Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: edema, fatigue major surgical or invasive procedure: none history of present illness: the pt is a 68 yo m with a h/o hemochromatosis and dm2 recently admitted to this hospital with a mssa bacteremia, pna, arf with creat 3.4, hyperk 6.0 and lactic acidosis with ag of 25. pt had fallen and was taking large amounts of motrin prior to that admission. on that admission, ekg showed right heart strain and possible lateral ischemic changes. pulmonary embolism was considered; v/q scan was read as low probability. he was admitted to the micu for hypotension and a pna and then sent to the floor, finished course oxacillin for bacteremia while in house. other issues during that hospitalization included: -cta showed an enlarged left-sided pleural effusion (fluid density) and a r-sided pleural effusion that was determined to be solid on thorocentesis (path result is pending) -hiv, and rf were sent and found to be negative; scleroderma ab neg - tte was obtained and showed a dilated rv with severe global free wall hypokinesis and abnormal septal movement; moderate-severe pulmonary artery systolic hypertension consistent with a primary pulmonary process. lvef was >55%. -seen on cta : 1. 3mm pulmonary nodule in the right middle lobe. 2. hypodense oval lesion approx 8mm at the liver dome. recommend follow-up ct in 1 year. . pt returned to with c/o increased fatigue and marked increase in peripheral edema. on home o2 since discharge home. states compliant with meds. denies cp/worsened sob/abd pain/ha/f/c. admits cough with small amt white sputum occasionally. . ccu course: pt was started on viagra for primary pulmonary htn, and aggressively diuresed with lasix gtt and diuril for several days with net 7.4l with symptomatic improvement in sob on 5lnc, also started on anticoagulation with lovenox for several days however switched to hep gtt and will transition to coumadin. thoracentesis on removed ~800 cc serosanguinous fluid which was negative for malignant cells. he was also started on steroids for copd. his o2 sats were stable on 5lnc. on pt's sats stable while working with physical therapy. pt also noted to go in and out of af/flutter, was started on digoxin, dosed by levels. his cri was followed closely and cr stable at 1.9 in setting of aggressive diuresis. pt was also started on dilt for better hr control in setting of af/flutter. pt was called out of micu to floor in stable condition. past medical history: pmh: * hemochromatosis with monthly phlebotomy; dx 15 yrs ago * cardiac involvement from hemochromatosis * dm * hx of colon polyps * gallstones (asx) * hypothyroidism * arf in setting of nsaid use 13 years ago, requiring 5 months of hd. social history: widowed, occ alcohol, no cigarettes, usually very active, plays golf, no tob use, can do all adls family history: parents died in their 50s, unknown cause, no fam hx of cad, dm, hemochromotosis, malignancy, hypercoaguable state physical exam: gen: elderly male, nad, full sentences vs: 97.6 98 152/80 16 89 ra 95% 5l nc heent: perrl, eomi, nl sclera, mmm neck: supple, no jvd cor: s1s2 rrr lungs: decreased bs bilat, scattered exp wheeze, ? crackles at l base abd: soft, nt/nd; eccymoses (fading) from previous heparin subq injections ext: 3+ pitting edema to knees bilat, +sacral edema neuro: a&ox3, cn intact, strength 5/5 prox & distal, no pronator drift, no asterixis skin: bronze color . . pertinent results: 05:41pm glucose-327* urea n-23* creat-1.3* sodium-131* potassium-4.5 chloride-96 total co2-27 anion gap-13 05:41pm wbc-7.5 rbc-3.79* hgb-11.0* hct-33.9* mcv-89 mch-29.1 mchc-32.5 rdw-18.7* 05:41pm neuts-79* bands-1 lymphs-7* monos-9 eos-3 basos-1 atyps-0 metas-0 myelos-0 05:41pm hypochrom-occasional anisocyt-2+ poikilocy-2+ macrocyt-2+ microcyt-1+ polychrom-1+ target-1+ burr-2+ 05:41pm pt-13.6* ptt-30.6 inr(pt)-1.2* 05:41pm plt smr-low plt count-80* . cxr : chest, pa and lateral: comparison is made to . the lung volumes are low. the picc line has been removed. allowing for low lung volumes, the cardiac and mediastinal contours are unchanged. there is a persistent right lower lobe opacity, probably some atelectasis in addition to effusion. there is also a left-sided effusion with parenchymal opacity obscuring the medial hemidiaphragm, which may represent atelectasis. right apical thickening is unchanged since a prior study from . impression: moderate bibasilar effusions and opacities, which are likely to represent atelectasis. underlying pneumonia cannot be excluded however. . cxr : preliminary read pa and