Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: nardil attending: chief complaint: severe headache, vertigo and unsteadiness major surgical or invasive procedure: cerebral angiography history of present illness: mr is aged 56 and is left handed. he had a history of depression on nardil who presents with a 3 day history of intermittent headaches and episodes of dizziness and imbalance, found to have a posterior sah on cerebral angiography showing diffuse vasospasm. the patient was in his usoh until tuesday (3 days pta) when he woke up and was getting ready for work, and felt the onset of a headache. he continued to get on the train for work, and over the next 20-30 minutes the headache got progressively worse to a headache that was almost making him cry because of the pain. he described the headache as a pounding/sharpness that was initially centered in his occiput and then radiated around to the front. he has a history of occasional headaches, but has never had symptoms like this before. he denied any associated nausea/vomiting, weakness or numbness, changes in vision, photophobia/phonophobia, or change in character with change in position or valsalva maneuver. the headaches were associated with a feeling of sleepiness. he got off the train and took 3 motrin with a decrease in the headache to . he proceeded to go to work, where his headache ressolved. later that day at work, he was headache-free, but then developed a "dizzy attack" in which the room was spinning and he felt imbalanced requiring him to hold onto the wall to walk. this lasted 20 minutes, was not associated with nausea, and improved after he sat down. on wednesday (2 days pta) he woke up at 4:00 am with a headache, took 3 motrin, and then went back to sleep when the headache ressolved. he woke up again at 8:00 am with another headache, which improved by the time he went to work. hedid not seek medical attention because he thought his headaches may have been secondary to his medications. on thursday (1 day pta), he presented to for a scheduled psychotherapy session. at that time he complained of dizziness and lightheadedness, and his sbp was found to be 98. he took the train back home, and as he was crossing the street in front of the train station he felt the intense sensation of dizziness come on and lost his balance. he returned to the train station, where he needed to hold onto the rails. regardless, he fell to the ground without head trauma or loc. he returned home, where a generalized feeling of weakness overcame him, and he again fell to the ground. his headache then returned, but ressolved with a bag of ice to the head. his partner wanted to take him to the hospital, but since he made a pcp appointment for the next day they decided to wait. he was seen at his pcp's office on the day of admission, and was referred for ct head given his symptoms. in the ed, vitals were temp 96.3, hr 72, bp 131/66, rr 18, sao2 95% on ra. he continued to have headache and was given morphine 4 mg iv x1. ct head showed subarachnoid blood within the posterior hemispheric sulci bilaterally. neurosurgery was consulted, and he was sent to cerebral angiography where he was found to have spasm of the r&l mca, r&l pca, l aca. he was admitted to the neuroicu. the nardil was felt to have contributed to his vasospasm, and so it was discontinued. past medical history: -hypercholesterolemia -depression, been hospitalized twice for depression, s/p 1 suicide attempt -anxiety -bipolar disorder -ocd -history of hepatitis b -c6 disc protrusion, low back pain -s/p incarcerated umbilical hernia repair -s/p pilonidal cyst removal at the -h/o right groin abscess -seborrheic keratoses -positive ppd in the early 70s, status post inh treatment x1 year social history: he stopped smoking cigarettes 8 months ago, but prior to that was smoking 1-1.5 ppd for 20-30 years. he denies a history of alcohol abuse. in the , he smoked pot and used coke, but denies any history of ivdu or any recent history of illicit drug use. he lives with his partner of 34 years, and has been married to him for the past 4 years. he works at as an assistant to the chief operating officer. family history: his mother had an ich while on anticoagulation, but there is no family history of cerebral aneurysms. his father and brother were alcoholics. mental illness in his family. physical exam: physical examination: vs: temp 95.2 (after ice chips had been in his mouth), bp 144/80, hr 54, rr 18, sao2 97% on 3l genl: awake, alert, nad heent: sclerae anicteric, right conjunctival injection, oropharynx clear cv: bradycardic, nl s1, s2, no murmurs, rubs, or gallops chest: cta bilaterally anteriorly and laterally, decreased breath sounds at the right base, no wheezes, rhonchi, rales abd: +bs, soft, ntnd abdomen ext: no lower extremity edema bilaterally neurologic examination: mental status: awake and alert, cooperative with exam, normal affect. oriented to person, place, and date. attentive, says backwards. speech is fluent with normal repetition. no dysarthria. registers , recalls (but with prompting)in 5 minutes. no