Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: worst headache of life, after exercising major surgical or invasive procedure: and : diagnostic angiogram(negative for aneurysm) history of present illness: patient is a 63 year old female with no significant pmh who presented with the worst ha of her life after exercising. she states she was at a fitness class and she was doing jumping jacks when she had the sudden onset of a severe occipital ha at around 6:45pm. the pain increased in severity to the point where she described it as the worst headache in her life. at this point her friends insisted that she call ems, and at this point she was taken to an osh. at that hospital a head ct was done and the patient was found to have a sah and she was transferred to for further evaluation. at the time of initial eval, she described the quality as a bifrontal severe, throbbing ha. she has significant nausea and has vomited a number of times. she does not feel that position changes the quality of the headache but she prefers to be still. there was no photo/phono-phobia and the pain is not made significantly worse with movement neuro ros, the pt denies loss of vision, blurred vision, diplopia, dysarthria, dysphagia, lightheadedness, vertigo, tinnitus or hearing difficulty. denies difficulties producing or comprehending speech. denies focal weakness, numbness, parasthesiae. no bowel or bladder incontinence or retention. past medical history: per patient she has no medical problems, s/p appy many years prior, and a d+c in for irregular menstrual bleeding social history: patient lives alone, has a long past smoking history (quit in ). 3 cig x 10 years, no etoh, no drugs. family history: non-contributory physical exam: on admission: t: 97.8 bp: 141/69 hr:55 r:18 97% on ra o2sats gen: patient lying in bed, moaning and uncomfortable heent: nc at neck: supple patient complains of mild inc in ha with neck movement lungs: cta bilaterally. cardiac: rrr. s1/s2. abd: soft, nt, bs+ extrem: warm and well-perfused. neuro: mental status: awake and alert, cooperative with exam, if not engaged frequently patient would fall asleep. orientation: oriented to person, place, and date. recall: objects at 5 minutes. language: speech fluent with good comprehension and repetition. naming intact. no dysarthria or paraphasic errors. cranial nerves: i: not tested ii: pupils equally round and reactive to light, 4 to 2 mm bilaterally. visual fields are full to confrontation. iii, iv, vi: extraocular movements intact bilaterally without nystagmus. v, vii: facial strength and sensation intact and symmetric. viii: hearing intact to voice. ix, x: palatal elevation symmetrical. : sternocleidomastoid and trapezius normal bilaterally. xii: tongue midline without fasciculations. motor: normal bulk, tone throughout. no pronator drift bilaterally. no adventitious movements, such as tremor, noted. no asterixis noted. delt bic tri wre ffl fe io ip quad ham ta edb l 5 5 5 5 5 5 5 5 5 5 5 5 5 5 r 5 5 5 5 5 5 5 5 5 5 5 5 5 5 -sensory: no deficits to light touch, pinprick, cold sensation, vibratory sense, proprioception throughout. no extinction to dss. -dtrs: tri pat ach l 2 2 2 2 1 r 2 2 2 2 1 toes downgoing bilaterally coordination: normal f-n-f. exam on discharge: xxxxxxxxxxx pertinent results: labs on admission: 10:37pm blood wbc-14.2*# rbc-4.69 hgb-12.3 hct-39.0 mcv-83 mch-26.3* mchc-31.7 rdw-13.0 plt ct-291 10:37pm blood neuts-87.9* lymphs-9.7* monos-1.7* eos-0.4 baso-0.2 10:37pm blood pt-12.6 ptt-22.3 inr(pt)-1.1 10:37pm blood glucose-151* urean-21* creat-0.8 na-136 k-3.8 cl-104 hco3-22 angap-14 01:56am blood alt-21 ast-23 ld(ldh)-128 alkphos-35* totbili-0.4 06:15am blood calcium-7.2* phos-3.1 mg-1.7 01:56am blood phenyto-10.5 labs on discharge: xxxxxxxxxxxxxx imaging: cta head : findings: head ct. diffuse subarachnoid hemorrhage identified in the basal cisterns, including the perimesencephalic cistern, suprasellar cistern and the right sylvian fissure as well as left parietal convexity sulci medially. there is no hydrocephalus seen. ct angiography of the head: ct angiography of the head demonstrates no evidence of a distinct aneurysm in the arteries of anterior and posterior circulation. no vascular stenosis is identified. impression: 1. cta head demonstrates diffuse subarachnoid hemorrhage. 