 When the anti-inflammatory signal phosphatidylserine usually found on apoptotic cells, is exposed on the surface of dead parasites, "L. major" switches off the oxidative burst, thereby preventing killing and degradation of the viable pathogen.In the case of "Leishmania", progeny are not generated in PMNs, but in this way they can survive and persist untangled in the primary site of infection.The promastigote forms also release "Leishmania" chemotactic factor (LCF) to actively recruit neutrophils, but not other leukocytes, for instance monocytes or NK cells.