Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: aspirin / nsaids attending: chief complaint: cc: major surgical or invasive procedure: orif history of present illness: . hpi: 83m with pmh significant for cad s/p cabg in , mvr in , chf, and copd, presents to the ed after experiencing r hip pain following a fall. he states that he was bending over, and became dizzy with blurred visual after standing up abruptly. he fell on his side. he denies loc or head trauma. films taken at his rehab () demonstrated r femoral neck fracture, and he was sent to ed. of note, cxr at rehab on suggested evidence of rll and lul infiltrate, and was started on levofloxacin 500mg po qd x 10 days. . in the ed, initial vs were bp 134/69, hr 71, rr 18, sao2 95% 2l nc. hip films confirmed r femoral neck fracture. initial labs significant for inr 5.4, on coumadin. ct head showed no evidence of hemorrhage. he was seen by orthopedic surgery, who recommended admission to medicine service for medical optimization prior to likely orif surgery . mr. also complained of mild flank pain. a ua was ordered once he reached the floor. past medical history: . pmh: cad: s/p cabg s/p mvr s/p ppm, placed at time of valve surgery, v-paced chf - ef 40% on tte pulmonary htn by cath tracheomalacia following prolonged intubation restrictive lung disease with pfts c/w neuromuscular disease, possibly diaphragmatic damage from previous cardiac surgeries h/o endocarditis h/o colon ca yrs ago, resected bph h/o gib social history: social history: the patient denies history of intravenous drug use or ethanol use. he has greater than 33 pack year history of tobacco use, discontinued . his wife recently died. his daughter died emphysema secondary to alpha i antitrypsin deficiency. the patient retired five years ago as a medical engineer. . family history: family history: father died of an mi at age 82, mother died of cancer at age 69. he is a carrier of alpha i antitrypsin gene. . physical exam: . pe: tl 97.1f bp: 135/60, hr: 79, rr: 30, sao2: 90% 2l (prior to neb treatment). gen: ill appearing gentleman, lying in bed, nad heent: perrl, sclerae anicteric, op clear neck: supple, no lad, previous orifice from trach visible cv: rrr, ii/vi sem lusb, mech valve click, +s3 chest: crackles r base, no w/r abd: soft, nt/nd, +bs extr: r leg externally rotated, 2+ dps bilaterally neuro: a&ox3 pertinent results: ecg : v-paced at 84bpm : atrial fibrillation. right axis deviation. compared to the previous tracing of there is deep t wave inversion in leads ii, iii, avf and v3-v6 consistent with active ischemic process. rule out infarction. clinical correlation is suggested. . imaging: cxr : cardiomegaly, s/p cabg and mvr, dual-lead ppm. elevation of right hemidiaphragm with volume loss and interstitial opacities c/w chf. also focal opactiy over r lung zone and fluid in fissure, could be c/w pna. small effusions, no ptx. . head ct : findings: there is no evidence of intra- or extra-axial hemorrhage. the ventricles, cisterns and sulci are mildly prominent, consistent with age-related involutional changes. multiple patchy areas of hypodensity in the white matter consistent with chronic small vessel ischemic disease, and include hypodensity which is more prominent, but unchanged within the left subinsular cortex. a bony protuberance about the ossicle may represent an osteoma which is unchanged or merely a congenital variant. impression: 1. no evidence of intracranial hemorrhage. 2. evidence of age related involutional changes and white matter disease, unchanged. . hip films: there is a comminuted right femoral neck fracture. subtle angulation is present. no other fracture is identified. impression: right femur fracture. . pfts: fvc 1.98l (51% predicted) fev1 1.25l (51% predicted) fev1/fvc: 63% (100% predicted) . tte: ef 40%. the left atrium is moderately dilated. the right atrium is moderately dilated. there is mild symmetric left ventricular hypertrophy. the left ventricular cavity size is normal. overall left ventricular systolic function is moderately depressed secondary to severe inferior and posterior hypokinesis and mild hypokinesis of the rest of the left ventricle; the ejection fraction is approximately 40 percent. there is moderate global right ventricular free wall hypokinesis. the number of aortic valve leaflets cannot be determined. the aortic valve leaflets are moderately thickened. there is no significant aortic valve stenosis. mild (1+) aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. a mitral valve annuloplasty ring is present. there is moderate thickening of the mitral valve chordae. moderate (2+) mitral regurgitation is seen. the mitral regurgitation jet is eccentric. the tricuspid valve leaflets are mildly thickened. there is mild pulmonary artery systolic hypertension. significant pulmonic regurgitation is seen. there is no pericardial effusion. . cath: (prior to mvr) 1. coronary angiography in this right dominant system revealed three vessel cad. the left main coronary artery had a 40% distal stenosis. the lad had an 80% mid-vessel stenosis, and there was competitive flow from the lima in the distal lad. the ramus intermedius branch had a 40% proximal stenosis. the left circumflex artery was totally occluded proximally. the rca had a diffusely diseased proximal segment and was totally occluded after the first acute marginal branch. 