Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: direct admit from hepatology outpatient office: hypotension. major surgical or invasive procedure: intubation and mechanical ventilation. history of present illness: the patient is a 44 yo f with pmh significant for alcoholic cirrhosis who was at dr. office today for routine evaluation and was noted to have bp ~80s/40s. per the patient's husband, she was in her usual state of health until approximately 2 years ago when she attempted to stop drinking for a family function. within a few days of stopping drinking (previously she drank pints gin/vodka / day) she had a seizure and was brought to the hospital. it was during that time, that she was told she had liver disease from alcohol. she tried to stop drinking but was unable to secondary to increased life stressors. she started and stopped drinking times and each time she stopped she had a seizure. her liver disease was well compensated until - when she noted increased ankle swelling and increased abdominal girth and jaundice. at that time an egd was done which was notable for large, portal gastropathy, nonbleeding varices. she was started on atenolol, lasix and spironolactone. she stopped drinking after that hospitalization. per her husband, she has remained weak, confused, and only intermittently lucent over the past few months. her atenolol was changed to nadolol ~1 month ago for unclear reasons per her husband. notes that she has been "yellow" for more than a month. they report a >10 pound weight loss in the last few months despite the patient attempting to eat (she drinks boost tid). they deny fevers, chills, night sweats. they deny pruritis. she reports easy bruising/bleeding. she had a fall secondary to instability in during which she hurt her back and continues to take tramadol. they deny loc/head trauma during the fall. the patient was seen at the liver clinic last month and her bp was 85/60. in the ed, the patient was given 3 liters of iv ns. ros: negative for lightheadedness/palpitations. negative for nausea/vomiting/abdominal pain/hematemesis. negative for melena/hematochezia. negative for dysuria/increased urinary frequency. past medical history: 1. cirrhosis - most likely from alcohol, further w/u pending, complicated by varices and portal gastropathy. she has never had a varicael bleed. 2. osteoporosis - -bone density study-notable for osteoporosis at the hip 3. compression fracture- 4. chronic alcoholism - no period of abstinence social history: the patient lives with her husband. she does not have children. she denies tobacco/iv drug use. she drank pints vodka/gin per day >15 years. she has not drank alcohol since . she used marijuana 3 years ago. family history: (+) for alcoholism. (-) for liver disease/cancer/dm physical exam: t: 97.1 bp: 81/44--90/50 hr: 71 rr 16 o2 sat: 100% ra genl: alert, confused, not oriented (baseline per husband), nad : (+) spider veins on face, (+) scleral icterus, perrl, eomi, oropharynx clear, moist mucus membranes (after 2 l ns) neck: supple, no jvd, no thyromegaly ln: no cervical, supraclavicular, axillary lad cv: rrr, normal s1, s2, (+) sm pulm: cta b/l, no w/c/r abd: soft, nondistended, nontender, no hsm, no r/g rectal: brown, guaic negative stool ext: no edema, 2+ dp pulses, no palmar erythema neuro: alert, cn 2-12 intact, motor ue/le bilaterally (limited effort) (+) asterixis pertinent results: ct head: impression: no acute intracranial abnormalities. findings consistent with bilateral cerebral and cerebellar atrophy. 11:23am wbc-9.2 rbc-3.34* hgb-9.4* hct-29.0* mcv-87 mch-28.0 mchc-32.3 rdw-16.6* 11:23am neuts-81.0* lymphs-11.1* monos-4.0 eos-3.4 basos-0.6 11:23am plt count-104* 11:23am pt-21.6* ptt-50.3* inr(pt)-2.9 11:23am alt(sgpt)-58* ast(sgot)-97* alk phos-191* amylase-25 tot bili-6.3* 12:51pm ammonia-18 12:25pm pth-16 11:23am glucose-153* urea n-10 creat-0.6 sodium-126* potassium-5.2* chloride-93* total co2-25 anion gap-13 05:20pm glucose-176* urea n-7 creat-0.5 sodium-130* potassium-4.3 chloride-101 total co2-25 anion gap-8 05:29pm urine color-yellow appear-clear sp -1.013 05:29pm urine blood-mod nitrite-pos protein-tr glucose-100 ketone-neg bilirubin-neg urobilngn-neg ph-5.0 leuk-mod brief hospital course: mrs. was admitted with known cirrhosis, thought secondary to chronic alcohol use, with hypotension and mild confusion. she had a long complicated course, which included hypotension, new-onset severe chf (of unclear etiology) and worsening liver failure. given her poor prognosis and responses to therapy, the goals of care were changed to comfort; the patient died comfortably with her husband at her bedside. an autopsy was requested by the family. again, mrs. had a known history of cirrhosis/esld, esophageal varices s/p hhemorrhage and banding (), hepatic encelphalopathy, active alcholism, idiopathic tachycardia who was admitted with confusion and was found to be hypotensive, tachycardic, and hyponatremic. she was admitted to the micu. hemoptysis then ensued in her early course and an egd and intubation were pursued. she under went variceal banding with success in regards to hemostasis. her hct nadired in the high 'teens' but responded to her banding procedure, along with prbc and ivf volume resucitation. she then (late ) developed oliguria in conjunction with increasing tachycardia and svt/af episodes. over the proceeding days, her uop improved, but the tachycardia along with new af persisted. an extensive search for infection was not fruitful (including hiv, blood, urine, peritoneal fluid and chest imaging evaluation). a tte showed marked lowering of the lvef (from >55%% to 25%). the etiology was unclear, but deemed to be either tachycardia-induced, viral, or ischemic; as evaluated by the ep cards and chf services. ces and tsh were normal. she was started on low-dose bb (nadolol) and acei. she was eventually extubated. she was called out to the floor but soon became hypotensive (sbps 70s-80s) with concominant redevelopment of oliguria. she was transferred to the ccu (). her bb and acei were stopped and she was given pressor support. cardiac catheterization, biopsy or ep evaluations were not purused during her course. given her dismal prognosis and poor responses to therapies, goals of care were changed to comfort alone - after many family discussions. she was given morphine for respiratory comfort and seemed peaceful and comfortable thereafter. her husband remained at her bedside and mrs. passed away. medications on admission: nadolol 20 mg daily lasix 20 mg daily spironolactone 100 mg daily mvi milk thistle folic acid 400 mg daily vitamin b1 100 mg daily tramadol prn back pain xanax 0.25 mg/day discharge medications: none discharge disposition: extended care facility: rehab discharge diagnosis: primary diagnosis: 1) alcholic end-stage liver disease. 2) idiopathich cardiomyopathy. 3) varriceal hemorrhage. discharge condition: deceased discharge instructions: none followup instructions: none procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours other endoscopy of small intestine insertion of endotracheal tube endoscopic excision or destruction of lesion or tissue of esophagus diagnoses: other primary cardiomyopathies thrombocytopenia, unspecified congestive heart failure, unspecified acute and subacute necrosis of liver alcoholic cirrhosis of liver acute respiratory failure osteoporosis, unspecified esophageal varices in diseases classified elsewhere, with bleeding hepatic encephalopathy other and unspecified alcohol dependence, continuous 

Answer: The patient is high likely exposed to