Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: the patient is a 77 year old man with a history of diabetes mellitus, coronary artery disease, status post three vessel coronary artery bypass grafting and status post mitral valve replacement, who presented to an outside hospital with ventricular tachycardia, progressing to a ventricular fibrillation arrest, with chronic defibrillation times two. the patient reports he was driving his car when he began to note some lightheadedness. he pulled over to the side of the road and then lost consciousness. prior to this, he had no symptoms of chest pain, shortness of breath, diaphoresis or any other anginal equivalent at that time. he was found a short time later, he does not know how long. emergency medical service was called and an electrocardiogram at that time reportedly revealed supraventricular tachycardia at a rate of 200 to 210, although no strips are available for review. the patient was given 6 mg of adenosine en route to an outside hospital, which had essentially no effect. upon arrival to the outside hospital, he was found to be in a tachycardia to approximately 200, of unknown etiology. he then rapidly progressed to monomorphic ventricular tachycardia, became pulseless and cyanotic, for which he was rapidly defibrillated at 200 joules, with an immediate resumption of normal sinus rhythm. the patient again went into ventricular tachycardia a short time later, with degeneration into ventricular fibrillation and was again defibrillated, this time with 300 joules, again returning to normal sinus rhythm immediately. at this time, he was given a 100 mg lidocaine bolus and a 2 mg/minute continuous intravenous drip was started. the patient remained in normal sinus rhythm after that and was transferred to for further management. of note, the patient denies ever having had an anginal equivalent or chest pain in the past. his initial coronary artery disease was picked up on a routine workup for another medical illness that he does not recall, ultimately resulting in stress, cardiac catheterization and then coronary artery bypass grafting. upon arrival to , the patient was without complaint. he had no chest pain or shortness of breath. past medical history: 1. coronary artery disease, status post three vessel coronary artery bypass grafting in ; also performed at the same time was a mitral valve repair which failed; patient then had a mitral valve replacement with a mechanical valve approximately in . 2. abdominal aortic aneurysm repair. 3. diabetes mellitus times ten years, controlled with glynase after diet management failed. 4. peptic ulcer disease. medications on admission: adalat 30 mg p.o.q.d., lopressor 50 mg p.o.b.i.d., lipitor 10 mg p.o.q.d., glynase 3 mg p.o.q.d., accupril 20 mg p.o.q.d., lanoxin 0.125 mg p.o.b.i.d. (patient verifies that his dosing is b.i.d.), coumadin 2.5 mg p.o.q.d., prevacid 30 mg p.o.q.d., zantac 150 mg p.o.q.d. allergies: penicillin (rash). social history: the patient does not currently smoke, he quit 20 years ago, and denies any alcohol intake. he lives with his wife in . he is a retired police officer. family history: noncontributory. physical examination: on physical examination on admission, the patient had a blood pressure of 143/103, pulse 69 and regular, respiratory rate 18 and oxygen saturation 98% in room air. general: well appearing, in no acute distress. head, eyes, ears, nose and throat: anicteric sclerae, oropharynx clear with moist mucous membranes. neck: jugular venous pressure to 6 cm, estimated central venous pressure of approximately 14. respiratory: lungs clear to auscultation bilaterally. cardiovascular: regular rate and rhythm, mechanical s1, normal s2, soft crescendo-decrescendo systolic murmur best heard at left sternal border, nonradiating. abdomen: old surgical scars, soft, benign. rectal: brown guaiac negative stool. extremities: no cyanosis, clubbing or edema, 2+ pulses bilaterally. laboratory data: electrocardiogram on admission showed normal sinus rhythm at 71 beats per minute, normal axis, normal intervals, borderline first degree a-v block, partial right bundle branch block, t wave inversions in ii, iii, v5 and v6. hospital course: 1. cardiovascular: given the patient's extensive history of coronary artery disease, it was suspected that he had had a primary arrhythmic event and this is what led to his monomorphic ventricular tachycardia and his need for defibrillation. the patient was continued on lidocaine overnight, which was stopped on hospital day number two, after he had been stable. he was scheduled to go to the electrophysiology laboratory for an electrophysiology study. cardiac enzymes were cycled and revealed cks of 187, 228 and 215 with mbs of 14, 18 and 16. we believed that this was a troponin leak secondary to his tachycardia and not a primary event. however, given the patient's extensive history, we could not rule out a primary cardiac vent leading to