Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: this is a 66-year-old man who presented with the worst headache of his life the morning of . he was seen in the emergency room at approximately 1:30 p.m. by neurosurgery. he was straining while trying to have a bowel movement. the patient has a history of bad headaches; however, this time the symptoms were much worse. past medical history: 1. hepatitis c related to transfusion. 2. benign prostatic hypertrophy. 3. thrombocytopenia. 4. atrial fibrillation. medications on admission: 1. proscar. 2. tylenol. 3. lisinopril 5 mg p.o. daily. 4. nadolol 40 mg p.o. daily. 5. interferon once a week for hepatitis c. past surgical history: gastrectomy 31 years ago. physical examination: the vital signs on admission revealed a blood pressure of 173/70, heart rate 70, respirations 16. on physical examination, he was awake, alert, oriented times three. cranial nerves ii through xii were intact. motor strength was in both his upper and lower extremities. he had no localized motor or sensory deficits. laboratory data: white count 3.1, hematocrit 39.8, platelets 63,000. sodium 141, potassium 4.6, 108 chloride, 25 bicarbonate, 17 bun, 0.8 creatinine, 117 glucose. his platelets on admission were 63. hospital course: given that his platelets were 63, he was transfused with platelets. he was obtained an immediate cta which showed a wide neck acom aneurysm. he was brought emergently to cerebral angiogram where he was showed to have a ruptured wide neck acom aneurysm 4-5 mm, a dominant left a1 and minimal filling from the right a1. he was then admitted to the icu where his blood pressure was kept at less than 120 at all times. hematology was consulted and asked to see the patient for a thrombocytopenia in anticipation of neurointerventional procedure for the subarachnoid hemorrhage which they felt that the thrombocytopenia was chronic secondary to splenic sequestration with moderate splenomegaly and they recommended transfusing platelets as needed. the attending from hematology felt that such patients do not usually bleed spontaneously post surgery and it was okay to complete the procedure with the use of heparin. he was monitored in the icu overnight for his first day of admission where he was started on nimodipine and nipride to keep his blood pressure less than 120. his systolic goal was 100-120. he remained neurologically intact overnight in anticipation of undergoing a clipping of his acom aneurysm. on , he underwent a clipping where he had without problems or complaints a vent drain placed without any difficulties. he continued to be followed by hematology for his hepatitis c. he also had atrial fibrillation which had been diagnosed three months prior to his hospitalization and his rate was well controlled in the 90s. on , his vital signs were 98.7, blood pressure 118-130s/60s-80s, heart rate in the 70s, atrial fibrillation, cvp was , icp 4-17. he was receiving some nipride to keep his blood pressure up at times. he was started with gentle fluids on that day to keep his goal cvp of 8. he had a chest x-ray to assess for chf, and his cvp level was , systolic blood pressure was in the range of the goal of 160. he was noted to have some high urine output from possible cerebral salt wasting. his sodium was 138. he had an echocardiogram which showed normal ejection fraction, chest x-ray had no acute changes. on , he remained awake, alert, completely neurologically intact. his urine input for 24 hours was 5,128, output was 10,100. he was started on one-to-one repletion therapy with saline due to cerebral salt wasting and a formal endocrine consult was obtained. his blood pressure goal of 150-160 was started. endocrine did not have any further recommendations other than to continue with his one-to-one repletion for urine and they followed the patient with us. his sodiums remained in the 138 range for . on , an echocardiogram showed a normal ejection fraction but mild pulmonary hypertension and mild left ventricular hypertrophy. on , he was awake, alert, and oriented times three. he was subarachnoid day five. his eoms were full. visual fields were intact. his hematocrit and platelets were stable. his drain was at 10 cm, put out 36 cc overnight. icps were in the 6 range. his drain was raised at 15 and his systolic blood pressure goals were increased from 170-180. he was also started on albumin to increase his cvp greater than 8. on , he was found to be awake, alert. the face was symmetric. he had a right drift, grips were . he continued to need one-to-one volume repletion for his urine output. his decadron doses were decreased. his blood pressure parameters were kept in the 160-180 range. his drain was raised to 20. on , a blood gas showed his ph at 7.44, pc02 24, and 02 of 192. he had crackles at the bases. a chest x-ray showed pulmonary edema. he was electively intubated for a low pac02. he required propofol in order to maintain an adequate ca02. on , his c02 was 30 even with propofol and our goal rate was 35-40. he needed significant