Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: sulfa (sulfonamides) / erythromycin / augmentin / codeine / phenytoin / ultram / morphine attending: chief complaint: near drowning major surgical or invasive procedure: intubation history of present illness: 58 y/o female with h/o dm type ii, asthma, copd, and arthritis admitted from osh after witnessed syncope and near drowning. she was fishing today with her friend on a canoe and was attempting to get out of the vessel. she was having difficulty, so she rolled into the water. she continued ot have difficulty so she thought deeper water would help her get to her feet, but her friend (on shore) witnessed that she suddenly was leanign back facing up in her life vest. she then turned over and was face down in the water. her partner called for help and the patient was pulled from the water and cpr was initiated by a civilian.. ems came and was found to be in pea arrest and was intubated in the field. she was brought to hospital and a perfusing rhythym was restored. she was transfered to for further care. . per reports, it was approximately 5 minutes from witnessed syncope to time of cpr initiation. ems arrived approximately 15 minutes therafter. in our ed initial vent settings were a/c 400/15 100%fio2, 5 peep. abg on floor 7.40/40/272/16. came to unit. past medical history: diabetes type ii - oral meds copd - takes "inhaler" asthma arthritis - darvocet h/o prior pneumonia requiriing hospitalization . social history: lives in with friend x 23 years. works @ shaws. family history: noncontributory physical exam: on admission: t: 92.5 bp: 154/88 hr: 63 sating 100% vent: 450/20 50%fio2 pip:29 peep:4 overbreathing @ 30/min gen: sedated heent: ncat cv: rrr s1, s2, no m/g/r resp: cta anteriorly abd: soft nt/nd, no massses ext: no edema neuro: minimally responsive pupils, no corneal reflex, no response to painful stimuli, no response to voice pertinent results: notable labs: . 06:40pm wbc-31.7* rbc-5.43* hgb-16.4* hct-50.2* mcv-93 mch-30.2 mchc-32.6 rdw-13.3 06:40pm neuts-75* bands-4 lymphs-10* monos-8 eos-1 basos-0 atyps-0 metas-1* myelos-1* 06:40pm plt count-374 06:40pm glucose-239* urea n-15 creat-1.0 sodium-140 potassium-4.3 chloride-104 total co2-14* anion gap-26* 06:40pm ck-mb-17* mb indx-2.2 ctropnt-0.34* 06:40pm alt(sgpt)-151* ast(sgot)-157* alk phos-123* amylase-180* tot bili-0.3 06:40pm ck-mb-17* mb indx-2.2 ctropnt-0.34* 06:40pm ck(cpk)-768* . notable studies: . # cxr 7:06 pm impression: no acute cardiopulmonary process. low lung volumes. . # ct head w/o contrast 6:49 pm impression: 1. no evidence of intracranial hemorrhage or mass effect. please note an mri is more sensitive for evaluation of acute ischemia. 2. diffuse sinus disease with air-fluid levels within the right maxillary and right sphenoid sinus which may relate to water inhalation or acute sinusitis. please correlate clinically. . # ct c-spine w/o contrast 6:49 pm impression: 1. limited examination due to technical artifacts as described above. no definite acute fracture or malalignment identified. 2. questionable hypoattenuating left thyroid lesion likely represents a benign nodule. this can be further evaluated with dedicated ultrasound on a nonemergent basis as clinically indicated. echo due to suboptimal technical quality, a focal wall motion abnormality cannot be fully excluded. overall left ventricular systolic function is normal (lvef>55%). there is no aortic valve stenosis. there is no pericardial effusion. # ct head findings: the patient remains intubated. there is newly apparent absence of distinction between the and white matter throughout both cerebral hemispheres, consistent with widespread anoxic brain injury. there is also relative hypodensity and effacement of sulci throughout both cerebral hemispheres, suggesting swelling. in addition, there is new hypodensity of the lentiform nuclei bilaterally, consistent with recent infarcts. the ethmoid and sphenoid sinuses are largely opacified and air-fluid levels and mucosal thickening are present among the maxillary sinuses. the mastoid air cells are clear. impression: 1. findings consistent with global ischemic brain injury. 2. considerable opacification among the paranasal sinuses, which can be seen in intubation. however, if the patient develops signs or symptoms of infection, the possibility of sinusitis could be considered. brief hospital course: a/p 58 y/o female s/p pea and near drowning admitted to the micu intubated and sedated. . # pea: estimate approximately 45min passed before perfusing rhythym restored. percipitant at this time not known. per story, appear syncopal event occured prior to her drowning episode, so can postuale that it was vaso-vagal, hypoglycmeia, or priamry arrhythmic event leading to drowining and hypoxia, leading to pea. no known underlying coronary disease. -ck/trop: anticipate elevation was cpr, trended downwards - echo was normal -serial neurologic exam showed little change -evidence for end organ perfusion problems from elevated lactate. . # metabolic acidosis: lactic acid from end organ ischemia. -iv fluids with bicarbonatex 2 l (vent can blow off co2) -mechanical ventilation -acidosis and lactate resolved over course . # near drowning: was intubated in the field. vented for airway protection and to assist in blowign off co2. -as sedation was weened pt did not increase responsiveness. - family made pt , and et tube was removed, and pt died approx 1 hour later. . # neurologic deficits: weened proprofol without improvement of exam. neuro consulted and eeg showed that pt did have some seizure activity, which resolved with propofol. -head ct consistent with diffuse anoxic brain damage, and incompatable with consciousness. - prognosis was presented to family, and after much discussion decided to make pt in accordance to the patients wishes. at that time, the et tube was removed and pt expired at 4:23 am # communication: , home ; ( daughter-in-law), cell . medications on admission: allergies: sulpha, codeine, cepheclor, augmentin, ultram, phenytoin, morphine, discharge medications: disceased discharge disposition: expired discharge diagnosis: respiratory arrest secondary to pea arrest discharge condition: desceased discharge instructions: none followup instructions: non procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours diagnoses: diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled acute respiratory failure anoxic brain damage chronic obstructive asthma, unspecified syncope and collapse accidental drowning and submersion while engaged in other sport or recreational activity without diving equipment drowning and nonfatal submersion 

Answer: The patient is high likely exposed to