is there any connection between this disease and aforementioned ltp?
Yes - some have suggested that the cognitive decline seen in individuals with Alzheimer's disease  may result from impaired Long-term potentiation
Long-term potentiation
LTP has received much attention among those who study Alzheimer's disease (AD), a neurodegenerative disease that causes marked cognitive decline and dementia. Much of this deterioration occurs in association with degenerative changes in the hippocampus and other medial temporal lobe structures. Because of the hippocampus' well established role in LTP, some have suggested that the cognitive decline seen in individuals with AD may result from impaired LTP.

In a 2003 review of the literature, Rowan "et al." proposed one model for how LTP might be affected in AD. AD appears to result, at least in part, from misprocessing of amyloid precursor protein (APP). The result of this abnormal processing is the accumulation of fragments of this protein, called amyloid β (Aβ). Aβ exists in both soluble and fibrillar forms. Misprocessing of APP results in the accumulation of soluble Aβ that, according to Rowan's hypothesis, impairs hippocampal LTP and may lead to the cognitive decline seen early in A
what is the best description for long-term potentiation (ltp) In neuroscience, long-term potentiation (LTP) is a persistent strengthening of synapses based on recent patterns of activity. These are patterns of synaptic activity that produce a long-lasting increase in signal transmission between two neurons. what is mean by the first mentioned science? It is the scientific study of the nervous system. regarding ltp; what is the earlier learning theory? The number of neurons in the adult brain (roughly 100 billion) did not increase significantly with age, memories were generally not the result of new neuron production, memories could form in the absence of new neurons. who discovered this? LTP was first observed by Terje Lømo who was this person? A Norwegian physiologist who specialized in neuroscience. what are the main branches of this science? Molecular neuroscience, Cellular neuroscience what is meant by the former one? A branch of neuroscience that observes concepts in molecular biology applied to the nervous systems of animals. is any disease observed in this regard? Neurodegenerative diseases like Alzheimer's, Parkinson's disease, Huntington's disease what are the possible causes of the first neurodegenerative disease? Abnormal amounts of proteins, amyloids and possibly tau proteins form in the brain. The resultant plaque disrupts normal functioning leading to a significant deficit of neurotransmitters, resulting in loss of brain function. what kind of protein is the mentioned one? The tau proteins  are a group of six highly soluble protein isoforms produced by alternative splicing from the gene "MAPT" (microtubule-associated protein tau). They have roles primarily in maintaining the stability of microtubules in axons and are abundant in the neurons of the central nervous system (CNS). is there any medicine available for the disease in context? Acetylcholinesterase inhibitors such as tacrine, rivastigmine, galantamine and donepezil. An NMDA receptor antagonist such as memantine.  No medication has been clearly shown to delay or halt the progression of the disease.
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Long-term potentiation [SEP] Alzheimer's disease
LTP has received much attention among those who study Alzheimer's disease (AD), a neurodegenerative disease that causes marked cognitive decline and dementia. Much of this deterioration occurs in association with degenerative changes in the hippocampus and other medial temporal lobe structures. Because of the hippocampus' well established role in LTP, some have suggested that the cognitive decline seen in individuals with AD may result from impaired LTP. In a 2003 review of the literature, Rowan "et al." proposed one model for how LTP might be affected in AD. AD appears to result, at least in part, from misprocessing of amyloid precursor protein (APP).