Furthermore, the potential mechanism was also explored using mechanism assays.


 Furthermore, the expression of miR-647 was upregulated via the silence of PROX1-AS1 in PC cells, while the expression of miR-647 was downregulated via the overexpression of PROX1-AS1 in PC cells.  Further mechanism assays showed that miR-647 was a direct target of PROX1-AS1 in PC. 
THERM