 All in all, then, compelling general arguments for massive modularity are hard to come by. This is not yet to dismiss the possibility of modularity in high-level cognition, but it invites skepticism, especially given the paucity of empirical evidence directly supporting the hypothesis (Robbins, 2013). For example, it has been suggested that the capacity to think about social exchanges is subserved by a domain-specific, functionally dissociable, and innate mechanism (Stone et al., 2002; Sugiyama et al., 2002). However, it appears that deficits in social exchange reasoning do not occur in isolation, but are accompanied by other social-cognitive impairments (Prinz, 2006). Skepticism about modularity in other areas of central cognition, such as high-level mindreading, also seems to be the order of the day (Currie &amp; Sterelny, 2000). The type of mindreading impairments characteristic of Asperger syndrome and high-functioning autism, for example, co-occur with sensory processing and executive function deficits (Frith, 2003). In general, there is little in the way of neuropsychological evidence to support the idea of high-level modularity. 