Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: found down major surgical or invasive procedure: picc placement hd tunneled line placement central venous line placement ultrasound guided percutaneous liver biopsy history of present illness: 54f with schizophrenia, htn, dm, who was found unresponsive by her son at approximately 3 am in the bathtub. he said he had seen the night before and she was okay, in no acute distress, though he notes she has not been taking her meds for many months. he said she was breathing shallowly and slapped her several times in the face with no response and then called ems. was given narcan in the field. in ed, hypotensive with sbp in 50s, hr 160s, was intubated and right subclav placed. pt. was hyperthermic to 106 and because patient was on antipsychotics at home, there was a question of overdose and both dantrolene (for nms) and physostigmine (for anticholinergic) were given. in addition, pt. got head ct (neg) and chest ct which showed bibasilar consolidation. pt. received vanc and ceftriaxone for coverage of pneumonia/meningitis. she was started on levophed and vasopressin for pressors. as they were attemtpting to lp patient at 6am, it was noted that she had a hematoma around subclavian site and patient was oozing blood there. coags were checked and showed inr 146. pt. transferred to micu. on arrival, pt. was very hypotensive and oozing from every orifice. past medical history: 1. dm on oral hypoglycemics: metformin and glyburide 2. htn 3. schizophrenia 4. asthma social history: lives with son, . extensive family. is originally from . per famiy, was drinking heavily several weeks ago but not recently. no other drugs. ?tob. family history: non-contributory physical exam: vs: t 91.7 hr 56 bp 86/40 vent: ac 15 x 550, fio2 100%, peep 5 gen: not responsive to painful stimuli heent: pupils constricted but reactive, og tube with brb cv: rrr s1s2 no mrg lung: coarse bs b/l abd: soft, nt, nd, with rectal bag with thin bloody liquid ext: no edema, ulcerations on legs neuro: not responding to painful stimulus lines: right subclavian oozing blood pertinent results: # at admission (): -glucose-299* urea n-19 creat-1.7* sodium-147* potassium-3.4 chloride-117* total co2-10* -ck(cpk)-2153*, ck-mb-147*, mb indx-6.8*, ctropnt-6.86* -calcium-6.7* phosphate-3.8 magnesium-2.0 -wbc-14.7 w/34 pmns, 8 bands. -hct 31->17 after ivf -plt 91->26 -inr(pt) >66 at admission -> -> 3.5 -fibrin <45, fdp >1280, d-dimer >10,000 -lactate-10.3* -asa-neg ethanol-neg acetmnphn-neg bnzodzpn-neg barbitrt-neg tricyclic-neg -ecg: sinus rhythm. wandering baseline. left axis deviation. q-t interval prolongation. a repeat tracing of diagnostic quality is suggested. there is low limb lead voltage. no previous tracing available for comparison -cxr: satisfactory positioning of tubes and lines as described. no pneumothorax detected. no definite consolidation. # other data: -alt peaked at 4377 on then stable in low 100s at discharge -ast peaked at 8458 on then stable in 60s at discharge -creatinine 1.4 at admission -> peaked at 8.0 on (then on dialysis) -peak tropt 9.88 on -peak ck 4104 on and ck-mb 149 on -total bili peaked at 13 on then trended down -alk phos peaked at 1436 on then trended down # imaging: -: sinus ct: no acute process seen. -: ct head: no evidence of intracranial hemorrhage or shift of normally midline structures. ventricles and cisterns are normal. hypodensities within both basal ganglia consistent with old lacunar infarcts. the -white differentiation is preserved. there are no abnormally enhancing regions within the brain. -: ct torso: no acute process; no evidence of abscess. -: le doppler u/s: no evidence of lower extremity dvt. -: : normal appearance of the liver, pancreas, pancreatic and common bile ducts. -: gallbladder scan: no evidence of cholecystitis. delayed gallbladder emptying following cck administration with ef of 12% after 30 minutes (normal range is more than 35%). -: left upper extrem doppler u/s: no evidence of deep venous thrombosis in the left upper extremity. -: echocardiogram: ef >=65%. . lv wall thickness, cavity size, and systolic function are normal (lvef>55%). regional left ventricular wall motion is normal. rv chamber size and free wall motion are normal. 1+ mr. mild pulmonary artery systolic hypertension. no pericardial effusion. -: eeg: abnormal eeg due to the slow and disorganized background and bursts of generalized slowing. these findings indicate a widespread encephalopathic condition affecting both cortical and subcortical structures. medications, metabolic disturbances, and infection are among the most common causes. there were no focal abnormalities although encephalopathies may obscure focal findings. there were no epileptiform features. -: mri head: areas of increased diffusion within the left internal capsule and bilateral frontal cortex is suspicious for a subacute infarct, as these are also seen as abnormal on t2 and flair. please correlate with the length of the patient's symptoms. sinus disease as described above. scalp hematoma. -: echocardiogram: ef 50%, no masses or thrombi seen in the lv. no vsd. rv chamber size is