evidence of apraxia or neglect. cranial nerves: pupils equally round and reactive to light, 4 to 3 mm bilaterally. visual fields are full in all fields. extraocular movements intact bilaterally without nystagmus. sensation intact v1-v3. facial movement symmetric. hearing intact to finger rub bilaterally. palate elevation symmetric. sternocleidomastoid and trapezius full strength bilaterally. tongue midline, movements intact. motor: normal bulk bilaterally. no observed myoclonus, asterixis, or tremor. no pronator drift. tri we fe ff ip h q df pf r 5 5 5 5 5 5 5 5 5 5 5 l 5 5 5 5 5 5 x x x 5 5 *unable to assess left deltoid, quad, or hamstring strength as patient was s/p cerebral angiography. sensation: intact to pinprick, vibration, position sense, and cold sensation throughout. no extinction to dss. reflexes: 2+ and symmetric in biceps, brachioradialis, triceps, knees, and ankles. toes downgoing on the right, equivocal on the left. coordination: finger-nose-finger, finger-to-nose, and fine finger movements normal. gait: unable to test prior to discharge, mr was walking normally, and had a completely normal clinical examination. pertinent results: 05:58pm glucose-73 urea n-20 creat-1.0 sodium-139 potassium-4.3 chloride-106 total co2-27 anion gap-10 05:58pm calcium-8.8 phosphate-3.2 magnesium-2.1 05:58pm -positive titer-1:40 05:58pm crp-1.3 05:58pm sed rate-11 10:58am glucose-97 urea n-26* creat-1.3* sodium-140 potassium-5.7* chloride-107 total co2-25 anion gap-14 10:58am estgfr-using this 10:58am ck(cpk)-166 10:58am ctropnt-<0.01 10:58am ck-mb-4 10:58am calcium-8.9 phosphate-2.7 magnesium-2.1 10:58am asa-neg* ethanol-neg acetmnphn-neg bnzodzpn-neg barbitrt-neg tricyclic-neg 10:58am wbc-7.2 rbc-4.80 hgb-14.2 hct-42.0 mcv-88 mch-29.6 mchc-33.8 rdw-14.6 10:58am neuts-60.0 lymphs-28.3 monos-5.1 eos-5.5* basos-1.2 10:58am plt count-243 10:58am pt-11.8 ptt-24.7 inr(pt)-1.0 mri and mra brain mr findings compatible with those demonstrated on the cta of . however, there is cortical swelling and hyperintensity along with vasospasm and focal areas of dilatation. these findings would be compatible with the suggested diagnosis of call . migraines may be a diagnostic alternative. ct of the head & cta from impression: 1. multiple, focal narrowings in the branches of the middle cerebral arteries bilaterally. an additional focus of narrowing is seen in the right posterior cerebral artery. these are concerning for vasculitis. 2. assymetric prominence of the cortical vasculature in the occipital region bilaterally. this may respresent hyperemia secondary to occipital subarachnoid hemorrhage. brief hospital course: mr was admitted with cerebral artery vasospasm (call ) and was found to have a posterior sah as a consequence of this vasospasm. he was in his usoh until tuesday (3 days pta) when he woke up and was getting ready for work, and felt the onset of a headache. he continued to get on the train for work, and over the next 20-30 minutes the headache got progressively worse to a headache that was almost making him cry because of the pain. he described the headache as a pounding or sharpness that was initially centered in his occiput. his headaches were controlled with dilaudid, however, three days prior to discharge, he did not need any analgesia (even tylenol). his call syndrome is being treated with verapamil. his vertiginous symptoms and unsteadiness have resolved. he saw psychiatry for his issues with depression, and the decision was made to hold off any medications for now. he will follow-up with his psychiatrist when he is discharged. prior to discharge, he mentioned that he felt well, and that he was back to his baseline. he no longer had a headache. he will have an outpatient cerebral angiogram to see whether his vasospasm has resolved, and he will follow-up with dr. as an outpatient. medications on admission: nardil 30 mg po bid, has been on for 2 months xanax 1 mg po tid (but takes it prn) ativan 1 mg po bid (but takes it prn) discharge medications: verapamil 180 mg tablet sustained release sig: one (1) tablet sustained release po q24h (every 24 hours). disp:*30 tablet sustained release(s)* refills:*2* discharge disposition: home discharge diagnosis: cerebral artery vasospasm subarachnoid hemorrhage discharge condition: dizziness and symtoms of unsteadiness have resolved completely. the headache is now controllable with oral analgesia. discharge instructions: if you have any of the following symptoms: worsening headache, alteration of consciousness, weakness on any one side of your body, please go to your nearest emergency department. followup instructions: 1. for out patient cerebral angiogram with dr on , monday at 11 am. 2. for follow-up with dr on mon :30 h with dr (, , tel: ). md procedure: arteriography of cerebral arteries diagnoses: pure hypercholesterolemia subarachnoid hemorrhage depressive disorder, not elsewhere classified unspecified transient cerebral ischemia chronic viral hepatitis b without mention of hepatic coma without mention of hepatitis delta 

Answer: The patient is high likely exposed to