Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: this is a 76 year old male with a history of atrial fibrillation, benign prostatic hypertrophy, history of left carotid bruit, history of seizure disorder, who was on a ladder the evening of admission and fell. he was noted to be pulseless and to be cyanotic afterwards. he was given epinephrine/atropine with heart rate in the 30s to 40s. he had been down for about fifty minutes. he had a history of such episodes as well in the past. the patient then had been intubated and then transferred for further treatment to . per family, he had a history of falls about twelve years ago and nine years ago, but not recently. past medical history: 1. history of basal cell carcinoma. 2. squamous cell carcinoma. 3. history of left carotid bruit. 4. seizure disorder, , started with episode of spinal meningitis. 5. history of sleep apnea, has been on cpap. 6. atrial fibrillation. 7. cataracts. 8. contractures. 9. benign prostatic hypertrophy. 10. history of osteoarthritis. 11. history of aortic and mitral regurgitation. 12. echocardiogram in , showed left atrium moderately dilated, right atrium markedly dilated, left ventricular systolic function 60%, 3+ aortic regurgitation, 1 to 2+ mitral regurgitation, 3+ tricuspid regurgitation, mild to moderate pulmonary artery systolic hypertension (37). 13. stress test in , nine minute modified protocol showed good exercise tolerance, no 2d echocardiogram evidence of inducible ischemia to the achieved workload. allergies: levaquin. family history: father with trigeminal neuralgia. siblings with parkinson's. no history of coronary artery disease, diabetes mellitus or hypertension otherwise. social history: active, gardener, walks, one glass per night of wine, no tobacco. physical examination: vital signs on admission showed a temperature of 100.1, pulse range of 101 to 115, blood pressure 128/59, respiratory rate 22, oxygen saturation 100% quantitative saturation. generally, the patient was in no acute distress, intubated. head, eyes, ears, nose and throat - diffuse contusions on face. cardiovascular is irregularly irregular. respiratory clear to auscultation bilaterally. the abdomen is soft, nontender, nondistended. extremities no edema. neurologically, intubated, sedated, moved extremities but not to command. laboratory data: on admission, white blood cell count was 8.5, hematocrit 37.3, platelet count 191,000. sodium 131, potassium 4.1, chloride 97, bicarbonate 20, blood urea nitrogen 35, creatinine 1.5. the patient had an anion gap of 17, glucose 240, lactate 7.2. inr 2.9, partial thromboplastin time 25.2. fibrinogen 194, amylase 59, ck 121, ck mb 4.0. urinalysis was negative. x-ray of the pelvis - the hips are normally aligned without fractures or dislocations. head ct showed no intracranial bleeding evidence or mass effect. low density lesions consistent with subacute or chronic infarct. mild brain atrophy. ct also showed no evidence of fractures or dislocations of cervical spine. mild wedge compression deformity and possible t2 evaluation of prevertebral soft tissue linked with secondary ett vacuum phenomenon present of disc c4-c5, c5-c6 and c6-c7. abdominal pelvic ct showed a small right pleural effusion, bilateral atelectasis, no intra-abdominal problems. electrocardiogram showed findings consistent with atrial fibrillation. urine toxicology screen was negative for all except positive for barbiturates. arterial blood gases while intubated on admission showed ph 7.52, co2 29, o2 295 and bicarbonate of 24. this was on a tidal volume of 700 and rate of 12. hospital course: the patient is now status post trauma. trauma surgery had evaluated the patient and there was no plan on treatment. the patient was transferred to ccu for observation. in terms of his neurological symptoms, etiology of his symptoms that may have led to the fall were unclear, but thought to be possibly secondary to transient ischemic attack/stroke. neurology was consulted. head ct as noted above, and he was continued on his tegretol. for his cardiac issues, given history of atrial fibrillation, he was rate controlled with vasotec, metoprolol, and his coumadin was held given the possibility of the patient going for cardiac catheterization. on , the patient was continued on his cervical collar and log roll precautions per trauma surgery advice until he was cleared by trauma surgery who is following peripherally. head ct again showed no bleed, has a t1, t2 fracture, new versus old, unsure but needs examination without sedation to be able to evaluate or a magnetic resonance scan. at this point, we are waiting for trauma team to clear his neck or a magnetic resonance scan which is planned for the day after. neurology had noted given the patient's past history of transient ischemic attack and falls, this could have been secondary to transient ischemic attack and cerebrovascular accident events. neurology was consulted and they recommended a magnetic resonance scan and a neural check q1hour. he also has a history of seizures and they recommended to continue his home medications mebaral and tegretol and levels pending and a.m. results. also, they ordered an electroencephalogram once the patient is off the cervical collar and stable. cardiac was continued as discussed before. his electrocardiogram had shown st depressions in v4 to v6. he was kept on aspirin and beta blockers for his cardiac care and we are cycling enzymes while the patient was on our service. there is a question of maybe the patient might go to cardiac catheterization laboratory once inr has decreased. at the time of admission, inr was 2.9. he was being rate controlled with vasotec and metoprolol as discussed previously. the morning before the patient had passed away the patient was continued still on the cervical collar and log rolling precautions. orthopedics had come on board to clear spine and there was a thought that the patient might have had a cerebellar infarct. per neurology recommendation, the patient was also started on steroids to decrease possible inflammatory processes that could contribute to the patient's symptoms. cardiac as before with no change. it was thought the patient was going through generalized seizures based on electroencephalogram results. on , there was no improvement in mental status when evaluated the patient. the patient was started on dilantin the day before the time of death, but no improvement was noticed in the patient's condition. on , dr. had discussed with daughters and wife regarding the patient's prognosis and recovery chances. at that time, the patient's family felt that he would not want to be resuscitated. also, the family had agreed that no more medical support should be necessary since that could prolong the patient's suffering while in the hospital. at 4:30 p.m. on , resident physician was called to bedside for the patient being unresponsive. the pupils were fixed, nonreactive, no audible heart sounds were felt, followed one minute, no sounds still, no spontaneous respirations, no response to sternal rub. the family was notified immediately and the attending physician was also notified immediately. time of death at that point was called at 7:25 p.m. on . dr., 12-703 dictated by: medquist36 procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more diagnoses: atrial fibrillation other convulsions pneumonitis due to inhalation of food or vomitus cardiac arrest anoxic brain damage accidental fall from ladder spinal stenosis in cervical region 

Answer: The patient is high likely exposed to