2. ct angiography fails to demonstrate a discrete aneurysm. however, with the presence of subarachnoid hemorrhage, suspicion for an aneurysm is high and further evaluation with cerebral angiography is recommended. mra/v of head/neck : technique: routine enhanced mr examination of the brain, including t1-weighted axial se and sagittal mp-rage sequences, post-gadolinium, the latter with coronal and axial reformations, unenhanced sagittal 2d-tof mrv with coronal and axial reformations, and 3d coronal gre acquisition of the neck vessels, following dynamic intravenous gadolinium administration, with rotational targeted and large field-of-view mip reconstructed images. findings: the study is compared with the enhanced cranial cta (with additional 3d-volume-rendered reconstructions), as well as conventional contrast angiography, both performed . the axial flair sequence is notable for persistent subarachnoid hemorrhage which again appears relatively localized to a few left paramedian occipital cortical sulci, the trigones of both lateral ventricles, and the right sylvian and suprasellar and interpeduncular cisterns, as on the presentation ct (, ). though there is global cortical atrophy, there is no evidence of progressive ventricular dilatation or periventricular interstitial edema to suggest the development of hydrocephalus. other than the expected demonstration of "blooming" susceptibility artifact at these sites, above, there is no specific focus of susceptibility abnormality to suggest a culprit vascular lesion as source of the hemorrhage. allowing for the susceptibility artifacts, above, there is no evidence of restricted diffusion to suggest an acute ischemic event. post-contrast imaging is similarly unremarkable, with normal enhancement of the principal vessels of the circle of and major deep and superficial cerebral venous structures (see mrv, below). there is no pathologic focus of parenchymal, leptomeningeal, or dural enhancement. the mrv demonstrates normal flow-related enhancement in the major dural venous sinuses, other than an apparent roughly 2.6-cm segmental flow gap within the proximal left transverse sinus at its junction with the torcular, demonstrated only on the reformatted images. this appears to relate to a "mipping" artifact as there is continuous flow signal throughout this portion of the transverse sinus on the source images and the sinus demonstrates a completely unremarkable appearance on post-contrast mp-rage sequence. similarly, there is normal flow-related and contrast enhancement in the major deep cerebral veins, concordant with the normal appearance of these structures on yesterday's cta. there is a normal appearing three-vessel aortic arch with normal origins of the great vessels. the common and cervical internal carotid arteries demonstrate normal course, caliber, contour, and enhancement from their origins through the level of skull base. there is similarly normal course, caliber, contour, and enhancement of roughly "co-dominant" vertebral arteries from their subclavian origins through the vertebrobasilar confluence. there is no significant mural irregularity, flow-limiting stenosis, or evidence of dissection involving these vessels. impression: 1. persistent subarachnoid hemorrhage, not significantly changed since the presentation ct of ; there is a small amount of intraventricular hemorrhage now layering in the trigones of both lateral ventricles. 2. no evidence of development of hydrocephalus or transependymal migration of csf. 3. no new hemorrhage seen. 4. no vascular lesion or pathologic focus of enhancement identified. 5. unremarkable cranial mrv, with no evidence of thrombosis, stenosis, or abnormal shunting; this is concordant with the results of yesterday's ct and catheter angiography. 