Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: slow responses, right sided weakness major surgical or invasive procedure: thoracentesis nasogastric tube placement history of present illness: 86yo right handed man with pmh significant for uncontrolled htn and hyperlipidemia who was in his usoh the night of presentation when he walked to the restroom at 8:30pm. he then sat on the couch but when his wife called to him to come to dinner, he was slow to respond, stood but could not walk due to right-sided weakness and his speech was slurred. she gave him a series of commands which he performed but his response time was significantly slowed and she had difficulty understanding his speech. at this point, she called ems. he is now transferred here after ct of the head at osh showed a 2.5cm x 2.4cm left thalamic ich with slight rupture into the ventricle. on presentation to , he was hypertensive to 231/88 and he was given lopressor, to lower his pressure to the current level of 204/64. past medical history: anemia, w/u pending - referred by pcp to hematologist. wife brought in letter stating the belief that he has a problem with "red cell production" htn x yrs, often uncontrolled to 200's hyperlipidemia gerd ankylosing spondylitis l ica carotid stenosis (complete occlusion) h/o tuberculosis "in his neck", s/p multidrug treatment x 6mos no mi, cad, or stroke social history: lives with his wife, retired sbo. quit smoking 8yrs ago after 20ppyr history. no other drug use. family history: brother died of mi at age 70 physical exam: exam on discharge: vs 98.5 204/64 16 98% 97 gen lying in bed in nad cv rrr pulm ctab abd soft ext l foot erythematous and swollen, warm to touch neuro ms lying in bed with eyes closed. opens them to voice. oriented to hospital and city, states it is . speech is very dysarthric (from normal baseline) and slow but fluent and without apparent errors. follows simple commands. slow response time. cn pupils anisocoric (b/l cataracts) - l 1.5mm and r 2mm; neither reacts. vff to confrontation. eomi including upgaze. facial sensation intact. r nlf flat. smile full. hearing intact. palate rises symmetrically. shrug . tongue midline motor normal bulk/tone. +r pronator drift d b t we fe ff ip q h df pf te coord decreased ffm/rams on right side, esp compared to non-dominant left side reflexes 2+ throughout, except for 1+ at ankles. toe up on r, down on l sensory intact to all primary modalities throughout, no extinction to lt gait deferred pertinent results: admission labs: cbc: wbc-3.5* rbc-3.28* hgb-10.0* hct-28.9* mcv-88 mch-30.4 mchc-34.5 rdw-19.0* plt ct-124* coags: pt-12.1 ptt-31.6 inr(pt)-1.0 chem10: glucose-130* urean-27* creat-1.4* na-133 k-3.9 cl-102 hco3-26 calcium-7.8* phos-3.8 mg-2.0 lfts: alt-33 ast-38 ck(cpk)-44 alkphos-473* totbili-0.5 albumin-3.3* lipase-54 ggt-321* cardiac enzymes negative x 3 other labs: probnp-7299* abg: type-art po2-100 pco2-53* ph-7.34* caltco2-30 base xs-0 repeat: type-art po2-103 pco2-49* ph-7.33* caltco2-27 base xs-0 pleural fluid: wbc-550* rbc-482* polys-9* lymphs-72* monos-0 meso-1* macro-18* totprot-1.9 glucose-127 ld(ldh)-89 albumin-1.3 cholest-37 gram stain-final; fluid culture-pending; anaerobic culture-pending; acid fast smear-final; acid fast culture-pending cxr: right-sided volume loss and right apical pleural thickening, unchanged. increased opacity involving the right lung may represent scarring versus atelectasis. comparison with previous radiographs would help to better assess for long-term interval change. loculated left-sided pleural effusion. increased air space opacity involving the left mid and lower lung is less conspicuous than seen previously. diagnostic considerations include asymmetric pulmonary edema and pneumonia. chest ct: small right and moderate left pleural effusions, probably not transudate, greater and maybe loculated in the left side. peripheral consolidation in the right upper lobe, largely scarring, but peribronchial thickening in the lower lobes, could be chronic or subacute infection. fusiform aneurysmal dilatation of the suprarrenal abdominal aorta. head ct : the left thalamic hemorrhage is similar in size, measuring 2.6 x 2.4 cm. there is interval increase in a small amount of intraventricular hemorrhage. there is no new mass effect, hydrocephalus, or major vascular territorial infarction. slight bulge of normally midline structures to the right is noted. surrounding osseous and soft tissue structures are again noted. mucosal thickening is seen in the sphenoid sinus. repeat : the left thalamic hemorrhage has decreased in size. there has been an increase in the amount of intraventricular blood. otherwise, no change. l leni: no evidence of intraluminal thrombus. abd u/s: 1. normal gallbladder and no biliary ductal dilatation. 