But if we actually use aceto acetoacete, we will not generate the NADH because we bypass this step. We only generate the two acetal coenzyme a molecule. So we see that if the liver forms these d three hydroxybutyrate, then ultimately that target cell will be able to produce more ATP molecules because of this NADH compared to the aceto acetatee, that will not be able to form the NADH inside the target cell. Now, another thing that I'd like to mention is the following. If the liver cell produces these ketone bodies, what stops the liver cells from actually generating back these acetyl acetal coenzyme a molecules? Well, inside the liver cells, it actually does not contain co enzyme a transphrase.