Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: seizure major surgical or invasive procedure: na history of present illness: 54 m with history of etoh abuse, transfer from osh p seizure with concern initially for sah, now disproven. briefly, patient with long h/o etoh abuse, drinks 6-12 beers per day, abruptly stopped drinking 2 days ago. was at home today and fell and hit his head while on the toilet. unknown if loc. found on ground and did not recall what happened. placed in c-collar and had ct ordered. on return from ct at osh, patient had gtc seizure, given 2 mg iv ativan and intubated for airway protection (required repeated attempts). also received labetalol for htn (up to 266/190 at osh). told ct showed sah and thus transferred to ed for neurosurgical management. however, on repeat ct here and review of osh ct, no sah seen. he was therefore admitted to micu for ongoing management of alcohol withdrawal seizures. vitals in ed 186/116, hr 82. currently he is intubated and sedated on propofol gtt. some history obtained from patient's old roommate . past medical history: alcohol abuse seizure x 1 4 months ago, c/b ich stroke x3 (one occurring in the last 4 months) ?stomach and pancreatic cancer per patient's old roommate other pmh unknown social history: etoh as above. per patient's friend, no other drug abuse. family history: unknown physical exam: vitals: t 99.8, p 79, bp 150/106, r 14, 100% vent settings: ac 0.40, tv 600, r 14, peep 5 general: intubated sedated. slightly disheveled, appears older than stated age. heent: pupils reactive to light, slight anisocoria (l>r), no icterus, few facial lacerations. neck: hard collar in place, jvd to 2 cm asa, no bruits appreciated. chest: cta bilat, ventilated heart: rrr s1 s2 no murmurs abdomen: soft, nt, nd, no masses, + mild hepatomegaly with easily palpable liver edge extrem: no edema, warm, 2+ pulses neuro: sedated, not response to voice, no current withdrawal to pain. pertinent results: 02:47am blood wbc-6.3 rbc-4.09* hgb-13.4* hct-37.8* mcv-92 mch-32.7* mchc-35.4* rdw-13.4 plt ct-163 07:30pm blood wbc-5.9 rbc-4.40* hgb-14.3 hct-41.8 mcv-95 mch-32.4* mchc-34.1 rdw-12.7 plt ct-155 07:30pm blood neuts-87.8* lymphs-6.9* monos-4.7 eos-0.4 baso-0.3 07:30pm blood pt-13.2 ptt-24.8 inr(pt)-1.1 02:47am blood glucose-124* urean-11 creat-1.0 na-138 k-3.6 cl-101 hco3-23 angap-18 07:30pm blood glucose-215* urean-9 creat-0.9 na-135 k-3.7 cl-99 hco3-20* angap-20 02:47am blood alt-55* ast-76* ld(ldh)-256* alkphos-72 amylase-48 totbili-0.8 07:30pm blood alt-60* ast-85* alkphos-74 totbili-0.9 02:47am blood lipase-22 02:47am blood lipase-22 07:30pm blood lipase-26 02:47am blood albumin-3.9 calcium-9.0 phos-3.3 mg-2.7* 07:30pm blood albumin-3.9 calcium-8.9 phos-3.0 mg-1.6 07:30pm blood asa-neg ethanol-neg acetmnp-neg bnzodzp-neg barbitr-neg tricycl-neg 04:15am blood type-art po2-151* pco2-39 ph-7.46* caltco2-29 base xs-4 <br>imaging: cxr: 1. high positioned endotracheal tube, roughly 8 cm above the carina. tube should be advanced for more optimal positioning. 2. possible right basilar atelectasis. repeat pa and lateral radiographs recommended with better inspiration when the patient is clinically able. <br> ct/cta head:noncontrast head: no definite evidence of sah. tiny punctate focus of hyperdensity in right frontal lobe most likely artifactual. left basal ganglia chronic lacunar infarcts. moderate bilateral mucosal sinus thickening, and small air-fluid levels in bilat. maxillary sinuses. cta: no occlusion, or aneurysm. atherosclerois, with roughly 50% narrowing in bilateral carotid siphons. brief hospital course: hospital course by problem: <br>seizure: given patient's known history and presentation the patient was thought to have likely etoh withdrawal seizure. also considered structural intracranial abnormalities given history of past seizure (though this may have been ?etoh related) and multiple cvas. he had no further seizure activity since getting iv ativan at osh. intubated for airway protection. patient was initially sedated on propofol gtt. on propofol was d/ced and patient was given valium 10-20mg every 15 minutes prn as this was most likely withdrawal seizure. patient was extubated on . he continued to have waxing and mental status, and a neuro consult was obtained. they were unable to examine the patient, and thought his mental status was likely due to the benzodiazepenes he had received. on , he was awake and answering questions, and his c-spine was clinically cleared. he continued to have variable alertness, and was again seen by neuro. he had a 24 hour eeg, which was finished on , and showed mild encephalopathy that could be toxic/metabolic in origin, but no seizure activity. . <br>hypertension: