Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: abnormal cxr, anemia major surgical or invasive procedure: intubation central line placement bronchoscopy arterial line placement history of present illness: the patient is an 84m with prostate ca, cad, htn who presents with 2 weeks dry cough. he saw his hematologist who ordered a cxr for the cough, and it showed extensive heterogenous opacification in the right lung. because of these abnormal findings, his pcp told him to come to the ed for evaluation. he has had the intermittent dry cough for about two weeks but has not had fevers, chills, weight loss, change in appetite, shortness of breath, nausea, vomiting, diarrhea. he has noticed some fatigue, as he used to walk two miles a day until the cough started, but notes he is limited by fatigue, not doe. no other symptoms. in the ed he was noted to have a dropping hct (25.4, down from 29.6 the previous day). he has not noted blood in his stool or had any lightheadedness. he was guaiac negative. he was being worked up for anemia and was not found to have evidence of iron, folate or b12 deficiency in . spep was also normal. after discussion with his pcp by the , he is being admitted for expedited workup of cxr findings and anemia. pulmonology was consulted in the ed. ros: -constitutional: wnl weight loss fatigue/malaise fever chills/rigors nightsweats anorexia -eyes: wnl blurry vision diplopia loss of vision photophobia -ent: wnl dry mouth oral ulcers bleeding gums/nose tinnitus sinus pain sore throat -cardiac: wnl chest pain palpitations le edema orthopnea/pnd doe -respiratory: wnl sob pleuritic pain hemoptysis cough -gastrointestinal: wnl nausea vomiting abdominal pain abdominal swelling diarrhea constipation hematemesis hematochezia melena -heme/lymph: wnl bleeding bruising lymphadenopathy -gu: wnl incontinence/retention dysuria hematuria discharge menorrhagia -skin: wnl rash pruritus -endocrine: wnl change in skin/hair loss of energy heat/cold intolerance -musculoskeletal: wnl myalgias arthralgias back pain -neurological: numbness of extremities weakness of extremities parasthesias dizziness/lightheaded vertigo confusion headache -psychiatric: wnl depression suicidal ideation -allergy/immunological: wnl seasonal allergies past medical history: prostate cancer diagnosed , getting treated with hormonal therapy, followed by dr. cad, s/p cabg dyslipidemia htn nsvt, sss s/p icd/pm severe left ventricular dysfunction (ef 20% in ) social history: he lives in with his wife. has a son who has had multiple bypass surgeries and significant cardiac disease. he had a daughter who passed away from cancer. he has no smoking history. reports drinking alcohol, drinking one glass of alcohol every night prior to dinner. he is retired. family history: son w/ multiple cabgs daughter w/ cancer physical exam: physical exam: appearance: nad vitals: t: 96.4 bp: 124/59 hr: 71 rr: 20 o2: 100% 2l eyes: eomi, perrl, conjunctiva clear, noninjected, anicteric, no exudate ent: moist neck: no jvd, no lad, no thyromegaly, no carotid bruits cardiovascular: rrr, nl s1/s2, no m/r/g respiratory: exam limited by coughing, poor air movement, diffusely wheezy gastrointestinal: soft, non-tender, non-distended, no hepatosplenomegaly, normal bowel sounds musculoskeletal/extremities: no clubbing, no cyanosis, no joint swelling, no edema in the bilateral extremities neurological: alert and oriented x3, fluent speech, no pronator drift, no asterixis, sensation wnl, cnii-xii intact, strength in upper and lower extremities bilaterally integument: warm, no rash, no ulcer psychiatric: appropriate, pleasant hematological/lymphatic: no cervical lymphadenopathy pertinent results: 11:15am glucose-116* urea n-23* creat-1.1 sodium-133 potassium-4.4 chloride-98 total co2-26 anion gap-13 11:15am ck(cpk)-117 11:15am ck-mb-4 11:30am ctropnt-0.01 11:15am wbc-7.9 rbc-2.85* hgb-8.5* hct-25.4* mcv-89 mch-29.9 mchc-33.6 rdw-13.9 11:15am plt count-274 10:10am ld(ldh)-259* tot bili-0.5 dir bili-0.2 indir bil-0.3 10:10am haptoglob-492* 10:10am igg-1510 iga-278 igm-58 10:10am ret aut-1.3 09:45am wbc-7.9 rbc-3.34* hgb-10.0* hct-29.6* mcv-89 mch-29.9 mchc-33.7 rdw-14.2 09:45am neuts-74.5* lymphs-11.7* monos-4.7 eos-9.0* basos-0.2 09:45am plt count-266# 03:30pm urine color-yellow appear-clear sp -1.021 03:30pm urine blood-sm nitrite-neg protein-30 glucose-neg ketone-neg bilirubin-neg urobilngn-1 ph-5.0 leuk-neg 03:30pm urine rbc-0-2 wbc-2 bacteria-few yeast-none epi-0-2 pa and lateral chest radiograph: multiple midline sternotomy wires are unchanged in position. small surgical clips are seen along the left cardiac border, compatible with prior cabg. the two-chamber pacemaker with icd is seen with leads in the right ventricle and right atrium, unchanged. there is mild cardiomegaly. the mediastinal and hilar