Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: dilantin / sulfa (sulfonamides) / azithromycin / trileptal attending: chief complaint: ha, seizures, left sided weakness major surgical or invasive procedure: picc line placement history of present illness: 30 yr old woman, 3 months post-partum, transferred from an osh to the neuro icu for evaluation of l-sided weakness and consideration of stroke vs. seizure vs infection. pt reports left-sided weakness began just yesterday and that was preceded by 3-4 days of nausea and malaise.+ photophobia. no temp was taken. no vomiting. she did have a headache which is similar to her migraine for the last few days. typically, her migraines headaches are throbbing, unilateral, and relieved with ibuprofen and sleep. no neck stiffness. she does not have any sick contacts at home. the left-sided weakness was predominantly in her arm and was sudden onset; she had difficulty putting on her pants or underwear yesterday. she also complains of parasthesias in her left arm and leg beginning yesterday. at the osh, her husband tells me he witnessed generalized whole body convulsions this morning for 15-20 seconds that the pt. does not remember. she did not lose bowel/bladder incontinence and pt tells me there was no tongue biting. he tells me movements were rhythmic and stereotyped. she was confused afterwards. she initially presented to hospital yesterday. lp there showed 365 wbc, 1 rbc, prot 105, glu 58. cbc and chem-8 were wnl. at the osh, records indicate that her l hand was twitching uncontrollably and she was conscious through event. ativan iv was given with questionable respone. acyclovir was then started for diagnosis of viral meningoencephalitis after mri, with osh prelim reports stating "right parietal infarct vs. inflammatory disease, inc. diffusion signal on l side, ? embolic dz." mra reportedly normal. she is transferred to for further workup and management. past medical history: migraine ha social history: patient is homemaker, recently given birth. no history of smoking or alcohol use. no history of drug abuse. family history: mother with stroke, lupus physical exam: vitals: t-100.5 bp-137/70 hr-112 rr-18 pt is pleasant young woman, lying in bed in no apparent distress. head, neck, cardiac, lung, abdominal, and extremity exam were within normal limits. neurologic exam: ms: oriented to person, place and time. alert to voice, but patient falls asleep if not spoken to. able to say months of year backwards in 15 seconds. fluent speech, repetition, naming intact. able to read and write. able to follow multi-step commands. memory registration, recall at 5 minutes. no apraxia, neglect, frontal signs. calculation intact. cn: visual fields intact to confrontation. pupils normal round 5mm->3mm with light. eomi without nystagmus. normal facial sensation. patient has l facial droop in umn pattern. hearing intact to finger rub. palate rises symmetrically. tongue midline. motor: normal tone and bulk. no tremors or fasciculations. pronator drift absent. strength full throughout. reflexes: 2+ and symmetric bilaterally. toes downgoing bilaterally. sensory: intact to pinprick, vibration, proprioception and temperature throughout. coordination: intact ftn b/l. intact . gait: normal. pertinent results: wbc-11.1* hgb-14.0 hct-37.4 plt ct-231 pt-13.0 ptt-34.7 inr(pt)-1.1 na-140 k-3.7 cl-108 hco3-24 bun-7 creat-0.7 gluc-117* ca-8.9 phos-2.8 mg-2.0 alt-13 ast-15 alkphos-64 totbili-0.4 osh: lp with 365 wbc, 1 rbc, prot 105, glu 58. mri prelim reports stating "right parietal infarct vs. inflammatory disease, increased diffusion signal on l side, ? embolic dz." mra reportedly normal. brief hospital course: impression: 30f, 3 months post-partum presents with headache/malaise x4 days, then left-sided paresis and seizures. mri with multiple t2 and diffusion abnormalities, lp with lymphocytosis. this is most likely secondary to viral menigoencephalitis. 1. neuro (meningoencephalitis, seizures, venous sinus thrombosis): given the concern for viral meningoencephalitis, pt was treated empirically with acyclovir pending hsv pcr results from csf. once results were negative, acyclovir was discontinued. because of the 99% lymphocyte predominance of the wbcs in the csf, no antibacterials were given. left-sided weakness rapidly improved over her first few days in the hospital, with only a mild left facial droop and mild left hand clumsiness remaining. at the osh, she initially had focal motor seizures (and one secondarily generalized) involving her left hand and dilantin was started there. while at , she developed paroxysmal paresthesias of her right face and hand, once accompanied by speech arrest as well. dilantin levels were low, and she was reloaded with dilantin. once therapeutic levels were reached, these paroxysmal episodes ceased. however, given her desire for furture pregnancies, it was decided to start lamictal so that dilantin could be discontinued in several months. when she began to have seizures again, since they were in a new location (right-sided as opposed to left), a repeat head mri was obtained which showed bilateral tubular areas of susceptibility effect and t1 hyperintensity with surrounding leptomeningeal enhancement. two of the regions in the right post-central gyrus and in the left frontal lobe are associated with underlying white matter edema suggesting vasogenic edema associated with elevated venous pressure/venous infarcts, although there is no restricted diffusion. compared with the mri from the osh, there was enlargement of the existing lesions, no new lesions and resolution of the dwi abnormalities. location of the lesions was entirely consistent with focal seizures causing her symptoms. however, the "tubular susceptibility lesions" in multiple areas of cortex were concerning for venous congestion or infarct. therefore, an mrv was obtained. cortical veins showed no evidence of thrombosis. however, the distal left transverse sinus, sigmoid sinus and proximal internal jugular vein was thrombosed. etiology was likely due to a combination of factors, including being 3 months post-partum, on birth control pills prior to admission and the viral meningoencephalitis. she was started on coumadin, and was maintained first on heparin and then on lovenox until inr was therapeutic. hypercoaguability workup was performed and all tests were negative. 