However, the standard operative approach to lower limb amputation at either the below knee (BKA) or above knee (AKA) level obliterates many of the dynamic relationships characteristic of the uninjured lower extremity. Initial exposure is accomplished through either a stair-step (BKA) or fishmouth (AKA) pattern incision, followed by progressive transection of muscles, vessels, nerves and bone at the level of the incision. Tissues distal to the site of structural transection are discarded, regardless of whether or not there may be viable segments, and the proximal residual muscles are layered over the distal transected bone in order to provide insulation to this exposed osseous surface. The surrounding skin is then advanced over the bone/muscle infrastructure in order to achieve definitive closure. The rudimentary approximation of tissues in the distal limb in these approaches results in a disorganized scar mass in which normal dynamic muscle relationships are destroyed. The uncoupling of native agonist/antagonist muscle pairings results in isometric contraction of residual muscle groups upon volitional activation, producing incomplete, unbalanced neural feedback to the brain that results in aberrant perception of residual limb position. Such disturbed feedback not only results in impaired ambulatory function with prosthetics, but also manifests as pathological sensory perception of the extremity in the form of phantom limb and phantom pain symptomatology.