Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no known allergies / adverse drug reactions attending: chief complaint: right sided weakness major surgical or invasive procedure: transesophageal echocardiogram history of present illness: the pt is a 81 year-old with pmh htn, hl, and possible tia who presents as a direct transfer from for evaluation of recurrent transient right hemibody plegia and slurred speech. the majority of the history is obtained from medical records as the patient is unable to relay a coherent history. mrs. initially sought medical attention earlier this month for right hemibody plegia and slurred speech. at that time she saw a neurologist, dr. , and reportedly had a mri brain, cta head, tte, and eeg, all wnl. she was discharged from the hospital with a diagnosis of tia and her previous daily aspirin was switched to plavix. on the morning of the patient again had right hemibody plegia. the daughter reported that it seemed to start with the right leg before also involving the right arm. the patient had slurred speech as well at this time. the right sided weakness last approximately 30 minutes prior to resolving. the patient currently does not recall these events and confabulates when asked. at lgh, they recorded initial bp to be 218/152. they repeated a nchct and mri and transferred her here for further eval. on neuro ros, the pt denies headache, loss of vision, blurred vision, diplopia, dysarthria, dysphagia, lightheadedness, vertigo, tinnitus or hearing difficulty. denies difficulties producing or comprehending speech. denies focal weakness, numbness, parasthesiae. no bowel or bladder incontinence or retention. denies difficulty with gait. on general review of systems, the pt denies recent fever or chills. no night sweats or recent weight loss or gain. denies cough, shortness of breath. denies chest pain or tightness, palpitations. denies nausea, vomiting, diarrhea, constipation or abdominal pain. no recent change in bowel or bladder habits. no dysuria. denies arthralgias or myalgias. denies rash. past medical history: htn hl depression ?tia - right sided plegia that resolved cognitve decline - reported by patient's family social history: no tobacco or drugs. lives adjacent to daughter. reports that she does all her own adls (not confirmed with family). occasional alcohol family history: not contributory physical exam: physical examination on admission vitals: t: p: 67 r: 15 bp: 148/77 sao2: 98%ra general: awake, cooperative, nad. heent: nc/at, no scleral icterus noted, mmm, no lesions noted in oropharynx neck: supple, no carotid bruits appreciated. no nuchal rigidity pulmonary: lungs cta bilaterally without r/r/w cardiac: rrr, nl. s1s2, no m/r/g noted abdomen: soft, nt/nd, normoactive bowel sounds, no masses or organomegaly noted. extremities: no c/c/e bilaterally, 2+ radial, dp pulses bilaterally. skin: no rashes or lesions noted. neurologic: -mental status: alert, oriented x 1. confabulates when relating recent history. inattentive, able to name forwards but not backwards. language is fluent with intact repetition and comprehension. normal prosody. +paraphasic errors when (read 1st 2 phrases on stroke card correctly before making errors on remaining phrases). pt. was able to name both high frequency objects on card but had difficulties with cactus and hammock (describes their function/descriptors). speech was not dysarthric. able to follow both midline and appendicular commands. prominent ideomotor apraxia, r hand > l. no evidence of neglect. -cranial nerves: i: olfaction not tested. ii: perrl 3 to 2mm and brisk. vff to confrontation. funduscopic exam revealed no papilledema, exudates, or hemorrhages. iii, iv, vi: eomi without nystagmus. normal saccades. v: facial sensation intact to light touch. vii: no facial droop, facial musculature symmetric. viii: hearing intact to finger-rub bilaterally. ix, x: palate elevates symmetrically. : 5/5 strength in trapezii and scm bilaterally. xii: tongue deviates to the right. -motor: normal bulk, tone throughout. no pronator drift bilaterally. no adventitious movements. delt bic tri wre ffl fe io ip quad ham ta edb l 5 5 5 5 5 5 5 5 5 5 5 5 5 5 r 5 5 5 5 5 5 5 5 5 5 5 5 5 5 -sensory: difficult to assess due to inattention. no deficits to light touch, pinprick, cold sensation, vibratory sense, proprioception throughout. no extinction to dss. -dtrs: tri pat ach l 2 2 2 2 1 r 2 2 2 2 1 plantar response was flexor bilaterally. -coordination: dysmetric on right fnf and toe-to-finger. left has no dysmetria. rams arrthythmic bilaterally, worse on right -gait: deferred pertinent results: admission labs: 10:40pm blood wbc-7.3 rbc-4.38 hgb-12.9 hct-41.7 mcv-95 mch-29.4 mchc-30.9* rdw-12.3 plt ct-199 10:40pm blood pt-10.7 ptt-29.6 inr(pt)-1.0 10:40pm blood glucose-106* urean-18 creat-0.9 na-142 k-3.3 cl-104 hco3-26 angap-15 10:40pm blood ck-mb-5 ctropnt-<0.01 05:05pm blood ctropnt-0.02* 10:40pm blood calcium-9.1 phos-3.9 mg-1.7 cholest-198 10:40pm blood triglyc-90 hdl-53 chol/hd-3.7 ldlcalc-127 10:40pm blood %hba1c-5.7 eag-117 10:40pm blood tsh-1.1 discharge labs: 05:05am blood wbc-9.7 rbc-4.40 hgb-13.0 hct-42.2 mcv-96 mch-29.6 mchc-30.8* rdw-12.6 plt ct-183 05:05am blood pt-10.5 ptt-29.6 inr(pt)-1.0 05:05am blood glucose-111* urean-22* creat-0.8 na-141 k-3.8 cl-108 hco3-25 angap-12 reports: ekg: sinus rhythm. left ventricular hypertrophy. q-t