6. normal cervical contrast mra, with no evidence of carotid or vertebral stenosis or dissection. brief hospital course: patient is a 63f who was admitted to after complaining of sudden onset, worst headache of life while exercising. ct of the head revealed a pericisternal subarachnoid hemorrhage. cta was performed and did not reveal aneurysm. urgent angiogram was done on for definitive diagnosis, and was interpreted as negative for aneurysmal causation of hemorrhage. mra/v of both head and neck were also done to further evaluate possible etiology of bleeding; studies were negative. during this time, she was admitted to the neurosurgical icu for continuous monitoring, systolic pressure control and nimodipine administration. on cta/perfusion study was done to evaluate for possible vasospasm, and was determined to be negative as well. she was cleared to transferred to the neurosurgical step down unit but beds were not available on . she was intermittantly nausea and receiving anti-emetics. she was transitioned out of bed. on day of discharge she was cleared by pt ambulating in the hallway occassionally her headaches were worse with ambulation. she was tolerating a regular diet and voiding without difficulty. neurologically she was intact. extensive discussion was had about sequelae of nonanuerysmal subarchnoid hemorrhage medications on admission: none discharge medications: 1. docusate sodium 100 mg capsule sig: one (1) capsule po bid (2 times a day): use while on dilaudid. 2. acetaminophen 325 mg tablet sig: two (2) tablet po q6h (every 6 hours) as needed for pain. 3. phenytoin sodium extended 100 mg capsule sig: one (1) capsule po tid (3 times a day) for 30 days. disp:*90 capsule(s)* refills:*0* 4. hydromorphone 2 mg tablet sig: 1-2 tablets po every 4-6 hours as needed for pain. disp:*60 tablet(s)* refills:*0* 5. ciprofloxacin 250 mg tablet sig: two (2) tablet po q12h (every 12 hours) for 2 days. disp:*8 tablet(s)* refills:*0* 6. nimodipine 30 mg capsule sig: two (2) capsule po q4h (every 4 hours) for 20 days. disp:*240 capsule(s)* refills:*0* 7. nimodipine 30 mg capsule sig: two (2) capsule po every four (4) hours for 1 days. disp:*12 capsule(s)* refills:*0* discharge disposition: home discharge diagnosis: pericisternal subarachnoid hemorrhage discharge condition: neurologically stable discharge instructions: general instructions ?????? take your pain medicine as prescribed. it is expected that you will continue to have some form of a headache for several weeks; though this should continually improve. ?????? exercise should be limited to walking; no lifting, straining, or excessive bending. ?????? increase your intake of fluids and fiber, as narcotic pain medicine can cause constipation. we generally recommend taking an over the counter stool softener, such as docusate (colace) while taking narcotic pain medication. ?????? unless directed by your doctor, do not take any anti-inflammatory medicines such as motrin, aspirin, advil, or ibuprofen etc. ?????? you have been prescribed dilantin (phenytoin) for anti-seizure medicine, take it as prescribed and follow up with laboratory blood drawing in one week. this can be drawn at your pcp??????s office, but please have the results faxed to . - do not take tub baths for one week due to angio site, showering is okay - if you develop any swelling in your groin or your right leg becomes cool,numb or swollen go to the nearest er call your surgeon immediately if you experience any of the following ?????? new onset of tremors or seizures. ?????? any confusion, lethargy or change in mental status. ?????? any numbness, tingling, weakness in your extremities. ?????? pain or headache that is continually increasing, or not relieved by pain medication. ?????? new onset of the loss of function, or decrease of function on one whole side of your body. followup instructions: follow-up appointment instructions ??????please call ( to schedule an appointment with dr. , to be seen in 4 weeks. ??????you will need a ct scan of the brain without contrast prior to your appointment. this can be scheduled when you call to make your office visit appointment. procedure: arteriography of cerebral arteries arteriography of cerebral arteries diagnoses: subarachnoid hemorrhage osteoporosis, unspecified encephalopathy, unspecified cervicalgia meningismus 

Answer: The patient is high likely exposed to