And these blood clots can basically aggregate right across that rupture and that seals off that rupture. So we have the polymerization of fiber monomers that forms blood clots that can seal off the ruptures that form as a result of that trauma. So we see that not only digestive enzymes are activated via the process of proteolytic cleavage, these enzymes that are part of the blood clot cascade are also activated via the process of proteolytic cleavage. And we see that proteolytic activation is a very dominant mechanism here because essentially the majority of all these different zymogens are activated via the process of proteolytic cleavage. So we have these zymogens, the blue ones here, these Zion's here, as well as the main Zion, the Prothrombin, which basically is activates the Thrombin and then goes on to activate the fibringen into fibrin, which ultimately forms those. Blood clots, those meshlike networks of fibrin monomers that essentially seal off and prevent the leaking of the blood plasma from within that blood vessel and into the extracellular environment found around that blood vessel.