Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: aspirin / sulfa (sulfonamides) / codeine attending: chief complaint: transfer from mwh for cardiac catheterization for cp with trops elevation to 0.79, likely nstemi (non st elevation myocardial infarction) major surgical or invasive procedure: cardiac catheterization history of present illness: 84yo male with cad - cabg x5 in (lim to lad, svg to da, svg to , svg to pda, svg to lt ventr branches), mi in , s/p aaa repair, s/p fem- bypass, crf on hd is transferred from mwh for cath for ?dx of mi. patient initially presented to mwh ed on with c/o continuous 7 out 10 shoulder to shoulder chest pain with no radiation. he denied sob or diaphoresis. took nitro at home x2 with no relief. in ed, he had +trop 0.79, ekg 100% paced, received iv nitro and morphine, plavix and heparin. no aspirin given (as per gi) because of the h/o severe gi bleed on aspitrin. pt had 2 subsequent episodes of cp overnight relieved by morphine. , pt was transferred to for cath. past medical history: cad - mi , cabg x5 in at s/p aaa repair ppm bilateral fem- bypass crf-hd on t-th-sat (last dialysis , tolerated well) severe duodenal ulcer bleed - received 11 prbc chrone's dx diverticulosis social history: past tobacco family history: . physical exam: pe: pt in bed, looks comfortable, no acute distress t 98.7 bp 130/72, hr 60, rr 18, 96% r/a heent: symm neck, mouth clear, no ln, flat jbp chest: limited exam, clear, gaeb cvs: rrr, n s1s2, syst gr ii-iii/vi murm over precordium : soft, n bs, nt extrem: no edema, varicose veins pulses: normal carotid, radial, doplerable pedal neuro: alert, oriented x3, grossly n lt groin: no hematoma (4pm) pertinent results: 06:55pm ck-mb-30* mb indx-13.8* ctropnt-0.89* 03:00am ck-mb-129* mb indx-20.4* 06:40am ck-mb-155* mb indx-20.9* ctropnt-2.54* 06:55pm wbc-7.1 rbc-3.25* hgb-11.4* hct-33.7* mcv-104* mch-35.0* mchc-33.7 rdw-15.8* 06:55pm plt smr-normal plt count-178 06:55pm glucose-74 urea n-52* creat-6.4* sodium-135 potassium-5.3* chloride-92* total co2-21* anion gap-27* cardiac cath:1. coronary and grft angiography showed a previous right dominant system. the lmca was diffusely disesed with no focal or critical lesions. the lad tapered off in the mid segment until a large s2 where it is totally occluded. the d1 and d2 are small vessels and are diffusely diseased. the d3 which recived a svg is not seen in the lmca injection. the mid and the distal lad receives the lima. the cx vessel it self has no lesions. it gives a lengthy collateral. the om1 arises close to the lmca and is small. the om2 too arises close to the lmca and is large. this has a proximal lesion of 80%. the om3 recives the svg and is not seen on lmca injection. the om4/postero latateral branch arises distally and is a small vessel. the rca is occluded proximally. the distal rca including the pda and the plv are collateralised by the left system. the pda is poorly filled and has a mid 60% lesion. the lima , the lima-lad anastomosis and the distal lad are free of disease. the lima fills the lad retrogradely to supply the proximal lad and the d3. the d3 has an ostial 70% lesion with timi iii flow. the sv grafts to the rca and the plb are occluded completely and are seen as stumps in the aorta. the graft to the diagonal could not be located, but is likely to be occluded given the other angiogaphic findings. the svg to the om3 shows diffuse disease with a mid lengthy lesion of 99 % and the whole vessel showed timi ii flow. there were no collaterals for this om. 2. left ventriculography was not performed. 3. predilation using 1.5 x 15 maverick balloon, stenting using 3.0 x 28 and 3.0 x 33 otw cypher stents and thrombus extraction using export catheter with gradual deterioration of flow of the svg to the om3. the flow deteriorated from timi i to timi 0. final diagnosis: 1. three vessel native coronary artery disease with functioning lima to the lad. 2. acute occlusion of the svg to the om and chronically occluded sv grafts to the pda, plb and diagonal. 3. unable to restore flow in the svg to the om despite stenting, pharmacotherapy and thrombus aspiration. . echo: . the left atrium is mildly dilated. 