Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: percocet / shellfish derived / levaquin attending: chief complaint: fevers and sob x 5 days major surgical or invasive procedure: : bronchoscopy with bronchoalveolar lavage history of present illness: the patient is a 63 yo m with nsclc and cll who originally p/w 5 days of fever and sob. the patient reports that until 5 days ago he was feeling well. he initially went to on for symptoms of shortness of breath and was given a course of levoquin. he reports that he developed a pruritic truncal rash while on the levaquin (thus it was discontinued on initial presentation to ). at home however, even while on the levaquin, he continued to be sob and was febrile to 100.5 x 3 days pta. 2 days pta, he coughed up 1/4 cup of blood. prior to that, cough was productive of moderate amounts of yellow sputum. hemoptysis and worsening "burning" rash is what prompted his presentation to ed. . on admission, cxr revealed b/l hazy opacities. he was initially started on cefepime/vanco/flagyl/bactrim for pulomonary infection unresponsive to levofloxacin. he was previously requiring o2 via nasal cannula to max of 4l, but since yesterday evening, has had increasing oxygen requirement overnight so that he was placed on a nrb. throughout the day, he has been maintaining o2 sats on nrb in the mid to upper 90s, however, with movement and with removal of mask he promptly desaturates to the 80s. he has had multiple coughing fits with posttussive emesis at which time he desaturates to the 80s. he continues to bring up small amounts of blood in his sputum. a ct scan was obtained today given his broad coverage, but persistent respiratory decompensation which showed b/l consolidations and ground glass opacities. id was consulted and recommended adding voriconazole to his above abx coverage and further recommended bronchoscopy. they also felt that bactrim was not indicated given low risk of pcp. the day, he became increasingly tachypneic to rr as high as 40s and although, recent abg revealed 7.44/37/141, he looked to be tiring with use of accessory respiratory muscles and was transferred to the icu for further respiratory monitoring and out of concern for possible need for intubation. . ros: denies chest pain, abdominal pain, blood in stool, urinary symptoms. +25lb weight loss x1 year. past medical history: # adenocarcinoma of the lung/ large cell lung cancer - diagnosed left , right , s/p wedge resection and rll lobectomy. previously treated with taxol/cisplatin, tarceva, and now navelbine x8 # cll # cad s/p 3v cabg # copd # s/p inguinal hernia repair social history: live swith wife. retired . smoking 30yrs x 1-1.5ppd; quit ; etoh: rare; illicits: none family history: mother died of breast cancer 66yrs, brother w/ h/o cad physical exam: vitals: t 96.2 hr 104 bp 110/59 rr 37 o2sat 90-98% nrb general: pale male, appears short of breath with nrb mask in place using accessory neck and abdominal muscles to breath, is however, able to complete full sentences, but appears tired. heent: perrl, op clear, mildy dry mm neck: supple cv: tachycardic, prominent heart sounds, no murmurs appreciated lungs: decreased breath sounds throughout right lung, specifically right base, left lung w/ decreased bs apically and with crackles from mid lung to base. abdomen: soft, nt/nd, +bs ext: 1+ b/l le edema, feet cool w/ palpable dp/pt pulses neuro: cn 2-12, strength, sensation grossly intact pertinent results: imaging: cxr ( ) - increased density in the r lung suspicous for pna. there are extensive post surgical changes in the chest. persistent focal areas of increased density n the lateral aspect of the lower right lung and in the left perihiklar region may represent residual or current tumor. these appear stable. micro: blood - pending sputum - gpc in pairs and clusters . bal : + malignant cells c/w nsclc adenocarcinoma; no viral agents, legionella, pcp, afb growth to date . chest ct: interval progression of dense left perihilar and right posterior lung consolidation with numerous new bilateral small areas of consolidation and diffuse ground-glass opacity. as was described in the past report, differential diagnosis for the chronic consolidation would include bronchoalveolar carcinoma or cryptogenic organizing pneumonia. the interval progression of consolidation in these two areas could at least in part represent progression of one of these processes with possible superimposed infectious pneumonia. primary considerations for the multifocal new areas of consolidation also includes acute infectious process. other considerations that would account for the diffuse ground-glass opacity would include hypersensitivity pneumonitis, drug reaction, or hydrostatic pulmonary edema. brief hospital course: the patient is a 63yo m with h/o nsclc, cll, and cad p/w fevers and shortness of breath