Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: the patient is a 69 year old, spanish speaking gentleman with a known history of coronary artery disease, peripheral vascular disease and diabetes mellitus type 2, who presented with approximately five to six weeks of dizziness and multiple falls, the last one about a week prior to admission. he also reported episodes of facial numbness and tingling and slurred speech at that time. he did have some blurry vision and diplopia several weeks ago. his dizziness did not appear to be vertiginous, but was postural. he had stable, chronic, paroxysmal nocturnal dyspnea and orthopnea without any chest pain with his falls and dizziness, but he did have some chronic angina. he used three to four nitro sublingually per day. he was admitted to the cardiology medical service on . past medical history: cad with three vessel disease. peripheral vascular disease status post axillo-bifemoral bypass graft and left below knee amputation. noninsulin dependent diabetes mellitus. history of mrsa. chf. chronic renal insufficiency. pulmonary hypertension. laboratory data: the patient was worked up for the cardiology service. labs on admission were as follows. white count 7.6, hematocrit 35.5, platelet count 264,000. pt 12.3, ptt 23.0, inr 1.0. admission ck 92, mb fraction 95, troponin 0.04. sodium 138, k 5.6, chloride 107, bicarb 22, bun 58, creatinine 2.7, blood sugar 123. anion gap 9. second ck was 90 with second troponin 0.03. chest x-ray showed old granuloma with no effusion, no infiltrate, no edema. ekg showed sinus brady with t wave inversions in leads 1 and l, no st depressions, also showed the presence of an old inferior myocardial infarction. hospital course: the patient was assessed also for bradycardia at that time. he was admitted to the c-med service. meds on admission were amitriptyline, aspirin, avandia, captopril, glyburide, imdur, lasix, lipitor, lopressor, triamcinolone, nitrostat p.r.n. he had a 40 pack year history of tobacco, quit 20 years ago. no history of alcohol. he was followed on the cardiology service. his dizziness was worked up. he was seen by the neurology service for workup of dizziness. they noted and assessed his multiple risk factors for stroke. he had presented with postural dizziness and unsteadiness. they recommended getting scans and giving him gentle hydration and to get physical therapy involved for gait evaluation. the patient was also seen by case management. he was followed every day by cardiology. he did start to have some episodes of what could be described as chest pain. his electrolytes were corrected as he was followed every day. lopressor was adjusted several times and then stopped when the patient had bradycardia on the morning of the 4th. he was also followed by his medical attending with plans to consult cardiology again. on the 4th he also had more shortness of breath with jaw pain in the morning. ekg was done and then repeated after sublingual nitroglycerin, which relieved his chest pain. he was still complaining of dizziness at that time whenever he sat up or was out of bed and felt like he was going to pass out or lose consciousness "as if a black shade came down." when he didn't move, he was okay. his blood pressure also rose into the 170s. nitrates were increased. the patient had a cardiology consult on the 5th. given his history, his symptoms, his bradycardia and his ekg evidence of an old inferior mi, they recommended getting cardiac catheterization done, but the issue was his creatinine at that time which was still 2.5 and 2.6. on again he had another episode of chest pain. they were going to perform a persantine mibi on the 6th in the afternoon, but it was not performed, given the fact that he had st segment depressions in inferior leads and in v4 to v6, so the plan was to then just proceed with cardiac cath. his pain was relieved with nitroglycerin. the patient's neuro followup exam on the 6th said that the mri showed right cerebellar encephalomalacia in the pica distribution. please read their final report dictated by cardiology and radiology. he was seen again by cardiology on the 7th. his creatinine had risen slightly to 2.8 with white count still normal at 9. his ekg from the 6th did show st depressions in v6 through v6 and 1 and avl as well as some lvh. decision was to start a nitroglycerin drip, give him mucomyst and the appropriate meds for premedication, to use a special dye to help protect his kidneys during cardiac catheterization. on