And this is known as negative feedback inhibition. Now, since the structure of the CTP, the citadine triphosphate, is nothing like the structure of these substrate molecules, that means this molecule does not bind to the active side, but it binds to some other regulatory site known as the allosteric site. And because it inhibits the activity of that enzyme, we call the CTP an allosteric inhibitor to this enzyme here. So we see that as low concentrations of the citadine triphosphate in our cells there is not enough CTP to actually bind onto the atcase. And so the activity of atcase will be high and the rate of production of the N carbon oil aspartate will also be high. And this is shown in the following curve.