Geroprotectors – agents/strategies which prevent or reverse the senescent state by preventing triggers of cellular senescence, such as DNA damage,[2][3][4] oxidative stress,[5] proteotoxic stress,[6] telomere shortening [7] (i.e. telomerase activators). SASP inhibitors – agents interfering with pro-inflammatory senescence‐associated secretory phenotype (SASP)[8][9] production, including: Glucocorticoids as potent suppressors of selected components of the SASP[10] Statins such as simvastatin, that can reduce the expression of pro-inflammatory cytokines (IL-6, IL-8, and MCP-1)[11] JAK1/2 inhibitors such as ruxolitinib[12][13] NF-κB and p38 inhibitors IL-1α blockers Mitochondrial depleters in the case of impaired mitophagy[14] Senolytics – small molecules that specifically induce cell death in senescent cells,[15][16] targeting survival pathways and anti-apoptotic mechanisms, antibodies and antibody-mediated drug delivery medications. Senomorphics – small molecules that suppress senescent phenotypes without cell killing[17] Gene therapy strategies – edit the genes of the cells of an organism in order to increase their resistance to aging, senile diseases and to prolong the life of the organism[3][18]