patency of the ductus arteriosus
Overall, our data support a role for genetic variations in transcription factor AP-2 beta, tumor necrosis factor receptor-associated factor 1, and prostacyclin synthase in the persistent patency of the ductus arteriosus seen in preterm infants.
Overall, our data support a role for genetic variations in transcription factor AP-2 beta, tumor necrosis factor receptor-associated factor 1, and <span class="gene" id="19336370-12-147-168">prostacyclin synthase</span> in the persistent <span class="disease" id="19336370-12-187-219">patency of the ductus arteriosus</span> seen in preterm infants.