contours are unchanged. pulmonary vasculature is unremarkable. there is no pneumothorax or large pleural effusion. new, extensive, heterogeneous opacification is seen predominately in the right lung, with peripheral consolidation and lesser degree of central opacification. right- sided volume loss suggests a chronic process. left basilar opacities are compatible with atelectasis, and the left lung is otherwise clear. impression: compared to study, new extensive heterogeneous opacification in the right lung with peripheral consolidation and lesser degree of central opacification. loss of right lung volume suggests a subacute process. differential is broad, including pneumonia, postinfectious and cryptogenic organizing pneumonia, multifocal bronchioloalveolar cell carcinoma, chronic eosinophilic pneumonia, churg- vasculitis, amiodarone toxicity. cxr: no interval change over one day in appearance of diffuse airspace opacity involving the right hemithorax. additional opacity seen in the left base, also unchanged. original differential diagnosis stands and multifocal pneumonia cannot be excluded. ct chest: findings the patient is intubated. pooling of secretions are above the cuff of the endotracheal tube. small layering bilateral non-hemorrhagic pleural effusions are increased on the right and new on the left. diffuse extensive areas of ground-glass opacity, peribronchial consolidation, and bronchiectasis have worsened in the left lung, minimally improved in the right upper lobe. right central catheter tip is in the mid svc. transvenous pacemaker lead terminates in a standard position. ng tube tip is out of view below the diaphragm. dense calcifications are in the native coronary arteries. there is mild-to-moderate cardiomegaly. the cardiac are hypodense. this suggests anemia. calcification in the aortic valve is of unknown hemodynamic significance. ap window and prevascular lymph nodes have increased in size; for instance, to 8 mm from 5 mm and to 5 mm from 3 mm respectively. this examination is not tailored for subdiaphragmatic evaluation, and the upper abdomen is unremarkable. there are no bone findings of malignancy. in the abdomen, previously described renal lesion is not included in this examination. impression: worsening of pre-existing opacity in the left lung and slight improvement of the abnormalities in the right upper lobe the left atrium is normal in size. left ventricular wall thicknesses are normal. the left ventricular cavity size is top normal/borderline dilated. there is severe regional left ventricular systolic dysfunction with akinesis of the anterior septum, anterior wall and apex. there is mild hypokinesis of the inferior septum and inferior wall. there is an anteroapical left ventricular aneurysm. there is no ventricular septal defect. right ventricular chamber size and free wall motion are normal. the aortic valve leaflets are mildly thickened (?#). there is no aortic valve stenosis. no aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. there is no mitral valve prolapse. trivial mitral regurgitation is seen. there is moderate pulmonary artery systolic hypertension. there is no pericardial effusion. impression: severe regional lv systolic dysfunction consistent with multi-vessel cad. no significant valvular abnormality seen. moderate pulmonary artery systolic hypertension. compared with the prior study (images reviewed) of , lv systolic dysfunction appears regional on the current study (as it did on study of ). estimated pulmonary artery systolic pressures are higher on the current study brief hospital course: mr. is an 85 yo m with cad s/p cabg/icd,chf ef20% prostate ca who presents with dry cough found to have diffuse right sided patchy peripheral opacities on cxr, transferred to the icu for hypoxia. he had a hospital course and was intubated with persistant hypoxic resp failure. etiology was unclear with concern for eosinophilic pneumonia, hap, pulm edema. due to progressive decline and poor prognosis, family decided to make him comfort measures only and patient passed away on 17:10. hospital course by problem: . #hypoxic resp failure: over his hospital course, he had continued decline of respiratory status of unclear etiology. initial bronchoscopy revealed high eosinophilia and he was felt to have eosinophilic pneumonia and treated with a course of steroids although he did not have a dramatic improvement in his resp status. shortly after this, he was transferred to the icu for worsening hypoxia and intubated. repeat ct chests showed worsening progression of disease adn well as new infiltrates concerning for infectious etiology. initially, it was also felt there was a component of pulm edema and he was aggressively diuresed. at this point, after meeting w/ family and ct surgery, it was felt that vats was not an