of the f actin and induction of the abnormal actin polymerization in cancer cells further the pt 262 possesses antitumor activity and can block survival pathway of the cancer cells resulting in cancer cells apoptosis and the pt 262 can induce growth arrest and inhibition of cell cycle pt 262 stabilizes cancer cells cytoskeleton that results in an irreversible cell elongation decreases the levels of cyclin b1 and phospho cdc2 proteins and inhibits the survival signal pathway of ras erk proteins the pt 262 also inhibits the mitochondrial membrane potential and induces the caspase 3 activation and apoptosis in the cancer cells application_number US 54880306 A description the present invention will be more clear from the following description when viewed together with the accompanying drawings which show for purpose of illustrations only the preferred embodiment in accordance with the present invention the cell culture of pt 262 the experimental procedures and the experiment results are described in conjunction with the accompanying drawings the a549 cell line was derived from lung carcinoma of a 58 year old male the h1299 cell line has a homozygous deletion of the p53 gene that was derived from a non small cell lung adenocarcinoma tumor mcf 7 cell line was derived from breast adenocarcinoma of a 69 year old caucasian female hela cell line was derived from cervical carcinoma of a 31 year old female these cell lines are cultured in rpmi 1640 medium supplemented with 10 fetal bovine serum 100 units ml penicillin 100 ug ml streptomycin and l glutamine 0 03 w v and cells were incubated.