Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: cardiogenic shock major surgical or invasive procedure: arterial line placement history of present illness: 79 f with insulin-dependent diabetes, cad, pvd, s/p rt cea in fairly good health had abdominal discomfort and generally feeling unwell for approximately 2 weeks. she states that she has been feeling "tired and weak", staying in bed for most of those two weeks. denies any cp, sob, n/v/d. her daughter visited her 2 days ago and reports that her mother was c/o mild periumbilical abdominal aching, no n/v/d and appeared to be dizzy. . her family brought her to the ed. in the ed, she was pale diaphoretic and c/o of abd pain. abd was found to be benign but she had new ekg changes with st elevations in anterior leads and cardiac enzyme elevations: trop 7.02, ck 200's, mb 47. bp mid 90's. left ij placed. she received approx 4l ivf for hypotension and then developed pulmonary edema. bnp 1854. she was diagnosed with cardiogenic shock and transfered to the ccu. . in the ccu, she was on neo at 40mcg per kg with improvement of sbp to 97. an echo revealed an ef of 20%. due to low bp, she was also placed on dobutatime. she subsequently became very tachycardic and was switched to dopamine. in addition, she was found to have a leukocytosis of 40,000 but had already been started on ceftriaxone given hx of uti's. past medical history: past medical history: hypertension status post r carotid endarterectomy in status post l second ray amputation for osteomyelitis in iddm . cardiac history: cabg: none percutaneous coronary intervention: none pacemaker/icd placed: none social history: no tobacco or etoh history. widowed. lives alone; meals on wheels and daughter helps with cooking/cleaning. manages her own adl's. worked in bank before retirement. . family history: n/a physical exam: admission: vitals: temp 98.6 bp 99/49 hr 109 sao2 87% on ra, 96% 6l nc gen: ill appearing, pale and clammy heent: minimally elevated jvp 2-3cm cvs: rrr, no mrg, minimally displace pmi resp: mildly labored breathing, no acessory muscle use, no crackles but dull at lll with egophany abd: soft nt/nd, nabs ext: no edema, cold clammy, diaphoretic neuro: aox2, drowsy but answering questions appropriately . discharge: expired pertinent results: cxr: cardiomegaly with moderate-to-severe pulmonary edema and bilateral pleural effusion indicates congestive heart failure. . echo: ef 25-20% the left atrium is mildly dilated. left ventricular wall thicknesses are normal. the left ventricular cavity size is normal. there is moderate to severe regional left ventricular systolic dysfunction with septal/anterior hypokinesis/akinesis and apical akinesis. right ventricular chamber size is normal. with focal hypokinesis of the apical free wall. the aortic valve leaflets (3) are mildly thickened. there is no aortic valve stenosis. the mitral valve leaflets are mildly thickened. mild (1+) mitral regurgitation is seen. there is a trivial/physiologic pericardial effusion. . abg: 01:56pm blood type-art po2-88 pco2-31* ph-7.32* caltco2-17* base xs--8 09:03pm blood type-art fio2-95 po2-107* pco2-27* ph-7.22* caltco2-12* base xs--15 aado2-568 req o2-90 intubat-not intuba 10:15pm blood type-mix ph-7.18* 11:39pm blood type-art po2-81* pco2-27* ph-7.17* caltco2-10* base xs--17 12:57am blood type-art po2-94 pco2-40 ph-7.10* caltco2-13* base xs--16 02:27am blood type-art fio2-100 po2-83* pco2-30* ph-7.20* caltco2-12* base xs--14 aado2-626 req o2-98 -assist/con intubat-intubated . lactate 01:56pm blood lactate-1.6 09:03pm blood lactate-1.7 10:15pm blood lactate-1.8 11:39pm blood lactate-2.9* 12:57am blood lactate-2.9* 02:27am blood lactate-4.3* . cardiac enzymes 02:06pm blood ck-mb-49* mb indx-15.4* ctropnt-3.31* 02:11am blood ck-mb-37* mb indx-19.3* ctropnt-3.43* 02:06pm blood alt-34 ast-58* ld(ldh)-455* ck(cpk)-318* alkphos-51 totbili-0.3 08:42pm blood ck(cpk)-226* 02:11am blood ck(cpk)-192* . chem 7 02:06pm blood glucose-108* urean-74* creat-2.6*# na-139 k-5.2* cl-109* hco3-18* angap-17 08:42pm blood glucose-217* urean-81* creat-2.8* na-137 k-5.6* cl-107 hco3-11* angap-25* 02:11am blood glucose-258* urean-84* creat-3.1* na-140 k-6.3* cl-106 hco3-11* angap-29* . cbc 02:06pm blood hypochr-normal anisocy-2+ poiklo-occasional macrocy-1+ microcy-normal polychr-occasional ovalocy-occasional schisto-occasional 02:06pm blood neuts-91* bands-1 lymphs-5* monos-3 eos-0 baso-0 atyps-0 metas-0 myelos-0 nrbc-1* 02:06pm blood wbc-52.7*# rbc-3.46* hgb-11.0* hct-33.0* mcv-95# mch-31.7# mchc-33.2 rdw-15.4 plt ct-333 02:11am blood wbc-70.1* rbc-3.60* hgb-11.1* hct-35.3* mcv-98 mch-30.9 mchc-31.5 rdw-15.3 plt ct-536*# brief hospital course: 79 year old female vasculopath, iddm with acute anterior mi, ef 20% in cardiogenic shock with pulmonary edema and leukocytosis. . # cardiogenic shock: the patient was admitted from osh diagnosed with cardiogenic shock with bp 99/40 on levophed. her ami was presumed to be completed with q waves as well as ste in v1-v4 although it was unclear if her had peaked. over the course of the next 16 hours, her ck's trended down. because she was initially stable, with presumed completed infarct and because of her elevated creatinine, she was not taken for pci immediately. an echo was performed showing ef 20-30%, severe regional left ventricular systolic dysfunction with septal/anterior hypokinesis/akinesis and apical akinesis. a cxr showed significant pulmonary edema. echo and cxr as well as ekg and ce c/w ami indicated that the patient was most likely in cardiogenic shock. she was started on a heparin drip for anterior wall akinesis. she was also started on a lasix drip. she was initially continued on levophed. as her mental status and bp declined, milrinone was added to levophed. 