can you tell me some researches that he has done?
His further researches include four other important endogenous active substances, prostaglandin, vesiglandin, piperidine and noradrenaline.
Ulf von Euler
e discovered with John H. Gaddum an important autopharmacological principle, substance P. After returning to Stockholm, von Euler pursued further this line of research, and successively discovered four other important endogenous active substances, prostaglandin, vesiglandin (1935), piperidine (1942) and noradrenaline (1946).
what role does the neurotransmitter norepinephrine have in the cardiac system It increases heart rate and blood pressure what is norepinephrine? It is an organic chemical in the catecholamine family that functions in the brain and body as a hormone and neurotransmitter. what kind of a structure does this organ have? Its structure differs from that of epinephrine only in that epinephrine has a methyl group attached to its nitrogen, whereas the methyl group is replaced by a hydrogen atom in norepinephrine. does it affect our sleep? Can cause sleeplessness does that chemical have any affects on our skin? It activates the afferent sensory neuron does it go through degradation? Yes, in mammals, norepinephrine is rapidly degraded to various metabolites. did anyone discover this? Ulf von Euler who was he? A Swedish physiologist and pharmacologist
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Ulf von Euler [SEP] Research
His short stay as a postdoctoral student in Dale's laboratory was very fruitful: in 1931 he discovered with John H. Gaddum an important autopharmacological principle, substance P. After returning to Stockholm, von Euler pursued further this line of research, and successively discovered four other important endogenous active substances, prostaglandin, vesiglandin (1935), piperidine (1942) and noradrenaline (1946). In 1939 von Euler was appointed Full Professor of Physiology at the Karolinska Institute, where he remained until 1971. His early collaboration with Liljestrand had led to an important discovery, which was named the Euler–Liljestrand mechanism (a physiological arterial shunt in response to the decrease in local oxygenation of the lungs).