Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: intracerebral hemorrhage major surgical or invasive procedure: endotracheal intubation radial arterial line nasogastric tube placement history of present illness: patient is an 82 yo woman with no significant pmh other than osteoporosis who presents as transfer from intubated with a left bg bleed. she has been in good health and takes no medications. she lives with her son and today at around 4:30pm she shreiked out three times, and then a loud thud was heard. her son found her on the floor, able to speak. other details about her state unavilable at this time. ems was called and noted that she was speaking to them. at the osh she was reported as having right facial droop and flacid right hemiparesis and speaking. a ct scan showed ich of the left bg 18 x 21 mm with intraventricular spread into the left lateral ventricle. she was intubated for transfer. it was noted that with administration of propofol she became hypotensive to 62/26, but bp stabilized within 5 minutes and has been 110-160 since. ros: unable to offer. past medical history: osteoporosis no known history of hypertension or other cerebrovascular risk factors. social history: lives with son, and daughter is very close by. no tob/etoh. very independent in her adls. family history: father died at 54 of bleeding ulcers. mother had stomach ca. physical exam: t- 97 bp- 132/75 hr- 97 rr- 16 o2sat 99% gen: lying in bed, intubated, sedated with versed. heent: nc/at, intubated neck: supple, no carotid or vertebral bruit back: cv: rrr, nl s1 and s2, no murmurs/gallops/rubs lung: clear to auscultation bilaterally abd: +bs soft. ext: no edema neurologic examination: mental status: intubated and sedated. intermittently writhing and spontaneously moving lue and lle antigravity and rle non antigravity. no spont movement of rue. grimacing. not following commands midline or appendicular. cranial nerves: pupils equally round and trace reactive to light, 2.5 to 2 mm bilaterally. no blink to threat. no corneals. no dolls. grimace appears symetric but difficult with intubation. motor: flaccid rue. intermittently writhing and spontaneously moving lue and lle antigravity and rle non antigravity. no spont movement of rue. with noxious stim withdraws lue and lle briskly. withdraws rle well but less strongly. extensory postures rigt ue. sensation: as above reflexes: 1+ in ue and symetric. difficult to elicit in legs. toes up bilaterally pertinent results: 11:12pm type-art po2-66* pco2-36 ph-7.42 total co2-24 base xs-0 11:12pm k+-3.3* 11:12pm freeca-1.18 02:55pm type-art po2-119* pco2-42 ph-7.42 total co2-28 base xs-3 02:55pm glucose-158* lactate-0.9 na+-131* k+-3.3* cl--100 02:55pm hgb-12.8 calchct-38 02:55pm freeca-1.11* 04:31am type-art po2-96 pco2-40 ph-7.35 total co2-23 base xs--3 intubated-intubated 04:31am freeca-1.13 04:20am glucose-182* urea n-13 creat-0.6 sodium-133 potassium-3.3 chloride-102 total co2-22 anion gap-12 04:20am ck(cpk)-108 04:20am ck-mb-8 ctropnt-0.06* 04:20am calcium-8.0* phosphate-2.7 magnesium-1.6 04:20am phenytoin-31.5* 04:20am wbc-10.7 rbc-4.01* hgb-12.7 hct-38.3 mcv-95 mch-31.5 mchc-33.1 rdw-13.8 04:20am plt count-186 04:20am pt-12.4 ptt-28.7 inr(pt)-1.1 09:39pm glucose-147* lactate-1.8 09:39pm hgb-13.9 calchct-42 09:20pm glucose-149* urea n-20 creat-0.8 sodium-129* potassium-3.9 chloride-92* total co2-25 anion gap-16 09:20pm ck(cpk)-102 09:20pm ctropnt-0.12* 09:20pm ck-mb-9 09:20pm calcium-9.0 phosphate-3.2 magnesium-2.0 09:20pm asa-neg ethanol-neg acetmnphn-neg bnzodzpn-neg barbitrt-neg tricyclic-neg 09:20pm urine bnzodzpn-pos barbitrt-neg opiates-neg cocaine-neg amphetmn-neg mthdone-neg 09:20pm wbc-9.2 rbc-4.42 hgb-14.5 hct-41.6 mcv-94 mch-32.8* mchc-34.8 rdw-13.8 09:20pm neuts-88.6* bands-0 lymphs-9.5* monos-1.6* eos-0.1 basos-0.1 09:20pm plt count-175 09:20pm pt-12.4 ptt-27.2 inr(pt)-1.1 09:20pm urine blood-mod nitrite-neg protein-neg glucose-neg ketone-tr bilirubin-neg urobilngn-neg ph-8.0 leuk-neg 09:20pm urine color-straw appear-clear sp -1.006 09:20pm urine rbc-* wbc-0-2 bacteria-occ yeast-none epi-<1 12:02pm blood %hba1c-5.5 06:38am