Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins attending: chief complaint: cardiac arrest major surgical or invasive procedure: none history of present illness: 78 yo female with h/o mi x2, right carotid endarterectomy, htn, prediabetes, dyslipidemia, stroke, cri who experienced a slow speed mva this morning (backed up into another car), and afterwards was pale and started vomiting. she was transported to the ed by her niece at 0825 with initial rhythm ventricular tachycardia (rate around 160bpm) with no palpable pulse/hypotension (88/50mmhg) and was found to be cool, clammy and diaphoretic. she was synchronized cardioverted x 3 at the osh. she underwent cpr and initially got bolus of amiodarone and then an infusion of it. patient was then intubated. patient continued in vt receiving 2 more synchronized cardioversion, a bolus of lidocaine and a new bolus of amiodarone. rhythm then converted to ventricular fibrillation (shocked once) and then pea; patient received 1 amp of epinephrine. rhythm converted to vfib again, receiving 2 amps of epi, 1 bolus of lidocaine and 2gm of magnesium. pulse was then found; patient was in vfib for at least 10 minutes. . ecg after the code blue was concerning for inferior stemi. patient had ice packs placed and she was then transported to . en route, patient became agitated, trying to pull her ett, and patient got fentanyl and versed for sedation. . in the ed ecg showed pattern compatible with vtach (multiple morphologies) patient has been stable with sbps 100s-110s even when she flips into vt, which she does intermittently. patient continued on amiodarone gtt (2 mg/min) and lidocaine gtt (2 mg/min). she was also started on iv heparin and given pr aspirin. patient moves all four extremities, but is not responding to commands. she got a bolus of midazolam at the time of her arrival; so it??????s difficult to assess the patient??????s neurologic status. she has underwent ct head, c-spine, chest, abdomen and pelvis, as well as a chest x-ray. cardiac catheterization was deferred because the patients neurologic was not intact, as well as the most likely cause of her monomorphic vt is a previous scar and not a current mi. right ij was placed before transfer. ng tube and foley were also placed. sedation was switched to propofol. . labs were significant for wbc count of 15k. creatinine was 1.8 from an unknown baseline. troponin was elevated to 0.21. vbg showed 7.05/50/52/15, with lactate 10.9, free calcium 1.08. glucose was elevated to 400s. rectal temperature on transfer is 35.3 c. p 76, bp 115/60, map 74, fio2 of 100, peep 5, rate 16, sat 100% on transfer. . on arrival to the ccu, patient was intubated and sedated. initial vs??????s: 94.1, 54 77/41 100 on ac 550x20 fio2 100%. also of note upon arrival to the ccu the patient??????s left lower limb was noted to be cold, mottled (progressing all the way to her hip at that time, but more prominent below the knee) and no distal pulses could be palpated. vascular surgery was called to evaluate the patient, the main diagnosis considered was an acute arterial thrombotic event so the patient was given an initial heparin bolus at 80u/kg iv and then a heparin infusion was started. anti-arrythmic gtts were d/cd without recurrence of vt. because of hypotension patient was started on a low dose of phenylephrine (other vasopressors were considered but due to risk of triggering arrhythmias phenylephrine was selected). an axillary line was placed. and arctic sun protocol was initiated. past medical history: diabetes hypertension hypothyroidism gerd gout tia status post right endarterectomy. degenerative joint disease in both hips. history of l4-l5 level of central spinal stenosis social history: social history; - - tobacco history: non-smoker - etoh: rare etoh - illicit drugs: - family history: no family history of early mi, arrhythmia, cardiomyopathies, or sudden cardiac death physical exam: admission exam: gen: intubated, sedated heent: atraumatic, normocephalic. did not assess pupils neck: unable to appreciate jvp due to right ij cv: no m/r/g, soft heart sounds lungs: ctab bilaterally on anterior exam abd: depressed bowel sounds, no tenderness, non-firm, obese extremities: mottled on left side of lower extremities and cold, extends up to abdomen, no palpable or dopplerable pulses dp/pt on left, dopplerable pt on right pertinent results: labs: 11:26am blood wbc-15.0* rbc-3.45* hgb-11.5* hct-36.1 mcv-105* mch-33.2* mchc-31.8 rdw-15.7* plt ct-167 02:20pm blood neuts-87* bands-1 lymphs-10* monos-1* eos-0 baso-0 atyps-0 metas-1* myelos-0 08:55pm blood hypochr-normal anisocy-normal poiklo-2+ macrocy-normal microcy-normal polychr-normal burr-2+ 02:20pm blood hypochr-1+ anisocy-normal poiklo-normal macrocy-2+ microcy-normal polychr-normal 