alcoholic cardiomyopathy
Alcohol-induced nitrative stress and apoptosis, which are mediated by angiotensin II interaction with AT1 and subsequent activation of a PKC-?1-dependent NOX pathway, are a causal factor in the development of alcoholic cardiomyopathy.
Alcohol-induced nitrative stress and apoptosis, which are mediated by <span class="gene" id="22497828-13-70-84">angiotensin II</span> interaction with AT1 and subsequent activation of a PKC-&beta;1-dependent NOX pathway, are a causal factor in the development of <span class="disease" id="22497828-13-209-233">alcoholic cardiomyopathy</span>.