Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: sulfa (sulfonamides) attending: chief complaint: cardiac arrest major surgical or invasive procedure: cardiac catheterization history of present illness: 80 year old male with cad, dm2, htn and aaa repair in presents from osh after cardiac arrest. per report and wife, the patient was in his usoh, very active at baseline, walking with his wife on the am of admission when he experienced a choking sensation and right arm pain after walking mile. he sat down to get some rest, but within minutes was found by his wife on the ground unresponsive. his wife started cpr and ems was called. ems arrived after 7-10 min and began resusitation efforts. he was found to be in pea, given epi x1, which converted to v.fib, was shocked w 200j and converted to sinus tachy. there was concern that the patient hit the back of his head when he fell. . on arrival to the osh, ph was 7.21, hct 26, k 3.2. ekg with deep st depr/twi in v1-v4. the patient went into vtach and was shocked back into sinus. pt was bolused with amio. he was hypotensive with sbps in the 70s and started on dopamine/neo which was switched to levophed (left subclavian) with good result. pt was given 1 unit prbcs, rectal asa. given concern for ruptured aaa, ct abd was performed and was negative for rupture. heparin gtt was also started. given acute cardiac issues, the patient was transferred to for further care. . on arrival to the ccu, the patient was intubated and sedated. with stimulation the patient was noted to have movement in all extremities with question of posturing. his levophed and heparin were continued. after reviewing the data, the decision was made to take the patient for cardiac cath. . according the the wife, ros prior to the event was as per above. in addition, the patient did complain of stomach discomfort on the night prior to admission. she otherwise denied ha, dizziness, lightheadedness, f/c, cough, sob, chest pain, n/v/d or change in bms, rashes . past medical history: cad s/p ptca lad and 1st diag at fusiform aaa s/p grafting and repair on diabetes mellitus type 2, diet controlled htn left facial spasm duodenal ulcer s/p right retinal detachment repair with macular degeneration, s/p left eye prosthesis rotator cuff repair c5-6 laminectomy s/p appy age 5 s/p left inguinal hernia repair s/p cholecystectomy . social history: patient is happily married with his wife for many years. he has multiple children. he is a retired postman. he is very active and independent in his adls. he does not smoke (quit many yrs ago). no etoh or recreational drugs . family history: there is no family history of premature coronary artery disease or sudden death. there is history of many gi malignancies in the family including colon, pancreas, stomach . physical exam: vs: t 95.6, bp 106/58, hr 80 rr 12 o2 100% gen: intubated, sedated, eyes closed. heent: ncat. sclera anicteric. conjunctiva were pink. no xanthalesma. neck: supple with jvp of 10 cm. + carotid bruits cv: rrr no r/g, nl s1 s2, + sem heard at base chest: no chest wall deformities, scoliosis or kyphosis. syymetric. clear bs ant/lat. decr bs at bases abd: firm, mild distention, well healed midline surgical scar. + bs appreciated ext: no c/c/e. no femoral bruits. skin: no stasis dermatitis, ulcers, scars, or xanthomas. neuro: intubated, sedated. could not appreciate cranial nerves. - corneal reflex, - gag reflex, chews ett to painful stimuli. equivocal plantar reflex. no fasciculations noted . pulses: right: carotid 2+ femoral 2+ dp palpable left: carotid 2+ femoral 2+ dp palpable . pertinent results: admission labs: 10:25am blood wbc-17.3* hgb-14.2 hct-42.1 plt ct-308 glucose-328* urean-16 creat-1.5* na-141 k-4.2 cl-108 hco3-22 angap-15 pt-18.4* ptt-150* inr(pt)-1.7* calcium-8.0* phos-3.5 mg-1.8 12:10pm blood wbc-19.5* hgb-13.2* hct-36.7* plt ct-234 neuts-86* bands-0 lymphs-5* monos-9 eos-0 baso-0 atyps-0 metas-0 myelos-0 vitb12-277 folate-7.7 ferritn-154 %hba1c-6.0* 05:09am blood triglyc-94 hdl-26 chol/hd-3.3 ldlcalc-42 . cardiac enzymes: 10:25am blood ck(cpk)-1008* ck-mb-93* mb indx-9.2* ctropnt-1.23* 12:10pm blood ck(cpk)-1205* 07:55pm blood ck(cpk)-1281* ck-mb-275* mb indx-21.5* ctropnt-5.68* 05:09am blood ck(cpk)-836* ck-mb-247* mb indx-29.5* ctropnt-4.50* . arterial blood gas: 11:14am blood type-art tidal v-600 peep-5 fio2-100 po2-422* pco2-44 ph-7.22* caltco2-19* base xs--9 04:42am blood type-art po2-118* pco2-40 ph-7.37 caltco2-24 base xs--1 . ct head: no evidence of intracranial hemorrhage or mass effect . cardiac cath: selective coronary angiography of this left dominant system demonstrated a severe three vessel cad. the lmca had a distal calcific 40% lesion. there was a twin lad system. the lad had a severe diffuse disease with an 80% tubular lesion at the origin into the d1/twin lad system. the twin lad/d1 had a 90% proximal hazy lesion. the distal lad was a small vessel. the lcx was a dominant vessel with origin calcifications. the ri was subtotally occluded and was small. the om2 was occluded with a left to left retrograde filling. the distal pda had a 70% proximal lesion. the rca was a non-dominant vessel with severe diffuse disease. resting hemodynamics revealed elevated left sided filling pressures with a mean pcwp of 22 mm hg. the cardiac index was preserved at 4.57 l/min/m2. left ventriculography was deferred. . tte: la is mildly dilated. ra is moderately dilated. lv wall thicknesses are normal. the lv cavity size is normal. due to suboptimal technical quality, a focal wall motion