Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: acute onset chest tightening, dizziness, diaphoresis, and shortness of breath major surgical or invasive procedure: coronary catherterization history of present illness: 81 y old male with hx of dyslipidemia, htn, cad s/p nqwmi in ' s/p bare metal stenting of proximal and mid lad as well as om1 presented to ed by ambulance with acute onset chest tightening, dizziness, diaphoresis, and shortness of breath, was found to have ste >5mm in ii, iii, avf, v4-v6 along with 3-4mm st depression in i and avl. hr was in the 40s. code stemi was called, pt was given asa 325mg, plavix 600mg (although takes plavix at home), heparin 5000 units x 1, integrillin 17mg ivx1 and then transferred to cath lab. in cath lab pt had successfull bare metal stenting to proximal rca and was also found to have new diffuse aneurysmla dilatation of his vessels. pt became bardycardic intermittently in the cath lab and required atropine x2. temporary pacer placed prior to the transfer to the floor. when pt seen on on the floor he denied any chets pain, sob, diaphoresis, nausea. states onset of chest pain was in the setting of the culmination of a 16 day editing project he had as a composer. pt quickly realized the urgency of the situation as the sx's very similar to his prior mi and therefore asked his friend to 911. of note, pt states he was on asa 325mg up until about 2 years ago when he was noted to have "blood from below". per pt he was told to stop taking the asa and never had a gi w/u for the bleeding as he states "it was assumed that the bleeding was due to apirin". his last colonoscopy was 7-8 years ago and was normal. he has never had an egd. at home pt exercises by "speed-walking" on a treadmill for 30 minutes almost every day and never experiences any anginal sx's or sob. he has never smoked, drinks occasionally and tries to adhere to a fairly low fat diet. . on review of symptoms, he denies any prior history of stroke, tia, deep venous thrombosis, pulmonary embolism, bleeding at the time of surgery, myalgias, joint pains, cough, hemoptysis, recent black stools or red stools. he denies recent fevers, chills or rigors. he denies exertional buttock or calf pain. all of the other review of systems were negative. . cardiac review of systems when seen on the floor is notable for absence of chest pain, dyspnea on exertion, paroxysmal nocturnal dyspnea, orthopnea, ankle edema, palpitations, syncope or presyncope. past medical history: # cad s/p nqwmi in ' s/p bare metal stenting of proximal and mid lad as well as om1 # htn # dyslipidemia # hx of ulcers on feet bilaterally # r eye blind after traumatic injury at age 11 social history: social history is significant for the absence of tobacco use. occasional alcohol. family history: there is no family history of premature coronary artery disease or sudden death. physical exam: vs: t 96.4, bp 98/69, hr 62, rr 19, sao2 100% on 2l gen: male appearing younger than stated age in nad. oriented x3. mood, affect appropriate. pleasant. heent: sclera anicteric. perrl, eomi. conjunctiva were pink, no pallor or cyanosis of the oral mucosa. neck: no jvd cv: rr, normal s1, s2. no s4, no s3. chest: resp were unlabored, no accessory muscle use. no crackles, wheeze, rhonchi. abd: obese, soft, ntnd, no hsm or tenderness. no abdominial bruits. ext: both feet with toes in dorsiflexion appearing like contractures. also with superficila fungal infections of toes and nails. both legs with brown discoloration of feet up to mid-calf. skin: no stasis dermatitis, ulcers, scars, or xanthomas. pulses: femoral 2+ without bruit bil; 1+ dp bil. pertinent results: 08:45pm glucose-126* urea n-20 creat-1.2 sodium-140 potassium-4.3 chloride-100 total co2-28 anion gap-16 08:45pm estgfr-using this 08:45pm ck(cpk)-115 08:45pm ctropnt-<0.01 08:45pm ck-mb-5 08:45pm wbc-7.9 rbc-5.18 hgb-16.4 hct-49.4 mcv-95 mch-31.7 mchc-33.2 rdw-14.5 08:45pm neuts-39.3* lymphs-51.5* monos-6.8 eos-2.0 basos-0.4 08:45pm plt count-189 08:45pm pt-13.7* ptt-27.3 inr(pt)-1.2* . echo (): the left atrium is mildly dilated. the estimated right atrial pressure is 0-5mmhg. there is mild symmetric left ventricular hypertrophy with normal cavity size. there is mild regional left ventricular systolic dysfunction with focal hypokinesis of the inferolateral wall. the remaining segments contract normally (lvef = 45-50 %). the aortic root is moderately dilated at the sinus level. the ascending aorta is moderately dilated. the aortic arch is mildly dilated. the aortic valve leaflets are mildly thickened. mild (1+) aortic regurgitation is seen. the mitral valve leaflets are structurally normal. there is no mitral valve prolapse. mild (1+) mitral regurgitation is seen. the pulmonary artery systolic pressure could not be determined. there is no pericardial effusion. impression: suboptimal image quality. mild