the 7th he underwent cardiac catheterization that showed lvedp of 19, normal left main, 80 percent lad lesion of diagonal one bifurcation, ostial 80 percent circumflex lesion with a taper to a 90 percent lesion at the bifurcation with om1 and om2 and inferior lesions distally in om2. the right was the dominant vessel which was occluded ostially and supplied by collaterals. the patient was referred to cardiac surgery. he was seen by dr. who noted the history of left and right femoral bypass grafts prior to left bka. meds at that time were as follows: amitriptyline 25 mg p.o. q.d., aspirin 325 mg p.o. q.d., avandia 4 mg p.o. q.d., captopril 50 mg p.o. t.i.d, glyburide 5 mg p.o. q.d., imdur 60 mg p.o. q.d., lasix 80 mg p.o. q.d., lipitor 20 mg p.o. q.d., lopressor 100 mg p.o. b.i.d. triamcinolone and nitrostat were also given, doses are not listed. the patient had no known allergies. echo on showed lv ejection fraction of 35 percent with 1 to 2+ mr. his severe three vessel disease and his depressed lv function, he was referred for cardiac surgery. prior to operations his labs were white count 8.7, hematocrit 29.4, platelet count 275,000. inr 2.0, pt 17.3, ptt greater than 150 on heparin. prior to heparin, previous inr was 1.0 to 1.2. sodium 136, k 5.0, chloride 110, co2 19, bun 37, creatinine 2.5, blood sugar 138. amylase 137. lfts were normal. ua was negative. chest x-ray showed no acute cardiopulmonary disease. neurology evaluation, in summary, said there was no acute stroke and it was likely vascular in origin. on exam lungs were clear bilaterally. heart was regular rate and rhythm, no murmur was noted. abdomen was softly distended, but nontender with positive bowel sounds. extremities were warm with no edema, noting, of course, the left bka. on the 8th he had carotid ultrasound done which showed no stenosis on the left internal carotid, but 40 to 60 percent right internal carotid stenosis. mra of the neck prior to operation showed mild atherosclerotic disease at the right proximal ica without high grade stenosis. bilateral vertebral arteries and left ica demonstrated normal flow. mra of the head showed left vertebral artery ended in the pica. he was seen by the stroke team for evaluation, but it was clear the patient was going to have to have bypass surgery at that time, given his severe coronary artery disease and his recurrent symptoms, necessitating nitroglycerin drip and heparin. the last creatinine prior to surgery was 2.7. coags were repeated. on the patient underwent coronary artery bypass grafting times three with lima to the lad, vein graft to the om and vein graft to the pda. the patient was transferred to the cardiothoracic icu on a milrinone drip of 0.5 mcg per kg per minute and a levophed drip of 0.02 mcg per kg per minute. in the first 12 hours the patient was unresponsive despite the fact that propofol had been off for 12 hours with no signs of waking. on exam his pupils were very sluggish on the evening prior and nonresponsive in the evening. the physicians caring for the patient were aware. there was no response to painful stimuli, turning, suctioning, etc. please refer to the nursing exam notes. on postoperative day one the patient remained on a levophed drip at 0.04 and a milrinone drip at 0.25 and a neo drip at 1.25 as well as an insulin drip at 8 units per hour. pulse was 132 in sinus tachycardia with blood pressure 121/50 with pa pressure of 33/21 and index of 2.1 and mixed venous of 65 percent. his blood gas was 7.30/40/79/20/-5. he was sating 96 percent with an fio2 of 50 percent on simv. the patient was seen by the neurology resident the following morning. postoperatively white count rose slightly to 13.2, hematocrit 34.2. bun 29, creatinine 2.5. he received two units of ffp, five units of packed red blood cells, one unit of platelets. following the neurology resident's exam, the patient was intubated off sedation approximately 16 hours at the time of exam with no response to verbal or physical stimuli, no spontaneous movements. pupils were 1 mm and not clearly reactive. he showed no gag, cough reflexes, etc., on exam, no withdrawal or posturing and he was flaccid in all four extremities, noting, of course, the left bka. he had received approximately one dose of morphine 2 mg and developed a low grade fever with some hypotension requiring levophed and neo-synephrine pressor support. given his issues, the ct surgery team was concerned the patient was not stable enough to go for ct or mri scanning at that