2. fever: when repeat mri showed enlargement of lesions, it was unclear if this represented the natural progression of the disease vs actual disease worsening. therefore, we consulted infectious disease for their input. they recommended repeat lp, which showed 79 wbc (99% lymphocytes) 19 rbc. acyclovir was restarted until repeat hsv was negative. vzv and bartonella were also negative. additionally, because venous sinus thrombosis can be caused by septic thrombophlebitis, they recommended empiric unasyn. a few days later, she developed a worsening rash and started spiking fevers. concern was for a drug rash, and unasyn and acyclovir were discontinued. pt had no localizing signs or symptoms of an infection, multiple cultures of blood and urine were negative. rash improved, but fevers persisted, and id felt that these were most likely secondary to dilantin. rheumatological diseases were also ruled out with antibody screen. therefore, she was started on trileptal for her seizures and dilantin was slowly tapered. a few days later she continued to have fevers to 104 and rash, and both lamictal and dilantin were stopped. fever began to improve, and she was discharged home with a diagnosis of dilantin-induced drug fever. 3. transaminitis: liver enzymes were checked as part of workup for fever, and both ast and alt were mildly elevated. hepatitis serologies were negative, except for hep b surface antibody positive, consistent with hep b vaccination. it was thought that the elevations were most likely secondary to medication toxicity, a combination of round the clock tylenol (total 4 gm per day) for fever and dilantin. tylenol dosing was decreased, and dilantin was discontinued with improvement in lfts. 4. hyponatremia: after starting trileptal, her sodium level decreased to 130 where it stabilized. this is almost certainly a side effect of the trileptal, was aymptomatic and does not need treatment. 5. leukopenia: while in the hospital, her wbc count decreased to a low of 2.1. etiology was unclear, but most likely represented a medication side effect. once dilantin wsa discontinued, her wbc count began to recover. at the time of discharge, pt remained febrile to ~101. neurologically, she had a very slight left facial droop and left hand clumsiness, otherwise intact. medications on admission: meds: on transfer zofran dilantin ativan tylenol asa discharge medications: 1. ibuprofen 400 mg tablet sig: 1-2 tablets po q8h (every 8 hours) as needed for fever, pain. 2. acetaminophen-caff-butalbital mg tablet sig: tablets po q4-6h (every 4 to 6 hours) as needed for migraine: if taking this then do not take tylenol at the same time. 3. outpatient occupational therapy outpatient ot for residual left hand weakness and clumsiness from viral meningoencephalitis 4. azithromycin 250 mg capsule sig: one (1) capsule po q24h (every 24 hours) for 2 days. disp:*2 capsule(s)* refills:*0* 5. trileptal 600 mg tablet sig: one (1) tablet po twice a day. disp:*60 tablet(s)* refills:*2* 6. warfarin sodium 1 mg tablet sig: three (3) tablet po at bedtime. disp:*90 tablet(s)* refills:*0* 7. acetaminophen 500 mg capsule sig: capsules po every eight (8) hours as needed for fever or pain: if taking fioricet, then do not take this at the same time. discharge disposition: home discharge diagnosis: meningoencephalitis, complicated by seizure, neurological deficits, likely viral left sigmoid sinus and internal jugular vein thrombosis migraine mild transaminitis, likely secondary to medication toxicity fever, likely secondary to phenytoin discharge condition: good with some very mild residual left facial droop and slight left hand clumsiness. discharge instructions: please seek medical attention if you have any symptoms of severe headache, neck stiffness, inability to move your arm or leg, abdominal pain, rash, itching or any concerning symptoms. also, please call dr. or dr. if you have any further tingling or uncontrolled twitching in your left arm, especially if it lasts longer than 10 minutes. the current impression of the infecious disease doctors is that your fever is being caused by one of your medications and that when you go home, you may care for your baby as usual. if your baby develops fever, rash or any concerning symptoms please seek medical attention right away. followup instructions: please follow up with your pcp . on friday at 9:40 am. call to reschedule if needed, though you should see her next week if possible. your inr will be followed by the anti-coagulation group at dr. office. please go to her office this monday to have your blood drawn and inr checked. they will let you know how much coumadin to take. additionally, they will draw blood to check your liver function tests, your white blood cell count, and your sodium. dr. is covering dr. while she is out of town. dr. nurse will be in touch to arrange for your blood. follow-up with dr. in clinic: provider: , .d. where: neurology phone: date/time: 1:00 get a follow-up mri a few days before you see dr. . call to schedule. please follow up with the dr. in the infecious disease clinic at . you have an appointment sceduled for at 3 pm. provider: md where: lm disease phone: date/time: 3:00 md, procedure: venous catheterization, not elsewhere classified spinal tap incision of lung diagnoses: other convulsions migraine, unspecified, without mention of intractable migraine without mention of status migrainosus hemorrhage of rectum and anus nonspecific elevation of levels of transaminase or lactic acid dehydrogenase [ldh] phlebitis and thrombophlebitis of intracranial venous sinuses other symptoms involving nervous and musculoskeletal systems other enterovirus diseases of central nervous system other phlebitis and thrombosis complicating pregnancy and the puerperium, postpartum condition or complication 

Answer: The patient is high likely exposed to