the arrhythmia. the plan was for the patient to go to the cardiac catheterization laboratory and, following his catheterization, go to the electrophysiology laboratory for an electrophysiology study and, most likely, an icd placement. on hospital day number two, however, the patient began to develop increasing blood pressure to approximately 200 systolic. he then developed rales bilaterally, approximately one-half way up, and his oxygen requirement began to increase. it was believed that the patient had flashed into pulmonary edema and he was diuresed with lasix. on hospital day number three, the patient's lungs were clear and his oxygen requirement had returned to , however, the patient's bun and creatinine had risen. his creatinine on hospital day two was in the mid-2s compared with 1.6 on admission. because of this rise in creatinine, it was believed it was not safe at the current time to send him to the catheterization laboratory, so catheterization was delayed. the electrophysiology service offered, in light of his delayed catheterization, to take the patient to the electrophysiology for an electrophysiology study to see if he had an ablatable focus. on , the patient was taken to the electrophysiology laboratory. a focus of atrial tachycardia was found, which was ablated during the electrophysiology study. a plan was made for the patient to have a pacemaker and icd placement after his catheterization. on the same day, an echocardiogram was performed which revealed mild symmetric left ventricular hypertrophy, normal left ventricular cavity size, severely depressed left ventricular function with a left ventricular ejection fraction of 25% to 30% and sever global left ventricular hypokinesis. the patient also showed a depressed right ventricular function, moderate tricuspid regurgitation, mitral valve prosthesis with normal function, no mitral regurgitation. the patient continued to be stable following his electrophysiology study and was transferred to the floor awaiting his catheterization. catheterization was performed and revealed a 100% occluded right coronary artery, left anterior descending artery and left circumflex. the patient also had three saphenous vein grafts. the superior saphenous vein graft to the obtuse marginal two was patent. saphenous vein graft to the distal right coronary artery was patent but the saphenous vein graft to the left anterior descending artery was occluded proximally with a mid- left anterior descending artery and distal graft filling via right-to-left collaterals. at the time, the decision was made to do no intervention and that medical management only would be preferred. the patient was sent back to the floor and, the following day, had an electrophysiology study in which an icd was implanted with ddd mode pacing capabilities. the procedure was uncomplicated and the patient was returned back to the floor in stable condition. upon returning back to the floor, the patient's coumadin was restarted, although he was continued on heparin for his mechanical valve. the patient remained in house for four days awaiting his inr to become therapeutic. on the day of discharge, his inr was 2.1 and it was deemed safe to send him home. the patient will have no medications make. i will tell him to return to his 2.5 mg daily of coumadin and he will follow up with his primary care physician, . , the day following discharge. additionally, per patient's discussion with the electrophysiology team, electrophysiology will see him today before he leaves and then, one month from now, he will be seen by dr. for follow-up on his icd pacemaker implantation. discharge diagnosis: ventricular fibrillation. coronary artery disease. flash pulmonary edema. anticoagulation for mechanical mitral valve. discharge medications: adalat 30 mg p.o.q.d. lopressor 50 mg p.o.b.i.d. lipitor 10 mg p.o.q.d. glynase 3 mg p.o.q.d. accupril 20 mg p.o.q.d. lanoxin 0.125 mg p.o.b.i.d. coumadin 2.5 mg p.o.q.d. prevacid 30 mg p.o.q.d. zantac 150 mg p.o.q.d. discharge status: to home. discharge condition: stable. follow-up: the patient will follow up with dr. , his primary care physician. , m.d. dictated by: medquist36 d: 10:21 t: 07:28 job#: procedure: combined right and left heart cardiac catheterization coronary arteriography using two catheters angiocardiography of left heart structures catheter based invasive electrophysiologic testing excision or destruction of other lesion or tissue of heart, endovascular approach implantation or replacement of automatic cardioverter/defibrillator, total system [aicd] diagnoses: urinary tract infection, site not specified congestive heart failure, unspecified diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled coronary atherosclerosis of autologous vein bypass graft aortocoronary bypass status paroxysmal ventricular tachycardia heart valve replaced by other means cardiac arrest ventricular fibrillation 

Answer: The patient is high likely exposed to