Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: vicodin / tramadol / codeine attending: chief complaint: iph major surgical or invasive procedure: intabuated, extubated on history of present illness: 47 yo m with history of htn, dm2, transferred from hospital after he was found to have large left basal ganglia hemorrhage. as per chart review, the patient collapsed when going to sit on his couch earlier this evening. at time of arrival to osh, gcs was 9. he was non-verbal, eyes would open to verbal commands and he had right-sided neglect. systolic bp was 216 at time of arrival and max of 250. an examination at 0050 noted a r pupil of 7 mm and nonreactive, l pupil 5mm and recative and he was noted to have non-purposeful movement of his right side. a ct head showed a left parietal / superior basal ganglia acute iph 6.4 cm x 2.5 cm x 4 cm with mild surrounding edema, creating mass effect on left lateral ventricule without significant midline shift. he was given 1g fosphenytoin, labetalol and naloxone and transferred to for further care. en route he was noted to decomponsate and was no longer tracking with his eyes and bradycardic to the 40s and was subseuqently paralyzed with veccuronium, fentanyl, and versed, and intubated. past medical history: -htn -dm -sciatica -gout -unspecified myalgia, myositis social history: occasional etoh, no tobacco or drugs, however was utox positive for cocaine family history: nc physical exam: vs; bp 130/80 p 56 rr 16 100% on vent gen; intubated, lying in bed heent; nc/at cv; rrr pulm; cta anteriorly abd; soft, nt, nd extr; no edema neuro; (had been paralyzed for intubation approximately one hour prior to examination) mental status; unarousable to noxious stimuli. does not grimace to pain. cn; eyes in midposition when held open. pupils 3mm and minimally reactive. no corneals or gag. motor; normal bulk and tone. no movement to noxious stimuli sensory; does not withdraw to noxious stimuli reflexes; toes mute pertinent results: 05:46am blood wbc-12.8*# rbc-3.70* hgb-11.8* hct-35.4* mcv-96 mch-31.9 mchc-33.2 rdw-12.3 plt ct-235 05:46am blood plt ct-235 05:46am blood pt-12.3 ptt-26.5 inr(pt)-1.0 05:46am blood glucose-150* urean-20 creat-1.7* na-141 k-3.8 cl-111* hco3-24 angap-10 09:20am blood alt-45* ast-68* ld(ldh)-207 alkphos-60 amylase-147* totbili-0.5 09:20am blood ck-mb-7 ctropnt-0.01 03:10am blood ck-mb-10 mb indx-0.8 ctropnt-<0.01 05:46am blood calcium-8.4 phos-3.3 mg-2.2 09:20am blood caltibc-247* ferritn-322 trf-190* 05:46am blood osmolal-305 03:10am blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 04:45am urine bnzodzp-neg barbitr-neg opiates-neg cocaine-pos amphetm-neg mthdone-neg imaging: ct head : slight interval decrease in size of left basal ganglia hemorrhage with surrounding edema. unchanged effacement of sulci with no new bleed or evidence of herniation present. ct head: 1. 6.4 x 2.6 cm left basal ganglionic acute hemorrhage with transpendymal dissection and intraventricular extension. likely etiology for this hemorrhage is hypertension; however, underlying mass or vascular abnormality cannot be excluded, and this should be correlated with clinical information. 2. edema surrounds the hemorrhage, with local mass effect, effacing the lateral ventricular body and overlying temporal and frontal sulci and gyri. 3. no significant midline shift and no evidence for herniation. ekg: sinus bradycardia. baseline artifact. rsr' pattern in lead v1. prominent qrs complex with probable left ventricular hypertrophy. q-t interval prolongation. j point and st segment elevation in the lateral precordial leads may be related to early repolarization or injury. clinical correlation is suggested. no previous tracing available for comparison. ct head impression: 1. no significant interval change in size of a left basal ganglial hemorrhage, though the surrounding edema appears to be slightly increased. 2. tiny focus of intraventricular hemorrhage within the occipital of the left lateral ventricle, decreased in extent from prior study. 3. stable local mass effect, with effacement of the left lateral ventricle. upper extremity ultrasound impression: 1. occlusive thrombus seen in the segment of the right cephalic vein at the antecubital fossa and in the segment of the left cephalic vein in the left forearm. 