insulin-dependent diabetes
In summary, inactivating mutations in SLC29A3 cause a syndromic form of insulin-dependent diabetes in humans and in Drosophila profoundly affect cell size/number through interactions with the insulin signaling pathway.
In summary, inactivating mutations in <span class="gene" id="19336477-8-38-45">SLC29A3</span> cause a syndromic form of <span class="disease" id="19336477-8-72-98">insulin-dependent diabetes</span> in humans and in Drosophila profoundly affect cell size/number through interactions with the insulin signaling pathway.