interval prolongation. non-specific st-t wave abnormalities. no previous tracing available for comparison. intervals axes rate pr qrs qt/qtc p qrs t 67 118 90 460/472 47 36 29 ct/cta head/neck: subacute infarction involving the left caudate body/internal capsule, best seen on the mri from with possible infarction in the right pons and questionable mesencephalon. repeat mri is suggested if clinically warranted. atherosclerotic disease with focal narrowing of the left v4 segment as well as the left p1 segment. nchct: new hypodensity in the left corona radiata may represent an evolving infarct. previously identified pontine lesion is not visualized on this study. no other areas of acute ischemia. no evidence of intracranial hemorrhage or mass effect. brief hospital course: ms. was admitted to the neurology wards of the for the management of her stuttering right sided weakness symptoms. she was initially admitted to the neuroicu as an icu-icu transfer, though she did not have any specific icu needs. she was quickly transferred out of the icu. her initial neurological evaluation showed the presence of signs of prior noted "cognitive decline", with features of inattention, poor orientation and naming. there was only mild ataxia on the right hemibody. she was placed on an antiplatelet regimen (plavix) and admitted to the floor. initial laboratory studies were unremarkable and her cardiac enzymes were negative. on our own ct imaging, we identified a subacute infarction of the left caudate/internal capsule, as well as a possible hypodensity in the left pons. she was risk stratified. her a1c returned normal, but ldl was elevated at 127. she was switched to simvastatin 40mg daily; it's not clear whether she had been really taking the atorvastatin prior to her admission. on her third hospital day, she was noted to have worsening weakness on the right. a stat nchct showed the presence of a completed infarction in the left internal capsule, which was expected given the typical evolution of lacunar infarctions. she was started on aggrenox therapy, as it may have some useful vasodilator properties. given the two hypodensities noted on ct scan, we were concerned of a possible embolic source. she was ordered for a transesophageal echocardiogram which was performed on , which identified simple atheroma in the aortic arch (and complex atheroma in the descending aortic arch). given this finding, we decided to once again switch back to a typical antiplatelet. she was discharged on plavix monotherapy. she was evaluated by our physical therapists who felt that she would best benefit from a course of acute rehabilitation. the family was agreeable to this. at the time of discharge, she had a dense right hemiplegia including face, arm, and leg without sensory involvement. while she was poorly oriented and inattentive, there were no obvious language deficiencies. medications on admission: atorvastatin 40' plavix 75' hctz 25' metoprolol 50'' sertaline 50' discharge medications: 1. sertraline 50 mg tablet sig: one (1) tablet po daily (daily). 2. simvastatin 40 mg tablet sig: one (1) tablet po daily (daily). 3. metoprolol tartrate 25 mg tablet sig: one (1) tablet po bid (2 times a day). 4. plavix 75 mg tablet sig: one (1) tablet po once a day. discharge disposition: extended care facility: - discharge diagnosis: ischemic stroke hypertension hyperlipidemia depression dementia discharge condition: mental status: confused - sometimes. level of consciousness: alert and interactive. activity status: ambulatory - requires assistance or aid (walker or cane). discharge instructions: dear ms. , it was a pleasure taking care of you during this hospitalization. you were admitted to the neurology wards of the for the evaluation of your new right sided weakness. through a series of interviews, physical examinations, laboratory tests and neuroimaging studies, we were able to show that your symptoms were likely related to an ischemic stroke that you sustained the left side of your brain. as you know, you have several risk factors for stroke, including high blood pressure and elevated cholesterol. - we made several adjustments to your medications. please be sure to take your medications as prescribed below. - our case managers were able to organize a brief stay at an acute rehabilitation facility for you. this will help significantly in improving your strength and balance. - it is important that you follow up with your primary care physician (dr. and dr. from the division of stroke neurology. - please be sure to contact us should you have any further questions or concerns pertaining to your neurological symptoms. - please be sure to come to the nearest ed should you experience any of the below listed unexplained signs or symptoms. followup instructions: please follow up with your primary care physician: . # 15 , ( @ 1:30pm please follow up the division of stroke neurology dr. building, , , ma: ( procedure: diagnostic ultrasound of heart diagnoses: unspecified essential hypertension atherosclerosis of aorta depressive disorder, not elsewhere classified other persistent mental disorders due to conditions classified elsewhere other and unspecified hyperlipidemia cerebral artery occlusion, unspecified with cerebral infarction hemiplegia, unspecified, affecting unspecified side lack of coordination 

Answer: The patient is high likely exposed to