And if we decrease the amount of oxyo acetate, then we won't have enough oxalo acetate to combine with the acetyl coenzyme A to help generate that intermediate of the citric acid cycle and ultimately generate ATP. And so low levels of oxyloacetate and high levels of acetyl coenzyme A molecules means the acetyl coenzyme A molecules cannot be fed into the citric acid cycle. And under such conditions, what will happen is that acetyl coenzyme A, produced via the beta oxidation pathway, will follow a different reaction pathway. It will follow the pathway we call ketonogenesis, the formation of ketone bodies. So once again, acetal Co enzyme A, generated via the beta oxidation of fatty acids, can only be used to form ATP via the citric acid cycle if we have enough oxyloacetate inside the cell. So if our carbohydrate intake is low, for example, we're fasting or in diabetics, and we'll talk more about diabetics and ketogenesis in the next lecture.