lateral chest. there are bilateral moderate-sized pleural effusions with possible partial posterior loculation. the cardiac silhouette is enlarged. i doubt the presence of vascular congestion although appearances suggest possible underlying chronic lung disease with stranding in the right lung. since exam one day previous the equivocal interstitial edema appears improved or resolved. the effusions are associated with bibasilar subsegmental atelectasis. impression: short interval probable improvement/resolution of chf. no change in effusions. . r-sided cardiac cath: hemodynamics results body surface area: 1.95 m2 hemoglobin: 11 gms % fick **pressures right atrium {a/v/m} 13/13/11 right ventricle {s/ed} 82/21 pulmonary artery {s/d/m} 82/43/63 pulmonary wedge {a/v/m} 13/13/11 **cardiac output o2 cons. ind {ml/min/m2} 125 a-v o2 difference {ml/ltr} 56 card. op/ind fick {l/mn/m2} 4.4/2.2 **resistances pulmonary vasc. resistance 946 **% saturation data (nl) svc low 57 pa main 54 **arterial blood gas inspired o2 concentr'n 21 . comments: 1. vasodilator challenge in this patient with pulmonary hypertension revealed baseline severe pulmonary hypertension with pressures of 82/43. the ra pressure was slightly elevated at mean of 14mmhg with a relatively normal left sided filling pressure with pcwp of 11mmhg. the cardiac index was preserved at 2.2. 2. with 100% oxygen and then nitric oxide there was no decrease in pulmonary pressures. with oxygen pa was measured at 83/38 mean 57 and pcwp of 13mmhg. with nitric oxygen pressure was 91/39 wit mean of 60. the cardiac index did improve on oxygen to 2.7 and on nitric oxide to 3.2 final diagnosis: 1. severe pulmonary hypertension with normal left sided filling pressures, not responsive to vasodilators. . echo bubble study: conclusions: no definite right-to-left passage of microbubbles identified at rest or with maneuvers (cough, post-valsalva). the right ventricle is dilated with prominent free wall hypokinesis. . cxr: indication: chf versus developing pneumonia. a right subclavian vascular catheter remains in place, terminating at the junction of the superior vena cava and right atrium. the cardiac silhouette is enlarged but stable. there is upper zone vascular redistribution and perihilar haziness, not significantly changed. there is partial atelectasis of the right lower lobe with inferomedial displacement of the right major fissure. an area of increased opacity is noted within the left retrocardiac region, with interval improvement since the recent study. this is probably due to a combination of effusion and atelectasis. . chest ct: bilateral pleural effusion, moderate and low density on the left, and small with high density contents and perimeter enhancement is unchanged in both hemithoraces. bibasilar consolidation is more pronounced in comparison to the previous studies and may represent pneumonia or secondary atelectasis. prominence of the interlobular septa as well as some engorgement of the pulmonary vasculature represent congestive heart failure. imaged part of the upper abdomen demonstrate several gallstones in othewise normal gallbladder, calcified liver granuloma and nodularity of the liver margin (the patient has a known history of cirrhosis ). the spleen, adrenals, kidneys and pancreas are unremarkable except for left renal cortical atrophy. no suspicious lytic or blastic lesions within the bones were shown. prominent gynecomastia is due to cirrhosis. impression: 1) increased, moderate left pleural effusion. smaller high density right pleural fluid collection is unchanged. 2) increased bibasilar pulmonary consolidations, which may represent secondary atelectasis or pneumonia. 3) mild congestive heart failure. prominent coronary artery calcifications and cardiomegaly are unchanged. 4) gallstones without evidence of acute cholecystitis. . thoracentesis fluid: negative for malignant cells. -mesothelial cells and lymphocytes. . echo tte: conclusions: there is moderate symmetric left ventricular hypertrophy. due to suboptimal technical quality, a focal wall motion abnormality cannot be fully excluded. overall left ventricular systolic function is normal (lvef>55%). the right ventricular cavity is markedly dilated. right ventricular systolic function appears depressed. there is abnormal septal motion/position consistent with right ventricular pressure/volume overload. the aortic valve leaflets are mildly thickened. there is no aortic valve stenosis. the mitral valve leaflets are mildly thickened. there is severe pulmonary artery systolic hypertension. there is no pericardial effusion. no vegetations seen (but cannot definitively exclude). . portable kub: impression: nonspecific bowel gas pattern. if clinically indicated, ct scan may be useful to further characterize this finding. this was discussed with dr. at approximately 3:30 a.m., . . abd u/s w/doppler: impression: . 