Consider the following medical report that includes the patient's historical health records. Use it to predict the disease that the patient is high likely exposed to. 

 Medical Report: history of present illness: this is a 78 year-old man who was admitted to the emergency room on . he has a history of coronary artery disease and is status post myocardial infarction, history of diabetes and he presented to the emergency room complaining of some confusion, a productive cough of yellow sputum, fatigue and malaise and abdominal distention. the patient woke up at 3:00 in the morning on the day of admission with an episode of coughing and choking with shortness of breath and appeared to his family to be confused. on arrival to the emergency department he was febrile to 102 and somewhat hypotensive with a blood pressure in the 80s. he had an oxygen saturation at 95% on 3 liters and a chest x-ray was suggestive of a left lower lobe pneumonia. he was administered intravenous fluids and levaquin in the emergency department. in addition, a foley catheter was placed and at some point at the catheter placement bright red blood was noted in the catheter tube. past medical history: significant for coronary artery disease, status post myocardial infarction one year ago. in of last year a catheterization showed a 70% occlusion of proximal d1 and a 60% occlusion of proximal obtuse marginal one, several occluded diagonal and marginal branches. he was not felt to be a candidate for intervention and was treated with medical therapy. he also has a history of transient ischemic attack for which he takes plavix, depression, mild congestive heart failure with an ejection fraction of 45%. medications: aspirin, plavix, glucophage, zestril, toprol xl, furosemide, spironolactone, hydrochlorothiazide, glipizide, lipitor, timoptic drops. allergies: no known drug allergies. social history: he did not use alcohol or tobacco. physical examination: this was a somewhat ill appearing man in no acute distress. temperature 102. heart rate 74. blood pressure 89/45. examination of the head, eyes, ears, nose and throat revealed no abnormalities. the neck was supple. lungs were clear to auscultation. heart was regular. abdomen was soft, slightly distended, nontender with mass. examination of the extremities revealed 2+ pitting edema bilaterally. he was oriented times three on admission, otherwise there were no significant neurological findings. laboratory findings on admission: white blood cell count of 10.4, hematocrit 35, sodium 137, potassium 4.3, chloride 101, bicarb 22, bun 48, creatinine 1.1, glucose 124, inr 1.7. chest x-ray showed cardiomegaly and a left lower lobe infiltrate and a kub showed a nonspecific bowel gas pattern. electrocardiogram showed a irregular rhythm without clear p waves and this was felt to represent atrial fibrillation. there was a right bundle branch block, q waves were present in the inferior leads and in v3 and v4. hospital course: the patient was initially admitted to the floor and was treated with levofloxacin. there was no evidence of cardiac ischemia at the time of admission. due to the patient's hematuria the foley catheter was left in place and hematuria initially appeared to clear, however, he did develop an obstruction in the catheter tube with bladder distention and the patient was evaluated by urology who removed the foley catheter and placed several foley catheters in order to irrigate clots and eventually placed a catheter for continuous bladder irrigations. per the recommendation of the urology team the patient underwent an abdominal ultrasound, which showed no abnormalities in the kidneys. the bladder was clasped from the foley tube. there was a significant amount of ascites in the abdomen. the patient received vitamin k for his elevated inr and the next day the patient seemed to be improving clinically with decreased hematuria, improved mental status and no difficulties with his breathing. however, at approximately 3:00 in the morning on the patient sustained a cardiac arrest. this appears to have initially begun as a bradycardic event. the patient was minimally responsive with poor respiratory effort with an irregular pulse in the 30s. the patient was given atropine and subsequently developed alternating pea and asystole. he was intubated and received cpr. he was given a total of three doses of epinephrine and two doses of atropine and dopamine and epinephrine drips were started. the patient did recover a pulse intermittently, but the total resuscitation lasted approximately an hour and twenty minutes and there were episodes as long as twenty minutes in which the patient was not noted to have any pulse. the patient was transferred to the micu and did have evidence of anoxic brain injury with sluggishly reactive pupils and decorticate posturing. the patient continued to be aggressively treated in the medical intensive care unit. he required high doses of nor-epinephrine and dopamine to maintain a mean arterial pressure of the high 50s. the epinephrine drip was weaned to off. he was continued on a morphine drip and ativan drip for sedation and received a bicarb drip for acidosis. he underwent a bedside echocardiogram, which showed severe globally depressed left ventricular function and minimal pericardial effusion. it was felt that the etiology of his hypotension was cardiogenic shock, but the etiology was unclear. his twelve lead electrocardiogram intermittently showed a ventricular escape rhythms and accelerated idioventricular rhythms, but there was no evidence of acute ischemic changes and his maximal troponin was 4.0. his inr continued to rise as high as 6 and liver function tests when checked showed transaminases in the 5000 range. when liver function tests were retrospectively added to the patient's admission laboratories they were normal. his antibiotic coverage was broadened to include ampicillin and flagyl and when his blood culture from came back positive for gram positive cocci the ampicillin was stopped and vancomycin was begun. the patient's white count did initially elevated as high as 19 after the event, but then decreased back down to 10 and his temperature did not exceed 100.5 and there did not appear to be evidence of active sepsis at this time. the patient underwent a head ct showing no abnormalities. an abdomen and pelvis ct performed without intravenous contrast showed a large amount of ascites and pleural effusions, but no evidence of perforation or obvious abscess. the patient remained significantly hemodynamically unstable. he was completely anuric. his creatinine rose to 2.8. the severity of the situation was discussed with the family extensively and they initially wished aggressive therapy to identify the underlying etiology of the patient's cardiac arrest as well as long as there was any chance of survival, however, as the patient continued to show a deteriorating course and multiorgan system failure and it was emphasized to the family that his prognosis for a meaningful survival was becoming extremely unlikely. they did agree that no further aggressive intervention should be performed. the patient underwent a ct angiogram of the lungs to rule out a pulmonary embolism and that showed no evidence of any pulmonary embolism. on the patient developed complete heart block and became progressively more bradycardic. he was placed on a do not resuscitate protocol and then developed asystole and was pronounced dead at 9:00 p.m. on . , m.d. dictated by: medquist36 d: 08:05 t: 06:38 job#: procedure: continuous invasive mechanical ventilation for less than 96 consecutive hours insertion of endotracheal tube diagnoses: pneumonia, organism unspecified coronary atherosclerosis of native coronary artery acute kidney failure with lesion of tubular necrosis diabetes mellitus without mention of complication, type ii or unspecified type, not stated as uncontrolled unspecified septicemia cardiac arrest cardiogenic shock old myocardial infarction 

Answer: The patient is high likely exposed to