x 5 days with worsening hypoxia despite broad coverage antibiotics. hospital course by problem is as follows: . # hypoxia: ct scan shows increased lung consolidation and diffuse ground glass opacities. in review of most recent chest ct in , appears to have worsening disease concerning for progression of lung cancer. ground glass opacities concerning for superimposed infection and even postobstructive pneumonia given progression of disease. although, by cell counts, not immunosuppressed, given cll he is functionally immunosuppressed. additionally, wbc count difficult to follow as indicator of infection due to cll. alimta is most recent therapeutic oncologic and does not appear to have increased risk of pneumonitis nor is there other clear medications cause of pneumonitis. despite abg without significant derangements, had increased wob w/ accessory muscle use and tachypnea on transfer concerning that the patient would tire. had episode of desat to 80s with increased wob secondary to likely mucus plugging as he improved s/p expectoration of small amount of blood-tinged sputum. trial of 20mg iv lasix on failed: pt dropped sbp to 80s from 100s with no improvement in o2 requirement. broad coverage antibiotics were continued with cefepime/vancomycin/ flagyl/ and voriconazole as per id recs with azithromycin for atypical coverage. bronchoalveolar lavage samples were sent with cultures pending, as above. due to poor respiratory status the patient was intubated on . . # nsclc: ct scan showed what appears to be progression of disease with consolidation secondary to pneumonia. unclear when patient last received alimta, but this appears to be his most recent treatment. the team had been in touch with dr. (outpt oncologist) regarding rec's, and it was felt that there were no further treatment options to treat his underlying disease. . # hemoptysis: most likely secondary to underlying infective process vs. progression of pulmonary oncologic process. hct were followed, which had been stable during admission. . # cll: again, status of disease was not entirely clear, but certainly lung ca is more active issue currently given advancement of disease. . # cad: s/p cabg in . on statin alone, not on asa (unclear reason, but potentially while undergoing tx) nor bb ( to hypotension in past). no active issues currently. the patient was maintained on a statin, with asa held given hemoptysis . # anemia: baseline appears to fluctuate some but hct appears mainly high 20s to mid 30s with nml mcv and elevated rdw. likely secondary to aocd given oncologic processes. hct was followed. stool was guaiac'ed regularly. . # rash: likely drug rash; ? to levoquin. asymptomatic, appears to be resolving. this was monitored, with benadryl as needed for pruritus . # hypothyroidism: recently diagnosed with hypothyroidism. the patient was maintained on thryroid hormone replacement . # fen: the patient was kept npo for intubation, and was maintained on d51/2ns at 50cc/hr maintenance fluid. . # ppx: sc heparin, ppi were used. . on am pt had an episode of bradycardic arrest with loss of pulse for 30-60seconds. pt received 1mg atropine given with hr 40 --> 130 and restoration of bp. on ventilator, fio2 changed to 1.0 and peep reduced to 10. ekg showed rbbb with sinus tachycardia in 140s. changes may be rate-related. etiology was unclear: perhaps vagal episode from ett tube, but this appears to be unlikely given proper placement on cxr. cxr not suggestive of ptx. ekg not consistent with inferior infarct. the family was notified and chose to pursue a dnr code status at that time. . at ~3:30pm the patient became bradycardic again. the family was called to the bedside. the patient continued to become bradycardic with decrease in respiratory rate until cardiopulmonary arrest. the family was at the bedside throughout. time of death = 3:52pm. medications on admission: lipitor 10mg daily foradil inhaler 12mcg aranesp 400mcg 1x/2 weeks b-12 1000mcg 1x/9weeks alimta 685mg 1x/3weels folic acid 400mg daily econpred plus 1% drops/left eye daily levoquin x 5 days discharge disposition: expired discharge diagnosis: nonsmall cell lung cancer post-obstructive pneumonia cll anemia cad hypothyroidism discharge condition: expired procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube closed [endoscopic] biopsy of bronchus diagnoses: unspecified acquired hypothyroidism chronic airway obstruction, not elsewhere classified aortocoronary bypass status acute respiratory failure malignant neoplasm of other parts of bronchus or lung dermatitis due to drugs and medicines taken internally other specified antibiotics causing adverse effects in therapeutic use chronic lymphoid leukemia, without mention of having achieved remission bronchopneumonia, organism unspecified anemia in neoplastic disease 

Answer: The patient is high likely exposed to