Now, at the same time, in our adipose tissue, in our fat cells, the inability of the glucose to be absorbed by these fat cells will stimulate the breakdown of triglycerides into fatty acids. And so these adipose cells will continue dumping these fatty acids into the bloodstream and these fatty acids will ultimately be absorbed by that liver. So when we increase the levels of fatty acids inside our liver, we're going to essentially increase the levels of acetyl coenzyme A, which comes from the beta degradation of fatty acids. So we have low levels of oxylo acetate in the liver cells and high levels of acetyl coenzyme A. And so this process by which we essentially feed the acetylcoenzyme a into the citric acid cycle will be blocked as a result of the low levels of oxalo acetate. So the only pathway that is left for these acetal co enzyme A molecules is ketogenesis, the production of ketone bodies.