2. graft arteriography revealed a patent lima to the lad. the svg to the rpda was widely patent, and the rpda distal to the anastamosis had a 70% stenosis. the svg to the obtuse marginal branch was ectatic but without significant stenosis and the marginal branch distal to the anastamosis supplied collaterals to the right postero-lateral branch. 3. resting hemodynamic measurements revealed severe pulmonary hypertension witha pa systolic pressure of 92 mmhg. there was increased right and left sided filling pressures with a mean ra pressure of 16 mmhg, a mean pcwp of 28 mmhg and an lvedp of 22 mmhg. the cardiac index was preserved at 2.3 l/min/m2. 4. left ventriculography revealed global hypokinesis with posterobasal wall akinesis and moderate-to-severe (3+) mitral regurgitation. the calculated lvef was 45%. final diagnosis: 1. three vessel coronary artery disease. 2. patent lima and svgs. 3. moderate-to-severe (3+) mitral regurgitation. 4. mild systolic ventricular dysfunction. 5. severe pulmonary hypertension 06:58am urine blood-neg nitrite-neg protein-neg glucose-neg ketone-neg bilirubin-neg urobilngn-neg ph-7.0 leuk-neg 06:05am calcium-8.8 phosphate-2.6* magnesium-2.3 06:05am wbc-5.4 rbc-3.18* hgb-10.2* hct-30.2* mcv-95 mch-32.2* mchc-33.9 rdw-16.7* 06:05am pt-43.8* ptt-37.1* inr(pt)-5.0* 01:30am glucose-90 urea n-28* creat-1.1 sodium-138 potassium-4.8 chloride-96 total co2-35* anion gap-12 01:30am ck(cpk)-39 01:30am ctropnt-0.02* 01:30am calcium-8.9 phosphate-2.8 magnesium-2.2 01:30am wbc-6.3# rbc-3.33* hgb-10.7* hct-31.6* mcv-95 mch-32.3* mchc-34.1 rdw-16.5* 01:30am pt-46.7* ptt-37.4* inr(pt)-5.4* brief hospital course: pt. was admitted for optimization of medical status prior to operation for hip fx. stable until early around 12:oo am when began desatting to the high 80s on 2l nc. vital signs o/w at the time: t 97.3 bps 90s-100s/30s-60s, hr 60s-70s, rr in the high 20s. pt was also noted to be increasingly somnolent and unresponsive. pt placed on 100% fm with improvement of sats. he had been given ms contin 45 mg at 11 am the day prior and was therefore given narcan 0.2 mg x1 and narcan 0.4 mg x1 several hours later. he was given lasix 10 mg iv x3 o/n. mental status improved somewhat with the early dose of narcan. cxr checked at the onset of the pt's change in status demonstrated worsened bibasilar pna. abg trend o/n was as follows: 12:20 am 7.33/68/70 4:00 am 7.23/90/65 6:00 am 7.27/79/60 at time of micu eval abg was checked and demonstrated 7.05/139/125. given worsening respiratory status pt transferred to the unit. code status confirmed with family to be dnr/dni. hcp felt that would be an in-line with the pt's wishes. pt. ekg showed new afib with st changes worrisome for ischemia and trop leak without elevation of ck in context of rapidly progressive arf. he was given trial of overnight without much improvement of ms. in discussion with pt.'s family, it was decided to choose comfort care interventions. he was placed on morphine drip and passed with family around medications on admission: meds: lopressor 25mg po bid prilosec 20mg po qd coumadin 3mg 5d/wk, 2mg 2d/wk azmacort 2 puffs tid levaquin ( last dose - ?pnemonia) lasix 10mg po qd lisinopril 5mg po qd albuterol neb atrovent neb combivent 2 puffs qid wellbutrin xl 150mg po qd dulcolax 10mg pr prn remeron 7.5mg po qhs . discharge medications: none discharge disposition: expired discharge diagnosis: hypercarbic respiratory failure cad: s/p cabg atrial fibrillation acute renal failure hip fracture chf pna discharge condition: deceased discharge instructions: none followup instructions: none procedure: non-invasive mechanical ventilation diagnoses: pneumonia, organism unspecified congestive heart failure, unspecified personal history of malignant neoplasm of gastrointestinal tract, unspecified acute kidney failure, unspecified unspecified septicemia severe sepsis obstructive chronic bronchitis with (acute) exacerbation aortocoronary bypass status unspecified fall heart valve replaced by other means acute respiratory failure pneumonitis due to inhalation of food or vomitus septic shock long-term (current) use of anticoagulants other alteration of consciousness closed fracture of unspecified part of neck of femur 

Answer: The patient is high likely exposed to