Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: the patient is a 63-year-old woman with stage iii, grade 3 papillary serous ovarian cancer (status post paclitaxel and carboplatin, taxotere, doxil, monthly taxol, gemcitabine, and topotecan weekly x2 cycles), who has been in the bahamas in the past two months receiving immuno-augmentive therapy. per report, she did relatively well until , when she developed nausea, vomiting, diarrhea, and altered mental status, and subsequently fell. she was ultimately flown back to on the day of admission, and was brought to ed, where she was confused and minimally responsive. she was hypotensive and started on dopamine. she was hypoxic and intubated for airway protection as well as hypoxia; she also reportedly had periods of apnea. a right femoral line was placed. levofloxacin and metronidazole were given, ct scans were obtained, and the patient was transferred to the fenard intensive care unit for further management. past medical history: 1. ovarian cancer as above. 2. hypertension. 3. diabetes mellitus type 2. 4. dyslipidemia. 5. viral encephalitis. 6. ovarian cystectomy in . 7. d&c in . 8. appendectomy. 9. neck lipoma excision. allergies: the patient is allergic to codeine and morphine. medications on admission: unknown. physical examination: on initial physical examination, the patient's temperature is 95 degrees, heart rate 115, blood pressure 71/50, respiratory rate 15, and oxygen saturation of 99% on mechanical ventilator. she was intubated. she had conjunctival edema, reactive pupils bilaterally. her heart rate was tachycardic, there are normal s1, s2 heart sounds, and there is a 2/6 systolic ejection murmur heard throughout the precordium. she had bronchial breath sounds at the left base, otherwise her lungs were clear to auscultation bilaterally. her abdomen was firm, distended, there were hypoactive bowel sounds, and a fluid wave was palpable. there was 2+ bilateral lower extremity pitting edema. the extremities were cool and peripheral pulses were barely palpable. she was moving her extremities purposely, and not responding to verbal or tactile stimuli. her skin was cool, modeled, and clammy. laboratories: initial laboratory evaluation demonstrated a white blood cell count of 16.3 (69% neutrophils, 21% bands, 8% lymphocytes, and 2% monocytes), hematocrit 29.8, and platelets of 224,000. pt was 17.2, ptt 33.7, and inr of 2.0. chemistries demonstrated a sodium of 128, potassium 3.4, chloride 86, bicarbonate 15, bun 46, creatinine 1.1, glucose 179, calcium 8.5, magnesium 2.3, and phosphorus 4.8. the albumin was 2.6. lfts were normal except for an ast of 63 and an alkaline phosphatase of 143. urinalysis was negative for uti. abg on the ventilator was 7.46/28/213. diagnostic paracentesis was consistent with spontaneous bacterial peritonitis; peritoneal fluid gram stain demonstrated 2+ polys, but no organisms. initial ct scan of the chest, abdomen, and pelvis demonstrated a large left sided pleural effusion, moderate right sided pleural effusion, bibasilar compressive atelectasis, interval enlargement of left sided axillary nodes, diffuse distention of multiple bowel loops, with the majority of the small bowel dilated and fluid filled, right sided colonic distention with air and fluid, ascites, and a soft tissue mass in the mid-transverse colon leading to relative decrease in the caliber of the remaining large bowel with air in the left sided colon and rectum. overall, there was significant worsening of omental disease with scalloping of the liver margin and falciform ligament as well as new amorphous mass growing into the porta hepatis and compressing the ivc and portal vein. the pancreas was also found to be invaded by a large adjacent hypodense mass. head ct demonstrated no intracranial hemorrhage or mass effect. hospital course: as noted above, the patient was critically ill at the time of admission. she was found to be in florid septic shock. she was initially aggressively resuscitated with multiple pressors to support her blood pressure as well as broad-spectrum antibiotics for her peritonitis. nearly immediately following her transfer to the intensive care unit, it became clear that the patient's chances of surviving her acute illness were essentially zero. after waiting for the patient's close family members to arrive at the bedside, supportive medical care was withdrawn on the day following admission. within 15 minutes of the withdraw of pressors, the patient became hypotensive and subsequently asystolic. she was pronounced dead at 8:20 p.m. on . the case was declared as nonreportable by the medical examiner, the admitting officer was notified, and the death certificate was completed. death diagnoses: 1. septic shock. 2. spontaneous bacterial peritonitis. 3. hypoxic ventilatory failure. 4. coagulopathy in the setting of sepsis and dic. 5. anemia of chronic inflammation. 6. blood loss anemia. 7. hyponatremia. the remainder of the patient's past medical history as noted above. , m.d. dictated by: medquist36 procedure: venous catheterization, not elsewhere classified insertion of endotracheal tube percutaneous abdominal drainage insertion of other (naso-)gastric tube transfusion of packed cells diagnoses: malignant neoplasm of liver, secondary unspecified septicemia acute respiratory failure defibrination syndrome other shock without mention of trauma septic shock secondary malignant neoplasm of retroperitoneum and peritoneum 

Answer: The patient is high likely exposed to