blood triglyc-64 hdl-48 chol/hd-2.5 ldlcalc-58 1:11 pm abscess source: r arm abscess. gram stain (final ): no polymorphonuclear leukocytes seen. 1+ (<1 per 1000x field): gram positive cocci. in pairs. fluid culture (final ): staph aureus coag +. moderate growth. staphylococcus species may develop resistance during prolonged therapy with quinolones. therefore, isolates that are initially susceptible may become resistant within three to four days after initiation of therapy. testing of repeat isolates may be warranted. sensitivities: mic expressed in mcg/ml _________________________________________________________ staph aureus coag + | clindamycin-----------<=0.25 s erythromycin----------<=0.25 s gentamicin------------ <=0.5 s levofloxacin---------- 0.25 s oxacillin------------- 0.5 s penicillin------------ =>0.5 r trimethoprim/sulfa---- <=0.5 s anaerobic culture (pending): ct head without contrast : an acute hemorrhage involving the left basal ganglion and thalamus measures approximately 18 x 21 mm with surrounding hypodense rim which likely represents edema. there is extensive intraventricular blood within the ventricle as well as layering blood within the occipital horns of the lateral ventricles bilaterally. there may be a small amount of blood within the third ventricle. there is no shift of normally midline structures, or evidence of acute major vascular territorial infarct. atherosclerotic calcification of the cavernous carotids is observed bilaterally. mucosal thickening of the ethmoid air cells and nasal mucosa is noted bilaterally. the mastoid air cells are well aerated. impression: left basal ganglion and thalamic intraparenchymal hemorrhage with ventricular extension as described. ct head without contrast : unchanged appearance of the previously seen left basal ganglia area of high attenuation extending into the lateral ventricle and the thalamus suggestive of acute intraparenchymal hemorrhage. no masses, mass effect, midline shift or new parenchymal hemorrhagic areas are seen. no areas of acute infarction. the visualized osseous and soft tissue structures are within normal limits. mild thickening of the mucosal lining of the paranasal sinuses is noted. impression: unchanged appearance of the left basal ganglia and thalamic parenchymal hemorrhage extending into the ventricles. no interval signs of progression or new hemorrhagic areas. no midline shift. non contrast head ct : significant image degradation due to patient motion limits evaluation, especially in the lower brain slices. unchanged appearance of the previously seen left thalamic hemorrhage extending into the ventricles. no evidence of ventricular dilation or hydrocephalus. unchanged appearance of the hypoattenuating rim surrounding the hemorrhage consistent with vasogenic edema. no masses, mass effect or midline shift is noted. the osseous and soft tissue structures appear unremarkable. mild mucosal thickening is noted in the bilateral sphenoid and ethmoid sinuses. air-fluid level is noted in the left maxillary sinus, indicating sinusitis. visualized portions of the orbits are unremarkable. impression: unchanged appearance of the left thalamic hemorrhage extending into the ventricles. no evidence of ventricular dilation or mass effect or midline shift. mri/mra head : mri head: there is an acute left thalamic hemorrhage identified extending to the left corona radiata and also extending to the left lateral ventricle. the hematoma in the left lateral ventricle demonstrated increased t1 signal. following gadolinium, no definite area of enhancement is identified within the left thalamic region or in the adjacent area. no abnormal flow voids are seen in this region. mild periventricular changes of small vessel disease are identified. no evidence of slow diffusion seen in the left thalamic region or in other parts of the brain. there are soft tissue changes in the visualized paranasal sinuses and left mastoid air cells. impression: left thalamic hemorrhage with intraventricular extension of the left lateral ventricle. no abnormal flow voids or enhancing lesion identified to indicate associated arteriovenous