Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no drug allergy information on file attending: chief complaint: vfib arrest major surgical or invasive procedure: echo x2 history of present illness: m with cad s/p 4v-cabg and cri had been doing well until this am when he was out walking with his wife. abruptly syncopized and a bystander started cpr quickly. the local fire department delivered two shocks without success. then ems came and gave two more shocks and he went back into sinus. it is unclear whether he regained consciousness. he was intubated but the et tube went into the esophagus. it was repositioned to the trachea, then brought to ed. . in the ed, his intial sbp was reported to be 110. labs show k 2.7 and hct 25. he was given 40meq of kcl. on repeat labs, his k normalized and his hct was 33 without any blood. it is unclear whether one of the labs was erroneous. ng lavage was repeatedly grossly positive. gi did not want to do egd until his hemodynamics were more stable. with fluids, his bp normalized and the vitals were recorded as: t=34.8, hr 62, 132/74, 18, 100% on ac 18x500, fio2 100%. he was briefly cooled by artic sun protocol but given his gib, this was stopped. . now the patient is still intubated and hemoadynamically stable and transferred to the ccu. gi is currently doing egd. . ros: per his wife, sons and pcp, was apparently in good health. he does not have chest pain or sob. he can walk a few blocks and up the stairs without symptoms. wife denies recent fevers, abd pain, n/v, hematemasis, hematachezia or brbpr. no orthopena, pnd or peripheral edema. no recent syncope or lightheadedness. no h/o stroke or bleeding disorder. the patient denies any chest pain or pressure, new exertional dyspnea, orthopnea, pnd or leg edema, palpitations or syncope, claudication-type symptoms, melena, rectal bleeding, or transient neurologic deficits. no change in weight, bowel habit or urinary symptoms. no cough, fever, night sweats, arthralgias, myalgias, headache or rash. all other review of systems negative. past medical history: # cad s/p mi and cabg -- lima to lad -- svg-d1 -- svg-pda -- svg-distal lcx -- ef 25% by cath # cardiomypathy -- echo : ef 30%, mid to distal anterior septal hk, inferoposterior wall hk, apex hk. trace ar, trace mr, +tr. # ccy, h/o gallstone pancreatitis # b12 deficiency, hct baseline 38 # restless legs # glaucoma social history: quit tobacco 20+ years ago, no alcohol. lives with wife. family history: no family of early cad or sudden death. physical exam: vitals: t=35, 82, 127/55, 22, 99% on ac 18x500 fi o2 100% gen: intubated and sedated, shivering heent: perrl, eyes edematous neck: no jvd cv: rrr, no m/g/r pulm: clear bilaterally abd: soft, nd, +bs ext: trace pedal edema. 2+ dp left, 1+ dp right. pt pulses not palpable. neuro: sedated. pertinent results: 09:28pm type-central ve temp-35 po2-29* pco2-39 ph-7.33* total co2-21 base xs--5 09:19pm ck(cpk)-395* 09:19pm ck-mb-9 ctropnt-0.26* 09:19pm hct-29.9* 04:14pm glucose-111* urea n-15 creat-1.3* sodium-143 potassium-4.2 chloride-116* total co2-21* anion gap-10 04:14pm ck(cpk)-319* 04:14pm ck-mb-7 ctropnt-0.27* 04:14pm calcium-6.8* phosphate-2.5* magnesium-1.4* 04:14pm wbc-8.5# rbc-3.12* hgb-9.7*# hct-29.6* mcv-95 mch-31.2 mchc-32.9# rdw-14.3 04:14pm plt count-106* 04:14pm pt-15.0* ptt-66.9* inr(pt)-1.3* 01:03pm hgb-11.0* calchct-33 12:15pm glucose-146* urea n-16 creat-1.5* sodium-142 potassium-4.5 chloride-113* total co2-22 anion gap-12 12:15pm estgfr-using this 12:15pm ck(cpk)-162 12:15pm ck-mb-3 11:45am po2-355* pco2-49* ph-7.25* total co2-23 base xs--5 11:45am glucose-151* lactate-4.3* na+-142 k+-3.1* cl--109 11:45am freeca-1.08* 11:23am type- po2-110* pco2-49* ph-7.17* total co2-19* base xs--10 comments-green top 11:23am glucose-612* lactate-4.3* na+-127* k+-2.7* cl--104 11:23am hgb-9.5* calchct-29 o2 sat-95 carboxyhb-2 met hgb-0 11:23am freeca-0.96* 11:22am ctropnt-<0.01 11:22am voidspec-suspect co 11:22am wbc-5.6 rbc-unable to hgb-5.6* hct-25.0* mcv-unable to mch-unable to mchc-24.7* rdw-unable to 11:22am neuts-47.6* lymphs-48.0* monos-2.6 eos-1.6 basos-0.2 11:10am urine rbc-* wbc-0-2 bacteria-occ yeast-none epi-0 trans epi- 11:10am urine blood-lg nitrite-neg protein-100 glucose-tr ketone-tr bilirubin-neg urobilngn-neg ph-6.0 leuk-neg 11:10am urine color-yellow appear-clear sp -1.013 11:10am urine bnzodzpn-neg barbitrt-neg opiates-neg cocaine-neg amphetmn-neg mthdone-neg 11:10am urine gr hold-hold 11:10am urine hours-random 11:22am fibrinoge-150 11:22am pt-17.3* ptt-43.8* inr(pt)-1.6* 11:22am plt smr-very low plt count-71* 11:22am neuts-47.6* lymphs-48.0* monos-2.6 eos-1.6 basos-0.2 the left atrium and right atrium are normal in cavity size. there is mild symmetric left ventricular hypertrophy with normal cavity size. there is mild global left ventricular hypokinesis (lvef = 45-50 %). the right ventricular free wall is hypertrophied. right ventricular chamber size is normal. with normal free wall contractility. the aortic root is mildly dilated at the sinus level. the ascending aorta is moderately dilated. