Considering the patient's current medication regimen and recurrent urinary tract infections, which mechanism could explain the potential interaction between acetaminophen and the patient's condition?
Dextromethorphan may reduce the effectiveness of acetaminophen in managing pain associated with infections. Acetaminophen metabolism pathways involving GSTP1 may affect liver function, potentially impacting the body's ability to clear infections. The combination of acetaminophen and doxylamine succinate could directly increase the risk of urinary tract infections. Acetaminophen's interaction with FAAH could increase stress levels, thereby exacerbating the patient's condition.