malformation or mass. however, if there is continued clinical suspicion for underlying lesion a followup mri can help. mra of the head: the head mra demonstrates normal flow signal within the arteries of anterior and posterior circulation. both posterior cerebral arteries are fetal in origin, a normal variation. there is no evidence of abnormal vascular structures in the region of left thalamic hemorrhage. impression: no significant abnormalities on mra of the head. cxr : the et tube tip is below the clavicular heads terminating about 1.8 cm above the carina. the ng tube tip is in the stomach. the heart size is enlarged to a moderate degree. the lung volumes are low. dextroscoliosis is noted most likely with element of kyphosis. a right chest deformity is demonstrated which may be related to old rib fractures with rounded opacity projecting over the mid right lung which may be related to pleural thickening or represent intraparenchymal lesion. in the absence of previous studies, the precise diagnosis is difficult. the left lung is grossly unremarkable. there is no evidence of congestive heart failure. small left pleural effusion cannot be excluded. cxr : distal end of the feeding tube overlies the region of the duodenojejunal junction. the opacity in the left paracardiac region likely represents a combination of elevated left hemidiaphragm andassociated atelectasis, but it is difficult to evaluate accurately in this single frontal view. a small ill- defined area of opacity at the right lung base is new since the prior film of and could be related to aspiration. no pneumothorax. impression: feeding tube tip remains in region of ligament of treitz. new area of opacity right lung base could be secondary to aspiration. persistent elevated left hemidiaphragm and associated left basilar opacity possibly related to atelectasis on this limited exam. rounded opacity projecting over the expected portion of the left ventricle may represent left ventricular enlargement. when the patient's condition improves, repeated pa and lateral radiographs are recommended for precise evaluation of cardiopulmonary findings. brief hospital course: ms. is an 82 year old woman with no significant past medical history other than osteoporosis who presented as a transfer from hospital intubated with a left thalamic hemorrhage with intraventricular spread. she was not on any medications such as aspirin or coumadin. she did not have a prior history of bleed. 1) left thalamic hemorrhage- after arriving intubated and sedated, her admission examination was notable for a partial right hemiparesis, arm greater than leg. repeat ct on admission revealed stable size of thalamic hemorrhage, but extension of blood from left lateral ventricle into the right lateral ventricle and small ammount into 3rd ventricle. neurosurgery evaluated the patient on admission as well as hd #2 and did not feel the pt would require an external ventricular drain. the hemorrhage was in a typical location for hypertensive bleed, but she had no clear history of this. she may have had occult hypertension nocturnally as she did have some episodes of hypertension while in hospital and mri/mra did not reveal a underlying lesion. she was admitted to the neurology icu and monitored closely for clinical change in her neurologic exam. her blood pressure was maintained for sbp 140-170 and map < 130. she was initially loaded on dilantin, but this was discontinued given lack of any cortical blood or other lesions. she was promptly extubated on hd #1. mri/mra head was obtained to search for underlying mass or vascular lesion to explain the patient's hemorrhage with apparent lack of risk factors. mri/mra head did not reveal mass or avm. the patient was able to follow simple commands, and remained with a dense r arm greater than leg hemiparesis. a dobhof tube was placed for ng feeds. she was stable for transfer to the neurology stepdown on . on her second day on the stepdown (), she was able to speak in a fluent, but dysarthric fashion. with the patient more alert and her speech returning, her swallowing was evaluated. however, the patient was aspirating and it was determined that she would need a peg tube to maintain long-term nutrition. the feeding tube was placed by interventional radiology on . 