pain
Our results establish that central CRF is a key modulator of pain behavior and indicates that CRF effects on nociception are largely independent of its mood modulating effect as well as its control of the HPA axis.
Our results establish that central <span class="gene" id="16495007-9-35-38">CRF</span> is a key modulator of <span class="disease" id="16495007-9-61-65">pain</span> behavior and indicates that <span class="gene" id="16495007-9-94-97">CRF</span> effects on nociception are largely independent of its mood modulating effect as well as its control of the HPA axis.