Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: 74 y/o f with cad, chf, copd admitted with pna/copd exacerbation and sepsis. . major surgical or invasive procedure: none history of present illness: 72 year old female with severe copd who presents with severe sob. intubated in ed for respiratory failure and cap. subsequent hypotension and and tachycardia, requiring ivf's and levophed in ed. also was autopeeping. started on ctx, vanc, solumedrol/bronchodilators. wbc 20 with 20% bands, arf, prerenal. no further information able to be obtained. past medical history: 1. copd on 2l home oxygen 2. dvt in past 3. cad status post mi 4. chf 5. htn 6. pnemonia 7. history of tachyarrhythmia social history: lives with husband. former heavy smoker. family history: noncontributory physical exam: vitals: 98.2, 112, 81/42, 30 gen: petite female in nad, intubated heent: perrla cor: rrr, nl s1 and s2, sem lungs: ventilator bs throughout, depressed on l abd: soft, ntnd, +bs ext/lines: r ij, no edema neuro: sedated pertinent results: 09:00pm wbc-19.9* rbc-4.44 hgb-12.7 hct-36.9 mcv-83 mch-28.5 mchc-34.4 rdw-14.8 09:00pm neuts-76* bands-21* lymphs-1* monos-1* eos-0 basos-0 atyps-0 metas-1* myelos-0 09:00pm pt-12.8 ptt-32.0 inr(pt)-1.1 09:00pm plt smr-normal plt count-157# 09:00pm lactate-3.6* 09:00pm tot prot-6.2* 09:00pm ck-mb-notdone 09:00pm ck(cpk)-87 09:00pm glucose-134* urea n-46* creat-1.4* sodium-131* potassium-3.5 chloride-82* total co2-36* anion gap-17 12:57am lactate-3.0* 01:40am urine blood-lge nitrite-neg protein-30 glucose-neg ketone-tr bilirubin-neg urobilngn-neg ph-6.5 leuk-mod 11:44pm type-art po2-53* pco2-64* ph-7.31* total co2-34* base xs-2 ........ cxr impression: asymmetric pulmonary edema superimposed on severe, bullous emphysema ......... ekg sinus tachycardia. other than a more rapid rate, no diagnostic change from the previous tracing of . the tracing continues to show right axis deviation, left ventricular hypertrophy by voltage in the precordial leads, and non-specific st-t wave abnormalities. .......... echo conclusions: the left atrium is normal in size. there is mild symmetric left ventricular hypertrophy. the left ventricular cavity size is normal. there is moderate global left ventricular hypokinesis (anterior wall appears slightly more hypokinetic). the aortic valve leaflets are severely thickened/deformed. there is severe aortic valve stenosis. trace aortic regurgitation is seen. the mitral valve leaflets are moderately thickened. moderate (2+) mitral regurgitation is seen. the mitral regurgitation jet is eccentric. the tricuspid valve leaflets are mildly thickened. there is moderate pulmonary artery systolic hypertension. there is no pericardial effusion. compared with the findings of the prior study (tape reviewed) of , the overall lvef has significantly decreased and the degree of aortic stenosis detected is now severe. ................ ct head today's examination is compared to the prior from . again, there are small areas of periventricular white matter hypoattenuation, which likely relates to chronic microvascular ischemic changes. however, there is no evidence of an intracranial hemorrhage. the -white matter differentiation is otherwise preserved. there is no midline shift, mass effect or hydrocephalus. impression: chronic microvascular ischemic changes without evidence of acute intracranial hemorrhage or infarct. no midline shift. .......... mri findings: the diffusion images demonstrate multiple small foci of slow diffusion involving the left frontal and both parietooccipital lobes. small foci of slow diffusion are also seen in both cerebellar hemispheres. several of these foci are also visualized on t2 and flair images. findings are indicative of multiple acute small cortical and subcortical infarcts. there is no mass effect or hydrocephalus. a small focus of low signal on susceptibility-weighted images in the left corona radiata basal ganglia region indicate a small area of chronic blood products from previous hemorrhage. there is no hydrocephalus or midline shift seen. impression: multiple small areas of acute infarcts in both cerebral and cerebellar hemispheres as described above. no mass effect or hydrocephalus. other changes as above brief hospital course: a/p: 1.)respiratory failure - patient presented with sob and respiratory failure and was found to have pna on cxr with bilateral lower lobe opacities, as well as mild chf, superimposed on severe emphysema. the patient was intubated and ventilated. empiric antibiotics were started to cover community acquired pna and sepsis. once blood, urine, and sputum cultures were obtained antibiotics were tailored appropriately. attempts at weaning the patient were unsuccessful. the patient also had severe as and mild chf contributing to the respiratory picture, and she was gently hydrated in the setting of sepsis. albuterol, atrovent, and solumedrol were given for treatment of copd. however, once the patient suffered a cva (see below), goals of care were reassessed and the patient was extubated, became apneic, and passed away from respiratory distress. . 2.)sepsis - patient was hypotensive and tachycardica. blood cultures grew mrsa in bottles. gpc were found on gram stain of the sputum, but nothing grew, and mrsa grew in the urine. she was treated with broad spectum empiric antibiotics but eventually tapered to vancomycin, as staph aureus was sensitive to this. hypotension was also treated with gentle fluid boluses in setting of as. there was a concern for endocarditis with her valve, and a tte was done that showed worsening as and ef, but no vegetations. however, the suspicion for endocarditis remained high, especially after her cva that showed evidence of multiple acute emboli. the patient required pressors throughout her hospital stay to maintain pressures. once it was determined that recovery from cva would be minimal, it was decided to withdraw all artificial support and the patient passed within one hour of extubating and removing pressors. . 3.) r-sided paralysis - no evidence of ich on ct, but mri showed multiple areas of acute infarct, thought to be d/t endocarditis in the setting of a dilated /t severe as. neuro was involved and made recommendations to start asa, maintain pressures 140-160, and obtain carotid u/s. while these maneuvers were attempted, it was difficult to maintain sbp in setting of sepsis. ultimately it was decided that the multiple new acute infarcts would be detremental to the patient's quality of life, and that she would not wish to live in such a compromised state, and pressors were withdrawn. . 4.)arf - initially was prerenal, but evolution led to muddy brown casts on sediment, suggestive of atn, likely d/t hypotension in setting of sepsis. uop markedly improved and cr normalized. . 5.)cad - ck and troponins elevated and peaked. elevated enzymes represented demand ischemia in setting of critical as, infection, and hypotension. dr. of cardiology saw the patient and determined that cardiac cath was not indicated at the time. . 6.) hyperglycemia - insulin gtt with good control . 7.) thrombocytopenia- stabalized at 65, normal. hit ab was negative. meds such as vancomycin and protonix were likely contributors. medications on admission: budesonide caco3 vit d3 mvi lipitor fomoterol asa dilt-sr prednisone lasix neurontin discharge medications: none discharge disposition: expired discharge diagnosis: sepsis discharge condition: deceased discharge instructions: none followup instructions: none procedure: continuous invasive mechanical ventilation for 96 consecutive hours or more insertion of endotracheal tube diagnoses: pneumonia, organism unspecified subendocardial infarction, initial episode of care coronary atherosclerosis of native coronary artery congestive heart failure, unspecified acute kidney failure, unspecified severe sepsis infection with microorganisms resistant to penicillins obstructive chronic bronchitis with (acute) exacerbation aortic valve disorders methicillin susceptible staphylococcus aureus septicemia acute respiratory failure cerebral embolism with cerebral infarction 

Answer: The patient is high likely exposed to