Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: sulfa (sulfonamides) / compazine / keflex / ativan attending: chief complaint: work-up of possible seizure disorder major surgical or invasive procedure: none history of present illness: 25 yo left-handed woman with seizures of unclear etiology since in the setting of a history of drug and alcohol abuse and a prior suicide attempt, and transferred here as a transfer from hospital for evaluation and management of increasing seizure frequency. the history is obtained from the patient and a review of records. according to the patient, she had her first seizure at the end of , in the context of drug abuse ("acid," cocaine, and marijuana). however, she does not know any further details regarding this event. records suggest that she may have been seen in the ed in for seizures dating back to . she did not experience the next episode until , when she began having monthly episodes after undergoing drug rehabilitation shortly after a close friend had died. prior to these events, the patient describes an aura of nausea and uneasiness. while she does not recall the episodes, others have told her that they involve shaking, eyes rolling back, foaming at the mouth, and occasionally associated with urinary incontinence. the events last a few minutes, and she awakens confused and tired for some time, able to speak but not move. she had initially attributed these seizures to drug use and possible subsequent withdrawal, but continued to have the single events every few months. she saw several physicians during (dr. a neurologist in and dr. , a family physician in ). she reports that she walked out of dr. office without medications or testing early in . however, she reportedly had a normal mri and eeg there in . dr. prescribed depakote at some point, which caused her hair to fall out. depakote was also too expensive and she stopped the medication after 30 days, a period in which she was seizure-free. she also saw dr. in , who performed a 24 hour ambulatory eeg; records report "some abnormality" although the patient reports a normal study. she was not offered anti-epileptic therapy at this time. the patient refused 24 video-eeg monitoring, as she did not want to spend time away from her new puppy. after she lost full-time work and thus her health insurance, she saw another physician, . , who gave her valium as needed for sleep later in the year. by early , the seizures were occurring every few weeks. she had been experiencing increasing seizure frequency since or , for the first time experiencing "back to back" episodes. the events were similar as before; however, between these consecutive episodes she can hear but not speak. she apparently reported to the ed in with her boyfriend on , after a night of heavy alcohol consumption while playing pool in . the patient only recalls seizing on the way home and apparently had 6 before arrival to the hospital. she could not offer further details. she was apparently sedated, paralyzed, and intubated for frequent, recurrent seizures. of particular note, blood alcohol level there was elevated at 138 but a tox screen was negative. the patient was loaded with fosphenytoin, and sent to the icu, where she was continued on 200 mg . she was extubated in the icu at ~10 am on , about 10 hours after she had been intubated. the patient reports that she seized 8 times while at . she received valium 5 mg prn for some of seizure episodes as well as agitation last evening. seizures were non-stereotyped. one, by neurologist's report, consisted of clonic movement of arms and legs in phase, eyes closed (not in forced nature), left gaze deviation with passive eye opening. she was non-verbal and blinking afterwards, then suddenly awoke and conversed on appropriate treatment. another, seen by a nurse, began with choking, then diffuse tremulousness, "like a panic attack." her eyes were open, gaze down and to the left. this episode lasted 13 minutes. on transport into the unit here, she was having an episode described by nursing as unusual oral movements with the production of saliva, with associated writhing and rolling on her side. the paramedics gave her 5 mg valium as she came into out trauma sicu. vitals were completely stable and the episode resolved after minutes. review of systems: reports recurrent head trauma in past, falling from a hose "more times than i can count." eating and sleeping well. recently stopped valium after her physician stopped prescribing it. denies assocoaition of events with menstruation or stress. no history meningitis. over past few months, left frontal throbbing headache, associated with photo- and phonophobia, relieved with rest in a dark room and excedrin migraine. this headache awakens her from sleep, but no postural component or worsening with valsalva. two weeks ago, had "sore throat" with low grade fewer to f. received "zpac." patient reports frequent urination, 10-15 times per day. bowel habits regular. a thorough review of neurological systems, including weakness, numbness, tingling, dysarthria, dysphagia is negative. she states she occasionally stutters. denies suicidal or homicidal ideation. past medical history: seizures as above drug and alcohol abuse (see below) has prior suicide attempt in 10th grade, overdose on ibuprofen. pneumomediastinum depression hpv, s/p scrapings/biopsy recurrent bladder infections social history: history of drug abuse including ecstasy, cocaine, acid, and marijuana since , but has reportedly abstained for 1.5 years. no ivdu. alcohol consumption has continued, generally on weekends "will have a few drinks." has been in rehab in both 10th grade and again in . denies history of tobacco use. begins working full ime at animal hospital on tuesday. family history: family history: years ago, her mother had several seizures as a young adult, requiring hospitalization. little else known. physical exam: afebrile general: well nourished and in no acute distress neck: supple lungs: clear to auscultation cv: regular rate and rhythm abdomen: non-tender, non-distended, bowel sounds present ext: warm, no edema neurologic examination: mental status: awake and alert, cooperative with exam, restricted affect oriented to person, place, time and president attention: can say days of week backward and forward language: fluent, no dysarthria, no paraphasic errors, naming intact fund of knowledge normal, can perform crossed body commands registration: items, recall items at 3 minutes no apraxia, no neglect calculations intact cranial nerves: optic disc