1. no evidence of perihepatic ascites. 2. stable appearance of hepatofugal portal venous flow. 3. limited examination secondary to cirrhotic heterogeneously echogenic liver. although no focal liver lesions are identified, given the technical difficulties of this ultrasound surveillance, ct or mri surveillance for lesions could be performed for future examinations. . cxr: findings: there has been interval removal of the left ij line. ng tube is in the stomach. there is hazy bilateral increased vasculature and increased bilateral pleural effusion suggesting chf. impression: worsening chf. . ecg: sinus rhythm with first degree a-v block. since the previous tracing of the rhythm has reverted from atrial fibrillation to no significant change and the rate has slowed. diffuse non-specific st-t wave abnormalities persist. . labs: -last set wbc rbc hgb hct mcv mch mchc rdw plt ct 10:11am 13.1* 2.64* 7.6* 22.6* 86 28.7 33.5 19.1* 147 . pt ptt plt smr plt ct inr(pt) 10:11am 147* 10:00am 18.7*1 33.6 1.8* glucose urean creat na k cl hco3 angap 04:16am 68* 87* 2.4* 141 3.4 97 34* 13 --- coags: fibrino d-dimer 08:19am 159 01:58am 176# 03:49am * 05:45am 293 09:50am 332 . hemolytic w/u: ret aut 03:19am 6.5* 06:17am 6.7* 04:35pm 3.5 . hit ab test=neg: heparin dependent antibodies test result ---- ------ heparin dependent antibodies negative comment: negative for heparin pf4 antibody by complete report on file in the laboratory. . . caltibc vitb12 folate hapto ferritn trf 08:19am <20* 03:19am <20* 03:49am 169* 1695* greater th1 69 130* added chem 11:50am 1 greater than 20 ng/ml 05:45am 39 09:50am <20 . -- lfts: alt ast ld(ldh) ck(cpk) alkphos amylase totbili dirbili indbili 04:16am 27 37 402* 103 125* 1.1 added osmo 8:24am 08:19am 26 39 441* 103 1.3 0.9* 0.4 03:19am 401* 1.2 05:27am 17 28 80 0.7 source: line-mlc 03:49am 21 38 346* 92 0.9 05:45am 353* 09:50am 466* 0.7 0.3 0.4 05:44am 21 36 429* 122* 0.8 . hbsag hbsab hbcab 06:55pm negative negative negative immunology dsdna 06:55pm negative protein and immunoelectrophoresis pep igg iga igm ife 06:55pm no specifi1 1054 557* 166 no monoclo2 . hcv ab 06:55pm negative . . c3 c4 06:55pm 83* 17 . --- micro: brief hospital course: the patient was admitted to the medical icu in critical condition in setting of severe pulmonary htn. his course was complicated by hemolytic anemia, gib, in setting of anticoagulation for his pulmonary htn. also c/b hepatic encephalopathy. per family and hcp-daughter , pt was made on and expired on . provided support to family towards the end of his complicated course of illness. . . medications on admission: spironolactone 25mg daily furosemide 20mg daily synthroid 0.1mg daily folic acid 1mg daily diltiazem 30mg daily mirtazapine 15mg at bedtime aspirin 81mg daily combivent 103-18 mcg/actuation aerosol 1 puff qid oxygen 2-3l via nasal cannula to keep o2 sat>94% insulin discharge medications: none discharge disposition: expired discharge diagnosis: -hemochromatosis, esld -pulmonary hypertension -expired discharge condition: deceased discharge instructions: none followup instructions: none md, procedure: venous catheterization, not elsewhere classified other endoscopy of small intestine diagnostic ultrasound of heart thoracentesis transfusion of packed cells right heart cardiac catheterization transfusion of other serum diagnoses: nephritis and nephropathy, not specified as acute or chronic, in diseases classified elsewhere congestive heart failure, unspecified cirrhosis of liver without mention of alcohol acute and subacute necrosis of liver acute kidney failure, unspecified acquired coagulation factor deficiency iron deficiency anemia secondary to blood loss (chronic) portal hypertension unspecified acquired hypothyroidism atrial fibrillation other chronic pulmonary heart diseases bacteremia methicillin susceptible staphylococcus aureus in conditions classified elsewhere and of unspecified site long-term (current) use of insulin infection and inflammatory reaction due to other vascular device, implant, and graft other disorders of plasma protein metabolism diabetes with renal manifestations, type ii or unspecified type, not stated as uncontrolled acute gastritis, with hemorrhage 

Answer: The patient is high likely exposed to