normal. rv systolic function appears depressed. lv inflow pattern suggests impaired relaxation. no pericardial effusion. brief hospital course: 54f w/ dm/htn/schizophrenia, p/w septic shock and oligouric arf, now on hd and has slow to improve ms. # fevers: patient initially admitted in presumed septic shock, although no infectious source identified. toxicology consult was obtained and felt that her presentation was consistent with septic shock. all cultures, including blood, urine, catheter tip, c.diff, have been with ngtd. urine cultures have grown out >100,000 cfu of albicans. in unit, she was given 14d course of ctx/vanc for empiric tx of pneumonia and meningitis but lp was deferred due to dic. this coverage was changed to clinda+levaquin for empiric tx of aspiration pneumonia, and then to levo + flagyl for cholangitis. antibiotics discontinued ~ and patient defervesced although she has had intermittent spikes and negative cultures. caspofungin started for fungal uti on but d/ced after 3 days. pan-ct of head, chest, abdomen, pelvis was negative for any source for fever. wbc scan was performed on and revealed possible uptake in the left lower extremity, but xrays in this area did not show an acute process. final read of wbc scan was read as normal. # lft abnormalities: lft??????s trended upwards in the micu, thought to be secondary to shock liver. transaminases peaked, but alk phos has been more or less persistently elevated and rising with elevated ggt. ruq ultrasounds shows no evidence of cholangitis or cholecystitis. hida scan shows normal gb flow, but shows low ??????ef?????? ?????? the significance of this is unclear. wnl. a liver biopsy was performed on , but results are negative for granulomatous hepatitis (i.e. fungal infx). # renal failure: the patinet had oliguric renal failure due to her shock/hypotension. in the micu, she was started on cvvh and then was transitioned to hemodialysis tue//sat. a tunneled line was placed on . however, by , her renal fxn improved off of dialysis so nephrology signed off. # mental status: mri with subacute l internal capsule, b/l frontal cortex infarct, scalp hematoma, sinus thickening. eegx2 showed toxic metabolic slowing. she has had decreased but waxing/ ms; she has been following commands in spanish, and slowly increasing her vocab. her expressie aphasia is improving. she needs speech therapy as an outpt. # heme: was found to have dic on admission, this was most likely secondary to sepsis. pt has had episodes of bleeding around hd cath site, she has responded to ddavp for uremic bleeding. hct stable by time of discharge. # cardiovascular: on presentation, the patient was found to have nstemi which can be attributed to demand ischemia in the setting of tachycardia and hypotension. the patient was started on a beta-blocker and this was titrated upwards. # endocrine: history of type ii diabetes, continue sliding scale insulin. oral hypoglycemics were held given her acute presentation. # fen: on tube feeds per ngt, nutrition input appreciated. peg to be placed . on -5 the patient developed hypernatremia due to being npo and having insufficient iv fluids. this was corrected with free water repletion via iv and ng. her last speech and swallow evaluation was which she failed. hopefully as her aphasia improves, so will her ability to protect her airway. she needs further assessment of this problem as an outpt. # access: tunneled hd cath placed for hemodialysis and removed as pt no longer needed hd. picc left arm (), ngt, foley. # psych: reinstitution of psychotropic meds was deferred given the patient's acute presentation. # communication: with her daughter (healtcare proxy), brother . contact information is: medications on admission: 1. geodon 2. cogentin 3. perphenazine 4. metformin 5. glyburide 6. lisinopril discharge disposition: extended care facility: & rehab center - discharge diagnosis: type ii diabetes mellitus hypertension schizophrenia acute renal failure hepatitis discharge condition: good discharge instructions: please take your medications as directed. if you have these symptoms, call your doctor: fever/chills/shortness of breath/ chest pain/ bleeding/ fainting she should start on a statin once her liver function stabilizes. followup instructions: please see your primary care physcian within 3 weeks procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more enteral infusion of concentrated nutritional substances hemodialysis closed (percutaneous) [needle] biopsy of liver venous catheterization for renal dialysis percutaneous [endoscopic] gastrostomy [peg] transfusion of packed cells diagnoses: acidosis hyperpotassemia subendocardial infarction, initial episode of care toxic encephalopathy unspecified essential hypertension acute posthemorrhagic anemia acute and subacute necrosis of liver acute kidney failure, unspecified unspecified septicemia severe sepsis unspecified schizophrenia, unspecified acute respiratory failure defibrination syndrome pneumonitis due to inhalation of food or vomitus septic shock cerebral artery occlusion, unspecified with cerebral infarction 

Answer: The patient is high likely exposed to