2) cardiac the patient was noted to have an elevated troponin enzyme of 0.12 on admission, which trended down to 0.06 on repeat testing. this was thought secondary to subendocardial demand related ischemia during a period of hypotension following administration of propofol at hospital prior to intubation. repeat ekg did not reveal ischemic changes. she was later rate controlled with diltiazem and metoprolol prn. 3) infectious disease- in the icu, the patient was noted to have air fluid level in her frontal sinuses by ct with clinical finding of erythema and ? tenderness over her left frontal sinus. she was completed a course of azithromycin for presumed sinusitis. her right antecubital fossa was noted to have marked warmth and erythema and she was started on vancomycin for cellulitis. the site underwent i and d. levaquin was later started in the place of vancomycin, then changed to clindamycin (to complete 5 day course of clinda). a repeat cxr was done on the stepdown unit after a coughing spell with concern for aspiration. although there was a question of a new opacity on the repeat cxr, no antibiotics were started in response, as there was no fever, wbc, or other indication of an aspiration pneumonia. it was suspected that there might be aspiration pneumonitis. on the day of discharge, the patient was in stable condition and ready for departure. medications on admission: none discharge medications: 1. famotidine 20 mg tablet sig: one (1) tablet po bid (2 times a day). 2. heparin (porcine) 5,000 unit/ml solution sig: one (1) injection tid (3 times a day). 3. diltiazem hcl 60 mg tablet sig: one (1) tablet po qid (4 times a day). 4. acetaminophen 325 mg tablet sig: one (1) tablet po q6h (every 6 hours) as needed for temperature > 100f or pain. 5. docusate sodium 50 mg/5 ml liquid sig: one (1) po bid (2 times a day). 6. senna 8.6 mg tablet sig: one (1) tablet po bid (2 times a day). 7. bisacodyl 5 mg tablet, delayed release (e.c.) sig: two (2) tablet, delayed release (e.c.) po daily (daily) as needed. 8. clindamycin hcl 150 mg capsule sig: two (2) capsule po q6h (every 6 hours) for 5 days: 5 more days of therapy after discharge from hospital - for treatment of thrombophlebitis. discharge disposition: extended care facility: medical center - discharge diagnosis: left thalamic intracerebral hemorrhage, likely hypertensive superficial thrombophlebitis s/p i&d sinusitis discharge condition: improved - verbal output increasing, able to answer questions and follow simple commands, fluent sentences and only minimally dysarthric speech; unable to move right arm, trace tripleflexion of right leg, right facial droop. discharge instructions: please return to er if you have new signs of cerebral hemorrhage or stroke - including new weakness or numbness, new trouble speaking or visual changes. please take all medications and keep all appointments. followup instructions: follow up with your primary care physician after discharge from rehab: , . follow up with neurologist/dr. ( medical center) after discharge from rehab - ( - call for appointment md procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours enteral infusion of concentrated nutritional substances percutaneous [endoscopic] gastrostomy [peg] other incision with drainage of skin and subcutaneous tissue diagnoses: unspecified essential hypertension hyposmolality and/or hyponatremia intracerebral hemorrhage methicillin susceptible staphylococcus aureus in conditions classified elsewhere and of unspecified site osteoporosis, unspecified cellulitis and abscess of upper arm and forearm other specified hemiplegia and hemiparesis affecting unspecified side phlebitis and thrombophlebitis of superficial veins of upper extremities other late effects of cerebrovascular disease, dysphagia thoracogenic scoliosis acute maxillary sinusitis late effects of cerebrovascular disease, speech and language deficit, unspecified 

Answer: The patient is high likely exposed to