Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: headache, language changes major surgical or invasive procedure: intubation history of present illness: 79 yo rh man with diet controlled dm, high cholesterol, memory loss who presents with not feeling well x 3 days. his daughter spoke with him this am and he stated he didn't feel well, daughter noted trouble articulating words. brought him to dr. office for an appt, thought he was doing reasonably well, reassured patient and sent him home. brother in law was not convinced of his wellness because he seemed to have things to say but unable to say them, thus brought him to the ed. patient walked into the ed around 11am. bp 145/75. head ct revealed ich left temporal lobe. during his ed stay, he became more confused, less fluent, more slurred (after dilantin infusion, bp 107/50 at this time). repeat head ct essentially shows no change (to my eye). 4x2cm bleed left temporal lobe, x 8 slices. after head ct, bp was 120's and he improved a bit in that he became more fluent (nonsensical words, but fluent at least). in ed got dilantin 1.3g iv and dex 10mg iv x 1. neurosurg consulted and rec'd the above plus mri, neuro c/s. considering taking him to the or. also, per son in law, he was was not able to carry a conversation 24 hrs ago on a walk outside. time of onset of symptoms is not clear - at minimum was 24 hrs ago, possibly more like 2-3 days. patient lives alone. past medical history: left corneal transplant on eye drops no other medical problems per daughters, but per pcp : diet controlled dm pmr high cholesterol memory loss anemia lbp chronic dizziness orthostatic hypotension no h/o stroke, cancers. social history: lives alone with daughter on , widowed, 3 daughters, smoker 1ppd x many years, no etoh/drugs. former worker for inspector. family history: no h/o stroke or ich physical exam: pe: vitals: 99 74 145/75 16 96% ra gen: elderly man, picking at sheets and lines, talking nonsensically, about to climb out of bed (quite strong) heent: nc/at, anicteric sclera, mmm neck: supple, no carotid bruits chest: cta bilat cv: rrr without mur abd: soft, nt/nd, +bs extrem: no edema, wwp neuro: mental status: initially, awake, not answering any questions including orientation questions, or following simple commands. babbling nonsensical words (one or two at a time), nonfluent, globally aphasic,unable to name, + neologisms. grabbing at lines, etc. later, after bp was a little higher, was fluent, albeit still talking nonsensically. would mimic commands but not follow any simple commands. does not appear to neglect either side as he orients to each side spontaneously. cranial nerves: pupil exam: left pupil is surgical, right is 1mm and min reactive. ? ptosis on the left. eom exam: intact to all movements facial symmetry: mild right lower face weakness gag reflex: present. tongue appears midline when he mimics me. motor: slightly increased tone throughout, moving all extremities vigorously, holds arms up x10 sec, holds legs up x 5 sec. no asymmetry. no drift. sensory: withdrawls and winces x4. he even wags his finger at me as if to say, "don't do that!" reflexes: 1+ ue, 2+ le, upgoing toe right, downgoing on the left. pertinent results: 10:31pm type-art temp-37.4 rates- tidal vol-650 peep-5 o2-50 po2-201* pco2-34* ph-7.43 total co2-23 base xs-0 intubated-intubated vent-controlled 10:31pm lactate-1.1 10:31pm freeca-1.09* 02:50pm urine color-yellow appear-clear sp -1.017 02:50pm urine blood-neg nitrite-neg protein-neg glucose-neg ketone-15 bilirubin-neg urobilngn-neg ph-5.0 leuk-neg 11:40am glucose-147* urea n-15 creat-1.2 sodium-137 potassium-4.2 chloride-101 total co2-23 anion gap-17 11:40am alt(sgpt)-11 ast(sgot)-14 alk phos-63 amylase-68 tot bili-1.4 11:40am lipase-17 11:40am albumin-4.7 calcium-9.8 phosphate-3.0 magnesium-1.7 11:40am wbc-10.7 rbc-3.89* hgb-12.1* hct-34.7* mcv-89 mch-31.1 mchc-34.8 rdw-13.7 11:40am neuts-76.4* lymphs-18.8 monos-3.4 eos-0.9 basos-0.5 11:40am plt count-241 11:40am pt-13.3* ptt-25.3 inr(pt)-1.2* ekg: sinus rhythm and occasional ventricular ectopy. compared to the previous tracing of the rate has increased. there is occasional ventricular ectopy. otherwise, no diagnostic interim change. cxr ap upright chest: the tip of an endotracheal tube terminates in a standard position, 6 cm above the level of the carina. the tip of a nasogastric tube terminates in the proximal stomach with its side port located at the gastroesophageal junction. lung volumes are low. the heart size is normal. the aorta is slightly tortuous without focal dilatation. alveolar airspace opacities are identified in the bilateral infrahilar region are concerning for aspiration/developing infiltrate. the pulmonary vasculature is prominent with upper zone redistribution reflecting likely mild interstitial edema. no pneumothorax is identified. the visualized soft tissue and osseous structures are unremarkable. impression: endotracheal tube in a standard position. no pneumothorax. patchy infrahilar opacities concerning for aspiration/developing infiltrate. mild pulmonary edema. ct brain initial:non-contrast head ct. findings: there is an approximately 4.4 x 2.1 cm intraparenchymal area of high