Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: this is a 77-year-old female with unclear history of prior cad, with history of a mass encapsulating right middle lobe and right lower lobe bronchi with no liver metastasis, presumed to be a possible malignancy, unclear brain mets (though noncontrast head ct showed no hemorrhage or midline shift), who was initially transferred from an outside hospital for concerns of an aorta dissection. by mri at , she was found to have aneurysmal thoracic and abdominal aorta with penetrating ulcers, but no evidence of a clear dissection. evaluation on the outside had revealed elevated cks to around 2200 with troponin t at peak 24. the outside hospital ecg had shown atrial fibrillation, rapid ventricular response up to 150s with st depressions. at the , on , the patient was in sinus rhythm without clear ischemia. however, until the a.m. of transfer on , the patient had an episode of polymorphic ventricular tachycardia degenerating into ventricular fibrillation, ultimately requiring dccv, lidocaine, and amiodarone drip. the patient was intubated, placed on dopamine for pressure support and then staged with propofol and transferred to the ccu for further evaluation. past medical history: breast cancer, diagnosed eight years ago, status post mastectomy. triple aorta repair approximately 5 to 10 years ago. history of cad with mi in the 80s. hyperlipidemia. presumed copd. question of psychiatric disorder. medications: synthroid unclear dose, also alternative medications. at the time of transfer, the patient was on amiodarone drip, dopamine of 13, aspirin, and propofol. allergies: no known drug allergies. social history: lived with retarded daughter and son. family history: unknown. physical examination: vital signs: afebrile, temperature 97.9, blood pressure in the systolic 110s/70s, actually in the 70s, heart rates in the 120s, respiratory of 20, on ac 600, saturating 100 percent. general: the patient was intubated and sedated. cardiovascular: she was tachycardic, irregular with a holosystolic murmur at the apex. abdomen exam: benign. extremities: the patient has 2+ lower extremity edema. laboratory data: from show a cbc of 13.2, hematocrit 34.9, and platelets of 36,000. chemistries, sodium 144, potassium 3.6, chloride 109, bicarbonate 29.5, bun 31, creatinine 0.8, glucose 129, lactate of 13.7, down to 6.3, coagulations 13.4, 28, 1.2; alt 118, ast 319, alkaline phosphatase 131, total bilirubin 0.7, troponin is 8 down from 24. cpk has been 2240, 1649, and 566 last. echocardiogram shows an ejection fraction of 25% with global hypokinesis, 1 plus mr, mild aortic stenosis, negative for an effusion. hospital course: review of systems: cad; the patient was admitted from an outside hospital with elevated ck and troponin in the setting of rapid atrial fibrillation. she also has an unknown history of cad. given her cks and troponin, the patient was treated with aspirin, statin, and plavix. she was not heparinized in the setting of a known malignancy with possible invasions to the brain. she was not placed on beta blockers given her hemodynamic instability. her enzymes were trended down throughout her hospital course. hypotension; the etiology of the patient's hypotension remains somewhat unclear during the hospital course. initially, she was transferred from the floor on dopamine following a polymorphic ventricular tachycardia, degenerative ventricular fibrillation arrest. the patient initially was treated for a possible cardiogenic insult. she was continued on dopamine. however, the patient also has fevers and there was a concern that the patient having possible pneumonia. as such, it was unclear if the patient's hypotension was secondary to cardiogenic versus septic shock. subsequently, during her hospital course, the patient had swan placed, which showed a ra pressure of 10, rv pressure of 30/7, pa pressure of 35/25, and a wedge of 15 with a cardiac index of 2.9, and svr of 10.20. ultimately, secondary to episodes of rapid atrial fibrillation and also episodes of nonsustained ventricular tachycardia, the patient was transitioned from dopamine to levofed. she did continue to have episodes of hypotension that appeared to be fluid responsive. as such, it was felt that the sepsis may then play a large role on the patient's hypotension. ultimately she remained pressor dependent, until her death on , at which point pressors had been discontinued for cmo status. chf; the patient had an echocardiogram from admission showing an ejection fraction of only 25%, in the setting of presumed cardiac ischemia. as mentioned above, she was initially thought to have a component of cardiogenic shock. however, her pa catheter numbers did not indicate cardiogenic shock. she actually did receive a questionable amount of fluid later in her hospital course for episodes of hypotension. it was felt later on the patient was grossly fluid overloaded, but still is exhibiting septic physiology. ultimately, she was gently diuresed