1-2 hours after starting milrinone her bp declined further to 80/30 with map's in 40's, lactate started rising and her urine output fell despite lasix. milrinone and lasix were then discontinued. dopamine was initiated, but was soon discontinued when she became tachycardic to hr 120's. vasopressin was then initiated in addition to levophed. during this time period, her ph fell from 7.32 to 7.1 and her lactate rose from 1.6 to 4.3. as pressors and ionotropes did not appear to be sufficient to maintain her perfusion, it was decided to take the patient to the cath lab for pci and balloon pump. initially, the family had decided that they would be comfortable with a short trial of aggressive measures but that the pt "did not want to live on machines". just prior to taking the patient to the cath lab, her family made the decision not to pursue catheterization or a balloon pump. they requested that she be made cmo. pressors were stopped. the patient died shortly thereafter. . # respiratory distress: on admission, the patient was in mild respiratory distress and slightly hypoxic. after settingly in and receiving morphine, the patient was comfortable on nc. a cxr was obtained showing pulmonary edema likely cardiogenic shock. she received several lasix boluses and was then put on a lasix drip. for several hours, she appeared relatively comfortable, but as her mental status declined and blood pressure fell, she became hypoxic and required increasing amounts of oxygen and a nrb. she was also tachypnic. she appeared to be increasing respiratory distress, with an ashen, cyanotic appearance. her abg's showed a metabolic acidosis with respiratory compensation. when her ph fell to 7.2, it was decided to semi-electively intubate her with agreement of her family. after she was make cmo, pressors were stopped. initially, the family requested that she remain intubated so that she would not be "gasping for air". however, after seeing the patient intubated and off pressors, they decided to have her extubated. a morphine drip was started. fentanyl and versed were discontinued. the patient was extubated and died. . # leukocytosis: at the osh, the patient's wbc was 33 with a left shift. her wbc count trended up from 33 to 55 to 70. intially, it was thought that with a leukocytosis of 33, that she could be having an inflammatory reaction secondary to mi. there was no evidence of infection w/o fever or focal symptoms. at the osh, she had been empirically started on ceftriaxone despite normal ua given a history of frequent uti's. her blood pressure and leukocytosis remained unchanged despite ceftriaxone. she was re-cultured here but was not continued on any antibiotics as her presentation was thought to be consistent with cardiogenic shock and not concerning for sepsis. as her wbc rose, it seemed less likely that a leukocytosis of 70 was due to ami. a review of the osh records, revealed a lab report stating that there were "rare auer rods" suggestive of aml. a blood smear was sent, but the patient was made cmo. after discussing the leukocytosis with her family, they requested an autospy to evaulated for an hematologic disturbance. . # metabolic acidosis: the patient's ph fell throughout the night from 7.31 t0 7.1 with co2 31->27 and bicarb 18->11, ag 17->25 suggestive of an evolving mixed gap and non-gap metabolic acidosis. intially, she had a non-gap acidosis likely due to acute renal failure. she was admitted on an insulin drip, but there was no report of dka. her blood glucose was 120's, no glucose or significant urine or serum ketones making dka unlikely. a serum beta-hydroxybutyrate level was sent, but was pending. as perfusion decreased and lactate rose, it became clearer that the ag metabolic acidosis was likely due to lactic acidosis. . # communication: hcp son : cell , home daughter: cell , home medications on admission: nifedical 50 cozaar 50 metoprolol 50 asa 81 insulin tid lumigan eye gtt alphagan eye gtt cosopt eye gtt discharge medications: n/a discharge disposition: expired discharge diagnosis: cardiogenic shock acute anterior myocardial infarction leukocytosis (unclear etiology) discharge condition: expired discharge instructions: n/a followup instructions: n/a procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube arterial catheterization diagnoses: coronary atherosclerosis of native coronary artery acute kidney failure with lesion of tubular necrosis congestive heart failure, unspecified unspecified essential hypertension diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled acute myocardial infarction of other anterior wall, initial episode of care peripheral vascular disease, unspecified cardiogenic shock acute on chronic systolic heart failure 

Answer: The patient is high likely exposed to