Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: allergies: no allergies/adrs on file attending: chief complaint: headache major surgical or invasive procedure: - evd placement - organ donation history of present illness: this is a 46 year old female with pmh of migraines on imitrex who experienced 2 day history of headaches. the patient was being driven to hospital emergency room to be seen for headache when on route she became agonal, pulseless, and unresponsive. the husband pulled over to the side of the road and the 12 year old daughter called 911. the patient was intubated, given epinephrine, cpr, 40 units vasopressin and was taken to hospital where she had a head ct consistent with extensive subarachnoid hemorrhage. the patient's gcs was 3t and her pupils were fixed and dilated. the patient was never paralyzed for intubation. she was given hypertonic saline/fentanyl/versed and med flighted to this emergency department. past medical history: migraines social history: lives at home with 12 year old daughter and husband family history: non-contributory physical exam: gen: unresponsive heent: pupils:4mm fixed neuro:gcs 3t- no cough/gag/or corneals mental status/orientation/recall/language:unresponsive/intubated cranial nerves: i: not tested ii: pupils 4mm and fixed v, vii,viii,ix, x,,xii:gcs 3t unable to test motor:no movement to noxious toes-mute : patient was declared brain dead after apnea testing. pertinent results: eeg: pending cta head & neck: impression: 1. fusiform aneurysm of the left intracranial vertebral artery, as well as pseudoaneurysm of the left internal carotid artery just proximal to the skull base. 2. diffuse subarachnoid hemorrhage with mildly dilated ventricles. 3. apical airspace disease likely reflects aspiration. nchct: no interval change compared with exam performed prior to this study and immediately after drainage placement, with diffuse severe subarachnoid hemorrhage resulting in severe cerebral edema with radiologic signs of tonsilar herniation. evd in stable position. note added at attending review: i would interpret this study somewhat differently than indicated above. there is new diffuse hemorrhage throughout the subarachnoid space. this is most extensive in the posterior fossa and the ambient cistern, consistent with the location of the aneurysm. there is also new intraventricular hemorrhage in the lateral and third ventricles. the frontal of the left lateral ventricle has enlarged slightly since the head ct of 15:49 on . i believe that the loss of sulci is best explained by a mixture of csf and sah, rendering the sulci isodense, rather than diffuse cortical swelling. this can be difficult to distinguish from early global infarction at this stage on ct. there appears to be preserved grey /white differentiation in the centrum semiovale, vs cortex and the internal capsule vs thalamus, although this is somewhat less clear than on the earlier ct. nchct: impression: 1. diffuse severe subarachnoid hemorrhage with no evidence of new bleeding. 2. new external ventricular drainage catheter ending in expected location in the right ventricle with slight decrease in ventricular caliber. cxr: the tubes are in adequate position. there is no evidence of aspiration. cxr: the new right-sided subclavian line is in adequate position. nchct: impression: no interval change compared with exam performed prior to this study and immediately after drainage placement, with diffuse severe subarachnoid hemorrhage resulting in severe cerebral edema with radiologic signs of tonsilar herniation. evd in stable position. note added at attending review: i would interpret this study somewhat differently than indicated above. there is new diffuse hemorrhage throughout the subarachnoid space. this is most extensive in the posterior fossa and the ambient cistern, consistent with the location of the aneurysm. there is also new intraventricular hemorrhage in the lateral and third ventricles. the frontal of the left lateral ventricle has enlarged slightly since the head ct of 15:49 on . i believe that the loss of sulci is best explained by a mixture of csf and sah, rendering the sulci isodense, rather than diffuse cortical swelling. this can be difficult to distinguish from early global infarction at this stage on ct. there appears to be preserved grey /white differentiation in the centrum semiovale, vs cortex and the internal capsule vs thalamus, although this is somewhat less clear than on the earlier ct. these findings were discussed by telephone with dr. at 10:15 am on , by dr. . at that time dr. informed me that the patient had expired. echo: impression: normal biventricular cavity size with low normal global left ventricular systolic function. no valvular pathology or pathologic flow identified. cxr: there is very mild new left lower lobe atelectasis. cardiac cath: pending brief hospital course: pt was admitted to the neurosurgery service, icu. upon arrival to the icu an evd was placed without complication. post placement ct revealed could catheter positioning and stable hemorrhage. a family meeting was held and the grave prognosis was discussed with the husband and daughter. eeg was ordered and placed to assess the brain conductivity. in the evening the patient was suddenly tachycardic to 140 and hypertensive to 200. also large amounts of clot were coming through the evd tubing requiring frequent flushing. cardiac enzymes were checked and positive (trop 1.14). she was given labetolol and propofol with good effect. shortly later her icp's were sustaining in the mid 20's. she was given mannitol x1 with good effect. a repeat head ct was performed and stable. her neurological exam remained poor but icp's were <20 and sbp<140 (on antihypertensives). on at approximately 1:30am her icp's increased/sustained in the 30's. she was given mannitol 25mg x1. this had no effect so she was given another 25mg. this also had no effect and the icp's continued to climb into the 40's. the patient's husband was brought to the bedside and updated of the situation. at approximately 0530 the patient's pupils were noted to be 6mm and nonreactive. code status was discussed and she was made dnr. chaplain was consulted per the families request. in the afternoon a multidicipilanary family meeting was held with social work present, patient's grave prognosis was discussed with the family and family decided to move forward with organ donation after cardiac death. a brain death and apnea exam was performed and the patient was pronounced around 1400. under the direction of the neob tests were performed to evaluate for organ donation. she was taken to the or on for harvesting. medications on admission: imitrex discharge medications: n/a discharge disposition: expired discharge diagnosis: subarachnoid hemorrhage increased intracranial pressure hydrocephalus myocardial infarction respiratory failure coma brain death discharge condition: expired discharge instructions: n/a followup instructions: n/a procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours venous catheterization, not elsewhere classified arterial catheterization insertion or replacement of external ventricular drain [evd] diagnoses: obstructive hydrocephalus subarachnoid hemorrhage compression of brain acute respiratory failure do not resuscitate status acute myocardial infarction of unspecified site, initial episode of care migraine, unspecified, without mention of intractable migraine without mention of status migrainosus coma brain death 

Answer: The patient is high likely exposed to