11:26am blood pt-11.5 ptt-45.9* inr(pt)-1.1 09:02pm blood pt-33.5* ptt->150 inr(pt)-3.3* 11:26am blood fibrino-232 09:58pm blood fibrino-105*# 09:58pm blood fdp-* 04:32am blood fibrino-172*# 04:32am blood fdp->1280* 02:20pm blood glucose-384* urean-39* creat-1.9* na-138 k-4.7 cl-106 hco3-14* angap-23* 04:32am blood glucose-264* urean-42* creat-2.2* na-135 k-4.8 cl-113* hco3-9* angap-18 04:32am blood alt-6255* ast-6553* alkphos-160* 11:26am blood ctropnt-0.21* 02:20pm blood ck-mb-24* mb indx-9.8* ctropnt-0.85* 02:20pm blood calcium-7.6* phos-5.0* mg-2.6 11:26am blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 04:32am blood d-dimer-greater th 11:27am blood type- temp-35.1 po2-52* pco2-50* ph-7.05* caltco2-15* base xs--17 intubat-intubated 04:49am blood type-art po2-165* pco2-22* ph-7.13* caltco2-8* base xs--20 11:27am blood glucose-429* lactate-10.9* na-136 k-4.3 cl-108 04:49am blood lactate-13.1* echo : the left atrium is normal in size. left ventricular wall thicknesses are normal. the left ventricular cavity is mildly dilated. there is severe regional left ventricular systolic dysfunction with focal thinning and akinesis of the inferior and inferolateral walls and hypokinesis of the inferior septum and apex. there is mild hypokinesis of the remaining segments (lvef = 25 %). right ventricular chamber size and free wall motion are normal. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. mild (1+) aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. there is no mitral valve prolapse. there is moderate thickening of the mitral valve chordae. mild (1+) mitral regurgitation is seen. there is borderline pulmonary artery systolic hypertension. there is an anterior space which most likely represents a prominent fat pad. impression: suboptimal image quality. severe left ventricular regional dysfunction with thinning/akinesis of the inferior and inferolateral walls, and hypokinesis of the inferior septum and apex c/w multivessel cad. mild mitral regurgitation. mild aortic regurgitation. borderline pulmonary hypertension. ct head : 1. no evidence of intracranial hemorrhage. 2. focal hypodensity involving the left caudate head. the chronicity of this finding is indeterminate, due to lack of previous exams, and it likely represents remote infarction or sequela of small vessel ischemic disease. ct spine : no evidence of acute fracture or malalignment. extensive degenerative disc disease of the cervical spine, most pronounced at c5-c6 and c6-c7. in the setting of high clinical suspicion for ligamentous or cord injury, mr may be considered for further assessment. abdominal xray : 1. distal end of nasogastric tube is seen within the stomach. a minimal amount of gas is seen within non-distended bowel with no evidence of bowel obstruction. no large amount of free area is seen, although detection is limited on a supine film. calcification of the right upper quadrant likely represents a gallstone. degenerative changes in the lower lumbar spine. bilateral hip replacements. followup imaging should be considered given the limited evaluation on this study. cxr : right ij catheter tip is in the upper right atrium. et tube tip is in standard position, 3.8 cm above the carina. ng tube tip is out of view below the diaphragm. there are low lung volumes. there is no evident pneumothorax. there is moderate cardiomegaly. widened mediastinum is unchanged from one hour earlier. diffuse opacity is in the left hemithorax suggests the presence of layering pleural effusion. left lung opacities could be atelectasis, contusion and/or aspiration. brief hospital course: hospital course: 78 yo female with h/o cad, cva, chronic renal insufficiency presents with cardiac arrest at osh, transferred here for concern of stemi, who underwent therapeutic hypothermia complicated by ischemic lle, dic, gi bleeding, and shock. patient was intubated on transfer and was cooled per protocol. given the patient's very poor prognosis and in accordance with family's wishes, care was withdrawn and the patient passed away. . # post cardiac arrest: pt was pulseless at osh with vt, vfib, and pea and underwent acls with multiple rounds of cardioversion/defibrillation and multiple rounds of epi before recovery of pulse. pt was in a pulseless rhythm for at least 10 minutes. vt was treated with lidocaine and amiodraone. the pt was transferred here for concern of stemi post resuscitation. she was initated on cooling protocol approximately 8 hours after being found pulseless. her course was complicated by cold and pulseless left lower extremity, gi bleed, multiorgan failure, lactic acidosis, and dic. the family understood the grave prognosis of the patient. in the setting of continued hypotension with max dose phenylephrine, multiorgan failure, and bleeding