is any disease observed in this regard?
Neurodegenerative diseases like Alzheimer's, Parkinson's disease, Huntington's disease
Molecular neuroscience
Alzheimer's disease is the most common neurodegenerative disease and is the most common form of dementia in the elderly. The disorder is characterized by progressive loss of memory and various cognitive functions. It is hypothesized that the deposition of amyloid-β peptide (40-42 amino acid residues) in the brain is integral in the incidence of Alzheimer's disease. Accumulation is purported to block hippocampal long-term potentiation. It is also possible that a receptor for amyloid-β oligomers could be a prion protein.
what is the best description for long-term potentiation (ltp) In neuroscience, long-term potentiation (LTP) is a persistent strengthening of synapses based on recent patterns of activity. These are patterns of synaptic activity that produce a long-lasting increase in signal transmission between two neurons. what is mean by the first mentioned science? It is the scientific study of the nervous system. regarding ltp; what is the earlier learning theory? The number of neurons in the adult brain (roughly 100 billion) did not increase significantly with age, memories were generally not the result of new neuron production, memories could form in the absence of new neurons. who discovered this? LTP was first observed by Terje Lømo who was this person? A Norwegian physiologist who specialized in neuroscience. what are the main branches of this science? Molecular neuroscience, Cellular neuroscience what is meant by the former one? A branch of neuroscience that observes concepts in molecular biology applied to the nervous systems of animals.
wiki:14528873
Molecular neuroscience [SEP] Alzheimer's disease
Alzheimer's disease is the most common neurodegenerative disease and is the most common form of dementia in the elderly. The disorder is characterized by progressive loss of memory and various cognitive functions. It is hypothesized that the deposition of amyloid-β peptide (40-42 amino acid residues) in the brain is integral in the incidence of Alzheimer's disease. Accumulation is purported to block hippocampal long-term potentiation. It is also possible that a receptor for amyloid-β oligomers could be a prion protein.