density within the left temporal lobe. surrounding hypodensity is consistent with edema. there are small amounts of mass effect exerted upon the frontal of the left lateral ventricle as well as focal effacement in the left temporal lobe. there is no shift of the normally midline structures. there is slight prominence of the contralateral ventricle, without frank hydrocephalus. the basal cisterns are well visualized. an approximately 1.1 cm area of hypodensity involving the cortex of the left frontal lobe is best seen on series 2, image 22. additionally, multiple hypodensities are seen within the periventricular white matter as well as within the left basal ganglia consistent with chronic small vessel infarction. the visualized paranasal sinuses and mastoid air cells are pneumatized and well aerated. atherosclerotic calcifications are noted within the distal vertebral arteries as well as within the intracranial and internal carotid arteries. the surrounding soft tissue structures including the orbits appear unremarkable. impression: 1. acute lobar hemorrhage in the left temporal lobe with surrounding edema. in elderly person, this constellation of findings raises the possibility of amyloid angiopathy. 2. wedge-shaped hypodensity in the left frontal lobe suggests a subacute or chronic infarction in this region. 3. extensive periventricular white matter chronic small vessel infarction as well as findings consistent with a lacunar infarction in the left basal ganglia. findings were relayed to the emergency department dashboard at 12 p.m. on . ct brain (repeat) non-contrast head ct scan. comparison: , at 11:41 a.m. findings: again seen is a 4.2 x 2.4 cm intraparenchymal area of high density within the left temporal lobe with a considerable amount of surrounding edema. this has not significantly changed compared to the prior examination. there is a small degree of mass effect exerted on the frontal of the left lateral ventricle as well as focal effacement of the left temporal lobe. there is slight trapping of the right lateral ventricle. again seen are multiple subcortical, periventricular, and left basal ganglia hypodensities consistent with chronic small vessel infarction. the osseous structures are unremarkable. the visualized paranasal sinuses and mastoid air cells are well aerated. atherosclerotic calcifications are noted within the distal vertebral arteries as well as the intracranial and internal carotid arteries. impression: acute lobar hemorrhage in the left temporal lobe with surrounding edema. the differential diagnosis includes hemorrhagic contusion and amyloid angiopathy. underlying neoplasm or avm cannot be excluded. correlation of the onset of the patient's symptoms to the initial head ct scan may help narrow the differential diagnosis. mri with contrast would better evaluate for neoplasm if this is of clinical concern. mr head w & w/o contrast; mra brain w/o contrast reason: evaluate head bleed, with and without contrast, please. pos contrast: magnevist medical condition: 79 year old man with confusion, ich on ct reason for this examination: evaluate head bleed, with and without contrast, please. possible mass. clinical history: confusion. hematoma on ct. technique: multiplanar pre and postcontrast t1-weighted images, axial t2- weighted, susceptibility flair and diffusion-weighted images were obtained. comparison is made to a ct examination performed at 1:30 p.m. on the same day. findings: there is a superficial hematoma in the anterolateral left temporal lobe, not significantly changed in size compared to the ct. it measures approximately 6 cm in greatest dimension on the sagittal t2-weighted images, unchanged allowing for the differences in technique. it consists largely of deoxyhemoglobin. there are few small areas of t1 hyperintense methemoglobin within it. there is surrounding flair hyperintensity. there is effacement of the left cerebral sulci and some flattening of the left lateral ventricle with minimal, if any, shift of the septum pellucidum. there is extensive flair hyperintensity in the deep cerebral white matter, corresponding to low attenuation seen on the ct from 11:40 a.m. the apparent small infarct in the left frontal lobe seen on that ct is not identified, perhaps the cavity of a chronic infarct has been compressed by the left-sided mass effect. there is no evidence of a recent infarct on the diffusion- weighted images. the brainstem and cerebellum are intact. no enhancing lesions are seen. there is susceptibility effect associated with the hematoma, as well as in the basal ganglia, which is normal in this aged patient. there may be minimal gyriform susceptibility effect in the right temporoparietal region. the ambient cisterns are roughly symmetric and no mass effect on the brainstem is seen. impression: 1. there is a 6 cm left temporal hematoma unchanged in size compared to studies dating back to 11:40 a.m. on the same day. 2. no enhancing lesions are seen elsewhere to suggest hemorrhagic metastatic disease. there may be minimal susceptibility effect superficially in the right temporoparietal region, but no diffuse abnormalities are seen to strongly suggest amyloid angiopathy, although obviously that diagnosis remains possible. 