Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: mr. is a year old man with a past medical history significant for atrial flutter, rotator cuff tendonitis, taking nsaids, prostatitis, and aortic stenosis, who presented to the emergency department and complained of dizziness, abdominal discomfort, nausea and multiple loose dark stools. he had developed lightheadedness, but no loss of consciousness. he was found on the toilet, pale, and close to passing out and was transported to the emergency . two to three weeks prior to this admission, he had developed a stiff shoulder for which he had been taking over-the-counter nsaids (naproxen two tablets twice a day per report). he did report occasional diarrhea during the two weeks prior to admission and had taken imodium leading to constipation. in the emergency department, he was hypotensive to 100/50 with a tachycardia to 110. he received fluids and one unit of packed red blood cells. ng lavage showed coffee grounds. he was started on protonix intravenously and admitted to the medical intensive care unit. past medical history: 1. aortic stenosis. 2. atrial flutter. 3. prostatitis, on bactrim starting . 4. rotator cuff injury. 5. cholecystectomy years prior. medications on admission: 1. aspirin 81 mg p.o. q. day. 2. multivitamins one tablet q. day. 3. naproxen. 4. bactrim, one double strength q. day since . 5. metamucil q. day. allergies: no known drug allergies. social history: occasional alcohol; no tobacco. lives at in . physical examination: on admission, vital signs with blood pressure 90/60; respiratory rate 24; heart rate 110; saturation at 93% on four liters nasal cannula. alert, oriented, pleasant, talking in full sentences, comfortable. anicteric. pupils equally round and reactive to light. extraocular muscles are intact. oral mucosa dry. no jaundice. heart rate irregularly irregular. ii/iii systolic murmur with radiation to the carotids noticed. lungs with bilateral coarse rhonchi; no crackles, no wheezing. abdomen soft, nontender, nondistended. positive bowel sounds. positive epigastric tenderness, no rebound, no guarding. rectal was ob positive. extremities with trace edema. no calf tenderness. neurological examination with intact strength five through five throughout; nonfocal otherwise. laboratory data: on admission, white blood cell count of 12.5, hemoglobin 7.2, hematocrit 21.8, platelets 186. sodium 136, potassium 5.2, chloride 104, bicarbonate 19, bun 46, creatinine 1.9, glucose 141, alt 23, ast 26, ck 53, alkaline phosphatase 29; amylase 393, total bilirubin 0.3. urinalysis, yellow, clear; otherwise negative. imaging: on admission, chest x-ray from , showing hyperinflated lungs with the question of congestive heart failure, prominent pulmonary arteries, heart mildly enlarged with left ventricular prominence consistent with aortic stenosis. no pleural effusion, no pneumothorax, no focal consolidation, no pulmonary edema. ekg on admission: atrial flutter with a heart rate of approximately 106, left axis deviation. no q waves. right bundle branch block, early r wave progression; no st changes. chest x-ray from , right prominent hilum, upper zone redistribution, no effusion. brief hospital course: mr. was initially admitted to the medical intensive care unit where he was transfused with two additional units of packed red blood cells. the following morning, he received an esophagogastroduodenoscopy showing ulcers in the pre-pyloric region, the anterior and posterior bulb, consistent with nsaid-induced ulcers. no active bleed was seen. while in the unit, for the subsequent 24 hours he continued to have guaiac positive stools, but no overt blood. h. pylori and gastrin levels were sent and h. pylori was positive and he was started on triple therapy of clarithromycin, amoxicillin and protonix. he will be continued on this as an outpatient for full therapy. he was called out of the medical intensive care unit the subsequent day to the medical floor with a stable hematocrit of 30.5. throughout his stay, his hematocrit remained stable at this level and he had no repeat episodes of bright red blood. the night following his transfer to the floor, he had an episode of shortness of breath not associated with any chest pain. he also complained of anxiety at this point. a routine ekg was checked and while unchanged significantly from prior ekgs in the medical intensive care unit, it was felt that there was an element of st depressions present in the precordial and lateral leads. this had developed while in the intensive care unit and was presumed secondary to rate related low-level ischemia. given these findings, cardiac enzymes were cycled. cks were negative times three, however, troponin i returned at 22. it was felt that mr. may have suffered an infarction in the setting of profound anemia, tachycardia and outflow obstruction from his aortic stenosis. however, given his age and his poor candidacy for either cardiac catheterization based revascularization or surgery, the decision was made not to pursue further work-up of this. two days later, the day of discharge, a repeat troponin was checked and had declined to 8.0. an echocardiogram was obtained to assess both the degree of aortic stenosis and assess for any potential wall motion abnormality. this was significant for mild symmetric left ventricular hypertrophy with a normal cavity size. left ventricular systolic function was assessed as normal. there was mild dilatation of the ascending aorta. the aortic valve leaflets were moderately thickened with probable moderate to severe aortic stenosis. moderate aortic regurgitation was seen. the mitral valve leaflets were mildly thickened with moderate to severe regurgitation. there was mild pulmonary artery systolic hypertension and no pericardial effusion. compared to a prior report of , the aortic stenosis was felt to be more severe. given the probable rate related changes, he was started on a low dose of beta blocker, lopressor 12.5 three times a day. this option had been discussed and initially held while in the intensive care unit secondary to the fact that he was felt to be dependent upon his rate for perfusion secondary to his profound anemia in the setting of blood loss. throughout his stay on the medical floor, he had no further shortness of breath and no further gi bleed or bright red blood per rectum. he will be followed up by the gastroenterology service and will need an esophagogastroduodenoscopy in two months at the clinic, phone number . again, he will be continued on triple therapy for h. pylori. he should avoid nsaids in the future and if need be take tylenol for pain. again, given his history of recent prostatitis, a routine urinalysis was again checked on the floor. it was negative for indications of infection and he was not continued on his bactrim. discharge diagnoses: 1. upper gastrointestinal bleed secondary to non-steroidal anti-inflammatories. 2. aortic stenosis. 3. atrial flutter. 4. prostatitis. discharge disposition: to rehabilitation. discharge medications: 1. multivitamin, one tablet q. day. 2. protonix 40 mg p.o. q. day. 3. atenolol 50 mg p.o. q. day. 4. amoxicillin 1 gram twice a day for ten days. 5. clarithromycin 500 mg p.o. twice a day times ten days. 6. ambien 5 mg p.o. q. h.s. p.r.n. while at rehabilitation. 7. tylenol 650 mg p.o. q. six hours p.r.n. discharge instructions: 1. the patient should follow-up with his primary care physician after discharge from rehabilitation. 2. should follow-up with the clinic for a repeat esophagogastroduodenoscopy and follow-up on his gastrin level. , m.d. dictated by: medquist36 procedure: other endoscopy of small intestine diagnostic ultrasound of heart gastric lavage diagnoses: congestive heart failure, unspecified iron deficiency anemia secondary to blood loss (chronic) atrial flutter other specified cardiac dysrhythmias mitral valve insufficiency and aortic valve stenosis chronic or unspecified gastric ulcer with hemorrhage, without mention of obstruction chronic or unspecified duodenal ulcer with hemorrhage, without mention of obstruction helicobacter pylori [h. pylori] unspecified analgesic and antipyretic causing adverse effects in therapeutic use 

Answer: The patient is high likely exposed to