pt is suspected to have baseline hypertension, as he has multiple lacunar infarcts on ct. blood pressures were initally consistently near or above 200 systolic, and he was initially controlled on labetalol. however, he required multiple re-dosings, and was started on a labetalol drip on . an ng tube was eventually placed, and the patient was begun on oral hypertensives. he was intially started on an ace, which was subsequently increased. he was then begun on low-dose metoprolol. when this combination failed to keep his blood pressure down, the metoprolol was increased, and hydralazine and amlodipine, which resulted in good control of blood pressure. . <br>arf: upon transfer from the icu the patient was noted to have acute renal failure, which fena and feurea suggesting prerenal. urine eosinophils were absent. the patient responded to small repeated ns boluses and the creatinine returned to baseline. . <br>dysarthria: pt's speech has been difficult to understand, though friends noted that his speech was garbled at baseline. pt was noted to be a difficult intubation at the osh prior to transfer, requiring multiple attemtps. he also was noted to have a diastasis between the body and the right cornu of the hyoid bone on neck ct, which ent reported was likley an incidental finding. ent also saw resolving bilateral hematoma of the true vocal cords (). . <br>ct evidence of internal carotid occlusion: the patient denied any symptoms of stroke. he however has a previous history of multiple strokes. we performed carotid us, which was negative for occlusion. vascular surgery was consulted without any additional recomendations. he was started on asa, lipitor and lisinopril for new stroke prevention. . <br>etoh abuse: patient had no etoh intake x 2 days per history. before that he had been drinking 6-12 beers a day, making this likely an alcohol withdrawal presentation. he was given iv folate/thiamine/mvi and treated for withdrawal seizure as per above. he has not required any benzodiazepines for several days as of . . <br>metabolic acidosis: patient initially had anion gap of 16, ketones on urinalysis, mildly elevated lactate level. he got 2 l ivfs in ed and d5 overnight. the anion gap was thought to be most likely alcoholic/malnourishment ketoacidosis as well as possible mild lactic acidosis seizure. he was treated with fluids as above and also put on insulin sliding scale for hyperglycemia. by , the anion gap had decreased to 11. . <br>?history of stomach and/or pancreatic cancer: per report of patient's friend patient had either/both of these malignancies. recent records were obtained from two outside hospitals, and no evidence of this history was found in these records. medications on admission: unknown discharge disposition: home discharge diagnosis: fall acute mental status change alcohol withrdrawl with seizure discharge condition: stable discharge instructions: you were admited to in after falling and were transfered to to be evaluated for possible subdural hematoma (bleeding in your head) and seizure activity. we did not find any evidence for subdural hematoma in your head or any evidence of new infarcts. although you may have had a seizure at hospital when you were here we did not observe any clinical behavior that was consisten with seizure. also, when we studied the electrical ectivity in your brain, we did not find any evidence of seizure activity. you must stop drinking alcohol. your primary care doctor can help you find an alcohol rehabilitation center if you would be willing to receive this help. please return to the ed or call your regular physician if you have any of the following. - headache, weakness on your extremities, confusion, nausea vommiting, chest pain, shortness of breath, cough, fever, chills, or any other symptoms that are unusual fo you. followup instructions: f/u ent in weeks for repeat fiber optic exam please make sure that you follow up with a regular physician to follow up. if you don't already have a regular physician, call at () to have a physician assigned to you. procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours spinal tap incision of lung alcohol detoxification diagnoses: acidosis toxic encephalopathy unspecified essential hypertension acute kidney failure, unspecified unspecified protein-calorie malnutrition other convulsions abnormality of gait occlusion and stenosis of carotid artery without mention of cerebral infarction pneumonitis due to inhalation of food or vomitus late effects of cerebrovascular disease, other speech and language deficits hypoxemia other and unspecified alcohol dependence, unspecified alcohol withdrawal other diseases of vocal cords 

Answer: The patient is high likely exposed to