hyperalgesia
Thus, increased sensitization and up-regulation of TRPV1 constitutes a potential mechanism by which TNFalpha mediates inflammatory hyperalgesia and pain.
Thus, increased sensitization and up-regulation of TRPV1 constitutes a potential mechanism by which <span class="gene" id="18582539-8-100-108">TNFalpha</span> mediates inflammatory <span class="disease" id="18582539-8-131-143">hyperalgesia</span> and pain.