and any medical uses?
General anesthetic and as a Neuroprotectant
Xenon
Xenon has been used as a general anesthetic, but it is more expensive than conventional anesthetics.

Xenon interacts with many different receptors and ion channels, and like many theoretically multi-modal inhalation anesthetics, these interactions are likely complementary. Xenon is a high-affinity glycine-site NMDA receptor antagonist. However, xenon is different from certain other NMDA receptor antagonists in that it is not neurotoxic and it inhibits the neurotoxicity of ketamine and nitrous oxide, while actually producing neuroprotective effects. Unlike ketamine and nitrous oxide, xenon does not stimulate a dopamine efflux in the nucleus accumbens.

Like nitrous oxide and cyclopropane, xenon activates the two-pore domain potassium channel TREK-1. A related channel TASK-3 also implicated in the actions of inhalation anesthetics is insensitive to xenon. Xenon inhibits nicotinic acetylcholine αβ receptors which contribute to spinally mediated analgesia. Xenon is an effective inhibitor 
why does xenon have a high boiling point UNANSWERABLE what kind of a substance is it? It is a colorless, dense, odorless noble gas found in Earth's atmosphere in trace amounts. by whom was it discovered? William Ramsay, Morris Travers where does it occur? In Earth's atmosphere how is it produced? By fractional distillation is it used in sports? Yes - Inhaling a xenon/oxygen mixture is known to increase red blood cell production and athletic performance. However, the World Anti Doping Agency added xenon to the list of prohibited substances
wiki:19669093
Xenon [SEP] Anesthesia
Xenon has been used as a general anesthetic, but it is more expensive than conventional anesthetics. Xenon interacts with many different receptors and ion channels, and like many theoretically multi-modal inhalation anesthetics, these interactions are likely complementary. Xenon is a high-affinity glycine-site NMDA receptor antagonist. However, xenon is different from certain other NMDA receptor antagonists in that it is not neurotoxic and it inhibits the neurotoxicity of ketamine and nitrous oxide, while actually producing neuroprotective effects. Unlike ketamine and nitrous oxide, xenon does not stimulate a dopamine efflux in the nucleus accumbens. Like nitrous oxide and cyclopropane, xenon activates the two-pore domain potassium channel TREK-1.