2. the left ventricular cavity size is normal. overall left ventricular systolic function is mildly depressed. basal inferior hypokinesis is present. 3. the aortic valve leaflets are severely thickened/deformed. there is moderate aortic valve stenosis. 4. the mitral valve leaflets are mildly thickened. 5. there is mild pulmonary artery systolic hypertension. . ct scan: 1. no evidence of intrahepatic gas as suggested on prior ultrasound. repeat ultrasound is suggested given this change in appearance. 2. bibasilar dependent atelectatic changes/consolidation with associated effusions. 3. gas distended loops of bowel with air-fluid levels but without transition suggesting ileus. stool distended rectum. 4. small infrarenal abdominal aortic aneurysm. brief hospital course: he was admitted with unstable angina, had total occlusion of all svg grafts with a patent lima to lad, and received 2 cypher stents. his catheterization was compicted by failed thrombus extractuib abd a timi 0. after catherization he had persistent cp and evidence of a nstemi. initially he was not treated with asa because of a past gi bleed, but with persistent ischemia, it was added to the plavix. he required a significant of morphine to controll his pain. through discussions between the micu team and the family pain control was determined to be the only option for him. his code status was changed to dnr/dni/. due to ongoing ischemia, he had persistent hypotension that required multiple pressors. his hd was changed to cvvh because of his low blood presssure. he also had intermittent nsvt. he had been transferred from the team to the micu team due to hypotension at hd after his cardiac catheterization out of concern for possible sepsis. there was a concern that he had an acute abdomen but he appeared to be impacted with stool. he was disimpacted and received an aggresive bowel regimen. his distension and pain improved. he had low grade temperatures and was initially treated for pneumonia because he was hypoxic. no source of infection was identified. it is more likely that he was in cardiogenic shock with fluid overload. a repeat bedside echo did not reveal worsening ventricular function. he required blood transfusions for persistently dropping hct in the setting of very frequent blood draws. he also had a coagulopathy which did not appear to be from dic. he required vitamin k supplementation. he expired at 6:45am on after an episode of severe chest pain. medications on admission: plavix 300mg x2 mg starting lopressor 12.5mg foslo 667mg x4 tid quinine 324mg daily pentasa 250mg x4 qid mvi mirtazapine 15mg qhs colace 100mg protonix 40mg daily morphine prn nitro prn discharge medications: none discharge disposition: expired discharge diagnosis: sinus tachycardia nsvt cardiogenic shock coagulopathy obstipation nstemi esrd discharge condition: expired discharge instructions: . followup instructions: . procedure: venous catheterization, not elsewhere classified coronary arteriography using two catheters left heart cardiac catheterization other and unspecified coronary arteriography enteral infusion of concentrated nutritional substances hemodialysis venous catheterization for renal dialysis transfusion of packed cells insertion of drug-eluting coronary artery stent(s) infusion of vasopressor agent transposition of cranial and peripheral nerves insertion of two vascular stents excision of lingual thyroid percutaneous transluminal coronary angioplasty [ptca] procedure on single vessel diagnoses: pneumonia, organism unspecified anemia in chronic kidney disease end stage renal disease subendocardial infarction, initial episode of care coronary atherosclerosis of native coronary artery congestive heart failure, unspecified coronary atherosclerosis of autologous vein bypass graft aortic valve disorders hypertensive chronic kidney disease, unspecified, with chronic kidney disease stage v or end stage renal disease constipation, unspecified cardiogenic shock hypovolemia other and unspecified coagulation defects hypotension of hemodialysis 

Answer: The patient is high likely exposed to