 and has not been on any anticoagulants. fibrinogen was normal, other dic labs not sent. likely largely nutrtional. he was given vitamin k in setting of gib and elevated inr. . # chf: has no previous h/o cad nor chf. last echo in with nml ef. bnp 1193 w/ clear e/o fluid overload on cxr, elevated jvd on exam nad peripheral edema. ces were negative x1. . # parkinson's disease: plan was to continue sinemet, entacapone, pramipaxole, and trihexyphenidyl. . # bph: foley was placed, tamsulosin was held in setting of severe hypotension. medications on admission: 1. acetaminophen 650 mg po q6h prn 2. entacapone 200 mg q4h w/ sinemet 3. carbidopa-levodopa 25-250 mg 1 po q 3am, 6am, 9am, noon, 3pm, 6 pm 4. carbidopa-levodopa 50-200 mg 1 po q9pm 5. pramipexole 0.25 mg tablet 1 po tid 6. trihexyphenidyl 2 mg tablet 1 po bid 7. tamsulosin 0.4 mg capsule 1 po qhs 8. lactulose 10 g/15 ml syrup 30ml po q8h prn 9. albuterol sulfate 0.083 % solution 1 neb q6h prn 10. docusate sodium 100 mg po bid 11. multivitamin,tx-minerals 1 po daily 12. aspirin 325 mg tablet 1 po daily 13. pantoprazole 40 mg tablet 1 po qdaily 14. calcium carbonate 500 mg tablet 1 po tid 15. cholecalciferol (vitamin d3) 800 unit daily 16. flagyl 500 mg tablet 1 po q8h (to complete on ) 17. augmentin 875-125 mg tablet 1 po bid (to complete ) . discharge disposition: expired discharge diagnosis: sepsis gi bleed discharge condition: expired followup instructions: none procedure: transfusion of packed cells diagnoses: esophageal reflux congestive heart failure, unspecified unspecified septicemia severe sepsis paralysis agitans acute respiratory failure pneumonitis due to inhalation of food or vomitus hemorrhage of gastrointestinal tract, unspecified 

Answer: The patient is high likely exposed to