Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: seizures, aphasia major surgical or invasive procedure: intubation/mechanical ventilation history of present illness: 66 yo m with a remote hx of eee resulting in a seizure disorder and etoh use, transferred to medicine from neurology for management of hypoxic respiratory distress. patient was initially presented to an osh on for evaluation of sudden onset aphasia that day, which was noted to occur in the past after seizures. on arrival to , he was noted to be in status epilepticus with subtherapeutic dilantin level, so he was intubated, loaded with dilantin, given iv valium, and transferred to neurology icu service. he was re-started on keppra, dilantin, and klonopin. no further episodes of status while in-house, although noted to have short episodes of seizures in lower extremities that would subside with ativan. hospital course complicated by nstemi with troponins to 0.9. cardiology was consulted and thought nstemi due to demand ischemia likely related to peri-intubation hypotension (transiently required neo gtt in icu). patient on heparin from and continued on cardioprotective medication. also noted to have a pna (fevers, leukocytosis, and rll consolidation), started on vancomycin and zosyn on at osh, which have been continued to date. patient self-extubated on , requiring bipap. resiratory status stabilized, and patient was called out to neuro floor on . called out to a negative pressure ?tuberculosis exposure (daughter's boyfriend died of tuberculosis). was ?receiving fluids when noted to be having difficulty with respiration. received 1 dose of solumedrol for wheezing on . med/ consult called for management of respiratory issues on , noted to be volume overloaded/with crackles on exam after getting maitenence fluids o/n. when evaluated by the primary team on the floor this afternoon, patient's breathing continued to be quite tachypneic/using accessory muscle to breathe despite being on 5 l o2 and 2 doses of lasix 40 mg iv, ipratroprium nebs, with -1 l diuresis in one hour. repeat abg shows increased hypoxia on same oxygen settings and worsening a-a gradient (570=>583, normal 19). patient also appeared slightly more confused, pulling off oxygen mask frequently during nebulizer treatment and insisting a 'cap' was on the floor, although nothing was noted on the floor. therefore, micu resident monitoring patient on the floor recommended immediate icu transfer. past medical history: seizure do s/p eee encephalitis # hx of etohism since # htn # hyperlipidemia # nstemi: echo with ef 20% to 25% (in setting of recent mi) social history: lives with daughter. daughter's boyfriend recently died of tuberculosis on . +etoh. denies illicit drug use or smoking hx. family history: noncontributory physical exam: vs: 97.6 110/80 76 28 95-97% on 5 l nc bs 105 ga: middle aged m lying in bed, labored breathing, aox2 (knows name and date) heent: perrla. mm dry. no lad. unable to assess jvd. neck supple. cards: distant hs heard. pulm: +expiratory wheezes scattered throughout all lung fields. +crackles at bases. abd: soft, obese, nt, +bs. no g/rt. neg hsm. neg sign. extremities: wwp, no edema. dps, pts 2+. skin: no rashes neuro/psych: cns ii-xii intact. 4/5 strength in u/l extremities. dtrs + bl (biceps, achilles). sensation grossly intact to lt. cerebellar fxn poor (unale to do ftn accurately). gait wnl. babinski upgoing bilaterally (r>l. spelled world backward "dlow". pertinent results: labs: 08:00am blood wbc-6.7 rbc-4.63 hgb-14.5 hct-39.6* mcv-86 mch-31.3 mchc-36.6* rdw-13.4 plt ct-362 07:20pm blood wbc-20.1* rbc-5.84 hgb-18.3* hct-48.4 mcv-83 mch-31.3 mchc-37.8* rdw-13.8 plt ct-339 07:20pm blood neuts-79.5* lymphs-14.6* monos-5.5 eos-0.1 baso-0.2 08:00am blood pt-13.8* ptt-22.4 inr(pt)-1.2* 08:00am blood glucose-114* urean-14 creat-1.0 na-141 k-3.9 cl-103 hco3-30 angap-12 07:20pm blood glucose-231* urean-14 creat-0.9 na-138 k-3.9 cl-101 hco3-23 angap-18 05:30am blood alt-18 ast-32 ld(ldh)-198 ck(cpk)-155 alkphos-101 totbili-0.3 12:20pm blood ck(cpk)-161 01:57pm blood ck(cpk)-118 10:47pm blood ck-mb-15* mb indx-12.4* ctropnt-0.91* 01:57pm blood ck-mb-3 ctropnt-0.14* 08:00am blood calcium-9.0 phos-3.1 mg-2.4 05:30am blood %hba1c-5.9 05:30am blood triglyc-452* hdl-21 chol/hd-7.1 05:30am blood tsh-5.3* 02:53am blood free t4-0.99 08:05am blood phenyto-20.8* 07:20pm blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg imaging: cxrs ~ : diffuse interstitial opacities are most likely due to pulmonary edema. repeat radiography following appropriate diuresis is