2. no evidence of deep vein thrombosis in either arm. brief hospital course: 47 yo m with history of htn and transferred from hospital with large basal ganglia hemorrhage, most likely hypertensive in etiology. with addition of a urine toxicology positive for cocaine. he had been intubated at an outside hospital. the patient was admitted to the neuro icu, for monitoring and blood pressure control. he was started on a nicardipine drip. he did well overnight and on the morning of he was noted to be awake and alert and following commands when off sedation. he was extubated that morning and did well in regards to his respiratory status. he was restarted on some oral hypertensive medications -> initially amlodipine and hydralazine and was slowly weaned for his nicardipine drip. he was given less fluids and the attempt was made to keep his fluid status at least 300-500cc negative to ensure that there was not significant edema. he had a repeat ct the following day, which appeared unchanged, with a mild amount of edema surrounding the lesion. the patient, after extubation was noted to have a significant right sided weakness. he has a right field cut with 0/5 strength in his right upper and lower extremities. he appeared to present with a mixed aphasia. he appears to have intact comprehension, some difficult with repetition and a decent level of fluency but a number of paraphasic errors. his speech has been continuing to improve throughout his hospital course. his blood pressure has been well-controlled and it is thought he may be able to begin to wean off some of his antihypertensive medications. renal- the patient presented with a creatinine of 2.0 which improved with hydration and has been fluctuating between 1.3-1.6. his baseline creatinine is unknown. his creatinine at the time of discharge was 1.6. of note, the patient was restarted on his home actos the day prior and has been receiving hydralazine for blood pressure control. he was given additional fluids and it is suggested his creatinine continue to be monitored closely upon transfer and he may benefit from studies of urine creatinine, electrolytes, and eosinophils for further evaluation if his creatinine continues to trend up. vasc/id- patient was noted to have erythema and a palpable cord on his forearms b/l at sites of prior ivs. he had low grade temperatures and initially started on keflex . two days later the sites appeared essentially unchanged and his antibiotic was changed to vancomycin. he underwent upper extremity ultrasounds which confirmed b/l cephalic vein thromboses but no evidence of dvt. he was continued on antibiotics to complete a seven-day course and warm compresses which provided good symptomatic relief. medications on admission: -actos 15 mg daily -flexeril 10 mg tid prn -ibuprofen 400 mg prn -percocet prn discharge medications: 1. amlodipine 5 mg tablet sig: two (2) tablet po daily (daily). 2. actos 15 mg tablet sig: one (1) tablet po once a day. 3. hydralazine 25 mg tablet sig: three (3) tablet po q6h (every 6 hours). 4. heparin (porcine) 5,000 unit/ml solution sig: one (1) injection tid (3 times a day). 5. clonidine 0.2 mg/24 hr patch weekly sig: one (1) patch weekly transdermal qsun (every sunday). 6. morphine 15 mg tablet sig: one (1) tablet po q4h (every 4 hours) as needed for pain. discharge disposition: extended care facility: state hospital discharge diagnosis: left basal ganglia hemorrhage cephalic vein thombosis b/l discharge condition: awake, alert, comprehension intact. occasional paraphasic errors. r facial droop, 0/5 strength in rue, rle. sensation intact to light touch. discharge instructions: you were admitted with a large bleed in a part of your brain called the basal ganglia. this was likely caused by high blood pressure as well as cocaine use. repeat imaging studies showed the bleed remained stable. you were also treated with antibiotics for superficial thrombophlebitis on your arms. followup instructions: provider: , .d. phone: date/time: 2:00 (neurology) please follow up with your pcp within one month. procedure: venous catheterization, not elsewhere classified enteral infusion of concentrated nutritional substances diagnoses: sciatica unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled acute kidney failure, unspecified gout, unspecified cocaine abuse, unspecified intracerebral hemorrhage cerebral edema unspecified disorder of kidney and ureter late effects of cerebrovascular disease, hemiplegia affecting unspecified side phlebitis and thrombophlebitis of superficial veins of upper extremities myalgia and myositis, unspecified late effects of cerebrovascular disease, aphasia muscle weakness (generalized) late effects of cerebrovascular disease, dysarthria 

Answer: The patient is high likely exposed to