optical measurements these changes can be interpreted as a reversal of the normal aging process of the lens the data for treatment with a non structure maker kcl when comprising 20 of a physiological saline show no comparable effect these results are given in table 1 eyes from the same donor were employed in the comparison of the structure maker to non structure maker i e one eye of the donor was exposed to the structure maker licl while the opposite eye was exposed to the non structure maker kcl referring to fig1 the substantially linear increase of the position of the photoluminescent peak of lenses by age is shown the linear progression establishes that as the lens ages a shift to longer wavelengths i e red shift in peak position occurs this is indicative of an increase in molecular defects within the lens system referring to fig2 the intensity of the peak photoluminescence is plotted as a function of age again showing an increase over time if log of intensity were plotted a linear progression would be seen establishing that as the lens gets older an increase in peak intensity occurs this is also indicative of an increase in molecular defects within the lens system fig3 shows the photoluminescence intensity plotted against wavelength for four human donor lenses of various ages 74 years 50 years 25 years and 7 years the exciting wavelength is 488 nm note that as the number of molecular defects increase in the lenses due to the increasing age of the donor there is an increase in the peak intensity a broadening of the spectral band i e the full width of the peak at half maximum photoluminescence intensity and a shift in the wavelength.