Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: patient recorded as having no known allergies to drugs attending: chief complaint: unresponsive episode major surgical or invasive procedure: ett tube placement cvl insertion history of present illness: the pt is an 86 year-old right-handed woman with a history of stroke who presented from her living facility with episodes of unresponsiveness and speech difficulties. . the pt was unable to offer a history at the time of my encounter. therefore, the following history is per the primary team, the medical record, and the pt's son who was present for the events. the pt had been in her usual state of health. her son was visiting her at her facility this morning. she was at her baseline and appeared well. at approximately 10am, she was receiving her medications and abruptly let out an "unusual cry" then became unresponsive for about thirty seconds. her son noted that her eyes were rolled back into her head. she then woke up, was speaking "gibberish" (by this the son means unintelligible, incomphrensible speech) and proceeded to drink the juice she was given with her medications. after another minute, she became unresponsive again. she remained in this state and ems was called. the pt was subsequently brought to the ed. the pt's sons noted that this sort of event had never happened before. there was no clear precipitant for the event. as above, the pt had voiced no complaints prior to this episode. . the pt was unable to offer a review of systems. past medical history: right mca territory stroke in with resultant left hemiplegia -seizure in (semiology unknown) -peripheral vascular disease, s/p abdominal aortogram with left lower extremity runoff, angioplasty of left superficial femoral artery, popliteal and anterior tibialis arteries, stenting of left superficial femoral artery, below knee popliteal, and anterior tibialis artery on . -hypertension -dementia; at baseline the pt is able to recognize her loved ones and carry out brief conversations, but has difficulty with short-term memory and executive functioning. she is an and requires help with adls due to underlying dementia and left hemiplegia. social history: pt lives in an facility. no history of tobacco, alcohol, illicit drug use family history: no history of seizure physical exam: vitals: t: 99.2f p: 88 r: 16 bp: 104/90 sao2: 100% nrb general: lying in bed with eyes closed. heent: nc/at, no scleral icterus noted, mmm, no lesions noted in oropharynx neck: supple, no jvd or carotid bruits appreciated. no nuchal rigidity pulmonary: lungs cta bilaterally without r/r/w cardiac: rrr, nl. s1s2, no m/r/g noted abdomen: soft, nt/nd, normoactive bowel sounds, no masses or organomegaly noted. extremities: no c/c/e bilaterally skin: no rashes . neurologic: -mental status: does not open eyes to verbal or noxious stimuli. no verbal output. does not follow commands. . -cranial nerves: perrl 3 to 2mm and brisk. funduscopic exam revealed no papilledema, exudates, or hemorrhages. eomi to oculocephalic maneuver. corneal reflex and nasal tickle present bilaterally. right facial droop. gag reflex intact. . -motor: normal bulk throughout. tone increased in the left arm and leg. withdraws to briskly in the right arm and leg. grimaces but does not withdraw to noxious stimuli on the left. on three occasions during the interview and exam, the pt demonstrated myoclonic jerks of the left arm. . -sensory: grimaces to noxious stimuli in all four extremities. . -dtrs: tri pat ach l 2 2 2 2 1 r 3 3 3 3 1 . plantar response was extensor bilaterally. pertinent results: admission labs: 7.7> 14.4 <370 42.5 . n:63.3 l:26.1 m:5.6 e:4.4 bas:0.6 . pt: 12.3 ptt: 20.8 inr: 1.1 . |104|14 /160 4.6| 26|0.7\ . ck: 19 mb: notdone trop-t: <0.01 . lactate: 3.3 . ekg: atrial fibrillation at 80bpm. lad. twi in v1-4. . admission head ct : 1. large area of encephalomalacia in the right frontal, temporal, and parietal lobes consistent with extensive remote mca territorial infarction, with multiple hyperdense areas within consistent with mineralization, and advanced wallerian degeneration. 2. no definite acute intracranial hemorrhage. 3. prominence of the right more than left lateral and third ventricles, that likely primarily reflects ex vacuo dilatation (no previous studies are available for comparison). 4. partial opacification of both external auditory canals with no obvious osseous destruction, which may simply reflect impacted cerumen; correlation clinically is recommended. . mri brain : 1. mri a new left thalamic infarction. 2. chronic changes from extensive remote middle cerebral artery territorial infarction with associated ex vacuo dilation of the ipsilateral lateral ventricle and wallerian degeneration. 