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. mild to moderate (+) aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. there is no mitral valve prolapse. mild (1+) mitral regurgitation is seen. the tricuspid valve leaflets are mildly thickened. the estimated pulmonary artery systolic pressure is normal. there is a trivial/physiologic pericardial effusion. impression: mildly depressed global left ventricular function. mild to moderate aortic regurgitation. mild mitral regurgitation. the left atrium is mildly dilated. there is mild symmetric left ventricular hypertrophy with normal cavity size. there is mild regional left ventricular systolic dysfunction with distal septal hypokinesis. the remaining segments contract normally (lvef = 50%). no masses or thrombi are seen in the left ventricle. right ventricular chamber size and free wall motion are normal. mild (1+) aortic regurgitation is seen. trivial mitral regurgitation is seen. there is mild pulmonary artery systolic hypertension. there is no pericardial effusion. impression: mild regional left ventricular systolic dysfunction, c/w cad. mild aortic regurgitation. mild pulmonary hypertension. compared with the prior study (images reviewed) of , the findings are similar. mild regional wall motion abnormalities are present on both cxr impression: 1. bilateral apical opacities, which are nonspecific but may be related to aspiration or infiltrate. 2. distal tip of the et tube 9 cm from the carina and may be advanced up to 6 cm. 3. distal tip of ng tube at gastroesophageal junction. recommend advancement. 4. multiple left-sided rib brief hospital course: m with cad s/p 4v-cabg, chf, cri admitted s/p vfib arrest with gib initially started on arctic sun cooling protocol. . # vfib arrest. unclear etiology for vfib arrest since was apparently well prior to this episode per family. possibly from scar since has h/o ischemia and mi in past. less likely from acute ischemia/acs since no focal wma on echo, ef improved c/w prior and no preceding symptoms of chest pain. not likely from decompensated chf since he is not on lasix at home and there is no h/o weight gain or chf symptoms. possibly from electolyte abnormality (k=2.7 on admission), but unclear if these lab results were accurate since repeat set was normalized. less likely from gib which was probably secondary to intubation rather than gib causing vfib arrest. he was initially started on arctic sun cooling protocol with conservative target t=35 given gib. given hypotension (discussed below) and bradycardia overnight, cooling protocol was stopped and he was allowed to rewarm. electrolytes were monitored closely and repleted prn. shock was managed as discussed below and given refractory hypotension, he was made dnr and terminally extubated per family request. . # hypotension: pt initially normotensive on arrival sbps 100s-110s. blood pressure trended down throughout night. low bp initially attributed to cooling so cooling protocol was discontinued and pt was allowed to rewarm. he was started on levophed, then neosynephrine then dopamine and maxed out on 3 pressors. he had worsening lactic acidosis so he was given bicarb and started on bicarb drip to correct acid base status to allow pressors to work more effectively. patient had rapid decompensation overnight and it was unclear why he was so hypotensive despite multiple pressors. possible etiologies include hypovolemic (secondary to volume/blood loss), cardiogenic (although relatively preserved ef on echo) or septic shock. he was started on broad spectrum antibiotics (vanco/zosyn) and also given steroids for possible adrenal insufficiency. he was tranfused 2 units prbc for concern for blood loss with gib although hct was stable. he remained hypotensive with maps in 40s and progressively worsening lactic acidosis despite multiple pressors. family meeting was held in am to discuss poor prognosis and decision was made to make pt dnr and terminally extubate when all family was present. he expired later that morning with family at bedside. # cad: s/p cabg as above. had reversible defects on stress in . no ekg change or +cardiac markers to suggest acs. asa held in setting of gib. held beta blockers given hypotension. # chf: ef 45-55%. relatively euvolemic on initial exam. cxr without overt pulm edema. . # gib: pt had bright red blood ngt output after traumatic esophageal intubation. upon arrival to ccu, he had egd which showed active bleed from tear vs. dieulafoy's ulcer. this was cauterized with resultant hemostasis. he was tranfused 2 units prbc when later became hypotensive and hct was monitored and remained relatively stable. he was started on ppi iv. . # resp failure: intubated at scene. kept intubated on cooling protocol then later terminally extubated per family request. . # htn: held antihypertensives given hypotension . # access: rij and 2 18-gauge piv, a line placed . # code: initially full confirmed with wife and children. code status then changed to dnr/dni and pt extubated per family request with family at bedside. medications on admission: cozaar 25 digoxin 0.125 flomax 0.4 carbidopa-levodopa 25-100 prn qhs xalantan eye gtt qhs both eyes asa 325 discharge medications: n/a discharge disposition: expired discharge diagnosis: vfib arrest cad hypotension sepsis discharge condition: deceased discharge instructions: n/a followup instructions: deceased procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours arterial catheterization insertion of other (naso-)gastric tube endoscopic excision or destruction of lesion or tissue of esophagus diagnoses: congestive heart failure, unspecified hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage i through stage iv, or unspecified coronary atherosclerosis of unspecified type of vessel, native or graft chronic kidney disease, unspecified acute respiratory failure hypotension, unspecified cardiac arrest ventricular fibrillation acute systolic heart failure other and unspecified coagulation defects gastroesophageal laceration-hemorrhage syndrome esophageal hemorrhage 

Answer: The patient is high likely exposed to