heart failure
The development of disturbed substrate utilization of FASN transgenic cardiomyocytes and signs of heart failure were retarded by the transgenic expression of GRKInh, a peptide inhibitor of GRK2.
The development of disturbed substrate utilization of <span class="gene" id="26670611-6-54-58">FASN</span> transgenic cardiomyocytes and signs of <span class="disease" id="26670611-6-98-111">heart failure</span> were retarded by the transgenic expression of GRKInh, a peptide inhibitor of GRK2.