pain
They also blocked pathological pain induced by sustained administration of morphine or spinal injection of dynorphin A, an endogenous mediator of chronic pain.
They also blocked pathological <span class="disease" id="17670969-3-31-35">pain</span> induced by sustained administration of morphine or spinal injection of <span class="gene" id="17670969-3-107-116">dynorphin</span> A, an endogenous mediator of chronic pain.