is there any model or theory based on this ltp?
BCM Theory, Hebbian learning model. Studies of dendritic spines, protruding structures on dendrites that physically grow and retract over the course of minutes or hours, have suggested a relationship between the electrical resistance of the spine and the effective synapse strength
Long-term potentiation
The physical and biological mechanism of LTP is still not understood, but some successful models have been developed. Studies of dendritic spines, protruding structures on dendrites that physically grow and retract over the course of minutes or hours, have suggested a relationship between the electrical resistance of the spine and the effective synapse strength, due to their relationship with intracellular calcium transients. Mathematical models such as BCM Theory, which depends also on intracellular calcium in relation to NMDA receptor voltage gates, have been developed since the 1980s and modify the traditional "a priori" Hebbian learning model with both biological and experimental justification. Still others have proposed re-arranging or synchronizing the relationship between receptor regulation, LTP, and synaptic strength.
what is the best description for long-term potentiation (ltp) In neuroscience, long-term potentiation (LTP) is a persistent strengthening of synapses based on recent patterns of activity. These are patterns of synaptic activity that produce a long-lasting increase in signal transmission between two neurons. what is mean by the first mentioned science? It is the scientific study of the nervous system. regarding ltp; what is the earlier learning theory? The number of neurons in the adult brain (roughly 100 billion) did not increase significantly with age, memories were generally not the result of new neuron production, memories could form in the absence of new neurons. who discovered this? LTP was first observed by Terje Lømo who was this person? A Norwegian physiologist who specialized in neuroscience. what are the main branches of this science? Molecular neuroscience, Cellular neuroscience what is meant by the former one? A branch of neuroscience that observes concepts in molecular biology applied to the nervous systems of animals. is any disease observed in this regard? Neurodegenerative diseases like Alzheimer's, Parkinson's disease, Huntington's disease what are the possible causes of the first neurodegenerative disease? Abnormal amounts of proteins, amyloids and possibly tau proteins form in the brain. The resultant plaque disrupts normal functioning leading to a significant deficit of neurotransmitters, resulting in loss of brain function. what kind of protein is the mentioned one? The tau proteins  are a group of six highly soluble protein isoforms produced by alternative splicing from the gene "MAPT" (microtubule-associated protein tau). They have roles primarily in maintaining the stability of microtubules in axons and are abundant in the neurons of the central nervous system (CNS). is there any medicine available for the disease in context? Acetylcholinesterase inhibitors such as tacrine, rivastigmine, galantamine and donepezil. An NMDA receptor antagonist such as memantine.  No medication has been clearly shown to delay or halt the progression of the disease. is there any connection between this disease and aforementioned ltp? Yes - some have suggested that the cognitive decline seen in individuals with Alzheimer's disease  may result from impaired Long-term potentiation
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Long-term potentiation [SEP] Models and theory
The physical and biological mechanism of LTP is still not understood, but some successful models have been developed. Studies of dendritic spines, protruding structures on dendrites that physically grow and retract over the course of minutes or hours, have suggested a relationship between the electrical resistance of the spine and the effective synapse strength, due to their relationship with intracellular calcium transients. Mathematical models such as BCM Theory, which depends also on intracellular calcium in relation to NMDA receptor voltage gates, have been developed since the 1980s and modify the traditional "a priori" Hebbian learning model with both biological and experimental justification.