2. no hydronephrosis. echo: the left atrium is mildly dilated. overall left ventricular systolic function is normal (lvef>55%). right ventricular chamber size and free wall motion are normal. the aortic root is moderately dilated athe sinus level. the ascending aorta is mildly dilated. the aortic valve leaflets (3) are mildly thickened. there is no aortic valve stenosis. no aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. no mitral regurgitation is seen. the left ventricular inflow pattern suggests impaired relaxation. there is moderate pulmonary artery systolic hypertension. there is a small pericardial effusion. there are no echocardiographic signs of tamponade. brief hospital course: impression: 86yo man with pmh significant for htn presents with dysarthria and subtle right-sided weakness, along with delayed response time cognitively, and was found to have left thalamic bleed with slight extension into the ventricle, likely secondary to hypertension. his hospital course was complicated by multiple medical problems as detailed below. he was eventually transferred to the micu for hypercarbic respiratory failure, made dnr/dni and expired. hospital course: 1. hemorrhage - he was initially admitted to the stroke service for management. his blood pressure was difficult to control (see below). his exam remained unchanged with severe dysarthria, slight right hemiparesis, and waxing and mental status most likely secondary to metabolic encephalopathy (see medical problems listed below). 2. hypertension - his blood pressure remained poorly controlled initially: metoprolol was initiated but was ineffective. hydralazine was added and was initially successful at controlling the blood pressure, but his blood pressure increased again when the metoprolol was weaned. ace inhibitor was not started due to mild acute renal failure. his hctz was continued. 3. respiratory difficulties - due to his bulbar weakness, the patient was unable to clear his secretions. he was treated with aggressive chest pt and deep suction (which was difficult due to deviated trachea). a cxr on admission showed a loculated pleural effusion on the left, which was not seen on previous x-rays. the pulmonary service was consulted, and performed a diagnostic thoracentesis, which was consistent with a transudative effusion. echo was performed, which showed... diuresis was started. for wheezing, he was treated with albuterol and atrovent. a chest ct showed emphysematous changes. he continued to decline and developed hypercarbic respiratory failure requiring transfer to the micu. he was made comfort care and expired. 4. elevated alkaline phos - his alk phos remained elevated during his hospitalization. this may be secondary to his ankylosing spondylitis, but was rather high for this explanation. ggt was elevated, but abdominal ultrasound was negative. 5. ?cellulitis - he was initially treated for concern for l foot cellulitis with cefazolin. however, suspicion remained low and the antibiotics were discontinued without effect. leni was negative. 6. renal failure - he was noted to have rising creatinine and bun during the beginning of his hospital stay, with low uop at times. fena was 0.22% and urine eosinophils were negative, no hydronephrosis on abdominal ultrasound. he was initially treated with ivf, and when uop picked up and renal function stabilized, diuresis was started. 7. fen - he failed his speech and swallow. an ngt was placed by ir on , and tube feeds started . 8. code status - pt was made dnr/dni by his family after worsening neurologic deficits manifested in the setting of respiratory failure. he expired at 11:45 pm on . medications on admission: toprol lipitor protonix iron sulfate isosorbide discharge disposition: expired discharge diagnosis: respiratory failure. stroke. renal failure. discharge condition: expired. discharge instructions: n/a followup instructions: n/a procedure: enteral infusion of concentrated nutritional substances thoracentesis diagnoses: anemia, unspecified esophageal reflux pure hypercholesterolemia unspecified pleural effusion unspecified essential hypertension acute kidney failure, unspecified intracerebral hemorrhage acute respiratory failure postinflammatory pulmonary fibrosis cellulitis and abscess of foot, except toes 

Answer: The patient is high likely exposed to