Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: found down, s/p cardiac arrest major surgical or invasive procedure: endotracheal intubation history of present illness: 28 y/o male with unknown pmh who was found down in after 911 call. his identity was initially unknown on admission on and he was identified on . physician was driving by the scene with his wife and responded by initiating cpr approximately 15 minutes after man went down. ems arrived on the scene soon after call was made by unknown observer. patient was a well-dressed male thought to be in his 30s without any evidence of track marks or drug use. his pupils were dilated. he was intubated in the field. he was found in pea arrest. according to rhythm strips obtained from ems, his first rhythm was sinus bradycardia and he was given epinephrine and atropine. he then went into wct followed by mmvt. he was then shocked with 200 j and rhythm became pmvt and was shocked for the second time with 300 j. he then went into st and recovery of pulse. he received a total of 2 mg of epinephrine and 2 mg of atropine with 2 shocks (200 and 300j). he was then transported to ed. . in the ed, his pupils were fixed and dilated b/l. fast was negative. cxr revealed distended stomach. ct of head, chest, abdomen, pelvis, c-spine were all negative. the patient was transferred to the ccu hemodynamically stable on the ventilator. past medical history: phm (obtained from family after patient was identified): hep b+ hep c+ h/o ivda . psh: none known social history: sh: has a h/o drug abuse and has served time in jail. he currently lives with his grandparents in . family history: nc physical exam: pe: vitals: 97.4 153/87 105 14 100% on ac tv 600 rr 14 peep 5 fio2 100 general: sedated, on vent, myoclonic jerking heent: pupils 4 mm fixed and dilated. mmm. et tube in place. neck: no jvd. cv: st. normal s1, s2 without m/r/g. pulm: ctab, no wheezes or crackles. abd: soft, nt/nd with normoactive bs. ext: no c/c/e. 2+ dp b/l. neuro: intermittent myoclonic jerking. gcs 3. pertinent results: wbc-18.1* rbc-4.90 hgb-14.2 hct-40.3 mcv-82 mch-28.9 mchc-35.1* rdw-13.0 plt ct-271 . wbc-26.0* rbc-5.60 hgb-15.2 hct-43.5 mcv-78* mch-27.1 mchc-34.9 rdw-13.1 plt ct-226 . wbc-19.9* rbc-5.08 hgb-14.3 hct-40.1 mcv-79* mch-28.1 mchc-35.7* rdw-13.3 plt ct-222 . wbc-20.7* rbc-4.53* hgb-13.4* hct-36.3* mcv-80* mch-29.5 mchc-36.9* rdw-13.5 plt ct-223 . wbc-34.6*# rbc-5.41 hgb-14.8 hct-42.6 mcv-79* mch-27.4 mchc-34.8 rdw-13.6 plt ct-214 . wbc-29.2* rbc-5.33 hgb-15.0 hct-42.5 mcv-80* mch-28.1 mchc-35.2* rdw-14.0 plt ct-217 . wbc-29.9* rbc-4.26* hgb-12.0* hct-35.5* mcv-83 mch-28.1 mchc-33.7 rdw-14.0 plt ct-240 . wbc-30.4* rbc-4.15* hgb-11.5* hct-34.1* mcv-82 mch-27.7 mchc-33.7 rdw-13.8 plt ct-201 neuts-84.1* lymphs-10.4* monos-4.6 eos-0.7 baso-0.2 neuts-85.5* lymphs-10.2* monos-3.7 eos-0.4 baso-0.2 fibrino-174 urean-16 creat-1.5* . glucose-207* urean-16 creat-1.2 na-136 k-3.8 cl-102 hco3-20* angap-18 glucose-122* urean-13 creat-0.8 na-136 k-3.7 cl-104 hco3-18* angap-18 glucose-120* k-3.9 glucose-102 urean-11 creat-1.0 na-142 k-5.2* cl-111* hco3-24 angap-12 k-5.2* k-4.6 glucose-121* urean-11 creat-1.4* na-146* k-4.3 cl-116* hco3-25 angap-9 glucose-129* urean-12 creat-1.3* na-150* k-2.5* cl-113* hco3-22 angap-18 glucose-172* urean-15 creat-1.2 na-152* k-3.1* cl-117* hco3-21* angap-17 na-149* k-3.1* glucose-193* urean-18 creat-1.5* na-144 k-4.1 cl-112* hco3-15* angap-21* urean-23* creat-2.7*# na-142 k-4.9 cl-111* hco3-19* angap-17 glucose-186* urean-36* creat-5.2* na-139 k-5.7* cl-108 hco3-14* angap-23* glucose-159* urean-38* creat-5.5* na-138 k-4.8 cl-106 hco3-15* angap-22* alt-398* ast-224* ld(ldh)-339* alkphos-133* amylase-131* totbili-1.0 ck(cpk)-79 alt-423* ast-252* ck(cpk)-524* alkphos-130* totbili-1.9* alt-392* ast-251* ck(cpk)-298* alkphos-112 totbili-1.6* ck(cpk)-1768* alt-242* ast-656* alkphos-105 totbili-0.5 alt-377* ast-1447* ld(ldh)-4377* alkphos-119* totbili-1.1 alt-427* ast-1832* ld(ldh)-5187* alkphos-123* totbili-1.4 ck-mb-notdone ctropnt-<0.01 ck-mb-82* mb indx-15.6* ctropnt-1.45* ck-mb-55* mb indx-18.5* calcium-6.5* phos-9.0* mg-2.1 calcium-8.5 phos-1.6* mg-1.8 calcium-6.5* phos-9.0* mg-2.1 tsh-4.0 hbsab-positive hbcab-positive hav ab-negative hbsag-negative asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg carbamz-<0.5* phenyto-<0.6* valproa-<3* hcv ab-positive hcv ab-positive freeca-1.11* freeca-1.09* . imaging studies: 1. cxr the et tube tip is positioned approximately 2 cm from the carina. there has been interval placement of an og tube, extending beyond the ge junction, though the tip is not visualized on this study. the cardiac and the mediastinal contours are within normal limits. the lungs are clear. no pleural effusion or pneumothorax seen. the soft tissue and osseous structures are stable in appearance. impression: the og tube extends beyond the ge junction, though the tip is not visualized on this image. . 2. ct head, c-spine, chest, abdomen, pelvis w/contrast the study is limited secondary to motion. no definite hemorrhage is identified. no fracture or subluxation. no evidence of traumatic injury. . 