3. the degree of mass effect is unchanged. 4. there are extensive microvascular changes in the cerebral white matter. mra of the head. technique: a 3d time-of-flight study was derived from overlapping axial slabs through the inferior cranium. findings: there is symmetric decreased signal intensity in the siphons, likely artifactual. the distal left vertebral artery is poorly seen where it curves inferiorly at the c1 level. the basilar artery is widely patent. the major branches of the cerebral arteries are normal. no abnormal vessels are seen in the region of the left temporal hematoma. impression: allowing for slight artifact, normal mra of the skull base/circle of levels. an atretic right a1 segment is noted. ct head:routine non-contrast head ct. comparison: . findings: again seen is intra-parenchymal hemorrhage within the left temporal lobe with slight interval increase in the surrounding hypodensity consistent with edema. there has been no change in the degree of mild mass effect on the lateral ventricle. again there is no shift of midline structures. no new foci of hemorrhage are identified. the surrounding osseous and soft tissue structures are unremarkable. the visualized paranasal sinuses show fluid within sphenoid sinus and mild maxillary sinus mucosal thickening. impression: left temporal intra-parenchymal hemorrhage, unchanged with mild increase in surrounding hypodensity consistent with edema. no change in the degree of mild mass effect without midline shift. brief hospital course: 79 yo man on asa who presents with 2-3 days of feeling unwell with headaches, found to have a left temporal lobe intracranial hemorrhage on head ct in the ed. on exam he initially had a wernickes type aphasia (fluent nonsensical words, neologisms, cannot repeat or name), and right lower facial weakness. he was intubated and admitted to the neurosurgical service/icu because of the proximity of the bleed to the cerebral peduncle, the differential diagnosis of which was thought to include amyloid angiopathy, hemorrhagic conversion of a stroke, mass/tumor, avm/aneurysm, less likely htn (has no h/o htn), less likely trauma (no history of this but patient lives alone), toxins, aspirin use, sinus thrombosis (less likely as only has one bleed, not multifocal). he was loaded with dilantin, and placed on maintenance dose at 100 mg 3x/d. mri/a was performed which showed no obvious underlying lesion (ie, vascular or neoplastic lesion). head ct was repeated and was unchanged. antiplatelets and anticoagulation was avoided. bp was kept between 130 and 150 systolic. his family declined surgical interventions. he was extubated on and transferred to the neurology service/floor for further workup of the cerebral hemorrhage. imaging was suggestive of amyloid angiopathy because of the lobar/cortical location of the bleed, although no microbleeds were present. however, on further history he recalled bumping his head on a car door two weeks before admission, and thus traumatic hemorrhage could not be ruled out. exam improved, and he made less paraphasic errors with improved comprehension. strength and fluency were normal. on the day of discharge, he felt well, was walking around his room, and had at least 90% comprehension, with some anomia and occasional paraphasic errors when asked to name, but otherwise normal. pt recommended home pt, and he was discharged in stable condition. medications on admission: none except asa 81 and eye drops for cornea transplant, occ sleeping pill discharge medications: 1. acetaminophen 325 mg tablet sig: 1-2 tablets po q4-6h (every 4 to 6 hours) as needed for pain;fever. 2. senna 8.6 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 3. cholecalciferol (vitamin d3) 400 unit tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 4. calcium carbonate 500 mg tablet, chewable sig: one (1) tablet, chewable po tid (3 times a day). disp:*90 tablet, chewable(s)* refills:*2* 5. phenytoin sodium extended 100 mg capsule sig: one (1) capsule po three times a day: take extra 60 mg with night time dose (see other prescription). disp:*90 capsule(s)* refills:*2* 6. phenytoin sodium extended 30 mg capsule sig: two (2) capsule po at bedtime: take with night-time dose to total 160 mg at night. disp:*60 capsule(s)* refills:*2* 7. outpatient physical therapy diagnosis: intracerebral hemorrhage discharge disposition: home with service facility: greater vna discharge diagnosis: intraparenchymal hemorrhage - left temporal lobe discharge condition: good - language/comprehension to at least 90% baseline discharge instructions: please md or return to ed if you experience new symptoms of speech or language problems, new weakness, numbness, visual or hearing changes, problems walking, severe headache or vertigo. followup instructions: please call dr. office for neurology appointment in 6 weeks; (. office is located on . provider: , apg (sb) date/time: 8:30 md, procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube diagnoses: anemia, unspecified pure hypercholesterolemia diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled unspecified intracranial hemorrhage 

Answer: The patient is high likely exposed to