arrhythmias
We show that myocytes from fructose-rich diet (FRD) animals exhibit arrhythmias produced by exacerbated Ca<sup>2+</sup>/calmodulin-protein kinase (CaMKII) activity, ryanodine receptor 2 (RyR2) phosphorylation and sarcoplasmic reticulum (SR) Ca<sup>2+</sup>leak.