3. echo the left atrium is normal in size. left ventricular wall thicknesses and cavity size are normal. overall left ventricular systolic function is mildly depressed with mild global hypokinesis more prominent in the distal inferior, distal septal and apical segments. no masses or thrombi are seen in the left ventricle. right ventricular chamber size and free wall motion are normal. the aortic valve leaflets (3) appear structurally normal with good leaflet excursion. there is no aortic valve stenosis. trace aortic regurgitation is seen. the mitral valve appears structurally normal with trivial mitral regurgitation. the pulmonary artery systolic pressure could not be determined. there is no pericardial effusion. ef 45% . 4. eeg this is a markedly abnormal electroencephalogram due to the presence of a suppressed background interrupted by rhythmic sharply contoured and evolving epileptiform activity. this eeg is consistent with brief periods of electrographic seizures on a suppressed background. the team was called and notified of this result. . 5. eeg this is an abnormal 24 hour bedside telemetry due to the presence of a low voltage, diffusely slow background activity. this suppressed activity may be seen with diffuse subcortical dysfunction, as occurs with cns depressant medications or diffuse cerebral ischemia. . culture results scx gram stain (final ): >25 pmns and <10 epithelial cells/100x field. 4+ (>10 per 1000x field): gram positive cocci. in pairs, chains, and clusters. 2+ (1-5 per 1000x field): gram positive rod(s). 1+ (<1 per 1000x field): gram negative rod(s). respiratory culture (final ): moderate growth oropharyngeal flora. staph aureus coag +. moderate growth. ucx urine culture (final ): staphylococcus, coagulase negative. 10,000-100,000 organisms/ml. all bcx were negative brief hospital course: 28 y/o male with no known pmh at the time found down s/p cardiac arrest. pea then vt then vf. successful resuscitation with 2 shocks. the patient suffered anoxic brain injury s/p cardiac arrest. . 1. s/p cardiac arrest the patient was initially found down in . please see above hpi for further details. after arriving at ed, he was ruled out for any traumatic injury. he was then transferred to the ccu for further evaluation and management after his cardiac arrest. due to his recent vf arrest, the hypothermia protocol was initiated and the patient was cooled for 18 hours. gcs before protocol was initiated was 3. the patient met all inclusion criteria and he had none of the exclusion criteria. following the cooling protocol, he was re-warmed. upon initial arrival to the ccu, his ekg was analyzed for any evidence of arrhythmia or abnormality that would explain his cardiac arrest. echocardiogram was performed soon after the patient arrived in the ccu which was negative for any structural abnormality of the heart. see full echo report above for details. initial tox screen was positive for opiates and benzos. the leading diagnosis after cardiac causes were ruled out were possible substance abuse causing respiratory suppression then hypoxia which may have induced the cardiac arrest. since the patient was never awake from presentation, obtaining any hpi was impossible. after talking with his family, they mentioned that he had a history of substance abuse and was most likely using heroin and/or benzos prior to being found down. the exact etiology of what caused the patient's cardiac arrest remained uncertain. due to the patient's anoxic brain injury and after consulting neurology, the family decided to withdraw support on and the patient expired soon afterwards. . 2. anoxic brain injury after the patient was transferred to the ccu, neurology was consulted so they could perform an exam while patient was off sedation prior to initiating cooling protocol. upon arrival to the ccu, the patient was having myoclonic jerking which was more consistent with anoxic brain injury than seizure activity. neurology recommended eeg and then continued eeg monitoring. on initial exam, the patient did have a gag reflex and corneal reflexes. however, as his hospital course progressed, all of his reflexes were absent, even off sedation for 48 hours. he was monitored continuously with eeg which showed no signs of activity. he was maintained initially on dilantin to control any seizures secondary to anoxic brain injury and that was eventually discontinued. organ bank saw the patient and determined that he was not a suitable candidate for organ donation. during the last two days of the patient's hospitalization, he began to show evidence of multi-organ failure and required pressor support to maintain his bp. after several discussions with the family, ccu team, and neurology, it was communicated that the patient would never return to normal functioning. the family decided in complete agreement to withdraw support on and the patient passed away soon after the night of the 16th at 21:15. the family declined autopsy. medications on admission: unknown discharge medications: the patient expired on . discharge disposition: expired discharge diagnosis: the patient suffered a cardiac arrest and subsequent anoxic brain injury. the family decided to withdraw support on and the patient expired later that evening at 21:15. discharge condition: the patient expired. discharge instructions: the patient expired. followup instructions: the patient expired. procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more diagnoses: unspecified essential hypertension acute kidney failure, unspecified other convulsions hypopotassemia cardiac arrest anoxic brain damage ventricular fibrillation hyperosmolality and/or hypernatremia opioid abuse, unspecified other specified disease of white blood cells disorders of magnesium metabolism hepatitis c carrier 

Answer: The patient is high likely exposed to