despite rewarming and cryoprecipitate, care was withdrawn per family wishes. . # shock/shock liver/acute renal failure: most likely mix between distributive and cardiogenic as suggested by wide pulse pressure despite alpha agonist therapy and elevated cvp (cardiogenic) with ef 25% on echo. phenylephrine chosen due to it being less likely to reintroduce vt. pt had clear evidence of end organ damage including shock liver, severe metabolic acidosis with elevated lactate to 13, no urine output, cold ischemic left leg. pt was given intermittent fluid boluses, prbc, ffp, cryoprecipitate, bicarbonate, hyperventilated, and started on vanc and zosyn to attempt to restore intravascular volume, treat the bleeding, and remedy the acidosis without avail. given grave prognosis, the family decided to not escalate care. . # ventricular tachycardia: most likely a result of scar causing monomorphic vt. resolved with lidocaine and amiodarone drip. these were discontinued on arrival to the floor without recurrence of vt. . #. dic: during the evening, pt began oozing at iv sites had brb per ng tube and brbpr. the patient was found to be in dic. plt dropped from 167->64, inr rose from 1.1 to 3.3, fdp rose to above 1280, and fibrinogen dropped from 232->105. the patient was rewarmed and heparin was stopped. the patient was given 1 unit of prbcs and 2 units of cryo and 2 units of ffp with some reversal of labs. . # gi bleed: pt with 400cc of brb via ngt, cleared slowly on lavage, hct stable. rectal exam demonstrated brbpr. the patient was started on iv pantoprazole. most likely this was ischemia reperfusion injury and dic. gi was consulted and agreed that endoscopy would not be of assistance in this case. . # coronary artery disease/? stemi: pt with inferior st elevations with reciprocal depressions on osh ecg that resolved. unclear if this was true event vs related to shock vs transient ischemic event that resolved. given unclear neurologic prognosis due to prolonged resuscitation efforts, she did not go to the cardiac catheterization lab. trop elevation non specific in setting of shock and arrest . # ischemic right leg: on arrival to the floor, left leg with non dopplerable dp/pt/popliteal pulse, cold, with mottling from lower extremity to all the way up the thigh. most likely ischemic from arrest and shock in setting of bad pvd. vascular surgery was consutled. patient was started on heparin gtt, which was stopped after development of dic. . # s/p mva: patient was in slow-moving car accident. there was no evidence of acute fractures/injuries on radiographic imaging. . # leukocytosis: patient with wbc count 15k which escalated to . most likely from stress induced post arrest but cannot rule out infection. potential etiologies included pneumonia (chest x-ray with left-sided infiltrate), uti, gastroenteritis (given h/o vomiting). pt pan cultured and started on vanc and zosyn . # diabetes mellitus: pt's blood sugars were very high >400s in setting of cooling and stress. pt was placed in insulin drip. . medications on admission: 1) lasix 20mg po bid 2) metoprolol 50mg po bid 3) quinipril 10mg po daily 4) levothyroxine 125mcg po daily 5) allopurinol 300mg po daily 6) vitamin d 1 tab po daily 7) multivitamin 1 tab po daily 8) aspirin 81mg po daily 9) protonix 20mg po bid 10) simvastatin 40mg po daily 11) aldactone 25mg po daily 12) plavix stopped last month 13) imdur 30mg po daily discharge disposition: expired discharge diagnosis: cardiopulmonary arrest dic ischemic hepatitis acute renal failure cardiogenic shock distributive shock ischemic left leg lactic acidosis discharge condition: n/a discharge instructions: n/a followup instructions: n/a procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours arterial catheterization central venous catheter placement with guidance diagnoses: acidosis esophageal reflux acute kidney failure with lesion of tubular necrosis diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled acute and subacute necrosis of liver unspecified acquired hypothyroidism hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage i through stage iv, or unspecified gout, unspecified chronic kidney disease, unspecified paroxysmal ventricular tachycardia other and unspecified hyperlipidemia defibrination syndrome cardiogenic shock old myocardial infarction ventricular fibrillation other motor vehicle traffic accident involving collision with motor vehicle injuring driver of motor vehicle other than motorcycle acute vascular insufficiency of intestine arterial embolism and thrombosis of lower extremity leukocytosis, unspecified osteoarthrosis, localized, not specified whether primary or secondary, pelvic region and thigh personal history of sudden cardiac arrest 

Answer: The patient is high likely exposed to