option given his tenuous status. repeat bronch on was w/o infectious results. sputum culture ultimately with sparse orpharyngeal flora. galactomannan, tularemia, strongyloides, and ige were unremarkable. he was treated with broad coverage antibiotics with no improvement in resp status and further worsening of disease. finally, it was decided by family, given his additional decline of other organ systems, to withdraw care. # ? vap pneumonia - s/p bronch, infectious studies with no obvious source. no microorganisms on gram stain from bal. sputum culture ultimately with sparse orpharyngeal flora. on ct, infiltrate had the appearance of progressive underlying process rather than new consolidation. fever and infiltrate on cxr may not really have been vap but rather part of underlying lung disease. # diffuse right peripheral infiltrates - bal 30% eosinophils, also with peripheral eosinophilia most likely acute eosinophilic pneumonia. pcp negative, respiratory viral antigens neg, bal neg for bacterial pathogens. legionella neg. per pulmonary consult, he may have "eosinophilic process secondary to malignancy or something like a non-steroid responsive boop". beta-glucan and galactomannan negative; mycolytic blood culture negative. histo negative. chest ct worse. # fever: during his hospital course, he intermittently had high fevers with no clear infectious etiology isolated. blood, urine, sputum, bal, stool all negative. ct sinuese negative. cvl were changed. # arf: he had waxing and course, but towards end of hospital stay, his creatinine was rapidly rising. urine sediment revealed muddy brown casts c/w atn. # lue dvt - increased welling l>r, confirmed on u/s to involve axillary and probably brachial veins. heparin gtt was started. # anemia: he was noted to have anemia since , slowly trending down but without any acute drops and no evidence of bleeding. he has been followed by / of hematology. work up thus far is negative for iron/folate/b12 deficient. thought possibly from hormone therapy for prostate ca. no evidence of hemolysis on admission. bone marrow biopsy showed anemia of chronic dz, maybe mds. transfused 1unit with good response. found to have warm autoimmune ab. # warm autoimmune ab: previously no evidence of hemolysis with elevated haptoglobin. hapto/ldh not suggestive of hemolysis. # vt/ nsvt, sss, s/p pm/icd: has had runs of vt; likely in setting of intubation. had 2 further episodes of svt , ecg w/ new t-wave changes but ces negative x3. ep was aware and felt this was most likely related to intubation. # cad s/p mi/cabg: no evidence of acute ischmia on arrival to icu, chest pain free. ekg with lbbb at baseline, no change. ecg w/ new t-wave changes but ces negative x3. #chronic systoic heart failure - likely ischemic cardiomyopathy - echo with ef of 20%, pcwp >18, 1+ mr, 1+tr. repeat tte unchanged. # h/o prostate cancer: receiving hormonal therapy q3 months but patient reports his treatment was skipped this month due to his symptoms. he is followed by dr. . medications on admission: lipitor 80mg daily lisinopril 10mg daily asa 325mg daily toprol xl 50mg daily flomax 0.4mg qhs discharge medications: patient expired discharge disposition: expired discharge diagnosis: patient expired discharge condition: patient expired discharge instructions: patient expired followup instructions: patient expired procedure: venous catheterization, not elsewhere classified venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more insertion of endotracheal tube enteral infusion of concentrated nutritional substances non-invasive mechanical ventilation biopsy of bone marrow arterial catheterization closed [endoscopic] biopsy of bronchus closed [endoscopic] biopsy of bronchus closed endoscopic biopsy of lung diagnoses: hyperpotassemia other iatrogenic hypotension anemia of other chronic disease coronary atherosclerosis of native coronary artery acute kidney failure with lesion of tubular necrosis urinary tract infection, site not specified congestive heart failure, unspecified unspecified essential hypertension gout, unspecified aortocoronary bypass status other chronic pulmonary heart diseases paroxysmal ventricular tachycardia other specified forms of chronic ischemic heart disease myelodysplastic syndrome, unspecified other and unspecified hyperlipidemia malignant neoplasm of prostate other left bundle branch block chronic systolic heart failure paralytic ileus automatic implantable cardiac defibrillator in situ cardiac pacemaker in situ unspecified hereditary and idiopathic peripheral neuropathy other and unspecified coagulation defects examination of participant in clinical trial pulmonary eosinophilia personal history of colonic polyps impotence of organic origin shock, unspecified hypertrophy of breast 

Answer: The patient is high likely exposed to