Psoriasis is an autoimmune disorder in which skin cells
proliferate out of control. For some hard-to-heal wounds, the
problem is just the opposite: Restorative skin cells don’t grow
well or fast enough. In a paper published in the June 21, 2012
issue of Immunity, researchers at the University of California,
San Diego School of Medicine describe a molecule that may lead
to new treatments for both problems. Researchers at the
University of California, San Diego School of Medicine have
uncovered a new signal transduction pathway specifically devoted
to the regulation of alternative RNA splicing, a process that
allows a single gene to produce or code multiple types of
protein variants. The discovery, published in the June 27, 2012
issue of Molecular Cell, suggests the new pathway might be a
fruitful target for new cancer drugs.