Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: the patient comes in with chest pain. he awoke at 4 a.m. in the morning and went to the bathroom. he returned, and then on returning he had chest pressure with radiation to his left arm which he considers his anginal equivalent. he took two sublingual nitroglycerin tablets, and the pain resolved. thirty minutes later while in bed he had chest pressure relieved with one sublingual nitroglycerin. the pain returned in 30 minutes again. this time it was to with diaphoresis. no nausea, vomiting, or shortness of breath. he called emergency medical service. they brought him to the emergency department at . he received three sublingual nitroglycerin in the ambulance and some morphine, and there was no resolution of the pain. in the emergency department he was given a nitroglycerin drip and heparin drip and had resolution of his pain. he has been pain free since. the patient normally can walk several blocks and greater than a flight of stairs. he has no paroxysmal nocturnal dyspnea. no orthopnea. review of systems: on review of systems he denied headache, dizziness, shortness of breath, cough, dysuria, nausea, vomiting, diarrhea. he has a little bit of constipation. past medical history: (his past medical history is significant for) 1. coronary artery disease with a non-q-wave myocardial infarction with 3-vessel coronary artery bypass graft back in . in had two stents in his right coronary artery. back in he had two stents in his saphenous vein grafts. 2. he also has had prostate cancer, status post radiation therapy which had led him to have consistently guaiac-positive stools. 3. he also has elevated cholesterol. 4. gout. 5. he has had a right hip replacement. allergies: he has no known drug allergies. social history: he does not smoke. he does not use ethanol; he does not really drink ethanol. no intravenous drug use. his wife lives in the home after having a cerebrovascular accident back in , which has been very stressful for the patient over the past few weeks. family history: his father died at the age of 65 from coronary artery disease. his brother had a myocardial infarction in his 60s and died. there is also hypertension in the family. physical examination on admission: the physical examination on admission showed a temperature of 97.8, pulse of 50, blood pressure 122/46, 99% on 2 liters, respiratory rate 13, weight was 155 pounds to 160 pounds. his head revealed pupils were equal, round, and reactive to light. anicteric. extraocular movements were intact. the oropharynx was clear and moist. his neck revealed there was no jugular venous distention. his neck was soft and supple, and there were no bruits. his lungs were clear to auscultation bilaterally. his heart had a regular rate and rhythm. at times he was in sinus bradycardia, s1/s2. on examination, the patient (mr. ) did have a 2/6 systolic murmur in his right upper sternal border. abdominal examination was nondistended. bowel sounds were positive. soft and nontender. groin revealed there were no bruits. extremities revealed no cyanosis, clubbing or edema. dorsalis pedis pulses bilaterally. his genitourinary examination showed guaiac-positive stool. cranial nerves ii through xii were grossly intact. he was nonfocal. his sensory was intact. motor was in the upper and lower extremities. laboratory values on admission: laboratory values on admission showed a first set of creatine kinases were actually negative at 45, troponin of less than 0.3. his sma-7 was unremarkable except for a glucose of 174. radiology/imaging: electrocardiogram showed sinus bradycardia with first-degree av block, st depressions in v4 to v6 which were new. hospital course: the patient was admitted. he was ruled out for a myocardial infarction by enzymes, and he was subjected to a persantine thallium test which showed a moderate reversible inferior wall defect with an ejection fraction of 65%. the patient was then taken to catheterization on which showed 2-vessel disease with left main coronary that was normal. the left anterior descending artery was 99%, occluded left internal mammary artery to left anterior descending artery was patent. the left circumflex was 80% proximally occluded, 80% mid before the first obtuse marginal. the right coronary artery with 50% mid. two stents were patent. the saphenous vein graft to first obtuse marginal was found to be 90% in-stent thrombosis. the saphenous vein graft to first diagonal showed a 50% in-stent occlusion, 50% in the saphenous vein graft. intervention of the obtuse marginal thrombus was removed and timi-iii flow was achieved. left ventricular ejection fraction was 57%. left ventricular end-diastolic pressure was 12, which was considered normal. the patient was returned to his hospital bed. on the night of , he began to experience chest pressure which was increasing in pain, and so the patient was taken for a re-look angiography. again, his second catheterization totally occluded saphenous vein graft to second obtuse marginal and third obtuse marginal at the site of prior intervention earlier that day. in addition, the percutaneous transluminal coronary angioplasty was complicated by what appeared to be perforation in the actual native vessel of the heart which appeared to be the second obtuse marginal; so, the patient was corrected with tamponade using balloon. the patient was admitted to the coronary care unit overnight. echocardiogram at this time showed no evidence of pericardial effusion, and echocardiogram on the next morning in the coronary care unit showed no additional effusions. the patient was transferred back to the floor on the morning of , . the patient continued to do experience chest pain which he has had since his second intervention. enzymes drawn that day showed evidence for a myocardial infarction with a creatine kinase of 1211, 97 mb fraction 8%, and the next subsequent creatine kinases were 1065, 1213. the patient also demonstrated st elevations in his electrocardiogram. his electrocardiogram actually showed over time evolving anterolateral non-q-wave myocardial infarction. the patient's creatine kinases continued to trend down from the time of his admission starting on after his myocardial infarction. on it was noted that his hematocrit had dropped from the lower 30s to 24%, and the patient also complained of new onset left shoulder pain; so, the patient underwent a ct angiography which showed no dissection. the aorta and a ct of his abdomen and pelvis showed no retroperitoneal bleeds, no femoral bleeds. he was guaiac was actually negative indicating no evidence for severe gastrointestinal bleeding. moreover, his hemolysis laboratory workup did not show evidence of intravascular hemolysis. the patient was transfused with a total of 4 units of blood. his hematocrit decreased isolated but then began to recover and improve to the point where on discharge he was below normal but above 30%. it was never determined what the source of the decrease in hematocrit was. the patient underwent physical therapy after his myocardial infarction and continued to improve. it was determined later that his shoulder pain which was ruled out for aortic dissection turned out was probably related to a fall which he had not reported against the side wall. there was no loss of consciousness at the time, and his pain improved with treatment with tylenol and nonsteroidal antiinflammatory drugs. condition at discharge/status: given that the patient's course improved after the myocardial infarction with continuing trending down enzymes, electrocardiogram resolving, and the patient's overall clinical picture improved, he was discharged on in improved status to the rehabilitation facility. medications on discharge: 1. enteric-coated aspirin 325 mg p.o. q.d. 2. plavix 75 mg p.o. q.d. times 23 more days. 3. lopressor 50 mg p.o. b.i.d. 4. mavik 4 mg p.o. q.d. 5. imdur 30 mg p.o. q.d. 6. serax 10 mg p.o. q.h.s. p.r.n. 7. colace 100 mg p.o. b.i.d. 8. tylenol 650 mg p.o. q.4-6h. p.r.n. discharge diagnoses: 1. non-q-wave myocardial infarction in the anterolateral region. 2. coronary artery disease. 3. hypertension. 4. prostate cancer, status post prostatectomy. code status: his code status is still full. , m.d. dictated by: medquist36 procedure: insertion of non-drug-eluting coronary artery stent(s) insertion of non-drug-eluting coronary artery stent(s) combined right and left heart cardiac catheterization angiocardiography of left heart structures left heart cardiac catheterization other and unspecified coronary arteriography other and unspecified coronary arteriography diagnoses: subendocardial infarction, initial episode of care coronary atherosclerosis of native coronary artery intermediate coronary syndrome unspecified essential hypertension cardiac complications, not elsewhere classified personal history of malignant neoplasm of prostate other complications due to other vascular device, implant, and graft 

Answer: The patient is high likely exposed to