It phosphorylates, that serene residue that inactivates the PFK two, and it activates that fructose bisphosphatase two. And so now what happens is in the presence of low blood glucose in our blood plasma, the fructose 26 bisphosphate molecule that we form here basically is transformed back into fructose six phosphate via the hydrolysis of that Esther Bond. And when this process takes place, this potent activator of phosphor kinase can no longer activate that phosphorinase. And because this concentration decreases, it can no longer inhibit the enzymes of gluconeogenesis. And so this basically means gluconeogenesis will essentially be activated, while glycolysis will basically decrease in the rate at which it actually takes place. Now, why does that actually make sense?