recommended to assess for underlying infection. ~ : in comparison with the earlier study of this date, the endotracheal tube has been removed. there is opacification at the left base silhouetting the hemidiaphragm. this most likely represents atelectasis and pleural effusion, though the possibility of supervening pneumonia cannot be definitely excluded. pulmonary vascularity is now essentially within normal limits. ~ : moderate cardiomegaly is exaggerated by lordotic positioning, but has probably increased consistent with an enlarging heart, and/or pericardial effusion. azygos distention indicates worsening elevated central venous pressure. there is no pulmonary edema, but left lower lobe consolidation persists, either atelectasis or pneumonia. left subclavian line tip projects over the junction of brachiocephalic veins. there is no pneumothorax. ~ : since earlier today, lung volumes remain low. left lower lobe consolidation, right basilar atelectasis, and small bilateral pleural effusions slightly improved. effusion in the minor fissure is not present anymore. echo: ~: severe left ventricular systolic dysfunction consistent with multivessel coronary artery disease or stress cardiomyopathy. ~: moderate regional left ventricular systolic dysfunction. mild pulmonary hypertension. micro: afb smear neg x 3, cultures pending urine and blood cultures neg brief hospital course: brief hospital summary: 66 yo m with seizure disorder after eee infection and former etoh abuser who was transferred from an osh in status epilepticus. intubated and admitted to the neuro icu service. hospital course complicated by an nstemi resulting in depressed lv ejection fraction, ventilator associated pneumonia treated with 8 day course of vancomycin and zosyn, and hypoxic respiratory distress due to volume overload from depressed lv ejection fraction. patient was also ruled out for tuberculosis due to a possible exposure within his family members. during this hospital course, patient was transferred to the neurology icu to the neurology floor to the medicine floor to the medical icu and finally back to the medicine floor. he was discharged home with physical therapy. hospital course by problem: # status epilepticus: patient initially presented to the neuro icu intubated in status epilepticus, likely in setting of subtherapeutic dilantin level. mr. daughter reports that he typically has seizures every 4-6 months and that these occur in the context of low or high levels of dilantin. his level at the osh was subtherapeutic. unlikely meningitis given lack of nuchal rigidity. unlikely alcohol withdrawal as patient reported he had not had etoh use recently and his toxicology screen was negative for etoh. his head ct from the osh shows stable l frontal encephalomalacia. the patient while in the micu became acutely agitated, attempted to leave the unit without any clothes, and a code purple was called. he received on dose of iv haldol 2mg and improved. his mental status exam eventually improved, and his seizure medications were changed to keppra 1500 mg po bid, dilantin 300 mg po qhs (with therapeutic dilantin levels), and klonopin 1.5 mg po qhs on discharge. given prn ativan for seizures > 3min. no further seizure activity on discharge. # hypoxia: patient became acute hypoxic on the floor after being called out from the icu, presumably in the setting of continous ivfs and decreased lv ejection fraction of 30%. aspiration pneumonia may also have contributed to hypoxia. he was transferred to the micu after remaining on the floor for 6 hours due to concern for hypoxic respiratory distress and aggressively diuresed with iv lasix and treated with standing nebulizers. he improved gradually and was transferred back to the floor. transitioned to oral lasix. continued beta-blocker and ace-inhibitor. continued albuterol and ipratroprum nebs as needed. room air sats were 94% on ra at rest and xx% on ambulation. cxr showed stable l lobar opacities. he was discharged with home physical therapy. # ventilator associated pneumonia: patient noted to have leukocytosis and left lower lobe infiltrate on cxr, likely vap versus aspiration pna during seizures 9could not distinguish between the two). unable to obtain speciation on sputum culture, so treated with 8 day course of vancomycin and zosyn. kept on aspiration precautions and diet per speech and swallow recommendations. # tuberculosis exposure: recent exposure to family member who died of tuberculosis of . afb sputum negative x3. kept in negative pressure room until completely ruled out. # nstemi: