rheumatoid arthritis
Because interleukin-1beta (IL-1beta) and tumor necrosis factor-alpha (TNFalpha) are central to the progression of inflammation and joint tissue injury in patients with rheumatoid arthritis, we investigated human monocyte IL-1beta and TNFalpha responses after the addition of AjA to cells in vitro.
Because interleukin-1beta (IL-1beta) and <span class="gene" id="12566094-2-41-68">tumor necrosis factor-alpha</span> (<span class="gene" id="12566094-2-70-78">TNFalpha</span>) are central to the progression of inflammation and joint tissue injury in patients with <span class="disease" id="12566094-2-168-188">rheumatoid arthritis</span>, we investigated human monocyte IL-1beta and <span class="gene" id="12566094-2-234-242">TNFalpha</span> responses after the addition of AjA to cells in vitro.