time and, hopefully, would be able to obtain a head ct when the patient was stable enough for transport. on postoperative day two eeg showed low voltage, no seizures. the patient received lasix and concentrated his drips. he was hemodynamically unstable, requiring bolusing and two units of packed red blood cells on milrinone at 0.25, neo 2.25. he had also been given aspirin and plavix and multiple doses of lasix at that time. chest x-ray showed an infiltrate in the left upper lobe and interstitial edema. he remained on simv ventilator support. creatinine rose to 3.5 from 2.5. white count rose to 15.9. on postoperative day three his drips were weaned down to neo 0.5. he was started on levaquin 250 q.48 for his pulmonary infiltrate, maintaining a mixed venous of 66 percent and remaining on simv ventilator support. renal consult was obtained. renal evaluated him and recommended holding on diuresis for now if he started making urine. no indication for dialysis at this time with the diagnosis of likely acute renal failure on top of chronic renal failure with atn probably related to hypotension. on the patient was still unresponsive. ct scan had been reviewed. the scan showed a total pca stroke causing lesions in the occipital cortices, thalamic, hypothalamic and brain stem and cerebellum. the patient's medulla probably was not affected as much or more as posterior circulation territories. the diagnosis by the neurology resident at that time was that the lesions were incompatible with life and they offered their services to talk to the family at that time. the patient remained completely neurologically unresponsive and the prognosis was grave. on the 13th this was discussed again by the stroke team with the cardiothoracic surgery team and the family wanted to withdraw care, but were awaiting family members from . the plan was agreed to by the neurology stroke team. the patient had another visit from the renal fellow on postoperative day five. note of the grave prognosis and catastrophic stroke was discussed as the family waited to gather all members. on postoperative day five white count was 14.3, hematocrit 25.8 with creatinine 3.9. the plan was again agreed to withdraw support at that time with no compressions or defibrillations. the swan-ganz catheter was pulled. at 2:30 in the afternoon discussion was held with the complete family, wife and children and dr. and it was decided that ventilatory support should be discontinued. the patient's wife stated that due to grave prognosis from stroke, the patient had stated that he would not want to live with no meaningful quality of life nor prolonged ventilatory support. the patient was extubated by the team at 14:34 with family at bedside. no spontaneous respirations were noted. the patient was asystolic at 14:41. the patient was pronounced at 14:41. the family was at the bedside during pronouncement of death by the cardiothoracic surgery team and dr. , the cardiothoracic surgery resident. again, the patient expired at 14:41 on . final diagnoses: 1. status post coronary artery bypass grafting times three. 2. status post axillo-bifemoral bypass grafting. 3. noninsulin dependent diabetes mellitus. 4. coronary artery disease. 5. hypertension. 6. hypercholesterolemia. 7. history of prior smoking. 8. chronic renal insufficiency. 9. peripheral vascular disease with additional left femoral bypass and left bka and right bypass grafting. 10. history of mrsa. 11. congestive heart failure. again, the patient expired in the cardiothoracic icu on . , m.d. dictated by: medquist36 procedure: extracorporeal circulation auxiliary to open heart surgery left heart cardiac catheterization coronary arteriography using a single catheter (aorto)coronary bypass of one coronary artery angiocardiography of right heart structures double internal mammary-coronary artery bypass transfusion of packed cells transfusion of other serum diagnoses: coronary atherosclerosis of native coronary artery intermediate coronary syndrome acute kidney failure with lesion of tubular necrosis congestive heart failure, unspecified diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled peripheral vascular disease, unspecified surgical operation with anastomosis, bypass, or graft, with natural or artificial tissues used as implant causing abnormal patient reaction, or later complication, without mention of misadventure at time of operation iatrogenic cerebrovascular infarction or hemorrhage 

Answer: The patient is high likely exposed to