3. mr decreased signal intensity in the right vertebral artery and right internal carotid and branches. there is also decreased signal in the left posterior cerebral artery. . eeg : markedly abnormal portable eeg due to the prominent focal slowing seen broadly over the right hemisphere particularly in more lateral areas and due to the occasional sharp waves in the more posterior areas on the right. the background rhythm was also slow and disorganized. the first abnormality signifies a large area of subcortical dysfunction and suggests a structural lesion. there were frequent sharp waves but no spike or sharp and slow wave complexes or repetitive discharges to suggest ongoing seizures at this time. the slow background indicates a more widespread encephalopathy. medications, metabolic disturbances, and infection are among the most common causes. . tte : the left atrium is elongated. the right atrium is moderately dilated. no atrial septal defect is seen by 2d or color doppler. there is mild symmetric left ventricular hypertrophy with normal cavity size and systolic function (lvef>55%). regional left ventricular wall motion is normal. right ventricular systolic function is borderline normal. the descending thoracic aorta is mildly dilated. the aortic valve leaflets (3) are mildly thickened but aortic stenosis is not present. mild (1+) aortic regurgitation is seen. the mitral valve leaflets are mildly thickened. there is no mitral valve prolapse. mild to moderate (+) mitral regurgitation is seen. the mitral regurgitation jet is eccentric. the tricuspid valve leaflets are mildly thickened. there is moderate pulmonary artery systolic hypertension. there is an anterior space which most likely represents a fat pad. . brief hospital course: pt. was admitted to the neurology and sicu services. she was loaded with dilantin in the ed and this was continued in the sicu. on hod #2 she was noted to be more somnolent, and to have a dilated and fixed l pupil. repeat mri head (see results above) showed new l thalamic infarct and decreased signal in the l pca on top of her old known r mca infarct. pt. was intubated for airway protection as she was not maintaining her secretions. an eeg was performed and showed encephalopathy but no evidence of seizure activity. pt. was noted to be in a fib in the ed and in the sicu on telemetry, and infarcts were felt to be to cardioemboli from paroxsymal atrial fibrillation. the team discussed with the family starting heparin and coumadin to prevent further cardioembolic strokes, but the family felt that given the extent of her known strokes and her risk for bleeding into old stroke that they did not want to pursue this therapy. tte was performed and showed no visible . . over the next two weeks pt. was maintained on the ventilator, which was weaned down to minimal settings (cpap 5). her exam improved with increased level of alertness and increased ability to follow commands. the neurology and sicu teams had multiple discussions with the family about prognosis and goals of care. it was felt that pt. would not maintain her airway if she were extubated and would need a tracheostomy to maintain ventilation long term. the family did not feel that this was compatible with the pt's wishes, and elected to extubate her and make her comfortable instead. . pt. was extubated on . her breathing became labored and her o2 sats dropped slowly over the next 24 hours. on 5:15 on she stopped breathing. pupils were fixed and dilated, carotid pulse was not palpable, and no heart or breath sounds were auscultated. family was at the bedside and declined autopsy. medications on admission: -nifedipine 30 mg po daily -clopidogrel 75 mg po daily -atorvastatin 40 mg po daily -quetiapine 12.5mg in am and 25mg before bedtime. -multivitamin po daily -vitamin b12 supplementation -metoprolol 25 po bid -recently finished a 10 day course of levofloxacin and linezolid post-operatively discharge medications: none discharge disposition: expired discharge diagnosis: primary: top of the basilar syndrome with a new l thalamic/midbrain infarction old r mca ischemic stroke paroxsymal atrial fibrillation . secondary: hypertension peripheral vascular disease dementia discharge condition: expired discharge instructions: none followup instructions: none md procedure: venous catheterization, not elsewhere classified continuous invasive mechanical ventilation for 96 consecutive hours or more insertion of endotracheal tube enteral infusion of concentrated nutritional substances diagnoses: unspecified essential hypertension atrial fibrillation peripheral vascular disease, unspecified percutaneous transluminal coronary angioplasty status other persistent mental disorders due to conditions classified elsewhere late effects of cerebrovascular disease, hemiplegia affecting unspecified side cerebral embolism with cerebral infarction 

Answer: The patient is high likely exposed to