margins sharp; visual fields are full to confrontation. pupils equally round and reactive to light, 5 to 3 mm bilaterally. extraocular movements intact, no nystagmus. facial sensation and facial movement normal bilaterally. hearing intact to finger rub bilaterally. tongue midline, no fasciculations. trapezius normal bilaterally. motor: normal bulk and tone throughout no adventitious movements d t b wf we fif ip gl q h af ae tf te right 5 * * 5 5 5 5 5 5 5 5 5 5 5 5 left 5 5 5 5 5 5 5 5 5 5 5 5 5 5 5 * not tested due to uncomfortable line in place sensation was intact to light touch, pin prick, temperature (cold), vibration, and proprioception reflexes: b t br pa pl right 3 2 3 2 1 left 3 2 3 2 1 toes were downgoing on left, mute on right coordination is normal on finger-nose-finger (left only tested give discomfort with line in right arm), ffms normal, heel to shin also normal pertinent results: outside hospital studies: wbc on admission was 11.7 (abs lymps high, 4.7) then 9.8, h/h 12.6 and 36. bal 138, tox screen was negative. review of chemistries, lfts, serum hcg, ua on admission was unremarkable. dilantin levels 17.8, then 21 on repeat. prolactin level 11. normal head ct, except partial opacification of ethmoid air cells. ekg, cxr essentially unremarkable. 08:15am blood wbc-9.1 rbc-4.02* hgb-12.7 hct-37.1 mcv-92 mch-31.6 mchc-34.2 rdw-13.1 plt ct-456* 08:15am blood plt ct-456* 08:15am blood glucose-80 urean-10 creat-0.7 na-138 k-4.5 cl-102 hco3-29 angap-12 08:15am blood alt-16 ast-16 amylase-54 03:04am blood alt-20 ast-25 ld(ldh)-154 alkphos-44 totbili-0.4 08:15am blood albumin-3.5 calcium-9.5 phos-4.0 mg-2.2 03:38am blood phenyto-15.1 eeg this telemetry captured a single pushbutton activation. this showed an episode of writhing in bed, without clinical features suggestive of a seizure. the eeg showed normal wakefulness at the time. routine sampling and automated detection showed no epileptiform features or electrographic seizures eeg report to follow. please refer to brief hospital course for preliminary results (of importance). brief hospital course: the patient was admitted, as outlined in detail in the history of present illness, to qualify the nature of the recurrent episodes concerning for seizures. on , a single pushbutton event was analyzed, but it was not a classical spell. on however, the patient had more than 7 episodes that, also upon review of the video, were slight different each time, but had a common pattern as follows: the patient would become restless first, with non-specific semi-purposeful movements, writhing in bed, rocking left to right, whimpering somewhat at times. her vital signs would remain stable. these behaviors could last up to several minutes, and did not have any stereotyped or rhytmic component to it. she would then arch her back lightly, and would have non-synchronous random movements of arms and legs, with intermittent bicycling movements. then, the behaviors would become more violent, with rhymthmic shakinng of all 4 limbs, however still with limited synchrony, and pelvic thrusting. frothing at the mouth was seen, and the patient would desaturate on occasion, but no tongue bite or urinary incontince. duration of this motor phase was again in the realm of 2 minutes, and she could desturate (but with poor quality signal on pulse-oximetry). afterwards, she would have a normal facial color, breath rapidly but regularly and not hypotonic. she would not make eye contact until her name was called repeatedly. on eeg, there were no epileptiform abnormalities, prior, during or after these events. just prior, during the writhing phase, a normal posterior dominant rhythm was identified. during the more pronounced motoric phase, there was prominent muscle artifact as well as large deflections atypical morphology were seen, representing movement artifact. at times, a normal background eeg could be seen through this artifact. post-ictally, there was an immediate return of normal background, with a well-formed reactive alpha-rhythm and a nice anterior-posterior frequency amplitude gradient. the family visited on saturday the 27th and the epilepsy fellow dr spoke to them and the patient at lenght, explaining the nature of these non-epileptic events. the patient was very understanding, and indicated that dr had also suggested such a thing. she expressed relief in the fact that we were finally able to clarify the nature and that they are not life-threatening. she felt like this good news would probably already decrease the severity of the spells and she may be able to fight them off better. the father was content and relieved, but did say he had some worries about being discharged without follow-up. we ensured him we could function as a backup and are available by phone. we prescribed a small dose of klonopin to bridge until follow-up. we will contact dr at the b&w hospital, and will provide the eeg data as well as our official read. there's a followup scheduled already on at 3pm. medications on admission: only on ortho-evra as outpatient discharge medications: 1. klonopin 0.5 mg tablet, rapid dissolve sig: tablet, rapid dissolves po prn maximum 3 times per day as needed for anxiety leading to non-epileptic seizures. disp:*30 tablet, rapid dissolve(s)* refills:*1* discharge disposition: home discharge diagnosis: non-epileptic seizures. discharge condition: stable, afebrile, normal neurological exam. discharge instructions: you have been admitted for seizures that have been shown to be of non-epileptic origin, as we have discussed. we have started you on a medication called klonopin (clonazepam) to decrease anxiety related to these episodes. if there is insufficient control of the episodes with klonopin, the knowledge that these are not life-threatening, the education we provied and the follow-up plans that are coming in place, please contact us at (ask for epilepsy person on call). the same applies with questions or concerns. as we have discussed, the approach to managing these events will be multimodal. you should probably be referred to a behavioral neurologist and/or a psychiatrist, to help you address these episodes and develop coping strategies for current stressors. followup instructions: please follow-up with your epileptologist, dr. , at the at 3pm on . call with questions. procedure: video and radio-telemetered electroencephalographic monitoring diagnoses: other, mixed, or unspecified drug abuse, unspecified other convulsions depressive disorder, not elsewhere classified 

Answer: The patient is high likely exposed to