Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: penicillins / sulfa (sulfonamides) attending: chief complaint: cc - acute hepatitis/subacute liver failure major surgical or invasive procedure: liver biopsy, history of present illness: hpi - this is a 39 y/o female with iddm who was in her usoh until 2 weeks prior to admission when she started feeling nauseous with decreased appetite, and extreme fatigue. she noted that she had been feeling fatigued for several months, but attributed it to work-related stress. she also reported a 5-lb weight loss over the last few months. she denied any fevers or chills. her mother noticed yellowish discoloration of skin and eyes approximately 1 week ago, prompting the patient to go to . there, she was noted to have abnormal lfts and on f/u with her pcp, findings were confirmed. she was referred to on for further work-up. there she had an abdominal u/s which demonstrated unexplained gb wall thickening at 3 mm, but no other abnormalities. lfts demonstrated elevated transaminases in the 1,000's and a total bilirubin greater than 8. she also had an mrcp which revealed a heterogenous liver with periportal edema compatible with acute hepatitis without any abnormality of the biliary system. work-up there also included viral hepatitis serologies which were negative; autoimmune labs were pending at the time of transfer. she was transferred to the micu on for the possible diagnosis of subacute liver failure, further dignostic workup and potential consideration of liver transplantation. past medical history: pmh - 1. dm1 w/ microalbuminuria for 25 years 2. nephrolitiasis 3. ibs social history: works as an attorney. no tobacco, occasional etoh, no ivdu. no tattoos, no h/o exposure to hep b/hep c. last traveled one year ago to central europe, no illness during that travel. no exposure to hepatotoxic drugs or herbs on repeat assessment. family history: nc physical exam: vs: tc 98.6, bp 95/68, hr 91, rr 18, sao2 98%/ra, fs 92 general: pleasant young woman in nad, ao x 3 heent: nc/at, perrl, eomi. sclerae icteric. mmm, op clear. neck: supple, no lad or tmg chest: cta-b, no w/r/r cv: rrr s1 s2 normal, no m/g/r abdomen: soft, nt/nd, nabs. no palpable hsm ext: no c/c/e, pulses 2+ b/l neuro: ao x 3, non-focal, no asterixis skin: +jaundice, no other stigmata of chronic liver disease . pertinent results: abd u/s - the liver demonstrates no focal or textural abnormalities. there is no intra- or extrahepatic biliary ductal dilatation. there is mild gallbladder wall thickening up to 3 mm with no evidence of cholelithiasis or adjacent pericholecystic fluid to indicate acute cholecystitis. the common bile duct measures 4 mm and is not dilated. the main portal vein is patent with forward flow. the aorta is of normal caliber throughout its visualized course. the pancreas and spleen are unremarkable. there is no ascites. the right kidney measures 9.7 cm and the left kidney measures 9.6 cm in length. there is no evidence of hydronephrosis, stones, or mass within either kidney. . liver, needle core biopsy: 1. marked portal, periportal, and moderate lobular, mixed inflammation with prominent plasma cells, scattered eosinophils and neutrophils. 2. foci of hepatocyte necrosis with drop-out and numerous apoptotic hepatocytes identified. evidence of submassive necrosis is not present; see note. 3. mild cholestasis and focal, mild pericentrivenular hemorrhage is identified. 4. results of trichrome, iron and reticulin stains will be reported separately in an addendum. note: the features are those of a chronic active hepatitis. the presence of a prominent plasmacytic component to the inflammatory infiltrate is suggestive of an autoimmune etiology, however, a drug effect (immune-mediated) or less likely, atypical variant of viral hepatitis, cannot be entirely excluded. the areas of hepatocyte necrosis with drop-out are estimated to comprise approximately 20-30% of the biopsy. correlation with clinical and serological findings is recommended. addendum: liver path conference as of : compatible with severe autoimmune hepatitis with massive collapse of hepatocyte plates; maximally stage 1 fibrosis. brief hospital course: this is a 39 y/o female with iddm who presented with subacute hepatitis that after an extensive work-up was found to be due to an autoimmune process. the following issues were investigated during this hospitalization: . # autoimmune hepatitis - confirmed with biopsy results. the patient was started on 40 mg of prednisone qd for two days, followed by imuran 50 mg. she had a dramatic improvement with these agents, with lfts decreasing to half their levels on