abnormality cannot be fully excluded. overall lv systolic function is normal (lvef>55%). rv chamber size and free wall motion are normal. the aortic valve leaflets are mildly thickened. there is no aortic valve stenosis. no ar is seen. the mitral valve appears structurally normal with trivial mr. there is no pericardial effusion. brief hospital course: pt is an 80 yo m with cad, htn, s/p aaa repair who presents with pea/v.fib cardiac arrest, hypotension, and acidosis, found to have severe 3 vessel cad, having seizures secondary to anoxic brain injury. # anoxic brain injury : pt. experienced cardiac arrest in the community with several minutes of presumed non-perfusion prior to cpr and ems arrival. pt has been intubated and unresponsive to all stimuli since admission. neurology was involved throughout the course of his hospitalization. over subsequent days, the patient did not recover any neurologic function requiring multiple anti-epileptic medications for seizure control. # cardiac arrest: likely related to his underlying severe cad +/- acute mi. no obvious acidosis, electrolyte or drug/toxin exposures. per cath, appeared to have stable heart function with adequate co/ci. ph/lactate improved. ce's peaked with ck of 1281 (). patient was s/p arctic cooling protocol. # myoclonus vs. seizure: on patient began to display myoclonus/ convulsive activity, especially with manipulation. neurology was consulted and performed an eeg which was inconclusive as to whether the etiology was anoxic brain injury versus epileptic foci. keppra was started on without resolution of myoclonus. a loading dose of dilantin was given on . due to good response with dilantin, another loading dose was given on and then transitioned to a maintence dose. sedation was initially avoided so as not to cloud a complete neurological exam, however, on due to poor response of antiepileptics and continued convulsive activity, a versed drip was begun. # cad: prior cath in with multivessel disease, s/p angioplasty. cath here with diffuse 3 vessel disease including lad, lcx, rca. no interventions performed. patient was continued on asa 325mg daily, heparin gtt, lipitor 80mg daily, and metoprolol 25mg tid. # pump/bp: osh course suggestive of cardiogenic shock secondary to acute event; however, patient had preserved co and ci at cath and preserved ef (ef > 55%) by echo. patient was diuresed with lasix. # htn: on admission patient was initially hypotensive and was placed on levophed; however, in the setting of manipulation he became hypertensive. levophed was weaned off; ntg gtt maintained for bp control, metoprolol po added. # leukocytosis/ fever: origin of fevers neurologic vs. infectious. sputum stain () was positive for gpc in pairs, chains, and clusters; sputum culture was positive for proteus mirabilis. patient was started on empiric treatment with ceftriaxone/ vancomycin which resulted in decreased fever and wbc count. on pt spiked a fever to 104 f, blood, sputum, and urine cx were done, and antibiotic coverage was changed to iv vancomycin and zosyn. sputum cx on positive for gram + cocci in pairs, afebrile on . # respiratory support: intubated in the setting of cardiac arrest. oxygenation maintained on ac settings. #? episode of vtach: currently in sinus. had ? episode of nsvt post-cath. patient was initially treated with amio gtt given concern for vtach, but was later switched to metoprolol. # acidemia: appears to have be metabolic. acidemic in the setting of cardiac arrest. had elevated lactate indicating probable tissue hypoperfusion. also received 4l ns. currently without an anion gap. ph improved post intubation. # dm2: diet controlled as an outpatient. sugars elevated in acute event. sugars were controlled with iss. # arf: mild elevation in cr in the setting of cardiac arrest. etiology likely decreased perfusion secondary to arrest. creatinine continued to trend down since admission. # fen: patient received tube feeds. six days after cardiac resusitation, there were no signs of neurologic improvement and difficult to control seizure activity continued. at this time the poor prognosis for any recovery was discussed with the patient's wife and children. the patient was made dnr. it was determined to await a visit by the daughter prior to further change in management. the patient was subsequently made comfort measures only on , extubated and died on . medications on admission: terazosin 2mg qhs omeprazole 20mg daily fosinopril 40mg daily toprol xl 25mg daily felodipine 20mg daily discharge medications: none discharge disposition: expired discharge diagnosis: cardiac arrest s/p cardiopulmonary resuscitation anoxic brain injury coronary artery disease myoclonic jerks seizures secondary to anoxic brain injury hypertension discharge condition: deceased discharge instructions: none followup instructions: none procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more combined right and left heart cardiac catheterization coronary arteriography using two catheters insertion of endotracheal tube arterial catheterization angiocardiography of right heart structures diagnoses: acidosis subendocardial infarction, initial episode of care coronary atherosclerosis of native coronary artery pneumonia due to other gram-negative bacteria unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled acute kidney failure, unspecified other convulsions paroxysmal ventricular tachycardia cardiac arrest anoxic brain damage cardiogenic shock ventricular fibrillation 

Answer: The patient is high likely exposed to