patient suffered an nstemi likely due to demand ischemia (hypotensive and tachycardic in the neuro icu after intubation requiring transient neosynephrine gtt) versus takatsubo/stress cardiomyopathy. therefore, decision was made not to do cardiac catherization. cardiac enzymes were cycled and were downtrending on discharge. continued cardioprotective meds (asa, statin, beta-blocker, started low dose ace-inhibitor). patient will need a stress test as outpatient # pump: patient had pulmonary edema with poor ef% (20-25%) which improved to 30% on repeat tte, likely due to recent myocardial insult. patient was diuresed and treated with beta blocker and afterload reduced with ace-inhibitor. he will need a repeat tte in weeks as outpatient. # rhythm: no arrythmias noted on telemetry. continued telemetry # etoh use: no signs of withdrawal. continue mvi, thiamine, folate medications on admission: - atenolol 25mg po qd - folate 1 mg qd - mvi qd - simvastatin 40mg po qd - keppra 1500 mg po bid - klonopin 1mg qam and 1.5 qhs po - amlodipine 5mg po qd - dilantin 200mg po qhs discharge medications: 1. simvastatin 40 mg tablet sig: one (1) tablet po daily (daily). 2. aspirin 325 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po daily (daily). 3. thiamine hcl 100 mg tablet sig: one (1) tablet po daily (daily). 4. folic acid 1 mg tablet sig: one (1) tablet po daily (daily). 5. levetiracetam 500 mg tablet sig: three (3) tablet po bid (2 times a day). 6. phenytoin sodium extended 100 mg capsule sig: three (3) capsule po hs (at bedtime). 7. clonazepam 0.5 mg tablet sig: three (3) tablet po qhs (once a day (at bedtime)). 8. furosemide 40 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 9. multivitamin tablet sig: one (1) tablet po daily (daily). 10. metoprolol tartrate 25 mg tablet sig: 1.5 tablets po bid (2 times a day). 11. lisinopril 5 mg tablet sig: 0.5 tablet po daily (daily). 12. albuterol 90 mcg/actuation aerosol sig: 1-2 puffs inhalation twice a day as needed for shortness of breath or wheezing. 13. amlodipine 5 mg tablet sig: one (1) tablet po once a day. discharge disposition: home with service facility: vna discharge diagnosis: primary: status epilepticus non-st elevation myocardial infarction ventilator associated pneumonia acute systolic congestive heart failure discharge condition: satting well on room air on discharge discharge instructions: you were admitted initially with a diagnosis of seizures (status epilepticus). in the course of your hospital stay, you suffered a heart attack that damaged the pumping function of your heart, a pneumonia that was treated with antibiotics, and volume overload and difficulty breathing. due a possible tuberculosis exposure, you were also ruled out for tuberculosis with 3 sets of sputums that were negative for tuberculosis on special stains. you were satting well on room air off of oxygen on discharge. please take your medications as directed. you were started on oral lasix 40 mg by mouth daily. your seizure medications were increased to: dilantin 300 mg by mouth at night klonopin 1.5 mg by mouth at night keppra 1500 mg by mouth twice a day your atenolol was changed to metoprolol 37.5 mg by mouth twice a day. lisinopril 2.5 mg by mouth was added to your medications to help with heart remodeling. please weigh yourself daily and call your pcp if your weight gain is > 3 lbs in one day. fluid restrict to < 2 l daily. please return to the ed or call your pcp if you experience shortness of breath, chest pain, fevers > 101 f, swelling in your legs, weight gain greater than 3 lbs in one day, or any symptoms concerning enough to you to warrant physician . followup instructions: please schedule a follow up appointment with your pcp weeks after discharge. if you have no pcp, call to schedule an appt at . cardiology: dr @ 1:00pm, center . . you should have an echo repeated and stress test with dr . please also follow up with your neurologist within 1-2 weeks after discharge. md procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified venous catheterization, not elsewhere classified non-invasive mechanical ventilation diagnoses: subendocardial infarction, initial episode of care congestive heart failure, unspecified unspecified essential hypertension other and unspecified hyperlipidemia pneumonitis due to inhalation of food or vomitus cardiogenic shock grand mal status ventilator associated pneumonia acute on chronic systolic heart failure aphasia alcohol abuse, in remission contact with or exposure to tuberculosis 

Answer: The patient is high likely exposed to