So notice in this blood clot cascade, we don't directly go from this factor to this. Well, we do go from this to this because the formation of this dimer, that CF seven dimer, directly affects the formation of prothrombinase. But we also have these other pathways. And what these all other pathways do is once again, they form more of these prothrombinases and these ultimately form more of our blood clots. So if we look at factor eleven, factor eleven, which is activated by factor twelve, goes on to activate nine, which ultimately forms more of these dimer protein complexes. So factor eleven is used to amplify the amount of factor nine form, which ultimately forms more active factor ten proteins and that forms more of these complexes leading to this final pathway that creates more blood clots.