sedation to control his respiratory rate in order to get an adequate pc02 level. on , the patient required levophed to keep his blood pressure goals in the 160-180 range. he was increased to cpap plus pressure support of 10 in order to maintain an adequate c02 and his nimodipine was discontinued in order to keep his blood pressure in the high range. he eventually had to be paralyzed on cisatracurium in order to increase his c02; however, that made his neurologic examination difficult to assess. on , he was still receiving a high amount of levophed and neo to maintain his blood pressure but at the same time he was noted to be cyanotic. his extremities started to become cyanotic on . on , it continued with cyanosis and the icu noted abdominal distention, worsening acidemia, rising lactate. the surgery team was asked for resuscitation for ischemic bowel. he had an emergent cat scan of his head and abdomen and also for a four vessel angiogram which showed minimal vasospasm which were in the bilateral ica and pca territories. his blood pressure parameters were now weaned down to 110. ct of the abdomen showed some moderate free fluid, mild rectal thickening, no pneumonitis or free air. they recommended a colonoscopy to exclude ischemia of the bowel but they felt more likely he was having some liver failure, hyperperfusion, or due to the existing hepatitis c. on , it appeared that the patient was developing ards. also, surgery recommended starting him on levo, flagyl, and vancomycin for prophylaxis. his abdomen remained enlarged and distended. it was difficult to get a neurological examination on him due to his sedation. his pupils were equal and reactive to light and accommodation and his corneal reflexes were intact on and cvps were 10- 17. his icps were in the 11-15 range. his ventriculostomy drain continued to work well. his sodium remained in the 147 range. his potassium was noted to be 6.0 and his creatinine had risen to 1.7. renal was asked to see the patient emergently for renal shock and this was felt due to marked increased phosphate, calcium, and lactate and that he had dense acute tubular necrosis. they recommended to start cvvhd/vf and to also replace 150 meq of bicarbonate per liter of fluid so he was to receive 300 meq of bicarbonate per hour. the liver service also saw the patient and felt that he did have acute shock liver and acute renal failure. they felt that he should start on dobutamine and avoid sedation and that his prognosis was poor, but he was now in multiorgan failure. a family meeting was done on to explain the grim prognosis due to multiorgan failure to the family. he was on a bicarbonate drip. his acidosis on did seem to improve. he started to receive insulin and calcium drips and cvvhd. on , he remained intubated and sedated. on examination, his pupil on the right was 6 mm, on the left 4 mm. he did not withdrawal to pain in his extremities. his blood pressure was maintained in the 100-110 range. he continued on cvvhd. on , cardiology also saw the patient due to myocardial depression. they were recommending transitioning his neo-synephrine to levophed, checking a cortisol, to stop his digoxin, keep his cvp 10-15 with a pa diastolic of 18-25 and a cardiac index greater than 2. on , the patient remained acutely ill with shock liver, increased acidosis, acute renal failure, multiorgan failure. he received blood products and factor vii for his hematocrit of 27 and his platelets of 74, inr of 1.7. he received lactulose to help increase his bicarbonate. on the afternoon of , a family meeting was convened and due to his poor prognosis and multiorgan system failure, his family requested withdrawal of care. he passed away at 1:05 on . discharge diagnosis: 1. subarachnoid hemorrhage from acom aneurysm. 2. multiorgan system failure. 3. shock liver. 4. hepatitis c. 5. hypertension. 6. benign prostatic hypertrophy. 7. acute renal failure. , procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more parenteral infusion of concentrated nutritional substances insertion of endotracheal tube enteral infusion of concentrated nutritional substances hemodialysis venous catheterization for renal dialysis clipping of aneurysm arteriography of cerebral arteries arteriography of cerebral arteries arteriography of cerebral arteries intravascular imaging of intrathoracic vessels arterial catheterization transfusion of packed cells transfusion of other serum transfusion of platelets transfusion of coagulation factors diagnoses: acidosis acute kidney failure with lesion of tubular necrosis unspecified essential hypertension chronic hepatitis c without mention of hepatic coma acute and subacute necrosis of liver acquired coagulation factor deficiency unspecified septicemia hyposmolality and/or hyponatremia severe sepsis atrial fibrillation subarachnoid hemorrhage cardiac arrest long-term (current) use of other medications acute vascular insufficiency of intestine other diseases of spleen chronic congestive splenomegaly 

Answer: The patient is high likely exposed to