admission and steadily trending downward. her synthetic function evidenced by coags and albumin, improved as well and her presenting symptoms resolved. she was discharged with instructions to follow up in hepatology for tapering of her steroid dosage. . # iddm: previously well-controlled, with a hba1c of 6.2. pt. was followed by private endocrinologist. during this hospitalization, her insulin requirement increased significantly as expected because of her steroid treatment. she was followed by and new recommendations on daily doses of glargine and a humalog sliding scale were made. she was discharged with these recommendations and instructions to follow with her endocrinologist for adjustments while her steroid treatment was tapered down. additionally, an appointment was made for her to follow up at for classes on carbohydrate counting and diabetic diet. a follow up appointment was made with the attending, dr. , at the liver outpatient clinic on , . medications on admission: - 1. humulin-n 10 units qam, 10 units qpm 2. tylenol prn (8 tabs per month) . meds on transfer - 1. protonix 40 mg qd 2. insulin nph , hss discharge medications: 1. prednisone 20 mg tablet sig: two (2) tablet po once a day for 1 days: take 2 pills together, for one day for a total of 40 mg. . disp:*2 tablet(s)* refills:*0* 2. azathioprine 50 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 3. hexavitamin tablet sig: one (1) cap po daily (daily). disp:*30 cap(s)* refills:*2* 4. cholecalciferol (vitamin d3) 400 unit tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 5. calcium carbonate 500 mg tablet, chewable sig: one (1) tablet, chewable po tid (3 times a day). disp:*90 tablet, chewable(s)* refills:*2* 6. prednisone 10 mg tablet sig: three (3) tablet po once a day for 10 days: take 3 pills together, once a day for a total of 30 mg every day. . disp:*30 tablet(s)* refills:*0* 7. lantus 100 unit/ml cartridge sig: 18 units subcutaneous once a day: administer 18 units each day at noon. disp:*qs * refills:*2* 8. humalog pen 100 unit/ml (75-25) insulin pen sig: as directed subcutaneous with meals: administer prescribed units with meals according to finger sticks and as recommended by sliding scale. disp:*qs * refills:*2* discharge disposition: home discharge diagnosis: primary 1. auto-immune hepatitis . secondary 1. dm type 1 w/ microalbuminuria 2. nephrolitiasis 3. ibs discharge condition: stable with liver enzymes trending downward, resolving jaundice. discharge instructions: you were seen and evaluated for acute hepatitis, which after being worked up with lab tests and a liver biopsy, was diagnosed as auto-immune hepatitis. for this condition, you were started on an immunosuppressant called imuran. additionally, you were started on prednisone, a steroid, which will slowly be tapered down while you continue the imuran. you should complete a week of prednisone at 40 mg, which would require you to take 1 more dose on . on , you will begin doses of 30 mg each day. continue this dose until you see dr. on . . steroids cause higher blood sugar levels which of course is problem for someone with diabetes, like yourself. for this reason, a new insulin regimen has been made for you and a copy of this regimen (the original one, given the reduced readings on your meter) has been included in your discharge paperwork. since you will be tapered down on prednisone, your insulin requirements will likewise need to be tapered down. please be sure to consult your endocrinologist for instructions on how to do this. . * please take all of your medications as directed. . * please keep all of your follow-up appointments . * call your doctor or go to the er for any of the following: abdominal pain, change in your bowel movements (i.e. color - tan/white, black or with frank blood; diarrhea, etc.), if you notice your skin, gums or eyes turning yellow, lightheadedness, jitters, nausea/vomiting, fevers/chills or any other concerning symptoms. followup instructions: * please see your endocrinologist for an appointment as soon as possible on or before you decrease your prednisone dosage, so that your insulin can be adjusted appropriately. . * you will be contact by the diabetes today program () with an appointment so that you can take classes for your diet to include carbohydrate counting. . provider: , md (gastroenterology) phone: date/time: 1:30 procedure: closed (percutaneous) [needle] biopsy of liver diagnoses: other chronic hepatitis diabetes mellitus without mention of complication, type i [juvenile type], not stated as uncontrolled other and unspecified coagulation defects 

Answer: The patient is high likely exposed to