symmetric left ventricular hypertrophy with regional systolic dysfunction c/w cad. mild aortic regurgitation. mild mitral regurgitation. dilated thoracic aorta. compared with the report of the prior study (images unavailable for review) of , the regional left ventricular wall motion abnormality is new and the ascending aorta and arch are now identified as dilated. clinical implications: based on aha endocarditis prophylaxis recommendations, the echo findings indicate prophylaxis is not recommended. clinical decisions regarding the need for prophylaxis should be based on clinical and echocardiographic data. brief hospital course: pt with presentation to as mentioned above, taken to cath; in cath lab pt had successfull bare metal stenting to proximal rca and was also found to have new diffuse aneurysmal dilatation of his vessels. pt became bardycardic intermittently in the cath lab and required atropine x2. temporary pacer placed prior to the transfer to the ccu. in the ccu where pt was placed on tele. on the second day the pace was briefly needed but then heart rate remained in to 60-s and 70s, therefore the pacer was removed after 48hrs, lopressor was again started after 72 hrs without a drop in the heart rate (bradycardia improved as expected since rca reperfused) enzymes were negative in the emergency room, but second set came back very elevated at ck 2713, trop t 11.23, ck-mb 363 and mb index of 13.4. ezymes thereafter trended down. medically, plavix 75 mg was continued, atorvastatin 80mg was started (for pleotropic effects, i.e. anti-inflammatory ect, and for mortality benefits), asa 325 mg was restarted in hosp on admission. the reason for pt not taking it the past 2 years prior to presentation was cleared up with pcp who stated this was b/c pt had nose bleeds during his performances, and therefore elected not to take asa anymore. pcp agrees pt needs to be on lifelong asa and plavix and will follow up closely in the case of another bleed. acei was held on presentation due to concern of droing bp with bradycardia but restarted on hod#2. an echo was done to r/o wall motion abnormalities determine ef demonstrating mild symmetric left ventricular hypertrophy with regional systolic dysfunction, and lvef = 45-50 % c/w cad. pt was evaluated by pt who found patient fit to go home since pt ambulated for 15 minutes at a fast rate without any cp or sob. upon discharge pt was asymptomatic, and ambulating, voiding, taking good po on own, and saturating well off oxygen. medications on admission: altace (ramipril) 5mg qday toprol xl 25 mg qday isosorbide mononitrate 30 mg qday lipitor 10mg qday plavix 75 mg qd discharge medications: 1. aspirin 325 mg tablet, delayed release (e.c.) sig: one (1) tablet, delayed release (e.c.) po daily (daily). disp:*30 tablet, delayed release (e.c.)(s)* refills:*2* 2. clopidogrel 75 mg tablet sig: one (1) tablet po daily (daily). 3. ramipril 5 mg capsule sig: one (1) capsule po daily (daily). 4. atorvastatin 80 mg tablet sig: one (1) tablet po daily (daily). disp:*30 tablet(s)* refills:*2* 5. toprol xl 25 mg tablet sustained release 24 hr sig: one (1) tablet sustained release 24 hr po once a day. discharge disposition: home discharge diagnosis: myocardial infarction (inferior stemi) discharge condition: stable discharge instructions: you were admitted to with an st elevation myocardial infarction. please take your previous medications as prescribed. the following changes has been made to your medications: - please start taking aspirin 325mg daily for secondary cardiovascular prevention (to prevent another heart attack) and atorvastatin 80mg daily for your heart and for your cholesterol. - please stop taking isosorbide mononitrate if you develop chest pain, jaw pain, or chest pressure with pain radiating into arm, or if you for any reason become concerned about your medical condition please call 911 or present to nearest ed. followup instructions: please call your pcp for an appointment within 1-2 weeks. procedure: insertion of non-drug-eluting coronary artery stent(s) coronary arteriography using two catheters angiocardiography of left heart structures left heart cardiac catheterization insertion of temporary transvenous pacemaker system cranial or peripheral nerve graft insertion of one vascular stent excision of lingual thyroid percutaneous transluminal coronary angioplasty [ptca] procedure on single vessel diagnoses: subendocardial infarction, initial episode of care coronary atherosclerosis of native coronary artery mitral valve disorders unspecified essential hypertension cardiac complications, not elsewhere classified other specified procedures as the cause of abnormal reaction of patient, or of later complication, without mention of misadventure at time of procedure other specified cardiac dysrhythmias heart disease, unspecified 

Answer: The patient is high likely exposed to