to help improve oxygenation with bilateral pleural effusions. this therapy continued until decision is to make the patient cmo. rhythm; the patient transferred to the ccu after following an episode of polymorphic ventricular tachycardia, degenerative ventricular fibrillation arrest requiring dc cardioversion, and in addition, iv amiodarone, lidocaine, epinephrine and bicarbonate. throughout the patient's ccu course, she remained extremely dependent upon both iv amiodarone and lidocaine. initially, the patient was transitioned to p.o. amiodarone and was attempted to have weaned off of lidocaine. however, the patient developed several episodes of rapid atrial fibrillation and also was felt to have some episodes of sustained ventricular tachycardia. ultimately, lidocaine was continued until the patient's family had desired to change the patient's status to cmo, at which point, lidocaine was discontinued. pulmonary: initially, the patient was transferred to the ccu intubated following her ventricular tachycardia/ventricular fibrillation arrest. she remained intubated in her entire course until her death on the 29th. the patient ended up spiking fevers throughout her hospital course. her sputum was positive for methicillin-sensitive staphylococcus. she was initially treated empirically with levaquin and clindamycin for possible postobstructive pneumonia given her history of presumed lung cancer. later on, vancomycin was added when staph was found in her sputum. meanwhile, the patient actually oxygenated reasonably well during much of her hospital course. later on, however, the patient developed increased oxygen requirements, as she became progressively more over fluid overloaded, secondary to the amounts of fluids she is requiring for hemodynamics. id; as i mentioned above, the patient spiked fevers throughout her ccu course. initially, she was treated with levaquin and clindamycin for presumed postobstructive pneumonia. vancomycin was added later on when sputum was found to be positive for staph. in addition, there was concern about possible line sepsis, so vancomycin was continued for possible line-type sepsis. she continued on antibiotics until the day of her death at which point she was changed to be cmo. of note, blood cultures remained negative throughout her hospital course. oncology; the patient with history of breast cancer, status post mastectomy. upon transfer to the hospital with having findings of her mass increasing in the right middle lobe, right lower bronchi. the patient had an abdominal ct, which also showed lesions in liver consistent with metastatic disease. the etiology of the patient's masses presumed to be malignant of unclear etiology. the patient did have a follow up head ct, which was negative for any intracranial malignancy, but did show diffuse mets throughout her skull. no further intervention was made at this point. however, given the patient's presumed metastatic disease, this was a key point in discussions with the patient's family given the patient's poor clinical progress in ccu. hematology; the patient's hematocrit was stable during the hospital course. her platelets remained under 100 and was felt to be stable possibly secondary to underlying malignancy. renal; the patient's renal function remained stable during her hospital course. as mentioned above, she ultimately found to be grossly fluid overloaded secondary to massive amounts of fluid required to maintain her blood pressure. disposition: ccu team had underwent multiple discussions with the patient 's family during her hospital course in the ccu. ultimately, given the patient's presumed metastatic malignancy, her ongoing arrhythmias requiring a persistent lidocaine drips, her recent mi, and prolonged hypotension with essential hypertension, and possible anoxic brain injury, it was decided to change the patient's code status from full code to eventually dnr/dni and to ultimately cmo. at 07:10 p.m., on , the patient became asystolic and hypertensive. she was found to be dead at 07:10 p.m. family was at the bedside. no request for autopsy. discharge diagnoses: presumed metastatic malignancy. status post polymorphic ventricular tachycardia/ventricular fibrillation arrest. coronary artery disease, status post myocardial infarction. fevers secondary to presumed pneumonia and questionable line sepsis. discharge condition: dead. , md dictated by: medquist36 d: 15:24:14 t: 10:20:43 job#: cc: procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more insertion of endotracheal tube pulmonary artery wedge monitoring diagnoses: pneumonia, organism unspecified malignant neoplasm of liver, secondary acute myocardial infarction of other anterior wall, initial episode of care chronic airway obstruction, not elsewhere classified atrial fibrillation paroxysmal ventricular tachycardia secondary malignant neoplasm of brain and spinal cord ventricular fibrillation malignant neoplasm of other parts of bronchus or lung 

Answer: The patient is high likely exposed to