But what happens if we don't have enough oxylacetate inside our cells? For example, if we're fasting, if we're fasting and we're not eating enough glucose molecules, then what the liver will do is it will begin the process of gluconeogenesis to ensure that the peripheral cells get enough glucose to use to produce ATP. So in the liver cells, the oxalo acetate will be used up to help form glucose via gluconeogenesis. And if we decrease the amount of oxyo acetate, then we won't have enough oxalo acetate to combine with the acetyl coenzyme A to help generate that intermediate of the citric acid cycle and ultimately generate ATP. And so low levels of oxyloacetate and high levels of acetyl coenzyme A molecules means the acetyl coenzyme A molecules cannot be fed into the citric acid cycle. And under such conditions, what will happen is that acetyl coenzyme A, produced via the beta oxidation pathway, will follow a different reaction pathway.